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You searched for subject:(Gut brain axis). Showing records 1 – 30 of 38 total matches.

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Boston University

1. Angelides, Sophia Morfea. The gut-brain axis and cognition.

Degree: MS, Medical Sciences, 2018, Boston University

 The gut and the brain are in constant communication through pathways that include the immune system, the nervous system, neurotransmitters, and hormones. Modifications in the… (more)

Subjects/Keywords: Neurosciences; Cognition; Microbiome; Gut brain axis

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APA (6th Edition):

Angelides, S. M. (2018). The gut-brain axis and cognition. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/32981

Chicago Manual of Style (16th Edition):

Angelides, Sophia Morfea. “The gut-brain axis and cognition.” 2018. Masters Thesis, Boston University. Accessed August 24, 2019. http://hdl.handle.net/2144/32981.

MLA Handbook (7th Edition):

Angelides, Sophia Morfea. “The gut-brain axis and cognition.” 2018. Web. 24 Aug 2019.

Vancouver:

Angelides SM. The gut-brain axis and cognition. [Internet] [Masters thesis]. Boston University; 2018. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/2144/32981.

Council of Science Editors:

Angelides SM. The gut-brain axis and cognition. [Masters Thesis]. Boston University; 2018. Available from: http://hdl.handle.net/2144/32981


University of Gothenburg / Göteborgs Universitet

2. Vallöf, Daniel. Glucagon-like peptide-1 and alcohol-mediated behaviors in rodents.

Degree: 2019, University of Gothenburg / Göteborgs Universitet

 Alcohol use disorder (AUD) is a serious cause of morbidity and mortality. However, due to the limited efficacy of existing pharmacotherapies, further investigations of potential… (more)

Subjects/Keywords: Addiction; Dopamine; Gut-brain axis; Reward

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APA (6th Edition):

Vallöf, D. (2019). Glucagon-like peptide-1 and alcohol-mediated behaviors in rodents. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/57960

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vallöf, Daniel. “Glucagon-like peptide-1 and alcohol-mediated behaviors in rodents.” 2019. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed August 24, 2019. http://hdl.handle.net/2077/57960.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vallöf, Daniel. “Glucagon-like peptide-1 and alcohol-mediated behaviors in rodents.” 2019. Web. 24 Aug 2019.

Vancouver:

Vallöf D. Glucagon-like peptide-1 and alcohol-mediated behaviors in rodents. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2019. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/2077/57960.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vallöf D. Glucagon-like peptide-1 and alcohol-mediated behaviors in rodents. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2019. Available from: http://hdl.handle.net/2077/57960

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

3. Horne, Rachael. Exploring host genetic differences in gastrointestinal microbiota and homeostasis, through the production of fecal miRNA.

Degree: MSc, 2018, McMaster University

Research has shown that our gut microbiota confers many beneficial functions, including aiding the development of the immune system, metabolism, modulating stress reactivity and behaviour.… (more)

Subjects/Keywords: Gut microbiota; Antibiotics; MicroRNA; Host Genetics: Gut-Brain Axis

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APA (6th Edition):

Horne, R. (2018). Exploring host genetic differences in gastrointestinal microbiota and homeostasis, through the production of fecal miRNA. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/24048

Chicago Manual of Style (16th Edition):

Horne, Rachael. “Exploring host genetic differences in gastrointestinal microbiota and homeostasis, through the production of fecal miRNA.” 2018. Masters Thesis, McMaster University. Accessed August 24, 2019. http://hdl.handle.net/11375/24048.

MLA Handbook (7th Edition):

Horne, Rachael. “Exploring host genetic differences in gastrointestinal microbiota and homeostasis, through the production of fecal miRNA.” 2018. Web. 24 Aug 2019.

Vancouver:

Horne R. Exploring host genetic differences in gastrointestinal microbiota and homeostasis, through the production of fecal miRNA. [Internet] [Masters thesis]. McMaster University; 2018. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/11375/24048.

Council of Science Editors:

Horne R. Exploring host genetic differences in gastrointestinal microbiota and homeostasis, through the production of fecal miRNA. [Masters Thesis]. McMaster University; 2018. Available from: http://hdl.handle.net/11375/24048

4. Kelly, John. The gut microbiota in depression.

Degree: 2016, University College Cork

 The accruing data linking the gut microbiota to the development and function of the central nervous system has been proposed as a paradigm shift in… (more)

Subjects/Keywords: Brain-gut axis; Depression; Inflammation; Intestinal barrier; Tryptophan; Psychobiotic; Gut microbiota

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APA (6th Edition):

Kelly, J. (2016). The gut microbiota in depression. (Thesis). University College Cork. Retrieved from http://hdl.handle.net/10468/3507

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kelly, John. “The gut microbiota in depression.” 2016. Thesis, University College Cork. Accessed August 24, 2019. http://hdl.handle.net/10468/3507.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kelly, John. “The gut microbiota in depression.” 2016. Web. 24 Aug 2019.

Vancouver:

Kelly J. The gut microbiota in depression. [Internet] [Thesis]. University College Cork; 2016. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10468/3507.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kelly J. The gut microbiota in depression. [Thesis]. University College Cork; 2016. Available from: http://hdl.handle.net/10468/3507

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Bowling Green State University

5. Choudhury, Sayantan Roy. The Role Of Gut Microbiome In 3,4 Methylene Dioxymethamphetamine (MDMA) Mediated Hyperthermia In Rats.

Degree: MS, Biological Sciences, 2018, Bowling Green State University

 The gut microbiome is known to be home to 105 to 106 micro-organisms which are known to be phylogenetically diverse. There is growing evidence that… (more)

Subjects/Keywords: Microbiology; Molecular Biology; Gut microbiome; MDMA; Gut-Brain axis; Proteus

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APA (6th Edition):

Choudhury, S. R. (2018). The Role Of Gut Microbiome In 3,4 Methylene Dioxymethamphetamine (MDMA) Mediated Hyperthermia In Rats. (Masters Thesis). Bowling Green State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1530889852900613

Chicago Manual of Style (16th Edition):

Choudhury, Sayantan Roy. “The Role Of Gut Microbiome In 3,4 Methylene Dioxymethamphetamine (MDMA) Mediated Hyperthermia In Rats.” 2018. Masters Thesis, Bowling Green State University. Accessed August 24, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1530889852900613.

MLA Handbook (7th Edition):

Choudhury, Sayantan Roy. “The Role Of Gut Microbiome In 3,4 Methylene Dioxymethamphetamine (MDMA) Mediated Hyperthermia In Rats.” 2018. Web. 24 Aug 2019.

Vancouver:

Choudhury SR. The Role Of Gut Microbiome In 3,4 Methylene Dioxymethamphetamine (MDMA) Mediated Hyperthermia In Rats. [Internet] [Masters thesis]. Bowling Green State University; 2018. [cited 2019 Aug 24]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1530889852900613.

Council of Science Editors:

Choudhury SR. The Role Of Gut Microbiome In 3,4 Methylene Dioxymethamphetamine (MDMA) Mediated Hyperthermia In Rats. [Masters Thesis]. Bowling Green State University; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1530889852900613


Georgia State University

6. Fields, Christopher. Defining Microbial Signatures of Gut Dysbiosis in Models of Anxiety-related Disorders.

Degree: PhD, Neuroscience Institute, 2019, Georgia State University

  The gut microbiome is a complex community featuring a bewildering array of microbial species. Over the past couple decades, there has been an explosion… (more)

Subjects/Keywords: LPS; TLR4; Immune system; Gut-brain axis; Gut microbiota; Discriminant analysis

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APA (6th Edition):

Fields, C. (2019). Defining Microbial Signatures of Gut Dysbiosis in Models of Anxiety-related Disorders. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/neurosci_diss/42

Chicago Manual of Style (16th Edition):

Fields, Christopher. “Defining Microbial Signatures of Gut Dysbiosis in Models of Anxiety-related Disorders.” 2019. Doctoral Dissertation, Georgia State University. Accessed August 24, 2019. https://scholarworks.gsu.edu/neurosci_diss/42.

MLA Handbook (7th Edition):

Fields, Christopher. “Defining Microbial Signatures of Gut Dysbiosis in Models of Anxiety-related Disorders.” 2019. Web. 24 Aug 2019.

Vancouver:

Fields C. Defining Microbial Signatures of Gut Dysbiosis in Models of Anxiety-related Disorders. [Internet] [Doctoral dissertation]. Georgia State University; 2019. [cited 2019 Aug 24]. Available from: https://scholarworks.gsu.edu/neurosci_diss/42.

Council of Science Editors:

Fields C. Defining Microbial Signatures of Gut Dysbiosis in Models of Anxiety-related Disorders. [Doctoral Dissertation]. Georgia State University; 2019. Available from: https://scholarworks.gsu.edu/neurosci_diss/42


Universiteit Utrecht

7. Veen, H. van. Autism and the gut-brain axis.

Degree: 2011, Universiteit Utrecht

 Autism is a disorder of neural development for which modern medicine has no cure. As many autism subjects also suffer from Gastro-Intestinal (GI) problems, gut-brain(more)

Subjects/Keywords: Autism; gut-brain axis; probiotics; mTOR; extreme male brain.

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APA (6th Edition):

Veen, H. v. (2011). Autism and the gut-brain axis. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/217767

Chicago Manual of Style (16th Edition):

Veen, H van. “Autism and the gut-brain axis.” 2011. Masters Thesis, Universiteit Utrecht. Accessed August 24, 2019. http://dspace.library.uu.nl:8080/handle/1874/217767.

MLA Handbook (7th Edition):

Veen, H van. “Autism and the gut-brain axis.” 2011. Web. 24 Aug 2019.

Vancouver:

Veen Hv. Autism and the gut-brain axis. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2019 Aug 24]. Available from: http://dspace.library.uu.nl:8080/handle/1874/217767.

Council of Science Editors:

Veen Hv. Autism and the gut-brain axis. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/217767


Wayne State University

8. Neff, Andrew. Intestinal Bacteria And Their Relationship With Dietary Fiber, Tryptophan Metabolism, And Appetite.

Degree: PhD, Psychiatry and Behavioral Neurosciences, 2019, Wayne State University

  Within the broad theoretical framework of physiological variability impacting psychology, researchers have begun to explore the role gastrointestinal microbes play in higher psychological functions.… (more)

Subjects/Keywords: fiber-adherent bacteria; gut-brain-axis; gut microbiome; resistant starch; RNA; tryptophan; Microbiology; Neurosciences

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APA (6th Edition):

Neff, A. (2019). Intestinal Bacteria And Their Relationship With Dietary Fiber, Tryptophan Metabolism, And Appetite. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/2179

Chicago Manual of Style (16th Edition):

Neff, Andrew. “Intestinal Bacteria And Their Relationship With Dietary Fiber, Tryptophan Metabolism, And Appetite.” 2019. Doctoral Dissertation, Wayne State University. Accessed August 24, 2019. https://digitalcommons.wayne.edu/oa_dissertations/2179.

MLA Handbook (7th Edition):

Neff, Andrew. “Intestinal Bacteria And Their Relationship With Dietary Fiber, Tryptophan Metabolism, And Appetite.” 2019. Web. 24 Aug 2019.

Vancouver:

Neff A. Intestinal Bacteria And Their Relationship With Dietary Fiber, Tryptophan Metabolism, And Appetite. [Internet] [Doctoral dissertation]. Wayne State University; 2019. [cited 2019 Aug 24]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/2179.

Council of Science Editors:

Neff A. Intestinal Bacteria And Their Relationship With Dietary Fiber, Tryptophan Metabolism, And Appetite. [Doctoral Dissertation]. Wayne State University; 2019. Available from: https://digitalcommons.wayne.edu/oa_dissertations/2179

9. Jargalsaikhan, Undarmaa. Ingestion of dried-bonito broth (dashi) facilitates parvalbumin-immunoreactive neurons in the brain, and affects emotional behaviors in mice : かつおだし摂取のマウスの情動行動及び脳内パルブアルブミン陽性ニューロンに及ぼす影響.

Degree: 博士(医学), 2017, University of Toyama / 富山大学

富山大学・富生命博甲第88号・Jargalsaikhan Undarmaa・2017/03/23・★論文非公開★

Nutritional Neuroscience,Published online:21 Jul 2016,1-16,doi:10.1080/1028415X.2016.1208429に掲載

2016

Subjects/Keywords: gut-brain axis; parvalbumin-immunoreactive neuron; amygdala; hippocampus; prefrontal cortex; emotion

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jargalsaikhan, U. (2017). Ingestion of dried-bonito broth (dashi) facilitates parvalbumin-immunoreactive neurons in the brain, and affects emotional behaviors in mice : かつおだし摂取のマウスの情動行動及び脳内パルブアルブミン陽性ニューロンに及ぼす影響. (Thesis). University of Toyama / 富山大学. Retrieved from http://hdl.handle.net/10110/00017464 ; http://dx.doi.org/10.15099/00017464

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jargalsaikhan, Undarmaa. “Ingestion of dried-bonito broth (dashi) facilitates parvalbumin-immunoreactive neurons in the brain, and affects emotional behaviors in mice : かつおだし摂取のマウスの情動行動及び脳内パルブアルブミン陽性ニューロンに及ぼす影響.” 2017. Thesis, University of Toyama / 富山大学. Accessed August 24, 2019. http://hdl.handle.net/10110/00017464 ; http://dx.doi.org/10.15099/00017464.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jargalsaikhan, Undarmaa. “Ingestion of dried-bonito broth (dashi) facilitates parvalbumin-immunoreactive neurons in the brain, and affects emotional behaviors in mice : かつおだし摂取のマウスの情動行動及び脳内パルブアルブミン陽性ニューロンに及ぼす影響.” 2017. Web. 24 Aug 2019.

Vancouver:

Jargalsaikhan U. Ingestion of dried-bonito broth (dashi) facilitates parvalbumin-immunoreactive neurons in the brain, and affects emotional behaviors in mice : かつおだし摂取のマウスの情動行動及び脳内パルブアルブミン陽性ニューロンに及ぼす影響. [Internet] [Thesis]. University of Toyama / 富山大学; 2017. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10110/00017464 ; http://dx.doi.org/10.15099/00017464.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jargalsaikhan U. Ingestion of dried-bonito broth (dashi) facilitates parvalbumin-immunoreactive neurons in the brain, and affects emotional behaviors in mice : かつおだし摂取のマウスの情動行動及び脳内パルブアルブミン陽性ニューロンに及ぼす影響. [Thesis]. University of Toyama / 富山大学; 2017. Available from: http://hdl.handle.net/10110/00017464 ; http://dx.doi.org/10.15099/00017464

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

10. Bouwhuis, N. Early life stress predisposes to food allergy via modulation of the brain-gut-axis.

Degree: 2014, Universiteit Utrecht

 The neural, immune and endocrine pathway communication between the brain and the gut, is referred to as the brain-gut-axis. The brain-gut-axis is a complex reflex… (more)

Subjects/Keywords: Early life stress; brain-gut-axis; food allergy; microbiota

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APA (6th Edition):

Bouwhuis, N. (2014). Early life stress predisposes to food allergy via modulation of the brain-gut-axis. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/297574

Chicago Manual of Style (16th Edition):

Bouwhuis, N. “Early life stress predisposes to food allergy via modulation of the brain-gut-axis.” 2014. Masters Thesis, Universiteit Utrecht. Accessed August 24, 2019. http://dspace.library.uu.nl:8080/handle/1874/297574.

MLA Handbook (7th Edition):

Bouwhuis, N. “Early life stress predisposes to food allergy via modulation of the brain-gut-axis.” 2014. Web. 24 Aug 2019.

Vancouver:

Bouwhuis N. Early life stress predisposes to food allergy via modulation of the brain-gut-axis. [Internet] [Masters thesis]. Universiteit Utrecht; 2014. [cited 2019 Aug 24]. Available from: http://dspace.library.uu.nl:8080/handle/1874/297574.

Council of Science Editors:

Bouwhuis N. Early life stress predisposes to food allergy via modulation of the brain-gut-axis. [Masters Thesis]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/297574


University of Illinois – Urbana-Champaign

11. Davis, Elizabeth A. Investigation of non-traditional roles of the neural gut-brain axis.

Degree: PhD, Neuroscience, 2018, University of Illinois – Urbana-Champaign

 The neural gut-brain axis is an important bidirectional pathway through which the gastrointestinal (GI) tract communicates with the central nervous system (CNS). This axis includes… (more)

Subjects/Keywords: gut-brain axis; autonomic; intestinal epithelium; stem cells

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APA (6th Edition):

Davis, E. A. (2018). Investigation of non-traditional roles of the neural gut-brain axis. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/102415

Chicago Manual of Style (16th Edition):

Davis, Elizabeth A. “Investigation of non-traditional roles of the neural gut-brain axis.” 2018. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed August 24, 2019. http://hdl.handle.net/2142/102415.

MLA Handbook (7th Edition):

Davis, Elizabeth A. “Investigation of non-traditional roles of the neural gut-brain axis.” 2018. Web. 24 Aug 2019.

Vancouver:

Davis EA. Investigation of non-traditional roles of the neural gut-brain axis. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2018. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/2142/102415.

Council of Science Editors:

Davis EA. Investigation of non-traditional roles of the neural gut-brain axis. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2018. Available from: http://hdl.handle.net/2142/102415


University of California – San Francisco

12. Bouagnon, Aude. Neural Regulatory Mechanisms that Link Metabolism and Behavior in Caenorhabditis elegans.

Degree: Biomedical Sciences, 2019, University of California – San Francisco

 The neurotransmitter 5-hydroxytryptamine or serotonin, functions as an indicator of food availability and is an ancient mechanism by which invertebrate and vertebrate species translate sensory… (more)

Subjects/Keywords: Neurosciences; Genetics; C. elegans; fat metabolism; gut-brain axis; serotonin

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APA (6th Edition):

Bouagnon, A. (2019). Neural Regulatory Mechanisms that Link Metabolism and Behavior in Caenorhabditis elegans. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/44q4r4jf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bouagnon, Aude. “Neural Regulatory Mechanisms that Link Metabolism and Behavior in Caenorhabditis elegans.” 2019. Thesis, University of California – San Francisco. Accessed August 24, 2019. http://www.escholarship.org/uc/item/44q4r4jf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bouagnon, Aude. “Neural Regulatory Mechanisms that Link Metabolism and Behavior in Caenorhabditis elegans.” 2019. Web. 24 Aug 2019.

Vancouver:

Bouagnon A. Neural Regulatory Mechanisms that Link Metabolism and Behavior in Caenorhabditis elegans. [Internet] [Thesis]. University of California – San Francisco; 2019. [cited 2019 Aug 24]. Available from: http://www.escholarship.org/uc/item/44q4r4jf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bouagnon A. Neural Regulatory Mechanisms that Link Metabolism and Behavior in Caenorhabditis elegans. [Thesis]. University of California – San Francisco; 2019. Available from: http://www.escholarship.org/uc/item/44q4r4jf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

13. Cowan, Caitlin. Microorganisms to the rescue: The use of a probiotic treatment to reduce developmental vulnerability in rats.

Degree: Psychology, 2017, University of New South Wales

 Recent findings have revealed that the community of microorganisms residing within the gastrointestinal tract, collectively known as the gut microbiota, is not only far more… (more)

Subjects/Keywords: Probiotic effects; Microorganisms; Probiotic treatment; Stress; Gut-brain axis; Physical development

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APA (6th Edition):

Cowan, C. (2017). Microorganisms to the rescue: The use of a probiotic treatment to reduce developmental vulnerability in rats. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/57830 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:44959/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Cowan, Caitlin. “Microorganisms to the rescue: The use of a probiotic treatment to reduce developmental vulnerability in rats.” 2017. Doctoral Dissertation, University of New South Wales. Accessed August 24, 2019. http://handle.unsw.edu.au/1959.4/57830 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:44959/SOURCE02?view=true.

MLA Handbook (7th Edition):

Cowan, Caitlin. “Microorganisms to the rescue: The use of a probiotic treatment to reduce developmental vulnerability in rats.” 2017. Web. 24 Aug 2019.

Vancouver:

Cowan C. Microorganisms to the rescue: The use of a probiotic treatment to reduce developmental vulnerability in rats. [Internet] [Doctoral dissertation]. University of New South Wales; 2017. [cited 2019 Aug 24]. Available from: http://handle.unsw.edu.au/1959.4/57830 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:44959/SOURCE02?view=true.

Council of Science Editors:

Cowan C. Microorganisms to the rescue: The use of a probiotic treatment to reduce developmental vulnerability in rats. [Doctoral Dissertation]. University of New South Wales; 2017. Available from: http://handle.unsw.edu.au/1959.4/57830 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:44959/SOURCE02?view=true


University College Cork

14. Peterson, Veronica L. Comparative compositional analysis of the gut microbiome in animal models of addiction and stress.

Degree: 2018, University College Cork

 Intro: The microbiome-gut-brain axis (MGBA) has been shown to be instrumental to brain and behaviour, including psychological disorders affecting stress, depression, and anxiety. Many of… (more)

Subjects/Keywords: Microbiome-gut-brain axis; Bacteriome; Psychological stress; Virome; Addiction

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APA (6th Edition):

Peterson, V. L. (2018). Comparative compositional analysis of the gut microbiome in animal models of addiction and stress. (Thesis). University College Cork. Retrieved from http://hdl.handle.net/10468/7941

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Peterson, Veronica L. “Comparative compositional analysis of the gut microbiome in animal models of addiction and stress.” 2018. Thesis, University College Cork. Accessed August 24, 2019. http://hdl.handle.net/10468/7941.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Peterson, Veronica L. “Comparative compositional analysis of the gut microbiome in animal models of addiction and stress.” 2018. Web. 24 Aug 2019.

Vancouver:

Peterson VL. Comparative compositional analysis of the gut microbiome in animal models of addiction and stress. [Internet] [Thesis]. University College Cork; 2018. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10468/7941.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Peterson VL. Comparative compositional analysis of the gut microbiome in animal models of addiction and stress. [Thesis]. University College Cork; 2018. Available from: http://hdl.handle.net/10468/7941

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

15. Koopman, H.J.M. Overview of Parkinson's Disease: Examing the role for the Gut-Brain-Gut axis and the use of Nutrients based on preclinical and clinical evidence.

Degree: 2013, Universiteit Utrecht

Subjects/Keywords: Parkinson's Disease Gut-Brain-Gut Axis Nutrients

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APA (6th Edition):

Koopman, H. J. M. (2013). Overview of Parkinson's Disease: Examing the role for the Gut-Brain-Gut axis and the use of Nutrients based on preclinical and clinical evidence. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/276841

Chicago Manual of Style (16th Edition):

Koopman, H J M. “Overview of Parkinson's Disease: Examing the role for the Gut-Brain-Gut axis and the use of Nutrients based on preclinical and clinical evidence.” 2013. Masters Thesis, Universiteit Utrecht. Accessed August 24, 2019. http://dspace.library.uu.nl:8080/handle/1874/276841.

MLA Handbook (7th Edition):

Koopman, H J M. “Overview of Parkinson's Disease: Examing the role for the Gut-Brain-Gut axis and the use of Nutrients based on preclinical and clinical evidence.” 2013. Web. 24 Aug 2019.

Vancouver:

Koopman HJM. Overview of Parkinson's Disease: Examing the role for the Gut-Brain-Gut axis and the use of Nutrients based on preclinical and clinical evidence. [Internet] [Masters thesis]. Universiteit Utrecht; 2013. [cited 2019 Aug 24]. Available from: http://dspace.library.uu.nl:8080/handle/1874/276841.

Council of Science Editors:

Koopman HJM. Overview of Parkinson's Disease: Examing the role for the Gut-Brain-Gut axis and the use of Nutrients based on preclinical and clinical evidence. [Masters Thesis]. Universiteit Utrecht; 2013. Available from: http://dspace.library.uu.nl:8080/handle/1874/276841


Linnaeus University

16. Göransson, Sara Eckered. Kan probiotika lindra depression?.

Degree: Chemistry and Biomedical Sciences, 2019, Linnaeus University

Tidigare forskning har visat ett samband mellan vår tarmflora och vår fysiska hälsa, och idag görs även mycket forskning på om den även kan… (more)

Subjects/Keywords: Probiotics; Gut microbiota; Depression; Gut-brain axis; Probiotika; Tarmflora; Depression; Tarm-hjärn axeln; Medical and Health Sciences; Medicin och hälsovetenskap

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APA (6th Edition):

Göransson, S. E. (2019). Kan probiotika lindra depression?. (Thesis). Linnaeus University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-84831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Göransson, Sara Eckered. “Kan probiotika lindra depression?.” 2019. Thesis, Linnaeus University. Accessed August 24, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-84831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Göransson, Sara Eckered. “Kan probiotika lindra depression?.” 2019. Web. 24 Aug 2019.

Vancouver:

Göransson SE. Kan probiotika lindra depression?. [Internet] [Thesis]. Linnaeus University; 2019. [cited 2019 Aug 24]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-84831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Göransson SE. Kan probiotika lindra depression?. [Thesis]. Linnaeus University; 2019. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-84831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kentucky

17. Hoffman, Jared D. THE PREBIOTIC INULIN BENEFICIALLY MODULATES THE GUT-BRAIN AXIS BY ENHANCING METABOLISM IN AN APOE4 MOUSE MODEL.

Degree: 2018, University of Kentucky

 Alzheimer’s disease (AD) is the most common form of dementia and a growing disease burden that has seen pharmacological interventions primarily fail. Instead, it has… (more)

Subjects/Keywords: Alzheimer’s Disease; APOE4; Gut Microbiota; Gut-Brain Axis; Prebiotic Inulin; Amyloid β; Biochemical Phenomena, Metabolism, and Nutrition; Neuroscience and Neurobiology; Nutrition

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APA (6th Edition):

Hoffman, J. D. (2018). THE PREBIOTIC INULIN BENEFICIALLY MODULATES THE GUT-BRAIN AXIS BY ENHANCING METABOLISM IN AN APOE4 MOUSE MODEL. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/pharmacol_etds/24

Chicago Manual of Style (16th Edition):

Hoffman, Jared D. “THE PREBIOTIC INULIN BENEFICIALLY MODULATES THE GUT-BRAIN AXIS BY ENHANCING METABOLISM IN AN APOE4 MOUSE MODEL.” 2018. Doctoral Dissertation, University of Kentucky. Accessed August 24, 2019. https://uknowledge.uky.edu/pharmacol_etds/24.

MLA Handbook (7th Edition):

Hoffman, Jared D. “THE PREBIOTIC INULIN BENEFICIALLY MODULATES THE GUT-BRAIN AXIS BY ENHANCING METABOLISM IN AN APOE4 MOUSE MODEL.” 2018. Web. 24 Aug 2019.

Vancouver:

Hoffman JD. THE PREBIOTIC INULIN BENEFICIALLY MODULATES THE GUT-BRAIN AXIS BY ENHANCING METABOLISM IN AN APOE4 MOUSE MODEL. [Internet] [Doctoral dissertation]. University of Kentucky; 2018. [cited 2019 Aug 24]. Available from: https://uknowledge.uky.edu/pharmacol_etds/24.

Council of Science Editors:

Hoffman JD. THE PREBIOTIC INULIN BENEFICIALLY MODULATES THE GUT-BRAIN AXIS BY ENHANCING METABOLISM IN AN APOE4 MOUSE MODEL. [Doctoral Dissertation]. University of Kentucky; 2018. Available from: https://uknowledge.uky.edu/pharmacol_etds/24


McMaster University

18. Louis-Auguste, Marc Philippe. The role of the gut microbiome in Major Depressive Disorder.

Degree: MSc, 2019, McMaster University

The aetiology of major depressive disorder (MDD) is poorly understood. Current evidence suggests immune activation and gut microbiota may play a role. Recent studies demonstrated… (more)

Subjects/Keywords: depression; microbiome; MDD; microbiota; Major depressive disorder; bacteria; gut brain axis; nervous system; GABA; metabolites

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APA (6th Edition):

Louis-Auguste, M. P. (2019). The role of the gut microbiome in Major Depressive Disorder. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/24008

Chicago Manual of Style (16th Edition):

Louis-Auguste, Marc Philippe. “The role of the gut microbiome in Major Depressive Disorder.” 2019. Masters Thesis, McMaster University. Accessed August 24, 2019. http://hdl.handle.net/11375/24008.

MLA Handbook (7th Edition):

Louis-Auguste, Marc Philippe. “The role of the gut microbiome in Major Depressive Disorder.” 2019. Web. 24 Aug 2019.

Vancouver:

Louis-Auguste MP. The role of the gut microbiome in Major Depressive Disorder. [Internet] [Masters thesis]. McMaster University; 2019. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/11375/24008.

Council of Science Editors:

Louis-Auguste MP. The role of the gut microbiome in Major Depressive Disorder. [Masters Thesis]. McMaster University; 2019. Available from: http://hdl.handle.net/11375/24008


University of Newcastle

19. Barouei, Javad. Maternal probiotic intervention as a prophylaxis against the impact of neonatal stress: implications for irritable bowel syndrome.

Degree: PhD, 2013, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Neonatal stress is a common early life event, reported in some instances to be associated with adverse physiological… (more)

Subjects/Keywords: early life stress; irritable bowel syndrome; rat models; probiotic; brain immune gut axis; neonatal stress

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APA (6th Edition):

Barouei, J. (2013). Maternal probiotic intervention as a prophylaxis against the impact of neonatal stress: implications for irritable bowel syndrome. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/937488

Chicago Manual of Style (16th Edition):

Barouei, Javad. “Maternal probiotic intervention as a prophylaxis against the impact of neonatal stress: implications for irritable bowel syndrome.” 2013. Doctoral Dissertation, University of Newcastle. Accessed August 24, 2019. http://hdl.handle.net/1959.13/937488.

MLA Handbook (7th Edition):

Barouei, Javad. “Maternal probiotic intervention as a prophylaxis against the impact of neonatal stress: implications for irritable bowel syndrome.” 2013. Web. 24 Aug 2019.

Vancouver:

Barouei J. Maternal probiotic intervention as a prophylaxis against the impact of neonatal stress: implications for irritable bowel syndrome. [Internet] [Doctoral dissertation]. University of Newcastle; 2013. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/1959.13/937488.

Council of Science Editors:

Barouei J. Maternal probiotic intervention as a prophylaxis against the impact of neonatal stress: implications for irritable bowel syndrome. [Doctoral Dissertation]. University of Newcastle; 2013. Available from: http://hdl.handle.net/1959.13/937488


UCLA

20. Videlock, Elizabeth Jane. Molecular Mechanisms in Irritable Bowel Syndrome.

Degree: Molec, Cell, & Integ Physiology, 2019, UCLA

 Irritable bowel syndrome (IBS) is a common functional gastrointestinal disorder characterized by abdominal pain associated with alteration in stool form and/or frequency. The pathophysiology of… (more)

Subjects/Keywords: Medicine; Epithelial cell; Gene expression; Gut brain axis; Irritable bowel syndrome; lncRNA; Permeability

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APA (6th Edition):

Videlock, E. J. (2019). Molecular Mechanisms in Irritable Bowel Syndrome. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/2wb0z1q6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Videlock, Elizabeth Jane. “Molecular Mechanisms in Irritable Bowel Syndrome.” 2019. Thesis, UCLA. Accessed August 24, 2019. http://www.escholarship.org/uc/item/2wb0z1q6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Videlock, Elizabeth Jane. “Molecular Mechanisms in Irritable Bowel Syndrome.” 2019. Web. 24 Aug 2019.

Vancouver:

Videlock EJ. Molecular Mechanisms in Irritable Bowel Syndrome. [Internet] [Thesis]. UCLA; 2019. [cited 2019 Aug 24]. Available from: http://www.escholarship.org/uc/item/2wb0z1q6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Videlock EJ. Molecular Mechanisms in Irritable Bowel Syndrome. [Thesis]. UCLA; 2019. Available from: http://www.escholarship.org/uc/item/2wb0z1q6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Northeastern University

21. Puzan, Marissa. Neuron-epithelial Crosstalk In The Gastrointestinal Tract.

Degree: PhD, Department of Chemical Engineering, 2018, Northeastern University

 The enteric nervous system (ENS) is gastrointestinal tract's intrinsic network of neurons, which controls aspects of intestinal motility, secretion, and homeostasis. This expansive network consists… (more)

Subjects/Keywords: enteric nervous system; gut-brain axis; intestinal stem cell; Bioengineering; Chemical engineering

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APA (6th Edition):

Puzan, M. (2018). Neuron-epithelial Crosstalk In The Gastrointestinal Tract. (Doctoral Dissertation). Northeastern University. Retrieved from http://hdl.handle.net/2047/D20316324

Chicago Manual of Style (16th Edition):

Puzan, Marissa. “Neuron-epithelial Crosstalk In The Gastrointestinal Tract.” 2018. Doctoral Dissertation, Northeastern University. Accessed August 24, 2019. http://hdl.handle.net/2047/D20316324.

MLA Handbook (7th Edition):

Puzan, Marissa. “Neuron-epithelial Crosstalk In The Gastrointestinal Tract.” 2018. Web. 24 Aug 2019.

Vancouver:

Puzan M. Neuron-epithelial Crosstalk In The Gastrointestinal Tract. [Internet] [Doctoral dissertation]. Northeastern University; 2018. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/2047/D20316324.

Council of Science Editors:

Puzan M. Neuron-epithelial Crosstalk In The Gastrointestinal Tract. [Doctoral Dissertation]. Northeastern University; 2018. Available from: http://hdl.handle.net/2047/D20316324


Universidade do Rio Grande do Sul

22. Lauffer, Adriana. Efeito do estresse agudo, crônico e ambos combinados na permeabilidade intestinal de ratos.

Degree: 2015, Universidade do Rio Grande do Sul

Introdução: o estresse psicológico aumenta a permeabilidade intestinal em roedores e humanos, potencialmente levando a inflamação de baixo grau e aos sintomas em distúrbios gastrintestinais… (more)

Subjects/Keywords: Gastroenteropatias; Brain-gut axis; Corticosterone; Corticosterona; Estresse psicológico; Intestinal permeability; Mast cells; Psychological stress; Tight junction

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APA (6th Edition):

Lauffer, A. (2015). Efeito do estresse agudo, crônico e ambos combinados na permeabilidade intestinal de ratos. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/143409

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lauffer, Adriana. “Efeito do estresse agudo, crônico e ambos combinados na permeabilidade intestinal de ratos.” 2015. Thesis, Universidade do Rio Grande do Sul. Accessed August 24, 2019. http://hdl.handle.net/10183/143409.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lauffer, Adriana. “Efeito do estresse agudo, crônico e ambos combinados na permeabilidade intestinal de ratos.” 2015. Web. 24 Aug 2019.

Vancouver:

Lauffer A. Efeito do estresse agudo, crônico e ambos combinados na permeabilidade intestinal de ratos. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2015. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/10183/143409.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lauffer A. Efeito do estresse agudo, crônico e ambos combinados na permeabilidade intestinal de ratos. [Thesis]. Universidade do Rio Grande do Sul; 2015. Available from: http://hdl.handle.net/10183/143409

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

23. Khoshdel, Amir. The Effects of Bifidobacterium Longum NCC3001 on AH Neuron Excitability and Slow Wave Activity of the Mouse Intestine.

Degree: MSc, 2012, McMaster University

  The small intestine holds an intrinsic ability to digest and absorb nutrients from the food we intake without intervention from the central nervous system.… (more)

Subjects/Keywords: gut-brain axis; probiotics; afferent neuron; Digestive, Oral, and Skin Physiology; Digestive, Oral, and Skin Physiology

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APA (6th Edition):

Khoshdel, A. (2012). The Effects of Bifidobacterium Longum NCC3001 on AH Neuron Excitability and Slow Wave Activity of the Mouse Intestine. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/11886

Chicago Manual of Style (16th Edition):

Khoshdel, Amir. “The Effects of Bifidobacterium Longum NCC3001 on AH Neuron Excitability and Slow Wave Activity of the Mouse Intestine.” 2012. Masters Thesis, McMaster University. Accessed August 24, 2019. http://hdl.handle.net/11375/11886.

MLA Handbook (7th Edition):

Khoshdel, Amir. “The Effects of Bifidobacterium Longum NCC3001 on AH Neuron Excitability and Slow Wave Activity of the Mouse Intestine.” 2012. Web. 24 Aug 2019.

Vancouver:

Khoshdel A. The Effects of Bifidobacterium Longum NCC3001 on AH Neuron Excitability and Slow Wave Activity of the Mouse Intestine. [Internet] [Masters thesis]. McMaster University; 2012. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/11375/11886.

Council of Science Editors:

Khoshdel A. The Effects of Bifidobacterium Longum NCC3001 on AH Neuron Excitability and Slow Wave Activity of the Mouse Intestine. [Masters Thesis]. McMaster University; 2012. Available from: http://hdl.handle.net/11375/11886

24. Perez Pardo, P. Targeting the Gut-Brain axis in Parkinson's disease.

Degree: 2017, University Utrecht

 Parkinson’s disease (PD) is the most common progressive movement disorder, with increasing age being the greatest risk factor for its development. PD is hallmarked by… (more)

Subjects/Keywords: Parkinson's disease; Gut-Brain axis; Gastrointestinal dysfunction; gut microbiome; dietary interventions

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APA (6th Edition):

Perez Pardo, P. (2017). Targeting the Gut-Brain axis in Parkinson's disease. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/354242 ; URN:NBN:NL:UI:10-1874-354242 ; urn:isbn:978-94-6233-709-1 ; URN:NBN:NL:UI:10-1874-354242 ; http://dspace.library.uu.nl/handle/1874/354242

Chicago Manual of Style (16th Edition):

Perez Pardo, P. “Targeting the Gut-Brain axis in Parkinson's disease.” 2017. Doctoral Dissertation, University Utrecht. Accessed August 24, 2019. http://dspace.library.uu.nl/handle/1874/354242 ; URN:NBN:NL:UI:10-1874-354242 ; urn:isbn:978-94-6233-709-1 ; URN:NBN:NL:UI:10-1874-354242 ; http://dspace.library.uu.nl/handle/1874/354242.

MLA Handbook (7th Edition):

Perez Pardo, P. “Targeting the Gut-Brain axis in Parkinson's disease.” 2017. Web. 24 Aug 2019.

Vancouver:

Perez Pardo P. Targeting the Gut-Brain axis in Parkinson's disease. [Internet] [Doctoral dissertation]. University Utrecht; 2017. [cited 2019 Aug 24]. Available from: http://dspace.library.uu.nl/handle/1874/354242 ; URN:NBN:NL:UI:10-1874-354242 ; urn:isbn:978-94-6233-709-1 ; URN:NBN:NL:UI:10-1874-354242 ; http://dspace.library.uu.nl/handle/1874/354242.

Council of Science Editors:

Perez Pardo P. Targeting the Gut-Brain axis in Parkinson's disease. [Doctoral Dissertation]. University Utrecht; 2017. Available from: http://dspace.library.uu.nl/handle/1874/354242 ; URN:NBN:NL:UI:10-1874-354242 ; urn:isbn:978-94-6233-709-1 ; URN:NBN:NL:UI:10-1874-354242 ; http://dspace.library.uu.nl/handle/1874/354242

25. Bavelaar, B.M. The microbiome-­gut‐brain‐axis in coeliac disease: mechanisms for depression and anxiety co‐morbidity.

Degree: 2013, Universiteit Utrecht

 Coeliac disease (CD) is an abundant autoimmune disease of the gastrointestinal (GI) tract. The pathogenesis of CD is triggered by administration of gluten, a common… (more)

Subjects/Keywords: Coeliac disease; microbiome-gut-brain axis; depression; anxiety

…COELIAC DISEASE AND THE MICROBIOME-­‐GUT-­‐BRAIN AXIS 7… …GUT-­‐BRAIN AXIS in CD (32). According to Ludvigsson… …et al., however, The microbiome-­‐gut-­‐brain axis can be of… …MICROBIOME-­‐GUT-­‐BRAIN AXIS al., the investigators found that… …COELIAC DISEASE AND THE MICROBIOME-­‐GUT-­‐BRAIN AXIS receptors… 

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APA (6th Edition):

Bavelaar, B. M. (2013). The microbiome-­gut‐brain‐axis in coeliac disease: mechanisms for depression and anxiety co‐morbidity. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/281761

Chicago Manual of Style (16th Edition):

Bavelaar, B M. “The microbiome-­gut‐brain‐axis in coeliac disease: mechanisms for depression and anxiety co‐morbidity.” 2013. Masters Thesis, Universiteit Utrecht. Accessed August 24, 2019. http://dspace.library.uu.nl:8080/handle/1874/281761.

MLA Handbook (7th Edition):

Bavelaar, B M. “The microbiome-­gut‐brain‐axis in coeliac disease: mechanisms for depression and anxiety co‐morbidity.” 2013. Web. 24 Aug 2019.

Vancouver:

Bavelaar BM. The microbiome-­gut‐brain‐axis in coeliac disease: mechanisms for depression and anxiety co‐morbidity. [Internet] [Masters thesis]. Universiteit Utrecht; 2013. [cited 2019 Aug 24]. Available from: http://dspace.library.uu.nl:8080/handle/1874/281761.

Council of Science Editors:

Bavelaar BM. The microbiome-­gut‐brain‐axis in coeliac disease: mechanisms for depression and anxiety co‐morbidity. [Masters Thesis]. Universiteit Utrecht; 2013. Available from: http://dspace.library.uu.nl:8080/handle/1874/281761

26. Delaere, Fabien. Détection des nutriments et contrôle central de la prise alimentaire : Nutrient sensing and central control of food intake.

Degree: Docteur es, Physiologie et neurosciences, 2009, Université Claude Bernard – Lyon I

En relation avec sa position anatomique, la détection portale de nutriments se situe au coeur de l’impact de la composition nutritionnelle d’un repas sur la… (more)

Subjects/Keywords: Détection des nutriments; Prise alimentaire; Activation cérébrale; Système nerveux périphérique; Interaction cerveau-périphérie; Nutrient sensing; Food intake; Central activation; Peripheral nervous system; Gut-brain axis; 612

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APA (6th Edition):

Delaere, F. (2009). Détection des nutriments et contrôle central de la prise alimentaire : Nutrient sensing and central control of food intake. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2009LYO10269

Chicago Manual of Style (16th Edition):

Delaere, Fabien. “Détection des nutriments et contrôle central de la prise alimentaire : Nutrient sensing and central control of food intake.” 2009. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed August 24, 2019. http://www.theses.fr/2009LYO10269.

MLA Handbook (7th Edition):

Delaere, Fabien. “Détection des nutriments et contrôle central de la prise alimentaire : Nutrient sensing and central control of food intake.” 2009. Web. 24 Aug 2019.

Vancouver:

Delaere F. Détection des nutriments et contrôle central de la prise alimentaire : Nutrient sensing and central control of food intake. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2009. [cited 2019 Aug 24]. Available from: http://www.theses.fr/2009LYO10269.

Council of Science Editors:

Delaere F. Détection des nutriments et contrôle central de la prise alimentaire : Nutrient sensing and central control of food intake. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2009. Available from: http://www.theses.fr/2009LYO10269

27. Voinot, Florian. Axe cerveau-intestin et contrôle de la prise alimentaire : exemple d'altérations chez un modèle animal de schizophrénie : Brain-gut axis and control of foodintake : example of alterations in an animal model of schizophrenia.

Degree: Docteur es, Physiologie et biologie des organismes, populations, interactions, 2012, Université de Strasbourg

L’axe cerveau-intestin désigne l’interaction bidirectionnelle entre le cerveau et le tube digestif. Bien que la leptine, hormone produite par le tissu adipeux, participe à la… (more)

Subjects/Keywords: Axe cerveau-intestin; Leptine; Système nerveux entérique; Schizophrénie; Inflammation; Fonctions gastro-intestinales; Nerf vague; Brain-gut axis; Leptin; Enteric nervous system; Schizophrenia; Inflammation; Gastrointestinal functions; Vagus nerve; 571.7; 616.89

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APA (6th Edition):

Voinot, F. (2012). Axe cerveau-intestin et contrôle de la prise alimentaire : exemple d'altérations chez un modèle animal de schizophrénie : Brain-gut axis and control of foodintake : example of alterations in an animal model of schizophrenia. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2012STRAJ058

Chicago Manual of Style (16th Edition):

Voinot, Florian. “Axe cerveau-intestin et contrôle de la prise alimentaire : exemple d'altérations chez un modèle animal de schizophrénie : Brain-gut axis and control of foodintake : example of alterations in an animal model of schizophrenia.” 2012. Doctoral Dissertation, Université de Strasbourg. Accessed August 24, 2019. http://www.theses.fr/2012STRAJ058.

MLA Handbook (7th Edition):

Voinot, Florian. “Axe cerveau-intestin et contrôle de la prise alimentaire : exemple d'altérations chez un modèle animal de schizophrénie : Brain-gut axis and control of foodintake : example of alterations in an animal model of schizophrenia.” 2012. Web. 24 Aug 2019.

Vancouver:

Voinot F. Axe cerveau-intestin et contrôle de la prise alimentaire : exemple d'altérations chez un modèle animal de schizophrénie : Brain-gut axis and control of foodintake : example of alterations in an animal model of schizophrenia. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2012. [cited 2019 Aug 24]. Available from: http://www.theses.fr/2012STRAJ058.

Council of Science Editors:

Voinot F. Axe cerveau-intestin et contrôle de la prise alimentaire : exemple d'altérations chez un modèle animal de schizophrénie : Brain-gut axis and control of foodintake : example of alterations in an animal model of schizophrenia. [Doctoral Dissertation]. Université de Strasbourg; 2012. Available from: http://www.theses.fr/2012STRAJ058

28. SONG GUANGHUI. Exploring the brain-gut axis in irritable bowel syndrome: Specific emphasis on stress and melatonin.

Degree: 2006, National University of Singapore

Subjects/Keywords: brain-gut axis; irritable bowel syndrome; stress; melatonin; sleep disturbance; pain

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

GUANGHUI, S. (2006). Exploring the brain-gut axis in irritable bowel syndrome: Specific emphasis on stress and melatonin. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/15308

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

GUANGHUI, SONG. “Exploring the brain-gut axis in irritable bowel syndrome: Specific emphasis on stress and melatonin.” 2006. Thesis, National University of Singapore. Accessed August 24, 2019. http://scholarbank.nus.edu.sg/handle/10635/15308.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

GUANGHUI, SONG. “Exploring the brain-gut axis in irritable bowel syndrome: Specific emphasis on stress and melatonin.” 2006. Web. 24 Aug 2019.

Vancouver:

GUANGHUI S. Exploring the brain-gut axis in irritable bowel syndrome: Specific emphasis on stress and melatonin. [Internet] [Thesis]. National University of Singapore; 2006. [cited 2019 Aug 24]. Available from: http://scholarbank.nus.edu.sg/handle/10635/15308.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

GUANGHUI S. Exploring the brain-gut axis in irritable bowel syndrome: Specific emphasis on stress and melatonin. [Thesis]. National University of Singapore; 2006. Available from: http://scholarbank.nus.edu.sg/handle/10635/15308

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Iowa State University

29. Ghaisas, Shivani. The effect of intestinal inflammation and enteric nervous system deregulation in the pathogenesis of Parkinsonian syndrome.

Degree: 2017, Iowa State University

 Increasing evidence from epidemiological studies, retrospective studies, clinical observations as well as pre-clinical in-vitro and in-vivo data suggest that Parkinson’s disease involves both the enteric… (more)

Subjects/Keywords: Enteric Glial cells; Enteric Nervous System; Environmental toxins; Gut-brain axis; Manganese toxicity; Parkinson's Disease; Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology; Neuroscience and Neurobiology; Toxicology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ghaisas, S. (2017). The effect of intestinal inflammation and enteric nervous system deregulation in the pathogenesis of Parkinsonian syndrome. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/16908

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ghaisas, Shivani. “The effect of intestinal inflammation and enteric nervous system deregulation in the pathogenesis of Parkinsonian syndrome.” 2017. Thesis, Iowa State University. Accessed August 24, 2019. https://lib.dr.iastate.edu/etd/16908.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ghaisas, Shivani. “The effect of intestinal inflammation and enteric nervous system deregulation in the pathogenesis of Parkinsonian syndrome.” 2017. Web. 24 Aug 2019.

Vancouver:

Ghaisas S. The effect of intestinal inflammation and enteric nervous system deregulation in the pathogenesis of Parkinsonian syndrome. [Internet] [Thesis]. Iowa State University; 2017. [cited 2019 Aug 24]. Available from: https://lib.dr.iastate.edu/etd/16908.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ghaisas S. The effect of intestinal inflammation and enteric nervous system deregulation in the pathogenesis of Parkinsonian syndrome. [Thesis]. Iowa State University; 2017. Available from: https://lib.dr.iastate.edu/etd/16908

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Iowa State University

30. Ghaisas, Shivani. The effect of intestinal inflammation and enteric nervous system deregulation in the pathogenesis of Parkinsonian syndrome.

Degree: 2017, Iowa State University

 Increasing evidence from epidemiological studies, retrospective studies, clinical observations as well as pre-clinical in-vitro and in-vivo data suggest that Parkinson’s disease involves both the enteric… (more)

Subjects/Keywords: Enteric Glial cells; Enteric Nervous System; Environmental toxins; Gut-brain axis; Manganese toxicity; Parkinson's Disease; Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology; Neuroscience and Neurobiology; Toxicology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ghaisas, S. (2017). The effect of intestinal inflammation and enteric nervous system deregulation in the pathogenesis of Parkinsonian syndrome. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/17190

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ghaisas, Shivani. “The effect of intestinal inflammation and enteric nervous system deregulation in the pathogenesis of Parkinsonian syndrome.” 2017. Thesis, Iowa State University. Accessed August 24, 2019. https://lib.dr.iastate.edu/etd/17190.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ghaisas, Shivani. “The effect of intestinal inflammation and enteric nervous system deregulation in the pathogenesis of Parkinsonian syndrome.” 2017. Web. 24 Aug 2019.

Vancouver:

Ghaisas S. The effect of intestinal inflammation and enteric nervous system deregulation in the pathogenesis of Parkinsonian syndrome. [Internet] [Thesis]. Iowa State University; 2017. [cited 2019 Aug 24]. Available from: https://lib.dr.iastate.edu/etd/17190.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ghaisas S. The effect of intestinal inflammation and enteric nervous system deregulation in the pathogenesis of Parkinsonian syndrome. [Thesis]. Iowa State University; 2017. Available from: https://lib.dr.iastate.edu/etd/17190

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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