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You searched for subject:(HDAC inhibitors). Showing records 1 – 30 of 39 total matches.

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Oregon State University

1. Nian, Hui. Dietary organosulfur and organoselenium compounds as HDAC inhibitors.

Degree: PhD, Biochemistry and Biophysics, 2010, Oregon State University

 Histone deacetylase (HDAC) inhibitors have the potential to de-repress epigenetically silenced genes in cancer cells, leading to cell cycle arrest and apoptosis. Dietary HDAC inhibitors(more)

Subjects/Keywords: HDAC; Histone deacetylase  – Inhibitors

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APA (6th Edition):

Nian, H. (2010). Dietary organosulfur and organoselenium compounds as HDAC inhibitors. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/15093

Chicago Manual of Style (16th Edition):

Nian, Hui. “Dietary organosulfur and organoselenium compounds as HDAC inhibitors.” 2010. Doctoral Dissertation, Oregon State University. Accessed October 28, 2020. http://hdl.handle.net/1957/15093.

MLA Handbook (7th Edition):

Nian, Hui. “Dietary organosulfur and organoselenium compounds as HDAC inhibitors.” 2010. Web. 28 Oct 2020.

Vancouver:

Nian H. Dietary organosulfur and organoselenium compounds as HDAC inhibitors. [Internet] [Doctoral dissertation]. Oregon State University; 2010. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1957/15093.

Council of Science Editors:

Nian H. Dietary organosulfur and organoselenium compounds as HDAC inhibitors. [Doctoral Dissertation]. Oregon State University; 2010. Available from: http://hdl.handle.net/1957/15093


University of Melbourne

2. Bishton, Mark. Deciphering the molecular and biological processes that mediate histone deacetylase inhibitor-induced thrombocytopenia.

Degree: 2012, University of Melbourne

 Somatic mutations in a variety of genes involved in cell cycle, signalling, differentiation, proliferation and apoptotic pathways can lead to the initiation and progression of… (more)

Subjects/Keywords: HDAC inhibitors; platelets; Rho-GTPases

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APA (6th Edition):

Bishton, M. (2012). Deciphering the molecular and biological processes that mediate histone deacetylase inhibitor-induced thrombocytopenia. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37587

Chicago Manual of Style (16th Edition):

Bishton, Mark. “Deciphering the molecular and biological processes that mediate histone deacetylase inhibitor-induced thrombocytopenia.” 2012. Doctoral Dissertation, University of Melbourne. Accessed October 28, 2020. http://hdl.handle.net/11343/37587.

MLA Handbook (7th Edition):

Bishton, Mark. “Deciphering the molecular and biological processes that mediate histone deacetylase inhibitor-induced thrombocytopenia.” 2012. Web. 28 Oct 2020.

Vancouver:

Bishton M. Deciphering the molecular and biological processes that mediate histone deacetylase inhibitor-induced thrombocytopenia. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/11343/37587.

Council of Science Editors:

Bishton M. Deciphering the molecular and biological processes that mediate histone deacetylase inhibitor-induced thrombocytopenia. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37587


Georgia Tech

3. Sodji, Quaovi Hemeka. Improving histone deacetylase inhibition therapy through isoform selectivity and targeted delivery.

Degree: PhD, Chemistry and Biochemistry, 2014, Georgia Tech

 Histone deacetylase (HDAC) inhibition has recently emerged as a novel therapy for cancer treatment. However, currently approved histone deacetylase inhibitors (HDACi) are pan-inhibitors thus inhibiting… (more)

Subjects/Keywords: Histone deacetylase (HDAC); HDAC inhibitors; Isoform selectivity; Targeted delivery

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APA (6th Edition):

Sodji, Q. H. (2014). Improving histone deacetylase inhibition therapy through isoform selectivity and targeted delivery. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/53445

Chicago Manual of Style (16th Edition):

Sodji, Quaovi Hemeka. “Improving histone deacetylase inhibition therapy through isoform selectivity and targeted delivery.” 2014. Doctoral Dissertation, Georgia Tech. Accessed October 28, 2020. http://hdl.handle.net/1853/53445.

MLA Handbook (7th Edition):

Sodji, Quaovi Hemeka. “Improving histone deacetylase inhibition therapy through isoform selectivity and targeted delivery.” 2014. Web. 28 Oct 2020.

Vancouver:

Sodji QH. Improving histone deacetylase inhibition therapy through isoform selectivity and targeted delivery. [Internet] [Doctoral dissertation]. Georgia Tech; 2014. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1853/53445.

Council of Science Editors:

Sodji QH. Improving histone deacetylase inhibition therapy through isoform selectivity and targeted delivery. [Doctoral Dissertation]. Georgia Tech; 2014. Available from: http://hdl.handle.net/1853/53445


University of Toledo

4. Al-Hamashi, Ayad Abed Ali Chiad A. Design, Synthesis, and Biological Evaluation of Novel Histone Deacetylase Inhibitors as Anti-Cancer Agents.

Degree: PhD, Medicinal Chemistry, 2018, University of Toledo

 Despite major advances in cancer treatment strategies in recent years, significant limitations still remain. Selectively targeting cancer cells without affecting normal cells is a challenging… (more)

Subjects/Keywords: Pharmacy Sciences; HDAC, Inhibitors, epigenetic, histone

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APA (6th Edition):

Al-Hamashi, A. A. A. C. A. (2018). Design, Synthesis, and Biological Evaluation of Novel Histone Deacetylase Inhibitors as Anti-Cancer Agents. (Doctoral Dissertation). University of Toledo. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=toledo1525945712448479

Chicago Manual of Style (16th Edition):

Al-Hamashi, Ayad Abed Ali Chiad A. “Design, Synthesis, and Biological Evaluation of Novel Histone Deacetylase Inhibitors as Anti-Cancer Agents.” 2018. Doctoral Dissertation, University of Toledo. Accessed October 28, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1525945712448479.

MLA Handbook (7th Edition):

Al-Hamashi, Ayad Abed Ali Chiad A. “Design, Synthesis, and Biological Evaluation of Novel Histone Deacetylase Inhibitors as Anti-Cancer Agents.” 2018. Web. 28 Oct 2020.

Vancouver:

Al-Hamashi AAACA. Design, Synthesis, and Biological Evaluation of Novel Histone Deacetylase Inhibitors as Anti-Cancer Agents. [Internet] [Doctoral dissertation]. University of Toledo; 2018. [cited 2020 Oct 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1525945712448479.

Council of Science Editors:

Al-Hamashi AAACA. Design, Synthesis, and Biological Evaluation of Novel Histone Deacetylase Inhibitors as Anti-Cancer Agents. [Doctoral Dissertation]. University of Toledo; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1525945712448479


University of Southern California

5. Jayathilaka, Nimanthi. Inhibition of the interaction between class IIA histone deacetylases and myocyte enhancer factor 2 by small molecules.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2012, University of Southern California

 Enzymes that modify epigenetic status provide attractive targets for therapy in various diseases including cardiac hypertrophy, cancer, neurodegenerative diseases, and immune dysfunction. The therapeutic development… (more)

Subjects/Keywords: histone deacetylases (HDAC); HDAC inhibitors; myocyte enhancer factor 2 (MEF2); transcription regulation

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APA (6th Edition):

Jayathilaka, N. (2012). Inhibition of the interaction between class IIA histone deacetylases and myocyte enhancer factor 2 by small molecules. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/649665/rec/3502

Chicago Manual of Style (16th Edition):

Jayathilaka, Nimanthi. “Inhibition of the interaction between class IIA histone deacetylases and myocyte enhancer factor 2 by small molecules.” 2012. Doctoral Dissertation, University of Southern California. Accessed October 28, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/649665/rec/3502.

MLA Handbook (7th Edition):

Jayathilaka, Nimanthi. “Inhibition of the interaction between class IIA histone deacetylases and myocyte enhancer factor 2 by small molecules.” 2012. Web. 28 Oct 2020.

Vancouver:

Jayathilaka N. Inhibition of the interaction between class IIA histone deacetylases and myocyte enhancer factor 2 by small molecules. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2020 Oct 28]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/649665/rec/3502.

Council of Science Editors:

Jayathilaka N. Inhibition of the interaction between class IIA histone deacetylases and myocyte enhancer factor 2 by small molecules. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/649665/rec/3502


Georgia State University

6. Rajan, Anubama. Histone acetylation modulates uncoupling protein1 expression in brown adipocytes.

Degree: MS, Biology, 2014, Georgia State University

  Uncoupling protein 1 (UCP1) is a classical feature of brown adipocytes and understanding its regulatory mechanism will help in the development of a pharmacological… (more)

Subjects/Keywords: Uncoupling protein 1; Brown fat; Epigenetics; Histone deacetylase (HDAC); HDAC inhibitors; Obesity

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APA (6th Edition):

Rajan, A. (2014). Histone acetylation modulates uncoupling protein1 expression in brown adipocytes. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/56

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rajan, Anubama. “Histone acetylation modulates uncoupling protein1 expression in brown adipocytes.” 2014. Thesis, Georgia State University. Accessed October 28, 2020. https://scholarworks.gsu.edu/biology_theses/56.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rajan, Anubama. “Histone acetylation modulates uncoupling protein1 expression in brown adipocytes.” 2014. Web. 28 Oct 2020.

Vancouver:

Rajan A. Histone acetylation modulates uncoupling protein1 expression in brown adipocytes. [Internet] [Thesis]. Georgia State University; 2014. [cited 2020 Oct 28]. Available from: https://scholarworks.gsu.edu/biology_theses/56.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rajan A. Histone acetylation modulates uncoupling protein1 expression in brown adipocytes. [Thesis]. Georgia State University; 2014. Available from: https://scholarworks.gsu.edu/biology_theses/56

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade Nova

7. Silva, Fernanda Maria Gonçalves da. Molecular mechanisms underlying the action of histone deacetylases inhibitors (HDACIs) in ovarian cancer.

Degree: 2017, Universidade Nova

 Epithelial ovarian cancer (EOC) is the most lethal gynecological malignancy, despite advances in treatment. The most common histological type, high grade serous carcinoma (HGSC) is… (more)

Subjects/Keywords: Carcinoma do ovário; Tratamento; Inibidores histona deacetilases; Inibidores de HDAC; Ovarian carcinoma; Treatment; Histone Deacetylase Inhibitors; HDAC Inhibitors; Ciências Médicas

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APA (6th Edition):

Silva, F. M. G. d. (2017). Molecular mechanisms underlying the action of histone deacetylases inhibitors (HDACIs) in ovarian cancer. (Thesis). Universidade Nova. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/21372

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Fernanda Maria Gonçalves da. “Molecular mechanisms underlying the action of histone deacetylases inhibitors (HDACIs) in ovarian cancer.” 2017. Thesis, Universidade Nova. Accessed October 28, 2020. http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/21372.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Fernanda Maria Gonçalves da. “Molecular mechanisms underlying the action of histone deacetylases inhibitors (HDACIs) in ovarian cancer.” 2017. Web. 28 Oct 2020.

Vancouver:

Silva FMGd. Molecular mechanisms underlying the action of histone deacetylases inhibitors (HDACIs) in ovarian cancer. [Internet] [Thesis]. Universidade Nova; 2017. [cited 2020 Oct 28]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/21372.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva FMGd. Molecular mechanisms underlying the action of histone deacetylases inhibitors (HDACIs) in ovarian cancer. [Thesis]. Universidade Nova; 2017. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/21372

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Li, Xiao Hui. Molecular Design, Synthesis and Simulation of Histone Deacetylase Inhibitors : ヒストン脱アセチル化酵素阻害剤の分子設計、合成及び結合シミュレーション.

Degree: 博士(工学), 2017, Kyushu Institute of Technology / 九州工業大学

九州工業大学博士学位論文 学位記番号:生工博乙第7号 学位授与年月日:平成22年12月31日

1 Histone Deacetylase and Histone Deacetylase Inhibitors|2 Molecular Simulation of Interactions between HDAC and Apicidin, Analogues|3 Synthesis of Non-natural Amino Acid|4 Molecular… (more)

Subjects/Keywords: Histone; Cycle Peptide HDAC Inhibitors; HDACs Inhibitors' Inhibitory Activity and Selectivity; Docking; Molecular Dynamics Simulation

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APA (6th Edition):

Li, X. H. (2017). Molecular Design, Synthesis and Simulation of Histone Deacetylase Inhibitors : ヒストン脱アセチル化酵素阻害剤の分子設計、合成及び結合シミュレーション. (Thesis). Kyushu Institute of Technology / 九州工業大学. Retrieved from http://hdl.handle.net/10228/5474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Xiao Hui. “Molecular Design, Synthesis and Simulation of Histone Deacetylase Inhibitors : ヒストン脱アセチル化酵素阻害剤の分子設計、合成及び結合シミュレーション.” 2017. Thesis, Kyushu Institute of Technology / 九州工業大学. Accessed October 28, 2020. http://hdl.handle.net/10228/5474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Xiao Hui. “Molecular Design, Synthesis and Simulation of Histone Deacetylase Inhibitors : ヒストン脱アセチル化酵素阻害剤の分子設計、合成及び結合シミュレーション.” 2017. Web. 28 Oct 2020.

Vancouver:

Li XH. Molecular Design, Synthesis and Simulation of Histone Deacetylase Inhibitors : ヒストン脱アセチル化酵素阻害剤の分子設計、合成及び結合シミュレーション. [Internet] [Thesis]. Kyushu Institute of Technology / 九州工業大学; 2017. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10228/5474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li XH. Molecular Design, Synthesis and Simulation of Histone Deacetylase Inhibitors : ヒストン脱アセチル化酵素阻害剤の分子設計、合成及び結合シミュレーション. [Thesis]. Kyushu Institute of Technology / 九州工業大学; 2017. Available from: http://hdl.handle.net/10228/5474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Harvard University

9. She, Angela A. Chemical Neurobiology of Progranulin-Deficient Frontotemporal Dementia.

Degree: PhD, 2017, Harvard University

Frontotemporal dementia (FTD) is a presenile dementia presenting with a variety of clinical phenotypes arising from Frontotemporal Lobar Degeneration (FTLD), a family of neurodegenrative pathologies… (more)

Subjects/Keywords: frontotemporal dementia; chemical neurobiology; chemical biology; epigenetic regulation; hdac inhibitors; bet inhibitors

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APA (6th Edition):

She, A. A. (2017). Chemical Neurobiology of Progranulin-Deficient Frontotemporal Dementia. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:41140292

Chicago Manual of Style (16th Edition):

She, Angela A. “Chemical Neurobiology of Progranulin-Deficient Frontotemporal Dementia.” 2017. Doctoral Dissertation, Harvard University. Accessed October 28, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:41140292.

MLA Handbook (7th Edition):

She, Angela A. “Chemical Neurobiology of Progranulin-Deficient Frontotemporal Dementia.” 2017. Web. 28 Oct 2020.

Vancouver:

She AA. Chemical Neurobiology of Progranulin-Deficient Frontotemporal Dementia. [Internet] [Doctoral dissertation]. Harvard University; 2017. [cited 2020 Oct 28]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:41140292.

Council of Science Editors:

She AA. Chemical Neurobiology of Progranulin-Deficient Frontotemporal Dementia. [Doctoral Dissertation]. Harvard University; 2017. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:41140292


Wayne State University

10. Nalawansha, Dhanusha Ashanthi. Studies Towards Broadening The Substrate Profile And Regulation Of Histone Deacetylase 1.

Degree: PhD, Chemistry, 2016, Wayne State University

  Aberrant expression of histone deacetylase 1 (HDAC1) is implicated in multiple diseases, including cancer. As a consequence, HDAC1 has emerged as an important therapeutic… (more)

Subjects/Keywords: Eg5; HDAC1; HDAC inhibitors; LSD1; SNP; Substrate trapping; Biochemistry; Cell Biology

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APA (6th Edition):

Nalawansha, D. A. (2016). Studies Towards Broadening The Substrate Profile And Regulation Of Histone Deacetylase 1. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1655

Chicago Manual of Style (16th Edition):

Nalawansha, Dhanusha Ashanthi. “Studies Towards Broadening The Substrate Profile And Regulation Of Histone Deacetylase 1.” 2016. Doctoral Dissertation, Wayne State University. Accessed October 28, 2020. https://digitalcommons.wayne.edu/oa_dissertations/1655.

MLA Handbook (7th Edition):

Nalawansha, Dhanusha Ashanthi. “Studies Towards Broadening The Substrate Profile And Regulation Of Histone Deacetylase 1.” 2016. Web. 28 Oct 2020.

Vancouver:

Nalawansha DA. Studies Towards Broadening The Substrate Profile And Regulation Of Histone Deacetylase 1. [Internet] [Doctoral dissertation]. Wayne State University; 2016. [cited 2020 Oct 28]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1655.

Council of Science Editors:

Nalawansha DA. Studies Towards Broadening The Substrate Profile And Regulation Of Histone Deacetylase 1. [Doctoral Dissertation]. Wayne State University; 2016. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1655


Deakin University

11. Fleming, Cassandra. Development of fluorescent and class selective HDAC Inhibitors.

Degree: School of Life and Environmental Sciences, 2015, Deakin University

  The inhibition of HDAC enzymes has proven beneficial for the treatment of a variety of diseases including cancer. This thesis details the development of class… (more)

Subjects/Keywords: histone deacetylase (HDAC) enzymes; cancer therapy; fluorescent inhibitors; cellular imaging

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APA (6th Edition):

Fleming, C. (2015). Development of fluorescent and class selective HDAC Inhibitors. (Thesis). Deakin University. Retrieved from http://hdl.handle.net/10536/DRO/DU:30074274

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fleming, Cassandra. “Development of fluorescent and class selective HDAC Inhibitors.” 2015. Thesis, Deakin University. Accessed October 28, 2020. http://hdl.handle.net/10536/DRO/DU:30074274.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fleming, Cassandra. “Development of fluorescent and class selective HDAC Inhibitors.” 2015. Web. 28 Oct 2020.

Vancouver:

Fleming C. Development of fluorescent and class selective HDAC Inhibitors. [Internet] [Thesis]. Deakin University; 2015. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10536/DRO/DU:30074274.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fleming C. Development of fluorescent and class selective HDAC Inhibitors. [Thesis]. Deakin University; 2015. Available from: http://hdl.handle.net/10536/DRO/DU:30074274

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

12. Groselj, Blaz. The histone deacetylase inhibitor panobinostat as a radiosensitiser in bladder cancer.

Degree: PhD, 2014, University of Oxford

 Muscle invasive bladder cancer (MIBC) has a poor prognosis. Currently, therapy consists of radical radiotherapy or cystectomy with or without chemotherapy. The average age of… (more)

Subjects/Keywords: 616.99; Biology; Oncology; HDAC inhibitors; bladder cancer; panobinostat; radiosensitisation

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APA (6th Edition):

Groselj, B. (2014). The histone deacetylase inhibitor panobinostat as a radiosensitiser in bladder cancer. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:40f48d00-8d74-41db-8630-ceacf22f3e78 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.635254

Chicago Manual of Style (16th Edition):

Groselj, Blaz. “The histone deacetylase inhibitor panobinostat as a radiosensitiser in bladder cancer.” 2014. Doctoral Dissertation, University of Oxford. Accessed October 28, 2020. http://ora.ox.ac.uk/objects/uuid:40f48d00-8d74-41db-8630-ceacf22f3e78 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.635254.

MLA Handbook (7th Edition):

Groselj, Blaz. “The histone deacetylase inhibitor panobinostat as a radiosensitiser in bladder cancer.” 2014. Web. 28 Oct 2020.

Vancouver:

Groselj B. The histone deacetylase inhibitor panobinostat as a radiosensitiser in bladder cancer. [Internet] [Doctoral dissertation]. University of Oxford; 2014. [cited 2020 Oct 28]. Available from: http://ora.ox.ac.uk/objects/uuid:40f48d00-8d74-41db-8630-ceacf22f3e78 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.635254.

Council of Science Editors:

Groselj B. The histone deacetylase inhibitor panobinostat as a radiosensitiser in bladder cancer. [Doctoral Dissertation]. University of Oxford; 2014. Available from: http://ora.ox.ac.uk/objects/uuid:40f48d00-8d74-41db-8630-ceacf22f3e78 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.635254


NSYSU

13. Peng, Yu-Ting. Studies on the regulatory mechanisms of GTN-induced protein kinase inhibitors in hepatocellular carcinoma-derived cells.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

 The study was to investigate the regulatory mechanisms of goniothalamin (GTN)-induced protein kinase inhibitors, which means cyclin-dependent kinase inhibitors (CKIs), in two hepatocellular carcinoma (HCC)-derived… (more)

Subjects/Keywords: E3-ligase inhibitor; HDAC inhibitor; Goniothalamin (GTN); CDKN1B; Cyclin-dependent kinase inhibitors (CKIs); CDKN1C

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APA (6th Edition):

Peng, Y. (2014). Studies on the regulatory mechanisms of GTN-induced protein kinase inhibitors in hepatocellular carcinoma-derived cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-004147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Peng, Yu-Ting. “Studies on the regulatory mechanisms of GTN-induced protein kinase inhibitors in hepatocellular carcinoma-derived cells.” 2014. Thesis, NSYSU. Accessed October 28, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-004147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Peng, Yu-Ting. “Studies on the regulatory mechanisms of GTN-induced protein kinase inhibitors in hepatocellular carcinoma-derived cells.” 2014. Web. 28 Oct 2020.

Vancouver:

Peng Y. Studies on the regulatory mechanisms of GTN-induced protein kinase inhibitors in hepatocellular carcinoma-derived cells. [Internet] [Thesis]. NSYSU; 2014. [cited 2020 Oct 28]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-004147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Peng Y. Studies on the regulatory mechanisms of GTN-induced protein kinase inhibitors in hepatocellular carcinoma-derived cells. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-004147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

14. Estrada-Rivadeneyra, Diego. Use of the ncRNA deletion and overexpression collections in Saccharomyces cerevisiae for the study of ncRNAs and mode of action of orphan drugs.

Degree: 2019, University of Manchester

 Analysis of eukaryotic transcriptomes has revealed the existence of thousands of previously unannotated noncoding RNAs (ncRNAs), most of them with unknown functions. Recent evidence suggests… (more)

Subjects/Keywords: ncRNA; yeast genetics; orphan drugs; chemogenomics; riluzole; lithium; HDAC inhibitors; drug screening

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APA (6th Edition):

Estrada-Rivadeneyra, D. (2019). Use of the ncRNA deletion and overexpression collections in Saccharomyces cerevisiae for the study of ncRNAs and mode of action of orphan drugs. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:322447

Chicago Manual of Style (16th Edition):

Estrada-Rivadeneyra, Diego. “Use of the ncRNA deletion and overexpression collections in Saccharomyces cerevisiae for the study of ncRNAs and mode of action of orphan drugs.” 2019. Doctoral Dissertation, University of Manchester. Accessed October 28, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:322447.

MLA Handbook (7th Edition):

Estrada-Rivadeneyra, Diego. “Use of the ncRNA deletion and overexpression collections in Saccharomyces cerevisiae for the study of ncRNAs and mode of action of orphan drugs.” 2019. Web. 28 Oct 2020.

Vancouver:

Estrada-Rivadeneyra D. Use of the ncRNA deletion and overexpression collections in Saccharomyces cerevisiae for the study of ncRNAs and mode of action of orphan drugs. [Internet] [Doctoral dissertation]. University of Manchester; 2019. [cited 2020 Oct 28]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:322447.

Council of Science Editors:

Estrada-Rivadeneyra D. Use of the ncRNA deletion and overexpression collections in Saccharomyces cerevisiae for the study of ncRNAs and mode of action of orphan drugs. [Doctoral Dissertation]. University of Manchester; 2019. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:322447


Wayne State University

15. Horton, Kyle Lawrence. Synthesis And Characterization Of Biodegradable Poly(vinyl Esters) With Hdac Inhibitory Activity.

Degree: MS, Biomedical Engineering, 2013, Wayne State University

HDAC inhibitors are known to have anti-inflammatory properties. HDAC inhibitors are used in combination with Oct4 to generate induced pluripotent stem cells. I hypothesized… (more)

Subjects/Keywords: anti-inflammatory; HDAC inhibitors; iPSC; microparticles; polymers; vinyl esters; Biomedical Engineering and Bioengineering; Chemistry

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APA (6th Edition):

Horton, K. L. (2013). Synthesis And Characterization Of Biodegradable Poly(vinyl Esters) With Hdac Inhibitory Activity. (Masters Thesis). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_theses/234

Chicago Manual of Style (16th Edition):

Horton, Kyle Lawrence. “Synthesis And Characterization Of Biodegradable Poly(vinyl Esters) With Hdac Inhibitory Activity.” 2013. Masters Thesis, Wayne State University. Accessed October 28, 2020. https://digitalcommons.wayne.edu/oa_theses/234.

MLA Handbook (7th Edition):

Horton, Kyle Lawrence. “Synthesis And Characterization Of Biodegradable Poly(vinyl Esters) With Hdac Inhibitory Activity.” 2013. Web. 28 Oct 2020.

Vancouver:

Horton KL. Synthesis And Characterization Of Biodegradable Poly(vinyl Esters) With Hdac Inhibitory Activity. [Internet] [Masters thesis]. Wayne State University; 2013. [cited 2020 Oct 28]. Available from: https://digitalcommons.wayne.edu/oa_theses/234.

Council of Science Editors:

Horton KL. Synthesis And Characterization Of Biodegradable Poly(vinyl Esters) With Hdac Inhibitory Activity. [Masters Thesis]. Wayne State University; 2013. Available from: https://digitalcommons.wayne.edu/oa_theses/234


University of South Florida

16. Sodre De Castro Laino, Andressa. Targeting Histone Deacetylases in Melanoma and T-cells to Improve Cancer Immunotherapy.

Degree: 2016, University of South Florida

 Histone deacetylases (HDACs) are key mediators of gene expression and, thus, major regulators of cell function. As such, HDACs play a role in orchestrating tumor… (more)

Subjects/Keywords: HDACs; HDAC Inhibitors; Lymphocytes; Immune Checkpoint Blockade; Immunology and Infectious Disease; Oncology

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APA (6th Edition):

Sodre De Castro Laino, A. (2016). Targeting Histone Deacetylases in Melanoma and T-cells to Improve Cancer Immunotherapy. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/6144

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sodre De Castro Laino, Andressa. “Targeting Histone Deacetylases in Melanoma and T-cells to Improve Cancer Immunotherapy.” 2016. Thesis, University of South Florida. Accessed October 28, 2020. https://scholarcommons.usf.edu/etd/6144.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sodre De Castro Laino, Andressa. “Targeting Histone Deacetylases in Melanoma and T-cells to Improve Cancer Immunotherapy.” 2016. Web. 28 Oct 2020.

Vancouver:

Sodre De Castro Laino A. Targeting Histone Deacetylases in Melanoma and T-cells to Improve Cancer Immunotherapy. [Internet] [Thesis]. University of South Florida; 2016. [cited 2020 Oct 28]. Available from: https://scholarcommons.usf.edu/etd/6144.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sodre De Castro Laino A. Targeting Histone Deacetylases in Melanoma and T-cells to Improve Cancer Immunotherapy. [Thesis]. University of South Florida; 2016. Available from: https://scholarcommons.usf.edu/etd/6144

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Monciino, Giulia. Synthesis and biological evaluation of pirrolidine, oxazolidin-2-imine and mercaptoketones derivates as HCV, HIV and HDAC inhibitors.

Degree: 2017, Università degli Studi di Catania

 The main objective of this thesis was the synthesis of modified azanucleosides potential ihibitors of non-structural proteins of HCV virus ((NS) NS3/4A, NS4B, NS5A and… (more)

Subjects/Keywords: Area 06 - Scienze mediche; pirrolidine, oxazolidin-2-imine and mercaptoketones, HCV, HIV, HDAC inhibitors

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APA (6th Edition):

Monciino, G. (2017). Synthesis and biological evaluation of pirrolidine, oxazolidin-2-imine and mercaptoketones derivates as HCV, HIV and HDAC inhibitors. (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/4173

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Monciino, Giulia. “Synthesis and biological evaluation of pirrolidine, oxazolidin-2-imine and mercaptoketones derivates as HCV, HIV and HDAC inhibitors.” 2017. Thesis, Università degli Studi di Catania. Accessed October 28, 2020. http://hdl.handle.net/10761/4173.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Monciino, Giulia. “Synthesis and biological evaluation of pirrolidine, oxazolidin-2-imine and mercaptoketones derivates as HCV, HIV and HDAC inhibitors.” 2017. Web. 28 Oct 2020.

Vancouver:

Monciino G. Synthesis and biological evaluation of pirrolidine, oxazolidin-2-imine and mercaptoketones derivates as HCV, HIV and HDAC inhibitors. [Internet] [Thesis]. Università degli Studi di Catania; 2017. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10761/4173.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Monciino G. Synthesis and biological evaluation of pirrolidine, oxazolidin-2-imine and mercaptoketones derivates as HCV, HIV and HDAC inhibitors. [Thesis]. Università degli Studi di Catania; 2017. Available from: http://hdl.handle.net/10761/4173

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

18. Estrada-Rivadeneyra, Diego. Use of the ncRNA deletion and overexpression collections in Saccharomyces cerevisiae for the study of ncRNAs and mode of action of orphan drugs.

Degree: PhD, 2019, University of Manchester

 Analysis of eukaryotic transcriptomes has revealed the existence of thousands of previously unannotated noncoding RNAs (ncRNAs), most of them with unknown functions. Recent evidence suggests… (more)

Subjects/Keywords: drug screening; HDAC inhibitors; lithium; riluzole; orphan drugs; yeast genetics; ncRNA; chemogenomics

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APA (6th Edition):

Estrada-Rivadeneyra, D. (2019). Use of the ncRNA deletion and overexpression collections in Saccharomyces cerevisiae for the study of ncRNAs and mode of action of orphan drugs. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/use-of-the-ncrna-deletion-and-overexpression-collections-in-saccharomyces-cerevisiae-for-the-study-of-ncrnas-and-mode-of-action-of-orphan-drugs(77f66a1a-9d7f-4285-8a19-598b665c2973).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.799327

Chicago Manual of Style (16th Edition):

Estrada-Rivadeneyra, Diego. “Use of the ncRNA deletion and overexpression collections in Saccharomyces cerevisiae for the study of ncRNAs and mode of action of orphan drugs.” 2019. Doctoral Dissertation, University of Manchester. Accessed October 28, 2020. https://www.research.manchester.ac.uk/portal/en/theses/use-of-the-ncrna-deletion-and-overexpression-collections-in-saccharomyces-cerevisiae-for-the-study-of-ncrnas-and-mode-of-action-of-orphan-drugs(77f66a1a-9d7f-4285-8a19-598b665c2973).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.799327.

MLA Handbook (7th Edition):

Estrada-Rivadeneyra, Diego. “Use of the ncRNA deletion and overexpression collections in Saccharomyces cerevisiae for the study of ncRNAs and mode of action of orphan drugs.” 2019. Web. 28 Oct 2020.

Vancouver:

Estrada-Rivadeneyra D. Use of the ncRNA deletion and overexpression collections in Saccharomyces cerevisiae for the study of ncRNAs and mode of action of orphan drugs. [Internet] [Doctoral dissertation]. University of Manchester; 2019. [cited 2020 Oct 28]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/use-of-the-ncrna-deletion-and-overexpression-collections-in-saccharomyces-cerevisiae-for-the-study-of-ncrnas-and-mode-of-action-of-orphan-drugs(77f66a1a-9d7f-4285-8a19-598b665c2973).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.799327.

Council of Science Editors:

Estrada-Rivadeneyra D. Use of the ncRNA deletion and overexpression collections in Saccharomyces cerevisiae for the study of ncRNAs and mode of action of orphan drugs. [Doctoral Dissertation]. University of Manchester; 2019. Available from: https://www.research.manchester.ac.uk/portal/en/theses/use-of-the-ncrna-deletion-and-overexpression-collections-in-saccharomyces-cerevisiae-for-the-study-of-ncrnas-and-mode-of-action-of-orphan-drugs(77f66a1a-9d7f-4285-8a19-598b665c2973).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.799327

19. Yevtushenko, Mariya Alexandra. Dual mTOR/HDAC Inhibition: Preclinical Development of a Novel Breast Cancer Therapy.

Degree: MS, Biological Sciences, 2013, Dominican University of California

  HER2 (human epidermal growth factor receptor-2) and/or estrogen receptor (ER) are overexpressed in ~80% of human breast cancers. Although modern therapeutics (e.g. Trastuzumab, Tamoxifen)… (more)

Subjects/Keywords: Breast Cancer; HER2; HDAC inhibitors; Laboratory and Basic Science Research; Life Sciences

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APA (6th Edition):

Yevtushenko, M. A. (2013). Dual mTOR/HDAC Inhibition: Preclinical Development of a Novel Breast Cancer Therapy. (Masters Thesis). Dominican University of California. Retrieved from https://scholar.dominican.edu/masters-theses/57

Chicago Manual of Style (16th Edition):

Yevtushenko, Mariya Alexandra. “Dual mTOR/HDAC Inhibition: Preclinical Development of a Novel Breast Cancer Therapy.” 2013. Masters Thesis, Dominican University of California. Accessed October 28, 2020. https://scholar.dominican.edu/masters-theses/57.

MLA Handbook (7th Edition):

Yevtushenko, Mariya Alexandra. “Dual mTOR/HDAC Inhibition: Preclinical Development of a Novel Breast Cancer Therapy.” 2013. Web. 28 Oct 2020.

Vancouver:

Yevtushenko MA. Dual mTOR/HDAC Inhibition: Preclinical Development of a Novel Breast Cancer Therapy. [Internet] [Masters thesis]. Dominican University of California; 2013. [cited 2020 Oct 28]. Available from: https://scholar.dominican.edu/masters-theses/57.

Council of Science Editors:

Yevtushenko MA. Dual mTOR/HDAC Inhibition: Preclinical Development of a Novel Breast Cancer Therapy. [Masters Thesis]. Dominican University of California; 2013. Available from: https://scholar.dominican.edu/masters-theses/57


Georgia State University

20. kondengaden, Shukkoor Muhammed. SMALL MOLECULE INHIBITORS OF G9a AND HDAC FOR TARGETED CANCER THERAPY AND DNA ENCODED GLYCAN LIBRARY (DEGL) FOR EARLY DETECTION OF CANCER.

Degree: PhD, Chemistry, 2018, Georgia State University

  This dissertation focuses on two aspects, targeted cancer therapy based on epigenetics and cancer diagnostics using DNA encoded glycan libraries. The first part addresses… (more)

Subjects/Keywords: G9a inhibitor; HDAC inhibitors; Dual inhibitors; MALDI-TOF assay; DNA encoded glycan library; Glyco-PCR; Carbohydrate oligonucleotide conjugates

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APA (6th Edition):

kondengaden, S. M. (2018). SMALL MOLECULE INHIBITORS OF G9a AND HDAC FOR TARGETED CANCER THERAPY AND DNA ENCODED GLYCAN LIBRARY (DEGL) FOR EARLY DETECTION OF CANCER. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/chemistry_diss/142

Chicago Manual of Style (16th Edition):

kondengaden, Shukkoor Muhammed. “SMALL MOLECULE INHIBITORS OF G9a AND HDAC FOR TARGETED CANCER THERAPY AND DNA ENCODED GLYCAN LIBRARY (DEGL) FOR EARLY DETECTION OF CANCER.” 2018. Doctoral Dissertation, Georgia State University. Accessed October 28, 2020. https://scholarworks.gsu.edu/chemistry_diss/142.

MLA Handbook (7th Edition):

kondengaden, Shukkoor Muhammed. “SMALL MOLECULE INHIBITORS OF G9a AND HDAC FOR TARGETED CANCER THERAPY AND DNA ENCODED GLYCAN LIBRARY (DEGL) FOR EARLY DETECTION OF CANCER.” 2018. Web. 28 Oct 2020.

Vancouver:

kondengaden SM. SMALL MOLECULE INHIBITORS OF G9a AND HDAC FOR TARGETED CANCER THERAPY AND DNA ENCODED GLYCAN LIBRARY (DEGL) FOR EARLY DETECTION OF CANCER. [Internet] [Doctoral dissertation]. Georgia State University; 2018. [cited 2020 Oct 28]. Available from: https://scholarworks.gsu.edu/chemistry_diss/142.

Council of Science Editors:

kondengaden SM. SMALL MOLECULE INHIBITORS OF G9a AND HDAC FOR TARGETED CANCER THERAPY AND DNA ENCODED GLYCAN LIBRARY (DEGL) FOR EARLY DETECTION OF CANCER. [Doctoral Dissertation]. Georgia State University; 2018. Available from: https://scholarworks.gsu.edu/chemistry_diss/142

21. Bahhaj, Fatima El. Nanosondes épigénétiques : Epigenetic nanoprobes.

Degree: Docteur es, Chimie organique, minérale, industrielle, 2014, Poitiers

Les cibles épigénétiques telles que les histones désacétylases (HDAC) sont étudiées comme nouveaux traitements anticancéreux, leur fonctionnement anormal étant associée aux processus cancéreux. De nombreux… (more)

Subjects/Keywords: Anticancéreux; Vectorisation; Inhibiteurs de HDAC; Prodrogues pH sensibles; Nanoparticules; Benzofuranones; Synthèse stéréosélective; Anticancer drugs; Vectorization; HDAC inhibitors; PH sensitive prodrugs; Nanoparticles; Benzofuranones; Stereoselective synthesis; 572

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APA (6th Edition):

Bahhaj, F. E. (2014). Nanosondes épigénétiques : Epigenetic nanoprobes. (Doctoral Dissertation). Poitiers. Retrieved from http://www.theses.fr/2014POIT2289

Chicago Manual of Style (16th Edition):

Bahhaj, Fatima El. “Nanosondes épigénétiques : Epigenetic nanoprobes.” 2014. Doctoral Dissertation, Poitiers. Accessed October 28, 2020. http://www.theses.fr/2014POIT2289.

MLA Handbook (7th Edition):

Bahhaj, Fatima El. “Nanosondes épigénétiques : Epigenetic nanoprobes.” 2014. Web. 28 Oct 2020.

Vancouver:

Bahhaj FE. Nanosondes épigénétiques : Epigenetic nanoprobes. [Internet] [Doctoral dissertation]. Poitiers; 2014. [cited 2020 Oct 28]. Available from: http://www.theses.fr/2014POIT2289.

Council of Science Editors:

Bahhaj FE. Nanosondes épigénétiques : Epigenetic nanoprobes. [Doctoral Dissertation]. Poitiers; 2014. Available from: http://www.theses.fr/2014POIT2289

22. Lamaa, Diana. Conception, synthèse et vectorisation de molécules apparentées à l'isocombrétastatine A-4 : Exploration de nouvelles réactivites des composés diazo-précurseurs : Design, synthesis and vectorization of isocombretastatin A-4 analogues : Exploration of new reactivities of diazo-precursor compounds.

Degree: Docteur es, Chimie thérapeutique, 2019, Université Paris-Saclay (ComUE)

Les travaux de thèse concernent la synthèse et la vectorisation d'analogues de la combrétastatine A-4, une molécule naturelle connue pour ses propriétés anti-vasculaires et cytotoxiques.… (more)

Subjects/Keywords: Inhibiteurs HDAC; Agents Antivasculaires; Composés diazo-Précurseurs; Vectorisation; Méthodologie de synthèse; Formulation; HDAC inhibitors; Antivascular agents; Diazo-Precursor compounds; Vectorization; Synthetic methodology

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APA (6th Edition):

Lamaa, D. (2019). Conception, synthèse et vectorisation de molécules apparentées à l'isocombrétastatine A-4 : Exploration de nouvelles réactivites des composés diazo-précurseurs : Design, synthesis and vectorization of isocombretastatin A-4 analogues : Exploration of new reactivities of diazo-precursor compounds. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2019SACLS448

Chicago Manual of Style (16th Edition):

Lamaa, Diana. “Conception, synthèse et vectorisation de molécules apparentées à l'isocombrétastatine A-4 : Exploration de nouvelles réactivites des composés diazo-précurseurs : Design, synthesis and vectorization of isocombretastatin A-4 analogues : Exploration of new reactivities of diazo-precursor compounds.” 2019. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed October 28, 2020. http://www.theses.fr/2019SACLS448.

MLA Handbook (7th Edition):

Lamaa, Diana. “Conception, synthèse et vectorisation de molécules apparentées à l'isocombrétastatine A-4 : Exploration de nouvelles réactivites des composés diazo-précurseurs : Design, synthesis and vectorization of isocombretastatin A-4 analogues : Exploration of new reactivities of diazo-precursor compounds.” 2019. Web. 28 Oct 2020.

Vancouver:

Lamaa D. Conception, synthèse et vectorisation de molécules apparentées à l'isocombrétastatine A-4 : Exploration de nouvelles réactivites des composés diazo-précurseurs : Design, synthesis and vectorization of isocombretastatin A-4 analogues : Exploration of new reactivities of diazo-precursor compounds. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2019. [cited 2020 Oct 28]. Available from: http://www.theses.fr/2019SACLS448.

Council of Science Editors:

Lamaa D. Conception, synthèse et vectorisation de molécules apparentées à l'isocombrétastatine A-4 : Exploration de nouvelles réactivites des composés diazo-précurseurs : Design, synthesis and vectorization of isocombretastatin A-4 analogues : Exploration of new reactivities of diazo-precursor compounds. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2019. Available from: http://www.theses.fr/2019SACLS448

23. Harttrampf, Anne Catherine. Molecular Profiling in Pediatric Oncology – the MOSCATO-01 Experience // Characterization of SMARCB1-Altered Soft Tissue Sarcomas in Response to Pharmacological HDAC Inhibition : Profilage moléculaire en oncologie pédiatrique – expérience de l'essai MOSCATO-01 // Caractérisation des sarcomes des tissus mous présentant des altérations de SMARCB1 traités par des inhibiteurs des HDAC.

Degree: Docteur es, Sciences de la vie et de la santé, 2018, Université Paris-Saclay (ComUE)

 1ère partie: Bien que les patients pédiatriques présentent généralement des taux de survie élevés, environ 20% d’entre eux ne peuvent être guéris avec des approches… (more)

Subjects/Keywords: Cancers pédiatriques; Médecine de précision; Sarcome épithélioïde; Tumeur rhabdoïde; Inhibiteurs des HDAC; Pediatric cancers; Precision medicine; Epithelioid sarcoma; Rhabdoid tumor; HDAC inhibitors

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APA (6th Edition):

Harttrampf, A. C. (2018). Molecular Profiling in Pediatric Oncology – the MOSCATO-01 Experience // Characterization of SMARCB1-Altered Soft Tissue Sarcomas in Response to Pharmacological HDAC Inhibition : Profilage moléculaire en oncologie pédiatrique – expérience de l'essai MOSCATO-01 // Caractérisation des sarcomes des tissus mous présentant des altérations de SMARCB1 traités par des inhibiteurs des HDAC. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2018SACLS449

Chicago Manual of Style (16th Edition):

Harttrampf, Anne Catherine. “Molecular Profiling in Pediatric Oncology – the MOSCATO-01 Experience // Characterization of SMARCB1-Altered Soft Tissue Sarcomas in Response to Pharmacological HDAC Inhibition : Profilage moléculaire en oncologie pédiatrique – expérience de l'essai MOSCATO-01 // Caractérisation des sarcomes des tissus mous présentant des altérations de SMARCB1 traités par des inhibiteurs des HDAC.” 2018. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed October 28, 2020. http://www.theses.fr/2018SACLS449.

MLA Handbook (7th Edition):

Harttrampf, Anne Catherine. “Molecular Profiling in Pediatric Oncology – the MOSCATO-01 Experience // Characterization of SMARCB1-Altered Soft Tissue Sarcomas in Response to Pharmacological HDAC Inhibition : Profilage moléculaire en oncologie pédiatrique – expérience de l'essai MOSCATO-01 // Caractérisation des sarcomes des tissus mous présentant des altérations de SMARCB1 traités par des inhibiteurs des HDAC.” 2018. Web. 28 Oct 2020.

Vancouver:

Harttrampf AC. Molecular Profiling in Pediatric Oncology – the MOSCATO-01 Experience // Characterization of SMARCB1-Altered Soft Tissue Sarcomas in Response to Pharmacological HDAC Inhibition : Profilage moléculaire en oncologie pédiatrique – expérience de l'essai MOSCATO-01 // Caractérisation des sarcomes des tissus mous présentant des altérations de SMARCB1 traités par des inhibiteurs des HDAC. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2018. [cited 2020 Oct 28]. Available from: http://www.theses.fr/2018SACLS449.

Council of Science Editors:

Harttrampf AC. Molecular Profiling in Pediatric Oncology – the MOSCATO-01 Experience // Characterization of SMARCB1-Altered Soft Tissue Sarcomas in Response to Pharmacological HDAC Inhibition : Profilage moléculaire en oncologie pédiatrique – expérience de l'essai MOSCATO-01 // Caractérisation des sarcomes des tissus mous présentant des altérations de SMARCB1 traités par des inhibiteurs des HDAC. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2018. Available from: http://www.theses.fr/2018SACLS449


University of Saskatchewan

24. PARK, MYUNG RYEOL 1979-. Metabolites from the plant pathogen Alternaria brassicicola: in vitro and in planta production and biosynthesis of brassicicolin A.

Degree: 2016, University of Saskatchewan

 The phytopathogenic fungus Alternaria brassicicola (Schwein.) Wiltshire together with A. brassicae cause Alternaria black spot (also called dark leaf spot) in Brassica species. During infection,… (more)

Subjects/Keywords: α-acetylorcinol; Alternaria brassicicola; HDAC inhibitors; host-selective; phomapyrone A; phytotoxins; secondary metabolites; siderophores; spore germination fluids; tyrosol

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APA (6th Edition):

PARK, M. R. 1. (2016). Metabolites from the plant pathogen Alternaria brassicicola: in vitro and in planta production and biosynthesis of brassicicolin A. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/12602

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

PARK, MYUNG RYEOL 1979-. “Metabolites from the plant pathogen Alternaria brassicicola: in vitro and in planta production and biosynthesis of brassicicolin A.” 2016. Thesis, University of Saskatchewan. Accessed October 28, 2020. http://hdl.handle.net/10388/12602.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

PARK, MYUNG RYEOL 1979-. “Metabolites from the plant pathogen Alternaria brassicicola: in vitro and in planta production and biosynthesis of brassicicolin A.” 2016. Web. 28 Oct 2020.

Vancouver:

PARK MR1. Metabolites from the plant pathogen Alternaria brassicicola: in vitro and in planta production and biosynthesis of brassicicolin A. [Internet] [Thesis]. University of Saskatchewan; 2016. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10388/12602.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

PARK MR1. Metabolites from the plant pathogen Alternaria brassicicola: in vitro and in planta production and biosynthesis of brassicicolin A. [Thesis]. University of Saskatchewan; 2016. Available from: http://hdl.handle.net/10388/12602

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wayne State University

25. Choi, Sun ea. The structural requirements of histone deacetylase (hdac) inhibitors: suberoylanilide hydroxamic acid (saha) analogues modified at c3, c6, and c7 positions enhance selectivity.

Degree: PhD, Chemistry, 2012, Wayne State University

  Histone deacetylase (HDAC) proteins are targets for drug design towards the treatment of cancers since overexpression of HDAC is linked to cancer. Several HDAC(more)

Subjects/Keywords: Dual selectivity, Gene transcription, HDAC inhibitors, Histone deacetylases, Isoform selectivity, Vorinostat; Biochemistry; Chemistry; Medicinal Chemistry and Pharmaceutics

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APA (6th Edition):

Choi, S. e. (2012). The structural requirements of histone deacetylase (hdac) inhibitors: suberoylanilide hydroxamic acid (saha) analogues modified at c3, c6, and c7 positions enhance selectivity. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/430

Chicago Manual of Style (16th Edition):

Choi, Sun ea. “The structural requirements of histone deacetylase (hdac) inhibitors: suberoylanilide hydroxamic acid (saha) analogues modified at c3, c6, and c7 positions enhance selectivity.” 2012. Doctoral Dissertation, Wayne State University. Accessed October 28, 2020. https://digitalcommons.wayne.edu/oa_dissertations/430.

MLA Handbook (7th Edition):

Choi, Sun ea. “The structural requirements of histone deacetylase (hdac) inhibitors: suberoylanilide hydroxamic acid (saha) analogues modified at c3, c6, and c7 positions enhance selectivity.” 2012. Web. 28 Oct 2020.

Vancouver:

Choi Se. The structural requirements of histone deacetylase (hdac) inhibitors: suberoylanilide hydroxamic acid (saha) analogues modified at c3, c6, and c7 positions enhance selectivity. [Internet] [Doctoral dissertation]. Wayne State University; 2012. [cited 2020 Oct 28]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/430.

Council of Science Editors:

Choi Se. The structural requirements of histone deacetylase (hdac) inhibitors: suberoylanilide hydroxamic acid (saha) analogues modified at c3, c6, and c7 positions enhance selectivity. [Doctoral Dissertation]. Wayne State University; 2012. Available from: https://digitalcommons.wayne.edu/oa_dissertations/430


The Ohio State University

26. Lindquist, Tera M. The development of zebrafish (<i>Danio rerio</i>) as a rapid and efficient model system for therapeutic drug screening for Spinal Muscular Atrophy.

Degree: MS, Pathology, 2011, The Ohio State University

  Spinal muscular atrophy (SMA) is the most common genetic cause of infant mortality. It is characterized by symmetrical weakness and atrophy of the proximal… (more)

Subjects/Keywords: Biomedical Research; Molecular Biology; Neurosciences; Pathology; Pharmacology; Spinal Muscular Atrophy; zebrafish; Danio rerio; HDAC inhibitors; Trichostatin A; drug discovery; transgenic model

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APA (6th Edition):

Lindquist, T. M. (2011). The development of zebrafish (<i>Danio rerio</i>) as a rapid and efficient model system for therapeutic drug screening for Spinal Muscular Atrophy. (Masters Thesis). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1311694979

Chicago Manual of Style (16th Edition):

Lindquist, Tera M. “The development of zebrafish (<i>Danio rerio</i>) as a rapid and efficient model system for therapeutic drug screening for Spinal Muscular Atrophy.” 2011. Masters Thesis, The Ohio State University. Accessed October 28, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1311694979.

MLA Handbook (7th Edition):

Lindquist, Tera M. “The development of zebrafish (<i>Danio rerio</i>) as a rapid and efficient model system for therapeutic drug screening for Spinal Muscular Atrophy.” 2011. Web. 28 Oct 2020.

Vancouver:

Lindquist TM. The development of zebrafish (<i>Danio rerio</i>) as a rapid and efficient model system for therapeutic drug screening for Spinal Muscular Atrophy. [Internet] [Masters thesis]. The Ohio State University; 2011. [cited 2020 Oct 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1311694979.

Council of Science Editors:

Lindquist TM. The development of zebrafish (<i>Danio rerio</i>) as a rapid and efficient model system for therapeutic drug screening for Spinal Muscular Atrophy. [Masters Thesis]. The Ohio State University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1311694979

27. Bouzelfen, Abdelilah. Étude du gène HACE1 dans les lymphomes B : Study of the HACE1 gene in B lymphomas.

Degree: Docteur es, Aspects moleculaires et cellulaires de la biologie, 2017, Normandie

Plusieurs lymphomes à cellules B présentent des anomalies génétiques qui sont importantes pour déterminer leurs caractéristiques biologiques et peuvent être utiles pour le diagnostic. Les… (more)

Subjects/Keywords: Oncologie; HACE1; Hématologie; Génétique; Épigénétique; Lymphomes B; Gène suppresseur de tumeur; B-cell lymphomas; Transcriptional regulation; HACE1; Deletions; Epigenetic mechanisms; HDAC inhibitors; DZNep inhibitors; 616.079 6; 616.079 8; QU 475

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APA (6th Edition):

Bouzelfen, A. (2017). Étude du gène HACE1 dans les lymphomes B : Study of the HACE1 gene in B lymphomas. (Doctoral Dissertation). Normandie. Retrieved from http://www.theses.fr/2017NORMR012

Chicago Manual of Style (16th Edition):

Bouzelfen, Abdelilah. “Étude du gène HACE1 dans les lymphomes B : Study of the HACE1 gene in B lymphomas.” 2017. Doctoral Dissertation, Normandie. Accessed October 28, 2020. http://www.theses.fr/2017NORMR012.

MLA Handbook (7th Edition):

Bouzelfen, Abdelilah. “Étude du gène HACE1 dans les lymphomes B : Study of the HACE1 gene in B lymphomas.” 2017. Web. 28 Oct 2020.

Vancouver:

Bouzelfen A. Étude du gène HACE1 dans les lymphomes B : Study of the HACE1 gene in B lymphomas. [Internet] [Doctoral dissertation]. Normandie; 2017. [cited 2020 Oct 28]. Available from: http://www.theses.fr/2017NORMR012.

Council of Science Editors:

Bouzelfen A. Étude du gène HACE1 dans les lymphomes B : Study of the HACE1 gene in B lymphomas. [Doctoral Dissertation]. Normandie; 2017. Available from: http://www.theses.fr/2017NORMR012

28. Alqahtani, Abdulateef, Alqarni. Synthesis and Biological Evaluation of New HDAC Inhibitors.

Degree: MS, Medicinal Chemistry, 2018, University of Toledo

 Cancer is the second leading cause of death in the United States of America and the entire world and the disease burden is exacerbated due… (more)

Subjects/Keywords: Chemistry; Pharmaceuticals; Pharmacy Sciences; HDAC, HDAC inhibitors, Zinc binding group, DNA, Gene expression

HDAC INHIBITORS …12 1.4 SYNTHISIZE of KETO-IMIDAZOLE HDAC… …5 Figure 5. Apoptosis by HDAC inhibitors… …7 Figure 6. Mechanism of anti-cancer effect of HDAC inhibitors… …15 Figure 9. Pharmacophore description of HDAC inhibitors and relevant examples… …results of acetylation of non-histone proteins.10 1.2 HDAC INHIBITORS AS EPIGENETIC REGULATORS… 

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APA (6th Edition):

Alqahtani, Abdulateef, A. (2018). Synthesis and Biological Evaluation of New HDAC Inhibitors. (Masters Thesis). University of Toledo. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=toledo1525448898248748

Chicago Manual of Style (16th Edition):

Alqahtani, Abdulateef, Alqarni. “Synthesis and Biological Evaluation of New HDAC Inhibitors.” 2018. Masters Thesis, University of Toledo. Accessed October 28, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1525448898248748.

MLA Handbook (7th Edition):

Alqahtani, Abdulateef, Alqarni. “Synthesis and Biological Evaluation of New HDAC Inhibitors.” 2018. Web. 28 Oct 2020.

Vancouver:

Alqahtani, Abdulateef A. Synthesis and Biological Evaluation of New HDAC Inhibitors. [Internet] [Masters thesis]. University of Toledo; 2018. [cited 2020 Oct 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1525448898248748.

Council of Science Editors:

Alqahtani, Abdulateef A. Synthesis and Biological Evaluation of New HDAC Inhibitors. [Masters Thesis]. University of Toledo; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1525448898248748


Freie Universität Berlin

29. Kawan, Lars. Histone deacetylase inhibitors (HDACi) induce apoptosis and proliferation inhibition in childhood B-cellprecursor acute lymphoblastic leukemia (BCP-ALL) cell lines.

Degree: 2012, Freie Universität Berlin

 Histone deacetylation is associated with transcriptional silencing. In many neoplasms, histone deacetylase inhibitors (HDACi) induce growth arrest, differentiation and apoptosis dependent on concentration and tumor… (more)

Subjects/Keywords: HDAC inhibitors; ALL; apoptosis; proliferation; Histon H4; MTT Assay; Western Blot; FACS; cell lines; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Kawan, L. (2012). Histone deacetylase inhibitors (HDACi) induce apoptosis and proliferation inhibition in childhood B-cellprecursor acute lymphoblastic leukemia (BCP-ALL) cell lines. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-5003

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kawan, Lars. “Histone deacetylase inhibitors (HDACi) induce apoptosis and proliferation inhibition in childhood B-cellprecursor acute lymphoblastic leukemia (BCP-ALL) cell lines.” 2012. Thesis, Freie Universität Berlin. Accessed October 28, 2020. http://dx.doi.org/10.17169/refubium-5003.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kawan, Lars. “Histone deacetylase inhibitors (HDACi) induce apoptosis and proliferation inhibition in childhood B-cellprecursor acute lymphoblastic leukemia (BCP-ALL) cell lines.” 2012. Web. 28 Oct 2020.

Vancouver:

Kawan L. Histone deacetylase inhibitors (HDACi) induce apoptosis and proliferation inhibition in childhood B-cellprecursor acute lymphoblastic leukemia (BCP-ALL) cell lines. [Internet] [Thesis]. Freie Universität Berlin; 2012. [cited 2020 Oct 28]. Available from: http://dx.doi.org/10.17169/refubium-5003.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kawan L. Histone deacetylase inhibitors (HDACi) induce apoptosis and proliferation inhibition in childhood B-cellprecursor acute lymphoblastic leukemia (BCP-ALL) cell lines. [Thesis]. Freie Universität Berlin; 2012. Available from: http://dx.doi.org/10.17169/refubium-5003

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Guerrant, William. Targeted histone deacetylase inhibition.

Degree: PhD, Chemistry and Biochemistry, 2012, Georgia Tech

 Histone deacetylase (HDAC) inhibitors (HDACi) have demonstrated a wealth of biological effects, including anti-proliferative, anti-inflammatory, anti-parasitic, and cognition-enhancing activities. The recent FDA approvals of the… (more)

Subjects/Keywords: HDAC inhibitors; Drug design; Targeted drug delivery; HDAC; Histone deacetylase; Bifunctional inhibitors; Cancer therapy; Drug delivery systems; Cancer Treatment; Enzymes

…of Radiolabeled Macrocyclic HDAC Inhibitors 125 4.5 Conclusion 131 4.6 Experimental 134… …Topo II-HDAC inhibitors. 58 Figure 3-3: Docked structures of Topo II-HDACi conjugates at… …tissue types. More importantly, most HDACi are non-selective “pan-inhibitors” of several HDAC… …HDAC and Topo II inhibitors SAHA and Daunorubicin, respectively. These compounds inhibit both… …tumors. xvi Chapter 1 Histone Deacetylase, HDAC Inhibitors, and Cancer Treatment 1.1… 

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APA (6th Edition):

Guerrant, W. (2012). Targeted histone deacetylase inhibition. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/44907

Chicago Manual of Style (16th Edition):

Guerrant, William. “Targeted histone deacetylase inhibition.” 2012. Doctoral Dissertation, Georgia Tech. Accessed October 28, 2020. http://hdl.handle.net/1853/44907.

MLA Handbook (7th Edition):

Guerrant, William. “Targeted histone deacetylase inhibition.” 2012. Web. 28 Oct 2020.

Vancouver:

Guerrant W. Targeted histone deacetylase inhibition. [Internet] [Doctoral dissertation]. Georgia Tech; 2012. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1853/44907.

Council of Science Editors:

Guerrant W. Targeted histone deacetylase inhibition. [Doctoral Dissertation]. Georgia Tech; 2012. Available from: http://hdl.handle.net/1853/44907

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