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You searched for subject:(K Ras). Showing records 1 – 30 of 56 total matches.

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The Ohio State University

1. Cantu, Daniel Vincenzo. Preliminary Characterization of K-Ras via Nuclear Magnetic Resonance.

Degree: MS, Biophysics, 2019, The Ohio State University

  For over 30 years, Ras GTPases have been intensively studied in cancer research. Membrane bound Ras proteins (H-Ras, N-Ras, K-Ras 4A, and K-Ras 4B)… (more)

Subjects/Keywords: Biophysics; Ras, K-Ras, NMR

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cantu, D. V. (2019). Preliminary Characterization of K-Ras via Nuclear Magnetic Resonance. (Masters Thesis). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1557171451783892

Chicago Manual of Style (16th Edition):

Cantu, Daniel Vincenzo. “Preliminary Characterization of K-Ras via Nuclear Magnetic Resonance.” 2019. Masters Thesis, The Ohio State University. Accessed October 27, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1557171451783892.

MLA Handbook (7th Edition):

Cantu, Daniel Vincenzo. “Preliminary Characterization of K-Ras via Nuclear Magnetic Resonance.” 2019. Web. 27 Oct 2020.

Vancouver:

Cantu DV. Preliminary Characterization of K-Ras via Nuclear Magnetic Resonance. [Internet] [Masters thesis]. The Ohio State University; 2019. [cited 2020 Oct 27]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1557171451783892.

Council of Science Editors:

Cantu DV. Preliminary Characterization of K-Ras via Nuclear Magnetic Resonance. [Masters Thesis]. The Ohio State University; 2019. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1557171451783892


University of Illinois – Chicago

2. Smith, Russell S. A Novel Affinity Reagent To RAS Perturbs Its Function In Cells.

Degree: 2017, University of Illinois – Chicago

RAS GTPases are mutated to the constitutively active state in around 30% of human cancers. These mutations lock RAS in a GTP-bound ‘active’ state and… (more)

Subjects/Keywords: NS1 monobody; H-RAS; K-RAS; RAS chimera; RAS inhibitor; RAS dimerization; RAS nanoclustering

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APA (6th Edition):

Smith, R. S. (2017). A Novel Affinity Reagent To RAS Perturbs Its Function In Cells. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21928

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Russell S. “A Novel Affinity Reagent To RAS Perturbs Its Function In Cells.” 2017. Thesis, University of Illinois – Chicago. Accessed October 27, 2020. http://hdl.handle.net/10027/21928.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Russell S. “A Novel Affinity Reagent To RAS Perturbs Its Function In Cells.” 2017. Web. 27 Oct 2020.

Vancouver:

Smith RS. A Novel Affinity Reagent To RAS Perturbs Its Function In Cells. [Internet] [Thesis]. University of Illinois – Chicago; 2017. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/10027/21928.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith RS. A Novel Affinity Reagent To RAS Perturbs Its Function In Cells. [Thesis]. University of Illinois – Chicago; 2017. Available from: http://hdl.handle.net/10027/21928

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Parker, Jillian Ann. The most oncogenic Ras isoform: specific insights into K-Ras structure, dimerization, and targeting.

Degree: PhD, Department of Chemistry and Chemical Biology, 2017, Northeastern University

 The small GTPase Ras is a bimolecular switch protein involved in a multitude of cellular signaling pathways, including those that control cell proliferation, differentiation, and… (more)

Subjects/Keywords: bimolecular switch proteins; guanine nucleotide exchange factors; Ras isoform; K-Ras

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APA (6th Edition):

Parker, J. A. (2017). The most oncogenic Ras isoform: specific insights into K-Ras structure, dimerization, and targeting. (Doctoral Dissertation). Northeastern University. Retrieved from http://hdl.handle.net/2047/D20240308

Chicago Manual of Style (16th Edition):

Parker, Jillian Ann. “The most oncogenic Ras isoform: specific insights into K-Ras structure, dimerization, and targeting.” 2017. Doctoral Dissertation, Northeastern University. Accessed October 27, 2020. http://hdl.handle.net/2047/D20240308.

MLA Handbook (7th Edition):

Parker, Jillian Ann. “The most oncogenic Ras isoform: specific insights into K-Ras structure, dimerization, and targeting.” 2017. Web. 27 Oct 2020.

Vancouver:

Parker JA. The most oncogenic Ras isoform: specific insights into K-Ras structure, dimerization, and targeting. [Internet] [Doctoral dissertation]. Northeastern University; 2017. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/2047/D20240308.

Council of Science Editors:

Parker JA. The most oncogenic Ras isoform: specific insights into K-Ras structure, dimerization, and targeting. [Doctoral Dissertation]. Northeastern University; 2017. Available from: http://hdl.handle.net/2047/D20240308


University of California – San Francisco

4. Ostrem, Jonathan Michael. Development of an Irreversible Mutant-Specific Inhibitor of Oncogenic K-Ras G12C.

Degree: Chemistry and Chemical Biology, 2013, University of California – San Francisco

 Somatic mutations in the small GTPase K-Ras are the most common activating lesions found in human cancer, and such mutations are generally associated with poor… (more)

Subjects/Keywords: Chemistry; G12C; Inhibitor; K-Ras; Lung Cancer

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APA (6th Edition):

Ostrem, J. M. (2013). Development of an Irreversible Mutant-Specific Inhibitor of Oncogenic K-Ras G12C. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/23h9s068

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ostrem, Jonathan Michael. “Development of an Irreversible Mutant-Specific Inhibitor of Oncogenic K-Ras G12C.” 2013. Thesis, University of California – San Francisco. Accessed October 27, 2020. http://www.escholarship.org/uc/item/23h9s068.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ostrem, Jonathan Michael. “Development of an Irreversible Mutant-Specific Inhibitor of Oncogenic K-Ras G12C.” 2013. Web. 27 Oct 2020.

Vancouver:

Ostrem JM. Development of an Irreversible Mutant-Specific Inhibitor of Oncogenic K-Ras G12C. [Internet] [Thesis]. University of California – San Francisco; 2013. [cited 2020 Oct 27]. Available from: http://www.escholarship.org/uc/item/23h9s068.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ostrem JM. Development of an Irreversible Mutant-Specific Inhibitor of Oncogenic K-Ras G12C. [Thesis]. University of California – San Francisco; 2013. Available from: http://www.escholarship.org/uc/item/23h9s068

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

5. Novotny, Chris J. Discovery and Design of Next Generation Inhibitors Targeting the HER2/HER3 heterodimer and K-Ras.

Degree: Chemistry and Chemical Biology, 2016, University of California – San Francisco

 Targeted inhibitors of driving oncogenes impose a selective pressure that benefits those cells who can most quickly and efficiently bypass the insult. While much attention,… (more)

Subjects/Keywords: Biology; Chemistry; HER2; HER3; Inhibitor; K-Ras

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APA (6th Edition):

Novotny, C. J. (2016). Discovery and Design of Next Generation Inhibitors Targeting the HER2/HER3 heterodimer and K-Ras. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/8zz805wx

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Novotny, Chris J. “Discovery and Design of Next Generation Inhibitors Targeting the HER2/HER3 heterodimer and K-Ras.” 2016. Thesis, University of California – San Francisco. Accessed October 27, 2020. http://www.escholarship.org/uc/item/8zz805wx.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Novotny, Chris J. “Discovery and Design of Next Generation Inhibitors Targeting the HER2/HER3 heterodimer and K-Ras.” 2016. Web. 27 Oct 2020.

Vancouver:

Novotny CJ. Discovery and Design of Next Generation Inhibitors Targeting the HER2/HER3 heterodimer and K-Ras. [Internet] [Thesis]. University of California – San Francisco; 2016. [cited 2020 Oct 27]. Available from: http://www.escholarship.org/uc/item/8zz805wx.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Novotny CJ. Discovery and Design of Next Generation Inhibitors Targeting the HER2/HER3 heterodimer and K-Ras. [Thesis]. University of California – San Francisco; 2016. Available from: http://www.escholarship.org/uc/item/8zz805wx

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

6. Yu, Yingzhe. Identification of small molecular inhibitors targeting G13D mutant K-ras.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

K-ras mutations can be frequently found in various cancers and are associated with resistance to treatment or poor prognosis. Thus far, however, there is no… (more)

Subjects/Keywords: inhibitors; targeting; G13D mutant K-ras

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APA (6th Edition):

Yu, Y. (2012). Identification of small molecular inhibitors targeting G13D mutant K-ras. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/66766/rec/3323

Chicago Manual of Style (16th Edition):

Yu, Yingzhe. “Identification of small molecular inhibitors targeting G13D mutant K-ras.” 2012. Masters Thesis, University of Southern California. Accessed October 27, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/66766/rec/3323.

MLA Handbook (7th Edition):

Yu, Yingzhe. “Identification of small molecular inhibitors targeting G13D mutant K-ras.” 2012. Web. 27 Oct 2020.

Vancouver:

Yu Y. Identification of small molecular inhibitors targeting G13D mutant K-ras. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2020 Oct 27]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/66766/rec/3323.

Council of Science Editors:

Yu Y. Identification of small molecular inhibitors targeting G13D mutant K-ras. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/66766/rec/3323

7. Turrini, Olivier. La mutation K-RAS détectée dans la marge de résection veineuse d'une pièce de duodénopancréatectomie céphalique définit la notion de "marge génique" et peut modifier la technique chirurgicale : Multi-referential analysis of attention : A comparative study of teacher knowledge, beliefs and practices.

Degree: Docteur es, Pathologie humaine. Oncologie, 2013, Aix Marseille Université

La technique d'une DPC pour adénocarcinome a évolué ces dernières années tant au niveau sécurité qu'au niveau carcinologique mais cela n'a pas suffit à faire… (more)

Subjects/Keywords: Pancréatectomie, K-ras, mutation, veine porte, marge; Pancreatectomy, K-ras, mutation, portal vein, margin

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APA (6th Edition):

Turrini, O. (2013). La mutation K-RAS détectée dans la marge de résection veineuse d'une pièce de duodénopancréatectomie céphalique définit la notion de "marge génique" et peut modifier la technique chirurgicale : Multi-referential analysis of attention : A comparative study of teacher knowledge, beliefs and practices. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2013AIXM5027

Chicago Manual of Style (16th Edition):

Turrini, Olivier. “La mutation K-RAS détectée dans la marge de résection veineuse d'une pièce de duodénopancréatectomie céphalique définit la notion de "marge génique" et peut modifier la technique chirurgicale : Multi-referential analysis of attention : A comparative study of teacher knowledge, beliefs and practices.” 2013. Doctoral Dissertation, Aix Marseille Université. Accessed October 27, 2020. http://www.theses.fr/2013AIXM5027.

MLA Handbook (7th Edition):

Turrini, Olivier. “La mutation K-RAS détectée dans la marge de résection veineuse d'une pièce de duodénopancréatectomie céphalique définit la notion de "marge génique" et peut modifier la technique chirurgicale : Multi-referential analysis of attention : A comparative study of teacher knowledge, beliefs and practices.” 2013. Web. 27 Oct 2020.

Vancouver:

Turrini O. La mutation K-RAS détectée dans la marge de résection veineuse d'une pièce de duodénopancréatectomie céphalique définit la notion de "marge génique" et peut modifier la technique chirurgicale : Multi-referential analysis of attention : A comparative study of teacher knowledge, beliefs and practices. [Internet] [Doctoral dissertation]. Aix Marseille Université 2013. [cited 2020 Oct 27]. Available from: http://www.theses.fr/2013AIXM5027.

Council of Science Editors:

Turrini O. La mutation K-RAS détectée dans la marge de résection veineuse d'une pièce de duodénopancréatectomie céphalique définit la notion de "marge génique" et peut modifier la technique chirurgicale : Multi-referential analysis of attention : A comparative study of teacher knowledge, beliefs and practices. [Doctoral Dissertation]. Aix Marseille Université 2013. Available from: http://www.theses.fr/2013AIXM5027

8. Zimmermann, Gunther. Design, synthesis and biophysical evaluation of small molecules targeting the K-Ras-PDEδ-interaction.

Degree: 2014, Technische Universität Dortmund

Ras-Proteine sind molekulare Schalter, die in gesunden Zellen Wachstum und Teilung regulieren. Mutationen in Ras-Genen können zu einem dauerhaft angeschalteten Zustand führen und im ungünstigsten… (more)

Subjects/Keywords: Protein-Protein-Wechselwirkungen; Ras; Inhibitoren; 570; 540; Protein-Protein-Wechselwirkung; Signaltransduktion; Tumor; Inhibitor; K-Ras

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APA (6th Edition):

Zimmermann, G. (2014). Design, synthesis and biophysical evaluation of small molecules targeting the K-Ras-PDEδ-interaction. (Doctoral Dissertation). Technische Universität Dortmund. Retrieved from http://dx.doi.org/10.17877/DE290R-6872

Chicago Manual of Style (16th Edition):

Zimmermann, Gunther. “Design, synthesis and biophysical evaluation of small molecules targeting the K-Ras-PDEδ-interaction.” 2014. Doctoral Dissertation, Technische Universität Dortmund. Accessed October 27, 2020. http://dx.doi.org/10.17877/DE290R-6872.

MLA Handbook (7th Edition):

Zimmermann, Gunther. “Design, synthesis and biophysical evaluation of small molecules targeting the K-Ras-PDEδ-interaction.” 2014. Web. 27 Oct 2020.

Vancouver:

Zimmermann G. Design, synthesis and biophysical evaluation of small molecules targeting the K-Ras-PDEδ-interaction. [Internet] [Doctoral dissertation]. Technische Universität Dortmund; 2014. [cited 2020 Oct 27]. Available from: http://dx.doi.org/10.17877/DE290R-6872.

Council of Science Editors:

Zimmermann G. Design, synthesis and biophysical evaluation of small molecules targeting the K-Ras-PDEδ-interaction. [Doctoral Dissertation]. Technische Universität Dortmund; 2014. Available from: http://dx.doi.org/10.17877/DE290R-6872

9. Vasseur, Romain. Etude de la régulation de la mucine MUC4 par l’oncogene K-ras dans le cancer du pancréas : Regulation of the mucin MUC4 by K-ras oncogene in pancreatic cancer.

Degree: Docteur es, Sciences de la vie et de la santé, 2015, Université Lille II – Droit et Santé

L’oncogène K-ras est une petite GTPase de la super famille RAS, fréquemment impliquée dans les cancers, en particulier celui du pancréas, l’un des plus mortels… (more)

Subjects/Keywords: Cancer du pancréas; Mucines-4; Oncogènes K-ras; Voies de signalisation; Régulation génétique; Pancreatic cancer; K-ras oncogene; MUC4

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APA (6th Edition):

Vasseur, R. (2015). Etude de la régulation de la mucine MUC4 par l’oncogene K-ras dans le cancer du pancréas : Regulation of the mucin MUC4 by K-ras oncogene in pancreatic cancer. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2015LIL2S051

Chicago Manual of Style (16th Edition):

Vasseur, Romain. “Etude de la régulation de la mucine MUC4 par l’oncogene K-ras dans le cancer du pancréas : Regulation of the mucin MUC4 by K-ras oncogene in pancreatic cancer.” 2015. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed October 27, 2020. http://www.theses.fr/2015LIL2S051.

MLA Handbook (7th Edition):

Vasseur, Romain. “Etude de la régulation de la mucine MUC4 par l’oncogene K-ras dans le cancer du pancréas : Regulation of the mucin MUC4 by K-ras oncogene in pancreatic cancer.” 2015. Web. 27 Oct 2020.

Vancouver:

Vasseur R. Etude de la régulation de la mucine MUC4 par l’oncogene K-ras dans le cancer du pancréas : Regulation of the mucin MUC4 by K-ras oncogene in pancreatic cancer. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2015. [cited 2020 Oct 27]. Available from: http://www.theses.fr/2015LIL2S051.

Council of Science Editors:

Vasseur R. Etude de la régulation de la mucine MUC4 par l’oncogene K-ras dans le cancer du pancréas : Regulation of the mucin MUC4 by K-ras oncogene in pancreatic cancer. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2015. Available from: http://www.theses.fr/2015LIL2S051


Miami University

10. Xu, Shenyuan. Nuclear Magnetic Resonance Spectroscopy Studies of At2g44920, a Pentapeptide Repeat Protein from Arabidopsis thaliana and X-ray Crystallography, Isothermal Titration Calorimetry Studies of K-Ras, a Human Oncogenic GTP-ase Signaling Protein.

Degree: PhD, Chemistry, 2017, Miami University

 At2g44920 from Arabidopsis thaliana is a pentapeptide-repeat protein (PRP) composed of 25 repeats capped by N- and C-terminal a-helices. PRP structures are dominated by four-sided… (more)

Subjects/Keywords: Biochemistry; Biophysics; AT2g44920; NMR; hydrogen exchange; dynamic; K-Ras; G12A-K-Ras mutant; ITC; X-Ray

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APA (6th Edition):

Xu, S. (2017). Nuclear Magnetic Resonance Spectroscopy Studies of At2g44920, a Pentapeptide Repeat Protein from Arabidopsis thaliana and X-ray Crystallography, Isothermal Titration Calorimetry Studies of K-Ras, a Human Oncogenic GTP-ase Signaling Protein. (Doctoral Dissertation). Miami University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=miami1500896385881469

Chicago Manual of Style (16th Edition):

Xu, Shenyuan. “Nuclear Magnetic Resonance Spectroscopy Studies of At2g44920, a Pentapeptide Repeat Protein from Arabidopsis thaliana and X-ray Crystallography, Isothermal Titration Calorimetry Studies of K-Ras, a Human Oncogenic GTP-ase Signaling Protein.” 2017. Doctoral Dissertation, Miami University. Accessed October 27, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=miami1500896385881469.

MLA Handbook (7th Edition):

Xu, Shenyuan. “Nuclear Magnetic Resonance Spectroscopy Studies of At2g44920, a Pentapeptide Repeat Protein from Arabidopsis thaliana and X-ray Crystallography, Isothermal Titration Calorimetry Studies of K-Ras, a Human Oncogenic GTP-ase Signaling Protein.” 2017. Web. 27 Oct 2020.

Vancouver:

Xu S. Nuclear Magnetic Resonance Spectroscopy Studies of At2g44920, a Pentapeptide Repeat Protein from Arabidopsis thaliana and X-ray Crystallography, Isothermal Titration Calorimetry Studies of K-Ras, a Human Oncogenic GTP-ase Signaling Protein. [Internet] [Doctoral dissertation]. Miami University; 2017. [cited 2020 Oct 27]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=miami1500896385881469.

Council of Science Editors:

Xu S. Nuclear Magnetic Resonance Spectroscopy Studies of At2g44920, a Pentapeptide Repeat Protein from Arabidopsis thaliana and X-ray Crystallography, Isothermal Titration Calorimetry Studies of K-Ras, a Human Oncogenic GTP-ase Signaling Protein. [Doctoral Dissertation]. Miami University; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=miami1500896385881469


Texas Medical Center

11. McCarthy, Michael. STRUCTURE BASED DRUG DESIGN OF HIGH AFFINITY KRAS INHIBITORS.

Degree: PhD, 2018, Texas Medical Center

RAS, one of the most well characterized membrane-associated small GTPases, is a notorious oncogene with >15% of all tumors harboring RAS mutations. When RAS(more)

Subjects/Keywords: KRAS; K-RAS; K-ras; drug design; molecular dynamics; biophysics; RAS inhibitor; KRAS inhibitor; small molecule; Medicinal Chemistry and Pharmaceutics; Medicine and Health Sciences; Pharmacology

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APA (6th Edition):

McCarthy, M. (2018). STRUCTURE BASED DRUG DESIGN OF HIGH AFFINITY KRAS INHIBITORS. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/829

Chicago Manual of Style (16th Edition):

McCarthy, Michael. “STRUCTURE BASED DRUG DESIGN OF HIGH AFFINITY KRAS INHIBITORS.” 2018. Doctoral Dissertation, Texas Medical Center. Accessed October 27, 2020. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/829.

MLA Handbook (7th Edition):

McCarthy, Michael. “STRUCTURE BASED DRUG DESIGN OF HIGH AFFINITY KRAS INHIBITORS.” 2018. Web. 27 Oct 2020.

Vancouver:

McCarthy M. STRUCTURE BASED DRUG DESIGN OF HIGH AFFINITY KRAS INHIBITORS. [Internet] [Doctoral dissertation]. Texas Medical Center; 2018. [cited 2020 Oct 27]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/829.

Council of Science Editors:

McCarthy M. STRUCTURE BASED DRUG DESIGN OF HIGH AFFINITY KRAS INHIBITORS. [Doctoral Dissertation]. Texas Medical Center; 2018. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/829

12. Asai, Takao. High frequency of TP53 but not K-ras gene mutations in Bolivian patients with gallbladder cancer : ボリビア胆嚢がん患者におけるTP53遺伝子およびK-ras遺伝子の変異頻度.

Degree: 博士(医学), 2014, Niigata University / 新潟大学

学位の種類: 博士(医学). 報告番号: 甲第3945号. 学位記番号: 新大院博(医)甲第612号.学位授与年月日: 平成26年9月22日

Asian Pacific Journal of Cancer Prevention. 2014, 15(13), 5449-5454.

Although genetic characteristics are considered to be a factor… (more)

Subjects/Keywords: gallbladder cancer; genetic alteration; TP53; K-ras; Bolivia

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APA (6th Edition):

Asai, T. (2014). High frequency of TP53 but not K-ras gene mutations in Bolivian patients with gallbladder cancer : ボリビア胆嚢がん患者におけるTP53遺伝子およびK-ras遺伝子の変異頻度. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/30973

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Asai, Takao. “High frequency of TP53 but not K-ras gene mutations in Bolivian patients with gallbladder cancer : ボリビア胆嚢がん患者におけるTP53遺伝子およびK-ras遺伝子の変異頻度.” 2014. Thesis, Niigata University / 新潟大学. Accessed October 27, 2020. http://hdl.handle.net/10191/30973.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Asai, Takao. “High frequency of TP53 but not K-ras gene mutations in Bolivian patients with gallbladder cancer : ボリビア胆嚢がん患者におけるTP53遺伝子およびK-ras遺伝子の変異頻度.” 2014. Web. 27 Oct 2020.

Vancouver:

Asai T. High frequency of TP53 but not K-ras gene mutations in Bolivian patients with gallbladder cancer : ボリビア胆嚢がん患者におけるTP53遺伝子およびK-ras遺伝子の変異頻度. [Internet] [Thesis]. Niigata University / 新潟大学; 2014. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/10191/30973.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Asai T. High frequency of TP53 but not K-ras gene mutations in Bolivian patients with gallbladder cancer : ボリビア胆嚢がん患者におけるTP53遺伝子およびK-ras遺伝子の変異頻度. [Thesis]. Niigata University / 新潟大学; 2014. Available from: http://hdl.handle.net/10191/30973

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

13. Jing, Hui. MULTIPLE ENZYMATIC FUNCTIONS OF SIRT2 AND ITS INVOLVEMENT IN CANCER.

Degree: PhD, Chemistry and Chemical Biology, 2017, Cornell University

 SIRT2 belongs to the mammalian sirtuin or NAD-dependent lysine deacylase family. Growing evidence suggests that SIRT2 plays important roles in cell cycle regulation, stress response,… (more)

Subjects/Keywords: c-Myc; K-Ras; Lysine fatty acylation; SIRT2; Sirtuin; Chemistry; cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jing, H. (2017). MULTIPLE ENZYMATIC FUNCTIONS OF SIRT2 AND ITS INVOLVEMENT IN CANCER. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/59083

Chicago Manual of Style (16th Edition):

Jing, Hui. “MULTIPLE ENZYMATIC FUNCTIONS OF SIRT2 AND ITS INVOLVEMENT IN CANCER.” 2017. Doctoral Dissertation, Cornell University. Accessed October 27, 2020. http://hdl.handle.net/1813/59083.

MLA Handbook (7th Edition):

Jing, Hui. “MULTIPLE ENZYMATIC FUNCTIONS OF SIRT2 AND ITS INVOLVEMENT IN CANCER.” 2017. Web. 27 Oct 2020.

Vancouver:

Jing H. MULTIPLE ENZYMATIC FUNCTIONS OF SIRT2 AND ITS INVOLVEMENT IN CANCER. [Internet] [Doctoral dissertation]. Cornell University; 2017. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/1813/59083.

Council of Science Editors:

Jing H. MULTIPLE ENZYMATIC FUNCTIONS OF SIRT2 AND ITS INVOLVEMENT IN CANCER. [Doctoral Dissertation]. Cornell University; 2017. Available from: http://hdl.handle.net/1813/59083


Vanderbilt University

14. Sun, Qi. Discovery of K-Ras inhibitors for the treatment of cancer.

Degree: PhD, Biochemistry, 2015, Vanderbilt University

Ras is a small GTPase that functions as a molecular switch, cycling between inactive (GDP-bound) and active (GTP-bound) states. Mutations in Ras fix the protein… (more)

Subjects/Keywords: Cancer; K-Ras; Fragment-based drug discovery; Structural-based drug design

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APA (6th Edition):

Sun, Q. (2015). Discovery of K-Ras inhibitors for the treatment of cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11334

Chicago Manual of Style (16th Edition):

Sun, Qi. “Discovery of K-Ras inhibitors for the treatment of cancer.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed October 27, 2020. http://hdl.handle.net/1803/11334.

MLA Handbook (7th Edition):

Sun, Qi. “Discovery of K-Ras inhibitors for the treatment of cancer.” 2015. Web. 27 Oct 2020.

Vancouver:

Sun Q. Discovery of K-Ras inhibitors for the treatment of cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/1803/11334.

Council of Science Editors:

Sun Q. Discovery of K-Ras inhibitors for the treatment of cancer. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/11334


University of Illinois – Chicago

15. Chavan, Tanmay S. Intra and Intermolecular Interactions of K-Ras4B: From Structure to Function.

Degree: 2015, University of Illinois – Chicago

Ras proteins are small GTPases that act as signal transducers between cell surface receptors and intracellular signaling cascades. According to the existing paradigm, Ras proteins… (more)

Subjects/Keywords: Ras; K-Ras4B; hypervariable region; protein-protein interactions; dimerization

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APA (6th Edition):

Chavan, T. S. (2015). Intra and Intermolecular Interactions of K-Ras4B: From Structure to Function. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/19405

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chavan, Tanmay S. “Intra and Intermolecular Interactions of K-Ras4B: From Structure to Function.” 2015. Thesis, University of Illinois – Chicago. Accessed October 27, 2020. http://hdl.handle.net/10027/19405.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chavan, Tanmay S. “Intra and Intermolecular Interactions of K-Ras4B: From Structure to Function.” 2015. Web. 27 Oct 2020.

Vancouver:

Chavan TS. Intra and Intermolecular Interactions of K-Ras4B: From Structure to Function. [Internet] [Thesis]. University of Illinois – Chicago; 2015. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/10027/19405.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chavan TS. Intra and Intermolecular Interactions of K-Ras4B: From Structure to Function. [Thesis]. University of Illinois – Chicago; 2015. Available from: http://hdl.handle.net/10027/19405

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Heinelt, Kaatje Friederike. Systems analysis of the spatial regulation of oncogenic Ras signalling.

Degree: 2017, Technische Universität Dortmund

Ras (Rat sarcoma) isoforms are small GTP-binding proteins that play a major role in the signalling networks controlling cell growth and survival. The Kras isoform… (more)

Subjects/Keywords: (K)Ras; PDEδ; Relocalization; Deltarasin; Deltazinone 1; 570

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APA (6th Edition):

Heinelt, K. F. (2017). Systems analysis of the spatial regulation of oncogenic Ras signalling. (Doctoral Dissertation). Technische Universität Dortmund. Retrieved from http://dx.doi.org/10.17877/DE290R-18036

Chicago Manual of Style (16th Edition):

Heinelt, Kaatje Friederike. “Systems analysis of the spatial regulation of oncogenic Ras signalling.” 2017. Doctoral Dissertation, Technische Universität Dortmund. Accessed October 27, 2020. http://dx.doi.org/10.17877/DE290R-18036.

MLA Handbook (7th Edition):

Heinelt, Kaatje Friederike. “Systems analysis of the spatial regulation of oncogenic Ras signalling.” 2017. Web. 27 Oct 2020.

Vancouver:

Heinelt KF. Systems analysis of the spatial regulation of oncogenic Ras signalling. [Internet] [Doctoral dissertation]. Technische Universität Dortmund; 2017. [cited 2020 Oct 27]. Available from: http://dx.doi.org/10.17877/DE290R-18036.

Council of Science Editors:

Heinelt KF. Systems analysis of the spatial regulation of oncogenic Ras signalling. [Doctoral Dissertation]. Technische Universität Dortmund; 2017. Available from: http://dx.doi.org/10.17877/DE290R-18036


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

17. Petanidis, Savvas. Μελέτη αλληλεπίδρασης μεταλλοϊόντων με Η-ras και Κ-ras ογκογονίδια: εστίαση στο καρκινογόνο κάδμιο και στην ανάπτυξη αντικαρκινικών μεταλλοφαρμάκων βαναδίου.

Degree: 2013, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

In this thesis we studied the interaction of metal ions with H-ras and K-ras oncogenes, focusing on the carcinogenic cadmium and the development of vanadium-based… (more)

Subjects/Keywords: Ογκογονίδια; Καρκίνος παχέος εντέρου; Βανάδιο; Μεταλλοφάρμακα; Ιντερλευκίνες; Κάδμιο; H-ras; K-ras; Oncogenes; Colorectal cancer; Cancer; Metallodrugs; Vanadium; Cadmium; Interleukins

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APA (6th Edition):

Petanidis, S. (2013). Μελέτη αλληλεπίδρασης μεταλλοϊόντων με Η-ras και Κ-ras ογκογονίδια: εστίαση στο καρκινογόνο κάδμιο και στην ανάπτυξη αντικαρκινικών μεταλλοφαρμάκων βαναδίου. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/43865

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Petanidis, Savvas. “Μελέτη αλληλεπίδρασης μεταλλοϊόντων με Η-ras και Κ-ras ογκογονίδια: εστίαση στο καρκινογόνο κάδμιο και στην ανάπτυξη αντικαρκινικών μεταλλοφαρμάκων βαναδίου.” 2013. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed October 27, 2020. http://hdl.handle.net/10442/hedi/43865.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Petanidis, Savvas. “Μελέτη αλληλεπίδρασης μεταλλοϊόντων με Η-ras και Κ-ras ογκογονίδια: εστίαση στο καρκινογόνο κάδμιο και στην ανάπτυξη αντικαρκινικών μεταλλοφαρμάκων βαναδίου.” 2013. Web. 27 Oct 2020.

Vancouver:

Petanidis S. Μελέτη αλληλεπίδρασης μεταλλοϊόντων με Η-ras και Κ-ras ογκογονίδια: εστίαση στο καρκινογόνο κάδμιο και στην ανάπτυξη αντικαρκινικών μεταλλοφαρμάκων βαναδίου. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2013. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/10442/hedi/43865.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Petanidis S. Μελέτη αλληλεπίδρασης μεταλλοϊόντων με Η-ras και Κ-ras ογκογονίδια: εστίαση στο καρκινογόνο κάδμιο και στην ανάπτυξη αντικαρκινικών μεταλλοφαρμάκων βαναδίου. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2013. Available from: http://hdl.handle.net/10442/hedi/43865

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Margetis, Nikolaos. Μελέτη των K-RAS μεταλλάξεων σε όλα τα στάδια ανάπτυξης του καρκίνου του παχέος εντέρου.

Degree: 2017, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

K-ras oncogene is a key factor in colorectal cancer. Based on published and our data we propose that K-ras could be the oncogene responsible for… (more)

Subjects/Keywords: K-ras μετάλλαξη; Φυσιολογικός βλεννογόνος; Καλοήθης πολύποδας; Βιοδείκτης; Ορθοκολικός καρκίνος; Πολύ πρώιμη μετάλλαξη; K-ras mutation; Normal mucosa; Benign polyp; Biomarker; Colorectal cancer; Earliest mutation

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APA (6th Edition):

Margetis, N. (2017). Μελέτη των K-RAS μεταλλάξεων σε όλα τα στάδια ανάπτυξης του καρκίνου του παχέος εντέρου. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/41286

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Margetis, Nikolaos. “Μελέτη των K-RAS μεταλλάξεων σε όλα τα στάδια ανάπτυξης του καρκίνου του παχέος εντέρου.” 2017. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed October 27, 2020. http://hdl.handle.net/10442/hedi/41286.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Margetis, Nikolaos. “Μελέτη των K-RAS μεταλλάξεων σε όλα τα στάδια ανάπτυξης του καρκίνου του παχέος εντέρου.” 2017. Web. 27 Oct 2020.

Vancouver:

Margetis N. Μελέτη των K-RAS μεταλλάξεων σε όλα τα στάδια ανάπτυξης του καρκίνου του παχέος εντέρου. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2017. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/10442/hedi/41286.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Margetis N. Μελέτη των K-RAS μεταλλάξεων σε όλα τα στάδια ανάπτυξης του καρκίνου του παχέος εντέρου. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2017. Available from: http://hdl.handle.net/10442/hedi/41286

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Balis, Vasileios. Εντοπισμός και μηχανισμοί δράσης του ΒΚ ιού στον καρκίνο του προστάτη και παγκρέατος του ανθρώπου.

Degree: 2012, University of Crete (UOC); Πανεπιστήμιο Κρήτης

The BKV was first identified in 1971 in a urine sample after a kidney transplant. The BK virus has been detected in various human tissues,… (more)

Subjects/Keywords: Προστάτης; Πάγκρεας; Πολυόμα ιοί; Πρωτο-ογκογονίδια; Κακοήθεια; BKV; H-K, N-Ras,; PCR; RFLP; Prostate; Pancreas; Polyoma virus; BKV; Proto-oncogene; H-, K- and N-ras; Malignancy; PCR

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Balis, V. (2012). Εντοπισμός και μηχανισμοί δράσης του ΒΚ ιού στον καρκίνο του προστάτη και παγκρέατος του ανθρώπου. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/27947

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Balis, Vasileios. “Εντοπισμός και μηχανισμοί δράσης του ΒΚ ιού στον καρκίνο του προστάτη και παγκρέατος του ανθρώπου.” 2012. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed October 27, 2020. http://hdl.handle.net/10442/hedi/27947.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Balis, Vasileios. “Εντοπισμός και μηχανισμοί δράσης του ΒΚ ιού στον καρκίνο του προστάτη και παγκρέατος του ανθρώπου.” 2012. Web. 27 Oct 2020.

Vancouver:

Balis V. Εντοπισμός και μηχανισμοί δράσης του ΒΚ ιού στον καρκίνο του προστάτη και παγκρέατος του ανθρώπου. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2012. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/10442/hedi/27947.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Balis V. Εντοπισμός και μηχανισμοί δράσης του ΒΚ ιού στον καρκίνο του προστάτη και παγκρέατος του ανθρώπου. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2012. Available from: http://hdl.handle.net/10442/hedi/27947

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

20. Iglesias, David A, M.D. DIRECT EFFECTS OF METFORMIN ON PI3K AND RAS SIGNALING IN ENDOMETRIAL CANCER.

Degree: MS, 2012, Texas Medical Center

  Metformin has antiproliferative effects through the activation of AMPK and has gained interest as an antineoplastic agent in several cancer types, although studies in… (more)

Subjects/Keywords: metformin; endometrial cancer; PI3K; K-Ras; Medical Molecular Biology; Medicine and Health Sciences

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APA (6th Edition):

Iglesias, David A, M. D. (2012). DIRECT EFFECTS OF METFORMIN ON PI3K AND RAS SIGNALING IN ENDOMETRIAL CANCER. (Thesis). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/260

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Iglesias, David A, M D. “DIRECT EFFECTS OF METFORMIN ON PI3K AND RAS SIGNALING IN ENDOMETRIAL CANCER.” 2012. Thesis, Texas Medical Center. Accessed October 27, 2020. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/260.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Iglesias, David A, M D. “DIRECT EFFECTS OF METFORMIN ON PI3K AND RAS SIGNALING IN ENDOMETRIAL CANCER.” 2012. Web. 27 Oct 2020.

Vancouver:

Iglesias, David A MD. DIRECT EFFECTS OF METFORMIN ON PI3K AND RAS SIGNALING IN ENDOMETRIAL CANCER. [Internet] [Thesis]. Texas Medical Center; 2012. [cited 2020 Oct 27]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/260.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Iglesias, David A MD. DIRECT EFFECTS OF METFORMIN ON PI3K AND RAS SIGNALING IN ENDOMETRIAL CANCER. [Thesis]. Texas Medical Center; 2012. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/260

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. 최, 희송. HeLa에서 K-ras 과발현시 활성산소 발생에 의한 핵모양의 이상의 발생과 이중핵/다핵 세포 증가 현상 연구.

Degree: 2014, Ajou University

Polyploidy and abnormal nuclear shapes are well known characteristics of cancer cells. K-Ras is frequently mutated in many cancers. One of the oncogene mutants, K-RasV12… (more)

Subjects/Keywords: ROS; K-Ras; bi/Multi-nucleated; abnormal nuclear shape; cytokinesis failure; aneuploidy; genomic instability

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APA (6th Edition):

최, . (2014). HeLa에서 K-ras 과발현시 활성산소 발생에 의한 핵모양의 이상의 발생과 이중핵/다핵 세포 증가 현상 연구. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/10862 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000017887

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

최, 희송. “HeLa에서 K-ras 과발현시 활성산소 발생에 의한 핵모양의 이상의 발생과 이중핵/다핵 세포 증가 현상 연구.” 2014. Thesis, Ajou University. Accessed October 27, 2020. http://repository.ajou.ac.kr/handle/201003/10862 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000017887.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

최, 희송. “HeLa에서 K-ras 과발현시 활성산소 발생에 의한 핵모양의 이상의 발생과 이중핵/다핵 세포 증가 현상 연구.” 2014. Web. 27 Oct 2020.

Vancouver:

최 . HeLa에서 K-ras 과발현시 활성산소 발생에 의한 핵모양의 이상의 발생과 이중핵/다핵 세포 증가 현상 연구. [Internet] [Thesis]. Ajou University; 2014. [cited 2020 Oct 27]. Available from: http://repository.ajou.ac.kr/handle/201003/10862 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000017887.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

최 . HeLa에서 K-ras 과발현시 활성산소 발생에 의한 핵모양의 이상의 발생과 이중핵/다핵 세포 증가 현상 연구. [Thesis]. Ajou University; 2014. Available from: http://repository.ajou.ac.kr/handle/201003/10862 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000017887

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Waters, Andrew Michael. KRAS 4B Silent Mutations in NIH/3T3 Cells Lead to a Tumorigenic Phenotype.

Degree: 2016, The Catholic University of America

Degree Awarded: Ph.D. Biology. The Catholic University of America

It has long been assumed that silent mutations do not affect the fate of a particular… (more)

Subjects/Keywords: Cellular biology; Molecular biology; Biology; Cancer; KRAS; K-RAS; Silent Mutation; Synonymous Codon; Synonymous Mutation

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APA (6th Edition):

Waters, A. M. (2016). KRAS 4B Silent Mutations in NIH/3T3 Cells Lead to a Tumorigenic Phenotype. (Thesis). The Catholic University of America. Retrieved from http://hdl.handle.net/1961/cuislandora:30422

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Waters, Andrew Michael. “KRAS 4B Silent Mutations in NIH/3T3 Cells Lead to a Tumorigenic Phenotype.” 2016. Thesis, The Catholic University of America. Accessed October 27, 2020. http://hdl.handle.net/1961/cuislandora:30422.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Waters, Andrew Michael. “KRAS 4B Silent Mutations in NIH/3T3 Cells Lead to a Tumorigenic Phenotype.” 2016. Web. 27 Oct 2020.

Vancouver:

Waters AM. KRAS 4B Silent Mutations in NIH/3T3 Cells Lead to a Tumorigenic Phenotype. [Internet] [Thesis]. The Catholic University of America; 2016. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/1961/cuislandora:30422.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Waters AM. KRAS 4B Silent Mutations in NIH/3T3 Cells Lead to a Tumorigenic Phenotype. [Thesis]. The Catholic University of America; 2016. Available from: http://hdl.handle.net/1961/cuislandora:30422

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas – Austin

23. Cammarata, Michael B. Development of top-down mass spectrometry methods for structural characterization of protein macromolecules utilizing 193nm ultraviolet photodissociation.

Degree: PhD, Chemistry, 2016, University of Texas – Austin

 The dissertation will discuss the advancement of informative structural biology techniques utilizing a top-down centric workflow with 193nm ultraviolet photodissociation (UVPD) mass spectrometry. Native electrospray… (more)

Subjects/Keywords: 193nm UVPD; Mass spectrometry; Protein structure; Dihydrofolate-reductase; K-Ras; Size exclusion chromatography

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APA (6th Edition):

Cammarata, M. B. (2016). Development of top-down mass spectrometry methods for structural characterization of protein macromolecules utilizing 193nm ultraviolet photodissociation. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/68584

Chicago Manual of Style (16th Edition):

Cammarata, Michael B. “Development of top-down mass spectrometry methods for structural characterization of protein macromolecules utilizing 193nm ultraviolet photodissociation.” 2016. Doctoral Dissertation, University of Texas – Austin. Accessed October 27, 2020. http://hdl.handle.net/2152/68584.

MLA Handbook (7th Edition):

Cammarata, Michael B. “Development of top-down mass spectrometry methods for structural characterization of protein macromolecules utilizing 193nm ultraviolet photodissociation.” 2016. Web. 27 Oct 2020.

Vancouver:

Cammarata MB. Development of top-down mass spectrometry methods for structural characterization of protein macromolecules utilizing 193nm ultraviolet photodissociation. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2016. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/2152/68584.

Council of Science Editors:

Cammarata MB. Development of top-down mass spectrometry methods for structural characterization of protein macromolecules utilizing 193nm ultraviolet photodissociation. [Doctoral Dissertation]. University of Texas – Austin; 2016. Available from: http://hdl.handle.net/2152/68584


Universitat de Barcelona

24. López Alcalá, Cristina. Regulación de K-Ras por Ca2+/CaM.

Degree: Departament de Biologia Cel·lular i Anatomia Patològica, 2006, Universitat de Barcelona

 Las proteínas de la superfamilia Ras son pequeñas proteínas G monoméricas de pesos moleculares de entre 20 y 40 kDa que actúan como interruptores moleculares… (more)

Subjects/Keywords: GTPasa; K-Ras; Funcions cel·lulars; Proteïnes; Superfamília Ras; Ciències de la Salut; 616

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APA (6th Edition):

López Alcalá, C. (2006). Regulación de K-Ras por Ca2+/CaM. (Thesis). Universitat de Barcelona. Retrieved from http://hdl.handle.net/10803/919

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

López Alcalá, Cristina. “Regulación de K-Ras por Ca2+/CaM.” 2006. Thesis, Universitat de Barcelona. Accessed October 27, 2020. http://hdl.handle.net/10803/919.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

López Alcalá, Cristina. “Regulación de K-Ras por Ca2+/CaM.” 2006. Web. 27 Oct 2020.

Vancouver:

López Alcalá C. Regulación de K-Ras por Ca2+/CaM. [Internet] [Thesis]. Universitat de Barcelona; 2006. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/10803/919.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

López Alcalá C. Regulación de K-Ras por Ca2+/CaM. [Thesis]. Universitat de Barcelona; 2006. Available from: http://hdl.handle.net/10803/919

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

25. Philip, Bincy. Acceleration of the PanIN Development in Mice Expressing Oncogenic K-Ras Due to a High Fat Diet.

Degree: MS, 2013, Texas Medical Center

  Obesity is postulated to be one of the major risk factors for pancreatic cancer, and recently it was indicated that an elevated body mass… (more)

Subjects/Keywords: K-Ras; Obesity; High Fat Diet; Cox-2; Pancreatic Cancer; PDAC; Biology; Cancer Biology; Medicine and Health Sciences; Other Nutrition

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APA (6th Edition):

Philip, B. (2013). Acceleration of the PanIN Development in Mice Expressing Oncogenic K-Ras Due to a High Fat Diet. (Thesis). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/356

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Philip, Bincy. “Acceleration of the PanIN Development in Mice Expressing Oncogenic K-Ras Due to a High Fat Diet.” 2013. Thesis, Texas Medical Center. Accessed October 27, 2020. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/356.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Philip, Bincy. “Acceleration of the PanIN Development in Mice Expressing Oncogenic K-Ras Due to a High Fat Diet.” 2013. Web. 27 Oct 2020.

Vancouver:

Philip B. Acceleration of the PanIN Development in Mice Expressing Oncogenic K-Ras Due to a High Fat Diet. [Internet] [Thesis]. Texas Medical Center; 2013. [cited 2020 Oct 27]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/356.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Philip B. Acceleration of the PanIN Development in Mice Expressing Oncogenic K-Ras Due to a High Fat Diet. [Thesis]. Texas Medical Center; 2013. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/356

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universitat Autònoma de Barcelona

26. Guerrero Caballero, Sílvia. Fenotips tumorals induïts per mutació puntual al codó 12 o 13 del gen k-ras humà.

Degree: Departament de Bioquímica i Biologia Molecular, 2002, Universitat Autònoma de Barcelona

K-ras gene is the most frequently mutated ras gene in human tumors. It's mutated in 40% of cases of colorectal carcinomas and 90% of pancreatic… (more)

Subjects/Keywords: K-ras; Mutació; Tumorogènesi; Ciències Experimentals; 616

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APA (6th Edition):

Guerrero Caballero, S. (2002). Fenotips tumorals induïts per mutació puntual al codó 12 o 13 del gen k-ras humà. (Thesis). Universitat Autònoma de Barcelona. Retrieved from http://hdl.handle.net/10803/3485

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guerrero Caballero, Sílvia. “Fenotips tumorals induïts per mutació puntual al codó 12 o 13 del gen k-ras humà.” 2002. Thesis, Universitat Autònoma de Barcelona. Accessed October 27, 2020. http://hdl.handle.net/10803/3485.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guerrero Caballero, Sílvia. “Fenotips tumorals induïts per mutació puntual al codó 12 o 13 del gen k-ras humà.” 2002. Web. 27 Oct 2020.

Vancouver:

Guerrero Caballero S. Fenotips tumorals induïts per mutació puntual al codó 12 o 13 del gen k-ras humà. [Internet] [Thesis]. Universitat Autònoma de Barcelona; 2002. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/10803/3485.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guerrero Caballero S. Fenotips tumorals induïts per mutació puntual al codó 12 o 13 del gen k-ras humà. [Thesis]. Universitat Autònoma de Barcelona; 2002. Available from: http://hdl.handle.net/10803/3485

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

27. Preston, Roman. Mutational analysis of K-ras in blood samples of patients with acute myeloid leukemia.

Degree: 2010, Freie Universität Berlin

 Mutations in K-ras are frequent in acute myeloid leukemia (AML). The association of these mutations to clinical features and their prognostic value are unclear. We… (more)

Subjects/Keywords: K-ras; acute myeloid leukemia; quantitative PCR; PNA; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Preston, R. (2010). Mutational analysis of K-ras in blood samples of patients with acute myeloid leukemia. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-14175

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Preston, Roman. “Mutational analysis of K-ras in blood samples of patients with acute myeloid leukemia.” 2010. Thesis, Freie Universität Berlin. Accessed October 27, 2020. http://dx.doi.org/10.17169/refubium-14175.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Preston, Roman. “Mutational analysis of K-ras in blood samples of patients with acute myeloid leukemia.” 2010. Web. 27 Oct 2020.

Vancouver:

Preston R. Mutational analysis of K-ras in blood samples of patients with acute myeloid leukemia. [Internet] [Thesis]. Freie Universität Berlin; 2010. [cited 2020 Oct 27]. Available from: http://dx.doi.org/10.17169/refubium-14175.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Preston R. Mutational analysis of K-ras in blood samples of patients with acute myeloid leukemia. [Thesis]. Freie Universität Berlin; 2010. Available from: http://dx.doi.org/10.17169/refubium-14175

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

28. Wahlström, Annika. Defining RCE1 and ICMT as Therapeutic Targets in K-RAS-induced Cancer.

Degree: 2009, University of Gothenburg / Göteborgs Universitet

 CAAX proteins, such as the RAS and RHO proteins, are recognized by a specific CAAX motif at the carboxyl terminus, which undergoes posttranslational modifications. First,… (more)

Subjects/Keywords: RCE1; ICMT; K-RAS; CAAX proteins; MPD; lung cancer

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APA (6th Edition):

Wahlström, A. (2009). Defining RCE1 and ICMT as Therapeutic Targets in K-RAS-induced Cancer. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/19643

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wahlström, Annika. “Defining RCE1 and ICMT as Therapeutic Targets in K-RAS-induced Cancer.” 2009. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed October 27, 2020. http://hdl.handle.net/2077/19643.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wahlström, Annika. “Defining RCE1 and ICMT as Therapeutic Targets in K-RAS-induced Cancer.” 2009. Web. 27 Oct 2020.

Vancouver:

Wahlström A. Defining RCE1 and ICMT as Therapeutic Targets in K-RAS-induced Cancer. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2009. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/2077/19643.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wahlström A. Defining RCE1 and ICMT as Therapeutic Targets in K-RAS-induced Cancer. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2009. Available from: http://hdl.handle.net/2077/19643

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arizona

29. Mehta, Dipti J. 15-deoxy-delta-12, 14-prostaglandin J2 (15d-PGJ2) Mediated Signaling in Colon Cancer .

Degree: 2006, University of Arizona

 Normal tissue structure and function are maintained by a dynamic interaction between epithelial cells and the stroma consisting of fibroblasts, adipose, vasculature and resident immune… (more)

Subjects/Keywords: 15d-PGJ2; K-RAS; Apoptosis; Adipogenesis; carcinogenesis; colon

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APA (6th Edition):

Mehta, D. J. (2006). 15-deoxy-delta-12, 14-prostaglandin J2 (15d-PGJ2) Mediated Signaling in Colon Cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194039

Chicago Manual of Style (16th Edition):

Mehta, Dipti J. “15-deoxy-delta-12, 14-prostaglandin J2 (15d-PGJ2) Mediated Signaling in Colon Cancer .” 2006. Doctoral Dissertation, University of Arizona. Accessed October 27, 2020. http://hdl.handle.net/10150/194039.

MLA Handbook (7th Edition):

Mehta, Dipti J. “15-deoxy-delta-12, 14-prostaglandin J2 (15d-PGJ2) Mediated Signaling in Colon Cancer .” 2006. Web. 27 Oct 2020.

Vancouver:

Mehta DJ. 15-deoxy-delta-12, 14-prostaglandin J2 (15d-PGJ2) Mediated Signaling in Colon Cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2006. [cited 2020 Oct 27]. Available from: http://hdl.handle.net/10150/194039.

Council of Science Editors:

Mehta DJ. 15-deoxy-delta-12, 14-prostaglandin J2 (15d-PGJ2) Mediated Signaling in Colon Cancer . [Doctoral Dissertation]. University of Arizona; 2006. Available from: http://hdl.handle.net/10150/194039

30. 김, 준형. Mechanism of mitochondrial respiratory dysfunction by oncogenic K-Ras.

Degree: 2015, Ajou University

 Among the several features of cancer cells, mitochondrial impairment in relation to altered metabolism has been extensively researched. Although the importance of mitochondrial impairment in… (more)

Subjects/Keywords: Mitochondrial dysfunction; K-RasV12; Mitophagy; Tumorigenesis; LC3-Ⅱ(microtubule associated protein 1 light chain 3); 미토콘드리아 기능이상; 형질전환; K-Ras; 미토콘드리아 자식작용; 대사전환

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APA (6th Edition):

김, . (2015). Mechanism of mitochondrial respiratory dysfunction by oncogenic K-Ras. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/11819 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020789

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

김, 준형. “Mechanism of mitochondrial respiratory dysfunction by oncogenic K-Ras.” 2015. Thesis, Ajou University. Accessed October 27, 2020. http://repository.ajou.ac.kr/handle/201003/11819 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020789.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

김, 준형. “Mechanism of mitochondrial respiratory dysfunction by oncogenic K-Ras.” 2015. Web. 27 Oct 2020.

Vancouver:

김 . Mechanism of mitochondrial respiratory dysfunction by oncogenic K-Ras. [Internet] [Thesis]. Ajou University; 2015. [cited 2020 Oct 27]. Available from: http://repository.ajou.ac.kr/handle/201003/11819 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020789.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

김 . Mechanism of mitochondrial respiratory dysfunction by oncogenic K-Ras. [Thesis]. Ajou University; 2015. Available from: http://repository.ajou.ac.kr/handle/201003/11819 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020789

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2]

.