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You searched for subject:(Insulin secretion). Showing records 1 – 30 of 243 total matches.

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University of Manchester

1. Basalem, Osama Salem. Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line.

Degree: 2016, University of Manchester

Introduction: Congenital Hyperinsulinism (CHI) is a rare neonatal syndrome associated with continuous inappropriate insulin secretion by the pancreatic β-cell in the presence of recurring hypoglycemia.… (more)

Subjects/Keywords: Insulin secretion; Rapamycin; mTOR

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Basalem, O. S. (2016). Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305154

Chicago Manual of Style (16th Edition):

Basalem, Osama Salem. “Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line.” 2016. Doctoral Dissertation, University of Manchester. Accessed April 13, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305154.

MLA Handbook (7th Edition):

Basalem, Osama Salem. “Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line.” 2016. Web. 13 Apr 2021.

Vancouver:

Basalem OS. Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 Apr 13]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305154.

Council of Science Editors:

Basalem OS. Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305154


Univerzitet u Beogradu

2. Cvijović, Goran M., 1971-. Sekrecija insulina i insulinska senzitivnost perifernih tkiva pre i nakon radikalnog lečenja primarnog hiperparatireoidizma.

Degree: Medicinski fakultet, 2016, Univerzitet u Beogradu

INTERNA MEDICINA - ENDOKRINOLOGIJA / INTERNAL MEDICINE - ENDOCRINOLOGY

Uvod i cilj: Prethodno je uočeno postojanje insulinske rezistencije i povećana prevalenca oštećene tolerancije na glikozu… (more)

Subjects/Keywords: primary hyperparathyroidism; insulin sensitivity; insulin secretion

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APA (6th Edition):

Cvijović, Goran M., 1. (2016). Sekrecija insulina i insulinska senzitivnost perifernih tkiva pre i nakon radikalnog lečenja primarnog hiperparatireoidizma. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:12592/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cvijović, Goran M., 1971-. “Sekrecija insulina i insulinska senzitivnost perifernih tkiva pre i nakon radikalnog lečenja primarnog hiperparatireoidizma.” 2016. Thesis, Univerzitet u Beogradu. Accessed April 13, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:12592/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cvijović, Goran M., 1971-. “Sekrecija insulina i insulinska senzitivnost perifernih tkiva pre i nakon radikalnog lečenja primarnog hiperparatireoidizma.” 2016. Web. 13 Apr 2021.

Vancouver:

Cvijović, Goran M. 1. Sekrecija insulina i insulinska senzitivnost perifernih tkiva pre i nakon radikalnog lečenja primarnog hiperparatireoidizma. [Internet] [Thesis]. Univerzitet u Beogradu; 2016. [cited 2021 Apr 13]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:12592/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cvijović, Goran M. 1. Sekrecija insulina i insulinska senzitivnost perifernih tkiva pre i nakon radikalnog lečenja primarnog hiperparatireoidizma. [Thesis]. Univerzitet u Beogradu; 2016. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:12592/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

3. Kloc, Noreen. Insulin Dynamic Measures and Weight Change.

Degree: MPH, Public Health, 2016, Georgia State University

  ABSTRACT Insulin Dynamic Measures and Weight Change By Noreen Kloc B.S. Computer Information Technology, Purdue University December 7, 2015 INTRODUCTION: Weight gain and obesity… (more)

Subjects/Keywords: insulin resistance; insulin sensitivity; insulin secretion; weight; metabolic syndrome; diabetes

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APA (6th Edition):

Kloc, N. (2016). Insulin Dynamic Measures and Weight Change. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/iph_theses/437

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kloc, Noreen. “Insulin Dynamic Measures and Weight Change.” 2016. Thesis, Georgia State University. Accessed April 13, 2021. https://scholarworks.gsu.edu/iph_theses/437.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kloc, Noreen. “Insulin Dynamic Measures and Weight Change.” 2016. Web. 13 Apr 2021.

Vancouver:

Kloc N. Insulin Dynamic Measures and Weight Change. [Internet] [Thesis]. Georgia State University; 2016. [cited 2021 Apr 13]. Available from: https://scholarworks.gsu.edu/iph_theses/437.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kloc N. Insulin Dynamic Measures and Weight Change. [Thesis]. Georgia State University; 2016. Available from: https://scholarworks.gsu.edu/iph_theses/437

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Moin Abu Saleh Md. Neuroendocrine regulatory peptide-2 stimulates glucose-induced insulin secretion in vivo and in vitro.

Degree: 博士(医学), 2015, University of Miyazaki / 宮崎大学

以下に掲載:Biochemical and Biophysical Research Communications. November 2012, Volume 428, Issue 4, Pages 512–517, doi:10.1016/j.bbrc.2012.10.073.

Subjects/Keywords: NERP-2; Insulin secretion; Ca^<; 2+>

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APA (6th Edition):

Md., M. A. S. (2015). Neuroendocrine regulatory peptide-2 stimulates glucose-induced insulin secretion in vivo and in vitro. (Thesis). University of Miyazaki / 宮崎大学. Retrieved from http://hdl.handle.net/10458/5418

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Md., Moin Abu Saleh. “Neuroendocrine regulatory peptide-2 stimulates glucose-induced insulin secretion in vivo and in vitro.” 2015. Thesis, University of Miyazaki / 宮崎大学. Accessed April 13, 2021. http://hdl.handle.net/10458/5418.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Md., Moin Abu Saleh. “Neuroendocrine regulatory peptide-2 stimulates glucose-induced insulin secretion in vivo and in vitro.” 2015. Web. 13 Apr 2021.

Vancouver:

Md. MAS. Neuroendocrine regulatory peptide-2 stimulates glucose-induced insulin secretion in vivo and in vitro. [Internet] [Thesis]. University of Miyazaki / 宮崎大学; 2015. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/10458/5418.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Md. MAS. Neuroendocrine regulatory peptide-2 stimulates glucose-induced insulin secretion in vivo and in vitro. [Thesis]. University of Miyazaki / 宮崎大学; 2015. Available from: http://hdl.handle.net/10458/5418

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. El Azzouny, Mahmoud. Development and Application of Metabolomics Techniques to Improve Understanding of Glucose and Fatty Acid Metabolism in ß-cells and their Role in Insulin Secretion.

Degree: PhD, Chemistry, 2014, University of Michigan

 The primary function of pancreatic beta cells is to secrete insulin in response to glucose metabolism. Decline in beta cell function contributes to the development… (more)

Subjects/Keywords: Metabolomics; Glucose Stimulated Insulin Secretion; Chemistry; Science

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APA (6th Edition):

El Azzouny, M. (2014). Development and Application of Metabolomics Techniques to Improve Understanding of Glucose and Fatty Acid Metabolism in ß-cells and their Role in Insulin Secretion. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/107168

Chicago Manual of Style (16th Edition):

El Azzouny, Mahmoud. “Development and Application of Metabolomics Techniques to Improve Understanding of Glucose and Fatty Acid Metabolism in ß-cells and their Role in Insulin Secretion.” 2014. Doctoral Dissertation, University of Michigan. Accessed April 13, 2021. http://hdl.handle.net/2027.42/107168.

MLA Handbook (7th Edition):

El Azzouny, Mahmoud. “Development and Application of Metabolomics Techniques to Improve Understanding of Glucose and Fatty Acid Metabolism in ß-cells and their Role in Insulin Secretion.” 2014. Web. 13 Apr 2021.

Vancouver:

El Azzouny M. Development and Application of Metabolomics Techniques to Improve Understanding of Glucose and Fatty Acid Metabolism in ß-cells and their Role in Insulin Secretion. [Internet] [Doctoral dissertation]. University of Michigan; 2014. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/2027.42/107168.

Council of Science Editors:

El Azzouny M. Development and Application of Metabolomics Techniques to Improve Understanding of Glucose and Fatty Acid Metabolism in ß-cells and their Role in Insulin Secretion. [Doctoral Dissertation]. University of Michigan; 2014. Available from: http://hdl.handle.net/2027.42/107168


University of Toronto

6. Koo, Ellen. Syntaxin-3 Regulates Biphasic Glucose Stimulated Insulin Secretion in the Pancreatic Beta Cell.

Degree: 2010, University of Toronto

Our study aims to investigate the role of Syntaxin-3 in glucose stimulated insulin secretion (GSIS) and how it regulates the recruitment to plasma membrane and/or… (more)

Subjects/Keywords: Insulin Secretion; Syntaxin; SNAREs; Exocytosis; 0719

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APA (6th Edition):

Koo, E. (2010). Syntaxin-3 Regulates Biphasic Glucose Stimulated Insulin Secretion in the Pancreatic Beta Cell. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25732

Chicago Manual of Style (16th Edition):

Koo, Ellen. “Syntaxin-3 Regulates Biphasic Glucose Stimulated Insulin Secretion in the Pancreatic Beta Cell.” 2010. Masters Thesis, University of Toronto. Accessed April 13, 2021. http://hdl.handle.net/1807/25732.

MLA Handbook (7th Edition):

Koo, Ellen. “Syntaxin-3 Regulates Biphasic Glucose Stimulated Insulin Secretion in the Pancreatic Beta Cell.” 2010. Web. 13 Apr 2021.

Vancouver:

Koo E. Syntaxin-3 Regulates Biphasic Glucose Stimulated Insulin Secretion in the Pancreatic Beta Cell. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1807/25732.

Council of Science Editors:

Koo E. Syntaxin-3 Regulates Biphasic Glucose Stimulated Insulin Secretion in the Pancreatic Beta Cell. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25732

7. Loweth, Anne C. Phospholipase A←2 expression and mechanisms of cell death in the endocrine pancreas.

Degree: PhD, 1995, Keele University

Subjects/Keywords: 572.8; Insulin secretion

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APA (6th Edition):

Loweth, A. C. (1995). Phospholipase A←2 expression and mechanisms of cell death in the endocrine pancreas. (Doctoral Dissertation). Keele University. Retrieved from https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.283255

Chicago Manual of Style (16th Edition):

Loweth, Anne C. “Phospholipase A←2 expression and mechanisms of cell death in the endocrine pancreas.” 1995. Doctoral Dissertation, Keele University. Accessed April 13, 2021. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.283255.

MLA Handbook (7th Edition):

Loweth, Anne C. “Phospholipase A←2 expression and mechanisms of cell death in the endocrine pancreas.” 1995. Web. 13 Apr 2021.

Vancouver:

Loweth AC. Phospholipase A←2 expression and mechanisms of cell death in the endocrine pancreas. [Internet] [Doctoral dissertation]. Keele University; 1995. [cited 2021 Apr 13]. Available from: https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.283255.

Council of Science Editors:

Loweth AC. Phospholipase A←2 expression and mechanisms of cell death in the endocrine pancreas. [Doctoral Dissertation]. Keele University; 1995. Available from: https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.283255


University of Edinburgh

8. Liu, Xiaoxia. Regulation and function of 11β-hydroxysteroid dehydrogenase (11β-HSD1) in pancreatic β-cells.

Degree: PhD, 2011, University of Edinburgh

 Diabetes Mellitus is characterized by high blood sugar and is caused by resistance to (type 2) or insufficiency of (type 1) the pancreatic β-cell hormone… (more)

Subjects/Keywords: 616.4; 11ß-HSD1; ß-cells; insulin; secretion

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APA (6th Edition):

Liu, X. (2011). Regulation and function of 11β-hydroxysteroid dehydrogenase (11β-HSD1) in pancreatic β-cells. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/5582

Chicago Manual of Style (16th Edition):

Liu, Xiaoxia. “Regulation and function of 11β-hydroxysteroid dehydrogenase (11β-HSD1) in pancreatic β-cells.” 2011. Doctoral Dissertation, University of Edinburgh. Accessed April 13, 2021. http://hdl.handle.net/1842/5582.

MLA Handbook (7th Edition):

Liu, Xiaoxia. “Regulation and function of 11β-hydroxysteroid dehydrogenase (11β-HSD1) in pancreatic β-cells.” 2011. Web. 13 Apr 2021.

Vancouver:

Liu X. Regulation and function of 11β-hydroxysteroid dehydrogenase (11β-HSD1) in pancreatic β-cells. [Internet] [Doctoral dissertation]. University of Edinburgh; 2011. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1842/5582.

Council of Science Editors:

Liu X. Regulation and function of 11β-hydroxysteroid dehydrogenase (11β-HSD1) in pancreatic β-cells. [Doctoral Dissertation]. University of Edinburgh; 2011. Available from: http://hdl.handle.net/1842/5582


University of New South Wales

9. Pearson, Gemma. THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS.

Degree: Garvan Institute of Medical Research, 2014, University of New South Wales

 Glucose-stimulated insulin secretion (GSIS) from pancreatic β-cells is vital for whole-bodyglucose homeostasis, and loss of β-cell function can result in type 2 diabetes(T2D), a major… (more)

Subjects/Keywords: Lipids; Pancreatic beta cells; Insulin secretion; Lysosomes

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APA (6th Edition):

Pearson, G. (2014). THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Pearson, Gemma. “THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS.” 2014. Doctoral Dissertation, University of New South Wales. Accessed April 13, 2021. http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true.

MLA Handbook (7th Edition):

Pearson, Gemma. “THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS.” 2014. Web. 13 Apr 2021.

Vancouver:

Pearson G. THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS. [Internet] [Doctoral dissertation]. University of New South Wales; 2014. [cited 2021 Apr 13]. Available from: http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true.

Council of Science Editors:

Pearson G. THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS. [Doctoral Dissertation]. University of New South Wales; 2014. Available from: http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true


Wright State University

10. Alshahrani, Saeed. Role of Na+K+2Cl¿¿¿¿¿¿ Co-transporters in Insulin Secretion.

Degree: MS, Pharmacology and Toxicology, 2012, Wright State University

  The objective of this study is to investigate the role of intracellular chloride concentration ([Cl¿¿¿¿¿¿]i) in insulin secretion in vivo and in vitro. The… (more)

Subjects/Keywords: Pharmacology; &946; -cell, Insulin Secretion, NKCC1, NKCC2

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APA (6th Edition):

Alshahrani, S. (2012). Role of Na+K+2Cl¿¿¿¿¿¿ Co-transporters in Insulin Secretion. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1347832847

Chicago Manual of Style (16th Edition):

Alshahrani, Saeed. “Role of Na+K+2Cl¿¿¿¿¿¿ Co-transporters in Insulin Secretion.” 2012. Masters Thesis, Wright State University. Accessed April 13, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=wright1347832847.

MLA Handbook (7th Edition):

Alshahrani, Saeed. “Role of Na+K+2Cl¿¿¿¿¿¿ Co-transporters in Insulin Secretion.” 2012. Web. 13 Apr 2021.

Vancouver:

Alshahrani S. Role of Na+K+2Cl¿¿¿¿¿¿ Co-transporters in Insulin Secretion. [Internet] [Masters thesis]. Wright State University; 2012. [cited 2021 Apr 13]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1347832847.

Council of Science Editors:

Alshahrani S. Role of Na+K+2Cl¿¿¿¿¿¿ Co-transporters in Insulin Secretion. [Masters Thesis]. Wright State University; 2012. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1347832847


University of Lund

11. Dayeh, Tasnim. The Human Pancreatic Islet Methylome and Its Role in Type 2 Diabetes.

Degree: 2016, University of Lund

 Islet dysfunction is central to the development and progression of type 2 diabetes (T2D). Epigenetic modifications are essential for establishing and maintaining cell identity and… (more)

Subjects/Keywords: Endocrinology and Diabetes; CpG-SNP; glucagon secretion; insulin secretion; human pancreatic islets; DNA methylation; Epigenetics

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APA (6th Edition):

Dayeh, T. (2016). The Human Pancreatic Islet Methylome and Its Role in Type 2 Diabetes. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/8515603 ; https://portal.research.lu.se/ws/files/3614834/8515627.pdf

Chicago Manual of Style (16th Edition):

Dayeh, Tasnim. “The Human Pancreatic Islet Methylome and Its Role in Type 2 Diabetes.” 2016. Doctoral Dissertation, University of Lund. Accessed April 13, 2021. https://lup.lub.lu.se/record/8515603 ; https://portal.research.lu.se/ws/files/3614834/8515627.pdf.

MLA Handbook (7th Edition):

Dayeh, Tasnim. “The Human Pancreatic Islet Methylome and Its Role in Type 2 Diabetes.” 2016. Web. 13 Apr 2021.

Vancouver:

Dayeh T. The Human Pancreatic Islet Methylome and Its Role in Type 2 Diabetes. [Internet] [Doctoral dissertation]. University of Lund; 2016. [cited 2021 Apr 13]. Available from: https://lup.lub.lu.se/record/8515603 ; https://portal.research.lu.se/ws/files/3614834/8515627.pdf.

Council of Science Editors:

Dayeh T. The Human Pancreatic Islet Methylome and Its Role in Type 2 Diabetes. [Doctoral Dissertation]. University of Lund; 2016. Available from: https://lup.lub.lu.se/record/8515603 ; https://portal.research.lu.se/ws/files/3614834/8515627.pdf


University of Georgia

12. Lobene, Andrea Jeanette. Zinc supplementation and insulin secretion in children.

Degree: 2017, University of Georgia

 Many children in the US are not meeting the Recommended Dietary Allowance (RDA) for zinc. Altered zinc status has been observed in prediabetic and diabetic… (more)

Subjects/Keywords: Zinc; insulin; insulin secretion; children; beta cell function; C-peptide; HOMA; pubertal growth

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APA (6th Edition):

Lobene, A. J. (2017). Zinc supplementation and insulin secretion in children. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/36838

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lobene, Andrea Jeanette. “Zinc supplementation and insulin secretion in children.” 2017. Thesis, University of Georgia. Accessed April 13, 2021. http://hdl.handle.net/10724/36838.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lobene, Andrea Jeanette. “Zinc supplementation and insulin secretion in children.” 2017. Web. 13 Apr 2021.

Vancouver:

Lobene AJ. Zinc supplementation and insulin secretion in children. [Internet] [Thesis]. University of Georgia; 2017. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/10724/36838.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lobene AJ. Zinc supplementation and insulin secretion in children. [Thesis]. University of Georgia; 2017. Available from: http://hdl.handle.net/10724/36838

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

13. Wang, Rong. PERK eIF2alpha kinase regulates cell proliferation, insulin synthesis and secretion in pancreatic beta cells.

Degree: 2014, Penn State University

Insulin synthesis and secretion, as well as cell proliferation are under tight regulation in pancreatic β-cells to maintain glucose homeostasis. Dysfunction in any of these… (more)

Subjects/Keywords: PERK eIF2alpha kinase; Ca2+ dynamics; insulin secretion; insulin biosynthesis; diabetes; ER stress; beta cell proliferation

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APA (6th Edition):

Wang, R. (2014). PERK eIF2alpha kinase regulates cell proliferation, insulin synthesis and secretion in pancreatic beta cells. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/21417

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Rong. “PERK eIF2alpha kinase regulates cell proliferation, insulin synthesis and secretion in pancreatic beta cells.” 2014. Thesis, Penn State University. Accessed April 13, 2021. https://submit-etda.libraries.psu.edu/catalog/21417.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Rong. “PERK eIF2alpha kinase regulates cell proliferation, insulin synthesis and secretion in pancreatic beta cells.” 2014. Web. 13 Apr 2021.

Vancouver:

Wang R. PERK eIF2alpha kinase regulates cell proliferation, insulin synthesis and secretion in pancreatic beta cells. [Internet] [Thesis]. Penn State University; 2014. [cited 2021 Apr 13]. Available from: https://submit-etda.libraries.psu.edu/catalog/21417.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang R. PERK eIF2alpha kinase regulates cell proliferation, insulin synthesis and secretion in pancreatic beta cells. [Thesis]. Penn State University; 2014. Available from: https://submit-etda.libraries.psu.edu/catalog/21417

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

14. Oakie, Amanda. The role of c-Kit and insulin receptor tyrosine kinases in beta cell function and insulin secretion.

Degree: 2019, University of Western Ontario

 The receptor tyrosine kinases (RTKs) c-Kit and insulin receptor (IR) initiate similar intracellular signalling pathways in pancreatic beta cells to regulate beta cell proliferation, survival,… (more)

Subjects/Keywords: c-Kit; insulin receptor; diabetes mellitus; insulin secretion; SNARE protein; Cre recombinase; Endocrinology

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APA (6th Edition):

Oakie, A. (2019). The role of c-Kit and insulin receptor tyrosine kinases in beta cell function and insulin secretion. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/6232

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oakie, Amanda. “The role of c-Kit and insulin receptor tyrosine kinases in beta cell function and insulin secretion.” 2019. Thesis, University of Western Ontario. Accessed April 13, 2021. https://ir.lib.uwo.ca/etd/6232.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oakie, Amanda. “The role of c-Kit and insulin receptor tyrosine kinases in beta cell function and insulin secretion.” 2019. Web. 13 Apr 2021.

Vancouver:

Oakie A. The role of c-Kit and insulin receptor tyrosine kinases in beta cell function and insulin secretion. [Internet] [Thesis]. University of Western Ontario; 2019. [cited 2021 Apr 13]. Available from: https://ir.lib.uwo.ca/etd/6232.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Oakie A. The role of c-Kit and insulin receptor tyrosine kinases in beta cell function and insulin secretion. [Thesis]. University of Western Ontario; 2019. Available from: https://ir.lib.uwo.ca/etd/6232

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade Estadual de Campinas

15. Branco, Renato Chaves Souto, 1986-. Desnutrição proteica impede os efeitos da taurina sobre o mecanismo de secreção e ação da insulina em camundongos obesos = participação do transportador de taurina = Protein malnutrition prevents the effects of taurine on secretion and action of insulin in obese mice : participation of the taurine transporter.

Degree: Instituto de Biologia; Programa de Pós-Graduação em Biologia Funcional e Molecular, 2016, Universidade Estadual de Campinas

Orientador: Everardo Magalhães Carneiro

Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia

Made available in DSpace on 2018-08-31T23:11:28Z (GMT). No. of bitstreams: 1… (more)

Subjects/Keywords: Obesidade; Desnutrição; Taurina; Insulina - Secreção; Insulina - Metabolismo; Obesity; Malnutrition; Taurine; Insulin - Secretion; Insulin - Metabolism

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APA (6th Edition):

Branco, Renato Chaves Souto, 1. (2016). Desnutrição proteica impede os efeitos da taurina sobre o mecanismo de secreção e ação da insulina em camundongos obesos = participação do transportador de taurina = Protein malnutrition prevents the effects of taurine on secretion and action of insulin in obese mice : participation of the taurine transporter. (Doctoral Dissertation). Universidade Estadual de Campinas. Retrieved from BRANCO, Renato Chaves Souto. Desnutrição proteica impede os efeitos da taurina sobre o mecanismo de secreção e ação da insulina em camundongos obesos: participação do transportador de taurina = Protein malnutrition prevents the effects of taurine on secretion and action of insulin in obese mice : participation of the taurine transporter. 2016. 1 recurso online ( 90 p.). Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia, Campinas, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/321561>. Acesso em: 31 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/321561

Chicago Manual of Style (16th Edition):

Branco, Renato Chaves Souto, 1986-. “Desnutrição proteica impede os efeitos da taurina sobre o mecanismo de secreção e ação da insulina em camundongos obesos = participação do transportador de taurina = Protein malnutrition prevents the effects of taurine on secretion and action of insulin in obese mice : participation of the taurine transporter.” 2016. Doctoral Dissertation, Universidade Estadual de Campinas. Accessed April 13, 2021. BRANCO, Renato Chaves Souto. Desnutrição proteica impede os efeitos da taurina sobre o mecanismo de secreção e ação da insulina em camundongos obesos: participação do transportador de taurina = Protein malnutrition prevents the effects of taurine on secretion and action of insulin in obese mice : participation of the taurine transporter. 2016. 1 recurso online ( 90 p.). Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia, Campinas, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/321561>. Acesso em: 31 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/321561.

MLA Handbook (7th Edition):

Branco, Renato Chaves Souto, 1986-. “Desnutrição proteica impede os efeitos da taurina sobre o mecanismo de secreção e ação da insulina em camundongos obesos = participação do transportador de taurina = Protein malnutrition prevents the effects of taurine on secretion and action of insulin in obese mice : participation of the taurine transporter.” 2016. Web. 13 Apr 2021.

Vancouver:

Branco, Renato Chaves Souto 1. Desnutrição proteica impede os efeitos da taurina sobre o mecanismo de secreção e ação da insulina em camundongos obesos = participação do transportador de taurina = Protein malnutrition prevents the effects of taurine on secretion and action of insulin in obese mice : participation of the taurine transporter. [Internet] [Doctoral dissertation]. Universidade Estadual de Campinas; 2016. [cited 2021 Apr 13]. Available from: BRANCO, Renato Chaves Souto. Desnutrição proteica impede os efeitos da taurina sobre o mecanismo de secreção e ação da insulina em camundongos obesos: participação do transportador de taurina = Protein malnutrition prevents the effects of taurine on secretion and action of insulin in obese mice : participation of the taurine transporter. 2016. 1 recurso online ( 90 p.). Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia, Campinas, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/321561>. Acesso em: 31 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/321561.

Council of Science Editors:

Branco, Renato Chaves Souto 1. Desnutrição proteica impede os efeitos da taurina sobre o mecanismo de secreção e ação da insulina em camundongos obesos = participação do transportador de taurina = Protein malnutrition prevents the effects of taurine on secretion and action of insulin in obese mice : participation of the taurine transporter. [Doctoral Dissertation]. Universidade Estadual de Campinas; 2016. Available from: BRANCO, Renato Chaves Souto. Desnutrição proteica impede os efeitos da taurina sobre o mecanismo de secreção e ação da insulina em camundongos obesos: participação do transportador de taurina = Protein malnutrition prevents the effects of taurine on secretion and action of insulin in obese mice : participation of the taurine transporter. 2016. 1 recurso online ( 90 p.). Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia, Campinas, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/321561>. Acesso em: 31 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/321561


University of Alberta

16. Kolic, Jelena. Distinct Roles of Class 1 PI3K Isoforms in the Regulation of Beta Cell Exocytosis and Insulin Secretion.

Degree: PhD, Department of Pharmacology, 2014, University of Alberta

 Type 2 diabetes (T2D) is characterized by peripheral insulin resistance and an insufficiency of insulin secretion from the pancreatic beta cell. The incidence of T2D… (more)

Subjects/Keywords: PI3K; Beta cell; Insulin secretion; Exocytosis; GIP; GLP-1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kolic, J. (2014). Distinct Roles of Class 1 PI3K Isoforms in the Regulation of Beta Cell Exocytosis and Insulin Secretion. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/cft848q912

Chicago Manual of Style (16th Edition):

Kolic, Jelena. “Distinct Roles of Class 1 PI3K Isoforms in the Regulation of Beta Cell Exocytosis and Insulin Secretion.” 2014. Doctoral Dissertation, University of Alberta. Accessed April 13, 2021. https://era.library.ualberta.ca/files/cft848q912.

MLA Handbook (7th Edition):

Kolic, Jelena. “Distinct Roles of Class 1 PI3K Isoforms in the Regulation of Beta Cell Exocytosis and Insulin Secretion.” 2014. Web. 13 Apr 2021.

Vancouver:

Kolic J. Distinct Roles of Class 1 PI3K Isoforms in the Regulation of Beta Cell Exocytosis and Insulin Secretion. [Internet] [Doctoral dissertation]. University of Alberta; 2014. [cited 2021 Apr 13]. Available from: https://era.library.ualberta.ca/files/cft848q912.

Council of Science Editors:

Kolic J. Distinct Roles of Class 1 PI3K Isoforms in the Regulation of Beta Cell Exocytosis and Insulin Secretion. [Doctoral Dissertation]. University of Alberta; 2014. Available from: https://era.library.ualberta.ca/files/cft848q912


University of Michigan

17. Cipolla, Cynthia Marie. Development and Application of Analytical Techniques for Evaluating Function in Pancreatic Islets of Langerhans.

Degree: PhD, Chemistry, 2015, University of Michigan

 Type 1 diabetes is caused by autoimmune destruction of insulin-secreting beta-cells found in the islets of Langerhans of the pancreas. Severe cases can be treated… (more)

Subjects/Keywords: Bioanalytical chemistry; Microfluidics; Metabolomics; Insulin secretion; Oxidative stress; Chemistry; Science

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cipolla, C. M. (2015). Development and Application of Analytical Techniques for Evaluating Function in Pancreatic Islets of Langerhans. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/113374

Chicago Manual of Style (16th Edition):

Cipolla, Cynthia Marie. “Development and Application of Analytical Techniques for Evaluating Function in Pancreatic Islets of Langerhans.” 2015. Doctoral Dissertation, University of Michigan. Accessed April 13, 2021. http://hdl.handle.net/2027.42/113374.

MLA Handbook (7th Edition):

Cipolla, Cynthia Marie. “Development and Application of Analytical Techniques for Evaluating Function in Pancreatic Islets of Langerhans.” 2015. Web. 13 Apr 2021.

Vancouver:

Cipolla CM. Development and Application of Analytical Techniques for Evaluating Function in Pancreatic Islets of Langerhans. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/2027.42/113374.

Council of Science Editors:

Cipolla CM. Development and Application of Analytical Techniques for Evaluating Function in Pancreatic Islets of Langerhans. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/113374


Vanderbilt University

18. Pound, Lynley Dayle. Characterization of islet genes implicated in human disease.

Degree: PhD, Molecular Physiology and Biophysics, 2011, Vanderbilt University

 Recent genome wide association (GWA) studies have linked single nucleotide polymorphisms (SNPs) in the G6PC2 gene with elevated fasting plasma glucose (FPG) and in the… (more)

Subjects/Keywords: fasting blood glucose; type 2 diabetes; insulin secretion; ZnT8; G6pc2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pound, L. D. (2011). Characterization of islet genes implicated in human disease. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14038

Chicago Manual of Style (16th Edition):

Pound, Lynley Dayle. “Characterization of islet genes implicated in human disease.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/14038.

MLA Handbook (7th Edition):

Pound, Lynley Dayle. “Characterization of islet genes implicated in human disease.” 2011. Web. 13 Apr 2021.

Vancouver:

Pound LD. Characterization of islet genes implicated in human disease. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/14038.

Council of Science Editors:

Pound LD. Characterization of islet genes implicated in human disease. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/14038


Vanderbilt University

19. Ustione, Alessandro. Dopaminergic regulation of insulin secretion from the pancreatic islet.

Degree: PhD, Molecular Physiology and Biophysics, 2012, Vanderbilt University

Insulin secretion is the natural response to hyperglycemia, and it is crucial to maintain glucose homeostasis in healthy individuals. Impairment in this regulation eventually results… (more)

Subjects/Keywords: pancreatic islet; dopamine; dopamine receptor; dopamine transporter; diabetes; insulin secretion

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APA (6th Edition):

Ustione, A. (2012). Dopaminergic regulation of insulin secretion from the pancreatic islet. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14923

Chicago Manual of Style (16th Edition):

Ustione, Alessandro. “Dopaminergic regulation of insulin secretion from the pancreatic islet.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/14923.

MLA Handbook (7th Edition):

Ustione, Alessandro. “Dopaminergic regulation of insulin secretion from the pancreatic islet.” 2012. Web. 13 Apr 2021.

Vancouver:

Ustione A. Dopaminergic regulation of insulin secretion from the pancreatic islet. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/14923.

Council of Science Editors:

Ustione A. Dopaminergic regulation of insulin secretion from the pancreatic islet. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/14923

20. Khalid, Parwaiz. Some biochemical studies on the islets of langerhans of rat pancreas with special reference to secretion of insulin;.

Degree: Biochemistry, 1988, Aligarh Muslim University

Abstract not available newline newline

Bibliography p. 135-160

Advisors/Committee Members: Kidwai, J R.

Subjects/Keywords: Biochemical; Secretion; Insulin; Langerhans; Pancreas

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APA (6th Edition):

Khalid, P. (1988). Some biochemical studies on the islets of langerhans of rat pancreas with special reference to secretion of insulin;. (Thesis). Aligarh Muslim University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/53889

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Khalid, Parwaiz. “Some biochemical studies on the islets of langerhans of rat pancreas with special reference to secretion of insulin;.” 1988. Thesis, Aligarh Muslim University. Accessed April 13, 2021. http://shodhganga.inflibnet.ac.in/handle/10603/53889.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Khalid, Parwaiz. “Some biochemical studies on the islets of langerhans of rat pancreas with special reference to secretion of insulin;.” 1988. Web. 13 Apr 2021.

Vancouver:

Khalid P. Some biochemical studies on the islets of langerhans of rat pancreas with special reference to secretion of insulin;. [Internet] [Thesis]. Aligarh Muslim University; 1988. [cited 2021 Apr 13]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/53889.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Khalid P. Some biochemical studies on the islets of langerhans of rat pancreas with special reference to secretion of insulin;. [Thesis]. Aligarh Muslim University; 1988. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/53889

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Waterloo

21. Pillai, Renjitha. Mechanistic Role of ARNT/HIF-1β in the Regulation of Glucose-Stimulated Insulin Secretion.

Degree: 2014, University of Waterloo

 Loss of glucose-stimulated insulin secretion (GSIS) from the pancreatic beta-cells is one of the earliest detectable defects in the pathogenesis of type 2 diabetes. However,… (more)

Subjects/Keywords: ARNT/HIF-1β; type 2 diabetes; insulin secretion; glucose metabolism

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APA (6th Edition):

Pillai, R. (2014). Mechanistic Role of ARNT/HIF-1β in the Regulation of Glucose-Stimulated Insulin Secretion. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/8376

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pillai, Renjitha. “Mechanistic Role of ARNT/HIF-1β in the Regulation of Glucose-Stimulated Insulin Secretion.” 2014. Thesis, University of Waterloo. Accessed April 13, 2021. http://hdl.handle.net/10012/8376.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pillai, Renjitha. “Mechanistic Role of ARNT/HIF-1β in the Regulation of Glucose-Stimulated Insulin Secretion.” 2014. Web. 13 Apr 2021.

Vancouver:

Pillai R. Mechanistic Role of ARNT/HIF-1β in the Regulation of Glucose-Stimulated Insulin Secretion. [Internet] [Thesis]. University of Waterloo; 2014. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/10012/8376.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pillai R. Mechanistic Role of ARNT/HIF-1β in the Regulation of Glucose-Stimulated Insulin Secretion. [Thesis]. University of Waterloo; 2014. Available from: http://hdl.handle.net/10012/8376

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

22. Erion, Karel Arnt. Nutrient regulation of insulin secretion: implications for hyperinsulinemia.

Degree: PhD, Medical Nutritional Sciences, 2016, Boston University

 Pancreatic beta-cells regulate blood glucose by secreting insulin in response to nutrients. The development of Type 2 Diabetes (T2D) is characterized by elevated insulin secretion(more)

Subjects/Keywords: Endocrinology; Beta-cell; Calcium; Diabetes; Fatty acid; Hyperinsulinemia; Insulin secretion

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APA (6th Edition):

Erion, K. A. (2016). Nutrient regulation of insulin secretion: implications for hyperinsulinemia. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/16726

Chicago Manual of Style (16th Edition):

Erion, Karel Arnt. “Nutrient regulation of insulin secretion: implications for hyperinsulinemia.” 2016. Doctoral Dissertation, Boston University. Accessed April 13, 2021. http://hdl.handle.net/2144/16726.

MLA Handbook (7th Edition):

Erion, Karel Arnt. “Nutrient regulation of insulin secretion: implications for hyperinsulinemia.” 2016. Web. 13 Apr 2021.

Vancouver:

Erion KA. Nutrient regulation of insulin secretion: implications for hyperinsulinemia. [Internet] [Doctoral dissertation]. Boston University; 2016. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/2144/16726.

Council of Science Editors:

Erion KA. Nutrient regulation of insulin secretion: implications for hyperinsulinemia. [Doctoral Dissertation]. Boston University; 2016. Available from: http://hdl.handle.net/2144/16726

23. Farret, Anne. Effets et mécanismes de l'activation des récepteurs purinergiques P2Y de la cellule beta pancréatique : Effects and mechanisms of activation of P2Y purinergic receptors of the pancreatic beta cell.

Degree: Docteur es, Anesthésiologie, réanimation, médecine d'urgence, pharmacologie et thérapeutique, 2010, Université Montpellier I

Les récepteurs purinergiques P2Y ont un rôle modulateur de la sécrétion d'insuline et constituent une cible potentielle pour la recherche de nouveaux antidiabétiques. Dans le… (more)

Subjects/Keywords: Récepteurs purinergiques P2Y; Insulinosécretion; Diabète; Purinergic receptors p2y; Insulin secretion; Diabetes

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APA (6th Edition):

Farret, A. (2010). Effets et mécanismes de l'activation des récepteurs purinergiques P2Y de la cellule beta pancréatique : Effects and mechanisms of activation of P2Y purinergic receptors of the pancreatic beta cell. (Doctoral Dissertation). Université Montpellier I. Retrieved from http://www.theses.fr/2010MON1T020

Chicago Manual of Style (16th Edition):

Farret, Anne. “Effets et mécanismes de l'activation des récepteurs purinergiques P2Y de la cellule beta pancréatique : Effects and mechanisms of activation of P2Y purinergic receptors of the pancreatic beta cell.” 2010. Doctoral Dissertation, Université Montpellier I. Accessed April 13, 2021. http://www.theses.fr/2010MON1T020.

MLA Handbook (7th Edition):

Farret, Anne. “Effets et mécanismes de l'activation des récepteurs purinergiques P2Y de la cellule beta pancréatique : Effects and mechanisms of activation of P2Y purinergic receptors of the pancreatic beta cell.” 2010. Web. 13 Apr 2021.

Vancouver:

Farret A. Effets et mécanismes de l'activation des récepteurs purinergiques P2Y de la cellule beta pancréatique : Effects and mechanisms of activation of P2Y purinergic receptors of the pancreatic beta cell. [Internet] [Doctoral dissertation]. Université Montpellier I; 2010. [cited 2021 Apr 13]. Available from: http://www.theses.fr/2010MON1T020.

Council of Science Editors:

Farret A. Effets et mécanismes de l'activation des récepteurs purinergiques P2Y de la cellule beta pancréatique : Effects and mechanisms of activation of P2Y purinergic receptors of the pancreatic beta cell. [Doctoral Dissertation]. Université Montpellier I; 2010. Available from: http://www.theses.fr/2010MON1T020


University of Adelaide

24. Huang, Weikun. [EMBARGOED] A microfluidic sensing system for simultaneous monitoring of extracellular Ca(II) and insulin secretion.

Degree: 2019, University of Adelaide

 Extracellular Ca²⁺ ([Ca²⁺]ex) is increasingly recognised as an important messenger for intercellular communications. Recent evidence suggests that [Ca²⁺]ex plays a critical role in mediating the… (more)

Subjects/Keywords: Extracellular Ca(ll); insulin secretion; microfluidic chip; multiplexed sensor

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APA (6th Edition):

Huang, W. (2019). [EMBARGOED] A microfluidic sensing system for simultaneous monitoring of extracellular Ca(II) and insulin secretion. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/120684

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Weikun. “[EMBARGOED] A microfluidic sensing system for simultaneous monitoring of extracellular Ca(II) and insulin secretion.” 2019. Thesis, University of Adelaide. Accessed April 13, 2021. http://hdl.handle.net/2440/120684.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Weikun. “[EMBARGOED] A microfluidic sensing system for simultaneous monitoring of extracellular Ca(II) and insulin secretion.” 2019. Web. 13 Apr 2021.

Vancouver:

Huang W. [EMBARGOED] A microfluidic sensing system for simultaneous monitoring of extracellular Ca(II) and insulin secretion. [Internet] [Thesis]. University of Adelaide; 2019. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/2440/120684.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang W. [EMBARGOED] A microfluidic sensing system for simultaneous monitoring of extracellular Ca(II) and insulin secretion. [Thesis]. University of Adelaide; 2019. Available from: http://hdl.handle.net/2440/120684

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Brigham Young University

25. Rowley, Thomas John. The Effect of Cocoa Flavanols on β-Cell Mass and Function.

Degree: MS, 2017, Brigham Young University

 A hallmark of type 2 diabetes (T2D) is β-cell dysfunction and the eventual loss of functional β-cell mass. Therefore, mechanisms that improve or preserve β-cell… (more)

Subjects/Keywords: cocoa; β-cell; catechin; insulin secretion; mitochondrial respiration; Nrf2; Nutrition

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APA (6th Edition):

Rowley, T. J. (2017). The Effect of Cocoa Flavanols on β-Cell Mass and Function. (Masters Thesis). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=7508&context=etd

Chicago Manual of Style (16th Edition):

Rowley, Thomas John. “The Effect of Cocoa Flavanols on β-Cell Mass and Function.” 2017. Masters Thesis, Brigham Young University. Accessed April 13, 2021. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=7508&context=etd.

MLA Handbook (7th Edition):

Rowley, Thomas John. “The Effect of Cocoa Flavanols on β-Cell Mass and Function.” 2017. Web. 13 Apr 2021.

Vancouver:

Rowley TJ. The Effect of Cocoa Flavanols on β-Cell Mass and Function. [Internet] [Masters thesis]. Brigham Young University; 2017. [cited 2021 Apr 13]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=7508&context=etd.

Council of Science Editors:

Rowley TJ. The Effect of Cocoa Flavanols on β-Cell Mass and Function. [Masters Thesis]. Brigham Young University; 2017. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=7508&context=etd


Boston University

26. Alsabeeh, Nour. Role of mitochondrial dysfunction in the development of nutrient-induced hyperinsulinemia.

Degree: PhD, Physiology, 2018, Boston University

 Pancreatic beta cells sense fluctuations in circulating nutrients and adjust the rate of insulin secretion to maintain glucose homeostasis. Mitochondria integrate changes in nutrient flux… (more)

Subjects/Keywords: Physiology; Cyclophilin D; Diabetes; Insulin secretion; Islet; Mitochondria; Proton leak

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APA (6th Edition):

Alsabeeh, N. (2018). Role of mitochondrial dysfunction in the development of nutrient-induced hyperinsulinemia. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/29271

Chicago Manual of Style (16th Edition):

Alsabeeh, Nour. “Role of mitochondrial dysfunction in the development of nutrient-induced hyperinsulinemia.” 2018. Doctoral Dissertation, Boston University. Accessed April 13, 2021. http://hdl.handle.net/2144/29271.

MLA Handbook (7th Edition):

Alsabeeh, Nour. “Role of mitochondrial dysfunction in the development of nutrient-induced hyperinsulinemia.” 2018. Web. 13 Apr 2021.

Vancouver:

Alsabeeh N. Role of mitochondrial dysfunction in the development of nutrient-induced hyperinsulinemia. [Internet] [Doctoral dissertation]. Boston University; 2018. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/2144/29271.

Council of Science Editors:

Alsabeeh N. Role of mitochondrial dysfunction in the development of nutrient-induced hyperinsulinemia. [Doctoral Dissertation]. Boston University; 2018. Available from: http://hdl.handle.net/2144/29271


Boston University

27. Rumala, Courtney. Metabolic regulation of insulin secretion: the link between excess glucose, mechanistic target of rapamycin complex 1 & hyperinsulinemia.

Degree: PhD, Nutrition and Metabolism, 2019, Boston University

 Obesity, a major risk factor in the development of Type 2 Diabetes (T2D), is commonly associated with insulin resistance and hyperinsulinemia. The long accepted view… (more)

Subjects/Keywords: Nutrition; Glucose; Hyperinsulinemia; Insulin secretion; mTORC1; Respiration; Type 2 diabetes

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rumala, C. (2019). Metabolic regulation of insulin secretion: the link between excess glucose, mechanistic target of rapamycin complex 1 & hyperinsulinemia. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/38590

Chicago Manual of Style (16th Edition):

Rumala, Courtney. “Metabolic regulation of insulin secretion: the link between excess glucose, mechanistic target of rapamycin complex 1 & hyperinsulinemia.” 2019. Doctoral Dissertation, Boston University. Accessed April 13, 2021. http://hdl.handle.net/2144/38590.

MLA Handbook (7th Edition):

Rumala, Courtney. “Metabolic regulation of insulin secretion: the link between excess glucose, mechanistic target of rapamycin complex 1 & hyperinsulinemia.” 2019. Web. 13 Apr 2021.

Vancouver:

Rumala C. Metabolic regulation of insulin secretion: the link between excess glucose, mechanistic target of rapamycin complex 1 & hyperinsulinemia. [Internet] [Doctoral dissertation]. Boston University; 2019. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/2144/38590.

Council of Science Editors:

Rumala C. Metabolic regulation of insulin secretion: the link between excess glucose, mechanistic target of rapamycin complex 1 & hyperinsulinemia. [Doctoral Dissertation]. Boston University; 2019. Available from: http://hdl.handle.net/2144/38590

28. Hadjivassiliou, Vicky. Cytokine inhibitory actions and gene expression in islets of Langerhans.

Degree: PhD, 2000, University of Sussex

Subjects/Keywords: 572; DNA damage; Insulin secretion

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APA (6th Edition):

Hadjivassiliou, V. (2000). Cytokine inhibitory actions and gene expression in islets of Langerhans. (Doctoral Dissertation). University of Sussex. Retrieved from https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.311334

Chicago Manual of Style (16th Edition):

Hadjivassiliou, Vicky. “Cytokine inhibitory actions and gene expression in islets of Langerhans.” 2000. Doctoral Dissertation, University of Sussex. Accessed April 13, 2021. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.311334.

MLA Handbook (7th Edition):

Hadjivassiliou, Vicky. “Cytokine inhibitory actions and gene expression in islets of Langerhans.” 2000. Web. 13 Apr 2021.

Vancouver:

Hadjivassiliou V. Cytokine inhibitory actions and gene expression in islets of Langerhans. [Internet] [Doctoral dissertation]. University of Sussex; 2000. [cited 2021 Apr 13]. Available from: https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.311334.

Council of Science Editors:

Hadjivassiliou V. Cytokine inhibitory actions and gene expression in islets of Langerhans. [Doctoral Dissertation]. University of Sussex; 2000. Available from: https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.311334

29. Delaney, Carol Ann. Cytokine and nitric oxide mediated DNA damage and inhibition of function in islets of Langerhans and insulin secreting cell lines.

Degree: PhD, 1995, University of Sussex

Subjects/Keywords: 572; Diabetes; Insulin secretion

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APA (6th Edition):

Delaney, C. A. (1995). Cytokine and nitric oxide mediated DNA damage and inhibition of function in islets of Langerhans and insulin secreting cell lines. (Doctoral Dissertation). University of Sussex. Retrieved from https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.283000

Chicago Manual of Style (16th Edition):

Delaney, Carol Ann. “Cytokine and nitric oxide mediated DNA damage and inhibition of function in islets of Langerhans and insulin secreting cell lines.” 1995. Doctoral Dissertation, University of Sussex. Accessed April 13, 2021. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.283000.

MLA Handbook (7th Edition):

Delaney, Carol Ann. “Cytokine and nitric oxide mediated DNA damage and inhibition of function in islets of Langerhans and insulin secreting cell lines.” 1995. Web. 13 Apr 2021.

Vancouver:

Delaney CA. Cytokine and nitric oxide mediated DNA damage and inhibition of function in islets of Langerhans and insulin secreting cell lines. [Internet] [Doctoral dissertation]. University of Sussex; 1995. [cited 2021 Apr 13]. Available from: https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.283000.

Council of Science Editors:

Delaney CA. Cytokine and nitric oxide mediated DNA damage and inhibition of function in islets of Langerhans and insulin secreting cell lines. [Doctoral Dissertation]. University of Sussex; 1995. Available from: https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.283000


Tartu University

30. Toots, Maarja. Pharmacological challenge in rodent models of Wolfram syndrome with emphasis on diabetic phenotype .

Degree: 2018, Tartu University

 Wolframi sündroom (WS) on autosomaalne retsessiivne haigus, mida põhjustavad mutatsioonid Wolframin1 (WFS1) geenis. Sündroomi põhisümptomiks on varajane suhkrudiabeet, millele järgneb optilise närvi atroofia, kurtus, psühhiaatrilised… (more)

Subjects/Keywords: animal models; Wolfram syndrome; diabetes; phenotype; secretion of insulin; enteroglucagon

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APA (6th Edition):

Toots, M. (2018). Pharmacological challenge in rodent models of Wolfram syndrome with emphasis on diabetic phenotype . (Thesis). Tartu University. Retrieved from http://hdl.handle.net/10062/62526

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Toots, Maarja. “Pharmacological challenge in rodent models of Wolfram syndrome with emphasis on diabetic phenotype .” 2018. Thesis, Tartu University. Accessed April 13, 2021. http://hdl.handle.net/10062/62526.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Toots, Maarja. “Pharmacological challenge in rodent models of Wolfram syndrome with emphasis on diabetic phenotype .” 2018. Web. 13 Apr 2021.

Vancouver:

Toots M. Pharmacological challenge in rodent models of Wolfram syndrome with emphasis on diabetic phenotype . [Internet] [Thesis]. Tartu University; 2018. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/10062/62526.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Toots M. Pharmacological challenge in rodent models of Wolfram syndrome with emphasis on diabetic phenotype . [Thesis]. Tartu University; 2018. Available from: http://hdl.handle.net/10062/62526

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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