subject:(Hypertriglyceridemia)

University of Hong Kong

1. Lynn, Edward G. The role of lipoproteins in the development of glomerulosclerosis: y Edward G. Lynn.

Degree: 2000, University of Hong Kong

URL: http://hdl.handle.net/10722/32031

Subjects/Keywords: Lipoproteins.; Hypertriglyceridemia.

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APA (6^th Edition):

Lynn, E. G. (2000). The role of lipoproteins in the development of glomerulosclerosis: y Edward G. Lynn. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/32031

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Lynn, Edward G. “The role of lipoproteins in the development of glomerulosclerosis: y Edward G. Lynn.” 2000. Thesis, University of Hong Kong. Accessed January 18, 2021. http://hdl.handle.net/10722/32031.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Lynn, Edward G. “The role of lipoproteins in the development of glomerulosclerosis: y Edward G. Lynn.” 2000. Web. 18 Jan 2021.

Vancouver:

Lynn EG. The role of lipoproteins in the development of glomerulosclerosis: y Edward G. Lynn. [Internet] [Thesis]. University of Hong Kong; 2000. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10722/32031.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lynn EG. The role of lipoproteins in the development of glomerulosclerosis: y Edward G. Lynn. [Thesis]. University of Hong Kong; 2000. Available from: http://hdl.handle.net/10722/32031

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of Maryland

2. Campbell, Eric. Fructose intake and circulating triglycerides: An examination of the role of APOC3.

Degree: Nutrition, 2014, University of Maryland

URL: http://hdl.handle.net/1903/15927

► Fructose consumption can lead to marked increases in plasma triglycerides in both humans and laboratory animals. We have observed that overnight access to a 16%… (more)

Subjects/Keywords: Nutrition; APOC3; Fructose; hypertriglyceridemia

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APA (6^th Edition):

Campbell, E. (2014). Fructose intake and circulating triglycerides: An examination of the role of APOC3. (Thesis). University of Maryland. Retrieved from http://hdl.handle.net/1903/15927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Campbell, Eric. “Fructose intake and circulating triglycerides: An examination of the role of APOC3.” 2014. Thesis, University of Maryland. Accessed January 18, 2021. http://hdl.handle.net/1903/15927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Campbell, Eric. “Fructose intake and circulating triglycerides: An examination of the role of APOC3.” 2014. Web. 18 Jan 2021.

Vancouver:

Campbell E. Fructose intake and circulating triglycerides: An examination of the role of APOC3. [Internet] [Thesis]. University of Maryland; 2014. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/1903/15927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Campbell E. Fructose intake and circulating triglycerides: An examination of the role of APOC3. [Thesis]. University of Maryland; 2014. Available from: http://hdl.handle.net/1903/15927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Universidade Estadual de Campinas

3. Golucci, Ana Paula Brigatto Simões, 1984-. Perfil lipídico e hepático de pacientes pediátricos e a terapia de nutrição parenteral total: Lipid and hepatic profile of pediatric patients and total parenteral nutrition therapy.

Degree: 2017, Universidade Estadual de Campinas

URL: http://repositorio.unicamp.br/jspui/handle/REPOSIP/325660

► Abstract: Total parenteral nutrition (TPN) consists of the intravenous administration of the necessary ingredients for the survival and maintenance of the patient's homeostasis. Over the… (more)

▼ Abstract: Total parenteral nutrition (TPN) consists of the intravenous administration of the necessary ingredients for the survival and maintenance of the patient's homeostasis. Over the years this technique has been increased and contributes to the treatment of several types of patients including the seriously ill. However, TPN may be related to alterations in the lipid and hepatic profile. The most common hepatic and biliary complications in PN are: cholestasis and hepatic steatosis. Changes in the lipid profile are more related to hypertriglyceridemia and low HDL values, mainly in patients in inflammatory response. The aim of this study was to evaluate the changes in the lipid and hepatic profile of pediatric patients submitted to TPN therapy hospitalized in the Clinical Hospital of the State University of Campinas - Unicamp. It is a descriptive and historical cohort study, in which 195 pediatric patients (January 2012 to February 2016, from 0 up to 19 years old) who received TPN were evaluated. The hepatic profile was assessed from alanine aminotransferase (ALT), aspartate aminotransferase (AST), alkaline phosphatase, gamma glutamyl tranpeptidase (GGT), total bilirubin. The albumin and transthyretin were also evaluated. The lipid profile was evaluated from values of cholesterol, HDL and triglycerides (TG). Laboratory and clinical nutritional status, evaluation and outcome were also measured. According to the hepatic profile, the majority of patients presented normal values of AST (79.2%), ALT (74.9%), GGT (56.4%) and alkaline phosphatase (68.1%). Total bilirubin was altered in 68.5% and transthyretin (87.3%) and albumin (65.1%) were low in most patients. The data regarding the lipid profile showed normal cholesterol in 84.6%, low HDL in 94.5%, high TG in 81.5% and high C-reactive protein in 92.7% of patients. With multivariate logistic regression it was possible to identify that the chances of patients under NPT with high GGT values are 2.46 times higher if the patient is older than two years of age, 2.26 times higher if not a surgical patient, 3 times higher if you have a high cholesterol and 2.39 times higher if you have high triglycerides. The chance of elevated ALT is 4.57 times higher in patients who present high cholesterol. It was concluded that the prevalence of elevated ALT, AST, GGT and alkaline phosphatase were within the percentages described in the literature. From the association of alterations of the hepatic and lipid profile it was possible to verify that patients with hypercholesterolemia are more likely to make alterations of ALT, and GGT. Patients with hypertriglyceridemia are more likely to have GGT changes. By knowing better the clinical and laboratory alterations associated with PN metabolic disorders, it is possible to optimize the PN prescription and ducts and to minimize complications and mortality Advisors/Committee Members: UNIVERSIDADE ESTADUAL DE CAMPINAS (CRUESP), Nogueira, Roberto José Negrão (advisor), Ribeiro, Antonio Fernando, 1948- (coadvisor), Universidade Estadual de Campinas. Faculdade de Ciências Médicas (institution), Programa de Pós-Graduação em Saúde da Criança e do Adolescente (nameofprogram), Brandão, Maria Angela Bellomo (committee member), Toro, Adyléia Aparecida Dalbo Contrera (committee member), Patin, Rose Vega (committee member), Guandalini, Valdete Regina (committee member).

Subjects/Keywords: Nutrição parenteral; Hipercolesterolemia; Hipertrigliceridemia; Parenteral nutrition; Hypercholesterolemia; Hypertriglyceridemia

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APA (6^th Edition):

Golucci, Ana Paula Brigatto Simões, 1. (2017). Perfil lipídico e hepático de pacientes pediátricos e a terapia de nutrição parenteral total: Lipid and hepatic profile of pediatric patients and total parenteral nutrition therapy. (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/325660

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Golucci, Ana Paula Brigatto Simões, 1984-. “Perfil lipídico e hepático de pacientes pediátricos e a terapia de nutrição parenteral total: Lipid and hepatic profile of pediatric patients and total parenteral nutrition therapy.” 2017. Thesis, Universidade Estadual de Campinas. Accessed January 18, 2021. http://repositorio.unicamp.br/jspui/handle/REPOSIP/325660.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Golucci, Ana Paula Brigatto Simões, 1984-. “Perfil lipídico e hepático de pacientes pediátricos e a terapia de nutrição parenteral total: Lipid and hepatic profile of pediatric patients and total parenteral nutrition therapy.” 2017. Web. 18 Jan 2021.

Vancouver:

Golucci, Ana Paula Brigatto Simões 1. Perfil lipídico e hepático de pacientes pediátricos e a terapia de nutrição parenteral total: Lipid and hepatic profile of pediatric patients and total parenteral nutrition therapy. [Internet] [Thesis]. Universidade Estadual de Campinas; 2017. [cited 2021 Jan 18]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/325660.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Golucci, Ana Paula Brigatto Simões 1. Perfil lipídico e hepático de pacientes pediátricos e a terapia de nutrição parenteral total: Lipid and hepatic profile of pediatric patients and total parenteral nutrition therapy. [Thesis]. Universidade Estadual de Campinas; 2017. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/325660

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Sato, Daisuke; MORINO, Katsutaro; Nakagawa, Fumiyuki; Murata, Koichiro; SEKINE, Osamu; Beppu, Fumiaki; Gotoh, Naohiro; UGI, Satoshi. Acute Effect of Metformin on Postprandial Hypertriglyceridemia through Delayed Gastric Emptying.

Degree: 博士（医学）, 2018, Shiga University of Medical Science / 滋賀医科大学

URL: http://hdl.handle.net/10422/00012406

Subjects/Keywords: Metformin; postprandial hypertriglyceridemia; gastric emptying

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APA (6^th Edition):

Sato, Daisuke; MORINO, Katsutaro; Nakagawa, Fumiyuki; Murata, Koichiro; SEKINE, Osamu; Beppu, Fumiaki; Gotoh, Naohiro; UGI, S. (2018). Acute Effect of Metformin on Postprandial Hypertriglyceridemia through Delayed Gastric Emptying. (Thesis). Shiga University of Medical Science / 滋賀医科大学. Retrieved from http://hdl.handle.net/10422/00012406

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Sato, Daisuke; MORINO, Katsutaro; Nakagawa, Fumiyuki; Murata, Koichiro; SEKINE, Osamu; Beppu, Fumiaki; Gotoh, Naohiro; UGI, Satoshi. “Acute Effect of Metformin on Postprandial Hypertriglyceridemia through Delayed Gastric Emptying.” 2018. Thesis, Shiga University of Medical Science / 滋賀医科大学. Accessed January 18, 2021. http://hdl.handle.net/10422/00012406.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Sato, Daisuke; MORINO, Katsutaro; Nakagawa, Fumiyuki; Murata, Koichiro; SEKINE, Osamu; Beppu, Fumiaki; Gotoh, Naohiro; UGI, Satoshi. “Acute Effect of Metformin on Postprandial Hypertriglyceridemia through Delayed Gastric Emptying.” 2018. Web. 18 Jan 2021.

Vancouver:

Sato, Daisuke; MORINO, Katsutaro; Nakagawa, Fumiyuki; Murata, Koichiro; SEKINE, Osamu; Beppu, Fumiaki; Gotoh, Naohiro; UGI S. Acute Effect of Metformin on Postprandial Hypertriglyceridemia through Delayed Gastric Emptying. [Internet] [Thesis]. Shiga University of Medical Science / 滋賀医科大学; 2018. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10422/00012406.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sato, Daisuke; MORINO, Katsutaro; Nakagawa, Fumiyuki; Murata, Koichiro; SEKINE, Osamu; Beppu, Fumiaki; Gotoh, Naohiro; UGI S. Acute Effect of Metformin on Postprandial Hypertriglyceridemia through Delayed Gastric Emptying. [Thesis]. Shiga University of Medical Science / 滋賀医科大学; 2018. Available from: http://hdl.handle.net/10422/00012406

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of Texas – Austin

5. -6219-9383. The effects of fluid milk in attenuating postprandial hyperglycemia and hypertriglyceridemia.

Degree: PhD, Kinesiology, 2016, University of Texas – Austin

URL: http://hdl.handle.net/2152/45581

► The increased risk of cardiovascular disease (CVD) and vascular dysfunction that is associated with postprandial hyperglycemia and hypertriglyceridemia may be reduced with consumption of non-fat… (more)

▼ The increased risk of cardiovascular disease (CVD) and vascular dysfunction that is associated with postprandial hyperglycemia and hypertriglyceridemia may be reduced with consumption of non-fat dairy products. Due to the insulinotropic effect of milk-derived proteins, postprandial hyperglycemia has been shown to be reduced with the addition of dairy products. The purpose of Study 1 was to determine if one serving of non-fat milk added to an oral glucose tolerance test (OGTT) could attenuate postprandial hyperglycemia in individuals with elevated android adiposity independent of the effects of the milk’s protein content and whether these improvements would be associated with metabolic and/or peripheral hemodynamic effects. In this placebo controlled, randomized, crossover experimental study, twenty-nine overweight/obese adults consumed an OGTT combined with either non-fat milk or a placebo control. In the whole sample, blood glucose and insulin concentrations increased over time in both trials with no differences between trials. When the subjects were divided into tertiles of android body fat, the highest tertile displayed attenuated hyperglycemic responses as well as improvements in flow-mediated dilation during the milk intervention. Repeated exposure to elevated postprandial triglycerides, as seen with typical Western diets, contributes to atherosclerosis and vascular disease. The purpose of Study 2 was to determine if a single serving of non-fat milk added to a high fat meal could attenuate postprandial hypertriglyceridemia in individuals who consume a high fat diet. In this placebo controlled, randomized, crossover experimental study, thirty overweight/obese adults consumed a high fat meal combined with either non-fat milk, carbohydrate control, or caloric control. In the whole sample, plasma triglycerides increased over time with no differences between interventions. When participants were ranked and divided based on habitual dietary fat intake, the high fat diet group exhibited reduced triglycerides when supplemented with non-fat milk. No differences in hemodynamic measures (brachial flow-mediated dilation and femoral vascular conductance) were observed between the milk and caloric control trials for either the low fat or high fat diet groups. Taken together, the results indicate that a single serving of non-fat milk may attenuate acute hyperglycemia and hypertriglyceridemia in individuals that present with specific risk factors for CVD. Advisors/Committee Members: Tanaka, Hirofumi, Ph. D. (advisor), Coyle, Edward F (committee member), Stone, Audrey (committee member), Davis, Jamie (committee member).

Subjects/Keywords: Non-fat milk; Postprandial; Hyperglycemia; Hypertriglyceridemia, Whole-limb perfusion; Endothelial function

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APA (6^th Edition):

-6219-9383. (2016). The effects of fluid milk in attenuating postprandial hyperglycemia and hypertriglyceridemia. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/45581

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16^th Edition):

-6219-9383. “The effects of fluid milk in attenuating postprandial hyperglycemia and hypertriglyceridemia.” 2016. Doctoral Dissertation, University of Texas – Austin. Accessed January 18, 2021. http://hdl.handle.net/2152/45581.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7^th Edition):

-6219-9383. “The effects of fluid milk in attenuating postprandial hyperglycemia and hypertriglyceridemia.” 2016. Web. 18 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-6219-9383. The effects of fluid milk in attenuating postprandial hyperglycemia and hypertriglyceridemia. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2016. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/2152/45581.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-6219-9383. The effects of fluid milk in attenuating postprandial hyperglycemia and hypertriglyceridemia. [Doctoral Dissertation]. University of Texas – Austin; 2016. Available from: http://hdl.handle.net/2152/45581

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

University of California – Berkeley

6. Mauldin, Kasuen. Structure-Function Analysis of Apolipoprotein A-V: Insight into Plasma Triglyceride Homeostasis.

Degree: Molecular & Biochemical Nutrition, 2010, University of California – Berkeley

URL: http://www.escholarship.org/uc/item/1115j5db

► Coronary heart disease is the leading cause of death in the United States, and epidemiological studies have shown that an elevated plasma triglyceride level is… (more)

Subjects/Keywords: Nutrition; Biochemistry; Molecular Biology; apoA-V; apolipoprotein A-V; hypertriglyceridemia; lipoprotein; structure-function; triglyceride

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APA (6^th Edition):

Mauldin, K. (2010). Structure-Function Analysis of Apolipoprotein A-V: Insight into Plasma Triglyceride Homeostasis. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/1115j5db

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Mauldin, Kasuen. “Structure-Function Analysis of Apolipoprotein A-V: Insight into Plasma Triglyceride Homeostasis.” 2010. Thesis, University of California – Berkeley. Accessed January 18, 2021. http://www.escholarship.org/uc/item/1115j5db.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Mauldin, Kasuen. “Structure-Function Analysis of Apolipoprotein A-V: Insight into Plasma Triglyceride Homeostasis.” 2010. Web. 18 Jan 2021.

Vancouver:

Mauldin K. Structure-Function Analysis of Apolipoprotein A-V: Insight into Plasma Triglyceride Homeostasis. [Internet] [Thesis]. University of California – Berkeley; 2010. [cited 2021 Jan 18]. Available from: http://www.escholarship.org/uc/item/1115j5db.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mauldin K. Structure-Function Analysis of Apolipoprotein A-V: Insight into Plasma Triglyceride Homeostasis. [Thesis]. University of California – Berkeley; 2010. Available from: http://www.escholarship.org/uc/item/1115j5db

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of Western Ontario

7. Dron, Jacqueline S. The Genetic Determinants of Complex Lipid and Lipoprotein Phenotypes.

Degree: 2020, University of Western Ontario

URL: https://ir.lib.uwo.ca/etd/7522

► Cardiovascular disease (CVD) is the primary cause of death globally and is estimated to cause one-third of deaths in Canada. Each year, millions of Canadians… (more)

▼ Cardiovascular disease (CVD) is the primary cause of death globally and is estimated to cause one-third of deaths in Canada. Each year, millions of Canadians are affected by CVD despite ongoing efforts to reduce risk through lifestyle modifications and pharmacological therapies. With the expected rise in CVD prevalence due to the obesity epidemic, we need to better understand the genetic basis of heritable, modifiable risk factors, including levels of high-density lipoprotein (HDL) cholesterol and triglyceride, for insights into future therapeutic treatments and risk prediction. Through the use of a targeted next-generation sequencing panel designed specifically to study lipid and metabolic disorders, I have explored a spectrum of genetic variation—including rare and common variants, single-nucleotide and copy-number variants—in over 3,000 DNA samples isolated from individuals with abnormal lipid phenotypes, including: (i) hypoalphalipoproteinemia; (ii) hyperalphalipoproteinemia; and (iii) hypertriglyceridemia. From my research efforts, I demonstrated that the majority of individuals with abnormal HDL cholesterol levels did not carry many phenotypically-relevant genetic factors, but in those who did, rare variants were more prevalent in individuals with extremely low HDL cholesterol levels, while both rare variants and the accumulation of common variants were approximately equal in individuals with extremely high HDL cholesterol levels. Meanwhile, hypertriglyceridemia had a stronger genetic basis, with common variant accumulation being the most prevalent genetic determinant. Further, I uncovered that genetic determinants are more prevalent as the hypertriglyceridemia phenotype becomes more severe, and a genetic locus, CREB3L3, may have an extremely important, previously unappreciated role in hypertriglyceridemia susceptibility. By better understanding the genetic underpinnings of abnormal levels of HDL cholesterol and triglyceride, future efforts can explore the relationship between these phenotypes and their genetic determinants, and how we might leverage this information to develop better therapeutics to lower levels of these risk factors or create screening methods to identify individuals who might be at higher risk for CVD.

Subjects/Keywords: dyslipidemia; HDL cholesterol; hypertriglyceridemia; genetic variation; next-generation sequencing; polygenic risk score; Genetics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Dron, J. S. (2020). The Genetic Determinants of Complex Lipid and Lipoprotein Phenotypes. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/7522

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Dron, Jacqueline S. “The Genetic Determinants of Complex Lipid and Lipoprotein Phenotypes.” 2020. Thesis, University of Western Ontario. Accessed January 18, 2021. https://ir.lib.uwo.ca/etd/7522.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Dron, Jacqueline S. “The Genetic Determinants of Complex Lipid and Lipoprotein Phenotypes.” 2020. Web. 18 Jan 2021.

Vancouver:

Dron JS. The Genetic Determinants of Complex Lipid and Lipoprotein Phenotypes. [Internet] [Thesis]. University of Western Ontario; 2020. [cited 2021 Jan 18]. Available from: https://ir.lib.uwo.ca/etd/7522.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dron JS. The Genetic Determinants of Complex Lipid and Lipoprotein Phenotypes. [Thesis]. University of Western Ontario; 2020. Available from: https://ir.lib.uwo.ca/etd/7522

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Universidade do Rio Grande do Sul

8. Riboldi, Bárbara Pelicioli. Carga alimentar em indivíduos com diabetes.

Degree: 2014, Universidade do Rio Grande do Sul

URL: http://hdl.handle.net/10183/97872

►

Objetivo Avaliar, em indivíduos com diabetes (DM), a resposta de glicose e triglicerídeos duas horas após a ingestão de uma carga alimentar (CA) e sua… (more)

▼

Objetivo Avaliar, em indivíduos com diabetes (DM), a resposta de glicose e triglicerídeos duas horas após a ingestão de uma carga alimentar (CA) e sua associação com características clínicas e a presença de doenças crônicas. Metodologia Foram incluídos 915 indivíduos com DM, participantes da linha de base do estudo ELSA-Brasil. Coleta de sangue foi realizada em jejum e duas horas após o consumo da CA, contendo 455 kcal; 14 g de gordura saturada e 47 g de carboidratos. Regressão linear foi utilizada para investigar preditores dos níveis de excursão (diferença entre níveis pós-carga e de jejum) de glicemia e trigliceridemia (variáveis dependentes). Regressão logística foi realizada para descrever a associação da excursão de glicemia e trigliceridemia (variáveis independentes) e a presença de complicações do DM (doença coronariana, infarto, angina, acidente vascular cerebral, ou insuficiência cardíaca). Resultados A mediana da glicemia de jejum foi de 150 (123–198) mg/dL e da excursão de glicose foi 45 (16–79) mg/dL, enquanto que a trigliceridemia de jejum foi de 140 (103–199) mg/dL e a excursão de triglicerídeos foi de 26 (11–45) mg/dL. Maiores excursões de glicemia foram associadas com duração do DM (incremento de 5,7 mg/dL, IC95% 3,7–7,6, a cada 5 anos de doença), uso de insulina (58,5 mg/dL, IC95% 45,43–71,55), outro medicamento para DM (13,6 mg/dL, IC95% 2,5–24,8), idade (4,1 mg/dL, IC95% 0,43–7,69, a cada 10 anos), nível de glicose basal (5,2 mg/dL, IC95% 1,0–4,1, a cada 25 mg/dL); o índice de massa corporal associou-se a menor excursão (-6,8 mg/dL, IC95% -9,7 a -3,85, a cada 5 kg/m²). Maior excursão de trigliceridemia esteve associada com glicemia em jejum (2,5 mg/dL, IC95% 1,64–3,38, a cada 25 mg/dL), e menor excursão associada com triglicerídeos de jejum (-2,97 mg/dL, IC95% -3,24 – -2,69, a cada 25 mg/dL). Conclusão A resposta pós-prandial esteve associada ao tempo de doença, à necessidade de insulina ou outro medicamento para DM, ao índice de massa corporal, além da idade e níveis basais de glicose e triglicerídeos. As excursões de glicose e triglicerídeos estiveram associadas a presença de comorbidades potencialmente tidas como complicações do DM.

Objective We aimed to assess, in individuals with diabetes (DM), the excursions of glucose and triglyceride levels two hours after consumption of a food load (FL) and their association with clinical characteristics and the presence of chronic diseases. Design and Methods We included 915 subjects with DM, participants of in the ELSA-Brazil cohort. Blood sampling was performed at fasting and two hours after consumption of a 455 kcal, 14 g saturated fat and 47 g carbohydrate FL. Linear regression was used to investigate predictors of levels of excursion (difference between post-load and fasting levels) of glucose and triglycerides (dependent variables). Logistic regression was performed to describe the association of glucose excursion and triglycerides excursion (independent variables) and the presence of DM complications (coronary heart disease, myocardial…

Advisors/Committee Members: Luft, Vivian Cristine.

Subjects/Keywords: Diabetes mellitus tipo 2; Post-prandial period; Hyperglycemia; Período pós-prandial; Hiperglicemia; Hypertriglyceridemia; Type 2 Diabetes; Hipertrigliceridemia; Epidemiologia; Cardiovascular diseases

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Riboldi, B. P. (2014). Carga alimentar em indivíduos com diabetes. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/97872

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Riboldi, Bárbara Pelicioli. “Carga alimentar em indivíduos com diabetes.” 2014. Thesis, Universidade do Rio Grande do Sul. Accessed January 18, 2021. http://hdl.handle.net/10183/97872.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Riboldi, Bárbara Pelicioli. “Carga alimentar em indivíduos com diabetes.” 2014. Web. 18 Jan 2021.

Vancouver:

Riboldi BP. Carga alimentar em indivíduos com diabetes. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2014. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10183/97872.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Riboldi BP. Carga alimentar em indivíduos com diabetes. [Thesis]. Universidade do Rio Grande do Sul; 2014. Available from: http://hdl.handle.net/10183/97872

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Sanjay,Kadnur V. Isolation of some active compounds from zingiber officinale for diabetes mellitus and its complications;.

Degree: Pharmacology, 2015, Gujarat University

URL: http://shodhganga.inflibnet.ac.in/handle/10603/46222

Abstract Available

Data not available

Advisors/Committee Members: Goyal,Ramesh K.

Subjects/Keywords: Diabetes; Hyperinsulinemia; Hypertriglyceridemia; Mellitus; Metabolic; Pharmacological; Phytochemical; Zingiber

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

V, S. (2015). Isolation of some active compounds from zingiber officinale for diabetes mellitus and its complications;. (Thesis). Gujarat University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/46222

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

V, Sanjay,Kadnur. “Isolation of some active compounds from zingiber officinale for diabetes mellitus and its complications;.” 2015. Thesis, Gujarat University. Accessed January 18, 2021. http://shodhganga.inflibnet.ac.in/handle/10603/46222.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

V, Sanjay,Kadnur. “Isolation of some active compounds from zingiber officinale for diabetes mellitus and its complications;.” 2015. Web. 18 Jan 2021.

Vancouver:

V S. Isolation of some active compounds from zingiber officinale for diabetes mellitus and its complications;. [Internet] [Thesis]. Gujarat University; 2015. [cited 2021 Jan 18]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/46222.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

V S. Isolation of some active compounds from zingiber officinale for diabetes mellitus and its complications;. [Thesis]. Gujarat University; 2015. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/46222

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Roberta da Silva Paula. Genótipos de apolipoproteínas e ingestão de macronutrientes em idosas modulam o risco cardiovascular expresso pela lipemia sérica de jejum.

Degree: 2009, Universidade Católica de Brasilia

URL: http://www.bdtd.ucb.br/tede/tde_busca/arquivo.php?codArquivo=1054

►

Vários aspectos do metabolismo lipídico são importantes fatores de risco para a aterogênese, e conseqüentemente, para o desenvolvimento de doenças cardiovasculares. Dislipidemias estão entre os… (more)

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Vários aspectos do metabolismo lipídico são importantes fatores de risco para a aterogênese, e conseqüentemente, para o desenvolvimento de doenças cardiovasculares. Dislipidemias estão entre os principais fatores de risco, e estas podem ocorrer devido ingestão inadequada de macronutrientes e/ou predisposição genética. Estudos mostram que proteínas responsáveis pelo transporte de lipídios para órgãos-alvo, principalmente apolipoproteínas, podem apresentar polimorfismos gênicos que predisponham aos distúrbios lipêmicos. Esse estudo se destina a investigar se há associação de genótipos das apolipoproteínas E, A-V e B e da ingestão de macronutrientes com variáveis lipêmicas de idosas brasileiras. Casuística: estudo transversal, com amostra de 252 idosas (60 anos ou mais), moradoras dos arredores de Brasília e submetidas a anamnese clínica para exclusão de fatores interferentes (tabagismo, etilismo, reposição hormonal, déficit cognitivo, prática de atividade física) e a exames laboratoriais para dosagens lipêmicas e glicêmicas. Registros alimentares foram realizados em 3 dias alternados, e a avaliação genotípica, conforme metodologia padronizada. Resultados: as freqüências dos genótipos apresentaram-se em proporções compatíveis com o equilíbrio de Hardy-Weinberg. Indivíduos portadores do alelo ε2 manifestaram níveis séricos médios de triglicerídeos (182.075.7) significativamente mais elevados que portadores do alelo ε4 (136.361.9; p<0.05); portadores do alelo ε2 apresentaram níveis séricos médios de VLDL (37.015.1) significativamente mais elevados comparados aos homozigotos do alelo ε3 (28.511.8; p<0.01) e portadores de ε4 (26.411.3; p<0.005). Ademais, observou-se uma forte interação gene-dieta tendo em vista que portadores do alelo ε4 em regime de ingestão elevada de lipídeos totais (TG: 144.867.1; VLDL: 29.013.4) ou sob uma dieta com ingestão proporcionalmente inferior de gordura polinsaturada perderam o efeito protetor para dislipidemias (TG: 139.558.2; VLDL: 27.711.7). Não foi observado associação entre os genótipos das apolipoproteínas A-V e B com os níveis lipêmicos, independentemente do regime de ingestão de lípídeos. Os resultados encontrados são sugestivos de um fenótipo semelhante à isbetalipoproteinemia na casuística. Conclusão: nossos resultados demonstram que a isoforma ε2 predispõe a hipertrigliceridemia em mulheres pós-menopausadas, conferindo risco a eventos ateroscleróticos neste segmento geriátrico. Em contrapartida, o alelo ε4 confere proteção contra este fenótipo, exceto quando seus portadores são submetidos a uma elevada ingestão de lipídeos totais ou a uma dieta relativamente pobre em gorduras insaturadas.

Introduction and Objective: Several aspects of lipid metabolism are important risk factors for atherogenesis and consequently to the development of cardiovascular diseases. Dyslipidaemia are among the main risk factors, and these may occur due to inadequate intake of nutrients and/or genetic predisposition. Studies show that proteins responsible for lipid…

Advisors/Committee Members: Clarice Sampaio Alho, Cláudio Olavo de Almeida Córdoba, Lucy Gomes Vianna, Otávio de Tolêdo Nóbrega.

Subjects/Keywords: apolipoproteínas; polimorfismos; hipertrigliceridemia; ingestão lipídica; idoso; Ciências da Saúde; apolipoprotein; polymorphisms; hypertriglyceridemia; fat intake; aged; Ciências da Saúde

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Paula, R. d. S. (2009). Genótipos de apolipoproteínas e ingestão de macronutrientes em idosas modulam o risco cardiovascular expresso pela lipemia sérica de jejum. (Masters Thesis). Universidade Católica de Brasilia. Retrieved from http://www.bdtd.ucb.br/tede/tde_busca/arquivo.php?codArquivo=1054

Chicago Manual of Style (16^th Edition):

Paula, Roberta da Silva. “Genótipos de apolipoproteínas e ingestão de macronutrientes em idosas modulam o risco cardiovascular expresso pela lipemia sérica de jejum.” 2009. Masters Thesis, Universidade Católica de Brasilia. Accessed January 18, 2021. http://www.bdtd.ucb.br/tede/tde_busca/arquivo.php?codArquivo=1054.

MLA Handbook (7^th Edition):

Paula, Roberta da Silva. “Genótipos de apolipoproteínas e ingestão de macronutrientes em idosas modulam o risco cardiovascular expresso pela lipemia sérica de jejum.” 2009. Web. 18 Jan 2021.

Vancouver:

Paula RdS. Genótipos de apolipoproteínas e ingestão de macronutrientes em idosas modulam o risco cardiovascular expresso pela lipemia sérica de jejum. [Internet] [Masters thesis]. Universidade Católica de Brasilia; 2009. [cited 2021 Jan 18]. Available from: http://www.bdtd.ucb.br/tede/tde_busca/arquivo.php?codArquivo=1054.

Council of Science Editors:

Paula RdS. Genótipos de apolipoproteínas e ingestão de macronutrientes em idosas modulam o risco cardiovascular expresso pela lipemia sérica de jejum. [Masters Thesis]. Universidade Católica de Brasilia; 2009. Available from: http://www.bdtd.ucb.br/tede/tde_busca/arquivo.php?codArquivo=1054

University of Western Ontario

11. Johansen, Christopher T. Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia.

Degree: 2011, University of Western Ontario

URL: https://ir.lib.uwo.ca/etd/219

► Cardiovascular disease (CVD) is the leading cause of morbidity and mortality in Canada. Among non-traditional risk factors, plasma triglyceride (TG) concentration is re-emerging as a… (more)

Subjects/Keywords: hypertriglyceridemia; hyperlipoproteinemia; genetic variation; cardiovascular disease; genome-wide association study; resequencing; Biochemical Phenomena, Metabolism, and Nutrition; Cardiovascular Diseases; Genetic Phenomena

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Johansen, C. T. (2011). Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/219

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Johansen, Christopher T. “Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia.” 2011. Thesis, University of Western Ontario. Accessed January 18, 2021. https://ir.lib.uwo.ca/etd/219.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Johansen, Christopher T. “Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia.” 2011. Web. 18 Jan 2021.

Vancouver:

Johansen CT. Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia. [Internet] [Thesis]. University of Western Ontario; 2011. [cited 2021 Jan 18]. Available from: https://ir.lib.uwo.ca/etd/219.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Johansen CT. Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia. [Thesis]. University of Western Ontario; 2011. Available from: https://ir.lib.uwo.ca/etd/219

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Caio Sussumu de Macedo Motoyama. frutose associada à fibra goma guar altera a tolerância à glicose em ratos.

Degree: 2009, Universidade Federal de São Paulo

URL: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=426

►

Introdução: O aumento da ingestão de dieta rica em frutose tem sido associado a alterações na homeostase energética e no metabolismo de lipídeos e carboidratos,… (more)

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Introdução: O aumento da ingestão de dieta rica em frutose tem sido associado a alterações na homeostase energética e no metabolismo de lipídeos e carboidratos, tal como resistência à insulina e dislipidemia. Contudo, a ingestão de fibra solúvel como goma guar pode apresentar mecanismos benéficos sobre tolerância à glicose e perfil lipídico. Objetivo: O alvo do presente estudo foi investigar os efeitos da suplementação com uma solução contendo goma guar parcialmente hidrolisada associada com frutose, sobre a tolerância à glicose (OGTT), concentração de triacilgliceróis no fígado, e concentração sérica de triacilgliceróis e colesterol total, quantificação do receptor de insulina (IR) no fígado e músculo gastrocnêmio, leptina sérica, conteúdo de lipídeos e proteína na carcaça. O estudo foi realizado em ratos Wistar, com trinta dias de idade, randomizados e divididos em quatro grupos: Controle (C) ou tratados com frutose (F- 20%), fibra goma guar (FB-5%) administradas na água isoladamente ou em associação (F-20% + FB-5% = FF) por 30 dias. Peso corporal e ingestão alimentar foram monitorados cuidadosamente em todos os grupos experimentais. Resultados: O ganho de massa corporal total não diferiu entre os grupos. Considerando a ingestão calórica total, no grupo F foi significantemente maior, e no grupo FB menor em relação aos outros grupos. O grupo fibra apresentou um significantemente maior conteúdo de proteínas da carcaça, comparado com o grupo controle. O grupo FF apresentou menor conteúdo de proteína comparado com os grupos F e FB. A concentração de triacilgliceróis no fígado do grupo FF foi maior em relação ao grupo F. A concentração sérica de triacilgliceróis foi maior no grupo F quando comparado com os outros grupos. O OGTT revelou um prejuízo na tolerância à glicose nos grupos F, FB e FF, quando comparados ao grupo controle. A concentração de IR no fígado foi menor nos grupos F, FB e FF comparados ao controle. Não foram observadas diferenças entre os grupos em relação à concentração de IR no músculo gastrocnêmio. Não foram observadas diferenças significantes entre os grupos no conteúdo de gordura da carcaça, leptina sérica e colesterol total. Conclusão: Frutose induz importantes alterações na tolerância à glicose e metabolismo lipídico. Com respeito à fibra, parcialmente promove reversão dessas alterações. A associação de frutose mais fibra parece diminuir a concentração do receptor de insulina no fígado, com conseqüente prejuízo na tolerância à glicose.

Introduction: The increased intake of dietary fructose can be associated with alterations on energy homeostasis and lipid/carbohydrate metabolism, such as insulin resistance and dislipidemia. However, soluble fiber gum guar intake could improve benefic mechanism on glucose tolerance and lipids profile. Objective: The aim of the present study were to investigate the effects of the supplemental feeding partially hydrolyzed gum guar plus fructose solution on glucose tolerance (OGTT), triglyceride concentration in the liver, and triglyceride and total cholesterol…

Advisors/Committee Members: Claudia Maria da Penha Oller do Nascimento, Lila Missae Oyama, Carlos Alexandre Habitante, Nadia Carla Cheik, Regina Lucia Harumi Watanabe Endo.

Subjects/Keywords: NUTRICAO; 1. Frutose 2. receptor de insulina 3. fibra goma guar 4. tolerância à glicose 5. hipertriacilglicerolemia; Fructose, gum guar hydrolysate, insulin receptor, glucose tolerance, hypertriglyceridemia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Motoyama, C. S. d. M. (2009). frutose associada à fibra goma guar altera a tolerância à glicose em ratos. (Thesis). Universidade Federal de São Paulo. Retrieved from http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=426

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Motoyama, Caio Sussumu de Macedo. “frutose associada à fibra goma guar altera a tolerância à glicose em ratos.” 2009. Thesis, Universidade Federal de São Paulo. Accessed January 18, 2021. http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=426.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Motoyama, Caio Sussumu de Macedo. “frutose associada à fibra goma guar altera a tolerância à glicose em ratos.” 2009. Web. 18 Jan 2021.

Vancouver:

Motoyama CSdM. frutose associada à fibra goma guar altera a tolerância à glicose em ratos. [Internet] [Thesis]. Universidade Federal de São Paulo; 2009. [cited 2021 Jan 18]. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=426.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Motoyama CSdM. frutose associada à fibra goma guar altera a tolerância à glicose em ratos. [Thesis]. Universidade Federal de São Paulo; 2009. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=426

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Marmontel, Oriane. Dysrégulations de la production et de la clairance des lipoprotéines riches en triglycérides : Dysregulations of production and clearance of triglyceride-rich lipoproteins.

Degree: Docteur es, Biologie, médecine et santé, 2018, Lyon

URL: http://www.theses.fr/2018LYSE1220

►

L’hypertriglycéridémie (HTG) correspond à une accumulation des lipoprotéines riches en triglycérides (LRTG) dans la circulation plasmatique, conséquence d’une augmentation de leur synthèse ou plus classiquement… (more)

▼

L’hypertriglycéridémie (HTG) correspond à une accumulation des lipoprotéines riches en triglycérides (LRTG) dans la circulation plasmatique, conséquence d’une augmentation de leur synthèse ou plus classiquement décrit, d’une diminution de leur catabolisme. Dans près de 50% des cas, aucune cause génétique n’est identifiée chez les patients présentant une présentant une HTG sévère, aussi bien dans le cadre du syndrome de chylomicronémie familiale (FCS) que dans celui du syndrome de chylomicronémie multifactorielle (MCS). Pour améliorer nos connaissances et la caractérisation de ces patients, la conduction de corrélations phénotypes-génotypes précises grâce à une collaboration clinico-biologique étroite, ainsi que le développement d’outils de diagnostic moléculaire performants, demeurent un enjeu majeur. Premièrement, l’évaluation de la concentration pré-héparinique en LPL et l’activité post-héparinique 60 minutes après l’injection d’héparine chez 62 patients MCS caractérises génétiquement a permis la mise en évidence deux sous-groupes chez ces patients. Deuxièmement, le développement d’une stratégie séquençage de nouvelle génération permettant d’explorer simultanément les 9 gènes les plus prévalents dans les hypercholestérolémies, les hypocholestérolémies et les hypertriglycéridémies, a permis de détecter les variants nucléotidiques avec une sensibilité équivalente au séquençage Sanger mais aussi de détecter des grands réarrangements. L’ensemble des résultats souligne la complexité des mécanismes de régulation du métabolisme des LRTG et l’intérêt de l’étude des interactions gène-gène. Ainsi, ces travaux ont permis de mettre en évidence de nouvelles hypothèses à explorer pour la compréhension des mécanismes physiopathologiques des HTG sévères et d’améliorer les outils disponibles pour les études de corrélation génotype-phénotype

Hypertriglyceridemia (HTG) correspond to an increase of triglyceride-rich lipoproteins (TGRL) circulating concentration, as a consequence of an increase in the synthesis of or a decrease in their catabolism, most classically described. In nearly 50% of patients with severe hypertriglyceridemia (HTG), no genetic cause is identified, either in familial chylomicronemia syndrome (FCS) or in multifactorial chylomicronemia syndrome (MCS). To gain new insights and to improve patient’s characterization, it remains important to conduct accurate phenotype-genotype association studies through close collaboration with referent lipidologists, and to develop high-performance tools for molecular diagnosis. Firstly, the assessment of pre-heparin LPL concentration as well as LPL activity 60 minutes after heparin injection, enabled the identification of two subgroups within 62 genotyped MCS patients Secondly, the development of a new sequencing generation workflow exploring simultaneously the 9 most prevalent genes in dyslipidemia, allowed the detection of single nucleotide variations with sensitivity equivalent to Sanger sequencing, but also allowed the detection of copy number variations. Collective…

Advisors/Committee Members: Moulin, Philippe (thesis director).

Subjects/Keywords: Lipoprotéine lipase; Hypertriglycéridémie; Chylomicrons; Lipolyse; Séquençage de nouvelle génération; Dyslipidémie; Diagnostic moléculaire; Grand réarrangement; Lipoprotein lipase; Hypertriglyceridemia; Chylomicrons; Lipolysis; Next-generation sequencing; Dyslipidemia; Molecular diagnosis; Copy number variation; 570

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Marmontel, O. (2018). Dysrégulations de la production et de la clairance des lipoprotéines riches en triglycérides : Dysregulations of production and clearance of triglyceride-rich lipoproteins. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2018LYSE1220

Chicago Manual of Style (16^th Edition):

Marmontel, Oriane. “Dysrégulations de la production et de la clairance des lipoprotéines riches en triglycérides : Dysregulations of production and clearance of triglyceride-rich lipoproteins.” 2018. Doctoral Dissertation, Lyon. Accessed January 18, 2021. http://www.theses.fr/2018LYSE1220.

MLA Handbook (7^th Edition):

Marmontel, Oriane. “Dysrégulations de la production et de la clairance des lipoprotéines riches en triglycérides : Dysregulations of production and clearance of triglyceride-rich lipoproteins.” 2018. Web. 18 Jan 2021.

Vancouver:

Marmontel O. Dysrégulations de la production et de la clairance des lipoprotéines riches en triglycérides : Dysregulations of production and clearance of triglyceride-rich lipoproteins. [Internet] [Doctoral dissertation]. Lyon; 2018. [cited 2021 Jan 18]. Available from: http://www.theses.fr/2018LYSE1220.

Council of Science Editors:

Marmontel O. Dysrégulations de la production et de la clairance des lipoprotéines riches en triglycérides : Dysregulations of production and clearance of triglyceride-rich lipoproteins. [Doctoral Dissertation]. Lyon; 2018. Available from: http://www.theses.fr/2018LYSE1220

East Carolina University

14. Rogers, Artie Carlyle. Disruption of mitochondrial fitness in human preadipocytes by fatty acids.

Degree: PhD, Pharmacology and Toxicology, 2013, East Carolina University

URL: http://hdl.handle.net/10342/4256

► Adipose tissue lies at the core of energy metabolism, by not only releasing and taking up fatty acids (FA) according to the individuals overall surplus… (more)

▼ Adipose tissue lies at the core of energy metabolism, by not only releasing and taking up fatty acids (FA) according to the individuals overall surplus or deficit of energy, but also signaling other tissues to regulate their own metabolism. In the event that adipose tissue function is lost, insulin resistance, hypertriglyceridemia and hepatic steatosis can develop. Despite the wealth of knowledge that exists for how mature adipocytes function in both normal and diseased states, preadipocytes, which can safe guard against adipocyte dysfunction by differentiating and joining the adipocyte pool, have been overlooked. Therefore, how human preadipocytes function in the presence of a mixture of FA (oleate, palmitate, stearate and linoleate) that resembles a healthy individual's adipose tissue microenvironment was investigated. Experiments using a novel real-time flow cytometry device show that the maximal rate of mitochondrial superoxide accumulation, hydrogen peroxide (H2O2) production, and lipid peroxidation occurred within seconds of exposure to FA. Although mitochondrial ROS occurred in a transient fashion (within 2 hours), preadipocytes still experienced a loss in respiration through depletion of NAD+ and NADH that manifested into overt oxidative stress and cell death after 24 hours of FA exposure. Despite observing cell death in 35% of our population, the majority of preadipocytes were able to mount a protective response through increases in the mitochondrial antioxidant buffering capacity. Investigation into FOXO1, one of the transcription factors that govern mitochondrial antioxidant gene transcription, revealed an increase in deacetylation as soon as six hours post FA exposure. Inhibition of FOXO1 or SIRT1, which deacetylates FOXO1, exacerbated the toxic effects of FA suggesting that the SIRT1-FOXO1 axis may play a beneficial role in protecting preadipocytes from constant exposure to FA. Furthermore, SIRT1, FOXO1 and mitochondrial antioxidant gene expression from human adipose tissue was correlated to insulin sensitivity further highlighting the potential importance of mitochondrial antioxidant buffering systems in adipose tissue function. Altogether, this study suggests that the preadipocyte survivability is centered on mitochondrial resistance to FA-induced ROS through up regulation of mitochondrial antioxidant systems.Â Â Advisors/Committee Members: Robidoux, Jacques (advisor).

Subjects/Keywords: Pharmacology; Adipose tissues; Fatty acids; FOXO1; Mitochondria; Preadipocytes; SIRT1; Adipose Tissue – growth & development; Adipocytes; Forkhead Transcription Factors; Humans; Insulin Resistance; Hypertriglyceridemia; Fatty Liver; Sirtuin 1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Rogers, A. C. (2013). Disruption of mitochondrial fitness in human preadipocytes by fatty acids. (Doctoral Dissertation). East Carolina University. Retrieved from http://hdl.handle.net/10342/4256

Chicago Manual of Style (16^th Edition):

Rogers, Artie Carlyle. “Disruption of mitochondrial fitness in human preadipocytes by fatty acids.” 2013. Doctoral Dissertation, East Carolina University. Accessed January 18, 2021. http://hdl.handle.net/10342/4256.

MLA Handbook (7^th Edition):

Rogers, Artie Carlyle. “Disruption of mitochondrial fitness in human preadipocytes by fatty acids.” 2013. Web. 18 Jan 2021.

Vancouver:

Rogers AC. Disruption of mitochondrial fitness in human preadipocytes by fatty acids. [Internet] [Doctoral dissertation]. East Carolina University; 2013. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10342/4256.

Council of Science Editors:

Rogers AC. Disruption of mitochondrial fitness in human preadipocytes by fatty acids. [Doctoral Dissertation]. East Carolina University; 2013. Available from: http://hdl.handle.net/10342/4256

15. Χρήστου, Γεώργιος. Ο ρόλος της retinol binding protein 4 (RBP4) σε άτομα με αυξημένο σωματικό βάρος και υπερτριγλυκεριδαιμία.

Degree: 2014, University of Ioannina; Πανεπιστήμιο Ιωαννίνων

URL: http://hdl.handle.net/10442/hedi/39914

► Objective: The objective of this study was the investigation of the relationship between retinol binding protein 4 (RBP4) and atherogenic apolipoprotein B (ApoB)-containing lipoproteins, as… (more)

▼ Objective: The objective of this study was the investigation of the relationship between retinol binding protein 4 (RBP4) and atherogenic apolipoprotein B (ApoB)-containing lipoproteins, as a possible mechanism underlying the association between RBP4 and cardiovascular disease. Moreover, this study aimed to investigate the relationship between high density lipoprotein (HDL) and adiponectin.Design: Forty six obese or overweight subjects [body mass index (BMI) ≥ 27 Kg/m2] with hypertriglyceridemia underwent one of the following interventions for 3 months: 1) Lowcalorie low-fat diet (n= 20), 2) Low-calorie low-fat diet plus fenofibrate (n= 18), 3) Low-calorie low-fat diet plus rimonabant (n= 8). At baseline, at one month and at 3 months of treatment the following measurements were performed: anthropometric measurements, serum glucose, serum insulin, lipidemic profile, analysis of low density lipoprotein-cholesterol (LDL-C) subfractions, plasma concentration of RBP4, plasma total adiponectin levels and plasma high molecular weight (HMW) adiponectin levels.Results for RBP4: Circulating RBP4 decreased over 3 months of diet. RBP4 levels at one month of diet were positively correlated with the percentage change of Apolipoprotein B (ApoB) and small dense LDL (sdLDL) over one month of diet. The percentage change of circulating RBP4 over 3 months of diet was positively correlated with the percentage change of serum triglycerides, very low density lipoprotein-cholesterol (VLDL-C), LDLC, ApoB and sdLDL-C. In multivariate analysis, the percentage change of circulating RBP4 over 3 months of diet was the most powerful independent predictor of the percentage changes of ApoB and sdLDL-C. Fenofibrate treatment induced an increase in circulating RBP4 over the first month, because of the fenofibrate-induced reduction in renal clearance of RBP4, and a subsequent reduction of plasma RBP4 levels over the later 2 months, that was attributed to the metabolic action of fenofibrate. Rimonabant treatment reduced circulating RBP4.Results for adiponectin: Circulating total adiponectin did not change significantly during diet. In the subgroup of patients whose HDL-C decreased over the first month of diet, a decrease in circulating total adiponectin was found. In the subgroup of patients whose HDL-C increased over the later 2 months of diet, an increase in circulating total adiponectin over the 2 months was found. The percentage change of HDL-C over the first month of diet was positively correlated with the percentage change of circulating total adiponectin. The percentage change of HDL-C over 3 months of diet was positively correlated with the percentage changes of circulating total adiponectin and HMW adiponectin. The change in circulating total adiponectin over the first month of diet was positively correlated with HDL-C at one month. The change in HMW adiponectin over 3 months of diet was positively correlated with HDL-C at 3 months. The percentage change of circulating HMW adiponectin over the first month of fenofibrate treatment was positively…

Subjects/Keywords: Συνδετική πρωτεΐνη 4 της ρετινόλης; Παχυσαρκία; Υπερτριγλυκεριδαιμία; Δίαιτα; Φαινοφιμπράτη; Ριμοναμπάντη; Απολιποπρωτεΐνη Β; Υψηλής πυκνότητας λιποπρωτεΐνες; Retinol binding protein 4; Obesity; Hypertriglyceridemia; Diet; Fenofibrate; Rimonabant; Apolipoprotein B; High density lipoprotein (HDL)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Χρήστου, . �. (2014). Ο ρόλος της retinol binding protein 4 (RBP4) σε άτομα με αυξημένο σωματικό βάρος και υπερτριγλυκεριδαιμία. (Thesis). University of Ioannina; Πανεπιστήμιο Ιωαννίνων. Retrieved from http://hdl.handle.net/10442/hedi/39914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Χρήστου, Γεώργιος. “Ο ρόλος της retinol binding protein 4 (RBP4) σε άτομα με αυξημένο σωματικό βάρος και υπερτριγλυκεριδαιμία.” 2014. Thesis, University of Ioannina; Πανεπιστήμιο Ιωαννίνων. Accessed January 18, 2021. http://hdl.handle.net/10442/hedi/39914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Χρήστου, Γεώργιος. “Ο ρόλος της retinol binding protein 4 (RBP4) σε άτομα με αυξημένο σωματικό βάρος και υπερτριγλυκεριδαιμία.” 2014. Web. 18 Jan 2021.

Vancouver:

Χρήστου �. Ο ρόλος της retinol binding protein 4 (RBP4) σε άτομα με αυξημένο σωματικό βάρος και υπερτριγλυκεριδαιμία. [Internet] [Thesis]. University of Ioannina; Πανεπιστήμιο Ιωαννίνων; 2014. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10442/hedi/39914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Χρήστου �. Ο ρόλος της retinol binding protein 4 (RBP4) σε άτομα με αυξημένο σωματικό βάρος και υπερτριγλυκεριδαιμία. [Thesis]. University of Ioannina; Πανεπιστήμιο Ιωαννίνων; 2014. Available from: http://hdl.handle.net/10442/hedi/39914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of Missouri – Columbia

16. Datz, Craig. Feeding studies of dietary diacylglycerol oil in normal and lipoprotein lipase-deficient cats.

Degree: 2008, University of Missouri – Columbia

URL: http://hdl.handle.net/10355/5696

► Diacylglycerol (DAG) oil has been investigated in humans and animals as a potential therapy for hypertriglyceridemia and related disorders. DAG oil was evaluated in healthy… (more)

Subjects/Keywords: Cats – Diseases – Diet therapy; Cats – Food; Hypertriglyceridemia – Diet therapy; Diglycerides – Therapeutic use

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APA (6^th Edition):

Datz, C. (2008). Feeding studies of dietary diacylglycerol oil in normal and lipoprotein lipase-deficient cats. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/5696

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Datz, Craig. “Feeding studies of dietary diacylglycerol oil in normal and lipoprotein lipase-deficient cats.” 2008. Thesis, University of Missouri – Columbia. Accessed January 18, 2021. http://hdl.handle.net/10355/5696.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Datz, Craig. “Feeding studies of dietary diacylglycerol oil in normal and lipoprotein lipase-deficient cats.” 2008. Web. 18 Jan 2021.

Vancouver:

Datz C. Feeding studies of dietary diacylglycerol oil in normal and lipoprotein lipase-deficient cats. [Internet] [Thesis]. University of Missouri – Columbia; 2008. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10355/5696.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Datz C. Feeding studies of dietary diacylglycerol oil in normal and lipoprotein lipase-deficient cats. [Thesis]. University of Missouri – Columbia; 2008. Available from: http://hdl.handle.net/10355/5696

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Granja, Luiz Antonio Raio. Análise da reatividade vascular no diabetes mellitus do tipo 2 e doença coronariana após sobrecarga lipídica.

Degree: PhD, Endocrinologia, 2005, University of São Paulo

URL: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-09102014-105903/ ;

► Para avaliar o efeito de uma refeição-teste rica em lipídeos, assim mimetizando o estado pós-prandial durante o dia, nos parâmetros metabólicos e na reatividade vascular,… (more)

▼ Para avaliar o efeito de uma refeição-teste rica em lipídeos, assim mimetizando o estado pós-prandial durante o dia, nos parâmetros metabólicos e na reatividade vascular, foram estudados quatro grupos de pacientes do sexo masculino, assim divididos: com diabetes mellitus Tipo 2 e sem doença arterial coronariana (DM, n = 10), com doença arterial coronariana e com diabetes mellitus Tipo 2 (DM + DAC, n = 11), com doença arterial coronariana e sem diabetes mellitus (DAC, n = 11) e Controle (n = 7). Todos os pacientes, após receberem uma dieta rica em gordura (60g) e pobre em hidratos de carbono (14g), foram avaliados em termos de perfil lipídico antes (0h) e 2h, 4h, 6h e 8h após a ingestão e realizada a avaliação da reatividade vascular nos tempos 0h, 4h e 8h. A reatividade vascular foi estudada por ultra-som de alta resolução, medindo-se a resposta vasodilatadora da artéria braquial durante hiperemia reativa (vasodilatação endotélio-dependente) e após administração de nitroglicerina (endotélio-independente). HbA1c não foi diferente entre os grupos do estudo, exceto, como seria de se esperar, nos Controles, que foi normal (média ± DP) (DAC: 6,3 ± 0,6% vs DM + DAC: 7,6 ± 1,6% vs DM: 6,8 ± 1,7% vs Controle: 5,43 ± 0,45%). Por outro lado, a glicemia e insulina de jejum foram significativamente menores dos DAC e grupo Controle, sendo similar nos dois grupos. Dos outros parâmetros metabólicos, apenas o ácido úrico foi significativamente maior no grupo DAC (p = 0,025) em comparação aos outros. Vasodilatação da artéria braquial pós-isquemia antes do teste de sobrecarga foi semelhante entre os grupos (DAC: 7,44 ± 4,30% vs 7,01 ± 4,53% no DM vs 10,34 ± 5,10% no DM+DAC vs 9,91 ± 2,97% no grupo Controle. A vasodilatação após administração de nitrato (realizada no dia anterior ao teste de sobrecarga lipídica) também se manteve dentro da normalidade nos quatro grupos, sem diferença entre eles (DAC: 19,85 ± 8,66% vs 15,68 ± 11,43% no DM vs 21,24 ± 11,82% no DM+DAC e 20,05 ± 3,73% no grupo Controle). HOMAIR (Homeostasis Model for Assessment of insulin resistance) aumentou progressivamente nos grupos DAC, DM e DM+DAC, respectivamente, sendo significativamente menor no grupo Controle. Após sobrecarga, os níveis de triglicerídeos aumentaram significativamente nos quatro grupos, com pico nos tempos 4h e 6h (p < 0,001 nos quatro grupos para 2h, 4h, 6h e 8h vs 0h). O colesterol total apresentou aumento, com p < 0,001 para 4h e 6h e p = 0,0019 para 8h, em comparação com 0h, nos quatro grupos. O LDL-colesterol (p = 0,001) e o HDL-colesterol (p < 0,001) apresentaram decréscimo em todos os tempos após o teste em relação ao basal. Não houve diferença significativa entre os quatro grupos no perfil lipídico após sobrecarga de gordura. A glicemia foi significativamente mais baixa nos grupos DAC e Controle do que nos grupos DM e DM+DAC. Nos grupos DAC e Controle, a glicemia manteve-se inalterada durante o teste. Os grupos DM e DM+DAC apresentaram um decréscimo significativo nos níveis glicêmicos nos tempos 4h, 6h e 8h. (p<0,001 para todos os grupos… Advisors/Committee Members: Wajchenberg, Bernardo Leo.

Subjects/Keywords: Diabetes Mellitus; Diabetes mellitus; Doenças cardiovasculares; Endotélio; Endothelium; Heart vascular disease; Hipertrigliceridemia; Hypertriglyceridemia; Lipídeos; Lipides; Reatividade vascular; Vascular reactivity

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Granja, L. A. R. (2005). Análise da reatividade vascular no diabetes mellitus do tipo 2 e doença coronariana após sobrecarga lipídica. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5135/tde-09102014-105903/ ;

Chicago Manual of Style (16^th Edition):

Granja, Luiz Antonio Raio. “Análise da reatividade vascular no diabetes mellitus do tipo 2 e doença coronariana após sobrecarga lipídica.” 2005. Doctoral Dissertation, University of São Paulo. Accessed January 18, 2021. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-09102014-105903/ ;.

MLA Handbook (7^th Edition):

Granja, Luiz Antonio Raio. “Análise da reatividade vascular no diabetes mellitus do tipo 2 e doença coronariana após sobrecarga lipídica.” 2005. Web. 18 Jan 2021.

Vancouver:

Granja LAR. Análise da reatividade vascular no diabetes mellitus do tipo 2 e doença coronariana após sobrecarga lipídica. [Internet] [Doctoral dissertation]. University of São Paulo; 2005. [cited 2021 Jan 18]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-09102014-105903/ ;.

Council of Science Editors:

Granja LAR. Análise da reatividade vascular no diabetes mellitus do tipo 2 e doença coronariana após sobrecarga lipídica. [Doctoral Dissertation]. University of São Paulo; 2005. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-09102014-105903/ ;

Université de Montréal

18. Guay, Simon-Pierre. Association de polymorphismes dans le gène GPIHBP1 avec l’hypertriglycéridémie.

Degree: 2011, Université de Montréal

URL: http://hdl.handle.net/1866/5003

Subjects/Keywords: GPIHBP1; Polymorphisme; Hypertriglycéridémie; Métabolisme lipidique-lipoprotéique; Lipoprotéine lipase; Polymorphism; Hypertriglyceridemia; Lipoprotein lipase; Lipid-lipoprotein metabolism; Biology - Genetics / Biologie - Génétique (UMI : 0369)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Guay, S. (2011). Association de polymorphismes dans le gène GPIHBP1 avec l’hypertriglycéridémie. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/5003

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Guay, Simon-Pierre. “Association de polymorphismes dans le gène GPIHBP1 avec l’hypertriglycéridémie.” 2011. Thesis, Université de Montréal. Accessed January 18, 2021. http://hdl.handle.net/1866/5003.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Guay, Simon-Pierre. “Association de polymorphismes dans le gène GPIHBP1 avec l’hypertriglycéridémie.” 2011. Web. 18 Jan 2021.

Vancouver:

Guay S. Association de polymorphismes dans le gène GPIHBP1 avec l’hypertriglycéridémie. [Internet] [Thesis]. Université de Montréal; 2011. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/1866/5003.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guay S. Association de polymorphismes dans le gène GPIHBP1 avec l’hypertriglycéridémie. [Thesis]. Université de Montréal; 2011. Available from: http://hdl.handle.net/1866/5003

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Xenoulis, Panagiotis. Investigations into Hyperlipidemia and its Possible Associations with Pancreatitis in Dogs.

Degree: PhD, Biomedical Sciences, 2012, Texas A&M University

URL: http://hdl.handle.net/1969.1/ETD-TAMU-2011-05-9169

► The relationship between hyperlipidemia and pancreatitis remains obscure in dogs. The aim of the present study was to investigate any possible association between hyperlipidemia and… (more)

▼ The relationship between hyperlipidemia and pancreatitis remains obscure in dogs. The aim of the present study was to investigate any possible association between hyperlipidemia and pancreatitis in dogs. In the first part of the study, Miniature Schnauzers with hypertriglyceridemia were found to have significantly higher serum cPLI concentrations than Miniature Schnauzers with normal serum triglyceride concentrations (P=0.0001). Also, Miniature Schnauzers with severe hypertriglyceridemia (>862 mg/dL) had 4.5 times higher odds (P=0.0343) for having a serum cPLI concentration consistent with pancreatitis. In the second part of the study, 17 Miniature Schnauzers prospectively enrolled with a history of pancreatitis were significantly more likely to have hypertriglyceridemia (71 percent) after resolution of pancreatitis than 34 age-matched Miniature Schnauzers without a history of pancreatitis (33 percent; odds ratio=5.02; P=0.0163). For the third part of the study, assessment of the feasibility and usefulness of a novel density gradient ultracentrifugation method using NaBiEDTA for lipoprotein profiling in dogs was attempted. Density gradient ultracentrifugation using NaBiEDTA was found to be useful for the study of lipoprotein profiles in dogs. Significant differences were detected in the lipoprotein profiles (mainly involving TRL and specific LDL fractions) among healthy Miniature Schnauzers, dogs of various other breeds, and hypertriglyceridemic Miniature Schnauzers. In the fourth part of the study, the effect of a commercially available low-fat diet on serum lipid and pancreas-specific lipase (Spec cPL) concentrations and lipoprotein profiles in Miniature Schnauzers with primary hypertriglyceridemia was evaluated. The study diet was found to be effective in significantly reducing serum triglyceride and cholesterol concentrations and changing the lipoprotein profiles of the dogs studied within 2 months. However, there was no significant effect of the study diet on serum Spec cPL concentrations. In the last part of the study, serum triglyceride and cholesterol concentrations and lipoprotein profiles were compared between dogs with naturally occurring pancreatitis and healthy dogs. The majority of dogs with naturally occurring pancreatitis had normal serum triglyceride and cholesterol concentrations. Important differences were identified in lipoprotein profiles between dogs with pancreatitis (higher LDL2, LDL3, and LDL4 fractions and lower TRL, HDL2a, and HDL3c fractions) and healthy control dogs. Advisors/Committee Members: Steiner, Jörg M. (advisor), Cohen, Noah D. (committee member), Ruaux, Craig G. (committee member), Suchodolski, Jan S. (committee member), Walzem, Rosemary L. (committee member).

Subjects/Keywords: Dog; Canine; hyperlipidemia; hypertriglyceridemia; pancreatitis; low-fat diet; lipoprotein; lipoprotein profile; ultracentrifugation; Miniature Schnauzer

…Miniature Schnauzers with hypertriglyceridemia (Group 2B)… …2A), and Miniature Schnauzers with hypertriglyceridemia (Group 2B)… …hypertriglyceridemia (Group 2B) ...................................................... 105 15 1… …106 16 Serum triglyceride concentrations in dogs with hypertriglyceridemia before and… …17 Serum cholesterol concentrations in dogs with hypertriglyceridemia before and after…

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Xenoulis, P. (2012). Investigations into Hyperlipidemia and its Possible Associations with Pancreatitis in Dogs. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2011-05-9169

Chicago Manual of Style (16^th Edition):

Xenoulis, Panagiotis. “Investigations into Hyperlipidemia and its Possible Associations with Pancreatitis in Dogs.” 2012. Doctoral Dissertation, Texas A&M University. Accessed January 18, 2021. http://hdl.handle.net/1969.1/ETD-TAMU-2011-05-9169.

MLA Handbook (7^th Edition):

Xenoulis, Panagiotis. “Investigations into Hyperlipidemia and its Possible Associations with Pancreatitis in Dogs.” 2012. Web. 18 Jan 2021.

Vancouver:

Xenoulis P. Investigations into Hyperlipidemia and its Possible Associations with Pancreatitis in Dogs. [Internet] [Doctoral dissertation]. Texas A&M University; 2012. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-05-9169.

Council of Science Editors:

Xenoulis P. Investigations into Hyperlipidemia and its Possible Associations with Pancreatitis in Dogs. [Doctoral Dissertation]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-05-9169

20. Sarkar, Samantha Khadija. The Regulation of PCSK9 Structure and Function Through Lipoprotein Interactions .

Degree: 2019, University of Ottawa

URL: http://hdl.handle.net/10393/39099

► Proprotein convertase subtilisin / kexin type 9 (PCSK9) is a negative regulator of the low-density lipoprotein receptor, and PCSK9 inhibition has become an important cholesterol-lowering… (more)

Subjects/Keywords: PCSK9; LDL; cholesterol; VLDL; IDL; lipoprotein; proprotein convertase; biochemistry; mutagenesis; hypercholesterolemia; hypertriglyceridemia; atherosclerosis; cardiovascular disease

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APA (6^th Edition):

Sarkar, S. K. (2019). The Regulation of PCSK9 Structure and Function Through Lipoprotein Interactions . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/39099

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Sarkar, Samantha Khadija. “The Regulation of PCSK9 Structure and Function Through Lipoprotein Interactions .” 2019. Thesis, University of Ottawa. Accessed January 18, 2021. http://hdl.handle.net/10393/39099.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Sarkar, Samantha Khadija. “The Regulation of PCSK9 Structure and Function Through Lipoprotein Interactions .” 2019. Web. 18 Jan 2021.

Vancouver:

Sarkar SK. The Regulation of PCSK9 Structure and Function Through Lipoprotein Interactions . [Internet] [Thesis]. University of Ottawa; 2019. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10393/39099.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sarkar SK. The Regulation of PCSK9 Structure and Function Through Lipoprotein Interactions . [Thesis]. University of Ottawa; 2019. Available from: http://hdl.handle.net/10393/39099

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Park, Sanghee. Effects of prolonged sitting and walking for two days on postprandial triglycerides in men : interaction with energy intake.

Degree: MSin Kinesiology, Kinesiology, 2011, University of Texas – Austin

URL: http://hdl.handle.net/2152/ETD-UT-2011-05-2708

► Postprandial hypertriglyceridemia (PPHT), an independent risk factor for atherosclerosis (Smyth and Heron 2006; Nordestgaard, Benn et al. 2007), is strongly associated with metabolic syndrome and… (more)

▼ Postprandial hypertriglyceridemia (PPHT), an independent risk factor for atherosclerosis (Smyth and Heron 2006; Nordestgaard, Benn et al. 2007), is strongly associated with metabolic syndrome and cardiovascular diseases (CVD) (Kolovou, Anagnostopoulou et al. 2005). It has been proposed that elevated triglycerides after a high-fat meal may be a postprandial phenomenon (Zilversmit 1979). PPHT are commonly concurrent with sedentary behaviors, such as extended sitting, which amplify PPHT (Levine, Vander Weg et al. 2006). The purpose of this study was to examine the effects of prolonged sitting with or without a balanced caloric diet and walking with a balanced diet on postprandial triglycerides (PPTG). Seven healthy, young men (age, 25.6 ± 3.7 y; height, 174 ± 5 cm; weight, 71.4 ± 6.2kg; VO2max, 49.3 ± 7.7 ml/kg/min) were recruited from a college and from within the Austin community. After 2 days of food and activity control (D1and D2), subjects performed one of three trials in a randomized, cross-over design for 2 days (D3 and D4); (1) active walking with a balanced diet (WB), (2) prolonged sitting with a hyper-caloric diet (SH), and (3) prolonged sitting with a balanced diet (SB). High fat tolerance tests (HFTT) were conducted on the following day, (D5), after 13 hour over-night fasting. Blood samples were obtained in the fasting state and every hour for 6 hours after subjects had eaten a high fat test meal consisting of 1.2 g fat, 1.1 g CHO, 0.2 g protein/kg body mass. All food was provided during the 5-day duration of the study. Body postures, heart rate, and daily steps were monitored. In both sit trials (SH and SB), subjects sat ~320 minutes longer and took 10 times fewer steps than WB. In WB, the total area under the curves for plasma triglycerides (AUC[subscript T] TG) was lower, compared to SH by 21.3% (p<0.001) and to SB by 19.7% (N.S.; p = 0.055), respectively. In WB, the incremental AUC TG (AUC[subscript I] TG), an index of postprandial response, was lower than both SH by 17.4% (p <0.005) and SB by 20.1% (p <0.05), respectively. Postprandial plasma insulin concentration was lower in WB, compared to SH by 19.4% (p <0.005) in AUC[scubscript T] and 18.8 % (p < 0.05) in AUC[subscript I]. No differences were shown in the metabolic responses between SB and SH despite the diet modifications. These findings indicate that two days of prolonged sitting significantly amplifies PPTG and suppresses insulin action. Advisors/Committee Members: Coyle, E. F. (Edward F.), 1952- (advisor), Brothers, Robert M. (committee member).

Subjects/Keywords: Postprandial triglycerides; Postprandial hypertriglyceridemia; Sedentary behavior; Health behavior; Sitting; Walking; Diet; Nutrition; Metabolism; Insulin

…Postprandial hypertriglyceridemia is an abnormal elevation in circulating plasma triglycerides… …between hypertriglyceridemia and atherosclerosis has remained unclear because the development of…

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Park, S. (2011). Effects of prolonged sitting and walking for two days on postprandial triglycerides in men : interaction with energy intake. (Masters Thesis). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/ETD-UT-2011-05-2708

Chicago Manual of Style (16^th Edition):

Park, Sanghee. “Effects of prolonged sitting and walking for two days on postprandial triglycerides in men : interaction with energy intake.” 2011. Masters Thesis, University of Texas – Austin. Accessed January 18, 2021. http://hdl.handle.net/2152/ETD-UT-2011-05-2708.

MLA Handbook (7^th Edition):

Park, Sanghee. “Effects of prolonged sitting and walking for two days on postprandial triglycerides in men : interaction with energy intake.” 2011. Web. 18 Jan 2021.

Vancouver:

Park S. Effects of prolonged sitting and walking for two days on postprandial triglycerides in men : interaction with energy intake. [Internet] [Masters thesis]. University of Texas – Austin; 2011. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/2152/ETD-UT-2011-05-2708.

Council of Science Editors:

Park S. Effects of prolonged sitting and walking for two days on postprandial triglycerides in men : interaction with energy intake. [Masters Thesis]. University of Texas – Austin; 2011. Available from: http://hdl.handle.net/2152/ETD-UT-2011-05-2708

22. Boer, den, A.M. Hepatic steatosis: metabolic consequences.

Degree: 2006, GildePrint B.V.

URL: http://hdl.handle.net/1887/4984

► In this thesis we focused on the causes and consequences of hepatic steatosis. Epidemiological studies in humans, as well as experimental studies in animal models,… (more)

Subjects/Keywords: Fatty acid; Fatty liver; Glucose metabolism; Hepatic steatosis; Hyperinsulinemic euglycemic clamp; Hypertriglyceridemia; Insulin resistance; Lipid metabolism; Mouse models; Triglycerides; Fatty acid; Fatty liver; Glucose metabolism; Hepatic steatosis; Hyperinsulinemic euglycemic clamp; Hypertriglyceridemia; Insulin resistance; Lipid metabolism; Mouse models; Triglycerides

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Boer, den, A. M. (2006). Hepatic steatosis: metabolic consequences. (Doctoral Dissertation). GildePrint B.V. Retrieved from http://hdl.handle.net/1887/4984

Chicago Manual of Style (16^th Edition):

Boer, den, A M. “Hepatic steatosis: metabolic consequences.” 2006. Doctoral Dissertation, GildePrint B.V. Accessed January 18, 2021. http://hdl.handle.net/1887/4984.

MLA Handbook (7^th Edition):

Boer, den, A M. “Hepatic steatosis: metabolic consequences.” 2006. Web. 18 Jan 2021.

Vancouver:

Boer, den AM. Hepatic steatosis: metabolic consequences. [Internet] [Doctoral dissertation]. GildePrint B.V.; 2006. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/1887/4984.

Council of Science Editors:

Boer, den AM. Hepatic steatosis: metabolic consequences. [Doctoral Dissertation]. GildePrint B.V.; 2006. Available from: http://hdl.handle.net/1887/4984

23. Τσιμιχοδήμος, Βασίλειος. Ενεργότητα της paf - ακετυλοϋδρολάσης σε ασθενείς με διαταραχές του μεταβολισμού των λιποπρωτεϊνών πριν και μετά τη χορήγηση υπολιπιδαιμικών φαρμάκων.

Degree: 2004, University of Ioannina; Πανεπιστήμιο Ιωαννίνων

URL: http://hdl.handle.net/10442/hedi/21147

► PAF-AH is a calcium independent phospholipase A2 that circulates in complex with lipoprotein particles. In human plasma, more than 90% of enzymatic activity is associated… (more)

▼ PAF-AH is a calcium independent phospholipase A2 that circulates in complex with lipoprotein particles. In human plasma, more than 90% of enzymatic activity is associated with low density lipoproteins (LDL) and the remainder with high density lipoproteins (HDL). Thus, the term plasma PAF-AH mainly reflects the LDL-associated proportion of the enzyme. Except for PAF, the enzyme can also effectively hydrolyze oxidized phospholipids with short acyl chains at sn-2 position. These phospholipids are formed during the oxidative modification of LDL and possibly contribute to their proinflammatory properties. From a pathophysiological point of view, PAF-AH may play either an antiatherogenic role (since it degrades PAF and oxidized phospholipids) or a proatherogenic role (since it generates lyso-PC and oxidized fatty acids, molecules that have been reported to exert several proinflammatory properties). The clinical studies that evaluated the role of PAF-AH in the pathogenesis of atherosclerosis failed to reveal consistent results. Thus, other studies have shown that patients with cardiovascular disease exhibit significantly higher plasma enzymatic activity as compared to healthy individuals, whereas recent studies in Japanese patients concluded that the loss of plasma enzymatic activity due to G994—»T mutation is a risk factor for ischemic events. However, a recent analysis of WOSCOPS data revealed that plasma PAF-AH is an independent risk factor for the development of cardiovascular disease. On the other hand, the HDL-associated proportion of the enzyme clearly plays an antiatherogenic role, since it protects LDL from oxidation and diminishes the biological action of oxidized LDL. The aim of this study was to determine and compare the PAF-AH activities in patients with different forms of primary dyslipidemias and to evaluate the effects of hypolipidemic drugs on plasma and HDL-associated enzymatic activity. Two hundred and seventy-five unrelated patients attending the outpatient Lipid Clinic of the University Hospital of Ioannina were screened. Secondary causes of dyslipidemia were excluded by history, clinical examination and appropriate laboratory tests. All patients instructed to follow a low fat-low cholesterol diet for at least 3 months. At the end of this period, all patients with abnormal lipid levels were included in the study. According to their baseline lipid values patients were subdivided into primary hypercholesterolemia or dyslipidemia type ILA group, consisted of 141 patients, combined hyperlipidemia or dyslipidemia type IIB group, of 84 patients, and hypertriglyceridemia or dyslipidemia type IV group of 50 patients. Furthermore, patients with138primary hypercholesterolemia were divided according to the underlying genetic defect into patients with homozygous familial hypercholesterolemia (HomoFH, n=5), patients with heterozygous familial hypercholesterolemia (HeteroFH, n=38), and patients with polygenic non-familial hypercholesterolemia (NonFH, n=98). Seventy-six patients received atorvastatin (55 type IIA and…

Subjects/Keywords: PAF-ακετυλοϋδρολάση ( PAF - AH); Υπερχοληστερολαιμία, οικογενής; Δυσλιπιδαιμίες, Μικτή; Υπερτριγλυκεριδαιμία; Ατορβαστατίνη; Φενοφιβράτη; PAF - Acetylhydrolase ( PAF-AH); Hypercholesterolemia, Familial; Dyslipidemia, Combined; Hypertriglyceridemia; Atorvastatin; Fenofibrate

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Τσιμιχοδήμος, . �. (2004). Ενεργότητα της paf - ακετυλοϋδρολάσης σε ασθενείς με διαταραχές του μεταβολισμού των λιποπρωτεϊνών πριν και μετά τη χορήγηση υπολιπιδαιμικών φαρμάκων. (Thesis). University of Ioannina; Πανεπιστήμιο Ιωαννίνων. Retrieved from http://hdl.handle.net/10442/hedi/21147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Τσιμιχοδήμος, Βασίλειος. “Ενεργότητα της paf - ακετυλοϋδρολάσης σε ασθενείς με διαταραχές του μεταβολισμού των λιποπρωτεϊνών πριν και μετά τη χορήγηση υπολιπιδαιμικών φαρμάκων.” 2004. Thesis, University of Ioannina; Πανεπιστήμιο Ιωαννίνων. Accessed January 18, 2021. http://hdl.handle.net/10442/hedi/21147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Τσιμιχοδήμος, Βασίλειος. “Ενεργότητα της paf - ακετυλοϋδρολάσης σε ασθενείς με διαταραχές του μεταβολισμού των λιποπρωτεϊνών πριν και μετά τη χορήγηση υπολιπιδαιμικών φαρμάκων.” 2004. Web. 18 Jan 2021.

Vancouver:

Τσιμιχοδήμος �. Ενεργότητα της paf - ακετυλοϋδρολάσης σε ασθενείς με διαταραχές του μεταβολισμού των λιποπρωτεϊνών πριν και μετά τη χορήγηση υπολιπιδαιμικών φαρμάκων. [Internet] [Thesis]. University of Ioannina; Πανεπιστήμιο Ιωαννίνων; 2004. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10442/hedi/21147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Τσιμιχοδήμος �. Ενεργότητα της paf - ακετυλοϋδρολάσης σε ασθενείς με διαταραχές του μεταβολισμού των λιποπρωτεϊνών πριν και μετά τη χορήγηση υπολιπιδαιμικών φαρμάκων. [Thesis]. University of Ioannina; Πανεπιστήμιο Ιωαννίνων; 2004. Available from: http://hdl.handle.net/10442/hedi/21147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Brigham Young University

24. Lewis, Amanda Gloria. Treatment of Hypertriglyceridemia with Omega-3 Fatty Acids: A Systematic Review.

Degree: MS, 2004, Brigham Young University

URL: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=1160&context=etd

► Purpose: To 1) critically appraise available randomized controlled trials (RTCs) addressing the efficacy of long-chain ω-3 fatty acids as a secondary prevention agent of… (more)

Subjects/Keywords: Long-chain omega-3 fatty acids; eicosapentaenoic acid (EPA); docosahexaenoic acid (DHA); hypertriglyceridemia; hyperlipidemia; systematic review; critical appraisal; alpha linolenic acid; omega-6 fatty acids; Nursing

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Lewis, A. G. (2004). Treatment of Hypertriglyceridemia with Omega-3 Fatty Acids: A Systematic Review. (Masters Thesis). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=1160&context=etd

Chicago Manual of Style (16^th Edition):

Lewis, Amanda Gloria. “Treatment of Hypertriglyceridemia with Omega-3 Fatty Acids: A Systematic Review.” 2004. Masters Thesis, Brigham Young University. Accessed January 18, 2021. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=1160&context=etd.

MLA Handbook (7^th Edition):

Lewis, Amanda Gloria. “Treatment of Hypertriglyceridemia with Omega-3 Fatty Acids: A Systematic Review.” 2004. Web. 18 Jan 2021.

Vancouver:

Lewis AG. Treatment of Hypertriglyceridemia with Omega-3 Fatty Acids: A Systematic Review. [Internet] [Masters thesis]. Brigham Young University; 2004. [cited 2021 Jan 18]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=1160&context=etd.

Council of Science Editors:

Lewis AG. Treatment of Hypertriglyceridemia with Omega-3 Fatty Acids: A Systematic Review. [Masters Thesis]. Brigham Young University; 2004. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=1160&context=etd

25. 舩津, 敏之. HMG-CoA還元酵素阻害薬のトリグリセリド低下作用およびその機序に関する研究 : Triglyceride lowering effect of HMG-CoA reductase inhibitors and study of the mechanism of action.

Degree: Chiba University / 千葉大学

URL: http://opac.ll.chiba-u.jp/da/curator/900022748/

研究科: 千葉大学大学院薬学研究科

学位:千大院薬博乙第246号

Subjects/Keywords: HMG-CoA reductase inhibitor; Triglyceride; Beta-Oxidation; Fatty acid; postprandial hypertriglyceridemia; HMG-CoA 還元酵素阻害薬; トリグリセリド; β酸化; 遊離脂肪酸; 食後高脂血症

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

舩津, �. (n.d.). HMG-CoA還元酵素阻害薬のトリグリセリド低下作用およびその機序に関する研究 : Triglyceride lowering effect of HMG-CoA reductase inhibitors and study of the mechanism of action. (Thesis). Chiba University / 千葉大学. Retrieved from http://opac.ll.chiba-u.jp/da/curator/900022748/

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

舩津, 敏之. “HMG-CoA還元酵素阻害薬のトリグリセリド低下作用およびその機序に関する研究 : Triglyceride lowering effect of HMG-CoA reductase inhibitors and study of the mechanism of action.” Thesis, Chiba University / 千葉大学. Accessed January 18, 2021. http://opac.ll.chiba-u.jp/da/curator/900022748/.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

舩津, 敏之. “HMG-CoA還元酵素阻害薬のトリグリセリド低下作用およびその機序に関する研究 : Triglyceride lowering effect of HMG-CoA reductase inhibitors and study of the mechanism of action.” Web. 18 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

舩津 �. HMG-CoA還元酵素阻害薬のトリグリセリド低下作用およびその機序に関する研究 : Triglyceride lowering effect of HMG-CoA reductase inhibitors and study of the mechanism of action. [Internet] [Thesis]. Chiba University / 千葉大学; [cited 2021 Jan 18]. Available from: http://opac.ll.chiba-u.jp/da/curator/900022748/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

舩津 �. HMG-CoA還元酵素阻害薬のトリグリセリド低下作用およびその機序に関する研究 : Triglyceride lowering effect of HMG-CoA reductase inhibitors and study of the mechanism of action. [Thesis]. Chiba University / 千葉大学; Available from: http://opac.ll.chiba-u.jp/da/curator/900022748/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

26. Dubois-Bouchard, Camélia. Identification de biomarqueurs de risque à la pancréatite aigüe récurrente dans l’hyperchylomicronémie familiale.

Degree: 2015, Université de Montréal

URL: http://hdl.handle.net/1866/13023

Subjects/Keywords: Lipoprotéine lipase; Lipoprotein lipase; Hyperchylomicronémie familiale; Familial hyperchylomicronemia; Déficience en lipoprotéine lipase; Lipoprotein lipase deficiency; Hypertriglycéridémie sévère; Severe hypertriglyceridemia; Pancréatite; Pancreatitis; SPINK1; CTRC; Biomarqueurs génétiques; Genetic biomarkers; Biology - Genetics / Biologie - Génétique (UMI : 0369)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Dubois-Bouchard, C. (2015). Identification de biomarqueurs de risque à la pancréatite aigüe récurrente dans l’hyperchylomicronémie familiale. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/13023

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Dubois-Bouchard, Camélia. “Identification de biomarqueurs de risque à la pancréatite aigüe récurrente dans l’hyperchylomicronémie familiale.” 2015. Thesis, Université de Montréal. Accessed January 18, 2021. http://hdl.handle.net/1866/13023.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Dubois-Bouchard, Camélia. “Identification de biomarqueurs de risque à la pancréatite aigüe récurrente dans l’hyperchylomicronémie familiale.” 2015. Web. 18 Jan 2021.

Vancouver:

Dubois-Bouchard C. Identification de biomarqueurs de risque à la pancréatite aigüe récurrente dans l’hyperchylomicronémie familiale. [Internet] [Thesis]. Université de Montréal; 2015. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/1866/13023.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dubois-Bouchard C. Identification de biomarqueurs de risque à la pancréatite aigüe récurrente dans l’hyperchylomicronémie familiale. [Thesis]. Université de Montréal; 2015. Available from: http://hdl.handle.net/1866/13023

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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Open Access Theses and Dissertations