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You searched for subject:(Interferon Type I). Showing records 1 – 30 of 75 total matches.

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NSYSU

1. Chang, Jenn-tzong. Genome and Infection Characteristics of Human Parechovirus Type 1.

Degree: PhD, Biological Sciences, 2015, NSYSU

 Human parechoviruses (HPeVs), members of the family Picornaviridae, are associated with severe human clinical conditions such as gastrointestinal disease, encephalitis, meningitis, respiratory disease and neonatal… (more)

Subjects/Keywords: type I interferon; parechovirus; HPeV; innate immunity

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APA (6th Edition):

Chang, J. (2015). Genome and Infection Characteristics of Human Parechovirus Type 1. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521115-005317

Chicago Manual of Style (16th Edition):

Chang, Jenn-tzong. “Genome and Infection Characteristics of Human Parechovirus Type 1.” 2015. Doctoral Dissertation, NSYSU. Accessed August 20, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521115-005317.

MLA Handbook (7th Edition):

Chang, Jenn-tzong. “Genome and Infection Characteristics of Human Parechovirus Type 1.” 2015. Web. 20 Aug 2019.

Vancouver:

Chang J. Genome and Infection Characteristics of Human Parechovirus Type 1. [Internet] [Doctoral dissertation]. NSYSU; 2015. [cited 2019 Aug 20]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521115-005317.

Council of Science Editors:

Chang J. Genome and Infection Characteristics of Human Parechovirus Type 1. [Doctoral Dissertation]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521115-005317

2. Smith, Nikaïa. Étude moléculaire du TNF-Related Apoptosis Induced Ligand (TRAIL) et de l’activation du Toll-Like Receptor 7 (TLR7) dans les cellules dendritiques plasmacytoïdes lors de la réponse antivirale : Molecular study of the TNF-Related Apoptosis Induced Ligand (TRAIL) and of Toll-Like Receptor 7 (TLR7) activation in plasmacytoid dendritic cells during viral infections.

Degree: Docteur es, Immunologie, 2015, Sorbonne Paris Cité

Les pDC représentent la première ligne de défense de l’organisme contre les pathogènes et établissent le lien essentiel entre l’immunité innée et adaptative. Les pDC… (more)

Subjects/Keywords: Cellules dendritiques plasmacytoïdes; Interferon de type I; Amines; CXCR4; SiRNA; Plasmacytoid dendritic cells; Type I interferon; Amines; CXCR4; SiRNA; 571.96

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APA (6th Edition):

Smith, N. (2015). Étude moléculaire du TNF-Related Apoptosis Induced Ligand (TRAIL) et de l’activation du Toll-Like Receptor 7 (TLR7) dans les cellules dendritiques plasmacytoïdes lors de la réponse antivirale : Molecular study of the TNF-Related Apoptosis Induced Ligand (TRAIL) and of Toll-Like Receptor 7 (TLR7) activation in plasmacytoid dendritic cells during viral infections. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2015USPCB145

Chicago Manual of Style (16th Edition):

Smith, Nikaïa. “Étude moléculaire du TNF-Related Apoptosis Induced Ligand (TRAIL) et de l’activation du Toll-Like Receptor 7 (TLR7) dans les cellules dendritiques plasmacytoïdes lors de la réponse antivirale : Molecular study of the TNF-Related Apoptosis Induced Ligand (TRAIL) and of Toll-Like Receptor 7 (TLR7) activation in plasmacytoid dendritic cells during viral infections.” 2015. Doctoral Dissertation, Sorbonne Paris Cité. Accessed August 20, 2019. http://www.theses.fr/2015USPCB145.

MLA Handbook (7th Edition):

Smith, Nikaïa. “Étude moléculaire du TNF-Related Apoptosis Induced Ligand (TRAIL) et de l’activation du Toll-Like Receptor 7 (TLR7) dans les cellules dendritiques plasmacytoïdes lors de la réponse antivirale : Molecular study of the TNF-Related Apoptosis Induced Ligand (TRAIL) and of Toll-Like Receptor 7 (TLR7) activation in plasmacytoid dendritic cells during viral infections.” 2015. Web. 20 Aug 2019.

Vancouver:

Smith N. Étude moléculaire du TNF-Related Apoptosis Induced Ligand (TRAIL) et de l’activation du Toll-Like Receptor 7 (TLR7) dans les cellules dendritiques plasmacytoïdes lors de la réponse antivirale : Molecular study of the TNF-Related Apoptosis Induced Ligand (TRAIL) and of Toll-Like Receptor 7 (TLR7) activation in plasmacytoid dendritic cells during viral infections. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2015. [cited 2019 Aug 20]. Available from: http://www.theses.fr/2015USPCB145.

Council of Science Editors:

Smith N. Étude moléculaire du TNF-Related Apoptosis Induced Ligand (TRAIL) et de l’activation du Toll-Like Receptor 7 (TLR7) dans les cellules dendritiques plasmacytoïdes lors de la réponse antivirale : Molecular study of the TNF-Related Apoptosis Induced Ligand (TRAIL) and of Toll-Like Receptor 7 (TLR7) activation in plasmacytoid dendritic cells during viral infections. [Doctoral Dissertation]. Sorbonne Paris Cité; 2015. Available from: http://www.theses.fr/2015USPCB145


Iowa State University

3. Ramanan, Parameshwaran. Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion.

Degree: 2012, Iowa State University

 Filoviruses are among the most deadly pathogens that cause acute disease in humans. Ebolavirus (EBOV) and marburgvirus (MARV) are the two members of this family,… (more)

Subjects/Keywords: dsRNA binding; immune evasion; Marburg virus; type I interferon; VP35; Biochemistry

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APA (6th Edition):

Ramanan, P. (2012). Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/12889

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ramanan, Parameshwaran. “Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion.” 2012. Thesis, Iowa State University. Accessed August 20, 2019. https://lib.dr.iastate.edu/etd/12889.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ramanan, Parameshwaran. “Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion.” 2012. Web. 20 Aug 2019.

Vancouver:

Ramanan P. Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion. [Internet] [Thesis]. Iowa State University; 2012. [cited 2019 Aug 20]. Available from: https://lib.dr.iastate.edu/etd/12889.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ramanan P. Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion. [Thesis]. Iowa State University; 2012. Available from: https://lib.dr.iastate.edu/etd/12889

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

4. Chang, Yu-Han. Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection.

Degree: 2016, University of Toronto

Co-stimulation of T cells through Glucocorticoid-Induced Tumour Necrosis Factor Receptor-Related Protein (GITR) contributes to viral control during persistent lymphocytic choriomeningitis virus (LCMV) infection. Herein, we… (more)

Subjects/Keywords: GITR; GITRL; LCMV; TNFRSF18; TNFSF18; Type I Interferon; 0982

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APA (6th Edition):

Chang, Y. (2016). Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91899

Chicago Manual of Style (16th Edition):

Chang, Yu-Han. “Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection.” 2016. Masters Thesis, University of Toronto. Accessed August 20, 2019. http://hdl.handle.net/1807/91899.

MLA Handbook (7th Edition):

Chang, Yu-Han. “Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection.” 2016. Web. 20 Aug 2019.

Vancouver:

Chang Y. Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Aug 20]. Available from: http://hdl.handle.net/1807/91899.

Council of Science Editors:

Chang Y. Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/91899


University of Pennsylvania

5. Iyer, Shilpa. Determinants Of Hiv-1 Transmission Fitness.

Degree: 2017, University of Pennsylvania

 DETERMINANTS OF HIV-1 TRANSMISSION FITNESS Shilpa S. Iyer Beatrice H. Hahn HIV-1 is predominantly transmitted by mucosal routes and almost 80 percent of new infections… (more)

Subjects/Keywords: Fitness; HIV-1; Mucosal Transmisison; Type I Interferon; Microbiology; Virology

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APA (6th Edition):

Iyer, S. (2017). Determinants Of Hiv-1 Transmission Fitness. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2355

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Iyer, Shilpa. “Determinants Of Hiv-1 Transmission Fitness.” 2017. Thesis, University of Pennsylvania. Accessed August 20, 2019. https://repository.upenn.edu/edissertations/2355.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Iyer, Shilpa. “Determinants Of Hiv-1 Transmission Fitness.” 2017. Web. 20 Aug 2019.

Vancouver:

Iyer S. Determinants Of Hiv-1 Transmission Fitness. [Internet] [Thesis]. University of Pennsylvania; 2017. [cited 2019 Aug 20]. Available from: https://repository.upenn.edu/edissertations/2355.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Iyer S. Determinants Of Hiv-1 Transmission Fitness. [Thesis]. University of Pennsylvania; 2017. Available from: https://repository.upenn.edu/edissertations/2355

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wright State University

6. Oulad Abdelati, Howaida A. Type I Interferon Activation of Natural Killer (NK) Cells by Cytomegalovirus (CMV) and Their Interaction with Dendritic (DC) and NKT Cells.

Degree: MS, Microbiology and Immunology, 2013, Wright State University

 In our current study the roles of natural killer (NK) cells in regulation of the acute phase of murine cytomegalovirus (MCMV) infection were demonstrated. NK… (more)

Subjects/Keywords: Microbiology; Immunology; NK cells; MCMV; NKT; PDCs; Type I interferon

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APA (6th Edition):

Oulad Abdelati, H. A. (2013). Type I Interferon Activation of Natural Killer (NK) Cells by Cytomegalovirus (CMV) and Their Interaction with Dendritic (DC) and NKT Cells. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1389625313

Chicago Manual of Style (16th Edition):

Oulad Abdelati, Howaida A. “Type I Interferon Activation of Natural Killer (NK) Cells by Cytomegalovirus (CMV) and Their Interaction with Dendritic (DC) and NKT Cells.” 2013. Masters Thesis, Wright State University. Accessed August 20, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1389625313.

MLA Handbook (7th Edition):

Oulad Abdelati, Howaida A. “Type I Interferon Activation of Natural Killer (NK) Cells by Cytomegalovirus (CMV) and Their Interaction with Dendritic (DC) and NKT Cells.” 2013. Web. 20 Aug 2019.

Vancouver:

Oulad Abdelati HA. Type I Interferon Activation of Natural Killer (NK) Cells by Cytomegalovirus (CMV) and Their Interaction with Dendritic (DC) and NKT Cells. [Internet] [Masters thesis]. Wright State University; 2013. [cited 2019 Aug 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1389625313.

Council of Science Editors:

Oulad Abdelati HA. Type I Interferon Activation of Natural Killer (NK) Cells by Cytomegalovirus (CMV) and Their Interaction with Dendritic (DC) and NKT Cells. [Masters Thesis]. Wright State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1389625313


Case Western Reserve University

7. Liu, Yi. Negative Regulation of Type I Interferon Induction in Dendritic Cells.

Degree: PhD, Pathology, 2011, Case Western Reserve University

Type I interferon (IFN) regulates innate and adaptive immunity. It protects host cells against viral infection. Many microorganisms have evolved successful mechanisms to inhibit host… (more)

Subjects/Keywords: Immunology; type I interferon; dendritic cells; pattern recognition receptors

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APA (6th Edition):

Liu, Y. (2011). Negative Regulation of Type I Interferon Induction in Dendritic Cells. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1310149509

Chicago Manual of Style (16th Edition):

Liu, Yi. “Negative Regulation of Type I Interferon Induction in Dendritic Cells.” 2011. Doctoral Dissertation, Case Western Reserve University. Accessed August 20, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1310149509.

MLA Handbook (7th Edition):

Liu, Yi. “Negative Regulation of Type I Interferon Induction in Dendritic Cells.” 2011. Web. 20 Aug 2019.

Vancouver:

Liu Y. Negative Regulation of Type I Interferon Induction in Dendritic Cells. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2011. [cited 2019 Aug 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1310149509.

Council of Science Editors:

Liu Y. Negative Regulation of Type I Interferon Induction in Dendritic Cells. [Doctoral Dissertation]. Case Western Reserve University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1310149509


University of Texas Southwestern Medical Center

8. Erickson, Andrea Kaup. The Role of Interferon Stimlated Genes in Resistance and Immunity to Hepatitis C Virus Infection.

Degree: 2009, University of Texas Southwestern Medical Center

 Hepatitis C Virus (HCV) is a global public health issue with 170 million people chronically infected. The only approved treatment for HCV infection is interferon-alpha… (more)

Subjects/Keywords: Hepacivirus; Interferon Type I; Hepatocytes

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APA (6th Edition):

Erickson, A. K. (2009). The Role of Interferon Stimlated Genes in Resistance and Immunity to Hepatitis C Virus Infection. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/290

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Erickson, Andrea Kaup. “The Role of Interferon Stimlated Genes in Resistance and Immunity to Hepatitis C Virus Infection.” 2009. Thesis, University of Texas Southwestern Medical Center. Accessed August 20, 2019. http://hdl.handle.net/2152.5/290.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Erickson, Andrea Kaup. “The Role of Interferon Stimlated Genes in Resistance and Immunity to Hepatitis C Virus Infection.” 2009. Web. 20 Aug 2019.

Vancouver:

Erickson AK. The Role of Interferon Stimlated Genes in Resistance and Immunity to Hepatitis C Virus Infection. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2009. [cited 2019 Aug 20]. Available from: http://hdl.handle.net/2152.5/290.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Erickson AK. The Role of Interferon Stimlated Genes in Resistance and Immunity to Hepatitis C Virus Infection. [Thesis]. University of Texas Southwestern Medical Center; 2009. Available from: http://hdl.handle.net/2152.5/290

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

9. Hagan, Kristan Andrea. Type I Interferon Signaling Pathway May Be Involved in Optimal Interleukin-2 Production in CD4+ T Cells.

Degree: 2013, University of Texas Southwestern Medical Center

 Previous studies demonstrate that interferon alpha (IFN-α) promotes human T helper 1 memory development by positively regulating interleukin-2 (IL-2) expression, a hallmark cytokine of central… (more)

Subjects/Keywords: Interferon Type I; CD4-Positive T-Lymphocytes; Receptors, Interleukin-2

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APA (6th Edition):

Hagan, K. A. (2013). Type I Interferon Signaling Pathway May Be Involved in Optimal Interleukin-2 Production in CD4+ T Cells. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1249

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hagan, Kristan Andrea. “Type I Interferon Signaling Pathway May Be Involved in Optimal Interleukin-2 Production in CD4+ T Cells.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed August 20, 2019. http://hdl.handle.net/2152.5/1249.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hagan, Kristan Andrea. “Type I Interferon Signaling Pathway May Be Involved in Optimal Interleukin-2 Production in CD4+ T Cells.” 2013. Web. 20 Aug 2019.

Vancouver:

Hagan KA. Type I Interferon Signaling Pathway May Be Involved in Optimal Interleukin-2 Production in CD4+ T Cells. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2019 Aug 20]. Available from: http://hdl.handle.net/2152.5/1249.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hagan KA. Type I Interferon Signaling Pathway May Be Involved in Optimal Interleukin-2 Production in CD4+ T Cells. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1249

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

10. Chowdhury, Fatema Zahra. Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12.

Degree: 2013, University of Texas Southwestern Medical Center

 CD8+ T cells or cytotoxic T lymphocytes (CTLs) play a major role in our defense against intracellular pathogens by secreting effector cytokines and directly killing… (more)

Subjects/Keywords: Gene Expression Regulation; CD8-Positive T-Lymphocytes; Interferon Type I

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APA (6th Edition):

Chowdhury, F. Z. (2013). Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chowdhury, Fatema Zahra. “Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed August 20, 2019. http://hdl.handle.net/2152.5/1729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chowdhury, Fatema Zahra. “Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12.” 2013. Web. 20 Aug 2019.

Vancouver:

Chowdhury FZ. Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2019 Aug 20]. Available from: http://hdl.handle.net/2152.5/1729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chowdhury FZ. Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

11. Zhang, Qingzhan. Innate immune evasion and interferon antagonism by porcine epidemic diarrhea virus.

Degree: PhD, VMS - Pathobiology, 2017, University of Illinois – Urbana-Champaign

 Porcine epidemic diarrhea (PED) is a highly contagious and acute enteric disease of swine featured by vomiting, watery diarrhea, and severe dehydration causing high mortality… (more)

Subjects/Keywords: PEDV; innate immunity; type I interferon; type III IFN; Intestinal epithelial cells; Nsp1

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APA (6th Edition):

Zhang, Q. (2017). Innate immune evasion and interferon antagonism by porcine epidemic diarrhea virus. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/99377

Chicago Manual of Style (16th Edition):

Zhang, Qingzhan. “Innate immune evasion and interferon antagonism by porcine epidemic diarrhea virus.” 2017. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed August 20, 2019. http://hdl.handle.net/2142/99377.

MLA Handbook (7th Edition):

Zhang, Qingzhan. “Innate immune evasion and interferon antagonism by porcine epidemic diarrhea virus.” 2017. Web. 20 Aug 2019.

Vancouver:

Zhang Q. Innate immune evasion and interferon antagonism by porcine epidemic diarrhea virus. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2017. [cited 2019 Aug 20]. Available from: http://hdl.handle.net/2142/99377.

Council of Science Editors:

Zhang Q. Innate immune evasion and interferon antagonism by porcine epidemic diarrhea virus. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2017. Available from: http://hdl.handle.net/2142/99377

12. Ribeiro, Aline Hunger. Transferência gênica de p19Arf e interferon-b em células de melanoma.

Degree: Mestrado, Biotecnologia, 2011, University of São Paulo

O melanoma maligno é uma forma de câncer com alto índice de morte devido, em parte, à falta de tratamentos eficazes e à sua tendência… (more)

Subjects/Keywords: Adenovirus; Adenovírus; Gene transfer; Interferon tipo I; Melanoma; Melanoma; Metástase neoplásica; Neoplasias; Neoplasm metastasis; Neoplasms; Transferência de genes; Type I interferon

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APA (6th Edition):

Ribeiro, A. H. (2011). Transferência gênica de p19Arf e interferon-b em células de melanoma. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/87/87131/tde-10022012-143841/ ;

Chicago Manual of Style (16th Edition):

Ribeiro, Aline Hunger. “Transferência gênica de p19Arf e interferon-b em células de melanoma.” 2011. Masters Thesis, University of São Paulo. Accessed August 20, 2019. http://www.teses.usp.br/teses/disponiveis/87/87131/tde-10022012-143841/ ;.

MLA Handbook (7th Edition):

Ribeiro, Aline Hunger. “Transferência gênica de p19Arf e interferon-b em células de melanoma.” 2011. Web. 20 Aug 2019.

Vancouver:

Ribeiro AH. Transferência gênica de p19Arf e interferon-b em células de melanoma. [Internet] [Masters thesis]. University of São Paulo; 2011. [cited 2019 Aug 20]. Available from: http://www.teses.usp.br/teses/disponiveis/87/87131/tde-10022012-143841/ ;.

Council of Science Editors:

Ribeiro AH. Transferência gênica de p19Arf e interferon-b em células de melanoma. [Masters Thesis]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/87/87131/tde-10022012-143841/ ;

13. Santillo, Bruna Tereso. Caracterização fenotípica e funcional de IFN-DCs derivadas de indivíduos infectados pelo HIV-1.

Degree: Mestrado, Imunologia, 2015, University of São Paulo

A imunoterapia baseada em MoDC constitui uma estratégia para tratamento de indivíduos HIV+. Protocolos para obtenção de MoDC em geral utilizam IL-4 e GM-CSF (IL4-DC).… (more)

Subjects/Keywords: Células dendríticas; Dendritic cells; HIV; HIV; Interferon tipo I; Therapeutic vaccine; Type I Interferon; Vacina terapêutica

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APA (6th Edition):

Santillo, B. T. (2015). Caracterização fenotípica e funcional de IFN-DCs derivadas de indivíduos infectados pelo HIV-1. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42133/tde-09122015-130521/ ;

Chicago Manual of Style (16th Edition):

Santillo, Bruna Tereso. “Caracterização fenotípica e funcional de IFN-DCs derivadas de indivíduos infectados pelo HIV-1.” 2015. Masters Thesis, University of São Paulo. Accessed August 20, 2019. http://www.teses.usp.br/teses/disponiveis/42/42133/tde-09122015-130521/ ;.

MLA Handbook (7th Edition):

Santillo, Bruna Tereso. “Caracterização fenotípica e funcional de IFN-DCs derivadas de indivíduos infectados pelo HIV-1.” 2015. Web. 20 Aug 2019.

Vancouver:

Santillo BT. Caracterização fenotípica e funcional de IFN-DCs derivadas de indivíduos infectados pelo HIV-1. [Internet] [Masters thesis]. University of São Paulo; 2015. [cited 2019 Aug 20]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-09122015-130521/ ;.

Council of Science Editors:

Santillo BT. Caracterização fenotípica e funcional de IFN-DCs derivadas de indivíduos infectados pelo HIV-1. [Masters Thesis]. University of São Paulo; 2015. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-09122015-130521/ ;

14. Alexandre, Yannick. Etude des fonctions des cellules dendritiques dans l'activation des lymphocytes cytotoxiques au cours d'infections in vivo : Investigating the functions of dendritic cells in activating cytotoxic lymphocytes during infections in vivo.

Degree: Docteur es, Immunologie, 2014, Aix Marseille Université

En réponse à une infection, un signal de danger, ou de cytokines inflammatoires, les cellules dendritiques subissent un programme de maturation augmentant leur capacité à… (more)

Subjects/Keywords: Cellule dendritique; Maturation; Interféron de type I; Réponse mémoire; Infections; Dendritic cell; Maturation; Type I interferon; Memory response; Infections; 571

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APA (6th Edition):

Alexandre, Y. (2014). Etude des fonctions des cellules dendritiques dans l'activation des lymphocytes cytotoxiques au cours d'infections in vivo : Investigating the functions of dendritic cells in activating cytotoxic lymphocytes during infections in vivo. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2014AIXM4038

Chicago Manual of Style (16th Edition):

Alexandre, Yannick. “Etude des fonctions des cellules dendritiques dans l'activation des lymphocytes cytotoxiques au cours d'infections in vivo : Investigating the functions of dendritic cells in activating cytotoxic lymphocytes during infections in vivo.” 2014. Doctoral Dissertation, Aix Marseille Université. Accessed August 20, 2019. http://www.theses.fr/2014AIXM4038.

MLA Handbook (7th Edition):

Alexandre, Yannick. “Etude des fonctions des cellules dendritiques dans l'activation des lymphocytes cytotoxiques au cours d'infections in vivo : Investigating the functions of dendritic cells in activating cytotoxic lymphocytes during infections in vivo.” 2014. Web. 20 Aug 2019.

Vancouver:

Alexandre Y. Etude des fonctions des cellules dendritiques dans l'activation des lymphocytes cytotoxiques au cours d'infections in vivo : Investigating the functions of dendritic cells in activating cytotoxic lymphocytes during infections in vivo. [Internet] [Doctoral dissertation]. Aix Marseille Université 2014. [cited 2019 Aug 20]. Available from: http://www.theses.fr/2014AIXM4038.

Council of Science Editors:

Alexandre Y. Etude des fonctions des cellules dendritiques dans l'activation des lymphocytes cytotoxiques au cours d'infections in vivo : Investigating the functions of dendritic cells in activating cytotoxic lymphocytes during infections in vivo. [Doctoral Dissertation]. Aix Marseille Université 2014. Available from: http://www.theses.fr/2014AIXM4038

15. Detilleux, Dylane. Functional characterization of the TRRAP pseudokinase and its chaperone TTT during transcriptional regulation in colorectal cancer : Etude du rôle de la pseudokinase TRRAP et de sa chaperone TTT sur la régulation de la transcription dans le cancer colorectal.

Degree: Docteur es, Biologie Santé, 2018, Montpellier

La régulation de l’expression des gènes est critique pour l’adaptation des cellules à leur environnement et pour leur homéostasie. La transcription, qui représente une étape… (more)

Subjects/Keywords: Transcription; Chromatine; Expression des gènes; Interféron type I; Cancer colorectal; Chaperonne; Transcription; Chromatine; Gene expression; Type I interferon; Colorectal cancer; Chaperone

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APA (6th Edition):

Detilleux, D. (2018). Functional characterization of the TRRAP pseudokinase and its chaperone TTT during transcriptional regulation in colorectal cancer : Etude du rôle de la pseudokinase TRRAP et de sa chaperone TTT sur la régulation de la transcription dans le cancer colorectal. (Doctoral Dissertation). Montpellier. Retrieved from http://www.theses.fr/2018MONTT092

Chicago Manual of Style (16th Edition):

Detilleux, Dylane. “Functional characterization of the TRRAP pseudokinase and its chaperone TTT during transcriptional regulation in colorectal cancer : Etude du rôle de la pseudokinase TRRAP et de sa chaperone TTT sur la régulation de la transcription dans le cancer colorectal.” 2018. Doctoral Dissertation, Montpellier. Accessed August 20, 2019. http://www.theses.fr/2018MONTT092.

MLA Handbook (7th Edition):

Detilleux, Dylane. “Functional characterization of the TRRAP pseudokinase and its chaperone TTT during transcriptional regulation in colorectal cancer : Etude du rôle de la pseudokinase TRRAP et de sa chaperone TTT sur la régulation de la transcription dans le cancer colorectal.” 2018. Web. 20 Aug 2019.

Vancouver:

Detilleux D. Functional characterization of the TRRAP pseudokinase and its chaperone TTT during transcriptional regulation in colorectal cancer : Etude du rôle de la pseudokinase TRRAP et de sa chaperone TTT sur la régulation de la transcription dans le cancer colorectal. [Internet] [Doctoral dissertation]. Montpellier; 2018. [cited 2019 Aug 20]. Available from: http://www.theses.fr/2018MONTT092.

Council of Science Editors:

Detilleux D. Functional characterization of the TRRAP pseudokinase and its chaperone TTT during transcriptional regulation in colorectal cancer : Etude du rôle de la pseudokinase TRRAP et de sa chaperone TTT sur la régulation de la transcription dans le cancer colorectal. [Doctoral Dissertation]. Montpellier; 2018. Available from: http://www.theses.fr/2018MONTT092

16. Perego, Jessica. Modulation pharmacologique des voies de signalisation des TLRs par le Guanabenz, un inhibiteur de la réponse au stress : Pharmacological inhibition of the TLRs signalling pathways by Guanabenz, an inhibitor of the stress response.

Degree: Docteur es, Biologie, 2016, Aix Marseille Université

On a récemment mis en évidence l'existence d'une étroite interconnexion entre la perception d'éléments d'origine microbienne (qui se fait à travers les récepteurs de l’immunité… (more)

Subjects/Keywords: Cellules dendritiques; Led; Gadd34; Interferon de type I; Choc septique; Gadd34; TLRs; Dendritic cells; Sle; Gadd34; Type I interferon; Septic shock; Gadd34; TLRs; 571

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APA (6th Edition):

Perego, J. (2016). Modulation pharmacologique des voies de signalisation des TLRs par le Guanabenz, un inhibiteur de la réponse au stress : Pharmacological inhibition of the TLRs signalling pathways by Guanabenz, an inhibitor of the stress response. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2016AIXM4066

Chicago Manual of Style (16th Edition):

Perego, Jessica. “Modulation pharmacologique des voies de signalisation des TLRs par le Guanabenz, un inhibiteur de la réponse au stress : Pharmacological inhibition of the TLRs signalling pathways by Guanabenz, an inhibitor of the stress response.” 2016. Doctoral Dissertation, Aix Marseille Université. Accessed August 20, 2019. http://www.theses.fr/2016AIXM4066.

MLA Handbook (7th Edition):

Perego, Jessica. “Modulation pharmacologique des voies de signalisation des TLRs par le Guanabenz, un inhibiteur de la réponse au stress : Pharmacological inhibition of the TLRs signalling pathways by Guanabenz, an inhibitor of the stress response.” 2016. Web. 20 Aug 2019.

Vancouver:

Perego J. Modulation pharmacologique des voies de signalisation des TLRs par le Guanabenz, un inhibiteur de la réponse au stress : Pharmacological inhibition of the TLRs signalling pathways by Guanabenz, an inhibitor of the stress response. [Internet] [Doctoral dissertation]. Aix Marseille Université 2016. [cited 2019 Aug 20]. Available from: http://www.theses.fr/2016AIXM4066.

Council of Science Editors:

Perego J. Modulation pharmacologique des voies de signalisation des TLRs par le Guanabenz, un inhibiteur de la réponse au stress : Pharmacological inhibition of the TLRs signalling pathways by Guanabenz, an inhibitor of the stress response. [Doctoral Dissertation]. Aix Marseille Université 2016. Available from: http://www.theses.fr/2016AIXM4066

17. Li, Zhi. Insights on type I IFN signaling and regulation : studies of disease-associated TYK2 variants and of the negative regulators USP18/ISG15 : Signalisation et régulation de l'interféron de type I : études de variants de TYK2 associés à des maladies et des régulateurs négatifs USP18 et ISG15.

Degree: Docteur es, Physiologie, Physiopathologie et Thérapeutique, 2017, Université Pierre et Marie Curie – Paris VI

L'action ubiquitaire de l'interféron de type I (IFN- alpha/beta , ici IFN) dans la physiologie et la pathologie est aujourd'hui certaine. Une réponse dérégulée à… (more)

Subjects/Keywords: Interferon de type I; Janus kinase; Maladies auto-Immunes; Polymorphisme nucleotidique simple; Usp18; Isg15; Type I interferon; Auto-immune disease; Janus kinase; 571.9

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APA (6th Edition):

Li, Z. (2017). Insights on type I IFN signaling and regulation : studies of disease-associated TYK2 variants and of the negative regulators USP18/ISG15 : Signalisation et régulation de l'interféron de type I : études de variants de TYK2 associés à des maladies et des régulateurs négatifs USP18 et ISG15. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2017PA066437

Chicago Manual of Style (16th Edition):

Li, Zhi. “Insights on type I IFN signaling and regulation : studies of disease-associated TYK2 variants and of the negative regulators USP18/ISG15 : Signalisation et régulation de l'interféron de type I : études de variants de TYK2 associés à des maladies et des régulateurs négatifs USP18 et ISG15.” 2017. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed August 20, 2019. http://www.theses.fr/2017PA066437.

MLA Handbook (7th Edition):

Li, Zhi. “Insights on type I IFN signaling and regulation : studies of disease-associated TYK2 variants and of the negative regulators USP18/ISG15 : Signalisation et régulation de l'interféron de type I : études de variants de TYK2 associés à des maladies et des régulateurs négatifs USP18 et ISG15.” 2017. Web. 20 Aug 2019.

Vancouver:

Li Z. Insights on type I IFN signaling and regulation : studies of disease-associated TYK2 variants and of the negative regulators USP18/ISG15 : Signalisation et régulation de l'interféron de type I : études de variants de TYK2 associés à des maladies et des régulateurs négatifs USP18 et ISG15. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. [cited 2019 Aug 20]. Available from: http://www.theses.fr/2017PA066437.

Council of Science Editors:

Li Z. Insights on type I IFN signaling and regulation : studies of disease-associated TYK2 variants and of the negative regulators USP18/ISG15 : Signalisation et régulation de l'interféron de type I : études de variants de TYK2 associés à des maladies et des régulateurs négatifs USP18 et ISG15. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. Available from: http://www.theses.fr/2017PA066437


University of Western Ontario

18. Hunt, Nina. Interferon-induced HERC5 is a novel and potent inhibitor of Ebola virus-like particle production.

Degree: 2018, University of Western Ontario

 Ebola’s severe pathogenesis can be attributed to its suppression of the Type I interferon (IFN) response, suggesting this pathway plays a role in restricting viral… (more)

Subjects/Keywords: Type I interferon response; HERC5; Ebola virus; antiviral; zinc finger antiviral protein; viral antagonism; Virology

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APA (6th Edition):

Hunt, N. (2018). Interferon-induced HERC5 is a novel and potent inhibitor of Ebola virus-like particle production. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/5922

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hunt, Nina. “Interferon-induced HERC5 is a novel and potent inhibitor of Ebola virus-like particle production.” 2018. Thesis, University of Western Ontario. Accessed August 20, 2019. https://ir.lib.uwo.ca/etd/5922.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hunt, Nina. “Interferon-induced HERC5 is a novel and potent inhibitor of Ebola virus-like particle production.” 2018. Web. 20 Aug 2019.

Vancouver:

Hunt N. Interferon-induced HERC5 is a novel and potent inhibitor of Ebola virus-like particle production. [Internet] [Thesis]. University of Western Ontario; 2018. [cited 2019 Aug 20]. Available from: https://ir.lib.uwo.ca/etd/5922.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hunt N. Interferon-induced HERC5 is a novel and potent inhibitor of Ebola virus-like particle production. [Thesis]. University of Western Ontario; 2018. Available from: https://ir.lib.uwo.ca/etd/5922

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

19. Chan, Jennifer. Pre-existing strain specific neutralising antibodies abrogates the induction of interferon type I and cytotoxic T cell responses to subsequent homotypic influenza A virus challenge.

Degree: 2013, University of Adelaide

 Current inactivated influenza vaccines target the generation of influenza-specific antibodies to provide homotypic protection. However, little is known about the effects of annual vaccinations on… (more)

Subjects/Keywords: immune response; virus challenge; B cells; influenza A virus; T cells; antibodies; interferon type I

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APA (6th Edition):

Chan, J. (2013). Pre-existing strain specific neutralising antibodies abrogates the induction of interferon type I and cytotoxic T cell responses to subsequent homotypic influenza A virus challenge. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/90751

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chan, Jennifer. “Pre-existing strain specific neutralising antibodies abrogates the induction of interferon type I and cytotoxic T cell responses to subsequent homotypic influenza A virus challenge.” 2013. Thesis, University of Adelaide. Accessed August 20, 2019. http://hdl.handle.net/2440/90751.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chan, Jennifer. “Pre-existing strain specific neutralising antibodies abrogates the induction of interferon type I and cytotoxic T cell responses to subsequent homotypic influenza A virus challenge.” 2013. Web. 20 Aug 2019.

Vancouver:

Chan J. Pre-existing strain specific neutralising antibodies abrogates the induction of interferon type I and cytotoxic T cell responses to subsequent homotypic influenza A virus challenge. [Internet] [Thesis]. University of Adelaide; 2013. [cited 2019 Aug 20]. Available from: http://hdl.handle.net/2440/90751.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chan J. Pre-existing strain specific neutralising antibodies abrogates the induction of interferon type I and cytotoxic T cell responses to subsequent homotypic influenza A virus challenge. [Thesis]. University of Adelaide; 2013. Available from: http://hdl.handle.net/2440/90751

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

20. Ranganath, Nischal. Oncolytic Viruses as a Potential Approach to Eliminate Cells That Constitute the Latent HIV Reservoir .

Degree: 2018, University of Ottawa

 HIV infection represents a major health and socioeconomic challenge worldwide. Despite significant advances in therapy, a cure for HIV continues to be elusive. The design… (more)

Subjects/Keywords: Latent HIV reservoir; Oncolytic viruses; Type I interferon; Vesicular stomatitis virus; Marba virus

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APA (6th Edition):

Ranganath, N. (2018). Oncolytic Viruses as a Potential Approach to Eliminate Cells That Constitute the Latent HIV Reservoir . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/37355

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ranganath, Nischal. “Oncolytic Viruses as a Potential Approach to Eliminate Cells That Constitute the Latent HIV Reservoir .” 2018. Thesis, University of Ottawa. Accessed August 20, 2019. http://hdl.handle.net/10393/37355.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ranganath, Nischal. “Oncolytic Viruses as a Potential Approach to Eliminate Cells That Constitute the Latent HIV Reservoir .” 2018. Web. 20 Aug 2019.

Vancouver:

Ranganath N. Oncolytic Viruses as a Potential Approach to Eliminate Cells That Constitute the Latent HIV Reservoir . [Internet] [Thesis]. University of Ottawa; 2018. [cited 2019 Aug 20]. Available from: http://hdl.handle.net/10393/37355.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ranganath N. Oncolytic Viruses as a Potential Approach to Eliminate Cells That Constitute the Latent HIV Reservoir . [Thesis]. University of Ottawa; 2018. Available from: http://hdl.handle.net/10393/37355

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

21. Machado Parrula, Maria Cecilia. Measles Virotherapy in Adult T cell Leukemia.

Degree: PhD, Veterinary Biosciences, 2009, The Ohio State University

 Adult T cell leukemia (ATL) is a proliferative disorder of CD4+ T cells caused by human T cell lymphotropic type I virus infection. ATL is… (more)

Subjects/Keywords: Oncology; Virology; HTLV-1; ATL; measles; immunodeficient mice; type I interferon; cotton rat

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APA (6th Edition):

Machado Parrula, M. C. (2009). Measles Virotherapy in Adult T cell Leukemia. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1261413581

Chicago Manual of Style (16th Edition):

Machado Parrula, Maria Cecilia. “Measles Virotherapy in Adult T cell Leukemia.” 2009. Doctoral Dissertation, The Ohio State University. Accessed August 20, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1261413581.

MLA Handbook (7th Edition):

Machado Parrula, Maria Cecilia. “Measles Virotherapy in Adult T cell Leukemia.” 2009. Web. 20 Aug 2019.

Vancouver:

Machado Parrula MC. Measles Virotherapy in Adult T cell Leukemia. [Internet] [Doctoral dissertation]. The Ohio State University; 2009. [cited 2019 Aug 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1261413581.

Council of Science Editors:

Machado Parrula MC. Measles Virotherapy in Adult T cell Leukemia. [Doctoral Dissertation]. The Ohio State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1261413581


UCLA

22. York, Autumn Gabrielle. Identification of a cholesterol metabolic-type I interferon inflammatory circuit.

Degree: Molecular and Medical Pharmacology, 2015, UCLA

 Cellular lipid requirements are achieved through a combination of biosynthesis and import programs. To date, the molecular mechanisms that regulate whether a cell preferentially scavenges… (more)

Subjects/Keywords: Immunology; Biochemistry; Innate Immunity; Lipid Metabolism; STING; Toll-like Receptors; Type I Interferon

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APA (6th Edition):

York, A. G. (2015). Identification of a cholesterol metabolic-type I interferon inflammatory circuit. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/7z09j9gj

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

York, Autumn Gabrielle. “Identification of a cholesterol metabolic-type I interferon inflammatory circuit.” 2015. Thesis, UCLA. Accessed August 20, 2019. http://www.escholarship.org/uc/item/7z09j9gj.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

York, Autumn Gabrielle. “Identification of a cholesterol metabolic-type I interferon inflammatory circuit.” 2015. Web. 20 Aug 2019.

Vancouver:

York AG. Identification of a cholesterol metabolic-type I interferon inflammatory circuit. [Internet] [Thesis]. UCLA; 2015. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/7z09j9gj.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

York AG. Identification of a cholesterol metabolic-type I interferon inflammatory circuit. [Thesis]. UCLA; 2015. Available from: http://www.escholarship.org/uc/item/7z09j9gj

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

23. Zhou, Zhicheng, Mr. CHARACTERIZATION OF THE ROLES OF CARMA3 AND BCL10 IN VIRUS-TRIGGERED RIG-I/MAVS SIGNALING PATHWAY.

Degree: PhD, 2015, Texas Medical Center

  After RNA virus infection, the innate immunity utilizes RIG-I family of receptors in the cytoplasm to initiate the production of type-I IFNs and proinflammatory… (more)

Subjects/Keywords: CARMA3; BCL10; RIG-I/MAVS signaling pathway; Type I interferon; Influenza virus; VSV; Poly(I:C); DsRNA;

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APA (6th Edition):

Zhou, Zhicheng, M. (2015). CHARACTERIZATION OF THE ROLES OF CARMA3 AND BCL10 IN VIRUS-TRIGGERED RIG-I/MAVS SIGNALING PATHWAY. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/546

Chicago Manual of Style (16th Edition):

Zhou, Zhicheng, Mr. “CHARACTERIZATION OF THE ROLES OF CARMA3 AND BCL10 IN VIRUS-TRIGGERED RIG-I/MAVS SIGNALING PATHWAY.” 2015. Doctoral Dissertation, Texas Medical Center. Accessed August 20, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/546.

MLA Handbook (7th Edition):

Zhou, Zhicheng, Mr. “CHARACTERIZATION OF THE ROLES OF CARMA3 AND BCL10 IN VIRUS-TRIGGERED RIG-I/MAVS SIGNALING PATHWAY.” 2015. Web. 20 Aug 2019.

Vancouver:

Zhou, Zhicheng M. CHARACTERIZATION OF THE ROLES OF CARMA3 AND BCL10 IN VIRUS-TRIGGERED RIG-I/MAVS SIGNALING PATHWAY. [Internet] [Doctoral dissertation]. Texas Medical Center; 2015. [cited 2019 Aug 20]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/546.

Council of Science Editors:

Zhou, Zhicheng M. CHARACTERIZATION OF THE ROLES OF CARMA3 AND BCL10 IN VIRUS-TRIGGERED RIG-I/MAVS SIGNALING PATHWAY. [Doctoral Dissertation]. Texas Medical Center; 2015. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/546


University of Toronto

24. Wang, Kuan Chung. Dichotomy of TNF Family Ligands Expression on Classical Dendritic Cells and Monocyte-derived Antigen Presenting Cells During Viral Infection.

Degree: 2018, University of Toronto

Tumor necrosis factor receptor (TNFR) superfamily members, including 4-1BB, OX40, GITR and CD27 contribute to T cell survival during viral infection. However, the cell types… (more)

Subjects/Keywords: Antigen presenting cell; B7 family ligands; lymphoytic choriomeningitis (LCMV) clone 13 infection; Tumor necrosis factor receptor (TNFR) superfamily members; type II interferon; type I interferon; 0982

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APA (6th Edition):

Wang, K. C. (2018). Dichotomy of TNF Family Ligands Expression on Classical Dendritic Cells and Monocyte-derived Antigen Presenting Cells During Viral Infection. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91345

Chicago Manual of Style (16th Edition):

Wang, Kuan Chung. “Dichotomy of TNF Family Ligands Expression on Classical Dendritic Cells and Monocyte-derived Antigen Presenting Cells During Viral Infection.” 2018. Masters Thesis, University of Toronto. Accessed August 20, 2019. http://hdl.handle.net/1807/91345.

MLA Handbook (7th Edition):

Wang, Kuan Chung. “Dichotomy of TNF Family Ligands Expression on Classical Dendritic Cells and Monocyte-derived Antigen Presenting Cells During Viral Infection.” 2018. Web. 20 Aug 2019.

Vancouver:

Wang KC. Dichotomy of TNF Family Ligands Expression on Classical Dendritic Cells and Monocyte-derived Antigen Presenting Cells During Viral Infection. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2019 Aug 20]. Available from: http://hdl.handle.net/1807/91345.

Council of Science Editors:

Wang KC. Dichotomy of TNF Family Ligands Expression on Classical Dendritic Cells and Monocyte-derived Antigen Presenting Cells During Viral Infection. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91345

25. Futsch, Nicolas. Caractérisation de l’activation des cellules dendritiques plasmacytoïdes par les virus HTLV-1 et HTLV-2 et de son importance dans la symptomatologie viro-induite : Characterization of the plasmacytoid dendritic cells activation by HTLV-1 or HTLV-2 and its importance on the viral-associated pathogenesis.

Degree: Docteur es, Sciences de la Vie, 2018, Lyon

Le virus T-lymphotrope humain de type 1 (HTLV-1) est l’agent étiologique de deux principales pathologies : la leucémie/lymphome à cellules T de l’adulte (ATLL) et… (more)

Subjects/Keywords: HTLV; Cellule dendritique plasmacytoïde; Interféron Type I; ATLL; Myélopathie associée au virus HTLV-1; HTLV; Plasmacytoid dendritic cells; Interferon Type I; ATLL; HTLV-I-Associated Myelopathy-Tropical Spastic Paraparesis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Futsch, N. (2018). Caractérisation de l’activation des cellules dendritiques plasmacytoïdes par les virus HTLV-1 et HTLV-2 et de son importance dans la symptomatologie viro-induite : Characterization of the plasmacytoid dendritic cells activation by HTLV-1 or HTLV-2 and its importance on the viral-associated pathogenesis. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2018LYSEN067

Chicago Manual of Style (16th Edition):

Futsch, Nicolas. “Caractérisation de l’activation des cellules dendritiques plasmacytoïdes par les virus HTLV-1 et HTLV-2 et de son importance dans la symptomatologie viro-induite : Characterization of the plasmacytoid dendritic cells activation by HTLV-1 or HTLV-2 and its importance on the viral-associated pathogenesis.” 2018. Doctoral Dissertation, Lyon. Accessed August 20, 2019. http://www.theses.fr/2018LYSEN067.

MLA Handbook (7th Edition):

Futsch, Nicolas. “Caractérisation de l’activation des cellules dendritiques plasmacytoïdes par les virus HTLV-1 et HTLV-2 et de son importance dans la symptomatologie viro-induite : Characterization of the plasmacytoid dendritic cells activation by HTLV-1 or HTLV-2 and its importance on the viral-associated pathogenesis.” 2018. Web. 20 Aug 2019.

Vancouver:

Futsch N. Caractérisation de l’activation des cellules dendritiques plasmacytoïdes par les virus HTLV-1 et HTLV-2 et de son importance dans la symptomatologie viro-induite : Characterization of the plasmacytoid dendritic cells activation by HTLV-1 or HTLV-2 and its importance on the viral-associated pathogenesis. [Internet] [Doctoral dissertation]. Lyon; 2018. [cited 2019 Aug 20]. Available from: http://www.theses.fr/2018LYSEN067.

Council of Science Editors:

Futsch N. Caractérisation de l’activation des cellules dendritiques plasmacytoïdes par les virus HTLV-1 et HTLV-2 et de son importance dans la symptomatologie viro-induite : Characterization of the plasmacytoid dendritic cells activation by HTLV-1 or HTLV-2 and its importance on the viral-associated pathogenesis. [Doctoral Dissertation]. Lyon; 2018. Available from: http://www.theses.fr/2018LYSEN067

26. Khoryati, Liliane. Propriétés immuno-modulatrices des IgE dans le lupus érythémateux systémique : impact sur la sécrétion d’interféron de type I par les cellules dendritiques plasmacytoïdes : Immunomodulatory properties of IgE in systemic lupus erythematosus : impact on type I interferon secretion by plasmacytoid dendritic cells.

Degree: Docteur es, Microbiologie-Immunologie, 2014, Bordeaux

Les cellules dendritiques plasmacytoïdes (pDCs) sont caractérisées par leur capacité unique de sécrétion massive d’interféron de type I (IFN-I) suite à la stimulation des Tolllike… (more)

Subjects/Keywords: Lupus érythémateux systémique; Cellule dendritique plasmacytoïde; Interféron de type I; Immunoglobuline E; Systemic lupus erythematosus; Plasmacytoid dendritic cell; Interferon type I; Immunoglobulin E

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Khoryati, L. (2014). Propriétés immuno-modulatrices des IgE dans le lupus érythémateux systémique : impact sur la sécrétion d’interféron de type I par les cellules dendritiques plasmacytoïdes : Immunomodulatory properties of IgE in systemic lupus erythematosus : impact on type I interferon secretion by plasmacytoid dendritic cells. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2014BORD0159

Chicago Manual of Style (16th Edition):

Khoryati, Liliane. “Propriétés immuno-modulatrices des IgE dans le lupus érythémateux systémique : impact sur la sécrétion d’interféron de type I par les cellules dendritiques plasmacytoïdes : Immunomodulatory properties of IgE in systemic lupus erythematosus : impact on type I interferon secretion by plasmacytoid dendritic cells.” 2014. Doctoral Dissertation, Bordeaux. Accessed August 20, 2019. http://www.theses.fr/2014BORD0159.

MLA Handbook (7th Edition):

Khoryati, Liliane. “Propriétés immuno-modulatrices des IgE dans le lupus érythémateux systémique : impact sur la sécrétion d’interféron de type I par les cellules dendritiques plasmacytoïdes : Immunomodulatory properties of IgE in systemic lupus erythematosus : impact on type I interferon secretion by plasmacytoid dendritic cells.” 2014. Web. 20 Aug 2019.

Vancouver:

Khoryati L. Propriétés immuno-modulatrices des IgE dans le lupus érythémateux systémique : impact sur la sécrétion d’interféron de type I par les cellules dendritiques plasmacytoïdes : Immunomodulatory properties of IgE in systemic lupus erythematosus : impact on type I interferon secretion by plasmacytoid dendritic cells. [Internet] [Doctoral dissertation]. Bordeaux; 2014. [cited 2019 Aug 20]. Available from: http://www.theses.fr/2014BORD0159.

Council of Science Editors:

Khoryati L. Propriétés immuno-modulatrices des IgE dans le lupus érythémateux systémique : impact sur la sécrétion d’interféron de type I par les cellules dendritiques plasmacytoïdes : Immunomodulatory properties of IgE in systemic lupus erythematosus : impact on type I interferon secretion by plasmacytoid dendritic cells. [Doctoral Dissertation]. Bordeaux; 2014. Available from: http://www.theses.fr/2014BORD0159

27. Chmiest, Daniela. Rôle du tri endosomal dans le trafic du récepteur de l'IFN de type I et dans la voie de signalisation JAK/STAT. : Role of endosomal sorting in IFN I receptor trafficking and JAK/STAT signaling.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2015, Paris Saclay

La voie JAK / STAT est une voie majeure de signalisation intracellulaire, activée par plusieurs cytokines, y compris par les interférons (IFNs). Mon laboratoire a… (more)

Subjects/Keywords: Ifnar; Rétromère; Signalisation JAK / STAT; Endocytose; Trafic intracellulaire; Interféron de type I; Ifnar; Retromer; JAK/STAT signaling; Endocytosis; Intracellular trafficking; Type I interferon

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chmiest, D. (2015). Rôle du tri endosomal dans le trafic du récepteur de l'IFN de type I et dans la voie de signalisation JAK/STAT. : Role of endosomal sorting in IFN I receptor trafficking and JAK/STAT signaling. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2015SACLS108

Chicago Manual of Style (16th Edition):

Chmiest, Daniela. “Rôle du tri endosomal dans le trafic du récepteur de l'IFN de type I et dans la voie de signalisation JAK/STAT. : Role of endosomal sorting in IFN I receptor trafficking and JAK/STAT signaling.” 2015. Doctoral Dissertation, Paris Saclay. Accessed August 20, 2019. http://www.theses.fr/2015SACLS108.

MLA Handbook (7th Edition):

Chmiest, Daniela. “Rôle du tri endosomal dans le trafic du récepteur de l'IFN de type I et dans la voie de signalisation JAK/STAT. : Role of endosomal sorting in IFN I receptor trafficking and JAK/STAT signaling.” 2015. Web. 20 Aug 2019.

Vancouver:

Chmiest D. Rôle du tri endosomal dans le trafic du récepteur de l'IFN de type I et dans la voie de signalisation JAK/STAT. : Role of endosomal sorting in IFN I receptor trafficking and JAK/STAT signaling. [Internet] [Doctoral dissertation]. Paris Saclay; 2015. [cited 2019 Aug 20]. Available from: http://www.theses.fr/2015SACLS108.

Council of Science Editors:

Chmiest D. Rôle du tri endosomal dans le trafic du récepteur de l'IFN de type I et dans la voie de signalisation JAK/STAT. : Role of endosomal sorting in IFN I receptor trafficking and JAK/STAT signaling. [Doctoral Dissertation]. Paris Saclay; 2015. Available from: http://www.theses.fr/2015SACLS108

28. Tokarz, Sara. Characterization of the SCFSkp2 substrate, UBP43, an ISG15 isopeptidase.

Degree: PhD, 2005, Oregon Health Sciences University

Subjects/Keywords: Ubiquitin-Protein Ligases; Interferon Type I; F-Box Proteins

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tokarz, S. (2005). Characterization of the SCFSkp2 substrate, UBP43, an ISG15 isopeptidase. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4V1232T ; http://digitalcommons.ohsu.edu/etd/3012

Chicago Manual of Style (16th Edition):

Tokarz, Sara. “Characterization of the SCFSkp2 substrate, UBP43, an ISG15 isopeptidase.” 2005. Doctoral Dissertation, Oregon Health Sciences University. Accessed August 20, 2019. doi:10.6083/M4V1232T ; http://digitalcommons.ohsu.edu/etd/3012.

MLA Handbook (7th Edition):

Tokarz, Sara. “Characterization of the SCFSkp2 substrate, UBP43, an ISG15 isopeptidase.” 2005. Web. 20 Aug 2019.

Vancouver:

Tokarz S. Characterization of the SCFSkp2 substrate, UBP43, an ISG15 isopeptidase. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2005. [cited 2019 Aug 20]. Available from: doi:10.6083/M4V1232T ; http://digitalcommons.ohsu.edu/etd/3012.

Council of Science Editors:

Tokarz S. Characterization of the SCFSkp2 substrate, UBP43, an ISG15 isopeptidase. [Doctoral Dissertation]. Oregon Health Sciences University; 2005. Available from: doi:10.6083/M4V1232T ; http://digitalcommons.ohsu.edu/etd/3012

29. Thompson, Mikayla R. Dissecting the Role of Cytosolic Nucleic Acid Sensors in the Type I Interferon Response to Herpes Simplex Virus-1 and other Ligands: A Dissertation.

Degree: Interdisciplinary Graduate Program, Medicine, 2014, U of Massachusetts : Med

  The innate immune system provides the first line of defense against infection. Pathogens are detected though a variety of Pattern Recognition Receptors (PRRs), which… (more)

Subjects/Keywords: Adaptive Immunity; Herpes Simplex; Immune System; Interferon Type I; Ligands; Immunologic Receptors; Receptors; Pattern Recognition; Immunity; Immunology of Infectious Disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Thompson, M. R. (2014). Dissecting the Role of Cytosolic Nucleic Acid Sensors in the Type I Interferon Response to Herpes Simplex Virus-1 and other Ligands: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/708

Chicago Manual of Style (16th Edition):

Thompson, Mikayla R. “Dissecting the Role of Cytosolic Nucleic Acid Sensors in the Type I Interferon Response to Herpes Simplex Virus-1 and other Ligands: A Dissertation.” 2014. Doctoral Dissertation, U of Massachusetts : Med. Accessed August 20, 2019. http://escholarship.umassmed.edu/gsbs_diss/708.

MLA Handbook (7th Edition):

Thompson, Mikayla R. “Dissecting the Role of Cytosolic Nucleic Acid Sensors in the Type I Interferon Response to Herpes Simplex Virus-1 and other Ligands: A Dissertation.” 2014. Web. 20 Aug 2019.

Vancouver:

Thompson MR. Dissecting the Role of Cytosolic Nucleic Acid Sensors in the Type I Interferon Response to Herpes Simplex Virus-1 and other Ligands: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2014. [cited 2019 Aug 20]. Available from: http://escholarship.umassmed.edu/gsbs_diss/708.

Council of Science Editors:

Thompson MR. Dissecting the Role of Cytosolic Nucleic Acid Sensors in the Type I Interferon Response to Herpes Simplex Virus-1 and other Ligands: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2014. Available from: http://escholarship.umassmed.edu/gsbs_diss/708

30. Urban, Stina L. The Role of Signal 3 Cytokine Timing in CD8 T Cell Activation: A Dissertation.

Degree: Immunology and Microbiology, Pathology, 2015, U of Massachusetts : Med

  During an acute virus infection, antigen-specific CD8 T cells undergo clonal expansion and differentiation into effector cells in order to control the infection. Efficient… (more)

Subjects/Keywords: CD8-Positive T-Lymphocytes; Cytokines; Interleukin-12; Lymphocyte Activation; Interferon Type I; Immunology and Infectious Disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Urban, S. L. (2015). The Role of Signal 3 Cytokine Timing in CD8 T Cell Activation: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/788

Chicago Manual of Style (16th Edition):

Urban, Stina L. “The Role of Signal 3 Cytokine Timing in CD8 T Cell Activation: A Dissertation.” 2015. Doctoral Dissertation, U of Massachusetts : Med. Accessed August 20, 2019. http://escholarship.umassmed.edu/gsbs_diss/788.

MLA Handbook (7th Edition):

Urban, Stina L. “The Role of Signal 3 Cytokine Timing in CD8 T Cell Activation: A Dissertation.” 2015. Web. 20 Aug 2019.

Vancouver:

Urban SL. The Role of Signal 3 Cytokine Timing in CD8 T Cell Activation: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2015. [cited 2019 Aug 20]. Available from: http://escholarship.umassmed.edu/gsbs_diss/788.

Council of Science Editors:

Urban SL. The Role of Signal 3 Cytokine Timing in CD8 T Cell Activation: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2015. Available from: http://escholarship.umassmed.edu/gsbs_diss/788

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