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You searched for subject:(HMGB1). Showing records 1 – 30 of 41 total matches.

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Université Laval

1. Azelmat, Jabrane. Rôle de la protéine HMGB1 et du récepteur RAGE dans l'étiopathogenèse des infections parodontales.

Degree: 2009, Université Laval

 Le but de ce projet est d'étudier le rôle de HMGB1 et son récepteur RAGE dans l'étiopathogenèse des maladies parodontales. Nos résultats montrent que les… (more)

Subjects/Keywords: Protéine HMGB1; Protéine HMGB1  – Récepteurs; Parodontopathies  – Microbiologie

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Azelmat, J. (2009). Rôle de la protéine HMGB1 et du récepteur RAGE dans l'étiopathogenèse des infections parodontales. (Thesis). Université Laval. Retrieved from http://hdl.handle.net/20.500.11794/21098

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Azelmat, Jabrane. “Rôle de la protéine HMGB1 et du récepteur RAGE dans l'étiopathogenèse des infections parodontales.” 2009. Thesis, Université Laval. Accessed January 20, 2020. http://hdl.handle.net/20.500.11794/21098.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Azelmat, Jabrane. “Rôle de la protéine HMGB1 et du récepteur RAGE dans l'étiopathogenèse des infections parodontales.” 2009. Web. 20 Jan 2020.

Vancouver:

Azelmat J. Rôle de la protéine HMGB1 et du récepteur RAGE dans l'étiopathogenèse des infections parodontales. [Internet] [Thesis]. Université Laval; 2009. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/20.500.11794/21098.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Azelmat J. Rôle de la protéine HMGB1 et du récepteur RAGE dans l'étiopathogenèse des infections parodontales. [Thesis]. Université Laval; 2009. Available from: http://hdl.handle.net/20.500.11794/21098

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Sigaut, Stéphanie. Activation microgliale : mécanismes et conséquences à long terme : Microglial activation : mechanisms and long term consequences.

Degree: Docteur es, Physiologie et biologie des organismes, populations, interactions. Neurosciences, 2017, Sorbonne Paris Cité

La neuro-inflammation induite par l'inflammation systémique ou générée en réponse à une lésion cérébrale aiguë a des conséquences cliniques néfastes : elle est mise en… (more)

Subjects/Keywords: HMGB1; Neuro-inflammation; Prematurity; DAMP; HMGB1

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APA (6th Edition):

Sigaut, S. (2017). Activation microgliale : mécanismes et conséquences à long terme : Microglial activation : mechanisms and long term consequences. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2017USPCC198

Chicago Manual of Style (16th Edition):

Sigaut, Stéphanie. “Activation microgliale : mécanismes et conséquences à long terme : Microglial activation : mechanisms and long term consequences.” 2017. Doctoral Dissertation, Sorbonne Paris Cité. Accessed January 20, 2020. http://www.theses.fr/2017USPCC198.

MLA Handbook (7th Edition):

Sigaut, Stéphanie. “Activation microgliale : mécanismes et conséquences à long terme : Microglial activation : mechanisms and long term consequences.” 2017. Web. 20 Jan 2020.

Vancouver:

Sigaut S. Activation microgliale : mécanismes et conséquences à long terme : Microglial activation : mechanisms and long term consequences. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2017. [cited 2020 Jan 20]. Available from: http://www.theses.fr/2017USPCC198.

Council of Science Editors:

Sigaut S. Activation microgliale : mécanismes et conséquences à long terme : Microglial activation : mechanisms and long term consequences. [Doctoral Dissertation]. Sorbonne Paris Cité; 2017. Available from: http://www.theses.fr/2017USPCC198


Bowling Green State University

3. Sarpong, Yaw Acheampong. Influence of HMGB1 on Nucleosome Structure and Estrogen Receptor Binding Affinity to Concensus Estrogen Response Element on Nucleosomal DNA.

Degree: PhD, Biological Sciences, 2010, Bowling Green State University

  Previous work from our lab has shown the high mobility group 1 protein (HMGB1) facilitates the binding of estrogen receptor (ER) to DNAs that… (more)

Subjects/Keywords: Biochemistry; nucleosomes; HMGB1

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APA (6th Edition):

Sarpong, Y. A. (2010). Influence of HMGB1 on Nucleosome Structure and Estrogen Receptor Binding Affinity to Concensus Estrogen Response Element on Nucleosomal DNA. (Doctoral Dissertation). Bowling Green State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1288629979

Chicago Manual of Style (16th Edition):

Sarpong, Yaw Acheampong. “Influence of HMGB1 on Nucleosome Structure and Estrogen Receptor Binding Affinity to Concensus Estrogen Response Element on Nucleosomal DNA.” 2010. Doctoral Dissertation, Bowling Green State University. Accessed January 20, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1288629979.

MLA Handbook (7th Edition):

Sarpong, Yaw Acheampong. “Influence of HMGB1 on Nucleosome Structure and Estrogen Receptor Binding Affinity to Concensus Estrogen Response Element on Nucleosomal DNA.” 2010. Web. 20 Jan 2020.

Vancouver:

Sarpong YA. Influence of HMGB1 on Nucleosome Structure and Estrogen Receptor Binding Affinity to Concensus Estrogen Response Element on Nucleosomal DNA. [Internet] [Doctoral dissertation]. Bowling Green State University; 2010. [cited 2020 Jan 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1288629979.

Council of Science Editors:

Sarpong YA. Influence of HMGB1 on Nucleosome Structure and Estrogen Receptor Binding Affinity to Concensus Estrogen Response Element on Nucleosomal DNA. [Doctoral Dissertation]. Bowling Green State University; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1288629979


University of Hawaii – Manoa

4. Napolitano, Andrea. Chronic Inflammation in Malignant Mesothelioma Pathogenesis: Focus on HMGB1 Isoforms and Germline BAP1 Mutations.

Degree: 2017, University of Hawaii – Manoa

Ph.D. University of Hawaii at Manoa 2015.

Malignant mesothelioma (MM) is an aggressive cancer arising from the mesothelial cells forming the lining of the pleural,… (more)

Subjects/Keywords: mesothelioma; inammation; BAP1; HMGB1

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APA (6th Edition):

Napolitano, A. (2017). Chronic Inflammation in Malignant Mesothelioma Pathogenesis: Focus on HMGB1 Isoforms and Germline BAP1 Mutations. (Thesis). University of Hawaii – Manoa. Retrieved from http://hdl.handle.net/10125/51634

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Napolitano, Andrea. “Chronic Inflammation in Malignant Mesothelioma Pathogenesis: Focus on HMGB1 Isoforms and Germline BAP1 Mutations.” 2017. Thesis, University of Hawaii – Manoa. Accessed January 20, 2020. http://hdl.handle.net/10125/51634.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Napolitano, Andrea. “Chronic Inflammation in Malignant Mesothelioma Pathogenesis: Focus on HMGB1 Isoforms and Germline BAP1 Mutations.” 2017. Web. 20 Jan 2020.

Vancouver:

Napolitano A. Chronic Inflammation in Malignant Mesothelioma Pathogenesis: Focus on HMGB1 Isoforms and Germline BAP1 Mutations. [Internet] [Thesis]. University of Hawaii – Manoa; 2017. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/10125/51634.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Napolitano A. Chronic Inflammation in Malignant Mesothelioma Pathogenesis: Focus on HMGB1 Isoforms and Germline BAP1 Mutations. [Thesis]. University of Hawaii – Manoa; 2017. Available from: http://hdl.handle.net/10125/51634

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Personnaz, Jean. Rôle de la protéine HMGB1 dans la stéatose hépatique associée à l'obésité : Role of HMGB1 protein in obesity-related liver steatosis.

Degree: Docteur es, Pharmacologie, 2018, Université Toulouse III – Paul Sabatier

Au cours de l'obésité, l'excès de lipides circulants, est stocké dans les organes périphériques, principalement dans le foie. Ce stockage ectopique de lipide peut avoir,… (more)

Subjects/Keywords: HMGB1; Stéatose hépatique; Synthèse des lipides hépatiques; HMGB1; Hepatic steatosis; Hepatic lipids synthesis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Personnaz, J. (2018). Rôle de la protéine HMGB1 dans la stéatose hépatique associée à l'obésité : Role of HMGB1 protein in obesity-related liver steatosis. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2018TOU30241

Chicago Manual of Style (16th Edition):

Personnaz, Jean. “Rôle de la protéine HMGB1 dans la stéatose hépatique associée à l'obésité : Role of HMGB1 protein in obesity-related liver steatosis.” 2018. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed January 20, 2020. http://www.theses.fr/2018TOU30241.

MLA Handbook (7th Edition):

Personnaz, Jean. “Rôle de la protéine HMGB1 dans la stéatose hépatique associée à l'obésité : Role of HMGB1 protein in obesity-related liver steatosis.” 2018. Web. 20 Jan 2020.

Vancouver:

Personnaz J. Rôle de la protéine HMGB1 dans la stéatose hépatique associée à l'obésité : Role of HMGB1 protein in obesity-related liver steatosis. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2018. [cited 2020 Jan 20]. Available from: http://www.theses.fr/2018TOU30241.

Council of Science Editors:

Personnaz J. Rôle de la protéine HMGB1 dans la stéatose hépatique associée à l'obésité : Role of HMGB1 protein in obesity-related liver steatosis. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2018. Available from: http://www.theses.fr/2018TOU30241


Temple University

6. Chakhtoura, Marita. Novel protocols to induce tolerance to solid organ transplants.

Degree: PhD, 2016, Temple University

Microbiology and Immunology

Dendritic cells (DCs) are the sentinels of the immune system. They mature at the encounter of the appropriate stimuli or danger signals,… (more)

Subjects/Keywords: Immunology; Medicine;

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APA (6th Edition):

Chakhtoura, M. (2016). Novel protocols to induce tolerance to solid organ transplants. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,383997

Chicago Manual of Style (16th Edition):

Chakhtoura, Marita. “Novel protocols to induce tolerance to solid organ transplants.” 2016. Doctoral Dissertation, Temple University. Accessed January 20, 2020. http://digital.library.temple.edu/u?/p245801coll10,383997.

MLA Handbook (7th Edition):

Chakhtoura, Marita. “Novel protocols to induce tolerance to solid organ transplants.” 2016. Web. 20 Jan 2020.

Vancouver:

Chakhtoura M. Novel protocols to induce tolerance to solid organ transplants. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Jan 20]. Available from: http://digital.library.temple.edu/u?/p245801coll10,383997.

Council of Science Editors:

Chakhtoura M. Novel protocols to induce tolerance to solid organ transplants. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,383997


NSYSU

7. Wang, Ting-ya. Suppression of High Mobility Group Box-1 (HMGB-1) by RNAi Might Alter the Inflammatory Response During Sepsis.

Degree: Master, Biological Sciences, 2008, NSYSU

 High mobility group box 1 (HMGB-1) protein is a non-histone chromosomal protein. As a DNA binding protein, HMGB-1 is involved in the maintenance of nucleosome… (more)

Subjects/Keywords: RNA interference (RNAi); sepsis; cytokine; High mobility group box 1 (HMGB1)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, T. (2008). Suppression of High Mobility Group Box-1 (HMGB-1) by RNAi Might Alter the Inflammatory Response During Sepsis. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0904108-164845

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Ting-ya. “Suppression of High Mobility Group Box-1 (HMGB-1) by RNAi Might Alter the Inflammatory Response During Sepsis.” 2008. Thesis, NSYSU. Accessed January 20, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0904108-164845.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Ting-ya. “Suppression of High Mobility Group Box-1 (HMGB-1) by RNAi Might Alter the Inflammatory Response During Sepsis.” 2008. Web. 20 Jan 2020.

Vancouver:

Wang T. Suppression of High Mobility Group Box-1 (HMGB-1) by RNAi Might Alter the Inflammatory Response During Sepsis. [Internet] [Thesis]. NSYSU; 2008. [cited 2020 Jan 20]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0904108-164845.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang T. Suppression of High Mobility Group Box-1 (HMGB-1) by RNAi Might Alter the Inflammatory Response During Sepsis. [Thesis]. NSYSU; 2008. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0904108-164845

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Louisiana State University

8. Dange, Rahul Babulal. Role of Toll-Like Receptor 4 in Pathophysiology of Hypertension.

Degree: PhD, Medicine and Health Sciences, 2014, Louisiana State University

 Hypertension is a chronic multifactorial condition with high morbidity and mortality rates, cur-rently affecting about 1 billion people worldwide. Currently available anti-hypertensive medica-tions are found… (more)

Subjects/Keywords: Hypertension; Toll-Like Receptor; Angiotensin II; cardiac hypertrophy; HMGB1; cytokine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dange, R. B. (2014). Role of Toll-Like Receptor 4 in Pathophysiology of Hypertension. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-11072014-223156 ; https://digitalcommons.lsu.edu/gradschool_dissertations/450

Chicago Manual of Style (16th Edition):

Dange, Rahul Babulal. “Role of Toll-Like Receptor 4 in Pathophysiology of Hypertension.” 2014. Doctoral Dissertation, Louisiana State University. Accessed January 20, 2020. etd-11072014-223156 ; https://digitalcommons.lsu.edu/gradschool_dissertations/450.

MLA Handbook (7th Edition):

Dange, Rahul Babulal. “Role of Toll-Like Receptor 4 in Pathophysiology of Hypertension.” 2014. Web. 20 Jan 2020.

Vancouver:

Dange RB. Role of Toll-Like Receptor 4 in Pathophysiology of Hypertension. [Internet] [Doctoral dissertation]. Louisiana State University; 2014. [cited 2020 Jan 20]. Available from: etd-11072014-223156 ; https://digitalcommons.lsu.edu/gradschool_dissertations/450.

Council of Science Editors:

Dange RB. Role of Toll-Like Receptor 4 in Pathophysiology of Hypertension. [Doctoral Dissertation]. Louisiana State University; 2014. Available from: etd-11072014-223156 ; https://digitalcommons.lsu.edu/gradschool_dissertations/450


University of Sydney

9. Chen, Xiaochen. Targeting innate immunity as therapeutic strategy in diabetic nephropathy .

Degree: 2017, University of Sydney

 Diabetic nephropathy (DN) is the most common cause of both chronic kidney disease and end-stage kidney disease worldwide, which affects 30-40% of people with Type… (more)

Subjects/Keywords: diabetic nephropathy; kidney disease; TLR; RAGE; HMGB1; SCFA

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APA (6th Edition):

Chen, X. (2017). Targeting innate immunity as therapeutic strategy in diabetic nephropathy . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/17265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Xiaochen. “Targeting innate immunity as therapeutic strategy in diabetic nephropathy .” 2017. Thesis, University of Sydney. Accessed January 20, 2020. http://hdl.handle.net/2123/17265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Xiaochen. “Targeting innate immunity as therapeutic strategy in diabetic nephropathy .” 2017. Web. 20 Jan 2020.

Vancouver:

Chen X. Targeting innate immunity as therapeutic strategy in diabetic nephropathy . [Internet] [Thesis]. University of Sydney; 2017. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/2123/17265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen X. Targeting innate immunity as therapeutic strategy in diabetic nephropathy . [Thesis]. University of Sydney; 2017. Available from: http://hdl.handle.net/2123/17265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

10. Petrović, Anja M., 1985-. Uloga HMGB1 proteina u modifikaciji i integraciji signalnih puteva apoptoze i autofagije u ćelijama jetre pacova sa indukovanim dijabetesom.

Degree: Biološki fakultet, 2018, Univerzitet u Beogradu

Biologija - Molekularna biologija / Biology - Molecular biology

Ćelijska smrt je jedan od uzroka patoloških promena u jetri tokom dijabetesa. Protein koji može imati… (more)

Subjects/Keywords: HMGB1 protein; diabetes; liver; oxidative stress; apoptosis; autophagy; necrosis

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APA (6th Edition):

Petrović, Anja M., 1. (2018). Uloga HMGB1 proteina u modifikaciji i integraciji signalnih puteva apoptoze i autofagije u ćelijama jetre pacova sa indukovanim dijabetesom. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:17626/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Petrović, Anja M., 1985-. “Uloga HMGB1 proteina u modifikaciji i integraciji signalnih puteva apoptoze i autofagije u ćelijama jetre pacova sa indukovanim dijabetesom.” 2018. Thesis, Univerzitet u Beogradu. Accessed January 20, 2020. https://fedorabg.bg.ac.rs/fedora/get/o:17626/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Petrović, Anja M., 1985-. “Uloga HMGB1 proteina u modifikaciji i integraciji signalnih puteva apoptoze i autofagije u ćelijama jetre pacova sa indukovanim dijabetesom.” 2018. Web. 20 Jan 2020.

Vancouver:

Petrović, Anja M. 1. Uloga HMGB1 proteina u modifikaciji i integraciji signalnih puteva apoptoze i autofagije u ćelijama jetre pacova sa indukovanim dijabetesom. [Internet] [Thesis]. Univerzitet u Beogradu; 2018. [cited 2020 Jan 20]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:17626/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Petrović, Anja M. 1. Uloga HMGB1 proteina u modifikaciji i integraciji signalnih puteva apoptoze i autofagije u ćelijama jetre pacova sa indukovanim dijabetesom. [Thesis]. Univerzitet u Beogradu; 2018. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:17626/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Colorado

11. Blair, Rebecca. Using Single Molecule FRET to Study the Mechanisms of DNA Bending by TBP and HMGB1.

Degree: PhD, Chemistry & Biochemistry, 2013, University of Colorado

  Defining mechanisms of transcriptional regulation is important for understanding how gene expression is controlled, which is essential to cellular viability. Outlined in this thesis… (more)

Subjects/Keywords: DNA bending; HMGB1; single molecule FRET; TBP; TIRF; Biochemistry; Molecular Genetics

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APA (6th Edition):

Blair, R. (2013). Using Single Molecule FRET to Study the Mechanisms of DNA Bending by TBP and HMGB1. (Doctoral Dissertation). University of Colorado. Retrieved from https://scholar.colorado.edu/chem_gradetds/106

Chicago Manual of Style (16th Edition):

Blair, Rebecca. “Using Single Molecule FRET to Study the Mechanisms of DNA Bending by TBP and HMGB1.” 2013. Doctoral Dissertation, University of Colorado. Accessed January 20, 2020. https://scholar.colorado.edu/chem_gradetds/106.

MLA Handbook (7th Edition):

Blair, Rebecca. “Using Single Molecule FRET to Study the Mechanisms of DNA Bending by TBP and HMGB1.” 2013. Web. 20 Jan 2020.

Vancouver:

Blair R. Using Single Molecule FRET to Study the Mechanisms of DNA Bending by TBP and HMGB1. [Internet] [Doctoral dissertation]. University of Colorado; 2013. [cited 2020 Jan 20]. Available from: https://scholar.colorado.edu/chem_gradetds/106.

Council of Science Editors:

Blair R. Using Single Molecule FRET to Study the Mechanisms of DNA Bending by TBP and HMGB1. [Doctoral Dissertation]. University of Colorado; 2013. Available from: https://scholar.colorado.edu/chem_gradetds/106


Univerzitet u Beogradu

12. Jovanović Stojanov, Sofija M., 1985-. Карактеризација структуре и функције HMGB1 протеина у јетри пацова током експериментално изазваног дијабетеса типа 1.

Degree: Biološki fakultet, 2018, Univerzitet u Beogradu

Биологија - Молекуларна биологија / Biology - Molecular biology

Оксидативни стрес и хронична инфламација сматрају се главним узроцима појаве дијабетичних компликација, међу којима су и… (more)

Subjects/Keywords: HMGB1 protein; diabetes; liver; oxidative stress; structural modifications; inflammation; regeneration

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APA (6th Edition):

Jovanović Stojanov, Sofija M., 1. (2018). Карактеризација структуре и функције HMGB1 протеина у јетри пацова током експериментално изазваног дијабетеса типа 1. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:18588/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jovanović Stojanov, Sofija M., 1985-. “Карактеризација структуре и функције HMGB1 протеина у јетри пацова током експериментално изазваног дијабетеса типа 1.” 2018. Thesis, Univerzitet u Beogradu. Accessed January 20, 2020. https://fedorabg.bg.ac.rs/fedora/get/o:18588/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jovanović Stojanov, Sofija M., 1985-. “Карактеризација структуре и функције HMGB1 протеина у јетри пацова током експериментално изазваног дијабетеса типа 1.” 2018. Web. 20 Jan 2020.

Vancouver:

Jovanović Stojanov, Sofija M. 1. Карактеризација структуре и функције HMGB1 протеина у јетри пацова током експериментално изазваног дијабетеса типа 1. [Internet] [Thesis]. Univerzitet u Beogradu; 2018. [cited 2020 Jan 20]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:18588/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jovanović Stojanov, Sofija M. 1. Карактеризација структуре и функције HMGB1 протеина у јетри пацова током експериментално изазваног дијабетеса типа 1. [Thesis]. Univerzitet u Beogradu; 2018. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:18588/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Medical Branch – Galveston

13. [No author]. The Eosinophil Granular Leukocyte; Source and Target of High Mobility Group Protein B1 .

Degree: University of Texas Medical Branch – Galveston

 Eosinophils are associated with various inflammatory diseases in which they participate through the release of mediators. Eosinophils thereby function as effectors and immunoregulators and contribute… (more)

Subjects/Keywords: Eosinophils; HMGB1; Proteomics; asthma

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

author], [. (n.d.). The Eosinophil Granular Leukocyte; Source and Target of High Mobility Group Protein B1 . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/861

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “The Eosinophil Granular Leukocyte; Source and Target of High Mobility Group Protein B1 .” Thesis, University of Texas Medical Branch – Galveston. Accessed January 20, 2020. http://hdl.handle.net/2152.3/861.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “The Eosinophil Granular Leukocyte; Source and Target of High Mobility Group Protein B1 .” Web. 20 Jan 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

author] [. The Eosinophil Granular Leukocyte; Source and Target of High Mobility Group Protein B1 . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; [cited 2020 Jan 20]. Available from: http://hdl.handle.net/2152.3/861.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

author] [. The Eosinophil Granular Leukocyte; Source and Target of High Mobility Group Protein B1 . [Thesis]. University of Texas Medical Branch – Galveston; Available from: http://hdl.handle.net/2152.3/861

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of California – San Francisco

14. Nehil, Michael. Novel roles for HMGB1 in cancer and its mechanisms of release during Adenovirus infection.

Degree: Cell Biology, 2011, University of California – San Francisco

 High mobility group box 1 (HMGB1) is a nuclear protein first discovered nearly 30 years ago. It was described to aid in transcription by forming… (more)

Subjects/Keywords: Cellular biology; Virology; adenovirus; cancer; chromatin; epigenetic; hmgb1; sema3a

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APA (6th Edition):

Nehil, M. (2011). Novel roles for HMGB1 in cancer and its mechanisms of release during Adenovirus infection. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/478704h8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nehil, Michael. “Novel roles for HMGB1 in cancer and its mechanisms of release during Adenovirus infection.” 2011. Thesis, University of California – San Francisco. Accessed January 20, 2020. http://www.escholarship.org/uc/item/478704h8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nehil, Michael. “Novel roles for HMGB1 in cancer and its mechanisms of release during Adenovirus infection.” 2011. Web. 20 Jan 2020.

Vancouver:

Nehil M. Novel roles for HMGB1 in cancer and its mechanisms of release during Adenovirus infection. [Internet] [Thesis]. University of California – San Francisco; 2011. [cited 2020 Jan 20]. Available from: http://www.escholarship.org/uc/item/478704h8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nehil M. Novel roles for HMGB1 in cancer and its mechanisms of release during Adenovirus infection. [Thesis]. University of California – San Francisco; 2011. Available from: http://www.escholarship.org/uc/item/478704h8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Βελεγράκη, Μαρία. Μελέτη της έκφρασης των toll-like receptors στα αιμοποιητικά και στρωματικά κύτταρα του μυελού των οστών ασθενών με μυελοδυσπλαστικά σύνδρομα και υποπλαστικού τύπου ουδετεροπενίες.

Degree: 2012, University of Crete (UOC); Πανεπιστήμιο Κρήτης

 Background: Excessive pro-inflammatory cytokine production in the bone marrow (BM) has been recognized as a prominent pathogenetic mechanism for the disturbed hematopoiesis in patients with… (more)

Subjects/Keywords: Υποδοχείς Toll-like; Μυελοδυσπλαστικά σύνδρομα; Χρόνια ιδιοπαθής ουδετεροπενία; Μακροφάγα; Πρωτεΐνη HMGB1; Φαγοκυττάρωση αποπτωτικών σωμάτων; Toll-like receptors; Myelodysplastic syndromes; Chronic idiopathic neutropenia; Macrophages; HMGB1; Phagocytosis of apoptotic bodies

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APA (6th Edition):

Βελεγράκη, . . (2012). Μελέτη της έκφρασης των toll-like receptors στα αιμοποιητικά και στρωματικά κύτταρα του μυελού των οστών ασθενών με μυελοδυσπλαστικά σύνδρομα και υποπλαστικού τύπου ουδετεροπενίες. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/29205

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Βελεγράκη, Μαρία. “Μελέτη της έκφρασης των toll-like receptors στα αιμοποιητικά και στρωματικά κύτταρα του μυελού των οστών ασθενών με μυελοδυσπλαστικά σύνδρομα και υποπλαστικού τύπου ουδετεροπενίες.” 2012. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed January 20, 2020. http://hdl.handle.net/10442/hedi/29205.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Βελεγράκη, Μαρία. “Μελέτη της έκφρασης των toll-like receptors στα αιμοποιητικά και στρωματικά κύτταρα του μυελού των οστών ασθενών με μυελοδυσπλαστικά σύνδρομα και υποπλαστικού τύπου ουδετεροπενίες.” 2012. Web. 20 Jan 2020.

Vancouver:

Βελεγράκη . Μελέτη της έκφρασης των toll-like receptors στα αιμοποιητικά και στρωματικά κύτταρα του μυελού των οστών ασθενών με μυελοδυσπλαστικά σύνδρομα και υποπλαστικού τύπου ουδετεροπενίες. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2012. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/10442/hedi/29205.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Βελεγράκη . Μελέτη της έκφρασης των toll-like receptors στα αιμοποιητικά και στρωματικά κύτταρα του μυελού των οστών ασθενών με μυελοδυσπλαστικά σύνδρομα και υποπλαστικού τύπου ουδετεροπενίες. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2012. Available from: http://hdl.handle.net/10442/hedi/29205

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Zhao, Liwei. Identification of pharmacological agents that induce HMGB1 release and inhibitors of conventional protein secretion : Ll'identification d'agents pharmacologiques qui induisent la libération de HMGB1 et les inhibiteurs de sécrétion de protéine classiques.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2019, Paris Saclay

 Le système RUSH, de l’anglais « Retention using selective hook » est un système développé récemment qui permet d'analyser et de quantifier en temps réel… (more)

Subjects/Keywords: Translocation de protéines; Test RUSH; Réticulum endoplasmique; Criblage à haut débit; Chimiothérapie; Hmgb1; Protein translocation; RUSH assay; Endoplasmic reticulum; Drug screening system; Chemotherapy; Hmgb1

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APA (6th Edition):

Zhao, L. (2019). Identification of pharmacological agents that induce HMGB1 release and inhibitors of conventional protein secretion : Ll'identification d'agents pharmacologiques qui induisent la libération de HMGB1 et les inhibiteurs de sécrétion de protéine classiques. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2019SACLS134

Chicago Manual of Style (16th Edition):

Zhao, Liwei. “Identification of pharmacological agents that induce HMGB1 release and inhibitors of conventional protein secretion : Ll'identification d'agents pharmacologiques qui induisent la libération de HMGB1 et les inhibiteurs de sécrétion de protéine classiques.” 2019. Doctoral Dissertation, Paris Saclay. Accessed January 20, 2020. http://www.theses.fr/2019SACLS134.

MLA Handbook (7th Edition):

Zhao, Liwei. “Identification of pharmacological agents that induce HMGB1 release and inhibitors of conventional protein secretion : Ll'identification d'agents pharmacologiques qui induisent la libération de HMGB1 et les inhibiteurs de sécrétion de protéine classiques.” 2019. Web. 20 Jan 2020.

Vancouver:

Zhao L. Identification of pharmacological agents that induce HMGB1 release and inhibitors of conventional protein secretion : Ll'identification d'agents pharmacologiques qui induisent la libération de HMGB1 et les inhibiteurs de sécrétion de protéine classiques. [Internet] [Doctoral dissertation]. Paris Saclay; 2019. [cited 2020 Jan 20]. Available from: http://www.theses.fr/2019SACLS134.

Council of Science Editors:

Zhao L. Identification of pharmacological agents that induce HMGB1 release and inhibitors of conventional protein secretion : Ll'identification d'agents pharmacologiques qui induisent la libération de HMGB1 et les inhibiteurs de sécrétion de protéine classiques. [Doctoral Dissertation]. Paris Saclay; 2019. Available from: http://www.theses.fr/2019SACLS134


Universidade do Rio Grande do Sul

17. Bonatto, Fernanda. Efeitos da proteína HMGB1 sobre fatores sensíveis a espécies reativas de oxigênio em macrófagos peritoneais de ratos : investigando um novo papel.

Degree: 2006, Universidade do Rio Grande do Sul

HMGB1é a proteína não-histona mais abundante no núcleo celular e foi inicialmente descrita como reguladora da transcrição gênica por ligar-se ao DNA promovendo alterações conformacionais… (more)

Subjects/Keywords: Proteína HMGB1; Macrófagos peritoneais; Radicais livres

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APA (6th Edition):

Bonatto, F. (2006). Efeitos da proteína HMGB1 sobre fatores sensíveis a espécies reativas de oxigênio em macrófagos peritoneais de ratos : investigando um novo papel. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/11125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bonatto, Fernanda. “Efeitos da proteína HMGB1 sobre fatores sensíveis a espécies reativas de oxigênio em macrófagos peritoneais de ratos : investigando um novo papel.” 2006. Thesis, Universidade do Rio Grande do Sul. Accessed January 20, 2020. http://hdl.handle.net/10183/11125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bonatto, Fernanda. “Efeitos da proteína HMGB1 sobre fatores sensíveis a espécies reativas de oxigênio em macrófagos peritoneais de ratos : investigando um novo papel.” 2006. Web. 20 Jan 2020.

Vancouver:

Bonatto F. Efeitos da proteína HMGB1 sobre fatores sensíveis a espécies reativas de oxigênio em macrófagos peritoneais de ratos : investigando um novo papel. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2006. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/10183/11125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bonatto F. Efeitos da proteína HMGB1 sobre fatores sensíveis a espécies reativas de oxigênio em macrófagos peritoneais de ratos : investigando um novo papel. [Thesis]. Universidade do Rio Grande do Sul; 2006. Available from: http://hdl.handle.net/10183/11125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Merced

18. Johnson, Larry. Periodontal Bacteria Influence Inflammasome Activation and Cytokine Secretion During Oral Infection.

Degree: Quantitative and Systems Biology, 2016, University of California – Merced

 Porphyromonas gingivalis and Fusobacterium nucleatum, Gram-negative anaerobic bacteria, have been identified as major contributing pathogens in the etiology of mild to acute periodontitis. They are… (more)

Subjects/Keywords: Molecular biology; Immunology; DAMPs; Fusobacterium nucleatum; HMGB1; Inflammasome; Oral Infection; Porphyromonas gingivalis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Johnson, L. (2016). Periodontal Bacteria Influence Inflammasome Activation and Cytokine Secretion During Oral Infection. (Thesis). University of California – Merced. Retrieved from http://www.escholarship.org/uc/item/70h4v8t7

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Johnson, Larry. “Periodontal Bacteria Influence Inflammasome Activation and Cytokine Secretion During Oral Infection.” 2016. Thesis, University of California – Merced. Accessed January 20, 2020. http://www.escholarship.org/uc/item/70h4v8t7.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Johnson, Larry. “Periodontal Bacteria Influence Inflammasome Activation and Cytokine Secretion During Oral Infection.” 2016. Web. 20 Jan 2020.

Vancouver:

Johnson L. Periodontal Bacteria Influence Inflammasome Activation and Cytokine Secretion During Oral Infection. [Internet] [Thesis]. University of California – Merced; 2016. [cited 2020 Jan 20]. Available from: http://www.escholarship.org/uc/item/70h4v8t7.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Johnson L. Periodontal Bacteria Influence Inflammasome Activation and Cytokine Secretion During Oral Infection. [Thesis]. University of California – Merced; 2016. Available from: http://www.escholarship.org/uc/item/70h4v8t7

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Feijóo Buján, Ana. Comprobación de una interacción física de HMGB1 con su diana .

Degree: 2017, Universidad da Coruña

 [Resumen] En estudios previos, mediante el sistema del doble híbrido en levaduras, se había descrito la interacción física entre la proteína humana HMGB1 y el… (more)

Subjects/Keywords: HMGB1; Immunoprecipitation; YY1; Prostate cancer; Biomarker; Inmunoprecipitación; Cáncer de próstata; Biomarcador; Cancro de próstata

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APA (6th Edition):

Feijóo Buján, A. (2017). Comprobación de una interacción física de HMGB1 con su diana . (Masters Thesis). Universidad da Coruña. Retrieved from http://hdl.handle.net/2183/19992

Chicago Manual of Style (16th Edition):

Feijóo Buján, Ana. “Comprobación de una interacción física de HMGB1 con su diana .” 2017. Masters Thesis, Universidad da Coruña. Accessed January 20, 2020. http://hdl.handle.net/2183/19992.

MLA Handbook (7th Edition):

Feijóo Buján, Ana. “Comprobación de una interacción física de HMGB1 con su diana .” 2017. Web. 20 Jan 2020.

Vancouver:

Feijóo Buján A. Comprobación de una interacción física de HMGB1 con su diana . [Internet] [Masters thesis]. Universidad da Coruña; 2017. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/2183/19992.

Council of Science Editors:

Feijóo Buján A. Comprobación de una interacción física de HMGB1 con su diana . [Masters Thesis]. Universidad da Coruña; 2017. Available from: http://hdl.handle.net/2183/19992

20. 최, 준영. Pathological mechanisms of the ischemic white matter injury in animal model of lacunar infarction.

Degree: 2014, Ajou University

뇌백질은 축삭과 수초로 이루어져 있으며, 수초는 희소돌기교세포에서 생성된다. 허혈성뇌졸중에서 뇌백질은 흔하게 침범되는 부위이며, 뇌백질의 허혈성손상은 궁극적으로 혈관성인지장애를 유발하는 것으로 알려져 있다. 허혈성백질손상으로 인한 신경학적 손상은… (more)

Subjects/Keywords: White matter; Oligodendrocyte; Ischemic stroke; Toll-like receptor 2; HMGB1; 백질; 희소돌기교세포; 허혈성뇌졸중; 톨유사수용체2

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APA (6th Edition):

최, . (2014). Pathological mechanisms of the ischemic white matter injury in animal model of lacunar infarction. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/10874 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000017413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

최, 준영. “Pathological mechanisms of the ischemic white matter injury in animal model of lacunar infarction.” 2014. Thesis, Ajou University. Accessed January 20, 2020. http://repository.ajou.ac.kr/handle/201003/10874 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000017413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

최, 준영. “Pathological mechanisms of the ischemic white matter injury in animal model of lacunar infarction.” 2014. Web. 20 Jan 2020.

Vancouver:

최 . Pathological mechanisms of the ischemic white matter injury in animal model of lacunar infarction. [Internet] [Thesis]. Ajou University; 2014. [cited 2020 Jan 20]. Available from: http://repository.ajou.ac.kr/handle/201003/10874 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000017413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

최 . Pathological mechanisms of the ischemic white matter injury in animal model of lacunar infarction. [Thesis]. Ajou University; 2014. Available from: http://repository.ajou.ac.kr/handle/201003/10874 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000017413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

21. Son, Susie. HMGB1 regulates the nuclear import of huntingtin in a ROS-dependent manner.

Degree: MSc, 2017, McMaster University

In healthy cells, huntingtin is primarily found in the cytoplasm; however, upon cellular stress, huntingtin is phosphorylated (phospho-huntingtin) at serines 13 and 16 of the… (more)

Subjects/Keywords: Huntington's disease; huntingtin; HMGB1; neurodegeneration; nuclear localization; N17; PY-NLS; ROS; oxidative stress; phosphorylation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Son, S. (2017). HMGB1 regulates the nuclear import of huntingtin in a ROS-dependent manner. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/22188

Chicago Manual of Style (16th Edition):

Son, Susie. “HMGB1 regulates the nuclear import of huntingtin in a ROS-dependent manner.” 2017. Masters Thesis, McMaster University. Accessed January 20, 2020. http://hdl.handle.net/11375/22188.

MLA Handbook (7th Edition):

Son, Susie. “HMGB1 regulates the nuclear import of huntingtin in a ROS-dependent manner.” 2017. Web. 20 Jan 2020.

Vancouver:

Son S. HMGB1 regulates the nuclear import of huntingtin in a ROS-dependent manner. [Internet] [Masters thesis]. McMaster University; 2017. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/11375/22188.

Council of Science Editors:

Son S. HMGB1 regulates the nuclear import of huntingtin in a ROS-dependent manner. [Masters Thesis]. McMaster University; 2017. Available from: http://hdl.handle.net/11375/22188


Univerzitet u Beogradu

22. Milić, Ljiljana, 1969-. Prognostički značaj proteina HMGB1 u difuznom sekundarnom peritonitisu.

Degree: Medicinski fakultet, 2016, Univerzitet u Beogradu

Medicina / Medicine

Sekundarni peritonitis je hirurško stanje koje iziskuje neodložno operativno lečenje. Često je praćen sepsom sa relativno visokom incidencom i stopom smrtnosti. Intraabdominalna… (more)

Subjects/Keywords: secondary peritonitis; HMGB1; C-reactive protein; procalcitonin; serum amyloid A; markers of inflammation; sepsis; SIRS

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APA (6th Edition):

Milić, Ljiljana, 1. (2016). Prognostički značaj proteina HMGB1 u difuznom sekundarnom peritonitisu. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:13291/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Milić, Ljiljana, 1969-. “Prognostički značaj proteina HMGB1 u difuznom sekundarnom peritonitisu.” 2016. Thesis, Univerzitet u Beogradu. Accessed January 20, 2020. https://fedorabg.bg.ac.rs/fedora/get/o:13291/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Milić, Ljiljana, 1969-. “Prognostički značaj proteina HMGB1 u difuznom sekundarnom peritonitisu.” 2016. Web. 20 Jan 2020.

Vancouver:

Milić, Ljiljana 1. Prognostički značaj proteina HMGB1 u difuznom sekundarnom peritonitisu. [Internet] [Thesis]. Univerzitet u Beogradu; 2016. [cited 2020 Jan 20]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:13291/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Milić, Ljiljana 1. Prognostički značaj proteina HMGB1 u difuznom sekundarnom peritonitisu. [Thesis]. Univerzitet u Beogradu; 2016. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:13291/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

23. Kashani, Hessam Hassanzadeh. Role of high mobility group box-1 in the pro-fibrotic response of human airway smooth muscle cells.

Degree: Physiology, 2014, University of Manitoba

 Asthma is a chronic disorder highlighted by intermittent airway inflammation and characterized by paroxysmal dyspnea and airway hyperresponsiveness (AHR). A key feature of severe asthma… (more)

Subjects/Keywords: high mobility group box-1 (HMGB1); airway smooth muscle; fibrosis; inflammation; airway remodeling; asthma

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kashani, H. H. (2014). Role of high mobility group box-1 in the pro-fibrotic response of human airway smooth muscle cells. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/23677

Chicago Manual of Style (16th Edition):

Kashani, Hessam Hassanzadeh. “Role of high mobility group box-1 in the pro-fibrotic response of human airway smooth muscle cells.” 2014. Masters Thesis, University of Manitoba. Accessed January 20, 2020. http://hdl.handle.net/1993/23677.

MLA Handbook (7th Edition):

Kashani, Hessam Hassanzadeh. “Role of high mobility group box-1 in the pro-fibrotic response of human airway smooth muscle cells.” 2014. Web. 20 Jan 2020.

Vancouver:

Kashani HH. Role of high mobility group box-1 in the pro-fibrotic response of human airway smooth muscle cells. [Internet] [Masters thesis]. University of Manitoba; 2014. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/1993/23677.

Council of Science Editors:

Kashani HH. Role of high mobility group box-1 in the pro-fibrotic response of human airway smooth muscle cells. [Masters Thesis]. University of Manitoba; 2014. Available from: http://hdl.handle.net/1993/23677


Uppsala University

24. Rensing, Merlin. Enzymatic cleavage of HMGB1.

Degree: Medical Biochemistry and Microbiology, 2017, Uppsala University

  Alarmins and damage associated molecular pattern (DAMP) are endogenous proteins with distinct and various intracellular roles that when released extracellularly act as startingsignals for… (more)

Subjects/Keywords: HMGB1; enzymatic cleavage; neutrophil Elastase; cathepsin G; matrix metalloproteinase-3; arthritis; Biological Sciences; Biologiska vetenskaper

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APA (6th Edition):

Rensing, M. (2017). Enzymatic cleavage of HMGB1. (Thesis). Uppsala University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-331333

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rensing, Merlin. “Enzymatic cleavage of HMGB1.” 2017. Thesis, Uppsala University. Accessed January 20, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-331333.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rensing, Merlin. “Enzymatic cleavage of HMGB1.” 2017. Web. 20 Jan 2020.

Vancouver:

Rensing M. Enzymatic cleavage of HMGB1. [Internet] [Thesis]. Uppsala University; 2017. [cited 2020 Jan 20]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-331333.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rensing M. Enzymatic cleavage of HMGB1. [Thesis]. Uppsala University; 2017. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-331333

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

25. Pullerits, Rille 1969-. The inflammatory and immunogenic properties of the receptor for advanced glycation end products and its ligand, high mobility group box chromosomal protein 1.

Degree: 2005, University of Gothenburg / Göteborgs Universitet

 Rheumatoid arthritis (RA) is a chronic systemic inflammatory joint disease, the pathogenesis of which is complex, involving a wide range of molecules. High mobility group… (more)

Subjects/Keywords: HMGB1; sRAGE; rheumatoid arthritis; inflammation; cytokines

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APA (6th Edition):

Pullerits, R. 1. (2005). The inflammatory and immunogenic properties of the receptor for advanced glycation end products and its ligand, high mobility group box chromosomal protein 1. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/16616

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pullerits, Rille 1969-. “The inflammatory and immunogenic properties of the receptor for advanced glycation end products and its ligand, high mobility group box chromosomal protein 1.” 2005. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed January 20, 2020. http://hdl.handle.net/2077/16616.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pullerits, Rille 1969-. “The inflammatory and immunogenic properties of the receptor for advanced glycation end products and its ligand, high mobility group box chromosomal protein 1.” 2005. Web. 20 Jan 2020.

Vancouver:

Pullerits R1. The inflammatory and immunogenic properties of the receptor for advanced glycation end products and its ligand, high mobility group box chromosomal protein 1. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2005. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/2077/16616.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pullerits R1. The inflammatory and immunogenic properties of the receptor for advanced glycation end products and its ligand, high mobility group box chromosomal protein 1. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2005. Available from: http://hdl.handle.net/2077/16616

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

26. Santos, João Paulo Almeida dos. Efeitos da modulação das proteínas HSP70 e HMGB1 na neuroinflamação provocada pela sepse em ratos.

Degree: 2013, Universidade do Rio Grande do Sul

A sepse é um conjunto complexo de interações moleculares e celulares mediado pela estimulação de receptores celulares envolvidos na inflamação. O controle desta rede de… (more)

Subjects/Keywords: Sistema nervoso central; Sepse; Inflamação; Proteínas de choque térmico HSP70; Proteína HMGB1; Modelos animais de doenças

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APA (6th Edition):

Santos, J. P. A. d. (2013). Efeitos da modulação das proteínas HSP70 e HMGB1 na neuroinflamação provocada pela sepse em ratos. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/80067

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Santos, João Paulo Almeida dos. “Efeitos da modulação das proteínas HSP70 e HMGB1 na neuroinflamação provocada pela sepse em ratos.” 2013. Thesis, Universidade do Rio Grande do Sul. Accessed January 20, 2020. http://hdl.handle.net/10183/80067.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Santos, João Paulo Almeida dos. “Efeitos da modulação das proteínas HSP70 e HMGB1 na neuroinflamação provocada pela sepse em ratos.” 2013. Web. 20 Jan 2020.

Vancouver:

Santos JPAd. Efeitos da modulação das proteínas HSP70 e HMGB1 na neuroinflamação provocada pela sepse em ratos. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2013. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/10183/80067.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Santos JPAd. Efeitos da modulação das proteínas HSP70 e HMGB1 na neuroinflamação provocada pela sepse em ratos. [Thesis]. Universidade do Rio Grande do Sul; 2013. Available from: http://hdl.handle.net/10183/80067

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Bowling Green State University

27. SARPONG, YAW A. THE BINDING OF ESTROGEN, PROGESTERONE AND GLUCOCORTICOID RECEPTORS TO THEIR RECOGNITION SITES IN A NUCLEOSOME AND THE EFFECT OF HMGB1 ON THE BINDING AFFINITY.

Degree: MS, Chemistry, 2006, Bowling Green State University

 The eukaryotic genome is packaged in a chain of nucleosomes called chromatin. For transcription to take place in chromatin, RNA polymerase II, transcriptional activators and… (more)

Subjects/Keywords: receptor binding in nucleosomes and effect of HMGB1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

SARPONG, Y. A. (2006). THE BINDING OF ESTROGEN, PROGESTERONE AND GLUCOCORTICOID RECEPTORS TO THEIR RECOGNITION SITES IN A NUCLEOSOME AND THE EFFECT OF HMGB1 ON THE BINDING AFFINITY. (Masters Thesis). Bowling Green State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1154368368

Chicago Manual of Style (16th Edition):

SARPONG, YAW A. “THE BINDING OF ESTROGEN, PROGESTERONE AND GLUCOCORTICOID RECEPTORS TO THEIR RECOGNITION SITES IN A NUCLEOSOME AND THE EFFECT OF HMGB1 ON THE BINDING AFFINITY.” 2006. Masters Thesis, Bowling Green State University. Accessed January 20, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1154368368.

MLA Handbook (7th Edition):

SARPONG, YAW A. “THE BINDING OF ESTROGEN, PROGESTERONE AND GLUCOCORTICOID RECEPTORS TO THEIR RECOGNITION SITES IN A NUCLEOSOME AND THE EFFECT OF HMGB1 ON THE BINDING AFFINITY.” 2006. Web. 20 Jan 2020.

Vancouver:

SARPONG YA. THE BINDING OF ESTROGEN, PROGESTERONE AND GLUCOCORTICOID RECEPTORS TO THEIR RECOGNITION SITES IN A NUCLEOSOME AND THE EFFECT OF HMGB1 ON THE BINDING AFFINITY. [Internet] [Masters thesis]. Bowling Green State University; 2006. [cited 2020 Jan 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1154368368.

Council of Science Editors:

SARPONG YA. THE BINDING OF ESTROGEN, PROGESTERONE AND GLUCOCORTICOID RECEPTORS TO THEIR RECOGNITION SITES IN A NUCLEOSOME AND THE EFFECT OF HMGB1 ON THE BINDING AFFINITY. [Masters Thesis]. Bowling Green State University; 2006. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1154368368


Bowling Green State University

28. Joshi, Sachindra Raj. Influence of Hmgb1 on Estrogen Responsive Gene Expression and Nucleosome Structure.

Degree: PhD, Biological Sciences, 2009, Bowling Green State University

  High mobility group box-1 protein (HMGB1) increases the in vitro binding affinity of estrogen receptor (ER) to the various estrogen response elements (EREs) such… (more)

Subjects/Keywords: Biology; Molecular Biology; HMGB1; estrogen receptor; non-conventional estrogen response elements; ERE; nucleosome; nucleosome remodeling; ATP-independent

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Joshi, S. R. (2009). Influence of Hmgb1 on Estrogen Responsive Gene Expression and Nucleosome Structure. (Doctoral Dissertation). Bowling Green State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1257186343

Chicago Manual of Style (16th Edition):

Joshi, Sachindra Raj. “Influence of Hmgb1 on Estrogen Responsive Gene Expression and Nucleosome Structure.” 2009. Doctoral Dissertation, Bowling Green State University. Accessed January 20, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1257186343.

MLA Handbook (7th Edition):

Joshi, Sachindra Raj. “Influence of Hmgb1 on Estrogen Responsive Gene Expression and Nucleosome Structure.” 2009. Web. 20 Jan 2020.

Vancouver:

Joshi SR. Influence of Hmgb1 on Estrogen Responsive Gene Expression and Nucleosome Structure. [Internet] [Doctoral dissertation]. Bowling Green State University; 2009. [cited 2020 Jan 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1257186343.

Council of Science Editors:

Joshi SR. Influence of Hmgb1 on Estrogen Responsive Gene Expression and Nucleosome Structure. [Doctoral Dissertation]. Bowling Green State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1257186343


Universidade Nova

29. Cunha, Maria Inês Fazendeiro. Exploring deregulated signals involved in motor neuron-microglia cross-talk in ALS.

Degree: 2014, Universidade Nova

Part of the results discussed in this thesis was presented in the following meetings: Cunha MI, Cunha C, Vaz AR, Brites D. Studying microglial-motoneuron cross-talk… (more)

Subjects/Keywords: Motor neuron dysfunction; Neuroinflammation; Microglia activation/deregulation; MN-microglia cross-talk; Fractalkine -CX3CR1 axis; HMGB1-TLRs signaling pathways

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cunha, M. I. F. (2014). Exploring deregulated signals involved in motor neuron-microglia cross-talk in ALS. (Thesis). Universidade Nova. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/13873

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cunha, Maria Inês Fazendeiro. “Exploring deregulated signals involved in motor neuron-microglia cross-talk in ALS.” 2014. Thesis, Universidade Nova. Accessed January 20, 2020. http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/13873.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cunha, Maria Inês Fazendeiro. “Exploring deregulated signals involved in motor neuron-microglia cross-talk in ALS.” 2014. Web. 20 Jan 2020.

Vancouver:

Cunha MIF. Exploring deregulated signals involved in motor neuron-microglia cross-talk in ALS. [Internet] [Thesis]. Universidade Nova; 2014. [cited 2020 Jan 20]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/13873.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cunha MIF. Exploring deregulated signals involved in motor neuron-microglia cross-talk in ALS. [Thesis]. Universidade Nova; 2014. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/13873

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Erin Khan, Melissa. Rôle de l'ADN dans l'activation du TLR9 lors de l'infection par Leishmania major : propriétés des séquences génomiques et implication des facteurs protéiques : TLR9 activation by Leishmania major DNA : role of genomic sequences and implication of DNA cofactor.

Degree: Docteur es, Immunologie, 2014, Université Pierre et Marie Curie – Paris VI

La plus grande sensibilité des souris TLR9-/- a révélé le rôle de ce récepteur dans l'infection par Leishmania major. Les cellules dendritiques (DCs) sont activées… (more)

Subjects/Keywords: Leishmania major; TLR9; Génome de Trypanosomatidae; Génome de vertébré; HMGB1; Cathepsine; Leishmania major; Trypanosomatidae genome; Vertebrate genome; 571.96

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APA (6th Edition):

Erin Khan, M. (2014). Rôle de l'ADN dans l'activation du TLR9 lors de l'infection par Leishmania major : propriétés des séquences génomiques et implication des facteurs protéiques : TLR9 activation by Leishmania major DNA : role of genomic sequences and implication of DNA cofactor. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2014PA066073

Chicago Manual of Style (16th Edition):

Erin Khan, Melissa. “Rôle de l'ADN dans l'activation du TLR9 lors de l'infection par Leishmania major : propriétés des séquences génomiques et implication des facteurs protéiques : TLR9 activation by Leishmania major DNA : role of genomic sequences and implication of DNA cofactor.” 2014. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed January 20, 2020. http://www.theses.fr/2014PA066073.

MLA Handbook (7th Edition):

Erin Khan, Melissa. “Rôle de l'ADN dans l'activation du TLR9 lors de l'infection par Leishmania major : propriétés des séquences génomiques et implication des facteurs protéiques : TLR9 activation by Leishmania major DNA : role of genomic sequences and implication of DNA cofactor.” 2014. Web. 20 Jan 2020.

Vancouver:

Erin Khan M. Rôle de l'ADN dans l'activation du TLR9 lors de l'infection par Leishmania major : propriétés des séquences génomiques et implication des facteurs protéiques : TLR9 activation by Leishmania major DNA : role of genomic sequences and implication of DNA cofactor. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2014. [cited 2020 Jan 20]. Available from: http://www.theses.fr/2014PA066073.

Council of Science Editors:

Erin Khan M. Rôle de l'ADN dans l'activation du TLR9 lors de l'infection par Leishmania major : propriétés des séquences génomiques et implication des facteurs protéiques : TLR9 activation by Leishmania major DNA : role of genomic sequences and implication of DNA cofactor. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2014. Available from: http://www.theses.fr/2014PA066073

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