Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Immunology AND Infectious Disease). Showing records 1 – 30 of 1032 total matches.

[1] [2] [3] [4] [5] … [35]

Search Limiters

Last 2 Years | English Only

Levels

Country

▼ Search Limiters


McMaster University

1. D'Agostino, Michael. Investigating the role of memory alveolar macrophages in early innate immune control of pulmonary tuberculosis.

Degree: MSc, 2019, McMaster University

Mycobacterium tuberculosis (M.tb) is the causative agent of pulmonary tuberculosis (TB). Over 25% of the world’s population is estimated to be infected with tuberculosis, yielding… (more)

Subjects/Keywords: Immunology; Infectious disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

D'Agostino, M. (2019). Investigating the role of memory alveolar macrophages in early innate immune control of pulmonary tuberculosis. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/24846

Chicago Manual of Style (16th Edition):

D'Agostino, Michael. “Investigating the role of memory alveolar macrophages in early innate immune control of pulmonary tuberculosis.” 2019. Masters Thesis, McMaster University. Accessed February 25, 2020. http://hdl.handle.net/11375/24846.

MLA Handbook (7th Edition):

D'Agostino, Michael. “Investigating the role of memory alveolar macrophages in early innate immune control of pulmonary tuberculosis.” 2019. Web. 25 Feb 2020.

Vancouver:

D'Agostino M. Investigating the role of memory alveolar macrophages in early innate immune control of pulmonary tuberculosis. [Internet] [Masters thesis]. McMaster University; 2019. [cited 2020 Feb 25]. Available from: http://hdl.handle.net/11375/24846.

Council of Science Editors:

D'Agostino M. Investigating the role of memory alveolar macrophages in early innate immune control of pulmonary tuberculosis. [Masters Thesis]. McMaster University; 2019. Available from: http://hdl.handle.net/11375/24846


University of Louisville

2. Hutcherson, Justin. Characterization of the innate response to the RagB protein of Porphyromonas gingivalis.

Degree: PhD, 2015, University of Louisville

  More than 64 million people in the US suffer from some form of periodontal disease. Periodontal diseases are bacterial infections of the hard and… (more)

Subjects/Keywords: Immunology and Infectious Disease; Microbiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hutcherson, J. (2015). Characterization of the innate response to the RagB protein of Porphyromonas gingivalis. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2077 ; https://ir.library.louisville.edu/etd/2077

Chicago Manual of Style (16th Edition):

Hutcherson, Justin. “Characterization of the innate response to the RagB protein of Porphyromonas gingivalis.” 2015. Doctoral Dissertation, University of Louisville. Accessed February 25, 2020. 10.18297/etd/2077 ; https://ir.library.louisville.edu/etd/2077.

MLA Handbook (7th Edition):

Hutcherson, Justin. “Characterization of the innate response to the RagB protein of Porphyromonas gingivalis.” 2015. Web. 25 Feb 2020.

Vancouver:

Hutcherson J. Characterization of the innate response to the RagB protein of Porphyromonas gingivalis. [Internet] [Doctoral dissertation]. University of Louisville; 2015. [cited 2020 Feb 25]. Available from: 10.18297/etd/2077 ; https://ir.library.louisville.edu/etd/2077.

Council of Science Editors:

Hutcherson J. Characterization of the innate response to the RagB protein of Porphyromonas gingivalis. [Doctoral Dissertation]. University of Louisville; 2015. Available from: 10.18297/etd/2077 ; https://ir.library.louisville.edu/etd/2077


University of Louisville

3. Camp, Jeremy V., 1980-. Critical insights into the pathogenesis of clinical isolates of pandemic influenza A(H1N1) 2009 virus in mouse and ferret models.

Degree: PhD, 2015, University of Louisville

  Influenza A virus (IAV) is a minus-sense, segmented, single-stranded RNA virus that infects the respiratory tract of humans and can cause severe illness. Novel… (more)

Subjects/Keywords: Immunology and Infectious Disease; Microbiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Camp, Jeremy V., 1. (2015). Critical insights into the pathogenesis of clinical isolates of pandemic influenza A(H1N1) 2009 virus in mouse and ferret models. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2079 ; https://ir.library.louisville.edu/etd/2079

Chicago Manual of Style (16th Edition):

Camp, Jeremy V., 1980-. “Critical insights into the pathogenesis of clinical isolates of pandemic influenza A(H1N1) 2009 virus in mouse and ferret models.” 2015. Doctoral Dissertation, University of Louisville. Accessed February 25, 2020. 10.18297/etd/2079 ; https://ir.library.louisville.edu/etd/2079.

MLA Handbook (7th Edition):

Camp, Jeremy V., 1980-. “Critical insights into the pathogenesis of clinical isolates of pandemic influenza A(H1N1) 2009 virus in mouse and ferret models.” 2015. Web. 25 Feb 2020.

Vancouver:

Camp, Jeremy V. 1. Critical insights into the pathogenesis of clinical isolates of pandemic influenza A(H1N1) 2009 virus in mouse and ferret models. [Internet] [Doctoral dissertation]. University of Louisville; 2015. [cited 2020 Feb 25]. Available from: 10.18297/etd/2079 ; https://ir.library.louisville.edu/etd/2079.

Council of Science Editors:

Camp, Jeremy V. 1. Critical insights into the pathogenesis of clinical isolates of pandemic influenza A(H1N1) 2009 virus in mouse and ferret models. [Doctoral Dissertation]. University of Louisville; 2015. Available from: 10.18297/etd/2079 ; https://ir.library.louisville.edu/etd/2079


University of Louisville

4. Gerlach, Rachael Lask. Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models.

Degree: PhD, 2015, University of Louisville

  Influenza A virus (IAV) subtypes and even genotypes within subtypes can show differences in tropism (host, cell type), magnitude of infection, immune response and… (more)

Subjects/Keywords: Immunology and Infectious Disease; Microbiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gerlach, R. L. (2015). Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2078 ; https://ir.library.louisville.edu/etd/2078

Chicago Manual of Style (16th Edition):

Gerlach, Rachael Lask. “Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models.” 2015. Doctoral Dissertation, University of Louisville. Accessed February 25, 2020. 10.18297/etd/2078 ; https://ir.library.louisville.edu/etd/2078.

MLA Handbook (7th Edition):

Gerlach, Rachael Lask. “Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models.” 2015. Web. 25 Feb 2020.

Vancouver:

Gerlach RL. Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models. [Internet] [Doctoral dissertation]. University of Louisville; 2015. [cited 2020 Feb 25]. Available from: 10.18297/etd/2078 ; https://ir.library.louisville.edu/etd/2078.

Council of Science Editors:

Gerlach RL. Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models. [Doctoral Dissertation]. University of Louisville; 2015. Available from: 10.18297/etd/2078 ; https://ir.library.louisville.edu/etd/2078


University of Louisville

5. Zhu, Yanfang. Regulation of macrophage inflammatory signaling pathways by AMP-activated protein kinase.

Degree: PhD, 2014, University of Louisville

  AMP-activated protein kinase, AMPK, is a conserved serine/threonine kinase with a critical function in the regulation of metabolic pathways in eukaryotic cells. Recently, AMPK… (more)

Subjects/Keywords: Immunology and Infectious Disease; Microbiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhu, Y. (2014). Regulation of macrophage inflammatory signaling pathways by AMP-activated protein kinase. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/1649 ; https://ir.library.louisville.edu/etd/1649

Chicago Manual of Style (16th Edition):

Zhu, Yanfang. “Regulation of macrophage inflammatory signaling pathways by AMP-activated protein kinase.” 2014. Doctoral Dissertation, University of Louisville. Accessed February 25, 2020. 10.18297/etd/1649 ; https://ir.library.louisville.edu/etd/1649.

MLA Handbook (7th Edition):

Zhu, Yanfang. “Regulation of macrophage inflammatory signaling pathways by AMP-activated protein kinase.” 2014. Web. 25 Feb 2020.

Vancouver:

Zhu Y. Regulation of macrophage inflammatory signaling pathways by AMP-activated protein kinase. [Internet] [Doctoral dissertation]. University of Louisville; 2014. [cited 2020 Feb 25]. Available from: 10.18297/etd/1649 ; https://ir.library.louisville.edu/etd/1649.

Council of Science Editors:

Zhu Y. Regulation of macrophage inflammatory signaling pathways by AMP-activated protein kinase. [Doctoral Dissertation]. University of Louisville; 2014. Available from: 10.18297/etd/1649 ; https://ir.library.louisville.edu/etd/1649


Loyola University Chicago

6. Krukowski, Karen. Psychosocial Distress Mediates Immune Dysregulation Through Alterations in Global Epigenetic Patterns and Chromatin Remodeling Proteins.

Degree: PhD, Microbiology and Immunology, 2013, Loyola University Chicago

  Psychosocial distress, characterized by increased perceived stress, anxiety and mood disturbance, is a common response of women to a diagnosis of breast cancer (Northouse,… (more)

Subjects/Keywords: Immunology and Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Krukowski, K. (2013). Psychosocial Distress Mediates Immune Dysregulation Through Alterations in Global Epigenetic Patterns and Chromatin Remodeling Proteins. (Doctoral Dissertation). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_diss/525

Chicago Manual of Style (16th Edition):

Krukowski, Karen. “Psychosocial Distress Mediates Immune Dysregulation Through Alterations in Global Epigenetic Patterns and Chromatin Remodeling Proteins.” 2013. Doctoral Dissertation, Loyola University Chicago. Accessed February 25, 2020. https://ecommons.luc.edu/luc_diss/525.

MLA Handbook (7th Edition):

Krukowski, Karen. “Psychosocial Distress Mediates Immune Dysregulation Through Alterations in Global Epigenetic Patterns and Chromatin Remodeling Proteins.” 2013. Web. 25 Feb 2020.

Vancouver:

Krukowski K. Psychosocial Distress Mediates Immune Dysregulation Through Alterations in Global Epigenetic Patterns and Chromatin Remodeling Proteins. [Internet] [Doctoral dissertation]. Loyola University Chicago; 2013. [cited 2020 Feb 25]. Available from: https://ecommons.luc.edu/luc_diss/525.

Council of Science Editors:

Krukowski K. Psychosocial Distress Mediates Immune Dysregulation Through Alterations in Global Epigenetic Patterns and Chromatin Remodeling Proteins. [Doctoral Dissertation]. Loyola University Chicago; 2013. Available from: https://ecommons.luc.edu/luc_diss/525


Loyola University Chicago

7. Nomellini, Vanessa. Aging Effects on Acute Lung Inflammation After Burn Injury.

Degree: PhD, Molecular and Cellular Biochemistry Program, 2010, Loyola University Chicago

  The risk of complications and death after a moderate sized burn injury is significantly higher in persons over the age of 65, while almost… (more)

Subjects/Keywords: Immunology and Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nomellini, V. (2010). Aging Effects on Acute Lung Inflammation After Burn Injury. (Doctoral Dissertation). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_diss/144

Chicago Manual of Style (16th Edition):

Nomellini, Vanessa. “Aging Effects on Acute Lung Inflammation After Burn Injury.” 2010. Doctoral Dissertation, Loyola University Chicago. Accessed February 25, 2020. https://ecommons.luc.edu/luc_diss/144.

MLA Handbook (7th Edition):

Nomellini, Vanessa. “Aging Effects on Acute Lung Inflammation After Burn Injury.” 2010. Web. 25 Feb 2020.

Vancouver:

Nomellini V. Aging Effects on Acute Lung Inflammation After Burn Injury. [Internet] [Doctoral dissertation]. Loyola University Chicago; 2010. [cited 2020 Feb 25]. Available from: https://ecommons.luc.edu/luc_diss/144.

Council of Science Editors:

Nomellini V. Aging Effects on Acute Lung Inflammation After Burn Injury. [Doctoral Dissertation]. Loyola University Chicago; 2010. Available from: https://ecommons.luc.edu/luc_diss/144

8. Hendrix, Emily K. Characterizing the early stages of a novel host shift using host fitness and metabolomics.

Degree: MS, Biology, 2018, Eastern Washington University

  The factors that contribute to successful colonization of a novel host species by a pathogen remain unclear. One likely factor determining host shift success… (more)

Subjects/Keywords: Biology; Immunology of Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hendrix, E. K. (2018). Characterizing the early stages of a novel host shift using host fitness and metabolomics. (Thesis). Eastern Washington University. Retrieved from https://dc.ewu.edu/theses/500

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hendrix, Emily K. “Characterizing the early stages of a novel host shift using host fitness and metabolomics.” 2018. Thesis, Eastern Washington University. Accessed February 25, 2020. https://dc.ewu.edu/theses/500.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hendrix, Emily K. “Characterizing the early stages of a novel host shift using host fitness and metabolomics.” 2018. Web. 25 Feb 2020.

Vancouver:

Hendrix EK. Characterizing the early stages of a novel host shift using host fitness and metabolomics. [Internet] [Thesis]. Eastern Washington University; 2018. [cited 2020 Feb 25]. Available from: https://dc.ewu.edu/theses/500.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hendrix EK. Characterizing the early stages of a novel host shift using host fitness and metabolomics. [Thesis]. Eastern Washington University; 2018. Available from: https://dc.ewu.edu/theses/500

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

9. Bush, Kristin. Role for Histone Deacetylases in Glucocorticoid Receptor Mediated Transpression of Natural Killer Cell Activity.

Degree: MS, Microbiology and Immunology, 2011, Loyola University Chicago

  During periods of psychosocial distress glucocorticoids (GCs) are known to reduce the lytic activity of natural killer cells (NKCA). Glucocorticoid treatment also reduces acetylation… (more)

Subjects/Keywords: Immunology and Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bush, K. (2011). Role for Histone Deacetylases in Glucocorticoid Receptor Mediated Transpression of Natural Killer Cell Activity. (Thesis). Loyola University Chicago. Retrieved from http://ecommons.luc.edu/luc_theses/567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bush, Kristin. “Role for Histone Deacetylases in Glucocorticoid Receptor Mediated Transpression of Natural Killer Cell Activity.” 2011. Thesis, Loyola University Chicago. Accessed February 25, 2020. http://ecommons.luc.edu/luc_theses/567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bush, Kristin. “Role for Histone Deacetylases in Glucocorticoid Receptor Mediated Transpression of Natural Killer Cell Activity.” 2011. Web. 25 Feb 2020.

Vancouver:

Bush K. Role for Histone Deacetylases in Glucocorticoid Receptor Mediated Transpression of Natural Killer Cell Activity. [Internet] [Thesis]. Loyola University Chicago; 2011. [cited 2020 Feb 25]. Available from: http://ecommons.luc.edu/luc_theses/567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bush K. Role for Histone Deacetylases in Glucocorticoid Receptor Mediated Transpression of Natural Killer Cell Activity. [Thesis]. Loyola University Chicago; 2011. Available from: http://ecommons.luc.edu/luc_theses/567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

10. Hlavin, Sara. Binge Ethanol Leads to Decreased Macrophage Accumulation in Infected Cutaneous Wounds.

Degree: MS, Microbiology and Immunology, 2012, Loyola University Chicago

  Trauma patients who consumed alcohol prior to sustaining injuries have higher rates of morbidity and mortality than those with comparable injuries who did not… (more)

Subjects/Keywords: Immunology and Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hlavin, S. (2012). Binge Ethanol Leads to Decreased Macrophage Accumulation in Infected Cutaneous Wounds. (Thesis). Loyola University Chicago. Retrieved from http://ecommons.luc.edu/luc_theses/720

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hlavin, Sara. “Binge Ethanol Leads to Decreased Macrophage Accumulation in Infected Cutaneous Wounds.” 2012. Thesis, Loyola University Chicago. Accessed February 25, 2020. http://ecommons.luc.edu/luc_theses/720.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hlavin, Sara. “Binge Ethanol Leads to Decreased Macrophage Accumulation in Infected Cutaneous Wounds.” 2012. Web. 25 Feb 2020.

Vancouver:

Hlavin S. Binge Ethanol Leads to Decreased Macrophage Accumulation in Infected Cutaneous Wounds. [Internet] [Thesis]. Loyola University Chicago; 2012. [cited 2020 Feb 25]. Available from: http://ecommons.luc.edu/luc_theses/720.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hlavin S. Binge Ethanol Leads to Decreased Macrophage Accumulation in Infected Cutaneous Wounds. [Thesis]. Loyola University Chicago; 2012. Available from: http://ecommons.luc.edu/luc_theses/720

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

11. Foley, Kendra. Tcr Modifications to Enhance Expression, Chain Pairing, and Antigen Recognition for Adoptive T Cell Transfer.

Degree: MS, Microbiology and Immunology, 2015, Loyola University Chicago

  T cell receptor (TCR) gene modified T cells for adoptive T cell transfer therapy have been shown to have clinical success in treating melanoma… (more)

Subjects/Keywords: Immunology and Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Foley, K. (2015). Tcr Modifications to Enhance Expression, Chain Pairing, and Antigen Recognition for Adoptive T Cell Transfer. (Thesis). Loyola University Chicago. Retrieved from http://ecommons.luc.edu/luc_theses/2784

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Foley, Kendra. “Tcr Modifications to Enhance Expression, Chain Pairing, and Antigen Recognition for Adoptive T Cell Transfer.” 2015. Thesis, Loyola University Chicago. Accessed February 25, 2020. http://ecommons.luc.edu/luc_theses/2784.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Foley, Kendra. “Tcr Modifications to Enhance Expression, Chain Pairing, and Antigen Recognition for Adoptive T Cell Transfer.” 2015. Web. 25 Feb 2020.

Vancouver:

Foley K. Tcr Modifications to Enhance Expression, Chain Pairing, and Antigen Recognition for Adoptive T Cell Transfer. [Internet] [Thesis]. Loyola University Chicago; 2015. [cited 2020 Feb 25]. Available from: http://ecommons.luc.edu/luc_theses/2784.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Foley K. Tcr Modifications to Enhance Expression, Chain Pairing, and Antigen Recognition for Adoptive T Cell Transfer. [Thesis]. Loyola University Chicago; 2015. Available from: http://ecommons.luc.edu/luc_theses/2784

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

12. Cannon, Abigail Rhea. Role of Heat Shock Proteins in Maintenance of the Gut Barrier Following Burn Injury.

Degree: MS, Microbiology and Immunology, 2015, Loyola University Chicago

  Introduction: Burn injury remains a prominent clinical problem. Patients suffering from burns often succumb to secondary infectious complications leading to sepsis and widespread tissue… (more)

Subjects/Keywords: Immunology and Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cannon, A. R. (2015). Role of Heat Shock Proteins in Maintenance of the Gut Barrier Following Burn Injury. (Thesis). Loyola University Chicago. Retrieved from http://ecommons.luc.edu/luc_theses/3127

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cannon, Abigail Rhea. “Role of Heat Shock Proteins in Maintenance of the Gut Barrier Following Burn Injury.” 2015. Thesis, Loyola University Chicago. Accessed February 25, 2020. http://ecommons.luc.edu/luc_theses/3127.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cannon, Abigail Rhea. “Role of Heat Shock Proteins in Maintenance of the Gut Barrier Following Burn Injury.” 2015. Web. 25 Feb 2020.

Vancouver:

Cannon AR. Role of Heat Shock Proteins in Maintenance of the Gut Barrier Following Burn Injury. [Internet] [Thesis]. Loyola University Chicago; 2015. [cited 2020 Feb 25]. Available from: http://ecommons.luc.edu/luc_theses/3127.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cannon AR. Role of Heat Shock Proteins in Maintenance of the Gut Barrier Following Burn Injury. [Thesis]. Loyola University Chicago; 2015. Available from: http://ecommons.luc.edu/luc_theses/3127

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Uprety, Tirth. Role of Bovine Ileal Sub-epithelial Myofibroblasts and Epithelial Cells in Innate Immunity.

Degree: MS, Biology and Microbiology, 2018, South Dakota State University

  Gastro-intestinal (GI) tract harbors largest number of microbiota as well as the largest number of immune cells for a given tissue. The host needs… (more)

Subjects/Keywords: Immunology and Infectious Disease; Microbiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Uprety, T. (2018). Role of Bovine Ileal Sub-epithelial Myofibroblasts and Epithelial Cells in Innate Immunity. (Masters Thesis). South Dakota State University. Retrieved from https://openprairie.sdstate.edu/etd/2942

Chicago Manual of Style (16th Edition):

Uprety, Tirth. “Role of Bovine Ileal Sub-epithelial Myofibroblasts and Epithelial Cells in Innate Immunity.” 2018. Masters Thesis, South Dakota State University. Accessed February 25, 2020. https://openprairie.sdstate.edu/etd/2942.

MLA Handbook (7th Edition):

Uprety, Tirth. “Role of Bovine Ileal Sub-epithelial Myofibroblasts and Epithelial Cells in Innate Immunity.” 2018. Web. 25 Feb 2020.

Vancouver:

Uprety T. Role of Bovine Ileal Sub-epithelial Myofibroblasts and Epithelial Cells in Innate Immunity. [Internet] [Masters thesis]. South Dakota State University; 2018. [cited 2020 Feb 25]. Available from: https://openprairie.sdstate.edu/etd/2942.

Council of Science Editors:

Uprety T. Role of Bovine Ileal Sub-epithelial Myofibroblasts and Epithelial Cells in Innate Immunity. [Masters Thesis]. South Dakota State University; 2018. Available from: https://openprairie.sdstate.edu/etd/2942


Loyola University Chicago

14. Keller, Taylor. Cell Intrinsic Characteristics of Cord Blood Naive CD4 T Cells.

Degree: MS, Microbiology and Immunology, 2018, Loyola University Chicago

  The neonatal immune system is functionally distinct from the adult immune system. Neonatal immune responses are less reactive than their adult counterparts, and as… (more)

Subjects/Keywords: Immunology and Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Keller, T. (2018). Cell Intrinsic Characteristics of Cord Blood Naive CD4 T Cells. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/3682

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Keller, Taylor. “Cell Intrinsic Characteristics of Cord Blood Naive CD4 T Cells.” 2018. Thesis, Loyola University Chicago. Accessed February 25, 2020. https://ecommons.luc.edu/luc_theses/3682.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Keller, Taylor. “Cell Intrinsic Characteristics of Cord Blood Naive CD4 T Cells.” 2018. Web. 25 Feb 2020.

Vancouver:

Keller T. Cell Intrinsic Characteristics of Cord Blood Naive CD4 T Cells. [Internet] [Thesis]. Loyola University Chicago; 2018. [cited 2020 Feb 25]. Available from: https://ecommons.luc.edu/luc_theses/3682.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Keller T. Cell Intrinsic Characteristics of Cord Blood Naive CD4 T Cells. [Thesis]. Loyola University Chicago; 2018. Available from: https://ecommons.luc.edu/luc_theses/3682

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

15. Armstrong, Cortney Linn. Characterization of Filifactor alocis and its immune evasion strategies employed against human neutrophils.

Degree: PhD, 2017, University of Louisville

  Periodontal disease is among the most common of inflammatory conditions and is caused by bacterial and host derived factors. The presence of bacteria drives… (more)

Subjects/Keywords: Immunology of Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Armstrong, C. L. (2017). Characterization of Filifactor alocis and its immune evasion strategies employed against human neutrophils. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2769 ; https://ir.library.louisville.edu/etd/2769

Chicago Manual of Style (16th Edition):

Armstrong, Cortney Linn. “Characterization of Filifactor alocis and its immune evasion strategies employed against human neutrophils.” 2017. Doctoral Dissertation, University of Louisville. Accessed February 25, 2020. 10.18297/etd/2769 ; https://ir.library.louisville.edu/etd/2769.

MLA Handbook (7th Edition):

Armstrong, Cortney Linn. “Characterization of Filifactor alocis and its immune evasion strategies employed against human neutrophils.” 2017. Web. 25 Feb 2020.

Vancouver:

Armstrong CL. Characterization of Filifactor alocis and its immune evasion strategies employed against human neutrophils. [Internet] [Doctoral dissertation]. University of Louisville; 2017. [cited 2020 Feb 25]. Available from: 10.18297/etd/2769 ; https://ir.library.louisville.edu/etd/2769.

Council of Science Editors:

Armstrong CL. Characterization of Filifactor alocis and its immune evasion strategies employed against human neutrophils. [Doctoral Dissertation]. University of Louisville; 2017. Available from: 10.18297/etd/2769 ; https://ir.library.louisville.edu/etd/2769


Wayne State University

16. Gibson, Heather. Ctla-4 transcriptional activation: regulation of induced expression.

Degree: PhD, Immunology and Microbiology, 2011, Wayne State University

  Cytotoxic T-lymphocyte antigen 4 (CTLA-4) is a T cell surface protein that is homologous to CD28 and binds to the B7 family of ligands.… (more)

Subjects/Keywords: Immunology and Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gibson, H. (2011). Ctla-4 transcriptional activation: regulation of induced expression. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/237

Chicago Manual of Style (16th Edition):

Gibson, Heather. “Ctla-4 transcriptional activation: regulation of induced expression.” 2011. Doctoral Dissertation, Wayne State University. Accessed February 25, 2020. https://digitalcommons.wayne.edu/oa_dissertations/237.

MLA Handbook (7th Edition):

Gibson, Heather. “Ctla-4 transcriptional activation: regulation of induced expression.” 2011. Web. 25 Feb 2020.

Vancouver:

Gibson H. Ctla-4 transcriptional activation: regulation of induced expression. [Internet] [Doctoral dissertation]. Wayne State University; 2011. [cited 2020 Feb 25]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/237.

Council of Science Editors:

Gibson H. Ctla-4 transcriptional activation: regulation of induced expression. [Doctoral Dissertation]. Wayne State University; 2011. Available from: https://digitalcommons.wayne.edu/oa_dissertations/237


Wayne State University

17. Rao, Pushpa Durgesh. Alterations In Ocular Surface System During The Pathogenesis Of Herpes Stromal Keratitis.

Degree: PhD, Anatomy and Cell Biology, 2019, Wayne State University

  Herpes stromal keratitis (HSK) is a chronic immuno-inflammatory ocular disease caused by Herpes simplex virus-1 (HSV-1) infection in the cornea. HSK is characterized by… (more)

Subjects/Keywords: Immunology and Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rao, P. D. (2019). Alterations In Ocular Surface System During The Pathogenesis Of Herpes Stromal Keratitis. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/2181

Chicago Manual of Style (16th Edition):

Rao, Pushpa Durgesh. “Alterations In Ocular Surface System During The Pathogenesis Of Herpes Stromal Keratitis.” 2019. Doctoral Dissertation, Wayne State University. Accessed February 25, 2020. https://digitalcommons.wayne.edu/oa_dissertations/2181.

MLA Handbook (7th Edition):

Rao, Pushpa Durgesh. “Alterations In Ocular Surface System During The Pathogenesis Of Herpes Stromal Keratitis.” 2019. Web. 25 Feb 2020.

Vancouver:

Rao PD. Alterations In Ocular Surface System During The Pathogenesis Of Herpes Stromal Keratitis. [Internet] [Doctoral dissertation]. Wayne State University; 2019. [cited 2020 Feb 25]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/2181.

Council of Science Editors:

Rao PD. Alterations In Ocular Surface System During The Pathogenesis Of Herpes Stromal Keratitis. [Doctoral Dissertation]. Wayne State University; 2019. Available from: https://digitalcommons.wayne.edu/oa_dissertations/2181


University of Iowa

18. Lifton, Samuel Robert. Retroviral mutagenesis in a newly developed myc transgenic mouse model of human B cell and plasma cell neoplasia.

Degree: MS, Interdisciplinary Studies in Immunology, 2013, University of Iowa

  Three potential driver genes were identified by use of retroviral mutagenesis in the newly developed iMyc model of B cell malignancy. To do so,… (more)

Subjects/Keywords: Immunology of Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lifton, S. R. (2013). Retroviral mutagenesis in a newly developed myc transgenic mouse model of human B cell and plasma cell neoplasia. (Masters Thesis). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/1479

Chicago Manual of Style (16th Edition):

Lifton, Samuel Robert. “Retroviral mutagenesis in a newly developed myc transgenic mouse model of human B cell and plasma cell neoplasia.” 2013. Masters Thesis, University of Iowa. Accessed February 25, 2020. https://ir.uiowa.edu/etd/1479.

MLA Handbook (7th Edition):

Lifton, Samuel Robert. “Retroviral mutagenesis in a newly developed myc transgenic mouse model of human B cell and plasma cell neoplasia.” 2013. Web. 25 Feb 2020.

Vancouver:

Lifton SR. Retroviral mutagenesis in a newly developed myc transgenic mouse model of human B cell and plasma cell neoplasia. [Internet] [Masters thesis]. University of Iowa; 2013. [cited 2020 Feb 25]. Available from: https://ir.uiowa.edu/etd/1479.

Council of Science Editors:

Lifton SR. Retroviral mutagenesis in a newly developed myc transgenic mouse model of human B cell and plasma cell neoplasia. [Masters Thesis]. University of Iowa; 2013. Available from: https://ir.uiowa.edu/etd/1479


University of Iowa

19. Parlet, Corey Patrick. Mechanisms by which chronic ethanol consumption impairs cutaneous immunity.

Degree: PhD, Immunology, 2014, University of Iowa

  The immunosuppressive effects of chronic alcohol abuse are profound, wide-ranging and readily apparent at the body's barriers. In the skin, alcoholism is associated with… (more)

Subjects/Keywords: Immunology of Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Parlet, C. P. (2014). Mechanisms by which chronic ethanol consumption impairs cutaneous immunity. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/4712

Chicago Manual of Style (16th Edition):

Parlet, Corey Patrick. “Mechanisms by which chronic ethanol consumption impairs cutaneous immunity.” 2014. Doctoral Dissertation, University of Iowa. Accessed February 25, 2020. https://ir.uiowa.edu/etd/4712.

MLA Handbook (7th Edition):

Parlet, Corey Patrick. “Mechanisms by which chronic ethanol consumption impairs cutaneous immunity.” 2014. Web. 25 Feb 2020.

Vancouver:

Parlet CP. Mechanisms by which chronic ethanol consumption impairs cutaneous immunity. [Internet] [Doctoral dissertation]. University of Iowa; 2014. [cited 2020 Feb 25]. Available from: https://ir.uiowa.edu/etd/4712.

Council of Science Editors:

Parlet CP. Mechanisms by which chronic ethanol consumption impairs cutaneous immunity. [Doctoral Dissertation]. University of Iowa; 2014. Available from: https://ir.uiowa.edu/etd/4712


University of Iowa

20. Vacaflores Salinas, Aldo Fabian. New roles for an old cytokine : characterizing how exposure to Il-12 alters human CD4 And CD8 T cell responses.

Degree: PhD, Immunology, 2016, University of Iowa

  CD4 and CD8 T cells are constantly exposed to inflammatory signals that influence diverse functional outcomes during infections and certain autoimmune disorders. One of… (more)

Subjects/Keywords: Immunology of Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vacaflores Salinas, A. F. (2016). New roles for an old cytokine : characterizing how exposure to Il-12 alters human CD4 And CD8 T cell responses. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/2157

Chicago Manual of Style (16th Edition):

Vacaflores Salinas, Aldo Fabian. “New roles for an old cytokine : characterizing how exposure to Il-12 alters human CD4 And CD8 T cell responses.” 2016. Doctoral Dissertation, University of Iowa. Accessed February 25, 2020. https://ir.uiowa.edu/etd/2157.

MLA Handbook (7th Edition):

Vacaflores Salinas, Aldo Fabian. “New roles for an old cytokine : characterizing how exposure to Il-12 alters human CD4 And CD8 T cell responses.” 2016. Web. 25 Feb 2020.

Vancouver:

Vacaflores Salinas AF. New roles for an old cytokine : characterizing how exposure to Il-12 alters human CD4 And CD8 T cell responses. [Internet] [Doctoral dissertation]. University of Iowa; 2016. [cited 2020 Feb 25]. Available from: https://ir.uiowa.edu/etd/2157.

Council of Science Editors:

Vacaflores Salinas AF. New roles for an old cytokine : characterizing how exposure to Il-12 alters human CD4 And CD8 T cell responses. [Doctoral Dissertation]. University of Iowa; 2016. Available from: https://ir.uiowa.edu/etd/2157


University of Iowa

21. Rosean, Timothy Robert. The tumor microenvironment is critical for the development of plasma cell neoplasia in mice.

Degree: PhD, Immunology, 2014, University of Iowa

  Plasma cell neoplasms (PCN), including multiple myeloma, are tumors of terminally differentiated B cells. Despite a significant research effort, and numerous advances in therapy,… (more)

Subjects/Keywords: publicabstract; Immunology of Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rosean, T. R. (2014). The tumor microenvironment is critical for the development of plasma cell neoplasia in mice. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/1497

Chicago Manual of Style (16th Edition):

Rosean, Timothy Robert. “The tumor microenvironment is critical for the development of plasma cell neoplasia in mice.” 2014. Doctoral Dissertation, University of Iowa. Accessed February 25, 2020. https://ir.uiowa.edu/etd/1497.

MLA Handbook (7th Edition):

Rosean, Timothy Robert. “The tumor microenvironment is critical for the development of plasma cell neoplasia in mice.” 2014. Web. 25 Feb 2020.

Vancouver:

Rosean TR. The tumor microenvironment is critical for the development of plasma cell neoplasia in mice. [Internet] [Doctoral dissertation]. University of Iowa; 2014. [cited 2020 Feb 25]. Available from: https://ir.uiowa.edu/etd/1497.

Council of Science Editors:

Rosean TR. The tumor microenvironment is critical for the development of plasma cell neoplasia in mice. [Doctoral Dissertation]. University of Iowa; 2014. Available from: https://ir.uiowa.edu/etd/1497


University of Iowa

22. Scorza, Breanna M. Interaction of human keratinocytes with Leishmania spp.: a comparative study of Leishmania infantum and Leishmania major.

Degree: PhD, Immunology, 2017, University of Iowa

  Leishmaniasis refers to the group of diseases caused by pathogenic protozoan parasites of the genus Leishmania. Nearly all human Leishmania spp. infections are initiated… (more)

Subjects/Keywords: Immunology of Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Scorza, B. M. (2017). Interaction of human keratinocytes with Leishmania spp.: a comparative study of Leishmania infantum and Leishmania major. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/5847

Chicago Manual of Style (16th Edition):

Scorza, Breanna M. “Interaction of human keratinocytes with Leishmania spp.: a comparative study of Leishmania infantum and Leishmania major.” 2017. Doctoral Dissertation, University of Iowa. Accessed February 25, 2020. https://ir.uiowa.edu/etd/5847.

MLA Handbook (7th Edition):

Scorza, Breanna M. “Interaction of human keratinocytes with Leishmania spp.: a comparative study of Leishmania infantum and Leishmania major.” 2017. Web. 25 Feb 2020.

Vancouver:

Scorza BM. Interaction of human keratinocytes with Leishmania spp.: a comparative study of Leishmania infantum and Leishmania major. [Internet] [Doctoral dissertation]. University of Iowa; 2017. [cited 2020 Feb 25]. Available from: https://ir.uiowa.edu/etd/5847.

Council of Science Editors:

Scorza BM. Interaction of human keratinocytes with Leishmania spp.: a comparative study of Leishmania infantum and Leishmania major. [Doctoral Dissertation]. University of Iowa; 2017. Available from: https://ir.uiowa.edu/etd/5847


University of Iowa

23. Gorman, Jacob. Regulation of T cell responses by the surface receptor Tim-3.

Degree: PhD, Immunology, 2014, University of Iowa

  Tim-3 (for T cell immunoglobulin and mucin domain 3) is a surface molecule expressed throughout the immune system that appears to mediate both stimulatory… (more)

Subjects/Keywords: Immunology of Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gorman, J. (2014). Regulation of T cell responses by the surface receptor Tim-3. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/1326

Chicago Manual of Style (16th Edition):

Gorman, Jacob. “Regulation of T cell responses by the surface receptor Tim-3.” 2014. Doctoral Dissertation, University of Iowa. Accessed February 25, 2020. https://ir.uiowa.edu/etd/1326.

MLA Handbook (7th Edition):

Gorman, Jacob. “Regulation of T cell responses by the surface receptor Tim-3.” 2014. Web. 25 Feb 2020.

Vancouver:

Gorman J. Regulation of T cell responses by the surface receptor Tim-3. [Internet] [Doctoral dissertation]. University of Iowa; 2014. [cited 2020 Feb 25]. Available from: https://ir.uiowa.edu/etd/1326.

Council of Science Editors:

Gorman J. Regulation of T cell responses by the surface receptor Tim-3. [Doctoral Dissertation]. University of Iowa; 2014. Available from: https://ir.uiowa.edu/etd/1326


University of Iowa

24. Janowski, Ann M. The protective roles of NLRs during infection and tumor progression.

Degree: PhD, Immunology, 2016, University of Iowa

  The immune system has evolved to fight off numerous pathogens. The first line of defense against these pathogens are innate immune cells. Innate immune… (more)

Subjects/Keywords: Immunology of Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Janowski, A. M. (2016). The protective roles of NLRs during infection and tumor progression. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/5518

Chicago Manual of Style (16th Edition):

Janowski, Ann M. “The protective roles of NLRs during infection and tumor progression.” 2016. Doctoral Dissertation, University of Iowa. Accessed February 25, 2020. https://ir.uiowa.edu/etd/5518.

MLA Handbook (7th Edition):

Janowski, Ann M. “The protective roles of NLRs during infection and tumor progression.” 2016. Web. 25 Feb 2020.

Vancouver:

Janowski AM. The protective roles of NLRs during infection and tumor progression. [Internet] [Doctoral dissertation]. University of Iowa; 2016. [cited 2020 Feb 25]. Available from: https://ir.uiowa.edu/etd/5518.

Council of Science Editors:

Janowski AM. The protective roles of NLRs during infection and tumor progression. [Doctoral Dissertation]. University of Iowa; 2016. Available from: https://ir.uiowa.edu/etd/5518

25. Liu, Runxia. Genetic Interference and Receptor Biology of Neglected Influenza Viruses.

Degree: PhD, Biology and Microbiology, 2017, South Dakota State University

  Influenza B virus (IBV) is an important pathogen that infects humans and causes seasonal influenza epidemics. By using next-generation sequencing (NGS) approach, we analyzed… (more)

Subjects/Keywords: Immunology and Infectious Disease; Microbiology; Virology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Liu, R. (2017). Genetic Interference and Receptor Biology of Neglected Influenza Viruses. (Doctoral Dissertation). South Dakota State University. Retrieved from https://openprairie.sdstate.edu/etd/2157

Chicago Manual of Style (16th Edition):

Liu, Runxia. “Genetic Interference and Receptor Biology of Neglected Influenza Viruses.” 2017. Doctoral Dissertation, South Dakota State University. Accessed February 25, 2020. https://openprairie.sdstate.edu/etd/2157.

MLA Handbook (7th Edition):

Liu, Runxia. “Genetic Interference and Receptor Biology of Neglected Influenza Viruses.” 2017. Web. 25 Feb 2020.

Vancouver:

Liu R. Genetic Interference and Receptor Biology of Neglected Influenza Viruses. [Internet] [Doctoral dissertation]. South Dakota State University; 2017. [cited 2020 Feb 25]. Available from: https://openprairie.sdstate.edu/etd/2157.

Council of Science Editors:

Liu R. Genetic Interference and Receptor Biology of Neglected Influenza Viruses. [Doctoral Dissertation]. South Dakota State University; 2017. Available from: https://openprairie.sdstate.edu/etd/2157


Loyola University Chicago

26. Zook, Erin Christine. Promoting Thymopoiesis with Age: Potential Role of the Transcription Factor Foxn1.

Degree: PhD, Cell Biology, Neurobiology and Anatomy, 2012, Loyola University Chicago

  It is known that the elderly are more susceptible to illnesses and infections and respond poorly to immunization. A contributing factor to a decrease… (more)

Subjects/Keywords: Aging; Foxn1; Thymus; Immunology and Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zook, E. C. (2012). Promoting Thymopoiesis with Age: Potential Role of the Transcription Factor Foxn1. (Doctoral Dissertation). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_diss/317

Chicago Manual of Style (16th Edition):

Zook, Erin Christine. “Promoting Thymopoiesis with Age: Potential Role of the Transcription Factor Foxn1.” 2012. Doctoral Dissertation, Loyola University Chicago. Accessed February 25, 2020. https://ecommons.luc.edu/luc_diss/317.

MLA Handbook (7th Edition):

Zook, Erin Christine. “Promoting Thymopoiesis with Age: Potential Role of the Transcription Factor Foxn1.” 2012. Web. 25 Feb 2020.

Vancouver:

Zook EC. Promoting Thymopoiesis with Age: Potential Role of the Transcription Factor Foxn1. [Internet] [Doctoral dissertation]. Loyola University Chicago; 2012. [cited 2020 Feb 25]. Available from: https://ecommons.luc.edu/luc_diss/317.

Council of Science Editors:

Zook EC. Promoting Thymopoiesis with Age: Potential Role of the Transcription Factor Foxn1. [Doctoral Dissertation]. Loyola University Chicago; 2012. Available from: https://ecommons.luc.edu/luc_diss/317

27. Tranchemontagne, Zachary Ronald. Community-Acquired Methicillin-Resistant Staphylococcus Aureus (CA-MRSA) USA300 Perturbs Acquisition Of Lysosomal Hydrolases And Requires Phagosomal Acidification For Survival In A Human Macrophage Cell Line.

Degree: MSin Biological Sciences, Biological Science, 2015, University of New England

  Community-acquired Methicillin-resistant Staphylococcus aureus (CA-MRSA) strain USA300 is a major cause of invasive drug-resistant skin and soft tissue infections in humans. Although S. aureus… (more)

Subjects/Keywords: Biology; Immunology and Infectious Disease; Microbiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tranchemontagne, Z. R. (2015). Community-Acquired Methicillin-Resistant Staphylococcus Aureus (CA-MRSA) USA300 Perturbs Acquisition Of Lysosomal Hydrolases And Requires Phagosomal Acidification For Survival In A Human Macrophage Cell Line. (Thesis). University of New England. Retrieved from https://dune.une.edu/theses/99

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tranchemontagne, Zachary Ronald. “Community-Acquired Methicillin-Resistant Staphylococcus Aureus (CA-MRSA) USA300 Perturbs Acquisition Of Lysosomal Hydrolases And Requires Phagosomal Acidification For Survival In A Human Macrophage Cell Line.” 2015. Thesis, University of New England. Accessed February 25, 2020. https://dune.une.edu/theses/99.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tranchemontagne, Zachary Ronald. “Community-Acquired Methicillin-Resistant Staphylococcus Aureus (CA-MRSA) USA300 Perturbs Acquisition Of Lysosomal Hydrolases And Requires Phagosomal Acidification For Survival In A Human Macrophage Cell Line.” 2015. Web. 25 Feb 2020.

Vancouver:

Tranchemontagne ZR. Community-Acquired Methicillin-Resistant Staphylococcus Aureus (CA-MRSA) USA300 Perturbs Acquisition Of Lysosomal Hydrolases And Requires Phagosomal Acidification For Survival In A Human Macrophage Cell Line. [Internet] [Thesis]. University of New England; 2015. [cited 2020 Feb 25]. Available from: https://dune.une.edu/theses/99.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tranchemontagne ZR. Community-Acquired Methicillin-Resistant Staphylococcus Aureus (CA-MRSA) USA300 Perturbs Acquisition Of Lysosomal Hydrolases And Requires Phagosomal Acidification For Survival In A Human Macrophage Cell Line. [Thesis]. University of New England; 2015. Available from: https://dune.une.edu/theses/99

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Iowa

28. Thalhofer, Colin Joseph. Leishmania infantum chagasi induces a dynamic cellular inflammatory response.

Degree: PhD, Immunology, 2011, University of Iowa

  Leishmania infantum chagasi (Lic) is a pathogenic protozoan parasite and one of the etiological agents of human visceral leishmaniasis (VL). VL is a potentially… (more)

Subjects/Keywords: Leishmania; Neutrophil; Immunology of Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Thalhofer, C. J. (2011). Leishmania infantum chagasi induces a dynamic cellular inflammatory response. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/1091

Chicago Manual of Style (16th Edition):

Thalhofer, Colin Joseph. “Leishmania infantum chagasi induces a dynamic cellular inflammatory response.” 2011. Doctoral Dissertation, University of Iowa. Accessed February 25, 2020. https://ir.uiowa.edu/etd/1091.

MLA Handbook (7th Edition):

Thalhofer, Colin Joseph. “Leishmania infantum chagasi induces a dynamic cellular inflammatory response.” 2011. Web. 25 Feb 2020.

Vancouver:

Thalhofer CJ. Leishmania infantum chagasi induces a dynamic cellular inflammatory response. [Internet] [Doctoral dissertation]. University of Iowa; 2011. [cited 2020 Feb 25]. Available from: https://ir.uiowa.edu/etd/1091.

Council of Science Editors:

Thalhofer CJ. Leishmania infantum chagasi induces a dynamic cellular inflammatory response. [Doctoral Dissertation]. University of Iowa; 2011. Available from: https://ir.uiowa.edu/etd/1091


University of Louisville

29. Guha Niyogi, Rajarshi. Cannabinoids suppress the innate immune response to periodontal pathogen Porphyromonas gingivalis in gingival epithelial cells.

Degree: MS, 2016, University of Louisville

  Marijuana is widely used in the United States for recreational and medicinal purposes. Despite the proposed beneficial effects of cannabis on certain medical conditions,… (more)

Subjects/Keywords: cannabinoids; inaate immunity; P. gingivalis; chronic periodontitis; Immunology and Infectious Disease; Immunology of Infectious Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Guha Niyogi, R. (2016). Cannabinoids suppress the innate immune response to periodontal pathogen Porphyromonas gingivalis in gingival epithelial cells. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/2503 ; https://ir.library.louisville.edu/etd/2503

Chicago Manual of Style (16th Edition):

Guha Niyogi, Rajarshi. “Cannabinoids suppress the innate immune response to periodontal pathogen Porphyromonas gingivalis in gingival epithelial cells.” 2016. Masters Thesis, University of Louisville. Accessed February 25, 2020. 10.18297/etd/2503 ; https://ir.library.louisville.edu/etd/2503.

MLA Handbook (7th Edition):

Guha Niyogi, Rajarshi. “Cannabinoids suppress the innate immune response to periodontal pathogen Porphyromonas gingivalis in gingival epithelial cells.” 2016. Web. 25 Feb 2020.

Vancouver:

Guha Niyogi R. Cannabinoids suppress the innate immune response to periodontal pathogen Porphyromonas gingivalis in gingival epithelial cells. [Internet] [Masters thesis]. University of Louisville; 2016. [cited 2020 Feb 25]. Available from: 10.18297/etd/2503 ; https://ir.library.louisville.edu/etd/2503.

Council of Science Editors:

Guha Niyogi R. Cannabinoids suppress the innate immune response to periodontal pathogen Porphyromonas gingivalis in gingival epithelial cells. [Masters Thesis]. University of Louisville; 2016. Available from: 10.18297/etd/2503 ; https://ir.library.louisville.edu/etd/2503


University of Pennsylvania

30. Whitmarsh, Ryan J. Innate Signals in the Macrophage Determine the Severity of Toxoplasma Gondii infection.

Degree: 2011, University of Pennsylvania

 Toxoplasma gondii is an obligate intracellular pathogen that is a cause of significant morbidity and mortality in humans and animals. The immune response necessary to… (more)

Subjects/Keywords: Toxoplasma; macrophage; SOCS3; STAT3; Immunology of Infectious Disease; Immunopathology; Other Immunology and Infectious Disease; Parasitology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Whitmarsh, R. J. (2011). Innate Signals in the Macrophage Determine the Severity of Toxoplasma Gondii infection. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Whitmarsh, Ryan J. “Innate Signals in the Macrophage Determine the Severity of Toxoplasma Gondii infection.” 2011. Thesis, University of Pennsylvania. Accessed February 25, 2020. https://repository.upenn.edu/edissertations/433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Whitmarsh, Ryan J. “Innate Signals in the Macrophage Determine the Severity of Toxoplasma Gondii infection.” 2011. Web. 25 Feb 2020.

Vancouver:

Whitmarsh RJ. Innate Signals in the Macrophage Determine the Severity of Toxoplasma Gondii infection. [Internet] [Thesis]. University of Pennsylvania; 2011. [cited 2020 Feb 25]. Available from: https://repository.upenn.edu/edissertations/433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Whitmarsh RJ. Innate Signals in the Macrophage Determine the Severity of Toxoplasma Gondii infection. [Thesis]. University of Pennsylvania; 2011. Available from: https://repository.upenn.edu/edissertations/433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] … [35]

.