Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Interleukin 1). Showing records 1 – 30 of 230 total matches.

[1] [2] [3] [4] [5] [6] [7] [8]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters


University of Utah

1. Gahring, Lorise Catherine. Effects of ultraviolet radiation on the production of epidermal-derived thymocyte activating factor/interleukin-1;.

Degree: PhD, Pathology;, 1986, University of Utah

 The effects of both a single exposure and multiple exposure to ultraviolet radiation (UVR) on the production and/or release of epidermal cell-derived thymocyte activating factor/interleukin-1(more)

Subjects/Keywords: Interleukin-1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gahring, L. C. (1986). Effects of ultraviolet radiation on the production of epidermal-derived thymocyte activating factor/interleukin-1;. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1198/rec/435

Chicago Manual of Style (16th Edition):

Gahring, Lorise Catherine. “Effects of ultraviolet radiation on the production of epidermal-derived thymocyte activating factor/interleukin-1;.” 1986. Doctoral Dissertation, University of Utah. Accessed July 23, 2019. http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1198/rec/435.

MLA Handbook (7th Edition):

Gahring, Lorise Catherine. “Effects of ultraviolet radiation on the production of epidermal-derived thymocyte activating factor/interleukin-1;.” 1986. Web. 23 Jul 2019.

Vancouver:

Gahring LC. Effects of ultraviolet radiation on the production of epidermal-derived thymocyte activating factor/interleukin-1;. [Internet] [Doctoral dissertation]. University of Utah; 1986. [cited 2019 Jul 23]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1198/rec/435.

Council of Science Editors:

Gahring LC. Effects of ultraviolet radiation on the production of epidermal-derived thymocyte activating factor/interleukin-1;. [Doctoral Dissertation]. University of Utah; 1986. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1198/rec/435


University of Manchester

2. Robinson, Emily. Mediators of inflammation in acute neurotoxicity.

Degree: PhD, 2013, University of Manchester

 Neuroinflammation is a major feature of most neurodegenerative conditions, and can leadto the exacerbation of neuronal injury. Inflammatory challenges in the central nervoussystem (CNS) have… (more)

Subjects/Keywords: 616.8; Interleukin-1; Inflammation; Excitotoxic

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Robinson, E. (2013). Mediators of inflammation in acute neurotoxicity. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/mediators-of-inflammation-in-acute-neurotoxicity(a657668f-f6de-4fb6-878d-65846fc18e66).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.576868

Chicago Manual of Style (16th Edition):

Robinson, Emily. “Mediators of inflammation in acute neurotoxicity.” 2013. Doctoral Dissertation, University of Manchester. Accessed July 23, 2019. https://www.research.manchester.ac.uk/portal/en/theses/mediators-of-inflammation-in-acute-neurotoxicity(a657668f-f6de-4fb6-878d-65846fc18e66).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.576868.

MLA Handbook (7th Edition):

Robinson, Emily. “Mediators of inflammation in acute neurotoxicity.” 2013. Web. 23 Jul 2019.

Vancouver:

Robinson E. Mediators of inflammation in acute neurotoxicity. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2019 Jul 23]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/mediators-of-inflammation-in-acute-neurotoxicity(a657668f-f6de-4fb6-878d-65846fc18e66).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.576868.

Council of Science Editors:

Robinson E. Mediators of inflammation in acute neurotoxicity. [Doctoral Dissertation]. University of Manchester; 2013. Available from: https://www.research.manchester.ac.uk/portal/en/theses/mediators-of-inflammation-in-acute-neurotoxicity(a657668f-f6de-4fb6-878d-65846fc18e66).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.576868


University of Manchester

3. Murray, Katie. Actions of interleukin-1 in cerebral ischaemia.

Degree: 2014, University of Manchester

 Cerebral ischaemia is characterised by an interruption in cerebral blood flow (CBF)leading to neuronal dysfunction and death. Pre-existing systemic inflammation isstrongly associated with exaggerated brain… (more)

Subjects/Keywords: interleukin-1; cerebral ischaemia

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Murray, K. (2014). Actions of interleukin-1 in cerebral ischaemia. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:241212

Chicago Manual of Style (16th Edition):

Murray, Katie. “Actions of interleukin-1 in cerebral ischaemia.” 2014. Doctoral Dissertation, University of Manchester. Accessed July 23, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:241212.

MLA Handbook (7th Edition):

Murray, Katie. “Actions of interleukin-1 in cerebral ischaemia.” 2014. Web. 23 Jul 2019.

Vancouver:

Murray K. Actions of interleukin-1 in cerebral ischaemia. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2019 Jul 23]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:241212.

Council of Science Editors:

Murray K. Actions of interleukin-1 in cerebral ischaemia. [Doctoral Dissertation]. University of Manchester; 2014. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:241212


University of Manchester

4. Murray, Katie. Actions of interleukin-1 in cerebral ischaemia.

Degree: PhD, 2014, University of Manchester

 Cerebral ischaemia is characterised by an interruption in cerebral blood flow (CBF) leading to neuronal dysfunction and death. Pre-existing systemic inflammation is strongly associated with… (more)

Subjects/Keywords: 616.8; interleukin-1; cerebral ischaemia

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Murray, K. (2014). Actions of interleukin-1 in cerebral ischaemia. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/actions-of-interleukin1-in-cerebral-ischaemia(76a8206b-4153-437d-9616-7d668a35db51).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.697735

Chicago Manual of Style (16th Edition):

Murray, Katie. “Actions of interleukin-1 in cerebral ischaemia.” 2014. Doctoral Dissertation, University of Manchester. Accessed July 23, 2019. https://www.research.manchester.ac.uk/portal/en/theses/actions-of-interleukin1-in-cerebral-ischaemia(76a8206b-4153-437d-9616-7d668a35db51).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.697735.

MLA Handbook (7th Edition):

Murray, Katie. “Actions of interleukin-1 in cerebral ischaemia.” 2014. Web. 23 Jul 2019.

Vancouver:

Murray K. Actions of interleukin-1 in cerebral ischaemia. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2019 Jul 23]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/actions-of-interleukin1-in-cerebral-ischaemia(76a8206b-4153-437d-9616-7d668a35db51).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.697735.

Council of Science Editors:

Murray K. Actions of interleukin-1 in cerebral ischaemia. [Doctoral Dissertation]. University of Manchester; 2014. Available from: https://www.research.manchester.ac.uk/portal/en/theses/actions-of-interleukin1-in-cerebral-ischaemia(76a8206b-4153-437d-9616-7d668a35db51).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.697735


University of Illinois – Urbana-Champaign

5. Chiu, Gabriel. Extracellular adenosine as a novel neuroimmune modulator.

Degree: PhD, 0191, 2014, University of Illinois – Urbana-Champaign

 Adenosine is a pleotropic purine nucleoside with actions ranging from neurotransmission to immunomodulation. Pathologic processes that trigger cellular stress and damage also cause significant increases… (more)

Subjects/Keywords: Adenosine; Interleukin-1; Caspase-1; Neuroimmunology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chiu, G. (2014). Extracellular adenosine as a novel neuroimmune modulator. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/49647

Chicago Manual of Style (16th Edition):

Chiu, Gabriel. “Extracellular adenosine as a novel neuroimmune modulator.” 2014. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed July 23, 2019. http://hdl.handle.net/2142/49647.

MLA Handbook (7th Edition):

Chiu, Gabriel. “Extracellular adenosine as a novel neuroimmune modulator.” 2014. Web. 23 Jul 2019.

Vancouver:

Chiu G. Extracellular adenosine as a novel neuroimmune modulator. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2014. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/2142/49647.

Council of Science Editors:

Chiu G. Extracellular adenosine as a novel neuroimmune modulator. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2014. Available from: http://hdl.handle.net/2142/49647

6. Besnard, Anne-Gaëlle. Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation.

Degree: Docteur es, Biologie cellulaire et moléculaire, 2010, Université d'Orléans

L’asthme est une maladie inflammatoire chronique des voies aériennes. Chez les individus sensibles, l’inhalation d’allergènes entraine une inflammation pulmonaire se traduisant par des épisodes récurrents… (more)

Subjects/Keywords: Interleukine-22; Interleukine-17; Interleukine-33; Interleukine-1; NLRP3 inflammasome; Interleukin-22; Interleukin-17; Interleukin-33; Interleukin-1; NLRP3 inflammasome

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Besnard, A. (2010). Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation. (Doctoral Dissertation). Université d'Orléans. Retrieved from http://www.theses.fr/2010ORLE2044

Chicago Manual of Style (16th Edition):

Besnard, Anne-Gaëlle. “Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation.” 2010. Doctoral Dissertation, Université d'Orléans. Accessed July 23, 2019. http://www.theses.fr/2010ORLE2044.

MLA Handbook (7th Edition):

Besnard, Anne-Gaëlle. “Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation.” 2010. Web. 23 Jul 2019.

Vancouver:

Besnard A. Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation. [Internet] [Doctoral dissertation]. Université d'Orléans; 2010. [cited 2019 Jul 23]. Available from: http://www.theses.fr/2010ORLE2044.

Council of Science Editors:

Besnard A. Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation. [Doctoral Dissertation]. Université d'Orléans; 2010. Available from: http://www.theses.fr/2010ORLE2044


University of Manchester

7. Giles, James. The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo.

Degree: PhD, 2012, University of Manchester

 Neuroinflammation is an important contributor to the pathogenesis of many neurological diseases. A key component of the innate immune response in the central nervous system… (more)

Subjects/Keywords: 616.8; neuro-inflammation; interleukin-1; platelet

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Giles, J. (2012). The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-plateletderived-interleukin1-alpha-as-a-driver-of-neutrophil-migration-in-vivo(8097504b-42c5-40c3-a18c-419bf79ebfb8).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559358

Chicago Manual of Style (16th Edition):

Giles, James. “The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo.” 2012. Doctoral Dissertation, University of Manchester. Accessed July 23, 2019. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-plateletderived-interleukin1-alpha-as-a-driver-of-neutrophil-migration-in-vivo(8097504b-42c5-40c3-a18c-419bf79ebfb8).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559358.

MLA Handbook (7th Edition):

Giles, James. “The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo.” 2012. Web. 23 Jul 2019.

Vancouver:

Giles J. The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2019 Jul 23]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-plateletderived-interleukin1-alpha-as-a-driver-of-neutrophil-migration-in-vivo(8097504b-42c5-40c3-a18c-419bf79ebfb8).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559358.

Council of Science Editors:

Giles J. The role of platelet-derived interleukin-1 alpha as a driver of neutrophil migration in vivo. [Doctoral Dissertation]. University of Manchester; 2012. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-plateletderived-interleukin1-alpha-as-a-driver-of-neutrophil-migration-in-vivo(8097504b-42c5-40c3-a18c-419bf79ebfb8).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559358


University of Manchester

8. Ogden, Stephanie. An investigation of Langerhans' cell function in aged skin.

Degree: PhD, 2013, University of Manchester

 With increasing age, aspects of the innate and adaptive immune systems show functional decline. In the skin this is associated with an increased incidence of… (more)

Subjects/Keywords: 616.97; Langerhans' cell; Ageing; Interleukin-1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ogden, S. (2013). An investigation of Langerhans' cell function in aged skin. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/an-investigation-of-langerhans-cell-function-in-aged-skin(a5d3967f-51bc-49fb-ab05-31e5aad4ae40).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.570266

Chicago Manual of Style (16th Edition):

Ogden, Stephanie. “An investigation of Langerhans' cell function in aged skin.” 2013. Doctoral Dissertation, University of Manchester. Accessed July 23, 2019. https://www.research.manchester.ac.uk/portal/en/theses/an-investigation-of-langerhans-cell-function-in-aged-skin(a5d3967f-51bc-49fb-ab05-31e5aad4ae40).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.570266.

MLA Handbook (7th Edition):

Ogden, Stephanie. “An investigation of Langerhans' cell function in aged skin.” 2013. Web. 23 Jul 2019.

Vancouver:

Ogden S. An investigation of Langerhans' cell function in aged skin. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2019 Jul 23]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/an-investigation-of-langerhans-cell-function-in-aged-skin(a5d3967f-51bc-49fb-ab05-31e5aad4ae40).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.570266.

Council of Science Editors:

Ogden S. An investigation of Langerhans' cell function in aged skin. [Doctoral Dissertation]. University of Manchester; 2013. Available from: https://www.research.manchester.ac.uk/portal/en/theses/an-investigation-of-langerhans-cell-function-in-aged-skin(a5d3967f-51bc-49fb-ab05-31e5aad4ae40).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.570266


University of Manchester

9. Edye, Michelle. Interleukin-1 signalling in disease.

Degree: 2015, University of Manchester

 The pro-inflammatory cytokine interleukin 1 (IL-1) is involved in numerous physiological and pathological processes. It contributes to thermoregulation, sleep, feeding behaviour and notably to the… (more)

Subjects/Keywords: Interleukin-1; Inflammation; Acidosis; Inflammasome; Seizure

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Edye, M. (2015). Interleukin-1 signalling in disease. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:293336

Chicago Manual of Style (16th Edition):

Edye, Michelle. “Interleukin-1 signalling in disease.” 2015. Doctoral Dissertation, University of Manchester. Accessed July 23, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:293336.

MLA Handbook (7th Edition):

Edye, Michelle. “Interleukin-1 signalling in disease.” 2015. Web. 23 Jul 2019.

Vancouver:

Edye M. Interleukin-1 signalling in disease. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2019 Jul 23]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:293336.

Council of Science Editors:

Edye M. Interleukin-1 signalling in disease. [Doctoral Dissertation]. University of Manchester; 2015. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:293336


Kansas State University

10. Liu, Ziyan. Induction of glioma stem cells by interleukin-1beta and transforming growth factor-beta.

Degree: MS, Department of Anatomy and Physiology, 2011, Kansas State University

 Transforming growth factor beta (TGF-β) and interleukin-1β (IL-1β) are both up-regulated in high grade gliomas and their elevated activities have been associated with prognosis in… (more)

Subjects/Keywords: stem; interleukin 1 beta; Biochemistry (0487)

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Liu, Z. (2011). Induction of glioma stem cells by interleukin-1beta and transforming growth factor-beta. (Masters Thesis). Kansas State University. Retrieved from http://hdl.handle.net/2097/8572

Chicago Manual of Style (16th Edition):

Liu, Ziyan. “Induction of glioma stem cells by interleukin-1beta and transforming growth factor-beta.” 2011. Masters Thesis, Kansas State University. Accessed July 23, 2019. http://hdl.handle.net/2097/8572.

MLA Handbook (7th Edition):

Liu, Ziyan. “Induction of glioma stem cells by interleukin-1beta and transforming growth factor-beta.” 2011. Web. 23 Jul 2019.

Vancouver:

Liu Z. Induction of glioma stem cells by interleukin-1beta and transforming growth factor-beta. [Internet] [Masters thesis]. Kansas State University; 2011. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/2097/8572.

Council of Science Editors:

Liu Z. Induction of glioma stem cells by interleukin-1beta and transforming growth factor-beta. [Masters Thesis]. Kansas State University; 2011. Available from: http://hdl.handle.net/2097/8572


University of Rochester

11. Wu, Michael Dacheng; O'Banion, M. Kerry. Inhibition of Adult Hippocampal Neurogenesis by Sustained Interleukin-1B.

Degree: PhD, 2013, University of Rochester

 Neurogenesis persists throughout adulthood in rodents and is speculated to be important in learning and memory, responses to stress, repair/regeneration, and normal maintenance of the… (more)

Subjects/Keywords: Inflammation; Interleukin-1; MyD88; Nestin; Neurogenesis; Running

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wu, Michael Dacheng; O'Banion, M. K. (2013). Inhibition of Adult Hippocampal Neurogenesis by Sustained Interleukin-1B. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/26794

Chicago Manual of Style (16th Edition):

Wu, Michael Dacheng; O'Banion, M Kerry. “Inhibition of Adult Hippocampal Neurogenesis by Sustained Interleukin-1B.” 2013. Doctoral Dissertation, University of Rochester. Accessed July 23, 2019. http://hdl.handle.net/1802/26794.

MLA Handbook (7th Edition):

Wu, Michael Dacheng; O'Banion, M Kerry. “Inhibition of Adult Hippocampal Neurogenesis by Sustained Interleukin-1B.” 2013. Web. 23 Jul 2019.

Vancouver:

Wu, Michael Dacheng; O'Banion MK. Inhibition of Adult Hippocampal Neurogenesis by Sustained Interleukin-1B. [Internet] [Doctoral dissertation]. University of Rochester; 2013. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1802/26794.

Council of Science Editors:

Wu, Michael Dacheng; O'Banion MK. Inhibition of Adult Hippocampal Neurogenesis by Sustained Interleukin-1B. [Doctoral Dissertation]. University of Rochester; 2013. Available from: http://hdl.handle.net/1802/26794


University of Aberdeen

12. Zhang, Chu. Elucidation of the role of IL-33 in type 2 immunity in an ex-vivo model of differentiated human airway epithelial cells.

Degree: PhD, 2019, University of Aberdeen

 Background: Airway epithelial cells play an important role in stimulating innate immune system responses, by producing DAMPs, which lead to asthma exacerbation and airway remodelling… (more)

Subjects/Keywords: Asthma; Interleukin-1; Interleukins; Epithelial cells

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, C. (2019). Elucidation of the role of IL-33 in type 2 immunity in an ex-vivo model of differentiated human airway epithelial cells. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=241179 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774044

Chicago Manual of Style (16th Edition):

Zhang, Chu. “Elucidation of the role of IL-33 in type 2 immunity in an ex-vivo model of differentiated human airway epithelial cells.” 2019. Doctoral Dissertation, University of Aberdeen. Accessed July 23, 2019. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=241179 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774044.

MLA Handbook (7th Edition):

Zhang, Chu. “Elucidation of the role of IL-33 in type 2 immunity in an ex-vivo model of differentiated human airway epithelial cells.” 2019. Web. 23 Jul 2019.

Vancouver:

Zhang C. Elucidation of the role of IL-33 in type 2 immunity in an ex-vivo model of differentiated human airway epithelial cells. [Internet] [Doctoral dissertation]. University of Aberdeen; 2019. [cited 2019 Jul 23]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=241179 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774044.

Council of Science Editors:

Zhang C. Elucidation of the role of IL-33 in type 2 immunity in an ex-vivo model of differentiated human airway epithelial cells. [Doctoral Dissertation]. University of Aberdeen; 2019. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=241179 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774044

13. Kousathana, Foteini. Μεταβολές της μη ειδικής ανοσιακής απάντησης κατά τη φυσική πορεία του σακχαρώδους διαβήτη τύπου 2: μια προοπτική μελέτη.

Degree: 2017, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

The underlying immune defect of susceptibility in diabetes mellitus type 2 to infections remains unknown. The qualitative changes in cytokine biosynthesis by circulating mononuclear cells… (more)

Subjects/Keywords: Ιντερλευκίνη - 1; Σακχαρώδης διαβήτης τύπου ΙΙ; interleukin 1; Diabetes mellitus type II; interleukin 6

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kousathana, F. (2017). Μεταβολές της μη ειδικής ανοσιακής απάντησης κατά τη φυσική πορεία του σακχαρώδους διαβήτη τύπου 2: μια προοπτική μελέτη. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/40499

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kousathana, Foteini. “Μεταβολές της μη ειδικής ανοσιακής απάντησης κατά τη φυσική πορεία του σακχαρώδους διαβήτη τύπου 2: μια προοπτική μελέτη.” 2017. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed July 23, 2019. http://hdl.handle.net/10442/hedi/40499.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kousathana, Foteini. “Μεταβολές της μη ειδικής ανοσιακής απάντησης κατά τη φυσική πορεία του σακχαρώδους διαβήτη τύπου 2: μια προοπτική μελέτη.” 2017. Web. 23 Jul 2019.

Vancouver:

Kousathana F. Μεταβολές της μη ειδικής ανοσιακής απάντησης κατά τη φυσική πορεία του σακχαρώδους διαβήτη τύπου 2: μια προοπτική μελέτη. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2017. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/10442/hedi/40499.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kousathana F. Μεταβολές της μη ειδικής ανοσιακής απάντησης κατά τη φυσική πορεία του σακχαρώδους διαβήτη τύπου 2: μια προοπτική μελέτη. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2017. Available from: http://hdl.handle.net/10442/hedi/40499

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Johannes Gutenberg Universität Mainz

14. Teschner, Daniel. Die Beeinflussung einer Th2-Zell-vermittelten Immunantwort durch Interleukin-1-alpha am Beispiel des murinen allergischen Asthmas.

Degree: 2010, Johannes Gutenberg Universität Mainz

Dass durch IL-1α die Th1-vermittelte Immunreaktion der kutanen Leishmaniasis beeinflusst werden kann, konnte unsere Arbeitsgruppe bereits zeigen. Daran anknüpfend war das Ziel meiner Dissertation zu… (more)

Subjects/Keywords: Interleukin-1, allergisches Asthma, Th1/Th2; Interleukin-1, allergic asthma, Th1/Th2; Medical sciences Medicine

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Teschner, D. (2010). Die Beeinflussung einer Th2-Zell-vermittelten Immunantwort durch Interleukin-1-alpha am Beispiel des murinen allergischen Asthmas. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2010/2213/

Chicago Manual of Style (16th Edition):

Teschner, Daniel. “Die Beeinflussung einer Th2-Zell-vermittelten Immunantwort durch Interleukin-1-alpha am Beispiel des murinen allergischen Asthmas.” 2010. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed July 23, 2019. http://ubm.opus.hbz-nrw.de/volltexte/2010/2213/.

MLA Handbook (7th Edition):

Teschner, Daniel. “Die Beeinflussung einer Th2-Zell-vermittelten Immunantwort durch Interleukin-1-alpha am Beispiel des murinen allergischen Asthmas.” 2010. Web. 23 Jul 2019.

Vancouver:

Teschner D. Die Beeinflussung einer Th2-Zell-vermittelten Immunantwort durch Interleukin-1-alpha am Beispiel des murinen allergischen Asthmas. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2010. [cited 2019 Jul 23]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2010/2213/.

Council of Science Editors:

Teschner D. Die Beeinflussung einer Th2-Zell-vermittelten Immunantwort durch Interleukin-1-alpha am Beispiel des murinen allergischen Asthmas. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2010. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2010/2213/


NSYSU

15. Wang, Shang-ying. Type-1 Interleukin-1 Receptor is Essential for Host Defense Against Pseudomonas aeruginosa-induced Pneumonia.

Degree: Master, Biological Sciences, 2009, NSYSU

 IL-1 is an essential pro-inflammatory factor in inflammation response. The effect of IL-1 is through binding to the IL-1 receptor that triggers the following signal… (more)

Subjects/Keywords: pseudomonas aeruginosa; pneumonia; Type-1 Interleukin-1 receptor

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, S. (2009). Type-1 Interleukin-1 Receptor is Essential for Host Defense Against Pseudomonas aeruginosa-induced Pneumonia. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0826109-152046

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Shang-ying. “Type-1 Interleukin-1 Receptor is Essential for Host Defense Against Pseudomonas aeruginosa-induced Pneumonia.” 2009. Thesis, NSYSU. Accessed July 23, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0826109-152046.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Shang-ying. “Type-1 Interleukin-1 Receptor is Essential for Host Defense Against Pseudomonas aeruginosa-induced Pneumonia.” 2009. Web. 23 Jul 2019.

Vancouver:

Wang S. Type-1 Interleukin-1 Receptor is Essential for Host Defense Against Pseudomonas aeruginosa-induced Pneumonia. [Internet] [Thesis]. NSYSU; 2009. [cited 2019 Jul 23]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0826109-152046.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang S. Type-1 Interleukin-1 Receptor is Essential for Host Defense Against Pseudomonas aeruginosa-induced Pneumonia. [Thesis]. NSYSU; 2009. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0826109-152046

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

16. Quiniou, Christiane. Développement de modulateurs allostériques peptidiques inhibiteurs de l’activité des récepteurs de l’interleukine 1 et de la vasopressine .

Degree: 2011, Université de Montréal

 L’approche Module X a été créée dans le but de concevoir de petits peptides modulateurs ayant des propriétés allostériques. Module X reproduit de petites parties… (more)

Subjects/Keywords: Allostérisme; Peptides; Allosteric; Inflammation; Interleukin-1; Récepteur; Allosterism; Receptor; Interleukine-1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Quiniou, C. (2011). Développement de modulateurs allostériques peptidiques inhibiteurs de l’activité des récepteurs de l’interleukine 1 et de la vasopressine . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/6097

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Quiniou, Christiane. “Développement de modulateurs allostériques peptidiques inhibiteurs de l’activité des récepteurs de l’interleukine 1 et de la vasopressine .” 2011. Thesis, Université de Montréal. Accessed July 23, 2019. http://hdl.handle.net/1866/6097.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Quiniou, Christiane. “Développement de modulateurs allostériques peptidiques inhibiteurs de l’activité des récepteurs de l’interleukine 1 et de la vasopressine .” 2011. Web. 23 Jul 2019.

Vancouver:

Quiniou C. Développement de modulateurs allostériques peptidiques inhibiteurs de l’activité des récepteurs de l’interleukine 1 et de la vasopressine . [Internet] [Thesis]. Université de Montréal; 2011. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1866/6097.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Quiniou C. Développement de modulateurs allostériques peptidiques inhibiteurs de l’activité des récepteurs de l’interleukine 1 et de la vasopressine . [Thesis]. Université de Montréal; 2011. Available from: http://hdl.handle.net/1866/6097

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

17. Raices, Raquel Marie. A Novel Role For Il-1 Cytokines And Tnfa In Ifn¿ Production, Which Is Mediated By I¿b¿.

Degree: PhD, Integrated Biomedical Science, 2008, The Ohio State University

  The human immune response to pathogen infection involves a complex network of immune cells, sensor molecules and signaling molecules that fight the invading organism… (more)

Subjects/Keywords: Immunology; Interleukin-1&946; Interleukin-18; Tumor Necrosis Factor &945; Interferon &947

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Raices, R. M. (2008). A Novel Role For Il-1 Cytokines And Tnfa In Ifn¿ Production, Which Is Mediated By I¿b¿. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1211994580

Chicago Manual of Style (16th Edition):

Raices, Raquel Marie. “A Novel Role For Il-1 Cytokines And Tnfa In Ifn¿ Production, Which Is Mediated By I¿b¿.” 2008. Doctoral Dissertation, The Ohio State University. Accessed July 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1211994580.

MLA Handbook (7th Edition):

Raices, Raquel Marie. “A Novel Role For Il-1 Cytokines And Tnfa In Ifn¿ Production, Which Is Mediated By I¿b¿.” 2008. Web. 23 Jul 2019.

Vancouver:

Raices RM. A Novel Role For Il-1 Cytokines And Tnfa In Ifn¿ Production, Which Is Mediated By I¿b¿. [Internet] [Doctoral dissertation]. The Ohio State University; 2008. [cited 2019 Jul 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1211994580.

Council of Science Editors:

Raices RM. A Novel Role For Il-1 Cytokines And Tnfa In Ifn¿ Production, Which Is Mediated By I¿b¿. [Doctoral Dissertation]. The Ohio State University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1211994580

18. 김, 순선. Association of the Interleukin-1 beta and Interleukin-1 Receptor Accessory Protein Gene Polymorphisms with Persistent Hepatitis B Virus Infection and Hepatocellular Carcinoma.

Degree: 2010, Ajou University

"Background & Aims: The reasons for persistent hepatitis B virus infection (HBV) are unknown, but they are probably related to host immune factors. Interleukin-1β (IL-1β)… (more)

Subjects/Keywords: Chronic hepatitis B; Hepatocellular carcinoma; Interleukin-1β; Interleukin-1 receptor accessory protein; Single nucleotide polymorphism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

김, . (2010). Association of the Interleukin-1 beta and Interleukin-1 Receptor Accessory Protein Gene Polymorphisms with Persistent Hepatitis B Virus Infection and Hepatocellular Carcinoma. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/1441 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010675

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

김, 순선. “Association of the Interleukin-1 beta and Interleukin-1 Receptor Accessory Protein Gene Polymorphisms with Persistent Hepatitis B Virus Infection and Hepatocellular Carcinoma.” 2010. Thesis, Ajou University. Accessed July 23, 2019. http://repository.ajou.ac.kr/handle/201003/1441 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010675.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

김, 순선. “Association of the Interleukin-1 beta and Interleukin-1 Receptor Accessory Protein Gene Polymorphisms with Persistent Hepatitis B Virus Infection and Hepatocellular Carcinoma.” 2010. Web. 23 Jul 2019.

Vancouver:

김 . Association of the Interleukin-1 beta and Interleukin-1 Receptor Accessory Protein Gene Polymorphisms with Persistent Hepatitis B Virus Infection and Hepatocellular Carcinoma. [Internet] [Thesis]. Ajou University; 2010. [cited 2019 Jul 23]. Available from: http://repository.ajou.ac.kr/handle/201003/1441 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010675.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

김 . Association of the Interleukin-1 beta and Interleukin-1 Receptor Accessory Protein Gene Polymorphisms with Persistent Hepatitis B Virus Infection and Hepatocellular Carcinoma. [Thesis]. Ajou University; 2010. Available from: http://repository.ajou.ac.kr/handle/201003/1441 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010675

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Tursich, Mischa. Relationships between Psychological Distress and Immune Function in Women with a History of Childhood Maltreatment.

Degree: 2012, Nova Southeastern University

 Exposure to traumatic events can lead to many varied psychological and physiological difficulties, including an increased risk for chronic physical health problems and chronic pain… (more)

Subjects/Keywords: Childhood Abuse; Depression; Dissociation; Interleukin-1; Interleukin-6; Posttraumatic Stress Disorder; Psychology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tursich, M. (2012). Relationships between Psychological Distress and Immune Function in Women with a History of Childhood Maltreatment. (Thesis). Nova Southeastern University. Retrieved from https://nsuworks.nova.edu/cps_stuetd/72

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tursich, Mischa. “Relationships between Psychological Distress and Immune Function in Women with a History of Childhood Maltreatment.” 2012. Thesis, Nova Southeastern University. Accessed July 23, 2019. https://nsuworks.nova.edu/cps_stuetd/72.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tursich, Mischa. “Relationships between Psychological Distress and Immune Function in Women with a History of Childhood Maltreatment.” 2012. Web. 23 Jul 2019.

Vancouver:

Tursich M. Relationships between Psychological Distress and Immune Function in Women with a History of Childhood Maltreatment. [Internet] [Thesis]. Nova Southeastern University; 2012. [cited 2019 Jul 23]. Available from: https://nsuworks.nova.edu/cps_stuetd/72.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tursich M. Relationships between Psychological Distress and Immune Function in Women with a History of Childhood Maltreatment. [Thesis]. Nova Southeastern University; 2012. Available from: https://nsuworks.nova.edu/cps_stuetd/72

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

20. Ward, Christie. Pulmonary expression of inflammatory cytokines in experimental Bovine pneumonic mannheimiosis.

Degree: PhD, Veterinary Medicine, 2009, University of Minnesota

 Bovine pneumonic mannheimiosis (BPM), an acute fibrinonecrotic pleuropneumonia caused by Mannheimia (Pasteurella) haemolytica, remains a leading source of economic losses to North American beef and… (more)

Subjects/Keywords: Cattle; Interleukin-1 beta; Interleukin-8; Mannheimia Haemolytica; Pneumonia; Tumor Necrosis Factor-alpha; Veterinary Medicine

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ward, C. (2009). Pulmonary expression of inflammatory cytokines in experimental Bovine pneumonic mannheimiosis. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/53837

Chicago Manual of Style (16th Edition):

Ward, Christie. “Pulmonary expression of inflammatory cytokines in experimental Bovine pneumonic mannheimiosis.” 2009. Doctoral Dissertation, University of Minnesota. Accessed July 23, 2019. http://purl.umn.edu/53837.

MLA Handbook (7th Edition):

Ward, Christie. “Pulmonary expression of inflammatory cytokines in experimental Bovine pneumonic mannheimiosis.” 2009. Web. 23 Jul 2019.

Vancouver:

Ward C. Pulmonary expression of inflammatory cytokines in experimental Bovine pneumonic mannheimiosis. [Internet] [Doctoral dissertation]. University of Minnesota; 2009. [cited 2019 Jul 23]. Available from: http://purl.umn.edu/53837.

Council of Science Editors:

Ward C. Pulmonary expression of inflammatory cytokines in experimental Bovine pneumonic mannheimiosis. [Doctoral Dissertation]. University of Minnesota; 2009. Available from: http://purl.umn.edu/53837


University of Oulu

21. Piira, O.-P. (Olli-Pekka). Effects of emotional excitement on cardiovascular regulation.

Degree: 2015, University of Oulu

Abstract The incidence of adverse cardiovascular events is higher among spectators of exciting sports events, particularly in patients with coronary artery disease (CAD), but the… (more)

Subjects/Keywords: autonomic regulation; coronary artery disease; emotional excitement; endothelin-1; exercise; interleukin-6; autonominen hermosto; endotelin-1; interleukin-6; jännitys; liikunta; sepelvaltimotauti

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Piira, O. -. (. (2015). Effects of emotional excitement on cardiovascular regulation. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789526209708

Chicago Manual of Style (16th Edition):

Piira, O -P (Olli-Pekka). “Effects of emotional excitement on cardiovascular regulation.” 2015. Doctoral Dissertation, University of Oulu. Accessed July 23, 2019. http://urn.fi/urn:isbn:9789526209708.

MLA Handbook (7th Edition):

Piira, O -P (Olli-Pekka). “Effects of emotional excitement on cardiovascular regulation.” 2015. Web. 23 Jul 2019.

Vancouver:

Piira O-(. Effects of emotional excitement on cardiovascular regulation. [Internet] [Doctoral dissertation]. University of Oulu; 2015. [cited 2019 Jul 23]. Available from: http://urn.fi/urn:isbn:9789526209708.

Council of Science Editors:

Piira O-(. Effects of emotional excitement on cardiovascular regulation. [Doctoral Dissertation]. University of Oulu; 2015. Available from: http://urn.fi/urn:isbn:9789526209708


Johannes Gutenberg Universität Mainz

22. Correll, Anita Luise Ellen. Untersuchungen zum Einfluss des malignen Melanoms auf die Frequenz und Funktion regulatorischer T-Zellen.

Degree: 2012, Johannes Gutenberg Universität Mainz

Im Immunsystem spielen regulatorische T-Zellen (Treg) eine essentielle Rolle bei der Unterdrückung und Eindämmung von überschüssigen oder schädlichen Immunreaktionen. Ihre suppressiven Eigenschaften wirken aber auch… (more)

Subjects/Keywords: Regulatorische T-Zelle; Melanom; Dendritische Zelle; Immuntherapie; Interleukin-1; regulatory T cell; melanoma; dendritic cell; immunotherapy; interleukin-1; Life sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Correll, A. L. E. (2012). Untersuchungen zum Einfluss des malignen Melanoms auf die Frequenz und Funktion regulatorischer T-Zellen. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2012/3111/

Chicago Manual of Style (16th Edition):

Correll, Anita Luise Ellen. “Untersuchungen zum Einfluss des malignen Melanoms auf die Frequenz und Funktion regulatorischer T-Zellen.” 2012. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed July 23, 2019. http://ubm.opus.hbz-nrw.de/volltexte/2012/3111/.

MLA Handbook (7th Edition):

Correll, Anita Luise Ellen. “Untersuchungen zum Einfluss des malignen Melanoms auf die Frequenz und Funktion regulatorischer T-Zellen.” 2012. Web. 23 Jul 2019.

Vancouver:

Correll ALE. Untersuchungen zum Einfluss des malignen Melanoms auf die Frequenz und Funktion regulatorischer T-Zellen. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2012. [cited 2019 Jul 23]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2012/3111/.

Council of Science Editors:

Correll ALE. Untersuchungen zum Einfluss des malignen Melanoms auf die Frequenz und Funktion regulatorischer T-Zellen. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2012. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2012/3111/

23. Lasoudris, Fanette. Immunosuppression associée à l’enzyme interleukine-4 induced gene 1 (IL4I1) : régulation de l’expression dans les cellules humaines et rôle dans l’échappement tumoral à la réponse immune dans un modèle murin : Immunosuppression induced by Interleukin-4 Induced gene 1 (IL4I1) : Regulation of expression in human cells and role in tumor escape from the immune response in a murine model.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2011, Université Paris-Est

La protéine IL4I1 est une enzyme sécrétée dont l’activité L-amino acide oxydase vis-à-visde la phénylalanine inhibe la prolifération des lymphocytes T in vitro (Boulland et… (more)

Subjects/Keywords: Interleukin-4 induced gene 1; Immunomodulation; Echappement tumoral; Enzyme immunosuppressive; Interleukin-4 induced gene 1; Immunomodulation; Tumor escape; Immunosuppressive Enzyme

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lasoudris, F. (2011). Immunosuppression associée à l’enzyme interleukine-4 induced gene 1 (IL4I1) : régulation de l’expression dans les cellules humaines et rôle dans l’échappement tumoral à la réponse immune dans un modèle murin : Immunosuppression induced by Interleukin-4 Induced gene 1 (IL4I1) : Regulation of expression in human cells and role in tumor escape from the immune response in a murine model. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2011PEST0089

Chicago Manual of Style (16th Edition):

Lasoudris, Fanette. “Immunosuppression associée à l’enzyme interleukine-4 induced gene 1 (IL4I1) : régulation de l’expression dans les cellules humaines et rôle dans l’échappement tumoral à la réponse immune dans un modèle murin : Immunosuppression induced by Interleukin-4 Induced gene 1 (IL4I1) : Regulation of expression in human cells and role in tumor escape from the immune response in a murine model.” 2011. Doctoral Dissertation, Université Paris-Est. Accessed July 23, 2019. http://www.theses.fr/2011PEST0089.

MLA Handbook (7th Edition):

Lasoudris, Fanette. “Immunosuppression associée à l’enzyme interleukine-4 induced gene 1 (IL4I1) : régulation de l’expression dans les cellules humaines et rôle dans l’échappement tumoral à la réponse immune dans un modèle murin : Immunosuppression induced by Interleukin-4 Induced gene 1 (IL4I1) : Regulation of expression in human cells and role in tumor escape from the immune response in a murine model.” 2011. Web. 23 Jul 2019.

Vancouver:

Lasoudris F. Immunosuppression associée à l’enzyme interleukine-4 induced gene 1 (IL4I1) : régulation de l’expression dans les cellules humaines et rôle dans l’échappement tumoral à la réponse immune dans un modèle murin : Immunosuppression induced by Interleukin-4 Induced gene 1 (IL4I1) : Regulation of expression in human cells and role in tumor escape from the immune response in a murine model. [Internet] [Doctoral dissertation]. Université Paris-Est; 2011. [cited 2019 Jul 23]. Available from: http://www.theses.fr/2011PEST0089.

Council of Science Editors:

Lasoudris F. Immunosuppression associée à l’enzyme interleukine-4 induced gene 1 (IL4I1) : régulation de l’expression dans les cellules humaines et rôle dans l’échappement tumoral à la réponse immune dans un modèle murin : Immunosuppression induced by Interleukin-4 Induced gene 1 (IL4I1) : Regulation of expression in human cells and role in tumor escape from the immune response in a murine model. [Doctoral Dissertation]. Université Paris-Est; 2011. Available from: http://www.theses.fr/2011PEST0089

24. Imbert, Paul. Multi-targeting of the innate immune system by Toll/interleukin-1 receptor domain-containing bacterial effectors and the consequences in bacterial immune-evasion : Ciblage multiple du système immunitaire inné par les effecteurs bactériens contenant un domaine Toll/interleukin-1 receptor (TIR) et les conséquences dans l’évasion immunitaire bactérienne.

Degree: Docteur es, Biologie, 2016, Lyon

Le domaine TIR (Toll/interleukin (IL)-1 receptor) est une composante essentielle du système immunitaire inné, celui-ci est présent dans les récepteurs TLR (Toll-like receptor) et les… (more)

Subjects/Keywords: Domaine Toll/interleukin-1 receptor; Évasion immunitaire bactérienne; Effecteur bactérien; Toll/interleukin-1 receptor domain; Bacterial immune-evasion; Bacterial effector; 616.9

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Imbert, P. (2016). Multi-targeting of the innate immune system by Toll/interleukin-1 receptor domain-containing bacterial effectors and the consequences in bacterial immune-evasion : Ciblage multiple du système immunitaire inné par les effecteurs bactériens contenant un domaine Toll/interleukin-1 receptor (TIR) et les conséquences dans l’évasion immunitaire bactérienne. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2016LYSE1226

Chicago Manual of Style (16th Edition):

Imbert, Paul. “Multi-targeting of the innate immune system by Toll/interleukin-1 receptor domain-containing bacterial effectors and the consequences in bacterial immune-evasion : Ciblage multiple du système immunitaire inné par les effecteurs bactériens contenant un domaine Toll/interleukin-1 receptor (TIR) et les conséquences dans l’évasion immunitaire bactérienne.” 2016. Doctoral Dissertation, Lyon. Accessed July 23, 2019. http://www.theses.fr/2016LYSE1226.

MLA Handbook (7th Edition):

Imbert, Paul. “Multi-targeting of the innate immune system by Toll/interleukin-1 receptor domain-containing bacterial effectors and the consequences in bacterial immune-evasion : Ciblage multiple du système immunitaire inné par les effecteurs bactériens contenant un domaine Toll/interleukin-1 receptor (TIR) et les conséquences dans l’évasion immunitaire bactérienne.” 2016. Web. 23 Jul 2019.

Vancouver:

Imbert P. Multi-targeting of the innate immune system by Toll/interleukin-1 receptor domain-containing bacterial effectors and the consequences in bacterial immune-evasion : Ciblage multiple du système immunitaire inné par les effecteurs bactériens contenant un domaine Toll/interleukin-1 receptor (TIR) et les conséquences dans l’évasion immunitaire bactérienne. [Internet] [Doctoral dissertation]. Lyon; 2016. [cited 2019 Jul 23]. Available from: http://www.theses.fr/2016LYSE1226.

Council of Science Editors:

Imbert P. Multi-targeting of the innate immune system by Toll/interleukin-1 receptor domain-containing bacterial effectors and the consequences in bacterial immune-evasion : Ciblage multiple du système immunitaire inné par les effecteurs bactériens contenant un domaine Toll/interleukin-1 receptor (TIR) et les conséquences dans l’évasion immunitaire bactérienne. [Doctoral Dissertation]. Lyon; 2016. Available from: http://www.theses.fr/2016LYSE1226

25. 王, 正道. Concurrent Generation of Nitric Oxide and Superoxide Inhibits Proteoglycan Synthesis in Bovine Articular Chondrocytes: Involvement of Peroxynitrite : 軟骨細胞におけるIL-1によるPeroxynitriteの誘導とその作用.

Degree: 1999, Kindai University / 近畿大学

[ABSTRACT] Objective. Nitric oxide (NO), widely assumed to be a mediator of interleukin 1 (IL-1), inhibits proteoglycan synthesis in articular chondrocytes. IL-1 also produces superoxide… (more)

Subjects/Keywords: CHONDROCYTES; INTERLEUKIN-1; RHEUMATOID ARTHRITIS

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

王, . . (1999). Concurrent Generation of Nitric Oxide and Superoxide Inhibits Proteoglycan Synthesis in Bovine Articular Chondrocytes: Involvement of Peroxynitrite : 軟骨細胞におけるIL-1によるPeroxynitriteの誘導とその作用. (Thesis). Kindai University / 近畿大学. Retrieved from http://id.nii.ac.jp/1391/00002103/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

王, 正道. “Concurrent Generation of Nitric Oxide and Superoxide Inhibits Proteoglycan Synthesis in Bovine Articular Chondrocytes: Involvement of Peroxynitrite : 軟骨細胞におけるIL-1によるPeroxynitriteの誘導とその作用.” 1999. Thesis, Kindai University / 近畿大学. Accessed July 23, 2019. http://id.nii.ac.jp/1391/00002103/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

王, 正道. “Concurrent Generation of Nitric Oxide and Superoxide Inhibits Proteoglycan Synthesis in Bovine Articular Chondrocytes: Involvement of Peroxynitrite : 軟骨細胞におけるIL-1によるPeroxynitriteの誘導とその作用.” 1999. Web. 23 Jul 2019.

Vancouver:

王 . Concurrent Generation of Nitric Oxide and Superoxide Inhibits Proteoglycan Synthesis in Bovine Articular Chondrocytes: Involvement of Peroxynitrite : 軟骨細胞におけるIL-1によるPeroxynitriteの誘導とその作用. [Internet] [Thesis]. Kindai University / 近畿大学; 1999. [cited 2019 Jul 23]. Available from: http://id.nii.ac.jp/1391/00002103/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

王 . Concurrent Generation of Nitric Oxide and Superoxide Inhibits Proteoglycan Synthesis in Bovine Articular Chondrocytes: Involvement of Peroxynitrite : 軟骨細胞におけるIL-1によるPeroxynitriteの誘導とその作用. [Thesis]. Kindai University / 近畿大学; 1999. Available from: http://id.nii.ac.jp/1391/00002103/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

26. Hooper, Kirsten Mary. PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS.

Degree: PhD, 2017, Temple University

Microbiology and Immunology

Interleukin-27 (p28/EBI3) is an immunomodulatory cytokine expressed by activated antigen presenting cells. Although first discovered to be involved in Th1 cell differentiation,… (more)

Subjects/Keywords: Immunology;

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hooper, K. M. (2017). PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,432693

Chicago Manual of Style (16th Edition):

Hooper, Kirsten Mary. “PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS.” 2017. Doctoral Dissertation, Temple University. Accessed July 23, 2019. http://digital.library.temple.edu/u?/p245801coll10,432693.

MLA Handbook (7th Edition):

Hooper, Kirsten Mary. “PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS.” 2017. Web. 23 Jul 2019.

Vancouver:

Hooper KM. PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2019 Jul 23]. Available from: http://digital.library.temple.edu/u?/p245801coll10,432693.

Council of Science Editors:

Hooper KM. PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,432693


Texas A&M University

27. Akoolo, Lavoisier. Role of Brucella Toll/Interleukin-1 Receptor (TIR) Domain Containing Protein (?TcpB) Deletion Mutant in Protective Immunity against Brucellosis.

Degree: 2015, Texas A&M University

 Brucellosis is an important zoonotic disease affecting about 500,000 people annually. The development of safer and more efficacious Brucella Live attenuated vaccines addresses safety concerns… (more)

Subjects/Keywords: Brucella; Toll interleukin-1 receptor domain containing protein; Immunity; vaccine

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Akoolo, L. (2015). Role of Brucella Toll/Interleukin-1 Receptor (TIR) Domain Containing Protein (?TcpB) Deletion Mutant in Protective Immunity against Brucellosis. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155602

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Akoolo, Lavoisier. “Role of Brucella Toll/Interleukin-1 Receptor (TIR) Domain Containing Protein (?TcpB) Deletion Mutant in Protective Immunity against Brucellosis.” 2015. Thesis, Texas A&M University. Accessed July 23, 2019. http://hdl.handle.net/1969.1/155602.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Akoolo, Lavoisier. “Role of Brucella Toll/Interleukin-1 Receptor (TIR) Domain Containing Protein (?TcpB) Deletion Mutant in Protective Immunity against Brucellosis.” 2015. Web. 23 Jul 2019.

Vancouver:

Akoolo L. Role of Brucella Toll/Interleukin-1 Receptor (TIR) Domain Containing Protein (?TcpB) Deletion Mutant in Protective Immunity against Brucellosis. [Internet] [Thesis]. Texas A&M University; 2015. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1969.1/155602.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Akoolo L. Role of Brucella Toll/Interleukin-1 Receptor (TIR) Domain Containing Protein (?TcpB) Deletion Mutant in Protective Immunity against Brucellosis. [Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155602

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Colorado

28. Walters, Ryan David. Mechanisms of Transcriptional Regulation by NFAT and AP-1 Transcriptional Activators.

Degree: PhD, Chemistry & Biochemistry, 2013, University of Colorado

  Transcription of mRNA by RNA Polymerase II (Pol II) is a fundamental step in gene expression. In mammalians, this is a highly regulated process… (more)

Subjects/Keywords: AP-1; cJun; Interleukin-2; NFAT; SELEX; Transcription; Biochemistry; Molecular Genetics

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Walters, R. D. (2013). Mechanisms of Transcriptional Regulation by NFAT and AP-1 Transcriptional Activators. (Doctoral Dissertation). University of Colorado. Retrieved from http://scholar.colorado.edu/chem_gradetds/101

Chicago Manual of Style (16th Edition):

Walters, Ryan David. “Mechanisms of Transcriptional Regulation by NFAT and AP-1 Transcriptional Activators.” 2013. Doctoral Dissertation, University of Colorado. Accessed July 23, 2019. http://scholar.colorado.edu/chem_gradetds/101.

MLA Handbook (7th Edition):

Walters, Ryan David. “Mechanisms of Transcriptional Regulation by NFAT and AP-1 Transcriptional Activators.” 2013. Web. 23 Jul 2019.

Vancouver:

Walters RD. Mechanisms of Transcriptional Regulation by NFAT and AP-1 Transcriptional Activators. [Internet] [Doctoral dissertation]. University of Colorado; 2013. [cited 2019 Jul 23]. Available from: http://scholar.colorado.edu/chem_gradetds/101.

Council of Science Editors:

Walters RD. Mechanisms of Transcriptional Regulation by NFAT and AP-1 Transcriptional Activators. [Doctoral Dissertation]. University of Colorado; 2013. Available from: http://scholar.colorado.edu/chem_gradetds/101


NSYSU

29. SU, HUEI-YU. Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

 Early cancers of the pancreas often cause no symptoms leading to pancreatic cancer often can't be detected until the cancer already reach to advanced stage.… (more)

Subjects/Keywords: EMT; TGF-β1/Smad4; Nestin; Interleukin-6; PDAC

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

SU, H. (2014). Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-021652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

SU, HUEI-YU. “Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells.” 2014. Thesis, NSYSU. Accessed July 23, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-021652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

SU, HUEI-YU. “Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells.” 2014. Web. 23 Jul 2019.

Vancouver:

SU H. Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells. [Internet] [Thesis]. NSYSU; 2014. [cited 2019 Jul 23]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-021652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

SU H. Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-021652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Karmarkar, Dipti. Modulators of the Acute Inflammatory Response: A Dissertation.

Degree: Immunology and Microbiology, Pathology, 2013, U of Massachusetts : Med

  Acute inflammatory response is caused by the rapid recruitment of leukocytes, mainly neutrophils and monocytes, from blood to the tissue site. Diverse agents, including… (more)

Subjects/Keywords: Inflammation; Leukocytes; Interleukin-1; Immunity; Immunology and Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Karmarkar, D. (2013). Modulators of the Acute Inflammatory Response: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/656

Chicago Manual of Style (16th Edition):

Karmarkar, Dipti. “Modulators of the Acute Inflammatory Response: A Dissertation.” 2013. Doctoral Dissertation, U of Massachusetts : Med. Accessed July 23, 2019. http://escholarship.umassmed.edu/gsbs_diss/656.

MLA Handbook (7th Edition):

Karmarkar, Dipti. “Modulators of the Acute Inflammatory Response: A Dissertation.” 2013. Web. 23 Jul 2019.

Vancouver:

Karmarkar D. Modulators of the Acute Inflammatory Response: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2013. [cited 2019 Jul 23]. Available from: http://escholarship.umassmed.edu/gsbs_diss/656.

Council of Science Editors:

Karmarkar D. Modulators of the Acute Inflammatory Response: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2013. Available from: http://escholarship.umassmed.edu/gsbs_diss/656

[1] [2] [3] [4] [5] [6] [7] [8]

.