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You searched for subject:(Immunology). Showing records 1 – 30 of 1051 total matches.

[1] [2] [3] [4] [5] … [36]

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University of Oxford

1. Nordlander, Sofia. Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation.

Degree: PhD, 2013, University of Oxford

 Flagellin is a highly immunogenic, bacterial protein considered to be abundant in the intestinal lumen. It has been reported to be an immunodominant antigen in… (more)

Subjects/Keywords: 616.07; Immunology; Mucosal immunology; inflammasome

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APA (6th Edition):

Nordlander, S. (2013). Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:775c4150-9b22-4008-9288-3a6053cbf4cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647535

Chicago Manual of Style (16th Edition):

Nordlander, Sofia. “Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation.” 2013. Doctoral Dissertation, University of Oxford. Accessed July 16, 2019. http://ora.ox.ac.uk/objects/uuid:775c4150-9b22-4008-9288-3a6053cbf4cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647535.

MLA Handbook (7th Edition):

Nordlander, Sofia. “Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation.” 2013. Web. 16 Jul 2019.

Vancouver:

Nordlander S. Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2019 Jul 16]. Available from: http://ora.ox.ac.uk/objects/uuid:775c4150-9b22-4008-9288-3a6053cbf4cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647535.

Council of Science Editors:

Nordlander S. Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:775c4150-9b22-4008-9288-3a6053cbf4cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647535


University of Oxford

2. Trendel, Nicola Charlotte. Phenotypic modelling of the polyfunctional T cell response.

Degree: PhD, 2018, University of Oxford

 T cells are important mediators of adaptive immunity and promising agents in immunotherapies. When T cell receptors (TCRs) on the surface of CD8+ T cells… (more)

Subjects/Keywords: Computational Immunology; Systems Biology; Immunology

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APA (6th Edition):

Trendel, N. C. (2018). Phenotypic modelling of the polyfunctional T cell response. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:31a57248-5825-4534-89ef-b63ec7e085cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.770561

Chicago Manual of Style (16th Edition):

Trendel, Nicola Charlotte. “Phenotypic modelling of the polyfunctional T cell response.” 2018. Doctoral Dissertation, University of Oxford. Accessed July 16, 2019. http://ora.ox.ac.uk/objects/uuid:31a57248-5825-4534-89ef-b63ec7e085cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.770561.

MLA Handbook (7th Edition):

Trendel, Nicola Charlotte. “Phenotypic modelling of the polyfunctional T cell response.” 2018. Web. 16 Jul 2019.

Vancouver:

Trendel NC. Phenotypic modelling of the polyfunctional T cell response. [Internet] [Doctoral dissertation]. University of Oxford; 2018. [cited 2019 Jul 16]. Available from: http://ora.ox.ac.uk/objects/uuid:31a57248-5825-4534-89ef-b63ec7e085cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.770561.

Council of Science Editors:

Trendel NC. Phenotypic modelling of the polyfunctional T cell response. [Doctoral Dissertation]. University of Oxford; 2018. Available from: http://ora.ox.ac.uk/objects/uuid:31a57248-5825-4534-89ef-b63ec7e085cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.770561


University of Nottingham

3. Mirza, Deeman. Probing the interaction of hepatitis C virus glycoproteins with putative receptors and neutralising antibodies.

Degree: 2012, University of Nottingham

 Hepatitis C virus (HCV) is a hepatotropic blood-borne virus which causes chronic hepatitis in the majority of cases and represents a global health burden. In… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Mirza, D. (2012). Probing the interaction of hepatitis C virus glycoproteins with putative receptors and neutralising antibodies. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/12685/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.564411

Chicago Manual of Style (16th Edition):

Mirza, Deeman. “Probing the interaction of hepatitis C virus glycoproteins with putative receptors and neutralising antibodies.” 2012. Doctoral Dissertation, University of Nottingham. Accessed July 16, 2019. http://eprints.nottingham.ac.uk/12685/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.564411.

MLA Handbook (7th Edition):

Mirza, Deeman. “Probing the interaction of hepatitis C virus glycoproteins with putative receptors and neutralising antibodies.” 2012. Web. 16 Jul 2019.

Vancouver:

Mirza D. Probing the interaction of hepatitis C virus glycoproteins with putative receptors and neutralising antibodies. [Internet] [Doctoral dissertation]. University of Nottingham; 2012. [cited 2019 Jul 16]. Available from: http://eprints.nottingham.ac.uk/12685/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.564411.

Council of Science Editors:

Mirza D. Probing the interaction of hepatitis C virus glycoproteins with putative receptors and neutralising antibodies. [Doctoral Dissertation]. University of Nottingham; 2012. Available from: http://eprints.nottingham.ac.uk/12685/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.564411


University of Glasgow

4. van der Ventel, Michelle. The role of IL-4Ra signalling in gene deficient mice during asexual-stage Plasmodium chabaudi AS infection.

Degree: PhD, 2012, University of Glasgow

 BALB/c mice infected with P. chabaudi AS develop immunity to erythrocytic-stage infection with early Th1 responses followed by a switch towards Th2 responses later to… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

van der Ventel, M. (2012). The role of IL-4Ra signalling in gene deficient mice during asexual-stage Plasmodium chabaudi AS infection. (Doctoral Dissertation). University of Glasgow. Retrieved from http://theses.gla.ac.uk/3438/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.552969

Chicago Manual of Style (16th Edition):

van der Ventel, Michelle. “The role of IL-4Ra signalling in gene deficient mice during asexual-stage Plasmodium chabaudi AS infection.” 2012. Doctoral Dissertation, University of Glasgow. Accessed July 16, 2019. http://theses.gla.ac.uk/3438/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.552969.

MLA Handbook (7th Edition):

van der Ventel, Michelle. “The role of IL-4Ra signalling in gene deficient mice during asexual-stage Plasmodium chabaudi AS infection.” 2012. Web. 16 Jul 2019.

Vancouver:

van der Ventel M. The role of IL-4Ra signalling in gene deficient mice during asexual-stage Plasmodium chabaudi AS infection. [Internet] [Doctoral dissertation]. University of Glasgow; 2012. [cited 2019 Jul 16]. Available from: http://theses.gla.ac.uk/3438/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.552969.

Council of Science Editors:

van der Ventel M. The role of IL-4Ra signalling in gene deficient mice during asexual-stage Plasmodium chabaudi AS infection. [Doctoral Dissertation]. University of Glasgow; 2012. Available from: http://theses.gla.ac.uk/3438/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.552969


University of Glasgow

5. Montgomery, Jennifer. Interrogation and modulation of the myeloid aspect of the inflammatory immune response in spinal cord injury.

Degree: PhD, 2013, University of Glasgow

 Spinal cord injury (SCI) affects approximately 40,000 people in the UK; the most common type of SCI is a contusion injury and the majority of… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Montgomery, J. (2013). Interrogation and modulation of the myeloid aspect of the inflammatory immune response in spinal cord injury. (Doctoral Dissertation). University of Glasgow. Retrieved from http://theses.gla.ac.uk/4820/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.591954

Chicago Manual of Style (16th Edition):

Montgomery, Jennifer. “Interrogation and modulation of the myeloid aspect of the inflammatory immune response in spinal cord injury.” 2013. Doctoral Dissertation, University of Glasgow. Accessed July 16, 2019. http://theses.gla.ac.uk/4820/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.591954.

MLA Handbook (7th Edition):

Montgomery, Jennifer. “Interrogation and modulation of the myeloid aspect of the inflammatory immune response in spinal cord injury.” 2013. Web. 16 Jul 2019.

Vancouver:

Montgomery J. Interrogation and modulation of the myeloid aspect of the inflammatory immune response in spinal cord injury. [Internet] [Doctoral dissertation]. University of Glasgow; 2013. [cited 2019 Jul 16]. Available from: http://theses.gla.ac.uk/4820/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.591954.

Council of Science Editors:

Montgomery J. Interrogation and modulation of the myeloid aspect of the inflammatory immune response in spinal cord injury. [Doctoral Dissertation]. University of Glasgow; 2013. Available from: http://theses.gla.ac.uk/4820/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.591954


University of Glasgow

6. Rodgers, David T. Investigation of the therapeutic potential of ES-62 in a murine model of SLE.

Degree: PhD, 2013, University of Glasgow

 Autoimmune inflammatory disorders such as systemic lupus erythematosus (SLE) remain debilitating conditions, as many patients are refractory to existing conventional and biologic therapies or suffer… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Rodgers, D. T. (2013). Investigation of the therapeutic potential of ES-62 in a murine model of SLE. (Doctoral Dissertation). University of Glasgow. Retrieved from http://theses.gla.ac.uk/4771/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.601541

Chicago Manual of Style (16th Edition):

Rodgers, David T. “Investigation of the therapeutic potential of ES-62 in a murine model of SLE.” 2013. Doctoral Dissertation, University of Glasgow. Accessed July 16, 2019. http://theses.gla.ac.uk/4771/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.601541.

MLA Handbook (7th Edition):

Rodgers, David T. “Investigation of the therapeutic potential of ES-62 in a murine model of SLE.” 2013. Web. 16 Jul 2019.

Vancouver:

Rodgers DT. Investigation of the therapeutic potential of ES-62 in a murine model of SLE. [Internet] [Doctoral dissertation]. University of Glasgow; 2013. [cited 2019 Jul 16]. Available from: http://theses.gla.ac.uk/4771/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.601541.

Council of Science Editors:

Rodgers DT. Investigation of the therapeutic potential of ES-62 in a murine model of SLE. [Doctoral Dissertation]. University of Glasgow; 2013. Available from: http://theses.gla.ac.uk/4771/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.601541


University of Glasgow

7. Bryce, Steven Alan. Regulation of the bioavailability of CCR7 ligands.

Degree: PhD, 2014, University of Glasgow

 The efficient functioning of the immune system is dependent on the coordinated movement and positioning of immune cells. These cells patrol the body and facilitate… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Bryce, S. A. (2014). Regulation of the bioavailability of CCR7 ligands. (Doctoral Dissertation). University of Glasgow. Retrieved from http://theses.gla.ac.uk/5904/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.732713

Chicago Manual of Style (16th Edition):

Bryce, Steven Alan. “Regulation of the bioavailability of CCR7 ligands.” 2014. Doctoral Dissertation, University of Glasgow. Accessed July 16, 2019. http://theses.gla.ac.uk/5904/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.732713.

MLA Handbook (7th Edition):

Bryce, Steven Alan. “Regulation of the bioavailability of CCR7 ligands.” 2014. Web. 16 Jul 2019.

Vancouver:

Bryce SA. Regulation of the bioavailability of CCR7 ligands. [Internet] [Doctoral dissertation]. University of Glasgow; 2014. [cited 2019 Jul 16]. Available from: http://theses.gla.ac.uk/5904/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.732713.

Council of Science Editors:

Bryce SA. Regulation of the bioavailability of CCR7 ligands. [Doctoral Dissertation]. University of Glasgow; 2014. Available from: http://theses.gla.ac.uk/5904/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.732713


University of Glasgow

8. Jaigirdar, Shafqat Ahrar. Investigating the molecular mechanisms of CD4 T cell persistence at inflamed peripheral tissues.

Degree: PhD, 2018, University of Glasgow

 CD4 T cells play an important role in the initiation and maintenance of inflammation in numerous inflammatory diseases. Rheumatoid arthritis (RA) is one such autoimmune… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Jaigirdar, S. A. (2018). Investigating the molecular mechanisms of CD4 T cell persistence at inflamed peripheral tissues. (Doctoral Dissertation). University of Glasgow. Retrieved from http://theses.gla.ac.uk/8952/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.739281

Chicago Manual of Style (16th Edition):

Jaigirdar, Shafqat Ahrar. “Investigating the molecular mechanisms of CD4 T cell persistence at inflamed peripheral tissues.” 2018. Doctoral Dissertation, University of Glasgow. Accessed July 16, 2019. http://theses.gla.ac.uk/8952/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.739281.

MLA Handbook (7th Edition):

Jaigirdar, Shafqat Ahrar. “Investigating the molecular mechanisms of CD4 T cell persistence at inflamed peripheral tissues.” 2018. Web. 16 Jul 2019.

Vancouver:

Jaigirdar SA. Investigating the molecular mechanisms of CD4 T cell persistence at inflamed peripheral tissues. [Internet] [Doctoral dissertation]. University of Glasgow; 2018. [cited 2019 Jul 16]. Available from: http://theses.gla.ac.uk/8952/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.739281.

Council of Science Editors:

Jaigirdar SA. Investigating the molecular mechanisms of CD4 T cell persistence at inflamed peripheral tissues. [Doctoral Dissertation]. University of Glasgow; 2018. Available from: http://theses.gla.ac.uk/8952/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.739281


University of Glasgow

9. Thirlwell, Kayleigh. Tissue origin dictates mesenchymal stromal cell chemokine and chemokine receptor repertoire and predicts in vitro chemotactic activity under homeostatic and inflammatory conditions.

Degree: PhD, 2018, University of Glasgow

 Due to their anti-inflammatory and immunomodulatory properties, mesenchymal stromal cells (MSCs) are under intense investigation in many pre-clinical and clinical trials as a potential cellular… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Thirlwell, K. (2018). Tissue origin dictates mesenchymal stromal cell chemokine and chemokine receptor repertoire and predicts in vitro chemotactic activity under homeostatic and inflammatory conditions. (Doctoral Dissertation). University of Glasgow. Retrieved from http://theses.gla.ac.uk/8951/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.739280

Chicago Manual of Style (16th Edition):

Thirlwell, Kayleigh. “Tissue origin dictates mesenchymal stromal cell chemokine and chemokine receptor repertoire and predicts in vitro chemotactic activity under homeostatic and inflammatory conditions.” 2018. Doctoral Dissertation, University of Glasgow. Accessed July 16, 2019. http://theses.gla.ac.uk/8951/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.739280.

MLA Handbook (7th Edition):

Thirlwell, Kayleigh. “Tissue origin dictates mesenchymal stromal cell chemokine and chemokine receptor repertoire and predicts in vitro chemotactic activity under homeostatic and inflammatory conditions.” 2018. Web. 16 Jul 2019.

Vancouver:

Thirlwell K. Tissue origin dictates mesenchymal stromal cell chemokine and chemokine receptor repertoire and predicts in vitro chemotactic activity under homeostatic and inflammatory conditions. [Internet] [Doctoral dissertation]. University of Glasgow; 2018. [cited 2019 Jul 16]. Available from: http://theses.gla.ac.uk/8951/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.739280.

Council of Science Editors:

Thirlwell K. Tissue origin dictates mesenchymal stromal cell chemokine and chemokine receptor repertoire and predicts in vitro chemotactic activity under homeostatic and inflammatory conditions. [Doctoral Dissertation]. University of Glasgow; 2018. Available from: http://theses.gla.ac.uk/8951/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.739280


University of Glasgow

10. Craig, Pauline Claire. Human recombinant Fc constructs: production and anti-osteoclastogenic effects.

Degree: PhD, 2015, University of Glasgow

 Staphylococcal protein A (SpA) produced by Staphylococcus aureus, binds Immunoglobulin G (IgG), forming SpA-IgG immune complexes (SIC), at a ratio of 2 SpA molecules to… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Craig, P. C. (2015). Human recombinant Fc constructs: production and anti-osteoclastogenic effects. (Doctoral Dissertation). University of Glasgow. Retrieved from http://theses.gla.ac.uk/6208/

Chicago Manual of Style (16th Edition):

Craig, Pauline Claire. “Human recombinant Fc constructs: production and anti-osteoclastogenic effects.” 2015. Doctoral Dissertation, University of Glasgow. Accessed July 16, 2019. http://theses.gla.ac.uk/6208/.

MLA Handbook (7th Edition):

Craig, Pauline Claire. “Human recombinant Fc constructs: production and anti-osteoclastogenic effects.” 2015. Web. 16 Jul 2019.

Vancouver:

Craig PC. Human recombinant Fc constructs: production and anti-osteoclastogenic effects. [Internet] [Doctoral dissertation]. University of Glasgow; 2015. [cited 2019 Jul 16]. Available from: http://theses.gla.ac.uk/6208/.

Council of Science Editors:

Craig PC. Human recombinant Fc constructs: production and anti-osteoclastogenic effects. [Doctoral Dissertation]. University of Glasgow; 2015. Available from: http://theses.gla.ac.uk/6208/


University of Glasgow

11. Jaigirdar, Shafqat Ahrar. Investigating the molecular mechanisms of CD4 T cell persistence at inflamed peripheral tissues.

Degree: PhD, 2018, University of Glasgow

 CD4 T cells play an important role in the initiation and maintenance of inflammation in numerous inflammatory diseases. Rheumatoid arthritis (RA) is one such autoimmune… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Jaigirdar, S. A. (2018). Investigating the molecular mechanisms of CD4 T cell persistence at inflamed peripheral tissues. (Doctoral Dissertation). University of Glasgow. Retrieved from http://theses.gla.ac.uk/8952/http://theses.gla.ac.uk/8952/7/Video1.mov ; http://theses.gla.ac.uk/8952/8/Video2.mov ; http://theses.gla.ac.uk/8952/9/Video3.mov ; http://theses.gla.ac.uk/8952/10/Video4.mov

Chicago Manual of Style (16th Edition):

Jaigirdar, Shafqat Ahrar. “Investigating the molecular mechanisms of CD4 T cell persistence at inflamed peripheral tissues.” 2018. Doctoral Dissertation, University of Glasgow. Accessed July 16, 2019. http://theses.gla.ac.uk/8952/http://theses.gla.ac.uk/8952/7/Video1.mov ; http://theses.gla.ac.uk/8952/8/Video2.mov ; http://theses.gla.ac.uk/8952/9/Video3.mov ; http://theses.gla.ac.uk/8952/10/Video4.mov.

MLA Handbook (7th Edition):

Jaigirdar, Shafqat Ahrar. “Investigating the molecular mechanisms of CD4 T cell persistence at inflamed peripheral tissues.” 2018. Web. 16 Jul 2019.

Vancouver:

Jaigirdar SA. Investigating the molecular mechanisms of CD4 T cell persistence at inflamed peripheral tissues. [Internet] [Doctoral dissertation]. University of Glasgow; 2018. [cited 2019 Jul 16]. Available from: http://theses.gla.ac.uk/8952/http://theses.gla.ac.uk/8952/7/Video1.mov ; http://theses.gla.ac.uk/8952/8/Video2.mov ; http://theses.gla.ac.uk/8952/9/Video3.mov ; http://theses.gla.ac.uk/8952/10/Video4.mov.

Council of Science Editors:

Jaigirdar SA. Investigating the molecular mechanisms of CD4 T cell persistence at inflamed peripheral tissues. [Doctoral Dissertation]. University of Glasgow; 2018. Available from: http://theses.gla.ac.uk/8952/http://theses.gla.ac.uk/8952/7/Video1.mov ; http://theses.gla.ac.uk/8952/8/Video2.mov ; http://theses.gla.ac.uk/8952/9/Video3.mov ; http://theses.gla.ac.uk/8952/10/Video4.mov


University of Oxford

12. Kendal, Adrian Reginald. The role of Foxp3+ regulatory T-cells in transplant tolerance.

Degree: PhD, 2011, University of Oxford

 A major conceptual shift in immunology has been the recent discovery of regulatory T-cells (Treg), of which CD4+Foxp3+ cells are already known to be essential… (more)

Subjects/Keywords: 617.954; Immunology

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APA (6th Edition):

Kendal, A. R. (2011). The role of Foxp3+ regulatory T-cells in transplant tolerance. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:22cd4a05-2412-4205-bb93-abd33c85335f ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558327

Chicago Manual of Style (16th Edition):

Kendal, Adrian Reginald. “The role of Foxp3+ regulatory T-cells in transplant tolerance.” 2011. Doctoral Dissertation, University of Oxford. Accessed July 16, 2019. http://ora.ox.ac.uk/objects/uuid:22cd4a05-2412-4205-bb93-abd33c85335f ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558327.

MLA Handbook (7th Edition):

Kendal, Adrian Reginald. “The role of Foxp3+ regulatory T-cells in transplant tolerance.” 2011. Web. 16 Jul 2019.

Vancouver:

Kendal AR. The role of Foxp3+ regulatory T-cells in transplant tolerance. [Internet] [Doctoral dissertation]. University of Oxford; 2011. [cited 2019 Jul 16]. Available from: http://ora.ox.ac.uk/objects/uuid:22cd4a05-2412-4205-bb93-abd33c85335f ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558327.

Council of Science Editors:

Kendal AR. The role of Foxp3+ regulatory T-cells in transplant tolerance. [Doctoral Dissertation]. University of Oxford; 2011. Available from: http://ora.ox.ac.uk/objects/uuid:22cd4a05-2412-4205-bb93-abd33c85335f ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558327


University of Oxford

13. Halliday, John Stuart. Vaccination for hepatitis C : characterising the host immune response and its implications for a successful therapeutic vaccine.

Degree: PhD, 2013, University of Oxford

 This thesis explores the first-ever administration of a prime/boost combination of adenovirus and MVA vector based vaccines that target hepatitis C virus (HCV) infection to… (more)

Subjects/Keywords: 616.3623; Immunology

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APA (6th Edition):

Halliday, J. S. (2013). Vaccination for hepatitis C : characterising the host immune response and its implications for a successful therapeutic vaccine. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:c5d0f8c2-a6d9-4f0d-9d68-bcd2dca5f0e4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.598044

Chicago Manual of Style (16th Edition):

Halliday, John Stuart. “Vaccination for hepatitis C : characterising the host immune response and its implications for a successful therapeutic vaccine.” 2013. Doctoral Dissertation, University of Oxford. Accessed July 16, 2019. http://ora.ox.ac.uk/objects/uuid:c5d0f8c2-a6d9-4f0d-9d68-bcd2dca5f0e4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.598044.

MLA Handbook (7th Edition):

Halliday, John Stuart. “Vaccination for hepatitis C : characterising the host immune response and its implications for a successful therapeutic vaccine.” 2013. Web. 16 Jul 2019.

Vancouver:

Halliday JS. Vaccination for hepatitis C : characterising the host immune response and its implications for a successful therapeutic vaccine. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2019 Jul 16]. Available from: http://ora.ox.ac.uk/objects/uuid:c5d0f8c2-a6d9-4f0d-9d68-bcd2dca5f0e4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.598044.

Council of Science Editors:

Halliday JS. Vaccination for hepatitis C : characterising the host immune response and its implications for a successful therapeutic vaccine. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:c5d0f8c2-a6d9-4f0d-9d68-bcd2dca5f0e4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.598044


University of Birmingham

14. Wall, Nadezhda Alexandrovna. Investigating the immune system in chrONIC kidney disease - the SONIC study.

Degree: d_ph, College of Medical & Dental Sciences, 2019, University of Birmingham

 Chronic kidney disease (CKD) is associated with a high infective burden and poorer vaccine responses. This thesis presents findings of a prospective observational study, where… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Wall, N. A. (2019). Investigating the immune system in chrONIC kidney disease - the SONIC study. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/8800/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wall, Nadezhda Alexandrovna. “Investigating the immune system in chrONIC kidney disease - the SONIC study.” 2019. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/8800/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wall, Nadezhda Alexandrovna. “Investigating the immune system in chrONIC kidney disease - the SONIC study.” 2019. Web. 16 Jul 2019.

Vancouver:

Wall NA. Investigating the immune system in chrONIC kidney disease - the SONIC study. [Internet] [Thesis]. University of Birmingham; 2019. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/8800/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wall NA. Investigating the immune system in chrONIC kidney disease - the SONIC study. [Thesis]. University of Birmingham; 2019. Available from: http://etheses.bham.ac.uk//id/eprint/8800/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

15. Keating, Thomas Oliver. An in vitro investigation of Mycobacterium Tuberculosis biofilm formation and its effect on the host innate immune response.

Degree: d_ph, College of Life & Environmental Sciences, 2018, University of Birmingham

 Mycobacterium tuberculosis is ostensibly an intracellular pathogen, which may form pellicle-like biofilms in the peripheries of tuberculosis cavities. Environment-induced cell wall modifications and extracellular polymeric… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Keating, T. O. (2018). An in vitro investigation of Mycobacterium Tuberculosis biofilm formation and its effect on the host innate immune response. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/8430/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Keating, Thomas Oliver. “An in vitro investigation of Mycobacterium Tuberculosis biofilm formation and its effect on the host innate immune response.” 2018. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/8430/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Keating, Thomas Oliver. “An in vitro investigation of Mycobacterium Tuberculosis biofilm formation and its effect on the host innate immune response.” 2018. Web. 16 Jul 2019.

Vancouver:

Keating TO. An in vitro investigation of Mycobacterium Tuberculosis biofilm formation and its effect on the host innate immune response. [Internet] [Thesis]. University of Birmingham; 2018. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/8430/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Keating TO. An in vitro investigation of Mycobacterium Tuberculosis biofilm formation and its effect on the host innate immune response. [Thesis]. University of Birmingham; 2018. Available from: http://etheses.bham.ac.uk//id/eprint/8430/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

16. Petrovic, Kristina. Exploring the therapeutic potential of CAR-engineered T-cells targeting endothelial markers on tumour and inflamed vasculature.

Degree: d_ph, College of Medical & Dental Sciences, 2018, University of Birmingham

 T-cells engineered to target tumour antigens through surface-expressed chimeric antigen receptors (CARs) are highly effective in treating some leukaemias. The challenge is to extend this… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Petrovic, K. (2018). Exploring the therapeutic potential of CAR-engineered T-cells targeting endothelial markers on tumour and inflamed vasculature. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/8468/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Petrovic, Kristina. “Exploring the therapeutic potential of CAR-engineered T-cells targeting endothelial markers on tumour and inflamed vasculature.” 2018. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/8468/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Petrovic, Kristina. “Exploring the therapeutic potential of CAR-engineered T-cells targeting endothelial markers on tumour and inflamed vasculature.” 2018. Web. 16 Jul 2019.

Vancouver:

Petrovic K. Exploring the therapeutic potential of CAR-engineered T-cells targeting endothelial markers on tumour and inflamed vasculature. [Internet] [Thesis]. University of Birmingham; 2018. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/8468/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Petrovic K. Exploring the therapeutic potential of CAR-engineered T-cells targeting endothelial markers on tumour and inflamed vasculature. [Thesis]. University of Birmingham; 2018. Available from: http://etheses.bham.ac.uk//id/eprint/8468/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

17. Hampson, Judith Anne. Immune activation in alpha-1 antitrypsin deficiency related and usual chronic obstructive pulmonary disease.

Degree: h_md, College of Medical & Dental Sciences, 2018, University of Birmingham

 A polyclonal increase in free light chains (FLCs) has been observed in a number of chronic inflammatory and autoimmune conditions and may be considered a… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Hampson, J. A. (2018). Immune activation in alpha-1 antitrypsin deficiency related and usual chronic obstructive pulmonary disease. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/8473/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hampson, Judith Anne. “Immune activation in alpha-1 antitrypsin deficiency related and usual chronic obstructive pulmonary disease.” 2018. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/8473/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hampson, Judith Anne. “Immune activation in alpha-1 antitrypsin deficiency related and usual chronic obstructive pulmonary disease.” 2018. Web. 16 Jul 2019.

Vancouver:

Hampson JA. Immune activation in alpha-1 antitrypsin deficiency related and usual chronic obstructive pulmonary disease. [Internet] [Thesis]. University of Birmingham; 2018. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/8473/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hampson JA. Immune activation in alpha-1 antitrypsin deficiency related and usual chronic obstructive pulmonary disease. [Thesis]. University of Birmingham; 2018. Available from: http://etheses.bham.ac.uk//id/eprint/8473/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

18. Than, Nwe Ni. Stem cell therapy in liver disease.

Degree: h_md, College of Medical & Dental Sciences, 2018, University of Birmingham

 Liver cirrhosis is the fifth leading cause of death worldwide and the definitive treatment for liver cirrhosis is liver transplantation although there are limitations such… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Than, N. N. (2018). Stem cell therapy in liver disease. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/8482/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Than, Nwe Ni. “Stem cell therapy in liver disease.” 2018. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/8482/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Than, Nwe Ni. “Stem cell therapy in liver disease.” 2018. Web. 16 Jul 2019.

Vancouver:

Than NN. Stem cell therapy in liver disease. [Internet] [Thesis]. University of Birmingham; 2018. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/8482/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Than NN. Stem cell therapy in liver disease. [Thesis]. University of Birmingham; 2018. Available from: http://etheses.bham.ac.uk//id/eprint/8482/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

19. Ruth, Nicola Dawn. Capturing T-cell receptors - a potential new modality for targeting hepatic tumours and post-transplantational lymphoproliferative disease (PLTD).

Degree: d_ph, College of Medical & Dental Sciences, 2018, University of Birmingham

 This project sought to identify paediatric tumour-specific phosphopeptide antigens on Post-transplantation lymphoproliferative disease samples (PTLD) and hepatic tumour samples. Tumour-specific T-cells are difficult to maintain… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Ruth, N. D. (2018). Capturing T-cell receptors - a potential new modality for targeting hepatic tumours and post-transplantational lymphoproliferative disease (PLTD). (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/8533/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ruth, Nicola Dawn. “Capturing T-cell receptors - a potential new modality for targeting hepatic tumours and post-transplantational lymphoproliferative disease (PLTD).” 2018. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/8533/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ruth, Nicola Dawn. “Capturing T-cell receptors - a potential new modality for targeting hepatic tumours and post-transplantational lymphoproliferative disease (PLTD).” 2018. Web. 16 Jul 2019.

Vancouver:

Ruth ND. Capturing T-cell receptors - a potential new modality for targeting hepatic tumours and post-transplantational lymphoproliferative disease (PLTD). [Internet] [Thesis]. University of Birmingham; 2018. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/8533/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ruth ND. Capturing T-cell receptors - a potential new modality for targeting hepatic tumours and post-transplantational lymphoproliferative disease (PLTD). [Thesis]. University of Birmingham; 2018. Available from: http://etheses.bham.ac.uk//id/eprint/8533/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

20. Lim, Teegan Reina. Metabolic effects of hypoxia and chronic hepatitis C.

Degree: d_ph, College of Medical & Dental Sciences, 2018, University of Birmingham

 Hypoxia has been linked to the pathogenesis of hepatic steatosis in murine and human models. There is an abundance of data suggesting that HIFs play… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Lim, T. R. (2018). Metabolic effects of hypoxia and chronic hepatitis C. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/8535/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lim, Teegan Reina. “Metabolic effects of hypoxia and chronic hepatitis C.” 2018. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/8535/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lim, Teegan Reina. “Metabolic effects of hypoxia and chronic hepatitis C.” 2018. Web. 16 Jul 2019.

Vancouver:

Lim TR. Metabolic effects of hypoxia and chronic hepatitis C. [Internet] [Thesis]. University of Birmingham; 2018. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/8535/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lim TR. Metabolic effects of hypoxia and chronic hepatitis C. [Thesis]. University of Birmingham; 2018. Available from: http://etheses.bham.ac.uk//id/eprint/8535/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

21. Preece, Shân. Harnessing CD4+ T cell effectors for lymphoma therapy.

Degree: m_sc, College of Medical & Dental Sciences, 2018, University of Birmingham

 CD4+ T cells play a pivotal role in orchestrating immune responses. It has now been shown that some cells display direct effector functioning in the… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Preece, S. (2018). Harnessing CD4+ T cell effectors for lymphoma therapy. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/8582/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Preece, Shân. “Harnessing CD4+ T cell effectors for lymphoma therapy.” 2018. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/8582/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Preece, Shân. “Harnessing CD4+ T cell effectors for lymphoma therapy.” 2018. Web. 16 Jul 2019.

Vancouver:

Preece S. Harnessing CD4+ T cell effectors for lymphoma therapy. [Internet] [Thesis]. University of Birmingham; 2018. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/8582/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Preece S. Harnessing CD4+ T cell effectors for lymphoma therapy. [Thesis]. University of Birmingham; 2018. Available from: http://etheses.bham.ac.uk//id/eprint/8582/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

22. Arcuri, Melissa. Novel Vi conjugate vaccines against typhoid.

Degree: d_ph, College of Medical & Dental Sciences, 2017, University of Birmingham

 Typhoid fever remains a major public health concern in low-income countries affecting millions of people each year. Research on effective vaccines against Salmonella Typhi has… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Arcuri, M. (2017). Novel Vi conjugate vaccines against typhoid. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/7736/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arcuri, Melissa. “Novel Vi conjugate vaccines against typhoid.” 2017. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/7736/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arcuri, Melissa. “Novel Vi conjugate vaccines against typhoid.” 2017. Web. 16 Jul 2019.

Vancouver:

Arcuri M. Novel Vi conjugate vaccines against typhoid. [Internet] [Thesis]. University of Birmingham; 2017. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/7736/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arcuri M. Novel Vi conjugate vaccines against typhoid. [Thesis]. University of Birmingham; 2017. Available from: http://etheses.bham.ac.uk//id/eprint/7736/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

23. Oo, Ye Htun. Recruitment and positioning of regulatory T cells and Th17/Tc17 in inflamed human liver.

Degree: d_ph, College of Medical & Dental Sciences, 2010, University of Birmingham

 The liver is a unique tolerogenic organ with dual blood supply. Both regulatory lymphocytes and effector lymphocytes are present in the normal and inflamed liver… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Oo, Y. H. (2010). Recruitment and positioning of regulatory T cells and Th17/Tc17 in inflamed human liver. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/1128/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oo, Ye Htun. “Recruitment and positioning of regulatory T cells and Th17/Tc17 in inflamed human liver.” 2010. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/1128/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oo, Ye Htun. “Recruitment and positioning of regulatory T cells and Th17/Tc17 in inflamed human liver.” 2010. Web. 16 Jul 2019.

Vancouver:

Oo YH. Recruitment and positioning of regulatory T cells and Th17/Tc17 in inflamed human liver. [Internet] [Thesis]. University of Birmingham; 2010. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/1128/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Oo YH. Recruitment and positioning of regulatory T cells and Th17/Tc17 in inflamed human liver. [Thesis]. University of Birmingham; 2010. Available from: http://etheses.bham.ac.uk//id/eprint/1128/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

24. Leszczynska, Katarzyna. Signalling and function of the small Rho GTPase RhoJ in endothelial cells.

Degree: d_ph, College of Medical & Dental Sciences, 2011, University of Birmingham

 RhoJ is an endothelial expressed Rho GTPase, and its knock-down impairs endothelial cell (EC) migration and tubulogenesis, increases stress fibre (SF) and focal adhesion (FA)… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Leszczynska, K. (2011). Signalling and function of the small Rho GTPase RhoJ in endothelial cells. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/1495/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Leszczynska, Katarzyna. “Signalling and function of the small Rho GTPase RhoJ in endothelial cells.” 2011. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/1495/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Leszczynska, Katarzyna. “Signalling and function of the small Rho GTPase RhoJ in endothelial cells.” 2011. Web. 16 Jul 2019.

Vancouver:

Leszczynska K. Signalling and function of the small Rho GTPase RhoJ in endothelial cells. [Internet] [Thesis]. University of Birmingham; 2011. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/1495/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Leszczynska K. Signalling and function of the small Rho GTPase RhoJ in endothelial cells. [Thesis]. University of Birmingham; 2011. Available from: http://etheses.bham.ac.uk//id/eprint/1495/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

25. Voice, Marie Ann. 1) Investigation of the clathrin-dependent internalisation of Cytotoxic T Lymphocyte Antigen-4 2) Analysis of the antigen-specific cytokine responses of CD4+ T lymphocytes.

Degree: m_rs, College of Medical & Dental Sciences, 2011, University of Birmingham

 Abstract 1) Cytotoxic T Lymphocyte Antigen-4 (CTLA-4) is a key inhibitory regulator of T-cell function. Its expression is predominantly intracellular however, many theories surrounding it’s… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Voice, M. A. (2011). 1) Investigation of the clathrin-dependent internalisation of Cytotoxic T Lymphocyte Antigen-4 2) Analysis of the antigen-specific cytokine responses of CD4+ T lymphocytes. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/2889/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Voice, Marie Ann. “1) Investigation of the clathrin-dependent internalisation of Cytotoxic T Lymphocyte Antigen-4 2) Analysis of the antigen-specific cytokine responses of CD4+ T lymphocytes.” 2011. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/2889/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Voice, Marie Ann. “1) Investigation of the clathrin-dependent internalisation of Cytotoxic T Lymphocyte Antigen-4 2) Analysis of the antigen-specific cytokine responses of CD4+ T lymphocytes.” 2011. Web. 16 Jul 2019.

Vancouver:

Voice MA. 1) Investigation of the clathrin-dependent internalisation of Cytotoxic T Lymphocyte Antigen-4 2) Analysis of the antigen-specific cytokine responses of CD4+ T lymphocytes. [Internet] [Thesis]. University of Birmingham; 2011. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/2889/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Voice MA. 1) Investigation of the clathrin-dependent internalisation of Cytotoxic T Lymphocyte Antigen-4 2) Analysis of the antigen-specific cytokine responses of CD4+ T lymphocytes. [Thesis]. University of Birmingham; 2011. Available from: http://etheses.bham.ac.uk//id/eprint/2889/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

26. Suresh, Shankar. 1) Neural differentiation of dental pulp stem cells 2) The effect of PDGF, FGF and TGF-β on the proliferation, differentiation and immunomodulatory functions of prospectively-isolated murine mesenchymal stem cells.

Degree: m_rs, College of Medical & Dental Sciences, 2011, University of Birmingham

 Abstract 1) A variety of stem/progenitor populations have been isolated from human dental tissue over the past decade. Of these, dental pulp stem cells (DPSCs)… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

Suresh, S. (2011). 1) Neural differentiation of dental pulp stem cells 2) The effect of PDGF, FGF and TGF-β on the proliferation, differentiation and immunomodulatory functions of prospectively-isolated murine mesenchymal stem cells. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/2893/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Suresh, Shankar. “1) Neural differentiation of dental pulp stem cells 2) The effect of PDGF, FGF and TGF-β on the proliferation, differentiation and immunomodulatory functions of prospectively-isolated murine mesenchymal stem cells.” 2011. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/2893/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Suresh, Shankar. “1) Neural differentiation of dental pulp stem cells 2) The effect of PDGF, FGF and TGF-β on the proliferation, differentiation and immunomodulatory functions of prospectively-isolated murine mesenchymal stem cells.” 2011. Web. 16 Jul 2019.

Vancouver:

Suresh S. 1) Neural differentiation of dental pulp stem cells 2) The effect of PDGF, FGF and TGF-β on the proliferation, differentiation and immunomodulatory functions of prospectively-isolated murine mesenchymal stem cells. [Internet] [Thesis]. University of Birmingham; 2011. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/2893/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Suresh S. 1) Neural differentiation of dental pulp stem cells 2) The effect of PDGF, FGF and TGF-β on the proliferation, differentiation and immunomodulatory functions of prospectively-isolated murine mesenchymal stem cells. [Thesis]. University of Birmingham; 2011. Available from: http://etheses.bham.ac.uk//id/eprint/2893/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

27. Rossiter, Amanda Eve. Deciphering the autotransporter pathway of Gram-negative bacteria; from regulation to secretion.

Degree: d_ph, College of Medical & Dental Sciences, 2011, University of Birmingham

 Autotransporters represent a diverse family of virulence effectors that are secreted from Gram-negative bacteria by the Type V Secretion System. Their initial description coined the… (more)

Subjects/Keywords: QR180 Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rossiter, A. E. (2011). Deciphering the autotransporter pathway of Gram-negative bacteria; from regulation to secretion. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/3046/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rossiter, Amanda Eve. “Deciphering the autotransporter pathway of Gram-negative bacteria; from regulation to secretion.” 2011. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/3046/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rossiter, Amanda Eve. “Deciphering the autotransporter pathway of Gram-negative bacteria; from regulation to secretion.” 2011. Web. 16 Jul 2019.

Vancouver:

Rossiter AE. Deciphering the autotransporter pathway of Gram-negative bacteria; from regulation to secretion. [Internet] [Thesis]. University of Birmingham; 2011. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/3046/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rossiter AE. Deciphering the autotransporter pathway of Gram-negative bacteria; from regulation to secretion. [Thesis]. University of Birmingham; 2011. Available from: http://etheses.bham.ac.uk//id/eprint/3046/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

28. Chimen, Myriam. Immunomodulation by adipokines in type 1 diabetes.

Degree: d_ph, College of Medical & Dental Sciences, 2012, University of Birmingham

 Type 1 diabetes (T1D) is a T cell-mediated autoimmune disease in which the immune system specifically targets and destroys the pancreatic insulin-producing beta-cells. We are… (more)

Subjects/Keywords: QR180 Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chimen, M. (2012). Immunomodulation by adipokines in type 1 diabetes. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/3581/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chimen, Myriam. “Immunomodulation by adipokines in type 1 diabetes.” 2012. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/3581/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chimen, Myriam. “Immunomodulation by adipokines in type 1 diabetes.” 2012. Web. 16 Jul 2019.

Vancouver:

Chimen M. Immunomodulation by adipokines in type 1 diabetes. [Internet] [Thesis]. University of Birmingham; 2012. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/3581/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chimen M. Immunomodulation by adipokines in type 1 diabetes. [Thesis]. University of Birmingham; 2012. Available from: http://etheses.bham.ac.uk//id/eprint/3581/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

29. George, Laura. Regulation of extrafollicular immunoglobulin class switch recombination.

Degree: d_ph, College of Medical & Dental Sciences, 2014, University of Birmingham

 The humoral immune response is characterised by the production of antibody secreting B cells. Some of these cells have cycled through the germinal centre, diversifying… (more)

Subjects/Keywords: QR180 Immunology

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APA (6th Edition):

George, L. (2014). Regulation of extrafollicular immunoglobulin class switch recombination. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/5109/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

George, Laura. “Regulation of extrafollicular immunoglobulin class switch recombination.” 2014. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/5109/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

George, Laura. “Regulation of extrafollicular immunoglobulin class switch recombination.” 2014. Web. 16 Jul 2019.

Vancouver:

George L. Regulation of extrafollicular immunoglobulin class switch recombination. [Internet] [Thesis]. University of Birmingham; 2014. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/5109/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

George L. Regulation of extrafollicular immunoglobulin class switch recombination. [Thesis]. University of Birmingham; 2014. Available from: http://etheses.bham.ac.uk//id/eprint/5109/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Birmingham

30. Jang, Boyun. Analysis of chromatin targeting modules in the chromatin remodelling enzyme NURF.

Degree: d_ph, College of Medical & Dental Sciences, 2014, University of Birmingham

 Drosophila nucleosome remodelling factor (NURF) is one of the founding members of the ISWI family of ATP-dependent chromatin remodelling enzymes and mediates energy-dependent nucleosome sliding… (more)

Subjects/Keywords: QR180 Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jang, B. (2014). Analysis of chromatin targeting modules in the chromatin remodelling enzyme NURF. (Thesis). University of Birmingham. Retrieved from http://etheses.bham.ac.uk//id/eprint/5204/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jang, Boyun. “Analysis of chromatin targeting modules in the chromatin remodelling enzyme NURF.” 2014. Thesis, University of Birmingham. Accessed July 16, 2019. http://etheses.bham.ac.uk//id/eprint/5204/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jang, Boyun. “Analysis of chromatin targeting modules in the chromatin remodelling enzyme NURF.” 2014. Web. 16 Jul 2019.

Vancouver:

Jang B. Analysis of chromatin targeting modules in the chromatin remodelling enzyme NURF. [Internet] [Thesis]. University of Birmingham; 2014. [cited 2019 Jul 16]. Available from: http://etheses.bham.ac.uk//id/eprint/5204/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jang B. Analysis of chromatin targeting modules in the chromatin remodelling enzyme NURF. [Thesis]. University of Birmingham; 2014. Available from: http://etheses.bham.ac.uk//id/eprint/5204/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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