Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Ischemia reperfusion I R ). Showing records 1 – 30 of 26512 total matches.

[1] [2] [3] [4] [5] … [884]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters


NSYSU

1. Chen, Kuan-hao. The protective effects of curcumin on the pancreatic juice induced lung injury.

Degree: PhD, Biological Sciences, 2015, NSYSU

 Abstract Emerging diseases such as middle east respiratory syndrome, severe acute respiratory syndrome or other infections induced by Ebola virus, Bacillus anthracis, and Legionnaireâs disease… (more)

Subjects/Keywords: nitrosative stress; Ischemia/ reperfusion (I/R); pulmonary capillary bronchial alveolar lavage (PCBAL); lung injury; curcumin; bronchoalveolar lavage fluid

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, K. (2015). The protective effects of curcumin on the pancreatic juice induced lung injury. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0802115-140256

Chicago Manual of Style (16th Edition):

Chen, Kuan-hao. “The protective effects of curcumin on the pancreatic juice induced lung injury.” 2015. Doctoral Dissertation, NSYSU. Accessed November 27, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0802115-140256.

MLA Handbook (7th Edition):

Chen, Kuan-hao. “The protective effects of curcumin on the pancreatic juice induced lung injury.” 2015. Web. 27 Nov 2020.

Vancouver:

Chen K. The protective effects of curcumin on the pancreatic juice induced lung injury. [Internet] [Doctoral dissertation]. NSYSU; 2015. [cited 2020 Nov 27]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0802115-140256.

Council of Science Editors:

Chen K. The protective effects of curcumin on the pancreatic juice induced lung injury. [Doctoral Dissertation]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0802115-140256


University of Alberta

2. Chow, Ava Kalyca. Intracellular regulation of matrix metalloproteinase-2 activity: the roles of caveolin-1 and troponin I phosphorylation.

Degree: PhD, Medical Sciences - Paediatrics, 2010, University of Alberta

 Matrix metalloproteinase‐2 (MMP‐2) was recently revealed to have targets and actions within the cardiac myocyte. In ischemia/reperfusion (I/R) injury, MMP‐2 is activated and degrades troponin… (more)

Subjects/Keywords: heart; ischemia-reperfusion; troponin I; caveolin; matrix metalloproteinase

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chow, A. K. (2010). Intracellular regulation of matrix metalloproteinase-2 activity: the roles of caveolin-1 and troponin I phosphorylation. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/8g84mm38c

Chicago Manual of Style (16th Edition):

Chow, Ava Kalyca. “Intracellular regulation of matrix metalloproteinase-2 activity: the roles of caveolin-1 and troponin I phosphorylation.” 2010. Doctoral Dissertation, University of Alberta. Accessed November 27, 2020. https://era.library.ualberta.ca/files/8g84mm38c.

MLA Handbook (7th Edition):

Chow, Ava Kalyca. “Intracellular regulation of matrix metalloproteinase-2 activity: the roles of caveolin-1 and troponin I phosphorylation.” 2010. Web. 27 Nov 2020.

Vancouver:

Chow AK. Intracellular regulation of matrix metalloproteinase-2 activity: the roles of caveolin-1 and troponin I phosphorylation. [Internet] [Doctoral dissertation]. University of Alberta; 2010. [cited 2020 Nov 27]. Available from: https://era.library.ualberta.ca/files/8g84mm38c.

Council of Science Editors:

Chow AK. Intracellular regulation of matrix metalloproteinase-2 activity: the roles of caveolin-1 and troponin I phosphorylation. [Doctoral Dissertation]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/8g84mm38c


Universiteit Utrecht

3. Soyal, M. Forkhead Box O in ischemia/reperfusion injury: a potential therapeutic target.

Degree: 2012, Universiteit Utrecht

 Acute myocardial infarction (AMI) is a major cause of morbidity. AMI is caused by a blockage of one or multiple coronary arteries, leading to an… (more)

Subjects/Keywords: AMI; FOXO; ischemia/reperfusion

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Soyal, M. (2012). Forkhead Box O in ischemia/reperfusion injury: a potential therapeutic target. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/254301

Chicago Manual of Style (16th Edition):

Soyal, M. “Forkhead Box O in ischemia/reperfusion injury: a potential therapeutic target.” 2012. Masters Thesis, Universiteit Utrecht. Accessed November 27, 2020. http://dspace.library.uu.nl:8080/handle/1874/254301.

MLA Handbook (7th Edition):

Soyal, M. “Forkhead Box O in ischemia/reperfusion injury: a potential therapeutic target.” 2012. Web. 27 Nov 2020.

Vancouver:

Soyal M. Forkhead Box O in ischemia/reperfusion injury: a potential therapeutic target. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2020 Nov 27]. Available from: http://dspace.library.uu.nl:8080/handle/1874/254301.

Council of Science Editors:

Soyal M. Forkhead Box O in ischemia/reperfusion injury: a potential therapeutic target. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/254301


University of Melbourne

4. Thacker, Michelle Anne. Structural damage to the enteric nervous system following ischemia / reperfusion injury.

Degree: 2012, University of Melbourne

Ischemia reperfusion (I/R) injury can occur in a number of organs, however the intestine is possibly the most susceptible to I/R damage. Intestinal I/R injury… (more)

Subjects/Keywords: enteric glia; ischemia reperfusion injury

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Thacker, M. A. (2012). Structural damage to the enteric nervous system following ischemia / reperfusion injury. (Masters Thesis). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37853

Chicago Manual of Style (16th Edition):

Thacker, Michelle Anne. “Structural damage to the enteric nervous system following ischemia / reperfusion injury.” 2012. Masters Thesis, University of Melbourne. Accessed November 27, 2020. http://hdl.handle.net/11343/37853.

MLA Handbook (7th Edition):

Thacker, Michelle Anne. “Structural damage to the enteric nervous system following ischemia / reperfusion injury.” 2012. Web. 27 Nov 2020.

Vancouver:

Thacker MA. Structural damage to the enteric nervous system following ischemia / reperfusion injury. [Internet] [Masters thesis]. University of Melbourne; 2012. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/11343/37853.

Council of Science Editors:

Thacker MA. Structural damage to the enteric nervous system following ischemia / reperfusion injury. [Masters Thesis]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37853


Universitat de Barcelona

5. Bejaoui, Mohamed. Polyethylene glycol conditioning: An effective strategy to protect against liver ischemia reperfusion injury.

Degree: Departament de Fisiologia (Biologia), 2015, Universitat de Barcelona

 La lesión por isquemia reperfusión (I/R) es un proceso complejo que tiene lugar cuando un órgano se ve privado del aporte sanguíneo (isquemia) y se… (more)

Subjects/Keywords: Isquèmia; Isquemia; Ischemia; Fetge; Hígado; Liver; Reperfusió (Fisiologia); Reperfusión (Fisiología); Reperfusion (Physiology); Ciències Experimentals i Matemàtiques; 612

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bejaoui, M. (2015). Polyethylene glycol conditioning: An effective strategy to protect against liver ischemia reperfusion injury. (Thesis). Universitat de Barcelona. Retrieved from http://hdl.handle.net/10803/385612

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bejaoui, Mohamed. “Polyethylene glycol conditioning: An effective strategy to protect against liver ischemia reperfusion injury.” 2015. Thesis, Universitat de Barcelona. Accessed November 27, 2020. http://hdl.handle.net/10803/385612.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bejaoui, Mohamed. “Polyethylene glycol conditioning: An effective strategy to protect against liver ischemia reperfusion injury.” 2015. Web. 27 Nov 2020.

Vancouver:

Bejaoui M. Polyethylene glycol conditioning: An effective strategy to protect against liver ischemia reperfusion injury. [Internet] [Thesis]. Universitat de Barcelona; 2015. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/10803/385612.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bejaoui M. Polyethylene glycol conditioning: An effective strategy to protect against liver ischemia reperfusion injury. [Thesis]. Universitat de Barcelona; 2015. Available from: http://hdl.handle.net/10803/385612

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

6. Ζαμπάς, Νεόφυτος. Μελέτη της προστατευτικής δράσης της αντιθρομβίνης ΙΙΙ στην καρδιά και στους πνεύμονες στο σύνδρομο οξείας ισχαιμίας - επαναιμάτωσης των οπισθίων άκρων: πειραματική μελέτη σε επίμυες.

Degree: 2010, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

 Introduction: Acute ischemia-reperfusion of a lower limb could cause injury to distal organs (remote ischemia-reperfusion) and systematic inflammation response resulting in high mortality rates. Lipid… (more)

Subjects/Keywords: Αντιθρομβίνη ΙΙΙ - (ΑΤ-ΙΙΙ); Ισχαιμία επαναιμάτωση (I-R) οπίσθιου άκρου; Ελεύθερες ρίζες οξυγόνου; Καρδιά; Πνεύμονες; Antithrombin III (AT-III); Ischemia-reperfusion (I-R) hind limb; Oxygen free radicals; Heart; Lungs

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ζαμπάς, . . (2010). Μελέτη της προστατευτικής δράσης της αντιθρομβίνης ΙΙΙ στην καρδιά και στους πνεύμονες στο σύνδρομο οξείας ισχαιμίας - επαναιμάτωσης των οπισθίων άκρων: πειραματική μελέτη σε επίμυες. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/28324

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ζαμπάς, Νεόφυτος. “Μελέτη της προστατευτικής δράσης της αντιθρομβίνης ΙΙΙ στην καρδιά και στους πνεύμονες στο σύνδρομο οξείας ισχαιμίας - επαναιμάτωσης των οπισθίων άκρων: πειραματική μελέτη σε επίμυες.” 2010. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed November 27, 2020. http://hdl.handle.net/10442/hedi/28324.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ζαμπάς, Νεόφυτος. “Μελέτη της προστατευτικής δράσης της αντιθρομβίνης ΙΙΙ στην καρδιά και στους πνεύμονες στο σύνδρομο οξείας ισχαιμίας - επαναιμάτωσης των οπισθίων άκρων: πειραματική μελέτη σε επίμυες.” 2010. Web. 27 Nov 2020.

Vancouver:

Ζαμπάς . Μελέτη της προστατευτικής δράσης της αντιθρομβίνης ΙΙΙ στην καρδιά και στους πνεύμονες στο σύνδρομο οξείας ισχαιμίας - επαναιμάτωσης των οπισθίων άκρων: πειραματική μελέτη σε επίμυες. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2010. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/10442/hedi/28324.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ζαμπάς . Μελέτη της προστατευτικής δράσης της αντιθρομβίνης ΙΙΙ στην καρδιά και στους πνεύμονες στο σύνδρομο οξείας ισχαιμίας - επαναιμάτωσης των οπισθίων άκρων: πειραματική μελέτη σε επίμυες. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2010. Available from: http://hdl.handle.net/10442/hedi/28324

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Olland, Anne. Intérêt des microparticules pour l'étude de l'ischémie reperfusion en tranplantation pulmonaire basé sur un modèle de perfusion ventilation pulmonaire ex vivo chez le rat : Relevance of microparticles for the study of ischemia reperfusion in lung transplantation using an experimental model of ex vivo rat lung reperfusion and ventilation.

Degree: Docteur es, Sciences de la vie et de la santé, 2016, Université de Strasbourg

L’ischémie reperfusion pulmonaire et sa traduction clinique la dysfonction primaire du greffon sont responsables d’une morbi-mortalité importante en transplantation pulmonaire aussi bien à court terme… (more)

Subjects/Keywords: Ischémie reperfusion; Reperfusion ex vivo; Microparticules; Ischemia reperfusion; Ex vivo reperfusion; Microparticles; 571.96; 617.9

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Olland, A. (2016). Intérêt des microparticules pour l'étude de l'ischémie reperfusion en tranplantation pulmonaire basé sur un modèle de perfusion ventilation pulmonaire ex vivo chez le rat : Relevance of microparticles for the study of ischemia reperfusion in lung transplantation using an experimental model of ex vivo rat lung reperfusion and ventilation. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2016STRAJ035

Chicago Manual of Style (16th Edition):

Olland, Anne. “Intérêt des microparticules pour l'étude de l'ischémie reperfusion en tranplantation pulmonaire basé sur un modèle de perfusion ventilation pulmonaire ex vivo chez le rat : Relevance of microparticles for the study of ischemia reperfusion in lung transplantation using an experimental model of ex vivo rat lung reperfusion and ventilation.” 2016. Doctoral Dissertation, Université de Strasbourg. Accessed November 27, 2020. http://www.theses.fr/2016STRAJ035.

MLA Handbook (7th Edition):

Olland, Anne. “Intérêt des microparticules pour l'étude de l'ischémie reperfusion en tranplantation pulmonaire basé sur un modèle de perfusion ventilation pulmonaire ex vivo chez le rat : Relevance of microparticles for the study of ischemia reperfusion in lung transplantation using an experimental model of ex vivo rat lung reperfusion and ventilation.” 2016. Web. 27 Nov 2020.

Vancouver:

Olland A. Intérêt des microparticules pour l'étude de l'ischémie reperfusion en tranplantation pulmonaire basé sur un modèle de perfusion ventilation pulmonaire ex vivo chez le rat : Relevance of microparticles for the study of ischemia reperfusion in lung transplantation using an experimental model of ex vivo rat lung reperfusion and ventilation. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2016. [cited 2020 Nov 27]. Available from: http://www.theses.fr/2016STRAJ035.

Council of Science Editors:

Olland A. Intérêt des microparticules pour l'étude de l'ischémie reperfusion en tranplantation pulmonaire basé sur un modèle de perfusion ventilation pulmonaire ex vivo chez le rat : Relevance of microparticles for the study of ischemia reperfusion in lung transplantation using an experimental model of ex vivo rat lung reperfusion and ventilation. [Doctoral Dissertation]. Université de Strasbourg; 2016. Available from: http://www.theses.fr/2016STRAJ035

8. 下内, 昭人. 網膜虚血再灌流障害における分散ヘスペレチンの神経保護効果 : Neuroprotective effect of water-dispersible hesperetin in retinal ischemia reperfusion injury.

Degree: 博士(医学), 2016, Asahikawa Medical University / 旭川医科大学

First online: 25 September 2015The final publication is available at Springer via http://dx.doi.org/10.1007/s10384-015-0415-z

PurposeTo determine whether water-dispersible hesperetin (WD-Hpt) can prevent degeneration of ganglion cell… (more)

Subjects/Keywords: Hesperetin; Ischemia–reperfusion; Neuroprotection; Retina; Inflammation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

下内, . (2016). 網膜虚血再灌流障害における分散ヘスペレチンの神経保護効果 : Neuroprotective effect of water-dispersible hesperetin in retinal ischemia reperfusion injury. (Thesis). Asahikawa Medical University / 旭川医科大学. Retrieved from http://amcor.asahikawa-med.ac.jp/modules/xoonips/detail.php?id=20160325_K492

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

下内, 昭人. “網膜虚血再灌流障害における分散ヘスペレチンの神経保護効果 : Neuroprotective effect of water-dispersible hesperetin in retinal ischemia reperfusion injury.” 2016. Thesis, Asahikawa Medical University / 旭川医科大学. Accessed November 27, 2020. http://amcor.asahikawa-med.ac.jp/modules/xoonips/detail.php?id=20160325_K492.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

下内, 昭人. “網膜虚血再灌流障害における分散ヘスペレチンの神経保護効果 : Neuroprotective effect of water-dispersible hesperetin in retinal ischemia reperfusion injury.” 2016. Web. 27 Nov 2020.

Vancouver:

下内 . 網膜虚血再灌流障害における分散ヘスペレチンの神経保護効果 : Neuroprotective effect of water-dispersible hesperetin in retinal ischemia reperfusion injury. [Internet] [Thesis]. Asahikawa Medical University / 旭川医科大学; 2016. [cited 2020 Nov 27]. Available from: http://amcor.asahikawa-med.ac.jp/modules/xoonips/detail.php?id=20160325_K492.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

下内 . 網膜虚血再灌流障害における分散ヘスペレチンの神経保護効果 : Neuroprotective effect of water-dispersible hesperetin in retinal ischemia reperfusion injury. [Thesis]. Asahikawa Medical University / 旭川医科大学; 2016. Available from: http://amcor.asahikawa-med.ac.jp/modules/xoonips/detail.php?id=20160325_K492

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. 畑地, 豪. The Poly(Adenosine Diphosphate-Ribose) Polymerase Inhibitor PJ34 Reduces Pulmonary Ischemia-Reperfusion Injury in Rats : ポリADPリボースポリメレース阻害薬(PJ34)はラットにおける肺虚血再灌流障害を軽減する.

Degree: 博士(医学), 2014, Nagasaki University / 長崎大学

 Background: Ischemia-reperfusion (I/R) injury after lung transplantation causes alveolar damage, lung edema, and acute rejection. Poly(adenosine diphosphate-ribose) polymerase (PARP) is a single-stranded DNA repair enzyme… (more)

Subjects/Keywords: Ischemia-reperfusion injury; PARP inhibitor; PJ34; Antioxidants

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

畑地, . (2014). The Poly(Adenosine Diphosphate-Ribose) Polymerase Inhibitor PJ34 Reduces Pulmonary Ischemia-Reperfusion Injury in Rats : ポリADPリボースポリメレース阻害薬(PJ34)はラットにおける肺虚血再灌流障害を軽減する. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/35827

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

畑地, 豪. “The Poly(Adenosine Diphosphate-Ribose) Polymerase Inhibitor PJ34 Reduces Pulmonary Ischemia-Reperfusion Injury in Rats : ポリADPリボースポリメレース阻害薬(PJ34)はラットにおける肺虚血再灌流障害を軽減する.” 2014. Thesis, Nagasaki University / 長崎大学. Accessed November 27, 2020. http://hdl.handle.net/10069/35827.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

畑地, 豪. “The Poly(Adenosine Diphosphate-Ribose) Polymerase Inhibitor PJ34 Reduces Pulmonary Ischemia-Reperfusion Injury in Rats : ポリADPリボースポリメレース阻害薬(PJ34)はラットにおける肺虚血再灌流障害を軽減する.” 2014. Web. 27 Nov 2020.

Vancouver:

畑地 . The Poly(Adenosine Diphosphate-Ribose) Polymerase Inhibitor PJ34 Reduces Pulmonary Ischemia-Reperfusion Injury in Rats : ポリADPリボースポリメレース阻害薬(PJ34)はラットにおける肺虚血再灌流障害を軽減する. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2014. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/10069/35827.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

畑地 . The Poly(Adenosine Diphosphate-Ribose) Polymerase Inhibitor PJ34 Reduces Pulmonary Ischemia-Reperfusion Injury in Rats : ポリADPリボースポリメレース阻害薬(PJ34)はラットにおける肺虚血再灌流障害を軽減する. [Thesis]. Nagasaki University / 長崎大学; 2014. Available from: http://hdl.handle.net/10069/35827

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

10. Qadhi, Rawabi. Docosahexaenoic Acid Induced Apoptosis In H9c2 Cells And Changed Cardiac Function After Ischemia-Reperfusion Injury.

Degree: MS, Faculty of Pharmacy and Pharmaceutical Sciences, 2013, University of Alberta

 Cardiovascular disease (CVD) remains one of the leading causes of death worldwide. As such, a vast amount of research has investigated novel therapies for preventing… (more)

Subjects/Keywords: H9c2 cells; Apoptosis; DHA; Ischemia/reperfusion injury

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Qadhi, R. (2013). Docosahexaenoic Acid Induced Apoptosis In H9c2 Cells And Changed Cardiac Function After Ischemia-Reperfusion Injury. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/fx719m76s

Chicago Manual of Style (16th Edition):

Qadhi, Rawabi. “Docosahexaenoic Acid Induced Apoptosis In H9c2 Cells And Changed Cardiac Function After Ischemia-Reperfusion Injury.” 2013. Masters Thesis, University of Alberta. Accessed November 27, 2020. https://era.library.ualberta.ca/files/fx719m76s.

MLA Handbook (7th Edition):

Qadhi, Rawabi. “Docosahexaenoic Acid Induced Apoptosis In H9c2 Cells And Changed Cardiac Function After Ischemia-Reperfusion Injury.” 2013. Web. 27 Nov 2020.

Vancouver:

Qadhi R. Docosahexaenoic Acid Induced Apoptosis In H9c2 Cells And Changed Cardiac Function After Ischemia-Reperfusion Injury. [Internet] [Masters thesis]. University of Alberta; 2013. [cited 2020 Nov 27]. Available from: https://era.library.ualberta.ca/files/fx719m76s.

Council of Science Editors:

Qadhi R. Docosahexaenoic Acid Induced Apoptosis In H9c2 Cells And Changed Cardiac Function After Ischemia-Reperfusion Injury. [Masters Thesis]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/fx719m76s

11. Güney, Türkan. Fukoidin’in rat aortik iskemi reperfüzyon modelinde böbrek ve akciğerler üzerine etkisi .

Degree: ESOGÜ, Tıp Fakültesi, Tıbbi Biyokimya, 2015, Eskisehir Osmangazi University

 Aort cerrahisinde abdominal aortun klemplenmesi ile oluşan iskemide birçok dokuda uzak organ hasarı meydana gelmektedir. Bu çalışmada fukoidinin rat aortik iskemi reperfüzyon modelinde böbrek ve… (more)

Subjects/Keywords: İskemi/Reperfüzyon; Fukoidin; Ischemia; Reperfusion; Fucoidin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Güney, T. (2015). Fukoidin’in rat aortik iskemi reperfüzyon modelinde böbrek ve akciğerler üzerine etkisi . (Thesis). Eskisehir Osmangazi University. Retrieved from http://hdl.handle.net/11684/631

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Güney, Türkan. “Fukoidin’in rat aortik iskemi reperfüzyon modelinde böbrek ve akciğerler üzerine etkisi .” 2015. Thesis, Eskisehir Osmangazi University. Accessed November 27, 2020. http://hdl.handle.net/11684/631.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Güney, Türkan. “Fukoidin’in rat aortik iskemi reperfüzyon modelinde böbrek ve akciğerler üzerine etkisi .” 2015. Web. 27 Nov 2020.

Vancouver:

Güney T. Fukoidin’in rat aortik iskemi reperfüzyon modelinde böbrek ve akciğerler üzerine etkisi . [Internet] [Thesis]. Eskisehir Osmangazi University; 2015. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/11684/631.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Güney T. Fukoidin’in rat aortik iskemi reperfüzyon modelinde böbrek ve akciğerler üzerine etkisi . [Thesis]. Eskisehir Osmangazi University; 2015. Available from: http://hdl.handle.net/11684/631

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

12. Lafreniere, Gina. mRNA Variants of a Neuronal Sodium Channel in Rat Cardiac Myocytes .

Degree: Physiology, 2015, Queens University

 Death due to ischemic heart disease (IHD) is the result of cardiac arrhythmias and loss of cardiac pump function. One of the putative underlying mechanisms… (more)

Subjects/Keywords: Ischemia/Reperfusion ; Heart ; Voltage Gated Sodium Channels

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lafreniere, G. (2015). mRNA Variants of a Neuronal Sodium Channel in Rat Cardiac Myocytes . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/12682

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lafreniere, Gina. “mRNA Variants of a Neuronal Sodium Channel in Rat Cardiac Myocytes .” 2015. Thesis, Queens University. Accessed November 27, 2020. http://hdl.handle.net/1974/12682.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lafreniere, Gina. “mRNA Variants of a Neuronal Sodium Channel in Rat Cardiac Myocytes .” 2015. Web. 27 Nov 2020.

Vancouver:

Lafreniere G. mRNA Variants of a Neuronal Sodium Channel in Rat Cardiac Myocytes . [Internet] [Thesis]. Queens University; 2015. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/1974/12682.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lafreniere G. mRNA Variants of a Neuronal Sodium Channel in Rat Cardiac Myocytes . [Thesis]. Queens University; 2015. Available from: http://hdl.handle.net/1974/12682

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

13. Rohailla, Sagar. A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies.

Degree: 2012, University of Toronto

Remote ischemic preconditioning (rIPC) through transient limb ischemia induces potent cardioprotection against ischemia reperfusion (IR) injury. I examined the delayed phase of protection that appears… (more)

Subjects/Keywords: Ischemia Reperfusion; Myocardial Infarction; Cardiology; Preconditioning; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rohailla, S. (2012). A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33511

Chicago Manual of Style (16th Edition):

Rohailla, Sagar. “A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies.” 2012. Masters Thesis, University of Toronto. Accessed November 27, 2020. http://hdl.handle.net/1807/33511.

MLA Handbook (7th Edition):

Rohailla, Sagar. “A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies.” 2012. Web. 27 Nov 2020.

Vancouver:

Rohailla S. A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/1807/33511.

Council of Science Editors:

Rohailla S. A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33511


University of Cambridge

14. DiRito, Jenna. Optimizing the delivery of pre-transplantation therapies during normothermic machine perfusion of human kidneys.

Degree: PhD, 2020, University of Cambridge

 Despite a severe organ shortage, thousands of kidneys from higher-risk donors go unused each year. These marginal kidneys have an increased susceptibility to ischemia reperfusion(more)

Subjects/Keywords: kidney; transplantation; nanoparticle; translational; ischemia reperfusion injury

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

DiRito, J. (2020). Optimizing the delivery of pre-transplantation therapies during normothermic machine perfusion of human kidneys. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/307976

Chicago Manual of Style (16th Edition):

DiRito, Jenna. “Optimizing the delivery of pre-transplantation therapies during normothermic machine perfusion of human kidneys.” 2020. Doctoral Dissertation, University of Cambridge. Accessed November 27, 2020. https://www.repository.cam.ac.uk/handle/1810/307976.

MLA Handbook (7th Edition):

DiRito, Jenna. “Optimizing the delivery of pre-transplantation therapies during normothermic machine perfusion of human kidneys.” 2020. Web. 27 Nov 2020.

Vancouver:

DiRito J. Optimizing the delivery of pre-transplantation therapies during normothermic machine perfusion of human kidneys. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2020 Nov 27]. Available from: https://www.repository.cam.ac.uk/handle/1810/307976.

Council of Science Editors:

DiRito J. Optimizing the delivery of pre-transplantation therapies during normothermic machine perfusion of human kidneys. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/307976


University of Toronto

15. Kwong, Wilson. Effect of Selective/Non-selective COX Inhibition on Rosuvastatin-Mediated Protection from Ischemia-reperfusion Induced Endothelial Dysfunction in the Human Forearm Vasculature.

Degree: 2011, University of Toronto

Statins can act as preconditioning agents against ischemia reperfusion (IR)-injury through a mechanism involving cyclooxygenase (COX)-2 and the upregulation of prostaglandin synthesis. The following study… (more)

Subjects/Keywords: statin; ischemia reperfusion injury; preconditioning; cox; 0419

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kwong, W. (2011). Effect of Selective/Non-selective COX Inhibition on Rosuvastatin-Mediated Protection from Ischemia-reperfusion Induced Endothelial Dysfunction in the Human Forearm Vasculature. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/29586

Chicago Manual of Style (16th Edition):

Kwong, Wilson. “Effect of Selective/Non-selective COX Inhibition on Rosuvastatin-Mediated Protection from Ischemia-reperfusion Induced Endothelial Dysfunction in the Human Forearm Vasculature.” 2011. Masters Thesis, University of Toronto. Accessed November 27, 2020. http://hdl.handle.net/1807/29586.

MLA Handbook (7th Edition):

Kwong, Wilson. “Effect of Selective/Non-selective COX Inhibition on Rosuvastatin-Mediated Protection from Ischemia-reperfusion Induced Endothelial Dysfunction in the Human Forearm Vasculature.” 2011. Web. 27 Nov 2020.

Vancouver:

Kwong W. Effect of Selective/Non-selective COX Inhibition on Rosuvastatin-Mediated Protection from Ischemia-reperfusion Induced Endothelial Dysfunction in the Human Forearm Vasculature. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/1807/29586.

Council of Science Editors:

Kwong W. Effect of Selective/Non-selective COX Inhibition on Rosuvastatin-Mediated Protection from Ischemia-reperfusion Induced Endothelial Dysfunction in the Human Forearm Vasculature. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/29586


Universidad de Extremadura

16. Ballestín Rodríguez, Alberto. Transferencia tisular libre en microcirugía reconstructiva: empleo de terapia celular para modular el daño por isquemia-reperfusión en colgajos cutáneos .

Degree: 2019, Universidad de Extremadura

 La microcirugía es una técnica quirúrgica empleada de forma rutinaria en transferencias tisulares libres en cirugía reconstructiva ante defectos causados por quemaduras, traumatismos o tras… (more)

Subjects/Keywords: Isquemia; Reperfusión; Microcirugía; Ischemia; Reperfusion; Microsurgery

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ballestín Rodríguez, A. (2019). Transferencia tisular libre en microcirugía reconstructiva: empleo de terapia celular para modular el daño por isquemia-reperfusión en colgajos cutáneos . (Thesis). Universidad de Extremadura. Retrieved from http://hdl.handle.net/10662/8978

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ballestín Rodríguez, Alberto. “Transferencia tisular libre en microcirugía reconstructiva: empleo de terapia celular para modular el daño por isquemia-reperfusión en colgajos cutáneos .” 2019. Thesis, Universidad de Extremadura. Accessed November 27, 2020. http://hdl.handle.net/10662/8978.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ballestín Rodríguez, Alberto. “Transferencia tisular libre en microcirugía reconstructiva: empleo de terapia celular para modular el daño por isquemia-reperfusión en colgajos cutáneos .” 2019. Web. 27 Nov 2020.

Vancouver:

Ballestín Rodríguez A. Transferencia tisular libre en microcirugía reconstructiva: empleo de terapia celular para modular el daño por isquemia-reperfusión en colgajos cutáneos . [Internet] [Thesis]. Universidad de Extremadura; 2019. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/10662/8978.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ballestín Rodríguez A. Transferencia tisular libre en microcirugía reconstructiva: empleo de terapia celular para modular el daño por isquemia-reperfusión en colgajos cutáneos . [Thesis]. Universidad de Extremadura; 2019. Available from: http://hdl.handle.net/10662/8978

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

17. DiRito, Jenna. Optimizing the delivery of pre-transplantation therapies during normothermic machine perfusion of human kidneys.

Degree: PhD, 2020, University of Cambridge

 Despite a severe organ shortage, thousands of kidneys from higher-risk donors go unused each year. These marginal kidneys have an increased susceptibility to ischemia reperfusion(more)

Subjects/Keywords: kidney; transplantation; nanoparticle; translational; ischemia reperfusion injury

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

DiRito, J. (2020). Optimizing the delivery of pre-transplantation therapies during normothermic machine perfusion of human kidneys. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.55068 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.810034

Chicago Manual of Style (16th Edition):

DiRito, Jenna. “Optimizing the delivery of pre-transplantation therapies during normothermic machine perfusion of human kidneys.” 2020. Doctoral Dissertation, University of Cambridge. Accessed November 27, 2020. https://doi.org/10.17863/CAM.55068 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.810034.

MLA Handbook (7th Edition):

DiRito, Jenna. “Optimizing the delivery of pre-transplantation therapies during normothermic machine perfusion of human kidneys.” 2020. Web. 27 Nov 2020.

Vancouver:

DiRito J. Optimizing the delivery of pre-transplantation therapies during normothermic machine perfusion of human kidneys. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2020 Nov 27]. Available from: https://doi.org/10.17863/CAM.55068 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.810034.

Council of Science Editors:

DiRito J. Optimizing the delivery of pre-transplantation therapies during normothermic machine perfusion of human kidneys. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://doi.org/10.17863/CAM.55068 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.810034

18. Katseni, Konstantina. Πειραματική μελέτη της επίδρασης των αντιοξειδωτικών φαρμάκων στο σύνδρομο ισχαιμίας - επαναιμάτωσης μετά από αποκλεισμό της υπονεφρικής μοίρας της κοιλιακής αορτής.

Degree: 2015, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

OBJECTIVES: The purpose of this study was to assess the inflammatory molecules released after infrarenal aortic clamping. In addition, we sought to investigate whether the… (more)

Subjects/Keywords: Σύνδρομο ισχαιμίας - επαναιμάτωσης; Ischemia - reperfusion syndrome

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Katseni, K. (2015). Πειραματική μελέτη της επίδρασης των αντιοξειδωτικών φαρμάκων στο σύνδρομο ισχαιμίας - επαναιμάτωσης μετά από αποκλεισμό της υπονεφρικής μοίρας της κοιλιακής αορτής. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/36710

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Katseni, Konstantina. “Πειραματική μελέτη της επίδρασης των αντιοξειδωτικών φαρμάκων στο σύνδρομο ισχαιμίας - επαναιμάτωσης μετά από αποκλεισμό της υπονεφρικής μοίρας της κοιλιακής αορτής.” 2015. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed November 27, 2020. http://hdl.handle.net/10442/hedi/36710.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Katseni, Konstantina. “Πειραματική μελέτη της επίδρασης των αντιοξειδωτικών φαρμάκων στο σύνδρομο ισχαιμίας - επαναιμάτωσης μετά από αποκλεισμό της υπονεφρικής μοίρας της κοιλιακής αορτής.” 2015. Web. 27 Nov 2020.

Vancouver:

Katseni K. Πειραματική μελέτη της επίδρασης των αντιοξειδωτικών φαρμάκων στο σύνδρομο ισχαιμίας - επαναιμάτωσης μετά από αποκλεισμό της υπονεφρικής μοίρας της κοιλιακής αορτής. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2015. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/10442/hedi/36710.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Katseni K. Πειραματική μελέτη της επίδρασης των αντιοξειδωτικών φαρμάκων στο σύνδρομο ισχαιμίας - επαναιμάτωσης μετά από αποκλεισμό της υπονεφρικής μοίρας της κοιλιακής αορτής. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2015. Available from: http://hdl.handle.net/10442/hedi/36710

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

19. Alrabadi, Nasr Nofal Salieba. Novel Pharmacological Approaches for Understanding and Modulating the Pathology of Ischemic Heart Disease.

Degree: 2016, University of Sydney

 Over the last century, ischemic heart diseases (IHD) have been the worldwide leading cause of death. Major improvement in the prevalence and prognosis of IHD… (more)

Subjects/Keywords: Ischemic Heart Disease; IHD; ischemia-reperfusion injury

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Alrabadi, N. N. S. (2016). Novel Pharmacological Approaches for Understanding and Modulating the Pathology of Ischemic Heart Disease. (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/15926

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alrabadi, Nasr Nofal Salieba. “Novel Pharmacological Approaches for Understanding and Modulating the Pathology of Ischemic Heart Disease. ” 2016. Thesis, University of Sydney. Accessed November 27, 2020. http://hdl.handle.net/2123/15926.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alrabadi, Nasr Nofal Salieba. “Novel Pharmacological Approaches for Understanding and Modulating the Pathology of Ischemic Heart Disease. ” 2016. Web. 27 Nov 2020.

Vancouver:

Alrabadi NNS. Novel Pharmacological Approaches for Understanding and Modulating the Pathology of Ischemic Heart Disease. [Internet] [Thesis]. University of Sydney; 2016. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/2123/15926.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alrabadi NNS. Novel Pharmacological Approaches for Understanding and Modulating the Pathology of Ischemic Heart Disease. [Thesis]. University of Sydney; 2016. Available from: http://hdl.handle.net/2123/15926

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Hoang, Quoc thang. Aspects diagnostiques et thérapeutiques du modèle d'ischémie - reperfusion mésentérique : Diagnostic and treatment aspects of mesenteric ischemia - reperfusion model.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie. Biothérapies, 2016, Sorbonne Paris Cité

Introduction: La lésion d'ischémie - reperfusion (I/R) intestinale est associée à une mortalité élevée.Le rôle de la transmigration des neutrophiles est probablement essentiel dans le… (more)

Subjects/Keywords: Ischémie - reperfusion mésentérique; P8RI; Mesenteric ischemia - reperfusion; P8RI

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hoang, Q. t. (2016). Aspects diagnostiques et thérapeutiques du modèle d'ischémie - reperfusion mésentérique : Diagnostic and treatment aspects of mesenteric ischemia - reperfusion model. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2016USPCC196

Chicago Manual of Style (16th Edition):

Hoang, Quoc thang. “Aspects diagnostiques et thérapeutiques du modèle d'ischémie - reperfusion mésentérique : Diagnostic and treatment aspects of mesenteric ischemia - reperfusion model.” 2016. Doctoral Dissertation, Sorbonne Paris Cité. Accessed November 27, 2020. http://www.theses.fr/2016USPCC196.

MLA Handbook (7th Edition):

Hoang, Quoc thang. “Aspects diagnostiques et thérapeutiques du modèle d'ischémie - reperfusion mésentérique : Diagnostic and treatment aspects of mesenteric ischemia - reperfusion model.” 2016. Web. 27 Nov 2020.

Vancouver:

Hoang Qt. Aspects diagnostiques et thérapeutiques du modèle d'ischémie - reperfusion mésentérique : Diagnostic and treatment aspects of mesenteric ischemia - reperfusion model. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2016. [cited 2020 Nov 27]. Available from: http://www.theses.fr/2016USPCC196.

Council of Science Editors:

Hoang Qt. Aspects diagnostiques et thérapeutiques du modèle d'ischémie - reperfusion mésentérique : Diagnostic and treatment aspects of mesenteric ischemia - reperfusion model. [Doctoral Dissertation]. Sorbonne Paris Cité; 2016. Available from: http://www.theses.fr/2016USPCC196

21. Sportouch-Dukhan, Catherine. Effets cardioprotecteurs du glutamate contre les lésions d'ischémie-reperfusion myocardique chez la souris. Evaluation échocardiographique. : Cardioprotective effects of glutamate against ischemia-reperfusion injury in the mouse heart. Echocardiographic evaluation.

Degree: Docteur es, Biochimie et biologie moléculaire, 2012, Université Montpellier I

L'infarctus du myocarde est la première cause de mortalité cardiovasculaire dans les pays occidentaux. La reperfusion la plus précoce possible est actuellement le seul traitement… (more)

Subjects/Keywords: Cardioprotection; Infarctus; Ischémie-reperfusion; Échocardiographie; Cardioprotection; Infarction; Ischemia-reperfusion; Echocardiography

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sportouch-Dukhan, C. (2012). Effets cardioprotecteurs du glutamate contre les lésions d'ischémie-reperfusion myocardique chez la souris. Evaluation échocardiographique. : Cardioprotective effects of glutamate against ischemia-reperfusion injury in the mouse heart. Echocardiographic evaluation. (Doctoral Dissertation). Université Montpellier I. Retrieved from http://www.theses.fr/2012MON1T021

Chicago Manual of Style (16th Edition):

Sportouch-Dukhan, Catherine. “Effets cardioprotecteurs du glutamate contre les lésions d'ischémie-reperfusion myocardique chez la souris. Evaluation échocardiographique. : Cardioprotective effects of glutamate against ischemia-reperfusion injury in the mouse heart. Echocardiographic evaluation.” 2012. Doctoral Dissertation, Université Montpellier I. Accessed November 27, 2020. http://www.theses.fr/2012MON1T021.

MLA Handbook (7th Edition):

Sportouch-Dukhan, Catherine. “Effets cardioprotecteurs du glutamate contre les lésions d'ischémie-reperfusion myocardique chez la souris. Evaluation échocardiographique. : Cardioprotective effects of glutamate against ischemia-reperfusion injury in the mouse heart. Echocardiographic evaluation.” 2012. Web. 27 Nov 2020.

Vancouver:

Sportouch-Dukhan C. Effets cardioprotecteurs du glutamate contre les lésions d'ischémie-reperfusion myocardique chez la souris. Evaluation échocardiographique. : Cardioprotective effects of glutamate against ischemia-reperfusion injury in the mouse heart. Echocardiographic evaluation. [Internet] [Doctoral dissertation]. Université Montpellier I; 2012. [cited 2020 Nov 27]. Available from: http://www.theses.fr/2012MON1T021.

Council of Science Editors:

Sportouch-Dukhan C. Effets cardioprotecteurs du glutamate contre les lésions d'ischémie-reperfusion myocardique chez la souris. Evaluation échocardiographique. : Cardioprotective effects of glutamate against ischemia-reperfusion injury in the mouse heart. Echocardiographic evaluation. [Doctoral Dissertation]. Université Montpellier I; 2012. Available from: http://www.theses.fr/2012MON1T021

22. Chevallier, Stéphane. Elastokines et Lactosylcéramide : cardioprotection et vieillissement : Elastokines et Lactosylceramide : cardioprotection et Ageing.

Degree: Docteur es, Pharmacologie, 2011, Reims

La maladie cardiovasculaire la plus courante dans les pays industrialisés est la maladie coronarienne responsable d’une ischémie du tissu cardiaque pouvant conduire à l’infarctus du… (more)

Subjects/Keywords: Ischémie; Reperfusion; Elastine; Lactosylcéramides; Ischemia; Reperfusion; Elastin; Lactosylceramides

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chevallier, S. (2011). Elastokines et Lactosylcéramide : cardioprotection et vieillissement : Elastokines et Lactosylceramide : cardioprotection et Ageing. (Doctoral Dissertation). Reims. Retrieved from http://www.theses.fr/2011REIMM203

Chicago Manual of Style (16th Edition):

Chevallier, Stéphane. “Elastokines et Lactosylcéramide : cardioprotection et vieillissement : Elastokines et Lactosylceramide : cardioprotection et Ageing.” 2011. Doctoral Dissertation, Reims. Accessed November 27, 2020. http://www.theses.fr/2011REIMM203.

MLA Handbook (7th Edition):

Chevallier, Stéphane. “Elastokines et Lactosylcéramide : cardioprotection et vieillissement : Elastokines et Lactosylceramide : cardioprotection et Ageing.” 2011. Web. 27 Nov 2020.

Vancouver:

Chevallier S. Elastokines et Lactosylcéramide : cardioprotection et vieillissement : Elastokines et Lactosylceramide : cardioprotection et Ageing. [Internet] [Doctoral dissertation]. Reims; 2011. [cited 2020 Nov 27]. Available from: http://www.theses.fr/2011REIMM203.

Council of Science Editors:

Chevallier S. Elastokines et Lactosylcéramide : cardioprotection et vieillissement : Elastokines et Lactosylceramide : cardioprotection et Ageing. [Doctoral Dissertation]. Reims; 2011. Available from: http://www.theses.fr/2011REIMM203

23. Paradis, Stephanie. Effet cardioprotecteur des ligands de la protéine translocatrice mitochondriale (TSPO) au cours de l'ischémie-reperfusion myocardique expérimentale : rôle du cholestérol. : Cardioprotective effect of mitochondrial translocator protein (TSPO) ligands during experimental myocardial ischemia-reperfusion : role of cholesterol.

Degree: Docteur es, Pharmacologie et Biothérapies, 2012, Université Paris-Est

Dans ce travail, nous avons montré qu'un nouveau ligand du TSPO, le TRO40303, possède des propriétés cardioprotectrices, confirmant que le TSPO joue un rôle important… (more)

Subjects/Keywords: Cholestérol; Ischémie reperfusion; Mitochondrie; Cardioprotection; Cholesterol; Ischemia reperfusion; Mitochondria; Cardioprotection

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Paradis, S. (2012). Effet cardioprotecteur des ligands de la protéine translocatrice mitochondriale (TSPO) au cours de l'ischémie-reperfusion myocardique expérimentale : rôle du cholestérol. : Cardioprotective effect of mitochondrial translocator protein (TSPO) ligands during experimental myocardial ischemia-reperfusion : role of cholesterol. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2012PEST0071

Chicago Manual of Style (16th Edition):

Paradis, Stephanie. “Effet cardioprotecteur des ligands de la protéine translocatrice mitochondriale (TSPO) au cours de l'ischémie-reperfusion myocardique expérimentale : rôle du cholestérol. : Cardioprotective effect of mitochondrial translocator protein (TSPO) ligands during experimental myocardial ischemia-reperfusion : role of cholesterol.” 2012. Doctoral Dissertation, Université Paris-Est. Accessed November 27, 2020. http://www.theses.fr/2012PEST0071.

MLA Handbook (7th Edition):

Paradis, Stephanie. “Effet cardioprotecteur des ligands de la protéine translocatrice mitochondriale (TSPO) au cours de l'ischémie-reperfusion myocardique expérimentale : rôle du cholestérol. : Cardioprotective effect of mitochondrial translocator protein (TSPO) ligands during experimental myocardial ischemia-reperfusion : role of cholesterol.” 2012. Web. 27 Nov 2020.

Vancouver:

Paradis S. Effet cardioprotecteur des ligands de la protéine translocatrice mitochondriale (TSPO) au cours de l'ischémie-reperfusion myocardique expérimentale : rôle du cholestérol. : Cardioprotective effect of mitochondrial translocator protein (TSPO) ligands during experimental myocardial ischemia-reperfusion : role of cholesterol. [Internet] [Doctoral dissertation]. Université Paris-Est; 2012. [cited 2020 Nov 27]. Available from: http://www.theses.fr/2012PEST0071.

Council of Science Editors:

Paradis S. Effet cardioprotecteur des ligands de la protéine translocatrice mitochondriale (TSPO) au cours de l'ischémie-reperfusion myocardique expérimentale : rôle du cholestérol. : Cardioprotective effect of mitochondrial translocator protein (TSPO) ligands during experimental myocardial ischemia-reperfusion : role of cholesterol. [Doctoral Dissertation]. Université Paris-Est; 2012. Available from: http://www.theses.fr/2012PEST0071

24. Noll, Éric. Ischémie-reperfusion musculaire squelettique expérimentale : place de la lactatémie capillaire dans le monitorage de la reperfusion et transposition au modèle intestinal : Experimental skeletal muscle ischemia-reperfusion : capillary lactate for reperfusion monitoring.

Degree: Docteur es, Recherche clinique et innovation technologique, 2016, Université de Strasbourg

Les objectifs de ce travail expérimental étaient : 1. Évaluer la place de la lactatémie capillaire comme dispositif de monitorage de l’ischémie et de la… (more)

Subjects/Keywords: Ischémie-Reperfusion; Lactate; Ischemia-Reperfusion; Lactate; 571.5; 616.13

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Noll, . (2016). Ischémie-reperfusion musculaire squelettique expérimentale : place de la lactatémie capillaire dans le monitorage de la reperfusion et transposition au modèle intestinal : Experimental skeletal muscle ischemia-reperfusion : capillary lactate for reperfusion monitoring. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2016STRAJ006

Chicago Manual of Style (16th Edition):

Noll, Éric. “Ischémie-reperfusion musculaire squelettique expérimentale : place de la lactatémie capillaire dans le monitorage de la reperfusion et transposition au modèle intestinal : Experimental skeletal muscle ischemia-reperfusion : capillary lactate for reperfusion monitoring.” 2016. Doctoral Dissertation, Université de Strasbourg. Accessed November 27, 2020. http://www.theses.fr/2016STRAJ006.

MLA Handbook (7th Edition):

Noll, Éric. “Ischémie-reperfusion musculaire squelettique expérimentale : place de la lactatémie capillaire dans le monitorage de la reperfusion et transposition au modèle intestinal : Experimental skeletal muscle ischemia-reperfusion : capillary lactate for reperfusion monitoring.” 2016. Web. 27 Nov 2020.

Vancouver:

Noll . Ischémie-reperfusion musculaire squelettique expérimentale : place de la lactatémie capillaire dans le monitorage de la reperfusion et transposition au modèle intestinal : Experimental skeletal muscle ischemia-reperfusion : capillary lactate for reperfusion monitoring. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2016. [cited 2020 Nov 27]. Available from: http://www.theses.fr/2016STRAJ006.

Council of Science Editors:

Noll . Ischémie-reperfusion musculaire squelettique expérimentale : place de la lactatémie capillaire dans le monitorage de la reperfusion et transposition au modèle intestinal : Experimental skeletal muscle ischemia-reperfusion : capillary lactate for reperfusion monitoring. [Doctoral Dissertation]. Université de Strasbourg; 2016. Available from: http://www.theses.fr/2016STRAJ006


University of Rochester

25. Mai, Nguyen. Lung-Brain Immunological Coupling Mediates Neutrophil De-Priming and Neuroprotection following Cerebral Ischemia-Reperfusion.

Degree: PhD, 2017, University of Rochester

 Systemic inflammation and multi-organ failure represent two hallmarks of the Post Cardiac Arrest Syndrome (PCAS), and when present, are associated with severe neurological injury and… (more)

Subjects/Keywords: Brain ischemia; Cardiac arrest; Ischemia-reperfusion; Neutrophil; Systematic inflammation; Neuroinflammation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mai, N. (2017). Lung-Brain Immunological Coupling Mediates Neutrophil De-Priming and Neuroprotection following Cerebral Ischemia-Reperfusion. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/32920

Chicago Manual of Style (16th Edition):

Mai, Nguyen. “Lung-Brain Immunological Coupling Mediates Neutrophil De-Priming and Neuroprotection following Cerebral Ischemia-Reperfusion.” 2017. Doctoral Dissertation, University of Rochester. Accessed November 27, 2020. http://hdl.handle.net/1802/32920.

MLA Handbook (7th Edition):

Mai, Nguyen. “Lung-Brain Immunological Coupling Mediates Neutrophil De-Priming and Neuroprotection following Cerebral Ischemia-Reperfusion.” 2017. Web. 27 Nov 2020.

Vancouver:

Mai N. Lung-Brain Immunological Coupling Mediates Neutrophil De-Priming and Neuroprotection following Cerebral Ischemia-Reperfusion. [Internet] [Doctoral dissertation]. University of Rochester; 2017. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/1802/32920.

Council of Science Editors:

Mai N. Lung-Brain Immunological Coupling Mediates Neutrophil De-Priming and Neuroprotection following Cerebral Ischemia-Reperfusion. [Doctoral Dissertation]. University of Rochester; 2017. Available from: http://hdl.handle.net/1802/32920


University of Florida

26. Kirkby, Kristin. The Effects of Low Level Laser Therapy on Intestinal Ischemia Reperfusion Injury.

Degree: PhD, Veterinary Medical Sciences - Veterinary Medicine, 2012, University of Florida

 Intestinal ischemia reperfusion injury (IRI) in animals andhumans is associated with mortality rates between 50-80%. Various pharmacologicagents have been studied as potential treatments for IRI;… (more)

Subjects/Keywords: Dosage; Ischemia; Laser therapy; Lasers; Lungs; Oxygen; Physical trauma; Rats; Reperfusion; Reperfusion injury; injury  – intestine  – ischemia  – laser  – lung  – rat  – reperfusion  – therapy

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kirkby, K. (2012). The Effects of Low Level Laser Therapy on Intestinal Ischemia Reperfusion Injury. (Doctoral Dissertation). University of Florida. Retrieved from https://ufdc.ufl.edu/UFE0044834

Chicago Manual of Style (16th Edition):

Kirkby, Kristin. “The Effects of Low Level Laser Therapy on Intestinal Ischemia Reperfusion Injury.” 2012. Doctoral Dissertation, University of Florida. Accessed November 27, 2020. https://ufdc.ufl.edu/UFE0044834.

MLA Handbook (7th Edition):

Kirkby, Kristin. “The Effects of Low Level Laser Therapy on Intestinal Ischemia Reperfusion Injury.” 2012. Web. 27 Nov 2020.

Vancouver:

Kirkby K. The Effects of Low Level Laser Therapy on Intestinal Ischemia Reperfusion Injury. [Internet] [Doctoral dissertation]. University of Florida; 2012. [cited 2020 Nov 27]. Available from: https://ufdc.ufl.edu/UFE0044834.

Council of Science Editors:

Kirkby K. The Effects of Low Level Laser Therapy on Intestinal Ischemia Reperfusion Injury. [Doctoral Dissertation]. University of Florida; 2012. Available from: https://ufdc.ufl.edu/UFE0044834

27. Sunu, Shawn Y. MITOCHONDRIAL THERAPEUTICS DURING ISCHEMIA-REPERFUSION; MODULATION OF COMPLEX I: EFFECT OF METFORMIN.

Degree: MS, Physiology and Biophysics, 2015, Virginia Commonwealth University

  The modulation of the electron transport during ischemia-reperfusion has been shown to be protective. We hypothesized that metformin, a Complex I inhibitor, may exhibit… (more)

Subjects/Keywords: Ischemia-Reperfusion; Mitochondria; Metformin; Complex I; Cellular and Molecular Physiology

…MITOCHONDRIAL THERAPEUTICS DURING ISCHEMIA-REPERFUSION; MODULATION OF COMPLEX I: EFFECT OF METFORMIN… …nitrosating agent before ischemia or during reperfusion rescued Complex I activity through… …during ischemia-reperfusion has been shown to be protective. We hypothesized that metformin, a… …long-term therapeutic intervention against ischemia-reperfusion injury. Mitochondria were… …to enable mitochondria to stay intact after ischemia-reperfusion in order for oxidative… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sunu, S. Y. (2015). MITOCHONDRIAL THERAPEUTICS DURING ISCHEMIA-REPERFUSION; MODULATION OF COMPLEX I: EFFECT OF METFORMIN. (Thesis). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/GWT4-0F13 ; https://scholarscompass.vcu.edu/etd/3822

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sunu, Shawn Y. “MITOCHONDRIAL THERAPEUTICS DURING ISCHEMIA-REPERFUSION; MODULATION OF COMPLEX I: EFFECT OF METFORMIN.” 2015. Thesis, Virginia Commonwealth University. Accessed November 27, 2020. https://doi.org/10.25772/GWT4-0F13 ; https://scholarscompass.vcu.edu/etd/3822.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sunu, Shawn Y. “MITOCHONDRIAL THERAPEUTICS DURING ISCHEMIA-REPERFUSION; MODULATION OF COMPLEX I: EFFECT OF METFORMIN.” 2015. Web. 27 Nov 2020.

Vancouver:

Sunu SY. MITOCHONDRIAL THERAPEUTICS DURING ISCHEMIA-REPERFUSION; MODULATION OF COMPLEX I: EFFECT OF METFORMIN. [Internet] [Thesis]. Virginia Commonwealth University; 2015. [cited 2020 Nov 27]. Available from: https://doi.org/10.25772/GWT4-0F13 ; https://scholarscompass.vcu.edu/etd/3822.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sunu SY. MITOCHONDRIAL THERAPEUTICS DURING ISCHEMIA-REPERFUSION; MODULATION OF COMPLEX I: EFFECT OF METFORMIN. [Thesis]. Virginia Commonwealth University; 2015. Available from: https://doi.org/10.25772/GWT4-0F13 ; https://scholarscompass.vcu.edu/etd/3822

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Sadek, Hesham A. REPERFUSION-INDUCED MODULATION OF CARDIAC MITOCHONDRIAL FUNCTION BY FREE RADICALS AND CALCIUM.

Degree: PhD, Physiology and Biophysics, 2004, Case Western Reserve University School of Graduate Studies

Reperfusion of an ischemic myocardium is associated with a number of deleterious events that are thought to contribute to myocardial dysfunction. Cellular events that predominate… (more)

Subjects/Keywords: Cardiac ischemia; Reperfusion; Mitochondria; Complex I; Free radicals; Superoxide; Calcium

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sadek, H. A. (2004). REPERFUSION-INDUCED MODULATION OF CARDIAC MITOCHONDRIAL FUNCTION BY FREE RADICALS AND CALCIUM. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1086333463

Chicago Manual of Style (16th Edition):

Sadek, Hesham A. “REPERFUSION-INDUCED MODULATION OF CARDIAC MITOCHONDRIAL FUNCTION BY FREE RADICALS AND CALCIUM.” 2004. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed November 27, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1086333463.

MLA Handbook (7th Edition):

Sadek, Hesham A. “REPERFUSION-INDUCED MODULATION OF CARDIAC MITOCHONDRIAL FUNCTION BY FREE RADICALS AND CALCIUM.” 2004. Web. 27 Nov 2020.

Vancouver:

Sadek HA. REPERFUSION-INDUCED MODULATION OF CARDIAC MITOCHONDRIAL FUNCTION BY FREE RADICALS AND CALCIUM. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2004. [cited 2020 Nov 27]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1086333463.

Council of Science Editors:

Sadek HA. REPERFUSION-INDUCED MODULATION OF CARDIAC MITOCHONDRIAL FUNCTION BY FREE RADICALS AND CALCIUM. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2004. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1086333463

29. Codas Duarte, Ricardo. Ischémie-reperfusion : impact de la perfusion rénale sur la fonction des greffons : Ischemia-reperfusion : impact of renal perfusion on grafts function.

Degree: Docteur es, Recherche clinique, innovation technologique, santé, 2013, Poitiers

La pénurie d'organes a amené les équipes de transplantation à élargir l'acceptation des greffons à des organes provenant de donneurs marginaux.Le recours aux greffons marginaux… (more)

Subjects/Keywords: Ischémie; Reperfusion; Machine de perfusion; Transplantation rénale; Ischemia; Reperfusion; Perfusion machine; Kidney transplantation; 617.9

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Codas Duarte, R. (2013). Ischémie-reperfusion : impact de la perfusion rénale sur la fonction des greffons : Ischemia-reperfusion : impact of renal perfusion on grafts function. (Doctoral Dissertation). Poitiers. Retrieved from http://www.theses.fr/2013POIT1404

Chicago Manual of Style (16th Edition):

Codas Duarte, Ricardo. “Ischémie-reperfusion : impact de la perfusion rénale sur la fonction des greffons : Ischemia-reperfusion : impact of renal perfusion on grafts function.” 2013. Doctoral Dissertation, Poitiers. Accessed November 27, 2020. http://www.theses.fr/2013POIT1404.

MLA Handbook (7th Edition):

Codas Duarte, Ricardo. “Ischémie-reperfusion : impact de la perfusion rénale sur la fonction des greffons : Ischemia-reperfusion : impact of renal perfusion on grafts function.” 2013. Web. 27 Nov 2020.

Vancouver:

Codas Duarte R. Ischémie-reperfusion : impact de la perfusion rénale sur la fonction des greffons : Ischemia-reperfusion : impact of renal perfusion on grafts function. [Internet] [Doctoral dissertation]. Poitiers; 2013. [cited 2020 Nov 27]. Available from: http://www.theses.fr/2013POIT1404.

Council of Science Editors:

Codas Duarte R. Ischémie-reperfusion : impact de la perfusion rénale sur la fonction des greffons : Ischemia-reperfusion : impact of renal perfusion on grafts function. [Doctoral Dissertation]. Poitiers; 2013. Available from: http://www.theses.fr/2013POIT1404

30. Amrouche, Lucile. Implication de miR-146a en physiopathologie rénale : The fine-tuning of CXCL8 protects kidney against ischemia-reperfusion injury in mice lacking microRNA-146a.

Degree: Docteur es, Biologie cellulaire, 2015, Sorbonne Paris Cité

Les microARNs (miARN) sont de petits ARN régulant l’expression des gènes au niveau post-transcriptionnel. Ils sont impliqués dans la régulation de nombreux processus biologiques nécessaires… (more)

Subjects/Keywords: MiR-146a; Inflammation; Ischémie-reperfusion rénale; MiR-146a; Inflammation; Renal ischemia-reperfusion; 571.96

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Amrouche, L. (2015). Implication de miR-146a en physiopathologie rénale : The fine-tuning of CXCL8 protects kidney against ischemia-reperfusion injury in mice lacking microRNA-146a. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2015USPCB105

Chicago Manual of Style (16th Edition):

Amrouche, Lucile. “Implication de miR-146a en physiopathologie rénale : The fine-tuning of CXCL8 protects kidney against ischemia-reperfusion injury in mice lacking microRNA-146a.” 2015. Doctoral Dissertation, Sorbonne Paris Cité. Accessed November 27, 2020. http://www.theses.fr/2015USPCB105.

MLA Handbook (7th Edition):

Amrouche, Lucile. “Implication de miR-146a en physiopathologie rénale : The fine-tuning of CXCL8 protects kidney against ischemia-reperfusion injury in mice lacking microRNA-146a.” 2015. Web. 27 Nov 2020.

Vancouver:

Amrouche L. Implication de miR-146a en physiopathologie rénale : The fine-tuning of CXCL8 protects kidney against ischemia-reperfusion injury in mice lacking microRNA-146a. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2015. [cited 2020 Nov 27]. Available from: http://www.theses.fr/2015USPCB105.

Council of Science Editors:

Amrouche L. Implication de miR-146a en physiopathologie rénale : The fine-tuning of CXCL8 protects kidney against ischemia-reperfusion injury in mice lacking microRNA-146a. [Doctoral Dissertation]. Sorbonne Paris Cité; 2015. Available from: http://www.theses.fr/2015USPCB105

[1] [2] [3] [4] [5] … [884]

.