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You searched for subject:(Gpnmb). Showing records 1 – 5 of 5 total matches.

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University of Rochester

1. Prizant, Hen. mTOR-mediated Leiomyoma Growth in Uterine-specific Tsc2-null Mice is Exclusively Dependent on Estradiol Signaling - Implications in the Treatment of Lymphangioleiomyomatosis (LAM).

Degree: PhD, 2016, University of Rochester

 Lymphangioleiomyomatosis (LAM) is a devastating rare lung disease in which adenomas consisting of abnormal smooth-muscle cells grow within the lungs and progressively lead to loss… (more)

Subjects/Keywords: Estrogen; GPNMB; Lympangioleiomyomatosis; metalloproteinase; mTOR; Uterus

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APA (6th Edition):

Prizant, H. (2016). mTOR-mediated Leiomyoma Growth in Uterine-specific Tsc2-null Mice is Exclusively Dependent on Estradiol Signaling - Implications in the Treatment of Lymphangioleiomyomatosis (LAM). (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/30560

Chicago Manual of Style (16th Edition):

Prizant, Hen. “mTOR-mediated Leiomyoma Growth in Uterine-specific Tsc2-null Mice is Exclusively Dependent on Estradiol Signaling - Implications in the Treatment of Lymphangioleiomyomatosis (LAM).” 2016. Doctoral Dissertation, University of Rochester. Accessed October 24, 2020. http://hdl.handle.net/1802/30560.

MLA Handbook (7th Edition):

Prizant, Hen. “mTOR-mediated Leiomyoma Growth in Uterine-specific Tsc2-null Mice is Exclusively Dependent on Estradiol Signaling - Implications in the Treatment of Lymphangioleiomyomatosis (LAM).” 2016. Web. 24 Oct 2020.

Vancouver:

Prizant H. mTOR-mediated Leiomyoma Growth in Uterine-specific Tsc2-null Mice is Exclusively Dependent on Estradiol Signaling - Implications in the Treatment of Lymphangioleiomyomatosis (LAM). [Internet] [Doctoral dissertation]. University of Rochester; 2016. [cited 2020 Oct 24]. Available from: http://hdl.handle.net/1802/30560.

Council of Science Editors:

Prizant H. mTOR-mediated Leiomyoma Growth in Uterine-specific Tsc2-null Mice is Exclusively Dependent on Estradiol Signaling - Implications in the Treatment of Lymphangioleiomyomatosis (LAM). [Doctoral Dissertation]. University of Rochester; 2016. Available from: http://hdl.handle.net/1802/30560

2. Smuczek, Basilio. Peptídeo C16, derivado da laminina, regulando a expressão de potenciais biomarcadorers do câncer de mama.

Degree: Mestrado, Biologia Celular e Tecidual, 2014, University of São Paulo

O câncer de mama é importante problema de saúde pública. O microambiente onde as células cancerígenas se encontram possui moléculas como a laminina e seus… (more)

Subjects/Keywords: Breast cancer; C16 peptide; Câncer de mama; Extracellular matrix; GPNMB; GPNMB; Laminin; Laminina; Matriz extracelular; Peptídeo C16; SPOCK; SPOCK

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APA (6th Edition):

Smuczek, B. (2014). Peptídeo C16, derivado da laminina, regulando a expressão de potenciais biomarcadorers do câncer de mama. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42134/tde-20022015-090950/ ;

Chicago Manual of Style (16th Edition):

Smuczek, Basilio. “Peptídeo C16, derivado da laminina, regulando a expressão de potenciais biomarcadorers do câncer de mama.” 2014. Masters Thesis, University of São Paulo. Accessed October 24, 2020. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-20022015-090950/ ;.

MLA Handbook (7th Edition):

Smuczek, Basilio. “Peptídeo C16, derivado da laminina, regulando a expressão de potenciais biomarcadorers do câncer de mama.” 2014. Web. 24 Oct 2020.

Vancouver:

Smuczek B. Peptídeo C16, derivado da laminina, regulando a expressão de potenciais biomarcadorers do câncer de mama. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2020 Oct 24]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42134/tde-20022015-090950/ ;.

Council of Science Editors:

Smuczek B. Peptídeo C16, derivado da laminina, regulando a expressão de potenciais biomarcadorers do câncer de mama. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/42/42134/tde-20022015-090950/ ;


Temple University

3. Belcher, Joyce Yvonne. Bone Cell Autonomous Effects of Osteoactivin In Vivo.

Degree: PhD, 2012, Temple University

Cell Biology

Osteoactivin (OA) is a type I transmembrane glycoprotein initially identified in bone in 2002. The protein is synthesized, processed and heavily glycosylated by… (more)

Subjects/Keywords: Cellular biology; Animal Model; Gpnmb; Osteoactivin; Osteoblasts; Osteoclasts; Osteopetrosis

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APA (6th Edition):

Belcher, J. Y. (2012). Bone Cell Autonomous Effects of Osteoactivin In Vivo. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,183061

Chicago Manual of Style (16th Edition):

Belcher, Joyce Yvonne. “Bone Cell Autonomous Effects of Osteoactivin In Vivo.” 2012. Doctoral Dissertation, Temple University. Accessed October 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,183061.

MLA Handbook (7th Edition):

Belcher, Joyce Yvonne. “Bone Cell Autonomous Effects of Osteoactivin In Vivo.” 2012. Web. 24 Oct 2020.

Vancouver:

Belcher JY. Bone Cell Autonomous Effects of Osteoactivin In Vivo. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Oct 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,183061.

Council of Science Editors:

Belcher JY. Bone Cell Autonomous Effects of Osteoactivin In Vivo. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,183061


Kent State University

4. Al-Adlaan, Asaad A. A NOVEL ANTI-INFLAMMATORY ROLE OF OSTEOACTIVIN/GPNMB INPOST-TRAUMATIC OSTEOARTHRITIS.

Degree: PhD, College of Arts and Sciences / School of Biomedical Sciences, 2017, Kent State University

 Osteoarthritis (OA) is a degenerative form of arthritis leading to joint disability. Ithas been estimated that more than 15% of world’s population have joint diseases,… (more)

Subjects/Keywords: Biomedical Research; Immunology; Molecular Biology; Osteoarthritis, inflammation, miRNA-150, CD44, GPNMB

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APA (6th Edition):

Al-Adlaan, A. A. (2017). A NOVEL ANTI-INFLAMMATORY ROLE OF OSTEOACTIVIN/GPNMB INPOST-TRAUMATIC OSTEOARTHRITIS. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent15114587826723

Chicago Manual of Style (16th Edition):

Al-Adlaan, Asaad A. “A NOVEL ANTI-INFLAMMATORY ROLE OF OSTEOACTIVIN/GPNMB INPOST-TRAUMATIC OSTEOARTHRITIS.” 2017. Doctoral Dissertation, Kent State University. Accessed October 24, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=kent15114587826723.

MLA Handbook (7th Edition):

Al-Adlaan, Asaad A. “A NOVEL ANTI-INFLAMMATORY ROLE OF OSTEOACTIVIN/GPNMB INPOST-TRAUMATIC OSTEOARTHRITIS.” 2017. Web. 24 Oct 2020.

Vancouver:

Al-Adlaan AA. A NOVEL ANTI-INFLAMMATORY ROLE OF OSTEOACTIVIN/GPNMB INPOST-TRAUMATIC OSTEOARTHRITIS. [Internet] [Doctoral dissertation]. Kent State University; 2017. [cited 2020 Oct 24]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent15114587826723.

Council of Science Editors:

Al-Adlaan AA. A NOVEL ANTI-INFLAMMATORY ROLE OF OSTEOACTIVIN/GPNMB INPOST-TRAUMATIC OSTEOARTHRITIS. [Doctoral Dissertation]. Kent State University; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent15114587826723

5. Stinnett, Hilary M. OSTEOACTIVIN IN SKELETON: CHARACTERIZATION OF OSTEOACTIVIN KNOCKOUT MICE & THERAPEUTIC IMPLICATIONS.

Degree: PhD, College of Arts and Sciences / School of Biomedical Sciences, 2015, Kent State University

 Osteoporosis impacts 55% of the population aged 50 and older, with post-menopausal, thin women of Caucasian descent at the highest risk for developing this disease.… (more)

Subjects/Keywords: Biology; Pharmacology; Cellular Biology; osteoactivin, osteoblast, osteoclast, gpnmb, osteoporosis, knockout mouse model, surgical model, ovariectomy

…x28;OA), also known as GPNMB in melanocytes or DC-HIL in dendritic cells (Shikano… 

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APA (6th Edition):

Stinnett, H. M. (2015). OSTEOACTIVIN IN SKELETON: CHARACTERIZATION OF OSTEOACTIVIN KNOCKOUT MICE & THERAPEUTIC IMPLICATIONS. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1428859108

Chicago Manual of Style (16th Edition):

Stinnett, Hilary M. “OSTEOACTIVIN IN SKELETON: CHARACTERIZATION OF OSTEOACTIVIN KNOCKOUT MICE & THERAPEUTIC IMPLICATIONS.” 2015. Doctoral Dissertation, Kent State University. Accessed October 24, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=kent1428859108.

MLA Handbook (7th Edition):

Stinnett, Hilary M. “OSTEOACTIVIN IN SKELETON: CHARACTERIZATION OF OSTEOACTIVIN KNOCKOUT MICE & THERAPEUTIC IMPLICATIONS.” 2015. Web. 24 Oct 2020.

Vancouver:

Stinnett HM. OSTEOACTIVIN IN SKELETON: CHARACTERIZATION OF OSTEOACTIVIN KNOCKOUT MICE & THERAPEUTIC IMPLICATIONS. [Internet] [Doctoral dissertation]. Kent State University; 2015. [cited 2020 Oct 24]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1428859108.

Council of Science Editors:

Stinnett HM. OSTEOACTIVIN IN SKELETON: CHARACTERIZATION OF OSTEOACTIVIN KNOCKOUT MICE & THERAPEUTIC IMPLICATIONS. [Doctoral Dissertation]. Kent State University; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1428859108

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