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You searched for subject:(Inflammasome). Showing records 1 – 11 of 11 total matches.

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Universiteit Utrecht

1. Diebolder, C.A. Structural studies by cryo-electron tomography of two initiation complexes of the immune system: C1-IgG6 and NAIP5-NLRC4.

Degree: 2015, Universiteit Utrecht

 Complement activation by antibodies bound to pathogens, tumors, and self antigens is a critical feature of natural immune defense, a number of disease processes, and… (more)

Subjects/Keywords: cryo-electron tomography; complement; IgG-Hexamer; inflammasome; conical Fourier shell correlation

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APA (6th Edition):

Diebolder, C. A. (2015). Structural studies by cryo-electron tomography of two initiation complexes of the immune system: C1-IgG6 and NAIP5-NLRC4. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/321426

Chicago Manual of Style (16th Edition):

Diebolder, C A. “Structural studies by cryo-electron tomography of two initiation complexes of the immune system: C1-IgG6 and NAIP5-NLRC4.” 2015. Doctoral Dissertation, Universiteit Utrecht. Accessed April 10, 2020. http://dspace.library.uu.nl:8080/handle/1874/321426.

MLA Handbook (7th Edition):

Diebolder, C A. “Structural studies by cryo-electron tomography of two initiation complexes of the immune system: C1-IgG6 and NAIP5-NLRC4.” 2015. Web. 10 Apr 2020.

Vancouver:

Diebolder CA. Structural studies by cryo-electron tomography of two initiation complexes of the immune system: C1-IgG6 and NAIP5-NLRC4. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2015. [cited 2020 Apr 10]. Available from: http://dspace.library.uu.nl:8080/handle/1874/321426.

Council of Science Editors:

Diebolder CA. Structural studies by cryo-electron tomography of two initiation complexes of the immune system: C1-IgG6 and NAIP5-NLRC4. [Doctoral Dissertation]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/321426


Universiteit Utrecht

2. Burgh, Robert van der. Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation.

Degree: 2015, Universiteit Utrecht

 Autoinflammatory disease (AID) is the uncontrolled and recurrent activation of the innate immune response, followed by complete resolution of the inflammation. Patients suffer from recurring… (more)

Subjects/Keywords: Autoinflammation; mitochondria; MKD; autoinflammatory-disease; Serpinb9; isoprenoid; interleukin-1β; inflammasome; caspase-1; GTPase

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APA (6th Edition):

Burgh, R. v. d. (2015). Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/317827

Chicago Manual of Style (16th Edition):

Burgh, Robert van der. “Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation.” 2015. Doctoral Dissertation, Universiteit Utrecht. Accessed April 10, 2020. http://dspace.library.uu.nl:8080/handle/1874/317827.

MLA Handbook (7th Edition):

Burgh, Robert van der. “Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation.” 2015. Web. 10 Apr 2020.

Vancouver:

Burgh Rvd. Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2015. [cited 2020 Apr 10]. Available from: http://dspace.library.uu.nl:8080/handle/1874/317827.

Council of Science Editors:

Burgh Rvd. Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation. [Doctoral Dissertation]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/317827


Universiteit Utrecht

3. Kuiper, J.J.W. Reactive Oxygen Species in Inflammasome Activation.

Degree: 2009, Universiteit Utrecht

 The fundamental role of the innate immune system is to form the first barrier in bacterial and viral defence. It is based upon the recognition… (more)

Subjects/Keywords: Geneeskunde; NOD-like receptor, P2x7-receptor, NLRP3, Reactive Oxygen Species, ROS, Caspase-1, IL-1beta, IL-18, Inflammasome, ERK1/2, Cryopyrinopathies, CAPS

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APA (6th Edition):

Kuiper, J. J. W. (2009). Reactive Oxygen Species in Inflammasome Activation. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/35211

Chicago Manual of Style (16th Edition):

Kuiper, J J W. “Reactive Oxygen Species in Inflammasome Activation.” 2009. Masters Thesis, Universiteit Utrecht. Accessed April 10, 2020. http://dspace.library.uu.nl:8080/handle/1874/35211.

MLA Handbook (7th Edition):

Kuiper, J J W. “Reactive Oxygen Species in Inflammasome Activation.” 2009. Web. 10 Apr 2020.

Vancouver:

Kuiper JJW. Reactive Oxygen Species in Inflammasome Activation. [Internet] [Masters thesis]. Universiteit Utrecht; 2009. [cited 2020 Apr 10]. Available from: http://dspace.library.uu.nl:8080/handle/1874/35211.

Council of Science Editors:

Kuiper JJW. Reactive Oxygen Species in Inflammasome Activation. [Masters Thesis]. Universiteit Utrecht; 2009. Available from: http://dspace.library.uu.nl:8080/handle/1874/35211

4. Chouri, E. Role of mitochondria in the development of autoinflammatory diseases.

Degree: 2014, Universiteit Utrecht

 Autoinflammatory diseases are hereditary inflammatory syndromes, which are characterized by unprovoked high activation of the innate immune response. The myeloid effector cells and the germline-encoded… (more)

Subjects/Keywords: mitochondria; NLPR3 inflammasome; innate immune system; autoinflammatory diseases

…demonstrated that ATP stimulation triggers ROS production and therefore NLPR3 inflammasome activation… …al 2008). When ROS production is inhibited, NLPR3 inflammasome activation is blocked… …inflammasome activation, as patients with chronic granulomatous diseases (CGDs), who cannot… …generate NADPH-dependent ROS, demonstrate increased inflammasome activation and also high levels… …inflammasome activation. Mitochondrial oxidative phosphorylation is a major source of ROS. Indeed, a… 

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APA (6th Edition):

Chouri, E. (2014). Role of mitochondria in the development of autoinflammatory diseases. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/291003

Chicago Manual of Style (16th Edition):

Chouri, E. “Role of mitochondria in the development of autoinflammatory diseases.” 2014. Masters Thesis, Universiteit Utrecht. Accessed April 10, 2020. http://dspace.library.uu.nl:8080/handle/1874/291003.

MLA Handbook (7th Edition):

Chouri, E. “Role of mitochondria in the development of autoinflammatory diseases.” 2014. Web. 10 Apr 2020.

Vancouver:

Chouri E. Role of mitochondria in the development of autoinflammatory diseases. [Internet] [Masters thesis]. Universiteit Utrecht; 2014. [cited 2020 Apr 10]. Available from: http://dspace.library.uu.nl:8080/handle/1874/291003.

Council of Science Editors:

Chouri E. Role of mitochondria in the development of autoinflammatory diseases. [Masters Thesis]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/291003

5. Burgh, Robert van der. Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation.

Degree: 2015, University Utrecht

 Autoinflammatory disease (AID) is the uncontrolled and recurrent activation of the innate immune response, followed by complete resolution of the inflammation. Patients suffer from recurring… (more)

Subjects/Keywords: Autoinflammation; mitochondria; MKD; autoinflammatory-disease; Serpinb9; isoprenoid; interleukin-1β; inflammasome; caspase-1; GTPase

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APA (6th Edition):

Burgh, R. v. d. (2015). Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/317827 ; URN:NBN:NL:UI:10-1874-317827 ; urn:isbn:978-94-6295-178-5 ; URN:NBN:NL:UI:10-1874-317827 ; http://dspace.library.uu.nl/handle/1874/317827

Chicago Manual of Style (16th Edition):

Burgh, Robert van der. “Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation.” 2015. Doctoral Dissertation, University Utrecht. Accessed April 10, 2020. http://dspace.library.uu.nl/handle/1874/317827 ; URN:NBN:NL:UI:10-1874-317827 ; urn:isbn:978-94-6295-178-5 ; URN:NBN:NL:UI:10-1874-317827 ; http://dspace.library.uu.nl/handle/1874/317827.

MLA Handbook (7th Edition):

Burgh, Robert van der. “Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation.” 2015. Web. 10 Apr 2020.

Vancouver:

Burgh Rvd. Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation. [Internet] [Doctoral dissertation]. University Utrecht; 2015. [cited 2020 Apr 10]. Available from: http://dspace.library.uu.nl/handle/1874/317827 ; URN:NBN:NL:UI:10-1874-317827 ; urn:isbn:978-94-6295-178-5 ; URN:NBN:NL:UI:10-1874-317827 ; http://dspace.library.uu.nl/handle/1874/317827.

Council of Science Editors:

Burgh Rvd. Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation. [Doctoral Dissertation]. University Utrecht; 2015. Available from: http://dspace.library.uu.nl/handle/1874/317827 ; URN:NBN:NL:UI:10-1874-317827 ; urn:isbn:978-94-6295-178-5 ; URN:NBN:NL:UI:10-1874-317827 ; http://dspace.library.uu.nl/handle/1874/317827

6. Bosch, Lena. Cooling down the inflamed heart.

Degree: 2018, University Utrecht

 Inflammation at the right time and adequate intensity is essential for cardiac repair after an acute myocardial infarction (MI). However, resolution of the inflammatory response… (more)

Subjects/Keywords: cardiovascular disease; heart failure; inflammation; inflammasome; animal models; transverse aortic constriction

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APA (6th Edition):

Bosch, L. (2018). Cooling down the inflamed heart. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/369698 ; URN:NBN:NL:UI:10-1874-369698 ; urn:isbn:978-94-9301-401-5 ; URN:NBN:NL:UI:10-1874-369698 ; http://dspace.library.uu.nl/handle/1874/369698

Chicago Manual of Style (16th Edition):

Bosch, Lena. “Cooling down the inflamed heart.” 2018. Doctoral Dissertation, University Utrecht. Accessed April 10, 2020. http://dspace.library.uu.nl/handle/1874/369698 ; URN:NBN:NL:UI:10-1874-369698 ; urn:isbn:978-94-9301-401-5 ; URN:NBN:NL:UI:10-1874-369698 ; http://dspace.library.uu.nl/handle/1874/369698.

MLA Handbook (7th Edition):

Bosch, Lena. “Cooling down the inflamed heart.” 2018. Web. 10 Apr 2020.

Vancouver:

Bosch L. Cooling down the inflamed heart. [Internet] [Doctoral dissertation]. University Utrecht; 2018. [cited 2020 Apr 10]. Available from: http://dspace.library.uu.nl/handle/1874/369698 ; URN:NBN:NL:UI:10-1874-369698 ; urn:isbn:978-94-9301-401-5 ; URN:NBN:NL:UI:10-1874-369698 ; http://dspace.library.uu.nl/handle/1874/369698.

Council of Science Editors:

Bosch L. Cooling down the inflamed heart. [Doctoral Dissertation]. University Utrecht; 2018. Available from: http://dspace.library.uu.nl/handle/1874/369698 ; URN:NBN:NL:UI:10-1874-369698 ; urn:isbn:978-94-9301-401-5 ; URN:NBN:NL:UI:10-1874-369698 ; http://dspace.library.uu.nl/handle/1874/369698

7. Burgh, Robert van der. Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation.

Degree: 2015, University Utrecht

 Autoinflammatory disease (AID) is the uncontrolled and recurrent activation of the innate immune response, followed by complete resolution of the inflammation. Patients suffer from recurring… (more)

Subjects/Keywords: Autoinflammation; mitochondria; MKD; autoinflammatory-disease; Serpinb9; isoprenoid; interleukin-1β; inflammasome; caspase-1; GTPase

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APA (6th Edition):

Burgh, R. v. d. (2015). Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/317827 ; URN:NBN:NL:UI:10-1874-317827 ; urn:isbn:978-94-6295-178-5 ; URN:NBN:NL:UI:10-1874-317827 ; http://dspace.library.uu.nl/handle/1874/317827

Chicago Manual of Style (16th Edition):

Burgh, Robert van der. “Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation.” 2015. Doctoral Dissertation, University Utrecht. Accessed April 10, 2020. http://dspace.library.uu.nl/handle/1874/317827 ; URN:NBN:NL:UI:10-1874-317827 ; urn:isbn:978-94-6295-178-5 ; URN:NBN:NL:UI:10-1874-317827 ; http://dspace.library.uu.nl/handle/1874/317827.

MLA Handbook (7th Edition):

Burgh, Robert van der. “Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation.” 2015. Web. 10 Apr 2020.

Vancouver:

Burgh Rvd. Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation. [Internet] [Doctoral dissertation]. University Utrecht; 2015. [cited 2020 Apr 10]. Available from: http://dspace.library.uu.nl/handle/1874/317827 ; URN:NBN:NL:UI:10-1874-317827 ; urn:isbn:978-94-6295-178-5 ; URN:NBN:NL:UI:10-1874-317827 ; http://dspace.library.uu.nl/handle/1874/317827.

Council of Science Editors:

Burgh Rvd. Mechanisms that Kindle Disease in Periodic Hereditary Diseases : How Isoprenoid Deficiency and SerpinB9 Mediate Autoinflammation. [Doctoral Dissertation]. University Utrecht; 2015. Available from: http://dspace.library.uu.nl/handle/1874/317827 ; URN:NBN:NL:UI:10-1874-317827 ; urn:isbn:978-94-6295-178-5 ; URN:NBN:NL:UI:10-1874-317827 ; http://dspace.library.uu.nl/handle/1874/317827

8. Diebolder, C.A. Structural studies by cryo-electron tomography of two initiation complexes of the immune system: C1-IgG6 and NAIP5-NLRC4.

Degree: 2015, University Utrecht

 Complement activation by antibodies bound to pathogens, tumors, and self antigens is a critical feature of natural immune defense, a number of disease processes, and… (more)

Subjects/Keywords: cryo-electron tomography; complement; IgG-Hexamer; inflammasome; conical Fourier shell correlation

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APA (6th Edition):

Diebolder, C. A. (2015). Structural studies by cryo-electron tomography of two initiation complexes of the immune system: C1-IgG6 and NAIP5-NLRC4. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/321426 ; URN:NBN:NL:UI:10-1874-321426 ; urn:isbn:978-94-6295-346-8 ; URN:NBN:NL:UI:10-1874-321426 ; http://dspace.library.uu.nl/handle/1874/321426

Chicago Manual of Style (16th Edition):

Diebolder, C A. “Structural studies by cryo-electron tomography of two initiation complexes of the immune system: C1-IgG6 and NAIP5-NLRC4.” 2015. Doctoral Dissertation, University Utrecht. Accessed April 10, 2020. http://dspace.library.uu.nl/handle/1874/321426 ; URN:NBN:NL:UI:10-1874-321426 ; urn:isbn:978-94-6295-346-8 ; URN:NBN:NL:UI:10-1874-321426 ; http://dspace.library.uu.nl/handle/1874/321426.

MLA Handbook (7th Edition):

Diebolder, C A. “Structural studies by cryo-electron tomography of two initiation complexes of the immune system: C1-IgG6 and NAIP5-NLRC4.” 2015. Web. 10 Apr 2020.

Vancouver:

Diebolder CA. Structural studies by cryo-electron tomography of two initiation complexes of the immune system: C1-IgG6 and NAIP5-NLRC4. [Internet] [Doctoral dissertation]. University Utrecht; 2015. [cited 2020 Apr 10]. Available from: http://dspace.library.uu.nl/handle/1874/321426 ; URN:NBN:NL:UI:10-1874-321426 ; urn:isbn:978-94-6295-346-8 ; URN:NBN:NL:UI:10-1874-321426 ; http://dspace.library.uu.nl/handle/1874/321426.

Council of Science Editors:

Diebolder CA. Structural studies by cryo-electron tomography of two initiation complexes of the immune system: C1-IgG6 and NAIP5-NLRC4. [Doctoral Dissertation]. University Utrecht; 2015. Available from: http://dspace.library.uu.nl/handle/1874/321426 ; URN:NBN:NL:UI:10-1874-321426 ; urn:isbn:978-94-6295-346-8 ; URN:NBN:NL:UI:10-1874-321426 ; http://dspace.library.uu.nl/handle/1874/321426

9. Burm, S.M. Inflammasome-mediated activation of microglia : Tissue-specific features of innate immunity.

Degree: 2016, University Utrecht

 Multiple sclerosis (MS) is a chronic neurodegenerative disease where lesions are found within the brain. Although the exact cause of MS is unknown, these lesions… (more)

Subjects/Keywords: Microglia; macrophages; tissue-specific; inflammasome; IL-1β; CNS; multiple sclerosis; 3R alternatives

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APA (6th Edition):

Burm, S. M. (2016). Inflammasome-mediated activation of microglia : Tissue-specific features of innate immunity. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/340540 ; URN:NBN:NL:UI:10-1874-340540 ; urn:isbn:978-94-6299-335-8 ; URN:NBN:NL:UI:10-1874-340540 ; http://dspace.library.uu.nl/handle/1874/340540

Chicago Manual of Style (16th Edition):

Burm, S M. “Inflammasome-mediated activation of microglia : Tissue-specific features of innate immunity.” 2016. Doctoral Dissertation, University Utrecht. Accessed April 10, 2020. http://dspace.library.uu.nl/handle/1874/340540 ; URN:NBN:NL:UI:10-1874-340540 ; urn:isbn:978-94-6299-335-8 ; URN:NBN:NL:UI:10-1874-340540 ; http://dspace.library.uu.nl/handle/1874/340540.

MLA Handbook (7th Edition):

Burm, S M. “Inflammasome-mediated activation of microglia : Tissue-specific features of innate immunity.” 2016. Web. 10 Apr 2020.

Vancouver:

Burm SM. Inflammasome-mediated activation of microglia : Tissue-specific features of innate immunity. [Internet] [Doctoral dissertation]. University Utrecht; 2016. [cited 2020 Apr 10]. Available from: http://dspace.library.uu.nl/handle/1874/340540 ; URN:NBN:NL:UI:10-1874-340540 ; urn:isbn:978-94-6299-335-8 ; URN:NBN:NL:UI:10-1874-340540 ; http://dspace.library.uu.nl/handle/1874/340540.

Council of Science Editors:

Burm SM. Inflammasome-mediated activation of microglia : Tissue-specific features of innate immunity. [Doctoral Dissertation]. University Utrecht; 2016. Available from: http://dspace.library.uu.nl/handle/1874/340540 ; URN:NBN:NL:UI:10-1874-340540 ; urn:isbn:978-94-6299-335-8 ; URN:NBN:NL:UI:10-1874-340540 ; http://dspace.library.uu.nl/handle/1874/340540

10. Bouwman, L.I. Campylobacter jejuni & Inflammation : Grilling the pathogen.

Degree: 2016, University Utrecht

 Campylobacter jejuni is the most common cause of bacterial foodborne disease. Yet, little is known about how this pathogen causes intestinal inflammation. The clinical pathology… (more)

Subjects/Keywords: Campylobacter jejuni; enteropathogenic bacteria; inflammation; innate; invasion; beta-glucan; inflammasome; intestine

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APA (6th Edition):

Bouwman, L. I. (2016). Campylobacter jejuni & Inflammation : Grilling the pathogen. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/331187 ; URN:NBN:NL:UI:10-1874-331187 ; urn:isbn:978-94-6182-664-0 ; URN:NBN:NL:UI:10-1874-331187 ; http://dspace.library.uu.nl/handle/1874/331187

Chicago Manual of Style (16th Edition):

Bouwman, L I. “Campylobacter jejuni & Inflammation : Grilling the pathogen.” 2016. Doctoral Dissertation, University Utrecht. Accessed April 10, 2020. http://dspace.library.uu.nl/handle/1874/331187 ; URN:NBN:NL:UI:10-1874-331187 ; urn:isbn:978-94-6182-664-0 ; URN:NBN:NL:UI:10-1874-331187 ; http://dspace.library.uu.nl/handle/1874/331187.

MLA Handbook (7th Edition):

Bouwman, L I. “Campylobacter jejuni & Inflammation : Grilling the pathogen.” 2016. Web. 10 Apr 2020.

Vancouver:

Bouwman LI. Campylobacter jejuni & Inflammation : Grilling the pathogen. [Internet] [Doctoral dissertation]. University Utrecht; 2016. [cited 2020 Apr 10]. Available from: http://dspace.library.uu.nl/handle/1874/331187 ; URN:NBN:NL:UI:10-1874-331187 ; urn:isbn:978-94-6182-664-0 ; URN:NBN:NL:UI:10-1874-331187 ; http://dspace.library.uu.nl/handle/1874/331187.

Council of Science Editors:

Bouwman LI. Campylobacter jejuni & Inflammation : Grilling the pathogen. [Doctoral Dissertation]. University Utrecht; 2016. Available from: http://dspace.library.uu.nl/handle/1874/331187 ; URN:NBN:NL:UI:10-1874-331187 ; urn:isbn:978-94-6182-664-0 ; URN:NBN:NL:UI:10-1874-331187 ; http://dspace.library.uu.nl/handle/1874/331187

11. Bosch, Lena. Cooling down the inflamed heart.

Degree: 2018, University Utrecht

 Inflammation at the right time and adequate intensity is essential for cardiac repair after an acute myocardial infarction (MI). However, resolution of the inflammatory response… (more)

Subjects/Keywords: cardiovascular disease; heart failure; inflammation; inflammasome; animal models; transverse aortic constriction

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APA (6th Edition):

Bosch, L. (2018). Cooling down the inflamed heart. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/369698 ; URN:NBN:NL:UI:10-1874-369698 ; urn:isbn:978-94-9301-401-5 ; URN:NBN:NL:UI:10-1874-369698 ; http://dspace.library.uu.nl/handle/1874/369698

Chicago Manual of Style (16th Edition):

Bosch, Lena. “Cooling down the inflamed heart.” 2018. Doctoral Dissertation, University Utrecht. Accessed April 10, 2020. http://dspace.library.uu.nl/handle/1874/369698 ; URN:NBN:NL:UI:10-1874-369698 ; urn:isbn:978-94-9301-401-5 ; URN:NBN:NL:UI:10-1874-369698 ; http://dspace.library.uu.nl/handle/1874/369698.

MLA Handbook (7th Edition):

Bosch, Lena. “Cooling down the inflamed heart.” 2018. Web. 10 Apr 2020.

Vancouver:

Bosch L. Cooling down the inflamed heart. [Internet] [Doctoral dissertation]. University Utrecht; 2018. [cited 2020 Apr 10]. Available from: http://dspace.library.uu.nl/handle/1874/369698 ; URN:NBN:NL:UI:10-1874-369698 ; urn:isbn:978-94-9301-401-5 ; URN:NBN:NL:UI:10-1874-369698 ; http://dspace.library.uu.nl/handle/1874/369698.

Council of Science Editors:

Bosch L. Cooling down the inflamed heart. [Doctoral Dissertation]. University Utrecht; 2018. Available from: http://dspace.library.uu.nl/handle/1874/369698 ; URN:NBN:NL:UI:10-1874-369698 ; urn:isbn:978-94-9301-401-5 ; URN:NBN:NL:UI:10-1874-369698 ; http://dspace.library.uu.nl/handle/1874/369698

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