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You searched for subject:(IL 1beta). Showing records 1 – 30 of 33 total matches.

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Queens University

1. Simmatis, Leif. The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia .

Degree: Neuroscience Studies, 2015, Queens University

 The pannexin-1 (panx1) hemichannel is associated with the Nod-Like Receptor Protein-1 (NLRP1) inflammasome following MCAO in a mouse model of focal ischaemic stroke. We investigated… (more)

Subjects/Keywords: Hemichannel; IL-1beta; neuroinflammation; NLRP1; stroke; ischemia; pannexin-1; interleukin-1beta

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Simmatis, L. (2015). The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/13868

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Simmatis, Leif. “The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia .” 2015. Thesis, Queens University. Accessed September 23, 2019. http://hdl.handle.net/1974/13868.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Simmatis, Leif. “The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia .” 2015. Web. 23 Sep 2019.

Vancouver:

Simmatis L. The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia . [Internet] [Thesis]. Queens University; 2015. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/1974/13868.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Simmatis L. The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia . [Thesis]. Queens University; 2015. Available from: http://hdl.handle.net/1974/13868

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

2. Chesnokov, Anton P, Mr. Modulation of NALP3 Inflammasome Genes by Estrogen.

Degree: MS, Biology, 2011, Georgia State University

  Immunity is known to be sexually dimorphic. This dimorphism may be attributed to the action of different hormones. Aluminum is a component of several… (more)

Subjects/Keywords: Nalp3 inflammasome; Estrogen; IL-1beta; Aluminum hydroxide

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APA (6th Edition):

Chesnokov, Anton P, M. (2011). Modulation of NALP3 Inflammasome Genes by Estrogen. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/33

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chesnokov, Anton P, Mr. “Modulation of NALP3 Inflammasome Genes by Estrogen.” 2011. Thesis, Georgia State University. Accessed September 23, 2019. https://scholarworks.gsu.edu/biology_theses/33.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chesnokov, Anton P, Mr. “Modulation of NALP3 Inflammasome Genes by Estrogen.” 2011. Web. 23 Sep 2019.

Vancouver:

Chesnokov, Anton P M. Modulation of NALP3 Inflammasome Genes by Estrogen. [Internet] [Thesis]. Georgia State University; 2011. [cited 2019 Sep 23]. Available from: https://scholarworks.gsu.edu/biology_theses/33.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chesnokov, Anton P M. Modulation of NALP3 Inflammasome Genes by Estrogen. [Thesis]. Georgia State University; 2011. Available from: https://scholarworks.gsu.edu/biology_theses/33

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Gómez Ochoa, Pablo. Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes.

Degree: 2011, Universidad de Zaragoza

 El diseño de este trabajo intenta establecer las condiciones de cultivo y la metodología para la evaluación del grupo de las Stem Cells adultas y… (more)

Subjects/Keywords: Campos electromagnéticos Pulsados; Stem Cells; TNF-alfa; IL-1beta; IL-10; PEMF; Stem Cells; TNF-alfa; IL-1beta; IL-10

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APA (6th Edition):

Gómez Ochoa, P. (2011). Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes. (Thesis). Universidad de Zaragoza. Retrieved from http://zaguan.unizar.es/record/4497

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gómez Ochoa, Pablo. “Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes.” 2011. Thesis, Universidad de Zaragoza. Accessed September 23, 2019. http://zaguan.unizar.es/record/4497.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gómez Ochoa, Pablo. “Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes.” 2011. Web. 23 Sep 2019.

Vancouver:

Gómez Ochoa P. Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes. [Internet] [Thesis]. Universidad de Zaragoza; 2011. [cited 2019 Sep 23]. Available from: http://zaguan.unizar.es/record/4497.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gómez Ochoa P. Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes. [Thesis]. Universidad de Zaragoza; 2011. Available from: http://zaguan.unizar.es/record/4497

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Case Western Reserve University

4. Shive, Carey Lynn. The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease.

Degree: PhD, Pathology, 2014, Case Western Reserve University

 HIV disease is characterized by the disruption of immune homeostasis and persistent immune activation. The use of anti-retroviral therapy dramatically reduces mortality caused by HIV,… (more)

Subjects/Keywords: Biomedical Research; Immunology; HIV; immune failure; IL-6; IL-1beta; IL-7; inflammation

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APA (6th Edition):

Shive, C. L. (2014). The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1396627515

Chicago Manual of Style (16th Edition):

Shive, Carey Lynn. “The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease.” 2014. Doctoral Dissertation, Case Western Reserve University. Accessed September 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1396627515.

MLA Handbook (7th Edition):

Shive, Carey Lynn. “The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease.” 2014. Web. 23 Sep 2019.

Vancouver:

Shive CL. The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2014. [cited 2019 Sep 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1396627515.

Council of Science Editors:

Shive CL. The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease. [Doctoral Dissertation]. Case Western Reserve University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1396627515


University of Manchester

5. Nguyen, Loan. The role of interleukin-1 receptors in brain cell signalling.

Degree: PhD, 2010, University of Manchester

IL-1α and IL-1β are two IL-1 agonists which signals at the same receptor complex composed of IL-1R1/IL-1RAcP. However, IL-1α and IL-1β exert differential actions. A… (more)

Subjects/Keywords: 573.8; IL-1 receptors, IL-1RAcPb, IL-1alpha, IL-1beta, cytokines, inflammation, IL-6, MAP kinase, Src kinase

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APA (6th Edition):

Nguyen, L. (2010). The role of interleukin-1 receptors in brain cell signalling. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239

Chicago Manual of Style (16th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Doctoral Dissertation, University of Manchester. Accessed September 23, 2019. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239.

MLA Handbook (7th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Web. 23 Sep 2019.

Vancouver:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Internet] [Doctoral dissertation]. University of Manchester; 2010. [cited 2019 Sep 23]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239.

Council of Science Editors:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Doctoral Dissertation]. University of Manchester; 2010. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239


University of Manchester

6. Nguyen, Loan. The role of interleukin-1 receptors in brain cell signalling.

Degree: 2010, University of Manchester

IL-1α and IL-1β are two IL-1 agonists which signals at the same receptor complex composed of IL-1R1/IL-1RAcP. However, IL-1α and IL-1β exert differential actions. A… (more)

Subjects/Keywords: IL-1 receptors; IL-1RAcPb; IL-1alpha; IL-1beta; cytokines; inflammation; IL-6; MAP kinase; Src kinase

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APA (6th Edition):

Nguyen, L. (2010). The role of interleukin-1 receptors in brain cell signalling. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038

Chicago Manual of Style (16th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Doctoral Dissertation, University of Manchester. Accessed September 23, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038.

MLA Handbook (7th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Web. 23 Sep 2019.

Vancouver:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Internet] [Doctoral dissertation]. University of Manchester; 2010. [cited 2019 Sep 23]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038.

Council of Science Editors:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Doctoral Dissertation]. University of Manchester; 2010. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038


University of Manchester

7. Englezou, Pavlos. P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages.

Degree: PhD, 2013, University of Manchester

 The P2X7R is a functionally distinct member of the P2X non-selective cation channels and has been implicated in the initiation of immune responses. One of… (more)

Subjects/Keywords: 616.07; P2X7R; Dendritic cells; Macrophages; IL-1beta; IL-1alpha; P2X7R splice variants

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APA (6th Edition):

Englezou, P. (2013). P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/p2x7rdriven-il1-responses-in-differentiated-murine-dendritic-cells-comparison-with-macrophages(81ddc8a9-6d43-4fe6-b9ec-e2b1b9a156a4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603121

Chicago Manual of Style (16th Edition):

Englezou, Pavlos. “P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages.” 2013. Doctoral Dissertation, University of Manchester. Accessed September 23, 2019. https://www.research.manchester.ac.uk/portal/en/theses/p2x7rdriven-il1-responses-in-differentiated-murine-dendritic-cells-comparison-with-macrophages(81ddc8a9-6d43-4fe6-b9ec-e2b1b9a156a4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603121.

MLA Handbook (7th Edition):

Englezou, Pavlos. “P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages.” 2013. Web. 23 Sep 2019.

Vancouver:

Englezou P. P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2019 Sep 23]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/p2x7rdriven-il1-responses-in-differentiated-murine-dendritic-cells-comparison-with-macrophages(81ddc8a9-6d43-4fe6-b9ec-e2b1b9a156a4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603121.

Council of Science Editors:

Englezou P. P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages. [Doctoral Dissertation]. University of Manchester; 2013. Available from: https://www.research.manchester.ac.uk/portal/en/theses/p2x7rdriven-il1-responses-in-differentiated-murine-dendritic-cells-comparison-with-macrophages(81ddc8a9-6d43-4fe6-b9ec-e2b1b9a156a4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603121


The Ohio State University

8. Norman, Greg. Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß.

Degree: MA, Psychology, 2009, The Ohio State University

  The physiological link between neuropathic pain and depression remains unknown despite a high co-morbidity between these two disorders. A mouse model of spared nerve… (more)

Subjects/Keywords: Psychobiology; Pain; oxytocin; social; IL-1beta; depression; frontal-cortex; inflammation

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APA (6th Edition):

Norman, G. (2009). Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß. (Masters Thesis). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1258626029

Chicago Manual of Style (16th Edition):

Norman, Greg. “Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß.” 2009. Masters Thesis, The Ohio State University. Accessed September 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1258626029.

MLA Handbook (7th Edition):

Norman, Greg. “Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß.” 2009. Web. 23 Sep 2019.

Vancouver:

Norman G. Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß. [Internet] [Masters thesis]. The Ohio State University; 2009. [cited 2019 Sep 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1258626029.

Council of Science Editors:

Norman G. Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß. [Masters Thesis]. The Ohio State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1258626029


University of Vienna

9. Quemada Garrido, Lucia. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.

Degree: 2017, University of Vienna

Rasmussen Enzephalitis (RE) ist eine seltene progressive Form der Epilepsie mit unbekannter Ätiologie. Die Hauptcharakteristika sind unilaterale hemisphärische Entzündung, arzneimittelresistente fokale Epilepsie und progressive neurologischer… (more)

Subjects/Keywords: 44.45 Immunologie; 42.13 Molekularbiologie; 44.90 Neurologie; Rasmussen Enzephalitis / IL-1beta / IL-1beta Rezeptor / Caspase-1 / Epilepsie / Krampfanfall / Neuroinflammation / Mikroglia; Rasmussen’s encephalitis / IL-1beta / IL-1beta receptor / Caspase-1 / epilepsy / epileptic seizure / neuroinflammation / microglia

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APA (6th Edition):

Quemada Garrido, L. (2017). Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/46783/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Quemada Garrido, Lucia. “Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.” 2017. Thesis, University of Vienna. Accessed September 23, 2019. http://othes.univie.ac.at/46783/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Quemada Garrido, Lucia. “Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.” 2017. Web. 23 Sep 2019.

Vancouver:

Quemada Garrido L. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. [Internet] [Thesis]. University of Vienna; 2017. [cited 2019 Sep 23]. Available from: http://othes.univie.ac.at/46783/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Quemada Garrido L. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. [Thesis]. University of Vienna; 2017. Available from: http://othes.univie.ac.at/46783/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Bernard, Marine. Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors.

Degree: Docteur es, Immunologie, 2014, Université Claude Bernard – Lyon I

La dermatite atopique (DA) est une dermatose inflammatoire dont la prévalence élevée est en constante augmentation dans les pays industrialisés. La physiopathologie de la DA… (more)

Subjects/Keywords: Dermatite atopique; Épiderme reconstruit humain; Apoptose; UV; IL-1beta; TSLP; Dermatophagoïdes farinae; Atopic dermatitis; Reconstructed human epidermis; Apoptosis; UV; IL-1beta; TSLP; Dermatophagoïdes farinae; 571.96

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APA (6th Edition):

Bernard, M. (2014). Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2014LYO10361

Chicago Manual of Style (16th Edition):

Bernard, Marine. “Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors.” 2014. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed September 23, 2019. http://www.theses.fr/2014LYO10361.

MLA Handbook (7th Edition):

Bernard, Marine. “Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors.” 2014. Web. 23 Sep 2019.

Vancouver:

Bernard M. Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2014. [cited 2019 Sep 23]. Available from: http://www.theses.fr/2014LYO10361.

Council of Science Editors:

Bernard M. Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2014. Available from: http://www.theses.fr/2014LYO10361

11. Williams, Clarence David. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2012, University of Kansas

 Acetaminophen (APAP) overdose is the leading cause of acute liver failure in the US. APAP is metabolized to a reactive metabolite that causes hepatotoxicity in… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Il-1beta; Neutrophil

…event in toxicity and if the production of IL-1β modulates neutrophil activity and function… …induced toxic insult the formation of cytokines such as TNF-α, IL-1β and others, has been well… …antibodies attenuated cytokine and chemokine (TNF-α, MCP-1, IL-6) formation (Antoine… …al., 2009). It was hypothesized that interleukin-1α and IL-1β are critical mediators… …of APAP hepatotoxicity based on the observation that IL-1 receptor-deficient (IL-1R1… 

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APA (6th Edition):

Williams, C. D. (2012). Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/10290

Chicago Manual of Style (16th Edition):

Williams, Clarence David. “Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.” 2012. Doctoral Dissertation, University of Kansas. Accessed September 23, 2019. http://hdl.handle.net/1808/10290.

MLA Handbook (7th Edition):

Williams, Clarence David. “Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.” 2012. Web. 23 Sep 2019.

Vancouver:

Williams CD. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/1808/10290.

Council of Science Editors:

Williams CD. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/10290


Universiteit Utrecht

12. Kleibeuker, W. GRK2 deficiency is a pain.

Degree: 2007, Universiteit Utrecht

 G protein-coupled receptor (GPCR) kinase (GRK)2 regulates the responsiveness of many GPCRS, e.g. CCR1, CCR2, CCR5, neurokinin-1 receptor, and metabotropic glutamate receptors, which are involved… (more)

Subjects/Keywords: Geneeskunde; GRK2; GPCR; hyperalgesia; allodynia; IL-1beta; signaling; inflammatory pain; spinal cord; nerve damage; astrocytes

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APA (6th Edition):

Kleibeuker, W. (2007). GRK2 deficiency is a pain. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/25012

Chicago Manual of Style (16th Edition):

Kleibeuker, W. “GRK2 deficiency is a pain.” 2007. Doctoral Dissertation, Universiteit Utrecht. Accessed September 23, 2019. http://dspace.library.uu.nl:8080/handle/1874/25012.

MLA Handbook (7th Edition):

Kleibeuker, W. “GRK2 deficiency is a pain.” 2007. Web. 23 Sep 2019.

Vancouver:

Kleibeuker W. GRK2 deficiency is a pain. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2007. [cited 2019 Sep 23]. Available from: http://dspace.library.uu.nl:8080/handle/1874/25012.

Council of Science Editors:

Kleibeuker W. GRK2 deficiency is a pain. [Doctoral Dissertation]. Universiteit Utrecht; 2007. Available from: http://dspace.library.uu.nl:8080/handle/1874/25012


Loyola University Chicago

13. Zack, Stephanie. Lidocaine Attenuates an Induced Inflammatory Response.

Degree: MS, Microbiology and Immunology, 2018, Loyola University Chicago

  Inflammation is common to the pathogenesis of a variety of diseases. Therapeutics which can regulate and reduce damaging inflammation are therefore valuable in the… (more)

Subjects/Keywords: IL-1beta; inflammation; lidocaine; NLRP3 inflammasome; toll-like receptor 4; Immunology and Infectious Disease

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APA (6th Edition):

Zack, S. (2018). Lidocaine Attenuates an Induced Inflammatory Response. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/3714

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zack, Stephanie. “Lidocaine Attenuates an Induced Inflammatory Response.” 2018. Thesis, Loyola University Chicago. Accessed September 23, 2019. https://ecommons.luc.edu/luc_theses/3714.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zack, Stephanie. “Lidocaine Attenuates an Induced Inflammatory Response.” 2018. Web. 23 Sep 2019.

Vancouver:

Zack S. Lidocaine Attenuates an Induced Inflammatory Response. [Internet] [Thesis]. Loyola University Chicago; 2018. [cited 2019 Sep 23]. Available from: https://ecommons.luc.edu/luc_theses/3714.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zack S. Lidocaine Attenuates an Induced Inflammatory Response. [Thesis]. Loyola University Chicago; 2018. Available from: https://ecommons.luc.edu/luc_theses/3714

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cleveland State University

14. Narayanan, Padmini. Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion.

Degree: PhD, College of Sciences and Health Professions, 2014, Cleveland State University

 Anucleate platelets alter their transcriptome through stimulated splicing of intron and exon containing hnRNA e.g. Interleukin-1ß (IL-1ß), BCL-3. Platelet TLR4 activation upon Lipopolysaccharide (LPS) binding… (more)

Subjects/Keywords: Biology; Cellular Biology; Immunology; Molecular Biology; Platelets; Interleukin1; IL-1R1; IL-1beta; microparticles; exosomes; inflammatory response; sterile inflammation

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APA (6th Edition):

Narayanan, P. (2014). Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion. (Doctoral Dissertation). Cleveland State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=csu1425911007

Chicago Manual of Style (16th Edition):

Narayanan, Padmini. “Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion.” 2014. Doctoral Dissertation, Cleveland State University. Accessed September 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=csu1425911007.

MLA Handbook (7th Edition):

Narayanan, Padmini. “Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion.” 2014. Web. 23 Sep 2019.

Vancouver:

Narayanan P. Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion. [Internet] [Doctoral dissertation]. Cleveland State University; 2014. [cited 2019 Sep 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1425911007.

Council of Science Editors:

Narayanan P. Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion. [Doctoral Dissertation]. Cleveland State University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1425911007

15. Dumont, Adélie. L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta.

Degree: Docteur es, Physiologie, 2018, Bourgogne Franche-Comté

Selon une étude précédente, une limitation à l'efficacité anticancéreuse du 5-Fluorouracile (5-FU) repose sur la sécrétion d'IL-1β par des cellules myéloïdes immunosuppressives (MDSC). La libération… (more)

Subjects/Keywords: Mdsc; 5 fluorouracil; Dha; IL-1 beta; Jnk; Mdsc; 5-Fluorouracil; Dha; IL-1beta; Jnk; 612

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APA (6th Edition):

Dumont, A. (2018). L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta. (Doctoral Dissertation). Bourgogne Franche-Comté. Retrieved from http://www.theses.fr/2018UBFCI013

Chicago Manual of Style (16th Edition):

Dumont, Adélie. “L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta.” 2018. Doctoral Dissertation, Bourgogne Franche-Comté. Accessed September 23, 2019. http://www.theses.fr/2018UBFCI013.

MLA Handbook (7th Edition):

Dumont, Adélie. “L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta.” 2018. Web. 23 Sep 2019.

Vancouver:

Dumont A. L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta. [Internet] [Doctoral dissertation]. Bourgogne Franche-Comté; 2018. [cited 2019 Sep 23]. Available from: http://www.theses.fr/2018UBFCI013.

Council of Science Editors:

Dumont A. L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta. [Doctoral Dissertation]. Bourgogne Franche-Comté; 2018. Available from: http://www.theses.fr/2018UBFCI013


University of Manitoba

16. DRAGON, Stephane. Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils.

Degree: Immunology, 2010, University of Manitoba

 Immunopathological disorders are no longer defined by dysregulated T-helper (Th) type 1/ Th2 responses but account for modulatory cell types such as regulatory and Th17… (more)

Subjects/Keywords: Human; Inflammation; Cytokines; IL-17; IL-1beta; IL-22; CXCL-8; airway smooth muscle cells; neutrophils; IL-17R; signal transduction; gene expression; apoptosis

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APA (6th Edition):

DRAGON, S. (2010). Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/3945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DRAGON, Stephane. “Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils.” 2010. Thesis, University of Manitoba. Accessed September 23, 2019. http://hdl.handle.net/1993/3945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DRAGON, Stephane. “Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils.” 2010. Web. 23 Sep 2019.

Vancouver:

DRAGON S. Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils. [Internet] [Thesis]. University of Manitoba; 2010. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/1993/3945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DRAGON S. Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils. [Thesis]. University of Manitoba; 2010. Available from: http://hdl.handle.net/1993/3945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Preira, Pascal. Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity.

Degree: Docteur es, Biophysique, 2012, Aix Marseille Université

Le Syndrome de Détresse Respiratoire Aiguë (SDRA) est une maladie inflammatoire courante en service de réanimation qui touche environ 10% des patients. Une augmentation pathologique… (more)

Subjects/Keywords: Syndrome de Détresse Respiratoire Aiguë (SDRA); Lésion Aigue Pulmonaire ALI; Microfluidique; Rigidité cellulaire; Cytokine; IL-1beta,IL-8; TNF-alpha; ARDS(acute respiratory,distress syndrome; Acute Lung Injyry; Microfluidique; Cell Stifness; Cytokine; IL-1beta,IL-8; TNF-alpha

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APA (6th Edition):

Preira, P. (2012). Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2012AIXM4020

Chicago Manual of Style (16th Edition):

Preira, Pascal. “Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity.” 2012. Doctoral Dissertation, Aix Marseille Université. Accessed September 23, 2019. http://www.theses.fr/2012AIXM4020.

MLA Handbook (7th Edition):

Preira, Pascal. “Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity.” 2012. Web. 23 Sep 2019.

Vancouver:

Preira P. Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity. [Internet] [Doctoral dissertation]. Aix Marseille Université 2012. [cited 2019 Sep 23]. Available from: http://www.theses.fr/2012AIXM4020.

Council of Science Editors:

Preira P. Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity. [Doctoral Dissertation]. Aix Marseille Université 2012. Available from: http://www.theses.fr/2012AIXM4020

18. Symington, Jane. Role of ATG16L1 in Uropathogenic E. Coli Pathogenesis.

Degree: PhD, Biology & Biomedical Sciences (Molecular Microbiology & Microbial Pathogenesis), 2016, Washington University in St. Louis

  Urinary tract infections (UTIs) are among the most common infectious diseases and are primarily caused by uropathogenic E. coli (UPEC). Given the greater incidence… (more)

Subjects/Keywords: ATG16L1, autophagy, bladder, IL-1beta, macrophage, UTI; Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology; Microbiology

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APA (6th Edition):

Symington, J. (2016). Role of ATG16L1 in Uropathogenic E. Coli Pathogenesis. (Doctoral Dissertation). Washington University in St. Louis. Retrieved from https://openscholarship.wustl.edu/art_sci_etds/764

Chicago Manual of Style (16th Edition):

Symington, Jane. “Role of ATG16L1 in Uropathogenic E. Coli Pathogenesis.” 2016. Doctoral Dissertation, Washington University in St. Louis. Accessed September 23, 2019. https://openscholarship.wustl.edu/art_sci_etds/764.

MLA Handbook (7th Edition):

Symington, Jane. “Role of ATG16L1 in Uropathogenic E. Coli Pathogenesis.” 2016. Web. 23 Sep 2019.

Vancouver:

Symington J. Role of ATG16L1 in Uropathogenic E. Coli Pathogenesis. [Internet] [Doctoral dissertation]. Washington University in St. Louis; 2016. [cited 2019 Sep 23]. Available from: https://openscholarship.wustl.edu/art_sci_etds/764.

Council of Science Editors:

Symington J. Role of ATG16L1 in Uropathogenic E. Coli Pathogenesis. [Doctoral Dissertation]. Washington University in St. Louis; 2016. Available from: https://openscholarship.wustl.edu/art_sci_etds/764


Kyoto University

19. Kanamori, Yohei. Transcriptional regulation of hepcidin by molecules mediating inflammatory responses .

Degree: 2018, Kyoto University

Subjects/Keywords: hepcidin; AP-1; activin B; IL-1beta; inflammation

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APA (6th Edition):

Kanamori, Y. (2018). Transcriptional regulation of hepcidin by molecules mediating inflammatory responses . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/232336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kanamori, Yohei. “Transcriptional regulation of hepcidin by molecules mediating inflammatory responses .” 2018. Thesis, Kyoto University. Accessed September 23, 2019. http://hdl.handle.net/2433/232336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kanamori, Yohei. “Transcriptional regulation of hepcidin by molecules mediating inflammatory responses .” 2018. Web. 23 Sep 2019.

Vancouver:

Kanamori Y. Transcriptional regulation of hepcidin by molecules mediating inflammatory responses . [Internet] [Thesis]. Kyoto University; 2018. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/2433/232336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kanamori Y. Transcriptional regulation of hepcidin by molecules mediating inflammatory responses . [Thesis]. Kyoto University; 2018. Available from: http://hdl.handle.net/2433/232336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

20. Kuiper, J.J.W. Reactive Oxygen Species in Inflammasome Activation.

Degree: 2009, Universiteit Utrecht

 The fundamental role of the innate immune system is to form the first barrier in bacterial and viral defence. It is based upon the recognition… (more)

Subjects/Keywords: Geneeskunde; NOD-like receptor, P2x7-receptor, NLRP3, Reactive Oxygen Species, ROS, Caspase-1, IL-1beta, IL-18, Inflammasome, ERK1/2, Cryopyrinopathies, CAPS

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APA (6th Edition):

Kuiper, J. J. W. (2009). Reactive Oxygen Species in Inflammasome Activation. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/35211

Chicago Manual of Style (16th Edition):

Kuiper, J J W. “Reactive Oxygen Species in Inflammasome Activation.” 2009. Masters Thesis, Universiteit Utrecht. Accessed September 23, 2019. http://dspace.library.uu.nl:8080/handle/1874/35211.

MLA Handbook (7th Edition):

Kuiper, J J W. “Reactive Oxygen Species in Inflammasome Activation.” 2009. Web. 23 Sep 2019.

Vancouver:

Kuiper JJW. Reactive Oxygen Species in Inflammasome Activation. [Internet] [Masters thesis]. Universiteit Utrecht; 2009. [cited 2019 Sep 23]. Available from: http://dspace.library.uu.nl:8080/handle/1874/35211.

Council of Science Editors:

Kuiper JJW. Reactive Oxygen Species in Inflammasome Activation. [Masters Thesis]. Universiteit Utrecht; 2009. Available from: http://dspace.library.uu.nl:8080/handle/1874/35211


Case Western Reserve University

21. Kahlenberg, Joanne Michelle. Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands.

Degree: PhD, Pathology, 2004, Case Western Reserve University

 Recent advances in understanding of the biology underlying hereditary inflammatory diseases have focused attention on the importance of regulating levels of the proinflammatory cytokine IL-1beta(more)

Subjects/Keywords: Caspase-1; IL-1beta; Inflammasome; Macrophage; Monocyte; Potassium; Toll-Like Receptors

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APA (6th Edition):

Kahlenberg, J. M. (2004). Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614

Chicago Manual of Style (16th Edition):

Kahlenberg, Joanne Michelle. “Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands.” 2004. Doctoral Dissertation, Case Western Reserve University. Accessed September 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614.

MLA Handbook (7th Edition):

Kahlenberg, Joanne Michelle. “Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands.” 2004. Web. 23 Sep 2019.

Vancouver:

Kahlenberg JM. Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2004. [cited 2019 Sep 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614.

Council of Science Editors:

Kahlenberg JM. Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands. [Doctoral Dissertation]. Case Western Reserve University; 2004. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614


University of Oxford

22. Yip, Ronald H. N. Mechanisms of induction and modulation of the pro-inflammatory cytokine interleukin-1beta.

Degree: PhD, 2012, University of Oxford

 Interleukin (IL)-1beta is a powerful pro-inflammatory cytokine with important roles in directing both innate and adaptive immunity. As a result, its production is tightly controlled,… (more)

Subjects/Keywords: 616.07; Life Sciences; Biology; Cell Biology (see also Plant sciences); Medical Sciences; Infectious diseases; Biology (medical sciences); Immunology; Viruses; Inflammasome; Serum Amyloid A; IL-1beta; iNKT cells; Influenza

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APA (6th Edition):

Yip, R. H. N. (2012). Mechanisms of induction and modulation of the pro-inflammatory cytokine interleukin-1beta. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:14e4bd37-d3f3-4419-b2f0-564c22df4d09 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.580980

Chicago Manual of Style (16th Edition):

Yip, Ronald H N. “Mechanisms of induction and modulation of the pro-inflammatory cytokine interleukin-1beta.” 2012. Doctoral Dissertation, University of Oxford. Accessed September 23, 2019. http://ora.ox.ac.uk/objects/uuid:14e4bd37-d3f3-4419-b2f0-564c22df4d09 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.580980.

MLA Handbook (7th Edition):

Yip, Ronald H N. “Mechanisms of induction and modulation of the pro-inflammatory cytokine interleukin-1beta.” 2012. Web. 23 Sep 2019.

Vancouver:

Yip RHN. Mechanisms of induction and modulation of the pro-inflammatory cytokine interleukin-1beta. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2019 Sep 23]. Available from: http://ora.ox.ac.uk/objects/uuid:14e4bd37-d3f3-4419-b2f0-564c22df4d09 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.580980.

Council of Science Editors:

Yip RHN. Mechanisms of induction and modulation of the pro-inflammatory cytokine interleukin-1beta. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:14e4bd37-d3f3-4419-b2f0-564c22df4d09 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.580980


University of New South Wales

23. Zhang, Ning. Transcriptional regulation of PDGF receptor alpha in fibroblasts.

Degree: Centre for Vascular Research, 2011, University of New South Wales

 Wound healing is a process of stimulating cell growth and proliferation to achieve the result of repair. This process is controlled by cytokines and growth… (more)

Subjects/Keywords: PDGF receptor alpha; fibroblasts; interleukin 1 beta (IL-1beta); tumour necrosis factor alpha (TNF-alpha); receptor alpha (PDGF-R-alpha); wound healing

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APA (6th Edition):

Zhang, N. (2011). Transcriptional regulation of PDGF receptor alpha in fibroblasts. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Zhang, Ning. “Transcriptional regulation of PDGF receptor alpha in fibroblasts.” 2011. Doctoral Dissertation, University of New South Wales. Accessed September 23, 2019. http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true.

MLA Handbook (7th Edition):

Zhang, Ning. “Transcriptional regulation of PDGF receptor alpha in fibroblasts.” 2011. Web. 23 Sep 2019.

Vancouver:

Zhang N. Transcriptional regulation of PDGF receptor alpha in fibroblasts. [Internet] [Doctoral dissertation]. University of New South Wales; 2011. [cited 2019 Sep 23]. Available from: http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true.

Council of Science Editors:

Zhang N. Transcriptional regulation of PDGF receptor alpha in fibroblasts. [Doctoral Dissertation]. University of New South Wales; 2011. Available from: http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true

24. Reed, Johanna Rae. Fibroblast growth factor Receptor 1-induced osteopontin regulates proinflammatory molecules to mediate cross-talk between breast cancer cells and the surrounding tumor microenvironment.

Degree: PhD, Microbiology, Immunology and Cancer Biology, 2012, University of Minnesota

 Tumor formation is an extensive process requiring complex interactions that involve both tumor cell-intrinsic pathways and soluble mediators within the microenvironment. Tumor cells exploit the… (more)

Subjects/Keywords: Breast cancer; Cell migration; CX3CL1; FGFR1; IL-1beta; Osteopontin

…stimulates gene and protein expression of IL-1β in macrophages 69 Activation of iFGFR1 in HC-11… …MECs results in decreased production of IL-1β in macrophages ..75… …dimerizer B/B...…...16 Figure 2.1: iFGFR1 activation induces IL-1β expression in the mammary gland… …of iFGFR1 and treatment with an IL-1β neutralizing antibody 39 Figure 2.3: IL-1β… …42 Figure 2.4: IL-1β and iFGFR1 induced expression of Cox-2 in HC-11/R1 cells …..45 Figure… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Reed, J. R. (2012). Fibroblast growth factor Receptor 1-induced osteopontin regulates proinflammatory molecules to mediate cross-talk between breast cancer cells and the surrounding tumor microenvironment. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/154196

Chicago Manual of Style (16th Edition):

Reed, Johanna Rae. “Fibroblast growth factor Receptor 1-induced osteopontin regulates proinflammatory molecules to mediate cross-talk between breast cancer cells and the surrounding tumor microenvironment.” 2012. Doctoral Dissertation, University of Minnesota. Accessed September 23, 2019. http://purl.umn.edu/154196.

MLA Handbook (7th Edition):

Reed, Johanna Rae. “Fibroblast growth factor Receptor 1-induced osteopontin regulates proinflammatory molecules to mediate cross-talk between breast cancer cells and the surrounding tumor microenvironment.” 2012. Web. 23 Sep 2019.

Vancouver:

Reed JR. Fibroblast growth factor Receptor 1-induced osteopontin regulates proinflammatory molecules to mediate cross-talk between breast cancer cells and the surrounding tumor microenvironment. [Internet] [Doctoral dissertation]. University of Minnesota; 2012. [cited 2019 Sep 23]. Available from: http://purl.umn.edu/154196.

Council of Science Editors:

Reed JR. Fibroblast growth factor Receptor 1-induced osteopontin regulates proinflammatory molecules to mediate cross-talk between breast cancer cells and the surrounding tumor microenvironment. [Doctoral Dissertation]. University of Minnesota; 2012. Available from: http://purl.umn.edu/154196


University of Lund

25. Westling, Sofie. Metabolic alterations in patients with self-inflicted aggressive behaviour.

Degree: 2009, University of Lund

 Self-inflicted aggressive behaviour is a cross diagnostic phenomenon of major clinical relevance in psychiatric setting. Accumulating evidence speaks for a role of metabolic and immunological… (more)

Subjects/Keywords: Psykiatri; Aggression; insulin; glucose; glucagon; deliberate self-harm; IL-1beta; leptin; suicide attempt.

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APA (6th Edition):

Westling, S. (2009). Metabolic alterations in patients with self-inflicted aggressive behaviour. (Doctoral Dissertation). University of Lund. Retrieved from http://lup.lub.lu.se/record/1397365 ; http://portal.research.lu.se/ws/files/3879343/1397428.pdf

Chicago Manual of Style (16th Edition):

Westling, Sofie. “Metabolic alterations in patients with self-inflicted aggressive behaviour.” 2009. Doctoral Dissertation, University of Lund. Accessed September 23, 2019. http://lup.lub.lu.se/record/1397365 ; http://portal.research.lu.se/ws/files/3879343/1397428.pdf.

MLA Handbook (7th Edition):

Westling, Sofie. “Metabolic alterations in patients with self-inflicted aggressive behaviour.” 2009. Web. 23 Sep 2019.

Vancouver:

Westling S. Metabolic alterations in patients with self-inflicted aggressive behaviour. [Internet] [Doctoral dissertation]. University of Lund; 2009. [cited 2019 Sep 23]. Available from: http://lup.lub.lu.se/record/1397365 ; http://portal.research.lu.se/ws/files/3879343/1397428.pdf.

Council of Science Editors:

Westling S. Metabolic alterations in patients with self-inflicted aggressive behaviour. [Doctoral Dissertation]. University of Lund; 2009. Available from: http://lup.lub.lu.se/record/1397365 ; http://portal.research.lu.se/ws/files/3879343/1397428.pdf

26. Shamaa, Obada. Intracellular and extracellular regulation of the inflammatory protease caspase-1.

Degree: PhD, Biomedical Sciences, 2014, The Ohio State University

 Caspase-1 is an inflammatory cysteine protease which cleaves the pro-inflammatory cytokines, interleukin-1beta (IL-1beta) and IL-18, into their mature, bioactive forms. In addition, caspase-1 plays a… (more)

Subjects/Keywords: Immunology; Medicine; Biochemistry; Biomedical Research; caspase-1; inflammasome; IL-1beta; IL-18; mitochondria; calcium; monocyte; THP1

…antagonist interleukin-1beta inhibitory CARD IL-1 receptor associated kinase lymphocyte activating… …9 1.2 Interleukin-1β (IL-1β)… …11 1.2.1 Structure of IL-1β… …14 1.2.2 Interleukin-18 (IL-18)… …8 Figure 1.2 Structure and caspase-1 cleavage site for IL-1β and IL-18… 

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APA (6th Edition):

Shamaa, O. (2014). Intracellular and extracellular regulation of the inflammatory protease caspase-1. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292

Chicago Manual of Style (16th Edition):

Shamaa, Obada. “Intracellular and extracellular regulation of the inflammatory protease caspase-1.” 2014. Doctoral Dissertation, The Ohio State University. Accessed September 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292.

MLA Handbook (7th Edition):

Shamaa, Obada. “Intracellular and extracellular regulation of the inflammatory protease caspase-1.” 2014. Web. 23 Sep 2019.

Vancouver:

Shamaa O. Intracellular and extracellular regulation of the inflammatory protease caspase-1. [Internet] [Doctoral dissertation]. The Ohio State University; 2014. [cited 2019 Sep 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292.

Council of Science Editors:

Shamaa O. Intracellular and extracellular regulation of the inflammatory protease caspase-1. [Doctoral Dissertation]. The Ohio State University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292

27. Englezou, Pavlos. P2X7R-driven IL-1 responses in differentiated murine Dendritic Cells; comparison with Macrophages.

Degree: 2013, University of Manchester

 The P2X7R is a functionally distinct member of the P2X non-selective cation channels and has been implicated in the initiation of immune responses. One of… (more)

Subjects/Keywords: P2X7R; Dendritic cells; Macrophages; IL-1beta; IL-1alpha; P2X7R splice variants

…Figure 5.9: The impact of treatment with A-740003 on IL-1β release from LPS-primed and… …230 Figure 5.10: The impact of treatment with A-740003 on IL-1α and IL-6 release from… …Figure 5.11: The effect of apyrase treatment on IL-1 release from LPS-primed and ATP challenged… …238 Figure 5.14: The effect of apyrase treatment on IL-1β release from LPS-pulsed day 8… …of IL-1β release in day 8 BM-DC; comparisons with 8 BM-MΦ… 

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APA (6th Edition):

Englezou, P. (2013). P2X7R-driven IL-1 responses in differentiated murine Dendritic Cells; comparison with Macrophages. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:213941

Chicago Manual of Style (16th Edition):

Englezou, Pavlos. “P2X7R-driven IL-1 responses in differentiated murine Dendritic Cells; comparison with Macrophages.” 2013. Doctoral Dissertation, University of Manchester. Accessed September 23, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:213941.

MLA Handbook (7th Edition):

Englezou, Pavlos. “P2X7R-driven IL-1 responses in differentiated murine Dendritic Cells; comparison with Macrophages.” 2013. Web. 23 Sep 2019.

Vancouver:

Englezou P. P2X7R-driven IL-1 responses in differentiated murine Dendritic Cells; comparison with Macrophages. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2019 Sep 23]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:213941.

Council of Science Editors:

Englezou P. P2X7R-driven IL-1 responses in differentiated murine Dendritic Cells; comparison with Macrophages. [Doctoral Dissertation]. University of Manchester; 2013. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:213941


Vanderbilt University

28. Li, Chunsheng. MyD88: central relay station of Interleukin 1 signaling pathway.

Degree: PhD, Microbiology and Immunology, 2005, Vanderbilt University

 As inflammation is a major mechanism of disease, we investigated the signal transduction processes induced by the key inflammatory cytokine Interleukin (IL) 1 beta as… (more)

Subjects/Keywords: IL-1beta signaling pathway; MyD88; gene expression profiling; inflammation; nuclear import; IL-1 receptor accessary protein; Interleukin-1; Cellular signal transduction

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APA (6th Edition):

Li, C. (2005). MyD88: central relay station of Interleukin 1 signaling pathway. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;

Chicago Manual of Style (16th Edition):

Li, Chunsheng. “MyD88: central relay station of Interleukin 1 signaling pathway.” 2005. Doctoral Dissertation, Vanderbilt University. Accessed September 23, 2019. http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;.

MLA Handbook (7th Edition):

Li, Chunsheng. “MyD88: central relay station of Interleukin 1 signaling pathway.” 2005. Web. 23 Sep 2019.

Vancouver:

Li C. MyD88: central relay station of Interleukin 1 signaling pathway. [Internet] [Doctoral dissertation]. Vanderbilt University; 2005. [cited 2019 Sep 23]. Available from: http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;.

Council of Science Editors:

Li C. MyD88: central relay station of Interleukin 1 signaling pathway. [Doctoral Dissertation]. Vanderbilt University; 2005. Available from: http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;

29. Beard, LaMonta L. Regulation of Distal Lung Fluid Absorption by Membrane Components.

Degree: PhD, College of Arts and Sciences / School of Biomedical Sciences, 2011, Kent State University

 Near the end of gestation, the direction of ion and fluid flow across the alveolar epithelium rapidly changes from secretion to absorption. Thus, the relative… (more)

Subjects/Keywords: Biomedical Research; Cl transport; distal lung fluid absorption; fetal lung development; IL-1beta; Na transport

…53 CHAPTER III. Fig. 3-1. IL-1/cortisol Pathway… …GD68 in the Lungs With and Without IL-1 Pretreatment… …ENaC in CRRs and Non-CRRs With and Without IL-1 Pretreatment .................. 71 x Fig… …3-4. Expression of CFTR in CRRs and Non-CRRs at GD61 and GD68 With and Without IL-1… …73 Fig. 3-5. Completed IL-1/cortisol Pathway… 

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APA (6th Edition):

Beard, L. L. (2011). Regulation of Distal Lung Fluid Absorption by Membrane Components. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1321989465

Chicago Manual of Style (16th Edition):

Beard, LaMonta L. “Regulation of Distal Lung Fluid Absorption by Membrane Components.” 2011. Doctoral Dissertation, Kent State University. Accessed September 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=kent1321989465.

MLA Handbook (7th Edition):

Beard, LaMonta L. “Regulation of Distal Lung Fluid Absorption by Membrane Components.” 2011. Web. 23 Sep 2019.

Vancouver:

Beard LL. Regulation of Distal Lung Fluid Absorption by Membrane Components. [Internet] [Doctoral dissertation]. Kent State University; 2011. [cited 2019 Sep 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1321989465.

Council of Science Editors:

Beard LL. Regulation of Distal Lung Fluid Absorption by Membrane Components. [Doctoral Dissertation]. Kent State University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1321989465

30. Kleibeuker, W. GRK2 deficiency is a pain.

Degree: 2007, University Utrecht

 G protein-coupled receptor (GPCR) kinase (GRK)2 regulates the responsiveness of many GPCRS, e.g. CCR1, CCR2, CCR5, neurokinin-1 receptor, and metabotropic glutamate receptors, which are involved… (more)

Subjects/Keywords: GRK2; GPCR; hyperalgesia; allodynia; IL-1beta; signaling; inflammatory pain; spinal cord; nerve damage; astrocytes

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APA (6th Edition):

Kleibeuker, W. (2007). GRK2 deficiency is a pain. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/25012 ; URN:NBN:NL:UI:10-1874-25012 ; urn:isbn:978-90-393-4685-3 ; URN:NBN:NL:UI:10-1874-25012 ; http://dspace.library.uu.nl/handle/1874/25012

Chicago Manual of Style (16th Edition):

Kleibeuker, W. “GRK2 deficiency is a pain.” 2007. Doctoral Dissertation, University Utrecht. Accessed September 23, 2019. http://dspace.library.uu.nl/handle/1874/25012 ; URN:NBN:NL:UI:10-1874-25012 ; urn:isbn:978-90-393-4685-3 ; URN:NBN:NL:UI:10-1874-25012 ; http://dspace.library.uu.nl/handle/1874/25012.

MLA Handbook (7th Edition):

Kleibeuker, W. “GRK2 deficiency is a pain.” 2007. Web. 23 Sep 2019.

Vancouver:

Kleibeuker W. GRK2 deficiency is a pain. [Internet] [Doctoral dissertation]. University Utrecht; 2007. [cited 2019 Sep 23]. Available from: http://dspace.library.uu.nl/handle/1874/25012 ; URN:NBN:NL:UI:10-1874-25012 ; urn:isbn:978-90-393-4685-3 ; URN:NBN:NL:UI:10-1874-25012 ; http://dspace.library.uu.nl/handle/1874/25012.

Council of Science Editors:

Kleibeuker W. GRK2 deficiency is a pain. [Doctoral Dissertation]. University Utrecht; 2007. Available from: http://dspace.library.uu.nl/handle/1874/25012 ; URN:NBN:NL:UI:10-1874-25012 ; urn:isbn:978-90-393-4685-3 ; URN:NBN:NL:UI:10-1874-25012 ; http://dspace.library.uu.nl/handle/1874/25012

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