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You searched for subject:(Immunology). Showing records 61 – 90 of 768 total matches.

[1] [2] [3] [4] [5] … [26]

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McGill University

61. Haghayeghi, Amirhossein. Pellino function in «Drosophila» innate immunity.

Degree: MS, Department of Biology, 2010, McGill University

The Toll pathway mediates innate immunity, the first line of defense against pathogens in vertebrates and invertebrates. In Drosophila the Toll pathway protects against Gram-positive… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Haghayeghi, A. (2010). Pellino function in «Drosophila» innate immunity. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile86930.pdf

Chicago Manual of Style (16th Edition):

Haghayeghi, Amirhossein. “Pellino function in «Drosophila» innate immunity.” 2010. Masters Thesis, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile86930.pdf.

MLA Handbook (7th Edition):

Haghayeghi, Amirhossein. “Pellino function in «Drosophila» innate immunity.” 2010. Web. 25 Aug 2019.

Vancouver:

Haghayeghi A. Pellino function in «Drosophila» innate immunity. [Internet] [Masters thesis]. McGill University; 2010. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile86930.pdf.

Council of Science Editors:

Haghayeghi A. Pellino function in «Drosophila» innate immunity. [Masters Thesis]. McGill University; 2010. Available from: http://digitool.library.mcgill.ca/thesisfile86930.pdf


McGill University

62. Shbat, Layla. Immune modulation in cardiovascular disease.

Degree: MS, Department of Medicine, 2011, McGill University

The importance of the adaptive immune response in cardiovascular disease has been increasingly appreciated. However, limited information is available on immune modulation in the context… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Shbat, L. (2011). Immune modulation in cardiovascular disease. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile103617.pdf

Chicago Manual of Style (16th Edition):

Shbat, Layla. “Immune modulation in cardiovascular disease.” 2011. Masters Thesis, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile103617.pdf.

MLA Handbook (7th Edition):

Shbat, Layla. “Immune modulation in cardiovascular disease.” 2011. Web. 25 Aug 2019.

Vancouver:

Shbat L. Immune modulation in cardiovascular disease. [Internet] [Masters thesis]. McGill University; 2011. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile103617.pdf.

Council of Science Editors:

Shbat L. Immune modulation in cardiovascular disease. [Masters Thesis]. McGill University; 2011. Available from: http://digitool.library.mcgill.ca/thesisfile103617.pdf


McGill University

63. Fielhaber, Jill. Regulation of signal tranducer and activator of transcription-1 and cell death by mammalian target of Rapamycin.

Degree: PhD, Department of Microbiology & Immunology, 2011, McGill University

Mammalian target of rapamycin (mTOR) is a highly-conserved serine/threonine kinase that permits anabolic responses to mitogens and metabolic substrates (e.g., insulin, amino acids, ATP). When… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Fielhaber, J. (2011). Regulation of signal tranducer and activator of transcription-1 and cell death by mammalian target of Rapamycin. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile104692.pdf

Chicago Manual of Style (16th Edition):

Fielhaber, Jill. “Regulation of signal tranducer and activator of transcription-1 and cell death by mammalian target of Rapamycin.” 2011. Doctoral Dissertation, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile104692.pdf.

MLA Handbook (7th Edition):

Fielhaber, Jill. “Regulation of signal tranducer and activator of transcription-1 and cell death by mammalian target of Rapamycin.” 2011. Web. 25 Aug 2019.

Vancouver:

Fielhaber J. Regulation of signal tranducer and activator of transcription-1 and cell death by mammalian target of Rapamycin. [Internet] [Doctoral dissertation]. McGill University; 2011. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile104692.pdf.

Council of Science Editors:

Fielhaber J. Regulation of signal tranducer and activator of transcription-1 and cell death by mammalian target of Rapamycin. [Doctoral Dissertation]. McGill University; 2011. Available from: http://digitool.library.mcgill.ca/thesisfile104692.pdf


McGill University

64. Flaczyk, Adam. Induction of the epithelial polarizing cytokine interleukin-33 by the fungus «Cryptococcus neoformans» in genetically susceptible mice.

Degree: MS, Department of Medicine, 2012, McGill University

With the progression of the AIDS epidemic, understanding the host responseto the opportunistic fungal pathogen Cryptococcus neoformans, responsible forapproximately 650,000 deaths in Sub-Saharan Africa each… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Flaczyk, A. (2012). Induction of the epithelial polarizing cytokine interleukin-33 by the fungus «Cryptococcus neoformans» in genetically susceptible mice. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile107706.pdf

Chicago Manual of Style (16th Edition):

Flaczyk, Adam. “Induction of the epithelial polarizing cytokine interleukin-33 by the fungus «Cryptococcus neoformans» in genetically susceptible mice.” 2012. Masters Thesis, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile107706.pdf.

MLA Handbook (7th Edition):

Flaczyk, Adam. “Induction of the epithelial polarizing cytokine interleukin-33 by the fungus «Cryptococcus neoformans» in genetically susceptible mice.” 2012. Web. 25 Aug 2019.

Vancouver:

Flaczyk A. Induction of the epithelial polarizing cytokine interleukin-33 by the fungus «Cryptococcus neoformans» in genetically susceptible mice. [Internet] [Masters thesis]. McGill University; 2012. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile107706.pdf.

Council of Science Editors:

Flaczyk A. Induction of the epithelial polarizing cytokine interleukin-33 by the fungus «Cryptococcus neoformans» in genetically susceptible mice. [Masters Thesis]. McGill University; 2012. Available from: http://digitool.library.mcgill.ca/thesisfile107706.pdf


McGill University

65. Ralph, Benjamin. SHP-1/PTP-1B Macrophage interactome upon «Leishmania mexicana» infection.

Degree: MS, Department of Microbiology & Immunology, 2012, McGill University

Leishmaniasis is a disease endemic to 88 countries that affects 12 million people worldwide. The causative agents are species of the genus Leishmania, with the… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Ralph, B. (2012). SHP-1/PTP-1B Macrophage interactome upon «Leishmania mexicana» infection. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile110597.pdf

Chicago Manual of Style (16th Edition):

Ralph, Benjamin. “SHP-1/PTP-1B Macrophage interactome upon «Leishmania mexicana» infection.” 2012. Masters Thesis, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile110597.pdf.

MLA Handbook (7th Edition):

Ralph, Benjamin. “SHP-1/PTP-1B Macrophage interactome upon «Leishmania mexicana» infection.” 2012. Web. 25 Aug 2019.

Vancouver:

Ralph B. SHP-1/PTP-1B Macrophage interactome upon «Leishmania mexicana» infection. [Internet] [Masters thesis]. McGill University; 2012. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile110597.pdf.

Council of Science Editors:

Ralph B. SHP-1/PTP-1B Macrophage interactome upon «Leishmania mexicana» infection. [Masters Thesis]. McGill University; 2012. Available from: http://digitool.library.mcgill.ca/thesisfile110597.pdf


McGill University

66. Williams, Patrick. The development and characterization of fusion cytokines of GMCSF with IL-21 and IFN-y for cancer immunotherapy.

Degree: PhD, Department of Medicine, 2011, McGill University

Different immunotherapeutic strategies have been attempted against cancer, ranging from the use of cytokines like IL-2, to the use of dendritic cells, like Provenge, or… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Williams, P. (2011). The development and characterization of fusion cytokines of GMCSF with IL-21 and IFN-y for cancer immunotherapy. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile96679.pdf

Chicago Manual of Style (16th Edition):

Williams, Patrick. “The development and characterization of fusion cytokines of GMCSF with IL-21 and IFN-y for cancer immunotherapy.” 2011. Doctoral Dissertation, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile96679.pdf.

MLA Handbook (7th Edition):

Williams, Patrick. “The development and characterization of fusion cytokines of GMCSF with IL-21 and IFN-y for cancer immunotherapy.” 2011. Web. 25 Aug 2019.

Vancouver:

Williams P. The development and characterization of fusion cytokines of GMCSF with IL-21 and IFN-y for cancer immunotherapy. [Internet] [Doctoral dissertation]. McGill University; 2011. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile96679.pdf.

Council of Science Editors:

Williams P. The development and characterization of fusion cytokines of GMCSF with IL-21 and IFN-y for cancer immunotherapy. [Doctoral Dissertation]. McGill University; 2011. Available from: http://digitool.library.mcgill.ca/thesisfile96679.pdf


McGill University

67. Bélanger, Simon. The role of natural killer cell receptors in the control of natural killer cell functions.

Degree: PhD, Department of Microbiology & Immunology, 2012, McGill University

Natural killer (NK) cells possess the ability to destroy abnormal cells such as cancerous and virally-infected cells. A common feature of these cells is the… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Bélanger, S. (2012). The role of natural killer cell receptors in the control of natural killer cell functions. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile107643.pdf

Chicago Manual of Style (16th Edition):

Bélanger, Simon. “The role of natural killer cell receptors in the control of natural killer cell functions.” 2012. Doctoral Dissertation, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile107643.pdf.

MLA Handbook (7th Edition):

Bélanger, Simon. “The role of natural killer cell receptors in the control of natural killer cell functions.” 2012. Web. 25 Aug 2019.

Vancouver:

Bélanger S. The role of natural killer cell receptors in the control of natural killer cell functions. [Internet] [Doctoral dissertation]. McGill University; 2012. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile107643.pdf.

Council of Science Editors:

Bélanger S. The role of natural killer cell receptors in the control of natural killer cell functions. [Doctoral Dissertation]. McGill University; 2012. Available from: http://digitool.library.mcgill.ca/thesisfile107643.pdf


McGill University

68. Labbe, Roman. Post transcriptional control of HIV-1 latency by the PKR and p53 pathways.

Degree: MS, Department of Microbiology & Immunology, 2017, McGill University

Latency represents the major obstacle currently faced in the development of a curative HIV therapy. Latency is responsible for the persistence of HIV in treated… (more)

Subjects/Keywords: Microbiology and Immunology

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APA (6th Edition):

Labbe, R. (2017). Post transcriptional control of HIV-1 latency by the PKR and p53 pathways. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile147057.pdf

Chicago Manual of Style (16th Edition):

Labbe, Roman. “Post transcriptional control of HIV-1 latency by the PKR and p53 pathways.” 2017. Masters Thesis, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile147057.pdf.

MLA Handbook (7th Edition):

Labbe, Roman. “Post transcriptional control of HIV-1 latency by the PKR and p53 pathways.” 2017. Web. 25 Aug 2019.

Vancouver:

Labbe R. Post transcriptional control of HIV-1 latency by the PKR and p53 pathways. [Internet] [Masters thesis]. McGill University; 2017. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile147057.pdf.

Council of Science Editors:

Labbe R. Post transcriptional control of HIV-1 latency by the PKR and p53 pathways. [Masters Thesis]. McGill University; 2017. Available from: http://digitool.library.mcgill.ca/thesisfile147057.pdf


McGill University

69. Baistrocchi, Shane. Posaconazole-loaded leukocytes as a novel treatment strategy targeting invasive pulmonary aspergillosis.

Degree: MS, Department of Microbiology & Immunology, 2017, McGill University

Invasive pulmonary aspergillosis (IPA) is a necrotizing infection of immunocompromised patients caused by the opportunistic fungus Aspergillus fumigatus and is associated with a high mortality… (more)

Subjects/Keywords: Microbiology and Immunology

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APA (6th Edition):

Baistrocchi, S. (2017). Posaconazole-loaded leukocytes as a novel treatment strategy targeting invasive pulmonary aspergillosis. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile146851.pdf

Chicago Manual of Style (16th Edition):

Baistrocchi, Shane. “Posaconazole-loaded leukocytes as a novel treatment strategy targeting invasive pulmonary aspergillosis.” 2017. Masters Thesis, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile146851.pdf.

MLA Handbook (7th Edition):

Baistrocchi, Shane. “Posaconazole-loaded leukocytes as a novel treatment strategy targeting invasive pulmonary aspergillosis.” 2017. Web. 25 Aug 2019.

Vancouver:

Baistrocchi S. Posaconazole-loaded leukocytes as a novel treatment strategy targeting invasive pulmonary aspergillosis. [Internet] [Masters thesis]. McGill University; 2017. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile146851.pdf.

Council of Science Editors:

Baistrocchi S. Posaconazole-loaded leukocytes as a novel treatment strategy targeting invasive pulmonary aspergillosis. [Masters Thesis]. McGill University; 2017. Available from: http://digitool.library.mcgill.ca/thesisfile146851.pdf


McGill University

70. Coulombe, François. NOD2 and Mycobacteria.

Degree: MS, Department of Microbiology and Immunology, 2010, McGill University

The genus Mycobacterium comprises a variety of highly successful intracellular pathogens. Mycobacterium tuberculosis is the causative agent of tuberculosis (TB) in humans and currently infects… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Coulombe, F. (2010). NOD2 and Mycobacteria. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile86837.pdf

Chicago Manual of Style (16th Edition):

Coulombe, François. “NOD2 and Mycobacteria.” 2010. Masters Thesis, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile86837.pdf.

MLA Handbook (7th Edition):

Coulombe, François. “NOD2 and Mycobacteria.” 2010. Web. 25 Aug 2019.

Vancouver:

Coulombe F. NOD2 and Mycobacteria. [Internet] [Masters thesis]. McGill University; 2010. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile86837.pdf.

Council of Science Editors:

Coulombe F. NOD2 and Mycobacteria. [Masters Thesis]. McGill University; 2010. Available from: http://digitool.library.mcgill.ca/thesisfile86837.pdf


McGill University

71. Drury, Gillian Louise. Expression, regulation and modulation of Fas Ligand during T lymphocyte activation.

Degree: PhD, Department of Physiology, 2010, McGill University

Auto-immune diseases stem from an inability to eliminate immune cells that react to "self" peptides. In particular, when T lymphocytes react to self peptides, or… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Drury, G. L. (2010). Expression, regulation and modulation of Fas Ligand during T lymphocyte activation. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile86656.pdf

Chicago Manual of Style (16th Edition):

Drury, Gillian Louise. “Expression, regulation and modulation of Fas Ligand during T lymphocyte activation.” 2010. Doctoral Dissertation, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile86656.pdf.

MLA Handbook (7th Edition):

Drury, Gillian Louise. “Expression, regulation and modulation of Fas Ligand during T lymphocyte activation.” 2010. Web. 25 Aug 2019.

Vancouver:

Drury GL. Expression, regulation and modulation of Fas Ligand during T lymphocyte activation. [Internet] [Doctoral dissertation]. McGill University; 2010. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile86656.pdf.

Council of Science Editors:

Drury GL. Expression, regulation and modulation of Fas Ligand during T lymphocyte activation. [Doctoral Dissertation]. McGill University; 2010. Available from: http://digitool.library.mcgill.ca/thesisfile86656.pdf


McGill University

72. Kozak, Robert Andrew. The role of RD2 and NOD2 in host-pathogen interactions of the BCG vaccine.

Degree: PhD, Department of Microbiology & Immunology, 2011, McGill University

Bacille Calmette-Guérin (BCG) is the only vaccine currently in use against tuberculosis. Despite its use for nearly a century, the scope of the global tuberculosis… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Kozak, R. A. (2011). The role of RD2 and NOD2 in host-pathogen interactions of the BCG vaccine. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile96670.pdf

Chicago Manual of Style (16th Edition):

Kozak, Robert Andrew. “The role of RD2 and NOD2 in host-pathogen interactions of the BCG vaccine.” 2011. Doctoral Dissertation, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile96670.pdf.

MLA Handbook (7th Edition):

Kozak, Robert Andrew. “The role of RD2 and NOD2 in host-pathogen interactions of the BCG vaccine.” 2011. Web. 25 Aug 2019.

Vancouver:

Kozak RA. The role of RD2 and NOD2 in host-pathogen interactions of the BCG vaccine. [Internet] [Doctoral dissertation]. McGill University; 2011. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile96670.pdf.

Council of Science Editors:

Kozak RA. The role of RD2 and NOD2 in host-pathogen interactions of the BCG vaccine. [Doctoral Dissertation]. McGill University; 2011. Available from: http://digitool.library.mcgill.ca/thesisfile96670.pdf


McGill University

73. Kamya, Nabukenya. Natural killer cells in HIV infected slow progressors.

Degree: PhD, Department of Medicine, 2011, McGill University

Acquired immunodeficiency syndrome AIDS-related illnesses are a leading cause of infectious disease mortality worldwide. The development of a safe and effective prophylactic HIV vaccine is… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Kamya, N. (2011). Natural killer cells in HIV infected slow progressors. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile104518.pdf

Chicago Manual of Style (16th Edition):

Kamya, Nabukenya. “Natural killer cells in HIV infected slow progressors.” 2011. Doctoral Dissertation, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile104518.pdf.

MLA Handbook (7th Edition):

Kamya, Nabukenya. “Natural killer cells in HIV infected slow progressors.” 2011. Web. 25 Aug 2019.

Vancouver:

Kamya N. Natural killer cells in HIV infected slow progressors. [Internet] [Doctoral dissertation]. McGill University; 2011. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile104518.pdf.

Council of Science Editors:

Kamya N. Natural killer cells in HIV infected slow progressors. [Doctoral Dissertation]. McGill University; 2011. Available from: http://digitool.library.mcgill.ca/thesisfile104518.pdf


McGill University

74. Al Heialy, Saba. Bidirectional crosstalk between airway smooth muscle cells and CD4+ T cells: implications in asthma pathology.

Degree: PhD, Department of Medicine, 2013, McGill University

 Asthma is a heterogeneous disease which affects 300 million people worldwide. Characterized by airway hyperresponsivness and inflammation, no curative therapy exists to date. Immune cell… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Al Heialy, S. (2013). Bidirectional crosstalk between airway smooth muscle cells and CD4+ T cells: implications in asthma pathology. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile116925.pdf

Chicago Manual of Style (16th Edition):

Al Heialy, Saba. “Bidirectional crosstalk between airway smooth muscle cells and CD4+ T cells: implications in asthma pathology.” 2013. Doctoral Dissertation, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile116925.pdf.

MLA Handbook (7th Edition):

Al Heialy, Saba. “Bidirectional crosstalk between airway smooth muscle cells and CD4+ T cells: implications in asthma pathology.” 2013. Web. 25 Aug 2019.

Vancouver:

Al Heialy S. Bidirectional crosstalk between airway smooth muscle cells and CD4+ T cells: implications in asthma pathology. [Internet] [Doctoral dissertation]. McGill University; 2013. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile116925.pdf.

Council of Science Editors:

Al Heialy S. Bidirectional crosstalk between airway smooth muscle cells and CD4+ T cells: implications in asthma pathology. [Doctoral Dissertation]. McGill University; 2013. Available from: http://digitool.library.mcgill.ca/thesisfile116925.pdf


McGill University

75. Lafferty, Erin. Functional characterization of a novel ENU-induced mutation in Unc93b1 and its role in single-stranded RNA virus infection.

Degree: PhD, Department of Medicine, 2014, McGill University

 Pattern recognition receptors (PRRs) of the innate immune system are on the front line of host defense against an invading pathogen. These proteins recognize highly… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Lafferty, E. (2014). Functional characterization of a novel ENU-induced mutation in Unc93b1 and its role in single-stranded RNA virus infection. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile127038.pdf

Chicago Manual of Style (16th Edition):

Lafferty, Erin. “Functional characterization of a novel ENU-induced mutation in Unc93b1 and its role in single-stranded RNA virus infection.” 2014. Doctoral Dissertation, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile127038.pdf.

MLA Handbook (7th Edition):

Lafferty, Erin. “Functional characterization of a novel ENU-induced mutation in Unc93b1 and its role in single-stranded RNA virus infection.” 2014. Web. 25 Aug 2019.

Vancouver:

Lafferty E. Functional characterization of a novel ENU-induced mutation in Unc93b1 and its role in single-stranded RNA virus infection. [Internet] [Doctoral dissertation]. McGill University; 2014. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile127038.pdf.

Council of Science Editors:

Lafferty E. Functional characterization of a novel ENU-induced mutation in Unc93b1 and its role in single-stranded RNA virus infection. [Doctoral Dissertation]. McGill University; 2014. Available from: http://digitool.library.mcgill.ca/thesisfile127038.pdf


McGill University

76. Roter, Evan. Beta2-glycoprotein I-specific T cells: antigen specificity and role in the induction of anti-phospholipid syndrome.

Degree: MS, Department of Medicine, 2010, McGill University

Antiphospholipid syndrome (APS) is an autoimmune disorder characterized by the presence of autoantibodies to phospholipid (PL)-binding proteins, such as β2-glycoprotein I (β2GPI), and clinical manifestations… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Roter, E. (2010). Beta2-glycoprotein I-specific T cells: antigen specificity and role in the induction of anti-phospholipid syndrome. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile86847.pdf

Chicago Manual of Style (16th Edition):

Roter, Evan. “Beta2-glycoprotein I-specific T cells: antigen specificity and role in the induction of anti-phospholipid syndrome.” 2010. Masters Thesis, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile86847.pdf.

MLA Handbook (7th Edition):

Roter, Evan. “Beta2-glycoprotein I-specific T cells: antigen specificity and role in the induction of anti-phospholipid syndrome.” 2010. Web. 25 Aug 2019.

Vancouver:

Roter E. Beta2-glycoprotein I-specific T cells: antigen specificity and role in the induction of anti-phospholipid syndrome. [Internet] [Masters thesis]. McGill University; 2010. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile86847.pdf.

Council of Science Editors:

Roter E. Beta2-glycoprotein I-specific T cells: antigen specificity and role in the induction of anti-phospholipid syndrome. [Masters Thesis]. McGill University; 2010. Available from: http://digitool.library.mcgill.ca/thesisfile86847.pdf


McGill University

77. Fortin, Geneviève Rose. Identifying new therapeutic targets for treatment of Crohn's disease: The role of CD47 and L-carnitine in the pathogenesis and treatment of a murine model of intestinal inflammation.

Degree: PhD, Department of Medicine, 2010, McGill University

Crohn's disease (CD) is a chronic, relapsing and remitting immune-mediated inflammatory disease of the gastrointestinal tract. While there is currently no cure for this disease,… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Fortin, G. R. (2010). Identifying new therapeutic targets for treatment of Crohn's disease: The role of CD47 and L-carnitine in the pathogenesis and treatment of a murine model of intestinal inflammation. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile86517.pdf

Chicago Manual of Style (16th Edition):

Fortin, Geneviève Rose. “Identifying new therapeutic targets for treatment of Crohn's disease: The role of CD47 and L-carnitine in the pathogenesis and treatment of a murine model of intestinal inflammation.” 2010. Doctoral Dissertation, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile86517.pdf.

MLA Handbook (7th Edition):

Fortin, Geneviève Rose. “Identifying new therapeutic targets for treatment of Crohn's disease: The role of CD47 and L-carnitine in the pathogenesis and treatment of a murine model of intestinal inflammation.” 2010. Web. 25 Aug 2019.

Vancouver:

Fortin GR. Identifying new therapeutic targets for treatment of Crohn's disease: The role of CD47 and L-carnitine in the pathogenesis and treatment of a murine model of intestinal inflammation. [Internet] [Doctoral dissertation]. McGill University; 2010. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile86517.pdf.

Council of Science Editors:

Fortin GR. Identifying new therapeutic targets for treatment of Crohn's disease: The role of CD47 and L-carnitine in the pathogenesis and treatment of a murine model of intestinal inflammation. [Doctoral Dissertation]. McGill University; 2010. Available from: http://digitool.library.mcgill.ca/thesisfile86517.pdf


McGill University

78. Al-Mot, Sawsan. Molecular signatures as a new classification scheme for chronic rhinosinusitus.

Degree: MS, Department of Surgery, 2013, McGill University

Chronic Rhinosinusitis (CRS), an inflammation of the paranasal sinus cavities, is a common disorder of uncertain etiology that affects the upper airways and paranasal sinuses.… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Al-Mot, S. (2013). Molecular signatures as a new classification scheme for chronic rhinosinusitus. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile114268.pdf

Chicago Manual of Style (16th Edition):

Al-Mot, Sawsan. “Molecular signatures as a new classification scheme for chronic rhinosinusitus.” 2013. Masters Thesis, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile114268.pdf.

MLA Handbook (7th Edition):

Al-Mot, Sawsan. “Molecular signatures as a new classification scheme for chronic rhinosinusitus.” 2013. Web. 25 Aug 2019.

Vancouver:

Al-Mot S. Molecular signatures as a new classification scheme for chronic rhinosinusitus. [Internet] [Masters thesis]. McGill University; 2013. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile114268.pdf.

Council of Science Editors:

Al-Mot S. Molecular signatures as a new classification scheme for chronic rhinosinusitus. [Masters Thesis]. McGill University; 2013. Available from: http://digitool.library.mcgill.ca/thesisfile114268.pdf


McGill University

79. Herscovitch, Kassey. The anti-inflammatory potential of vitamin D in cystic fibrosis.

Degree: MS, Department of Medicine, 2013, McGill University

 Cystic fibrosis (CF) is a fatal genetic disorder characterized by chronic inflammation in the airways, involving an accumulation of neutrophils induced by increased production of… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Herscovitch, K. (2013). The anti-inflammatory potential of vitamin D in cystic fibrosis. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile119599.pdf

Chicago Manual of Style (16th Edition):

Herscovitch, Kassey. “The anti-inflammatory potential of vitamin D in cystic fibrosis.” 2013. Masters Thesis, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile119599.pdf.

MLA Handbook (7th Edition):

Herscovitch, Kassey. “The anti-inflammatory potential of vitamin D in cystic fibrosis.” 2013. Web. 25 Aug 2019.

Vancouver:

Herscovitch K. The anti-inflammatory potential of vitamin D in cystic fibrosis. [Internet] [Masters thesis]. McGill University; 2013. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile119599.pdf.

Council of Science Editors:

Herscovitch K. The anti-inflammatory potential of vitamin D in cystic fibrosis. [Masters Thesis]. McGill University; 2013. Available from: http://digitool.library.mcgill.ca/thesisfile119599.pdf

80. Perez Quintero, Luis Alberto. Activation mechanisms of the SAP family adaptor EAT-2 in natural killer cells.

Degree: PhD, Department of Medicine, 2015, McGill University

Natural killer (NK) cells express several members of the SLAM family of receptors. When stimulated, these receptor molecules can inhibit or activate NK cell function.… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Perez Quintero, L. A. (2015). Activation mechanisms of the SAP family adaptor EAT-2 in natural killer cells. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile130367.pdf

Chicago Manual of Style (16th Edition):

Perez Quintero, Luis Alberto. “Activation mechanisms of the SAP family adaptor EAT-2 in natural killer cells.” 2015. Doctoral Dissertation, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile130367.pdf.

MLA Handbook (7th Edition):

Perez Quintero, Luis Alberto. “Activation mechanisms of the SAP family adaptor EAT-2 in natural killer cells.” 2015. Web. 25 Aug 2019.

Vancouver:

Perez Quintero LA. Activation mechanisms of the SAP family adaptor EAT-2 in natural killer cells. [Internet] [Doctoral dissertation]. McGill University; 2015. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile130367.pdf.

Council of Science Editors:

Perez Quintero LA. Activation mechanisms of the SAP family adaptor EAT-2 in natural killer cells. [Doctoral Dissertation]. McGill University; 2015. Available from: http://digitool.library.mcgill.ca/thesisfile130367.pdf


McGill University

81. Lambert, Caroline. Distinct regulation of recruitment and inflammatory potential of human microglia vs. blood-derived myeloid cells.

Degree: PhD, Department of Physiology, 2010, McGill University

Microglia are myeloid cells that are long-term residents of the central nervous system (CNS). The active lesions in the CNS in multiple sclerosis (MS) show… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Lambert, C. (2010). Distinct regulation of recruitment and inflammatory potential of human microglia vs. blood-derived myeloid cells. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile92238.pdf

Chicago Manual of Style (16th Edition):

Lambert, Caroline. “Distinct regulation of recruitment and inflammatory potential of human microglia vs. blood-derived myeloid cells.” 2010. Doctoral Dissertation, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile92238.pdf.

MLA Handbook (7th Edition):

Lambert, Caroline. “Distinct regulation of recruitment and inflammatory potential of human microglia vs. blood-derived myeloid cells.” 2010. Web. 25 Aug 2019.

Vancouver:

Lambert C. Distinct regulation of recruitment and inflammatory potential of human microglia vs. blood-derived myeloid cells. [Internet] [Doctoral dissertation]. McGill University; 2010. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile92238.pdf.

Council of Science Editors:

Lambert C. Distinct regulation of recruitment and inflammatory potential of human microglia vs. blood-derived myeloid cells. [Doctoral Dissertation]. McGill University; 2010. Available from: http://digitool.library.mcgill.ca/thesisfile92238.pdf


McGill University

82. Camateros, Pierre. The genetic control of airway responsiveness and the effect of resiquimod treatment on allergic asthma.

Degree: PhD, Department of Medicine, 2010, McGill University

Asthma is a heterogeneous airway disease caused by a mixture of genetic and environmental factors which result in improper immune responses to innocuous antigens. Toll-like… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Camateros, P. (2010). The genetic control of airway responsiveness and the effect of resiquimod treatment on allergic asthma. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile92166.pdf

Chicago Manual of Style (16th Edition):

Camateros, Pierre. “The genetic control of airway responsiveness and the effect of resiquimod treatment on allergic asthma.” 2010. Doctoral Dissertation, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile92166.pdf.

MLA Handbook (7th Edition):

Camateros, Pierre. “The genetic control of airway responsiveness and the effect of resiquimod treatment on allergic asthma.” 2010. Web. 25 Aug 2019.

Vancouver:

Camateros P. The genetic control of airway responsiveness and the effect of resiquimod treatment on allergic asthma. [Internet] [Doctoral dissertation]. McGill University; 2010. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile92166.pdf.

Council of Science Editors:

Camateros P. The genetic control of airway responsiveness and the effect of resiquimod treatment on allergic asthma. [Doctoral Dissertation]. McGill University; 2010. Available from: http://digitool.library.mcgill.ca/thesisfile92166.pdf


McGill University

83. Contreras Garcia, Irazú. Modulation of the host innate immune response by «Leishmania» parasites.

Degree: PhD, Department of Microbiology & Immunology, 2010, McGill University

Leishmania parasites have evolved sophisticated mechanisms to subvert macrophage immune responses in order to survive inside the mammalian host. Among these mechanisms are the rapid… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Contreras Garcia, I. (2010). Modulation of the host innate immune response by «Leishmania» parasites. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile95096.pdf

Chicago Manual of Style (16th Edition):

Contreras Garcia, Irazú. “Modulation of the host innate immune response by «Leishmania» parasites.” 2010. Doctoral Dissertation, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile95096.pdf.

MLA Handbook (7th Edition):

Contreras Garcia, Irazú. “Modulation of the host innate immune response by «Leishmania» parasites.” 2010. Web. 25 Aug 2019.

Vancouver:

Contreras Garcia I. Modulation of the host innate immune response by «Leishmania» parasites. [Internet] [Doctoral dissertation]. McGill University; 2010. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile95096.pdf.

Council of Science Editors:

Contreras Garcia I. Modulation of the host innate immune response by «Leishmania» parasites. [Doctoral Dissertation]. McGill University; 2010. Available from: http://digitool.library.mcgill.ca/thesisfile95096.pdf


McGill University

84. Zhang, Zhanguang. Signaling mechanism of DNAM-1, a natural killer cell cytotoxicity receptor.

Degree: MS, Department of Medicine, 2014, McGill University

DNAM-1 (CD226) was discovered in 1996 and first defined as an adhesion molecule expressed by lymphocytes. For the past few years, DNAM-1 has attracted more… (more)

Subjects/Keywords: Health Sciences - Immunology

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APA (6th Edition):

Zhang, Z. (2014). Signaling mechanism of DNAM-1, a natural killer cell cytotoxicity receptor. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile130585.pdf

Chicago Manual of Style (16th Edition):

Zhang, Zhanguang. “Signaling mechanism of DNAM-1, a natural killer cell cytotoxicity receptor.” 2014. Masters Thesis, McGill University. Accessed August 25, 2019. http://digitool.library.mcgill.ca/thesisfile130585.pdf.

MLA Handbook (7th Edition):

Zhang, Zhanguang. “Signaling mechanism of DNAM-1, a natural killer cell cytotoxicity receptor.” 2014. Web. 25 Aug 2019.

Vancouver:

Zhang Z. Signaling mechanism of DNAM-1, a natural killer cell cytotoxicity receptor. [Internet] [Masters thesis]. McGill University; 2014. [cited 2019 Aug 25]. Available from: http://digitool.library.mcgill.ca/thesisfile130585.pdf.

Council of Science Editors:

Zhang Z. Signaling mechanism of DNAM-1, a natural killer cell cytotoxicity receptor. [Masters Thesis]. McGill University; 2014. Available from: http://digitool.library.mcgill.ca/thesisfile130585.pdf


University of Manitoba

85. Rahman, Rahmat Naureen. JNK- associated leucine zipper (JLP) scaffolding protein regulates natural killer cell migrations.

Degree: Immunology, 2019, University of Manitoba

 JNK-associated leucine zipper protein (JLP) is a scaffold protein in the Mitogen-activated protein kinase (MAPK) known to be essential for mediating MAPK functions= such as… (more)

Subjects/Keywords: Immunology; Innate Immunity

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APA (6th Edition):

Rahman, R. N. (2019). JNK- associated leucine zipper (JLP) scaffolding protein regulates natural killer cell migrations. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/33778

Chicago Manual of Style (16th Edition):

Rahman, Rahmat Naureen. “JNK- associated leucine zipper (JLP) scaffolding protein regulates natural killer cell migrations.” 2019. Masters Thesis, University of Manitoba. Accessed August 25, 2019. http://hdl.handle.net/1993/33778.

MLA Handbook (7th Edition):

Rahman, Rahmat Naureen. “JNK- associated leucine zipper (JLP) scaffolding protein regulates natural killer cell migrations.” 2019. Web. 25 Aug 2019.

Vancouver:

Rahman RN. JNK- associated leucine zipper (JLP) scaffolding protein regulates natural killer cell migrations. [Internet] [Masters thesis]. University of Manitoba; 2019. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/1993/33778.

Council of Science Editors:

Rahman RN. JNK- associated leucine zipper (JLP) scaffolding protein regulates natural killer cell migrations. [Masters Thesis]. University of Manitoba; 2019. Available from: http://hdl.handle.net/1993/33778


McMaster University

86. Marcinko, Josip. EVOLUTION OF ALLERGEN RESPONSIVENESS DURING DEVELOPMENT.

Degree: MSMS, 2011, McMaster University

  Background: Early infancy is a critical period during which the interplay between host and environmental factors influences susceptibility to allergic sensitization, a process that… (more)

Subjects/Keywords: allergic asthma; immunology; development; Medical Immunology; Medical Immunology

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APA (6th Edition):

Marcinko, J. (2011). EVOLUTION OF ALLERGEN RESPONSIVENESS DURING DEVELOPMENT. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/10971

Chicago Manual of Style (16th Edition):

Marcinko, Josip. “EVOLUTION OF ALLERGEN RESPONSIVENESS DURING DEVELOPMENT.” 2011. Masters Thesis, McMaster University. Accessed August 25, 2019. http://hdl.handle.net/11375/10971.

MLA Handbook (7th Edition):

Marcinko, Josip. “EVOLUTION OF ALLERGEN RESPONSIVENESS DURING DEVELOPMENT.” 2011. Web. 25 Aug 2019.

Vancouver:

Marcinko J. EVOLUTION OF ALLERGEN RESPONSIVENESS DURING DEVELOPMENT. [Internet] [Masters thesis]. McMaster University; 2011. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/11375/10971.

Council of Science Editors:

Marcinko J. EVOLUTION OF ALLERGEN RESPONSIVENESS DURING DEVELOPMENT. [Masters Thesis]. McMaster University; 2011. Available from: http://hdl.handle.net/11375/10971


University of Toronto

87. Gupta, Sandeep. Fcγ receptor signaling in response to soluble versus immobilized IgG .

Degree: Immunology, 2009, University of Toronto

 Fcγ receptors present on monocytes and macrophages clear pathogens engaged by antibodies through the process of endocytosis. Upon being clustered by immune complexes, Fcγ receptors… (more)

Subjects/Keywords: Health and environmental sciences; immunology

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APA (6th Edition):

Gupta, S. (2009). Fcγ receptor signaling in response to soluble versus immobilized IgG . (Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/80232

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gupta, Sandeep. “Fcγ receptor signaling in response to soluble versus immobilized IgG .” 2009. Thesis, University of Toronto. Accessed August 25, 2019. http://hdl.handle.net/1807/80232.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gupta, Sandeep. “Fcγ receptor signaling in response to soluble versus immobilized IgG .” 2009. Web. 25 Aug 2019.

Vancouver:

Gupta S. Fcγ receptor signaling in response to soluble versus immobilized IgG . [Internet] [Thesis]. University of Toronto; 2009. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/1807/80232.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gupta S. Fcγ receptor signaling in response to soluble versus immobilized IgG . [Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/80232

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

88. Hockley, Deanna L. Co-stimulator contributions in CD8+ T cell differentiation.

Degree: PhD, Department of Medical Microbiology and Immunology, 2012, University of Alberta

 The adaptive immune response against intracellular pathogens is largely mediated by CD8+ T lymphocytes. The clonal expansion and expression of cytolytic and immune stimulatory proteins… (more)

Subjects/Keywords: Immunology; T cells; Co-stimulation

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APA (6th Edition):

Hockley, D. L. (2012). Co-stimulator contributions in CD8+ T cell differentiation. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/f4752h04p

Chicago Manual of Style (16th Edition):

Hockley, Deanna L. “Co-stimulator contributions in CD8+ T cell differentiation.” 2012. Doctoral Dissertation, University of Alberta. Accessed August 25, 2019. https://era.library.ualberta.ca/files/f4752h04p.

MLA Handbook (7th Edition):

Hockley, Deanna L. “Co-stimulator contributions in CD8+ T cell differentiation.” 2012. Web. 25 Aug 2019.

Vancouver:

Hockley DL. Co-stimulator contributions in CD8+ T cell differentiation. [Internet] [Doctoral dissertation]. University of Alberta; 2012. [cited 2019 Aug 25]. Available from: https://era.library.ualberta.ca/files/f4752h04p.

Council of Science Editors:

Hockley DL. Co-stimulator contributions in CD8+ T cell differentiation. [Doctoral Dissertation]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/f4752h04p


University of Alberta

89. Samrat, Subodh Kr. MODULATION OF IMMUNE RESPONSE BY HCV-DERIVED F, CORE AND NS3 PROTEINS.

Degree: PhD, Department of Surgery, 2013, University of Alberta

 The hepatitis C virus leads to chronic infection in the majority of infected individuals; however, in a minority of patients, acute infection is followed by… (more)

Subjects/Keywords: HCV; Immunology; DCs modulation

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APA (6th Edition):

Samrat, S. K. (2013). MODULATION OF IMMUNE RESPONSE BY HCV-DERIVED F, CORE AND NS3 PROTEINS. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/vm40xt06s

Chicago Manual of Style (16th Edition):

Samrat, Subodh Kr. “MODULATION OF IMMUNE RESPONSE BY HCV-DERIVED F, CORE AND NS3 PROTEINS.” 2013. Doctoral Dissertation, University of Alberta. Accessed August 25, 2019. https://era.library.ualberta.ca/files/vm40xt06s.

MLA Handbook (7th Edition):

Samrat, Subodh Kr. “MODULATION OF IMMUNE RESPONSE BY HCV-DERIVED F, CORE AND NS3 PROTEINS.” 2013. Web. 25 Aug 2019.

Vancouver:

Samrat SK. MODULATION OF IMMUNE RESPONSE BY HCV-DERIVED F, CORE AND NS3 PROTEINS. [Internet] [Doctoral dissertation]. University of Alberta; 2013. [cited 2019 Aug 25]. Available from: https://era.library.ualberta.ca/files/vm40xt06s.

Council of Science Editors:

Samrat SK. MODULATION OF IMMUNE RESPONSE BY HCV-DERIVED F, CORE AND NS3 PROTEINS. [Doctoral Dissertation]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/vm40xt06s


University of Saskatchewan

90. Hamilton, Duane Howard. Strategies for exploiting the immune system to achieve prevention and improve therapy of cancer.

Degree: 2007, University of Saskatchewan

 It has steadily become more recognized that even patients with progressively growing tumors are often mounting substantial immune responses against their tumor. The reasons why… (more)

Subjects/Keywords: cancer; Immunology

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APA (6th Edition):

Hamilton, D. H. (2007). Strategies for exploiting the immune system to achieve prevention and improve therapy of cancer. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/etd-06252007-095221

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hamilton, Duane Howard. “Strategies for exploiting the immune system to achieve prevention and improve therapy of cancer.” 2007. Thesis, University of Saskatchewan. Accessed August 25, 2019. http://hdl.handle.net/10388/etd-06252007-095221.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hamilton, Duane Howard. “Strategies for exploiting the immune system to achieve prevention and improve therapy of cancer.” 2007. Web. 25 Aug 2019.

Vancouver:

Hamilton DH. Strategies for exploiting the immune system to achieve prevention and improve therapy of cancer. [Internet] [Thesis]. University of Saskatchewan; 2007. [cited 2019 Aug 25]. Available from: http://hdl.handle.net/10388/etd-06252007-095221.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hamilton DH. Strategies for exploiting the immune system to achieve prevention and improve therapy of cancer. [Thesis]. University of Saskatchewan; 2007. Available from: http://hdl.handle.net/10388/etd-06252007-095221

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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