Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(HDAC6). Showing records 1 – 27 of 27 total matches.

Search Limiters

Last 2 Years | English Only

No search limiters apply to these results.

▼ Search Limiters

1. Riolo, Matthew T. HDAC6 restrict acetylated survivin nuclear export.

Degree: PhD, Pathobiology, 2012, Brown University

 Survivin is an oncofetal protein expressed in most tumors. It is a unique member of the inhibitor of apoptosis family of proteins having a dual… (more)

Subjects/Keywords: HDAC6

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Riolo, M. T. (2012). HDAC6 restrict acetylated survivin nuclear export. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:297559/

Chicago Manual of Style (16th Edition):

Riolo, Matthew T. “HDAC6 restrict acetylated survivin nuclear export.” 2012. Doctoral Dissertation, Brown University. Accessed January 19, 2021. https://repository.library.brown.edu/studio/item/bdr:297559/.

MLA Handbook (7th Edition):

Riolo, Matthew T. “HDAC6 restrict acetylated survivin nuclear export.” 2012. Web. 19 Jan 2021.

Vancouver:

Riolo MT. HDAC6 restrict acetylated survivin nuclear export. [Internet] [Doctoral dissertation]. Brown University; 2012. [cited 2021 Jan 19]. Available from: https://repository.library.brown.edu/studio/item/bdr:297559/.

Council of Science Editors:

Riolo MT. HDAC6 restrict acetylated survivin nuclear export. [Doctoral Dissertation]. Brown University; 2012. Available from: https://repository.library.brown.edu/studio/item/bdr:297559/


Université de Sherbrooke

2. Arsenault, Dominique. Rôle du microenvironnement hypoxique dans la formation des métastases : impact de la relocalisation intracellulaire de la furine dans l'invasion cellulaire.

Degree: 2013, Université de Sherbrooke

 La compréhension des mécanismes impliqués dans la formation des métastases est l’un des défis majeurs de la recherche sur le cancer. En effet, la formation… (more)

Subjects/Keywords: TGFß; Smad3; Invasion; Invadopode; Hypoxie; HDAC6; Furine

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Arsenault, D. (2013). Rôle du microenvironnement hypoxique dans la formation des métastases : impact de la relocalisation intracellulaire de la furine dans l'invasion cellulaire. (Doctoral Dissertation). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/6221

Chicago Manual of Style (16th Edition):

Arsenault, Dominique. “Rôle du microenvironnement hypoxique dans la formation des métastases : impact de la relocalisation intracellulaire de la furine dans l'invasion cellulaire.” 2013. Doctoral Dissertation, Université de Sherbrooke. Accessed January 19, 2021. http://hdl.handle.net/11143/6221.

MLA Handbook (7th Edition):

Arsenault, Dominique. “Rôle du microenvironnement hypoxique dans la formation des métastases : impact de la relocalisation intracellulaire de la furine dans l'invasion cellulaire.” 2013. Web. 19 Jan 2021.

Vancouver:

Arsenault D. Rôle du microenvironnement hypoxique dans la formation des métastases : impact de la relocalisation intracellulaire de la furine dans l'invasion cellulaire. [Internet] [Doctoral dissertation]. Université de Sherbrooke; 2013. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/11143/6221.

Council of Science Editors:

Arsenault D. Rôle du microenvironnement hypoxique dans la formation des métastases : impact de la relocalisation intracellulaire de la furine dans l'invasion cellulaire. [Doctoral Dissertation]. Université de Sherbrooke; 2013. Available from: http://hdl.handle.net/11143/6221


University of South Florida

3. Williams, Kendra Allana. Phosphorylation of Histone Deacetylase 6 within its C-terminal Region by Extracellular Signal Regulated Kinase 1.

Degree: 2013, University of South Florida

http://upload.etdadmin.com/etdadmin/pdfout/222759_supp_undefined_2A63E500-E9D7-11E2-925E-BE522E1BA5B1.PDF

Subjects/Keywords: Deacetylation; HDAC6; Phosphorylation; Biochemistry; Cell Biology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Williams, K. A. (2013). Phosphorylation of Histone Deacetylase 6 within its C-terminal Region by Extracellular Signal Regulated Kinase 1. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/4792

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Williams, Kendra Allana. “Phosphorylation of Histone Deacetylase 6 within its C-terminal Region by Extracellular Signal Regulated Kinase 1.” 2013. Thesis, University of South Florida. Accessed January 19, 2021. https://scholarcommons.usf.edu/etd/4792.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Williams, Kendra Allana. “Phosphorylation of Histone Deacetylase 6 within its C-terminal Region by Extracellular Signal Regulated Kinase 1.” 2013. Web. 19 Jan 2021.

Vancouver:

Williams KA. Phosphorylation of Histone Deacetylase 6 within its C-terminal Region by Extracellular Signal Regulated Kinase 1. [Internet] [Thesis]. University of South Florida; 2013. [cited 2021 Jan 19]. Available from: https://scholarcommons.usf.edu/etd/4792.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Williams KA. Phosphorylation of Histone Deacetylase 6 within its C-terminal Region by Extracellular Signal Regulated Kinase 1. [Thesis]. University of South Florida; 2013. Available from: https://scholarcommons.usf.edu/etd/4792

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. CHI, ZHEXI. Inhibition of HDAC6 attenuates progression of hypertension.

Degree: 2017, Ajou University

고혈압은 선진국에서 노인인구의 사망 및 유병율에서 가장 빈도가 높은 심혈관계질환의 주요 위험 인자이며 심부전의 원인이 되기도 한다. 황화수소(H2S)는 혈관내에서 주요 가스 신호 전달물질(gasotransmitters) 중의 하나이며… (more)

Subjects/Keywords: Angiotensin Ⅱ; HDAC6; CSE; H2S; 고혈압

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

CHI, Z. (2017). Inhibition of HDAC6 attenuates progression of hypertension. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/16486 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000025778

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

CHI, ZHEXI. “Inhibition of HDAC6 attenuates progression of hypertension.” 2017. Thesis, Ajou University. Accessed January 19, 2021. http://repository.ajou.ac.kr/handle/201003/16486 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000025778.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

CHI, ZHEXI. “Inhibition of HDAC6 attenuates progression of hypertension.” 2017. Web. 19 Jan 2021.

Vancouver:

CHI Z. Inhibition of HDAC6 attenuates progression of hypertension. [Internet] [Thesis]. Ajou University; 2017. [cited 2021 Jan 19]. Available from: http://repository.ajou.ac.kr/handle/201003/16486 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000025778.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

CHI Z. Inhibition of HDAC6 attenuates progression of hypertension. [Thesis]. Ajou University; 2017. Available from: http://repository.ajou.ac.kr/handle/201003/16486 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000025778

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Aznar, Nicolas. L’histone déacétylase HDAC6, un nouvel effecteur du suppresseur de tumeur LKB1 : Histone deacetylase HDAC6 : a new effector of tumor suppressor LKB1.

Degree: Docteur es, Cancérologie. Biologie cellulaire, 2011, Université Claude Bernard – Lyon I

Le gène suppresseur de tumeur LKB1 code une sérine/thréonine kinase qui régule le métabolisme énergétique et la polarité cellulaire. Son action biologique s'exerce en partie… (more)

Subjects/Keywords: LKB1; HDAC6; Granules de stress; Aggrésomes; LKB1; HDAC6; Stress granules; Aggresomes; 616.99

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Aznar, N. (2011). L’histone déacétylase HDAC6, un nouvel effecteur du suppresseur de tumeur LKB1 : Histone deacetylase HDAC6 : a new effector of tumor suppressor LKB1. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2011LYO10043

Chicago Manual of Style (16th Edition):

Aznar, Nicolas. “L’histone déacétylase HDAC6, un nouvel effecteur du suppresseur de tumeur LKB1 : Histone deacetylase HDAC6 : a new effector of tumor suppressor LKB1.” 2011. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed January 19, 2021. http://www.theses.fr/2011LYO10043.

MLA Handbook (7th Edition):

Aznar, Nicolas. “L’histone déacétylase HDAC6, un nouvel effecteur du suppresseur de tumeur LKB1 : Histone deacetylase HDAC6 : a new effector of tumor suppressor LKB1.” 2011. Web. 19 Jan 2021.

Vancouver:

Aznar N. L’histone déacétylase HDAC6, un nouvel effecteur du suppresseur de tumeur LKB1 : Histone deacetylase HDAC6 : a new effector of tumor suppressor LKB1. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2011. [cited 2021 Jan 19]. Available from: http://www.theses.fr/2011LYO10043.

Council of Science Editors:

Aznar N. L’histone déacétylase HDAC6, un nouvel effecteur du suppresseur de tumeur LKB1 : Histone deacetylase HDAC6 : a new effector of tumor suppressor LKB1. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2011. Available from: http://www.theses.fr/2011LYO10043

6. Forcioli-Conti, Nicolas. Rôle du cil primaire au cours de la différenciation adipocytaire : Role of the primary cilium during adipocyte differentiation.

Degree: Docteur es, Interactions moléculaires et cellulaires, 2015, Nice

Le cil primaire (CP) est une organelle présente chez l’Homme dans la grande majorité des cellules. Lors du développement le CP est d’une importance capitale,… (more)

Subjects/Keywords: Cil primaire; Différenciation adipocytaire; Hedgehog; HDAC6; Primary cilium; Adipocyte differentiation; Hedgehog; HDAC6

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Forcioli-Conti, N. (2015). Rôle du cil primaire au cours de la différenciation adipocytaire : Role of the primary cilium during adipocyte differentiation. (Doctoral Dissertation). Nice. Retrieved from http://www.theses.fr/2015NICE4119

Chicago Manual of Style (16th Edition):

Forcioli-Conti, Nicolas. “Rôle du cil primaire au cours de la différenciation adipocytaire : Role of the primary cilium during adipocyte differentiation.” 2015. Doctoral Dissertation, Nice. Accessed January 19, 2021. http://www.theses.fr/2015NICE4119.

MLA Handbook (7th Edition):

Forcioli-Conti, Nicolas. “Rôle du cil primaire au cours de la différenciation adipocytaire : Role of the primary cilium during adipocyte differentiation.” 2015. Web. 19 Jan 2021.

Vancouver:

Forcioli-Conti N. Rôle du cil primaire au cours de la différenciation adipocytaire : Role of the primary cilium during adipocyte differentiation. [Internet] [Doctoral dissertation]. Nice; 2015. [cited 2021 Jan 19]. Available from: http://www.theses.fr/2015NICE4119.

Council of Science Editors:

Forcioli-Conti N. Rôle du cil primaire au cours de la différenciation adipocytaire : Role of the primary cilium during adipocyte differentiation. [Doctoral Dissertation]. Nice; 2015. Available from: http://www.theses.fr/2015NICE4119


Université de Grenoble

7. Pernet, Lydia. Rôle d'HDAC6 et de VCP dans la réponse au stress thermique. Implications dans l'IBMPFD : Role of HDAC6 and VCP in the regulation of the stress response.Implication in IBMPFD myopathy.

Degree: Docteur es, Biologie, 2014, Université de Grenoble

Lors d'un stress, les cellules activent un mécanisme de défense appelé “la réponse au stress”. Ce mécanisme empêche notamment l'accumulation de protéines mal enroulées grâce… (more)

Subjects/Keywords: VCP; HDAC6; HSF1; Réponse au stress; Myopathie IBMPFD; HSP; VCP; HDAC6; HSF1; Stress response; IBMPFD myopathy; HSP; 570

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pernet, L. (2014). Rôle d'HDAC6 et de VCP dans la réponse au stress thermique. Implications dans l'IBMPFD : Role of HDAC6 and VCP in the regulation of the stress response.Implication in IBMPFD myopathy. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2014GRENV005

Chicago Manual of Style (16th Edition):

Pernet, Lydia. “Rôle d'HDAC6 et de VCP dans la réponse au stress thermique. Implications dans l'IBMPFD : Role of HDAC6 and VCP in the regulation of the stress response.Implication in IBMPFD myopathy.” 2014. Doctoral Dissertation, Université de Grenoble. Accessed January 19, 2021. http://www.theses.fr/2014GRENV005.

MLA Handbook (7th Edition):

Pernet, Lydia. “Rôle d'HDAC6 et de VCP dans la réponse au stress thermique. Implications dans l'IBMPFD : Role of HDAC6 and VCP in the regulation of the stress response.Implication in IBMPFD myopathy.” 2014. Web. 19 Jan 2021.

Vancouver:

Pernet L. Rôle d'HDAC6 et de VCP dans la réponse au stress thermique. Implications dans l'IBMPFD : Role of HDAC6 and VCP in the regulation of the stress response.Implication in IBMPFD myopathy. [Internet] [Doctoral dissertation]. Université de Grenoble; 2014. [cited 2021 Jan 19]. Available from: http://www.theses.fr/2014GRENV005.

Council of Science Editors:

Pernet L. Rôle d'HDAC6 et de VCP dans la réponse au stress thermique. Implications dans l'IBMPFD : Role of HDAC6 and VCP in the regulation of the stress response.Implication in IBMPFD myopathy. [Doctoral Dissertation]. Université de Grenoble; 2014. Available from: http://www.theses.fr/2014GRENV005


Texas A&M University

8. Perkins, Ashley Lyn. Examining the Roles of Beta-Catenin and HDAC6 in Primary Cilia Signaling in ccRCC and CRC.

Degree: MS, Medical Sciences, 2015, Texas A&M University

 Primary cilia are single hair-like organelles found on the apical surface of growth arrested and differentiated cells and can be found in almost every cell… (more)

Subjects/Keywords: primary cilia; ciliogenesis; VHL; AURKA; HDAC6; APC; beta-catenin; renal; colon

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Perkins, A. L. (2015). Examining the Roles of Beta-Catenin and HDAC6 in Primary Cilia Signaling in ccRCC and CRC. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155658

Chicago Manual of Style (16th Edition):

Perkins, Ashley Lyn. “Examining the Roles of Beta-Catenin and HDAC6 in Primary Cilia Signaling in ccRCC and CRC.” 2015. Masters Thesis, Texas A&M University. Accessed January 19, 2021. http://hdl.handle.net/1969.1/155658.

MLA Handbook (7th Edition):

Perkins, Ashley Lyn. “Examining the Roles of Beta-Catenin and HDAC6 in Primary Cilia Signaling in ccRCC and CRC.” 2015. Web. 19 Jan 2021.

Vancouver:

Perkins AL. Examining the Roles of Beta-Catenin and HDAC6 in Primary Cilia Signaling in ccRCC and CRC. [Internet] [Masters thesis]. Texas A&M University; 2015. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1969.1/155658.

Council of Science Editors:

Perkins AL. Examining the Roles of Beta-Catenin and HDAC6 in Primary Cilia Signaling in ccRCC and CRC. [Masters Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155658

9. De Vreese, Rob. Synthesis of thiaheterocyclic benzohydroxamic acids and evaluation of their HDAC6 inhibitory activity.

Degree: 2017, Ghent University

 The hydroxamic acid functionality is an important group in different chemistry disciplines, such as coordination chemistry and medicinal chemistry, due to its excellent metal-chelating properties.… (more)

Subjects/Keywords: Chemistry; Biology and Life Sciences; HDAC6; inhibitors; malaria

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

De Vreese, R. (2017). Synthesis of thiaheterocyclic benzohydroxamic acids and evaluation of their HDAC6 inhibitory activity. (Thesis). Ghent University. Retrieved from http://hdl.handle.net/1854/LU-8519305

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

De Vreese, Rob. “Synthesis of thiaheterocyclic benzohydroxamic acids and evaluation of their HDAC6 inhibitory activity.” 2017. Thesis, Ghent University. Accessed January 19, 2021. http://hdl.handle.net/1854/LU-8519305.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

De Vreese, Rob. “Synthesis of thiaheterocyclic benzohydroxamic acids and evaluation of their HDAC6 inhibitory activity.” 2017. Web. 19 Jan 2021.

Vancouver:

De Vreese R. Synthesis of thiaheterocyclic benzohydroxamic acids and evaluation of their HDAC6 inhibitory activity. [Internet] [Thesis]. Ghent University; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1854/LU-8519305.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

De Vreese R. Synthesis of thiaheterocyclic benzohydroxamic acids and evaluation of their HDAC6 inhibitory activity. [Thesis]. Ghent University; 2017. Available from: http://hdl.handle.net/1854/LU-8519305

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan Technological University

10. Charbonneau, Morgan. ESTABLISHMENT AND CHARACTERIZATION OF ERASTIN-RESISTANT TRIPLE NEGATIVE BREAST CANCER CELL MODELS.

Degree: MS, Department of Biological Sciences, 2019, Michigan Technological University

  Breast cancer is a collection of heterogeneous diseases and the most common cancer in females worldwide. Breast cancer is often categorized by the distinctive… (more)

Subjects/Keywords: Triple-negative breast cancer; erastin; erastin-resistant; xCT; HDAC6

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Charbonneau, M. (2019). ESTABLISHMENT AND CHARACTERIZATION OF ERASTIN-RESISTANT TRIPLE NEGATIVE BREAST CANCER CELL MODELS. (Masters Thesis). Michigan Technological University. Retrieved from https://digitalcommons.mtu.edu/etdr/785

Chicago Manual of Style (16th Edition):

Charbonneau, Morgan. “ESTABLISHMENT AND CHARACTERIZATION OF ERASTIN-RESISTANT TRIPLE NEGATIVE BREAST CANCER CELL MODELS.” 2019. Masters Thesis, Michigan Technological University. Accessed January 19, 2021. https://digitalcommons.mtu.edu/etdr/785.

MLA Handbook (7th Edition):

Charbonneau, Morgan. “ESTABLISHMENT AND CHARACTERIZATION OF ERASTIN-RESISTANT TRIPLE NEGATIVE BREAST CANCER CELL MODELS.” 2019. Web. 19 Jan 2021.

Vancouver:

Charbonneau M. ESTABLISHMENT AND CHARACTERIZATION OF ERASTIN-RESISTANT TRIPLE NEGATIVE BREAST CANCER CELL MODELS. [Internet] [Masters thesis]. Michigan Technological University; 2019. [cited 2021 Jan 19]. Available from: https://digitalcommons.mtu.edu/etdr/785.

Council of Science Editors:

Charbonneau M. ESTABLISHMENT AND CHARACTERIZATION OF ERASTIN-RESISTANT TRIPLE NEGATIVE BREAST CANCER CELL MODELS. [Masters Thesis]. Michigan Technological University; 2019. Available from: https://digitalcommons.mtu.edu/etdr/785


Boston University

11. Trengrove, Chelsea Brais. Autophagy and stress granules: the merging of two pathways in Parkinson's disease.

Degree: PhD, Neuroscience, 2016, Boston University

 Autophagy is compromised in Parkinson’s disease (PD) with a number of PD-associated genetic mutations leading to its dysregulation. Leucine-rich repeat kinase (LRRK2) mutations, causative of… (more)

Subjects/Keywords: Pharmacology; Autophagy; HDAC6; LRRK2; Parkinson's; RNA-binding proteins

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Trengrove, C. B. (2016). Autophagy and stress granules: the merging of two pathways in Parkinson's disease. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/14619

Chicago Manual of Style (16th Edition):

Trengrove, Chelsea Brais. “Autophagy and stress granules: the merging of two pathways in Parkinson's disease.” 2016. Doctoral Dissertation, Boston University. Accessed January 19, 2021. http://hdl.handle.net/2144/14619.

MLA Handbook (7th Edition):

Trengrove, Chelsea Brais. “Autophagy and stress granules: the merging of two pathways in Parkinson's disease.” 2016. Web. 19 Jan 2021.

Vancouver:

Trengrove CB. Autophagy and stress granules: the merging of two pathways in Parkinson's disease. [Internet] [Doctoral dissertation]. Boston University; 2016. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2144/14619.

Council of Science Editors:

Trengrove CB. Autophagy and stress granules: the merging of two pathways in Parkinson's disease. [Doctoral Dissertation]. Boston University; 2016. Available from: http://hdl.handle.net/2144/14619


Duke University

12. Kapur, Meghan Danielle. The Regulation of Lipid Metabolism and Mitochondrial Quality Control in Health and Disease .

Degree: 2015, Duke University

  Advances in modern medicine have helped to prolong human life. These advancements coupled with an ever-increasing population means that diseases associated with aging will… (more)

Subjects/Keywords: Cellular biology; HDAC6; lipid droplets; mitochondria; neurodegeneration; obesity; Parkin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kapur, M. D. (2015). The Regulation of Lipid Metabolism and Mitochondrial Quality Control in Health and Disease . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/9852

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kapur, Meghan Danielle. “The Regulation of Lipid Metabolism and Mitochondrial Quality Control in Health and Disease .” 2015. Thesis, Duke University. Accessed January 19, 2021. http://hdl.handle.net/10161/9852.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kapur, Meghan Danielle. “The Regulation of Lipid Metabolism and Mitochondrial Quality Control in Health and Disease .” 2015. Web. 19 Jan 2021.

Vancouver:

Kapur MD. The Regulation of Lipid Metabolism and Mitochondrial Quality Control in Health and Disease . [Internet] [Thesis]. Duke University; 2015. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10161/9852.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kapur MD. The Regulation of Lipid Metabolism and Mitochondrial Quality Control in Health and Disease . [Thesis]. Duke University; 2015. Available from: http://hdl.handle.net/10161/9852

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Froehlich, Jeanne. Caractérisation de Fam65b, un nouvel effecteur de FoxO1 dans la régulation de la quiescence : Characterization of Fam65b, a new effector of FoxO1 in the regulation of quiescence.

Degree: Docteur es, Immunologie, 2016, Sorbonne Paris Cité

Le comportement et le devenir des lymphocytes T (LT) est conditionné par l’intégration de nombreux signaux solubles et cellulaires. Lorsque les LT ne sont pas… (more)

Subjects/Keywords: Prolifération cellulaire; Fam65b; RhoGTPase; HDAC6; Cycle cellulaire et signalisation; Voie PI3K/Akt/FoxO; Proliferation; Fam65b; RhoGTPase; HDAC6; Cell cycle and signalisation; PI3K/Akt/FoxO; 571.84

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Froehlich, J. (2016). Caractérisation de Fam65b, un nouvel effecteur de FoxO1 dans la régulation de la quiescence : Characterization of Fam65b, a new effector of FoxO1 in the regulation of quiescence. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2016USPCB075

Chicago Manual of Style (16th Edition):

Froehlich, Jeanne. “Caractérisation de Fam65b, un nouvel effecteur de FoxO1 dans la régulation de la quiescence : Characterization of Fam65b, a new effector of FoxO1 in the regulation of quiescence.” 2016. Doctoral Dissertation, Sorbonne Paris Cité. Accessed January 19, 2021. http://www.theses.fr/2016USPCB075.

MLA Handbook (7th Edition):

Froehlich, Jeanne. “Caractérisation de Fam65b, un nouvel effecteur de FoxO1 dans la régulation de la quiescence : Characterization of Fam65b, a new effector of FoxO1 in the regulation of quiescence.” 2016. Web. 19 Jan 2021.

Vancouver:

Froehlich J. Caractérisation de Fam65b, un nouvel effecteur de FoxO1 dans la régulation de la quiescence : Characterization of Fam65b, a new effector of FoxO1 in the regulation of quiescence. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2016. [cited 2021 Jan 19]. Available from: http://www.theses.fr/2016USPCB075.

Council of Science Editors:

Froehlich J. Caractérisation de Fam65b, un nouvel effecteur de FoxO1 dans la régulation de la quiescence : Characterization of Fam65b, a new effector of FoxO1 in the regulation of quiescence. [Doctoral Dissertation]. Sorbonne Paris Cité; 2016. Available from: http://www.theses.fr/2016USPCB075

14. Ratti, Francesca. Role of HDAC6 in Skeletal Muscle Atrophy : Rôle de l’Histone Deacetylase 6 au cours de l’atrophie musculaire.

Degree: Docteur es, Sciences de la Vie, 2014, Lyon, École normale supérieure

HDAC6 est une histone déacétylase hautement conservée, principalement cytoplasmique. Contrairement à d'autres désacétylases, HDAC6 a une spécificité de substrat unique pour les protéines non -… (more)

Subjects/Keywords: Atrophie musculaire; Histone deacetylase 6; Dégradation; Ubiquitine; Muscle atrophy; HDAC6; Degradation; Ubiquitin; 570

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ratti, F. (2014). Role of HDAC6 in Skeletal Muscle Atrophy : Rôle de l’Histone Deacetylase 6 au cours de l’atrophie musculaire. (Doctoral Dissertation). Lyon, École normale supérieure. Retrieved from http://www.theses.fr/2014ENSL0887

Chicago Manual of Style (16th Edition):

Ratti, Francesca. “Role of HDAC6 in Skeletal Muscle Atrophy : Rôle de l’Histone Deacetylase 6 au cours de l’atrophie musculaire.” 2014. Doctoral Dissertation, Lyon, École normale supérieure. Accessed January 19, 2021. http://www.theses.fr/2014ENSL0887.

MLA Handbook (7th Edition):

Ratti, Francesca. “Role of HDAC6 in Skeletal Muscle Atrophy : Rôle de l’Histone Deacetylase 6 au cours de l’atrophie musculaire.” 2014. Web. 19 Jan 2021.

Vancouver:

Ratti F. Role of HDAC6 in Skeletal Muscle Atrophy : Rôle de l’Histone Deacetylase 6 au cours de l’atrophie musculaire. [Internet] [Doctoral dissertation]. Lyon, École normale supérieure; 2014. [cited 2021 Jan 19]. Available from: http://www.theses.fr/2014ENSL0887.

Council of Science Editors:

Ratti F. Role of HDAC6 in Skeletal Muscle Atrophy : Rôle de l’Histone Deacetylase 6 au cours de l’atrophie musculaire. [Doctoral Dissertation]. Lyon, École normale supérieure; 2014. Available from: http://www.theses.fr/2014ENSL0887


University of Western Ontario

15. Salemi, Louisa M. Characterization of RanBPM Subcellular Localization and Function in HDAC6 Regulation.

Degree: 2016, University of Western Ontario

 RanBPM has been shown to interact with numerous proteins implicating it in a variety of cellular processes including apoptosis, transcription regulation, cell migration, adhesion and… (more)

Subjects/Keywords: RanBPM; CTLH complex; subcellular localization; microtubules; α-tubulin; aggresome; HDAC6; cell migration; tumour suppressor; Biochemistry

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Salemi, L. M. (2016). Characterization of RanBPM Subcellular Localization and Function in HDAC6 Regulation. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4301

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Salemi, Louisa M. “Characterization of RanBPM Subcellular Localization and Function in HDAC6 Regulation.” 2016. Thesis, University of Western Ontario. Accessed January 19, 2021. https://ir.lib.uwo.ca/etd/4301.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Salemi, Louisa M. “Characterization of RanBPM Subcellular Localization and Function in HDAC6 Regulation.” 2016. Web. 19 Jan 2021.

Vancouver:

Salemi LM. Characterization of RanBPM Subcellular Localization and Function in HDAC6 Regulation. [Internet] [Thesis]. University of Western Ontario; 2016. [cited 2021 Jan 19]. Available from: https://ir.lib.uwo.ca/etd/4301.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Salemi LM. Characterization of RanBPM Subcellular Localization and Function in HDAC6 Regulation. [Thesis]. University of Western Ontario; 2016. Available from: https://ir.lib.uwo.ca/etd/4301

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Ren, Jingjing. The Role of Histone Deacetylase 6 Inhibition on Systemic Lupus Erythematosus.

Degree: PhD, Biomedical and Veterinary Sciences, 2019, Virginia Tech

 Systemic lupus erythematosus (SLE) is a chronic inflammatory autoimmune disease by which immune cells mistakenly attacks healthy self-cells in different organs. Kidney inflammation occurs in… (more)

Subjects/Keywords: Systemic lupus erythematosus; Lupus nephritis; B cell; Germinal center; Histone deacetylase 6 (HDAC6); ACY738

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ren, J. (2019). The Role of Histone Deacetylase 6 Inhibition on Systemic Lupus Erythematosus. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/93590

Chicago Manual of Style (16th Edition):

Ren, Jingjing. “The Role of Histone Deacetylase 6 Inhibition on Systemic Lupus Erythematosus.” 2019. Doctoral Dissertation, Virginia Tech. Accessed January 19, 2021. http://hdl.handle.net/10919/93590.

MLA Handbook (7th Edition):

Ren, Jingjing. “The Role of Histone Deacetylase 6 Inhibition on Systemic Lupus Erythematosus.” 2019. Web. 19 Jan 2021.

Vancouver:

Ren J. The Role of Histone Deacetylase 6 Inhibition on Systemic Lupus Erythematosus. [Internet] [Doctoral dissertation]. Virginia Tech; 2019. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10919/93590.

Council of Science Editors:

Ren J. The Role of Histone Deacetylase 6 Inhibition on Systemic Lupus Erythematosus. [Doctoral Dissertation]. Virginia Tech; 2019. Available from: http://hdl.handle.net/10919/93590

17. Aldana Masangkay, Grace Idalia. Targeted Therapies for Acute Leukemia.

Degree: Biochemistry & Molecular Biology, 2013, UCLA

 Small molecules that can disrupt cell signaling by inhibiting protein-protein interactions hold promise for the development of therapeutics against leukemia. This study tested the effect… (more)

Subjects/Keywords: Biochemistry; ALL; AML; CBP; CREB; HDAC6; Tubacin

…Characteristics of HDAC6 ... .73 C. HDAC6 and tubacin interaction …. ... ..74 D… …The functional domains of HDAC6 ........ …...74 The proposed deacetylation… …Tubacin inhibits binding of HDAC6 with dynein and synergistically induces apoptosis… …HDAC6 in Cancer. J. Biomed and Biotech. 2011; 2011:875824. Sakamoto, K. M., Aldana-Masangkay… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Aldana Masangkay, G. I. (2013). Targeted Therapies for Acute Leukemia. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/6jr809df

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aldana Masangkay, Grace Idalia. “Targeted Therapies for Acute Leukemia.” 2013. Thesis, UCLA. Accessed January 19, 2021. http://www.escholarship.org/uc/item/6jr809df.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aldana Masangkay, Grace Idalia. “Targeted Therapies for Acute Leukemia.” 2013. Web. 19 Jan 2021.

Vancouver:

Aldana Masangkay GI. Targeted Therapies for Acute Leukemia. [Internet] [Thesis]. UCLA; 2013. [cited 2021 Jan 19]. Available from: http://www.escholarship.org/uc/item/6jr809df.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aldana Masangkay GI. Targeted Therapies for Acute Leukemia. [Thesis]. UCLA; 2013. Available from: http://www.escholarship.org/uc/item/6jr809df

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wayne State University

18. Negmeldin, Ahmed. Design, Synthesis And Biological Evaluation Of Histone Deacetylase (hdac) Inhibitors: Saha (vorinostat) Analogs And Biaryl Indolyl Benzamide Inhibitors Display Isoform Selectivity.

Degree: PhD, Chemistry, 2017, Wayne State University

  HDAC proteins have emerged as interesting targets for anti-cancer drugs due to their involvement in cancers, as well as several other diseases. Several HDAC… (more)

Subjects/Keywords: HDAC6 HDAC8 selective inhibitor; HDAC inhibitor; HDAC isoform selectivity; Histone Deacetylase; SAHA; Vorinostat; Biochemistry; Chemistry; Organic Chemistry

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Negmeldin, A. (2017). Design, Synthesis And Biological Evaluation Of Histone Deacetylase (hdac) Inhibitors: Saha (vorinostat) Analogs And Biaryl Indolyl Benzamide Inhibitors Display Isoform Selectivity. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1853

Chicago Manual of Style (16th Edition):

Negmeldin, Ahmed. “Design, Synthesis And Biological Evaluation Of Histone Deacetylase (hdac) Inhibitors: Saha (vorinostat) Analogs And Biaryl Indolyl Benzamide Inhibitors Display Isoform Selectivity.” 2017. Doctoral Dissertation, Wayne State University. Accessed January 19, 2021. https://digitalcommons.wayne.edu/oa_dissertations/1853.

MLA Handbook (7th Edition):

Negmeldin, Ahmed. “Design, Synthesis And Biological Evaluation Of Histone Deacetylase (hdac) Inhibitors: Saha (vorinostat) Analogs And Biaryl Indolyl Benzamide Inhibitors Display Isoform Selectivity.” 2017. Web. 19 Jan 2021.

Vancouver:

Negmeldin A. Design, Synthesis And Biological Evaluation Of Histone Deacetylase (hdac) Inhibitors: Saha (vorinostat) Analogs And Biaryl Indolyl Benzamide Inhibitors Display Isoform Selectivity. [Internet] [Doctoral dissertation]. Wayne State University; 2017. [cited 2021 Jan 19]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1853.

Council of Science Editors:

Negmeldin A. Design, Synthesis And Biological Evaluation Of Histone Deacetylase (hdac) Inhibitors: Saha (vorinostat) Analogs And Biaryl Indolyl Benzamide Inhibitors Display Isoform Selectivity. [Doctoral Dissertation]. Wayne State University; 2017. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1853


Freie Universität Berlin

19. Brix, Sarah. Die Funktion der Histon-Deacetylase 6 (HDAC6) in der Entstehung und Progression kardialer pathologischer Hypertrophie.

Degree: 2017, Freie Universität Berlin

 Histondeacetylasen (HDACs) wurden als zentrale Regulatoren mal-adaptiver Hypertrophie identifiziert. Insbesondere die Aktivität von HDAC6 scheint eine wichtige Rolle bei der Entstehung kardialer Hypertrophie zu spielen.… (more)

Subjects/Keywords: HDAC6; Tubacin; cardiac Hypertrophy; TAC; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit::610 Medizin und Gesundheit

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brix, S. (2017). Die Funktion der Histon-Deacetylase 6 (HDAC6) in der Entstehung und Progression kardialer pathologischer Hypertrophie. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/10851

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brix, Sarah. “Die Funktion der Histon-Deacetylase 6 (HDAC6) in der Entstehung und Progression kardialer pathologischer Hypertrophie.” 2017. Thesis, Freie Universität Berlin. Accessed January 19, 2021. https://refubium.fu-berlin.de/handle/fub188/10851.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brix, Sarah. “Die Funktion der Histon-Deacetylase 6 (HDAC6) in der Entstehung und Progression kardialer pathologischer Hypertrophie.” 2017. Web. 19 Jan 2021.

Vancouver:

Brix S. Die Funktion der Histon-Deacetylase 6 (HDAC6) in der Entstehung und Progression kardialer pathologischer Hypertrophie. [Internet] [Thesis]. Freie Universität Berlin; 2017. [cited 2021 Jan 19]. Available from: https://refubium.fu-berlin.de/handle/fub188/10851.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brix S. Die Funktion der Histon-Deacetylase 6 (HDAC6) in der Entstehung und Progression kardialer pathologischer Hypertrophie. [Thesis]. Freie Universität Berlin; 2017. Available from: https://refubium.fu-berlin.de/handle/fub188/10851

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Otago

20. Hussain, Mazhar. The anti-influenza viral mechanism and viral antagonism of host histone deacetylase 6 .

Degree: University of Otago

 Influenza virus is a causative agent of respiratory illness called "flu" and is a prototypic member of Orthomyxoviridae family. Influenza viruses are enveloped, negative-sense single… (more)

Subjects/Keywords: influenza; HDAC6; ISG15; Viperin; IFITM3; antiviral; apoptosis; caspases; STAT1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hussain, M. (n.d.). The anti-influenza viral mechanism and viral antagonism of host histone deacetylase 6 . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/9874

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Hussain, Mazhar. “The anti-influenza viral mechanism and viral antagonism of host histone deacetylase 6 .” Doctoral Dissertation, University of Otago. Accessed January 19, 2021. http://hdl.handle.net/10523/9874.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Hussain, Mazhar. “The anti-influenza viral mechanism and viral antagonism of host histone deacetylase 6 .” Web. 19 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Hussain M. The anti-influenza viral mechanism and viral antagonism of host histone deacetylase 6 . [Internet] [Doctoral dissertation]. University of Otago; [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10523/9874.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Hussain M. The anti-influenza viral mechanism and viral antagonism of host histone deacetylase 6 . [Doctoral Dissertation]. University of Otago; Available from: http://hdl.handle.net/10523/9874

Note: this citation may be lacking information needed for this citation format:
No year of publication.


Texas Medical Center

21. White, Matthew C. Mechanisms Underlying the Heterogeneous Sensitivities of Cancer Cells to Proteasome Inhibitors.

Degree: PhD, 2013, Texas Medical Center

  The mechanisms underlying cellular response to proteasome inhibitors have not been clearly elucidated in solid tumor models. Evidence suggests that the ability of a… (more)

Subjects/Keywords: proteasome; bortezomib; pancreatic cancer; bladder cancer; eIF2alpha; HRI; Hsp70; HDAC6; proteotoxicity; Cancer Biology; Cell Biology; Medicine and Health Sciences; Molecular Biology; Neoplasms

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

White, M. C. (2013). Mechanisms Underlying the Heterogeneous Sensitivities of Cancer Cells to Proteasome Inhibitors. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/336

Chicago Manual of Style (16th Edition):

White, Matthew C. “Mechanisms Underlying the Heterogeneous Sensitivities of Cancer Cells to Proteasome Inhibitors.” 2013. Doctoral Dissertation, Texas Medical Center. Accessed January 19, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/336.

MLA Handbook (7th Edition):

White, Matthew C. “Mechanisms Underlying the Heterogeneous Sensitivities of Cancer Cells to Proteasome Inhibitors.” 2013. Web. 19 Jan 2021.

Vancouver:

White MC. Mechanisms Underlying the Heterogeneous Sensitivities of Cancer Cells to Proteasome Inhibitors. [Internet] [Doctoral dissertation]. Texas Medical Center; 2013. [cited 2021 Jan 19]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/336.

Council of Science Editors:

White MC. Mechanisms Underlying the Heterogeneous Sensitivities of Cancer Cells to Proteasome Inhibitors. [Doctoral Dissertation]. Texas Medical Center; 2013. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/336


Freie Universität Berlin

22. Winkler, Robin. The importance of the histone deacetylase 6 for the glucocorticod receptor mediated glucose metabolism.

Degree: 2012, Freie Universität Berlin

 The glucocorticoid receptor (GR) is an important regulator of insulin sensitivity and glucose tolerance. In view of the fact that in vitro the histone deacetylase… (more)

Subjects/Keywords: histone deacetylase 6; HDAC6; heat shock protein 90; HSP90; glucocorticoid receptor; GR; steroid diabetes; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie::572 Biochemie

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Winkler, R. (2012). The importance of the histone deacetylase 6 for the glucocorticod receptor mediated glucose metabolism. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-4337

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Winkler, Robin. “The importance of the histone deacetylase 6 for the glucocorticod receptor mediated glucose metabolism.” 2012. Thesis, Freie Universität Berlin. Accessed January 19, 2021. http://dx.doi.org/10.17169/refubium-4337.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Winkler, Robin. “The importance of the histone deacetylase 6 for the glucocorticod receptor mediated glucose metabolism.” 2012. Web. 19 Jan 2021.

Vancouver:

Winkler R. The importance of the histone deacetylase 6 for the glucocorticod receptor mediated glucose metabolism. [Internet] [Thesis]. Freie Universität Berlin; 2012. [cited 2021 Jan 19]. Available from: http://dx.doi.org/10.17169/refubium-4337.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Winkler R. The importance of the histone deacetylase 6 for the glucocorticod receptor mediated glucose metabolism. [Thesis]. Freie Universität Berlin; 2012. Available from: http://dx.doi.org/10.17169/refubium-4337

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Castaneda, Adrian Lance. Selective histone deacetlyase inhibition decreases disease in lupus-prone mice.

Degree: MS, Biomedical and Veterinary Sciences, 2016, Virginia Tech

 Histone deacetylase 6 (HDAC6) is a cytoplasmic enzyme that acetylates several proteins that are involved in the immune response. HDAC6 inhibition has been shown in… (more)

Subjects/Keywords: SLE; T cells; α -tubulin; Autoimmunity; HDACi; HDAC6

…of HDAC6 can ameliorate autoimmune disease and in transplantation (de Zoeten et… …Horwitz, 2008). Selective HDAC6 inhibition treatment has been shown to increase T… …2016). Previous work in our laboratory has been testing the selective HDAC6 inhibitor… …differentiation within the bone marrow (Regna et al., 2016). This suggests that HDAC6… …within the BM during normal B cell development. HDAC6 inhibitors have been shown to increase… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Castaneda, A. L. (2016). Selective histone deacetlyase inhibition decreases disease in lupus-prone mice. (Masters Thesis). Virginia Tech. Retrieved from http://hdl.handle.net/10919/72952

Chicago Manual of Style (16th Edition):

Castaneda, Adrian Lance. “Selective histone deacetlyase inhibition decreases disease in lupus-prone mice.” 2016. Masters Thesis, Virginia Tech. Accessed January 19, 2021. http://hdl.handle.net/10919/72952.

MLA Handbook (7th Edition):

Castaneda, Adrian Lance. “Selective histone deacetlyase inhibition decreases disease in lupus-prone mice.” 2016. Web. 19 Jan 2021.

Vancouver:

Castaneda AL. Selective histone deacetlyase inhibition decreases disease in lupus-prone mice. [Internet] [Masters thesis]. Virginia Tech; 2016. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10919/72952.

Council of Science Editors:

Castaneda AL. Selective histone deacetlyase inhibition decreases disease in lupus-prone mice. [Masters Thesis]. Virginia Tech; 2016. Available from: http://hdl.handle.net/10919/72952

24. Mak, Anthony. Functional Characterization of the Membrane Glycoprotein CD133.

Degree: 2012, University of Toronto

The AC133 epitope of the pentaspan transmembrane glycoprotein CD133 has been used as a cell-surface marker for normal and cancer stem cells from a broad… (more)

Subjects/Keywords: CD133; N-glycosylation; AF4; HDAC6; β-catenin; 0307; 0369; 0379

HDAC6....................................................................................89… …Figure 4.2. CD133 specifically interacts with HDAC6… …92 Figure 4.3. HDAC6 negatively regulates CD133 protein expression via its deacetylase… …regulated by HDAC6 deacetylase activity… …103 Figure 4.8. CD133, HDAC6 and !-catenin form a ternary complex… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mak, A. (2012). Functional Characterization of the Membrane Glycoprotein CD133. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/34797

Chicago Manual of Style (16th Edition):

Mak, Anthony. “Functional Characterization of the Membrane Glycoprotein CD133.” 2012. Doctoral Dissertation, University of Toronto. Accessed January 19, 2021. http://hdl.handle.net/1807/34797.

MLA Handbook (7th Edition):

Mak, Anthony. “Functional Characterization of the Membrane Glycoprotein CD133.” 2012. Web. 19 Jan 2021.

Vancouver:

Mak A. Functional Characterization of the Membrane Glycoprotein CD133. [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1807/34797.

Council of Science Editors:

Mak A. Functional Characterization of the Membrane Glycoprotein CD133. [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/34797

25. Li, Wenjiao. Autophagy Enhanced by RASSF1A Suppresses Diethylnitrosamine (DEN)-Induced Hepatocarcinogenesis.

Degree: PhD, Medical Sciences, 2017, Texas A&M University

 Hepatocellular carcinoma (HCC) is the most common type of human liver cancer and it is now the second leading cause of cancer death worldwide. In… (more)

Subjects/Keywords: AKT; HDAC6; Hippo pathway; MAP1S; microtubule; Mst1; mTOR; p27; survival

…Histone deacetylase 4 HDAC6 Histone deacetylase 6 HEK Human embryonic kidney HRP… …HDAC6 .................................................... 62 Figure 14 RASSF1A interacts with… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, W. (2017). Autophagy Enhanced by RASSF1A Suppresses Diethylnitrosamine (DEN)-Induced Hepatocarcinogenesis. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/173170

Chicago Manual of Style (16th Edition):

Li, Wenjiao. “Autophagy Enhanced by RASSF1A Suppresses Diethylnitrosamine (DEN)-Induced Hepatocarcinogenesis.” 2017. Doctoral Dissertation, Texas A&M University. Accessed January 19, 2021. http://hdl.handle.net/1969.1/173170.

MLA Handbook (7th Edition):

Li, Wenjiao. “Autophagy Enhanced by RASSF1A Suppresses Diethylnitrosamine (DEN)-Induced Hepatocarcinogenesis.” 2017. Web. 19 Jan 2021.

Vancouver:

Li W. Autophagy Enhanced by RASSF1A Suppresses Diethylnitrosamine (DEN)-Induced Hepatocarcinogenesis. [Internet] [Doctoral dissertation]. Texas A&M University; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1969.1/173170.

Council of Science Editors:

Li W. Autophagy Enhanced by RASSF1A Suppresses Diethylnitrosamine (DEN)-Induced Hepatocarcinogenesis. [Doctoral Dissertation]. Texas A&M University; 2017. Available from: http://hdl.handle.net/1969.1/173170


Université de Lorraine

26. Losson, Hélène. Combinaisons de nouveaux inhibiteurs de désacétylase d’histones 6 avec des inhibiteurs de tyrosine kinase pour le traitement de la leucémie myéloïde chronique : Combinations of novel HDAC6 inhibitors with tyrosine kinase inhibitors for chronic myeloid leukemia treatment.

Degree: Docteur es, Sciences de la vie et de la santé, 2020, Université de Lorraine

Les patients atteints de leucémie myéloïde chronique (LMC) breakpoint cluster region-Abelson (BCR-ABL)+ sont traités avec des inhibiteurs de tyrosine kinase (ITK), comme l’imatinib, cependant certains… (more)

Subjects/Keywords: Inhibiteur d’HDAC6; Inhibiteur de tyrosine kinase; Traitements combinés; Effet synergique cytotoxique; Résistances à l’imatinib; Cellules souches cancéreuses; HDAC6 inhibitor; Tyrosine kinase inhibitor; Combination treatments; Synergistic cytotoxic effect; Imatinib resistance; Cancer stem cells; 615.3; 616.994 19

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Losson, H. (2020). Combinaisons de nouveaux inhibiteurs de désacétylase d’histones 6 avec des inhibiteurs de tyrosine kinase pour le traitement de la leucémie myéloïde chronique : Combinations of novel HDAC6 inhibitors with tyrosine kinase inhibitors for chronic myeloid leukemia treatment. (Doctoral Dissertation). Université de Lorraine. Retrieved from http://www.theses.fr/2020LORR0003

Chicago Manual of Style (16th Edition):

Losson, Hélène. “Combinaisons de nouveaux inhibiteurs de désacétylase d’histones 6 avec des inhibiteurs de tyrosine kinase pour le traitement de la leucémie myéloïde chronique : Combinations of novel HDAC6 inhibitors with tyrosine kinase inhibitors for chronic myeloid leukemia treatment.” 2020. Doctoral Dissertation, Université de Lorraine. Accessed January 19, 2021. http://www.theses.fr/2020LORR0003.

MLA Handbook (7th Edition):

Losson, Hélène. “Combinaisons de nouveaux inhibiteurs de désacétylase d’histones 6 avec des inhibiteurs de tyrosine kinase pour le traitement de la leucémie myéloïde chronique : Combinations of novel HDAC6 inhibitors with tyrosine kinase inhibitors for chronic myeloid leukemia treatment.” 2020. Web. 19 Jan 2021.

Vancouver:

Losson H. Combinaisons de nouveaux inhibiteurs de désacétylase d’histones 6 avec des inhibiteurs de tyrosine kinase pour le traitement de la leucémie myéloïde chronique : Combinations of novel HDAC6 inhibitors with tyrosine kinase inhibitors for chronic myeloid leukemia treatment. [Internet] [Doctoral dissertation]. Université de Lorraine; 2020. [cited 2021 Jan 19]. Available from: http://www.theses.fr/2020LORR0003.

Council of Science Editors:

Losson H. Combinaisons de nouveaux inhibiteurs de désacétylase d’histones 6 avec des inhibiteurs de tyrosine kinase pour le traitement de la leucémie myéloïde chronique : Combinations of novel HDAC6 inhibitors with tyrosine kinase inhibitors for chronic myeloid leukemia treatment. [Doctoral Dissertation]. Université de Lorraine; 2020. Available from: http://www.theses.fr/2020LORR0003

27. Mortenson, Jeffrey Benjamin. Histone Deacetylase 6 (HDAC6) Is Critical for Tumor Cell Survival and Promotes the Pro-Survival Activity of 14-3-3ζ viaDeacetylation of Lysines Within the14-3-3ζ Binding Pocket.

Degree: MS, 2015, Brigham Young University

 Our understanding of non-histone acetylation as a means of cellular regulation is in its infancy. Using a mass spectrometry approach we identified acetylated lysine residues… (more)

Subjects/Keywords: Non-histone acetylation; cell survival; 14-3-3; HDAC6; Biochemistry; Chemistry

…means to treat various cancers (13). Histone deacetylase 6 (HDAC6) is one… …cancer. HDAC6 is overexpressed in several cancer types (14) and is found primarily in… …the cytosol closely associated with the cytoskeleton. Indeed HDAC6 has been shown to… …directly deacetylate alpha-tubulin which promotes cytoskeletal stability (15). HDAC6… …is important to cell survival under stress conditions (16). HDAC6 has few known… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mortenson, J. B. (2015). Histone Deacetylase 6 (HDAC6) Is Critical for Tumor Cell Survival and Promotes the Pro-Survival Activity of 14-3-3ζ viaDeacetylation of Lysines Within the14-3-3ζ Binding Pocket. (Masters Thesis). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=6567&context=etd

Chicago Manual of Style (16th Edition):

Mortenson, Jeffrey Benjamin. “Histone Deacetylase 6 (HDAC6) Is Critical for Tumor Cell Survival and Promotes the Pro-Survival Activity of 14-3-3ζ viaDeacetylation of Lysines Within the14-3-3ζ Binding Pocket.” 2015. Masters Thesis, Brigham Young University. Accessed January 19, 2021. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=6567&context=etd.

MLA Handbook (7th Edition):

Mortenson, Jeffrey Benjamin. “Histone Deacetylase 6 (HDAC6) Is Critical for Tumor Cell Survival and Promotes the Pro-Survival Activity of 14-3-3ζ viaDeacetylation of Lysines Within the14-3-3ζ Binding Pocket.” 2015. Web. 19 Jan 2021.

Vancouver:

Mortenson JB. Histone Deacetylase 6 (HDAC6) Is Critical for Tumor Cell Survival and Promotes the Pro-Survival Activity of 14-3-3ζ viaDeacetylation of Lysines Within the14-3-3ζ Binding Pocket. [Internet] [Masters thesis]. Brigham Young University; 2015. [cited 2021 Jan 19]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=6567&context=etd.

Council of Science Editors:

Mortenson JB. Histone Deacetylase 6 (HDAC6) Is Critical for Tumor Cell Survival and Promotes the Pro-Survival Activity of 14-3-3ζ viaDeacetylation of Lysines Within the14-3-3ζ Binding Pocket. [Masters Thesis]. Brigham Young University; 2015. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=6567&context=etd

.