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You searched for subject:(IL 22). Showing records 1 – 30 of 31 total matches.

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Université Catholique de Louvain

1. Cochez, Perrine. AhR and Ccr6 are not required for IL-22 production in the imiquimod-induced psoriasis model.

Degree: 2016, Université Catholique de Louvain

IL-22 is a crucial cytokine in skin homeostasis by its action on keratinocytes. It induces the production of antimicrobial peptides, promotes epithelial regeneration and therefore… (more)

Subjects/Keywords: IL-22; AhR; Ccr6; Psoriasis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cochez, P. (2016). AhR and Ccr6 are not required for IL-22 production in the imiquimod-induced psoriasis model. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/176811

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cochez, Perrine. “AhR and Ccr6 are not required for IL-22 production in the imiquimod-induced psoriasis model.” 2016. Thesis, Université Catholique de Louvain. Accessed October 22, 2019. http://hdl.handle.net/2078.1/176811.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cochez, Perrine. “AhR and Ccr6 are not required for IL-22 production in the imiquimod-induced psoriasis model.” 2016. Web. 22 Oct 2019.

Vancouver:

Cochez P. AhR and Ccr6 are not required for IL-22 production in the imiquimod-induced psoriasis model. [Internet] [Thesis]. Université Catholique de Louvain; 2016. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/2078.1/176811.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cochez P. AhR and Ccr6 are not required for IL-22 production in the imiquimod-induced psoriasis model. [Thesis]. Université Catholique de Louvain; 2016. Available from: http://hdl.handle.net/2078.1/176811

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Segueni, Noria. Etude des relations hôte-pathogène lors de l’infection par Mycobacterium tuberculosis : implication des voies de signalisation IL-36, TNF et IL-17/IL-22 : Host-pathogen interactions during Mycobacterium tuberculosis infection : role of IL-36, TNF and IL-17/IL-22 pathways.

Degree: Docteur es, Biologie moléculaire et cellulaire. Immunologie, 2015, Université d'Orléans

La tuberculose est encore aujourd’hui un problème de santé majeur et l’augmentation des cas de tuberculoses résistantes au niveau mondial, associé au dépistage et diagnostic… (more)

Subjects/Keywords: Mycobacterium tuberculosis; IL-17; IL-22; IL-36; TNF; Anticorps; Mycobacterium tuberculosis; IL-17; IL-22; IL-36; TNF; Antibodies; 571.96

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APA (6th Edition):

Segueni, N. (2015). Etude des relations hôte-pathogène lors de l’infection par Mycobacterium tuberculosis : implication des voies de signalisation IL-36, TNF et IL-17/IL-22 : Host-pathogen interactions during Mycobacterium tuberculosis infection : role of IL-36, TNF and IL-17/IL-22 pathways. (Doctoral Dissertation). Université d'Orléans. Retrieved from http://www.theses.fr/2015ORLE2075

Chicago Manual of Style (16th Edition):

Segueni, Noria. “Etude des relations hôte-pathogène lors de l’infection par Mycobacterium tuberculosis : implication des voies de signalisation IL-36, TNF et IL-17/IL-22 : Host-pathogen interactions during Mycobacterium tuberculosis infection : role of IL-36, TNF and IL-17/IL-22 pathways.” 2015. Doctoral Dissertation, Université d'Orléans. Accessed October 22, 2019. http://www.theses.fr/2015ORLE2075.

MLA Handbook (7th Edition):

Segueni, Noria. “Etude des relations hôte-pathogène lors de l’infection par Mycobacterium tuberculosis : implication des voies de signalisation IL-36, TNF et IL-17/IL-22 : Host-pathogen interactions during Mycobacterium tuberculosis infection : role of IL-36, TNF and IL-17/IL-22 pathways.” 2015. Web. 22 Oct 2019.

Vancouver:

Segueni N. Etude des relations hôte-pathogène lors de l’infection par Mycobacterium tuberculosis : implication des voies de signalisation IL-36, TNF et IL-17/IL-22 : Host-pathogen interactions during Mycobacterium tuberculosis infection : role of IL-36, TNF and IL-17/IL-22 pathways. [Internet] [Doctoral dissertation]. Université d'Orléans; 2015. [cited 2019 Oct 22]. Available from: http://www.theses.fr/2015ORLE2075.

Council of Science Editors:

Segueni N. Etude des relations hôte-pathogène lors de l’infection par Mycobacterium tuberculosis : implication des voies de signalisation IL-36, TNF et IL-17/IL-22 : Host-pathogen interactions during Mycobacterium tuberculosis infection : role of IL-36, TNF and IL-17/IL-22 pathways. [Doctoral Dissertation]. Université d'Orléans; 2015. Available from: http://www.theses.fr/2015ORLE2075

3. Michaudel, Chloé. Pollution à l'ozone : maintien de la barrière pulmonaire via l'IL-33, implication des autres membres de la famille IL-1 et régulation cytokinique via AhR : Ozone pollution : pulmonary barrier maintenance via IL-33, involvement of IL-1 family members and AhR-dependent cytokine regulation.

Degree: Docteur es, Biologie des sciences du vivant et immunologie, 2017, Université d'Orléans

L’ozone est un des polluants présents dans l’air que nous respirons. Les pics de ce polluant entrainent une augmentation des hospitalisations et des cas d’exacerbations… (more)

Subjects/Keywords: Ozone; Inflammation; IL-33; IL-1α; IL-22; IL-17A; AhR; Ozone; Inflammation; IL-33; IL-1α; IL-22; IL-17A; AhR; 571.96

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APA (6th Edition):

Michaudel, C. (2017). Pollution à l'ozone : maintien de la barrière pulmonaire via l'IL-33, implication des autres membres de la famille IL-1 et régulation cytokinique via AhR : Ozone pollution : pulmonary barrier maintenance via IL-33, involvement of IL-1 family members and AhR-dependent cytokine regulation. (Doctoral Dissertation). Université d'Orléans. Retrieved from http://www.theses.fr/2017ORLE2040

Chicago Manual of Style (16th Edition):

Michaudel, Chloé. “Pollution à l'ozone : maintien de la barrière pulmonaire via l'IL-33, implication des autres membres de la famille IL-1 et régulation cytokinique via AhR : Ozone pollution : pulmonary barrier maintenance via IL-33, involvement of IL-1 family members and AhR-dependent cytokine regulation.” 2017. Doctoral Dissertation, Université d'Orléans. Accessed October 22, 2019. http://www.theses.fr/2017ORLE2040.

MLA Handbook (7th Edition):

Michaudel, Chloé. “Pollution à l'ozone : maintien de la barrière pulmonaire via l'IL-33, implication des autres membres de la famille IL-1 et régulation cytokinique via AhR : Ozone pollution : pulmonary barrier maintenance via IL-33, involvement of IL-1 family members and AhR-dependent cytokine regulation.” 2017. Web. 22 Oct 2019.

Vancouver:

Michaudel C. Pollution à l'ozone : maintien de la barrière pulmonaire via l'IL-33, implication des autres membres de la famille IL-1 et régulation cytokinique via AhR : Ozone pollution : pulmonary barrier maintenance via IL-33, involvement of IL-1 family members and AhR-dependent cytokine regulation. [Internet] [Doctoral dissertation]. Université d'Orléans; 2017. [cited 2019 Oct 22]. Available from: http://www.theses.fr/2017ORLE2040.

Council of Science Editors:

Michaudel C. Pollution à l'ozone : maintien de la barrière pulmonaire via l'IL-33, implication des autres membres de la famille IL-1 et régulation cytokinique via AhR : Ozone pollution : pulmonary barrier maintenance via IL-33, involvement of IL-1 family members and AhR-dependent cytokine regulation. [Doctoral Dissertation]. Université d'Orléans; 2017. Available from: http://www.theses.fr/2017ORLE2040

4. Oliveira Neto, Mario de. Espalhamento de raios-X a baixo ângulo aplicado ao estudo estrutural de proteínas.

Degree: PhD, Física Aplicada, 2008, University of São Paulo

O espalhamento de raios X a baixo ângulo tem se mostrado uma poderosa ferramenta na ánalise estrutural de proteínas em solução. Estudos em condições próximas… (more)

Subjects/Keywords: SAXS. nuclear receptor. IL-22/IL-22R1. ferredoxin; SAXS. receptores nucleares. IL-22/IL-22R1. ferredoxina.

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APA (6th Edition):

Oliveira Neto, M. d. (2008). Espalhamento de raios-X a baixo ângulo aplicado ao estudo estrutural de proteínas. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/76/76132/tde-18112008-132202/ ;

Chicago Manual of Style (16th Edition):

Oliveira Neto, Mario de. “Espalhamento de raios-X a baixo ângulo aplicado ao estudo estrutural de proteínas.” 2008. Doctoral Dissertation, University of São Paulo. Accessed October 22, 2019. http://www.teses.usp.br/teses/disponiveis/76/76132/tde-18112008-132202/ ;.

MLA Handbook (7th Edition):

Oliveira Neto, Mario de. “Espalhamento de raios-X a baixo ângulo aplicado ao estudo estrutural de proteínas.” 2008. Web. 22 Oct 2019.

Vancouver:

Oliveira Neto Md. Espalhamento de raios-X a baixo ângulo aplicado ao estudo estrutural de proteínas. [Internet] [Doctoral dissertation]. University of São Paulo; 2008. [cited 2019 Oct 22]. Available from: http://www.teses.usp.br/teses/disponiveis/76/76132/tde-18112008-132202/ ;.

Council of Science Editors:

Oliveira Neto Md. Espalhamento de raios-X a baixo ângulo aplicado ao estudo estrutural de proteínas. [Doctoral Dissertation]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/76/76132/tde-18112008-132202/ ;


Virginia Tech

5. Kim, Sungwon. Functional Characterization of the Avian Inflammatory Mediators Nod1, MIF and IL-22.

Degree: PhD, Animal and Poultry Sciences, 2011, Virginia Tech

 Inflammation can be initiated by an innate immune sensor, followed by activation of a signal mediator, resulting in control of immune response by a signal… (more)

Subjects/Keywords: Nod1; MIF; IL-22; inflammation; cytokines

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APA (6th Edition):

Kim, S. (2011). Functional Characterization of the Avian Inflammatory Mediators Nod1, MIF and IL-22. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/77232

Chicago Manual of Style (16th Edition):

Kim, Sungwon. “Functional Characterization of the Avian Inflammatory Mediators Nod1, MIF and IL-22.” 2011. Doctoral Dissertation, Virginia Tech. Accessed October 22, 2019. http://hdl.handle.net/10919/77232.

MLA Handbook (7th Edition):

Kim, Sungwon. “Functional Characterization of the Avian Inflammatory Mediators Nod1, MIF and IL-22.” 2011. Web. 22 Oct 2019.

Vancouver:

Kim S. Functional Characterization of the Avian Inflammatory Mediators Nod1, MIF and IL-22. [Internet] [Doctoral dissertation]. Virginia Tech; 2011. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/10919/77232.

Council of Science Editors:

Kim S. Functional Characterization of the Avian Inflammatory Mediators Nod1, MIF and IL-22. [Doctoral Dissertation]. Virginia Tech; 2011. Available from: http://hdl.handle.net/10919/77232

6. 조, 옥기. The Effect of Endocytosis of Interferon-lambda and The Genotype of IL-10RB on The Cellular Responses to Cytokines.

Degree: 2010, Ajou University

"Endocytosis pathway는 세포내 신호전달과 기능에 관련되므로 중요하게 연구 되고있다. Endocytosis는 크게 clathrin-dependent/-independent, caveolae-dependent /-independent pathway와 그 외의 다른 pathway들로 분류 된다. 또한 dynamin II 관련에… (more)

Subjects/Keywords: IFN-lambda; HBV; IL-10R2; IL-22; endocytosis; cholesterol; dynamin

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APA (6th Edition):

조, . (2010). The Effect of Endocytosis of Interferon-lambda and The Genotype of IL-10RB on The Cellular Responses to Cytokines. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/1762 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010565

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

조, 옥기. “The Effect of Endocytosis of Interferon-lambda and The Genotype of IL-10RB on The Cellular Responses to Cytokines.” 2010. Thesis, Ajou University. Accessed October 22, 2019. http://repository.ajou.ac.kr/handle/201003/1762 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010565.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

조, 옥기. “The Effect of Endocytosis of Interferon-lambda and The Genotype of IL-10RB on The Cellular Responses to Cytokines.” 2010. Web. 22 Oct 2019.

Vancouver:

조 . The Effect of Endocytosis of Interferon-lambda and The Genotype of IL-10RB on The Cellular Responses to Cytokines. [Internet] [Thesis]. Ajou University; 2010. [cited 2019 Oct 22]. Available from: http://repository.ajou.ac.kr/handle/201003/1762 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010565.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

조 . The Effect of Endocytosis of Interferon-lambda and The Genotype of IL-10RB on The Cellular Responses to Cytokines. [Thesis]. Ajou University; 2010. Available from: http://repository.ajou.ac.kr/handle/201003/1762 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000010565

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Sertorio, Mathieu. Contribution à l'analyse immunogénétique de la susceptibilité aux bilharzioses hépatospléniques : The service offshoring phenomenon : a theoretical approach and empirical : an application to the countries development.

Degree: Docteur es, Pathologie humaine, 2012, Aix Marseille Université

Les bilharzioses hépatospléniques sont des maladies parasitaires provoquées essentiellement par Schistosoma japonicum et Schistosoma mansoni. Ces infections provoquent une fibrose hépatique sévère chez 5 à… (more)

Subjects/Keywords: Bilharziose; Fibrose Hépatique; Il-22; Génétique; Immunologie; Vaccin; Schistosomiasis; Hepatic Fibrosis; Il-22; Genetic, Immunology, Vaccine

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APA (6th Edition):

Sertorio, M. (2012). Contribution à l'analyse immunogénétique de la susceptibilité aux bilharzioses hépatospléniques : The service offshoring phenomenon : a theoretical approach and empirical : an application to the countries development. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2012AIXM5001

Chicago Manual of Style (16th Edition):

Sertorio, Mathieu. “Contribution à l'analyse immunogénétique de la susceptibilité aux bilharzioses hépatospléniques : The service offshoring phenomenon : a theoretical approach and empirical : an application to the countries development.” 2012. Doctoral Dissertation, Aix Marseille Université. Accessed October 22, 2019. http://www.theses.fr/2012AIXM5001.

MLA Handbook (7th Edition):

Sertorio, Mathieu. “Contribution à l'analyse immunogénétique de la susceptibilité aux bilharzioses hépatospléniques : The service offshoring phenomenon : a theoretical approach and empirical : an application to the countries development.” 2012. Web. 22 Oct 2019.

Vancouver:

Sertorio M. Contribution à l'analyse immunogénétique de la susceptibilité aux bilharzioses hépatospléniques : The service offshoring phenomenon : a theoretical approach and empirical : an application to the countries development. [Internet] [Doctoral dissertation]. Aix Marseille Université 2012. [cited 2019 Oct 22]. Available from: http://www.theses.fr/2012AIXM5001.

Council of Science Editors:

Sertorio M. Contribution à l'analyse immunogénétique de la susceptibilité aux bilharzioses hépatospléniques : The service offshoring phenomenon : a theoretical approach and empirical : an application to the countries development. [Doctoral Dissertation]. Aix Marseille Université 2012. Available from: http://www.theses.fr/2012AIXM5001

8. Picard, Marion. Étude de l'implication d’une voie MyD88/IL-22 dans le contrôle de la colonisation par la bactérie segmentée filamenteuse : Implication of a MyD88/IL-22 pathway in the control of colonisation by segmented filamentous bacteria.

Degree: Docteur es, Immunologie, 2017, Sorbonne Paris Cité

L’intestin des mammifères est colonisé par une dense communauté microbienne avec lequel il a établi, au cours d’une longue co-évolution, des relations mutualistes. Ces relations,… (more)

Subjects/Keywords: SFB; Intestin; TLR; IL-22; ILC; Peptides anti-microbiens; Microbiote; SFB; Intestine; TLR; IL-22; ILC; Antimicrobials peptides; Microbiota; 571.96

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APA (6th Edition):

Picard, M. (2017). Étude de l'implication d’une voie MyD88/IL-22 dans le contrôle de la colonisation par la bactérie segmentée filamenteuse : Implication of a MyD88/IL-22 pathway in the control of colonisation by segmented filamentous bacteria. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2017USPCB107

Chicago Manual of Style (16th Edition):

Picard, Marion. “Étude de l'implication d’une voie MyD88/IL-22 dans le contrôle de la colonisation par la bactérie segmentée filamenteuse : Implication of a MyD88/IL-22 pathway in the control of colonisation by segmented filamentous bacteria.” 2017. Doctoral Dissertation, Sorbonne Paris Cité. Accessed October 22, 2019. http://www.theses.fr/2017USPCB107.

MLA Handbook (7th Edition):

Picard, Marion. “Étude de l'implication d’une voie MyD88/IL-22 dans le contrôle de la colonisation par la bactérie segmentée filamenteuse : Implication of a MyD88/IL-22 pathway in the control of colonisation by segmented filamentous bacteria.” 2017. Web. 22 Oct 2019.

Vancouver:

Picard M. Étude de l'implication d’une voie MyD88/IL-22 dans le contrôle de la colonisation par la bactérie segmentée filamenteuse : Implication of a MyD88/IL-22 pathway in the control of colonisation by segmented filamentous bacteria. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2017. [cited 2019 Oct 22]. Available from: http://www.theses.fr/2017USPCB107.

Council of Science Editors:

Picard M. Étude de l'implication d’une voie MyD88/IL-22 dans le contrôle de la colonisation par la bactérie segmentée filamenteuse : Implication of a MyD88/IL-22 pathway in the control of colonisation by segmented filamentous bacteria. [Doctoral Dissertation]. Sorbonne Paris Cité; 2017. Available from: http://www.theses.fr/2017USPCB107


The Ohio State University

9. Victor, Aaron Robert. Regulation of IL-22 Production by Immature Natural Killer Cells and CD16 Expression during their Maturation.

Degree: PhD, Integrated Biomedical Science Graduate Program, 2016, The Ohio State University

 Natural killer (NK) cells are innate immune cells that play different roles depending on their stage of development. NK cells develop through five stages that… (more)

Subjects/Keywords: Biomedical Research; natural killer cells; innate immunity; cell development; IL-18; IL-22; CD16; FCGR3A

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APA (6th Edition):

Victor, A. R. (2016). Regulation of IL-22 Production by Immature Natural Killer Cells and CD16 Expression during their Maturation. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1460744760

Chicago Manual of Style (16th Edition):

Victor, Aaron Robert. “Regulation of IL-22 Production by Immature Natural Killer Cells and CD16 Expression during their Maturation.” 2016. Doctoral Dissertation, The Ohio State University. Accessed October 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1460744760.

MLA Handbook (7th Edition):

Victor, Aaron Robert. “Regulation of IL-22 Production by Immature Natural Killer Cells and CD16 Expression during their Maturation.” 2016. Web. 22 Oct 2019.

Vancouver:

Victor AR. Regulation of IL-22 Production by Immature Natural Killer Cells and CD16 Expression during their Maturation. [Internet] [Doctoral dissertation]. The Ohio State University; 2016. [cited 2019 Oct 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1460744760.

Council of Science Editors:

Victor AR. Regulation of IL-22 Production by Immature Natural Killer Cells and CD16 Expression during their Maturation. [Doctoral Dissertation]. The Ohio State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1460744760


Loyola University Chicago

10. Rendon, Juan L. Effects of Acute Alcohol Exposure on Post Burn Intestinal Immunity: Role of IL-23.

Degree: PhD, Cell Biology, Neurobiology and Anatomy, 2013, Loyola University Chicago

  More than one million burn injuries are reported yearly within the United States. These injuries result in approximately 500,000 emergency room visits and 40,000… (more)

Subjects/Keywords: Burn; Ethanol; IL-17; IL-22; Peyer's patch; T cell; Cell and Developmental Biology

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APA (6th Edition):

Rendon, J. L. (2013). Effects of Acute Alcohol Exposure on Post Burn Intestinal Immunity: Role of IL-23. (Doctoral Dissertation). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_diss/543

Chicago Manual of Style (16th Edition):

Rendon, Juan L. “Effects of Acute Alcohol Exposure on Post Burn Intestinal Immunity: Role of IL-23.” 2013. Doctoral Dissertation, Loyola University Chicago. Accessed October 22, 2019. https://ecommons.luc.edu/luc_diss/543.

MLA Handbook (7th Edition):

Rendon, Juan L. “Effects of Acute Alcohol Exposure on Post Burn Intestinal Immunity: Role of IL-23.” 2013. Web. 22 Oct 2019.

Vancouver:

Rendon JL. Effects of Acute Alcohol Exposure on Post Burn Intestinal Immunity: Role of IL-23. [Internet] [Doctoral dissertation]. Loyola University Chicago; 2013. [cited 2019 Oct 22]. Available from: https://ecommons.luc.edu/luc_diss/543.

Council of Science Editors:

Rendon JL. Effects of Acute Alcohol Exposure on Post Burn Intestinal Immunity: Role of IL-23. [Doctoral Dissertation]. Loyola University Chicago; 2013. Available from: https://ecommons.luc.edu/luc_diss/543


University of Texas Medical Branch – Galveston

11. [No author]. Innate Interleukin-17 and Interleukin-22 play important roles in viral hepatitis .

Degree: University of Texas Medical Branch – Galveston

 Intrahepatic cell-derived IL-17 and IL-22 are important for T cell priming and hepatoprotection in viral hepatitis, respectively; however, the source and regulation of these cytokines… (more)

Subjects/Keywords: intrahepatic; IL-17; IL-22; hepatitis

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APA (6th Edition):

author], [. (n.d.). Innate Interleukin-17 and Interleukin-22 play important roles in viral hepatitis . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/700

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Innate Interleukin-17 and Interleukin-22 play important roles in viral hepatitis .” Thesis, University of Texas Medical Branch – Galveston. Accessed October 22, 2019. http://hdl.handle.net/2152.3/700.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Innate Interleukin-17 and Interleukin-22 play important roles in viral hepatitis .” Web. 22 Oct 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

author] [. Innate Interleukin-17 and Interleukin-22 play important roles in viral hepatitis . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; [cited 2019 Oct 22]. Available from: http://hdl.handle.net/2152.3/700.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

author] [. Innate Interleukin-17 and Interleukin-22 play important roles in viral hepatitis . [Thesis]. University of Texas Medical Branch – Galveston; Available from: http://hdl.handle.net/2152.3/700

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Universiteit Utrecht

12. Coppens, C.J.C. Role of Th17 cells in health and disease.

Degree: 2011, Universiteit Utrecht

 Th17 cells are a recently discovered novel subset of helper T cells. Th17 cells differentiate along a different lineage than Th1 and Th2 cells. Already… (more)

Subjects/Keywords: Th17; regulation; function; helper T; IL-17; IL-6; TGF-β; IL-21; IL-23; STAT3; RORγt; RORα; IL-22; neutrophil; extracellular; bacteria; fungi; autoimmune; allergy; cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Coppens, C. J. C. (2011). Role of Th17 cells in health and disease. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/193877

Chicago Manual of Style (16th Edition):

Coppens, C J C. “Role of Th17 cells in health and disease.” 2011. Masters Thesis, Universiteit Utrecht. Accessed October 22, 2019. http://dspace.library.uu.nl:8080/handle/1874/193877.

MLA Handbook (7th Edition):

Coppens, C J C. “Role of Th17 cells in health and disease.” 2011. Web. 22 Oct 2019.

Vancouver:

Coppens CJC. Role of Th17 cells in health and disease. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2019 Oct 22]. Available from: http://dspace.library.uu.nl:8080/handle/1874/193877.

Council of Science Editors:

Coppens CJC. Role of Th17 cells in health and disease. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/193877


University of Manitoba

13. DRAGON, Stephane. Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils.

Degree: Immunology, 2010, University of Manitoba

 Immunopathological disorders are no longer defined by dysregulated T-helper (Th) type 1/ Th2 responses but account for modulatory cell types such as regulatory and Th17… (more)

Subjects/Keywords: Human; Inflammation; Cytokines; IL-17; IL-1beta; IL-22; CXCL-8; airway smooth muscle cells; neutrophils; IL-17R; signal transduction; gene expression; apoptosis

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APA (6th Edition):

DRAGON, S. (2010). Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/3945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DRAGON, Stephane. “Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils.” 2010. Thesis, University of Manitoba. Accessed October 22, 2019. http://hdl.handle.net/1993/3945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DRAGON, Stephane. “Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils.” 2010. Web. 22 Oct 2019.

Vancouver:

DRAGON S. Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils. [Internet] [Thesis]. University of Manitoba; 2010. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/1993/3945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DRAGON S. Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils. [Thesis]. University of Manitoba; 2010. Available from: http://hdl.handle.net/1993/3945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Jönköping University

14. Andersson, Lisa. Expertskattens förutsägbarhet.

Degree: Commercial Law, 2011, Jönköping University

  In 1984 the Swedish government introduced a special law which gave tax benefits to foreign researchers who for a limited time was engaged in… (more)

Subjects/Keywords: Expertskatt; förutsägbarhet; 11:22-23a IL; Law; Juridik; Law and Society; Juridik och samhälle

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APA (6th Edition):

Andersson, L. (2011). Expertskattens förutsägbarhet. (Thesis). Jönköping University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-15052

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Andersson, Lisa. “Expertskattens förutsägbarhet.” 2011. Thesis, Jönköping University. Accessed October 22, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-15052.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Andersson, Lisa. “Expertskattens förutsägbarhet.” 2011. Web. 22 Oct 2019.

Vancouver:

Andersson L. Expertskattens förutsägbarhet. [Internet] [Thesis]. Jönköping University; 2011. [cited 2019 Oct 22]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-15052.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Andersson L. Expertskattens förutsägbarhet. [Thesis]. Jönköping University; 2011. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-15052

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

15. Morgan, Nadine N. Characterization of Il-22-Producing Cells in the Human Thymus and the Mechanism by Which Il-22 Expression Is Modulated by Thymic Epithelial Cells.

Degree: MS, Microbiology and Immunology, 2016, Loyola University Chicago

  Thymic epithelial cells (TECs) are indispensable for T cell development and maturation. Therefore, damages to the thymic epithelial cells are detrimental to thymic function… (more)

Subjects/Keywords: IL-22; ILC3s; Innate lymphoid cells; Regeneration; Thymic epithelial cells; Thymus; Immunology and Infectious Disease

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APA (6th Edition):

Morgan, N. N. (2016). Characterization of Il-22-Producing Cells in the Human Thymus and the Mechanism by Which Il-22 Expression Is Modulated by Thymic Epithelial Cells. (Thesis). Loyola University Chicago. Retrieved from http://ecommons.luc.edu/luc_theses/3564

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Morgan, Nadine N. “Characterization of Il-22-Producing Cells in the Human Thymus and the Mechanism by Which Il-22 Expression Is Modulated by Thymic Epithelial Cells.” 2016. Thesis, Loyola University Chicago. Accessed October 22, 2019. http://ecommons.luc.edu/luc_theses/3564.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Morgan, Nadine N. “Characterization of Il-22-Producing Cells in the Human Thymus and the Mechanism by Which Il-22 Expression Is Modulated by Thymic Epithelial Cells.” 2016. Web. 22 Oct 2019.

Vancouver:

Morgan NN. Characterization of Il-22-Producing Cells in the Human Thymus and the Mechanism by Which Il-22 Expression Is Modulated by Thymic Epithelial Cells. [Internet] [Thesis]. Loyola University Chicago; 2016. [cited 2019 Oct 22]. Available from: http://ecommons.luc.edu/luc_theses/3564.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Morgan NN. Characterization of Il-22-Producing Cells in the Human Thymus and the Mechanism by Which Il-22 Expression Is Modulated by Thymic Epithelial Cells. [Thesis]. Loyola University Chicago; 2016. Available from: http://ecommons.luc.edu/luc_theses/3564

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Helsinki

16. Kilpeläinen, Niko. The effects of interleukin-22 and -17 on beta-cell destruction in vitro.

Degree: Department of Food and Environmental Sciences; Helsingfors universitet, Agrikultur- och forstvetenskapliga fakulteten, Institutionen för livsmedels- och miljövetenskaper, 2014, University of Helsinki

 Type 1 diabetes (T1D) is a chronic autoimmune disease in which insulin-producing beta-cells in pancreatic islets are destroyed by the body's own immune system. Patients… (more)

Subjects/Keywords: T1D; beta-cell; Th17; IL-17; IL-22; MIN6; human pancreatic islet; apoptosis; Bioteknik (EYT); Biotechnology (EYT); Biotekniikka (EYT); T1D; beta-cell; Th17; IL-17; IL-22; MIN6; human pancreatic islet; apoptosis

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APA (6th Edition):

Kilpeläinen, N. (2014). The effects of interleukin-22 and -17 on beta-cell destruction in vitro. (Masters Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/135939

Chicago Manual of Style (16th Edition):

Kilpeläinen, Niko. “The effects of interleukin-22 and -17 on beta-cell destruction in vitro.” 2014. Masters Thesis, University of Helsinki. Accessed October 22, 2019. http://hdl.handle.net/10138/135939.

MLA Handbook (7th Edition):

Kilpeläinen, Niko. “The effects of interleukin-22 and -17 on beta-cell destruction in vitro.” 2014. Web. 22 Oct 2019.

Vancouver:

Kilpeläinen N. The effects of interleukin-22 and -17 on beta-cell destruction in vitro. [Internet] [Masters thesis]. University of Helsinki; 2014. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/10138/135939.

Council of Science Editors:

Kilpeläinen N. The effects of interleukin-22 and -17 on beta-cell destruction in vitro. [Masters Thesis]. University of Helsinki; 2014. Available from: http://hdl.handle.net/10138/135939

17. Koné, Bachirou. Mécanismes cellulaires et moléculaires de la susceptibilité à l'infection au cours de la bronchopneumopathie chronique obstructive (BPCO) : Cellular and molecular mechanisms of susceptibility to infection in chronic obstructive pulmonary disease (COPD).

Degree: Docteur es, Immunologie, 2017, Université Lille II – Droit et Santé

 La BPCO se traduit rapidement par l'apparition d'une susceptibilité aux infections liées aux atteintes des mécanismes de défense du poumon. Les travaux antérieurs de l’équipe… (more)

Subjects/Keywords: Broncho-pneumopathie chronique obstructive; BPCO; Exacerbation; Streptococcus pneumoniae; Haemophilus influenzae non typable; Flagelline; Cytokines IL-20; IL-17/22; Chronic obstructive pulmonary disease; COPD; Exacerbation; Streptococcus pneumoniae; Non typable Haemophilus influenza; Flagellin; IL-20 cytokines; IL-17/22

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APA (6th Edition):

Koné, B. (2017). Mécanismes cellulaires et moléculaires de la susceptibilité à l'infection au cours de la bronchopneumopathie chronique obstructive (BPCO) : Cellular and molecular mechanisms of susceptibility to infection in chronic obstructive pulmonary disease (COPD). (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2017LIL2S028

Chicago Manual of Style (16th Edition):

Koné, Bachirou. “Mécanismes cellulaires et moléculaires de la susceptibilité à l'infection au cours de la bronchopneumopathie chronique obstructive (BPCO) : Cellular and molecular mechanisms of susceptibility to infection in chronic obstructive pulmonary disease (COPD).” 2017. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed October 22, 2019. http://www.theses.fr/2017LIL2S028.

MLA Handbook (7th Edition):

Koné, Bachirou. “Mécanismes cellulaires et moléculaires de la susceptibilité à l'infection au cours de la bronchopneumopathie chronique obstructive (BPCO) : Cellular and molecular mechanisms of susceptibility to infection in chronic obstructive pulmonary disease (COPD).” 2017. Web. 22 Oct 2019.

Vancouver:

Koné B. Mécanismes cellulaires et moléculaires de la susceptibilité à l'infection au cours de la bronchopneumopathie chronique obstructive (BPCO) : Cellular and molecular mechanisms of susceptibility to infection in chronic obstructive pulmonary disease (COPD). [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2017. [cited 2019 Oct 22]. Available from: http://www.theses.fr/2017LIL2S028.

Council of Science Editors:

Koné B. Mécanismes cellulaires et moléculaires de la susceptibilité à l'infection au cours de la bronchopneumopathie chronique obstructive (BPCO) : Cellular and molecular mechanisms of susceptibility to infection in chronic obstructive pulmonary disease (COPD). [Doctoral Dissertation]. Université Lille II – Droit et Santé 2017. Available from: http://www.theses.fr/2017LIL2S028

18. VILLAR, Kamila de Melo. Associação de polimorfismos no gene IL22RA1 como os níveis séricos de IL-22 e com parâmetros clínicos de pacientes portadores de artrite reumatoide .

Degree: 2015, Universidade Federal de Pernambuco

 O processo inflamatório associado à liberação de citocinas está diretamente envolvido na patogênese da artrite reumatóide (AR). Um estudo anterior do nosso grupo relatou que… (more)

Subjects/Keywords: Artrite Reumatóide, IL-22, IL22RA1, SNP (Polimorfismo de Nucleotídeo Único).; Rheumatoid Arthritis,IL22, IL22RA1. Polymorphisms, SNP (Single Nucleotide Polymorphism

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APA (6th Edition):

VILLAR, K. d. M. (2015). Associação de polimorfismos no gene IL22RA1 como os níveis séricos de IL-22 e com parâmetros clínicos de pacientes portadores de artrite reumatoide . (Thesis). Universidade Federal de Pernambuco. Retrieved from http://repositorio.ufpe.br/handle/123456789/17169

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

VILLAR, Kamila de Melo. “Associação de polimorfismos no gene IL22RA1 como os níveis séricos de IL-22 e com parâmetros clínicos de pacientes portadores de artrite reumatoide .” 2015. Thesis, Universidade Federal de Pernambuco. Accessed October 22, 2019. http://repositorio.ufpe.br/handle/123456789/17169.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

VILLAR, Kamila de Melo. “Associação de polimorfismos no gene IL22RA1 como os níveis séricos de IL-22 e com parâmetros clínicos de pacientes portadores de artrite reumatoide .” 2015. Web. 22 Oct 2019.

Vancouver:

VILLAR KdM. Associação de polimorfismos no gene IL22RA1 como os níveis séricos de IL-22 e com parâmetros clínicos de pacientes portadores de artrite reumatoide . [Internet] [Thesis]. Universidade Federal de Pernambuco; 2015. [cited 2019 Oct 22]. Available from: http://repositorio.ufpe.br/handle/123456789/17169.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

VILLAR KdM. Associação de polimorfismos no gene IL22RA1 como os níveis séricos de IL-22 e com parâmetros clínicos de pacientes portadores de artrite reumatoide . [Thesis]. Universidade Federal de Pernambuco; 2015. Available from: http://repositorio.ufpe.br/handle/123456789/17169

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

19. Gimblet-Ochieng, Ciara. Regulating Immune Mediated Pathology In Cutaneous Leishmaniasis: Roles For Il-22 And Skin Microbiota.

Degree: 2017, University of Pennsylvania

 Cutaneous leishmaniasis is a chronic disease characterized by ulcerating and disfiguring skin lesions. Infection with different species of Leishmania parasites is responsible for the initiation… (more)

Subjects/Keywords: IL-22; Inflammation; Leishmania; Microbiome; Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology; Microbiology; Parasitology

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APA (6th Edition):

Gimblet-Ochieng, C. (2017). Regulating Immune Mediated Pathology In Cutaneous Leishmaniasis: Roles For Il-22 And Skin Microbiota. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2305

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gimblet-Ochieng, Ciara. “Regulating Immune Mediated Pathology In Cutaneous Leishmaniasis: Roles For Il-22 And Skin Microbiota.” 2017. Thesis, University of Pennsylvania. Accessed October 22, 2019. https://repository.upenn.edu/edissertations/2305.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gimblet-Ochieng, Ciara. “Regulating Immune Mediated Pathology In Cutaneous Leishmaniasis: Roles For Il-22 And Skin Microbiota.” 2017. Web. 22 Oct 2019.

Vancouver:

Gimblet-Ochieng C. Regulating Immune Mediated Pathology In Cutaneous Leishmaniasis: Roles For Il-22 And Skin Microbiota. [Internet] [Thesis]. University of Pennsylvania; 2017. [cited 2019 Oct 22]. Available from: https://repository.upenn.edu/edissertations/2305.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gimblet-Ochieng C. Regulating Immune Mediated Pathology In Cutaneous Leishmaniasis: Roles For Il-22 And Skin Microbiota. [Thesis]. University of Pennsylvania; 2017. Available from: https://repository.upenn.edu/edissertations/2305

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lund

20. Rattik, Sara. Regulatory Immune Responses and Repair Mechanisms in Atherosclerosis.

Degree: 2015, University of Lund

 Atherosclerosis is a chronic inflammatory disease characterized by the formation of lipid rich plaques in the arterial wall. Rupture of a plaque results in clinical… (more)

Subjects/Keywords: Kardiologi; Atherosclerosis; Cardiovascular disease; T helper 2 cells; ApoB100; Regulatory B cells; Regulatory T cells; Smooth muscle cells; IL-22.

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APA (6th Edition):

Rattik, S. (2015). Regulatory Immune Responses and Repair Mechanisms in Atherosclerosis. (Doctoral Dissertation). University of Lund. Retrieved from http://lup.lub.lu.se/record/4927061 ; http://portal.research.lu.se/ws/files/3245838/4935460.pdf

Chicago Manual of Style (16th Edition):

Rattik, Sara. “Regulatory Immune Responses and Repair Mechanisms in Atherosclerosis.” 2015. Doctoral Dissertation, University of Lund. Accessed October 22, 2019. http://lup.lub.lu.se/record/4927061 ; http://portal.research.lu.se/ws/files/3245838/4935460.pdf.

MLA Handbook (7th Edition):

Rattik, Sara. “Regulatory Immune Responses and Repair Mechanisms in Atherosclerosis.” 2015. Web. 22 Oct 2019.

Vancouver:

Rattik S. Regulatory Immune Responses and Repair Mechanisms in Atherosclerosis. [Internet] [Doctoral dissertation]. University of Lund; 2015. [cited 2019 Oct 22]. Available from: http://lup.lub.lu.se/record/4927061 ; http://portal.research.lu.se/ws/files/3245838/4935460.pdf.

Council of Science Editors:

Rattik S. Regulatory Immune Responses and Repair Mechanisms in Atherosclerosis. [Doctoral Dissertation]. University of Lund; 2015. Available from: http://lup.lub.lu.se/record/4927061 ; http://portal.research.lu.se/ws/files/3245838/4935460.pdf

21. Goupil, Mathieu. Analyse des altérations de l'immunité T-dépendante à l'égard de Candida albicans chez la souris transgénique exprimant le génome du VIH-1 .

Degree: 2014, Université de Montréal

 La candidose oro-pharyngée (COP) est l’infection fongique opportuniste la plus commune chez les individus infectés par le VIH-1. La production des cytokines Il-17 et Il-22(more)

Subjects/Keywords: Candidose oro-pharyngée; Cytokine; Il-17; Il-22; Modèle animal; Th17; Lymphocyte T régulateur; VIH-1; Animal model; HIV-1; Oropharyngeal candidiasis; Regulatory T-cell

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APA (6th Edition):

Goupil, M. (2014). Analyse des altérations de l'immunité T-dépendante à l'égard de Candida albicans chez la souris transgénique exprimant le génome du VIH-1 . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/12738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Goupil, Mathieu. “Analyse des altérations de l'immunité T-dépendante à l'égard de Candida albicans chez la souris transgénique exprimant le génome du VIH-1 .” 2014. Thesis, Université de Montréal. Accessed October 22, 2019. http://hdl.handle.net/1866/12738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Goupil, Mathieu. “Analyse des altérations de l'immunité T-dépendante à l'égard de Candida albicans chez la souris transgénique exprimant le génome du VIH-1 .” 2014. Web. 22 Oct 2019.

Vancouver:

Goupil M. Analyse des altérations de l'immunité T-dépendante à l'égard de Candida albicans chez la souris transgénique exprimant le génome du VIH-1 . [Internet] [Thesis]. Université de Montréal; 2014. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/1866/12738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Goupil M. Analyse des altérations de l'immunité T-dépendante à l'égard de Candida albicans chez la souris transgénique exprimant le génome du VIH-1 . [Thesis]. Université de Montréal; 2014. Available from: http://hdl.handle.net/1866/12738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

22. Fung, Thomas Chia Ting. Defining Interactions Between Lymphoid Tissue-Resident Commensal Bacteria and the Host Immune System.

Degree: 2016, University of Pennsylvania

 The mammalian gastro-intestinal tract is colonized by trillions of beneficial commensal bacteria that are essential for promoting normal host physiology. While the majority of commensal… (more)

Subjects/Keywords: commensal bacteria; group 3 innate lymphoid cells; IL-10; IL-22; intestinal-associated lymphoid tissues; T helper 17 cells; Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology; Microbiology

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APA (6th Edition):

Fung, T. C. T. (2016). Defining Interactions Between Lymphoid Tissue-Resident Commensal Bacteria and the Host Immune System. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1727

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fung, Thomas Chia Ting. “Defining Interactions Between Lymphoid Tissue-Resident Commensal Bacteria and the Host Immune System.” 2016. Thesis, University of Pennsylvania. Accessed October 22, 2019. https://repository.upenn.edu/edissertations/1727.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fung, Thomas Chia Ting. “Defining Interactions Between Lymphoid Tissue-Resident Commensal Bacteria and the Host Immune System.” 2016. Web. 22 Oct 2019.

Vancouver:

Fung TCT. Defining Interactions Between Lymphoid Tissue-Resident Commensal Bacteria and the Host Immune System. [Internet] [Thesis]. University of Pennsylvania; 2016. [cited 2019 Oct 22]. Available from: https://repository.upenn.edu/edissertations/1727.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fung TCT. Defining Interactions Between Lymphoid Tissue-Resident Commensal Bacteria and the Host Immune System. [Thesis]. University of Pennsylvania; 2016. Available from: https://repository.upenn.edu/edissertations/1727

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

23. Mantei, Andrej. Molekulare Analyse von pDZ-vermittelter Induktion von IL-10- und IL-22-Expression in humanen T-Zellen.

Degree: 2015, Freie Universität Berlin

IL-10 und IL-22 gehören zu einer Zytokinfamilie, spielen jedoch unterschiedliche Rollen in der Immunantwort. IL-10 ist einer der wichtigsten immunsuppressiven Faktoren, während IL-22 in die… (more)

Subjects/Keywords: T cell; IL-10; IL-22; IL10; IL22; STAT3; AhR; MAF; STAT1; IFN-alpha; pDC; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie

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APA (6th Edition):

Mantei, A. (2015). Molekulare Analyse von pDZ-vermittelter Induktion von IL-10- und IL-22-Expression in humanen T-Zellen. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/13946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mantei, Andrej. “Molekulare Analyse von pDZ-vermittelter Induktion von IL-10- und IL-22-Expression in humanen T-Zellen.” 2015. Thesis, Freie Universität Berlin. Accessed October 22, 2019. https://refubium.fu-berlin.de/handle/fub188/13946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mantei, Andrej. “Molekulare Analyse von pDZ-vermittelter Induktion von IL-10- und IL-22-Expression in humanen T-Zellen.” 2015. Web. 22 Oct 2019.

Vancouver:

Mantei A. Molekulare Analyse von pDZ-vermittelter Induktion von IL-10- und IL-22-Expression in humanen T-Zellen. [Internet] [Thesis]. Freie Universität Berlin; 2015. [cited 2019 Oct 22]. Available from: https://refubium.fu-berlin.de/handle/fub188/13946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mantei A. Molekulare Analyse von pDZ-vermittelter Induktion von IL-10- und IL-22-Expression in humanen T-Zellen. [Thesis]. Freie Universität Berlin; 2015. Available from: https://refubium.fu-berlin.de/handle/fub188/13946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

24. Fabre, Thomas. Étude in vitro de l’implication des cytokines de type Th17 dans la fibrose hépatique .

Degree: 2013, Université de Montréal

Subjects/Keywords: Hépatite; virus de l'hépatite C; Lymphocytes Th17; Lymphocytes T régulateurs; cellules stellaires hépatique; IL-17A; IL-22; Fibrose hépatique; Hepatitis; Liver fibrosis; Hepatitis C virus; Th17 lymphocytes; regulatory T lymphocytes; Hepatic stellate cells; IL-17A; IL-22

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fabre, T. (2013). Étude in vitro de l’implication des cytokines de type Th17 dans la fibrose hépatique . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/9848

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fabre, Thomas. “Étude in vitro de l’implication des cytokines de type Th17 dans la fibrose hépatique .” 2013. Thesis, Université de Montréal. Accessed October 22, 2019. http://hdl.handle.net/1866/9848.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fabre, Thomas. “Étude in vitro de l’implication des cytokines de type Th17 dans la fibrose hépatique .” 2013. Web. 22 Oct 2019.

Vancouver:

Fabre T. Étude in vitro de l’implication des cytokines de type Th17 dans la fibrose hépatique . [Internet] [Thesis]. Université de Montréal; 2013. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/1866/9848.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fabre T. Étude in vitro de l’implication des cytokines de type Th17 dans la fibrose hépatique . [Thesis]. Université de Montréal; 2013. Available from: http://hdl.handle.net/1866/9848

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Porte, Rémi. Caractérisation des propriétés anti-infectieuses de la flagelline, agoniste du Toll-like receptor 5 : Characterization of anti-infectious properties of flagellin, Toll-like receptor 5 agonist.

Degree: Docteur es, Immunologie (Médecine), 2015, Université Lille II – Droit et Santé

 De par sa capacité à détecter les microorganismes et à mettre en place une défense anti-infectieuse rapide, l’immunité innée représente la première ligne de défense… (more)

Subjects/Keywords: Immunité innée; TLR5; Agoniste; Flagelline; Infection intestinale; Infection respiratoire; Antibiotique; Thérapie; Prophylaxie; IL-22; Muqueuse; Toll-like receptor 5; Antibiotics; Flagellin; Innate immune

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APA (6th Edition):

Porte, R. (2015). Caractérisation des propriétés anti-infectieuses de la flagelline, agoniste du Toll-like receptor 5 : Characterization of anti-infectious properties of flagellin, Toll-like receptor 5 agonist. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2015LIL2S065

Chicago Manual of Style (16th Edition):

Porte, Rémi. “Caractérisation des propriétés anti-infectieuses de la flagelline, agoniste du Toll-like receptor 5 : Characterization of anti-infectious properties of flagellin, Toll-like receptor 5 agonist.” 2015. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed October 22, 2019. http://www.theses.fr/2015LIL2S065.

MLA Handbook (7th Edition):

Porte, Rémi. “Caractérisation des propriétés anti-infectieuses de la flagelline, agoniste du Toll-like receptor 5 : Characterization of anti-infectious properties of flagellin, Toll-like receptor 5 agonist.” 2015. Web. 22 Oct 2019.

Vancouver:

Porte R. Caractérisation des propriétés anti-infectieuses de la flagelline, agoniste du Toll-like receptor 5 : Characterization of anti-infectious properties of flagellin, Toll-like receptor 5 agonist. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2015. [cited 2019 Oct 22]. Available from: http://www.theses.fr/2015LIL2S065.

Council of Science Editors:

Porte R. Caractérisation des propriétés anti-infectieuses de la flagelline, agoniste du Toll-like receptor 5 : Characterization of anti-infectious properties of flagellin, Toll-like receptor 5 agonist. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2015. Available from: http://www.theses.fr/2015LIL2S065


Freie Universität Berlin

26. Winsauer, Caroline. TNF, das von verschiedenen Zelltypen produziert wird, und dessen Rolle in autoimmuner Colitis.

Degree: 2015, Freie Universität Berlin

 Als chronisch-entzündliche Darmerkrankungen (CED) werden persistierende Entzündungen der Schleimhaut des Gastrointestinaltraktes bezeichnet, die mit einer erhöhten Produktion von Zytokinen assoziiert sind. Die systemische medikamentöse Behandlung… (more)

Subjects/Keywords: TNF; colitis; autoimmunity; anti-TNF therapy; tissue repair; IL-22; 500 Naturwissenschaften und Mathematik; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie::570 Biowissenschaften; Biologie

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APA (6th Edition):

Winsauer, C. (2015). TNF, das von verschiedenen Zelltypen produziert wird, und dessen Rolle in autoimmuner Colitis. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-12427

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Winsauer, Caroline. “TNF, das von verschiedenen Zelltypen produziert wird, und dessen Rolle in autoimmuner Colitis.” 2015. Thesis, Freie Universität Berlin. Accessed October 22, 2019. http://dx.doi.org/10.17169/refubium-12427.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Winsauer, Caroline. “TNF, das von verschiedenen Zelltypen produziert wird, und dessen Rolle in autoimmuner Colitis.” 2015. Web. 22 Oct 2019.

Vancouver:

Winsauer C. TNF, das von verschiedenen Zelltypen produziert wird, und dessen Rolle in autoimmuner Colitis. [Internet] [Thesis]. Freie Universität Berlin; 2015. [cited 2019 Oct 22]. Available from: http://dx.doi.org/10.17169/refubium-12427.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Winsauer C. TNF, das von verschiedenen Zelltypen produziert wird, und dessen Rolle in autoimmuner Colitis. [Thesis]. Freie Universität Berlin; 2015. Available from: http://dx.doi.org/10.17169/refubium-12427

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Uppsala University

27. Edin, Josephine Lystedt. Omfattningen av begreppet delägare i handelsbolag i 22:3 och 23:5 IL : En kritisk granskning av HFD 2015 ref. 30.

Degree: Law, 2016, Uppsala University

Subjects/Keywords: uttagsbeskattning; indirekt ägt handelsbolag; HFD 2015 ref. 30; 22:3 IL; 23:5 IL

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APA (6th Edition):

Edin, J. L. (2016). Omfattningen av begreppet delägare i handelsbolag i 22:3 och 23:5 IL : En kritisk granskning av HFD 2015 ref. 30. (Thesis). Uppsala University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-272538

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Edin, Josephine Lystedt. “Omfattningen av begreppet delägare i handelsbolag i 22:3 och 23:5 IL : En kritisk granskning av HFD 2015 ref. 30.” 2016. Thesis, Uppsala University. Accessed October 22, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-272538.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Edin, Josephine Lystedt. “Omfattningen av begreppet delägare i handelsbolag i 22:3 och 23:5 IL : En kritisk granskning av HFD 2015 ref. 30.” 2016. Web. 22 Oct 2019.

Vancouver:

Edin JL. Omfattningen av begreppet delägare i handelsbolag i 22:3 och 23:5 IL : En kritisk granskning av HFD 2015 ref. 30. [Internet] [Thesis]. Uppsala University; 2016. [cited 2019 Oct 22]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-272538.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Edin JL. Omfattningen av begreppet delägare i handelsbolag i 22:3 och 23:5 IL : En kritisk granskning av HFD 2015 ref. 30. [Thesis]. Uppsala University; 2016. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-272538

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

28. LAVOIE,TEGAN N. Expression and Effects of Interleukin-22 in Human Sjogren's Syndrome and Mouse Models.

Degree: MS, Medical Sciences - Medicine, 2011, University of Florida

 Sj?gren's syndrome (SjS) is a complex autoimmune disease which targets the exocrine glands resulting in xerostomia/ keratoconjunctivitis sicca. Presently, we examined the presence, source and… (more)

Subjects/Keywords: Cells; Cytokines; Diseases; Mice; Natural killer cells; Receptors; Saliva; Salivary glands; Sjogrens syndrome; Spleen; 22  – AEC1R1AEC2  – APOPTOSIS  – C57BL6  – HSG  – IL  – INTERLEUKIN  – LTI  – LYMPHOID  – MODEL  – MOUSE  – NK  – NKP46  – PROLIFERATION  – ROR  – RORGAMMAT  – SJOGREN  – SJOGRENS  – SYNDROME

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APA (6th Edition):

N, L. (2011). Expression and Effects of Interleukin-22 in Human Sjogren's Syndrome and Mouse Models. (Masters Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0042955

Chicago Manual of Style (16th Edition):

N, LAVOIE,TEGAN. “Expression and Effects of Interleukin-22 in Human Sjogren's Syndrome and Mouse Models.” 2011. Masters Thesis, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0042955.

MLA Handbook (7th Edition):

N, LAVOIE,TEGAN. “Expression and Effects of Interleukin-22 in Human Sjogren's Syndrome and Mouse Models.” 2011. Web. 22 Oct 2019.

Vancouver:

N L. Expression and Effects of Interleukin-22 in Human Sjogren's Syndrome and Mouse Models. [Internet] [Masters thesis]. University of Florida; 2011. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0042955.

Council of Science Editors:

N L. Expression and Effects of Interleukin-22 in Human Sjogren's Syndrome and Mouse Models. [Masters Thesis]. University of Florida; 2011. Available from: http://ufdc.ufl.edu/UFE0042955

29. Bondeson, Johan. Den nya expertskatten : Innebär de nya reglerna en ökad förutsebarhet för personer i nyckelposition?.

Degree: Commercial Law, 2012, Jönköping University

  Inom ramen för den svenska inkomstskattelagen finns en expertskatt som ger möjlighet för svenska företag att anställa utländska experter som kan beviljas skattelättnader. 2001… (more)

Subjects/Keywords: Internationell skatterätt; individbeskattning; expertskatt; nyckelpersoner; 11 kap. 22-23a §§ IL; Law; Juridik

…ska 11 kap. 22 § 1 st. IL anses vara uppfylld. I 11 kap. 22 § 1 st. IL ställs ett antal… …omfattas av expertskatten. Genom bestämmelsen i 11 kap. 22 § 3 st. IL är expertskattens… …Syftet är att utreda om förutsebarheten har ökats genom ändringarna i 11 kap. 22-23a §§ IL. I… …Gällande rätt Inledning Reglerna om expertskatten finns stadgade i 11 kap. 22-23a §§ IL. Här… …forskare I 11 kap. 22 § 1 st. IL definieras de olika typer av kategorier av personer som kan få… 

Page 1 Page 2 Page 3 Page 4 Page 5 Page 6 Page 7

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APA (6th Edition):

Bondeson, J. (2012). Den nya expertskatten : Innebär de nya reglerna en ökad förutsebarhet för personer i nyckelposition?. (Thesis). Jönköping University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-19266

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bondeson, Johan. “Den nya expertskatten : Innebär de nya reglerna en ökad förutsebarhet för personer i nyckelposition?.” 2012. Thesis, Jönköping University. Accessed October 22, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-19266.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bondeson, Johan. “Den nya expertskatten : Innebär de nya reglerna en ökad förutsebarhet för personer i nyckelposition?.” 2012. Web. 22 Oct 2019.

Vancouver:

Bondeson J. Den nya expertskatten : Innebär de nya reglerna en ökad förutsebarhet för personer i nyckelposition?. [Internet] [Thesis]. Jönköping University; 2012. [cited 2019 Oct 22]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-19266.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bondeson J. Den nya expertskatten : Innebär de nya reglerna en ökad förutsebarhet för personer i nyckelposition?. [Thesis]. Jönköping University; 2012. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-19266

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

30. Sonnenberg, Gregory Field. Interleukin-22 Regulates Immunity, Inflammation and Tissue Homeostasis at Mucosal Sites.

Degree: 2011, University of Pennsylvania

 At mucosal sites, a single layer of epithelial cells separates the connective tissues of the mammalian body from the external environment. The gastrointestinal tract is… (more)

Subjects/Keywords: IL-22; innate lymphoid cell; mucosal immunology; Th17 cell; intestinal barrier function; intestinal homeostasis; Cell Biology; Immunity; Immunology of Infectious Disease; Immunopathology; Immunoprophylaxis and Therapy; Microbiology; Other Immunology and Infectious Disease

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APA (6th Edition):

Sonnenberg, G. F. (2011). Interleukin-22 Regulates Immunity, Inflammation and Tissue Homeostasis at Mucosal Sites. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/963

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sonnenberg, Gregory Field. “Interleukin-22 Regulates Immunity, Inflammation and Tissue Homeostasis at Mucosal Sites.” 2011. Thesis, University of Pennsylvania. Accessed October 22, 2019. https://repository.upenn.edu/edissertations/963.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sonnenberg, Gregory Field. “Interleukin-22 Regulates Immunity, Inflammation and Tissue Homeostasis at Mucosal Sites.” 2011. Web. 22 Oct 2019.

Vancouver:

Sonnenberg GF. Interleukin-22 Regulates Immunity, Inflammation and Tissue Homeostasis at Mucosal Sites. [Internet] [Thesis]. University of Pennsylvania; 2011. [cited 2019 Oct 22]. Available from: https://repository.upenn.edu/edissertations/963.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sonnenberg GF. Interleukin-22 Regulates Immunity, Inflammation and Tissue Homeostasis at Mucosal Sites. [Thesis]. University of Pennsylvania; 2011. Available from: https://repository.upenn.edu/edissertations/963

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2]

.