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You searched for subject:(Gut peptides). Showing records 1 – 11 of 11 total matches.

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1. Maziarz, Mindy Patterson. High-Amylose Maize Resistant Starch Type 2 (HAM-RS2) Intake, Body Composition, and Satiety in Overweight Adults.

Degree: 2014, Texas Woman's University

 The purpose of this dissertation determined the impact of high-amylose maize resistant starch type 2 (HAM-RS2) consumption on objective and subjective satiety and body composition… (more)

Subjects/Keywords: Nutrition; Body composition; Fiber; Gut peptides; Overweight; Resistant starch; Satiety

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Maziarz, M. P. (2014). High-Amylose Maize Resistant Starch Type 2 (HAM-RS2) Intake, Body Composition, and Satiety in Overweight Adults. (Thesis). Texas Woman's University. Retrieved from http://hdl.handle.net/11274/3664

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maziarz, Mindy Patterson. “High-Amylose Maize Resistant Starch Type 2 (HAM-RS2) Intake, Body Composition, and Satiety in Overweight Adults.” 2014. Thesis, Texas Woman's University. Accessed December 04, 2020. http://hdl.handle.net/11274/3664.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maziarz, Mindy Patterson. “High-Amylose Maize Resistant Starch Type 2 (HAM-RS2) Intake, Body Composition, and Satiety in Overweight Adults.” 2014. Web. 04 Dec 2020.

Vancouver:

Maziarz MP. High-Amylose Maize Resistant Starch Type 2 (HAM-RS2) Intake, Body Composition, and Satiety in Overweight Adults. [Internet] [Thesis]. Texas Woman's University; 2014. [cited 2020 Dec 04]. Available from: http://hdl.handle.net/11274/3664.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maziarz MP. High-Amylose Maize Resistant Starch Type 2 (HAM-RS2) Intake, Body Composition, and Satiety in Overweight Adults. [Thesis]. Texas Woman's University; 2014. Available from: http://hdl.handle.net/11274/3664

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

2. Earley, Zachary. Role of the Intestinal Microbiota in Gut Barrier Dysfunction Following Burn Injury.

Degree: MS, Cell Biology, Neurobiology and Anatomy, 2014, Loyola University Chicago

  Burn injury represents a major medical problem with half a million cases requiring medical attention and 4,000 deaths reported annually. Sepsis and multiple organ… (more)

Subjects/Keywords: Antimicrobial Peptides; Barrier; Burn; Gut; Intestine; Microbiota; Immunology and Infectious Disease

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APA (6th Edition):

Earley, Z. (2014). Role of the Intestinal Microbiota in Gut Barrier Dysfunction Following Burn Injury. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/2619

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Earley, Zachary. “Role of the Intestinal Microbiota in Gut Barrier Dysfunction Following Burn Injury.” 2014. Thesis, Loyola University Chicago. Accessed December 04, 2020. https://ecommons.luc.edu/luc_theses/2619.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Earley, Zachary. “Role of the Intestinal Microbiota in Gut Barrier Dysfunction Following Burn Injury.” 2014. Web. 04 Dec 2020.

Vancouver:

Earley Z. Role of the Intestinal Microbiota in Gut Barrier Dysfunction Following Burn Injury. [Internet] [Thesis]. Loyola University Chicago; 2014. [cited 2020 Dec 04]. Available from: https://ecommons.luc.edu/luc_theses/2619.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Earley Z. Role of the Intestinal Microbiota in Gut Barrier Dysfunction Following Burn Injury. [Thesis]. Loyola University Chicago; 2014. Available from: https://ecommons.luc.edu/luc_theses/2619

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Poudel, Prakash. Effects of Commercial Feed Additives on the Gut Microbiota of Food Animals.

Degree: PhD, Animal Science, 2019, South Dakota State University

  Weaning is a stressful event of newborn animals which can lead to dysbiosis in the GIT causing invasion of pathogens, retarded growth, high incidence… (more)

Subjects/Keywords: Dairy calves; Essential oils; Feed additives; Gut microbiota; Peptides; Piglets; Animal Sciences

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APA (6th Edition):

Poudel, P. (2019). Effects of Commercial Feed Additives on the Gut Microbiota of Food Animals. (Doctoral Dissertation). South Dakota State University. Retrieved from https://openprairie.sdstate.edu/etd/3640

Chicago Manual of Style (16th Edition):

Poudel, Prakash. “Effects of Commercial Feed Additives on the Gut Microbiota of Food Animals.” 2019. Doctoral Dissertation, South Dakota State University. Accessed December 04, 2020. https://openprairie.sdstate.edu/etd/3640.

MLA Handbook (7th Edition):

Poudel, Prakash. “Effects of Commercial Feed Additives on the Gut Microbiota of Food Animals.” 2019. Web. 04 Dec 2020.

Vancouver:

Poudel P. Effects of Commercial Feed Additives on the Gut Microbiota of Food Animals. [Internet] [Doctoral dissertation]. South Dakota State University; 2019. [cited 2020 Dec 04]. Available from: https://openprairie.sdstate.edu/etd/3640.

Council of Science Editors:

Poudel P. Effects of Commercial Feed Additives on the Gut Microbiota of Food Animals. [Doctoral Dissertation]. South Dakota State University; 2019. Available from: https://openprairie.sdstate.edu/etd/3640


University of Cambridge

4. Billing, Lawrence. Characterisation of L-cell secretory mechanisms and colonic enteroendocrine cell subpopulations.

Degree: PhD, 2019, University of Cambridge

 Enteroendocrine cells (EECs) are chemosensitive cells of the gastrointestinal epithelium that exert a wide range of physiological effects via production and secretion of hormones in… (more)

Subjects/Keywords: INSL5; GLP-1; Enteroendocrine cells; Intestinal Organoids; scRNA-seq; L-cells; Gut peptides; Diabetes; Obesity; Colon; Small intestine; Neurotensin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Billing, L. (2019). Characterisation of L-cell secretory mechanisms and colonic enteroendocrine cell subpopulations. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/290641

Chicago Manual of Style (16th Edition):

Billing, Lawrence. “Characterisation of L-cell secretory mechanisms and colonic enteroendocrine cell subpopulations.” 2019. Doctoral Dissertation, University of Cambridge. Accessed December 04, 2020. https://www.repository.cam.ac.uk/handle/1810/290641.

MLA Handbook (7th Edition):

Billing, Lawrence. “Characterisation of L-cell secretory mechanisms and colonic enteroendocrine cell subpopulations.” 2019. Web. 04 Dec 2020.

Vancouver:

Billing L. Characterisation of L-cell secretory mechanisms and colonic enteroendocrine cell subpopulations. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Dec 04]. Available from: https://www.repository.cam.ac.uk/handle/1810/290641.

Council of Science Editors:

Billing L. Characterisation of L-cell secretory mechanisms and colonic enteroendocrine cell subpopulations. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/290641


University of Cambridge

5. Billing, Lawrence. Characterisation of L-cell secretory mechanisms and colonic enteroendocrine cell subpopulations.

Degree: PhD, 2019, University of Cambridge

 Enteroendocrine cells (EECs) are chemosensitive cells of the gastrointestinal epithelium that exert a wide range of physiological effects via production and secretion of hormones in… (more)

Subjects/Keywords: INSL5; GLP-1; Enteroendocrine cells; Intestinal Organoids; scRNA-seq; L-cells; Gut peptides; Diabetes; Obesity; Colon; Small intestine; Neurotensin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Billing, L. (2019). Characterisation of L-cell secretory mechanisms and colonic enteroendocrine cell subpopulations. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.37850 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774620

Chicago Manual of Style (16th Edition):

Billing, Lawrence. “Characterisation of L-cell secretory mechanisms and colonic enteroendocrine cell subpopulations.” 2019. Doctoral Dissertation, University of Cambridge. Accessed December 04, 2020. https://doi.org/10.17863/CAM.37850 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774620.

MLA Handbook (7th Edition):

Billing, Lawrence. “Characterisation of L-cell secretory mechanisms and colonic enteroendocrine cell subpopulations.” 2019. Web. 04 Dec 2020.

Vancouver:

Billing L. Characterisation of L-cell secretory mechanisms and colonic enteroendocrine cell subpopulations. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Dec 04]. Available from: https://doi.org/10.17863/CAM.37850 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774620.

Council of Science Editors:

Billing L. Characterisation of L-cell secretory mechanisms and colonic enteroendocrine cell subpopulations. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://doi.org/10.17863/CAM.37850 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774620


Universidade do Estado do Rio de Janeiro

6. Fernanda Torres Quitete. Perfil dos peptídeos gastrointestinais na obesidade programada pelo desmame precoce e após a terapia anti-obesidade com cálcio.

Degree: Master, 2014, Universidade do Estado do Rio de Janeiro

O desmame precoce (DP) leva ao desenvolvimento tardio de obesidade e de resistência insulínica (RI), sendo essas alterações prevenidas quando os animais são suplementados com… (more)

Subjects/Keywords: Lactação; Desmame precoce; Programação; Peptídeos gastrointestinais; Lactation; Early weaning; Programming; Gut peptides; NUTRICAO; Lactentes - Desmame Teses; Lactação - Teses; Resistência à insulina - Teses; Obesidade Teses

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Quitete, F. T. (2014). Perfil dos peptídeos gastrointestinais na obesidade programada pelo desmame precoce e após a terapia anti-obesidade com cálcio. (Masters Thesis). Universidade do Estado do Rio de Janeiro. Retrieved from http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=8991 ;

Chicago Manual of Style (16th Edition):

Quitete, Fernanda Torres. “Perfil dos peptídeos gastrointestinais na obesidade programada pelo desmame precoce e após a terapia anti-obesidade com cálcio.” 2014. Masters Thesis, Universidade do Estado do Rio de Janeiro. Accessed December 04, 2020. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=8991 ;.

MLA Handbook (7th Edition):

Quitete, Fernanda Torres. “Perfil dos peptídeos gastrointestinais na obesidade programada pelo desmame precoce e após a terapia anti-obesidade com cálcio.” 2014. Web. 04 Dec 2020.

Vancouver:

Quitete FT. Perfil dos peptídeos gastrointestinais na obesidade programada pelo desmame precoce e após a terapia anti-obesidade com cálcio. [Internet] [Masters thesis]. Universidade do Estado do Rio de Janeiro; 2014. [cited 2020 Dec 04]. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=8991 ;.

Council of Science Editors:

Quitete FT. Perfil dos peptídeos gastrointestinais na obesidade programada pelo desmame precoce e após a terapia anti-obesidade com cálcio. [Masters Thesis]. Universidade do Estado do Rio de Janeiro; 2014. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=8991 ;


Virginia Commonwealth University

7. Nadkarni, Priya. PULMONARY DELIVERY OF ANORECTIC GUT SECRETED PEPTIDES FOR APPETITE SUPPRESSION IN RATS.

Degree: PhD, Pharmaceutics, 2009, Virginia Commonwealth University

  This dissertation project aimed to demonstrate that pulmonary delivery of two anorectic gut secreted peptides, peptide YY (PYY) and oxyntomodulin (OXM) enabled food intake… (more)

Subjects/Keywords: Gut secreted peptides; Drug delivery; Inhalation; Pharmacokinetics; Preclinical; Obesity; Medicine and Health Sciences; Pharmacy and Pharmaceutical Sciences

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APA (6th Edition):

Nadkarni, P. (2009). PULMONARY DELIVERY OF ANORECTIC GUT SECRETED PEPTIDES FOR APPETITE SUPPRESSION IN RATS. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/7CGF-6T32 ; https://scholarscompass.vcu.edu/etd/1941

Chicago Manual of Style (16th Edition):

Nadkarni, Priya. “PULMONARY DELIVERY OF ANORECTIC GUT SECRETED PEPTIDES FOR APPETITE SUPPRESSION IN RATS.” 2009. Doctoral Dissertation, Virginia Commonwealth University. Accessed December 04, 2020. https://doi.org/10.25772/7CGF-6T32 ; https://scholarscompass.vcu.edu/etd/1941.

MLA Handbook (7th Edition):

Nadkarni, Priya. “PULMONARY DELIVERY OF ANORECTIC GUT SECRETED PEPTIDES FOR APPETITE SUPPRESSION IN RATS.” 2009. Web. 04 Dec 2020.

Vancouver:

Nadkarni P. PULMONARY DELIVERY OF ANORECTIC GUT SECRETED PEPTIDES FOR APPETITE SUPPRESSION IN RATS. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2009. [cited 2020 Dec 04]. Available from: https://doi.org/10.25772/7CGF-6T32 ; https://scholarscompass.vcu.edu/etd/1941.

Council of Science Editors:

Nadkarni P. PULMONARY DELIVERY OF ANORECTIC GUT SECRETED PEPTIDES FOR APPETITE SUPPRESSION IN RATS. [Doctoral Dissertation]. Virginia Commonwealth University; 2009. Available from: https://doi.org/10.25772/7CGF-6T32 ; https://scholarscompass.vcu.edu/etd/1941


The Ohio State University

8. Relling, Alejandro Enrique. EFFECT OF GUT PEPTIDES ON HYPOTHALAMIC mRNA CONCENTRATION AND DRY MATTER INTAKE IN RUMINANTS.

Degree: PhD, OSU Nutrition, 2009, The Ohio State University

 A series of experiments were conducted to gain a better understanding of mechanisms involved in feed intake regulation by ruminants. Specifically, the effect of changes… (more)

Subjects/Keywords: Anatomy and Physiology; Animals; Nutrition; Gut peptides; insulin; sheep; dairy cows; dry matter intake; ruminants; NPY; AgRP; POMC; hypothalamic neuropeptides; appetite

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APA (6th Edition):

Relling, A. E. (2009). EFFECT OF GUT PEPTIDES ON HYPOTHALAMIC mRNA CONCENTRATION AND DRY MATTER INTAKE IN RUMINANTS. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1243026530

Chicago Manual of Style (16th Edition):

Relling, Alejandro Enrique. “EFFECT OF GUT PEPTIDES ON HYPOTHALAMIC mRNA CONCENTRATION AND DRY MATTER INTAKE IN RUMINANTS.” 2009. Doctoral Dissertation, The Ohio State University. Accessed December 04, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1243026530.

MLA Handbook (7th Edition):

Relling, Alejandro Enrique. “EFFECT OF GUT PEPTIDES ON HYPOTHALAMIC mRNA CONCENTRATION AND DRY MATTER INTAKE IN RUMINANTS.” 2009. Web. 04 Dec 2020.

Vancouver:

Relling AE. EFFECT OF GUT PEPTIDES ON HYPOTHALAMIC mRNA CONCENTRATION AND DRY MATTER INTAKE IN RUMINANTS. [Internet] [Doctoral dissertation]. The Ohio State University; 2009. [cited 2020 Dec 04]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1243026530.

Council of Science Editors:

Relling AE. EFFECT OF GUT PEPTIDES ON HYPOTHALAMIC mRNA CONCENTRATION AND DRY MATTER INTAKE IN RUMINANTS. [Doctoral Dissertation]. The Ohio State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1243026530


University of Queensland

9. Pluschke, Anton. The impact of soluble dietary fibre on elements of digestive physiology.

Degree: Queensland Alliance for Agriculture and Food Innovation, 2015, University of Queensland

Subjects/Keywords: Soluble dietary fibres; Arabinoxylan; β-glucan; Pectin; Pigs; Digestive enzymes; Gut peptides; Retention time; Transit time; Digestion; 0908 Food Sciences; 1111 Nutrition and Dietetics

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APA (6th Edition):

Pluschke, A. (2015). The impact of soluble dietary fibre on elements of digestive physiology. (Thesis). University of Queensland. Retrieved from http://espace.library.uq.edu.au/view/UQ:361435

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pluschke, Anton. “The impact of soluble dietary fibre on elements of digestive physiology.” 2015. Thesis, University of Queensland. Accessed December 04, 2020. http://espace.library.uq.edu.au/view/UQ:361435.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pluschke, Anton. “The impact of soluble dietary fibre on elements of digestive physiology.” 2015. Web. 04 Dec 2020.

Vancouver:

Pluschke A. The impact of soluble dietary fibre on elements of digestive physiology. [Internet] [Thesis]. University of Queensland; 2015. [cited 2020 Dec 04]. Available from: http://espace.library.uq.edu.au/view/UQ:361435.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pluschke A. The impact of soluble dietary fibre on elements of digestive physiology. [Thesis]. University of Queensland; 2015. Available from: http://espace.library.uq.edu.au/view/UQ:361435

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Hoddy, Kristin K. Meal Timing during Alternate Day Fasting: Effect on Body Weight and Cardiovascular Disease Risk.

Degree: 2016, University of Illinois – Chicago

 Background and aims: Alternate day fasting (ADF) involves the cycling between consuming 25% energy needs on the “fast day” with ad libitum intake on the… (more)

Subjects/Keywords: Calorie restriction; alternate day fasting; safety; adverse events; eating disorder behaviors; obese adults; hunger; fullness; gut peptides; meal timing; meal frequency; coronary heart disease; insulin resistance

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APA (6th Edition):

Hoddy, K. K. (2016). Meal Timing during Alternate Day Fasting: Effect on Body Weight and Cardiovascular Disease Risk. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21175

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hoddy, Kristin K. “Meal Timing during Alternate Day Fasting: Effect on Body Weight and Cardiovascular Disease Risk.” 2016. Thesis, University of Illinois – Chicago. Accessed December 04, 2020. http://hdl.handle.net/10027/21175.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hoddy, Kristin K. “Meal Timing during Alternate Day Fasting: Effect on Body Weight and Cardiovascular Disease Risk.” 2016. Web. 04 Dec 2020.

Vancouver:

Hoddy KK. Meal Timing during Alternate Day Fasting: Effect on Body Weight and Cardiovascular Disease Risk. [Internet] [Thesis]. University of Illinois – Chicago; 2016. [cited 2020 Dec 04]. Available from: http://hdl.handle.net/10027/21175.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hoddy KK. Meal Timing during Alternate Day Fasting: Effect on Body Weight and Cardiovascular Disease Risk. [Thesis]. University of Illinois – Chicago; 2016. Available from: http://hdl.handle.net/10027/21175

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

11. Grenier, Émilie. Intestin et défauts métaboliques dans la résistance à l'insuline.

Degree: 2014, Université de Montréal

Subjects/Keywords: Résistance à l'insuline; Diabète de type 2; Intestin; Dyslipidémie diabétique; Transporteurs du cholestérol; Peptides gastro-intestinaux; Insulin resistance; Type 2 diabetes; Intestine; Diabetic dyslipidemia; Cholesterol transporters; Gut peptides; Health Sciences - Nutrition / Sciences de la santé - Alimentation et nutrition (UMI : 0570)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Grenier, . (2014). Intestin et défauts métaboliques dans la résistance à l'insuline. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/10891

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Grenier, Émilie. “Intestin et défauts métaboliques dans la résistance à l'insuline.” 2014. Thesis, Université de Montréal. Accessed December 04, 2020. http://hdl.handle.net/1866/10891.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Grenier, Émilie. “Intestin et défauts métaboliques dans la résistance à l'insuline.” 2014. Web. 04 Dec 2020.

Vancouver:

Grenier . Intestin et défauts métaboliques dans la résistance à l'insuline. [Internet] [Thesis]. Université de Montréal; 2014. [cited 2020 Dec 04]. Available from: http://hdl.handle.net/1866/10891.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Grenier . Intestin et défauts métaboliques dans la résistance à l'insuline. [Thesis]. Université de Montréal; 2014. Available from: http://hdl.handle.net/1866/10891

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.