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You searched for subject:(Inflammasome). Showing records 1 – 30 of 188 total matches.

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1. Riteau, Nicolas. Immunité innée et inflammasome : rôle des signaux de dangers endogènes : Innate immunity and inflammasome : role of endogenous danger signals.

Degree: Docteur es, Science du vivant. Immunologie, 2011, Université d'Orléans

La théorie du danger développée par Polly Matzinger stipule que l’attrait principal du système immunitaire ne réside pas dans la distinction entre le soi «… (more)

Subjects/Keywords: Inflammasome; Inflammasome

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Riteau, N. (2011). Immunité innée et inflammasome : rôle des signaux de dangers endogènes : Innate immunity and inflammasome : role of endogenous danger signals. (Doctoral Dissertation). Université d'Orléans. Retrieved from http://www.theses.fr/2011ORLE2038

Chicago Manual of Style (16th Edition):

Riteau, Nicolas. “Immunité innée et inflammasome : rôle des signaux de dangers endogènes : Innate immunity and inflammasome : role of endogenous danger signals.” 2011. Doctoral Dissertation, Université d'Orléans. Accessed February 16, 2020. http://www.theses.fr/2011ORLE2038.

MLA Handbook (7th Edition):

Riteau, Nicolas. “Immunité innée et inflammasome : rôle des signaux de dangers endogènes : Innate immunity and inflammasome : role of endogenous danger signals.” 2011. Web. 16 Feb 2020.

Vancouver:

Riteau N. Immunité innée et inflammasome : rôle des signaux de dangers endogènes : Innate immunity and inflammasome : role of endogenous danger signals. [Internet] [Doctoral dissertation]. Université d'Orléans; 2011. [cited 2020 Feb 16]. Available from: http://www.theses.fr/2011ORLE2038.

Council of Science Editors:

Riteau N. Immunité innée et inflammasome : rôle des signaux de dangers endogènes : Innate immunity and inflammasome : role of endogenous danger signals. [Doctoral Dissertation]. Université d'Orléans; 2011. Available from: http://www.theses.fr/2011ORLE2038

2. Molyvdas, Adam. Το NLRP3 φλεγμονόσωμα στην χρόνια ιογενή ηπατική φλεγμονή.

Degree: 2017, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας

Background: Chronic viral hepatitis is a prevalent disease with major health implications. The mechanisms that are involved in the pathophysiology of chronic viral hepatitis are… (more)

Subjects/Keywords: Φλεγμονόσωμα; Ηπατίτιτδα; inflammasome; Hepatitis

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APA (6th Edition):

Molyvdas, A. (2017). Το NLRP3 φλεγμονόσωμα στην χρόνια ιογενή ηπατική φλεγμονή. (Thesis). University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Retrieved from http://hdl.handle.net/10442/hedi/41928

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Molyvdas, Adam. “Το NLRP3 φλεγμονόσωμα στην χρόνια ιογενή ηπατική φλεγμονή.” 2017. Thesis, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Accessed February 16, 2020. http://hdl.handle.net/10442/hedi/41928.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Molyvdas, Adam. “Το NLRP3 φλεγμονόσωμα στην χρόνια ιογενή ηπατική φλεγμονή.” 2017. Web. 16 Feb 2020.

Vancouver:

Molyvdas A. Το NLRP3 φλεγμονόσωμα στην χρόνια ιογενή ηπατική φλεγμονή. [Internet] [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2017. [cited 2020 Feb 16]. Available from: http://hdl.handle.net/10442/hedi/41928.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Molyvdas A. Το NLRP3 φλεγμονόσωμα στην χρόνια ιογενή ηπατική φλεγμονή. [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2017. Available from: http://hdl.handle.net/10442/hedi/41928

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

3. Nordlander, Sofia. Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation.

Degree: PhD, 2013, University of Oxford

 Flagellin is a highly immunogenic, bacterial protein considered to be abundant in the intestinal lumen. It has been reported to be an immunodominant antigen in… (more)

Subjects/Keywords: 616.07; Immunology; Mucosal immunology; inflammasome

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APA (6th Edition):

Nordlander, S. (2013). Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:775c4150-9b22-4008-9288-3a6053cbf4cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647535

Chicago Manual of Style (16th Edition):

Nordlander, Sofia. “Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation.” 2013. Doctoral Dissertation, University of Oxford. Accessed February 16, 2020. http://ora.ox.ac.uk/objects/uuid:775c4150-9b22-4008-9288-3a6053cbf4cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647535.

MLA Handbook (7th Edition):

Nordlander, Sofia. “Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation.” 2013. Web. 16 Feb 2020.

Vancouver:

Nordlander S. Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2020 Feb 16]. Available from: http://ora.ox.ac.uk/objects/uuid:775c4150-9b22-4008-9288-3a6053cbf4cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647535.

Council of Science Editors:

Nordlander S. Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:775c4150-9b22-4008-9288-3a6053cbf4cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647535


University of Texas Medical Branch – Galveston

4. [No author]. The Activation of the Inflammasome during Rickettsial Infection .

Degree: University of Texas Medical Branch – Galveston

 Rickettsiae are Gram-negative, obligately intracellular bacteria that infect endothelial cells and macrophages. The molecular mechanisms involved in interaction of rickettsiae with macrophages, however, remain poorly… (more)

Subjects/Keywords: Rickettsia; Inflammasome

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APA (6th Edition):

author], [. (n.d.). The Activation of the Inflammasome during Rickettsial Infection . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/11179

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “The Activation of the Inflammasome during Rickettsial Infection .” Thesis, University of Texas Medical Branch – Galveston. Accessed February 16, 2020. http://hdl.handle.net/2152.3/11179.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “The Activation of the Inflammasome during Rickettsial Infection .” Web. 16 Feb 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

author] [. The Activation of the Inflammasome during Rickettsial Infection . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; [cited 2020 Feb 16]. Available from: http://hdl.handle.net/2152.3/11179.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

author] [. The Activation of the Inflammasome during Rickettsial Infection . [Thesis]. University of Texas Medical Branch – Galveston; Available from: http://hdl.handle.net/2152.3/11179

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Newcastle

5. Williams, Evan James. Strategies for reducing systemic inflammation in obesity.

Degree: PhD, 2019, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

The prevalence of overweight and obese individuals in Australia is currently 63% of adults according to current data… (more)

Subjects/Keywords: obesity; systemic inflammation; inflammasome; macrophages

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APA (6th Edition):

Williams, E. J. (2019). Strategies for reducing systemic inflammation in obesity. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1401110

Chicago Manual of Style (16th Edition):

Williams, Evan James. “Strategies for reducing systemic inflammation in obesity.” 2019. Doctoral Dissertation, University of Newcastle. Accessed February 16, 2020. http://hdl.handle.net/1959.13/1401110.

MLA Handbook (7th Edition):

Williams, Evan James. “Strategies for reducing systemic inflammation in obesity.” 2019. Web. 16 Feb 2020.

Vancouver:

Williams EJ. Strategies for reducing systemic inflammation in obesity. [Internet] [Doctoral dissertation]. University of Newcastle; 2019. [cited 2020 Feb 16]. Available from: http://hdl.handle.net/1959.13/1401110.

Council of Science Editors:

Williams EJ. Strategies for reducing systemic inflammation in obesity. [Doctoral Dissertation]. University of Newcastle; 2019. Available from: http://hdl.handle.net/1959.13/1401110

6. Groslambert, Marine. Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation.

Degree: Docteur es, Sciences de la Vie, 2019, Lyon

L'inflammation est un processus déclenché suite à la détection de pathogènes et de dommages tissulaires. Elle conduit à la sécrétion de cytokines pro-inflammatoires par les… (more)

Subjects/Keywords: NLRP3; Inflammasome; Ubiquitination; Phosphorylation; NLRP3; Inflammasome; Ubiquitination; Phosphorylation

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APA (6th Edition):

Groslambert, M. (2019). Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2019LYSEN022

Chicago Manual of Style (16th Edition):

Groslambert, Marine. “Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation.” 2019. Doctoral Dissertation, Lyon. Accessed February 16, 2020. http://www.theses.fr/2019LYSEN022.

MLA Handbook (7th Edition):

Groslambert, Marine. “Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation.” 2019. Web. 16 Feb 2020.

Vancouver:

Groslambert M. Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation. [Internet] [Doctoral dissertation]. Lyon; 2019. [cited 2020 Feb 16]. Available from: http://www.theses.fr/2019LYSEN022.

Council of Science Editors:

Groslambert M. Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation. [Doctoral Dissertation]. Lyon; 2019. Available from: http://www.theses.fr/2019LYSEN022

7. Paoletti, Audrey. Étude des étapes précoces de l’infection par le VIH-1 : identification d’un nouveau point de contrôle immunitaire immunitaire impliquant le récepteur P2Y2 et la protéine NLRP3 : Study Of The Early Steps Of HIV-1 Infection : Identification Of A New Immune Checkpoint Involving P2Y2 And NLRP3.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2015, Université Paris-Saclay (ComUE)

Plus de 34 millions de personnes dans le monde vivent avec le virus de l’immunodéficience humaine de type 1 (VIH-1). Cette pandémie est partiellement contrôlée… (more)

Subjects/Keywords: Vih-1; Récepteur purinergique; Inflammasome; Hiv-1; Purinergic receptor; Inflammasome

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APA (6th Edition):

Paoletti, A. (2015). Étude des étapes précoces de l’infection par le VIH-1 : identification d’un nouveau point de contrôle immunitaire immunitaire impliquant le récepteur P2Y2 et la protéine NLRP3 : Study Of The Early Steps Of HIV-1 Infection : Identification Of A New Immune Checkpoint Involving P2Y2 And NLRP3. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2015SACLS240

Chicago Manual of Style (16th Edition):

Paoletti, Audrey. “Étude des étapes précoces de l’infection par le VIH-1 : identification d’un nouveau point de contrôle immunitaire immunitaire impliquant le récepteur P2Y2 et la protéine NLRP3 : Study Of The Early Steps Of HIV-1 Infection : Identification Of A New Immune Checkpoint Involving P2Y2 And NLRP3.” 2015. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed February 16, 2020. http://www.theses.fr/2015SACLS240.

MLA Handbook (7th Edition):

Paoletti, Audrey. “Étude des étapes précoces de l’infection par le VIH-1 : identification d’un nouveau point de contrôle immunitaire immunitaire impliquant le récepteur P2Y2 et la protéine NLRP3 : Study Of The Early Steps Of HIV-1 Infection : Identification Of A New Immune Checkpoint Involving P2Y2 And NLRP3.” 2015. Web. 16 Feb 2020.

Vancouver:

Paoletti A. Étude des étapes précoces de l’infection par le VIH-1 : identification d’un nouveau point de contrôle immunitaire immunitaire impliquant le récepteur P2Y2 et la protéine NLRP3 : Study Of The Early Steps Of HIV-1 Infection : Identification Of A New Immune Checkpoint Involving P2Y2 And NLRP3. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2015. [cited 2020 Feb 16]. Available from: http://www.theses.fr/2015SACLS240.

Council of Science Editors:

Paoletti A. Étude des étapes précoces de l’infection par le VIH-1 : identification d’un nouveau point de contrôle immunitaire immunitaire impliquant le récepteur P2Y2 et la protéine NLRP3 : Study Of The Early Steps Of HIV-1 Infection : Identification Of A New Immune Checkpoint Involving P2Y2 And NLRP3. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2015. Available from: http://www.theses.fr/2015SACLS240


University of Oxford

8. Srinivasan, N. The role of inflammasomes in intestinal inflammation.

Degree: PhD, 2014, University of Oxford

 Single Nucleotide Polymorphisms (SNPs) in the intracellular pattern recognition receptor gene NLRP3 are associated with susceptibility to Crohn’s disease, a form of inflammatory bowel disease… (more)

Subjects/Keywords: 616.3; Immunology; Inflammasome; intestinal inflammation; mucosal immunity

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APA (6th Edition):

Srinivasan, N. (2014). The role of inflammasomes in intestinal inflammation. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:04ad577c-a8dd-46eb-811a-79a3980ff806 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596000

Chicago Manual of Style (16th Edition):

Srinivasan, N. “The role of inflammasomes in intestinal inflammation.” 2014. Doctoral Dissertation, University of Oxford. Accessed February 16, 2020. http://ora.ox.ac.uk/objects/uuid:04ad577c-a8dd-46eb-811a-79a3980ff806 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596000.

MLA Handbook (7th Edition):

Srinivasan, N. “The role of inflammasomes in intestinal inflammation.” 2014. Web. 16 Feb 2020.

Vancouver:

Srinivasan N. The role of inflammasomes in intestinal inflammation. [Internet] [Doctoral dissertation]. University of Oxford; 2014. [cited 2020 Feb 16]. Available from: http://ora.ox.ac.uk/objects/uuid:04ad577c-a8dd-46eb-811a-79a3980ff806 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596000.

Council of Science Editors:

Srinivasan N. The role of inflammasomes in intestinal inflammation. [Doctoral Dissertation]. University of Oxford; 2014. Available from: http://ora.ox.ac.uk/objects/uuid:04ad577c-a8dd-46eb-811a-79a3980ff806 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596000


University of Manchester

9. Edye, Michelle. Interleukin-1 signalling in disease.

Degree: 2015, University of Manchester

 The pro-inflammatory cytokine interleukin 1 (IL-1) is involved in numerous physiological and pathological processes. It contributes to thermoregulation, sleep, feeding behaviour and notably to the… (more)

Subjects/Keywords: Interleukin-1; Inflammation; Acidosis; Inflammasome; Seizure

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APA (6th Edition):

Edye, M. (2015). Interleukin-1 signalling in disease. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:293336

Chicago Manual of Style (16th Edition):

Edye, Michelle. “Interleukin-1 signalling in disease.” 2015. Doctoral Dissertation, University of Manchester. Accessed February 16, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:293336.

MLA Handbook (7th Edition):

Edye, Michelle. “Interleukin-1 signalling in disease.” 2015. Web. 16 Feb 2020.

Vancouver:

Edye M. Interleukin-1 signalling in disease. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2020 Feb 16]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:293336.

Council of Science Editors:

Edye M. Interleukin-1 signalling in disease. [Doctoral Dissertation]. University of Manchester; 2015. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:293336

10. 김, 은형. Expression and function of the NLRP3 inflammasome in Behçet’s disease.

Degree: 2016, Ajou University

연구배경: 베체트병운 점막 및피부, 눈, 혈관, 신경 및 기타 여러 장기를 침범할 수 있는 임상적 특징을 가지는 만성, 재발성의 전신질환이다. 현재까지 베체트병에서 보이는 지속적인 염증… (more)

Subjects/Keywords: Behçet’s disease; inflammasome; NLRP3; interleukin-1β

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APA (6th Edition):

김, . (2016). Expression and function of the NLRP3 inflammasome in Behçet’s disease. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/13061 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

김, 은형. “Expression and function of the NLRP3 inflammasome in Behçet’s disease.” 2016. Thesis, Ajou University. Accessed February 16, 2020. http://repository.ajou.ac.kr/handle/201003/13061 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

김, 은형. “Expression and function of the NLRP3 inflammasome in Behçet’s disease.” 2016. Web. 16 Feb 2020.

Vancouver:

김 . Expression and function of the NLRP3 inflammasome in Behçet’s disease. [Internet] [Thesis]. Ajou University; 2016. [cited 2020 Feb 16]. Available from: http://repository.ajou.ac.kr/handle/201003/13061 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

김 . Expression and function of the NLRP3 inflammasome in Behçet’s disease. [Thesis]. Ajou University; 2016. Available from: http://repository.ajou.ac.kr/handle/201003/13061 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

11. Li, Yujin. Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy.

Degree: MSc, 2016, McMaster University

As a front-line treatment for cardiovascular disease, statins are among some of the most widely prescribed drugs worldwide. Statins are effective at lowering cholesterol, but… (more)

Subjects/Keywords: Inflammasome; Statins; Myopathy; Prenylation; Caspase-1; Atrogenes

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APA (6th Edition):

Li, Y. (2016). Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/20268

Chicago Manual of Style (16th Edition):

Li, Yujin. “Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy.” 2016. Masters Thesis, McMaster University. Accessed February 16, 2020. http://hdl.handle.net/11375/20268.

MLA Handbook (7th Edition):

Li, Yujin. “Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy.” 2016. Web. 16 Feb 2020.

Vancouver:

Li Y. Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy. [Internet] [Masters thesis]. McMaster University; 2016. [cited 2020 Feb 16]. Available from: http://hdl.handle.net/11375/20268.

Council of Science Editors:

Li Y. Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy. [Masters Thesis]. McMaster University; 2016. Available from: http://hdl.handle.net/11375/20268


Universitat de Valencia

12. Alegre Guerra, Fernando. Acute inflammatory and fibrogenic responses induced in liver cells by efavirenz .

Degree: 2017, Universitat de Valencia

 RESUMEN INTRODUCCIÓN Diversos agentes hepatotóxicos y enfermedades hepáticas pueden conducir al desarrollo de fibrosis hepática debido a la presencia de respuestas inflamatorias desreguladas (Kubes and… (more)

Subjects/Keywords: liver; hiv; efavirenz; inflammation; inflammasome; fibrogenesis

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APA (6th Edition):

Alegre Guerra, F. (2017). Acute inflammatory and fibrogenic responses induced in liver cells by efavirenz . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/58371

Chicago Manual of Style (16th Edition):

Alegre Guerra, Fernando. “Acute inflammatory and fibrogenic responses induced in liver cells by efavirenz .” 2017. Doctoral Dissertation, Universitat de Valencia. Accessed February 16, 2020. http://hdl.handle.net/10550/58371.

MLA Handbook (7th Edition):

Alegre Guerra, Fernando. “Acute inflammatory and fibrogenic responses induced in liver cells by efavirenz .” 2017. Web. 16 Feb 2020.

Vancouver:

Alegre Guerra F. Acute inflammatory and fibrogenic responses induced in liver cells by efavirenz . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2017. [cited 2020 Feb 16]. Available from: http://hdl.handle.net/10550/58371.

Council of Science Editors:

Alegre Guerra F. Acute inflammatory and fibrogenic responses induced in liver cells by efavirenz . [Doctoral Dissertation]. Universitat de Valencia; 2017. Available from: http://hdl.handle.net/10550/58371


University of Vermont

13. Hoyt, Laura Rose. Inhibitory Effects of Ethanol on the NLRP3 Inflammasome.

Degree: Medicine, 2015, University of Vermont

  Immunosuppression is a major complication of alcoholism and contributes to increased rates of opportunistic infections and sepsis associated with the addiction. The NLRP3 inflammasome(more)

Subjects/Keywords: Ethanol; Alcohol; NLRP3; Inflammasome; Inflammation; Immunosuppression

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APA (6th Edition):

Hoyt, L. R. (2015). Inhibitory Effects of Ethanol on the NLRP3 Inflammasome. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/hcoltheses/194

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hoyt, Laura Rose. “Inhibitory Effects of Ethanol on the NLRP3 Inflammasome.” 2015. Thesis, University of Vermont. Accessed February 16, 2020. https://scholarworks.uvm.edu/hcoltheses/194.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hoyt, Laura Rose. “Inhibitory Effects of Ethanol on the NLRP3 Inflammasome.” 2015. Web. 16 Feb 2020.

Vancouver:

Hoyt LR. Inhibitory Effects of Ethanol on the NLRP3 Inflammasome. [Internet] [Thesis]. University of Vermont; 2015. [cited 2020 Feb 16]. Available from: https://scholarworks.uvm.edu/hcoltheses/194.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hoyt LR. Inhibitory Effects of Ethanol on the NLRP3 Inflammasome. [Thesis]. University of Vermont; 2015. Available from: https://scholarworks.uvm.edu/hcoltheses/194

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

14. Maloy, Jeffrey Phillip. Characterization of an Intracellular Flagellar System in Pathogenic Burkholderia Species.

Degree: Microbiology, Immunology, & Molecular Genetics, 2017, UCLA

 Burkholderia pseudomallei and Burkholderia thailandensis are related facultative intracellular pathogens that infect a variety of cell types, escape into the cytoplasm of host cells, and… (more)

Subjects/Keywords: Microbiology; Immunology; Burkholderia; Flagella; Inflammasome; Melioidosis; NLRC4

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APA (6th Edition):

Maloy, J. P. (2017). Characterization of an Intracellular Flagellar System in Pathogenic Burkholderia Species. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/11m9037f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maloy, Jeffrey Phillip. “Characterization of an Intracellular Flagellar System in Pathogenic Burkholderia Species.” 2017. Thesis, UCLA. Accessed February 16, 2020. http://www.escholarship.org/uc/item/11m9037f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maloy, Jeffrey Phillip. “Characterization of an Intracellular Flagellar System in Pathogenic Burkholderia Species.” 2017. Web. 16 Feb 2020.

Vancouver:

Maloy JP. Characterization of an Intracellular Flagellar System in Pathogenic Burkholderia Species. [Internet] [Thesis]. UCLA; 2017. [cited 2020 Feb 16]. Available from: http://www.escholarship.org/uc/item/11m9037f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maloy JP. Characterization of an Intracellular Flagellar System in Pathogenic Burkholderia Species. [Thesis]. UCLA; 2017. Available from: http://www.escholarship.org/uc/item/11m9037f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

15. Wynick, Christopher. IL-27 Enhances LPS-Induced Proinflammatory Responses in Human Monocytes: Augmented Inflammasome Activity and IL-23 Expression .

Degree: Microbiology and Immunology, 2014, Queens University

 Inflammation plays an important role in responding to injury and combating infections. In this thesis, I examine how inflammation is regulated by cytokines responsible for… (more)

Subjects/Keywords: Inflammation; IL-27; Monocytes; Inflammasome; IL-23

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APA (6th Edition):

Wynick, C. (2014). IL-27 Enhances LPS-Induced Proinflammatory Responses in Human Monocytes: Augmented Inflammasome Activity and IL-23 Expression . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/12256

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wynick, Christopher. “IL-27 Enhances LPS-Induced Proinflammatory Responses in Human Monocytes: Augmented Inflammasome Activity and IL-23 Expression .” 2014. Thesis, Queens University. Accessed February 16, 2020. http://hdl.handle.net/1974/12256.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wynick, Christopher. “IL-27 Enhances LPS-Induced Proinflammatory Responses in Human Monocytes: Augmented Inflammasome Activity and IL-23 Expression .” 2014. Web. 16 Feb 2020.

Vancouver:

Wynick C. IL-27 Enhances LPS-Induced Proinflammatory Responses in Human Monocytes: Augmented Inflammasome Activity and IL-23 Expression . [Internet] [Thesis]. Queens University; 2014. [cited 2020 Feb 16]. Available from: http://hdl.handle.net/1974/12256.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wynick C. IL-27 Enhances LPS-Induced Proinflammatory Responses in Human Monocytes: Augmented Inflammasome Activity and IL-23 Expression . [Thesis]. Queens University; 2014. Available from: http://hdl.handle.net/1974/12256

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Tasmania

16. Ranson, NM. Inflammasome activity in the inflammatory bowel diseases.

Degree: 2018, University of Tasmania

 Ulcerative colitis (UC) and Crohn’s disease (CD) are characterised by chronic and recurrent inflammation of the gastrointestinal tract. The development of Inflammatory Bowel Diseases (IBD)… (more)

Subjects/Keywords: inflammation; inflammasome complexes; ulcerative colitis; Crohn's disease

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APA (6th Edition):

Ranson, N. (2018). Inflammasome activity in the inflammatory bowel diseases. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/29986/1/Ranson_whole_thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ranson, NM. “Inflammasome activity in the inflammatory bowel diseases.” 2018. Thesis, University of Tasmania. Accessed February 16, 2020. https://eprints.utas.edu.au/29986/1/Ranson_whole_thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ranson, NM. “Inflammasome activity in the inflammatory bowel diseases.” 2018. Web. 16 Feb 2020.

Vancouver:

Ranson N. Inflammasome activity in the inflammatory bowel diseases. [Internet] [Thesis]. University of Tasmania; 2018. [cited 2020 Feb 16]. Available from: https://eprints.utas.edu.au/29986/1/Ranson_whole_thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ranson N. Inflammasome activity in the inflammatory bowel diseases. [Thesis]. University of Tasmania; 2018. Available from: https://eprints.utas.edu.au/29986/1/Ranson_whole_thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

17. Tong, Yanzheng. Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses.

Degree: PhD, Medical Sciences, 2014, Texas A&M University

 The NOD-like receptor protein NLRC5 inhibits TLR-induced NF-ҡB and viral infection-mediated type I interferon signaling in vitro. In the first project, we generated NLRC5 knockout… (more)

Subjects/Keywords: innate immunity; NLRC5; inflammasome; NF-kB signaling

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APA (6th Edition):

Tong, Y. (2014). Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/186991

Chicago Manual of Style (16th Edition):

Tong, Yanzheng. “Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses.” 2014. Doctoral Dissertation, Texas A&M University. Accessed February 16, 2020. http://hdl.handle.net/1969.1/186991.

MLA Handbook (7th Edition):

Tong, Yanzheng. “Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses.” 2014. Web. 16 Feb 2020.

Vancouver:

Tong Y. Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses. [Internet] [Doctoral dissertation]. Texas A&M University; 2014. [cited 2020 Feb 16]. Available from: http://hdl.handle.net/1969.1/186991.

Council of Science Editors:

Tong Y. Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses. [Doctoral Dissertation]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/186991


Georgia State University

18. Chesnokov, Anton P, Mr. Modulation of NALP3 Inflammasome Genes by Estrogen.

Degree: MS, Biology, 2011, Georgia State University

  Immunity is known to be sexually dimorphic. This dimorphism may be attributed to the action of different hormones. Aluminum is a component of several… (more)

Subjects/Keywords: Nalp3 inflammasome; Estrogen; IL-1beta; Aluminum hydroxide

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APA (6th Edition):

Chesnokov, Anton P, M. (2011). Modulation of NALP3 Inflammasome Genes by Estrogen. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/33

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chesnokov, Anton P, Mr. “Modulation of NALP3 Inflammasome Genes by Estrogen.” 2011. Thesis, Georgia State University. Accessed February 16, 2020. https://scholarworks.gsu.edu/biology_theses/33.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chesnokov, Anton P, Mr. “Modulation of NALP3 Inflammasome Genes by Estrogen.” 2011. Web. 16 Feb 2020.

Vancouver:

Chesnokov, Anton P M. Modulation of NALP3 Inflammasome Genes by Estrogen. [Internet] [Thesis]. Georgia State University; 2011. [cited 2020 Feb 16]. Available from: https://scholarworks.gsu.edu/biology_theses/33.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chesnokov, Anton P M. Modulation of NALP3 Inflammasome Genes by Estrogen. [Thesis]. Georgia State University; 2011. Available from: https://scholarworks.gsu.edu/biology_theses/33

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Bergin, Ronan. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.

Degree: 2015, RIAN

 The innate immune system is the first line of defence against invading pathogens and is responsible for both initial pathogen recognition and mounting an effective… (more)

Subjects/Keywords: Analysis; role; Pellino2; Activation; NLRP3 Inflammasome

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APA (6th Edition):

Bergin, R. (2015). Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. (Thesis). RIAN. Retrieved from http://eprints.maynoothuniversity.ie/10393/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bergin, Ronan. “Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.” 2015. Thesis, RIAN. Accessed February 16, 2020. http://eprints.maynoothuniversity.ie/10393/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bergin, Ronan. “Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.” 2015. Web. 16 Feb 2020.

Vancouver:

Bergin R. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. [Internet] [Thesis]. RIAN; 2015. [cited 2020 Feb 16]. Available from: http://eprints.maynoothuniversity.ie/10393/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bergin R. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. [Thesis]. RIAN; 2015. Available from: http://eprints.maynoothuniversity.ie/10393/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Iowa

20. Clay, Gwendolyn Mary. The role of NLR proteins in Leishmaniasis.

Degree: PhD, Molecular and Cell Biology, 2016, University of Iowa

  Leishmania species are vector-borne protozoan parasites that cause a spectrum of human diseases, with an estimated 12 million people infected in 88 countries. Inflammation… (more)

Subjects/Keywords: Inflammasome; Leishmania; Leishmaniasis; NLR; Cell Biology

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APA (6th Edition):

Clay, G. M. (2016). The role of NLR proteins in Leishmaniasis. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/5442

Chicago Manual of Style (16th Edition):

Clay, Gwendolyn Mary. “The role of NLR proteins in Leishmaniasis.” 2016. Doctoral Dissertation, University of Iowa. Accessed February 16, 2020. https://ir.uiowa.edu/etd/5442.

MLA Handbook (7th Edition):

Clay, Gwendolyn Mary. “The role of NLR proteins in Leishmaniasis.” 2016. Web. 16 Feb 2020.

Vancouver:

Clay GM. The role of NLR proteins in Leishmaniasis. [Internet] [Doctoral dissertation]. University of Iowa; 2016. [cited 2020 Feb 16]. Available from: https://ir.uiowa.edu/etd/5442.

Council of Science Editors:

Clay GM. The role of NLR proteins in Leishmaniasis. [Doctoral Dissertation]. University of Iowa; 2016. Available from: https://ir.uiowa.edu/etd/5442


University of Toronto

21. Neiman-Zenevich, Jana. Characterization of NLRP1B Inflammasome Activation.

Degree: PhD, 2017, University of Toronto

An inflammasome is a multi-protein complex that consists of a sensor protein, caspase-1, and often an adaptor protein ASC. An inflammasome assembles in response to… (more)

Subjects/Keywords: ATP depletion; Inflammasome; Listeria; NLRP1B; 0306

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APA (6th Edition):

Neiman-Zenevich, J. (2017). Characterization of NLRP1B Inflammasome Activation. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/98773

Chicago Manual of Style (16th Edition):

Neiman-Zenevich, Jana. “Characterization of NLRP1B Inflammasome Activation.” 2017. Doctoral Dissertation, University of Toronto. Accessed February 16, 2020. http://hdl.handle.net/1807/98773.

MLA Handbook (7th Edition):

Neiman-Zenevich, Jana. “Characterization of NLRP1B Inflammasome Activation.” 2017. Web. 16 Feb 2020.

Vancouver:

Neiman-Zenevich J. Characterization of NLRP1B Inflammasome Activation. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2020 Feb 16]. Available from: http://hdl.handle.net/1807/98773.

Council of Science Editors:

Neiman-Zenevich J. Characterization of NLRP1B Inflammasome Activation. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/98773

22. Benaoudia, Sacha. Détection du LPS cytosolique : un crible CRISPR/Cas9 pangénomique identifie IRF2 comme régulateur de l’inflammasome non-canonique caspase-4 : Detection of cytosolic LPS : a genome-wide CRISPR/Cas9 screen identifies IRF2 as a regulator of the non-canonical inflammasome caspase-4.

Degree: Docteur es, Science de la vie, 2018, Lyon

Le lipopolysaccharide (LPS), un composant de la membrane des bactéries à Gram-négatif, active l’inflammasome non-canonique constitué chez l’homme des caspase-4/5 et chez la souris de… (more)

Subjects/Keywords: Caspase; Inflammasome; Mort cellulaire; LPS; Immunité innée; Caspase; Inflammasome; Cell death; LPS; Innate immunity; 570

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Benaoudia, S. (2018). Détection du LPS cytosolique : un crible CRISPR/Cas9 pangénomique identifie IRF2 comme régulateur de l’inflammasome non-canonique caspase-4 : Detection of cytosolic LPS : a genome-wide CRISPR/Cas9 screen identifies IRF2 as a regulator of the non-canonical inflammasome caspase-4. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2018LYSE1219

Chicago Manual of Style (16th Edition):

Benaoudia, Sacha. “Détection du LPS cytosolique : un crible CRISPR/Cas9 pangénomique identifie IRF2 comme régulateur de l’inflammasome non-canonique caspase-4 : Detection of cytosolic LPS : a genome-wide CRISPR/Cas9 screen identifies IRF2 as a regulator of the non-canonical inflammasome caspase-4.” 2018. Doctoral Dissertation, Lyon. Accessed February 16, 2020. http://www.theses.fr/2018LYSE1219.

MLA Handbook (7th Edition):

Benaoudia, Sacha. “Détection du LPS cytosolique : un crible CRISPR/Cas9 pangénomique identifie IRF2 comme régulateur de l’inflammasome non-canonique caspase-4 : Detection of cytosolic LPS : a genome-wide CRISPR/Cas9 screen identifies IRF2 as a regulator of the non-canonical inflammasome caspase-4.” 2018. Web. 16 Feb 2020.

Vancouver:

Benaoudia S. Détection du LPS cytosolique : un crible CRISPR/Cas9 pangénomique identifie IRF2 comme régulateur de l’inflammasome non-canonique caspase-4 : Detection of cytosolic LPS : a genome-wide CRISPR/Cas9 screen identifies IRF2 as a regulator of the non-canonical inflammasome caspase-4. [Internet] [Doctoral dissertation]. Lyon; 2018. [cited 2020 Feb 16]. Available from: http://www.theses.fr/2018LYSE1219.

Council of Science Editors:

Benaoudia S. Détection du LPS cytosolique : un crible CRISPR/Cas9 pangénomique identifie IRF2 comme régulateur de l’inflammasome non-canonique caspase-4 : Detection of cytosolic LPS : a genome-wide CRISPR/Cas9 screen identifies IRF2 as a regulator of the non-canonical inflammasome caspase-4. [Doctoral Dissertation]. Lyon; 2018. Available from: http://www.theses.fr/2018LYSE1219

23. Awad, Fawaz. Pathophysiology of hereditary recurrent fever syndromes : cellular and molecular approaches : Pathophysiologie des fièvres récurrentes héréditaires : approches cellulaires et moléculaires.

Degree: Docteur es, Génétique humaine, 2014, Université Pierre et Marie Curie – Paris VI

Les fièvres récurrentes héréditaires (FRH) sont des maladies auto-inflammatoires transmises selon un mode mendélien. Elles se caractérisent par des accès fébriles récurrents spontanément résolutifs accompagnés… (more)

Subjects/Keywords: Fièvres récurrentes héréditaires; Fièvre méditerranéenne familiale; Inflammasome; Nlrp3; Pyrine; Macrophages; Hereditary recurrent fevers; Inflammasome; 570

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APA (6th Edition):

Awad, F. (2014). Pathophysiology of hereditary recurrent fever syndromes : cellular and molecular approaches : Pathophysiologie des fièvres récurrentes héréditaires : approches cellulaires et moléculaires. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2014PA066394

Chicago Manual of Style (16th Edition):

Awad, Fawaz. “Pathophysiology of hereditary recurrent fever syndromes : cellular and molecular approaches : Pathophysiologie des fièvres récurrentes héréditaires : approches cellulaires et moléculaires.” 2014. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed February 16, 2020. http://www.theses.fr/2014PA066394.

MLA Handbook (7th Edition):

Awad, Fawaz. “Pathophysiology of hereditary recurrent fever syndromes : cellular and molecular approaches : Pathophysiologie des fièvres récurrentes héréditaires : approches cellulaires et moléculaires.” 2014. Web. 16 Feb 2020.

Vancouver:

Awad F. Pathophysiology of hereditary recurrent fever syndromes : cellular and molecular approaches : Pathophysiologie des fièvres récurrentes héréditaires : approches cellulaires et moléculaires. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2014. [cited 2020 Feb 16]. Available from: http://www.theses.fr/2014PA066394.

Council of Science Editors:

Awad F. Pathophysiology of hereditary recurrent fever syndromes : cellular and molecular approaches : Pathophysiologie des fièvres récurrentes héréditaires : approches cellulaires et moléculaires. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2014. Available from: http://www.theses.fr/2014PA066394

24. Baron, Ludivine. Mécanismes d’activation de l’inflammasome NLRP3 par les micro- et les nano- particules dans un modèle d’inflammation pulmonaire chez la souris : Mechanisms of activation of the NLRP3 inflammasome by micro- and nano- particles in a murine model of lung inflammation.

Degree: Docteur es, Sciences du vivant. Immunologie, 2013, Université d'Orléans

Les mécanismes de défense de l’organisme contre des pathogènes ou des particules toxiques sont regroupés sous le terme d’Immunité. L’Immunité a développé plusieurs familles de… (more)

Subjects/Keywords: Adénosine; ATP; Inflammasome; Interleukines (IL); Cristaux; Nanoparticules; Adenosine; ATP; Inflammasome; Interleukines (IL); Crystals; Nanoparticles

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APA (6th Edition):

Baron, L. (2013). Mécanismes d’activation de l’inflammasome NLRP3 par les micro- et les nano- particules dans un modèle d’inflammation pulmonaire chez la souris : Mechanisms of activation of the NLRP3 inflammasome by micro- and nano- particles in a murine model of lung inflammation. (Doctoral Dissertation). Université d'Orléans. Retrieved from http://www.theses.fr/2013ORLE2037

Chicago Manual of Style (16th Edition):

Baron, Ludivine. “Mécanismes d’activation de l’inflammasome NLRP3 par les micro- et les nano- particules dans un modèle d’inflammation pulmonaire chez la souris : Mechanisms of activation of the NLRP3 inflammasome by micro- and nano- particles in a murine model of lung inflammation.” 2013. Doctoral Dissertation, Université d'Orléans. Accessed February 16, 2020. http://www.theses.fr/2013ORLE2037.

MLA Handbook (7th Edition):

Baron, Ludivine. “Mécanismes d’activation de l’inflammasome NLRP3 par les micro- et les nano- particules dans un modèle d’inflammation pulmonaire chez la souris : Mechanisms of activation of the NLRP3 inflammasome by micro- and nano- particles in a murine model of lung inflammation.” 2013. Web. 16 Feb 2020.

Vancouver:

Baron L. Mécanismes d’activation de l’inflammasome NLRP3 par les micro- et les nano- particules dans un modèle d’inflammation pulmonaire chez la souris : Mechanisms of activation of the NLRP3 inflammasome by micro- and nano- particles in a murine model of lung inflammation. [Internet] [Doctoral dissertation]. Université d'Orléans; 2013. [cited 2020 Feb 16]. Available from: http://www.theses.fr/2013ORLE2037.

Council of Science Editors:

Baron L. Mécanismes d’activation de l’inflammasome NLRP3 par les micro- et les nano- particules dans un modèle d’inflammation pulmonaire chez la souris : Mechanisms of activation of the NLRP3 inflammasome by micro- and nano- particles in a murine model of lung inflammation. [Doctoral Dissertation]. Université d'Orléans; 2013. Available from: http://www.theses.fr/2013ORLE2037

25. Poli, Caroline. Il-26 : une cytokine pro-inflammatoire stimulant les cellules immunitaires innées myéloïdes : Il-26 : a pro-inflammatory cytokine that stimulates innate myeloid cells.

Degree: Docteur es, Immunologie, 2017, Angers

Physiologiquement, l’ADN, séquestré dans les noyaux, n’est pas détecté par les récepteurs de danger du système immunitaire. En revanche, au cours d’inflammations chroniques, une rupture… (more)

Subjects/Keywords: Inflammation; Mort cellulaire; Acides nucléiques; Sting; Inflammasome; Inflammation; Cell death; Nucleic acids; Sting; Inflammasome; 616.079

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APA (6th Edition):

Poli, C. (2017). Il-26 : une cytokine pro-inflammatoire stimulant les cellules immunitaires innées myéloïdes : Il-26 : a pro-inflammatory cytokine that stimulates innate myeloid cells. (Doctoral Dissertation). Angers. Retrieved from http://www.theses.fr/2017ANGE0068

Chicago Manual of Style (16th Edition):

Poli, Caroline. “Il-26 : une cytokine pro-inflammatoire stimulant les cellules immunitaires innées myéloïdes : Il-26 : a pro-inflammatory cytokine that stimulates innate myeloid cells.” 2017. Doctoral Dissertation, Angers. Accessed February 16, 2020. http://www.theses.fr/2017ANGE0068.

MLA Handbook (7th Edition):

Poli, Caroline. “Il-26 : une cytokine pro-inflammatoire stimulant les cellules immunitaires innées myéloïdes : Il-26 : a pro-inflammatory cytokine that stimulates innate myeloid cells.” 2017. Web. 16 Feb 2020.

Vancouver:

Poli C. Il-26 : une cytokine pro-inflammatoire stimulant les cellules immunitaires innées myéloïdes : Il-26 : a pro-inflammatory cytokine that stimulates innate myeloid cells. [Internet] [Doctoral dissertation]. Angers; 2017. [cited 2020 Feb 16]. Available from: http://www.theses.fr/2017ANGE0068.

Council of Science Editors:

Poli C. Il-26 : une cytokine pro-inflammatoire stimulant les cellules immunitaires innées myéloïdes : Il-26 : a pro-inflammatory cytokine that stimulates innate myeloid cells. [Doctoral Dissertation]. Angers; 2017. Available from: http://www.theses.fr/2017ANGE0068

26. Eichholz, Karsten. Influence de protéin[e]s de l'hôte sur la réponse immunitaire innée face aux adénovirus humains dans les phagocytes humains : Influence of host proteins on the innate immune response to human adenoviruses in human phagocytes.

Degree: Docteur es, Biologie Santé, 2015, Montpellier

Les adénovirus humains (HAdV) provoquent un large spectre de maladies cliniques chez les patients immunodéprimés et immunocompétents et sont également des outils polyvalents pour le… (more)

Subjects/Keywords: Cellule dendritique; Inflammasome; Adenoviridae; Innate sensors; Facteurs de coagulation; Dendritic cells; Inflammasome; Adenovirus; Innate sensors; Coagulation factors

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APA (6th Edition):

Eichholz, K. (2015). Influence de protéin[e]s de l'hôte sur la réponse immunitaire innée face aux adénovirus humains dans les phagocytes humains : Influence of host proteins on the innate immune response to human adenoviruses in human phagocytes. (Doctoral Dissertation). Montpellier. Retrieved from http://www.theses.fr/2015MONTS059

Chicago Manual of Style (16th Edition):

Eichholz, Karsten. “Influence de protéin[e]s de l'hôte sur la réponse immunitaire innée face aux adénovirus humains dans les phagocytes humains : Influence of host proteins on the innate immune response to human adenoviruses in human phagocytes.” 2015. Doctoral Dissertation, Montpellier. Accessed February 16, 2020. http://www.theses.fr/2015MONTS059.

MLA Handbook (7th Edition):

Eichholz, Karsten. “Influence de protéin[e]s de l'hôte sur la réponse immunitaire innée face aux adénovirus humains dans les phagocytes humains : Influence of host proteins on the innate immune response to human adenoviruses in human phagocytes.” 2015. Web. 16 Feb 2020.

Vancouver:

Eichholz K. Influence de protéin[e]s de l'hôte sur la réponse immunitaire innée face aux adénovirus humains dans les phagocytes humains : Influence of host proteins on the innate immune response to human adenoviruses in human phagocytes. [Internet] [Doctoral dissertation]. Montpellier; 2015. [cited 2020 Feb 16]. Available from: http://www.theses.fr/2015MONTS059.

Council of Science Editors:

Eichholz K. Influence de protéin[e]s de l'hôte sur la réponse immunitaire innée face aux adénovirus humains dans les phagocytes humains : Influence of host proteins on the innate immune response to human adenoviruses in human phagocytes. [Doctoral Dissertation]. Montpellier; 2015. Available from: http://www.theses.fr/2015MONTS059

27. Gicquel, Thomas. Implication des récepteurs purinergiques dans l'activation de l'inflammasome NLRP3 dans les macrophages : Involvement of purinergic receptors in NLRP3-inflammasome pathway from macrophages.

Degree: Docteur es, Biologie et sciences de la santé, 2014, Rennes 1

L’inflammasome NLRP3 est très impliqué dans de nombreuses pathologies inflammatoires comme la fibrose pulmonaire, la polyarthrite rhumatoïde, la goutte ou la maladie de Crohn. Cette… (more)

Subjects/Keywords: Inflammasome NLRP3; Récepteurs purinergiques; Atp; Macrophages; Interleukines; Inflammation; Immunité innée; NLRP3 inflammasome; Purinergic receptors; Atp; Macrophages; Interleukines; Inflammation; Innate immunity

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gicquel, T. (2014). Implication des récepteurs purinergiques dans l'activation de l'inflammasome NLRP3 dans les macrophages : Involvement of purinergic receptors in NLRP3-inflammasome pathway from macrophages. (Doctoral Dissertation). Rennes 1. Retrieved from http://www.theses.fr/2014REN1B012

Chicago Manual of Style (16th Edition):

Gicquel, Thomas. “Implication des récepteurs purinergiques dans l'activation de l'inflammasome NLRP3 dans les macrophages : Involvement of purinergic receptors in NLRP3-inflammasome pathway from macrophages.” 2014. Doctoral Dissertation, Rennes 1. Accessed February 16, 2020. http://www.theses.fr/2014REN1B012.

MLA Handbook (7th Edition):

Gicquel, Thomas. “Implication des récepteurs purinergiques dans l'activation de l'inflammasome NLRP3 dans les macrophages : Involvement of purinergic receptors in NLRP3-inflammasome pathway from macrophages.” 2014. Web. 16 Feb 2020.

Vancouver:

Gicquel T. Implication des récepteurs purinergiques dans l'activation de l'inflammasome NLRP3 dans les macrophages : Involvement of purinergic receptors in NLRP3-inflammasome pathway from macrophages. [Internet] [Doctoral dissertation]. Rennes 1; 2014. [cited 2020 Feb 16]. Available from: http://www.theses.fr/2014REN1B012.

Council of Science Editors:

Gicquel T. Implication des récepteurs purinergiques dans l'activation de l'inflammasome NLRP3 dans les macrophages : Involvement of purinergic receptors in NLRP3-inflammasome pathway from macrophages. [Doctoral Dissertation]. Rennes 1; 2014. Available from: http://www.theses.fr/2014REN1B012

28. Robert, Sacha. Caractérisation des signaux de danger et de la signalisation cellulaire dans le développement de la fibrose hépatique : Characterisation of danger signals and cell signaling in the development of liver fibrosis.

Degree: Docteur es, Biologie, 2016, Rennes 1

La voie de signalisation de l’inflammasome est impliquée dans plusieurs pathologies inflammatoires dont la fibrose pulmonaire et plus récemment, elle a été mise en lumière… (more)

Subjects/Keywords: Inflammasome; Fibrose hépatique; Cytokine; Macrophage; Fibroblaste; Inflammation; Fibrolyse; Inflammasome; Liver fibrosis; Cytokine; Macrophage; Fibroblast; Inflammation; Fibrolysis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Robert, S. (2016). Caractérisation des signaux de danger et de la signalisation cellulaire dans le développement de la fibrose hépatique : Characterisation of danger signals and cell signaling in the development of liver fibrosis. (Doctoral Dissertation). Rennes 1. Retrieved from http://www.theses.fr/2016REN1B016

Chicago Manual of Style (16th Edition):

Robert, Sacha. “Caractérisation des signaux de danger et de la signalisation cellulaire dans le développement de la fibrose hépatique : Characterisation of danger signals and cell signaling in the development of liver fibrosis.” 2016. Doctoral Dissertation, Rennes 1. Accessed February 16, 2020. http://www.theses.fr/2016REN1B016.

MLA Handbook (7th Edition):

Robert, Sacha. “Caractérisation des signaux de danger et de la signalisation cellulaire dans le développement de la fibrose hépatique : Characterisation of danger signals and cell signaling in the development of liver fibrosis.” 2016. Web. 16 Feb 2020.

Vancouver:

Robert S. Caractérisation des signaux de danger et de la signalisation cellulaire dans le développement de la fibrose hépatique : Characterisation of danger signals and cell signaling in the development of liver fibrosis. [Internet] [Doctoral dissertation]. Rennes 1; 2016. [cited 2020 Feb 16]. Available from: http://www.theses.fr/2016REN1B016.

Council of Science Editors:

Robert S. Caractérisation des signaux de danger et de la signalisation cellulaire dans le développement de la fibrose hépatique : Characterisation of danger signals and cell signaling in the development of liver fibrosis. [Doctoral Dissertation]. Rennes 1; 2016. Available from: http://www.theses.fr/2016REN1B016

29. Huck, Olivier. Infection et stimulation de cellules endothéliales par Porphyromonas gingivalis et son lipopolysaccharide : lien entre maladies parodontales et athérosclérose : Infection and stimulation of endothelial cells with porphyromonas gingivalis and its lipopolysaccharide : link between periodontal diseases and atherosclerosis.

Degree: Docteur es, Sciences de la vie et de la santé, 2013, Université de Strasbourg

Depuis plusieurs années, l’influence des pathologies parodontales sur certaines pathologies systémiques, notamment les maladies cardio-vasculaires et l’athérosclérose apparait de plus en plus évidente. Dans notre… (more)

Subjects/Keywords: Porphyromonas gingivalis; Maladies parodontales; Athérosclérose; Cathepsine B; Inflammasome; Porphyromonas gingivalis; Periodontal diseases; Atherosclerosis; Cathepsin B; Inflammasome; 616.13; 617.6

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Huck, O. (2013). Infection et stimulation de cellules endothéliales par Porphyromonas gingivalis et son lipopolysaccharide : lien entre maladies parodontales et athérosclérose : Infection and stimulation of endothelial cells with porphyromonas gingivalis and its lipopolysaccharide : link between periodontal diseases and atherosclerosis. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2013STRAJ021

Chicago Manual of Style (16th Edition):

Huck, Olivier. “Infection et stimulation de cellules endothéliales par Porphyromonas gingivalis et son lipopolysaccharide : lien entre maladies parodontales et athérosclérose : Infection and stimulation of endothelial cells with porphyromonas gingivalis and its lipopolysaccharide : link between periodontal diseases and atherosclerosis.” 2013. Doctoral Dissertation, Université de Strasbourg. Accessed February 16, 2020. http://www.theses.fr/2013STRAJ021.

MLA Handbook (7th Edition):

Huck, Olivier. “Infection et stimulation de cellules endothéliales par Porphyromonas gingivalis et son lipopolysaccharide : lien entre maladies parodontales et athérosclérose : Infection and stimulation of endothelial cells with porphyromonas gingivalis and its lipopolysaccharide : link between periodontal diseases and atherosclerosis.” 2013. Web. 16 Feb 2020.

Vancouver:

Huck O. Infection et stimulation de cellules endothéliales par Porphyromonas gingivalis et son lipopolysaccharide : lien entre maladies parodontales et athérosclérose : Infection and stimulation of endothelial cells with porphyromonas gingivalis and its lipopolysaccharide : link between periodontal diseases and atherosclerosis. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2013. [cited 2020 Feb 16]. Available from: http://www.theses.fr/2013STRAJ021.

Council of Science Editors:

Huck O. Infection et stimulation de cellules endothéliales par Porphyromonas gingivalis et son lipopolysaccharide : lien entre maladies parodontales et athérosclérose : Infection and stimulation of endothelial cells with porphyromonas gingivalis and its lipopolysaccharide : link between periodontal diseases and atherosclerosis. [Doctoral Dissertation]. Université de Strasbourg; 2013. Available from: http://www.theses.fr/2013STRAJ021

30. Besnard, Anne-Gaëlle. Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation.

Degree: Docteur es, Biologie cellulaire et moléculaire, 2010, Université d'Orléans

L’asthme est une maladie inflammatoire chronique des voies aériennes. Chez les individus sensibles, l’inhalation d’allergènes entraine une inflammation pulmonaire se traduisant par des épisodes récurrents… (more)

Subjects/Keywords: Interleukine-22; Interleukine-17; Interleukine-33; Interleukine-1; NLRP3 inflammasome; Interleukin-22; Interleukin-17; Interleukin-33; Interleukin-1; NLRP3 inflammasome

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Besnard, A. (2010). Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation. (Doctoral Dissertation). Université d'Orléans. Retrieved from http://www.theses.fr/2010ORLE2044

Chicago Manual of Style (16th Edition):

Besnard, Anne-Gaëlle. “Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation.” 2010. Doctoral Dissertation, Université d'Orléans. Accessed February 16, 2020. http://www.theses.fr/2010ORLE2044.

MLA Handbook (7th Edition):

Besnard, Anne-Gaëlle. “Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation.” 2010. Web. 16 Feb 2020.

Vancouver:

Besnard A. Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation. [Internet] [Doctoral dissertation]. Université d'Orléans; 2010. [cited 2020 Feb 16]. Available from: http://www.theses.fr/2010ORLE2044.

Council of Science Editors:

Besnard A. Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation. [Doctoral Dissertation]. Université d'Orléans; 2010. Available from: http://www.theses.fr/2010ORLE2044

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