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You searched for subject:(Hypoxia inducible factor 1). Showing records 1 – 30 of 19442 total matches.

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Queens University

1. Sullivan, Richard. The multifactorial nature of hypoxia-induced drug resistance in cancer: involvement of hypoxia-inducible factor 1 .

Degree: Anatomy & Cell Biology, 2008, Queens University

 The development of intratumoral hypoxia is associated with resistance to therapy in many forms of human cancer, and pre-exposure of tumor cells to hypoxia confers… (more)

Subjects/Keywords: Hypoxia; Drug resistance; Cancer; Hypoxia-inducible factor 1

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APA (6th Edition):

Sullivan, R. (2008). The multifactorial nature of hypoxia-induced drug resistance in cancer: involvement of hypoxia-inducible factor 1 . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/1394

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sullivan, Richard. “The multifactorial nature of hypoxia-induced drug resistance in cancer: involvement of hypoxia-inducible factor 1 .” 2008. Thesis, Queens University. Accessed September 22, 2017. http://hdl.handle.net/1974/1394.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sullivan, Richard. “The multifactorial nature of hypoxia-induced drug resistance in cancer: involvement of hypoxia-inducible factor 1 .” 2008. Web. 22 Sep 2017.

Vancouver:

Sullivan R. The multifactorial nature of hypoxia-induced drug resistance in cancer: involvement of hypoxia-inducible factor 1 . [Internet] [Thesis]. Queens University; 2008. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/1974/1394.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sullivan R. The multifactorial nature of hypoxia-induced drug resistance in cancer: involvement of hypoxia-inducible factor 1 . [Thesis]. Queens University; 2008. Available from: http://hdl.handle.net/1974/1394

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

2. Nguyen, Andrew Tuan Duc. Oxygen-Mediated Regulation of Cholesterol Synthesis Through Accelerated Degradation of HMG COA Reductase.

Degree: 2009, University of Texas Southwestern Medical Center

 Endoplasmic reticulum-associated degradation of the enzyme 3-hydroxy-3-methylglutaryl CoA reductase represents one mechanism by which cholesterol synthesis is controlled in mammalian cells. The key reaction in… (more)

Subjects/Keywords: Hydroxymethylglutaryl CoA Reductases; Hypoxia-Inducible Factor 1, alpha Subunit; Cell Hypoxia

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APA (6th Edition):

Nguyen, A. T. D. (2009). Oxygen-Mediated Regulation of Cholesterol Synthesis Through Accelerated Degradation of HMG COA Reductase. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/514

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nguyen, Andrew Tuan Duc. “Oxygen-Mediated Regulation of Cholesterol Synthesis Through Accelerated Degradation of HMG COA Reductase.” 2009. Thesis, University of Texas Southwestern Medical Center. Accessed September 22, 2017. http://hdl.handle.net/2152.5/514.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nguyen, Andrew Tuan Duc. “Oxygen-Mediated Regulation of Cholesterol Synthesis Through Accelerated Degradation of HMG COA Reductase.” 2009. Web. 22 Sep 2017.

Vancouver:

Nguyen ATD. Oxygen-Mediated Regulation of Cholesterol Synthesis Through Accelerated Degradation of HMG COA Reductase. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2009. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/2152.5/514.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nguyen ATD. Oxygen-Mediated Regulation of Cholesterol Synthesis Through Accelerated Degradation of HMG COA Reductase. [Thesis]. University of Texas Southwestern Medical Center; 2009. Available from: http://hdl.handle.net/2152.5/514

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

3. Kim, Judy. A MECHANISM OF REGULATION OF HYPOXIA-INDUCIBLE FACTOR 1 ALPHA BY NITRIC OXIDE .

Degree: Anatomy & Cell Biology, 2014, Queens University

 Low tissue oxygen levels, known as hypoxia, characterize many solid cancers. Both clinical and experimental studies have demonstrated that intratumoral hypoxia is associated with malignant… (more)

Subjects/Keywords: Glyceryl trinitrate; Cyclic guanosine monophosphate; Tumour hypoxia; Nitric oxide; Calpain; Hypoxia-inducible factor 1

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APA (6th Edition):

Kim, J. (2014). A MECHANISM OF REGULATION OF HYPOXIA-INDUCIBLE FACTOR 1 ALPHA BY NITRIC OXIDE . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/12441

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kim, Judy. “A MECHANISM OF REGULATION OF HYPOXIA-INDUCIBLE FACTOR 1 ALPHA BY NITRIC OXIDE .” 2014. Thesis, Queens University. Accessed September 22, 2017. http://hdl.handle.net/1974/12441.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kim, Judy. “A MECHANISM OF REGULATION OF HYPOXIA-INDUCIBLE FACTOR 1 ALPHA BY NITRIC OXIDE .” 2014. Web. 22 Sep 2017.

Vancouver:

Kim J. A MECHANISM OF REGULATION OF HYPOXIA-INDUCIBLE FACTOR 1 ALPHA BY NITRIC OXIDE . [Internet] [Thesis]. Queens University; 2014. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/1974/12441.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kim J. A MECHANISM OF REGULATION OF HYPOXIA-INDUCIBLE FACTOR 1 ALPHA BY NITRIC OXIDE . [Thesis]. Queens University; 2014. Available from: http://hdl.handle.net/1974/12441

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wright State University

4. Kulkarni, Kashmira. HIF-1 alpha: a master regulator of trophoblast differentiation and placental development.

Degree: PhD, Biomedical Sciences PhD, 2009, Wright State University

 Early embryonic development occurs under low oxygen levels. The placenta is an organ transiently formed during pregnancy and plays a crucial role during development of… (more)

Subjects/Keywords: Cellular Biology; Molecular Biology; Hypoxia; Hypoxia inducible factor-1 alpha; Trophoblast; Differentiation; Placenta.

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APA (6th Edition):

Kulkarni, K. (2009). HIF-1 alpha: a master regulator of trophoblast differentiation and placental development. (Doctoral Dissertation). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1247186848

Chicago Manual of Style (16th Edition):

Kulkarni, Kashmira. “HIF-1 alpha: a master regulator of trophoblast differentiation and placental development.” 2009. Doctoral Dissertation, Wright State University. Accessed September 22, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=wright1247186848.

MLA Handbook (7th Edition):

Kulkarni, Kashmira. “HIF-1 alpha: a master regulator of trophoblast differentiation and placental development.” 2009. Web. 22 Sep 2017.

Vancouver:

Kulkarni K. HIF-1 alpha: a master regulator of trophoblast differentiation and placental development. [Internet] [Doctoral dissertation]. Wright State University; 2009. [cited 2017 Sep 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1247186848.

Council of Science Editors:

Kulkarni K. HIF-1 alpha: a master regulator of trophoblast differentiation and placental development. [Doctoral Dissertation]. Wright State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1247186848


Duke University

5. Moon, Eui Jung. The role of hypoxia-inducible factor-1 in hyperthermia-induced tumor reoxygenation and therapy resistance.

Degree: 2010, Duke University

  Imbalance between oxygen consumption and supply often makes tumors hypoxic (Bristow and Hill 2008). Tumor hypoxia is significantly correlated with aggressive tumor growth, ineffective… (more)

Subjects/Keywords: Biology, Physiology; Biology, Molecular; Hyperthermia; Hypoxia; Hypoxia-inducible factor-1; Reactive oxygen species; Reoxygenation; Tumor

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APA (6th Edition):

Moon, E. J. (2010). The role of hypoxia-inducible factor-1 in hyperthermia-induced tumor reoxygenation and therapy resistance. (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/3000

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Moon, Eui Jung. “The role of hypoxia-inducible factor-1 in hyperthermia-induced tumor reoxygenation and therapy resistance.” 2010. Thesis, Duke University. Accessed September 22, 2017. http://hdl.handle.net/10161/3000.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Moon, Eui Jung. “The role of hypoxia-inducible factor-1 in hyperthermia-induced tumor reoxygenation and therapy resistance.” 2010. Web. 22 Sep 2017.

Vancouver:

Moon EJ. The role of hypoxia-inducible factor-1 in hyperthermia-induced tumor reoxygenation and therapy resistance. [Internet] [Thesis]. Duke University; 2010. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/10161/3000.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Moon EJ. The role of hypoxia-inducible factor-1 in hyperthermia-induced tumor reoxygenation and therapy resistance. [Thesis]. Duke University; 2010. Available from: http://hdl.handle.net/10161/3000

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. L. Signorini. INTERACTION BETWEEN THE HYPOXIA INDUCIBLE FACTOR 1 ALPHA AND THE HUMAN POLYOMAVIRUS BK: A RISK FACTOR FOR THE DEVELOPMENT OF POLYOMAVIRUS ASSOCIATED NEPHROPATHY.

Degree: 2015, Università degli Studi di Milano

 Polyomaviruses are nonenveloped viruses with an icosahedral capsid of about 40-45 nm in diameter. The human polyomavirus BK (BKV) is a member of the Polyomaviridae… (more)

Subjects/Keywords: POLYOMAVIRUS; HUMAN POLYOMAVIRUS BK; HYPOXIA; HYPOXIA INDUCIBLE FACTOR 1 ALPHA; Settore MED/07 - Microbiologia e Microbiologia Clinica

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APA (6th Edition):

Signorini, L. (2015). INTERACTION BETWEEN THE HYPOXIA INDUCIBLE FACTOR 1 ALPHA AND THE HUMAN POLYOMAVIRUS BK: A RISK FACTOR FOR THE DEVELOPMENT OF POLYOMAVIRUS ASSOCIATED NEPHROPATHY. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/339318

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Signorini, L.. “INTERACTION BETWEEN THE HYPOXIA INDUCIBLE FACTOR 1 ALPHA AND THE HUMAN POLYOMAVIRUS BK: A RISK FACTOR FOR THE DEVELOPMENT OF POLYOMAVIRUS ASSOCIATED NEPHROPATHY.” 2015. Thesis, Università degli Studi di Milano. Accessed September 22, 2017. http://hdl.handle.net/2434/339318.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Signorini, L.. “INTERACTION BETWEEN THE HYPOXIA INDUCIBLE FACTOR 1 ALPHA AND THE HUMAN POLYOMAVIRUS BK: A RISK FACTOR FOR THE DEVELOPMENT OF POLYOMAVIRUS ASSOCIATED NEPHROPATHY.” 2015. Web. 22 Sep 2017.

Vancouver:

Signorini L. INTERACTION BETWEEN THE HYPOXIA INDUCIBLE FACTOR 1 ALPHA AND THE HUMAN POLYOMAVIRUS BK: A RISK FACTOR FOR THE DEVELOPMENT OF POLYOMAVIRUS ASSOCIATED NEPHROPATHY. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/2434/339318.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Signorini L. INTERACTION BETWEEN THE HYPOXIA INDUCIBLE FACTOR 1 ALPHA AND THE HUMAN POLYOMAVIRUS BK: A RISK FACTOR FOR THE DEVELOPMENT OF POLYOMAVIRUS ASSOCIATED NEPHROPATHY. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/339318

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

7. Tam, Kimberley Kai Yen. The role of hypoxia inducible factors in regulating ovarian function.

Degree: 2010, University of Adelaide

Hypoxia inducible factors (HIFs) are heterodimeric bHLH-PAS domain transcriptional factors that mediate physiological responses to low oxygen. There are 3 known isoforms. In normoxic conditions,… (more)

Subjects/Keywords: hypoxia inducible factor; ovary; ovulation; luteinisation; granulosa

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APA (6th Edition):

Tam, K. K. Y. (2010). The role of hypoxia inducible factors in regulating ovarian function. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/61245

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tam, Kimberley Kai Yen. “The role of hypoxia inducible factors in regulating ovarian function.” 2010. Thesis, University of Adelaide. Accessed September 22, 2017. http://hdl.handle.net/2440/61245.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tam, Kimberley Kai Yen. “The role of hypoxia inducible factors in regulating ovarian function.” 2010. Web. 22 Sep 2017.

Vancouver:

Tam KKY. The role of hypoxia inducible factors in regulating ovarian function. [Internet] [Thesis]. University of Adelaide; 2010. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/2440/61245.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tam KKY. The role of hypoxia inducible factors in regulating ovarian function. [Thesis]. University of Adelaide; 2010. Available from: http://hdl.handle.net/2440/61245

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

8. Lao, Taotao. OS-9 regulates hypoxia-inducible factor 1a in the endoplasmic reticulum.

Degree: 2011, Drexel University

Hypoxia-inducible factor 1 (HIF-1) is the master regulator of oxygen homeostasis and plays critical roles in cells’ responses to multiple signaling by activating proangiogenic progress… (more)

Subjects/Keywords: Biology; Hypoxia-inducible factor; Endoplasmic reticulum

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APA (6th Edition):

Lao, T. (2011). OS-9 regulates hypoxia-inducible factor 1a in the endoplasmic reticulum. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/3981

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lao, Taotao. “OS-9 regulates hypoxia-inducible factor 1a in the endoplasmic reticulum.” 2011. Thesis, Drexel University. Accessed September 22, 2017. http://hdl.handle.net/1860/3981.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lao, Taotao. “OS-9 regulates hypoxia-inducible factor 1a in the endoplasmic reticulum.” 2011. Web. 22 Sep 2017.

Vancouver:

Lao T. OS-9 regulates hypoxia-inducible factor 1a in the endoplasmic reticulum. [Internet] [Thesis]. Drexel University; 2011. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/1860/3981.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lao T. OS-9 regulates hypoxia-inducible factor 1a in the endoplasmic reticulum. [Thesis]. Drexel University; 2011. Available from: http://hdl.handle.net/1860/3981

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Paris-Sud – Paris XI

9. Noman, Muhammad zaeem. Influence of hypoxia on tumour cell susceptibility to cytotoxic T lymphocyte mediated lysis : Influence de l’hypoxie sur la susceptibilité des cellules tumorales à la lyse induite par les lymphocytes T cytotoxiques.

Degree: Docteur es, Cancérologie, 2012, Université Paris-Sud – Paris XI

 L’hypoxie est une caractéristique commune des tumeurs solides et l’une des spécificités du micro environnement tumoral. L’hypoxie tumorale joue un rôle important dans l’angio génèse,… (more)

Subjects/Keywords: Hypoxie; Lymphocytes T cytotoxiques; Autophagie; MicroRNA-210; Hypoxia Inducible Factor 1 alpha; Susceptibilité des cellules tumorale; Signal transducer and activator of transcription 3; Hypoxia; Cytotoxic T lymphocytes; Autophagy; MicroRNA-210; Hypoxia Inducible Factor 1 alpha; Tumor cell susceptibility; Signal transducer and activator of transcription 3

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APA (6th Edition):

Noman, M. z. (2012). Influence of hypoxia on tumour cell susceptibility to cytotoxic T lymphocyte mediated lysis : Influence de l’hypoxie sur la susceptibilité des cellules tumorales à la lyse induite par les lymphocytes T cytotoxiques. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2012PA11T051

Chicago Manual of Style (16th Edition):

Noman, Muhammad zaeem. “Influence of hypoxia on tumour cell susceptibility to cytotoxic T lymphocyte mediated lysis : Influence de l’hypoxie sur la susceptibilité des cellules tumorales à la lyse induite par les lymphocytes T cytotoxiques.” 2012. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed September 22, 2017. http://www.theses.fr/2012PA11T051.

MLA Handbook (7th Edition):

Noman, Muhammad zaeem. “Influence of hypoxia on tumour cell susceptibility to cytotoxic T lymphocyte mediated lysis : Influence de l’hypoxie sur la susceptibilité des cellules tumorales à la lyse induite par les lymphocytes T cytotoxiques.” 2012. Web. 22 Sep 2017.

Vancouver:

Noman Mz. Influence of hypoxia on tumour cell susceptibility to cytotoxic T lymphocyte mediated lysis : Influence de l’hypoxie sur la susceptibilité des cellules tumorales à la lyse induite par les lymphocytes T cytotoxiques. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2012. [cited 2017 Sep 22]. Available from: http://www.theses.fr/2012PA11T051.

Council of Science Editors:

Noman Mz. Influence of hypoxia on tumour cell susceptibility to cytotoxic T lymphocyte mediated lysis : Influence de l’hypoxie sur la susceptibilité des cellules tumorales à la lyse induite par les lymphocytes T cytotoxiques. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2012. Available from: http://www.theses.fr/2012PA11T051


Texas Medical Center

10. Philip, Kemly Mary; and#60;pand#62;and#60;a href="http://orcid.org/0000-0002-7749-9781" target="_blank"and#62;0000-0002-7749-9781and#60;/aand#62;and#60;/pand#62. The Hypoxic Adenosine Response Modulates Macrophage Differentiation and Contributes to Lung Disease.

Degree: PhD, 2017, Texas Medical Center

  Idiopathic pulmonary fibrosis (IPF) is a chronic lung disease which affects 5 - 8 million individuals worldwide and 200,000 individuals in the United States… (more)

Subjects/Keywords: idiopathic pulmonary fibrosis; adenosine; alternatively-activated macrophages; hypoxia-inducible factor 1-alpha; acute lung injury; Medicine and Health Sciences

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APA (6th Edition):

Philip, K. M. a. h. o. t. (2017). The Hypoxic Adenosine Response Modulates Macrophage Differentiation and Contributes to Lung Disease. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/734

Chicago Manual of Style (16th Edition):

Philip, Kemly Mary; and#60;pand#62;and#60;a href="http://orcid org/0000-0002-7749-9781" target="_blank"and#62;0000-0002-7749-9781and#60;/aand#62;and#60;/pand#62. “The Hypoxic Adenosine Response Modulates Macrophage Differentiation and Contributes to Lung Disease.” 2017. Doctoral Dissertation, Texas Medical Center. Accessed September 22, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/734.

MLA Handbook (7th Edition):

Philip, Kemly Mary; and#60;pand#62;and#60;a href="http://orcid org/0000-0002-7749-9781" target="_blank"and#62;0000-0002-7749-9781and#60;/aand#62;and#60;/pand#62. “The Hypoxic Adenosine Response Modulates Macrophage Differentiation and Contributes to Lung Disease.” 2017. Web. 22 Sep 2017.

Vancouver:

Philip KMahot. The Hypoxic Adenosine Response Modulates Macrophage Differentiation and Contributes to Lung Disease. [Internet] [Doctoral dissertation]. Texas Medical Center; 2017. [cited 2017 Sep 22]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/734.

Council of Science Editors:

Philip KMahot. The Hypoxic Adenosine Response Modulates Macrophage Differentiation and Contributes to Lung Disease. [Doctoral Dissertation]. Texas Medical Center; 2017. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/734


University of Oulu

11. Peurala, E. (Emmi). Regulators of hypoxia response and the cell cycle in breast cancer.

Degree: 2013, University of Oulu

Abstract Breast cancer is the most common cancer affecting the female population of the Western world. It is a heterogeneous disease entity that encompasses tumors… (more)

Subjects/Keywords: breast cancer; cyclin D1; hypoxia-inducible factor (HIF); hypoxiassa indusoituva tekijä (HIF); rintasyöpä; sykliini D1; CDK4; PHDs 1-3; p16

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APA (6th Edition):

Peurala, E. (. (2013). Regulators of hypoxia response and the cell cycle in breast cancer. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789526202709

Chicago Manual of Style (16th Edition):

Peurala, E (Emmi). “Regulators of hypoxia response and the cell cycle in breast cancer.” 2013. Doctoral Dissertation, University of Oulu. Accessed September 22, 2017. http://urn.fi/urn:isbn:9789526202709.

MLA Handbook (7th Edition):

Peurala, E (Emmi). “Regulators of hypoxia response and the cell cycle in breast cancer.” 2013. Web. 22 Sep 2017.

Vancouver:

Peurala E(. Regulators of hypoxia response and the cell cycle in breast cancer. [Internet] [Doctoral dissertation]. University of Oulu; 2013. [cited 2017 Sep 22]. Available from: http://urn.fi/urn:isbn:9789526202709.

Council of Science Editors:

Peurala E(. Regulators of hypoxia response and the cell cycle in breast cancer. [Doctoral Dissertation]. University of Oulu; 2013. Available from: http://urn.fi/urn:isbn:9789526202709

12. Mimouna, Sanda. E. coli adhérentes et invasives et pathogénèse de la maladie de Crohn : rôle du facteur hypoxique HIF-1 : Reduced-order statistical models of cardiac growth, motion and blood flow : application to the tetralogy of Fallot heart.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2013, Nice

La maladie de Crohn (MC) est une maladie inflammatoire chronique intestinale (MICI). Son incidence et sa prévalence ont augmenté en Europe au cours des dix… (more)

Subjects/Keywords: HIF-1α; Maladie de Crohn; Xénophagie; E.coli adhérentes et invasives; Hypoxia Inducible Factor 1; Crohn Disease; Xenophagy; Adherent and Invasive E.coli

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APA (6th Edition):

Mimouna, S. (2013). E. coli adhérentes et invasives et pathogénèse de la maladie de Crohn : rôle du facteur hypoxique HIF-1 : Reduced-order statistical models of cardiac growth, motion and blood flow : application to the tetralogy of Fallot heart. (Doctoral Dissertation). Nice. Retrieved from http://www.theses.fr/2013NICE4085

Chicago Manual of Style (16th Edition):

Mimouna, Sanda. “E. coli adhérentes et invasives et pathogénèse de la maladie de Crohn : rôle du facteur hypoxique HIF-1 : Reduced-order statistical models of cardiac growth, motion and blood flow : application to the tetralogy of Fallot heart.” 2013. Doctoral Dissertation, Nice. Accessed September 22, 2017. http://www.theses.fr/2013NICE4085.

MLA Handbook (7th Edition):

Mimouna, Sanda. “E. coli adhérentes et invasives et pathogénèse de la maladie de Crohn : rôle du facteur hypoxique HIF-1 : Reduced-order statistical models of cardiac growth, motion and blood flow : application to the tetralogy of Fallot heart.” 2013. Web. 22 Sep 2017.

Vancouver:

Mimouna S. E. coli adhérentes et invasives et pathogénèse de la maladie de Crohn : rôle du facteur hypoxique HIF-1 : Reduced-order statistical models of cardiac growth, motion and blood flow : application to the tetralogy of Fallot heart. [Internet] [Doctoral dissertation]. Nice; 2013. [cited 2017 Sep 22]. Available from: http://www.theses.fr/2013NICE4085.

Council of Science Editors:

Mimouna S. E. coli adhérentes et invasives et pathogénèse de la maladie de Crohn : rôle du facteur hypoxique HIF-1 : Reduced-order statistical models of cardiac growth, motion and blood flow : application to the tetralogy of Fallot heart. [Doctoral Dissertation]. Nice; 2013. Available from: http://www.theses.fr/2013NICE4085

13. WONG YUAN SY. Applying side effects of anti fibrotic compounds to promote neovascularization for tissue engineering.

Degree: 2008, National University of Singapore

Subjects/Keywords: neovascularization; prolyl hydroxylase inhibitors; hypoxia inducible factor-1

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APA (6th Edition):

SY, W. Y. (2008). Applying side effects of anti fibrotic compounds to promote neovascularization for tissue engineering. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/13184 ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/3/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/4/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/1/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/2/bitstream

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

SY, WONG YUAN. “Applying side effects of anti fibrotic compounds to promote neovascularization for tissue engineering.” 2008. Thesis, National University of Singapore. Accessed September 22, 2017. http://scholarbank.nus.edu.sg/handle/10635/13184 ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/3/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/4/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/1/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/2/bitstream.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

SY, WONG YUAN. “Applying side effects of anti fibrotic compounds to promote neovascularization for tissue engineering.” 2008. Web. 22 Sep 2017.

Vancouver:

SY WY. Applying side effects of anti fibrotic compounds to promote neovascularization for tissue engineering. [Internet] [Thesis]. National University of Singapore; 2008. [cited 2017 Sep 22]. Available from: http://scholarbank.nus.edu.sg/handle/10635/13184 ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/3/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/4/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/1/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/2/bitstream.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

SY WY. Applying side effects of anti fibrotic compounds to promote neovascularization for tissue engineering. [Thesis]. National University of Singapore; 2008. Available from: http://scholarbank.nus.edu.sg/handle/10635/13184 ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/3/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/4/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/1/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F13184/2/bitstream

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Iowa State University

14. Shao, Zhiyong. Roles of Caenorhabditis elegans EGL-9 in HIF-1 regulation and pathogen resistance.

Degree: 2010, Iowa State University

 Oxygen is required for most organisms to survive, and plays a critical role in the evolution of life. However, oxygen deprivation (hypoxia) may occur in… (more)

Subjects/Keywords: Caenorhabditis elegans; Gene expression; Hypoxia-inducible factor; Oxygen; Pseudomonas aeruginosa; SWAN-1; Cell and Developmental Biology; Genetics and Genomics

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APA (6th Edition):

Shao, Z. (2010). Roles of Caenorhabditis elegans EGL-9 in HIF-1 regulation and pathogen resistance. (Thesis). Iowa State University. Retrieved from http://lib.dr.iastate.edu/etd/11676

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shao, Zhiyong. “Roles of Caenorhabditis elegans EGL-9 in HIF-1 regulation and pathogen resistance.” 2010. Thesis, Iowa State University. Accessed September 22, 2017. http://lib.dr.iastate.edu/etd/11676.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shao, Zhiyong. “Roles of Caenorhabditis elegans EGL-9 in HIF-1 regulation and pathogen resistance.” 2010. Web. 22 Sep 2017.

Vancouver:

Shao Z. Roles of Caenorhabditis elegans EGL-9 in HIF-1 regulation and pathogen resistance. [Internet] [Thesis]. Iowa State University; 2010. [cited 2017 Sep 22]. Available from: http://lib.dr.iastate.edu/etd/11676.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shao Z. Roles of Caenorhabditis elegans EGL-9 in HIF-1 regulation and pathogen resistance. [Thesis]. Iowa State University; 2010. Available from: http://lib.dr.iastate.edu/etd/11676

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Ikemori, Rafael Yamashita. Análise da expressão de galectina-3 em células de glioma expostas a condições hipóxicas e seu papel no desenvolvimento de tumores in vivo.

Degree: PhD, Oncologia, 2014, University of São Paulo

 A galectina-3 (gal-3) pertence a uma família de proteínas com domínios de ligação a beta-galactosídeos e está relacionada com diversos aspectos tumorais, como proliferação e… (more)

Subjects/Keywords: Autofagia; Autophagy; Cell death; Fator 1 induzível por hipóxia; Galectin-3; Galectina-3; Glioma; Glioma; Hipóxia; Hypoxia; Hypoxia inducible factor 1; Morte celular

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APA (6th Edition):

Ikemori, R. Y. (2014). Análise da expressão de galectina-3 em células de glioma expostas a condições hipóxicas e seu papel no desenvolvimento de tumores in vivo. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5155/tde-14082014-105241/ ;

Chicago Manual of Style (16th Edition):

Ikemori, Rafael Yamashita. “Análise da expressão de galectina-3 em células de glioma expostas a condições hipóxicas e seu papel no desenvolvimento de tumores in vivo.” 2014. Doctoral Dissertation, University of São Paulo. Accessed September 22, 2017. http://www.teses.usp.br/teses/disponiveis/5/5155/tde-14082014-105241/ ;.

MLA Handbook (7th Edition):

Ikemori, Rafael Yamashita. “Análise da expressão de galectina-3 em células de glioma expostas a condições hipóxicas e seu papel no desenvolvimento de tumores in vivo.” 2014. Web. 22 Sep 2017.

Vancouver:

Ikemori RY. Análise da expressão de galectina-3 em células de glioma expostas a condições hipóxicas e seu papel no desenvolvimento de tumores in vivo. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2017 Sep 22]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-14082014-105241/ ;.

Council of Science Editors:

Ikemori RY. Análise da expressão de galectina-3 em células de glioma expostas a condições hipóxicas e seu papel no desenvolvimento de tumores in vivo. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-14082014-105241/ ;


Universidad de Chile

16. Sanhueza Muñoz, Carlos Joaquín. Caveolina-1 reduce la transcripción dependiente de hif1α en un mecanismo dependiente del óxido nítrico en células tumorales .

Degree: 2013, Universidad de Chile

 El cáncer es la 2° causa de muerte en Chile. El desarrollo del cáncer se ha propuesto que es consecuencia de la pérdida de función… (more)

Subjects/Keywords: Caveolinas; Factor 1 Inducible por Hipoxia

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APA (6th Edition):

Sanhueza Muñoz, C. J. (2013). Caveolina-1 reduce la transcripción dependiente de hif1α en un mecanismo dependiente del óxido nítrico en células tumorales . (Thesis). Universidad de Chile. Retrieved from http://www.repositorio.uchile.cl/handle/2250/114871

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sanhueza Muñoz, Carlos Joaquín. “Caveolina-1 reduce la transcripción dependiente de hif1α en un mecanismo dependiente del óxido nítrico en células tumorales .” 2013. Thesis, Universidad de Chile. Accessed September 22, 2017. http://www.repositorio.uchile.cl/handle/2250/114871.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sanhueza Muñoz, Carlos Joaquín. “Caveolina-1 reduce la transcripción dependiente de hif1α en un mecanismo dependiente del óxido nítrico en células tumorales .” 2013. Web. 22 Sep 2017.

Vancouver:

Sanhueza Muñoz CJ. Caveolina-1 reduce la transcripción dependiente de hif1α en un mecanismo dependiente del óxido nítrico en células tumorales . [Internet] [Thesis]. Universidad de Chile; 2013. [cited 2017 Sep 22]. Available from: http://www.repositorio.uchile.cl/handle/2250/114871.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sanhueza Muñoz CJ. Caveolina-1 reduce la transcripción dependiente de hif1α en un mecanismo dependiente del óxido nítrico en células tumorales . [Thesis]. Universidad de Chile; 2013. Available from: http://www.repositorio.uchile.cl/handle/2250/114871

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queensland University of Technology

17. Zhang, Xufang. Pro-angiogenic and anti-angiogenic factors in the degradation of osteoarthritic cartilage.

Degree: 2015, Queensland University of Technology

This project has determined angiogenic and anti-angiogenic factors in osteoarthritis cartilage. The work has expanded our knowledge and understanding of the importance of anti-angiogenic factors in maintaining cartilage homeostasis. This study also tested the concept of topical application of anti-angiogenic treatment strategy for osteoarthritis.

Subjects/Keywords: angiogenesis inhibitors; rticular cartilage chondrocytes; cartilage degradation; chondrocyte hypertrophy; chondromodulin-1; hypoxia-inducible factor; osteoarthritis; vascular endothelial growth factor; angiogenesis; anti-angiogenesis

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APA (6th Edition):

Zhang, X. (2015). Pro-angiogenic and anti-angiogenic factors in the degradation of osteoarthritic cartilage. (Thesis). Queensland University of Technology. Retrieved from https://eprints.qut.edu.au/87318/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Xufang. “Pro-angiogenic and anti-angiogenic factors in the degradation of osteoarthritic cartilage.” 2015. Thesis, Queensland University of Technology. Accessed September 22, 2017. https://eprints.qut.edu.au/87318/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Xufang. “Pro-angiogenic and anti-angiogenic factors in the degradation of osteoarthritic cartilage.” 2015. Web. 22 Sep 2017.

Vancouver:

Zhang X. Pro-angiogenic and anti-angiogenic factors in the degradation of osteoarthritic cartilage. [Internet] [Thesis]. Queensland University of Technology; 2015. [cited 2017 Sep 22]. Available from: https://eprints.qut.edu.au/87318/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang X. Pro-angiogenic and anti-angiogenic factors in the degradation of osteoarthritic cartilage. [Thesis]. Queensland University of Technology; 2015. Available from: https://eprints.qut.edu.au/87318/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Thalyta Amanda Pinheiro Ferreira. Hypoxia-Inducible Factor-1α (HIF-1α) expression in radicular and dentigerous cysts: Immunohistochemical evaluation .

Degree: Master, 2017, Universidade Federal do Ceará

Os cistos odontogÃnicos sÃo classificados conforme sua origem em: cistos de desenvolvimento, dos quais faz parte o cisto dentÃgero (CD), ou em cistos inflamatÃrios, que… (more)

Subjects/Keywords: ODONTOLOGIA; Cistos OdontogÃnicos; Cisto Radicular; Cisto DentÃgero; Fator 1 InduzÃvel por HipÃxia; Odontogenic Cysts; Radicular Cyst; Dentigerous Cyst; Hypoxia-Inducible Factor 1

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APA (6th Edition):

Ferreira, T. A. P. (2017). Hypoxia-Inducible Factor-1α (HIF-1α) expression in radicular and dentigerous cysts: Immunohistochemical evaluation . (Masters Thesis). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=19110 ;

Chicago Manual of Style (16th Edition):

Ferreira, Thalyta Amanda Pinheiro. “Hypoxia-Inducible Factor-1α (HIF-1α) expression in radicular and dentigerous cysts: Immunohistochemical evaluation .” 2017. Masters Thesis, Universidade Federal do Ceará. Accessed September 22, 2017. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=19110 ;.

MLA Handbook (7th Edition):

Ferreira, Thalyta Amanda Pinheiro. “Hypoxia-Inducible Factor-1α (HIF-1α) expression in radicular and dentigerous cysts: Immunohistochemical evaluation .” 2017. Web. 22 Sep 2017.

Vancouver:

Ferreira TAP. Hypoxia-Inducible Factor-1α (HIF-1α) expression in radicular and dentigerous cysts: Immunohistochemical evaluation . [Internet] [Masters thesis]. Universidade Federal do Ceará 2017. [cited 2017 Sep 22]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=19110 ;.

Council of Science Editors:

Ferreira TAP. Hypoxia-Inducible Factor-1α (HIF-1α) expression in radicular and dentigerous cysts: Immunohistochemical evaluation . [Masters Thesis]. Universidade Federal do Ceará 2017. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=19110 ;


Humboldt University of Berlin

19. Wiesener, Michael S. Mechanismen und Konsequenzen sauerstoffabhängiger Genregulation.

Degree: 2003, Humboldt University of Berlin

 Die ständige Verfügbarkeit von molekularem Sauerstoff (O2) ist ein elementarer Bestandteil multizellulärer Lebensformen. Zur Aufrechterhaltung der Homöostase sind diese auf die Bildung des Energiesubstrates ATP… (more)

Subjects/Keywords: Medizin; Ischämie; Hypoxie; Sauerstoff; Tumor; Transkriptionsfaktor; HIF-1; EPAS-1; Hypoxia-inducible Factor-1; von Hippel Lindau; Ischemia; Hypoxia; HIF-1; EPAS-1; Hypoxia-inducible Factor-1; Transcription factor; von Hippel Lindau; Oxygen; Tumor; Medizin; ddc:610

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APA (6th Edition):

Wiesener, M. S. (2003). Mechanismen und Konsequenzen sauerstoffabhängiger Genregulation. (Doctoral Dissertation). Humboldt University of Berlin. Retrieved from http://edoc.hu-berlin.de/docviews/abstract.php?id=20187 ; http://edoc.hu-berlin.de/habilitationen/wiesener-michael-s-2003-10-23/PDF/Wiesener.pdf ; http://www.nbn-resolving.de/urn:nbn:de:kobv:11-10024752

Chicago Manual of Style (16th Edition):

Wiesener, Michael S. “Mechanismen und Konsequenzen sauerstoffabhängiger Genregulation.” 2003. Doctoral Dissertation, Humboldt University of Berlin. Accessed September 22, 2017. http://edoc.hu-berlin.de/docviews/abstract.php?id=20187 ; http://edoc.hu-berlin.de/habilitationen/wiesener-michael-s-2003-10-23/PDF/Wiesener.pdf ; http://www.nbn-resolving.de/urn:nbn:de:kobv:11-10024752.

MLA Handbook (7th Edition):

Wiesener, Michael S. “Mechanismen und Konsequenzen sauerstoffabhängiger Genregulation.” 2003. Web. 22 Sep 2017.

Vancouver:

Wiesener MS. Mechanismen und Konsequenzen sauerstoffabhängiger Genregulation. [Internet] [Doctoral dissertation]. Humboldt University of Berlin; 2003. [cited 2017 Sep 22]. Available from: http://edoc.hu-berlin.de/docviews/abstract.php?id=20187 ; http://edoc.hu-berlin.de/habilitationen/wiesener-michael-s-2003-10-23/PDF/Wiesener.pdf ; http://www.nbn-resolving.de/urn:nbn:de:kobv:11-10024752.

Council of Science Editors:

Wiesener MS. Mechanismen und Konsequenzen sauerstoffabhängiger Genregulation. [Doctoral Dissertation]. Humboldt University of Berlin; 2003. Available from: http://edoc.hu-berlin.de/docviews/abstract.php?id=20187 ; http://edoc.hu-berlin.de/habilitationen/wiesener-michael-s-2003-10-23/PDF/Wiesener.pdf ; http://www.nbn-resolving.de/urn:nbn:de:kobv:11-10024752

20. Lima, Camila Bárbara Cantalupo. Estudo da invasão de hepatócitos de rato por Shigella flexneri: análise da influência da hipóxia sobre a injúria celular.

Degree: Mestrado, Doenças Infecciosas e Parasitárias, 2012, University of São Paulo

O presente estudo avaliou a capacidade de invasão de hepatócitos de rato por Shigella flexneri (S. flexneri) nas condições de normóxia e hipóxia. O estudo… (more)

Subjects/Keywords: Apoptose; Apoptosis; Fator de necrose tumoral alfa; Hepatócitos; Hepatocytes; Hipóxia; Hypoxia; Hypoxia-inducible factor 1 alpha subunit; Ratos Wistar; Rats Wistar; Shigella flexneri; Shigella flexneri; Subunidade alfa do fator 1 induzível por hipóxia; Tumor necrosis factor alpha

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APA (6th Edition):

Lima, C. B. C. (2012). Estudo da invasão de hepatócitos de rato por Shigella flexneri: análise da influência da hipóxia sobre a injúria celular. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5134/tde-24042012-163857/ ;

Chicago Manual of Style (16th Edition):

Lima, Camila Bárbara Cantalupo. “Estudo da invasão de hepatócitos de rato por Shigella flexneri: análise da influência da hipóxia sobre a injúria celular.” 2012. Masters Thesis, University of São Paulo. Accessed September 22, 2017. http://www.teses.usp.br/teses/disponiveis/5/5134/tde-24042012-163857/ ;.

MLA Handbook (7th Edition):

Lima, Camila Bárbara Cantalupo. “Estudo da invasão de hepatócitos de rato por Shigella flexneri: análise da influência da hipóxia sobre a injúria celular.” 2012. Web. 22 Sep 2017.

Vancouver:

Lima CBC. Estudo da invasão de hepatócitos de rato por Shigella flexneri: análise da influência da hipóxia sobre a injúria celular. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2017 Sep 22]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5134/tde-24042012-163857/ ;.

Council of Science Editors:

Lima CBC. Estudo da invasão de hepatócitos de rato por Shigella flexneri: análise da influência da hipóxia sobre a injúria celular. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5134/tde-24042012-163857/ ;


University of New South Wales

21. Cheng, Kim. First phase insulin secretion.

Degree: Garvan Institute of Medical Research, 2012, University of New South Wales

 Type 2 diabetes (T2D) is a metabolic disorder characterised by the inability of β-cells to secrete enough insulin to maintain glucose homeostasis. Pancreatic β-cells secrete… (more)

Subjects/Keywords: Hypoxia inducible factor; Type 2 Diabetes; Insulin Secretion

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APA (6th Edition):

Cheng, K. (2012). First phase insulin secretion. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/52259

Chicago Manual of Style (16th Edition):

Cheng, Kim. “First phase insulin secretion.” 2012. Doctoral Dissertation, University of New South Wales. Accessed September 22, 2017. http://handle.unsw.edu.au/1959.4/52259.

MLA Handbook (7th Edition):

Cheng, Kim. “First phase insulin secretion.” 2012. Web. 22 Sep 2017.

Vancouver:

Cheng K. First phase insulin secretion. [Internet] [Doctoral dissertation]. University of New South Wales; 2012. [cited 2017 Sep 22]. Available from: http://handle.unsw.edu.au/1959.4/52259.

Council of Science Editors:

Cheng K. First phase insulin secretion. [Doctoral Dissertation]. University of New South Wales; 2012. Available from: http://handle.unsw.edu.au/1959.4/52259

22. Groenman, Freek. Hypoxia Inducible Factor: a breath of fresh air in lung development.

Degree: Department of Pediatric Surgery, 2007, Erasmus University Medical Center

 textabstractChapter 1 is a general introduction to normal and abnormal lung development. It outlines the current state of knowledge of the molecular basis of normal… (more)

Subjects/Keywords: hypoxia-inducible factor; lung development

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APA (6th Edition):

Groenman, F. (2007). Hypoxia Inducible Factor: a breath of fresh air in lung development. (Doctoral Dissertation). Erasmus University Medical Center. Retrieved from http://hdl.handle.net/1765/10347

Chicago Manual of Style (16th Edition):

Groenman, Freek. “Hypoxia Inducible Factor: a breath of fresh air in lung development.” 2007. Doctoral Dissertation, Erasmus University Medical Center. Accessed September 22, 2017. http://hdl.handle.net/1765/10347.

MLA Handbook (7th Edition):

Groenman, Freek. “Hypoxia Inducible Factor: a breath of fresh air in lung development.” 2007. Web. 22 Sep 2017.

Vancouver:

Groenman F. Hypoxia Inducible Factor: a breath of fresh air in lung development. [Internet] [Doctoral dissertation]. Erasmus University Medical Center; 2007. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/1765/10347.

Council of Science Editors:

Groenman F. Hypoxia Inducible Factor: a breath of fresh air in lung development. [Doctoral Dissertation]. Erasmus University Medical Center; 2007. Available from: http://hdl.handle.net/1765/10347

23. Matsuyama, Tomonori. Midazolam inhibits the hypoxia-induced up-regulation of erythropoietin in the central nervous system .

Degree: 2015, Kyoto University

Subjects/Keywords: Midazolam; Erythropoietin; Hypoxia-inducible factor

Page 1 Page 2

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APA (6th Edition):

Matsuyama, T. (2015). Midazolam inhibits the hypoxia-induced up-regulation of erythropoietin in the central nervous system . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/202803

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Matsuyama, Tomonori. “Midazolam inhibits the hypoxia-induced up-regulation of erythropoietin in the central nervous system .” 2015. Thesis, Kyoto University. Accessed September 22, 2017. http://hdl.handle.net/2433/202803.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Matsuyama, Tomonori. “Midazolam inhibits the hypoxia-induced up-regulation of erythropoietin in the central nervous system .” 2015. Web. 22 Sep 2017.

Vancouver:

Matsuyama T. Midazolam inhibits the hypoxia-induced up-regulation of erythropoietin in the central nervous system . [Internet] [Thesis]. Kyoto University; 2015. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/2433/202803.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Matsuyama T. Midazolam inhibits the hypoxia-induced up-regulation of erythropoietin in the central nervous system . [Thesis]. Kyoto University; 2015. Available from: http://hdl.handle.net/2433/202803

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University / 京都大学

24. Matsuyama, Tomonori. Midazolam inhibits the hypoxia-induced up-regulation of erythropoietin in the central nervous system : ミダゾラムは低酸素に誘導される脳内エリスロポイエチン発現上昇を抑制する.

Degree: 博士(医学), 2015, Kyoto University / 京都大学

新制・課程博士

甲第19369号

医博第4046号

Subjects/Keywords: Midazolam; Erythropoietin; Hypoxia-inducible factor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Matsuyama, T. (2015). Midazolam inhibits the hypoxia-induced up-regulation of erythropoietin in the central nervous system : ミダゾラムは低酸素に誘導される脳内エリスロポイエチン発現上昇を抑制する. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/202803 ; http://dx.doi.org/10.14989/doctor.k19369

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Matsuyama, Tomonori. “Midazolam inhibits the hypoxia-induced up-regulation of erythropoietin in the central nervous system : ミダゾラムは低酸素に誘導される脳内エリスロポイエチン発現上昇を抑制する.” 2015. Thesis, Kyoto University / 京都大学. Accessed September 22, 2017. http://hdl.handle.net/2433/202803 ; http://dx.doi.org/10.14989/doctor.k19369.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Matsuyama, Tomonori. “Midazolam inhibits the hypoxia-induced up-regulation of erythropoietin in the central nervous system : ミダゾラムは低酸素に誘導される脳内エリスロポイエチン発現上昇を抑制する.” 2015. Web. 22 Sep 2017.

Vancouver:

Matsuyama T. Midazolam inhibits the hypoxia-induced up-regulation of erythropoietin in the central nervous system : ミダゾラムは低酸素に誘導される脳内エリスロポイエチン発現上昇を抑制する. [Internet] [Thesis]. Kyoto University / 京都大学; 2015. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/2433/202803 ; http://dx.doi.org/10.14989/doctor.k19369.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Matsuyama T. Midazolam inhibits the hypoxia-induced up-regulation of erythropoietin in the central nervous system : ミダゾラムは低酸素に誘導される脳内エリスロポイエチン発現上昇を抑制する. [Thesis]. Kyoto University / 京都大学; 2015. Available from: http://hdl.handle.net/2433/202803 ; http://dx.doi.org/10.14989/doctor.k19369

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

25. Chen, Shuyang. AMPK-HDAC5 Pathway Facilitates Nuclear Accumulation of HIF-1alpha and Functional Activation of HIF-1 by Deacetylating Hsp70 in the Cytosol.

Degree: 2015, Drexel University

Hypoxia-inducible factor 1 (HIF-1) is a major transcriptional regulator of adaptation to hypoxia at both cellular and tissue levels. As such, HIF-1 plays an important… (more)

Subjects/Keywords: Biological sciences; Hypoxia-inducible factor; Coronary heart disease

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APA (6th Edition):

Chen, S. (2015). AMPK-HDAC5 Pathway Facilitates Nuclear Accumulation of HIF-1alpha and Functional Activation of HIF-1 by Deacetylating Hsp70 in the Cytosol. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:6331

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Shuyang. “AMPK-HDAC5 Pathway Facilitates Nuclear Accumulation of HIF-1alpha and Functional Activation of HIF-1 by Deacetylating Hsp70 in the Cytosol.” 2015. Thesis, Drexel University. Accessed September 22, 2017. http://hdl.handle.net/1860/idea:6331.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Shuyang. “AMPK-HDAC5 Pathway Facilitates Nuclear Accumulation of HIF-1alpha and Functional Activation of HIF-1 by Deacetylating Hsp70 in the Cytosol.” 2015. Web. 22 Sep 2017.

Vancouver:

Chen S. AMPK-HDAC5 Pathway Facilitates Nuclear Accumulation of HIF-1alpha and Functional Activation of HIF-1 by Deacetylating Hsp70 in the Cytosol. [Internet] [Thesis]. Drexel University; 2015. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/1860/idea:6331.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen S. AMPK-HDAC5 Pathway Facilitates Nuclear Accumulation of HIF-1alpha and Functional Activation of HIF-1 by Deacetylating Hsp70 in the Cytosol. [Thesis]. Drexel University; 2015. Available from: http://hdl.handle.net/1860/idea:6331

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

26. Asplund, Annika. The effect of hypoxia on macrophage proteoglycans: potential role in atherosclerosis.

Degree: 2009, University of Gothenburg / Göteborgs Universitet

 A critical step in the development of atherosclerotic lesions appears to be the retention of low density lipoproteins (LDL) in the arterial wall, mediated by… (more)

Subjects/Keywords: atherosclerosis; proteoglycans; glycosaminoglycans; macrophages; hypoxia; hypoxia-inducible transcription factor; LDL binding; cell motility

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APA (6th Edition):

Asplund, A. (2009). The effect of hypoxia on macrophage proteoglycans: potential role in atherosclerosis. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/21174

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Asplund, Annika. “The effect of hypoxia on macrophage proteoglycans: potential role in atherosclerosis.” 2009. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed September 22, 2017. http://hdl.handle.net/2077/21174.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Asplund, Annika. “The effect of hypoxia on macrophage proteoglycans: potential role in atherosclerosis.” 2009. Web. 22 Sep 2017.

Vancouver:

Asplund A. The effect of hypoxia on macrophage proteoglycans: potential role in atherosclerosis. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2009. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/2077/21174.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Asplund A. The effect of hypoxia on macrophage proteoglycans: potential role in atherosclerosis. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2009. Available from: http://hdl.handle.net/2077/21174

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

27. Li, Zhizhong. Differential Angiogenic Capability and Hypoxia Responses in Glioma Stem Cells.

Degree: 2009, Duke University

  Malignant gliomas are highly lethal cancers characterized by florid angiogenesis. Glioma stem cells (GSCs), enriched through CD133 (Prominin1) selection, are highly tumorigenic and therapy… (more)

Subjects/Keywords: Biology, Molecular; Angiogenesis; Cancer Stem Cell; Glioma; HIF; alpha; Hypoxia; Hypoxia Inducible Factor

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APA (6th Edition):

Li, Z. (2009). Differential Angiogenic Capability and Hypoxia Responses in Glioma Stem Cells. (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/1149

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Zhizhong. “Differential Angiogenic Capability and Hypoxia Responses in Glioma Stem Cells.” 2009. Thesis, Duke University. Accessed September 22, 2017. http://hdl.handle.net/10161/1149.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Zhizhong. “Differential Angiogenic Capability and Hypoxia Responses in Glioma Stem Cells.” 2009. Web. 22 Sep 2017.

Vancouver:

Li Z. Differential Angiogenic Capability and Hypoxia Responses in Glioma Stem Cells. [Internet] [Thesis]. Duke University; 2009. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/10161/1149.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li Z. Differential Angiogenic Capability and Hypoxia Responses in Glioma Stem Cells. [Thesis]. Duke University; 2009. Available from: http://hdl.handle.net/10161/1149

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lund

28. Rehn, Matilda. THE HYPOXIC HEMATOPOIETIC STEM CELL NICHE Consequences of Hypoxia-induced Transcription on Stem Cell Fate.

Degree: 2011, University of Lund

 Hematopoiesis is the process of blood formation that originates with the hematopoietic stem cell (HSC), a cell type that is responsible for the life-long supply… (more)

Subjects/Keywords: Hematologi; Hematopoietic Stem Cells; Stem Cell Niche; Hypoxia; Hypoxia-Inducible Factor; Vegfa.

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APA (6th Edition):

Rehn, M. (2011). THE HYPOXIC HEMATOPOIETIC STEM CELL NICHE Consequences of Hypoxia-induced Transcription on Stem Cell Fate. (Doctoral Dissertation). University of Lund. Retrieved from http://lup.lub.lu.se/record/2063863 ; http://portal.research.lu.se/ws/files/3578010/2063903.pdf

Chicago Manual of Style (16th Edition):

Rehn, Matilda. “THE HYPOXIC HEMATOPOIETIC STEM CELL NICHE Consequences of Hypoxia-induced Transcription on Stem Cell Fate.” 2011. Doctoral Dissertation, University of Lund. Accessed September 22, 2017. http://lup.lub.lu.se/record/2063863 ; http://portal.research.lu.se/ws/files/3578010/2063903.pdf.

MLA Handbook (7th Edition):

Rehn, Matilda. “THE HYPOXIC HEMATOPOIETIC STEM CELL NICHE Consequences of Hypoxia-induced Transcription on Stem Cell Fate.” 2011. Web. 22 Sep 2017.

Vancouver:

Rehn M. THE HYPOXIC HEMATOPOIETIC STEM CELL NICHE Consequences of Hypoxia-induced Transcription on Stem Cell Fate. [Internet] [Doctoral dissertation]. University of Lund; 2011. [cited 2017 Sep 22]. Available from: http://lup.lub.lu.se/record/2063863 ; http://portal.research.lu.se/ws/files/3578010/2063903.pdf.

Council of Science Editors:

Rehn M. THE HYPOXIC HEMATOPOIETIC STEM CELL NICHE Consequences of Hypoxia-induced Transcription on Stem Cell Fate. [Doctoral Dissertation]. University of Lund; 2011. Available from: http://lup.lub.lu.se/record/2063863 ; http://portal.research.lu.se/ws/files/3578010/2063903.pdf


Kyoto University / 京都大学

29. Sowa, Terumasa. Hypoxia-inducible factor 1 promotes chemoresistance of lung cancer by inducing carbonic anhydrase IX expression : 低酸素誘導性因子は、炭酸脱水素酵素IXの誘導により、肺がんの抗がん剤耐性を惹起する.

Degree: 博士(医学), 2017, Kyoto University / 京都大学

新制・課程博士

甲第20614号

医博第4263号

Subjects/Keywords: carbonic anhydrase IX; chemoresistance; Hypoxia-inducible factor 1; induction chemoradiotherapy; lung cancer

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APA (6th Edition):

Sowa, T. (2017). Hypoxia-inducible factor 1 promotes chemoresistance of lung cancer by inducing carbonic anhydrase IX expression : 低酸素誘導性因子は、炭酸脱水素酵素IXの誘導により、肺がんの抗がん剤耐性を惹起する. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/226768 ; http://dx.doi.org/10.14989/doctor.k20614

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sowa, Terumasa. “Hypoxia-inducible factor 1 promotes chemoresistance of lung cancer by inducing carbonic anhydrase IX expression : 低酸素誘導性因子は、炭酸脱水素酵素IXの誘導により、肺がんの抗がん剤耐性を惹起する.” 2017. Thesis, Kyoto University / 京都大学. Accessed September 22, 2017. http://hdl.handle.net/2433/226768 ; http://dx.doi.org/10.14989/doctor.k20614.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sowa, Terumasa. “Hypoxia-inducible factor 1 promotes chemoresistance of lung cancer by inducing carbonic anhydrase IX expression : 低酸素誘導性因子は、炭酸脱水素酵素IXの誘導により、肺がんの抗がん剤耐性を惹起する.” 2017. Web. 22 Sep 2017.

Vancouver:

Sowa T. Hypoxia-inducible factor 1 promotes chemoresistance of lung cancer by inducing carbonic anhydrase IX expression : 低酸素誘導性因子は、炭酸脱水素酵素IXの誘導により、肺がんの抗がん剤耐性を惹起する. [Internet] [Thesis]. Kyoto University / 京都大学; 2017. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/2433/226768 ; http://dx.doi.org/10.14989/doctor.k20614.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sowa T. Hypoxia-inducible factor 1 promotes chemoresistance of lung cancer by inducing carbonic anhydrase IX expression : 低酸素誘導性因子は、炭酸脱水素酵素IXの誘導により、肺がんの抗がん剤耐性を惹起する. [Thesis]. Kyoto University / 京都大学; 2017. Available from: http://hdl.handle.net/2433/226768 ; http://dx.doi.org/10.14989/doctor.k20614

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

30. Sowa, Terumasa. Hypoxia-inducible factor 1 promotes chemoresistance of lung cancer by inducing carbonic anhydrase IX expression .

Degree: 2017, Kyoto University

Subjects/Keywords: carbonic anhydrase IX; chemoresistance; Hypoxia-inducible factor 1; induction chemoradiotherapy; lung cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sowa, T. (2017). Hypoxia-inducible factor 1 promotes chemoresistance of lung cancer by inducing carbonic anhydrase IX expression . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/226768

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sowa, Terumasa. “Hypoxia-inducible factor 1 promotes chemoresistance of lung cancer by inducing carbonic anhydrase IX expression .” 2017. Thesis, Kyoto University. Accessed September 22, 2017. http://hdl.handle.net/2433/226768.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sowa, Terumasa. “Hypoxia-inducible factor 1 promotes chemoresistance of lung cancer by inducing carbonic anhydrase IX expression .” 2017. Web. 22 Sep 2017.

Vancouver:

Sowa T. Hypoxia-inducible factor 1 promotes chemoresistance of lung cancer by inducing carbonic anhydrase IX expression . [Internet] [Thesis]. Kyoto University; 2017. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/2433/226768.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sowa T. Hypoxia-inducible factor 1 promotes chemoresistance of lung cancer by inducing carbonic anhydrase IX expression . [Thesis]. Kyoto University; 2017. Available from: http://hdl.handle.net/2433/226768

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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