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You searched for subject:(Inflammation models). Showing records 1 – 30 of 97 total matches.

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1. Millon, Antoine. Imagerie haute résolution morphologique et fonctionnelle de la plaque d'athérosclérose : Morphological and functional imaging of the atherosclerotic plaque.

Degree: Docteur es, Sciences de la santé, 2013, Université Claude Bernard – Lyon I

La compréhension des mécanismes précis conduisant à la formation d'une plaque d'athérosclérose et à sa déstabilisation provoquant infarctus du myocarde ou accident vasculaire cérébral est… (more)

Subjects/Keywords: Athérosclérose; Modèles animaux; Imagerie; Inflammation; Atherosclerosis; Animal models; Imaging; Inflammation; 616.136

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APA (6th Edition):

Millon, A. (2013). Imagerie haute résolution morphologique et fonctionnelle de la plaque d'athérosclérose : Morphological and functional imaging of the atherosclerotic plaque. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2013LYO10140

Chicago Manual of Style (16th Edition):

Millon, Antoine. “Imagerie haute résolution morphologique et fonctionnelle de la plaque d'athérosclérose : Morphological and functional imaging of the atherosclerotic plaque.” 2013. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed April 18, 2021. http://www.theses.fr/2013LYO10140.

MLA Handbook (7th Edition):

Millon, Antoine. “Imagerie haute résolution morphologique et fonctionnelle de la plaque d'athérosclérose : Morphological and functional imaging of the atherosclerotic plaque.” 2013. Web. 18 Apr 2021.

Vancouver:

Millon A. Imagerie haute résolution morphologique et fonctionnelle de la plaque d'athérosclérose : Morphological and functional imaging of the atherosclerotic plaque. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2013. [cited 2021 Apr 18]. Available from: http://www.theses.fr/2013LYO10140.

Council of Science Editors:

Millon A. Imagerie haute résolution morphologique et fonctionnelle de la plaque d'athérosclérose : Morphological and functional imaging of the atherosclerotic plaque. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2013. Available from: http://www.theses.fr/2013LYO10140


University of Manchester

2. Sridharan, Sujata. Quantitative analysis of positron emission tomography (PET) with the second generation translocator protein (TSPO) ligand [18F]GE-180.

Degree: 2016, University of Manchester

 Background: The 18 kDa translocator protein (TSPO), expressed at a low level in the healthy human central nervous system (CNS), is upregulated in inflammatory brain… (more)

Subjects/Keywords: TSPO; PET; Inflammation; Animal models; Multiple sclerosis

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APA (6th Edition):

Sridharan, S. (2016). Quantitative analysis of positron emission tomography (PET) with the second generation translocator protein (TSPO) ligand [18F]GE-180. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306013

Chicago Manual of Style (16th Edition):

Sridharan, Sujata. “Quantitative analysis of positron emission tomography (PET) with the second generation translocator protein (TSPO) ligand [18F]GE-180.” 2016. Doctoral Dissertation, University of Manchester. Accessed April 18, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306013.

MLA Handbook (7th Edition):

Sridharan, Sujata. “Quantitative analysis of positron emission tomography (PET) with the second generation translocator protein (TSPO) ligand [18F]GE-180.” 2016. Web. 18 Apr 2021.

Vancouver:

Sridharan S. Quantitative analysis of positron emission tomography (PET) with the second generation translocator protein (TSPO) ligand [18F]GE-180. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 Apr 18]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306013.

Council of Science Editors:

Sridharan S. Quantitative analysis of positron emission tomography (PET) with the second generation translocator protein (TSPO) ligand [18F]GE-180. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306013

3. Dormont, Flavio. Development of nanomedicines for inflammation disorders : evaluation of pharmacological efficacy on preclinical models : Développement de nanomédicaments pour les troubles inflammatoires : évaluation de l’effet protecteur dans des modèles précliniques.

Degree: Docteur es, Pharmacotechnie et biopharmacie, 2019, Université Paris-Saclay (ComUE)

 Les syndromes inflammatoires, marqués par une réaction excessive du système immunitaire, sont associés à de nombreuses conditions pathologiques comme le sepsis, les accidents d’ischémie/reperfusion ou… (more)

Subjects/Keywords: Squalène; Adénosine; Antioxydants; Modèles précliniques; Nanothérapie; Inflammation; Inflammation; Nanotherapy; Preclinical models; Antioxidants; Adenosine; Squalene

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APA (6th Edition):

Dormont, F. (2019). Development of nanomedicines for inflammation disorders : evaluation of pharmacological efficacy on preclinical models : Développement de nanomédicaments pour les troubles inflammatoires : évaluation de l’effet protecteur dans des modèles précliniques. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2019SACLS451

Chicago Manual of Style (16th Edition):

Dormont, Flavio. “Development of nanomedicines for inflammation disorders : evaluation of pharmacological efficacy on preclinical models : Développement de nanomédicaments pour les troubles inflammatoires : évaluation de l’effet protecteur dans des modèles précliniques.” 2019. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed April 18, 2021. http://www.theses.fr/2019SACLS451.

MLA Handbook (7th Edition):

Dormont, Flavio. “Development of nanomedicines for inflammation disorders : evaluation of pharmacological efficacy on preclinical models : Développement de nanomédicaments pour les troubles inflammatoires : évaluation de l’effet protecteur dans des modèles précliniques.” 2019. Web. 18 Apr 2021.

Vancouver:

Dormont F. Development of nanomedicines for inflammation disorders : evaluation of pharmacological efficacy on preclinical models : Développement de nanomédicaments pour les troubles inflammatoires : évaluation de l’effet protecteur dans des modèles précliniques. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2019. [cited 2021 Apr 18]. Available from: http://www.theses.fr/2019SACLS451.

Council of Science Editors:

Dormont F. Development of nanomedicines for inflammation disorders : evaluation of pharmacological efficacy on preclinical models : Développement de nanomédicaments pour les troubles inflammatoires : évaluation de l’effet protecteur dans des modèles précliniques. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2019. Available from: http://www.theses.fr/2019SACLS451


Michigan State University

4. Iqbal, Tania Riyan. Elucidating mechanisms of neurogenic inflammation in the mouse olfactory epithelium.

Degree: 2015, Michigan State University

ABSTRACTELUCIDATING MECHANISMS OF NEUROGENIC INFLAMMATION IN THE MOUSE OLFACTORY EPITHELIUMByTania Riyan Iqbal Inflammation in the nervous system is linked to neurological disorders and neurodegenerative disease,… (more)

Subjects/Keywords: Inflammation – Animal models; Inflammation – Environmental aspects; Epithelium; Developmental neurobiology; Mice – Physiology; Neurosciences; Cellular biology; Molecular biology; Neurogenic inflammation; Olfactory epithelium

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APA (6th Edition):

Iqbal, T. R. (2015). Elucidating mechanisms of neurogenic inflammation in the mouse olfactory epithelium. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:3218

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Iqbal, Tania Riyan. “Elucidating mechanisms of neurogenic inflammation in the mouse olfactory epithelium.” 2015. Thesis, Michigan State University. Accessed April 18, 2021. http://etd.lib.msu.edu/islandora/object/etd:3218.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Iqbal, Tania Riyan. “Elucidating mechanisms of neurogenic inflammation in the mouse olfactory epithelium.” 2015. Web. 18 Apr 2021.

Vancouver:

Iqbal TR. Elucidating mechanisms of neurogenic inflammation in the mouse olfactory epithelium. [Internet] [Thesis]. Michigan State University; 2015. [cited 2021 Apr 18]. Available from: http://etd.lib.msu.edu/islandora/object/etd:3218.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Iqbal TR. Elucidating mechanisms of neurogenic inflammation in the mouse olfactory epithelium. [Thesis]. Michigan State University; 2015. Available from: http://etd.lib.msu.edu/islandora/object/etd:3218

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

5. Saxon, Jamie Ausborn. Investigation of epithelial canonical and non-canonical NF-κB signaling in lung adenocarcinoma.

Degree: PhD, Cancer Biology, 2016, Vanderbilt University

 Nuclear factor κ-light chain enhancer of activated B cells (NF-κB) is a transcription factor that can be activated through canonical or non-canonical signaling pathways, and… (more)

Subjects/Keywords: macrophages; cell cycle; inflammation; lung cancer; mouse models; ARDS; apoptosis

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APA (6th Edition):

Saxon, J. A. (2016). Investigation of epithelial canonical and non-canonical NF-κB signaling in lung adenocarcinoma. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10550

Chicago Manual of Style (16th Edition):

Saxon, Jamie Ausborn. “Investigation of epithelial canonical and non-canonical NF-κB signaling in lung adenocarcinoma.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed April 18, 2021. http://hdl.handle.net/1803/10550.

MLA Handbook (7th Edition):

Saxon, Jamie Ausborn. “Investigation of epithelial canonical and non-canonical NF-κB signaling in lung adenocarcinoma.” 2016. Web. 18 Apr 2021.

Vancouver:

Saxon JA. Investigation of epithelial canonical and non-canonical NF-κB signaling in lung adenocarcinoma. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1803/10550.

Council of Science Editors:

Saxon JA. Investigation of epithelial canonical and non-canonical NF-κB signaling in lung adenocarcinoma. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/10550


University of Texas Southwestern Medical Center

6. Luitel, Krishna. Investigating the Effects of Particle Radiation Exposure on Lung Carcinogenesis.

Degree: 2019, University of Texas Southwestern Medical Center

 Lung cancer accounts for more cancer-related deaths than any other cancer type among both men and women. The overall increase in radiation risk for human… (more)

Subjects/Keywords: Disease Models, Animal; Inflammation; Lung Neoplasms; Neoplasms, Radiation-Induced; Protons

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APA (6th Edition):

Luitel, K. (2019). Investigating the Effects of Particle Radiation Exposure on Lung Carcinogenesis. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Luitel, Krishna. “Investigating the Effects of Particle Radiation Exposure on Lung Carcinogenesis.” 2019. Thesis, University of Texas Southwestern Medical Center. Accessed April 18, 2021. http://hdl.handle.net/2152.5/6625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Luitel, Krishna. “Investigating the Effects of Particle Radiation Exposure on Lung Carcinogenesis.” 2019. Web. 18 Apr 2021.

Vancouver:

Luitel K. Investigating the Effects of Particle Radiation Exposure on Lung Carcinogenesis. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2019. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2152.5/6625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Luitel K. Investigating the Effects of Particle Radiation Exposure on Lung Carcinogenesis. [Thesis]. University of Texas Southwestern Medical Center; 2019. Available from: http://hdl.handle.net/2152.5/6625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Guelph

7. Pinelli, Christopher. Role of Macrophages in the Development of Castration-Resistant Prostate Cancer.

Degree: PhD, Department of Pathobiology, 2016, University of Guelph

 Prostate cancer (PCa) is the second most common cancer affecting men in Canada and USA, and accounts for thousands of deaths each year. While most… (more)

Subjects/Keywords: Prostate; Cancer; Animal Models; Inflammation; Macrophages; Cytokines; Pathology

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APA (6th Edition):

Pinelli, C. (2016). Role of Macrophages in the Development of Castration-Resistant Prostate Cancer. (Doctoral Dissertation). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9689

Chicago Manual of Style (16th Edition):

Pinelli, Christopher. “Role of Macrophages in the Development of Castration-Resistant Prostate Cancer.” 2016. Doctoral Dissertation, University of Guelph. Accessed April 18, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9689.

MLA Handbook (7th Edition):

Pinelli, Christopher. “Role of Macrophages in the Development of Castration-Resistant Prostate Cancer.” 2016. Web. 18 Apr 2021.

Vancouver:

Pinelli C. Role of Macrophages in the Development of Castration-Resistant Prostate Cancer. [Internet] [Doctoral dissertation]. University of Guelph; 2016. [cited 2021 Apr 18]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9689.

Council of Science Editors:

Pinelli C. Role of Macrophages in the Development of Castration-Resistant Prostate Cancer. [Doctoral Dissertation]. University of Guelph; 2016. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9689


University of Manchester

8. Sridharan, Sujata. Quantitative analysis of positron emission tomography (PET) with the second generation translocator protein (TSPO) ligand [18F]GE-180.

Degree: PhD, 2016, University of Manchester

 Background: The 18 kDa translocator protein (TSPO), expressed at a low level in the healthy human central nervous system (CNS), is upregulated in inflammatory brain… (more)

Subjects/Keywords: 616.07; Multiple sclerosis; TSPO; PET; Inflammation; Animal models

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APA (6th Edition):

Sridharan, S. (2016). Quantitative analysis of positron emission tomography (PET) with the second generation translocator protein (TSPO) ligand [18F]GE-180. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/quantitative-analysis-of-positron-emission-tomography-pet-with-the-second-generation-translocator-protein-tspo-ligand-18fge180(01bdc97f-90f7-4db3-8268-f65e013640ab).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.719307

Chicago Manual of Style (16th Edition):

Sridharan, Sujata. “Quantitative analysis of positron emission tomography (PET) with the second generation translocator protein (TSPO) ligand [18F]GE-180.” 2016. Doctoral Dissertation, University of Manchester. Accessed April 18, 2021. https://www.research.manchester.ac.uk/portal/en/theses/quantitative-analysis-of-positron-emission-tomography-pet-with-the-second-generation-translocator-protein-tspo-ligand-18fge180(01bdc97f-90f7-4db3-8268-f65e013640ab).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.719307.

MLA Handbook (7th Edition):

Sridharan, Sujata. “Quantitative analysis of positron emission tomography (PET) with the second generation translocator protein (TSPO) ligand [18F]GE-180.” 2016. Web. 18 Apr 2021.

Vancouver:

Sridharan S. Quantitative analysis of positron emission tomography (PET) with the second generation translocator protein (TSPO) ligand [18F]GE-180. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 Apr 18]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/quantitative-analysis-of-positron-emission-tomography-pet-with-the-second-generation-translocator-protein-tspo-ligand-18fge180(01bdc97f-90f7-4db3-8268-f65e013640ab).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.719307.

Council of Science Editors:

Sridharan S. Quantitative analysis of positron emission tomography (PET) with the second generation translocator protein (TSPO) ligand [18F]GE-180. [Doctoral Dissertation]. University of Manchester; 2016. Available from: https://www.research.manchester.ac.uk/portal/en/theses/quantitative-analysis-of-positron-emission-tomography-pet-with-the-second-generation-translocator-protein-tspo-ligand-18fge180(01bdc97f-90f7-4db3-8268-f65e013640ab).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.719307


University of Toronto

9. Hanna, Daniel. Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis.

Degree: PhD, 2018, University of Toronto

 Liver cancer is the third most common cause of cancer-related death in humans and is 2-4 times more common in men than in women for… (more)

Subjects/Keywords: Hepatotoxicity; Inflammation; Liver cancer; Mouse models; Procarcinogens; Sex differences; 0419

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APA (6th Edition):

Hanna, D. (2018). Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/91754

Chicago Manual of Style (16th Edition):

Hanna, Daniel. “Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis.” 2018. Doctoral Dissertation, University of Toronto. Accessed April 18, 2021. http://hdl.handle.net/1807/91754.

MLA Handbook (7th Edition):

Hanna, Daniel. “Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis.” 2018. Web. 18 Apr 2021.

Vancouver:

Hanna D. Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis. [Internet] [Doctoral dissertation]. University of Toronto; 2018. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1807/91754.

Council of Science Editors:

Hanna D. Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis. [Doctoral Dissertation]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91754


University of Melbourne

10. Laskaris, Liliana Ellin. Inflammation in psychosis: Impact on brain structure and symptoms in animal models and humans.

Degree: 2019, University of Melbourne

 Schizophrenia is a severe neuropsychiatric disorder, arising in adolescence and early adulthood and characterised by hallucinations, delusions, blunted affect and disorganised thought patterns. One of… (more)

Subjects/Keywords: schizophrenia; psychosis; inflammation; peripheral biomarkers; mGluR5; imaging; animal models

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APA (6th Edition):

Laskaris, L. E. (2019). Inflammation in psychosis: Impact on brain structure and symptoms in animal models and humans. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/234029

Chicago Manual of Style (16th Edition):

Laskaris, Liliana Ellin. “Inflammation in psychosis: Impact on brain structure and symptoms in animal models and humans.” 2019. Doctoral Dissertation, University of Melbourne. Accessed April 18, 2021. http://hdl.handle.net/11343/234029.

MLA Handbook (7th Edition):

Laskaris, Liliana Ellin. “Inflammation in psychosis: Impact on brain structure and symptoms in animal models and humans.” 2019. Web. 18 Apr 2021.

Vancouver:

Laskaris LE. Inflammation in psychosis: Impact on brain structure and symptoms in animal models and humans. [Internet] [Doctoral dissertation]. University of Melbourne; 2019. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/11343/234029.

Council of Science Editors:

Laskaris LE. Inflammation in psychosis: Impact on brain structure and symptoms in animal models and humans. [Doctoral Dissertation]. University of Melbourne; 2019. Available from: http://hdl.handle.net/11343/234029


Rutgers University

11. Foteinou, Panagiota, 1981-. A multiscale model for the assessment of autonomic dysfunction in human endotoxemia.

Degree: PhD, Biomedical Engineering, 2010, Rutgers University

The overall goal of this dissertation is the development of a semi-mechanistic, physiology-based, multiscale model of human endotoxemia, as a prototype model of acute inflammation(more)

Subjects/Keywords: Endotoxemia; Inflammation – Mathematical models

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APA (6th Edition):

Foteinou, Panagiota, 1. (2010). A multiscale model for the assessment of autonomic dysfunction in human endotoxemia. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000053028

Chicago Manual of Style (16th Edition):

Foteinou, Panagiota, 1981-. “A multiscale model for the assessment of autonomic dysfunction in human endotoxemia.” 2010. Doctoral Dissertation, Rutgers University. Accessed April 18, 2021. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000053028.

MLA Handbook (7th Edition):

Foteinou, Panagiota, 1981-. “A multiscale model for the assessment of autonomic dysfunction in human endotoxemia.” 2010. Web. 18 Apr 2021.

Vancouver:

Foteinou, Panagiota 1. A multiscale model for the assessment of autonomic dysfunction in human endotoxemia. [Internet] [Doctoral dissertation]. Rutgers University; 2010. [cited 2021 Apr 18]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000053028.

Council of Science Editors:

Foteinou, Panagiota 1. A multiscale model for the assessment of autonomic dysfunction in human endotoxemia. [Doctoral Dissertation]. Rutgers University; 2010. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000053028

12. Restagno, Damien. Contribution à l’étude du choc septique à l’aide de modèles animaux : de l’immunoparalysie à la cachexie : Studying septic shock using animal models : from immunoparalysis to cachexia.

Degree: Docteur es, Physiologie, 2016, Lyon

Le choc septique, première cause de mortalité intra hospitalière en France, comporte deux phases chronologiques. Lors de la première phase, la mortalité est essentiellement liée… (more)

Subjects/Keywords: Sepsis; Choc; Modèles; Inflammation; Immunoparalysies; Cachexie; Muscles; Ghréline; Sepsis; Shock; Models; Inflammation; Immunoparalysie; Cachexia; Muscles; Ghrelin; 571

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APA (6th Edition):

Restagno, D. (2016). Contribution à l’étude du choc septique à l’aide de modèles animaux : de l’immunoparalysie à la cachexie : Studying septic shock using animal models : from immunoparalysis to cachexia. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2016LYSE1096

Chicago Manual of Style (16th Edition):

Restagno, Damien. “Contribution à l’étude du choc septique à l’aide de modèles animaux : de l’immunoparalysie à la cachexie : Studying septic shock using animal models : from immunoparalysis to cachexia.” 2016. Doctoral Dissertation, Lyon. Accessed April 18, 2021. http://www.theses.fr/2016LYSE1096.

MLA Handbook (7th Edition):

Restagno, Damien. “Contribution à l’étude du choc septique à l’aide de modèles animaux : de l’immunoparalysie à la cachexie : Studying septic shock using animal models : from immunoparalysis to cachexia.” 2016. Web. 18 Apr 2021.

Vancouver:

Restagno D. Contribution à l’étude du choc septique à l’aide de modèles animaux : de l’immunoparalysie à la cachexie : Studying septic shock using animal models : from immunoparalysis to cachexia. [Internet] [Doctoral dissertation]. Lyon; 2016. [cited 2021 Apr 18]. Available from: http://www.theses.fr/2016LYSE1096.

Council of Science Editors:

Restagno D. Contribution à l’étude du choc septique à l’aide de modèles animaux : de l’immunoparalysie à la cachexie : Studying septic shock using animal models : from immunoparalysis to cachexia. [Doctoral Dissertation]. Lyon; 2016. Available from: http://www.theses.fr/2016LYSE1096


University of Florida

13. Davids, Jennifer A. Myxomaviral Immune Modulating Proteins Alter Gene Expression of Monocyte Apoptotic Pathways.

Degree: PhD, Medical Sciences - Immunology and Microbiology (IDP), 2013, University of Florida

 Atherosclerosis ischaracterized by chronic inflammation and cell death (apoptosis) whichinitiates plaque growth and thrombotic occlusion. Serine protease inhibitors, or serpins, regulate apoptotic, inflammatory, and coagulation… (more)

Subjects/Keywords: Animal models; Apoptosis; Cells; Chemokines; Homologous transplantation; Inflammation; Macrophages; Mice; Physical trauma; Serpins; atherosclerosis  – davids  – dissertation  – inflammation  – serp-1  – serp-2

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APA (6th Edition):

Davids, J. A. (2013). Myxomaviral Immune Modulating Proteins Alter Gene Expression of Monocyte Apoptotic Pathways. (Doctoral Dissertation). University of Florida. Retrieved from https://ufdc.ufl.edu/UFE0045042

Chicago Manual of Style (16th Edition):

Davids, Jennifer A. “Myxomaviral Immune Modulating Proteins Alter Gene Expression of Monocyte Apoptotic Pathways.” 2013. Doctoral Dissertation, University of Florida. Accessed April 18, 2021. https://ufdc.ufl.edu/UFE0045042.

MLA Handbook (7th Edition):

Davids, Jennifer A. “Myxomaviral Immune Modulating Proteins Alter Gene Expression of Monocyte Apoptotic Pathways.” 2013. Web. 18 Apr 2021.

Vancouver:

Davids JA. Myxomaviral Immune Modulating Proteins Alter Gene Expression of Monocyte Apoptotic Pathways. [Internet] [Doctoral dissertation]. University of Florida; 2013. [cited 2021 Apr 18]. Available from: https://ufdc.ufl.edu/UFE0045042.

Council of Science Editors:

Davids JA. Myxomaviral Immune Modulating Proteins Alter Gene Expression of Monocyte Apoptotic Pathways. [Doctoral Dissertation]. University of Florida; 2013. Available from: https://ufdc.ufl.edu/UFE0045042


University of Hong Kong

14. Cheung, Oi-fung, Stephanie. A study into the anti-inflammatory effects of silver nanoparticles andtheir potential clinical application.

Degree: 2008, University of Hong Kong

Subjects/Keywords: Nanoparticles.; Cytokines.; Inflammation - Animal models.; Silver.

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APA (6th Edition):

Cheung, Oi-fung, S. (2008). A study into the anti-inflammatory effects of silver nanoparticles andtheir potential clinical application. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/54466

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cheung, Oi-fung, Stephanie. “A study into the anti-inflammatory effects of silver nanoparticles andtheir potential clinical application.” 2008. Thesis, University of Hong Kong. Accessed April 18, 2021. http://hdl.handle.net/10722/54466.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cheung, Oi-fung, Stephanie. “A study into the anti-inflammatory effects of silver nanoparticles andtheir potential clinical application.” 2008. Web. 18 Apr 2021.

Vancouver:

Cheung, Oi-fung S. A study into the anti-inflammatory effects of silver nanoparticles andtheir potential clinical application. [Internet] [Thesis]. University of Hong Kong; 2008. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10722/54466.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cheung, Oi-fung S. A study into the anti-inflammatory effects of silver nanoparticles andtheir potential clinical application. [Thesis]. University of Hong Kong; 2008. Available from: http://hdl.handle.net/10722/54466

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Otago

15. Rivers Auty, Jack Rocky-Jay. An investigation into the cannabinoid receptor type 2 as a therapeutic target for childhood cerebral hypoxia .

Degree: 2013, University of Otago

 Cannabis sativa is an herbaceous plant that when consumed produces a number of well-known effects on the body, primarily elicited by two G-protein coupled receptors:… (more)

Subjects/Keywords: Marijuana; Cannabinoid; receptor; stroke; brain damage; neuroscience; neuroprotection; inflammation; microglia; animal models; CB2; pharmacology

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APA (6th Edition):

Rivers Auty, J. R. (2013). An investigation into the cannabinoid receptor type 2 as a therapeutic target for childhood cerebral hypoxia . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/4350

Chicago Manual of Style (16th Edition):

Rivers Auty, Jack Rocky-Jay. “An investigation into the cannabinoid receptor type 2 as a therapeutic target for childhood cerebral hypoxia .” 2013. Doctoral Dissertation, University of Otago. Accessed April 18, 2021. http://hdl.handle.net/10523/4350.

MLA Handbook (7th Edition):

Rivers Auty, Jack Rocky-Jay. “An investigation into the cannabinoid receptor type 2 as a therapeutic target for childhood cerebral hypoxia .” 2013. Web. 18 Apr 2021.

Vancouver:

Rivers Auty JR. An investigation into the cannabinoid receptor type 2 as a therapeutic target for childhood cerebral hypoxia . [Internet] [Doctoral dissertation]. University of Otago; 2013. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10523/4350.

Council of Science Editors:

Rivers Auty JR. An investigation into the cannabinoid receptor type 2 as a therapeutic target for childhood cerebral hypoxia . [Doctoral Dissertation]. University of Otago; 2013. Available from: http://hdl.handle.net/10523/4350


University of Western Ontario

16. Lai-Zhao, Yue. The Role of Synovium and Synovial Macrophages in Experimental Post-Traumatic Knee Osteoarthritis.

Degree: 2020, University of Western Ontario

 In osteoarthritis (OA), synovitis is associated with symptom severity. As synovium secretes both catabolic and anabolic factors into the joint, the impact of synovitis in… (more)

Subjects/Keywords: Animal models of disease; chondrocyte; inflammation; liposomal drug delivery; STAT inhibitors; tissue culture.; Musculoskeletal Diseases

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APA (6th Edition):

Lai-Zhao, Y. (2020). The Role of Synovium and Synovial Macrophages in Experimental Post-Traumatic Knee Osteoarthritis. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/7075

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lai-Zhao, Yue. “The Role of Synovium and Synovial Macrophages in Experimental Post-Traumatic Knee Osteoarthritis.” 2020. Thesis, University of Western Ontario. Accessed April 18, 2021. https://ir.lib.uwo.ca/etd/7075.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lai-Zhao, Yue. “The Role of Synovium and Synovial Macrophages in Experimental Post-Traumatic Knee Osteoarthritis.” 2020. Web. 18 Apr 2021.

Vancouver:

Lai-Zhao Y. The Role of Synovium and Synovial Macrophages in Experimental Post-Traumatic Knee Osteoarthritis. [Internet] [Thesis]. University of Western Ontario; 2020. [cited 2021 Apr 18]. Available from: https://ir.lib.uwo.ca/etd/7075.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lai-Zhao Y. The Role of Synovium and Synovial Macrophages in Experimental Post-Traumatic Knee Osteoarthritis. [Thesis]. University of Western Ontario; 2020. Available from: https://ir.lib.uwo.ca/etd/7075

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

17. Abimosleh, Suzanne Mashtoub. Emu oil promotes intestinal repair in rat models of enteric inflammation.

Degree: 2013, University of Adelaide

 Several disorders of the gastrointestinal (GI) tract including ulcerative colitis, chemotherapy-induced mucositis and non-steroidal anti-inflammatory drug (NSAID)- induced enteropathy, are characterised by inflammation, ulceration, mucosal… (more)

Subjects/Keywords: emu oil; ulcerative colitis; chemotherapy-induced mucositis; NSAID-induced enteropathy; inflammation; intestinal; rat models; repair

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APA (6th Edition):

Abimosleh, S. M. (2013). Emu oil promotes intestinal repair in rat models of enteric inflammation. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/90798

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Abimosleh, Suzanne Mashtoub. “Emu oil promotes intestinal repair in rat models of enteric inflammation.” 2013. Thesis, University of Adelaide. Accessed April 18, 2021. http://hdl.handle.net/2440/90798.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Abimosleh, Suzanne Mashtoub. “Emu oil promotes intestinal repair in rat models of enteric inflammation.” 2013. Web. 18 Apr 2021.

Vancouver:

Abimosleh SM. Emu oil promotes intestinal repair in rat models of enteric inflammation. [Internet] [Thesis]. University of Adelaide; 2013. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2440/90798.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Abimosleh SM. Emu oil promotes intestinal repair in rat models of enteric inflammation. [Thesis]. University of Adelaide; 2013. Available from: http://hdl.handle.net/2440/90798

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

18. Shimp, Samuel Kline. Inhibition of Heat Shock Protein 90 Reduces Inflammatory Signal Transduction in Murine J774 Macrophage Cells and Lessens Disease in Autoimmune MRL/lpr Mice: What in vitro, in vivo, and in silico Models Reveal.

Degree: PhD, Biomedical Engineering, 2012, Virginia Tech

 Heat shock protein 90 (HSP90) is a molecular chaperone protein that protects proteins from degradation, repairs damaged proteins, and assists proteins in carrying out their… (more)

Subjects/Keywords: inflammation; heat shock protein 90; computational models; geldanamycin; 17-DMAG; nuclear factor-κB

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APA (6th Edition):

Shimp, S. K. (2012). Inhibition of Heat Shock Protein 90 Reduces Inflammatory Signal Transduction in Murine J774 Macrophage Cells and Lessens Disease in Autoimmune MRL/lpr Mice: What in vitro, in vivo, and in silico Models Reveal. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/77080

Chicago Manual of Style (16th Edition):

Shimp, Samuel Kline. “Inhibition of Heat Shock Protein 90 Reduces Inflammatory Signal Transduction in Murine J774 Macrophage Cells and Lessens Disease in Autoimmune MRL/lpr Mice: What in vitro, in vivo, and in silico Models Reveal.” 2012. Doctoral Dissertation, Virginia Tech. Accessed April 18, 2021. http://hdl.handle.net/10919/77080.

MLA Handbook (7th Edition):

Shimp, Samuel Kline. “Inhibition of Heat Shock Protein 90 Reduces Inflammatory Signal Transduction in Murine J774 Macrophage Cells and Lessens Disease in Autoimmune MRL/lpr Mice: What in vitro, in vivo, and in silico Models Reveal.” 2012. Web. 18 Apr 2021.

Vancouver:

Shimp SK. Inhibition of Heat Shock Protein 90 Reduces Inflammatory Signal Transduction in Murine J774 Macrophage Cells and Lessens Disease in Autoimmune MRL/lpr Mice: What in vitro, in vivo, and in silico Models Reveal. [Internet] [Doctoral dissertation]. Virginia Tech; 2012. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10919/77080.

Council of Science Editors:

Shimp SK. Inhibition of Heat Shock Protein 90 Reduces Inflammatory Signal Transduction in Murine J774 Macrophage Cells and Lessens Disease in Autoimmune MRL/lpr Mice: What in vitro, in vivo, and in silico Models Reveal. [Doctoral Dissertation]. Virginia Tech; 2012. Available from: http://hdl.handle.net/10919/77080


Linköping University

19. Wu, Simon. Mathematical Modelling of Insulin Resistance Development Caused by Chronic Inflammation.

Degree: Biomedical Engineering, 2019, Linköping University

  Obesity has in recent times become a more serious health issue and was estimated to affect over 650 million people world-wide in 2016. Furthermore,… (more)

Subjects/Keywords: Mathematical models; Modelling; Chronic inflammation; Macrophages; Adipocytes; Insulin resistance; Other Medical Engineering; Annan medicinteknik

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APA (6th Edition):

Wu, S. (2019). Mathematical Modelling of Insulin Resistance Development Caused by Chronic Inflammation. (Thesis). Linköping University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-158671

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Simon. “Mathematical Modelling of Insulin Resistance Development Caused by Chronic Inflammation.” 2019. Thesis, Linköping University. Accessed April 18, 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-158671.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Simon. “Mathematical Modelling of Insulin Resistance Development Caused by Chronic Inflammation.” 2019. Web. 18 Apr 2021.

Vancouver:

Wu S. Mathematical Modelling of Insulin Resistance Development Caused by Chronic Inflammation. [Internet] [Thesis]. Linköping University; 2019. [cited 2021 Apr 18]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-158671.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu S. Mathematical Modelling of Insulin Resistance Development Caused by Chronic Inflammation. [Thesis]. Linköping University; 2019. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-158671

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


East Carolina University

20. McPeek, Matthew. THE ROLE OF THE ALVEOLAR MACROPHAGE IN CARBON NANOTUBE ELICITED MURINE MODEL OF PULMONARY GRANULOMATOUS INFLAMMATION.

Degree: PhD, PHD-Interdisc Biological Sci, 2018, East Carolina University

 Pulmonary sarcoidosis is a debilitating inflammatory condition characterized by the presence of granulomatous lesions throughout the lung. Granulomas are a physiological response to inhaled antigens… (more)

Subjects/Keywords: Pulmonary Sarcoidosis; Lipid Transporters; Macrophages, Alveolar; Mice; Disease Models, Animal; Nanotubes, Carbon; Pneumonia; Granuloma; Inflammation

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APA (6th Edition):

McPeek, M. (2018). THE ROLE OF THE ALVEOLAR MACROPHAGE IN CARBON NANOTUBE ELICITED MURINE MODEL OF PULMONARY GRANULOMATOUS INFLAMMATION. (Doctoral Dissertation). East Carolina University. Retrieved from http://hdl.handle.net/10342/7055

Chicago Manual of Style (16th Edition):

McPeek, Matthew. “THE ROLE OF THE ALVEOLAR MACROPHAGE IN CARBON NANOTUBE ELICITED MURINE MODEL OF PULMONARY GRANULOMATOUS INFLAMMATION.” 2018. Doctoral Dissertation, East Carolina University. Accessed April 18, 2021. http://hdl.handle.net/10342/7055.

MLA Handbook (7th Edition):

McPeek, Matthew. “THE ROLE OF THE ALVEOLAR MACROPHAGE IN CARBON NANOTUBE ELICITED MURINE MODEL OF PULMONARY GRANULOMATOUS INFLAMMATION.” 2018. Web. 18 Apr 2021.

Vancouver:

McPeek M. THE ROLE OF THE ALVEOLAR MACROPHAGE IN CARBON NANOTUBE ELICITED MURINE MODEL OF PULMONARY GRANULOMATOUS INFLAMMATION. [Internet] [Doctoral dissertation]. East Carolina University; 2018. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10342/7055.

Council of Science Editors:

McPeek M. THE ROLE OF THE ALVEOLAR MACROPHAGE IN CARBON NANOTUBE ELICITED MURINE MODEL OF PULMONARY GRANULOMATOUS INFLAMMATION. [Doctoral Dissertation]. East Carolina University; 2018. Available from: http://hdl.handle.net/10342/7055

21. Charlie-Silva, Ives [UNESP]. Imunomodulação da ciclofosfamida na reação inflamatória em tilapias do Nilo (Oreochromis niloticus).

Degree: 2017, Universidade Estadual Paulista (UNESP)

Submitted by Ives Charlie Da Silva ([email protected]) on 2019-11-14T15:04:59Z No. of bitstreams: 2 TESE_ Parcial.pdf: 822621 bytes, checksum: b6a1f19aaea6335859f88df95bb828cc (MD5) Tese_Vet_COMPLETA.pdf: 3549562 bytes, checksum: 8eb7b3c02a7e6b5e23cee46598fbe90c… (more)

Subjects/Keywords: Imunologia; Modelos experimentais; Proteína de fase aguda; Inflamação; Inflammation; Immunology; Experimental models; Acute-phase protein

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APA (6th Edition):

Charlie-Silva, I. [. (2017). Imunomodulação da ciclofosfamida na reação inflamatória em tilapias do Nilo (Oreochromis niloticus). (Doctoral Dissertation). Universidade Estadual Paulista (UNESP). Retrieved from http://hdl.handle.net/11449/191099

Chicago Manual of Style (16th Edition):

Charlie-Silva, Ives [UNESP]. “Imunomodulação da ciclofosfamida na reação inflamatória em tilapias do Nilo (Oreochromis niloticus).” 2017. Doctoral Dissertation, Universidade Estadual Paulista (UNESP). Accessed April 18, 2021. http://hdl.handle.net/11449/191099.

MLA Handbook (7th Edition):

Charlie-Silva, Ives [UNESP]. “Imunomodulação da ciclofosfamida na reação inflamatória em tilapias do Nilo (Oreochromis niloticus).” 2017. Web. 18 Apr 2021.

Vancouver:

Charlie-Silva I[. Imunomodulação da ciclofosfamida na reação inflamatória em tilapias do Nilo (Oreochromis niloticus). [Internet] [Doctoral dissertation]. Universidade Estadual Paulista (UNESP); 2017. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/11449/191099.

Council of Science Editors:

Charlie-Silva I[. Imunomodulação da ciclofosfamida na reação inflamatória em tilapias do Nilo (Oreochromis niloticus). [Doctoral Dissertation]. Universidade Estadual Paulista (UNESP); 2017. Available from: http://hdl.handle.net/11449/191099


Michigan State University

22. Sharma, Deepika. Multifaceted role of beta-arrestins in inflammation.

Degree: 2014, Michigan State University

Thesis Ph. D. Michigan State University. Microbiology and Molecular Genetics 2014.

The overall goal of the study is to understand the role of β-arrestins (intracellular… (more)

Subjects/Keywords: Arrestins – Mechanism of action; Inflammation – Animal models; Septicemia – Pathogenesis; Colitis – Pathogenesis; Immunology; Microbiology; Physiology

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APA (6th Edition):

Sharma, D. (2014). Multifaceted role of beta-arrestins in inflammation. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:2833

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sharma, Deepika. “Multifaceted role of beta-arrestins in inflammation.” 2014. Thesis, Michigan State University. Accessed April 18, 2021. http://etd.lib.msu.edu/islandora/object/etd:2833.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sharma, Deepika. “Multifaceted role of beta-arrestins in inflammation.” 2014. Web. 18 Apr 2021.

Vancouver:

Sharma D. Multifaceted role of beta-arrestins in inflammation. [Internet] [Thesis]. Michigan State University; 2014. [cited 2021 Apr 18]. Available from: http://etd.lib.msu.edu/islandora/object/etd:2833.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sharma D. Multifaceted role of beta-arrestins in inflammation. [Thesis]. Michigan State University; 2014. Available from: http://etd.lib.msu.edu/islandora/object/etd:2833

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Miyamoto, Takeshi. Site-specific elevation of interleukin-1β and matrix metalloproteinase-9 in the Willis circle by hemodynamic changes is associated with rupture in a novel rat cerebral aneurysm model : 新規脳動脈瘤ラットモデルにおける血行力学的変化によるウィリス動脈輪内の部位特異的なIL-1βとMMP-9の上昇は破裂に関連する.

Degree: 博士(医学), 2017, Tokushima University / 徳島大学

Subjects/Keywords: Animal Models; Cerebral Hemodynamics; Inflammation; Intracranial Aneurysm; Subarachnoid Hemorrhage

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APA (6th Edition):

Miyamoto, T. (2017). Site-specific elevation of interleukin-1β and matrix metalloproteinase-9 in the Willis circle by hemodynamic changes is associated with rupture in a novel rat cerebral aneurysm model : 新規脳動脈瘤ラットモデルにおける血行力学的変化によるウィリス動脈輪内の部位特異的なIL-1βとMMP-9の上昇は破裂に関連する. (Thesis). Tokushima University / 徳島大学. Retrieved from http://repo.lib.tokushima-u.ac.jp/110032

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Miyamoto, Takeshi. “Site-specific elevation of interleukin-1β and matrix metalloproteinase-9 in the Willis circle by hemodynamic changes is associated with rupture in a novel rat cerebral aneurysm model : 新規脳動脈瘤ラットモデルにおける血行力学的変化によるウィリス動脈輪内の部位特異的なIL-1βとMMP-9の上昇は破裂に関連する.” 2017. Thesis, Tokushima University / 徳島大学. Accessed April 18, 2021. http://repo.lib.tokushima-u.ac.jp/110032.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Miyamoto, Takeshi. “Site-specific elevation of interleukin-1β and matrix metalloproteinase-9 in the Willis circle by hemodynamic changes is associated with rupture in a novel rat cerebral aneurysm model : 新規脳動脈瘤ラットモデルにおける血行力学的変化によるウィリス動脈輪内の部位特異的なIL-1βとMMP-9の上昇は破裂に関連する.” 2017. Web. 18 Apr 2021.

Vancouver:

Miyamoto T. Site-specific elevation of interleukin-1β and matrix metalloproteinase-9 in the Willis circle by hemodynamic changes is associated with rupture in a novel rat cerebral aneurysm model : 新規脳動脈瘤ラットモデルにおける血行力学的変化によるウィリス動脈輪内の部位特異的なIL-1βとMMP-9の上昇は破裂に関連する. [Internet] [Thesis]. Tokushima University / 徳島大学; 2017. [cited 2021 Apr 18]. Available from: http://repo.lib.tokushima-u.ac.jp/110032.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Miyamoto T. Site-specific elevation of interleukin-1β and matrix metalloproteinase-9 in the Willis circle by hemodynamic changes is associated with rupture in a novel rat cerebral aneurysm model : 新規脳動脈瘤ラットモデルにおける血行力学的変化によるウィリス動脈輪内の部位特異的なIL-1βとMMP-9の上昇は破裂に関連する. [Thesis]. Tokushima University / 徳島大学; 2017. Available from: http://repo.lib.tokushima-u.ac.jp/110032

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Stellenbosch University

24. Mentoor, Ilze. Chemoresistance in a breast cancer animal model: the role of obesity and inflammation.

Degree: PhD, Physiological Sciences, 2019, Stellenbosch University

 ENGLISH ABSTRACT: Background: Globally an increase in obesity incidence represent a major health concern amongst a rising incidence of impaired treatment outcomes of breast cancer… (more)

Subjects/Keywords: Breast  – Cancer  – Chemotherapy; Obesity  – Animal models  – Exercise therapy; Breast  – Cancer  – Treatment  – Evaluation; Fatty acids  – Metabolism; Inflammation  – Diagnosis; Chemotherapy  – Analysis; UCTD

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mentoor, I. (2019). Chemoresistance in a breast cancer animal model: the role of obesity and inflammation. (Doctoral Dissertation). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/107009

Chicago Manual of Style (16th Edition):

Mentoor, Ilze. “Chemoresistance in a breast cancer animal model: the role of obesity and inflammation.” 2019. Doctoral Dissertation, Stellenbosch University. Accessed April 18, 2021. http://hdl.handle.net/10019.1/107009.

MLA Handbook (7th Edition):

Mentoor, Ilze. “Chemoresistance in a breast cancer animal model: the role of obesity and inflammation.” 2019. Web. 18 Apr 2021.

Vancouver:

Mentoor I. Chemoresistance in a breast cancer animal model: the role of obesity and inflammation. [Internet] [Doctoral dissertation]. Stellenbosch University; 2019. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10019.1/107009.

Council of Science Editors:

Mentoor I. Chemoresistance in a breast cancer animal model: the role of obesity and inflammation. [Doctoral Dissertation]. Stellenbosch University; 2019. Available from: http://hdl.handle.net/10019.1/107009


Freie Universität Berlin

25. Elkhrbash, Kamal. Die Rolle von Angiotensin AT1 und AT2 Rezeptoren in der Entwicklung von Apoptose und Entzündung nach einem experimentellen Myokardinfarkt.

Degree: 2015, Freie Universität Berlin

 Die Studie wurde konzipiert, um die Rolle des Angiotensin AT1 Rezeptor-Subtyp 1 (AT1 ) und Subtyp 2 ( AT2 ) Rezeptor in der Apoptose und… (more)

Subjects/Keywords: Angiotensin; receptors; apoptosis; inflammation; myocardial infarction; rats; animal models; 600 Technik, Medizin, angewandte Wissenschaften::630 Landwirtschaft::630 Landwirtschaft und verwandte Bereiche

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APA (6th Edition):

Elkhrbash, K. (2015). Die Rolle von Angiotensin AT1 und AT2 Rezeptoren in der Entwicklung von Apoptose und Entzündung nach einem experimentellen Myokardinfarkt. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-10413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Elkhrbash, Kamal. “Die Rolle von Angiotensin AT1 und AT2 Rezeptoren in der Entwicklung von Apoptose und Entzündung nach einem experimentellen Myokardinfarkt.” 2015. Thesis, Freie Universität Berlin. Accessed April 18, 2021. http://dx.doi.org/10.17169/refubium-10413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Elkhrbash, Kamal. “Die Rolle von Angiotensin AT1 und AT2 Rezeptoren in der Entwicklung von Apoptose und Entzündung nach einem experimentellen Myokardinfarkt.” 2015. Web. 18 Apr 2021.

Vancouver:

Elkhrbash K. Die Rolle von Angiotensin AT1 und AT2 Rezeptoren in der Entwicklung von Apoptose und Entzündung nach einem experimentellen Myokardinfarkt. [Internet] [Thesis]. Freie Universität Berlin; 2015. [cited 2021 Apr 18]. Available from: http://dx.doi.org/10.17169/refubium-10413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Elkhrbash K. Die Rolle von Angiotensin AT1 und AT2 Rezeptoren in der Entwicklung von Apoptose und Entzündung nach einem experimentellen Myokardinfarkt. [Thesis]. Freie Universität Berlin; 2015. Available from: http://dx.doi.org/10.17169/refubium-10413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Paris-Sud – Paris XI

26. Tu, Ly. Innovation thérapeutique dans la dysfonction endothéliale associée à l'hypertension artérielle pulmonaire : Novel molecular targets for correcting endothelial dysfunction associated with pulmonary arterial hypertension.

Degree: Docteur es, Physiopathologie cellulaire et moléculaire, 2012, Université Paris-Sud – Paris XI

L’hypertension artérielle pulmonaire (HTAP) est une maladie grave caractérisée par une obstruction progressive des petites artères pulmonaires, conduisant à une augmentation progressive des résistances vasculaires… (more)

Subjects/Keywords: Dysfonction endothéliale; Facteurs de croissance; Signalisation cellulaire; PAH; Endothelial Dysfunction; Therapeutic Innovations; Growth Factors; Inflammation; Cell Signaling; Proliferation; Apoptosis; Animal Models

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APA (6th Edition):

Tu, L. (2012). Innovation thérapeutique dans la dysfonction endothéliale associée à l'hypertension artérielle pulmonaire : Novel molecular targets for correcting endothelial dysfunction associated with pulmonary arterial hypertension. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2012PA114823

Chicago Manual of Style (16th Edition):

Tu, Ly. “Innovation thérapeutique dans la dysfonction endothéliale associée à l'hypertension artérielle pulmonaire : Novel molecular targets for correcting endothelial dysfunction associated with pulmonary arterial hypertension.” 2012. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed April 18, 2021. http://www.theses.fr/2012PA114823.

MLA Handbook (7th Edition):

Tu, Ly. “Innovation thérapeutique dans la dysfonction endothéliale associée à l'hypertension artérielle pulmonaire : Novel molecular targets for correcting endothelial dysfunction associated with pulmonary arterial hypertension.” 2012. Web. 18 Apr 2021.

Vancouver:

Tu L. Innovation thérapeutique dans la dysfonction endothéliale associée à l'hypertension artérielle pulmonaire : Novel molecular targets for correcting endothelial dysfunction associated with pulmonary arterial hypertension. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2012. [cited 2021 Apr 18]. Available from: http://www.theses.fr/2012PA114823.

Council of Science Editors:

Tu L. Innovation thérapeutique dans la dysfonction endothéliale associée à l'hypertension artérielle pulmonaire : Novel molecular targets for correcting endothelial dysfunction associated with pulmonary arterial hypertension. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2012. Available from: http://www.theses.fr/2012PA114823


University of Gothenburg / Göteborgs Universitet

27. Almén Wranning, Caiza. Surgery and Ischemia in Uterus Transplantation. Studies in various experimental models.

Degree: 2007, University of Gothenburg / Göteborgs Universitet

 The use of transplantation to enhance quality of life is a growing clinical field that also includes transplantation of reproductive organs. Transplantation of the uterus… (more)

Subjects/Keywords: animal models; cold ischemia-reperfusion injury; inflammation; transplantation; uterus

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APA (6th Edition):

Almén Wranning, C. (2007). Surgery and Ischemia in Uterus Transplantation. Studies in various experimental models. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/4753

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Almén Wranning, Caiza. “Surgery and Ischemia in Uterus Transplantation. Studies in various experimental models.” 2007. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed April 18, 2021. http://hdl.handle.net/2077/4753.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Almén Wranning, Caiza. “Surgery and Ischemia in Uterus Transplantation. Studies in various experimental models.” 2007. Web. 18 Apr 2021.

Vancouver:

Almén Wranning C. Surgery and Ischemia in Uterus Transplantation. Studies in various experimental models. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2007. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2077/4753.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Almén Wranning C. Surgery and Ischemia in Uterus Transplantation. Studies in various experimental models. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2007. Available from: http://hdl.handle.net/2077/4753

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Wohn, Christopher. Mechanisms of Psoriatic Plaque Formation in Mice.

Degree: 2015, Erasmus University Medical Center

 markdownabstract__Abstract__ Psoriasis is an inflammatory skin disease of un known etiology. Dendritic cells (DC) are the key antigen-presenting cells of immune system with the capacity… (more)

Subjects/Keywords: Psoriasis; mouse models; dendritic cells; monocytes; IL-17A; inflammation

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APA (6th Edition):

Wohn, C. (2015). Mechanisms of Psoriatic Plaque Formation in Mice. (Doctoral Dissertation). Erasmus University Medical Center. Retrieved from http://hdl.handle.net/1765/78325

Chicago Manual of Style (16th Edition):

Wohn, Christopher. “Mechanisms of Psoriatic Plaque Formation in Mice.” 2015. Doctoral Dissertation, Erasmus University Medical Center. Accessed April 18, 2021. http://hdl.handle.net/1765/78325.

MLA Handbook (7th Edition):

Wohn, Christopher. “Mechanisms of Psoriatic Plaque Formation in Mice.” 2015. Web. 18 Apr 2021.

Vancouver:

Wohn C. Mechanisms of Psoriatic Plaque Formation in Mice. [Internet] [Doctoral dissertation]. Erasmus University Medical Center; 2015. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1765/78325.

Council of Science Editors:

Wohn C. Mechanisms of Psoriatic Plaque Formation in Mice. [Doctoral Dissertation]. Erasmus University Medical Center; 2015. Available from: http://hdl.handle.net/1765/78325


Stellenbosch University

29. Boodhoo, Kiara. A comparison of the wound healing dynamics in an acute and newly developed chronic wound model using Neutral endopeptidase, an inhibitor of Substance P.

Degree: MSc, Physiological Sciences, 2020, Stellenbosch University

 ENGLISH ABSTRACT: Chronic wounds affect millions of people with diabetes world-wide. Various diabetic wound healing animal models exist, none of which currently replicate chronic wounds… (more)

Subjects/Keywords: Diabetes; Chronic wounds; Substance P  – Physiological effect; Inflammation  – Mediators; Neutral endopeptidase; Diabetes  – Treatment  – Animal models; Diabetes  – Patients; UCTD

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APA (6th Edition):

Boodhoo, K. (2020). A comparison of the wound healing dynamics in an acute and newly developed chronic wound model using Neutral endopeptidase, an inhibitor of Substance P. (Masters Thesis). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/108407

Chicago Manual of Style (16th Edition):

Boodhoo, Kiara. “A comparison of the wound healing dynamics in an acute and newly developed chronic wound model using Neutral endopeptidase, an inhibitor of Substance P.” 2020. Masters Thesis, Stellenbosch University. Accessed April 18, 2021. http://hdl.handle.net/10019.1/108407.

MLA Handbook (7th Edition):

Boodhoo, Kiara. “A comparison of the wound healing dynamics in an acute and newly developed chronic wound model using Neutral endopeptidase, an inhibitor of Substance P.” 2020. Web. 18 Apr 2021.

Vancouver:

Boodhoo K. A comparison of the wound healing dynamics in an acute and newly developed chronic wound model using Neutral endopeptidase, an inhibitor of Substance P. [Internet] [Masters thesis]. Stellenbosch University; 2020. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10019.1/108407.

Council of Science Editors:

Boodhoo K. A comparison of the wound healing dynamics in an acute and newly developed chronic wound model using Neutral endopeptidase, an inhibitor of Substance P. [Masters Thesis]. Stellenbosch University; 2020. Available from: http://hdl.handle.net/10019.1/108407


Rutgers University

30. Baldassari, James, 1977-. Effects of quercetin on exercise potential and exercise-induced cytokines in the horse.

Degree: MS, Animal Sciences, 2011, Rutgers University

Inflammation, the common physiological response to a wide variety of injuries and irritations, has been studied in one form or another for literally thousands of… (more)

Subjects/Keywords: Equine sports medicine; Inflammation – Animal models; Cytokines

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APA (6th Edition):

Baldassari, James, 1. (2011). Effects of quercetin on exercise potential and exercise-induced cytokines in the horse. (Masters Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057505

Chicago Manual of Style (16th Edition):

Baldassari, James, 1977-. “Effects of quercetin on exercise potential and exercise-induced cytokines in the horse.” 2011. Masters Thesis, Rutgers University. Accessed April 18, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057505.

MLA Handbook (7th Edition):

Baldassari, James, 1977-. “Effects of quercetin on exercise potential and exercise-induced cytokines in the horse.” 2011. Web. 18 Apr 2021.

Vancouver:

Baldassari, James 1. Effects of quercetin on exercise potential and exercise-induced cytokines in the horse. [Internet] [Masters thesis]. Rutgers University; 2011. [cited 2021 Apr 18]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057505.

Council of Science Editors:

Baldassari, James 1. Effects of quercetin on exercise potential and exercise-induced cytokines in the horse. [Masters Thesis]. Rutgers University; 2011. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057505

[1] [2] [3] [4]

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