subject:(Hemodynamics)

University of Cape Town

1. Resnekov, Leon Zundel. Synchronised capacitor discharge in the management of cardiac arrhythmias with particular reference to the haemodynamic significance of atrial systole.

Degree: Doctoral, Medicine, 1965, University of Cape Town

URL: http://hdl.handle.net/11427/31962

► In December 1962 a boy of 15 years was referred to the National Heart Hospital because of a ventricular tachycardia, which had proved resistant to… (more)

Subjects/Keywords: Hemodynamics

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APA (6^th Edition):

Resnekov, L. Z. (1965). Synchronised capacitor discharge in the management of cardiac arrhythmias with particular reference to the haemodynamic significance of atrial systole. (Doctoral Dissertation). University of Cape Town. Retrieved from http://hdl.handle.net/11427/31962

Chicago Manual of Style (16^th Edition):

Resnekov, Leon Zundel. “Synchronised capacitor discharge in the management of cardiac arrhythmias with particular reference to the haemodynamic significance of atrial systole.” 1965. Doctoral Dissertation, University of Cape Town. Accessed March 01, 2021. http://hdl.handle.net/11427/31962.

MLA Handbook (7^th Edition):

Resnekov, Leon Zundel. “Synchronised capacitor discharge in the management of cardiac arrhythmias with particular reference to the haemodynamic significance of atrial systole.” 1965. Web. 01 Mar 2021.

Vancouver:

Resnekov LZ. Synchronised capacitor discharge in the management of cardiac arrhythmias with particular reference to the haemodynamic significance of atrial systole. [Internet] [Doctoral dissertation]. University of Cape Town; 1965. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/11427/31962.

Council of Science Editors:

Resnekov LZ. Synchronised capacitor discharge in the management of cardiac arrhythmias with particular reference to the haemodynamic significance of atrial systole. [Doctoral Dissertation]. University of Cape Town; 1965. Available from: http://hdl.handle.net/11427/31962

University of Illinois – Urbana-Champaign

2. Kistler, Brandon. Acute effects of intradialytic nutrition supplementation on hemodynamics, treatment efficiency, and gastrointestinal symptoms in maintenance hemodialysis patients.

Degree: PhD, Kinesiology, 2015, University of Illinois – Urbana-Champaign

URL: http://hdl.handle.net/2142/89194

► Poor nutritional status is common and among the strongest predictors of mortality in patients undergoing maintenance hemodialysis (HD) treatment. Reduced dietary intake, especially on days… (more)

▼ Poor nutritional status is common and among the strongest predictors of mortality in patients undergoing maintenance hemodialysis (HD) treatment. Reduced dietary intake, especially on days in which patients undergo treatment, is one factor that contributes to this poor nutritional status. Providing nutritional supplements or allowing patients to eat during HD treatment can help restore dietary intake, improve nutritional status, and significantly reduce mortality in malnourished patients. However, in the United States this practice is frequently restricted due to concerns about patient safety. To better understand practice patterns related to eating during HD treatment we conducted an international survey. We received 73 responses representing clinics in six continents. Among this cohort, 61 of 73 clinicians (85%) were at clinics that allowed patients to eat during treatment and 53 (73%) were at clinics that provided food during treatment. Interestingly, none of the nine clinics from North America provided food during treatment. In the second part of this survey, we asked the 61 clinicians who were at clinics which allowed patients to eat during treatment about their experience with the six most-cited reasons to restrict eating during treatment using a four point scale. Clinicians responded that they observed choking (98%), reduced Kt/V (98%), infection control issues (96%), spills or pests (83%), gastrointestinal issues (71%), and hypotension (62%) either “rarely” or “never.” The results of our survey suggested that gastrointestinal (GI) issues were amongst the conditions most frequently attributed to eating during HD treatment. However, there have been no studies directly examining the effects of providing nutrition on these symptoms. One reason for this lack of data may have been the absence of a validated tool to measure GI symptoms. Therefore, we developed and validated a questionnaire to measure GI symptoms associated with a single HD treatment. Following a brief face validation with renal dietitians, we recruited 50 maintenance HD patients and administered our survey following a mid-week HD treatment. During the same treatment we measured dietary intake by diet recall. Three weeks later we repeated this process. In general, we found good agreement between items in each domain and repeatability among individual domains. Prevalence of GI symptoms during treatment (77.1%) was much higher than previously reported and associated with the intake of fat (r=.318, p = 0.027) and fiber (.386, p = 0.007) during treatment. At the same time as our validation study, we also sought to determine the effect that liquid supplements had on the symptoms commonly associated with eating during treatment. We used a within-subjects design (n=8) to compare standard HD treatment (HD) to a standard HD treatment in which patients ingested 30 grams of whey protein starting 30 minutes into their treatment (HD + Protein). We found no interaction between groups in any hemodynamic variable (p>0.05). However, there was a main-effect of… Advisors/Committee Members: Wilund, Kenneth (advisor), Wilund, Kenneth (Committee Chair), De Lisio, Mike (committee member), Chapman-Novakofski, Karen (committee member), Heffernan, Kevin (committee member), Ikizler, Alp (committee member).

Subjects/Keywords: Hemodynamics; Intradialytic Nutrition

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APA (6^th Edition):

Kistler, B. (2015). Acute effects of intradialytic nutrition supplementation on hemodynamics, treatment efficiency, and gastrointestinal symptoms in maintenance hemodialysis patients. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/89194

Chicago Manual of Style (16^th Edition):

Kistler, Brandon. “Acute effects of intradialytic nutrition supplementation on hemodynamics, treatment efficiency, and gastrointestinal symptoms in maintenance hemodialysis patients.” 2015. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed March 01, 2021. http://hdl.handle.net/2142/89194.

MLA Handbook (7^th Edition):

Kistler, Brandon. “Acute effects of intradialytic nutrition supplementation on hemodynamics, treatment efficiency, and gastrointestinal symptoms in maintenance hemodialysis patients.” 2015. Web. 01 Mar 2021.

Vancouver:

Kistler B. Acute effects of intradialytic nutrition supplementation on hemodynamics, treatment efficiency, and gastrointestinal symptoms in maintenance hemodialysis patients. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2015. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/2142/89194.

Council of Science Editors:

Kistler B. Acute effects of intradialytic nutrition supplementation on hemodynamics, treatment efficiency, and gastrointestinal symptoms in maintenance hemodialysis patients. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2015. Available from: http://hdl.handle.net/2142/89194

Rutgers University

3. Hegyi, Gina N., 1991-. Arterial hemodynamics and its interaction with the left ventricle in heart failure.

Degree: MS, Biomedical Engineering, 2015, Rutgers University

URL: https://rucore.libraries.rutgers.edu/rutgers-lib/48508/

► Monitoring heart-arterial system (LV-AS) interactions of cardiovascular patients is important in achieving successful diagnosis and treatment. This is particularly true in the cases of hypertension… (more)

▼ Monitoring heart-arterial system (LV-AS) interactions of cardiovascular patients is important in achieving successful diagnosis and treatment. This is particularly true in the cases of hypertension and heart failure. However, there has been limited quantitative hemodynamic analysis aimed at improving the outcome for the 70-million hypertensive patients and millions of heart failure patients. This thesis develops a model-based strategy to provide a comprehensive evaluation of the state of the vascular system and its coupling to the heart, and ultimately aims to distinguish patient groups of heart failure with preserved or reduced ejection fraction (HFpEF and HFrEF). The first aspect of this study is analysis of arterial hemodynamics from animal experimental data collected during normotensive, vasodilation, and induced hypertension conditions. Total arterial compliance (C), peripheral resistance (Rs), and aortic characteristic impedance (Z0) based on the 3-element Windkessel model were obtained, together with frequency-domain impedance, wave reflection information, and parameters for assessing global cardiac function. Subsequently, clinical human data were collected and analyzed, focusing on the LV-AS interactions. We identified critical parameters in LV-AS interaction, i.e. effective arterial system elastance, Ea, LV systolic elastance, Emax, LV-AS coupling index, k (=Ea/Emax), and C. Results characterized hypertension by a significantly reduced C and increased Rs, confirmed by increased vascular impedance and wave reflections. Unloading the heart reversed these observations via vasodilator treatment. For human studies, we found C to be more effective than Ea in distinguishing groups of HFpEF and HFrEF patients. It was found that HFpEF patients tended to have a relatively independent coupling index, k, with respect to arterial parameter changes. The findings in this work can be incorporated into larger scale clinical studies that implement the use of pulse waveform analysis and the identified critical LV-AS interaction parameters to improve patient treatment and outcome. Advisors/Committee Members: Li, John K-J. (chair).

Subjects/Keywords: Heart failure; Hemodynamics

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APA (6^th Edition):

Hegyi, Gina N., 1. (2015). Arterial hemodynamics and its interaction with the left ventricle in heart failure. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/48508/

Chicago Manual of Style (16^th Edition):

Hegyi, Gina N., 1991-. “Arterial hemodynamics and its interaction with the left ventricle in heart failure.” 2015. Masters Thesis, Rutgers University. Accessed March 01, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/48508/.

MLA Handbook (7^th Edition):

Hegyi, Gina N., 1991-. “Arterial hemodynamics and its interaction with the left ventricle in heart failure.” 2015. Web. 01 Mar 2021.

Vancouver:

Hegyi, Gina N. 1. Arterial hemodynamics and its interaction with the left ventricle in heart failure. [Internet] [Masters thesis]. Rutgers University; 2015. [cited 2021 Mar 01]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/48508/.

Council of Science Editors:

Hegyi, Gina N. 1. Arterial hemodynamics and its interaction with the left ventricle in heart failure. [Masters Thesis]. Rutgers University; 2015. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/48508/

University of Utah

4. Doig, Alexa K. Graphical cardiovascular display for hemodynamic monitoring;.

Degree: PhD, Nursing;, 2006, University of Utah

URL: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/573/rec/595

► During emergent situations critical care nurses must recognize changes in the patient's condition, rapidly select relevant hemodynamic variables, then interpret the data in order to… (more)

▼ During emergent situations critical care nurses must recognize changes in the patient's condition, rapidly select relevant hemodynamic variables, then interpret the data in order to intervene appropriately. Studies in anesthesia have shown that graphical monitoring displays have the potential to improve detection and management of adverse physiological events. Despite the fact that nurses are the primary users of patient monitoring technologies, the application of graphical displays has not been well explored within the domain of nursing practice. The first aim of this research was to describe the cognitive aspects of the hemodynamic monitoring task of critical care nurses. The data, collected during interviews with 13 nurses, supported the use of graphical displays in hemodynamic monitoring and the findings were used as the basis for a series of preliminary display design strategy recommendations. The second aim was to compare the performance of critical care nurses using a new integrated graphical cardiovascular display versus a traditional numerical-waveform display to interpret hemodynamic data using a computerized, scenario-based test (n = 15/group). Although use of the graphical display did not improve accuracy of data interpretation, it did not significantly reduce it, despite limited training on the new graphical display technology. Response times among the two display conditions were equivalent. On subjective measures, the graphical display was favorably rated in terms of acceptance and usability. Interviews conducted following the display testing sessions found that nurses using the graphical display used fewer variables in their assessment, as well as more conceptual but less precise language during the interview. Nurses who used imprecise language scored lower on the scenario-based test, regardless of display type. The most common sources of error among nurses in both display conditions were inattention to key variables, misinterpretation of variables, and being misled by the patient's history. Graphical displays for hemodynamic monitoring may be able to enhance nursing practice with relevant, cue-based design development and additional training prior to testing. Advancing the design of monitoring displays for use by critical care nurses also requires further study of the cognitive tasks involved in patient monitoring and the sources of error during data interpretation.

Subjects/Keywords: Nursing; Hemodynamics

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APA (6^th Edition):

Doig, A. K. (2006). Graphical cardiovascular display for hemodynamic monitoring;. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/573/rec/595

Chicago Manual of Style (16^th Edition):

Doig, Alexa K. “Graphical cardiovascular display for hemodynamic monitoring;.” 2006. Doctoral Dissertation, University of Utah. Accessed March 01, 2021. http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/573/rec/595.

MLA Handbook (7^th Edition):

Doig, Alexa K. “Graphical cardiovascular display for hemodynamic monitoring;.” 2006. Web. 01 Mar 2021.

Vancouver:

Doig AK. Graphical cardiovascular display for hemodynamic monitoring;. [Internet] [Doctoral dissertation]. University of Utah; 2006. [cited 2021 Mar 01]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/573/rec/595.

Council of Science Editors:

Doig AK. Graphical cardiovascular display for hemodynamic monitoring;. [Doctoral Dissertation]. University of Utah; 2006. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/573/rec/595

Rochester Institute of Technology

5. Varble, Nicole. A Hemodynamic investigation of a complete arteriovenous model of the arm, arteriovenous fistula, and distal revascularization and interval ligation.

Degree: Mechanical Engineering, 2011, Rochester Institute of Technology

URL: https://scholarworks.rit.edu/theses/5824

► One of the largest problems facing vascular surgeons today is malfunctioning arteriovenous fistulas (AVF) in patients on hemodialysis. A radial- cephalic AVF is a vessel… (more)

▼ One of the largest problems facing vascular surgeons today is malfunctioning arteriovenous fistulas (AVF) in patients on hemodialysis. A radial- cephalic AVF is a vessel in the arm that directs blood flow from the high pressure arteries to the low pressure veins and is put in place so blood can be withdrawn from a patient at a point where high volume blood flow occurs. After a period of maturation, the fistula often expands to adapt for the increase flow and can therefore be accessed repeatedly for hemodialysis. However, in a small percentage of cases, access related steal can lead to ischemia of the hand and threaten the patient with gangrene and amputation. This is a recognizable problem and is currently being treated with a method called Distal Revascularization and Interval Ligation (DRIL) in which a vast majority of cases see improved blood flow and pressure to the digits all while maintaining the AVF. The DRIL procedure has been widely accepted as an effective method to prevent the onset of steal, however the flow mechanisms and reasoning behind its success is not well understood. Thus far, attempts of explaining the procedure have been sparse and incomplete, often disregarding factors such as the compliance of the vessels and the pressures and flows on the venous side of the vasculature. To study this problem, a physical model was built using tubing with comparable resistances and compliances to that of native vessels. The native circulation was first built, including the venous side, which will provide a unique baseline for the study. An emphasis was placed on creating a system with physiologic pressures on both the arterial side and venous side. Once the model successfully represented the native blood flow, it was adapted for an AVF. Upon this adaptation, variations of radius, length, and position were tested. Furthermore, this model can easily be adapted to model hypertensive patients, much like those on hemodialysis and can offer additional insight as to if those patients are more susceptible to access related ischemia. Observations of pressures and flows led to a comprehensive study of the hemodynamics of the native arm circulation, an AVF and the DRIL procedure. When the model is integrated into the hemodynamic simulator loop, physiologic pressures have been produced in the arterial side of 125/55 mmHg and a damped waveform with a mean pressure of 18 mmHg was found on the venous side. Mean aortic flow was 4.7 L/min. Advisors/Committee Members: Day, Steven.

Subjects/Keywords: AVF; DRIL; Engineering; Hemodynamics; Vascular

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APA (6^th Edition):

Varble, N. (2011). A Hemodynamic investigation of a complete arteriovenous model of the arm, arteriovenous fistula, and distal revascularization and interval ligation. (Thesis). Rochester Institute of Technology. Retrieved from https://scholarworks.rit.edu/theses/5824

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Varble, Nicole. “A Hemodynamic investigation of a complete arteriovenous model of the arm, arteriovenous fistula, and distal revascularization and interval ligation.” 2011. Thesis, Rochester Institute of Technology. Accessed March 01, 2021. https://scholarworks.rit.edu/theses/5824.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Varble, Nicole. “A Hemodynamic investigation of a complete arteriovenous model of the arm, arteriovenous fistula, and distal revascularization and interval ligation.” 2011. Web. 01 Mar 2021.

Vancouver:

Varble N. A Hemodynamic investigation of a complete arteriovenous model of the arm, arteriovenous fistula, and distal revascularization and interval ligation. [Internet] [Thesis]. Rochester Institute of Technology; 2011. [cited 2021 Mar 01]. Available from: https://scholarworks.rit.edu/theses/5824.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Varble N. A Hemodynamic investigation of a complete arteriovenous model of the arm, arteriovenous fistula, and distal revascularization and interval ligation. [Thesis]. Rochester Institute of Technology; 2011. Available from: https://scholarworks.rit.edu/theses/5824

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Cornell University

6. Lindsey, Stephanie. Hemodynamic Regulation Of Cardiac Outflow Morphogenesis.

Degree: PhD, Biomedical Engineering, 2016, Cornell University

URL: http://hdl.handle.net/1813/43703

► Throughout development, blood flow guides cardiac morphogenesis, sculpting tissue by promoting growth in response to increased mechanical demands. Alterations in flow patterns during critical stages… (more)

▼ Throughout development, blood flow guides cardiac morphogenesis, sculpting tissue by promoting growth in response to increased mechanical demands. Alterations in flow patterns during critical stages of development may therefore lead to adverse tissue remodeling and subsequent functional cardiac defects. Distinguishing the specific effects due to hemodynamics and genetic mutations is a current challenge, as the role of hemodynamics in outflow tract and pharyngeal arch artery morphogenesis is poorly understood. There exists a need for more clinically relevant animal models that allow for the study of disease pathogenesis both structurally and molecularly. My work begins to delineate the effects of structural changes resulting from abnormal hemodynamic patterning and elucidate their effects in the creation of congenital heart defects through a combination of experimental interventions and computational modeling. Two- photon microscopy guided femtosecond laser pulses were used to nucleate and control the growth of cavitation bubbles within developing outflow vessels without disturbing surrounding tissues. These cavitation bubbles temporarily occluded the vessel, while a more stable occlusion while a more stable occlusion was formed by ablating the circulating thrombocytes that accumulated behind the bubble. Using this approach, I examined the effects of PAA vessel occlusions on embryonic viability, hemodynamic rearrangement, and downstream outflow tract morphogenesis (Chapter 3). A zero-dimensional (0D) electric analog model that allows for global tuning of the embryo's vasculature relative to the arches was developed (Chapter 4). A 3D-0D model was used to characterize natural variation in day3, day4, and day 5 arch artery pressure, flow, and shape changes. These 0D bounds served as a basis for prediction of flow distribution to the cranial and caudal outlets when switching between normal and aberrant flow (Chapter 5). Results revealed distinct morphology dynamics for day3, day4, and day 5 geometries, as well as natural shifts in arch artery dominance at different stages and across individual days. Immediate remodeling of the arch artery vasculature took place following day 3 experimental occlusions. In some embryos, that remodeling worked to lessen the severity of increased pressure magnitude in the cranial part of the aortic sac. My findings contribute a more detailed picture of arch artery growth and adaptation across a critical window of development. Advisors/Committee Members: Butcher,Jonathan T. (chair), Noden,Drew Morrison (committee member), Schaffer,Chris (committee member), Vignon-Clementel,Irene (committee member).

Subjects/Keywords: hemodynamics; cardiogenesis; mathematical modeling

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APA (6^th Edition):

Lindsey, S. (2016). Hemodynamic Regulation Of Cardiac Outflow Morphogenesis. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/43703

Chicago Manual of Style (16^th Edition):

Lindsey, Stephanie. “Hemodynamic Regulation Of Cardiac Outflow Morphogenesis.” 2016. Doctoral Dissertation, Cornell University. Accessed March 01, 2021. http://hdl.handle.net/1813/43703.

MLA Handbook (7^th Edition):

Lindsey, Stephanie. “Hemodynamic Regulation Of Cardiac Outflow Morphogenesis.” 2016. Web. 01 Mar 2021.

Vancouver:

Lindsey S. Hemodynamic Regulation Of Cardiac Outflow Morphogenesis. [Internet] [Doctoral dissertation]. Cornell University; 2016. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/1813/43703.

Council of Science Editors:

Lindsey S. Hemodynamic Regulation Of Cardiac Outflow Morphogenesis. [Doctoral Dissertation]. Cornell University; 2016. Available from: http://hdl.handle.net/1813/43703

Clemson University

7. Ghobrial, Nardine M. Using Heparin-Coated Nanoparticles in the Treatment of Neointimal Hyperplasia.

Degree: PhD, Bioengineering, 2020, Clemson University

URL: https://tigerprints.clemson.edu/all_dissertations/2674

► The use of stents in the treatment of atherosclerosis leads to a potential risk of restenosis, caused by neointimal hyperplasia. Neointimal hyperplasia is mainly… (more)

▼ The use of stents in the treatment of atherosclerosis leads to a potential risk of restenosis, caused by neointimal hyperplasia. Neointimal hyperplasia is mainly caused by an injury to the endothelial layer of the blood vessel followed by the proliferation of smooth muscle cells into the lumen of the blood vessel. To address this, we designed a magnetically-guided drug delivery system to locally deliver heparin to a stented artery. The nanoparticles were synthesized, characterized, and tested on relevant human cell lines. The particles were non-toxic to human smooth muscle cells, endothelial cells, and fibroblasts. They reduced the proliferation of the smooth muscle cells and increased the proliferation of endothelial cells at concentrations as low as 10 μg/mL. The particles also shifted the smooth muscle cells from their synthetic phenotype to their contractile phenotype. The capture of the nanoparticles by the stent struts, under relevant magnetic field and blood velocity was modeled using COMSOL Multiphysics. The coronary artery was modeled using a 2D axisymmetric model with stainless steel stent struts. A Magnetic field of 1 T was applied to magnetize the stent struts. Three different strut geometries were compared for their effect of the capture efficiency. The model had a capture efficiency 0f 34-42%, which is comparable to models using the same particle sizes. Ex vivo organ culture studies using porcine right coronary arteries were performed. The arteries were conditioned either statically in cell culture flasks or dynamically in an organ culture bioreactor. Nanoparticles reduced intimal thickening in and expressed contractile properties in the treated arteries compared to the controls. We were successfully able to synthesize heparin-coated magnetic nanoparticles and achieve high heparin loading. Particle capture efficiency around the stent in the ex vivo porcine artery model was found to be similar to that predicted by the computational model. Consistent with the prior results of systemic heparin delivery, the nanoparticles reduce the proliferation and dedifferentiation of vascular smooth muscle cells while promoting endothelialization, both in vitro and ex vivo. Thus, these particles may be a promising treatment option for neointimal hyperplasia. . Advisors/Committee Members: Delphine Dean, Olin T Mefford, Jeoungsoo Lee, Ulf Schiller.

Subjects/Keywords: Cardiovascular; Hemodynamics; Heparin; Magnetic; Nanoparticles

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APA (6^th Edition):

Ghobrial, N. M. (2020). Using Heparin-Coated Nanoparticles in the Treatment of Neointimal Hyperplasia. (Doctoral Dissertation). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_dissertations/2674

Chicago Manual of Style (16^th Edition):

Ghobrial, Nardine M. “Using Heparin-Coated Nanoparticles in the Treatment of Neointimal Hyperplasia.” 2020. Doctoral Dissertation, Clemson University. Accessed March 01, 2021. https://tigerprints.clemson.edu/all_dissertations/2674.

MLA Handbook (7^th Edition):

Ghobrial, Nardine M. “Using Heparin-Coated Nanoparticles in the Treatment of Neointimal Hyperplasia.” 2020. Web. 01 Mar 2021.

Vancouver:

Ghobrial NM. Using Heparin-Coated Nanoparticles in the Treatment of Neointimal Hyperplasia. [Internet] [Doctoral dissertation]. Clemson University; 2020. [cited 2021 Mar 01]. Available from: https://tigerprints.clemson.edu/all_dissertations/2674.

Council of Science Editors:

Ghobrial NM. Using Heparin-Coated Nanoparticles in the Treatment of Neointimal Hyperplasia. [Doctoral Dissertation]. Clemson University; 2020. Available from: https://tigerprints.clemson.edu/all_dissertations/2674

Texas A&M University

8. Nguyen, Phuc Hoang. Integrating Biomechanics, Hemodynamics, and Vascular Adaptation to Relate Mechanisms of Vascular Adaptation to Arterial Pulsatile Pressure in Health and Disease.

Degree: PhD, Biomedical Sciences, 2014, Texas A&M University

URL: http://hdl.handle.net/1969.1/153567

► The inherent complexity of the mammalian systemic arterial system has presented numerous challenges to relating basic vascular biology to clinically-relevant derangements of blood pressures and… (more)

▼ The inherent complexity of the mammalian systemic arterial system has presented numerous challenges to relating basic vascular biology to clinically-relevant derangements of blood pressures and flows. The field of biomechanics has identified how local changes in pulsatile blood pressures and flows lead to changes in local endothelial shear stress and circumferential wall stress. The field of mechanobiology has identified how local changes in wall circumferential stress and endothelial shear stress lead to changes in arterial radii, wall thicknesses and stiffnesses. The field of pulsatile hemodynamics has identified how changes in local radii, wall thicknesses and stiffnesses lead to changes in the complex distributions of pressures and flows throughout an arterial network. These three fields have primarily been studied in isolation, and yet the properties of a single vessel emerge from the interaction of these three processes. The effect of adaptation of one artery on hemodynamics, stress, and structure of all other vessels in the network makes the arterial system a complex adaptive system that is difficult to study experimentally. This dissertation addresses this unmet need by integrating hemodynamics, vessel mechanics, and vascular adaptation by developing a novel framework with mathematical models at different scales. Allowing arteries simultaneously to adapt to mechanical stresses in a computational model of the human systemic arterial system, the present work illustrated that simple arterial adaptation to wall circumferential and endothelial shear stresses are sufficient to explain nine salient features of the cardiovascular system when traversing away from the aortic root towards the peripheral arteries: decrease in lumen radii, wall thicknesses, vessel compliances, shear stresses, wall stresses and pulsatile flows, and increase in wall stiffnesses, pulse wave velocities, and pulsatile pressures. In addition, it revealed that pulse pressure homeostasis emerges to mechanical perturbations such as reduced ejection fraction, increased peripheral resistance and aortic coarctation. Finally, it illustrated how changes in sensitivity of arterial adaptation to pulsatile wall stress can lead to manifestations of disease states such as increased pulse wave velocity and isolated systolic hypertension. The governing principles leading to the emergence of complex, adaptive behavior in the systemic arterial system have thus been identified. Advisors/Committee Members: Quick, Christopher M. (advisor), Stewart, Randolph H. (committee member), Criscione, John C. (committee member), Dongaonkar, Ranjeet M. (committee member).

Subjects/Keywords: hemodynamics; vascular adaptation; biomechanics

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APA (6^th Edition):

Nguyen, P. H. (2014). Integrating Biomechanics, Hemodynamics, and Vascular Adaptation to Relate Mechanisms of Vascular Adaptation to Arterial Pulsatile Pressure in Health and Disease. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/153567

Chicago Manual of Style (16^th Edition):

Nguyen, Phuc Hoang. “Integrating Biomechanics, Hemodynamics, and Vascular Adaptation to Relate Mechanisms of Vascular Adaptation to Arterial Pulsatile Pressure in Health and Disease.” 2014. Doctoral Dissertation, Texas A&M University. Accessed March 01, 2021. http://hdl.handle.net/1969.1/153567.

MLA Handbook (7^th Edition):

Nguyen, Phuc Hoang. “Integrating Biomechanics, Hemodynamics, and Vascular Adaptation to Relate Mechanisms of Vascular Adaptation to Arterial Pulsatile Pressure in Health and Disease.” 2014. Web. 01 Mar 2021.

Vancouver:

Nguyen PH. Integrating Biomechanics, Hemodynamics, and Vascular Adaptation to Relate Mechanisms of Vascular Adaptation to Arterial Pulsatile Pressure in Health and Disease. [Internet] [Doctoral dissertation]. Texas A&M University; 2014. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/1969.1/153567.

Council of Science Editors:

Nguyen PH. Integrating Biomechanics, Hemodynamics, and Vascular Adaptation to Relate Mechanisms of Vascular Adaptation to Arterial Pulsatile Pressure in Health and Disease. [Doctoral Dissertation]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/153567

University of Toronto

9. Tan, Luke. A Newborn Sex-related Response to Hyperoxia.

Degree: 2012, University of Toronto

URL: http://hdl.handle.net/1807/33867

►

Exposure to hyperoxia has been shown to have detrimental effects on newborn hemodynamics and antioxidant activities in males. These effects are unknown in the newborn… (more)

Subjects/Keywords: Hyperoxia; Hemodynamics; sex-difference; 0719

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APA (6^th Edition):

Tan, L. (2012). A Newborn Sex-related Response to Hyperoxia. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33867

Chicago Manual of Style (16^th Edition):

Tan, Luke. “A Newborn Sex-related Response to Hyperoxia.” 2012. Masters Thesis, University of Toronto. Accessed March 01, 2021. http://hdl.handle.net/1807/33867.

MLA Handbook (7^th Edition):

Tan, Luke. “A Newborn Sex-related Response to Hyperoxia.” 2012. Web. 01 Mar 2021.

Vancouver:

Tan L. A Newborn Sex-related Response to Hyperoxia. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/1807/33867.

Council of Science Editors:

Tan L. A Newborn Sex-related Response to Hyperoxia. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33867

10. Moberg, Niklas. Hemodynamic Model of the Cardiovascular System during Valsalva Maneuver and Orthostatic Changes.

Degree: Radiation Physics, 2011, Umeå University

URL: http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-50044

► The goal of the Master's Thesis was to extend an existing cardiovascular model to include the mechanics of the lung, thus allowing to simulate… (more)

Subjects/Keywords: Hemodynamics; model; Valsalva; cardiovascular

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Moberg, N. (2011). Hemodynamic Model of the Cardiovascular System during Valsalva Maneuver and Orthostatic Changes. (Thesis). Umeå University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-50044

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Moberg, Niklas. “Hemodynamic Model of the Cardiovascular System during Valsalva Maneuver and Orthostatic Changes.” 2011. Thesis, Umeå University. Accessed March 01, 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-50044.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Moberg, Niklas. “Hemodynamic Model of the Cardiovascular System during Valsalva Maneuver and Orthostatic Changes.” 2011. Web. 01 Mar 2021.

Vancouver:

Moberg N. Hemodynamic Model of the Cardiovascular System during Valsalva Maneuver and Orthostatic Changes. [Internet] [Thesis]. Umeå University; 2011. [cited 2021 Mar 01]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-50044.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Moberg N. Hemodynamic Model of the Cardiovascular System during Valsalva Maneuver and Orthostatic Changes. [Thesis]. Umeå University; 2011. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-50044

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Colorado State University

11. Moore, Brandon L. Influence of anatomic valve conditions and coronary flow on aortic sinus hemodynamics.

Degree: PhD, Mechanical Engineering, 2015, Colorado State University

URL: http://hdl.handle.net/10217/170289

► Heart disease is the leading cause of death in the US, and aortic valve stenosis represents a significant portion of this disease. While the specific… (more)

▼ Heart disease is the leading cause of death in the US, and aortic valve stenosis represents a significant portion of this disease. While the specific causes of stenosis are not entirely clear, its development has been strongly linked to mechanical factors such as localized solid and fluid stresses and strains, especially on the aortic side of the valve leaflets. These mechanical cues can be tied to valvular hemodynamics, however the factors regulating these hemodynamics are relatively unknown. Therefore, the overarching hypothesis of this research is that aortic valve sinus hemodynamics are regulated by anatomic valve conditions and presence of coronary flow. This hypothesis is explored through three specific aims: 1) to develop methodologies for quantifying hemodynamics within the aortic sinuses, 2) to characterize the differences in native valve flow patterns that occur due to patient and sinus variability, and 3) to evaluate the hemodynamic impacts of different prosthetic aortic valve implantations. In this work, experimental methods have been developed to study a broad range of aortic valve conditions, and computational models were also employed to validate and enhance experimental findings. An in vitro setup is presented using a surgical bioprosthesis as a native aortic valve model, while additional valve implantations were also tested. Physiological pressures and flow rates were imposed across these valves via an in-house pumping loop, which included a novel coronary flow branch. Two-dimensional time-resolved particle image velocimetry (PIV) protocols were developed and employed to analyze sinus vorticity dynamics. Computationally, both 2D and 3D simulations were run in ANSYS Fluent to enhance experimental findings. Results from this research demonstrate that aortic sinus hemodynamics are indeed regulated by anatomic valve conditions and coronary flow. From a clinical perspective, average valve geometric parameters tend to produce hemodynamics that are least likely to initiate disease than those near the upper or lower anatomical limits. Coronary flow was likewise found to increase sinus velocities and shear stresses near the leaflets, which is also beneficial for valve health. The prosthetic valves tested – transcatheter and sutureless – both severely limited sinus perfusion, which could help explain an increased risk of thrombus formation in the transcatheter case and suggests similar risk for sutureless valves. These findings could help educate clinicians on proper courses of treatment based on patient-specific valve parameters, and could also provide useful information for engineers when designing new valve prostheses. Advisors/Committee Members: Dasi, Lakshmi Prasad (advisor), Gao, Xinfeng (advisor), Kirkpatrick, Allan (committee member), Orton, Christopher (committee member).

Subjects/Keywords: aortic valve; cardiovascular; heart; hemodynamics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Moore, B. L. (2015). Influence of anatomic valve conditions and coronary flow on aortic sinus hemodynamics. (Doctoral Dissertation). Colorado State University. Retrieved from http://hdl.handle.net/10217/170289

Chicago Manual of Style (16^th Edition):

Moore, Brandon L. “Influence of anatomic valve conditions and coronary flow on aortic sinus hemodynamics.” 2015. Doctoral Dissertation, Colorado State University. Accessed March 01, 2021. http://hdl.handle.net/10217/170289.

MLA Handbook (7^th Edition):

Moore, Brandon L. “Influence of anatomic valve conditions and coronary flow on aortic sinus hemodynamics.” 2015. Web. 01 Mar 2021.

Vancouver:

Moore BL. Influence of anatomic valve conditions and coronary flow on aortic sinus hemodynamics. [Internet] [Doctoral dissertation]. Colorado State University; 2015. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/10217/170289.

Council of Science Editors:

Moore BL. Influence of anatomic valve conditions and coronary flow on aortic sinus hemodynamics. [Doctoral Dissertation]. Colorado State University; 2015. Available from: http://hdl.handle.net/10217/170289

University of Southern California

12. Henley, Brandon C. Parametric and nonparametric modeling of cerebral hemodynamics.

Degree: PhD, Biomedical Engineering, 2016, University of Southern California

URL: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/639702/rec/4918

► This manuscript focuses on the application of mathematical modeling to the study of cerebral hemodynamics. The central goal is to contribute to the pursuit of… (more)

▼ This manuscript focuses on the application of mathematical modeling to the study of cerebral hemodynamics. The central goal is to contribute to the pursuit of providing a reliable and practical approach to the management of clinical conditions relating to cerebral vascular and cognitive dysfunction. In particular, we study two fundamental processes of cerebral hemodynamics, cerebral autoregulation (CA) and vasomotor reactivity (VR) to CO₂ tension, using modeling techniques that provide quantitative information about the two processes. Quantitative assessment of CA and VR in humans serves as an important modeling goal, due to the observed association between cerebrovascular structure and function in the outcome of head injury, hypertension, stroke, vascular dementia, Alzheimer’s disease, and other forms of cognitive impairment. The availability of non-invasive measurements of relevant hemodynamic data has provided a means to obtain objective measures of CA and VR. It is believed that the relationship between time variations in cerebral perfusion pressure (CPP) and cerebral blood flow velocity can reflect relevant information about the status of CA in humans. In addition, it is well-known that CO₂ tension significantly influences the cerebral vasculature under physiological conditions, and the relationship between variations in CO₂ tension and cerebral perfusion is accepted as an approximation of the status of VR. Mathematical models of various forms have been employs as the methodological framework in which information about cerebral vascular status is summarized, analyzed, and interpreted. These models can be classified generally as “hypothesis-based” (or mechanistic) and “data-based” (or inductive). In the hypothesis-based approach, parametric models are constructed in the form of differential and/or algebraic equations from first hemodynamic principles in attempt to explicate the observed data. The main benefit of these models is the ability to derive physiological meaning, since the parameters which characterize the model are given meaning a priori. However, one important challenge is the virtually unavoidable oversimplification of the true depth of system, risking an incomplete and superficial picture of the actual system. This disadvantage can be supplemented by the use of inductive models, which are not hindered by simplifying assumptions about the system, but are instead obtained directly from measured data. Thus the data-based models are nonparametric in nature and their depth of the system depends on the richness of the data. These models are typically much easier to obtain (using a computer), and knowledge about the system is not required. However, the challenge of the data-based approach is the difficulty in achieving clear physiological interpretation for the model, which says nothing explicitly about the system. Advisors/Committee Members: Marmarelis, Vasilis (Committee Chair), Nation, Daniel (Committee Member), Khoo, Michael (Committee Member), King, Kevin (Committee Member).

Subjects/Keywords: cerebral autoregulation; vasomotor reactivity; hemodynamics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Henley, B. C. (2016). Parametric and nonparametric modeling of cerebral hemodynamics. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/639702/rec/4918

Chicago Manual of Style (16^th Edition):

Henley, Brandon C. “Parametric and nonparametric modeling of cerebral hemodynamics.” 2016. Doctoral Dissertation, University of Southern California. Accessed March 01, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/639702/rec/4918.

MLA Handbook (7^th Edition):

Henley, Brandon C. “Parametric and nonparametric modeling of cerebral hemodynamics.” 2016. Web. 01 Mar 2021.

Vancouver:

Henley BC. Parametric and nonparametric modeling of cerebral hemodynamics. [Internet] [Doctoral dissertation]. University of Southern California; 2016. [cited 2021 Mar 01]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/639702/rec/4918.

Council of Science Editors:

Henley BC. Parametric and nonparametric modeling of cerebral hemodynamics. [Doctoral Dissertation]. University of Southern California; 2016. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/639702/rec/4918

McMaster University

13. Noguchi, Kenneth. Arterial Stiffness and Central Hemodynamic Response and Recovery in Individuals Post-Stroke.

Degree: MSc, 2020, McMaster University

URL: http://hdl.handle.net/11375/25806

►

Background. Stroke affects over 80 million individuals worldwide. Elevated arterial stiffness has emerged as a novel independent risk marker for stroke. While arterial stiffness is… (more)

▼

Background. Stroke affects over 80 million individuals worldwide. Elevated arterial stiffness has emerged as a novel independent risk marker for stroke. While arterial stiffness is improved after chronic aerobic training, a single bout of aerobic exercise leads to transient increases that typically resolve within 5 minutes of recovery. Elevated arterial stiffness may persist for up to 30 minutes following exercise in populations with cardiovascular disease. However, no study has examined the effect of acute aerobic exercise on arterial stiffness and central hemodynamics in individuals with stroke. Moreover, no study has explored the clinical significance of these responses. Objectives. The primary objective of this thesis was to characterize the response and recovery of arterial stiffness and central hemodynamics to peak aerobic exercise in individuals ≥ 6 months post-stroke. The secondary objective was to explore the relationships between the exercise response and recovery of arterial stiffness and central hemodynamics, with cardiorespiratory fitness and walking ability. Results. This cross-sectional study recruited 10 adults with stroke (mean ± SD age=56.9 ± 11.8; median [IQR]= 2.9 [1.9] years post-stroke; n=4 females). After peak aerobic exercise, cfPWV increased from rest and remained elevated for 20 minutes (p<0.05). Heart rate increased and remained elevated for 10 minutes post-exercise (p<0.05), while systolic blood pressure decreased and remained reduced for 15 minutes (p<0.05). Positive associations were found between cardiorespiratory fitness and heart rate reserve (r=0.74, p=0.02), and with each phase of heart rate recovery (HR60s r=0.80, p=0.005, HR120s r=0.79, p=0.006; HR300s r=0.72, p=0.02; and HR600s r=0.75, p=0.01). There were no relationships between response and recovery of hemodynamic variables with walking ability. Conclusion. Individuals with chronic stroke may have impaired arterial stiffness and heart rate recovery following peak aerobic exercise. Moreover, heart rate reserve and all phases of heart rate recovery were related to cardiorespiratory fitness, but not walking ability.

Thesis

Master of Science Rehabilitation Science (MSc)

Arterial stiffness has been recently identified as an important risk marker for stroke. Aerobic exercise reduces the risk of stroke by lowering arterial stiffness. But during exercise, there is an increase in arterial stiffness that usually subsides by 5 minutes. Lengthy exposure to arterial stiffness can cause damage to organs like the kidneys and liver. The purpose of this thesis was to measure the arterial stiffness and cardiovascular response to exercise in people with stroke. We also studied the relationship between the responses, fitness and walking ability. Ten people with stroke participated in this study. After aerobic exercise, arterial stiffness stayed high above resting levels and did not recover after 20 minutes. Also, heart rate recovery was related to fitness but not walking ability. This study tells us that people with stroke have an…

Advisors/Committee Members: Tang, Ada, Rehabilitation Science.

Subjects/Keywords: Stroke; Arterial Stiffness; Hemodynamics; Exercise

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Noguchi, K. (2020). Arterial Stiffness and Central Hemodynamic Response and Recovery in Individuals Post-Stroke. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/25806

Chicago Manual of Style (16^th Edition):

Noguchi, Kenneth. “Arterial Stiffness and Central Hemodynamic Response and Recovery in Individuals Post-Stroke.” 2020. Masters Thesis, McMaster University. Accessed March 01, 2021. http://hdl.handle.net/11375/25806.

MLA Handbook (7^th Edition):

Noguchi, Kenneth. “Arterial Stiffness and Central Hemodynamic Response and Recovery in Individuals Post-Stroke.” 2020. Web. 01 Mar 2021.

Vancouver:

Noguchi K. Arterial Stiffness and Central Hemodynamic Response and Recovery in Individuals Post-Stroke. [Internet] [Masters thesis]. McMaster University; 2020. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/11375/25806.

Council of Science Editors:

Noguchi K. Arterial Stiffness and Central Hemodynamic Response and Recovery in Individuals Post-Stroke. [Masters Thesis]. McMaster University; 2020. Available from: http://hdl.handle.net/11375/25806

Université Catholique de Louvain

14. De Magnee, Catherine. Liver and systemic hemodynamics in cirrhotic children : contributions to the physiopathology and to the surgical algorithm in pediatric liver transplantation.

Degree: 2018, Université Catholique de Louvain

URL: http://hdl.handle.net/2078.1/199568

►

Liver and systemic hemodynamics are altered in cirrhotic adults. This work attempts to determine if these hemodynamic parameters are perturbed in cirrhotic children as in… (more)

Subjects/Keywords: Liver hemodynamics; Children; Cirrhosis; Pediatric liver transplantation; Systemic hemodynamics

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APA (6^th Edition):

De Magnee, C. (2018). Liver and systemic hemodynamics in cirrhotic children : contributions to the physiopathology and to the surgical algorithm in pediatric liver transplantation. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/199568

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

De Magnee, Catherine. “Liver and systemic hemodynamics in cirrhotic children : contributions to the physiopathology and to the surgical algorithm in pediatric liver transplantation.” 2018. Thesis, Université Catholique de Louvain. Accessed March 01, 2021. http://hdl.handle.net/2078.1/199568.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

De Magnee, Catherine. “Liver and systemic hemodynamics in cirrhotic children : contributions to the physiopathology and to the surgical algorithm in pediatric liver transplantation.” 2018. Web. 01 Mar 2021.

Vancouver:

De Magnee C. Liver and systemic hemodynamics in cirrhotic children : contributions to the physiopathology and to the surgical algorithm in pediatric liver transplantation. [Internet] [Thesis]. Université Catholique de Louvain; 2018. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/2078.1/199568.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

De Magnee C. Liver and systemic hemodynamics in cirrhotic children : contributions to the physiopathology and to the surgical algorithm in pediatric liver transplantation. [Thesis]. Université Catholique de Louvain; 2018. Available from: http://hdl.handle.net/2078.1/199568

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of Zambia

15. Mubyana, Maswabi Mukela. Some aspects of hemodynamics on blood flow .

Degree: 2012, University of Zambia

URL: http://hdl.handle.net/123456789/1688

Subjects/Keywords: Blood flow.; Hemodynamics.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Mubyana, M. M. (2012). Some aspects of hemodynamics on blood flow . (Thesis). University of Zambia. Retrieved from http://hdl.handle.net/123456789/1688

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Mubyana, Maswabi Mukela. “Some aspects of hemodynamics on blood flow .” 2012. Thesis, University of Zambia. Accessed March 01, 2021. http://hdl.handle.net/123456789/1688.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Mubyana, Maswabi Mukela. “Some aspects of hemodynamics on blood flow .” 2012. Web. 01 Mar 2021.

Vancouver:

Mubyana MM. Some aspects of hemodynamics on blood flow . [Internet] [Thesis]. University of Zambia; 2012. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/123456789/1688.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mubyana MM. Some aspects of hemodynamics on blood flow . [Thesis]. University of Zambia; 2012. Available from: http://hdl.handle.net/123456789/1688

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of Alberta

16. Urquhart, Gayle. Timing of hemodynamic pressure measurements and thermodilutional cardiac outputs on derived hemodynamic parameters.

Degree: Master of Nursing, Faculty of Nursing, 1999, University of Alberta

URL: https://era.library.ualberta.ca/files/kp78gj50q

Subjects/Keywords: Cardiac output.; Hemodynamics.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Urquhart, G. (1999). Timing of hemodynamic pressure measurements and thermodilutional cardiac outputs on derived hemodynamic parameters. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/kp78gj50q

Chicago Manual of Style (16^th Edition):

Urquhart, Gayle. “Timing of hemodynamic pressure measurements and thermodilutional cardiac outputs on derived hemodynamic parameters.” 1999. Masters Thesis, University of Alberta. Accessed March 01, 2021. https://era.library.ualberta.ca/files/kp78gj50q.

MLA Handbook (7^th Edition):

Urquhart, Gayle. “Timing of hemodynamic pressure measurements and thermodilutional cardiac outputs on derived hemodynamic parameters.” 1999. Web. 01 Mar 2021.

Vancouver:

Urquhart G. Timing of hemodynamic pressure measurements and thermodilutional cardiac outputs on derived hemodynamic parameters. [Internet] [Masters thesis]. University of Alberta; 1999. [cited 2021 Mar 01]. Available from: https://era.library.ualberta.ca/files/kp78gj50q.

Council of Science Editors:

Urquhart G. Timing of hemodynamic pressure measurements and thermodilutional cardiac outputs on derived hemodynamic parameters. [Masters Thesis]. University of Alberta; 1999. Available from: https://era.library.ualberta.ca/files/kp78gj50q

17. Vedula, Vijay. Image based computational modeling of intracardiac flows.

Degree: 2015, Johns Hopkins University

URL: http://jhir.library.jhu.edu/handle/1774.2/37858

► With continuous advancements in four-dimensional medical imaging technologies, increasing computational speeds, and widespread availability of high performance computing facilities, computational modeling of intracardiac flows is… (more)

▼ With continuous advancements in four-dimensional medical imaging technologies, increasing computational speeds, and widespread availability of high performance computing facilities, computational modeling of intracardiac flows is becoming increasingly viable and has the potential to become a powerful non-invasive diagnostic tool for the diagnosis and treatment of cardiovascular disease. The motive of the current study is to develop a modeling framework that facilitates image-based analysis of intracardiac flows in health as well as disease and to use this framework to gain fundamental insights into intracardiac hemodynamics. A procedure is developed for constructing computational fluid dynamics (CFD) – ready models from in vivo imaging data. The key components of this procedure are the registration and segmentation of the 4D data for several (~20) key frames, template based mapping to ensure surface grid conformality and high-fidelity simulations using a sharp-interface immersed boundary solver. A physiologically representative, kinematic model of the mitral valve is also developed for use in these simulations. As a precursor, a comprehensive quantitative validation of the flow solver is performed using experimental data in a simple model of the left ventricle. A quantitative comparison of the phase-averaged velocity and vorticity fields between the simulation and the experiment shows a reasonable agreement. The detailed assessment of this comparison is used to identify and discuss the key challenges and uncertainties associated in conducting such a validation study. The vast majority of computational investigations of intracardiac flows have focused either on the left or the right ventricles while the corresponding atria were modeled in highly simplistic ways. However, the impact of this simplification on the hemodynamics of the ventricular filling has not been clearly understood. Additionally, the surface of the ventricle has been assumed to be smooth although it is well known that the left ventricle is highly corrugated with surface protrusions or trabeculae and papillary muscles extending deep into the ventricular cavity. Hence, separate studies were conducted to understand the effect of complex atrial flows on the intraventricular flow development and also to understand and quantify the impact of the trabeculae and papillary muscles on ventricular hemodynamics Results indicate that the trabeculae and papillary muscles significantly impact ventricular flow resulting in a deeper penetration of the mitral jet into the ventricle during filling. These anatomical features are also found to produce a “squeezing” effect that enhances apical washout. It is also demonstrated that the complex flow dynamics developed inside the left atrium have minimal influence on the flow inside the left ventricle, which is primarily governed by the mitral valve leaflets configuration. The complex vortical structures inside the left atrium are rapidly dissipated due to the complex interaction of multiple vortex rings leading to breakup,… Advisors/Committee Members: Mittal, Rajat (advisor).

Subjects/Keywords: Cardiac hemodynamics; computational modeling; immersed boundary methods

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APA (6^th Edition):

Vedula, V. (2015). Image based computational modeling of intracardiac flows. (Thesis). Johns Hopkins University. Retrieved from http://jhir.library.jhu.edu/handle/1774.2/37858

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Vedula, Vijay. “Image based computational modeling of intracardiac flows.” 2015. Thesis, Johns Hopkins University. Accessed March 01, 2021. http://jhir.library.jhu.edu/handle/1774.2/37858.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Vedula, Vijay. “Image based computational modeling of intracardiac flows.” 2015. Web. 01 Mar 2021.

Vancouver:

Vedula V. Image based computational modeling of intracardiac flows. [Internet] [Thesis]. Johns Hopkins University; 2015. [cited 2021 Mar 01]. Available from: http://jhir.library.jhu.edu/handle/1774.2/37858.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vedula V. Image based computational modeling of intracardiac flows. [Thesis]. Johns Hopkins University; 2015. Available from: http://jhir.library.jhu.edu/handle/1774.2/37858

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Cornell University

18. Nguyen, John. Femtosecond Laser Pulses To Model And Treat Brain Diseases.

Degree: PhD, Biomedical Engineering, 2011, Cornell University

URL: http://hdl.handle.net/1813/30763

► Tightly focusing femtosecond laser pulses into bulk media, such as brain tissue, can generate high intensities within the focal volume. With sufficient laser energies, nonlinear… (more)

▼ Tightly focusing femtosecond laser pulses into bulk media, such as brain tissue, can generate high intensities within the focal volume. With sufficient laser energies, nonlinear absorption of femtosecond laser pulses by the media can take place. This thesis takes advantages of these nonlinear processes to image the cortical vasculature in live anesthetized rodent, and induce localized tissue disruption. We used two-photon excited fluorescence (2PEF) microscopy to image fluorescently labeled cortical vasculature in vivo. In this technique, two-photons are simultaneously absorbed to generate fluorescence that is localized within the focal volume, where intensity is greatest. Scanning the focus enables three-dimensional imaging since out-of-focus fluorescence is not generated. Higher-order nonlinear absorption processes that lead to plasma and cavitation bubble formation are used to induce localized tissue disruption. Together these nonlinear tools enable fine-detailed studies of the cortical vasculature and precise ablation of tissue. To understand the potential role of small venule strokes in the development of cognitive disorders, we investigated the blood flow and vascular effects in individual vessels after inducing single venule occlusions using femtosecond laser ablation. To better understand the blood flow and metabolic responses during an epileptic seizure, we use a several optical techniques to investigate the neurovascular and neurometabolic activity during pre-ictal and ictal phases of focal seizures. Using 2PEF microscopy we reveal the presence of vasoconstriction in arterioles surrounding the focus prior to seizure onset. The deposition of energy within the focal volume can also used to make incisions in bulk brain to disrupt tissue. Using our laser scalpel, we produced sub-surface cortical incisions and characterized the cut width and depth as a function of different laser energies. We then employed this cutting technique to determine the efficacy of sub-surface cuts in stopping seizure propagation. We then showcased a technique to produce welds at the unexposed interface between two pieces of glass and characterized their size as a function of multiple experimental parameters. Joint strength of the fused glass was tested in addition to investigating the changes in optical properties induced by the weld. Lastly, future experiments and advantages of nonlinear optical techniques are discussed. Advisors/Committee Members: Schaffer, Chris (chair), Fetcho, Joseph R. (committee member), Zipfel, Warren R. (committee member).

Subjects/Keywords: two-photon microscopy; femtosecond laser ablation; hemodynamics

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APA (6^th Edition):

Nguyen, J. (2011). Femtosecond Laser Pulses To Model And Treat Brain Diseases. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/30763

Chicago Manual of Style (16^th Edition):

Nguyen, John. “Femtosecond Laser Pulses To Model And Treat Brain Diseases.” 2011. Doctoral Dissertation, Cornell University. Accessed March 01, 2021. http://hdl.handle.net/1813/30763.

MLA Handbook (7^th Edition):

Nguyen, John. “Femtosecond Laser Pulses To Model And Treat Brain Diseases.” 2011. Web. 01 Mar 2021.

Vancouver:

Nguyen J. Femtosecond Laser Pulses To Model And Treat Brain Diseases. [Internet] [Doctoral dissertation]. Cornell University; 2011. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/1813/30763.

Council of Science Editors:

Nguyen J. Femtosecond Laser Pulses To Model And Treat Brain Diseases. [Doctoral Dissertation]. Cornell University; 2011. Available from: http://hdl.handle.net/1813/30763

Penn State University

19. Huo, Bingxing. Control of cerebral hemodynamics during voluntary locomotion in rodents.

Degree: 2014, Penn State University

URL: https://submit-etda.libraries.psu.edu/catalog/23641

► Increases in cerebral blood flow are widely used as surrogate indicators of neural activity, making the understanding of the precision and regularity of cerebral hemodynamic… (more)

▼ Increases in cerebral blood flow are widely used as surrogate indicators of neural activity, making the understanding of the precision and regularity of cerebral hemodynamic signals of fundamental importance. To determine whether cerebral blood flow is tightly regulated spatially and temporally, we measured hemodynamic signals in the mouse cortex during voluntary locomotion, which drives large changes in neural activity and in the peripheral cardiovascular system. Locomotion drives the increase of neural activity in both frontal and parietal cortices in mice. Parietal cortex, especially within the forelimb and hindlimb representations, showed significant increases of blood flow and volume, supporting neurovascular coupling. But frontal cortex showed little or no hemodynamic response, suggesting a neurovascular decoupling mechanism within the region. To quantify the spatial and temporal characters of cerebral hemodynamics, we developed a novel empirical model that is based on microscopic dynamics of artery and vein dilations. This model reliably fits the cerebral hemodynamics in the parietal cortex and quantitatively separated out arterial and venous blood volume. Using this model, we showed that the cerebral hemodynamics in regions presenting neurovascular coupling are highly predictable in space and time. Further, by pharmacologically manipulating the heart rate, we separately tested the sympathetic and parasympathetic influences on the cerebral hemodynamics. We found that the cerebral arterial response and blood flow are highly robust to cardiovascular perturbations; but venous blood volume changes were largely compromised by adrenergic blockade. We concluded that neurovascular coupling during voluntary locomotion is a region-specific, rather than a global phenomenon. The arterial response within the region presenting neurovascular coupling is tightly controlled by a central nervous system, while the venous response is under the influence of the peripheral system. Advisors/Committee Members: Patrick James Drew, Dissertation Advisor/Co-Advisor, Steven Schiff, Committee Member, Corina Stefania Drapaca, Committee Member, Dezhe Jin, Committee Member.

Subjects/Keywords: cerebral hemodynamics; neurovascular coupling; in vivo imaging

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APA (6^th Edition):

Huo, B. (2014). Control of cerebral hemodynamics during voluntary locomotion in rodents. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/23641

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Huo, Bingxing. “Control of cerebral hemodynamics during voluntary locomotion in rodents.” 2014. Thesis, Penn State University. Accessed March 01, 2021. https://submit-etda.libraries.psu.edu/catalog/23641.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Huo, Bingxing. “Control of cerebral hemodynamics during voluntary locomotion in rodents.” 2014. Web. 01 Mar 2021.

Vancouver:

Huo B. Control of cerebral hemodynamics during voluntary locomotion in rodents. [Internet] [Thesis]. Penn State University; 2014. [cited 2021 Mar 01]. Available from: https://submit-etda.libraries.psu.edu/catalog/23641.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huo B. Control of cerebral hemodynamics during voluntary locomotion in rodents. [Thesis]. Penn State University; 2014. Available from: https://submit-etda.libraries.psu.edu/catalog/23641

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Penn State University

20. Winder, Aaron Thomas. Origins of Spontaneous Hemodynamic Signals in the Awake Brain.

Degree: 2017, Penn State University

URL: https://submit-etda.libraries.psu.edu/catalog/14417atw155

► Cerebral blood flow is dynamically regulated through signaling from active neural populations. This neurovascular coupling allows the brain to satisfy its metabolic needs and enables… (more)

▼ Cerebral blood flow is dynamically regulated through signaling from active neural populations. This neurovascular coupling allows the brain to satisfy its metabolic needs and enables the use of non-invasive imaging techniques, such as fMRI, to study brain function through hemodynamic measurements. In particular, hemodynamic fluctuations, which occur in the absence of a task or overt sensory stimulation, are used to infer ongoing local neural activity and study intrinsic functional brain connectivity. However, the neural underpinnings of these spontaneous hemodynamic signals are not clear. We tested how well spontaneous hemodynamic signals correspond to local neural activity by making simultaneous cerebral blood volume (CBV) and electrophysiological measurements from the somatosensory cortex of awake, head-fixed mice during periods of rest, sensory stimulation, and volitional movement. Large spontaneous CBV changes in the absence of sensory input were primarily driven by volitional whisker and body movements, indicating that unmonitored behaviors could contribute to hemodynamic variance in resting state neuroimaging studies. We then quantified neurovascular coupling as the transfer function between measured neural activity and CBV. We showed that the temporal dynamics of the vascular response to increased neural activity were similar across behavioral states. However, individual CBV changes were weakly predictive of the corresponding local neural activity during periods of rest. To discern the origin of resting state CBV, we perturbed local neural activity and observed that spontaneous fluctuations in CBV and vessel diameter persisted when local neural spiking, glutamatergic input, and noradrenergic modulation were blocked. These results indicated that spontaneous hemodynamic signals observed in resting-state imaging data reflect a combination of behavior, local neural activity, and putatively non-neural processes. Advisors/Committee Members: Patrick James Drew, Dissertation Advisor/Co-Advisor, Patrick James Drew, Committee Chair/Co-Chair, Bruce Gluckman, Committee Member, Steven Schiff, Committee Member, Nanyin Zhang, Outside Member.

Subjects/Keywords: Neurovascular Coupling; Behavior; Barrel Cortex; Spontaneous Hemodynamics

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APA (6^th Edition):

Winder, A. T. (2017). Origins of Spontaneous Hemodynamic Signals in the Awake Brain. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/14417atw155

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Winder, Aaron Thomas. “Origins of Spontaneous Hemodynamic Signals in the Awake Brain.” 2017. Thesis, Penn State University. Accessed March 01, 2021. https://submit-etda.libraries.psu.edu/catalog/14417atw155.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Winder, Aaron Thomas. “Origins of Spontaneous Hemodynamic Signals in the Awake Brain.” 2017. Web. 01 Mar 2021.

Vancouver:

Winder AT. Origins of Spontaneous Hemodynamic Signals in the Awake Brain. [Internet] [Thesis]. Penn State University; 2017. [cited 2021 Mar 01]. Available from: https://submit-etda.libraries.psu.edu/catalog/14417atw155.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Winder AT. Origins of Spontaneous Hemodynamic Signals in the Awake Brain. [Thesis]. Penn State University; 2017. Available from: https://submit-etda.libraries.psu.edu/catalog/14417atw155

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of Wisconsin – La Cross

21. Haugen, Alyson R. Screening for psychosocial distress in patients with hematological malignancies and identifying specific factors associated with distress throughout phase of disease.

Degree: 2011, University of Wisconsin – La Cross

URL: http://digital.library.wisc.edu/1793/57136

► The diagnosis of a hematological malignancy and its treatment can lead to symptoms and problems that affect quality of life. An important task of the… (more)

Subjects/Keywords: Cancer, Psychological aspects; Stress (Physiology); Hemodynamics

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APA (6^th Edition):

Haugen, A. R. (2011). Screening for psychosocial distress in patients with hematological malignancies and identifying specific factors associated with distress throughout phase of disease. (Thesis). University of Wisconsin – La Cross. Retrieved from http://digital.library.wisc.edu/1793/57136

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Haugen, Alyson R. “Screening for psychosocial distress in patients with hematological malignancies and identifying specific factors associated with distress throughout phase of disease.” 2011. Thesis, University of Wisconsin – La Cross. Accessed March 01, 2021. http://digital.library.wisc.edu/1793/57136.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Haugen, Alyson R. “Screening for psychosocial distress in patients with hematological malignancies and identifying specific factors associated with distress throughout phase of disease.” 2011. Web. 01 Mar 2021.

Vancouver:

Haugen AR. Screening for psychosocial distress in patients with hematological malignancies and identifying specific factors associated with distress throughout phase of disease. [Internet] [Thesis]. University of Wisconsin – La Cross; 2011. [cited 2021 Mar 01]. Available from: http://digital.library.wisc.edu/1793/57136.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Haugen AR. Screening for psychosocial distress in patients with hematological malignancies and identifying specific factors associated with distress throughout phase of disease. [Thesis]. University of Wisconsin – La Cross; 2011. Available from: http://digital.library.wisc.edu/1793/57136

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of Wisconsin – La Cross

22. Motto, Rosemary E. Comparison of the Cardiovascular hemodynamic adaptations of angina pectoris to "second wind".

Degree: 1979, University of Wisconsin – La Cross

URL: http://digital.library.wisc.edu/1793/48852

► Heart rate (HR), systolic blood pressure (SBP), diastolic blood pressure (DBP ), rate-pressure product (RPP), the mean maximal MET level and the mean maximal painfree… (more)

Subjects/Keywords: Hemodynamics; Angina pectoris

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APA (6^th Edition):

Motto, R. E. (1979). Comparison of the Cardiovascular hemodynamic adaptations of angina pectoris to "second wind". (Thesis). University of Wisconsin – La Cross. Retrieved from http://digital.library.wisc.edu/1793/48852

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Motto, Rosemary E. “Comparison of the Cardiovascular hemodynamic adaptations of angina pectoris to "second wind".” 1979. Thesis, University of Wisconsin – La Cross. Accessed March 01, 2021. http://digital.library.wisc.edu/1793/48852.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Motto, Rosemary E. “Comparison of the Cardiovascular hemodynamic adaptations of angina pectoris to "second wind".” 1979. Web. 01 Mar 2021.

Vancouver:

Motto RE. Comparison of the Cardiovascular hemodynamic adaptations of angina pectoris to "second wind". [Internet] [Thesis]. University of Wisconsin – La Cross; 1979. [cited 2021 Mar 01]. Available from: http://digital.library.wisc.edu/1793/48852.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Motto RE. Comparison of the Cardiovascular hemodynamic adaptations of angina pectoris to "second wind". [Thesis]. University of Wisconsin – La Cross; 1979. Available from: http://digital.library.wisc.edu/1793/48852

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Hong Kong University of Science and Technology

23. Ou, Chubin MAE. Computational modeling of thrombosis in cerebral aneurysms.

Degree: 2016, Hong Kong University of Science and Technology

URL: http://repository.ust.hk/ir/Record/1783.1-97121 ; https://doi.org/10.14711/thesis-b1585093 ; http://repository.ust.hk/ir/bitstream/1783.1-97121/1/th_redirect.html

► Thrombus plays an important role in the progression, rupture and healing of cerebral aneurysms. Nowadays, cerebral aneurysms are usually treated with endovascular techniques including coiling… (more)

Subjects/Keywords: Intracranial aneurysms ; Thrombosis ; Computer simulation ; Hemodynamics

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APA (6^th Edition):

Ou, C. M. (2016). Computational modeling of thrombosis in cerebral aneurysms. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-97121 ; https://doi.org/10.14711/thesis-b1585093 ; http://repository.ust.hk/ir/bitstream/1783.1-97121/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Ou, Chubin MAE. “Computational modeling of thrombosis in cerebral aneurysms.” 2016. Thesis, Hong Kong University of Science and Technology. Accessed March 01, 2021. http://repository.ust.hk/ir/Record/1783.1-97121 ; https://doi.org/10.14711/thesis-b1585093 ; http://repository.ust.hk/ir/bitstream/1783.1-97121/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Ou, Chubin MAE. “Computational modeling of thrombosis in cerebral aneurysms.” 2016. Web. 01 Mar 2021.

Vancouver:

Ou CM. Computational modeling of thrombosis in cerebral aneurysms. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2016. [cited 2021 Mar 01]. Available from: http://repository.ust.hk/ir/Record/1783.1-97121 ; https://doi.org/10.14711/thesis-b1585093 ; http://repository.ust.hk/ir/bitstream/1783.1-97121/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ou CM. Computational modeling of thrombosis in cerebral aneurysms. [Thesis]. Hong Kong University of Science and Technology; 2016. Available from: http://repository.ust.hk/ir/Record/1783.1-97121 ; https://doi.org/10.14711/thesis-b1585093 ; http://repository.ust.hk/ir/bitstream/1783.1-97121/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of Gothenburg / Göteborgs Universitet

24. Malmcrona, Raoul, 1931-. Haemodynamics in myocardial infarction.

Degree: 1964, University of Gothenburg / Göteborgs Universitet

URL: http://hdl.handle.net/2077/12938

Subjects/Keywords: Myocardial infarction; hemodynamics

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APA (6^th Edition):

Malmcrona, Raoul, 1. (1964). Haemodynamics in myocardial infarction. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/12938

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Malmcrona, Raoul, 1931-. “Haemodynamics in myocardial infarction.” 1964. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed March 01, 2021. http://hdl.handle.net/2077/12938.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Malmcrona, Raoul, 1931-. “Haemodynamics in myocardial infarction.” 1964. Web. 01 Mar 2021.

Vancouver:

Malmcrona, Raoul 1. Haemodynamics in myocardial infarction. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 1964. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/2077/12938.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Malmcrona, Raoul 1. Haemodynamics in myocardial infarction. [Thesis]. University of Gothenburg / Göteborgs Universitet; 1964. Available from: http://hdl.handle.net/2077/12938

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of Gothenburg / Göteborgs Universitet

25. Sivertsson, Ramon, 1936-. The hemodynamic importance of structural vascular changes in essential hypertension.

Degree: 1970, University of Gothenburg / Göteborgs Universitet

URL: http://hdl.handle.net/2077/14140

Subjects/Keywords: Blood vessels; Hemodynamics

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APA (6^th Edition):

Sivertsson, Ramon, 1. (1970). The hemodynamic importance of structural vascular changes in essential hypertension. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/14140

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Sivertsson, Ramon, 1936-. “The hemodynamic importance of structural vascular changes in essential hypertension.” 1970. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed March 01, 2021. http://hdl.handle.net/2077/14140.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Sivertsson, Ramon, 1936-. “The hemodynamic importance of structural vascular changes in essential hypertension.” 1970. Web. 01 Mar 2021.

Vancouver:

Sivertsson, Ramon 1. The hemodynamic importance of structural vascular changes in essential hypertension. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 1970. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/2077/14140.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sivertsson, Ramon 1. The hemodynamic importance of structural vascular changes in essential hypertension. [Thesis]. University of Gothenburg / Göteborgs Universitet; 1970. Available from: http://hdl.handle.net/2077/14140

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Iowa State University

26. Ladwig, Matthew A. Dorsolateral prefrontal cortical hemodynamics, cognitive inhibition, and affective responses to exercise among children: Implications for pediatric exercise prescription.

Degree: 2019, Iowa State University

URL: https://lib.dr.iastate.edu/etd/17492

► Preliminary evidence suggests that children may report decreasing pleasure throughout incremental exercise tests. This pattern of affective responses might be related to the deviation of… (more)

▼ Preliminary evidence suggests that children may report decreasing pleasure throughout incremental exercise tests. This pattern of affective responses might be related to the deviation of incremental and prolonged patterns of exercise from the innate propensity of children for intermittent movement. Furthermore, the self-regulation of affective responses during exercise may require cognitive strategies underpinned by cortical networks still under development during childhood. Consequently, the purpose of this study was to examine (1) whether dorsolateral prefrontal cortical (dlPFC) activity is associated with affective responses during an incremental cycling exercise test, and (2) whether theoretically relevant covariates moderate affective responses and dlPFC activation during exercise. Secondary outcomes of interest included (3) whether affective responses, dlPFC activation, and time spent exercising at intensities above ventilatory threshold (VT) are associated with lower postexercise scores on the inhibition component of executive function as assessed by a pediatric adaptation of the Eriksen Flanker Task. Fifty-six healthy girls (n = 27) and boys (n = 29) aged 7-9 years participated in the study. There was a positive association between left dlPFC activity and affective responses above VT to the end of exercise, whereby dlPFC activity declined concurrently with affective responses. Age and body mass index (BMI) moderated the slopes of change in right dlPFC activity below VT, while age moderated the slope of affective responses and BMI moderated the slope of left dlPFC activity below VT. There were no moderators of affective responses or dlPFC activity from VT to the end of exercise. These results suggest that children experience declines in pleasure that accelerate during incremental exercise and that these declines may reflect unsuccessful prefrontally mediated efforts to self-regulate affect. Children may not yet be able to effectively self-regulate affective responses during exercise without additional biological maturation and/or deliberate practice.

Subjects/Keywords: Affect; Exercise; Hemodynamics; Pediatrics; Prefrontal cortex; Kinesiology

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APA (6^th Edition):

Ladwig, M. A. (2019). Dorsolateral prefrontal cortical hemodynamics, cognitive inhibition, and affective responses to exercise among children: Implications for pediatric exercise prescription. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/17492

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Ladwig, Matthew A. “Dorsolateral prefrontal cortical hemodynamics, cognitive inhibition, and affective responses to exercise among children: Implications for pediatric exercise prescription.” 2019. Thesis, Iowa State University. Accessed March 01, 2021. https://lib.dr.iastate.edu/etd/17492.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Ladwig, Matthew A. “Dorsolateral prefrontal cortical hemodynamics, cognitive inhibition, and affective responses to exercise among children: Implications for pediatric exercise prescription.” 2019. Web. 01 Mar 2021.

Vancouver:

Ladwig MA. Dorsolateral prefrontal cortical hemodynamics, cognitive inhibition, and affective responses to exercise among children: Implications for pediatric exercise prescription. [Internet] [Thesis]. Iowa State University; 2019. [cited 2021 Mar 01]. Available from: https://lib.dr.iastate.edu/etd/17492.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ladwig MA. Dorsolateral prefrontal cortical hemodynamics, cognitive inhibition, and affective responses to exercise among children: Implications for pediatric exercise prescription. [Thesis]. Iowa State University; 2019. Available from: https://lib.dr.iastate.edu/etd/17492

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Visser, Willy. Hemodynamic studies in preeclampsia: implications for management.

Degree: Department of Gynaecology & Obstetrics, 1995, Erasmus University Medical Center

URL: http://hdl.handle.net/1765/22043

► textabstractPreeclampsia is a condition unique to pregnancy, characterized by hypertension and proteinuria. Nulliparous pregnancies and those affected by preexisting hypertension, diabetes and renal disease are… (more)

Subjects/Keywords: hemodynamics; preeclampsia; vasodilatation

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APA (6^th Edition):

Visser, W. (1995). Hemodynamic studies in preeclampsia: implications for management. (Doctoral Dissertation). Erasmus University Medical Center. Retrieved from http://hdl.handle.net/1765/22043

Chicago Manual of Style (16^th Edition):

Visser, Willy. “Hemodynamic studies in preeclampsia: implications for management.” 1995. Doctoral Dissertation, Erasmus University Medical Center. Accessed March 01, 2021. http://hdl.handle.net/1765/22043.

MLA Handbook (7^th Edition):

Visser, Willy. “Hemodynamic studies in preeclampsia: implications for management.” 1995. Web. 01 Mar 2021.

Vancouver:

Visser W. Hemodynamic studies in preeclampsia: implications for management. [Internet] [Doctoral dissertation]. Erasmus University Medical Center; 1995. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/1765/22043.

Council of Science Editors:

Visser W. Hemodynamic studies in preeclampsia: implications for management. [Doctoral Dissertation]. Erasmus University Medical Center; 1995. Available from: http://hdl.handle.net/1765/22043

University of South Florida

28. Zhang, Jie. Glomerular Hyperfiltration and Hypertension in Diabetes.

Degree: 2017, University of South Florida

URL: https://scholarcommons.usf.edu/etd/7455

► In the present study, we investigated the pathophysiological mechanisms of the hemodynamic alteration in diabetes. Glomerular hyperfiltration occurs in the early stage of diabetes mellitus… (more)

Subjects/Keywords: Diabetes,; Hyperfiltration; Hypertension; Renal Hemodynamics; Physiology

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APA (6^th Edition):

Zhang, J. (2017). Glomerular Hyperfiltration and Hypertension in Diabetes. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/7455

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Zhang, Jie. “Glomerular Hyperfiltration and Hypertension in Diabetes.” 2017. Thesis, University of South Florida. Accessed March 01, 2021. https://scholarcommons.usf.edu/etd/7455.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Zhang, Jie. “Glomerular Hyperfiltration and Hypertension in Diabetes.” 2017. Web. 01 Mar 2021.

Vancouver:

Zhang J. Glomerular Hyperfiltration and Hypertension in Diabetes. [Internet] [Thesis]. University of South Florida; 2017. [cited 2021 Mar 01]. Available from: https://scholarcommons.usf.edu/etd/7455.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang J. Glomerular Hyperfiltration and Hypertension in Diabetes. [Thesis]. University of South Florida; 2017. Available from: https://scholarcommons.usf.edu/etd/7455

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Duke University

29. Patterson, Sarah Elizabeth Ritchey. Extensions of the Immersed Interface Method to Open Tube Interfaces and Hemodynamic Models .

Degree: 2019, Duke University

URL: http://hdl.handle.net/10161/19869

► Blood flow can be modeled as a fluid-structure interaction problem in which the vessel is represented as an infinitely thin elastic interface that exerts… (more)

Subjects/Keywords: Mathematics; Immersed Interface Method; Renal Hemodynamics

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APA (6^th Edition):

Patterson, S. E. R. (2019). Extensions of the Immersed Interface Method to Open Tube Interfaces and Hemodynamic Models . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/19869

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Patterson, Sarah Elizabeth Ritchey. “Extensions of the Immersed Interface Method to Open Tube Interfaces and Hemodynamic Models .” 2019. Thesis, Duke University. Accessed March 01, 2021. http://hdl.handle.net/10161/19869.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Patterson, Sarah Elizabeth Ritchey. “Extensions of the Immersed Interface Method to Open Tube Interfaces and Hemodynamic Models .” 2019. Web. 01 Mar 2021.

Vancouver:

Patterson SER. Extensions of the Immersed Interface Method to Open Tube Interfaces and Hemodynamic Models . [Internet] [Thesis]. Duke University; 2019. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/10161/19869.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Patterson SER. Extensions of the Immersed Interface Method to Open Tube Interfaces and Hemodynamic Models . [Thesis]. Duke University; 2019. Available from: http://hdl.handle.net/10161/19869

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Georgia Tech

30. Rathan, Swetha. Aortic valve mechanobiology- role of altered hemodynamics in mediating aortic valve inflammation and calcification.

Degree: PhD, Chemical and Biomolecular Engineering, 2016, Georgia Tech

URL: http://hdl.handle.net/1853/58144

► Calcific aortic valve (AV) disease is a strong risk factor for cardiovascular related deaths and is a significant source of mortality worldwide, with the number… (more)

▼ Calcific aortic valve (AV) disease is a strong risk factor for cardiovascular related deaths and is a significant source of mortality worldwide, with the number of patients requiring AV surgery expected to increase from 250,000 to 850,000 by 2050. However, the molecular mechanisms underlying AV disease have not been well studied or understood. Further, identification of biomarkers that can be used to detect early stage AV disease is also understudied but vital to successfully preventing and/or treating AV disease. It was hypothesized that sclerosis, inflammation and calcification preferentially occurs in the fibrosa compared to the ventricularis due to differential gene expression and oscillatory shear stress. Freshly isolated porcine AV leaflets and an ex vivo shear stress bioreactor was used to test this hypothesis. The low magnitude oscillatory shear stress (OS) appeared to predispose fibrosa to side-dependent calcification via increasing collagen turnover (Col1a1), and thickening of the extracellular matrix (ECM) (fibrosis) and decreasing the expression of genes that protect endothelial function (Klf4 and Enos). The unidirectional pulsatile shear, LS, however, preserved the ECM and gene expression in the ventricularis. The involvement of miRNAs in OS mediated AV pathogenesis was also investigated in a shear- and side-dependent manner. The miR-214 was found to play a role in this OS induced pathogenesis in fibrosa but not ventricularis. Using an ex vivo miRNA silencing protocol, anti-miRNA was delivered to both endothelial and interstitial cells of the AV tissue without compromising the cell viability. Silencing of miR-214 showed that the OS induced pathology in the fibrosa is likely to be mediated via miR-214, klf4 and Tgfβ1 dependent pathway that can lead to AV fibrosis, endothelial-to-mesenchymal transition and eventually sclerosis. The miR-214, however, did not play a role in shear-induced inflammation and calcification. The miR-214, as such, is likely to play a key role in the early onset of side- and shear- dependent AV disease and has a potential to serve as a disease biomarker. Further, an ex vivo AV calcification model was also developed to understand the role of endogenous pro- and anti-calcification factors, such as inorganic pyrophosphate, orthophosphate, and alkaline phosphatase. The functional studies carried out in this dissertation aim to link the mechanosensitive miRNAs to the genes involved in inflammation, endothelial-to-mesenchymal transition, and cell apoptosis etc, which eventually causes AV leaflets to calcify. Thus improved understanding of AV disease mechanisms under different hemodynamic conditions will enable us to improve the design of tissue-engineered valves and develop non-surgical treatment options. Advisors/Committee Members: Yoganathan, Ajit P. (advisor), Jo, Hanjoong (committee member), Taylor, W. Robert (committee member), Champion, Julie A. (committee member), Nerem, Robert M. (committee member).

Subjects/Keywords: Aortic valve; Hemodynamics; Mechanobiology; MicroRNA; Calcification

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APA (6^th Edition):

Rathan, S. (2016). Aortic valve mechanobiology- role of altered hemodynamics in mediating aortic valve inflammation and calcification. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/58144

Chicago Manual of Style (16^th Edition):

Rathan, Swetha. “Aortic valve mechanobiology- role of altered hemodynamics in mediating aortic valve inflammation and calcification.” 2016. Doctoral Dissertation, Georgia Tech. Accessed March 01, 2021. http://hdl.handle.net/1853/58144.

MLA Handbook (7^th Edition):

Rathan, Swetha. “Aortic valve mechanobiology- role of altered hemodynamics in mediating aortic valve inflammation and calcification.” 2016. Web. 01 Mar 2021.

Vancouver:

Rathan S. Aortic valve mechanobiology- role of altered hemodynamics in mediating aortic valve inflammation and calcification. [Internet] [Doctoral dissertation]. Georgia Tech; 2016. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/1853/58144.

Council of Science Editors:

Rathan S. Aortic valve mechanobiology- role of altered hemodynamics in mediating aortic valve inflammation and calcification. [Doctoral Dissertation]. Georgia Tech; 2016. Available from: http://hdl.handle.net/1853/58144

Open Access Theses and Dissertations