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University: Texas Medical Center

You searched for subject:(Health AND Medical Administration). Showing records 1 – 30 of 105 total matches.

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Texas Medical Center

1. Han, Lixia. SOCIAL-DEMOGRAPHICS, HEALTH BEHAVIORS, TELOMERE LENGTH IN MEXICAN-AMERICAN COHORT AND CIRCULATING DNA METHYLATION IN BLADDER CANCER.

Degree: MS, 2015, Texas Medical Center

  I. SOCIAL-DEMOGRAPHICS, HEALTH BEHAVIORS, AND TELOMERE LENGTH IN MEXICAN AMERICANS: A COHORT STUDY Recent studies using a prospective cohort design have suggested that telomere… (more)

Subjects/Keywords: Medicine and Health Sciences; Translational Medical Research

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APA (6th Edition):

Han, L. (2015). SOCIAL-DEMOGRAPHICS, HEALTH BEHAVIORS, TELOMERE LENGTH IN MEXICAN-AMERICAN COHORT AND CIRCULATING DNA METHYLATION IN BLADDER CANCER. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/573

Chicago Manual of Style (16th Edition):

Han, Lixia. “SOCIAL-DEMOGRAPHICS, HEALTH BEHAVIORS, TELOMERE LENGTH IN MEXICAN-AMERICAN COHORT AND CIRCULATING DNA METHYLATION IN BLADDER CANCER.” 2015. Masters Thesis, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/573.

MLA Handbook (7th Edition):

Han, Lixia. “SOCIAL-DEMOGRAPHICS, HEALTH BEHAVIORS, TELOMERE LENGTH IN MEXICAN-AMERICAN COHORT AND CIRCULATING DNA METHYLATION IN BLADDER CANCER.” 2015. Web. 23 Oct 2019.

Vancouver:

Han L. SOCIAL-DEMOGRAPHICS, HEALTH BEHAVIORS, TELOMERE LENGTH IN MEXICAN-AMERICAN COHORT AND CIRCULATING DNA METHYLATION IN BLADDER CANCER. [Internet] [Masters thesis]. Texas Medical Center; 2015. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/573.

Council of Science Editors:

Han L. SOCIAL-DEMOGRAPHICS, HEALTH BEHAVIORS, TELOMERE LENGTH IN MEXICAN-AMERICAN COHORT AND CIRCULATING DNA METHYLATION IN BLADDER CANCER. [Masters Thesis]. Texas Medical Center; 2015. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/573


Texas Medical Center

2. Hansen, Jean M. Clinical Significance of Homologous Recombination Deficiency Score Testing in Endometrial Cancer.

Degree: MS, 2016, Texas Medical Center

  Objectives: Homologous recombination deficiency (HRD) score is related to chemotherapy response in breast and ovarian cancers. The role of HRD is unknown in endometrial… (more)

Subjects/Keywords: Medicine and Health Sciences; Translational Medical Research

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APA (6th Edition):

Hansen, J. M. (2016). Clinical Significance of Homologous Recombination Deficiency Score Testing in Endometrial Cancer. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/683

Chicago Manual of Style (16th Edition):

Hansen, Jean M. “Clinical Significance of Homologous Recombination Deficiency Score Testing in Endometrial Cancer.” 2016. Masters Thesis, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/683.

MLA Handbook (7th Edition):

Hansen, Jean M. “Clinical Significance of Homologous Recombination Deficiency Score Testing in Endometrial Cancer.” 2016. Web. 23 Oct 2019.

Vancouver:

Hansen JM. Clinical Significance of Homologous Recombination Deficiency Score Testing in Endometrial Cancer. [Internet] [Masters thesis]. Texas Medical Center; 2016. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/683.

Council of Science Editors:

Hansen JM. Clinical Significance of Homologous Recombination Deficiency Score Testing in Endometrial Cancer. [Masters Thesis]. Texas Medical Center; 2016. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/683


Texas Medical Center

3. Voice, Roy L, III. Differential regulation of IRESs in the Aurora A mRNA by bFGF through the mTOR complex TORC2 modulates Aurora A kinase expression.

Degree: MS, 2014, Texas Medical Center

  Identifying the mechanisms that contribute to tumorigenesis is a major area of focus in our fight against cancer. Epithelial malignant tumors, such as breast,… (more)

Subjects/Keywords: Aurora A kinase; mTOR; FGF; over-express; protein; regulation; Medical Biochemistry; Medical Cell Biology; Medical Genetics; Medicine and Health Sciences

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APA (6th Edition):

Voice, Roy L, I. (2014). Differential regulation of IRESs in the Aurora A mRNA by bFGF through the mTOR complex TORC2 modulates Aurora A kinase expression. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/424

Chicago Manual of Style (16th Edition):

Voice, Roy L, III. “Differential regulation of IRESs in the Aurora A mRNA by bFGF through the mTOR complex TORC2 modulates Aurora A kinase expression.” 2014. Masters Thesis, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/424.

MLA Handbook (7th Edition):

Voice, Roy L, III. “Differential regulation of IRESs in the Aurora A mRNA by bFGF through the mTOR complex TORC2 modulates Aurora A kinase expression.” 2014. Web. 23 Oct 2019.

Vancouver:

Voice, Roy L I. Differential regulation of IRESs in the Aurora A mRNA by bFGF through the mTOR complex TORC2 modulates Aurora A kinase expression. [Internet] [Masters thesis]. Texas Medical Center; 2014. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/424.

Council of Science Editors:

Voice, Roy L I. Differential regulation of IRESs in the Aurora A mRNA by bFGF through the mTOR complex TORC2 modulates Aurora A kinase expression. [Masters Thesis]. Texas Medical Center; 2014. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/424


Texas Medical Center

4. Falugi, Christina. Effect of Photographs of Visible Genetic Conditions on Quality of Life Perceptions.

Degree: MS, 2016, Texas Medical Center

  Historically, medical photographs are used to demonstrate dysmorphic features and characteristic presentations of genetic conditions. Traditional, pictorial depictions of genetic conditions typically involve nude… (more)

Subjects/Keywords: medical photographs; textbook photographs; perceptions; quality of life; healthcare education; medical education; Medical Education; Medicine and Health Sciences

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APA (6th Edition):

Falugi, C. (2016). Effect of Photographs of Visible Genetic Conditions on Quality of Life Perceptions. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/673

Chicago Manual of Style (16th Edition):

Falugi, Christina. “Effect of Photographs of Visible Genetic Conditions on Quality of Life Perceptions.” 2016. Masters Thesis, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/673.

MLA Handbook (7th Edition):

Falugi, Christina. “Effect of Photographs of Visible Genetic Conditions on Quality of Life Perceptions.” 2016. Web. 23 Oct 2019.

Vancouver:

Falugi C. Effect of Photographs of Visible Genetic Conditions on Quality of Life Perceptions. [Internet] [Masters thesis]. Texas Medical Center; 2016. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/673.

Council of Science Editors:

Falugi C. Effect of Photographs of Visible Genetic Conditions on Quality of Life Perceptions. [Masters Thesis]. Texas Medical Center; 2016. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/673


Texas Medical Center

5. Wagner, Chelsea; and#60;pand#62;0000-0002-5730-1360and#60;/pand#62. Genesurance Counseling: Patient Perspectives.

Degree: MS, 2017, Texas Medical Center

  Genetic counselors (GCs) have recently reported an increase in the discussion of insurance-related, or “genesurance,” topics during genetic counseling sessions. Despite increasing frequency, little… (more)

Subjects/Keywords: genesurance counseling; genetic counseling; efficiency; health insurance literacy; Medical Genetics; Medicine and Health Sciences

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APA (6th Edition):

Wagner, C. a. (2017). Genesurance Counseling: Patient Perspectives. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/759

Chicago Manual of Style (16th Edition):

Wagner, Chelsea; and#60;pand#62;0000-0002-5730-1360and#60;/pand#62. “Genesurance Counseling: Patient Perspectives.” 2017. Masters Thesis, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/759.

MLA Handbook (7th Edition):

Wagner, Chelsea; and#60;pand#62;0000-0002-5730-1360and#60;/pand#62. “Genesurance Counseling: Patient Perspectives.” 2017. Web. 23 Oct 2019.

Vancouver:

Wagner Ca. Genesurance Counseling: Patient Perspectives. [Internet] [Masters thesis]. Texas Medical Center; 2017. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/759.

Council of Science Editors:

Wagner Ca. Genesurance Counseling: Patient Perspectives. [Masters Thesis]. Texas Medical Center; 2017. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/759


Texas Medical Center

6. Penney, Samantha. Evaluation of Knowledge Regarding Diagnostic Strategies for Genetic Diseases in Select Residents.

Degree: MS, 2012, Texas Medical Center

  Genetics education for physicians has been a popular publication topic in the United States and in Europe for over 20 years. Decreasing numbers of… (more)

Subjects/Keywords: CMA; resident knowledge; genetics education; medical genetics; microarray; genetic testing; clinical genetics primary care; genetics; pediatrics; physician knowledge; medical education; Medical Education; Medical Genetics; Medicine and Health Sciences

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APA (6th Edition):

Penney, S. (2012). Evaluation of Knowledge Regarding Diagnostic Strategies for Genetic Diseases in Select Residents. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/257

Chicago Manual of Style (16th Edition):

Penney, Samantha. “Evaluation of Knowledge Regarding Diagnostic Strategies for Genetic Diseases in Select Residents.” 2012. Masters Thesis, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/257.

MLA Handbook (7th Edition):

Penney, Samantha. “Evaluation of Knowledge Regarding Diagnostic Strategies for Genetic Diseases in Select Residents.” 2012. Web. 23 Oct 2019.

Vancouver:

Penney S. Evaluation of Knowledge Regarding Diagnostic Strategies for Genetic Diseases in Select Residents. [Internet] [Masters thesis]. Texas Medical Center; 2012. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/257.

Council of Science Editors:

Penney S. Evaluation of Knowledge Regarding Diagnostic Strategies for Genetic Diseases in Select Residents. [Masters Thesis]. Texas Medical Center; 2012. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/257


Texas Medical Center

7. Park, Peter. DEVELOPMENT OF A BEAM-SPECIFIC PLANNING TARGET VOLUME AND A ROBUST PLAN ANALYSIS TOOL FOR PROTON THERAPY.

Degree: PhD, 2012, Texas Medical Center

  Proton therapy is growing increasingly popular due to its superior dose characteristics compared to conventional photon therapy. Protons travel a finite range in the… (more)

Subjects/Keywords: Proton Therapy; Medical Biophysics; Medicine and Health Sciences

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APA (6th Edition):

Park, P. (2012). DEVELOPMENT OF A BEAM-SPECIFIC PLANNING TARGET VOLUME AND A ROBUST PLAN ANALYSIS TOOL FOR PROTON THERAPY. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/325

Chicago Manual of Style (16th Edition):

Park, Peter. “DEVELOPMENT OF A BEAM-SPECIFIC PLANNING TARGET VOLUME AND A ROBUST PLAN ANALYSIS TOOL FOR PROTON THERAPY.” 2012. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/325.

MLA Handbook (7th Edition):

Park, Peter. “DEVELOPMENT OF A BEAM-SPECIFIC PLANNING TARGET VOLUME AND A ROBUST PLAN ANALYSIS TOOL FOR PROTON THERAPY.” 2012. Web. 23 Oct 2019.

Vancouver:

Park P. DEVELOPMENT OF A BEAM-SPECIFIC PLANNING TARGET VOLUME AND A ROBUST PLAN ANALYSIS TOOL FOR PROTON THERAPY. [Internet] [Doctoral dissertation]. Texas Medical Center; 2012. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/325.

Council of Science Editors:

Park P. DEVELOPMENT OF A BEAM-SPECIFIC PLANNING TARGET VOLUME AND A ROBUST PLAN ANALYSIS TOOL FOR PROTON THERAPY. [Doctoral Dissertation]. Texas Medical Center; 2012. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/325


Texas Medical Center

8. Salazar, Katrina. TAZ AS A REGULATOR OF MESENCHYMAL TRANSFORMATION AND CLINICAL AGGRESSIVENESS IN GLIOMAS.

Degree: PhD, 2012, Texas Medical Center

  Glioblastoma multiforme (GBM) is an aggressive, high grade brain tumor. Microarray studies have shown a subset of GBMs with a mesenchymal gene signature. This… (more)

Subjects/Keywords: Glioblastoma; TAZ; TEAD; Hippo pathway; mesenchymal transition; proneural; Medical Molecular Biology; Medical Pathology; Medicine and Health Sciences; Neoplasms; Oncology; Pathology

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APA (6th Edition):

Salazar, K. (2012). TAZ AS A REGULATOR OF MESENCHYMAL TRANSFORMATION AND CLINICAL AGGRESSIVENESS IN GLIOMAS. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/229

Chicago Manual of Style (16th Edition):

Salazar, Katrina. “TAZ AS A REGULATOR OF MESENCHYMAL TRANSFORMATION AND CLINICAL AGGRESSIVENESS IN GLIOMAS.” 2012. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/229.

MLA Handbook (7th Edition):

Salazar, Katrina. “TAZ AS A REGULATOR OF MESENCHYMAL TRANSFORMATION AND CLINICAL AGGRESSIVENESS IN GLIOMAS.” 2012. Web. 23 Oct 2019.

Vancouver:

Salazar K. TAZ AS A REGULATOR OF MESENCHYMAL TRANSFORMATION AND CLINICAL AGGRESSIVENESS IN GLIOMAS. [Internet] [Doctoral dissertation]. Texas Medical Center; 2012. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/229.

Council of Science Editors:

Salazar K. TAZ AS A REGULATOR OF MESENCHYMAL TRANSFORMATION AND CLINICAL AGGRESSIVENESS IN GLIOMAS. [Doctoral Dissertation]. Texas Medical Center; 2012. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/229


Texas Medical Center

9. Sun, Hua. THE NEW ROLE OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9: A CONNECTION OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9, APOLIPOPROTEIN B, and AUTOPHAGY.

Degree: PhD, 2012, Texas Medical Center

  Plasma low-density lipoprotein (LDL) levels are positively correlated with the incidence of coronary artery disease. In the circulation, the plasma LDL clearance is mainly… (more)

Subjects/Keywords: PCSK9; apolipoprotein; cholesterol; mouse model; autophgy; atherosclerosis; LDL; Biochemical Phenomena, Metabolism, and Nutrition; Medical Biochemistry; Medical Pathology; Medicine and Health Sciences

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APA (6th Edition):

Sun, H. (2012). THE NEW ROLE OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9: A CONNECTION OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9, APOLIPOPROTEIN B, and AUTOPHAGY. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/277

Chicago Manual of Style (16th Edition):

Sun, Hua. “THE NEW ROLE OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9: A CONNECTION OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9, APOLIPOPROTEIN B, and AUTOPHAGY.” 2012. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/277.

MLA Handbook (7th Edition):

Sun, Hua. “THE NEW ROLE OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9: A CONNECTION OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9, APOLIPOPROTEIN B, and AUTOPHAGY.” 2012. Web. 23 Oct 2019.

Vancouver:

Sun H. THE NEW ROLE OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9: A CONNECTION OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9, APOLIPOPROTEIN B, and AUTOPHAGY. [Internet] [Doctoral dissertation]. Texas Medical Center; 2012. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/277.

Council of Science Editors:

Sun H. THE NEW ROLE OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9: A CONNECTION OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9, APOLIPOPROTEIN B, and AUTOPHAGY. [Doctoral Dissertation]. Texas Medical Center; 2012. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/277


Texas Medical Center

10. Huang, Olivia. Evaluation of PRESAGE® dosimeters for brachytherapy sources and the 3D dosimetry and characterization of the new AgX100 125I seed model.

Degree: MS, 2013, Texas Medical Center

  With continuous new improvements in brachytherapy source designs and techniques, method of 3D dosimetry for treatment dose verifications would better ensure accurate patient radiotherapy… (more)

Subjects/Keywords: Brachytherapy; PRESAGE; Radiation therapy; dosimeters; 3D; Amersham; AgX100; Medical Biophysics; Medical Sciences; Medicine and Health Sciences

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APA (6th Edition):

Huang, O. (2013). Evaluation of PRESAGE® dosimeters for brachytherapy sources and the 3D dosimetry and characterization of the new AgX100 125I seed model. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/393

Chicago Manual of Style (16th Edition):

Huang, Olivia. “Evaluation of PRESAGE® dosimeters for brachytherapy sources and the 3D dosimetry and characterization of the new AgX100 125I seed model.” 2013. Masters Thesis, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/393.

MLA Handbook (7th Edition):

Huang, Olivia. “Evaluation of PRESAGE® dosimeters for brachytherapy sources and the 3D dosimetry and characterization of the new AgX100 125I seed model.” 2013. Web. 23 Oct 2019.

Vancouver:

Huang O. Evaluation of PRESAGE® dosimeters for brachytherapy sources and the 3D dosimetry and characterization of the new AgX100 125I seed model. [Internet] [Masters thesis]. Texas Medical Center; 2013. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/393.

Council of Science Editors:

Huang O. Evaluation of PRESAGE® dosimeters for brachytherapy sources and the 3D dosimetry and characterization of the new AgX100 125I seed model. [Masters Thesis]. Texas Medical Center; 2013. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/393


Texas Medical Center

11. Liao, Hsin-Wei. Methylation of EGFR by Arginine Methyltransferase PRMT1 Enhances EGFR Signaling and Cetuximab resistance.

Degree: PhD, 2015, Texas Medical Center

  Protein modifications of epidermal growth factor receptor (EGFR) intracellular domain are well known regulators of EGFR functions whereas those of its extracellular domain remain… (more)

Subjects/Keywords: PRMT1; EGFR; methylation; cetuximab; colorectal cancer; Medical Cell Biology; Medical Molecular Biology; Medicine and Health Sciences

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APA (6th Edition):

Liao, H. (2015). Methylation of EGFR by Arginine Methyltransferase PRMT1 Enhances EGFR Signaling and Cetuximab resistance. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/615

Chicago Manual of Style (16th Edition):

Liao, Hsin-Wei. “Methylation of EGFR by Arginine Methyltransferase PRMT1 Enhances EGFR Signaling and Cetuximab resistance.” 2015. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/615.

MLA Handbook (7th Edition):

Liao, Hsin-Wei. “Methylation of EGFR by Arginine Methyltransferase PRMT1 Enhances EGFR Signaling and Cetuximab resistance.” 2015. Web. 23 Oct 2019.

Vancouver:

Liao H. Methylation of EGFR by Arginine Methyltransferase PRMT1 Enhances EGFR Signaling and Cetuximab resistance. [Internet] [Doctoral dissertation]. Texas Medical Center; 2015. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/615.

Council of Science Editors:

Liao H. Methylation of EGFR by Arginine Methyltransferase PRMT1 Enhances EGFR Signaling and Cetuximab resistance. [Doctoral Dissertation]. Texas Medical Center; 2015. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/615


Texas Medical Center

12. Lin, Jing. Post-transcriptional Regulation of Mammalian Gene Expression in Non-coding Region of Target RNA.

Degree: PhD, 2012, Texas Medical Center

  Tumor Suppressor Candidate 2 (TUSC2) is a novel tumor suppressor gene located in the human chromosome 3p21.3 region. TUSC2 mRNA transcripts could be detected… (more)

Subjects/Keywords: Post-transcriptional Regulation; non-coding region of RNA; miRNA; UTR; SLA-RT-PCR; Medical Genetics; Medical Molecular Biology; Medical Sciences; Medicine and Health Sciences

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APA (6th Edition):

Lin, J. (2012). Post-transcriptional Regulation of Mammalian Gene Expression in Non-coding Region of Target RNA. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/299

Chicago Manual of Style (16th Edition):

Lin, Jing. “Post-transcriptional Regulation of Mammalian Gene Expression in Non-coding Region of Target RNA.” 2012. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/299.

MLA Handbook (7th Edition):

Lin, Jing. “Post-transcriptional Regulation of Mammalian Gene Expression in Non-coding Region of Target RNA.” 2012. Web. 23 Oct 2019.

Vancouver:

Lin J. Post-transcriptional Regulation of Mammalian Gene Expression in Non-coding Region of Target RNA. [Internet] [Doctoral dissertation]. Texas Medical Center; 2012. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/299.

Council of Science Editors:

Lin J. Post-transcriptional Regulation of Mammalian Gene Expression in Non-coding Region of Target RNA. [Doctoral Dissertation]. Texas Medical Center; 2012. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/299


Texas Medical Center

13. Thompson, Stephanie. LATINAS AND THE TRADITIONAL GENETIC COUNSELING MODEL: A QUALITATIVE STUDY.

Degree: MS, 2014, Texas Medical Center

  The traditional genetic counseling model reflects an individualized counseling session that includes the presentation of information about genes, chromosomes, personalized risk assessment, and genetic… (more)

Subjects/Keywords: prenatal genetic counseling; traditional model; group model; Latinas; Counseling; Genetic Phenomena; Health Services Research; Medical Education; Medical Genetics; Medicine and Health Sciences; Obstetrics and Gynecology

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APA (6th Edition):

Thompson, S. (2014). LATINAS AND THE TRADITIONAL GENETIC COUNSELING MODEL: A QUALITATIVE STUDY. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/466

Chicago Manual of Style (16th Edition):

Thompson, Stephanie. “LATINAS AND THE TRADITIONAL GENETIC COUNSELING MODEL: A QUALITATIVE STUDY.” 2014. Masters Thesis, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/466.

MLA Handbook (7th Edition):

Thompson, Stephanie. “LATINAS AND THE TRADITIONAL GENETIC COUNSELING MODEL: A QUALITATIVE STUDY.” 2014. Web. 23 Oct 2019.

Vancouver:

Thompson S. LATINAS AND THE TRADITIONAL GENETIC COUNSELING MODEL: A QUALITATIVE STUDY. [Internet] [Masters thesis]. Texas Medical Center; 2014. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/466.

Council of Science Editors:

Thompson S. LATINAS AND THE TRADITIONAL GENETIC COUNSELING MODEL: A QUALITATIVE STUDY. [Masters Thesis]. Texas Medical Center; 2014. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/466


Texas Medical Center

14. Nabilsi, Nancy H. Regulation of survivin gene expression in the human endometrium and endometrial cancer.

Degree: PhD, 2009, Texas Medical Center

 In the United States, endometrial cancer is the leading cancer of the female reproductive tract. There are 40,100 new cases and 7,470 deaths from endometrial… (more)

Subjects/Keywords: Endometrial Cancer; Genes; Molecular biomarkers; Survivin; Medical Genetics; Medicine and Health Sciences; Oncology

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APA (6th Edition):

Nabilsi, N. H. (2009). Regulation of survivin gene expression in the human endometrium and endometrial cancer. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/1

Chicago Manual of Style (16th Edition):

Nabilsi, Nancy H. “Regulation of survivin gene expression in the human endometrium and endometrial cancer.” 2009. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/1.

MLA Handbook (7th Edition):

Nabilsi, Nancy H. “Regulation of survivin gene expression in the human endometrium and endometrial cancer.” 2009. Web. 23 Oct 2019.

Vancouver:

Nabilsi NH. Regulation of survivin gene expression in the human endometrium and endometrial cancer. [Internet] [Doctoral dissertation]. Texas Medical Center; 2009. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/1.

Council of Science Editors:

Nabilsi NH. Regulation of survivin gene expression in the human endometrium and endometrial cancer. [Doctoral Dissertation]. Texas Medical Center; 2009. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/1


Texas Medical Center

15. Iglesias, David A, M.D. DIRECT EFFECTS OF METFORMIN ON PI3K AND RAS SIGNALING IN ENDOMETRIAL CANCER.

Degree: MS, 2012, Texas Medical Center

  Metformin has antiproliferative effects through the activation of AMPK and has gained interest as an antineoplastic agent in several cancer types, although studies in… (more)

Subjects/Keywords: metformin; endometrial cancer; PI3K; K-Ras; Medical Molecular Biology; Medicine and Health Sciences

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APA (6th Edition):

Iglesias, David A, M. D. (2012). DIRECT EFFECTS OF METFORMIN ON PI3K AND RAS SIGNALING IN ENDOMETRIAL CANCER. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/260

Chicago Manual of Style (16th Edition):

Iglesias, David A, M D. “DIRECT EFFECTS OF METFORMIN ON PI3K AND RAS SIGNALING IN ENDOMETRIAL CANCER.” 2012. Masters Thesis, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/260.

MLA Handbook (7th Edition):

Iglesias, David A, M D. “DIRECT EFFECTS OF METFORMIN ON PI3K AND RAS SIGNALING IN ENDOMETRIAL CANCER.” 2012. Web. 23 Oct 2019.

Vancouver:

Iglesias, David A MD. DIRECT EFFECTS OF METFORMIN ON PI3K AND RAS SIGNALING IN ENDOMETRIAL CANCER. [Internet] [Masters thesis]. Texas Medical Center; 2012. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/260.

Council of Science Editors:

Iglesias, David A MD. DIRECT EFFECTS OF METFORMIN ON PI3K AND RAS SIGNALING IN ENDOMETRIAL CANCER. [Masters Thesis]. Texas Medical Center; 2012. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/260


Texas Medical Center

16. Wang, Feng. LOW REACTIVE OXYGEN SPECIES AND HIGH GLYCOLYSIS IN GLIOBLASTOMA STEM CELLS:MECHANISMS AND THERAPEUTIC IMPLICATIONS.

Degree: PhD, 2012, Texas Medical Center

  Glioblastoma multiforme (GBM) is the most common and aggressive primary brain tumor with poor prognosis due in part to drug resistance and high incidence… (more)

Subjects/Keywords: glioblastoma stem cells; metabolic; glycolysis; reactive oxygen species; Medical Sciences; Medicine and Health Sciences

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APA (6th Edition):

Wang, F. (2012). LOW REACTIVE OXYGEN SPECIES AND HIGH GLYCOLYSIS IN GLIOBLASTOMA STEM CELLS:MECHANISMS AND THERAPEUTIC IMPLICATIONS. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/279

Chicago Manual of Style (16th Edition):

Wang, Feng. “LOW REACTIVE OXYGEN SPECIES AND HIGH GLYCOLYSIS IN GLIOBLASTOMA STEM CELLS:MECHANISMS AND THERAPEUTIC IMPLICATIONS.” 2012. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/279.

MLA Handbook (7th Edition):

Wang, Feng. “LOW REACTIVE OXYGEN SPECIES AND HIGH GLYCOLYSIS IN GLIOBLASTOMA STEM CELLS:MECHANISMS AND THERAPEUTIC IMPLICATIONS.” 2012. Web. 23 Oct 2019.

Vancouver:

Wang F. LOW REACTIVE OXYGEN SPECIES AND HIGH GLYCOLYSIS IN GLIOBLASTOMA STEM CELLS:MECHANISMS AND THERAPEUTIC IMPLICATIONS. [Internet] [Doctoral dissertation]. Texas Medical Center; 2012. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/279.

Council of Science Editors:

Wang F. LOW REACTIVE OXYGEN SPECIES AND HIGH GLYCOLYSIS IN GLIOBLASTOMA STEM CELLS:MECHANISMS AND THERAPEUTIC IMPLICATIONS. [Doctoral Dissertation]. Texas Medical Center; 2012. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/279


Texas Medical Center

17. Mohrbacher, Nicole D. THE IMPACT OF FAMILY HISTORY ON MEDULLARY THYROID CANCER IN MEN2A PATIENTS.

Degree: MS, 2013, Texas Medical Center

  The American Thyroid Association recently classified all MEN2A-associated codons into increasing risk levels A-C and stated that some patients may delay prophylactic thyroidectomy if… (more)

Subjects/Keywords: Medullary Thyroid Cancer; Genetic Counseling; MEN2A; Medical Genetics; Medicine and Health Sciences; Oncology; Pediatrics

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APA (6th Edition):

Mohrbacher, N. D. (2013). THE IMPACT OF FAMILY HISTORY ON MEDULLARY THYROID CANCER IN MEN2A PATIENTS. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/353

Chicago Manual of Style (16th Edition):

Mohrbacher, Nicole D. “THE IMPACT OF FAMILY HISTORY ON MEDULLARY THYROID CANCER IN MEN2A PATIENTS.” 2013. Masters Thesis, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/353.

MLA Handbook (7th Edition):

Mohrbacher, Nicole D. “THE IMPACT OF FAMILY HISTORY ON MEDULLARY THYROID CANCER IN MEN2A PATIENTS.” 2013. Web. 23 Oct 2019.

Vancouver:

Mohrbacher ND. THE IMPACT OF FAMILY HISTORY ON MEDULLARY THYROID CANCER IN MEN2A PATIENTS. [Internet] [Masters thesis]. Texas Medical Center; 2013. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/353.

Council of Science Editors:

Mohrbacher ND. THE IMPACT OF FAMILY HISTORY ON MEDULLARY THYROID CANCER IN MEN2A PATIENTS. [Masters Thesis]. Texas Medical Center; 2013. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/353


Texas Medical Center

18. Anfossi, Simone. ROLE OF MIR-19A RELEASED BY INFLAMMATORY BREAST CANCER CELLS IN THE REGULATION OF DENDRITIC CELL FUNCTIONS: IN VITRO MODEL OF CROSSTALK IN THE TUMOR MICROENVIRONMENT OF INFLAMMATORY BREAST CANCER.

Degree: PhD, 2013, Texas Medical Center

  Inflammatory breast cancer (IBC) is a rare but very aggressive form of locally advanced breast cancer (1-6% of total breast cancer patients in United… (more)

Subjects/Keywords: Inflammatory breast cancer; miRNAs; DCs; tumor microenvironment; inflammation; Medical Immunology; Medicine and Health Sciences

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APA (6th Edition):

Anfossi, S. (2013). ROLE OF MIR-19A RELEASED BY INFLAMMATORY BREAST CANCER CELLS IN THE REGULATION OF DENDRITIC CELL FUNCTIONS: IN VITRO MODEL OF CROSSTALK IN THE TUMOR MICROENVIRONMENT OF INFLAMMATORY BREAST CANCER. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/368

Chicago Manual of Style (16th Edition):

Anfossi, Simone. “ROLE OF MIR-19A RELEASED BY INFLAMMATORY BREAST CANCER CELLS IN THE REGULATION OF DENDRITIC CELL FUNCTIONS: IN VITRO MODEL OF CROSSTALK IN THE TUMOR MICROENVIRONMENT OF INFLAMMATORY BREAST CANCER.” 2013. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/368.

MLA Handbook (7th Edition):

Anfossi, Simone. “ROLE OF MIR-19A RELEASED BY INFLAMMATORY BREAST CANCER CELLS IN THE REGULATION OF DENDRITIC CELL FUNCTIONS: IN VITRO MODEL OF CROSSTALK IN THE TUMOR MICROENVIRONMENT OF INFLAMMATORY BREAST CANCER.” 2013. Web. 23 Oct 2019.

Vancouver:

Anfossi S. ROLE OF MIR-19A RELEASED BY INFLAMMATORY BREAST CANCER CELLS IN THE REGULATION OF DENDRITIC CELL FUNCTIONS: IN VITRO MODEL OF CROSSTALK IN THE TUMOR MICROENVIRONMENT OF INFLAMMATORY BREAST CANCER. [Internet] [Doctoral dissertation]. Texas Medical Center; 2013. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/368.

Council of Science Editors:

Anfossi S. ROLE OF MIR-19A RELEASED BY INFLAMMATORY BREAST CANCER CELLS IN THE REGULATION OF DENDRITIC CELL FUNCTIONS: IN VITRO MODEL OF CROSSTALK IN THE TUMOR MICROENVIRONMENT OF INFLAMMATORY BREAST CANCER. [Doctoral Dissertation]. Texas Medical Center; 2013. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/368


Texas Medical Center

19. Parham, Emily A. EVALUATION OF CURRENT CLINICAL CRITERIA FOR LI-FRAUMENI SYNDROME IN A DIVERSE SAMPLE OF TP53 MUTATION CARRIERS.

Degree: MS, 2014, Texas Medical Center

  Li-Fraumeni syndrome (LFS) is a hereditary cancer predisposition syndrome caused by heterozyogous germline mutations in the TP53 gene and characterized by an excess of… (more)

Subjects/Keywords: Li fraumeni syndrome; sarcoma; tp53; genetic testing; Medical Genetics; Medicine and Health Sciences; Neoplasms; Oncology

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APA (6th Edition):

Parham, E. A. (2014). EVALUATION OF CURRENT CLINICAL CRITERIA FOR LI-FRAUMENI SYNDROME IN A DIVERSE SAMPLE OF TP53 MUTATION CARRIERS. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/459

Chicago Manual of Style (16th Edition):

Parham, Emily A. “EVALUATION OF CURRENT CLINICAL CRITERIA FOR LI-FRAUMENI SYNDROME IN A DIVERSE SAMPLE OF TP53 MUTATION CARRIERS.” 2014. Masters Thesis, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/459.

MLA Handbook (7th Edition):

Parham, Emily A. “EVALUATION OF CURRENT CLINICAL CRITERIA FOR LI-FRAUMENI SYNDROME IN A DIVERSE SAMPLE OF TP53 MUTATION CARRIERS.” 2014. Web. 23 Oct 2019.

Vancouver:

Parham EA. EVALUATION OF CURRENT CLINICAL CRITERIA FOR LI-FRAUMENI SYNDROME IN A DIVERSE SAMPLE OF TP53 MUTATION CARRIERS. [Internet] [Masters thesis]. Texas Medical Center; 2014. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/459.

Council of Science Editors:

Parham EA. EVALUATION OF CURRENT CLINICAL CRITERIA FOR LI-FRAUMENI SYNDROME IN A DIVERSE SAMPLE OF TP53 MUTATION CARRIERS. [Masters Thesis]. Texas Medical Center; 2014. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/459


Texas Medical Center

20. Thokala, Radhika. REDIRECTING T CELLS WITH CHIMERIC ANTIGEN RECEPTORS TO TARGET CD123+ LEUKEMIA.

Degree: PhD, 2015, Texas Medical Center

 <h1>ABSTRAC</h1> <h1> REDIRECTING T CELLS WITH CHIMERIC ANTIGEN RECEPTORS TO TARGET CD123+ LEUKEMIA</h1> Radhika Thokala, Ph.D* Advisory Professor: Dean Anthony Lee, M.D, Ph.D CD123 or… (more)

Subjects/Keywords: CD123; Chimeric Antigen Receptors; Sleeping Beauty System; Medicine and Health Sciences; Translational Medical Research

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APA (6th Edition):

Thokala, R. (2015). REDIRECTING T CELLS WITH CHIMERIC ANTIGEN RECEPTORS TO TARGET CD123+ LEUKEMIA. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/644

Chicago Manual of Style (16th Edition):

Thokala, Radhika. “REDIRECTING T CELLS WITH CHIMERIC ANTIGEN RECEPTORS TO TARGET CD123+ LEUKEMIA.” 2015. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/644.

MLA Handbook (7th Edition):

Thokala, Radhika. “REDIRECTING T CELLS WITH CHIMERIC ANTIGEN RECEPTORS TO TARGET CD123+ LEUKEMIA.” 2015. Web. 23 Oct 2019.

Vancouver:

Thokala R. REDIRECTING T CELLS WITH CHIMERIC ANTIGEN RECEPTORS TO TARGET CD123+ LEUKEMIA. [Internet] [Doctoral dissertation]. Texas Medical Center; 2015. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/644.

Council of Science Editors:

Thokala R. REDIRECTING T CELLS WITH CHIMERIC ANTIGEN RECEPTORS TO TARGET CD123+ LEUKEMIA. [Doctoral Dissertation]. Texas Medical Center; 2015. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/644


Texas Medical Center

21. Lim, Tze Yee. Encapsulated Contrast Agent Markers for MRI-based Post-implant Dosimetry.

Degree: PhD, 2016, Texas Medical Center

  Low-dose-rate prostate brachytherapy involves the implantation of tiny radioactive seeds into the prostate to treat prostate cancer. The current standard post-implant imaging modality is… (more)

Subjects/Keywords: low dose rate; prostate; brachytherapy; MRI; markers; Medical Biophysics; Medicine and Health Sciences

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APA (6th Edition):

Lim, T. Y. (2016). Encapsulated Contrast Agent Markers for MRI-based Post-implant Dosimetry. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/691

Chicago Manual of Style (16th Edition):

Lim, Tze Yee. “Encapsulated Contrast Agent Markers for MRI-based Post-implant Dosimetry.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/691.

MLA Handbook (7th Edition):

Lim, Tze Yee. “Encapsulated Contrast Agent Markers for MRI-based Post-implant Dosimetry.” 2016. Web. 23 Oct 2019.

Vancouver:

Lim TY. Encapsulated Contrast Agent Markers for MRI-based Post-implant Dosimetry. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/691.

Council of Science Editors:

Lim TY. Encapsulated Contrast Agent Markers for MRI-based Post-implant Dosimetry. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/691


Texas Medical Center

22. Weygand, Joseph; and#60;pand#62;0000-0002-1508-9162and#60;/pand#62. IDENTIFYING THE IMMUNE RELATED METABOLIC PROPERTIES OF PANCREATIC CANCER USING NUCLEAR MAGNETIC RESONANCE SPECTROSCOPY AND DYNAMIC MAGNETIC RESONANCE SPECTROSCOPIC IMAGING WITH HYPERPOLARIZED PYRUVATE.

Degree: MS, 2017, Texas Medical Center

  Despite its relatively low incidence, pancreatic cancer was the fourth leading cause of cancer-related death in the US in 2015. This is due in… (more)

Subjects/Keywords: Hyperpolarization pyruvate lactate pancreatic cancer MRI NMR spectroscopy; Medical Biophysics; Medicine and Health Sciences

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APA (6th Edition):

Weygand, J. a. (2017). IDENTIFYING THE IMMUNE RELATED METABOLIC PROPERTIES OF PANCREATIC CANCER USING NUCLEAR MAGNETIC RESONANCE SPECTROSCOPY AND DYNAMIC MAGNETIC RESONANCE SPECTROSCOPIC IMAGING WITH HYPERPOLARIZED PYRUVATE. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/783

Chicago Manual of Style (16th Edition):

Weygand, Joseph; and#60;pand#62;0000-0002-1508-9162and#60;/pand#62. “IDENTIFYING THE IMMUNE RELATED METABOLIC PROPERTIES OF PANCREATIC CANCER USING NUCLEAR MAGNETIC RESONANCE SPECTROSCOPY AND DYNAMIC MAGNETIC RESONANCE SPECTROSCOPIC IMAGING WITH HYPERPOLARIZED PYRUVATE.” 2017. Masters Thesis, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/783.

MLA Handbook (7th Edition):

Weygand, Joseph; and#60;pand#62;0000-0002-1508-9162and#60;/pand#62. “IDENTIFYING THE IMMUNE RELATED METABOLIC PROPERTIES OF PANCREATIC CANCER USING NUCLEAR MAGNETIC RESONANCE SPECTROSCOPY AND DYNAMIC MAGNETIC RESONANCE SPECTROSCOPIC IMAGING WITH HYPERPOLARIZED PYRUVATE.” 2017. Web. 23 Oct 2019.

Vancouver:

Weygand Ja. IDENTIFYING THE IMMUNE RELATED METABOLIC PROPERTIES OF PANCREATIC CANCER USING NUCLEAR MAGNETIC RESONANCE SPECTROSCOPY AND DYNAMIC MAGNETIC RESONANCE SPECTROSCOPIC IMAGING WITH HYPERPOLARIZED PYRUVATE. [Internet] [Masters thesis]. Texas Medical Center; 2017. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/783.

Council of Science Editors:

Weygand Ja. IDENTIFYING THE IMMUNE RELATED METABOLIC PROPERTIES OF PANCREATIC CANCER USING NUCLEAR MAGNETIC RESONANCE SPECTROSCOPY AND DYNAMIC MAGNETIC RESONANCE SPECTROSCOPIC IMAGING WITH HYPERPOLARIZED PYRUVATE. [Masters Thesis]. Texas Medical Center; 2017. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/783


Texas Medical Center

23. Patrizz, Anthony; and#60;pand#62;https://orcid.org/0000-0001-7153-9028and#60;/pand#62. Modeling Post Stroke Respiratory Dysfunction, Apneas and Cognitive Decline.

Degree: PhD, 2017, Texas Medical Center

  Modeling Post Stroke Respiratory Dysfunction, Apneas and Cognitive Decline Anthony Patrizz, B.A. Advisory Professor: Louise McCullough M.D., Ph.D. Stroke is a major cause of… (more)

Subjects/Keywords: stroke; disordered breathing; apnea; cognitive decline; Medical Neurobiology; Medicine and Health Sciences; Neurosciences

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APA (6th Edition):

Patrizz, A. a. o. (2017). Modeling Post Stroke Respiratory Dysfunction, Apneas and Cognitive Decline. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/825

Chicago Manual of Style (16th Edition):

Patrizz, Anthony; and#60;pand#62;https://orcid org/0000-0001-7153-9028and#60;/pand#62. “Modeling Post Stroke Respiratory Dysfunction, Apneas and Cognitive Decline.” 2017. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/825.

MLA Handbook (7th Edition):

Patrizz, Anthony; and#60;pand#62;https://orcid org/0000-0001-7153-9028and#60;/pand#62. “Modeling Post Stroke Respiratory Dysfunction, Apneas and Cognitive Decline.” 2017. Web. 23 Oct 2019.

Vancouver:

Patrizz Aao. Modeling Post Stroke Respiratory Dysfunction, Apneas and Cognitive Decline. [Internet] [Doctoral dissertation]. Texas Medical Center; 2017. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/825.

Council of Science Editors:

Patrizz Aao. Modeling Post Stroke Respiratory Dysfunction, Apneas and Cognitive Decline. [Doctoral Dissertation]. Texas Medical Center; 2017. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/825


Texas Medical Center

24. Wagner, Victoria. No Difference in Health Related Quality of Life Between Therapeutic Options for Type 1 Gaucher Disease.

Degree: MS, 2016, Texas Medical Center

  Type 1 Gaucher disease (GD) is the most common lysosomal storage disorder. Previously, treatment for GD was limited to intravenous enzyme replacement therapy (ERT).… (more)

Subjects/Keywords: Gaucher disease; treatment options; health related quality of life; substrate reduction therapy; enzyme replacement therapy; Medical Genetics; Medicine and Health Sciences

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APA (6th Edition):

Wagner, V. (2016). No Difference in Health Related Quality of Life Between Therapeutic Options for Type 1 Gaucher Disease. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/654

Chicago Manual of Style (16th Edition):

Wagner, Victoria. “No Difference in Health Related Quality of Life Between Therapeutic Options for Type 1 Gaucher Disease.” 2016. Masters Thesis, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/654.

MLA Handbook (7th Edition):

Wagner, Victoria. “No Difference in Health Related Quality of Life Between Therapeutic Options for Type 1 Gaucher Disease.” 2016. Web. 23 Oct 2019.

Vancouver:

Wagner V. No Difference in Health Related Quality of Life Between Therapeutic Options for Type 1 Gaucher Disease. [Internet] [Masters thesis]. Texas Medical Center; 2016. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/654.

Council of Science Editors:

Wagner V. No Difference in Health Related Quality of Life Between Therapeutic Options for Type 1 Gaucher Disease. [Masters Thesis]. Texas Medical Center; 2016. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/654


Texas Medical Center

25. Jaiswal, Ashvin; and#60;pand#62;https://orcid.org/0000-0002-5550-5589and#60;/pand#62. TUMOR IMMUNOTHERAPY: MECHANISMS OF ACQUIRED RESISTANCE AND CHARACTERIZATION OF IMMUNE RELATED TOXICITIES.

Degree: PhD, 2018, Texas Medical Center

  Tumor immunotherapy has shown very promising clinical benefit across an array of cancers; however, two major challenges remain unresolved in the field. First, many… (more)

Subjects/Keywords: Immunotherapy Resistance; Immunooncology; CTLA-4; PD-1; PD-L1; 4-1BB; Immune Related Adverse Effects (IRAEs); Immunometabolism; Checkpoint Blockade Immunotherapy; Hepatotoxicity; Genetic Processes; Immunity; Immunology and Infectious Disease; Immunopathology; Immunoprophylaxis and Therapy; Medical Biochemistry; Medical Biotechnology; Medical Genetics; Medical Immunology; Medicine and Health Sciences

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APA (6th Edition):

Jaiswal, A. a. o. (2018). TUMOR IMMUNOTHERAPY: MECHANISMS OF ACQUIRED RESISTANCE AND CHARACTERIZATION OF IMMUNE RELATED TOXICITIES. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/832

Chicago Manual of Style (16th Edition):

Jaiswal, Ashvin; and#60;pand#62;https://orcid org/0000-0002-5550-5589and#60;/pand#62. “TUMOR IMMUNOTHERAPY: MECHANISMS OF ACQUIRED RESISTANCE AND CHARACTERIZATION OF IMMUNE RELATED TOXICITIES.” 2018. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/832.

MLA Handbook (7th Edition):

Jaiswal, Ashvin; and#60;pand#62;https://orcid org/0000-0002-5550-5589and#60;/pand#62. “TUMOR IMMUNOTHERAPY: MECHANISMS OF ACQUIRED RESISTANCE AND CHARACTERIZATION OF IMMUNE RELATED TOXICITIES.” 2018. Web. 23 Oct 2019.

Vancouver:

Jaiswal Aao. TUMOR IMMUNOTHERAPY: MECHANISMS OF ACQUIRED RESISTANCE AND CHARACTERIZATION OF IMMUNE RELATED TOXICITIES. [Internet] [Doctoral dissertation]. Texas Medical Center; 2018. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/832.

Council of Science Editors:

Jaiswal Aao. TUMOR IMMUNOTHERAPY: MECHANISMS OF ACQUIRED RESISTANCE AND CHARACTERIZATION OF IMMUNE RELATED TOXICITIES. [Doctoral Dissertation]. Texas Medical Center; 2018. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/832


Texas Medical Center

26. Liu, Qian. ABERRATIONS OF A PUTATIVE TUMOR SUPPRESSOR GENE SEL1L IN PANCREATIC DUCTAL ADENOCARCINOMA.

Degree: PhD, 2011, Texas Medical Center

  Introduction: Pancreatic cancer is the fourth leading cause of cancer-related death among males and females in the United States. Sel-1-like (SEL1L) is a putative… (more)

Subjects/Keywords: pancreatic cancer; tumor suppressor gene; SEL1L; pancreatic ductal adenocarcinoma; loss of heterozygosity; methylation; mutation; aberrant splicing; microRNA; Medical Cell Biology; Medical Genetics; Medicine and Health Sciences

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APA (6th Edition):

Liu, Q. (2011). ABERRATIONS OF A PUTATIVE TUMOR SUPPRESSOR GENE SEL1L IN PANCREATIC DUCTAL ADENOCARCINOMA. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/178

Chicago Manual of Style (16th Edition):

Liu, Qian. “ABERRATIONS OF A PUTATIVE TUMOR SUPPRESSOR GENE SEL1L IN PANCREATIC DUCTAL ADENOCARCINOMA.” 2011. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/178.

MLA Handbook (7th Edition):

Liu, Qian. “ABERRATIONS OF A PUTATIVE TUMOR SUPPRESSOR GENE SEL1L IN PANCREATIC DUCTAL ADENOCARCINOMA.” 2011. Web. 23 Oct 2019.

Vancouver:

Liu Q. ABERRATIONS OF A PUTATIVE TUMOR SUPPRESSOR GENE SEL1L IN PANCREATIC DUCTAL ADENOCARCINOMA. [Internet] [Doctoral dissertation]. Texas Medical Center; 2011. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/178.

Council of Science Editors:

Liu Q. ABERRATIONS OF A PUTATIVE TUMOR SUPPRESSOR GENE SEL1L IN PANCREATIC DUCTAL ADENOCARCINOMA. [Doctoral Dissertation]. Texas Medical Center; 2011. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/178


Texas Medical Center

27. Ow, Thomas J, MD. TP53 as a Biomarker in Head and Neck Squamous Cell Carcinoma.

Degree: MS, 2011, Texas Medical Center

  Currently, there are no molecular biomarkers that guide treatment decisions for patients with head and neck squamous cell carcinoma (HNSCC). Several retrospective studies have… (more)

Subjects/Keywords: TP53; Head and Neck Squamous Cell Carcinoma; Biomarker; Analytical, Diagnostic and Therapeutic Techniques and Equipment; Medical Molecular Biology; Medicine and Health Sciences; Oncology; Otolaryngology; Translational Medical Research

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APA (6th Edition):

Ow, Thomas J, M. (2011). TP53 as a Biomarker in Head and Neck Squamous Cell Carcinoma. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/217

Chicago Manual of Style (16th Edition):

Ow, Thomas J, MD. “TP53 as a Biomarker in Head and Neck Squamous Cell Carcinoma.” 2011. Masters Thesis, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/217.

MLA Handbook (7th Edition):

Ow, Thomas J, MD. “TP53 as a Biomarker in Head and Neck Squamous Cell Carcinoma.” 2011. Web. 23 Oct 2019.

Vancouver:

Ow, Thomas J M. TP53 as a Biomarker in Head and Neck Squamous Cell Carcinoma. [Internet] [Masters thesis]. Texas Medical Center; 2011. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/217.

Council of Science Editors:

Ow, Thomas J M. TP53 as a Biomarker in Head and Neck Squamous Cell Carcinoma. [Masters Thesis]. Texas Medical Center; 2011. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/217


Texas Medical Center

28. Hurton, Lenka. TETHERED IL-15 TO AUGMENT THE THERAPEUTIC POTENTIAL OF T CELLS EXPRESSING CHIMERIC ANTIGEN RECEPTOR: MAINTAINING MEMORY POTENTIAL, PERSISTENCE, AND ANTITUMOR ACTIVITY.

Degree: PhD, 2014, Texas Medical Center

  Tethered IL-15 to augment the therapeutic potential of T cells expressing chimeric antigen receptor: Maintaining memory potential, persistence, and antitumor activity Adoptive immunotherapy can… (more)

Subjects/Keywords: immunotherapy; cytokines; IL-15; chimeric antigen receptor; T-cell persistence; memory; memory stem cell; leukemia; B-ALL; Medical Biotechnology; Medical Immunology; Medicine and Health Sciences; Other Immunology and Infectious Disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hurton, L. (2014). TETHERED IL-15 TO AUGMENT THE THERAPEUTIC POTENTIAL OF T CELLS EXPRESSING CHIMERIC ANTIGEN RECEPTOR: MAINTAINING MEMORY POTENTIAL, PERSISTENCE, AND ANTITUMOR ACTIVITY. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/421

Chicago Manual of Style (16th Edition):

Hurton, Lenka. “TETHERED IL-15 TO AUGMENT THE THERAPEUTIC POTENTIAL OF T CELLS EXPRESSING CHIMERIC ANTIGEN RECEPTOR: MAINTAINING MEMORY POTENTIAL, PERSISTENCE, AND ANTITUMOR ACTIVITY.” 2014. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/421.

MLA Handbook (7th Edition):

Hurton, Lenka. “TETHERED IL-15 TO AUGMENT THE THERAPEUTIC POTENTIAL OF T CELLS EXPRESSING CHIMERIC ANTIGEN RECEPTOR: MAINTAINING MEMORY POTENTIAL, PERSISTENCE, AND ANTITUMOR ACTIVITY.” 2014. Web. 23 Oct 2019.

Vancouver:

Hurton L. TETHERED IL-15 TO AUGMENT THE THERAPEUTIC POTENTIAL OF T CELLS EXPRESSING CHIMERIC ANTIGEN RECEPTOR: MAINTAINING MEMORY POTENTIAL, PERSISTENCE, AND ANTITUMOR ACTIVITY. [Internet] [Doctoral dissertation]. Texas Medical Center; 2014. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/421.

Council of Science Editors:

Hurton L. TETHERED IL-15 TO AUGMENT THE THERAPEUTIC POTENTIAL OF T CELLS EXPRESSING CHIMERIC ANTIGEN RECEPTOR: MAINTAINING MEMORY POTENTIAL, PERSISTENCE, AND ANTITUMOR ACTIVITY. [Doctoral Dissertation]. Texas Medical Center; 2014. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/421


Texas Medical Center

29. Minter, Lindsey C. DISSECTING THE ROLES OF TRIM24 IN REGULATION OF HEPATIC LIPID METABOLISM AND INFLAMMATION.

Degree: PhD, 2014, Texas Medical Center

  DISSECTING THE ROLES OF TRIM24 IN REGULATION OF HEPATIC LIPID METABOLISM AND INFLAMMATION Lindsey Cauthen Minter, B.S., B.A. Advisory Professor: Michelle C. Barton, Ph.D.… (more)

Subjects/Keywords: Trim24; Non-alcoholic fatty liver disease; Non-alcohlic steatohepatitis; Hepatocellular carcinoma; Hepatic lipid metabolism; Inflammation; Medical Genetics; Medical Sciences; Medicine and Health Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Minter, L. C. (2014). DISSECTING THE ROLES OF TRIM24 IN REGULATION OF HEPATIC LIPID METABOLISM AND INFLAMMATION. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/497

Chicago Manual of Style (16th Edition):

Minter, Lindsey C. “DISSECTING THE ROLES OF TRIM24 IN REGULATION OF HEPATIC LIPID METABOLISM AND INFLAMMATION.” 2014. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/497.

MLA Handbook (7th Edition):

Minter, Lindsey C. “DISSECTING THE ROLES OF TRIM24 IN REGULATION OF HEPATIC LIPID METABOLISM AND INFLAMMATION.” 2014. Web. 23 Oct 2019.

Vancouver:

Minter LC. DISSECTING THE ROLES OF TRIM24 IN REGULATION OF HEPATIC LIPID METABOLISM AND INFLAMMATION. [Internet] [Doctoral dissertation]. Texas Medical Center; 2014. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/497.

Council of Science Editors:

Minter LC. DISSECTING THE ROLES OF TRIM24 IN REGULATION OF HEPATIC LIPID METABOLISM AND INFLAMMATION. [Doctoral Dissertation]. Texas Medical Center; 2014. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/497


Texas Medical Center

30. Vishwamitra, Deeksha. MULTILEVEL DEREGULATION OF SURVIVAL MECHANISMS IN NPM-ALK+ T-CELL LYMPHOMA.

Degree: PhD, 2015, Texas Medical Center

  The anaplastic lymphoma kinase (ALK) is a single chain transmembrane receptor tyrosine kinase that belongs to the insulin receptor superfamily. Other members of this… (more)

Subjects/Keywords: IGF-IR; NPM-ALK; lymphoma; cancer signaling; miRNA; SUMOylation; gene regulation; cancer; Hematology; Laboratory and Basic Science Research; Medical Cell Biology; Medical Molecular Biology; Medicine and Health Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vishwamitra, D. (2015). MULTILEVEL DEREGULATION OF SURVIVAL MECHANISMS IN NPM-ALK+ T-CELL LYMPHOMA. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/554

Chicago Manual of Style (16th Edition):

Vishwamitra, Deeksha. “MULTILEVEL DEREGULATION OF SURVIVAL MECHANISMS IN NPM-ALK+ T-CELL LYMPHOMA.” 2015. Doctoral Dissertation, Texas Medical Center. Accessed October 23, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/554.

MLA Handbook (7th Edition):

Vishwamitra, Deeksha. “MULTILEVEL DEREGULATION OF SURVIVAL MECHANISMS IN NPM-ALK+ T-CELL LYMPHOMA.” 2015. Web. 23 Oct 2019.

Vancouver:

Vishwamitra D. MULTILEVEL DEREGULATION OF SURVIVAL MECHANISMS IN NPM-ALK+ T-CELL LYMPHOMA. [Internet] [Doctoral dissertation]. Texas Medical Center; 2015. [cited 2019 Oct 23]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/554.

Council of Science Editors:

Vishwamitra D. MULTILEVEL DEREGULATION OF SURVIVAL MECHANISMS IN NPM-ALK+ T-CELL LYMPHOMA. [Doctoral Dissertation]. Texas Medical Center; 2015. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/554

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