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You searched for subject:(Hepatotoxicity). Showing records 1 – 30 of 89 total matches.

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Penn State University

1. James, Karma Delores. Effect of Dietary Pretreatment and Obesity on (-)- Epigallocatechin-3-gallate (egcg) Mediated Hepatotoxicity and the Underlying Mechanism.

Degree: PhD, Food Science, 2016, Penn State University

 A growing body of evidence suggests the hepatotoxic potential of high doses of EGCG. In vitro several mechanisms of EGCG cytotoxicity have been examined; however… (more)

Subjects/Keywords: EGCG; hepatotoxicity; bioavailability; mitochondria; antioxidant

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APA (6th Edition):

James, K. D. (2016). Effect of Dietary Pretreatment and Obesity on (-)- Epigallocatechin-3-gallate (egcg) Mediated Hepatotoxicity and the Underlying Mechanism. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/28937

Chicago Manual of Style (16th Edition):

James, Karma Delores. “Effect of Dietary Pretreatment and Obesity on (-)- Epigallocatechin-3-gallate (egcg) Mediated Hepatotoxicity and the Underlying Mechanism.” 2016. Doctoral Dissertation, Penn State University. Accessed May 20, 2019. https://etda.libraries.psu.edu/catalog/28937.

MLA Handbook (7th Edition):

James, Karma Delores. “Effect of Dietary Pretreatment and Obesity on (-)- Epigallocatechin-3-gallate (egcg) Mediated Hepatotoxicity and the Underlying Mechanism.” 2016. Web. 20 May 2019.

Vancouver:

James KD. Effect of Dietary Pretreatment and Obesity on (-)- Epigallocatechin-3-gallate (egcg) Mediated Hepatotoxicity and the Underlying Mechanism. [Internet] [Doctoral dissertation]. Penn State University; 2016. [cited 2019 May 20]. Available from: https://etda.libraries.psu.edu/catalog/28937.

Council of Science Editors:

James KD. Effect of Dietary Pretreatment and Obesity on (-)- Epigallocatechin-3-gallate (egcg) Mediated Hepatotoxicity and the Underlying Mechanism. [Doctoral Dissertation]. Penn State University; 2016. Available from: https://etda.libraries.psu.edu/catalog/28937


Universitat de Valencia

2. García Cañaveras, Juan Carlos. Metabolomics as a tool for the study of drug-induced hepatotoxicity .

Degree: 2015, Universitat de Valencia

 Drug-induced liver injury (DILI) is a major health and economic problem and the leading cause of hepatic dysfunction, drug failure during clinical testing and post-market… (more)

Subjects/Keywords: metabolomics; hepatotoxicity; lc-ms

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APA (6th Edition):

García Cañaveras, J. C. (2015). Metabolomics as a tool for the study of drug-induced hepatotoxicity . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/42776

Chicago Manual of Style (16th Edition):

García Cañaveras, Juan Carlos. “Metabolomics as a tool for the study of drug-induced hepatotoxicity .” 2015. Doctoral Dissertation, Universitat de Valencia. Accessed May 20, 2019. http://hdl.handle.net/10550/42776.

MLA Handbook (7th Edition):

García Cañaveras, Juan Carlos. “Metabolomics as a tool for the study of drug-induced hepatotoxicity .” 2015. Web. 20 May 2019.

Vancouver:

García Cañaveras JC. Metabolomics as a tool for the study of drug-induced hepatotoxicity . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2015. [cited 2019 May 20]. Available from: http://hdl.handle.net/10550/42776.

Council of Science Editors:

García Cañaveras JC. Metabolomics as a tool for the study of drug-induced hepatotoxicity . [Doctoral Dissertation]. Universitat de Valencia; 2015. Available from: http://hdl.handle.net/10550/42776


Boise State University

3. Lamb, Cheri Lou. Consequences of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) on Hepatic Stellate Cell Activation and Extracellular Matrix Remodeling During Chronic Liver Injury.

Degree: 2016, Boise State University

 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) is a persistent environmental pollutant and high-affinity ligand for the aryl hydrocarbon receptor (AhR). Exposure to TCDD elicits a spectrum of toxic effects,… (more)

Subjects/Keywords: Wound Healing; Inflammation; Hepatotoxicity; Toxicology

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APA (6th Edition):

Lamb, C. L. (2016). Consequences of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) on Hepatic Stellate Cell Activation and Extracellular Matrix Remodeling During Chronic Liver Injury. (Thesis). Boise State University. Retrieved from https://scholarworks.boisestate.edu/td/1114

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lamb, Cheri Lou. “Consequences of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) on Hepatic Stellate Cell Activation and Extracellular Matrix Remodeling During Chronic Liver Injury.” 2016. Thesis, Boise State University. Accessed May 20, 2019. https://scholarworks.boisestate.edu/td/1114.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lamb, Cheri Lou. “Consequences of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) on Hepatic Stellate Cell Activation and Extracellular Matrix Remodeling During Chronic Liver Injury.” 2016. Web. 20 May 2019.

Vancouver:

Lamb CL. Consequences of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) on Hepatic Stellate Cell Activation and Extracellular Matrix Remodeling During Chronic Liver Injury. [Internet] [Thesis]. Boise State University; 2016. [cited 2019 May 20]. Available from: https://scholarworks.boisestate.edu/td/1114.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lamb CL. Consequences of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) on Hepatic Stellate Cell Activation and Extracellular Matrix Remodeling During Chronic Liver Injury. [Thesis]. Boise State University; 2016. Available from: https://scholarworks.boisestate.edu/td/1114

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Addis Ababa University

4. Getnet, Yimer. Anti Tuberculosis Drug Induced Hepatotoxicity in HIV Positive and Negative Patients .

Degree: 2005, Addis Ababa University

 Anti-tuberculosis drug induced hepatotoxicity (DIH) is a common problem in the management of tuberculosis. This study was intended to identify possible risk factors for development… (more)

Subjects/Keywords: Tuberculosis; HIV; Hepatotoxicity

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APA (6th Edition):

Getnet, Y. (2005). Anti Tuberculosis Drug Induced Hepatotoxicity in HIV Positive and Negative Patients . (Thesis). Addis Ababa University. Retrieved from http://etd.aau.edu.et/dspace/handle/123456789/5674

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Getnet, Yimer. “Anti Tuberculosis Drug Induced Hepatotoxicity in HIV Positive and Negative Patients .” 2005. Thesis, Addis Ababa University. Accessed May 20, 2019. http://etd.aau.edu.et/dspace/handle/123456789/5674.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Getnet, Yimer. “Anti Tuberculosis Drug Induced Hepatotoxicity in HIV Positive and Negative Patients .” 2005. Web. 20 May 2019.

Vancouver:

Getnet Y. Anti Tuberculosis Drug Induced Hepatotoxicity in HIV Positive and Negative Patients . [Internet] [Thesis]. Addis Ababa University; 2005. [cited 2019 May 20]. Available from: http://etd.aau.edu.et/dspace/handle/123456789/5674.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Getnet Y. Anti Tuberculosis Drug Induced Hepatotoxicity in HIV Positive and Negative Patients . [Thesis]. Addis Ababa University; 2005. Available from: http://etd.aau.edu.et/dspace/handle/123456789/5674

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

5. Petronijević, Marija M., 1978-. Farmakoepidemiološka studija spontano prijavljenih hepatotoksičnih reakcija na lekove i biljne dijetetske suplemente.

Degree: Farmaceutski fakultet, 2014, Univerzitet u Beogradu

Farmakologija - Farmakoepidemiologija / Pharmacology - Pharmacoepidemiology

Usled mogućih veoma ozbiljnih kliničkih posledica i velikih poteškoća pri otkrivanju i dijagnostikovanju, hepatotoksičnost lekova ili lekovima izazvano… (more)

Subjects/Keywords: hepatotoxicity; adverse reactions; spontaneous reporting; pharmacovigilance

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APA (6th Edition):

Petronijević, Marija M., 1. (2014). Farmakoepidemiološka studija spontano prijavljenih hepatotoksičnih reakcija na lekove i biljne dijetetske suplemente. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:8493/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Petronijević, Marija M., 1978-. “Farmakoepidemiološka studija spontano prijavljenih hepatotoksičnih reakcija na lekove i biljne dijetetske suplemente.” 2014. Thesis, Univerzitet u Beogradu. Accessed May 20, 2019. https://fedorabg.bg.ac.rs/fedora/get/o:8493/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Petronijević, Marija M., 1978-. “Farmakoepidemiološka studija spontano prijavljenih hepatotoksičnih reakcija na lekove i biljne dijetetske suplemente.” 2014. Web. 20 May 2019.

Vancouver:

Petronijević, Marija M. 1. Farmakoepidemiološka studija spontano prijavljenih hepatotoksičnih reakcija na lekove i biljne dijetetske suplemente. [Internet] [Thesis]. Univerzitet u Beogradu; 2014. [cited 2019 May 20]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:8493/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Petronijević, Marija M. 1. Farmakoepidemiološka studija spontano prijavljenih hepatotoksičnih reakcija na lekove i biljne dijetetske suplemente. [Thesis]. Univerzitet u Beogradu; 2014. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:8493/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

6. Henderson, Neil C. Molecular mechanisms of hepatic injury and repair.

Degree: 2007, University of Edinburgh

 In this thesis I examined molecular mechanisms involved in acute and chronic liver injury, and also studied basic pathways mediating tumour promotion. Acute hepatic failure… (more)

Subjects/Keywords: 616.2; paracetamol hepatotoxicity; acute liver failure

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APA (6th Edition):

Henderson, N. C. (2007). Molecular mechanisms of hepatic injury and repair. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/1554

Chicago Manual of Style (16th Edition):

Henderson, Neil C. “Molecular mechanisms of hepatic injury and repair.” 2007. Doctoral Dissertation, University of Edinburgh. Accessed May 20, 2019. http://hdl.handle.net/1842/1554.

MLA Handbook (7th Edition):

Henderson, Neil C. “Molecular mechanisms of hepatic injury and repair.” 2007. Web. 20 May 2019.

Vancouver:

Henderson NC. Molecular mechanisms of hepatic injury and repair. [Internet] [Doctoral dissertation]. University of Edinburgh; 2007. [cited 2019 May 20]. Available from: http://hdl.handle.net/1842/1554.

Council of Science Editors:

Henderson NC. Molecular mechanisms of hepatic injury and repair. [Doctoral Dissertation]. University of Edinburgh; 2007. Available from: http://hdl.handle.net/1842/1554


University of California – San Francisco

7. Chang, Shiow-Huey. Adherence Issues of Isoniazid Therapy Among Children.

Degree: Nursing, 2013, University of California – San Francisco

 An estimated 8.7 million individuals worldwide are infected with the bacillus mycobacterium tuberculosis, with 1.4 million deaths from tuberculosis (TB) in 2011. A 9-month course… (more)

Subjects/Keywords: Nursing; hepatotoxicity; LTBI; stigma; transaminase; tuberculosis

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APA (6th Edition):

Chang, S. (2013). Adherence Issues of Isoniazid Therapy Among Children. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/1tt2t0t4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chang, Shiow-Huey. “Adherence Issues of Isoniazid Therapy Among Children.” 2013. Thesis, University of California – San Francisco. Accessed May 20, 2019. http://www.escholarship.org/uc/item/1tt2t0t4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chang, Shiow-Huey. “Adherence Issues of Isoniazid Therapy Among Children.” 2013. Web. 20 May 2019.

Vancouver:

Chang S. Adherence Issues of Isoniazid Therapy Among Children. [Internet] [Thesis]. University of California – San Francisco; 2013. [cited 2019 May 20]. Available from: http://www.escholarship.org/uc/item/1tt2t0t4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chang S. Adherence Issues of Isoniazid Therapy Among Children. [Thesis]. University of California – San Francisco; 2013. Available from: http://www.escholarship.org/uc/item/1tt2t0t4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Martins, Nádia Maria. Avaliação do estresse oxidativo e estado redox mitocondrial na hepatotoxicidade induzida pela cisplatina em ratos \'Wistar\': efeito protetor da dimetiltiouréia.

Degree: PhD, Toxicologia, 2007, University of São Paulo

A cisplatina ainda é um dos agentes quimioterápicos mais efetivos. No entanto, em elevadas doses pode ocorrer hepatotoxicidade. Alguns antioxidantes têm sido mostrado amenizar a… (more)

Subjects/Keywords: cisplatin; cisplatina; citoproteção; cytoprotection; hepatotoxicidade; hepatotoxicity; mitochondria; mitocôndria e EROs; ROS

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APA (6th Edition):

Martins, N. M. (2007). Avaliação do estresse oxidativo e estado redox mitocondrial na hepatotoxicidade induzida pela cisplatina em ratos \'Wistar\': efeito protetor da dimetiltiouréia. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/60/60134/tde-24072007-095608/ ;

Chicago Manual of Style (16th Edition):

Martins, Nádia Maria. “Avaliação do estresse oxidativo e estado redox mitocondrial na hepatotoxicidade induzida pela cisplatina em ratos \'Wistar\': efeito protetor da dimetiltiouréia.” 2007. Doctoral Dissertation, University of São Paulo. Accessed May 20, 2019. http://www.teses.usp.br/teses/disponiveis/60/60134/tde-24072007-095608/ ;.

MLA Handbook (7th Edition):

Martins, Nádia Maria. “Avaliação do estresse oxidativo e estado redox mitocondrial na hepatotoxicidade induzida pela cisplatina em ratos \'Wistar\': efeito protetor da dimetiltiouréia.” 2007. Web. 20 May 2019.

Vancouver:

Martins NM. Avaliação do estresse oxidativo e estado redox mitocondrial na hepatotoxicidade induzida pela cisplatina em ratos \'Wistar\': efeito protetor da dimetiltiouréia. [Internet] [Doctoral dissertation]. University of São Paulo; 2007. [cited 2019 May 20]. Available from: http://www.teses.usp.br/teses/disponiveis/60/60134/tde-24072007-095608/ ;.

Council of Science Editors:

Martins NM. Avaliação do estresse oxidativo e estado redox mitocondrial na hepatotoxicidade induzida pela cisplatina em ratos \'Wistar\': efeito protetor da dimetiltiouréia. [Doctoral Dissertation]. University of São Paulo; 2007. Available from: http://www.teses.usp.br/teses/disponiveis/60/60134/tde-24072007-095608/ ;

9. Umar, Shahid. Studies on some aspects of acute phase protein metabolism; -.

Degree: Biochemistry, 1992, Aligarh Muslim University

Abstract not available newline newline

Summary 107-112, Bibliography p. 93-106

Advisors/Committee Members: Sahib, M K.

Subjects/Keywords: Metabolism; Immunologic; Biochemical; Mechanism; Hepatotoxicity

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APA (6th Edition):

Umar, S. (1992). Studies on some aspects of acute phase protein metabolism; -. (Thesis). Aligarh Muslim University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/54043

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Umar, Shahid. “Studies on some aspects of acute phase protein metabolism; -.” 1992. Thesis, Aligarh Muslim University. Accessed May 20, 2019. http://shodhganga.inflibnet.ac.in/handle/10603/54043.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Umar, Shahid. “Studies on some aspects of acute phase protein metabolism; -.” 1992. Web. 20 May 2019.

Vancouver:

Umar S. Studies on some aspects of acute phase protein metabolism; -. [Internet] [Thesis]. Aligarh Muslim University; 1992. [cited 2019 May 20]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/54043.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Umar S. Studies on some aspects of acute phase protein metabolism; -. [Thesis]. Aligarh Muslim University; 1992. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/54043

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Παπαστεφάνου, Βασίλειος. Έκφραση αγγειογενετικών παραγόντων κατά την ηπατική αναγέννηση μετά από χορήγηση τοξικής δόσης ακεταμινοφαινής σε επίμυς.

Degree: 2009, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 Liver has the capacity to regenerate after partial hepatectomy or after the induction of damage of toxic or viral etiology both inducing a reduction in… (more)

Subjects/Keywords: Ακεταμινοφαίνη; Ηπατική αναγέννηση; Αγγειογένεση; Ηπατοτοξικότητα; Acetaminophen; Liver regeneration; Angiogenesis; Hepatotoxicity

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APA (6th Edition):

Παπαστεφάνου, . . (2009). Έκφραση αγγειογενετικών παραγόντων κατά την ηπατική αναγέννηση μετά από χορήγηση τοξικής δόσης ακεταμινοφαινής σε επίμυς. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/24353

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Παπαστεφάνου, Βασίλειος. “Έκφραση αγγειογενετικών παραγόντων κατά την ηπατική αναγέννηση μετά από χορήγηση τοξικής δόσης ακεταμινοφαινής σε επίμυς.” 2009. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed May 20, 2019. http://hdl.handle.net/10442/hedi/24353.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Παπαστεφάνου, Βασίλειος. “Έκφραση αγγειογενετικών παραγόντων κατά την ηπατική αναγέννηση μετά από χορήγηση τοξικής δόσης ακεταμινοφαινής σε επίμυς.” 2009. Web. 20 May 2019.

Vancouver:

Παπαστεφάνου . Έκφραση αγγειογενετικών παραγόντων κατά την ηπατική αναγέννηση μετά από χορήγηση τοξικής δόσης ακεταμινοφαινής σε επίμυς. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2009. [cited 2019 May 20]. Available from: http://hdl.handle.net/10442/hedi/24353.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Παπαστεφάνου . Έκφραση αγγειογενετικών παραγόντων κατά την ηπατική αναγέννηση μετά από χορήγηση τοξικής δόσης ακεταμινοφαινής σε επίμυς. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2009. Available from: http://hdl.handle.net/10442/hedi/24353

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pretoria

11. Van Tonder, Jacob John. Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes.

Degree: Pharmacology, 2012, University of Pretoria

 In vitro testing includes both cell-based and cell-free systems that can be used to detect toxicity induced by xenobiotics. In vitro methods are especially useful… (more)

Subjects/Keywords: Apoptosis; Cyp1a1; Ddd; Dde; Ddt; Hepatotoxicity; Mechanistic profiling; UCTD

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APA (6th Edition):

Van Tonder, J. J. (2012). Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes. (Doctoral Dissertation). University of Pretoria. Retrieved from http://hdl.handle.net/2263/24162

Chicago Manual of Style (16th Edition):

Van Tonder, Jacob John. “Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes.” 2012. Doctoral Dissertation, University of Pretoria. Accessed May 20, 2019. http://hdl.handle.net/2263/24162.

MLA Handbook (7th Edition):

Van Tonder, Jacob John. “Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes.” 2012. Web. 20 May 2019.

Vancouver:

Van Tonder JJ. Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes. [Internet] [Doctoral dissertation]. University of Pretoria; 2012. [cited 2019 May 20]. Available from: http://hdl.handle.net/2263/24162.

Council of Science Editors:

Van Tonder JJ. Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes. [Doctoral Dissertation]. University of Pretoria; 2012. Available from: http://hdl.handle.net/2263/24162


University of Pretoria

12. Van Tonder, Jacob John. Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes .

Degree: 2012, University of Pretoria

 In vitro testing includes both cell-based and cell-free systems that can be used to detect toxicity induced by xenobiotics. In vitro methods are especially useful… (more)

Subjects/Keywords: Apoptosis; Cyp1a1; Ddd; Dde; Ddt; Hepatotoxicity; Mechanistic profiling; UCTD

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APA (6th Edition):

Van Tonder, J. J. (2012). Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes . (Doctoral Dissertation). University of Pretoria. Retrieved from http://upetd.up.ac.za/thesis/available/etd-04262012-112100/

Chicago Manual of Style (16th Edition):

Van Tonder, Jacob John. “Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes .” 2012. Doctoral Dissertation, University of Pretoria. Accessed May 20, 2019. http://upetd.up.ac.za/thesis/available/etd-04262012-112100/.

MLA Handbook (7th Edition):

Van Tonder, Jacob John. “Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes .” 2012. Web. 20 May 2019.

Vancouver:

Van Tonder JJ. Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes . [Internet] [Doctoral dissertation]. University of Pretoria; 2012. [cited 2019 May 20]. Available from: http://upetd.up.ac.za/thesis/available/etd-04262012-112100/.

Council of Science Editors:

Van Tonder JJ. Development of an in vitro mechanistic toxicity screening model using cultured hepatocytes . [Doctoral Dissertation]. University of Pretoria; 2012. Available from: http://upetd.up.ac.za/thesis/available/etd-04262012-112100/


Universidade do Rio Grande do Sul

13. Rodrigues, Daniela. Transplante de hepatócitos no modelo experimental de hepatotoxicidade aguda induzida por paracetamol em ratos.

Degree: 2012, Universidade do Rio Grande do Sul

O transplante de hepatócitos é uma modalidade terapêutica atrativa para as doenças hepáticas, assim como uma alternativa para o transplante hepático. O objetivo do presente… (more)

Subjects/Keywords: Acetaminophen; Acetaminofen; Hepatotoxicity; Gastroenterologia; Toxicidade; Rat hepatocyte transplantation; Hepatócitos; Ratos

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APA (6th Edition):

Rodrigues, D. (2012). Transplante de hepatócitos no modelo experimental de hepatotoxicidade aguda induzida por paracetamol em ratos. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/66678

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rodrigues, Daniela. “Transplante de hepatócitos no modelo experimental de hepatotoxicidade aguda induzida por paracetamol em ratos.” 2012. Thesis, Universidade do Rio Grande do Sul. Accessed May 20, 2019. http://hdl.handle.net/10183/66678.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rodrigues, Daniela. “Transplante de hepatócitos no modelo experimental de hepatotoxicidade aguda induzida por paracetamol em ratos.” 2012. Web. 20 May 2019.

Vancouver:

Rodrigues D. Transplante de hepatócitos no modelo experimental de hepatotoxicidade aguda induzida por paracetamol em ratos. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2012. [cited 2019 May 20]. Available from: http://hdl.handle.net/10183/66678.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rodrigues D. Transplante de hepatócitos no modelo experimental de hepatotoxicidade aguda induzida por paracetamol em ratos. [Thesis]. Universidade do Rio Grande do Sul; 2012. Available from: http://hdl.handle.net/10183/66678

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

14. Mach, John. The effect of ageing on drug induced liver injury: insights from animal models .

Degree: 2015, University of Sydney

 Historically, older people (>65 years) were thought to be at increased risk of drug induced liver injury (DILI). This has not been well established empirically.… (more)

Subjects/Keywords: Drug induced liver injury; paracetamol; isoniazid; hepatotoxicity; ageing

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APA (6th Edition):

Mach, J. (2015). The effect of ageing on drug induced liver injury: insights from animal models . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/14689

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mach, John. “The effect of ageing on drug induced liver injury: insights from animal models .” 2015. Thesis, University of Sydney. Accessed May 20, 2019. http://hdl.handle.net/2123/14689.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mach, John. “The effect of ageing on drug induced liver injury: insights from animal models .” 2015. Web. 20 May 2019.

Vancouver:

Mach J. The effect of ageing on drug induced liver injury: insights from animal models . [Internet] [Thesis]. University of Sydney; 2015. [cited 2019 May 20]. Available from: http://hdl.handle.net/2123/14689.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mach J. The effect of ageing on drug induced liver injury: insights from animal models . [Thesis]. University of Sydney; 2015. Available from: http://hdl.handle.net/2123/14689

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Dimou, Pantelis. Πειραματική διερύνηση της ηπατοτοξικότητας της δαναζόλης μετά από μερική ηπατεκτομή και συγχορήγηση φαρμάκων.

Degree: 2014, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

In this paper, it has been studies the hepatotoxicity og danazol after a partial hepatectomy, as well as after an intramuscular drug administration, which both… (more)

Subjects/Keywords: Δαναζόλη; Ηπατοτοξικότητα; Ηπατεκτομή; Φάρμακα; Θειοκεταμίδη; Danazol; Hepatotoxicity; Hepatectomy; Drugs; Thioakeatmidi

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APA (6th Edition):

Dimou, P. (2014). Πειραματική διερύνηση της ηπατοτοξικότητας της δαναζόλης μετά από μερική ηπατεκτομή και συγχορήγηση φαρμάκων. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/43669

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dimou, Pantelis. “Πειραματική διερύνηση της ηπατοτοξικότητας της δαναζόλης μετά από μερική ηπατεκτομή και συγχορήγηση φαρμάκων.” 2014. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed May 20, 2019. http://hdl.handle.net/10442/hedi/43669.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dimou, Pantelis. “Πειραματική διερύνηση της ηπατοτοξικότητας της δαναζόλης μετά από μερική ηπατεκτομή και συγχορήγηση φαρμάκων.” 2014. Web. 20 May 2019.

Vancouver:

Dimou P. Πειραματική διερύνηση της ηπατοτοξικότητας της δαναζόλης μετά από μερική ηπατεκτομή και συγχορήγηση φαρμάκων. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2014. [cited 2019 May 20]. Available from: http://hdl.handle.net/10442/hedi/43669.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dimou P. Πειραματική διερύνηση της ηπατοτοξικότητας της δαναζόλης μετά από μερική ηπατεκτομή και συγχορήγηση φαρμάκων. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2014. Available from: http://hdl.handle.net/10442/hedi/43669

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Kyriakou, Loukas. Μελέτη του μηχανισμού ηπατοτοξικής δράσης του καδμίου σε επίμυς.

Degree: 2015, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Cadmium is a known industrial and environmental pollutant. It causes hepatotoxicity uponacute administration.Features of cadmium-induced acute hepatoxicity encompass necrosis,apoptosis, peliosis and inflammatory infiltration.Gadolinium chloride (GdCl3)… (more)

Subjects/Keywords: Ηπατοτοξικότητα; Κάδμιο; Γαδολίνιο; Νέκρωση; Ηπατοκύτταρα; Hepatotoxicity; Cadmium; Gadolinium; Necrosis; Hepatocytes

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APA (6th Edition):

Kyriakou, L. (2015). Μελέτη του μηχανισμού ηπατοτοξικής δράσης του καδμίου σε επίμυς. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/42861

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kyriakou, Loukas. “Μελέτη του μηχανισμού ηπατοτοξικής δράσης του καδμίου σε επίμυς.” 2015. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed May 20, 2019. http://hdl.handle.net/10442/hedi/42861.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kyriakou, Loukas. “Μελέτη του μηχανισμού ηπατοτοξικής δράσης του καδμίου σε επίμυς.” 2015. Web. 20 May 2019.

Vancouver:

Kyriakou L. Μελέτη του μηχανισμού ηπατοτοξικής δράσης του καδμίου σε επίμυς. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2015. [cited 2019 May 20]. Available from: http://hdl.handle.net/10442/hedi/42861.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kyriakou L. Μελέτη του μηχανισμού ηπατοτοξικής δράσης του καδμίου σε επίμυς. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2015. Available from: http://hdl.handle.net/10442/hedi/42861

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

17. Dinić, Miroslav R., 1988-. Uticaj bioaktivnih molekula laktobacila na procese autofagije i inflamacije u in vitro i in vivo sistemima.

Degree: Farmaceutski fakultet, 2018, Univerzitet u Beogradu

Farmacija - Farmaceutska mikrobiologija / Pharmacy - Pharmaceutical Microbiology

Izolacija i karakterizacija bakterijskih biomolekula koji interaguju sa receptorima ćelija domaćina predstavlja kljuĉ za razumevanje mehanizama… (more)

Subjects/Keywords: lactobacilli; exopolysaccharide; postbiotics; autophagy; hepatotoxicity; hyperalgesia; inflammatory mediators; immunosuppression

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APA (6th Edition):

Dinić, Miroslav R., 1. (2018). Uticaj bioaktivnih molekula laktobacila na procese autofagije i inflamacije u in vitro i in vivo sistemima. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:18343/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dinić, Miroslav R., 1988-. “Uticaj bioaktivnih molekula laktobacila na procese autofagije i inflamacije u in vitro i in vivo sistemima.” 2018. Thesis, Univerzitet u Beogradu. Accessed May 20, 2019. https://fedorabg.bg.ac.rs/fedora/get/o:18343/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dinić, Miroslav R., 1988-. “Uticaj bioaktivnih molekula laktobacila na procese autofagije i inflamacije u in vitro i in vivo sistemima.” 2018. Web. 20 May 2019.

Vancouver:

Dinić, Miroslav R. 1. Uticaj bioaktivnih molekula laktobacila na procese autofagije i inflamacije u in vitro i in vivo sistemima. [Internet] [Thesis]. Univerzitet u Beogradu; 2018. [cited 2019 May 20]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:18343/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dinić, Miroslav R. 1. Uticaj bioaktivnih molekula laktobacila na procese autofagije i inflamacije u in vitro i in vivo sistemima. [Thesis]. Univerzitet u Beogradu; 2018. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:18343/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

18. Chan, Katherine. Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems.

Degree: 2008, University of Toronto

Hepatotoxicity is a serious adverse health effect caused by drugs and other chemical toxins generally detected in the later stages of drug development or in… (more)

Subjects/Keywords: xenobiotic metabolism; hepatotoxicity; QSAR; 0383

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APA (6th Edition):

Chan, K. (2008). Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/17290

Chicago Manual of Style (16th Edition):

Chan, Katherine. “Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems.” 2008. Doctoral Dissertation, University of Toronto. Accessed May 20, 2019. http://hdl.handle.net/1807/17290.

MLA Handbook (7th Edition):

Chan, Katherine. “Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems.” 2008. Web. 20 May 2019.

Vancouver:

Chan K. Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems. [Internet] [Doctoral dissertation]. University of Toronto; 2008. [cited 2019 May 20]. Available from: http://hdl.handle.net/1807/17290.

Council of Science Editors:

Chan K. Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems. [Doctoral Dissertation]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/17290


University of Connecticut

19. Rohrer, Philip. The Role of Nrf2 in the Differential Susceptibility of Male and Female Mice to Acetaminophen-Induced Hepatotoxicity.

Degree: MS, Pharmaceutical Science, 2014, University of Connecticut

Subjects/Keywords: Nrf2; APAP; liver; gender; hepatotoxicity

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APA (6th Edition):

Rohrer, P. (2014). The Role of Nrf2 in the Differential Susceptibility of Male and Female Mice to Acetaminophen-Induced Hepatotoxicity. (Masters Thesis). University of Connecticut. Retrieved from https://opencommons.uconn.edu/gs_theses/574

Chicago Manual of Style (16th Edition):

Rohrer, Philip. “The Role of Nrf2 in the Differential Susceptibility of Male and Female Mice to Acetaminophen-Induced Hepatotoxicity.” 2014. Masters Thesis, University of Connecticut. Accessed May 20, 2019. https://opencommons.uconn.edu/gs_theses/574.

MLA Handbook (7th Edition):

Rohrer, Philip. “The Role of Nrf2 in the Differential Susceptibility of Male and Female Mice to Acetaminophen-Induced Hepatotoxicity.” 2014. Web. 20 May 2019.

Vancouver:

Rohrer P. The Role of Nrf2 in the Differential Susceptibility of Male and Female Mice to Acetaminophen-Induced Hepatotoxicity. [Internet] [Masters thesis]. University of Connecticut; 2014. [cited 2019 May 20]. Available from: https://opencommons.uconn.edu/gs_theses/574.

Council of Science Editors:

Rohrer P. The Role of Nrf2 in the Differential Susceptibility of Male and Female Mice to Acetaminophen-Induced Hepatotoxicity. [Masters Thesis]. University of Connecticut; 2014. Available from: https://opencommons.uconn.edu/gs_theses/574


University of Toronto

20. Hanna, Daniel. Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis.

Degree: PhD, 2018, University of Toronto

 Liver cancer is the third most common cause of cancer-related death in humans and is 2-4 times more common in men than in women for… (more)

Subjects/Keywords: Hepatotoxicity; Inflammation; Liver cancer; Mouse models; Procarcinogens; Sex differences; 0419

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APA (6th Edition):

Hanna, D. (2018). Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/91754

Chicago Manual of Style (16th Edition):

Hanna, Daniel. “Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis.” 2018. Doctoral Dissertation, University of Toronto. Accessed May 20, 2019. http://hdl.handle.net/1807/91754.

MLA Handbook (7th Edition):

Hanna, Daniel. “Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis.” 2018. Web. 20 May 2019.

Vancouver:

Hanna D. Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis. [Internet] [Doctoral dissertation]. University of Toronto; 2018. [cited 2019 May 20]. Available from: http://hdl.handle.net/1807/91754.

Council of Science Editors:

Hanna D. Sex Differences in Hepatotoxic, Inflammatory and Proliferative Responses in Mouse Models of Liver Carcinogenesis. [Doctoral Dissertation]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91754

21. Singh, Padam. Hepatotoxicity and Haemato Biochemical alteration in fresh water teleost clarias Batrachus along with some insecticides endosulfan Malathion and rogor exposure;.

Degree: Zoology, 2012, Chaudhary Charan Singh University

None newline

Summary p. 51-65, Refrences p. 66-70

Advisors/Committee Members: Malhotra, N M.

Subjects/Keywords: Hepatotoxicity; Haemato Biochemical; Teleost Clarias Batrachus

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APA (6th Edition):

Singh, P. (2012). Hepatotoxicity and Haemato Biochemical alteration in fresh water teleost clarias Batrachus along with some insecticides endosulfan Malathion and rogor exposure;. (Thesis). Chaudhary Charan Singh University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/40371

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Singh, Padam. “Hepatotoxicity and Haemato Biochemical alteration in fresh water teleost clarias Batrachus along with some insecticides endosulfan Malathion and rogor exposure;.” 2012. Thesis, Chaudhary Charan Singh University. Accessed May 20, 2019. http://shodhganga.inflibnet.ac.in/handle/10603/40371.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Singh, Padam. “Hepatotoxicity and Haemato Biochemical alteration in fresh water teleost clarias Batrachus along with some insecticides endosulfan Malathion and rogor exposure;.” 2012. Web. 20 May 2019.

Vancouver:

Singh P. Hepatotoxicity and Haemato Biochemical alteration in fresh water teleost clarias Batrachus along with some insecticides endosulfan Malathion and rogor exposure;. [Internet] [Thesis]. Chaudhary Charan Singh University; 2012. [cited 2019 May 20]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/40371.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Singh P. Hepatotoxicity and Haemato Biochemical alteration in fresh water teleost clarias Batrachus along with some insecticides endosulfan Malathion and rogor exposure;. [Thesis]. Chaudhary Charan Singh University; 2012. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/40371

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Fernandes, Fábio Henrique [UNESP]. Potencial toxicogenômico e carcinogênico de efluentes da indústria têxtil e dos corantes Disperse Red 1 e Disperse Blue 291 em roedores.

Degree: 2017, Universidade Estadual Paulista

As consequências da poluição ambiental não se limitam a danos ao ambiente, mas também atingem diretamente a saúde humana. Recentemente, a Organização Mundial de Saúde… (more)

Subjects/Keywords: Azo corantes; Genotoxicidade; Hepatotoxicidade; Mutagênese ambiental; Azo dyes; Environmental mutagenesis; Genotoxicity; Hepatotoxicity

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APA (6th Edition):

Fernandes, F. H. [. (2017). Potencial toxicogenômico e carcinogênico de efluentes da indústria têxtil e dos corantes Disperse Red 1 e Disperse Blue 291 em roedores. (Thesis). Universidade Estadual Paulista. Retrieved from http://hdl.handle.net/11449/148643

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fernandes, Fábio Henrique [UNESP]. “Potencial toxicogenômico e carcinogênico de efluentes da indústria têxtil e dos corantes Disperse Red 1 e Disperse Blue 291 em roedores.” 2017. Thesis, Universidade Estadual Paulista. Accessed May 20, 2019. http://hdl.handle.net/11449/148643.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fernandes, Fábio Henrique [UNESP]. “Potencial toxicogenômico e carcinogênico de efluentes da indústria têxtil e dos corantes Disperse Red 1 e Disperse Blue 291 em roedores.” 2017. Web. 20 May 2019.

Vancouver:

Fernandes FH[. Potencial toxicogenômico e carcinogênico de efluentes da indústria têxtil e dos corantes Disperse Red 1 e Disperse Blue 291 em roedores. [Internet] [Thesis]. Universidade Estadual Paulista; 2017. [cited 2019 May 20]. Available from: http://hdl.handle.net/11449/148643.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fernandes FH[. Potencial toxicogenômico e carcinogênico de efluentes da indústria têxtil e dos corantes Disperse Red 1 e Disperse Blue 291 em roedores. [Thesis]. Universidade Estadual Paulista; 2017. Available from: http://hdl.handle.net/11449/148643

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Gulmez, Sinem. EPIHAM Drug-induced liver injury leading to hospital admission : a study in national healthcare insurance databases : EPIHAM : Epidémiologie d’Hépatites aiguës médicamenteuses.

Degree: Docteur es, Pharmacologie. Option : Pharmaco-épidémiologie, pharmaco-vigilance, 2017, Bordeaux

L’objectif principal de l’étude était d’identifier les principaux médicaments associés aux hépatites aiguës (HA) associé aux médicaments (HAM) en France. Trois approches méthodologiques ont été… (more)

Subjects/Keywords: Cas-population; DILI; Hépatotoxicité médicamenteuse; Case-population study; DILI; Drug-exposed hepatotoxicity

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gulmez, S. (2017). EPIHAM Drug-induced liver injury leading to hospital admission : a study in national healthcare insurance databases : EPIHAM : Epidémiologie d’Hépatites aiguës médicamenteuses. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2017BORD0592

Chicago Manual of Style (16th Edition):

Gulmez, Sinem. “EPIHAM Drug-induced liver injury leading to hospital admission : a study in national healthcare insurance databases : EPIHAM : Epidémiologie d’Hépatites aiguës médicamenteuses.” 2017. Doctoral Dissertation, Bordeaux. Accessed May 20, 2019. http://www.theses.fr/2017BORD0592.

MLA Handbook (7th Edition):

Gulmez, Sinem. “EPIHAM Drug-induced liver injury leading to hospital admission : a study in national healthcare insurance databases : EPIHAM : Epidémiologie d’Hépatites aiguës médicamenteuses.” 2017. Web. 20 May 2019.

Vancouver:

Gulmez S. EPIHAM Drug-induced liver injury leading to hospital admission : a study in national healthcare insurance databases : EPIHAM : Epidémiologie d’Hépatites aiguës médicamenteuses. [Internet] [Doctoral dissertation]. Bordeaux; 2017. [cited 2019 May 20]. Available from: http://www.theses.fr/2017BORD0592.

Council of Science Editors:

Gulmez S. EPIHAM Drug-induced liver injury leading to hospital admission : a study in national healthcare insurance databases : EPIHAM : Epidémiologie d’Hépatites aiguës médicamenteuses. [Doctoral Dissertation]. Bordeaux; 2017. Available from: http://www.theses.fr/2017BORD0592

24. Alexandre Garcia Marques. DA AUSÊNCIA DE DIVULGAÇÃO OSTENSIVA DO B24 RISCO DO PARACETAMOL E DA RESPONSABILIDADE CIVIL.

Degree: 2010, Universidade Católica de Goiás

O estudo apresenta uma análise crítica sobre a ausência de divulgação ostensiva do risco potencial a que está submetido o consumidor de fármaco à base… (more)

Subjects/Keywords: Security; Paracetamol; Ostensividade; Hepatotoxidade, Segurança; ECOLOGIA; Paracetamol; Ostensivicity; Hepatotoxicity; Civil Responsibility; Responsabilidade Civil

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APA (6th Edition):

Marques, A. G. (2010). DA AUSÊNCIA DE DIVULGAÇÃO OSTENSIVA DO B24 RISCO DO PARACETAMOL E DA RESPONSABILIDADE CIVIL. (Thesis). Universidade Católica de Goiás. Retrieved from http://tede.biblioteca.ucg.br/tde_busca/arquivo.php?codArquivo=1011

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marques, Alexandre Garcia. “DA AUSÊNCIA DE DIVULGAÇÃO OSTENSIVA DO B24 RISCO DO PARACETAMOL E DA RESPONSABILIDADE CIVIL.” 2010. Thesis, Universidade Católica de Goiás. Accessed May 20, 2019. http://tede.biblioteca.ucg.br/tde_busca/arquivo.php?codArquivo=1011.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marques, Alexandre Garcia. “DA AUSÊNCIA DE DIVULGAÇÃO OSTENSIVA DO B24 RISCO DO PARACETAMOL E DA RESPONSABILIDADE CIVIL.” 2010. Web. 20 May 2019.

Vancouver:

Marques AG. DA AUSÊNCIA DE DIVULGAÇÃO OSTENSIVA DO B24 RISCO DO PARACETAMOL E DA RESPONSABILIDADE CIVIL. [Internet] [Thesis]. Universidade Católica de Goiás; 2010. [cited 2019 May 20]. Available from: http://tede.biblioteca.ucg.br/tde_busca/arquivo.php?codArquivo=1011.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marques AG. DA AUSÊNCIA DE DIVULGAÇÃO OSTENSIVA DO B24 RISCO DO PARACETAMOL E DA RESPONSABILIDADE CIVIL. [Thesis]. Universidade Católica de Goiás; 2010. Available from: http://tede.biblioteca.ucg.br/tde_busca/arquivo.php?codArquivo=1011

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

25. Schemitt, Elizângela Gonçalves. Ação da glutamina sobre o estresse oxidativo e processo inflamatório na insuficiência hepática aguda grave.

Degree: 2014, Universidade do Rio Grande do Sul

Introdução: A Insuficiência Hepática Aguda Grave (IHAG) é uma síndrome clínica rara, caracterizada por uma disfunção grave e súbita das células do fígado. A tioacetamida… (more)

Subjects/Keywords: Fígado; Hepatotoxicity; Lipid peroxidation; Toxicidade; Free radicals; Falência hepática aguda; Antioxidants; Estresse oxidativo; Glutamina; Liver

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APA (6th Edition):

Schemitt, E. G. (2014). Ação da glutamina sobre o estresse oxidativo e processo inflamatório na insuficiência hepática aguda grave. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/110198

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schemitt, Elizângela Gonçalves. “Ação da glutamina sobre o estresse oxidativo e processo inflamatório na insuficiência hepática aguda grave.” 2014. Thesis, Universidade do Rio Grande do Sul. Accessed May 20, 2019. http://hdl.handle.net/10183/110198.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schemitt, Elizângela Gonçalves. “Ação da glutamina sobre o estresse oxidativo e processo inflamatório na insuficiência hepática aguda grave.” 2014. Web. 20 May 2019.

Vancouver:

Schemitt EG. Ação da glutamina sobre o estresse oxidativo e processo inflamatório na insuficiência hepática aguda grave. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2014. [cited 2019 May 20]. Available from: http://hdl.handle.net/10183/110198.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schemitt EG. Ação da glutamina sobre o estresse oxidativo e processo inflamatório na insuficiência hepática aguda grave. [Thesis]. Universidade do Rio Grande do Sul; 2014. Available from: http://hdl.handle.net/10183/110198

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. 馬渡, 力. A Combination Polymorphism of the Glutathione Synthesis Genes Can Be a Predictive Biomarker for Anti-Tuberculosis Drug-Induced Hepatotoxicity in Japanese Patients with Pulmonary Tuberculosis : グルタチオン合成関連遺伝子の多型の組み合わせは日本人肺結核患者における抗結核薬誘発性肝障害の予測バイオマーカーとなり得る.

Degree: 博士(薬学), 2016, Nagasaki University / 長崎大学

 Background: To identify certain genes related to anti-tuberculosis drug-induced hepatotoxicity (ATDH) for Japanese patients with pulmonary tuberculosis (TB), we examined an association study of single… (more)

Subjects/Keywords: Tuberculosis; Anti-tuberculosis drug-induced hepatotoxicity; Single nucleotide polymorphism; Glutathione synthesis; Case-control association study

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APA (6th Edition):

馬渡, . (2016). A Combination Polymorphism of the Glutathione Synthesis Genes Can Be a Predictive Biomarker for Anti-Tuberculosis Drug-Induced Hepatotoxicity in Japanese Patients with Pulmonary Tuberculosis : グルタチオン合成関連遺伝子の多型の組み合わせは日本人肺結核患者における抗結核薬誘発性肝障害の予測バイオマーカーとなり得る. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/36832

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

馬渡, 力. “A Combination Polymorphism of the Glutathione Synthesis Genes Can Be a Predictive Biomarker for Anti-Tuberculosis Drug-Induced Hepatotoxicity in Japanese Patients with Pulmonary Tuberculosis : グルタチオン合成関連遺伝子の多型の組み合わせは日本人肺結核患者における抗結核薬誘発性肝障害の予測バイオマーカーとなり得る.” 2016. Thesis, Nagasaki University / 長崎大学. Accessed May 20, 2019. http://hdl.handle.net/10069/36832.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

馬渡, 力. “A Combination Polymorphism of the Glutathione Synthesis Genes Can Be a Predictive Biomarker for Anti-Tuberculosis Drug-Induced Hepatotoxicity in Japanese Patients with Pulmonary Tuberculosis : グルタチオン合成関連遺伝子の多型の組み合わせは日本人肺結核患者における抗結核薬誘発性肝障害の予測バイオマーカーとなり得る.” 2016. Web. 20 May 2019.

Vancouver:

馬渡 . A Combination Polymorphism of the Glutathione Synthesis Genes Can Be a Predictive Biomarker for Anti-Tuberculosis Drug-Induced Hepatotoxicity in Japanese Patients with Pulmonary Tuberculosis : グルタチオン合成関連遺伝子の多型の組み合わせは日本人肺結核患者における抗結核薬誘発性肝障害の予測バイオマーカーとなり得る. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2016. [cited 2019 May 20]. Available from: http://hdl.handle.net/10069/36832.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

馬渡 . A Combination Polymorphism of the Glutathione Synthesis Genes Can Be a Predictive Biomarker for Anti-Tuberculosis Drug-Induced Hepatotoxicity in Japanese Patients with Pulmonary Tuberculosis : グルタチオン合成関連遺伝子の多型の組み合わせは日本人肺結核患者における抗結核薬誘発性肝障害の予測バイオマーカーとなり得る. [Thesis]. Nagasaki University / 長崎大学; 2016. Available from: http://hdl.handle.net/10069/36832

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Acar, Özge. Sıçanlarda siklofosfamid nedenli hepatotoksisitede oksidatif stres ve karaciğer hasarına karşı selenyumun koruyucu etkisi .

Degree: ESOGÜ, Fen Edebiyat Fakültesi, Biyoloji, 2015, Eskisehir Osmangazi University

 Siklofosfamid (CP) klinikte kanser ve non-malignant hastalıkların tedavisinde yaygın olarak kullanılan, yüksek derecede etkili, alkilleyici sitotoksik bir ilaçtır. CP’nin antitümoral etkinliği yüksek dozda kullanılmasına bağlıdır.… (more)

Subjects/Keywords: Siklofosfamid; Oksidatif Stres; Hepatotoksisite; Selenyum; Sitoprotektivite; Sıçan; Cyclophosphamide; Oxidative Stress; Hepatotoxicity; Selenium; Cytoprotectivity; Rat

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APA (6th Edition):

Acar, . (2015). Sıçanlarda siklofosfamid nedenli hepatotoksisitede oksidatif stres ve karaciğer hasarına karşı selenyumun koruyucu etkisi . (Thesis). Eskisehir Osmangazi University. Retrieved from http://hdl.handle.net/11684/444

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Acar, Özge. “Sıçanlarda siklofosfamid nedenli hepatotoksisitede oksidatif stres ve karaciğer hasarına karşı selenyumun koruyucu etkisi .” 2015. Thesis, Eskisehir Osmangazi University. Accessed May 20, 2019. http://hdl.handle.net/11684/444.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Acar, Özge. “Sıçanlarda siklofosfamid nedenli hepatotoksisitede oksidatif stres ve karaciğer hasarına karşı selenyumun koruyucu etkisi .” 2015. Web. 20 May 2019.

Vancouver:

Acar . Sıçanlarda siklofosfamid nedenli hepatotoksisitede oksidatif stres ve karaciğer hasarına karşı selenyumun koruyucu etkisi . [Internet] [Thesis]. Eskisehir Osmangazi University; 2015. [cited 2019 May 20]. Available from: http://hdl.handle.net/11684/444.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Acar . Sıçanlarda siklofosfamid nedenli hepatotoksisitede oksidatif stres ve karaciğer hasarına karşı selenyumun koruyucu etkisi . [Thesis]. Eskisehir Osmangazi University; 2015. Available from: http://hdl.handle.net/11684/444

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

28. Hanna, Daniel. Sex and Strain Differences in Acute Hepatotoxic and Inflammatory Responses to Liver Procarcinogens in the Developing Mouse.

Degree: 2013, University of Toronto

We previously observed that postnatal exposure of mice to the procarcinogen 4-aminobiphenyl (ABP) produced liver tumors only in wild-type males, while arylamine N-acetyltransferase deficient males… (more)

Subjects/Keywords: 4-aminobiphenyl; Aromatic amines; Diethylnitrosamine; Carcinogenesis; Hepatocellular carcinoma; Hepatotoxicity; Inflammation; 0419; 0383; 0433; 0307

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APA (6th Edition):

Hanna, D. (2013). Sex and Strain Differences in Acute Hepatotoxic and Inflammatory Responses to Liver Procarcinogens in the Developing Mouse. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/35616

Chicago Manual of Style (16th Edition):

Hanna, Daniel. “Sex and Strain Differences in Acute Hepatotoxic and Inflammatory Responses to Liver Procarcinogens in the Developing Mouse.” 2013. Masters Thesis, University of Toronto. Accessed May 20, 2019. http://hdl.handle.net/1807/35616.

MLA Handbook (7th Edition):

Hanna, Daniel. “Sex and Strain Differences in Acute Hepatotoxic and Inflammatory Responses to Liver Procarcinogens in the Developing Mouse.” 2013. Web. 20 May 2019.

Vancouver:

Hanna D. Sex and Strain Differences in Acute Hepatotoxic and Inflammatory Responses to Liver Procarcinogens in the Developing Mouse. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2019 May 20]. Available from: http://hdl.handle.net/1807/35616.

Council of Science Editors:

Hanna D. Sex and Strain Differences in Acute Hepatotoxic and Inflammatory Responses to Liver Procarcinogens in the Developing Mouse. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/35616


University of Helsinki

29. Mustonen, Merja. Introduction of high-content screening method for studying drug-induced mitochondrial dysfunction in hepatocytes.

Degree: Farmaceutiska fakulteten, 2011, University of Helsinki

 Lääkkeitä kehitettäessä on pitkään jo pyritty löytämään tehokkaampia, nopeampia ja kalliita eläinkokeita korvaavia toksisuuden tutkimismenetelmiä. High Content Screening (HCS)-laitteisto koostuu yksinkertaistetusti tietokoneeseen yhdistetystä fluoresenssimikroskoopista. Koneessa… (more)

Subjects/Keywords: High Content Screening (HCS); Mitochondria; Hepatotoxicity; Fluorescent Microscopy; mitokondrio; maksatoksisuus; fluoresoivat leimat; Farmakologi; Pharmacology; Farmakologia

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APA (6th Edition):

Mustonen, M. (2011). Introduction of high-content screening method for studying drug-induced mitochondrial dysfunction in hepatocytes. (Masters Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/26646

Chicago Manual of Style (16th Edition):

Mustonen, Merja. “Introduction of high-content screening method for studying drug-induced mitochondrial dysfunction in hepatocytes.” 2011. Masters Thesis, University of Helsinki. Accessed May 20, 2019. http://hdl.handle.net/10138/26646.

MLA Handbook (7th Edition):

Mustonen, Merja. “Introduction of high-content screening method for studying drug-induced mitochondrial dysfunction in hepatocytes.” 2011. Web. 20 May 2019.

Vancouver:

Mustonen M. Introduction of high-content screening method for studying drug-induced mitochondrial dysfunction in hepatocytes. [Internet] [Masters thesis]. University of Helsinki; 2011. [cited 2019 May 20]. Available from: http://hdl.handle.net/10138/26646.

Council of Science Editors:

Mustonen M. Introduction of high-content screening method for studying drug-induced mitochondrial dysfunction in hepatocytes. [Masters Thesis]. University of Helsinki; 2011. Available from: http://hdl.handle.net/10138/26646


University of Washington

30. Wahlin, Michelle Diane. Defining the Relationship between the Bioactivation of Lapatinib by CYP3A and Lapatinib-Induced Hepatotoxicity.

Degree: PhD, 2014, University of Washington

 Lapatinib was the first orally active dual tyrosine kinase inhibitor (TKI) of the epidermal growth factor receptor (EGFR, ErbB1) and human epidermal receptor 2 (HER2,… (more)

Subjects/Keywords: bioactivation; CYP3A4; CYP3A5; hepatotoxicity; lapatinib; metabolism; Pharmaceutical sciences; Chemistry; Toxicology; medicinal chemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wahlin, M. D. (2014). Defining the Relationship between the Bioactivation of Lapatinib by CYP3A and Lapatinib-Induced Hepatotoxicity. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/26061

Chicago Manual of Style (16th Edition):

Wahlin, Michelle Diane. “Defining the Relationship between the Bioactivation of Lapatinib by CYP3A and Lapatinib-Induced Hepatotoxicity.” 2014. Doctoral Dissertation, University of Washington. Accessed May 20, 2019. http://hdl.handle.net/1773/26061.

MLA Handbook (7th Edition):

Wahlin, Michelle Diane. “Defining the Relationship between the Bioactivation of Lapatinib by CYP3A and Lapatinib-Induced Hepatotoxicity.” 2014. Web. 20 May 2019.

Vancouver:

Wahlin MD. Defining the Relationship between the Bioactivation of Lapatinib by CYP3A and Lapatinib-Induced Hepatotoxicity. [Internet] [Doctoral dissertation]. University of Washington; 2014. [cited 2019 May 20]. Available from: http://hdl.handle.net/1773/26061.

Council of Science Editors:

Wahlin MD. Defining the Relationship between the Bioactivation of Lapatinib by CYP3A and Lapatinib-Induced Hepatotoxicity. [Doctoral Dissertation]. University of Washington; 2014. Available from: http://hdl.handle.net/1773/26061

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