Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(HMOX1). Showing records 1 – 7 of 7 total matches.

Search Limiters

Last 2 Years | English Only

No search limiters apply to these results.

▼ Search Limiters


University of Helsinki

1. Lim, Nancy. The Role of Heme Oxygenase 1 Gene Promoter Variant in Shaping the Clinical Phenotype of Carotid Artery Disease.

Degree: Medicinska fakulteten, 2017, University of Helsinki

 Background: Atherosclerosis is a common pathological process in arteries causing significant morbidity and mortality due to stroke and myocardial infarction. The progression of atherosclerosis is… (more)

Subjects/Keywords: stroke; atherosclerosis; carotid artery disease; HMOX1; HMOX1 promoter variants; HO1; Molecular neurology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lim, N. (2017). The Role of Heme Oxygenase 1 Gene Promoter Variant in Shaping the Clinical Phenotype of Carotid Artery Disease. (Masters Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/228717

Chicago Manual of Style (16th Edition):

Lim, Nancy. “The Role of Heme Oxygenase 1 Gene Promoter Variant in Shaping the Clinical Phenotype of Carotid Artery Disease.” 2017. Masters Thesis, University of Helsinki. Accessed January 20, 2021. http://hdl.handle.net/10138/228717.

MLA Handbook (7th Edition):

Lim, Nancy. “The Role of Heme Oxygenase 1 Gene Promoter Variant in Shaping the Clinical Phenotype of Carotid Artery Disease.” 2017. Web. 20 Jan 2021.

Vancouver:

Lim N. The Role of Heme Oxygenase 1 Gene Promoter Variant in Shaping the Clinical Phenotype of Carotid Artery Disease. [Internet] [Masters thesis]. University of Helsinki; 2017. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/10138/228717.

Council of Science Editors:

Lim N. The Role of Heme Oxygenase 1 Gene Promoter Variant in Shaping the Clinical Phenotype of Carotid Artery Disease. [Masters Thesis]. University of Helsinki; 2017. Available from: http://hdl.handle.net/10138/228717


University of Iowa

2. Boylston, Jennifer A. FHIT inactivation combined with cigarette smoke enhances the oxidative stress response.

Degree: PhD, Molecular and Cellular Biology, 2013, University of Iowa

  The FHIT gene is located on the most fragile site in the human genome. FHIT gene deletions are among the earliest and most frequent… (more)

Subjects/Keywords: Bach1; Bronchial Epithelial Cells; Cigarette Smoke; Fhit; Hmox1; Oxidative Stress; Cell Biology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Boylston, J. A. (2013). FHIT inactivation combined with cigarette smoke enhances the oxidative stress response. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/1295

Chicago Manual of Style (16th Edition):

Boylston, Jennifer A. “FHIT inactivation combined with cigarette smoke enhances the oxidative stress response.” 2013. Doctoral Dissertation, University of Iowa. Accessed January 20, 2021. https://ir.uiowa.edu/etd/1295.

MLA Handbook (7th Edition):

Boylston, Jennifer A. “FHIT inactivation combined with cigarette smoke enhances the oxidative stress response.” 2013. Web. 20 Jan 2021.

Vancouver:

Boylston JA. FHIT inactivation combined with cigarette smoke enhances the oxidative stress response. [Internet] [Doctoral dissertation]. University of Iowa; 2013. [cited 2021 Jan 20]. Available from: https://ir.uiowa.edu/etd/1295.

Council of Science Editors:

Boylston JA. FHIT inactivation combined with cigarette smoke enhances the oxidative stress response. [Doctoral Dissertation]. University of Iowa; 2013. Available from: https://ir.uiowa.edu/etd/1295


University of Gothenburg / Göteborgs Universitet

3. Andersson, Heléne. Reactive gliosis in the injured brain: The effect of cell communication and Nrf2-mediated cellular defence.

Degree: 2011, University of Gothenburg / Göteborgs Universitet

 Stroke and other brain injuries trigger an extensive glial cell response referred to as reactive gliosis. Reactive gliosis is characterized by hypertrophic and proliferating astrocytes,… (more)

Subjects/Keywords: Keywords: astrocyte; gap junction; Hmox1; microglia; Nrf2; Nqo1; oxidative stress; reactive gliosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Andersson, H. (2011). Reactive gliosis in the injured brain: The effect of cell communication and Nrf2-mediated cellular defence. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/23937

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Andersson, Heléne. “Reactive gliosis in the injured brain: The effect of cell communication and Nrf2-mediated cellular defence.” 2011. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed January 20, 2021. http://hdl.handle.net/2077/23937.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Andersson, Heléne. “Reactive gliosis in the injured brain: The effect of cell communication and Nrf2-mediated cellular defence.” 2011. Web. 20 Jan 2021.

Vancouver:

Andersson H. Reactive gliosis in the injured brain: The effect of cell communication and Nrf2-mediated cellular defence. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2011. [cited 2021 Jan 20]. Available from: http://hdl.handle.net/2077/23937.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Andersson H. Reactive gliosis in the injured brain: The effect of cell communication and Nrf2-mediated cellular defence. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2011. Available from: http://hdl.handle.net/2077/23937

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

4. Ferguson, Jeanette M. An examination of genetic polymorphisms in the enzyme heme oxygenase-1 and their relationship to cardiovascular disease.

Degree: PhD, Pathology, 2005, The Ohio State University

  Heme oxygenase-1 (HO-1) plays a critical role in protecting the cardiovascular system from the damaging effects of oxidative stress. The anti-inflammatory, antioxidant, antiproliferative and… (more)

Subjects/Keywords: Heme Oxygenase-1; HMOX1 Microsatellite Polymorphism; HMOX1 SNP-413; Cardiovascular Disease; Genetic Polymorphisms in African Americans

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ferguson, J. M. (2005). An examination of genetic polymorphisms in the enzyme heme oxygenase-1 and their relationship to cardiovascular disease. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1124123173

Chicago Manual of Style (16th Edition):

Ferguson, Jeanette M. “An examination of genetic polymorphisms in the enzyme heme oxygenase-1 and their relationship to cardiovascular disease.” 2005. Doctoral Dissertation, The Ohio State University. Accessed January 20, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=osu1124123173.

MLA Handbook (7th Edition):

Ferguson, Jeanette M. “An examination of genetic polymorphisms in the enzyme heme oxygenase-1 and their relationship to cardiovascular disease.” 2005. Web. 20 Jan 2021.

Vancouver:

Ferguson JM. An examination of genetic polymorphisms in the enzyme heme oxygenase-1 and their relationship to cardiovascular disease. [Internet] [Doctoral dissertation]. The Ohio State University; 2005. [cited 2021 Jan 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1124123173.

Council of Science Editors:

Ferguson JM. An examination of genetic polymorphisms in the enzyme heme oxygenase-1 and their relationship to cardiovascular disease. [Doctoral Dissertation]. The Ohio State University; 2005. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1124123173


University of Oulu

5. Lämsä, V. (Virpi). Regulation of murine hepatic Cytochrome P450 2a5 expression by transcription factor Nuclear factor (erythroid-derived 2)-like 2.

Degree: 2012, University of Oulu

Abstract The hepatic inducible Cytochrome P450s (CYPs) generally prime xenobiotics for elimination. Murine CYP2A5 and human CYP2A6 share similar xenobiotic substrates and some regulatory features.… (more)

Subjects/Keywords: Bach1; CYP2A5; HMOX1; Nrf2; PGC-1α; heavy metals; heme homeostasis; hepatotoxicity; liver; redox homeostasis; transcriptional regulation; xenobiotic metabolism; aineenvaihdunta; antioksidantit; entsyymit; geeniekspressio; geenitutkimus; hapettuminen; maksa; markkerit; mitokondriot; oksidantit; raskasmetallit; sytokromit; toksikologia; transkriptio; vierasaineet

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lämsä, V. (. (2012). Regulation of murine hepatic Cytochrome P450 2a5 expression by transcription factor Nuclear factor (erythroid-derived 2)-like 2. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789514299278

Chicago Manual of Style (16th Edition):

Lämsä, V (Virpi). “Regulation of murine hepatic Cytochrome P450 2a5 expression by transcription factor Nuclear factor (erythroid-derived 2)-like 2.” 2012. Doctoral Dissertation, University of Oulu. Accessed January 20, 2021. http://urn.fi/urn:isbn:9789514299278.

MLA Handbook (7th Edition):

Lämsä, V (Virpi). “Regulation of murine hepatic Cytochrome P450 2a5 expression by transcription factor Nuclear factor (erythroid-derived 2)-like 2.” 2012. Web. 20 Jan 2021.

Vancouver:

Lämsä V(. Regulation of murine hepatic Cytochrome P450 2a5 expression by transcription factor Nuclear factor (erythroid-derived 2)-like 2. [Internet] [Doctoral dissertation]. University of Oulu; 2012. [cited 2021 Jan 20]. Available from: http://urn.fi/urn:isbn:9789514299278.

Council of Science Editors:

Lämsä V(. Regulation of murine hepatic Cytochrome P450 2a5 expression by transcription factor Nuclear factor (erythroid-derived 2)-like 2. [Doctoral Dissertation]. University of Oulu; 2012. Available from: http://urn.fi/urn:isbn:9789514299278


University of Cincinnati

6. MA, CI, Miss. THE TRANSCRIPTION FACTOR BTB AND CNC HOMOLOG 1 IN THE REGULATION OF CELL DIFFERENTIATION AND ORGANOGENESIS.

Degree: MS, Medicine : Toxicology (Environmental Health), 2007, University of Cincinnati

 BACH1 (BTB and CNC homolog 1) is a transcription factor that belongs to the CNC (cap'n'collar)-type bZip (basic region leucine zipper) transcription factor family. A… (more)

Subjects/Keywords: Health Sciences, Toxicology; BACH1; HMOX1; NQO1; transcription factor; stably-transfection; real-time PCR; western blotting

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

MA, CI, M. (2007). THE TRANSCRIPTION FACTOR BTB AND CNC HOMOLOG 1 IN THE REGULATION OF CELL DIFFERENTIATION AND ORGANOGENESIS. (Masters Thesis). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1185995222

Chicago Manual of Style (16th Edition):

MA, CI, Miss. “THE TRANSCRIPTION FACTOR BTB AND CNC HOMOLOG 1 IN THE REGULATION OF CELL DIFFERENTIATION AND ORGANOGENESIS.” 2007. Masters Thesis, University of Cincinnati. Accessed January 20, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1185995222.

MLA Handbook (7th Edition):

MA, CI, Miss. “THE TRANSCRIPTION FACTOR BTB AND CNC HOMOLOG 1 IN THE REGULATION OF CELL DIFFERENTIATION AND ORGANOGENESIS.” 2007. Web. 20 Jan 2021.

Vancouver:

MA, CI M. THE TRANSCRIPTION FACTOR BTB AND CNC HOMOLOG 1 IN THE REGULATION OF CELL DIFFERENTIATION AND ORGANOGENESIS. [Internet] [Masters thesis]. University of Cincinnati; 2007. [cited 2021 Jan 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1185995222.

Council of Science Editors:

MA, CI M. THE TRANSCRIPTION FACTOR BTB AND CNC HOMOLOG 1 IN THE REGULATION OF CELL DIFFERENTIATION AND ORGANOGENESIS. [Masters Thesis]. University of Cincinnati; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1185995222


University of Queensland

7. Hu, Hao. Transcriptional regulation of human stress responsive cytochrome P450 2A6 (CYP2A6) by p53.

Degree: School of Medicine, 2016, University of Queensland

Subjects/Keywords: Cytochrome P450 2A6 (CYP2A6); Bilirubin (BR); Haem oxygenase1 (HMOX1); Benzo[α]pyrene (BaP); Nuclear factor erythroid 2-like 2 (Nrf-2); Tumour suppressor p53; Constitutive androstane receptor (CAR); CEBP α; Octamer-1 (Oct-1); Hepatocyte nuclear factor-4α (HNF-4α); 060107 Enzymes; 060111 Signal Transduction; 060199 Biochemistry and Cell Biology not elsewhere classified

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hu, H. (2016). Transcriptional regulation of human stress responsive cytochrome P450 2A6 (CYP2A6) by p53. (Thesis). University of Queensland. Retrieved from http://espace.library.uq.edu.au/view/UQ:405743

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hu, Hao. “Transcriptional regulation of human stress responsive cytochrome P450 2A6 (CYP2A6) by p53.” 2016. Thesis, University of Queensland. Accessed January 20, 2021. http://espace.library.uq.edu.au/view/UQ:405743.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hu, Hao. “Transcriptional regulation of human stress responsive cytochrome P450 2A6 (CYP2A6) by p53.” 2016. Web. 20 Jan 2021.

Vancouver:

Hu H. Transcriptional regulation of human stress responsive cytochrome P450 2A6 (CYP2A6) by p53. [Internet] [Thesis]. University of Queensland; 2016. [cited 2021 Jan 20]. Available from: http://espace.library.uq.edu.au/view/UQ:405743.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hu H. Transcriptional regulation of human stress responsive cytochrome P450 2A6 (CYP2A6) by p53. [Thesis]. University of Queensland; 2016. Available from: http://espace.library.uq.edu.au/view/UQ:405743

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.