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You searched for subject:(Interferon Type I). Showing records 1 – 30 of 93 total matches.

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University of Texas Southwestern Medical Center

1. Perelman, Sofya. Identification and Characterization of Interferon-Stimulated Regulators of Bacterial Infection.

Degree: 2017, University of Texas Southwestern Medical Center

 The type I interferon activated transcriptional response is a critical antiviral defense mechanism, yet its role in bacterial pathogenesis remains less well characterized. Using an… (more)

Subjects/Keywords: Interferon Type I; Listeriosis; Virulence

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APA (6th Edition):

Perelman, S. (2017). Identification and Characterization of Interferon-Stimulated Regulators of Bacterial Infection. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7187

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Perelman, Sofya. “Identification and Characterization of Interferon-Stimulated Regulators of Bacterial Infection.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed September 30, 2020. http://hdl.handle.net/2152.5/7187.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Perelman, Sofya. “Identification and Characterization of Interferon-Stimulated Regulators of Bacterial Infection.” 2017. Web. 30 Sep 2020.

Vancouver:

Perelman S. Identification and Characterization of Interferon-Stimulated Regulators of Bacterial Infection. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Sep 30]. Available from: http://hdl.handle.net/2152.5/7187.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Perelman S. Identification and Characterization of Interferon-Stimulated Regulators of Bacterial Infection. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7187

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

2. Chang, Jenn-tzong. Genome and Infection Characteristics of Human Parechovirus Type 1.

Degree: PhD, Biological Sciences, 2015, NSYSU

 Human parechoviruses (HPeVs), members of the family Picornaviridae, are associated with severe human clinical conditions such as gastrointestinal disease, encephalitis, meningitis, respiratory disease and neonatal… (more)

Subjects/Keywords: type I interferon; parechovirus; HPeV; innate immunity

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APA (6th Edition):

Chang, J. (2015). Genome and Infection Characteristics of Human Parechovirus Type 1. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521115-005317

Chicago Manual of Style (16th Edition):

Chang, Jenn-tzong. “Genome and Infection Characteristics of Human Parechovirus Type 1.” 2015. Doctoral Dissertation, NSYSU. Accessed September 30, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521115-005317.

MLA Handbook (7th Edition):

Chang, Jenn-tzong. “Genome and Infection Characteristics of Human Parechovirus Type 1.” 2015. Web. 30 Sep 2020.

Vancouver:

Chang J. Genome and Infection Characteristics of Human Parechovirus Type 1. [Internet] [Doctoral dissertation]. NSYSU; 2015. [cited 2020 Sep 30]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521115-005317.

Council of Science Editors:

Chang J. Genome and Infection Characteristics of Human Parechovirus Type 1. [Doctoral Dissertation]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521115-005317

3. Smith, Nikaïa. Étude moléculaire du TNF-Related Apoptosis Induced Ligand (TRAIL) et de l’activation du Toll-Like Receptor 7 (TLR7) dans les cellules dendritiques plasmacytoïdes lors de la réponse antivirale : Molecular study of the TNF-Related Apoptosis Induced Ligand (TRAIL) and of Toll-Like Receptor 7 (TLR7) activation in plasmacytoid dendritic cells during viral infections.

Degree: Docteur es, Immunologie, 2015, Sorbonne Paris Cité

Les pDC représentent la première ligne de défense de l’organisme contre les pathogènes et établissent le lien essentiel entre l’immunité innée et adaptative. Les pDC… (more)

Subjects/Keywords: Cellules dendritiques plasmacytoïdes; Interferon de type I; Amines; CXCR4; SiRNA; Plasmacytoid dendritic cells; Type I interferon; Amines; CXCR4; SiRNA; 571.96

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APA (6th Edition):

Smith, N. (2015). Étude moléculaire du TNF-Related Apoptosis Induced Ligand (TRAIL) et de l’activation du Toll-Like Receptor 7 (TLR7) dans les cellules dendritiques plasmacytoïdes lors de la réponse antivirale : Molecular study of the TNF-Related Apoptosis Induced Ligand (TRAIL) and of Toll-Like Receptor 7 (TLR7) activation in plasmacytoid dendritic cells during viral infections. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2015USPCB145

Chicago Manual of Style (16th Edition):

Smith, Nikaïa. “Étude moléculaire du TNF-Related Apoptosis Induced Ligand (TRAIL) et de l’activation du Toll-Like Receptor 7 (TLR7) dans les cellules dendritiques plasmacytoïdes lors de la réponse antivirale : Molecular study of the TNF-Related Apoptosis Induced Ligand (TRAIL) and of Toll-Like Receptor 7 (TLR7) activation in plasmacytoid dendritic cells during viral infections.” 2015. Doctoral Dissertation, Sorbonne Paris Cité. Accessed September 30, 2020. http://www.theses.fr/2015USPCB145.

MLA Handbook (7th Edition):

Smith, Nikaïa. “Étude moléculaire du TNF-Related Apoptosis Induced Ligand (TRAIL) et de l’activation du Toll-Like Receptor 7 (TLR7) dans les cellules dendritiques plasmacytoïdes lors de la réponse antivirale : Molecular study of the TNF-Related Apoptosis Induced Ligand (TRAIL) and of Toll-Like Receptor 7 (TLR7) activation in plasmacytoid dendritic cells during viral infections.” 2015. Web. 30 Sep 2020.

Vancouver:

Smith N. Étude moléculaire du TNF-Related Apoptosis Induced Ligand (TRAIL) et de l’activation du Toll-Like Receptor 7 (TLR7) dans les cellules dendritiques plasmacytoïdes lors de la réponse antivirale : Molecular study of the TNF-Related Apoptosis Induced Ligand (TRAIL) and of Toll-Like Receptor 7 (TLR7) activation in plasmacytoid dendritic cells during viral infections. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2015. [cited 2020 Sep 30]. Available from: http://www.theses.fr/2015USPCB145.

Council of Science Editors:

Smith N. Étude moléculaire du TNF-Related Apoptosis Induced Ligand (TRAIL) et de l’activation du Toll-Like Receptor 7 (TLR7) dans les cellules dendritiques plasmacytoïdes lors de la réponse antivirale : Molecular study of the TNF-Related Apoptosis Induced Ligand (TRAIL) and of Toll-Like Receptor 7 (TLR7) activation in plasmacytoid dendritic cells during viral infections. [Doctoral Dissertation]. Sorbonne Paris Cité; 2015. Available from: http://www.theses.fr/2015USPCB145


Case Western Reserve University

4. Liu, Yi. Negative Regulation of Type I Interferon Induction in Dendritic Cells.

Degree: PhD, Pathology, 2011, Case Western Reserve University

Type I interferon (IFN) regulates innate and adaptive immunity. It protects host cells against viral infection. Many microorganisms have evolved successful mechanisms to inhibit host… (more)

Subjects/Keywords: Immunology; type I interferon; dendritic cells; pattern recognition receptors

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APA (6th Edition):

Liu, Y. (2011). Negative Regulation of Type I Interferon Induction in Dendritic Cells. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1310149509

Chicago Manual of Style (16th Edition):

Liu, Yi. “Negative Regulation of Type I Interferon Induction in Dendritic Cells.” 2011. Doctoral Dissertation, Case Western Reserve University. Accessed September 30, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1310149509.

MLA Handbook (7th Edition):

Liu, Yi. “Negative Regulation of Type I Interferon Induction in Dendritic Cells.” 2011. Web. 30 Sep 2020.

Vancouver:

Liu Y. Negative Regulation of Type I Interferon Induction in Dendritic Cells. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2011. [cited 2020 Sep 30]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1310149509.

Council of Science Editors:

Liu Y. Negative Regulation of Type I Interferon Induction in Dendritic Cells. [Doctoral Dissertation]. Case Western Reserve University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1310149509


Penn State University

5. Hsu, Paul. CARDIOLIPIN REMODELING AND MITOPHAGY: INVESTIGATING THE ROLE OF MITOCHONDRIAL QUALITY CONTROL IN DISEASE.

Degree: 2016, Penn State University

 Metabolic disorders and aging-related diseases are characterized by an accumulation of dysfunctional mitochondria and a gradual decline of the aging organism. Mitophagy, the selective degradation… (more)

Subjects/Keywords: Mitophagy; Cardiolipin; Barth Syndrome; Hypertension; Type I interferon

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APA (6th Edition):

Hsu, P. (2016). CARDIOLIPIN REMODELING AND MITOPHAGY: INVESTIGATING THE ROLE OF MITOCHONDRIAL QUALITY CONTROL IN DISEASE. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13165pxh200

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hsu, Paul. “CARDIOLIPIN REMODELING AND MITOPHAGY: INVESTIGATING THE ROLE OF MITOCHONDRIAL QUALITY CONTROL IN DISEASE.” 2016. Thesis, Penn State University. Accessed September 30, 2020. https://submit-etda.libraries.psu.edu/catalog/13165pxh200.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hsu, Paul. “CARDIOLIPIN REMODELING AND MITOPHAGY: INVESTIGATING THE ROLE OF MITOCHONDRIAL QUALITY CONTROL IN DISEASE.” 2016. Web. 30 Sep 2020.

Vancouver:

Hsu P. CARDIOLIPIN REMODELING AND MITOPHAGY: INVESTIGATING THE ROLE OF MITOCHONDRIAL QUALITY CONTROL IN DISEASE. [Internet] [Thesis]. Penn State University; 2016. [cited 2020 Sep 30]. Available from: https://submit-etda.libraries.psu.edu/catalog/13165pxh200.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hsu P. CARDIOLIPIN REMODELING AND MITOPHAGY: INVESTIGATING THE ROLE OF MITOCHONDRIAL QUALITY CONTROL IN DISEASE. [Thesis]. Penn State University; 2016. Available from: https://submit-etda.libraries.psu.edu/catalog/13165pxh200

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

6. Iyer, Shilpa. Determinants Of Hiv-1 Transmission Fitness.

Degree: 2017, University of Pennsylvania

 DETERMINANTS OF HIV-1 TRANSMISSION FITNESS Shilpa S. Iyer Beatrice H. Hahn HIV-1 is predominantly transmitted by mucosal routes and almost 80 percent of new infections… (more)

Subjects/Keywords: Fitness; HIV-1; Mucosal Transmisison; Type I Interferon; Microbiology; Virology

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APA (6th Edition):

Iyer, S. (2017). Determinants Of Hiv-1 Transmission Fitness. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2355

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Iyer, Shilpa. “Determinants Of Hiv-1 Transmission Fitness.” 2017. Thesis, University of Pennsylvania. Accessed September 30, 2020. https://repository.upenn.edu/edissertations/2355.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Iyer, Shilpa. “Determinants Of Hiv-1 Transmission Fitness.” 2017. Web. 30 Sep 2020.

Vancouver:

Iyer S. Determinants Of Hiv-1 Transmission Fitness. [Internet] [Thesis]. University of Pennsylvania; 2017. [cited 2020 Sep 30]. Available from: https://repository.upenn.edu/edissertations/2355.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Iyer S. Determinants Of Hiv-1 Transmission Fitness. [Thesis]. University of Pennsylvania; 2017. Available from: https://repository.upenn.edu/edissertations/2355

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

7. Chang, Yu-Han. Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection.

Degree: 2016, University of Toronto

Co-stimulation of T cells through Glucocorticoid-Induced Tumour Necrosis Factor Receptor-Related Protein (GITR) contributes to viral control during persistent lymphocytic choriomeningitis virus (LCMV) infection. Herein, we… (more)

Subjects/Keywords: GITR; GITRL; LCMV; TNFRSF18; TNFSF18; Type I Interferon; 0982

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APA (6th Edition):

Chang, Y. (2016). Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91899

Chicago Manual of Style (16th Edition):

Chang, Yu-Han. “Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection.” 2016. Masters Thesis, University of Toronto. Accessed September 30, 2020. http://hdl.handle.net/1807/91899.

MLA Handbook (7th Edition):

Chang, Yu-Han. “Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection.” 2016. Web. 30 Sep 2020.

Vancouver:

Chang Y. Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2020 Sep 30]. Available from: http://hdl.handle.net/1807/91899.

Council of Science Editors:

Chang Y. Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/91899


Wright State University

8. Oulad Abdelati, Howaida A. Type I Interferon Activation of Natural Killer (NK) Cells by Cytomegalovirus (CMV) and Their Interaction with Dendritic (DC) and NKT Cells.

Degree: MS, Microbiology and Immunology, 2013, Wright State University

 In our current study the roles of natural killer (NK) cells in regulation of the acute phase of murine cytomegalovirus (MCMV) infection were demonstrated. NK… (more)

Subjects/Keywords: Microbiology; Immunology; NK cells; MCMV; NKT; PDCs; Type I interferon

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APA (6th Edition):

Oulad Abdelati, H. A. (2013). Type I Interferon Activation of Natural Killer (NK) Cells by Cytomegalovirus (CMV) and Their Interaction with Dendritic (DC) and NKT Cells. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1389625313

Chicago Manual of Style (16th Edition):

Oulad Abdelati, Howaida A. “Type I Interferon Activation of Natural Killer (NK) Cells by Cytomegalovirus (CMV) and Their Interaction with Dendritic (DC) and NKT Cells.” 2013. Masters Thesis, Wright State University. Accessed September 30, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1389625313.

MLA Handbook (7th Edition):

Oulad Abdelati, Howaida A. “Type I Interferon Activation of Natural Killer (NK) Cells by Cytomegalovirus (CMV) and Their Interaction with Dendritic (DC) and NKT Cells.” 2013. Web. 30 Sep 2020.

Vancouver:

Oulad Abdelati HA. Type I Interferon Activation of Natural Killer (NK) Cells by Cytomegalovirus (CMV) and Their Interaction with Dendritic (DC) and NKT Cells. [Internet] [Masters thesis]. Wright State University; 2013. [cited 2020 Sep 30]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1389625313.

Council of Science Editors:

Oulad Abdelati HA. Type I Interferon Activation of Natural Killer (NK) Cells by Cytomegalovirus (CMV) and Their Interaction with Dendritic (DC) and NKT Cells. [Masters Thesis]. Wright State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1389625313


University of Lund

9. Olsson, Peter. Sjögren's syndrome: epidemiological risk factors and biomarkers.

Degree: 2019, University of Lund

 Primary Sjögren’s syndrome (pSS) is an autoimmune disease, primarliy affecting women, characterised by inflammation and destruction of exocrine glands with a prevalence of 0.01-0.09%. About… (more)

Subjects/Keywords: Rheumatology and Autoimmunity; Sjögren syndrome; cytokine; Interferon Type I; Cigarette smoking

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APA (6th Edition):

Olsson, P. (2019). Sjögren's syndrome: epidemiological risk factors and biomarkers. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/dc31e909-3f96-46f4-a53a-91ea3dc8925c ; https://portal.research.lu.se/ws/files/68410569/Avhandling_Peter_Olsson.pdf

Chicago Manual of Style (16th Edition):

Olsson, Peter. “Sjögren's syndrome: epidemiological risk factors and biomarkers.” 2019. Doctoral Dissertation, University of Lund. Accessed September 30, 2020. https://lup.lub.lu.se/record/dc31e909-3f96-46f4-a53a-91ea3dc8925c ; https://portal.research.lu.se/ws/files/68410569/Avhandling_Peter_Olsson.pdf.

MLA Handbook (7th Edition):

Olsson, Peter. “Sjögren's syndrome: epidemiological risk factors and biomarkers.” 2019. Web. 30 Sep 2020.

Vancouver:

Olsson P. Sjögren's syndrome: epidemiological risk factors and biomarkers. [Internet] [Doctoral dissertation]. University of Lund; 2019. [cited 2020 Sep 30]. Available from: https://lup.lub.lu.se/record/dc31e909-3f96-46f4-a53a-91ea3dc8925c ; https://portal.research.lu.se/ws/files/68410569/Avhandling_Peter_Olsson.pdf.

Council of Science Editors:

Olsson P. Sjögren's syndrome: epidemiological risk factors and biomarkers. [Doctoral Dissertation]. University of Lund; 2019. Available from: https://lup.lub.lu.se/record/dc31e909-3f96-46f4-a53a-91ea3dc8925c ; https://portal.research.lu.se/ws/files/68410569/Avhandling_Peter_Olsson.pdf


University of Melbourne

10. Rahman, Tania. Regulation of the immune responses to Citrobacter rodentium.

Degree: 2016, University of Melbourne

 Pathogenic strains of E. coli including enteropathogenic E. coli (EPEC), enterohemorrhagic E. coli (EHEC), Enterotoxigenic E. coli (ETEC) are principle causes for diarrhoea in many… (more)

Subjects/Keywords: Citrobacter rodentium; plasmacytoid dendritic cell; regulatory T cell; type I interferon

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APA (6th Edition):

Rahman, T. (2016). Regulation of the immune responses to Citrobacter rodentium. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/121805

Chicago Manual of Style (16th Edition):

Rahman, Tania. “Regulation of the immune responses to Citrobacter rodentium.” 2016. Doctoral Dissertation, University of Melbourne. Accessed September 30, 2020. http://hdl.handle.net/11343/121805.

MLA Handbook (7th Edition):

Rahman, Tania. “Regulation of the immune responses to Citrobacter rodentium.” 2016. Web. 30 Sep 2020.

Vancouver:

Rahman T. Regulation of the immune responses to Citrobacter rodentium. [Internet] [Doctoral dissertation]. University of Melbourne; 2016. [cited 2020 Sep 30]. Available from: http://hdl.handle.net/11343/121805.

Council of Science Editors:

Rahman T. Regulation of the immune responses to Citrobacter rodentium. [Doctoral Dissertation]. University of Melbourne; 2016. Available from: http://hdl.handle.net/11343/121805


University of Texas Southwestern Medical Center

11. Hagan, Kristan Andrea. Type I Interferon Signaling Pathway May Be Involved in Optimal Interleukin-2 Production in CD4+ T Cells.

Degree: 2013, University of Texas Southwestern Medical Center

 Previous studies demonstrate that interferon alpha (IFN-α) promotes human T helper 1 memory development by positively regulating interleukin-2 (IL-2) expression, a hallmark cytokine of central… (more)

Subjects/Keywords: Interferon Type I; CD4-Positive T-Lymphocytes; Receptors, Interleukin-2

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APA (6th Edition):

Hagan, K. A. (2013). Type I Interferon Signaling Pathway May Be Involved in Optimal Interleukin-2 Production in CD4+ T Cells. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1249

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hagan, Kristan Andrea. “Type I Interferon Signaling Pathway May Be Involved in Optimal Interleukin-2 Production in CD4+ T Cells.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed September 30, 2020. http://hdl.handle.net/2152.5/1249.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hagan, Kristan Andrea. “Type I Interferon Signaling Pathway May Be Involved in Optimal Interleukin-2 Production in CD4+ T Cells.” 2013. Web. 30 Sep 2020.

Vancouver:

Hagan KA. Type I Interferon Signaling Pathway May Be Involved in Optimal Interleukin-2 Production in CD4+ T Cells. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Sep 30]. Available from: http://hdl.handle.net/2152.5/1249.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hagan KA. Type I Interferon Signaling Pathway May Be Involved in Optimal Interleukin-2 Production in CD4+ T Cells. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1249

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

12. Chowdhury, Fatema Zahra. Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12.

Degree: 2013, University of Texas Southwestern Medical Center

 CD8+ T cells or cytotoxic T lymphocytes (CTLs) play a major role in our defense against intracellular pathogens by secreting effector cytokines and directly killing… (more)

Subjects/Keywords: Gene Expression Regulation; CD8-Positive T-Lymphocytes; Interferon Type I

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APA (6th Edition):

Chowdhury, F. Z. (2013). Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chowdhury, Fatema Zahra. “Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed September 30, 2020. http://hdl.handle.net/2152.5/1729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chowdhury, Fatema Zahra. “Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12.” 2013. Web. 30 Sep 2020.

Vancouver:

Chowdhury FZ. Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Sep 30]. Available from: http://hdl.handle.net/2152.5/1729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chowdhury FZ. Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

13. Erickson, Andrea Kaup. The Role of Interferon Stimlated Genes in Resistance and Immunity to Hepatitis C Virus Infection.

Degree: 2009, University of Texas Southwestern Medical Center

 Hepatitis C Virus (HCV) is a global public health issue with 170 million people chronically infected. The only approved treatment for HCV infection is interferon-alpha… (more)

Subjects/Keywords: Hepacivirus; Interferon Type I; Hepatocytes

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APA (6th Edition):

Erickson, A. K. (2009). The Role of Interferon Stimlated Genes in Resistance and Immunity to Hepatitis C Virus Infection. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/290

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Erickson, Andrea Kaup. “The Role of Interferon Stimlated Genes in Resistance and Immunity to Hepatitis C Virus Infection.” 2009. Thesis, University of Texas Southwestern Medical Center. Accessed September 30, 2020. http://hdl.handle.net/2152.5/290.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Erickson, Andrea Kaup. “The Role of Interferon Stimlated Genes in Resistance and Immunity to Hepatitis C Virus Infection.” 2009. Web. 30 Sep 2020.

Vancouver:

Erickson AK. The Role of Interferon Stimlated Genes in Resistance and Immunity to Hepatitis C Virus Infection. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2009. [cited 2020 Sep 30]. Available from: http://hdl.handle.net/2152.5/290.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Erickson AK. The Role of Interferon Stimlated Genes in Resistance and Immunity to Hepatitis C Virus Infection. [Thesis]. University of Texas Southwestern Medical Center; 2009. Available from: http://hdl.handle.net/2152.5/290

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Iowa State University

14. Ramanan, Parameshwaran. Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion.

Degree: 2012, Iowa State University

 Filoviruses are among the most deadly pathogens that cause acute disease in humans. Ebolavirus (EBOV) and marburgvirus (MARV) are the two members of this family,… (more)

Subjects/Keywords: dsRNA binding; immune evasion; Marburg virus; type I interferon; VP35; Biochemistry

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APA (6th Edition):

Ramanan, P. (2012). Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/12889

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ramanan, Parameshwaran. “Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion.” 2012. Thesis, Iowa State University. Accessed September 30, 2020. https://lib.dr.iastate.edu/etd/12889.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ramanan, Parameshwaran. “Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion.” 2012. Web. 30 Sep 2020.

Vancouver:

Ramanan P. Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion. [Internet] [Thesis]. Iowa State University; 2012. [cited 2020 Sep 30]. Available from: https://lib.dr.iastate.edu/etd/12889.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ramanan P. Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion. [Thesis]. Iowa State University; 2012. Available from: https://lib.dr.iastate.edu/etd/12889

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Ribeiro, Aline Hunger. Transferência gênica de p19Arf e interferon-b em células de melanoma.

Degree: Mestrado, Biotecnologia, 2011, University of São Paulo

O melanoma maligno é uma forma de câncer com alto índice de morte devido, em parte, à falta de tratamentos eficazes e à sua tendência… (more)

Subjects/Keywords: Adenovirus; Adenovírus; Gene transfer; Interferon tipo I; Melanoma; Melanoma; Metástase neoplásica; Neoplasias; Neoplasm metastasis; Neoplasms; Transferência de genes; Type I interferon

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APA (6th Edition):

Ribeiro, A. H. (2011). Transferência gênica de p19Arf e interferon-b em células de melanoma. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/87/87131/tde-10022012-143841/ ;

Chicago Manual of Style (16th Edition):

Ribeiro, Aline Hunger. “Transferência gênica de p19Arf e interferon-b em células de melanoma.” 2011. Masters Thesis, University of São Paulo. Accessed September 30, 2020. http://www.teses.usp.br/teses/disponiveis/87/87131/tde-10022012-143841/ ;.

MLA Handbook (7th Edition):

Ribeiro, Aline Hunger. “Transferência gênica de p19Arf e interferon-b em células de melanoma.” 2011. Web. 30 Sep 2020.

Vancouver:

Ribeiro AH. Transferência gênica de p19Arf e interferon-b em células de melanoma. [Internet] [Masters thesis]. University of São Paulo; 2011. [cited 2020 Sep 30]. Available from: http://www.teses.usp.br/teses/disponiveis/87/87131/tde-10022012-143841/ ;.

Council of Science Editors:

Ribeiro AH. Transferência gênica de p19Arf e interferon-b em células de melanoma. [Masters Thesis]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/87/87131/tde-10022012-143841/ ;

16. Santillo, Bruna Tereso. Caracterização fenotípica e funcional de IFN-DCs derivadas de indivíduos infectados pelo HIV-1.

Degree: Mestrado, Imunologia, 2015, University of São Paulo

A imunoterapia baseada em MoDC constitui uma estratégia para tratamento de indivíduos HIV+. Protocolos para obtenção de MoDC em geral utilizam IL-4 e GM-CSF (IL4-DC).… (more)

Subjects/Keywords: Células dendríticas; Dendritic cells; HIV; HIV; Interferon tipo I; Therapeutic vaccine; Type I Interferon; Vacina terapêutica

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APA (6th Edition):

Santillo, B. T. (2015). Caracterização fenotípica e funcional de IFN-DCs derivadas de indivíduos infectados pelo HIV-1. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42133/tde-09122015-130521/ ;

Chicago Manual of Style (16th Edition):

Santillo, Bruna Tereso. “Caracterização fenotípica e funcional de IFN-DCs derivadas de indivíduos infectados pelo HIV-1.” 2015. Masters Thesis, University of São Paulo. Accessed September 30, 2020. http://www.teses.usp.br/teses/disponiveis/42/42133/tde-09122015-130521/ ;.

MLA Handbook (7th Edition):

Santillo, Bruna Tereso. “Caracterização fenotípica e funcional de IFN-DCs derivadas de indivíduos infectados pelo HIV-1.” 2015. Web. 30 Sep 2020.

Vancouver:

Santillo BT. Caracterização fenotípica e funcional de IFN-DCs derivadas de indivíduos infectados pelo HIV-1. [Internet] [Masters thesis]. University of São Paulo; 2015. [cited 2020 Sep 30]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-09122015-130521/ ;.

Council of Science Editors:

Santillo BT. Caracterização fenotípica e funcional de IFN-DCs derivadas de indivíduos infectados pelo HIV-1. [Masters Thesis]. University of São Paulo; 2015. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-09122015-130521/ ;

17. Frémond, Marie-Louise. Clinical and molecular characterisation of the type I interferonopathies and approaches to therapy : Caractéristiques cliniques et moléculaires et approches thérapeutiques des interféronopathies de type I.

Degree: Docteur es, Génétique et biologie cellulaire, 2018, Sorbonne Paris Cité

 Le concept d'interféronopathie de type I émerge en 2011 et fait référence à un ensemble de pathologies Mendéliennes caractérisées par une hyperactivation des interférons (IFN)… (more)

Subjects/Keywords: Interférons; Interféronopathies de type I; Sting; Savi; Copa; Inhibiteurs de JAK1; Interferon; Type I interferonopathies; Sting; Savi; Copa; JAK1 inhibitors; 606.079

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APA (6th Edition):

Frémond, M. (2018). Clinical and molecular characterisation of the type I interferonopathies and approaches to therapy : Caractéristiques cliniques et moléculaires et approches thérapeutiques des interféronopathies de type I. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2018USPCB098

Chicago Manual of Style (16th Edition):

Frémond, Marie-Louise. “Clinical and molecular characterisation of the type I interferonopathies and approaches to therapy : Caractéristiques cliniques et moléculaires et approches thérapeutiques des interféronopathies de type I.” 2018. Doctoral Dissertation, Sorbonne Paris Cité. Accessed September 30, 2020. http://www.theses.fr/2018USPCB098.

MLA Handbook (7th Edition):

Frémond, Marie-Louise. “Clinical and molecular characterisation of the type I interferonopathies and approaches to therapy : Caractéristiques cliniques et moléculaires et approches thérapeutiques des interféronopathies de type I.” 2018. Web. 30 Sep 2020.

Vancouver:

Frémond M. Clinical and molecular characterisation of the type I interferonopathies and approaches to therapy : Caractéristiques cliniques et moléculaires et approches thérapeutiques des interféronopathies de type I. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2018. [cited 2020 Sep 30]. Available from: http://www.theses.fr/2018USPCB098.

Council of Science Editors:

Frémond M. Clinical and molecular characterisation of the type I interferonopathies and approaches to therapy : Caractéristiques cliniques et moléculaires et approches thérapeutiques des interféronopathies de type I. [Doctoral Dissertation]. Sorbonne Paris Cité; 2018. Available from: http://www.theses.fr/2018USPCB098

18. Alexandre, Yannick. Etude des fonctions des cellules dendritiques dans l'activation des lymphocytes cytotoxiques au cours d'infections in vivo : Investigating the functions of dendritic cells in activating cytotoxic lymphocytes during infections in vivo.

Degree: Docteur es, Immunologie, 2014, Aix Marseille Université

En réponse à une infection, un signal de danger, ou de cytokines inflammatoires, les cellules dendritiques subissent un programme de maturation augmentant leur capacité à… (more)

Subjects/Keywords: Cellule dendritique; Maturation; Interféron de type I; Réponse mémoire; Infections; Dendritic cell; Maturation; Type I interferon; Memory response; Infections; 571

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APA (6th Edition):

Alexandre, Y. (2014). Etude des fonctions des cellules dendritiques dans l'activation des lymphocytes cytotoxiques au cours d'infections in vivo : Investigating the functions of dendritic cells in activating cytotoxic lymphocytes during infections in vivo. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2014AIXM4038

Chicago Manual of Style (16th Edition):

Alexandre, Yannick. “Etude des fonctions des cellules dendritiques dans l'activation des lymphocytes cytotoxiques au cours d'infections in vivo : Investigating the functions of dendritic cells in activating cytotoxic lymphocytes during infections in vivo.” 2014. Doctoral Dissertation, Aix Marseille Université. Accessed September 30, 2020. http://www.theses.fr/2014AIXM4038.

MLA Handbook (7th Edition):

Alexandre, Yannick. “Etude des fonctions des cellules dendritiques dans l'activation des lymphocytes cytotoxiques au cours d'infections in vivo : Investigating the functions of dendritic cells in activating cytotoxic lymphocytes during infections in vivo.” 2014. Web. 30 Sep 2020.

Vancouver:

Alexandre Y. Etude des fonctions des cellules dendritiques dans l'activation des lymphocytes cytotoxiques au cours d'infections in vivo : Investigating the functions of dendritic cells in activating cytotoxic lymphocytes during infections in vivo. [Internet] [Doctoral dissertation]. Aix Marseille Université 2014. [cited 2020 Sep 30]. Available from: http://www.theses.fr/2014AIXM4038.

Council of Science Editors:

Alexandre Y. Etude des fonctions des cellules dendritiques dans l'activation des lymphocytes cytotoxiques au cours d'infections in vivo : Investigating the functions of dendritic cells in activating cytotoxic lymphocytes during infections in vivo. [Doctoral Dissertation]. Aix Marseille Université 2014. Available from: http://www.theses.fr/2014AIXM4038

19. Detilleux, Dylane. Functional characterization of the TRRAP pseudokinase and its chaperone TTT during transcriptional regulation in colorectal cancer : Etude du rôle de la pseudokinase TRRAP et de sa chaperone TTT sur la régulation de la transcription dans le cancer colorectal.

Degree: Docteur es, Biologie Santé, 2018, Montpellier

La régulation de l’expression des gènes est critique pour l’adaptation des cellules à leur environnement et pour leur homéostasie. La transcription, qui représente une étape… (more)

Subjects/Keywords: Transcription; Chromatine; Expression des gènes; Interféron type I; Cancer colorectal; Chaperonne; Transcription; Chromatine; Gene expression; Type I interferon; Colorectal cancer; Chaperone

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APA (6th Edition):

Detilleux, D. (2018). Functional characterization of the TRRAP pseudokinase and its chaperone TTT during transcriptional regulation in colorectal cancer : Etude du rôle de la pseudokinase TRRAP et de sa chaperone TTT sur la régulation de la transcription dans le cancer colorectal. (Doctoral Dissertation). Montpellier. Retrieved from http://www.theses.fr/2018MONTT092

Chicago Manual of Style (16th Edition):

Detilleux, Dylane. “Functional characterization of the TRRAP pseudokinase and its chaperone TTT during transcriptional regulation in colorectal cancer : Etude du rôle de la pseudokinase TRRAP et de sa chaperone TTT sur la régulation de la transcription dans le cancer colorectal.” 2018. Doctoral Dissertation, Montpellier. Accessed September 30, 2020. http://www.theses.fr/2018MONTT092.

MLA Handbook (7th Edition):

Detilleux, Dylane. “Functional characterization of the TRRAP pseudokinase and its chaperone TTT during transcriptional regulation in colorectal cancer : Etude du rôle de la pseudokinase TRRAP et de sa chaperone TTT sur la régulation de la transcription dans le cancer colorectal.” 2018. Web. 30 Sep 2020.

Vancouver:

Detilleux D. Functional characterization of the TRRAP pseudokinase and its chaperone TTT during transcriptional regulation in colorectal cancer : Etude du rôle de la pseudokinase TRRAP et de sa chaperone TTT sur la régulation de la transcription dans le cancer colorectal. [Internet] [Doctoral dissertation]. Montpellier; 2018. [cited 2020 Sep 30]. Available from: http://www.theses.fr/2018MONTT092.

Council of Science Editors:

Detilleux D. Functional characterization of the TRRAP pseudokinase and its chaperone TTT during transcriptional regulation in colorectal cancer : Etude du rôle de la pseudokinase TRRAP et de sa chaperone TTT sur la régulation de la transcription dans le cancer colorectal. [Doctoral Dissertation]. Montpellier; 2018. Available from: http://www.theses.fr/2018MONTT092

20. Maria, Naomi. The Toll of too much Interferon: The systemic interferon signature in the pathogenesis of Sjögren’s syndrome.

Degree: 2015, Erasmus University Medical Center

 markdownabstractAbstract Primary Sjögren's Syndrome (pSS) is an autoimmune disease characterized by accumulation of white blood cells in the salivary and lachrymal glands. Characteristic symptoms are… (more)

Subjects/Keywords: Sjögren's syndrome; autoimmunity; Interferon type I; Interferon signature

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APA (6th Edition):

Maria, N. (2015). The Toll of too much Interferon: The systemic interferon signature in the pathogenesis of Sjögren’s syndrome. (Doctoral Dissertation). Erasmus University Medical Center. Retrieved from http://hdl.handle.net/1765/79230

Chicago Manual of Style (16th Edition):

Maria, Naomi. “The Toll of too much Interferon: The systemic interferon signature in the pathogenesis of Sjögren’s syndrome.” 2015. Doctoral Dissertation, Erasmus University Medical Center. Accessed September 30, 2020. http://hdl.handle.net/1765/79230.

MLA Handbook (7th Edition):

Maria, Naomi. “The Toll of too much Interferon: The systemic interferon signature in the pathogenesis of Sjögren’s syndrome.” 2015. Web. 30 Sep 2020.

Vancouver:

Maria N. The Toll of too much Interferon: The systemic interferon signature in the pathogenesis of Sjögren’s syndrome. [Internet] [Doctoral dissertation]. Erasmus University Medical Center; 2015. [cited 2020 Sep 30]. Available from: http://hdl.handle.net/1765/79230.

Council of Science Editors:

Maria N. The Toll of too much Interferon: The systemic interferon signature in the pathogenesis of Sjögren’s syndrome. [Doctoral Dissertation]. Erasmus University Medical Center; 2015. Available from: http://hdl.handle.net/1765/79230

21. Perego, Jessica. Modulation pharmacologique des voies de signalisation des TLRs par le Guanabenz, un inhibiteur de la réponse au stress : Pharmacological inhibition of the TLRs signalling pathways by Guanabenz, an inhibitor of the stress response.

Degree: Docteur es, Biologie, 2016, Aix Marseille Université

On a récemment mis en évidence l'existence d'une étroite interconnexion entre la perception d'éléments d'origine microbienne (qui se fait à travers les récepteurs de l’immunité… (more)

Subjects/Keywords: Cellules dendritiques; Led; Gadd34; Interferon de type I; Choc septique; Gadd34; TLRs; Dendritic cells; Sle; Gadd34; Type I interferon; Septic shock; Gadd34; TLRs; 571

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APA (6th Edition):

Perego, J. (2016). Modulation pharmacologique des voies de signalisation des TLRs par le Guanabenz, un inhibiteur de la réponse au stress : Pharmacological inhibition of the TLRs signalling pathways by Guanabenz, an inhibitor of the stress response. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2016AIXM4066

Chicago Manual of Style (16th Edition):

Perego, Jessica. “Modulation pharmacologique des voies de signalisation des TLRs par le Guanabenz, un inhibiteur de la réponse au stress : Pharmacological inhibition of the TLRs signalling pathways by Guanabenz, an inhibitor of the stress response.” 2016. Doctoral Dissertation, Aix Marseille Université. Accessed September 30, 2020. http://www.theses.fr/2016AIXM4066.

MLA Handbook (7th Edition):

Perego, Jessica. “Modulation pharmacologique des voies de signalisation des TLRs par le Guanabenz, un inhibiteur de la réponse au stress : Pharmacological inhibition of the TLRs signalling pathways by Guanabenz, an inhibitor of the stress response.” 2016. Web. 30 Sep 2020.

Vancouver:

Perego J. Modulation pharmacologique des voies de signalisation des TLRs par le Guanabenz, un inhibiteur de la réponse au stress : Pharmacological inhibition of the TLRs signalling pathways by Guanabenz, an inhibitor of the stress response. [Internet] [Doctoral dissertation]. Aix Marseille Université 2016. [cited 2020 Sep 30]. Available from: http://www.theses.fr/2016AIXM4066.

Council of Science Editors:

Perego J. Modulation pharmacologique des voies de signalisation des TLRs par le Guanabenz, un inhibiteur de la réponse au stress : Pharmacological inhibition of the TLRs signalling pathways by Guanabenz, an inhibitor of the stress response. [Doctoral Dissertation]. Aix Marseille Université 2016. Available from: http://www.theses.fr/2016AIXM4066

22. Li, Zhi. Insights on type I IFN signaling and regulation : studies of disease-associated TYK2 variants and of the negative regulators USP18/ISG15 : Signalisation et régulation de l'interféron de type I : études de variants de TYK2 associés à des maladies et des régulateurs négatifs USP18 et ISG15.

Degree: Docteur es, Physiologie, Physiopathologie et Thérapeutique, 2017, Université Pierre et Marie Curie – Paris VI

L'action ubiquitaire de l'interféron de type I (IFN- alpha/beta , ici IFN) dans la physiologie et la pathologie est aujourd'hui certaine. Une réponse dérégulée à… (more)

Subjects/Keywords: Interferon de type I; Janus kinase; Maladies auto-Immunes; Polymorphisme nucleotidique simple; Usp18; Isg15; Type I interferon; Auto-immune disease; Janus kinase; 571.9

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APA (6th Edition):

Li, Z. (2017). Insights on type I IFN signaling and regulation : studies of disease-associated TYK2 variants and of the negative regulators USP18/ISG15 : Signalisation et régulation de l'interféron de type I : études de variants de TYK2 associés à des maladies et des régulateurs négatifs USP18 et ISG15. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2017PA066437

Chicago Manual of Style (16th Edition):

Li, Zhi. “Insights on type I IFN signaling and regulation : studies of disease-associated TYK2 variants and of the negative regulators USP18/ISG15 : Signalisation et régulation de l'interféron de type I : études de variants de TYK2 associés à des maladies et des régulateurs négatifs USP18 et ISG15.” 2017. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed September 30, 2020. http://www.theses.fr/2017PA066437.

MLA Handbook (7th Edition):

Li, Zhi. “Insights on type I IFN signaling and regulation : studies of disease-associated TYK2 variants and of the negative regulators USP18/ISG15 : Signalisation et régulation de l'interféron de type I : études de variants de TYK2 associés à des maladies et des régulateurs négatifs USP18 et ISG15.” 2017. Web. 30 Sep 2020.

Vancouver:

Li Z. Insights on type I IFN signaling and regulation : studies of disease-associated TYK2 variants and of the negative regulators USP18/ISG15 : Signalisation et régulation de l'interféron de type I : études de variants de TYK2 associés à des maladies et des régulateurs négatifs USP18 et ISG15. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. [cited 2020 Sep 30]. Available from: http://www.theses.fr/2017PA066437.

Council of Science Editors:

Li Z. Insights on type I IFN signaling and regulation : studies of disease-associated TYK2 variants and of the negative regulators USP18/ISG15 : Signalisation et régulation de l'interféron de type I : études de variants de TYK2 associés à des maladies et des régulateurs négatifs USP18 et ISG15. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. Available from: http://www.theses.fr/2017PA066437


UCLA

23. York, Autumn Gabrielle. Identification of a cholesterol metabolic-type I interferon inflammatory circuit.

Degree: Molecular and Medical Pharmacology, 2015, UCLA

 Cellular lipid requirements are achieved through a combination of biosynthesis and import programs. To date, the molecular mechanisms that regulate whether a cell preferentially scavenges… (more)

Subjects/Keywords: Immunology; Biochemistry; Innate Immunity; Lipid Metabolism; STING; Toll-like Receptors; Type I Interferon

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APA (6th Edition):

York, A. G. (2015). Identification of a cholesterol metabolic-type I interferon inflammatory circuit. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/7z09j9gj

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

York, Autumn Gabrielle. “Identification of a cholesterol metabolic-type I interferon inflammatory circuit.” 2015. Thesis, UCLA. Accessed September 30, 2020. http://www.escholarship.org/uc/item/7z09j9gj.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

York, Autumn Gabrielle. “Identification of a cholesterol metabolic-type I interferon inflammatory circuit.” 2015. Web. 30 Sep 2020.

Vancouver:

York AG. Identification of a cholesterol metabolic-type I interferon inflammatory circuit. [Internet] [Thesis]. UCLA; 2015. [cited 2020 Sep 30]. Available from: http://www.escholarship.org/uc/item/7z09j9gj.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

York AG. Identification of a cholesterol metabolic-type I interferon inflammatory circuit. [Thesis]. UCLA; 2015. Available from: http://www.escholarship.org/uc/item/7z09j9gj

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Brkic, Zana. Interferon Type I Driven Immune Activation in Generalized Autoimmune Diseases.

Degree: 2013, Erasmus University Medical Center

 textabstractThis thesis describes research performed on several generalized autoimmune diseases with the main focus on primary Sjögren’s syndrome. Interferon type I has been implicated in… (more)

Subjects/Keywords: Interferon type I; autoimmune diseases; immunology

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APA (6th Edition):

Brkic, Z. (2013). Interferon Type I Driven Immune Activation in Generalized Autoimmune Diseases. (Doctoral Dissertation). Erasmus University Medical Center. Retrieved from http://hdl.handle.net/1765/41397

Chicago Manual of Style (16th Edition):

Brkic, Zana. “Interferon Type I Driven Immune Activation in Generalized Autoimmune Diseases.” 2013. Doctoral Dissertation, Erasmus University Medical Center. Accessed September 30, 2020. http://hdl.handle.net/1765/41397.

MLA Handbook (7th Edition):

Brkic, Zana. “Interferon Type I Driven Immune Activation in Generalized Autoimmune Diseases.” 2013. Web. 30 Sep 2020.

Vancouver:

Brkic Z. Interferon Type I Driven Immune Activation in Generalized Autoimmune Diseases. [Internet] [Doctoral dissertation]. Erasmus University Medical Center; 2013. [cited 2020 Sep 30]. Available from: http://hdl.handle.net/1765/41397.

Council of Science Editors:

Brkic Z. Interferon Type I Driven Immune Activation in Generalized Autoimmune Diseases. [Doctoral Dissertation]. Erasmus University Medical Center; 2013. Available from: http://hdl.handle.net/1765/41397


The Ohio State University

25. Machado Parrula, Maria Cecilia. Measles Virotherapy in Adult T cell Leukemia.

Degree: PhD, Veterinary Biosciences, 2009, The Ohio State University

 Adult T cell leukemia (ATL) is a proliferative disorder of CD4+ T cells caused by human T cell lymphotropic type I virus infection. ATL is… (more)

Subjects/Keywords: Oncology; Virology; HTLV-1; ATL; measles; immunodeficient mice; type I interferon; cotton rat

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APA (6th Edition):

Machado Parrula, M. C. (2009). Measles Virotherapy in Adult T cell Leukemia. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1261413581

Chicago Manual of Style (16th Edition):

Machado Parrula, Maria Cecilia. “Measles Virotherapy in Adult T cell Leukemia.” 2009. Doctoral Dissertation, The Ohio State University. Accessed September 30, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1261413581.

MLA Handbook (7th Edition):

Machado Parrula, Maria Cecilia. “Measles Virotherapy in Adult T cell Leukemia.” 2009. Web. 30 Sep 2020.

Vancouver:

Machado Parrula MC. Measles Virotherapy in Adult T cell Leukemia. [Internet] [Doctoral dissertation]. The Ohio State University; 2009. [cited 2020 Sep 30]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1261413581.

Council of Science Editors:

Machado Parrula MC. Measles Virotherapy in Adult T cell Leukemia. [Doctoral Dissertation]. The Ohio State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1261413581


University of Adelaide

26. Chan, Jennifer. Pre-existing strain specific neutralising antibodies abrogates the induction of interferon type I and cytotoxic T cell responses to subsequent homotypic influenza A virus challenge.

Degree: 2013, University of Adelaide

 Current inactivated influenza vaccines target the generation of influenza-specific antibodies to provide homotypic protection. However, little is known about the effects of annual vaccinations on… (more)

Subjects/Keywords: immune response; virus challenge; B cells; influenza A virus; T cells; antibodies; interferon type I

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APA (6th Edition):

Chan, J. (2013). Pre-existing strain specific neutralising antibodies abrogates the induction of interferon type I and cytotoxic T cell responses to subsequent homotypic influenza A virus challenge. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/90751

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chan, Jennifer. “Pre-existing strain specific neutralising antibodies abrogates the induction of interferon type I and cytotoxic T cell responses to subsequent homotypic influenza A virus challenge.” 2013. Thesis, University of Adelaide. Accessed September 30, 2020. http://hdl.handle.net/2440/90751.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chan, Jennifer. “Pre-existing strain specific neutralising antibodies abrogates the induction of interferon type I and cytotoxic T cell responses to subsequent homotypic influenza A virus challenge.” 2013. Web. 30 Sep 2020.

Vancouver:

Chan J. Pre-existing strain specific neutralising antibodies abrogates the induction of interferon type I and cytotoxic T cell responses to subsequent homotypic influenza A virus challenge. [Internet] [Thesis]. University of Adelaide; 2013. [cited 2020 Sep 30]. Available from: http://hdl.handle.net/2440/90751.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chan J. Pre-existing strain specific neutralising antibodies abrogates the induction of interferon type I and cytotoxic T cell responses to subsequent homotypic influenza A virus challenge. [Thesis]. University of Adelaide; 2013. Available from: http://hdl.handle.net/2440/90751

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Ranganath, Nischal. Oncolytic Viruses as a Potential Approach to Eliminate Cells That Constitute the Latent HIV Reservoir .

Degree: 2018, University of Ottawa

 HIV infection represents a major health and socioeconomic challenge worldwide. Despite significant advances in therapy, a cure for HIV continues to be elusive. The design… (more)

Subjects/Keywords: Latent HIV reservoir; Oncolytic viruses; Type I interferon; Vesicular stomatitis virus; Marba virus

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ranganath, N. (2018). Oncolytic Viruses as a Potential Approach to Eliminate Cells That Constitute the Latent HIV Reservoir . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/37355

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ranganath, Nischal. “Oncolytic Viruses as a Potential Approach to Eliminate Cells That Constitute the Latent HIV Reservoir .” 2018. Thesis, University of Ottawa. Accessed September 30, 2020. http://hdl.handle.net/10393/37355.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ranganath, Nischal. “Oncolytic Viruses as a Potential Approach to Eliminate Cells That Constitute the Latent HIV Reservoir .” 2018. Web. 30 Sep 2020.

Vancouver:

Ranganath N. Oncolytic Viruses as a Potential Approach to Eliminate Cells That Constitute the Latent HIV Reservoir . [Internet] [Thesis]. University of Ottawa; 2018. [cited 2020 Sep 30]. Available from: http://hdl.handle.net/10393/37355.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ranganath N. Oncolytic Viruses as a Potential Approach to Eliminate Cells That Constitute the Latent HIV Reservoir . [Thesis]. University of Ottawa; 2018. Available from: http://hdl.handle.net/10393/37355

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

28. Rostom, Raghd. Human Cellular Genetics of Innate Immunity.

Degree: PhD, 2020, University of Cambridge

 The type I interferon response is a key part of the innate immune system, responding to infection and inducing an antiviral intracellular state. While there… (more)

Subjects/Keywords: Single cell RNA-sequencing; Single cell genomics; Type I interferon response; Innate immunity

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rostom, R. (2020). Human Cellular Genetics of Innate Immunity. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/299466

Chicago Manual of Style (16th Edition):

Rostom, Raghd. “Human Cellular Genetics of Innate Immunity.” 2020. Doctoral Dissertation, University of Cambridge. Accessed September 30, 2020. https://www.repository.cam.ac.uk/handle/1810/299466.

MLA Handbook (7th Edition):

Rostom, Raghd. “Human Cellular Genetics of Innate Immunity.” 2020. Web. 30 Sep 2020.

Vancouver:

Rostom R. Human Cellular Genetics of Innate Immunity. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2020 Sep 30]. Available from: https://www.repository.cam.ac.uk/handle/1810/299466.

Council of Science Editors:

Rostom R. Human Cellular Genetics of Innate Immunity. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/299466


University of Washington

29. Chen, Helen C. Control of endosomal Toll-like receptor (TLR) signaling by nanoparticles and applications in cancer therapy.

Degree: PhD, 2014, University of Washington

 Toll-like receptors (TLRs) play critical roles in the activation of innate and adaptive immune responses. TLRs 3 and 9 are expressed in endosomal and lysosomal… (more)

Subjects/Keywords: immunotherapy; inflammation; tissue engineering; tumorigenesis; type I interferon; vaccine; Biomedical engineering; Immunology; Nanotechnology; chemical engineering

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, H. C. (2014). Control of endosomal Toll-like receptor (TLR) signaling by nanoparticles and applications in cancer therapy. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/25368

Chicago Manual of Style (16th Edition):

Chen, Helen C. “Control of endosomal Toll-like receptor (TLR) signaling by nanoparticles and applications in cancer therapy.” 2014. Doctoral Dissertation, University of Washington. Accessed September 30, 2020. http://hdl.handle.net/1773/25368.

MLA Handbook (7th Edition):

Chen, Helen C. “Control of endosomal Toll-like receptor (TLR) signaling by nanoparticles and applications in cancer therapy.” 2014. Web. 30 Sep 2020.

Vancouver:

Chen HC. Control of endosomal Toll-like receptor (TLR) signaling by nanoparticles and applications in cancer therapy. [Internet] [Doctoral dissertation]. University of Washington; 2014. [cited 2020 Sep 30]. Available from: http://hdl.handle.net/1773/25368.

Council of Science Editors:

Chen HC. Control of endosomal Toll-like receptor (TLR) signaling by nanoparticles and applications in cancer therapy. [Doctoral Dissertation]. University of Washington; 2014. Available from: http://hdl.handle.net/1773/25368


University of Western Ontario

30. Hunt, Nina. Interferon-induced HERC5 is a novel and potent inhibitor of Ebola virus-like particle production.

Degree: 2018, University of Western Ontario

 Ebola’s severe pathogenesis can be attributed to its suppression of the Type I interferon (IFN) response, suggesting this pathway plays a role in restricting viral… (more)

Subjects/Keywords: Type I interferon response; HERC5; Ebola virus; antiviral; zinc finger antiviral protein; viral antagonism; Virology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hunt, N. (2018). Interferon-induced HERC5 is a novel and potent inhibitor of Ebola virus-like particle production. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/5922

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hunt, Nina. “Interferon-induced HERC5 is a novel and potent inhibitor of Ebola virus-like particle production.” 2018. Thesis, University of Western Ontario. Accessed September 30, 2020. https://ir.lib.uwo.ca/etd/5922.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hunt, Nina. “Interferon-induced HERC5 is a novel and potent inhibitor of Ebola virus-like particle production.” 2018. Web. 30 Sep 2020.

Vancouver:

Hunt N. Interferon-induced HERC5 is a novel and potent inhibitor of Ebola virus-like particle production. [Internet] [Thesis]. University of Western Ontario; 2018. [cited 2020 Sep 30]. Available from: https://ir.lib.uwo.ca/etd/5922.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hunt N. Interferon-induced HERC5 is a novel and potent inhibitor of Ebola virus-like particle production. [Thesis]. University of Western Ontario; 2018. Available from: https://ir.lib.uwo.ca/etd/5922

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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