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You searched for subject:(I B Kinase ). Showing records 1 – 30 of 30746 total matches.

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University of Cambridge

1. Prescott, Jack. Interrogating novel functions of the I kappa B kinases via CRISPR-Cas9 gene editing and small molecule inhibition.

Degree: PhD, 2018, University of Cambridge

 The NF-kB signalling pathway is a critical mediator of the cellular responses to inflammatory cytokines. The IκB kinase (IKK) complex, which is composed of two… (more)

Subjects/Keywords: 616.99; NF-kappa B; I kappa B kinase; NEMO

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APA (6th Edition):

Prescott, J. (2018). Interrogating novel functions of the I kappa B kinases via CRISPR-Cas9 gene editing and small molecule inhibition. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.24323 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744930

Chicago Manual of Style (16th Edition):

Prescott, Jack. “Interrogating novel functions of the I kappa B kinases via CRISPR-Cas9 gene editing and small molecule inhibition.” 2018. Doctoral Dissertation, University of Cambridge. Accessed January 18, 2021. https://doi.org/10.17863/CAM.24323 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744930.

MLA Handbook (7th Edition):

Prescott, Jack. “Interrogating novel functions of the I kappa B kinases via CRISPR-Cas9 gene editing and small molecule inhibition.” 2018. Web. 18 Jan 2021.

Vancouver:

Prescott J. Interrogating novel functions of the I kappa B kinases via CRISPR-Cas9 gene editing and small molecule inhibition. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2021 Jan 18]. Available from: https://doi.org/10.17863/CAM.24323 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744930.

Council of Science Editors:

Prescott J. Interrogating novel functions of the I kappa B kinases via CRISPR-Cas9 gene editing and small molecule inhibition. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://doi.org/10.17863/CAM.24323 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744930


University of Texas Southwestern Medical Center

2. Skaug, Brian. Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20.

Degree: 2013, University of Texas Southwestern Medical Center

 A20 is a potent anti-inflammatory protein that inhibits NF-ΚB, and A20 dysfunction is associated with autoimmunity and B-cell lymphoma. A20 harbors a deubiquitination enzyme domain… (more)

Subjects/Keywords: I-kappa B Kinase; Inflammation; Intracellular Signaling Peptides and Proteins; NF-kappa B

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APA (6th Edition):

Skaug, B. (2013). Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1706

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Skaug, Brian. “Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed January 18, 2021. http://hdl.handle.net/2152.5/1706.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Skaug, Brian. “Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20.” 2013. Web. 18 Jan 2021.

Vancouver:

Skaug B. Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/2152.5/1706.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Skaug B. Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1706

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade Estadual de Campinas

3. Weissmann, Laís, 1989-. A exposição ao material particulado MP 2,5 induz a resistência à leptina no hipotálamo e estimula a hiperfagia e a obesidade em camundongos : PM 2,5 exposure induces leptin resistance in the hypothalamus and drives hyperphagia and obesity in mice: PM 2,5 exposure induces leptin resistance in the hypothalamus and drives hyperphagia and obesity in mice.

Degree: 2018, Universidade Estadual de Campinas

 Abstract: Background: The exposure to environmental pollution is increasing overall affecting human health and global climate. The particulate matter with less than 2.5 diameters (PM2.5)… (more)

Subjects/Keywords: Ar - Poluição; Material particulado; Quinase I-kappa B epsilon; Obesidade; Hipotálamo; Air pollution; Particulate matter; I-kappa B Kinase epsilon; Obesity; Hypothalamus

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APA (6th Edition):

Weissmann, Laís, 1. (2018). A exposição ao material particulado MP 2,5 induz a resistência à leptina no hipotálamo e estimula a hiperfagia e a obesidade em camundongos : PM 2,5 exposure induces leptin resistance in the hypothalamus and drives hyperphagia and obesity in mice: PM 2,5 exposure induces leptin resistance in the hypothalamus and drives hyperphagia and obesity in mice. (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/335778

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Weissmann, Laís, 1989-. “A exposição ao material particulado MP 2,5 induz a resistência à leptina no hipotálamo e estimula a hiperfagia e a obesidade em camundongos : PM 2,5 exposure induces leptin resistance in the hypothalamus and drives hyperphagia and obesity in mice: PM 2,5 exposure induces leptin resistance in the hypothalamus and drives hyperphagia and obesity in mice.” 2018. Thesis, Universidade Estadual de Campinas. Accessed January 18, 2021. http://repositorio.unicamp.br/jspui/handle/REPOSIP/335778.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Weissmann, Laís, 1989-. “A exposição ao material particulado MP 2,5 induz a resistência à leptina no hipotálamo e estimula a hiperfagia e a obesidade em camundongos : PM 2,5 exposure induces leptin resistance in the hypothalamus and drives hyperphagia and obesity in mice: PM 2,5 exposure induces leptin resistance in the hypothalamus and drives hyperphagia and obesity in mice.” 2018. Web. 18 Jan 2021.

Vancouver:

Weissmann, Laís 1. A exposição ao material particulado MP 2,5 induz a resistência à leptina no hipotálamo e estimula a hiperfagia e a obesidade em camundongos : PM 2,5 exposure induces leptin resistance in the hypothalamus and drives hyperphagia and obesity in mice: PM 2,5 exposure induces leptin resistance in the hypothalamus and drives hyperphagia and obesity in mice. [Internet] [Thesis]. Universidade Estadual de Campinas; 2018. [cited 2021 Jan 18]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/335778.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Weissmann, Laís 1. A exposição ao material particulado MP 2,5 induz a resistência à leptina no hipotálamo e estimula a hiperfagia e a obesidade em camundongos : PM 2,5 exposure induces leptin resistance in the hypothalamus and drives hyperphagia and obesity in mice: PM 2,5 exposure induces leptin resistance in the hypothalamus and drives hyperphagia and obesity in mice. [Thesis]. Universidade Estadual de Campinas; 2018. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/335778

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Rajurkar, Mihir S. GLI-IKBKE Requirement In KRAS-Induced Pancreatic Tumorigenesis: A Dissertation.

Degree: Cancer Biology, Molecular, Cell and Cancer Biology Department, 2014, U of Massachusetts : Med

  Pancreatic ductal adenocarcinoma (PDAC), one of the most aggressive human malignancies, is thought to be initiated by KRAS activation. Here, we find that transcriptional… (more)

Subjects/Keywords: Pancreatic Ductal Carcinoma; I-kappa B Kinase; Pancreatic Neoplasms; Phosphatidylinositol 3-Kinases; Transcription Factors; Carcinogenesis; Transcriptional Activation; Cancer Biology; Neoplasms

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APA (6th Edition):

Rajurkar, M. S. (2014). GLI-IKBKE Requirement In KRAS-Induced Pancreatic Tumorigenesis: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/753

Chicago Manual of Style (16th Edition):

Rajurkar, Mihir S. “GLI-IKBKE Requirement In KRAS-Induced Pancreatic Tumorigenesis: A Dissertation.” 2014. Doctoral Dissertation, U of Massachusetts : Med. Accessed January 18, 2021. http://escholarship.umassmed.edu/gsbs_diss/753.

MLA Handbook (7th Edition):

Rajurkar, Mihir S. “GLI-IKBKE Requirement In KRAS-Induced Pancreatic Tumorigenesis: A Dissertation.” 2014. Web. 18 Jan 2021.

Vancouver:

Rajurkar MS. GLI-IKBKE Requirement In KRAS-Induced Pancreatic Tumorigenesis: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2014. [cited 2021 Jan 18]. Available from: http://escholarship.umassmed.edu/gsbs_diss/753.

Council of Science Editors:

Rajurkar MS. GLI-IKBKE Requirement In KRAS-Induced Pancreatic Tumorigenesis: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2014. Available from: http://escholarship.umassmed.edu/gsbs_diss/753


Queen Mary, University of London

5. Johnson, Florence Lilian. The late inhibition of IκB kinase attenuates acute kidney injury and the subsequent development of renal fibrosis in animal models of ischaemia-reperfusion injury and unilateral ureteral obstruction.

Degree: PhD, 2016, Queen Mary, University of London

 Acute kidney injury (AKI) is a major risk factor for chronic kidney disease (CKD). For patients who recover from AKI, there is a 25% increase… (more)

Subjects/Keywords: Translational Medicine and Therapeutics; Acute kidney injury; chronic kidney disease; Inhibitor of I?B kinase; renal fibrosis

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APA (6th Edition):

Johnson, F. L. (2016). The late inhibition of IκB kinase attenuates acute kidney injury and the subsequent development of renal fibrosis in animal models of ischaemia-reperfusion injury and unilateral ureteral obstruction. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/36706 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765842

Chicago Manual of Style (16th Edition):

Johnson, Florence Lilian. “The late inhibition of IκB kinase attenuates acute kidney injury and the subsequent development of renal fibrosis in animal models of ischaemia-reperfusion injury and unilateral ureteral obstruction.” 2016. Doctoral Dissertation, Queen Mary, University of London. Accessed January 18, 2021. http://qmro.qmul.ac.uk/xmlui/handle/123456789/36706 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765842.

MLA Handbook (7th Edition):

Johnson, Florence Lilian. “The late inhibition of IκB kinase attenuates acute kidney injury and the subsequent development of renal fibrosis in animal models of ischaemia-reperfusion injury and unilateral ureteral obstruction.” 2016. Web. 18 Jan 2021.

Vancouver:

Johnson FL. The late inhibition of IκB kinase attenuates acute kidney injury and the subsequent development of renal fibrosis in animal models of ischaemia-reperfusion injury and unilateral ureteral obstruction. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2016. [cited 2021 Jan 18]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/36706 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765842.

Council of Science Editors:

Johnson FL. The late inhibition of IκB kinase attenuates acute kidney injury and the subsequent development of renal fibrosis in animal models of ischaemia-reperfusion injury and unilateral ureteral obstruction. [Doctoral Dissertation]. Queen Mary, University of London; 2016. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/36706 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765842


IUPUI

6. Fears, Sharry L. Effect of Inhibition of S-Nitrosoglutathione Reductase on the NF-κB Pathway.

Degree: 2009, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

S-nitrosoglutathione reductase (GSNOR) also known as glutathione- dependent formaldehyde dehydrogenase (FDH), is a zinc-dependent dehydrogenase. GSNOR oxidizes long chain alcohols… (more)

Subjects/Keywords: I kappa B; I kappa B kinase beta; NF-kappa B; Nitric Oxide; S-Nitrosoglutathione reductase; Nitric oxide; Dehyrdogenases; Phosphorylation

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APA (6th Edition):

Fears, S. L. (2009). Effect of Inhibition of S-Nitrosoglutathione Reductase on the NF-κB Pathway. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/1949

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fears, Sharry L. “Effect of Inhibition of S-Nitrosoglutathione Reductase on the NF-κB Pathway.” 2009. Thesis, IUPUI. Accessed January 18, 2021. http://hdl.handle.net/1805/1949.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fears, Sharry L. “Effect of Inhibition of S-Nitrosoglutathione Reductase on the NF-κB Pathway.” 2009. Web. 18 Jan 2021.

Vancouver:

Fears SL. Effect of Inhibition of S-Nitrosoglutathione Reductase on the NF-κB Pathway. [Internet] [Thesis]. IUPUI; 2009. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/1805/1949.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fears SL. Effect of Inhibition of S-Nitrosoglutathione Reductase on the NF-κB Pathway. [Thesis]. IUPUI; 2009. Available from: http://hdl.handle.net/1805/1949

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Ea, Chee-Kwee. Ubiquitination-Dependent Activation of IKK.

Degree: 2005, University of Texas Southwestern Medical Center

 Ubiquitination plays two different roles in the nuclear factor κΒ (NF-κΒ ) pathway, the traditional K48-linked polyubiquitination-mediated IκΒ degradation and the non-traditional K63-linked polyubiquitination-mediated IKK… (more)

Subjects/Keywords: TNF Receptor-Associated Factor 6; Ubiquitination; I-kappa B Kinase

…TABLE OF CONTENTS TITLE i DEDICATION… …xiv CHAPTER I: GENERAL INTRODUCTION… …21 I.B.3.b Ubiquitination and processing of p100 and p105… …66 III.B.7 IKK kinase assay… …77 III.C.4 Polyubiquitination of RIP1 is Required for Recruitment of TAK1 and IKK Kinase… 

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APA (6th Edition):

Ea, C. (2005). Ubiquitination-Dependent Activation of IKK. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/382

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ea, Chee-Kwee. “Ubiquitination-Dependent Activation of IKK.” 2005. Thesis, University of Texas Southwestern Medical Center. Accessed January 18, 2021. http://hdl.handle.net/2152.5/382.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ea, Chee-Kwee. “Ubiquitination-Dependent Activation of IKK.” 2005. Web. 18 Jan 2021.

Vancouver:

Ea C. Ubiquitination-Dependent Activation of IKK. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2005. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/2152.5/382.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ea C. Ubiquitination-Dependent Activation of IKK. [Thesis]. University of Texas Southwestern Medical Center; 2005. Available from: http://hdl.handle.net/2152.5/382

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Paquette, Nicholas Paul. Caspase Mediated Cleavage, IAP Binding, Ubiquitination and Kinase Activation : Defining the Molecular Mechanisms Required for Drosophila NF-кB Signaling: A Dissertation.

Degree: Interdisciplinary Graduate Program, Medicine, 2009, U of Massachusetts : Med

  Innate immunity is the first line of defense against invading pathogens. Vertebrate innate immunity provides both initial protection, and activates adaptive immune responses, including… (more)

Subjects/Keywords: Immunity; Innate; I-kappa B Kinase; Drosophila Proteins; Bacterial Proteins; Yersinia pestis; Amino Acids, Peptides, and Proteins; Animal Experimentation and Research; Bacteria; Enzymes and Coenzymes; Hemic and Immune Systems

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APA (6th Edition):

Paquette, N. P. (2009). Caspase Mediated Cleavage, IAP Binding, Ubiquitination and Kinase Activation : Defining the Molecular Mechanisms Required for Drosophila NF-кB Signaling: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/444

Chicago Manual of Style (16th Edition):

Paquette, Nicholas Paul. “Caspase Mediated Cleavage, IAP Binding, Ubiquitination and Kinase Activation : Defining the Molecular Mechanisms Required for Drosophila NF-кB Signaling: A Dissertation.” 2009. Doctoral Dissertation, U of Massachusetts : Med. Accessed January 18, 2021. https://escholarship.umassmed.edu/gsbs_diss/444.

MLA Handbook (7th Edition):

Paquette, Nicholas Paul. “Caspase Mediated Cleavage, IAP Binding, Ubiquitination and Kinase Activation : Defining the Molecular Mechanisms Required for Drosophila NF-кB Signaling: A Dissertation.” 2009. Web. 18 Jan 2021.

Vancouver:

Paquette NP. Caspase Mediated Cleavage, IAP Binding, Ubiquitination and Kinase Activation : Defining the Molecular Mechanisms Required for Drosophila NF-кB Signaling: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2009. [cited 2021 Jan 18]. Available from: https://escholarship.umassmed.edu/gsbs_diss/444.

Council of Science Editors:

Paquette NP. Caspase Mediated Cleavage, IAP Binding, Ubiquitination and Kinase Activation : Defining the Molecular Mechanisms Required for Drosophila NF-кB Signaling: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2009. Available from: https://escholarship.umassmed.edu/gsbs_diss/444


Colorado State University

9. Caldas, Gina V. Kinetochore protein KNL1 links kinetochore-microtubule attachment and checkpoint signaling during mitosis, The.

Degree: PhD, Biochemistry and Molecular Biology, 2014, Colorado State University

 Mitosis is the phase of the cell cycle in which replicated chromosomes physically separate, resulting in the formation of two genetically identical daughter cells. This… (more)

Subjects/Keywords: Hec1; Aurora B kinase; kinetochore; KNL1; mitosis

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APA (6th Edition):

Caldas, G. V. (2014). Kinetochore protein KNL1 links kinetochore-microtubule attachment and checkpoint signaling during mitosis, The. (Doctoral Dissertation). Colorado State University. Retrieved from http://hdl.handle.net/10217/85026

Chicago Manual of Style (16th Edition):

Caldas, Gina V. “Kinetochore protein KNL1 links kinetochore-microtubule attachment and checkpoint signaling during mitosis, The.” 2014. Doctoral Dissertation, Colorado State University. Accessed January 18, 2021. http://hdl.handle.net/10217/85026.

MLA Handbook (7th Edition):

Caldas, Gina V. “Kinetochore protein KNL1 links kinetochore-microtubule attachment and checkpoint signaling during mitosis, The.” 2014. Web. 18 Jan 2021.

Vancouver:

Caldas GV. Kinetochore protein KNL1 links kinetochore-microtubule attachment and checkpoint signaling during mitosis, The. [Internet] [Doctoral dissertation]. Colorado State University; 2014. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10217/85026.

Council of Science Editors:

Caldas GV. Kinetochore protein KNL1 links kinetochore-microtubule attachment and checkpoint signaling during mitosis, The. [Doctoral Dissertation]. Colorado State University; 2014. Available from: http://hdl.handle.net/10217/85026


University of Sydney

10. Yeo, Reichelle. Redox Regulation of Protein Kinase B/Akt Function by an Allosteric Disulphide Bond .

Degree: 2019, University of Sydney

 Most proteins in nature are chemically modified after they are made to control how, when and where they function. One type of chemical modification is… (more)

Subjects/Keywords: protein kinase B/Akt; metabolism; cancer; mechanism

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APA (6th Edition):

Yeo, R. (2019). Redox Regulation of Protein Kinase B/Akt Function by an Allosteric Disulphide Bond . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/20816

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yeo, Reichelle. “Redox Regulation of Protein Kinase B/Akt Function by an Allosteric Disulphide Bond .” 2019. Thesis, University of Sydney. Accessed January 18, 2021. http://hdl.handle.net/2123/20816.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yeo, Reichelle. “Redox Regulation of Protein Kinase B/Akt Function by an Allosteric Disulphide Bond .” 2019. Web. 18 Jan 2021.

Vancouver:

Yeo R. Redox Regulation of Protein Kinase B/Akt Function by an Allosteric Disulphide Bond . [Internet] [Thesis]. University of Sydney; 2019. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/2123/20816.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yeo R. Redox Regulation of Protein Kinase B/Akt Function by an Allosteric Disulphide Bond . [Thesis]. University of Sydney; 2019. Available from: http://hdl.handle.net/2123/20816

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

11. Huff, Belinda. The development of pyrrolopyrimidines as kinase inhibitors.

Degree: Chemistry, 2011, University of New South Wales

 This thesis is focussed on the synthesis of various pyrrolopyrimidines, which will be examined for their kinase inhibitory potential. In Chapter 2, the synthesis of… (more)

Subjects/Keywords: Pyrrolopyrimidine; Kinase Inhibitors; Variolin B; Pyrrolopyrimidines

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APA (6th Edition):

Huff, B. (2011). The development of pyrrolopyrimidines as kinase inhibitors. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/51897 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10566/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Huff, Belinda. “The development of pyrrolopyrimidines as kinase inhibitors.” 2011. Doctoral Dissertation, University of New South Wales. Accessed January 18, 2021. http://handle.unsw.edu.au/1959.4/51897 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10566/SOURCE02?view=true.

MLA Handbook (7th Edition):

Huff, Belinda. “The development of pyrrolopyrimidines as kinase inhibitors.” 2011. Web. 18 Jan 2021.

Vancouver:

Huff B. The development of pyrrolopyrimidines as kinase inhibitors. [Internet] [Doctoral dissertation]. University of New South Wales; 2011. [cited 2021 Jan 18]. Available from: http://handle.unsw.edu.au/1959.4/51897 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10566/SOURCE02?view=true.

Council of Science Editors:

Huff B. The development of pyrrolopyrimidines as kinase inhibitors. [Doctoral Dissertation]. University of New South Wales; 2011. Available from: http://handle.unsw.edu.au/1959.4/51897 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10566/SOURCE02?view=true


Université de Sherbrooke

12. Ear, Thornin. Signalisation en amont de la voie NF-[kappa]B et son impact sur la production de cytokines chez les neutrophiles humains.

Degree: 2008, Université de Sherbrooke

 En premier lieu, en utilisant des inhibiteurs pharmacologiques du NF-[kappa]B, nous avons constaté que l'inhibition du facteur de transcription NF-[kappa]B chez ces cellules diminue de… (more)

Subjects/Keywords: Génération de cytokines et chimiokines; Facteurs de transcription NF-[kappa]B; Cascade de signalisation IKK/I[kappa]B/NF-[kappa]B nucléaire; Protéine kinase TAK1; Neutrophiles humains

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APA (6th Edition):

Ear, T. (2008). Signalisation en amont de la voie NF-[kappa]B et son impact sur la production de cytokines chez les neutrophiles humains. (Doctoral Dissertation). Université de Sherbrooke. Retrieved from http://savoirs.usherbrooke.ca/handle/11143/4274

Chicago Manual of Style (16th Edition):

Ear, Thornin. “Signalisation en amont de la voie NF-[kappa]B et son impact sur la production de cytokines chez les neutrophiles humains.” 2008. Doctoral Dissertation, Université de Sherbrooke. Accessed January 18, 2021. http://savoirs.usherbrooke.ca/handle/11143/4274.

MLA Handbook (7th Edition):

Ear, Thornin. “Signalisation en amont de la voie NF-[kappa]B et son impact sur la production de cytokines chez les neutrophiles humains.” 2008. Web. 18 Jan 2021.

Vancouver:

Ear T. Signalisation en amont de la voie NF-[kappa]B et son impact sur la production de cytokines chez les neutrophiles humains. [Internet] [Doctoral dissertation]. Université de Sherbrooke; 2008. [cited 2021 Jan 18]. Available from: http://savoirs.usherbrooke.ca/handle/11143/4274.

Council of Science Editors:

Ear T. Signalisation en amont de la voie NF-[kappa]B et son impact sur la production de cytokines chez les neutrophiles humains. [Doctoral Dissertation]. Université de Sherbrooke; 2008. Available from: http://savoirs.usherbrooke.ca/handle/11143/4274

13. Al Mohamad, Hazar. Etude de la balance réactivation/apoptose des cellules B infectées par l' EBV suite au traitement par un HDACi, le vorinostat : Study of the balance reactivation / apoptosis of B cells infected with EBV following treatment with a HDACi, vorinostat.

Degree: Docteur es, Immunologie, oncologie et infectiologie, 2016, Limoges

Les inhibiteurs des histones désacétylase (HDACi) constituent une classe prometteuse de médicaments anticancéreux. Ils peuvent déclencher la voie apoptotique et sont proposés pour le traitement… (more)

Subjects/Keywords: EBV; Réactivation; Apoptose; Vorinostat; MAP kinase p38; Lymphocytes B; EBV; Reactivation; Apoptosis; Vorinostat; P38 MAP Kinase; B lymphocytes; 615.37

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APA (6th Edition):

Al Mohamad, H. (2016). Etude de la balance réactivation/apoptose des cellules B infectées par l' EBV suite au traitement par un HDACi, le vorinostat : Study of the balance reactivation / apoptosis of B cells infected with EBV following treatment with a HDACi, vorinostat. (Doctoral Dissertation). Limoges. Retrieved from http://www.theses.fr/2016LIMO0014

Chicago Manual of Style (16th Edition):

Al Mohamad, Hazar. “Etude de la balance réactivation/apoptose des cellules B infectées par l' EBV suite au traitement par un HDACi, le vorinostat : Study of the balance reactivation / apoptosis of B cells infected with EBV following treatment with a HDACi, vorinostat.” 2016. Doctoral Dissertation, Limoges. Accessed January 18, 2021. http://www.theses.fr/2016LIMO0014.

MLA Handbook (7th Edition):

Al Mohamad, Hazar. “Etude de la balance réactivation/apoptose des cellules B infectées par l' EBV suite au traitement par un HDACi, le vorinostat : Study of the balance reactivation / apoptosis of B cells infected with EBV following treatment with a HDACi, vorinostat.” 2016. Web. 18 Jan 2021.

Vancouver:

Al Mohamad H. Etude de la balance réactivation/apoptose des cellules B infectées par l' EBV suite au traitement par un HDACi, le vorinostat : Study of the balance reactivation / apoptosis of B cells infected with EBV following treatment with a HDACi, vorinostat. [Internet] [Doctoral dissertation]. Limoges; 2016. [cited 2021 Jan 18]. Available from: http://www.theses.fr/2016LIMO0014.

Council of Science Editors:

Al Mohamad H. Etude de la balance réactivation/apoptose des cellules B infectées par l' EBV suite au traitement par un HDACi, le vorinostat : Study of the balance reactivation / apoptosis of B cells infected with EBV following treatment with a HDACi, vorinostat. [Doctoral Dissertation]. Limoges; 2016. Available from: http://www.theses.fr/2016LIMO0014

14. Bendjeddou, Lyamin. Synthèse et évaluation biologique de nouveaux inhibiteurs de kinases : identification d‘inhibiteurs de kinases parasitaires : Synthesis and biological evaluation of new kinase inhibitors : identification of inhibitors of several parasite protein kinases.

Degree: Docteur es, Chimie thérapeutique, 2014, Université Paris Descartes – Paris V

La phosphorylation des protéines par les kinases est l’une plus importantes modification post-traductionnelle dans les processus cellulaires tels que la division, la différenciation, la prolifération… (more)

Subjects/Keywords: Inhibiteur de protéine kinase; Imidazo[1,2-b]pyridazine; Imidazo[4,5-b]pyridine; Kinase cycline-dépendante (CDKs); CDC-like kinase (CLKs); Dual specificity tyrosine-phosphorylation-regulated kinase (DYRKs); Parasite unicellulaire; Maladie d’Alzheimer; Trisomie 21; Kinase inhibitor; Imidazo[1,2-b]pyridazine; Imidazo[4,5-b]pyridine; Dual specificity tyrosine-phosphorylation-regulated kinase (DYRKs); Cyclin-dependent kinase (CDKs); Unicellular parasite; Alzheimer’s disease; Down syndrome; 615.19

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APA (6th Edition):

Bendjeddou, L. (2014). Synthèse et évaluation biologique de nouveaux inhibiteurs de kinases : identification d‘inhibiteurs de kinases parasitaires : Synthesis and biological evaluation of new kinase inhibitors : identification of inhibitors of several parasite protein kinases. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2014PA05P615

Chicago Manual of Style (16th Edition):

Bendjeddou, Lyamin. “Synthèse et évaluation biologique de nouveaux inhibiteurs de kinases : identification d‘inhibiteurs de kinases parasitaires : Synthesis and biological evaluation of new kinase inhibitors : identification of inhibitors of several parasite protein kinases.” 2014. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed January 18, 2021. http://www.theses.fr/2014PA05P615.

MLA Handbook (7th Edition):

Bendjeddou, Lyamin. “Synthèse et évaluation biologique de nouveaux inhibiteurs de kinases : identification d‘inhibiteurs de kinases parasitaires : Synthesis and biological evaluation of new kinase inhibitors : identification of inhibitors of several parasite protein kinases.” 2014. Web. 18 Jan 2021.

Vancouver:

Bendjeddou L. Synthèse et évaluation biologique de nouveaux inhibiteurs de kinases : identification d‘inhibiteurs de kinases parasitaires : Synthesis and biological evaluation of new kinase inhibitors : identification of inhibitors of several parasite protein kinases. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2014. [cited 2021 Jan 18]. Available from: http://www.theses.fr/2014PA05P615.

Council of Science Editors:

Bendjeddou L. Synthèse et évaluation biologique de nouveaux inhibiteurs de kinases : identification d‘inhibiteurs de kinases parasitaires : Synthesis and biological evaluation of new kinase inhibitors : identification of inhibitors of several parasite protein kinases. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2014. Available from: http://www.theses.fr/2014PA05P615

15. Jiang, Shaoning. Molecular mechanisms of hepatic insulin resistance following injury.

Degree: PhD, 2010, University of Alabama – Birmingham

Insulin resistance commonly occurs following injuries or critical illness independent of previous diabetic status. The development of insulin resistance and hyperglycemia is associated with increased… (more)

Subjects/Keywords: Adenoviridae – metabolism<; br>; Adenoviridae Infections – metabolism<; br>; Diabetes Mellitus, Type 2 – physiopathology<; br>; Glucose – metabolism<; br>; I-kappa B Kinase – physiology <; br>; Insulin – metabolism<; br>; JNK Mitogen-Activated Protein Kinases – physiology <; br>; Liver

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APA (6th Edition):

Jiang, S. (2010). Molecular mechanisms of hepatic insulin resistance following injury. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1374

Chicago Manual of Style (16th Edition):

Jiang, Shaoning. “Molecular mechanisms of hepatic insulin resistance following injury.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 18, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1374.

MLA Handbook (7th Edition):

Jiang, Shaoning. “Molecular mechanisms of hepatic insulin resistance following injury.” 2010. Web. 18 Jan 2021.

Vancouver:

Jiang S. Molecular mechanisms of hepatic insulin resistance following injury. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2021 Jan 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1374.

Council of Science Editors:

Jiang S. Molecular mechanisms of hepatic insulin resistance following injury. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1374

16. Reyftmann, Romina. Studies of the Ubiquitin-NFκβ pathway in women and baboons with endometriosis .

Degree: 2012, University of Sydney

 Background: Our aim was to determine if changes in ubiquitin are correlated with proteins known to be affected directly or indirectly by the ubiquitin-proteasome pathway.… (more)

Subjects/Keywords: Endometriosis; Baboon; Ubiquitin; Nuclear factor-kappa beta; I kappa B kinase

…IKK Inhibitor of kappa beta kinase IKK1 I-kappa-B kinase 1 IKK2 I-kappa-B kinase 2… …IKKα I-kappa-B-kinase-alpha IKKβ I-kappa-B-kinase-beta IKKγ I-kappa-B-kinase-gamma IL… …kappa B xiv Iκβ Inhibitor of kappa beta Iκβα I-kappa-B-alpha IκBβ I-kappa-B-beta IκBγ… …I-kappa-B-gamma IκBKα Inhibitor of kappa light polypeptide gene enhancer in B cells… …kinase of alpha IKBKB Inhibitor of kappa light polypeptide gene enhancer in B-cells, kinase… 

Page 1 Page 2 Page 3 Page 4 Page 5 Page 6 Page 7

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APA (6th Edition):

Reyftmann, R. (2012). Studies of the Ubiquitin-NFκβ pathway in women and baboons with endometriosis . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/8688

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Reyftmann, Romina. “Studies of the Ubiquitin-NFκβ pathway in women and baboons with endometriosis .” 2012. Thesis, University of Sydney. Accessed January 18, 2021. http://hdl.handle.net/2123/8688.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Reyftmann, Romina. “Studies of the Ubiquitin-NFκβ pathway in women and baboons with endometriosis .” 2012. Web. 18 Jan 2021.

Vancouver:

Reyftmann R. Studies of the Ubiquitin-NFκβ pathway in women and baboons with endometriosis . [Internet] [Thesis]. University of Sydney; 2012. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/2123/8688.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Reyftmann R. Studies of the Ubiquitin-NFκβ pathway in women and baboons with endometriosis . [Thesis]. University of Sydney; 2012. Available from: http://hdl.handle.net/2123/8688

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Mississippi State University

17. Leach, Charles Andrew. Efficacy of novel pyridinium oximes in preventing neural damage.

Degree: MS, Veterinary Medicine, College of, 2017, Mississippi State University

  Organophosphates are neurotoxic compounds that inhibit acetylcholinesterase producing excess cholinergic stimulation. This produces various toxic signs including excitotoxic neuronal damage. Oximes can be used… (more)

Subjects/Keywords: MAP Kinase; histopathology; Fluoro-Jade B; gene expression; pyridinium oxime; organophosphate

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APA (6th Edition):

Leach, C. A. (2017). Efficacy of novel pyridinium oximes in preventing neural damage. (Masters Thesis). Mississippi State University. Retrieved from http://sun.library.msstate.edu/ETD-db/theses/available/etd-10232017-093523/ ;

Chicago Manual of Style (16th Edition):

Leach, Charles Andrew. “Efficacy of novel pyridinium oximes in preventing neural damage.” 2017. Masters Thesis, Mississippi State University. Accessed January 18, 2021. http://sun.library.msstate.edu/ETD-db/theses/available/etd-10232017-093523/ ;.

MLA Handbook (7th Edition):

Leach, Charles Andrew. “Efficacy of novel pyridinium oximes in preventing neural damage.” 2017. Web. 18 Jan 2021.

Vancouver:

Leach CA. Efficacy of novel pyridinium oximes in preventing neural damage. [Internet] [Masters thesis]. Mississippi State University; 2017. [cited 2021 Jan 18]. Available from: http://sun.library.msstate.edu/ETD-db/theses/available/etd-10232017-093523/ ;.

Council of Science Editors:

Leach CA. Efficacy of novel pyridinium oximes in preventing neural damage. [Masters Thesis]. Mississippi State University; 2017. Available from: http://sun.library.msstate.edu/ETD-db/theses/available/etd-10232017-093523/ ;


University of Tasmania

18. Ng, JMJ. Elucidating the roles of aurora B kinase in neurons.

Degree: 2012, University of Tasmania

 To reveal molecular determinates that underlie the intrinsic molecular pathways within neurons that support regeneration after injury, a DNA microarray study was performed on axotomized… (more)

Subjects/Keywords: neurite regeneration; microarray; Aurora B kinase; neurite outgrowth; neurons; brain; proteomics

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APA (6th Edition):

Ng, J. (2012). Elucidating the roles of aurora B kinase in neurons. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/15014/3/whole-ng-thesis-incl-pub-mat.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ng, JMJ. “Elucidating the roles of aurora B kinase in neurons.” 2012. Thesis, University of Tasmania. Accessed January 18, 2021. https://eprints.utas.edu.au/15014/3/whole-ng-thesis-incl-pub-mat.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ng, JMJ. “Elucidating the roles of aurora B kinase in neurons.” 2012. Web. 18 Jan 2021.

Vancouver:

Ng J. Elucidating the roles of aurora B kinase in neurons. [Internet] [Thesis]. University of Tasmania; 2012. [cited 2021 Jan 18]. Available from: https://eprints.utas.edu.au/15014/3/whole-ng-thesis-incl-pub-mat.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ng J. Elucidating the roles of aurora B kinase in neurons. [Thesis]. University of Tasmania; 2012. Available from: https://eprints.utas.edu.au/15014/3/whole-ng-thesis-incl-pub-mat.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

19. Tsantikos, Evelyn. Dissecting autoimmune disease susceptibility in Lyn-deficient mice.

Degree: 2011, University of Melbourne

 This thesis dissects the nature of autoimmune disease development in mice deficient in the tyrosine kinase Lyn. In particular, the role of cellular defects, genetics,… (more)

Subjects/Keywords: autoimmune disease; B cells; inflammation; genetic interaction; Lyn kinase

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APA (6th Edition):

Tsantikos, E. (2011). Dissecting autoimmune disease susceptibility in Lyn-deficient mice. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/36280

Chicago Manual of Style (16th Edition):

Tsantikos, Evelyn. “Dissecting autoimmune disease susceptibility in Lyn-deficient mice.” 2011. Doctoral Dissertation, University of Melbourne. Accessed January 18, 2021. http://hdl.handle.net/11343/36280.

MLA Handbook (7th Edition):

Tsantikos, Evelyn. “Dissecting autoimmune disease susceptibility in Lyn-deficient mice.” 2011. Web. 18 Jan 2021.

Vancouver:

Tsantikos E. Dissecting autoimmune disease susceptibility in Lyn-deficient mice. [Internet] [Doctoral dissertation]. University of Melbourne; 2011. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/11343/36280.

Council of Science Editors:

Tsantikos E. Dissecting autoimmune disease susceptibility in Lyn-deficient mice. [Doctoral Dissertation]. University of Melbourne; 2011. Available from: http://hdl.handle.net/11343/36280


University of Arizona

20. Dietrich, Justin David. The Design, Synthesis, and Evaluation of Novel DFG-out Allosteric Kinase Inhibitors .

Degree: 2008, University of Arizona

 Today, current drug discovery and lead generation efforts focus on high throughput screening of large chemical libraries as the primary source of lead candidates. A… (more)

Subjects/Keywords: Allosteric; B-Raf; Kinase; p38

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APA (6th Edition):

Dietrich, J. D. (2008). The Design, Synthesis, and Evaluation of Novel DFG-out Allosteric Kinase Inhibitors . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195661

Chicago Manual of Style (16th Edition):

Dietrich, Justin David. “The Design, Synthesis, and Evaluation of Novel DFG-out Allosteric Kinase Inhibitors .” 2008. Doctoral Dissertation, University of Arizona. Accessed January 18, 2021. http://hdl.handle.net/10150/195661.

MLA Handbook (7th Edition):

Dietrich, Justin David. “The Design, Synthesis, and Evaluation of Novel DFG-out Allosteric Kinase Inhibitors .” 2008. Web. 18 Jan 2021.

Vancouver:

Dietrich JD. The Design, Synthesis, and Evaluation of Novel DFG-out Allosteric Kinase Inhibitors . [Internet] [Doctoral dissertation]. University of Arizona; 2008. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10150/195661.

Council of Science Editors:

Dietrich JD. The Design, Synthesis, and Evaluation of Novel DFG-out Allosteric Kinase Inhibitors . [Doctoral Dissertation]. University of Arizona; 2008. Available from: http://hdl.handle.net/10150/195661


University of Tennessee – Knoxville

21. Bai, Xiaofei. Investigating the Roles of Master Cell Cycle Regulators during Cytokinesis and Embryonic Development in <i>Caenorhabditis elegans</i>.

Degree: 2018, University of Tennessee – Knoxville

 Faithful cell division is required to maintain ploidy and generate daughter cells with necessary genetic components for life. During mitosis, dividing cells face the challenge… (more)

Subjects/Keywords: Caenorhabditis elegans; Cell Division; Cytokinesis; RAB-11; Separase; Aurora B kinase

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APA (6th Edition):

Bai, X. (2018). Investigating the Roles of Master Cell Cycle Regulators during Cytokinesis and Embryonic Development in <i>Caenorhabditis elegans</i>. (Doctoral Dissertation). University of Tennessee – Knoxville. Retrieved from https://trace.tennessee.edu/utk_graddiss/4919

Chicago Manual of Style (16th Edition):

Bai, Xiaofei. “Investigating the Roles of Master Cell Cycle Regulators during Cytokinesis and Embryonic Development in <i>Caenorhabditis elegans</i>.” 2018. Doctoral Dissertation, University of Tennessee – Knoxville. Accessed January 18, 2021. https://trace.tennessee.edu/utk_graddiss/4919.

MLA Handbook (7th Edition):

Bai, Xiaofei. “Investigating the Roles of Master Cell Cycle Regulators during Cytokinesis and Embryonic Development in <i>Caenorhabditis elegans</i>.” 2018. Web. 18 Jan 2021.

Vancouver:

Bai X. Investigating the Roles of Master Cell Cycle Regulators during Cytokinesis and Embryonic Development in <i>Caenorhabditis elegans</i>. [Internet] [Doctoral dissertation]. University of Tennessee – Knoxville; 2018. [cited 2021 Jan 18]. Available from: https://trace.tennessee.edu/utk_graddiss/4919.

Council of Science Editors:

Bai X. Investigating the Roles of Master Cell Cycle Regulators during Cytokinesis and Embryonic Development in <i>Caenorhabditis elegans</i>. [Doctoral Dissertation]. University of Tennessee – Knoxville; 2018. Available from: https://trace.tennessee.edu/utk_graddiss/4919


University of Georgia

22. McNeill, Brian Wayne. Adenosine receptor mediated inhibition of tumor necrosis factor alpha in human monocytic cells.

Degree: 2014, University of Georgia

 Sepsis and septic shock is a continuing problem in the United States with an estimated 200,000 deaths annually. Septic shock occurs when conserved pathogen-derived mediators… (more)

Subjects/Keywords: LPS; sepsis; adenosine; TNFα; monocyte; NFκ; B; MAP kinase

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APA (6th Edition):

McNeill, B. W. (2014). Adenosine receptor mediated inhibition of tumor necrosis factor alpha in human monocytic cells. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/21588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McNeill, Brian Wayne. “Adenosine receptor mediated inhibition of tumor necrosis factor alpha in human monocytic cells.” 2014. Thesis, University of Georgia. Accessed January 18, 2021. http://hdl.handle.net/10724/21588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McNeill, Brian Wayne. “Adenosine receptor mediated inhibition of tumor necrosis factor alpha in human monocytic cells.” 2014. Web. 18 Jan 2021.

Vancouver:

McNeill BW. Adenosine receptor mediated inhibition of tumor necrosis factor alpha in human monocytic cells. [Internet] [Thesis]. University of Georgia; 2014. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10724/21588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McNeill BW. Adenosine receptor mediated inhibition of tumor necrosis factor alpha in human monocytic cells. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/21588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. PIMENTA FILHO, Adenor Almeida. Impacto da esquistossomose mansônica no perfil lipídico e nas concentrações plasmáticas das apolipoproteínas A-I e B .

Degree: 2009, Universidade Federal de Pernambuco

 A esquistossomose é uma doença parasitária intravascular presente em regiões de climas tropical e subtropical que acomete, aproximadamente, 200 milhões de pessoas no mundo. Estudos… (more)

Subjects/Keywords: Apolipoproteína B; Apolipoproteína A-I; lipídios; esquistossomose

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APA (6th Edition):

PIMENTA FILHO, A. A. (2009). Impacto da esquistossomose mansônica no perfil lipídico e nas concentrações plasmáticas das apolipoproteínas A-I e B . (Thesis). Universidade Federal de Pernambuco. Retrieved from http://repositorio.ufpe.br/handle/123456789/1419

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

PIMENTA FILHO, Adenor Almeida. “Impacto da esquistossomose mansônica no perfil lipídico e nas concentrações plasmáticas das apolipoproteínas A-I e B .” 2009. Thesis, Universidade Federal de Pernambuco. Accessed January 18, 2021. http://repositorio.ufpe.br/handle/123456789/1419.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

PIMENTA FILHO, Adenor Almeida. “Impacto da esquistossomose mansônica no perfil lipídico e nas concentrações plasmáticas das apolipoproteínas A-I e B .” 2009. Web. 18 Jan 2021.

Vancouver:

PIMENTA FILHO AA. Impacto da esquistossomose mansônica no perfil lipídico e nas concentrações plasmáticas das apolipoproteínas A-I e B . [Internet] [Thesis]. Universidade Federal de Pernambuco; 2009. [cited 2021 Jan 18]. Available from: http://repositorio.ufpe.br/handle/123456789/1419.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

PIMENTA FILHO AA. Impacto da esquistossomose mansônica no perfil lipídico e nas concentrações plasmáticas das apolipoproteínas A-I e B . [Thesis]. Universidade Federal de Pernambuco; 2009. Available from: http://repositorio.ufpe.br/handle/123456789/1419

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Aziz, Faisal. Contextual Analysis of IT-Business Alignment.

Degree: 2011, , School of Management

  IT-Business alignment is an imperative process in the success of any business to use the competitive advantage of the information technology. In this work,… (more)

Subjects/Keywords: Business; IT-Business Alignment; B/I Alignment

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APA (6th Edition):

Aziz, F. (2011). Contextual Analysis of IT-Business Alignment. (Thesis). , School of Management. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:bth-6020

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aziz, Faisal. “Contextual Analysis of IT-Business Alignment.” 2011. Thesis, , School of Management. Accessed January 18, 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:bth-6020.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aziz, Faisal. “Contextual Analysis of IT-Business Alignment.” 2011. Web. 18 Jan 2021.

Vancouver:

Aziz F. Contextual Analysis of IT-Business Alignment. [Internet] [Thesis]. , School of Management; 2011. [cited 2021 Jan 18]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:bth-6020.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aziz F. Contextual Analysis of IT-Business Alignment. [Thesis]. , School of Management; 2011. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:bth-6020

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Quetglas, Maria Daniela. Efeito do bloqueio meiótico na expressão, atividade e distribuição do fator promotor da meiose (MPF) e da proteína cinase ativada por mitógenos (MAPK) em oócitos bovinos.

Degree: PhD, Qualidade e Produtividade Animal, 2008, University of São Paulo

Apesar dos grandes avanços na produção in vitro (PIV) de embriões bovinos, a produção de blastocistos se mantém ainda aquém do observado in vivo, indicando… (more)

Subjects/Keywords: Butirolactona I; Butyrolactone I; Competence; Competência; Expressão; Expression; Maturação; Maturation; Protein kinase; Proteína cinase

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APA (6th Edition):

Quetglas, M. D. (2008). Efeito do bloqueio meiótico na expressão, atividade e distribuição do fator promotor da meiose (MPF) e da proteína cinase ativada por mitógenos (MAPK) em oócitos bovinos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/74/74131/tde-11032008-102815/ ;

Chicago Manual of Style (16th Edition):

Quetglas, Maria Daniela. “Efeito do bloqueio meiótico na expressão, atividade e distribuição do fator promotor da meiose (MPF) e da proteína cinase ativada por mitógenos (MAPK) em oócitos bovinos.” 2008. Doctoral Dissertation, University of São Paulo. Accessed January 18, 2021. http://www.teses.usp.br/teses/disponiveis/74/74131/tde-11032008-102815/ ;.

MLA Handbook (7th Edition):

Quetglas, Maria Daniela. “Efeito do bloqueio meiótico na expressão, atividade e distribuição do fator promotor da meiose (MPF) e da proteína cinase ativada por mitógenos (MAPK) em oócitos bovinos.” 2008. Web. 18 Jan 2021.

Vancouver:

Quetglas MD. Efeito do bloqueio meiótico na expressão, atividade e distribuição do fator promotor da meiose (MPF) e da proteína cinase ativada por mitógenos (MAPK) em oócitos bovinos. [Internet] [Doctoral dissertation]. University of São Paulo; 2008. [cited 2021 Jan 18]. Available from: http://www.teses.usp.br/teses/disponiveis/74/74131/tde-11032008-102815/ ;.

Council of Science Editors:

Quetglas MD. Efeito do bloqueio meiótico na expressão, atividade e distribuição do fator promotor da meiose (MPF) e da proteína cinase ativada por mitógenos (MAPK) em oócitos bovinos. [Doctoral Dissertation]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/74/74131/tde-11032008-102815/ ;


University of Kentucky

26. Johnson, Jeremy Andrew. Examining the Role of Metabolic Pathways as Therapeutic Modalities for Triple Negative Breast Cancer.

Degree: 2020, University of Kentucky

 Triple negative breast cancer (TNBC) comprises 15-20% of breast cancers, affects a younger patient population than other subtypes, and is very aggressive. TNBC is comprised… (more)

Subjects/Keywords: Triple Negative Breast Cancer; AMP-Activated Protein Kinase; Protein Kinase B; Chemotherapy; Radiotherapy; Medical Cell Biology; Medical Molecular Biology; Oncology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Johnson, J. A. (2020). Examining the Role of Metabolic Pathways as Therapeutic Modalities for Triple Negative Breast Cancer. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/toxicology_etds/32

Chicago Manual of Style (16th Edition):

Johnson, Jeremy Andrew. “Examining the Role of Metabolic Pathways as Therapeutic Modalities for Triple Negative Breast Cancer.” 2020. Doctoral Dissertation, University of Kentucky. Accessed January 18, 2021. https://uknowledge.uky.edu/toxicology_etds/32.

MLA Handbook (7th Edition):

Johnson, Jeremy Andrew. “Examining the Role of Metabolic Pathways as Therapeutic Modalities for Triple Negative Breast Cancer.” 2020. Web. 18 Jan 2021.

Vancouver:

Johnson JA. Examining the Role of Metabolic Pathways as Therapeutic Modalities for Triple Negative Breast Cancer. [Internet] [Doctoral dissertation]. University of Kentucky; 2020. [cited 2021 Jan 18]. Available from: https://uknowledge.uky.edu/toxicology_etds/32.

Council of Science Editors:

Johnson JA. Examining the Role of Metabolic Pathways as Therapeutic Modalities for Triple Negative Breast Cancer. [Doctoral Dissertation]. University of Kentucky; 2020. Available from: https://uknowledge.uky.edu/toxicology_etds/32


University of Illinois – Chicago

27. Taglieri, Domenico M. P21-Activated Kinase-1 Signaling in Myocardial Hypertrophy, Ischemic Heart Disease and Remodeling.

Degree: 2012, University of Illinois – Chicago

 P21-Activated Kinase-1 Signaling in Myocardial Hypertrophy, Ischemic Heart Disease and Remodeling Domenico M. Taglieri, M.D., Ph.D. Department of Physiology and Biophysics University of Illinois at… (more)

Subjects/Keywords: Myocardial hypertrophy; P-21-Activated kinase-1Erk 1/2Cardiac Troponin I

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Taglieri, D. M. (2012). P21-Activated Kinase-1 Signaling in Myocardial Hypertrophy, Ischemic Heart Disease and Remodeling. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9091

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Taglieri, Domenico M. “P21-Activated Kinase-1 Signaling in Myocardial Hypertrophy, Ischemic Heart Disease and Remodeling.” 2012. Thesis, University of Illinois – Chicago. Accessed January 18, 2021. http://hdl.handle.net/10027/9091.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Taglieri, Domenico M. “P21-Activated Kinase-1 Signaling in Myocardial Hypertrophy, Ischemic Heart Disease and Remodeling.” 2012. Web. 18 Jan 2021.

Vancouver:

Taglieri DM. P21-Activated Kinase-1 Signaling in Myocardial Hypertrophy, Ischemic Heart Disease and Remodeling. [Internet] [Thesis]. University of Illinois – Chicago; 2012. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10027/9091.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Taglieri DM. P21-Activated Kinase-1 Signaling in Myocardial Hypertrophy, Ischemic Heart Disease and Remodeling. [Thesis]. University of Illinois – Chicago; 2012. Available from: http://hdl.handle.net/10027/9091

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Bordeaux I

28. Tauzin, Sébastien. Association oncogénique de Lyn et PAG dans les lymphomes B non Hodgkinien : Statistical problems encountered in the study of antiretroviral treatment of adults infected with HIV in sub-Saharan Africa.

Degree: Docteur es, Biologie cellulaire et physiopathologie, 2008, Université de Bordeaux I

Les lymphomes B non-Hodgkiniens sont caractérisés par des altérations génétiques à l’origine du syndrôme néoplasique. Dans ce travail, nous démontrons que l’association dans les microdomaines… (more)

Subjects/Keywords: Lymphomes B non-Hodgkiniens; Lyn; Rafts; Signalisation; PAG; Oncogène; PI3K; Kinase; STAT3; B non Hodgkin lymphomas; Kinase; Lyn; Signaling; Oncogene; PAG; PI3K; STAT3; Rafts

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tauzin, S. (2008). Association oncogénique de Lyn et PAG dans les lymphomes B non Hodgkinien : Statistical problems encountered in the study of antiretroviral treatment of adults infected with HIV in sub-Saharan Africa. (Doctoral Dissertation). Université de Bordeaux I. Retrieved from http://www.theses.fr/2008BOR13619

Chicago Manual of Style (16th Edition):

Tauzin, Sébastien. “Association oncogénique de Lyn et PAG dans les lymphomes B non Hodgkinien : Statistical problems encountered in the study of antiretroviral treatment of adults infected with HIV in sub-Saharan Africa.” 2008. Doctoral Dissertation, Université de Bordeaux I. Accessed January 18, 2021. http://www.theses.fr/2008BOR13619.

MLA Handbook (7th Edition):

Tauzin, Sébastien. “Association oncogénique de Lyn et PAG dans les lymphomes B non Hodgkinien : Statistical problems encountered in the study of antiretroviral treatment of adults infected with HIV in sub-Saharan Africa.” 2008. Web. 18 Jan 2021.

Vancouver:

Tauzin S. Association oncogénique de Lyn et PAG dans les lymphomes B non Hodgkinien : Statistical problems encountered in the study of antiretroviral treatment of adults infected with HIV in sub-Saharan Africa. [Internet] [Doctoral dissertation]. Université de Bordeaux I; 2008. [cited 2021 Jan 18]. Available from: http://www.theses.fr/2008BOR13619.

Council of Science Editors:

Tauzin S. Association oncogénique de Lyn et PAG dans les lymphomes B non Hodgkinien : Statistical problems encountered in the study of antiretroviral treatment of adults infected with HIV in sub-Saharan Africa. [Doctoral Dissertation]. Université de Bordeaux I; 2008. Available from: http://www.theses.fr/2008BOR13619


Université Montpellier II

29. Brioudes, Estelle. RSK2 et Greatwall, deux AGC kinases actrices de la mitose : RSK2 and Greatwall, two AGC kinases involved in the regulation of mitosis.

Degree: Docteur es, Biologie cellulaire et moléculaire. Biochimie, 2010, Université Montpellier II

La mitose est une phase importante du cycle cellulaire. Les mécanismes de surveillance s'assurent de l'ordre et de l'exécution correcte des événements du cycle cellulaire… (more)

Subjects/Keywords: Mitose; Point de contrôle du fuseau mitotique; Rsk2; Greatwall; AGC kinase; Cycline B/cdk1; Mitosis; Spindle assembly checkpoint; Rsk2; Greatwall; AGC kinase; Cyclin B/Cdk1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brioudes, E. (2010). RSK2 et Greatwall, deux AGC kinases actrices de la mitose : RSK2 and Greatwall, two AGC kinases involved in the regulation of mitosis. (Doctoral Dissertation). Université Montpellier II. Retrieved from http://www.theses.fr/2010MON20251

Chicago Manual of Style (16th Edition):

Brioudes, Estelle. “RSK2 et Greatwall, deux AGC kinases actrices de la mitose : RSK2 and Greatwall, two AGC kinases involved in the regulation of mitosis.” 2010. Doctoral Dissertation, Université Montpellier II. Accessed January 18, 2021. http://www.theses.fr/2010MON20251.

MLA Handbook (7th Edition):

Brioudes, Estelle. “RSK2 et Greatwall, deux AGC kinases actrices de la mitose : RSK2 and Greatwall, two AGC kinases involved in the regulation of mitosis.” 2010. Web. 18 Jan 2021.

Vancouver:

Brioudes E. RSK2 et Greatwall, deux AGC kinases actrices de la mitose : RSK2 and Greatwall, two AGC kinases involved in the regulation of mitosis. [Internet] [Doctoral dissertation]. Université Montpellier II; 2010. [cited 2021 Jan 18]. Available from: http://www.theses.fr/2010MON20251.

Council of Science Editors:

Brioudes E. RSK2 et Greatwall, deux AGC kinases actrices de la mitose : RSK2 and Greatwall, two AGC kinases involved in the regulation of mitosis. [Doctoral Dissertation]. Université Montpellier II; 2010. Available from: http://www.theses.fr/2010MON20251


Columbia University

30. De, Arnab. Understanding two inhibitors of NF-κB: A20 and IκBβ.

Degree: 2014, Columbia University

 While prompt activation of NF-κB is essential for optimal immune response, it is equally important to terminate the response to avoid tissue damage and perhaps… (more)

Subjects/Keywords: Glycogen synthase kinase-3; NF-kappa B (DNA-binding protein); Immunology; Biochemistry; Chemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

De, A. (2014). Understanding two inhibitors of NF-κB: A20 and IκBβ. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8QC01JX

Chicago Manual of Style (16th Edition):

De, Arnab. “Understanding two inhibitors of NF-κB: A20 and IκBβ.” 2014. Doctoral Dissertation, Columbia University. Accessed January 18, 2021. https://doi.org/10.7916/D8QC01JX.

MLA Handbook (7th Edition):

De, Arnab. “Understanding two inhibitors of NF-κB: A20 and IκBβ.” 2014. Web. 18 Jan 2021.

Vancouver:

De A. Understanding two inhibitors of NF-κB: A20 and IκBβ. [Internet] [Doctoral dissertation]. Columbia University; 2014. [cited 2021 Jan 18]. Available from: https://doi.org/10.7916/D8QC01JX.

Council of Science Editors:

De A. Understanding two inhibitors of NF-κB: A20 and IκBβ. [Doctoral Dissertation]. Columbia University; 2014. Available from: https://doi.org/10.7916/D8QC01JX

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