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You searched for subject:(Immunology AND Infectious Disease). Showing records 1 – 30 of 1108 total matches.

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McMaster University

1. D'Agostino, Michael. Investigating the role of memory alveolar macrophages in early innate immune control of pulmonary tuberculosis.

Degree: MSc, 2019, McMaster University

Mycobacterium tuberculosis (M.tb) is the causative agent of pulmonary tuberculosis (TB). Over 25% of the world’s population is estimated to be infected with tuberculosis, yielding… (more)

Subjects/Keywords: Immunology; Infectious disease

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APA (6th Edition):

D'Agostino, M. (2019). Investigating the role of memory alveolar macrophages in early innate immune control of pulmonary tuberculosis. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/24846

Chicago Manual of Style (16th Edition):

D'Agostino, Michael. “Investigating the role of memory alveolar macrophages in early innate immune control of pulmonary tuberculosis.” 2019. Masters Thesis, McMaster University. Accessed February 27, 2021. http://hdl.handle.net/11375/24846.

MLA Handbook (7th Edition):

D'Agostino, Michael. “Investigating the role of memory alveolar macrophages in early innate immune control of pulmonary tuberculosis.” 2019. Web. 27 Feb 2021.

Vancouver:

D'Agostino M. Investigating the role of memory alveolar macrophages in early innate immune control of pulmonary tuberculosis. [Internet] [Masters thesis]. McMaster University; 2019. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/11375/24846.

Council of Science Editors:

D'Agostino M. Investigating the role of memory alveolar macrophages in early innate immune control of pulmonary tuberculosis. [Masters Thesis]. McMaster University; 2019. Available from: http://hdl.handle.net/11375/24846


Loyola University Chicago

2. Krukowski, Karen. Psychosocial Distress Mediates Immune Dysregulation Through Alterations in Global Epigenetic Patterns and Chromatin Remodeling Proteins.

Degree: PhD, Microbiology and Immunology, 2013, Loyola University Chicago

  Psychosocial distress, characterized by increased perceived stress, anxiety and mood disturbance, is a common response of women to a diagnosis of breast cancer (Northouse,… (more)

Subjects/Keywords: Immunology and Infectious Disease

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APA (6th Edition):

Krukowski, K. (2013). Psychosocial Distress Mediates Immune Dysregulation Through Alterations in Global Epigenetic Patterns and Chromatin Remodeling Proteins. (Doctoral Dissertation). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_diss/525

Chicago Manual of Style (16th Edition):

Krukowski, Karen. “Psychosocial Distress Mediates Immune Dysregulation Through Alterations in Global Epigenetic Patterns and Chromatin Remodeling Proteins.” 2013. Doctoral Dissertation, Loyola University Chicago. Accessed February 27, 2021. https://ecommons.luc.edu/luc_diss/525.

MLA Handbook (7th Edition):

Krukowski, Karen. “Psychosocial Distress Mediates Immune Dysregulation Through Alterations in Global Epigenetic Patterns and Chromatin Remodeling Proteins.” 2013. Web. 27 Feb 2021.

Vancouver:

Krukowski K. Psychosocial Distress Mediates Immune Dysregulation Through Alterations in Global Epigenetic Patterns and Chromatin Remodeling Proteins. [Internet] [Doctoral dissertation]. Loyola University Chicago; 2013. [cited 2021 Feb 27]. Available from: https://ecommons.luc.edu/luc_diss/525.

Council of Science Editors:

Krukowski K. Psychosocial Distress Mediates Immune Dysregulation Through Alterations in Global Epigenetic Patterns and Chromatin Remodeling Proteins. [Doctoral Dissertation]. Loyola University Chicago; 2013. Available from: https://ecommons.luc.edu/luc_diss/525


Loyola University Chicago

3. Bush, Kristin. Role for Histone Deacetylases in Glucocorticoid Receptor Mediated Transpression of Natural Killer Cell Activity.

Degree: MS, Microbiology and Immunology, 2011, Loyola University Chicago

  During periods of psychosocial distress glucocorticoids (GCs) are known to reduce the lytic activity of natural killer cells (NKCA). Glucocorticoid treatment also reduces acetylation… (more)

Subjects/Keywords: Immunology and Infectious Disease

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APA (6th Edition):

Bush, K. (2011). Role for Histone Deacetylases in Glucocorticoid Receptor Mediated Transpression of Natural Killer Cell Activity. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bush, Kristin. “Role for Histone Deacetylases in Glucocorticoid Receptor Mediated Transpression of Natural Killer Cell Activity.” 2011. Thesis, Loyola University Chicago. Accessed February 27, 2021. https://ecommons.luc.edu/luc_theses/567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bush, Kristin. “Role for Histone Deacetylases in Glucocorticoid Receptor Mediated Transpression of Natural Killer Cell Activity.” 2011. Web. 27 Feb 2021.

Vancouver:

Bush K. Role for Histone Deacetylases in Glucocorticoid Receptor Mediated Transpression of Natural Killer Cell Activity. [Internet] [Thesis]. Loyola University Chicago; 2011. [cited 2021 Feb 27]. Available from: https://ecommons.luc.edu/luc_theses/567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bush K. Role for Histone Deacetylases in Glucocorticoid Receptor Mediated Transpression of Natural Killer Cell Activity. [Thesis]. Loyola University Chicago; 2011. Available from: https://ecommons.luc.edu/luc_theses/567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

4. Armstrong, Cortney Linn. Characterization of Filifactor alocis and its immune evasion strategies employed against human neutrophils.

Degree: PhD, 2017, University of Louisville

  Periodontal disease is among the most common of inflammatory conditions and is caused by bacterial and host derived factors. The presence of bacteria drives… (more)

Subjects/Keywords: Immunology of Infectious Disease

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APA (6th Edition):

Armstrong, C. L. (2017). Characterization of Filifactor alocis and its immune evasion strategies employed against human neutrophils. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2769 ; https://ir.library.louisville.edu/etd/2769

Chicago Manual of Style (16th Edition):

Armstrong, Cortney Linn. “Characterization of Filifactor alocis and its immune evasion strategies employed against human neutrophils.” 2017. Doctoral Dissertation, University of Louisville. Accessed February 27, 2021. 10.18297/etd/2769 ; https://ir.library.louisville.edu/etd/2769.

MLA Handbook (7th Edition):

Armstrong, Cortney Linn. “Characterization of Filifactor alocis and its immune evasion strategies employed against human neutrophils.” 2017. Web. 27 Feb 2021.

Vancouver:

Armstrong CL. Characterization of Filifactor alocis and its immune evasion strategies employed against human neutrophils. [Internet] [Doctoral dissertation]. University of Louisville; 2017. [cited 2021 Feb 27]. Available from: 10.18297/etd/2769 ; https://ir.library.louisville.edu/etd/2769.

Council of Science Editors:

Armstrong CL. Characterization of Filifactor alocis and its immune evasion strategies employed against human neutrophils. [Doctoral Dissertation]. University of Louisville; 2017. Available from: 10.18297/etd/2769 ; https://ir.library.louisville.edu/etd/2769


Wayne State University

5. Gibson, Heather. Ctla-4 transcriptional activation: regulation of induced expression.

Degree: PhD, Immunology and Microbiology, 2011, Wayne State University

  Cytotoxic T-lymphocyte antigen 4 (CTLA-4) is a T cell surface protein that is homologous to CD28 and binds to the B7 family of ligands.… (more)

Subjects/Keywords: Immunology and Infectious Disease

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APA (6th Edition):

Gibson, H. (2011). Ctla-4 transcriptional activation: regulation of induced expression. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/237

Chicago Manual of Style (16th Edition):

Gibson, Heather. “Ctla-4 transcriptional activation: regulation of induced expression.” 2011. Doctoral Dissertation, Wayne State University. Accessed February 27, 2021. https://digitalcommons.wayne.edu/oa_dissertations/237.

MLA Handbook (7th Edition):

Gibson, Heather. “Ctla-4 transcriptional activation: regulation of induced expression.” 2011. Web. 27 Feb 2021.

Vancouver:

Gibson H. Ctla-4 transcriptional activation: regulation of induced expression. [Internet] [Doctoral dissertation]. Wayne State University; 2011. [cited 2021 Feb 27]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/237.

Council of Science Editors:

Gibson H. Ctla-4 transcriptional activation: regulation of induced expression. [Doctoral Dissertation]. Wayne State University; 2011. Available from: https://digitalcommons.wayne.edu/oa_dissertations/237


Wayne State University

6. Rao, Pushpa Durgesh. Alterations In Ocular Surface System During The Pathogenesis Of Herpes Stromal Keratitis.

Degree: PhD, Anatomy and Cell Biology, 2019, Wayne State University

  Herpes stromal keratitis (HSK) is a chronic immuno-inflammatory ocular disease caused by Herpes simplex virus-1 (HSV-1) infection in the cornea. HSK is characterized by… (more)

Subjects/Keywords: Immunology and Infectious Disease

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APA (6th Edition):

Rao, P. D. (2019). Alterations In Ocular Surface System During The Pathogenesis Of Herpes Stromal Keratitis. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/2181

Chicago Manual of Style (16th Edition):

Rao, Pushpa Durgesh. “Alterations In Ocular Surface System During The Pathogenesis Of Herpes Stromal Keratitis.” 2019. Doctoral Dissertation, Wayne State University. Accessed February 27, 2021. https://digitalcommons.wayne.edu/oa_dissertations/2181.

MLA Handbook (7th Edition):

Rao, Pushpa Durgesh. “Alterations In Ocular Surface System During The Pathogenesis Of Herpes Stromal Keratitis.” 2019. Web. 27 Feb 2021.

Vancouver:

Rao PD. Alterations In Ocular Surface System During The Pathogenesis Of Herpes Stromal Keratitis. [Internet] [Doctoral dissertation]. Wayne State University; 2019. [cited 2021 Feb 27]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/2181.

Council of Science Editors:

Rao PD. Alterations In Ocular Surface System During The Pathogenesis Of Herpes Stromal Keratitis. [Doctoral Dissertation]. Wayne State University; 2019. Available from: https://digitalcommons.wayne.edu/oa_dissertations/2181


University of Iowa

7. Rosean, Timothy Robert. The tumor microenvironment is critical for the development of plasma cell neoplasia in mice.

Degree: PhD, Immunology, 2014, University of Iowa

  Plasma cell neoplasms (PCN), including multiple myeloma, are tumors of terminally differentiated B cells. Despite a significant research effort, and numerous advances in therapy,… (more)

Subjects/Keywords: publicabstract; Immunology of Infectious Disease

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APA (6th Edition):

Rosean, T. R. (2014). The tumor microenvironment is critical for the development of plasma cell neoplasia in mice. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/1497

Chicago Manual of Style (16th Edition):

Rosean, Timothy Robert. “The tumor microenvironment is critical for the development of plasma cell neoplasia in mice.” 2014. Doctoral Dissertation, University of Iowa. Accessed February 27, 2021. https://ir.uiowa.edu/etd/1497.

MLA Handbook (7th Edition):

Rosean, Timothy Robert. “The tumor microenvironment is critical for the development of plasma cell neoplasia in mice.” 2014. Web. 27 Feb 2021.

Vancouver:

Rosean TR. The tumor microenvironment is critical for the development of plasma cell neoplasia in mice. [Internet] [Doctoral dissertation]. University of Iowa; 2014. [cited 2021 Feb 27]. Available from: https://ir.uiowa.edu/etd/1497.

Council of Science Editors:

Rosean TR. The tumor microenvironment is critical for the development of plasma cell neoplasia in mice. [Doctoral Dissertation]. University of Iowa; 2014. Available from: https://ir.uiowa.edu/etd/1497


University of Iowa

8. Scorza, Breanna M. Interaction of human keratinocytes with Leishmania spp.: a comparative study of Leishmania infantum and Leishmania major.

Degree: PhD, Immunology, 2017, University of Iowa

  Leishmaniasis refers to the group of diseases caused by pathogenic protozoan parasites of the genus Leishmania. Nearly all human Leishmania spp. infections are initiated… (more)

Subjects/Keywords: Immunology of Infectious Disease

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APA (6th Edition):

Scorza, B. M. (2017). Interaction of human keratinocytes with Leishmania spp.: a comparative study of Leishmania infantum and Leishmania major. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/5847

Chicago Manual of Style (16th Edition):

Scorza, Breanna M. “Interaction of human keratinocytes with Leishmania spp.: a comparative study of Leishmania infantum and Leishmania major.” 2017. Doctoral Dissertation, University of Iowa. Accessed February 27, 2021. https://ir.uiowa.edu/etd/5847.

MLA Handbook (7th Edition):

Scorza, Breanna M. “Interaction of human keratinocytes with Leishmania spp.: a comparative study of Leishmania infantum and Leishmania major.” 2017. Web. 27 Feb 2021.

Vancouver:

Scorza BM. Interaction of human keratinocytes with Leishmania spp.: a comparative study of Leishmania infantum and Leishmania major. [Internet] [Doctoral dissertation]. University of Iowa; 2017. [cited 2021 Feb 27]. Available from: https://ir.uiowa.edu/etd/5847.

Council of Science Editors:

Scorza BM. Interaction of human keratinocytes with Leishmania spp.: a comparative study of Leishmania infantum and Leishmania major. [Doctoral Dissertation]. University of Iowa; 2017. Available from: https://ir.uiowa.edu/etd/5847


University of Iowa

9. Gorman, Jacob. Regulation of T cell responses by the surface receptor Tim-3.

Degree: PhD, Immunology, 2014, University of Iowa

  Tim-3 (for T cell immunoglobulin and mucin domain 3) is a surface molecule expressed throughout the immune system that appears to mediate both stimulatory… (more)

Subjects/Keywords: Immunology of Infectious Disease

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APA (6th Edition):

Gorman, J. (2014). Regulation of T cell responses by the surface receptor Tim-3. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/1326

Chicago Manual of Style (16th Edition):

Gorman, Jacob. “Regulation of T cell responses by the surface receptor Tim-3.” 2014. Doctoral Dissertation, University of Iowa. Accessed February 27, 2021. https://ir.uiowa.edu/etd/1326.

MLA Handbook (7th Edition):

Gorman, Jacob. “Regulation of T cell responses by the surface receptor Tim-3.” 2014. Web. 27 Feb 2021.

Vancouver:

Gorman J. Regulation of T cell responses by the surface receptor Tim-3. [Internet] [Doctoral dissertation]. University of Iowa; 2014. [cited 2021 Feb 27]. Available from: https://ir.uiowa.edu/etd/1326.

Council of Science Editors:

Gorman J. Regulation of T cell responses by the surface receptor Tim-3. [Doctoral Dissertation]. University of Iowa; 2014. Available from: https://ir.uiowa.edu/etd/1326


University of Iowa

10. Janowski, Ann M. The protective roles of NLRs during infection and tumor progression.

Degree: PhD, Immunology, 2016, University of Iowa

  The immune system has evolved to fight off numerous pathogens. The first line of defense against these pathogens are innate immune cells. Innate immune… (more)

Subjects/Keywords: Immunology of Infectious Disease

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APA (6th Edition):

Janowski, A. M. (2016). The protective roles of NLRs during infection and tumor progression. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/5518

Chicago Manual of Style (16th Edition):

Janowski, Ann M. “The protective roles of NLRs during infection and tumor progression.” 2016. Doctoral Dissertation, University of Iowa. Accessed February 27, 2021. https://ir.uiowa.edu/etd/5518.

MLA Handbook (7th Edition):

Janowski, Ann M. “The protective roles of NLRs during infection and tumor progression.” 2016. Web. 27 Feb 2021.

Vancouver:

Janowski AM. The protective roles of NLRs during infection and tumor progression. [Internet] [Doctoral dissertation]. University of Iowa; 2016. [cited 2021 Feb 27]. Available from: https://ir.uiowa.edu/etd/5518.

Council of Science Editors:

Janowski AM. The protective roles of NLRs during infection and tumor progression. [Doctoral Dissertation]. University of Iowa; 2016. Available from: https://ir.uiowa.edu/etd/5518

11. Hendrix, Emily K. Characterizing the early stages of a novel host shift using host fitness and metabolomics.

Degree: MS, Biology, 2018, Eastern Washington University

  The factors that contribute to successful colonization of a novel host species by a pathogen remain unclear. One likely factor determining host shift success… (more)

Subjects/Keywords: Biology; Immunology of Infectious Disease

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APA (6th Edition):

Hendrix, E. K. (2018). Characterizing the early stages of a novel host shift using host fitness and metabolomics. (Thesis). Eastern Washington University. Retrieved from https://dc.ewu.edu/theses/500

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hendrix, Emily K. “Characterizing the early stages of a novel host shift using host fitness and metabolomics.” 2018. Thesis, Eastern Washington University. Accessed February 27, 2021. https://dc.ewu.edu/theses/500.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hendrix, Emily K. “Characterizing the early stages of a novel host shift using host fitness and metabolomics.” 2018. Web. 27 Feb 2021.

Vancouver:

Hendrix EK. Characterizing the early stages of a novel host shift using host fitness and metabolomics. [Internet] [Thesis]. Eastern Washington University; 2018. [cited 2021 Feb 27]. Available from: https://dc.ewu.edu/theses/500.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hendrix EK. Characterizing the early stages of a novel host shift using host fitness and metabolomics. [Thesis]. Eastern Washington University; 2018. Available from: https://dc.ewu.edu/theses/500

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Iowa

12. Lifton, Samuel Robert. Retroviral mutagenesis in a newly developed myc transgenic mouse model of human B cell and plasma cell neoplasia.

Degree: MS, Interdisciplinary Studies in Immunology, 2013, University of Iowa

  Three potential driver genes were identified by use of retroviral mutagenesis in the newly developed iMyc model of B cell malignancy. To do so,… (more)

Subjects/Keywords: Immunology of Infectious Disease

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APA (6th Edition):

Lifton, S. R. (2013). Retroviral mutagenesis in a newly developed myc transgenic mouse model of human B cell and plasma cell neoplasia. (Masters Thesis). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/1479

Chicago Manual of Style (16th Edition):

Lifton, Samuel Robert. “Retroviral mutagenesis in a newly developed myc transgenic mouse model of human B cell and plasma cell neoplasia.” 2013. Masters Thesis, University of Iowa. Accessed February 27, 2021. https://ir.uiowa.edu/etd/1479.

MLA Handbook (7th Edition):

Lifton, Samuel Robert. “Retroviral mutagenesis in a newly developed myc transgenic mouse model of human B cell and plasma cell neoplasia.” 2013. Web. 27 Feb 2021.

Vancouver:

Lifton SR. Retroviral mutagenesis in a newly developed myc transgenic mouse model of human B cell and plasma cell neoplasia. [Internet] [Masters thesis]. University of Iowa; 2013. [cited 2021 Feb 27]. Available from: https://ir.uiowa.edu/etd/1479.

Council of Science Editors:

Lifton SR. Retroviral mutagenesis in a newly developed myc transgenic mouse model of human B cell and plasma cell neoplasia. [Masters Thesis]. University of Iowa; 2013. Available from: https://ir.uiowa.edu/etd/1479


University of Iowa

13. Parlet, Corey Patrick. Mechanisms by which chronic ethanol consumption impairs cutaneous immunity.

Degree: PhD, Immunology, 2014, University of Iowa

  The immunosuppressive effects of chronic alcohol abuse are profound, wide-ranging and readily apparent at the body's barriers. In the skin, alcoholism is associated with… (more)

Subjects/Keywords: Immunology of Infectious Disease

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APA (6th Edition):

Parlet, C. P. (2014). Mechanisms by which chronic ethanol consumption impairs cutaneous immunity. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/4712

Chicago Manual of Style (16th Edition):

Parlet, Corey Patrick. “Mechanisms by which chronic ethanol consumption impairs cutaneous immunity.” 2014. Doctoral Dissertation, University of Iowa. Accessed February 27, 2021. https://ir.uiowa.edu/etd/4712.

MLA Handbook (7th Edition):

Parlet, Corey Patrick. “Mechanisms by which chronic ethanol consumption impairs cutaneous immunity.” 2014. Web. 27 Feb 2021.

Vancouver:

Parlet CP. Mechanisms by which chronic ethanol consumption impairs cutaneous immunity. [Internet] [Doctoral dissertation]. University of Iowa; 2014. [cited 2021 Feb 27]. Available from: https://ir.uiowa.edu/etd/4712.

Council of Science Editors:

Parlet CP. Mechanisms by which chronic ethanol consumption impairs cutaneous immunity. [Doctoral Dissertation]. University of Iowa; 2014. Available from: https://ir.uiowa.edu/etd/4712


University of Iowa

14. Vacaflores Salinas, Aldo Fabian. New roles for an old cytokine : characterizing how exposure to Il-12 alters human CD4 And CD8 T cell responses.

Degree: PhD, Immunology, 2016, University of Iowa

  CD4 and CD8 T cells are constantly exposed to inflammatory signals that influence diverse functional outcomes during infections and certain autoimmune disorders. One of… (more)

Subjects/Keywords: Immunology of Infectious Disease

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APA (6th Edition):

Vacaflores Salinas, A. F. (2016). New roles for an old cytokine : characterizing how exposure to Il-12 alters human CD4 And CD8 T cell responses. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/2157

Chicago Manual of Style (16th Edition):

Vacaflores Salinas, Aldo Fabian. “New roles for an old cytokine : characterizing how exposure to Il-12 alters human CD4 And CD8 T cell responses.” 2016. Doctoral Dissertation, University of Iowa. Accessed February 27, 2021. https://ir.uiowa.edu/etd/2157.

MLA Handbook (7th Edition):

Vacaflores Salinas, Aldo Fabian. “New roles for an old cytokine : characterizing how exposure to Il-12 alters human CD4 And CD8 T cell responses.” 2016. Web. 27 Feb 2021.

Vancouver:

Vacaflores Salinas AF. New roles for an old cytokine : characterizing how exposure to Il-12 alters human CD4 And CD8 T cell responses. [Internet] [Doctoral dissertation]. University of Iowa; 2016. [cited 2021 Feb 27]. Available from: https://ir.uiowa.edu/etd/2157.

Council of Science Editors:

Vacaflores Salinas AF. New roles for an old cytokine : characterizing how exposure to Il-12 alters human CD4 And CD8 T cell responses. [Doctoral Dissertation]. University of Iowa; 2016. Available from: https://ir.uiowa.edu/etd/2157


University of Melbourne

15. Wang, Huimeng. Mucosal associated invariant T cell-mediated vaccination and protection against pathogenic bacteria.

Degree: 2018, University of Melbourne

 Mucosal Associated Invariant T (MAIT) cells are a subset of innate-like T cells, which are abundant in humans. They recognize antigens that are derived from… (more)

Subjects/Keywords: cellular immunology; infectious disease

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APA (6th Edition):

Wang, H. (2018). Mucosal associated invariant T cell-mediated vaccination and protection against pathogenic bacteria. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/214694

Chicago Manual of Style (16th Edition):

Wang, Huimeng. “Mucosal associated invariant T cell-mediated vaccination and protection against pathogenic bacteria.” 2018. Doctoral Dissertation, University of Melbourne. Accessed February 27, 2021. http://hdl.handle.net/11343/214694.

MLA Handbook (7th Edition):

Wang, Huimeng. “Mucosal associated invariant T cell-mediated vaccination and protection against pathogenic bacteria.” 2018. Web. 27 Feb 2021.

Vancouver:

Wang H. Mucosal associated invariant T cell-mediated vaccination and protection against pathogenic bacteria. [Internet] [Doctoral dissertation]. University of Melbourne; 2018. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/11343/214694.

Council of Science Editors:

Wang H. Mucosal associated invariant T cell-mediated vaccination and protection against pathogenic bacteria. [Doctoral Dissertation]. University of Melbourne; 2018. Available from: http://hdl.handle.net/11343/214694

16. Singh, Varan J. Development and Maintenance of Thymic Epithelial Microenvironment.

Degree: MS(M.S.), Biology, 2015, City University of New York

  Thymus is a primary lymphoid organ responsible for proper development and selection Of T-­‐cells. The thymic microenvironment, however, undergoes age related involution, Resulting in… (more)

Subjects/Keywords: Biology; Immunology and Infectious Disease

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APA (6th Edition):

Singh, V. J. (2015). Development and Maintenance of Thymic Epithelial Microenvironment. (Thesis). City University of New York. Retrieved from https://academicworks.cuny.edu/cc_etds_theses/549

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Singh, Varan J. “Development and Maintenance of Thymic Epithelial Microenvironment.” 2015. Thesis, City University of New York. Accessed February 27, 2021. https://academicworks.cuny.edu/cc_etds_theses/549.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Singh, Varan J. “Development and Maintenance of Thymic Epithelial Microenvironment.” 2015. Web. 27 Feb 2021.

Vancouver:

Singh VJ. Development and Maintenance of Thymic Epithelial Microenvironment. [Internet] [Thesis]. City University of New York; 2015. [cited 2021 Feb 27]. Available from: https://academicworks.cuny.edu/cc_etds_theses/549.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Singh VJ. Development and Maintenance of Thymic Epithelial Microenvironment. [Thesis]. City University of New York; 2015. Available from: https://academicworks.cuny.edu/cc_etds_theses/549

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

17. Hutcherson, Justin. Characterization of the innate response to the RagB protein of Porphyromonas gingivalis.

Degree: PhD, 2015, University of Louisville

  More than 64 million people in the US suffer from some form of periodontal disease. Periodontal diseases are bacterial infections of the hard and… (more)

Subjects/Keywords: Immunology and Infectious Disease; Microbiology

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APA (6th Edition):

Hutcherson, J. (2015). Characterization of the innate response to the RagB protein of Porphyromonas gingivalis. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2077 ; https://ir.library.louisville.edu/etd/2077

Chicago Manual of Style (16th Edition):

Hutcherson, Justin. “Characterization of the innate response to the RagB protein of Porphyromonas gingivalis.” 2015. Doctoral Dissertation, University of Louisville. Accessed February 27, 2021. 10.18297/etd/2077 ; https://ir.library.louisville.edu/etd/2077.

MLA Handbook (7th Edition):

Hutcherson, Justin. “Characterization of the innate response to the RagB protein of Porphyromonas gingivalis.” 2015. Web. 27 Feb 2021.

Vancouver:

Hutcherson J. Characterization of the innate response to the RagB protein of Porphyromonas gingivalis. [Internet] [Doctoral dissertation]. University of Louisville; 2015. [cited 2021 Feb 27]. Available from: 10.18297/etd/2077 ; https://ir.library.louisville.edu/etd/2077.

Council of Science Editors:

Hutcherson J. Characterization of the innate response to the RagB protein of Porphyromonas gingivalis. [Doctoral Dissertation]. University of Louisville; 2015. Available from: 10.18297/etd/2077 ; https://ir.library.louisville.edu/etd/2077


University of Louisville

18. Gerlach, Rachael Lask. Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models.

Degree: PhD, 2015, University of Louisville

  Influenza A virus (IAV) subtypes and even genotypes within subtypes can show differences in tropism (host, cell type), magnitude of infection, immune response and… (more)

Subjects/Keywords: Immunology and Infectious Disease; Microbiology

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APA (6th Edition):

Gerlach, R. L. (2015). Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2078 ; https://ir.library.louisville.edu/etd/2078

Chicago Manual of Style (16th Edition):

Gerlach, Rachael Lask. “Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models.” 2015. Doctoral Dissertation, University of Louisville. Accessed February 27, 2021. 10.18297/etd/2078 ; https://ir.library.louisville.edu/etd/2078.

MLA Handbook (7th Edition):

Gerlach, Rachael Lask. “Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models.” 2015. Web. 27 Feb 2021.

Vancouver:

Gerlach RL. Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models. [Internet] [Doctoral dissertation]. University of Louisville; 2015. [cited 2021 Feb 27]. Available from: 10.18297/etd/2078 ; https://ir.library.louisville.edu/etd/2078.

Council of Science Editors:

Gerlach RL. Early host response and immune signaling to 2009 pandemic influenza A (H1N1) viruses in primary cell culture models. [Doctoral Dissertation]. University of Louisville; 2015. Available from: 10.18297/etd/2078 ; https://ir.library.louisville.edu/etd/2078


University of Louisville

19. Zhu, Yanfang. Regulation of macrophage inflammatory signaling pathways by AMP-activated protein kinase.

Degree: PhD, 2014, University of Louisville

  AMP-activated protein kinase, AMPK, is a conserved serine/threonine kinase with a critical function in the regulation of metabolic pathways in eukaryotic cells. Recently, AMPK… (more)

Subjects/Keywords: Immunology and Infectious Disease; Microbiology

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APA (6th Edition):

Zhu, Y. (2014). Regulation of macrophage inflammatory signaling pathways by AMP-activated protein kinase. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/1649 ; https://ir.library.louisville.edu/etd/1649

Chicago Manual of Style (16th Edition):

Zhu, Yanfang. “Regulation of macrophage inflammatory signaling pathways by AMP-activated protein kinase.” 2014. Doctoral Dissertation, University of Louisville. Accessed February 27, 2021. 10.18297/etd/1649 ; https://ir.library.louisville.edu/etd/1649.

MLA Handbook (7th Edition):

Zhu, Yanfang. “Regulation of macrophage inflammatory signaling pathways by AMP-activated protein kinase.” 2014. Web. 27 Feb 2021.

Vancouver:

Zhu Y. Regulation of macrophage inflammatory signaling pathways by AMP-activated protein kinase. [Internet] [Doctoral dissertation]. University of Louisville; 2014. [cited 2021 Feb 27]. Available from: 10.18297/etd/1649 ; https://ir.library.louisville.edu/etd/1649.

Council of Science Editors:

Zhu Y. Regulation of macrophage inflammatory signaling pathways by AMP-activated protein kinase. [Doctoral Dissertation]. University of Louisville; 2014. Available from: 10.18297/etd/1649 ; https://ir.library.louisville.edu/etd/1649


University of Louisville

20. Camp, Jeremy V., 1980-. Critical insights into the pathogenesis of clinical isolates of pandemic influenza A(H1N1) 2009 virus in mouse and ferret models.

Degree: PhD, 2015, University of Louisville

  Influenza A virus (IAV) is a minus-sense, segmented, single-stranded RNA virus that infects the respiratory tract of humans and can cause severe illness. Novel… (more)

Subjects/Keywords: Immunology and Infectious Disease; Microbiology

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APA (6th Edition):

Camp, Jeremy V., 1. (2015). Critical insights into the pathogenesis of clinical isolates of pandemic influenza A(H1N1) 2009 virus in mouse and ferret models. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2079 ; https://ir.library.louisville.edu/etd/2079

Chicago Manual of Style (16th Edition):

Camp, Jeremy V., 1980-. “Critical insights into the pathogenesis of clinical isolates of pandemic influenza A(H1N1) 2009 virus in mouse and ferret models.” 2015. Doctoral Dissertation, University of Louisville. Accessed February 27, 2021. 10.18297/etd/2079 ; https://ir.library.louisville.edu/etd/2079.

MLA Handbook (7th Edition):

Camp, Jeremy V., 1980-. “Critical insights into the pathogenesis of clinical isolates of pandemic influenza A(H1N1) 2009 virus in mouse and ferret models.” 2015. Web. 27 Feb 2021.

Vancouver:

Camp, Jeremy V. 1. Critical insights into the pathogenesis of clinical isolates of pandemic influenza A(H1N1) 2009 virus in mouse and ferret models. [Internet] [Doctoral dissertation]. University of Louisville; 2015. [cited 2021 Feb 27]. Available from: 10.18297/etd/2079 ; https://ir.library.louisville.edu/etd/2079.

Council of Science Editors:

Camp, Jeremy V. 1. Critical insights into the pathogenesis of clinical isolates of pandemic influenza A(H1N1) 2009 virus in mouse and ferret models. [Doctoral Dissertation]. University of Louisville; 2015. Available from: 10.18297/etd/2079 ; https://ir.library.louisville.edu/etd/2079


Loyola University Chicago

21. Hlavin, Sara. Binge Ethanol Leads to Decreased Macrophage Accumulation in Infected Cutaneous Wounds.

Degree: MS, Microbiology and Immunology, 2012, Loyola University Chicago

  Trauma patients who consumed alcohol prior to sustaining injuries have higher rates of morbidity and mortality than those with comparable injuries who did not… (more)

Subjects/Keywords: Immunology and Infectious Disease

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APA (6th Edition):

Hlavin, S. (2012). Binge Ethanol Leads to Decreased Macrophage Accumulation in Infected Cutaneous Wounds. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/720

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hlavin, Sara. “Binge Ethanol Leads to Decreased Macrophage Accumulation in Infected Cutaneous Wounds.” 2012. Thesis, Loyola University Chicago. Accessed February 27, 2021. https://ecommons.luc.edu/luc_theses/720.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hlavin, Sara. “Binge Ethanol Leads to Decreased Macrophage Accumulation in Infected Cutaneous Wounds.” 2012. Web. 27 Feb 2021.

Vancouver:

Hlavin S. Binge Ethanol Leads to Decreased Macrophage Accumulation in Infected Cutaneous Wounds. [Internet] [Thesis]. Loyola University Chicago; 2012. [cited 2021 Feb 27]. Available from: https://ecommons.luc.edu/luc_theses/720.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hlavin S. Binge Ethanol Leads to Decreased Macrophage Accumulation in Infected Cutaneous Wounds. [Thesis]. Loyola University Chicago; 2012. Available from: https://ecommons.luc.edu/luc_theses/720

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

22. Ochoa, Karina. Selective Expansion of B Cells by Intestinal Microbiota.

Degree: MS, Microbiology and Immunology, 2013, Loyola University Chicago

  In rabbits, antibody diversity and B cell expansion are generated in gut-associated lymphoid tissues (GALT), in an antigen- and T cell-independent mechanism, and require… (more)

Subjects/Keywords: Immunology and Infectious Disease

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APA (6th Edition):

Ochoa, K. (2013). Selective Expansion of B Cells by Intestinal Microbiota. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/1468

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ochoa, Karina. “Selective Expansion of B Cells by Intestinal Microbiota.” 2013. Thesis, Loyola University Chicago. Accessed February 27, 2021. https://ecommons.luc.edu/luc_theses/1468.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ochoa, Karina. “Selective Expansion of B Cells by Intestinal Microbiota.” 2013. Web. 27 Feb 2021.

Vancouver:

Ochoa K. Selective Expansion of B Cells by Intestinal Microbiota. [Internet] [Thesis]. Loyola University Chicago; 2013. [cited 2021 Feb 27]. Available from: https://ecommons.luc.edu/luc_theses/1468.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ochoa K. Selective Expansion of B Cells by Intestinal Microbiota. [Thesis]. Loyola University Chicago; 2013. Available from: https://ecommons.luc.edu/luc_theses/1468

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

23. Foley, Kendra. Tcr Modifications to Enhance Expression, Chain Pairing, and Antigen Recognition for Adoptive T Cell Transfer.

Degree: MS, Microbiology and Immunology, 2015, Loyola University Chicago

  T cell receptor (TCR) gene modified T cells for adoptive T cell transfer therapy have been shown to have clinical success in treating melanoma… (more)

Subjects/Keywords: Immunology and Infectious Disease

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APA (6th Edition):

Foley, K. (2015). Tcr Modifications to Enhance Expression, Chain Pairing, and Antigen Recognition for Adoptive T Cell Transfer. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/2784

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Foley, Kendra. “Tcr Modifications to Enhance Expression, Chain Pairing, and Antigen Recognition for Adoptive T Cell Transfer.” 2015. Thesis, Loyola University Chicago. Accessed February 27, 2021. https://ecommons.luc.edu/luc_theses/2784.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Foley, Kendra. “Tcr Modifications to Enhance Expression, Chain Pairing, and Antigen Recognition for Adoptive T Cell Transfer.” 2015. Web. 27 Feb 2021.

Vancouver:

Foley K. Tcr Modifications to Enhance Expression, Chain Pairing, and Antigen Recognition for Adoptive T Cell Transfer. [Internet] [Thesis]. Loyola University Chicago; 2015. [cited 2021 Feb 27]. Available from: https://ecommons.luc.edu/luc_theses/2784.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Foley K. Tcr Modifications to Enhance Expression, Chain Pairing, and Antigen Recognition for Adoptive T Cell Transfer. [Thesis]. Loyola University Chicago; 2015. Available from: https://ecommons.luc.edu/luc_theses/2784

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

24. Cannon, Abigail Rhea. Role of Heat Shock Proteins in Maintenance of the Gut Barrier Following Burn Injury.

Degree: MS, Microbiology and Immunology, 2015, Loyola University Chicago

  Introduction: Burn injury remains a prominent clinical problem. Patients suffering from burns often succumb to secondary infectious complications leading to sepsis and widespread tissue… (more)

Subjects/Keywords: Immunology and Infectious Disease

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APA (6th Edition):

Cannon, A. R. (2015). Role of Heat Shock Proteins in Maintenance of the Gut Barrier Following Burn Injury. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/3127

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cannon, Abigail Rhea. “Role of Heat Shock Proteins in Maintenance of the Gut Barrier Following Burn Injury.” 2015. Thesis, Loyola University Chicago. Accessed February 27, 2021. https://ecommons.luc.edu/luc_theses/3127.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cannon, Abigail Rhea. “Role of Heat Shock Proteins in Maintenance of the Gut Barrier Following Burn Injury.” 2015. Web. 27 Feb 2021.

Vancouver:

Cannon AR. Role of Heat Shock Proteins in Maintenance of the Gut Barrier Following Burn Injury. [Internet] [Thesis]. Loyola University Chicago; 2015. [cited 2021 Feb 27]. Available from: https://ecommons.luc.edu/luc_theses/3127.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cannon AR. Role of Heat Shock Proteins in Maintenance of the Gut Barrier Following Burn Injury. [Thesis]. Loyola University Chicago; 2015. Available from: https://ecommons.luc.edu/luc_theses/3127

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

25. Keller, Taylor. Cell Intrinsic Characteristics of Cord Blood Naive CD4 T Cells.

Degree: MS, Microbiology and Immunology, 2018, Loyola University Chicago

  The neonatal immune system is functionally distinct from the adult immune system. Neonatal immune responses are less reactive than their adult counterparts, and as… (more)

Subjects/Keywords: Immunology and Infectious Disease

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APA (6th Edition):

Keller, T. (2018). Cell Intrinsic Characteristics of Cord Blood Naive CD4 T Cells. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/3682

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Keller, Taylor. “Cell Intrinsic Characteristics of Cord Blood Naive CD4 T Cells.” 2018. Thesis, Loyola University Chicago. Accessed February 27, 2021. https://ecommons.luc.edu/luc_theses/3682.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Keller, Taylor. “Cell Intrinsic Characteristics of Cord Blood Naive CD4 T Cells.” 2018. Web. 27 Feb 2021.

Vancouver:

Keller T. Cell Intrinsic Characteristics of Cord Blood Naive CD4 T Cells. [Internet] [Thesis]. Loyola University Chicago; 2018. [cited 2021 Feb 27]. Available from: https://ecommons.luc.edu/luc_theses/3682.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Keller T. Cell Intrinsic Characteristics of Cord Blood Naive CD4 T Cells. [Thesis]. Loyola University Chicago; 2018. Available from: https://ecommons.luc.edu/luc_theses/3682

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

26. Haque, Tamara T. FLUOXETINE ATTENUATES MAST CELL FUNCTION BY TARGETING PURINERGIC SIGNALING.

Degree: PhD, Microbiology & Immunology, 2019, Virginia Commonwealth University

  Mast cells are tissue resident, innate immune cells that provide protection against parasitic and bacterial infections and venom poisoning. Mast cells also play a… (more)

Subjects/Keywords: Immunology and Infectious Disease

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APA (6th Edition):

Haque, T. T. (2019). FLUOXETINE ATTENUATES MAST CELL FUNCTION BY TARGETING PURINERGIC SIGNALING. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/6924-QD95 ; https://scholarscompass.vcu.edu/etd/5996

Chicago Manual of Style (16th Edition):

Haque, Tamara T. “FLUOXETINE ATTENUATES MAST CELL FUNCTION BY TARGETING PURINERGIC SIGNALING.” 2019. Doctoral Dissertation, Virginia Commonwealth University. Accessed February 27, 2021. https://doi.org/10.25772/6924-QD95 ; https://scholarscompass.vcu.edu/etd/5996.

MLA Handbook (7th Edition):

Haque, Tamara T. “FLUOXETINE ATTENUATES MAST CELL FUNCTION BY TARGETING PURINERGIC SIGNALING.” 2019. Web. 27 Feb 2021.

Vancouver:

Haque TT. FLUOXETINE ATTENUATES MAST CELL FUNCTION BY TARGETING PURINERGIC SIGNALING. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2019. [cited 2021 Feb 27]. Available from: https://doi.org/10.25772/6924-QD95 ; https://scholarscompass.vcu.edu/etd/5996.

Council of Science Editors:

Haque TT. FLUOXETINE ATTENUATES MAST CELL FUNCTION BY TARGETING PURINERGIC SIGNALING. [Doctoral Dissertation]. Virginia Commonwealth University; 2019. Available from: https://doi.org/10.25772/6924-QD95 ; https://scholarscompass.vcu.edu/etd/5996

27. Uprety, Tirth. Role of Bovine Ileal Sub-epithelial Myofibroblasts and Epithelial Cells in Innate Immunity.

Degree: MS, Biology and Microbiology, 2018, South Dakota State University

  Gastro-intestinal (GI) tract harbors largest number of microbiota as well as the largest number of immune cells for a given tissue. The host needs… (more)

Subjects/Keywords: Immunology and Infectious Disease; Microbiology

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APA (6th Edition):

Uprety, T. (2018). Role of Bovine Ileal Sub-epithelial Myofibroblasts and Epithelial Cells in Innate Immunity. (Masters Thesis). South Dakota State University. Retrieved from https://openprairie.sdstate.edu/etd/2942

Chicago Manual of Style (16th Edition):

Uprety, Tirth. “Role of Bovine Ileal Sub-epithelial Myofibroblasts and Epithelial Cells in Innate Immunity.” 2018. Masters Thesis, South Dakota State University. Accessed February 27, 2021. https://openprairie.sdstate.edu/etd/2942.

MLA Handbook (7th Edition):

Uprety, Tirth. “Role of Bovine Ileal Sub-epithelial Myofibroblasts and Epithelial Cells in Innate Immunity.” 2018. Web. 27 Feb 2021.

Vancouver:

Uprety T. Role of Bovine Ileal Sub-epithelial Myofibroblasts and Epithelial Cells in Innate Immunity. [Internet] [Masters thesis]. South Dakota State University; 2018. [cited 2021 Feb 27]. Available from: https://openprairie.sdstate.edu/etd/2942.

Council of Science Editors:

Uprety T. Role of Bovine Ileal Sub-epithelial Myofibroblasts and Epithelial Cells in Innate Immunity. [Masters Thesis]. South Dakota State University; 2018. Available from: https://openprairie.sdstate.edu/etd/2942


Loyola University Chicago

28. Zook, Erin Christine. Promoting Thymopoiesis with Age: Potential Role of the Transcription Factor Foxn1.

Degree: PhD, Cell Biology, Neurobiology and Anatomy, 2012, Loyola University Chicago

  It is known that the elderly are more susceptible to illnesses and infections and respond poorly to immunization. A contributing factor to a decrease… (more)

Subjects/Keywords: Aging; Foxn1; Thymus; Immunology and Infectious Disease

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APA (6th Edition):

Zook, E. C. (2012). Promoting Thymopoiesis with Age: Potential Role of the Transcription Factor Foxn1. (Doctoral Dissertation). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_diss/317

Chicago Manual of Style (16th Edition):

Zook, Erin Christine. “Promoting Thymopoiesis with Age: Potential Role of the Transcription Factor Foxn1.” 2012. Doctoral Dissertation, Loyola University Chicago. Accessed February 27, 2021. https://ecommons.luc.edu/luc_diss/317.

MLA Handbook (7th Edition):

Zook, Erin Christine. “Promoting Thymopoiesis with Age: Potential Role of the Transcription Factor Foxn1.” 2012. Web. 27 Feb 2021.

Vancouver:

Zook EC. Promoting Thymopoiesis with Age: Potential Role of the Transcription Factor Foxn1. [Internet] [Doctoral dissertation]. Loyola University Chicago; 2012. [cited 2021 Feb 27]. Available from: https://ecommons.luc.edu/luc_diss/317.

Council of Science Editors:

Zook EC. Promoting Thymopoiesis with Age: Potential Role of the Transcription Factor Foxn1. [Doctoral Dissertation]. Loyola University Chicago; 2012. Available from: https://ecommons.luc.edu/luc_diss/317


Loyola University Chicago

29. Fleming-Trujillo, Erica. Modulating the Tumor Microenvironment to Induce Cross-Priming for Cancer Immunotherapy.

Degree: PhD, Microbiology and Immunology, 2019, Loyola University Chicago

  Adoptive cell transfer (ACT) using T cells engineered to express tumor-specific T cell receptors (TCR) holds great promise in treating cancer patients. ACT involves… (more)

Subjects/Keywords: Cancer; Immunotherapy; Immunology of Infectious Disease

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APA (6th Edition):

Fleming-Trujillo, E. (2019). Modulating the Tumor Microenvironment to Induce Cross-Priming for Cancer Immunotherapy. (Doctoral Dissertation). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_diss/3332

Chicago Manual of Style (16th Edition):

Fleming-Trujillo, Erica. “Modulating the Tumor Microenvironment to Induce Cross-Priming for Cancer Immunotherapy.” 2019. Doctoral Dissertation, Loyola University Chicago. Accessed February 27, 2021. https://ecommons.luc.edu/luc_diss/3332.

MLA Handbook (7th Edition):

Fleming-Trujillo, Erica. “Modulating the Tumor Microenvironment to Induce Cross-Priming for Cancer Immunotherapy.” 2019. Web. 27 Feb 2021.

Vancouver:

Fleming-Trujillo E. Modulating the Tumor Microenvironment to Induce Cross-Priming for Cancer Immunotherapy. [Internet] [Doctoral dissertation]. Loyola University Chicago; 2019. [cited 2021 Feb 27]. Available from: https://ecommons.luc.edu/luc_diss/3332.

Council of Science Editors:

Fleming-Trujillo E. Modulating the Tumor Microenvironment to Induce Cross-Priming for Cancer Immunotherapy. [Doctoral Dissertation]. Loyola University Chicago; 2019. Available from: https://ecommons.luc.edu/luc_diss/3332

30. Tranchemontagne, Zachary Ronald. Community-Acquired Methicillin-Resistant Staphylococcus Aureus (CA-MRSA) USA300 Perturbs Acquisition Of Lysosomal Hydrolases And Requires Phagosomal Acidification For Survival In A Human Macrophage Cell Line.

Degree: MSin Biological Sciences, Biological Science, 2015, University of New England

  Community-acquired Methicillin-resistant Staphylococcus aureus (CA-MRSA) strain USA300 is a major cause of invasive drug-resistant skin and soft tissue infections in humans. Although S. aureus… (more)

Subjects/Keywords: Biology; Immunology and Infectious Disease; Microbiology

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APA (6th Edition):

Tranchemontagne, Z. R. (2015). Community-Acquired Methicillin-Resistant Staphylococcus Aureus (CA-MRSA) USA300 Perturbs Acquisition Of Lysosomal Hydrolases And Requires Phagosomal Acidification For Survival In A Human Macrophage Cell Line. (Thesis). University of New England. Retrieved from https://dune.une.edu/theses/99

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tranchemontagne, Zachary Ronald. “Community-Acquired Methicillin-Resistant Staphylococcus Aureus (CA-MRSA) USA300 Perturbs Acquisition Of Lysosomal Hydrolases And Requires Phagosomal Acidification For Survival In A Human Macrophage Cell Line.” 2015. Thesis, University of New England. Accessed February 27, 2021. https://dune.une.edu/theses/99.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tranchemontagne, Zachary Ronald. “Community-Acquired Methicillin-Resistant Staphylococcus Aureus (CA-MRSA) USA300 Perturbs Acquisition Of Lysosomal Hydrolases And Requires Phagosomal Acidification For Survival In A Human Macrophage Cell Line.” 2015. Web. 27 Feb 2021.

Vancouver:

Tranchemontagne ZR. Community-Acquired Methicillin-Resistant Staphylococcus Aureus (CA-MRSA) USA300 Perturbs Acquisition Of Lysosomal Hydrolases And Requires Phagosomal Acidification For Survival In A Human Macrophage Cell Line. [Internet] [Thesis]. University of New England; 2015. [cited 2021 Feb 27]. Available from: https://dune.une.edu/theses/99.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tranchemontagne ZR. Community-Acquired Methicillin-Resistant Staphylococcus Aureus (CA-MRSA) USA300 Perturbs Acquisition Of Lysosomal Hydrolases And Requires Phagosomal Acidification For Survival In A Human Macrophage Cell Line. [Thesis]. University of New England; 2015. Available from: https://dune.une.edu/theses/99

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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