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You searched for subject:(IL 1beta). Showing records 1 – 30 of 37 total matches.

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1. Beard, LaMonta L. Regulation of Distal Lung Fluid Absorption by Membrane Components.

Degree: PhD, College of Arts and Sciences / School of Biomedical Sciences, 2011, Kent State University

 Near the end of gestation, the direction of ion and fluid flow across the alveolar epithelium rapidly changes from secretion to absorption. Thus, the relative… (more)

Subjects/Keywords: Biomedical Research; Cl transport; distal lung fluid absorption; fetal lung development; IL-1beta; Na transport

…53 CHAPTER III. Fig. 3-1. IL-1/cortisol Pathway… …GD68 in the Lungs With and Without IL-1 Pretreatment… …ENaC in CRRs and Non-CRRs With and Without IL-1 Pretreatment .................. 71 x Fig… …3-4. Expression of CFTR in CRRs and Non-CRRs at GD61 and GD68 With and Without IL-1… …73 Fig. 3-5. Completed IL-1/cortisol Pathway… 

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APA (6th Edition):

Beard, L. L. (2011). Regulation of Distal Lung Fluid Absorption by Membrane Components. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1321989465

Chicago Manual of Style (16th Edition):

Beard, LaMonta L. “Regulation of Distal Lung Fluid Absorption by Membrane Components.” 2011. Doctoral Dissertation, Kent State University. Accessed October 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=kent1321989465.

MLA Handbook (7th Edition):

Beard, LaMonta L. “Regulation of Distal Lung Fluid Absorption by Membrane Components.” 2011. Web. 16 Oct 2019.

Vancouver:

Beard LL. Regulation of Distal Lung Fluid Absorption by Membrane Components. [Internet] [Doctoral dissertation]. Kent State University; 2011. [cited 2019 Oct 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1321989465.

Council of Science Editors:

Beard LL. Regulation of Distal Lung Fluid Absorption by Membrane Components. [Doctoral Dissertation]. Kent State University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1321989465

2. Bernard, Marine. Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors.

Degree: Docteur es, Immunologie, 2014, Université Claude Bernard – Lyon I

La dermatite atopique (DA) est une dermatose inflammatoire dont la prévalence élevée est en constante augmentation dans les pays industrialisés. La physiopathologie de la DA… (more)

Subjects/Keywords: Dermatite atopique; Épiderme reconstruit humain; Apoptose; UV; IL-1beta; TSLP; Dermatophagoïdes farinae; Atopic dermatitis; Reconstructed human epidermis; Apoptosis; UV; IL-1beta; TSLP; Dermatophagoïdes farinae; 571.96

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APA (6th Edition):

Bernard, M. (2014). Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2014LYO10361

Chicago Manual of Style (16th Edition):

Bernard, Marine. “Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors.” 2014. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed October 16, 2019. http://www.theses.fr/2014LYO10361.

MLA Handbook (7th Edition):

Bernard, Marine. “Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors.” 2014. Web. 16 Oct 2019.

Vancouver:

Bernard M. Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2014. [cited 2019 Oct 16]. Available from: http://www.theses.fr/2014LYO10361.

Council of Science Editors:

Bernard M. Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2014. Available from: http://www.theses.fr/2014LYO10361


Freie Universität Berlin

3. Busch, Katharina. The role of the innate immune system in the inflammation-mediated cardiomyopathy.

Degree: 2019, Freie Universität Berlin

 Sepsis is one of the leading causes for increased mortality of critically ill patients in the hospital. In sepsis, elevated serum levels of inflammatory cytokines… (more)

Subjects/Keywords: inflammation; cardiomyopathy; NaLP3-inflammasom; IL-1beta; MuRF1; atrophy; autophagy; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit::610 Medizin und Gesundheit

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APA (6th Edition):

Busch, K. (2019). The role of the innate immune system in the inflammation-mediated cardiomyopathy. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-1849

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Busch, Katharina. “The role of the innate immune system in the inflammation-mediated cardiomyopathy.” 2019. Thesis, Freie Universität Berlin. Accessed October 16, 2019. http://dx.doi.org/10.17169/refubium-1849.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Busch, Katharina. “The role of the innate immune system in the inflammation-mediated cardiomyopathy.” 2019. Web. 16 Oct 2019.

Vancouver:

Busch K. The role of the innate immune system in the inflammation-mediated cardiomyopathy. [Internet] [Thesis]. Freie Universität Berlin; 2019. [cited 2019 Oct 16]. Available from: http://dx.doi.org/10.17169/refubium-1849.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Busch K. The role of the innate immune system in the inflammation-mediated cardiomyopathy. [Thesis]. Freie Universität Berlin; 2019. Available from: http://dx.doi.org/10.17169/refubium-1849

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

4. Chesnokov, Anton P, Mr. Modulation of NALP3 Inflammasome Genes by Estrogen.

Degree: MS, Biology, 2011, Georgia State University

  Immunity is known to be sexually dimorphic. This dimorphism may be attributed to the action of different hormones. Aluminum is a component of several… (more)

Subjects/Keywords: Nalp3 inflammasome; Estrogen; IL-1beta; Aluminum hydroxide

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APA (6th Edition):

Chesnokov, Anton P, M. (2011). Modulation of NALP3 Inflammasome Genes by Estrogen. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/33

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chesnokov, Anton P, Mr. “Modulation of NALP3 Inflammasome Genes by Estrogen.” 2011. Thesis, Georgia State University. Accessed October 16, 2019. https://scholarworks.gsu.edu/biology_theses/33.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chesnokov, Anton P, Mr. “Modulation of NALP3 Inflammasome Genes by Estrogen.” 2011. Web. 16 Oct 2019.

Vancouver:

Chesnokov, Anton P M. Modulation of NALP3 Inflammasome Genes by Estrogen. [Internet] [Thesis]. Georgia State University; 2011. [cited 2019 Oct 16]. Available from: https://scholarworks.gsu.edu/biology_theses/33.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chesnokov, Anton P M. Modulation of NALP3 Inflammasome Genes by Estrogen. [Thesis]. Georgia State University; 2011. Available from: https://scholarworks.gsu.edu/biology_theses/33

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

5. DRAGON, Stephane. Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils.

Degree: Immunology, 2010, University of Manitoba

 Immunopathological disorders are no longer defined by dysregulated T-helper (Th) type 1/ Th2 responses but account for modulatory cell types such as regulatory and Th17… (more)

Subjects/Keywords: Human; Inflammation; Cytokines; IL-17; IL-1beta; IL-22; CXCL-8; airway smooth muscle cells; neutrophils; IL-17R; signal transduction; gene expression; apoptosis

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APA (6th Edition):

DRAGON, S. (2010). Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/3945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DRAGON, Stephane. “Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils.” 2010. Thesis, University of Manitoba. Accessed October 16, 2019. http://hdl.handle.net/1993/3945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DRAGON, Stephane. “Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils.” 2010. Web. 16 Oct 2019.

Vancouver:

DRAGON S. Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils. [Internet] [Thesis]. University of Manitoba; 2010. [cited 2019 Oct 16]. Available from: http://hdl.handle.net/1993/3945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DRAGON S. Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils. [Thesis]. University of Manitoba; 2010. Available from: http://hdl.handle.net/1993/3945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Dumont, Adélie. L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta.

Degree: Docteur es, Physiologie, 2018, Bourgogne Franche-Comté

Selon une étude précédente, une limitation à l'efficacité anticancéreuse du 5-Fluorouracile (5-FU) repose sur la sécrétion d'IL-1β par des cellules myéloïdes immunosuppressives (MDSC). La libération… (more)

Subjects/Keywords: Mdsc; 5 fluorouracil; Dha; IL-1 beta; Jnk; Mdsc; 5-Fluorouracil; Dha; IL-1beta; Jnk; 612

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APA (6th Edition):

Dumont, A. (2018). L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta. (Doctoral Dissertation). Bourgogne Franche-Comté. Retrieved from http://www.theses.fr/2018UBFCI013

Chicago Manual of Style (16th Edition):

Dumont, Adélie. “L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta.” 2018. Doctoral Dissertation, Bourgogne Franche-Comté. Accessed October 16, 2019. http://www.theses.fr/2018UBFCI013.

MLA Handbook (7th Edition):

Dumont, Adélie. “L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta.” 2018. Web. 16 Oct 2019.

Vancouver:

Dumont A. L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta. [Internet] [Doctoral dissertation]. Bourgogne Franche-Comté; 2018. [cited 2019 Oct 16]. Available from: http://www.theses.fr/2018UBFCI013.

Council of Science Editors:

Dumont A. L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta. [Doctoral Dissertation]. Bourgogne Franche-Comté; 2018. Available from: http://www.theses.fr/2018UBFCI013


University of Manchester

7. Englezou, Pavlos. P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages.

Degree: PhD, 2013, University of Manchester

 The P2X7R is a functionally distinct member of the P2X non-selective cation channels and has been implicated in the initiation of immune responses. One of… (more)

Subjects/Keywords: 616.07; P2X7R; Dendritic cells; Macrophages; IL-1beta; IL-1alpha; P2X7R splice variants

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APA (6th Edition):

Englezou, P. (2013). P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/p2x7rdriven-il1-responses-in-differentiated-murine-dendritic-cells-comparison-with-macrophages(81ddc8a9-6d43-4fe6-b9ec-e2b1b9a156a4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603121

Chicago Manual of Style (16th Edition):

Englezou, Pavlos. “P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages.” 2013. Doctoral Dissertation, University of Manchester. Accessed October 16, 2019. https://www.research.manchester.ac.uk/portal/en/theses/p2x7rdriven-il1-responses-in-differentiated-murine-dendritic-cells-comparison-with-macrophages(81ddc8a9-6d43-4fe6-b9ec-e2b1b9a156a4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603121.

MLA Handbook (7th Edition):

Englezou, Pavlos. “P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages.” 2013. Web. 16 Oct 2019.

Vancouver:

Englezou P. P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2019 Oct 16]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/p2x7rdriven-il1-responses-in-differentiated-murine-dendritic-cells-comparison-with-macrophages(81ddc8a9-6d43-4fe6-b9ec-e2b1b9a156a4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603121.

Council of Science Editors:

Englezou P. P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages. [Doctoral Dissertation]. University of Manchester; 2013. Available from: https://www.research.manchester.ac.uk/portal/en/theses/p2x7rdriven-il1-responses-in-differentiated-murine-dendritic-cells-comparison-with-macrophages(81ddc8a9-6d43-4fe6-b9ec-e2b1b9a156a4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603121

8. Englezou, Pavlos. P2X7R-driven IL-1 responses in differentiated murine Dendritic Cells; comparison with Macrophages.

Degree: 2013, University of Manchester

 The P2X7R is a functionally distinct member of the P2X non-selective cation channels and has been implicated in the initiation of immune responses. One of… (more)

Subjects/Keywords: P2X7R; Dendritic cells; Macrophages; IL-1beta; IL-1alpha; P2X7R splice variants

…Figure 5.9: The impact of treatment with A-740003 on IL-1β release from LPS-primed and… …230 Figure 5.10: The impact of treatment with A-740003 on IL-1α and IL-6 release from… …Figure 5.11: The effect of apyrase treatment on IL-1 release from LPS-primed and ATP challenged… …238 Figure 5.14: The effect of apyrase treatment on IL-1β release from LPS-pulsed day 8… …of IL-1β release in day 8 BM-DC; comparisons with 8 BM-MΦ… 

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APA (6th Edition):

Englezou, P. (2013). P2X7R-driven IL-1 responses in differentiated murine Dendritic Cells; comparison with Macrophages. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:213941

Chicago Manual of Style (16th Edition):

Englezou, Pavlos. “P2X7R-driven IL-1 responses in differentiated murine Dendritic Cells; comparison with Macrophages.” 2013. Doctoral Dissertation, University of Manchester. Accessed October 16, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:213941.

MLA Handbook (7th Edition):

Englezou, Pavlos. “P2X7R-driven IL-1 responses in differentiated murine Dendritic Cells; comparison with Macrophages.” 2013. Web. 16 Oct 2019.

Vancouver:

Englezou P. P2X7R-driven IL-1 responses in differentiated murine Dendritic Cells; comparison with Macrophages. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2019 Oct 16]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:213941.

Council of Science Editors:

Englezou P. P2X7R-driven IL-1 responses in differentiated murine Dendritic Cells; comparison with Macrophages. [Doctoral Dissertation]. University of Manchester; 2013. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:213941


University of Lund

9. Gewert, Karin. Molecular targets for glucocorticoids in macrophages: cytosolic phospholipase A2 and cytokine formation.

Degree: 2000, University of Lund

 The aim of this investigation has been to study mechanisms by which glucocorticoids inhibit inflammatory processes in vitro. Type IV cytosolic 85 kDa phospholipase A2… (more)

Subjects/Keywords: Infektionsmedicin; pharmacognosy; Pharmacological sciences; signal transduction; dexamethasone; cytosolic phospholipase A2; glucocorticoids; macrophages; protein kinase C; mitogen-activated protein kinases; TNF-alpha; IL-1beta; protein phosphatases; okadaic acid; pharmacy; toxicology; Farmakologi; farmakognosi; farmaci; toxikologi

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APA (6th Edition):

Gewert, K. (2000). Molecular targets for glucocorticoids in macrophages: cytosolic phospholipase A2 and cytokine formation. (Doctoral Dissertation). University of Lund. Retrieved from http://lup.lub.lu.se/record/40246 ; http://portal.research.lu.se/ws/files/5922756/1693387.pdf

Chicago Manual of Style (16th Edition):

Gewert, Karin. “Molecular targets for glucocorticoids in macrophages: cytosolic phospholipase A2 and cytokine formation.” 2000. Doctoral Dissertation, University of Lund. Accessed October 16, 2019. http://lup.lub.lu.se/record/40246 ; http://portal.research.lu.se/ws/files/5922756/1693387.pdf.

MLA Handbook (7th Edition):

Gewert, Karin. “Molecular targets for glucocorticoids in macrophages: cytosolic phospholipase A2 and cytokine formation.” 2000. Web. 16 Oct 2019.

Vancouver:

Gewert K. Molecular targets for glucocorticoids in macrophages: cytosolic phospholipase A2 and cytokine formation. [Internet] [Doctoral dissertation]. University of Lund; 2000. [cited 2019 Oct 16]. Available from: http://lup.lub.lu.se/record/40246 ; http://portal.research.lu.se/ws/files/5922756/1693387.pdf.

Council of Science Editors:

Gewert K. Molecular targets for glucocorticoids in macrophages: cytosolic phospholipase A2 and cytokine formation. [Doctoral Dissertation]. University of Lund; 2000. Available from: http://lup.lub.lu.se/record/40246 ; http://portal.research.lu.se/ws/files/5922756/1693387.pdf

10. Gómez Ochoa, Pablo. Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes.

Degree: 2011, Universidad de Zaragoza

 El diseño de este trabajo intenta establecer las condiciones de cultivo y la metodología para la evaluación del grupo de las Stem Cells adultas y… (more)

Subjects/Keywords: Campos electromagnéticos Pulsados; Stem Cells; TNF-alfa; IL-1beta; IL-10; PEMF; Stem Cells; TNF-alfa; IL-1beta; IL-10

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APA (6th Edition):

Gómez Ochoa, P. (2011). Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes. (Thesis). Universidad de Zaragoza. Retrieved from http://zaguan.unizar.es/record/4497

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gómez Ochoa, Pablo. “Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes.” 2011. Thesis, Universidad de Zaragoza. Accessed October 16, 2019. http://zaguan.unizar.es/record/4497.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gómez Ochoa, Pablo. “Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes.” 2011. Web. 16 Oct 2019.

Vancouver:

Gómez Ochoa P. Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes. [Internet] [Thesis]. Universidad de Zaragoza; 2011. [cited 2019 Oct 16]. Available from: http://zaguan.unizar.es/record/4497.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gómez Ochoa P. Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes. [Thesis]. Universidad de Zaragoza; 2011. Available from: http://zaguan.unizar.es/record/4497

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

11. Huang, Nora. Nlrp3-Deletion schützt vor inflammationsvermittelter Skelettmuskelatrophie.

Degree: 2019, Freie Universität Berlin

 Background Intensive care unit-acquired weakness (ICUAW) is a common neuromuscular complication occurring in critically ill ICU-patients and is characterized by generalized muscle weakness. The associated… (more)

Subjects/Keywords: NLRP3; Sepsis; IL-1beta; muscle atrophy; MuRF1; inflammation; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit::610 Medizin und Gesundheit

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APA (6th Edition):

Huang, N. (2019). Nlrp3-Deletion schützt vor inflammationsvermittelter Skelettmuskelatrophie. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-4170

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Nora. “Nlrp3-Deletion schützt vor inflammationsvermittelter Skelettmuskelatrophie.” 2019. Thesis, Freie Universität Berlin. Accessed October 16, 2019. http://dx.doi.org/10.17169/refubium-4170.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Nora. “Nlrp3-Deletion schützt vor inflammationsvermittelter Skelettmuskelatrophie.” 2019. Web. 16 Oct 2019.

Vancouver:

Huang N. Nlrp3-Deletion schützt vor inflammationsvermittelter Skelettmuskelatrophie. [Internet] [Thesis]. Freie Universität Berlin; 2019. [cited 2019 Oct 16]. Available from: http://dx.doi.org/10.17169/refubium-4170.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang N. Nlrp3-Deletion schützt vor inflammationsvermittelter Skelettmuskelatrophie. [Thesis]. Freie Universität Berlin; 2019. Available from: http://dx.doi.org/10.17169/refubium-4170

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Case Western Reserve University

12. Kahlenberg, Joanne Michelle. Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands.

Degree: PhD, Pathology, 2004, Case Western Reserve University

 Recent advances in understanding of the biology underlying hereditary inflammatory diseases have focused attention on the importance of regulating levels of the proinflammatory cytokine IL-1beta(more)

Subjects/Keywords: Caspase-1; IL-1beta; Inflammasome; Macrophage; Monocyte; Potassium; Toll-Like Receptors

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APA (6th Edition):

Kahlenberg, J. M. (2004). Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614

Chicago Manual of Style (16th Edition):

Kahlenberg, Joanne Michelle. “Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands.” 2004. Doctoral Dissertation, Case Western Reserve University. Accessed October 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614.

MLA Handbook (7th Edition):

Kahlenberg, Joanne Michelle. “Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands.” 2004. Web. 16 Oct 2019.

Vancouver:

Kahlenberg JM. Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2004. [cited 2019 Oct 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614.

Council of Science Editors:

Kahlenberg JM. Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands. [Doctoral Dissertation]. Case Western Reserve University; 2004. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614


Freie Universität Berlin

13. Kalb, Alexander Christof. Acetylcholinesterase inhibitors reduce neuroinflammation and -degeneration in the cortex and hippocampus of a surgery stress rat model.

Degree: 2014, Freie Universität Berlin

 Exogenous stress like tissue damage and pathogen invasion during surgical trauma could lead to a peripheral inflammatory response and induce neuroinflammation, which can result in… (more)

Subjects/Keywords: neuroinflammation; POCD; acetylcholinesterase-inhibitor; rat; il-1beta; neurodegeneration; physostigmine; neostigmine; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Kalb, A. C. (2014). Acetylcholinesterase inhibitors reduce neuroinflammation and -degeneration in the cortex and hippocampus of a surgery stress rat model. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/11511

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kalb, Alexander Christof. “Acetylcholinesterase inhibitors reduce neuroinflammation and -degeneration in the cortex and hippocampus of a surgery stress rat model.” 2014. Thesis, Freie Universität Berlin. Accessed October 16, 2019. https://refubium.fu-berlin.de/handle/fub188/11511.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kalb, Alexander Christof. “Acetylcholinesterase inhibitors reduce neuroinflammation and -degeneration in the cortex and hippocampus of a surgery stress rat model.” 2014. Web. 16 Oct 2019.

Vancouver:

Kalb AC. Acetylcholinesterase inhibitors reduce neuroinflammation and -degeneration in the cortex and hippocampus of a surgery stress rat model. [Internet] [Thesis]. Freie Universität Berlin; 2014. [cited 2019 Oct 16]. Available from: https://refubium.fu-berlin.de/handle/fub188/11511.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kalb AC. Acetylcholinesterase inhibitors reduce neuroinflammation and -degeneration in the cortex and hippocampus of a surgery stress rat model. [Thesis]. Freie Universität Berlin; 2014. Available from: https://refubium.fu-berlin.de/handle/fub188/11511

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

14. Kanamori, Yohei. Transcriptional regulation of hepcidin by molecules mediating inflammatory responses .

Degree: 2018, Kyoto University

Subjects/Keywords: hepcidin; AP-1; activin B; IL-1beta; inflammation

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APA (6th Edition):

Kanamori, Y. (2018). Transcriptional regulation of hepcidin by molecules mediating inflammatory responses . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/232336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kanamori, Yohei. “Transcriptional regulation of hepcidin by molecules mediating inflammatory responses .” 2018. Thesis, Kyoto University. Accessed October 16, 2019. http://hdl.handle.net/2433/232336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kanamori, Yohei. “Transcriptional regulation of hepcidin by molecules mediating inflammatory responses .” 2018. Web. 16 Oct 2019.

Vancouver:

Kanamori Y. Transcriptional regulation of hepcidin by molecules mediating inflammatory responses . [Internet] [Thesis]. Kyoto University; 2018. [cited 2019 Oct 16]. Available from: http://hdl.handle.net/2433/232336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kanamori Y. Transcriptional regulation of hepcidin by molecules mediating inflammatory responses . [Thesis]. Kyoto University; 2018. Available from: http://hdl.handle.net/2433/232336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Kannan, Yashaswini. Functional Characterization Of Human IkappaBzeta In Modulating Inflammatory Responses.

Degree: PhD, Biophysics, 2011, The Ohio State University

  Innate immunity primarily mediated by germline-encoded pathogen recognition receptors (PRRs), recognize the pathogen and activate immune signaling. Signaling mediated by the innate immune toll… (more)

Subjects/Keywords: Biochemistry; Biology; Biophysics; Cellular Biology; Molecular Biology; IkappaBzeta; Pro-inflammatory response; Innate Immunity; TLRs; Monocytes; NK cells; IL-6; IL-1beta; IFN-gamma; Post-transcriptional regulation; Atherosclerosis; oxLDL

…Wei, Michael A. Caligiuri and Mark D. Wewers. I%B! augments IL-12– and IL-18–mediated IFN… …regulates human monocyte IL-6 production. J Immunol., October 2009, 15, 183(8), 5358… …Anasuya Sarkar, Vedavathi BellamkondaAthmaram and Mark D. Wewers. A synergistic role for IL-1… …8 1.3 IL-1 cytokines and the IL-1R family… …11 1.4 TLR/IL-1R signaling… 

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APA (6th Edition):

Kannan, Y. (2011). Functional Characterization Of Human IkappaBzeta In Modulating Inflammatory Responses. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1314564642

Chicago Manual of Style (16th Edition):

Kannan, Yashaswini. “Functional Characterization Of Human IkappaBzeta In Modulating Inflammatory Responses.” 2011. Doctoral Dissertation, The Ohio State University. Accessed October 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1314564642.

MLA Handbook (7th Edition):

Kannan, Yashaswini. “Functional Characterization Of Human IkappaBzeta In Modulating Inflammatory Responses.” 2011. Web. 16 Oct 2019.

Vancouver:

Kannan Y. Functional Characterization Of Human IkappaBzeta In Modulating Inflammatory Responses. [Internet] [Doctoral dissertation]. The Ohio State University; 2011. [cited 2019 Oct 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1314564642.

Council of Science Editors:

Kannan Y. Functional Characterization Of Human IkappaBzeta In Modulating Inflammatory Responses. [Doctoral Dissertation]. The Ohio State University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1314564642


Universiteit Utrecht

16. Kleibeuker, W. GRK2 deficiency is a pain.

Degree: 2007, Universiteit Utrecht

 G protein-coupled receptor (GPCR) kinase (GRK)2 regulates the responsiveness of many GPCRS, e.g. CCR1, CCR2, CCR5, neurokinin-1 receptor, and metabotropic glutamate receptors, which are involved… (more)

Subjects/Keywords: Geneeskunde; GRK2; GPCR; hyperalgesia; allodynia; IL-1beta; signaling; inflammatory pain; spinal cord; nerve damage; astrocytes

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APA (6th Edition):

Kleibeuker, W. (2007). GRK2 deficiency is a pain. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/25012

Chicago Manual of Style (16th Edition):

Kleibeuker, W. “GRK2 deficiency is a pain.” 2007. Doctoral Dissertation, Universiteit Utrecht. Accessed October 16, 2019. http://dspace.library.uu.nl:8080/handle/1874/25012.

MLA Handbook (7th Edition):

Kleibeuker, W. “GRK2 deficiency is a pain.” 2007. Web. 16 Oct 2019.

Vancouver:

Kleibeuker W. GRK2 deficiency is a pain. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2007. [cited 2019 Oct 16]. Available from: http://dspace.library.uu.nl:8080/handle/1874/25012.

Council of Science Editors:

Kleibeuker W. GRK2 deficiency is a pain. [Doctoral Dissertation]. Universiteit Utrecht; 2007. Available from: http://dspace.library.uu.nl:8080/handle/1874/25012

17. Kleibeuker, W. GRK2 deficiency is a pain.

Degree: 2007, University Utrecht

 G protein-coupled receptor (GPCR) kinase (GRK)2 regulates the responsiveness of many GPCRS, e.g. CCR1, CCR2, CCR5, neurokinin-1 receptor, and metabotropic glutamate receptors, which are involved… (more)

Subjects/Keywords: GRK2; GPCR; hyperalgesia; allodynia; IL-1beta; signaling; inflammatory pain; spinal cord; nerve damage; astrocytes

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APA (6th Edition):

Kleibeuker, W. (2007). GRK2 deficiency is a pain. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/25012 ; URN:NBN:NL:UI:10-1874-25012 ; urn:isbn:978-90-393-4685-3 ; URN:NBN:NL:UI:10-1874-25012 ; http://dspace.library.uu.nl/handle/1874/25012

Chicago Manual of Style (16th Edition):

Kleibeuker, W. “GRK2 deficiency is a pain.” 2007. Doctoral Dissertation, University Utrecht. Accessed October 16, 2019. http://dspace.library.uu.nl/handle/1874/25012 ; URN:NBN:NL:UI:10-1874-25012 ; urn:isbn:978-90-393-4685-3 ; URN:NBN:NL:UI:10-1874-25012 ; http://dspace.library.uu.nl/handle/1874/25012.

MLA Handbook (7th Edition):

Kleibeuker, W. “GRK2 deficiency is a pain.” 2007. Web. 16 Oct 2019.

Vancouver:

Kleibeuker W. GRK2 deficiency is a pain. [Internet] [Doctoral dissertation]. University Utrecht; 2007. [cited 2019 Oct 16]. Available from: http://dspace.library.uu.nl/handle/1874/25012 ; URN:NBN:NL:UI:10-1874-25012 ; urn:isbn:978-90-393-4685-3 ; URN:NBN:NL:UI:10-1874-25012 ; http://dspace.library.uu.nl/handle/1874/25012.

Council of Science Editors:

Kleibeuker W. GRK2 deficiency is a pain. [Doctoral Dissertation]. University Utrecht; 2007. Available from: http://dspace.library.uu.nl/handle/1874/25012 ; URN:NBN:NL:UI:10-1874-25012 ; urn:isbn:978-90-393-4685-3 ; URN:NBN:NL:UI:10-1874-25012 ; http://dspace.library.uu.nl/handle/1874/25012

18. Kleibeuker, W. GRK2 deficiency is a pain.

Degree: 2007, University Utrecht

 G protein-coupled receptor (GPCR) kinase (GRK)2 regulates the responsiveness of many GPCRS, e.g. CCR1, CCR2, CCR5, neurokinin-1 receptor, and metabotropic glutamate receptors, which are involved… (more)

Subjects/Keywords: GRK2; GPCR; hyperalgesia; allodynia; IL-1beta; signaling; inflammatory pain; spinal cord; nerve damage; astrocytes

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kleibeuker, W. (2007). GRK2 deficiency is a pain. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/25012 ; URN:NBN:NL:UI:10-1874-25012 ; urn:isbn:978-90-393-4685-3 ; URN:NBN:NL:UI:10-1874-25012 ; http://dspace.library.uu.nl/handle/1874/25012

Chicago Manual of Style (16th Edition):

Kleibeuker, W. “GRK2 deficiency is a pain.” 2007. Doctoral Dissertation, University Utrecht. Accessed October 16, 2019. http://dspace.library.uu.nl/handle/1874/25012 ; URN:NBN:NL:UI:10-1874-25012 ; urn:isbn:978-90-393-4685-3 ; URN:NBN:NL:UI:10-1874-25012 ; http://dspace.library.uu.nl/handle/1874/25012.

MLA Handbook (7th Edition):

Kleibeuker, W. “GRK2 deficiency is a pain.” 2007. Web. 16 Oct 2019.

Vancouver:

Kleibeuker W. GRK2 deficiency is a pain. [Internet] [Doctoral dissertation]. University Utrecht; 2007. [cited 2019 Oct 16]. Available from: http://dspace.library.uu.nl/handle/1874/25012 ; URN:NBN:NL:UI:10-1874-25012 ; urn:isbn:978-90-393-4685-3 ; URN:NBN:NL:UI:10-1874-25012 ; http://dspace.library.uu.nl/handle/1874/25012.

Council of Science Editors:

Kleibeuker W. GRK2 deficiency is a pain. [Doctoral Dissertation]. University Utrecht; 2007. Available from: http://dspace.library.uu.nl/handle/1874/25012 ; URN:NBN:NL:UI:10-1874-25012 ; urn:isbn:978-90-393-4685-3 ; URN:NBN:NL:UI:10-1874-25012 ; http://dspace.library.uu.nl/handle/1874/25012


Universiteit Utrecht

19. Kuiper, J.J.W. Reactive Oxygen Species in Inflammasome Activation.

Degree: 2009, Universiteit Utrecht

 The fundamental role of the innate immune system is to form the first barrier in bacterial and viral defence. It is based upon the recognition… (more)

Subjects/Keywords: Geneeskunde; NOD-like receptor, P2x7-receptor, NLRP3, Reactive Oxygen Species, ROS, Caspase-1, IL-1beta, IL-18, Inflammasome, ERK1/2, Cryopyrinopathies, CAPS

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APA (6th Edition):

Kuiper, J. J. W. (2009). Reactive Oxygen Species in Inflammasome Activation. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/35211

Chicago Manual of Style (16th Edition):

Kuiper, J J W. “Reactive Oxygen Species in Inflammasome Activation.” 2009. Masters Thesis, Universiteit Utrecht. Accessed October 16, 2019. http://dspace.library.uu.nl:8080/handle/1874/35211.

MLA Handbook (7th Edition):

Kuiper, J J W. “Reactive Oxygen Species in Inflammasome Activation.” 2009. Web. 16 Oct 2019.

Vancouver:

Kuiper JJW. Reactive Oxygen Species in Inflammasome Activation. [Internet] [Masters thesis]. Universiteit Utrecht; 2009. [cited 2019 Oct 16]. Available from: http://dspace.library.uu.nl:8080/handle/1874/35211.

Council of Science Editors:

Kuiper JJW. Reactive Oxygen Species in Inflammasome Activation. [Masters Thesis]. Universiteit Utrecht; 2009. Available from: http://dspace.library.uu.nl:8080/handle/1874/35211


Vanderbilt University

20. Li, Chunsheng. MyD88: central relay station of Interleukin 1 signaling pathway.

Degree: PhD, Microbiology and Immunology, 2005, Vanderbilt University

 As inflammation is a major mechanism of disease, we investigated the signal transduction processes induced by the key inflammatory cytokine Interleukin (IL) 1 beta as… (more)

Subjects/Keywords: IL-1beta signaling pathway; MyD88; gene expression profiling; inflammation; nuclear import; IL-1 receptor accessary protein; Interleukin-1; Cellular signal transduction

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APA (6th Edition):

Li, C. (2005). MyD88: central relay station of Interleukin 1 signaling pathway. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;

Chicago Manual of Style (16th Edition):

Li, Chunsheng. “MyD88: central relay station of Interleukin 1 signaling pathway.” 2005. Doctoral Dissertation, Vanderbilt University. Accessed October 16, 2019. http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;.

MLA Handbook (7th Edition):

Li, Chunsheng. “MyD88: central relay station of Interleukin 1 signaling pathway.” 2005. Web. 16 Oct 2019.

Vancouver:

Li C. MyD88: central relay station of Interleukin 1 signaling pathway. [Internet] [Doctoral dissertation]. Vanderbilt University; 2005. [cited 2019 Oct 16]. Available from: http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;.

Council of Science Editors:

Li C. MyD88: central relay station of Interleukin 1 signaling pathway. [Doctoral Dissertation]. Vanderbilt University; 2005. Available from: http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;


Cleveland State University

21. Narayanan, Padmini. Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion.

Degree: PhD, College of Sciences and Health Professions, 2014, Cleveland State University

 Anucleate platelets alter their transcriptome through stimulated splicing of intron and exon containing hnRNA e.g. Interleukin-1ß (IL-1ß), BCL-3. Platelet TLR4 activation upon Lipopolysaccharide (LPS) binding… (more)

Subjects/Keywords: Biology; Cellular Biology; Immunology; Molecular Biology; Platelets; Interleukin1; IL-1R1; IL-1beta; microparticles; exosomes; inflammatory response; sterile inflammation

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APA (6th Edition):

Narayanan, P. (2014). Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion. (Doctoral Dissertation). Cleveland State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=csu1425911007

Chicago Manual of Style (16th Edition):

Narayanan, Padmini. “Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion.” 2014. Doctoral Dissertation, Cleveland State University. Accessed October 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=csu1425911007.

MLA Handbook (7th Edition):

Narayanan, Padmini. “Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion.” 2014. Web. 16 Oct 2019.

Vancouver:

Narayanan P. Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion. [Internet] [Doctoral dissertation]. Cleveland State University; 2014. [cited 2019 Oct 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1425911007.

Council of Science Editors:

Narayanan P. Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion. [Doctoral Dissertation]. Cleveland State University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1425911007


University of Manchester

22. Nguyen, Loan. The role of interleukin-1 receptors in brain cell signalling.

Degree: PhD, 2010, University of Manchester

IL-1α and IL-1β are two IL-1 agonists which signals at the same receptor complex composed of IL-1R1/IL-1RAcP. However, IL-1α and IL-1β exert differential actions. A… (more)

Subjects/Keywords: 573.8; IL-1 receptors, IL-1RAcPb, IL-1alpha, IL-1beta, cytokines, inflammation, IL-6, MAP kinase, Src kinase

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APA (6th Edition):

Nguyen, L. (2010). The role of interleukin-1 receptors in brain cell signalling. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239

Chicago Manual of Style (16th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Doctoral Dissertation, University of Manchester. Accessed October 16, 2019. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239.

MLA Handbook (7th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Web. 16 Oct 2019.

Vancouver:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Internet] [Doctoral dissertation]. University of Manchester; 2010. [cited 2019 Oct 16]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239.

Council of Science Editors:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Doctoral Dissertation]. University of Manchester; 2010. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239


University of Manchester

23. Nguyen, Loan. The role of interleukin-1 receptors in brain cell signalling.

Degree: 2010, University of Manchester

IL-1α and IL-1β are two IL-1 agonists which signals at the same receptor complex composed of IL-1R1/IL-1RAcP. However, IL-1α and IL-1β exert differential actions. A… (more)

Subjects/Keywords: IL-1 receptors; IL-1RAcPb; IL-1alpha; IL-1beta; cytokines; inflammation; IL-6; MAP kinase; Src kinase

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APA (6th Edition):

Nguyen, L. (2010). The role of interleukin-1 receptors in brain cell signalling. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038

Chicago Manual of Style (16th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Doctoral Dissertation, University of Manchester. Accessed October 16, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038.

MLA Handbook (7th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Web. 16 Oct 2019.

Vancouver:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Internet] [Doctoral dissertation]. University of Manchester; 2010. [cited 2019 Oct 16]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038.

Council of Science Editors:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Doctoral Dissertation]. University of Manchester; 2010. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038


The Ohio State University

24. Norman, Greg. Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß.

Degree: MA, Psychology, 2009, The Ohio State University

  The physiological link between neuropathic pain and depression remains unknown despite a high co-morbidity between these two disorders. A mouse model of spared nerve… (more)

Subjects/Keywords: Psychobiology; Pain; oxytocin; social; IL-1beta; depression; frontal-cortex; inflammation

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APA (6th Edition):

Norman, G. (2009). Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß. (Masters Thesis). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1258626029

Chicago Manual of Style (16th Edition):

Norman, Greg. “Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß.” 2009. Masters Thesis, The Ohio State University. Accessed October 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1258626029.

MLA Handbook (7th Edition):

Norman, Greg. “Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß.” 2009. Web. 16 Oct 2019.

Vancouver:

Norman G. Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß. [Internet] [Masters thesis]. The Ohio State University; 2009. [cited 2019 Oct 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1258626029.

Council of Science Editors:

Norman G. Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß. [Masters Thesis]. The Ohio State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1258626029

25. Preira, Pascal. Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity.

Degree: Docteur es, Biophysique, 2012, Aix Marseille Université

Le Syndrome de Détresse Respiratoire Aiguë (SDRA) est une maladie inflammatoire courante en service de réanimation qui touche environ 10% des patients. Une augmentation pathologique… (more)

Subjects/Keywords: Syndrome de Détresse Respiratoire Aiguë (SDRA); Lésion Aigue Pulmonaire ALI; Microfluidique; Rigidité cellulaire; Cytokine; IL-1beta,IL-8; TNF-alpha; ARDS(acute respiratory,distress syndrome; Acute Lung Injyry; Microfluidique; Cell Stifness; Cytokine; IL-1beta,IL-8; TNF-alpha

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APA (6th Edition):

Preira, P. (2012). Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2012AIXM4020

Chicago Manual of Style (16th Edition):

Preira, Pascal. “Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity.” 2012. Doctoral Dissertation, Aix Marseille Université. Accessed October 16, 2019. http://www.theses.fr/2012AIXM4020.

MLA Handbook (7th Edition):

Preira, Pascal. “Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity.” 2012. Web. 16 Oct 2019.

Vancouver:

Preira P. Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity. [Internet] [Doctoral dissertation]. Aix Marseille Université 2012. [cited 2019 Oct 16]. Available from: http://www.theses.fr/2012AIXM4020.

Council of Science Editors:

Preira P. Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity. [Doctoral Dissertation]. Aix Marseille Université 2012. Available from: http://www.theses.fr/2012AIXM4020


University of Vienna

26. Quemada Garrido, Lucia. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.

Degree: 2017, University of Vienna

Rasmussen Enzephalitis (RE) ist eine seltene progressive Form der Epilepsie mit unbekannter Ätiologie. Die Hauptcharakteristika sind unilaterale hemisphärische Entzündung, arzneimittelresistente fokale Epilepsie und progressive neurologischer… (more)

Subjects/Keywords: 44.45 Immunologie; 42.13 Molekularbiologie; 44.90 Neurologie; Rasmussen Enzephalitis / IL-1beta / IL-1beta Rezeptor / Caspase-1 / Epilepsie / Krampfanfall / Neuroinflammation / Mikroglia; Rasmussen’s encephalitis / IL-1beta / IL-1beta receptor / Caspase-1 / epilepsy / epileptic seizure / neuroinflammation / microglia

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APA (6th Edition):

Quemada Garrido, L. (2017). Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/46783/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Quemada Garrido, Lucia. “Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.” 2017. Thesis, University of Vienna. Accessed October 16, 2019. http://othes.univie.ac.at/46783/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Quemada Garrido, Lucia. “Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.” 2017. Web. 16 Oct 2019.

Vancouver:

Quemada Garrido L. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. [Internet] [Thesis]. University of Vienna; 2017. [cited 2019 Oct 16]. Available from: http://othes.univie.ac.at/46783/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Quemada Garrido L. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. [Thesis]. University of Vienna; 2017. Available from: http://othes.univie.ac.at/46783/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Reed, Johanna Rae. Fibroblast growth factor Receptor 1-induced osteopontin regulates proinflammatory molecules to mediate cross-talk between breast cancer cells and the surrounding tumor microenvironment.

Degree: PhD, Microbiology, Immunology and Cancer Biology, 2012, University of Minnesota

 Tumor formation is an extensive process requiring complex interactions that involve both tumor cell-intrinsic pathways and soluble mediators within the microenvironment. Tumor cells exploit the… (more)

Subjects/Keywords: Breast cancer; Cell migration; CX3CL1; FGFR1; IL-1beta; Osteopontin

…stimulates gene and protein expression of IL-1β in macrophages 69 Activation of iFGFR1 in HC-11… …MECs results in decreased production of IL-1β in macrophages ..75… …dimerizer B/B...…...16 Figure 2.1: iFGFR1 activation induces IL-1β expression in the mammary gland… …of iFGFR1 and treatment with an IL-1β neutralizing antibody 39 Figure 2.3: IL-1β… …42 Figure 2.4: IL-1β and iFGFR1 induced expression of Cox-2 in HC-11/R1 cells …..45 Figure… 

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APA (6th Edition):

Reed, J. R. (2012). Fibroblast growth factor Receptor 1-induced osteopontin regulates proinflammatory molecules to mediate cross-talk between breast cancer cells and the surrounding tumor microenvironment. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/154196

Chicago Manual of Style (16th Edition):

Reed, Johanna Rae. “Fibroblast growth factor Receptor 1-induced osteopontin regulates proinflammatory molecules to mediate cross-talk between breast cancer cells and the surrounding tumor microenvironment.” 2012. Doctoral Dissertation, University of Minnesota. Accessed October 16, 2019. http://purl.umn.edu/154196.

MLA Handbook (7th Edition):

Reed, Johanna Rae. “Fibroblast growth factor Receptor 1-induced osteopontin regulates proinflammatory molecules to mediate cross-talk between breast cancer cells and the surrounding tumor microenvironment.” 2012. Web. 16 Oct 2019.

Vancouver:

Reed JR. Fibroblast growth factor Receptor 1-induced osteopontin regulates proinflammatory molecules to mediate cross-talk between breast cancer cells and the surrounding tumor microenvironment. [Internet] [Doctoral dissertation]. University of Minnesota; 2012. [cited 2019 Oct 16]. Available from: http://purl.umn.edu/154196.

Council of Science Editors:

Reed JR. Fibroblast growth factor Receptor 1-induced osteopontin regulates proinflammatory molecules to mediate cross-talk between breast cancer cells and the surrounding tumor microenvironment. [Doctoral Dissertation]. University of Minnesota; 2012. Available from: http://purl.umn.edu/154196

28. Shamaa, Obada. Intracellular and extracellular regulation of the inflammatory protease caspase-1.

Degree: PhD, Biomedical Sciences, 2014, The Ohio State University

 Caspase-1 is an inflammatory cysteine protease which cleaves the pro-inflammatory cytokines, interleukin-1beta (IL-1beta) and IL-18, into their mature, bioactive forms. In addition, caspase-1 plays a… (more)

Subjects/Keywords: Immunology; Medicine; Biochemistry; Biomedical Research; caspase-1; inflammasome; IL-1beta; IL-18; mitochondria; calcium; monocyte; THP1

…antagonist interleukin-1beta inhibitory CARD IL-1 receptor associated kinase lymphocyte activating… …9 1.2 Interleukin-1β (IL-1β)… …11 1.2.1 Structure of IL-1β… …14 1.2.2 Interleukin-18 (IL-18)… …8 Figure 1.2 Structure and caspase-1 cleavage site for IL-1β and IL-18… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shamaa, O. (2014). Intracellular and extracellular regulation of the inflammatory protease caspase-1. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292

Chicago Manual of Style (16th Edition):

Shamaa, Obada. “Intracellular and extracellular regulation of the inflammatory protease caspase-1.” 2014. Doctoral Dissertation, The Ohio State University. Accessed October 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292.

MLA Handbook (7th Edition):

Shamaa, Obada. “Intracellular and extracellular regulation of the inflammatory protease caspase-1.” 2014. Web. 16 Oct 2019.

Vancouver:

Shamaa O. Intracellular and extracellular regulation of the inflammatory protease caspase-1. [Internet] [Doctoral dissertation]. The Ohio State University; 2014. [cited 2019 Oct 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292.

Council of Science Editors:

Shamaa O. Intracellular and extracellular regulation of the inflammatory protease caspase-1. [Doctoral Dissertation]. The Ohio State University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292


Case Western Reserve University

29. Shive, Carey Lynn. The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease.

Degree: PhD, Pathology, 2014, Case Western Reserve University

 HIV disease is characterized by the disruption of immune homeostasis and persistent immune activation. The use of anti-retroviral therapy dramatically reduces mortality caused by HIV,… (more)

Subjects/Keywords: Biomedical Research; Immunology; HIV; immune failure; IL-6; IL-1beta; IL-7; inflammation

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APA (6th Edition):

Shive, C. L. (2014). The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1396627515

Chicago Manual of Style (16th Edition):

Shive, Carey Lynn. “The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease.” 2014. Doctoral Dissertation, Case Western Reserve University. Accessed October 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1396627515.

MLA Handbook (7th Edition):

Shive, Carey Lynn. “The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease.” 2014. Web. 16 Oct 2019.

Vancouver:

Shive CL. The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2014. [cited 2019 Oct 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1396627515.

Council of Science Editors:

Shive CL. The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease. [Doctoral Dissertation]. Case Western Reserve University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1396627515


Queens University

30. Simmatis, Leif. The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia .

Degree: Neuroscience Studies, 2015, Queens University

 The pannexin-1 (panx1) hemichannel is associated with the Nod-Like Receptor Protein-1 (NLRP1) inflammasome following MCAO in a mouse model of focal ischaemic stroke. We investigated… (more)

Subjects/Keywords: Hemichannel; IL-1beta; neuroinflammation; NLRP1; stroke; ischemia; pannexin-1; interleukin-1beta

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APA (6th Edition):

Simmatis, L. (2015). The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/13868

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Simmatis, Leif. “The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia .” 2015. Thesis, Queens University. Accessed October 16, 2019. http://hdl.handle.net/1974/13868.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Simmatis, Leif. “The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia .” 2015. Web. 16 Oct 2019.

Vancouver:

Simmatis L. The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia . [Internet] [Thesis]. Queens University; 2015. [cited 2019 Oct 16]. Available from: http://hdl.handle.net/1974/13868.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Simmatis L. The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia . [Thesis]. Queens University; 2015. Available from: http://hdl.handle.net/1974/13868

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2]

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