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You searched for subject:(Glucotoxicity). Showing records 1 – 11 of 11 total matches.

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Vanderbilt University

1. Bryant, Nora Kayton. Glutotoxic and Lipotoxic Consequences for Human β Cell Function In Vivo.

Degree: PhD, Molecular Physiology and Biophysics, 2015, Vanderbilt University

 Diabetes is characterized by excess levels of glucose and lipid in the blood, which have been proposed to directly and negatively impact islet β cells,… (more)

Subjects/Keywords: human islets; Islet biology; endocrinology; insulin; diabetes; glucotoxicity; lipotoxicity

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bryant, N. K. (2015). Glutotoxic and Lipotoxic Consequences for Human β Cell Function In Vivo. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-10162015-113112/ ;

Chicago Manual of Style (16th Edition):

Bryant, Nora Kayton. “Glutotoxic and Lipotoxic Consequences for Human β Cell Function In Vivo.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed April 19, 2019. http://etd.library.vanderbilt.edu/available/etd-10162015-113112/ ;.

MLA Handbook (7th Edition):

Bryant, Nora Kayton. “Glutotoxic and Lipotoxic Consequences for Human β Cell Function In Vivo.” 2015. Web. 19 Apr 2019.

Vancouver:

Bryant NK. Glutotoxic and Lipotoxic Consequences for Human β Cell Function In Vivo. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2019 Apr 19]. Available from: http://etd.library.vanderbilt.edu/available/etd-10162015-113112/ ;.

Council of Science Editors:

Bryant NK. Glutotoxic and Lipotoxic Consequences for Human β Cell Function In Vivo. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-10162015-113112/ ;


Université Catholique de Louvain

2. Balteau, Magali. Mechanisms of glucotoxicity in the heart.

Degree: 2012, Université Catholique de Louvain

Hyperglycemia is a powerful indicator of mortality in patients with acute myocardial infarction. Hyperglycemia might also result in toxic effects in cardiomyocytes. Indeed, it modifies… (more)

Subjects/Keywords: Glucotoxicity; Glucose metabolism; Oxidative stress; NADPH oxidase; SGLT

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APA (6th Edition):

Balteau, M. (2012). Mechanisms of glucotoxicity in the heart. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/106878

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Balteau, Magali. “Mechanisms of glucotoxicity in the heart.” 2012. Thesis, Université Catholique de Louvain. Accessed April 19, 2019. http://hdl.handle.net/2078.1/106878.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Balteau, Magali. “Mechanisms of glucotoxicity in the heart.” 2012. Web. 19 Apr 2019.

Vancouver:

Balteau M. Mechanisms of glucotoxicity in the heart. [Internet] [Thesis]. Université Catholique de Louvain; 2012. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/2078.1/106878.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Balteau M. Mechanisms of glucotoxicity in the heart. [Thesis]. Université Catholique de Louvain; 2012. Available from: http://hdl.handle.net/2078.1/106878

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

3. Tang, Christine. Mechanisms of High Glucose-induced Decrease in β-cell Function.

Degree: 2010, University of Toronto

Chronic hyperglycemia, a hallmark of type 2 diabetes, can decrease β-cell function and mass (β-cell glucotoxicity); however, the mechanisms are incompletely understood. The objective was… (more)

Subjects/Keywords: Glucotoxicity; Oxidative Stress; Type 2 Diabetes; Endoplasmic Reticulum Stress; c-jun N-terminal kinase; 0719

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APA (6th Edition):

Tang, C. (2010). Mechanisms of High Glucose-induced Decrease in β-cell Function. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/26331

Chicago Manual of Style (16th Edition):

Tang, Christine. “Mechanisms of High Glucose-induced Decrease in β-cell Function.” 2010. Doctoral Dissertation, University of Toronto. Accessed April 19, 2019. http://hdl.handle.net/1807/26331.

MLA Handbook (7th Edition):

Tang, Christine. “Mechanisms of High Glucose-induced Decrease in β-cell Function.” 2010. Web. 19 Apr 2019.

Vancouver:

Tang C. Mechanisms of High Glucose-induced Decrease in β-cell Function. [Internet] [Doctoral dissertation]. University of Toronto; 2010. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1807/26331.

Council of Science Editors:

Tang C. Mechanisms of High Glucose-induced Decrease in β-cell Function. [Doctoral Dissertation]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/26331


Université de Bordeaux I

4. Papin, Julien. Bases moléculaires des défauts sécrétoires des cellules ß pancréatiques lors de la glucotoxicité : Modelling of a cultural dictionnary for students of French as a foreign language in Colombia : cultural differences between French and Colombian universes.

Degree: Docteur es, Biologie cellulaire et Physiopathologie, 2009, Université de Bordeaux I

La glucotoxicité, ou exposition prolongée à de hautes teneurs en glucose, altère la fonction des cellules ß-pancréatiques et participe au développement du diabète. Il a… (more)

Subjects/Keywords: Glucotoxicité; AMPc; Voies de signalisation; Insuline; Calcium; Apoptose; Exocytose; Glucotoxicity; CAMP; Signalling pathways; Insulin; Calcium; Apoptosis; Exocytosis

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APA (6th Edition):

Papin, J. (2009). Bases moléculaires des défauts sécrétoires des cellules ß pancréatiques lors de la glucotoxicité : Modelling of a cultural dictionnary for students of French as a foreign language in Colombia : cultural differences between French and Colombian universes. (Doctoral Dissertation). Université de Bordeaux I. Retrieved from http://www.theses.fr/2009BOR13986

Chicago Manual of Style (16th Edition):

Papin, Julien. “Bases moléculaires des défauts sécrétoires des cellules ß pancréatiques lors de la glucotoxicité : Modelling of a cultural dictionnary for students of French as a foreign language in Colombia : cultural differences between French and Colombian universes.” 2009. Doctoral Dissertation, Université de Bordeaux I. Accessed April 19, 2019. http://www.theses.fr/2009BOR13986.

MLA Handbook (7th Edition):

Papin, Julien. “Bases moléculaires des défauts sécrétoires des cellules ß pancréatiques lors de la glucotoxicité : Modelling of a cultural dictionnary for students of French as a foreign language in Colombia : cultural differences between French and Colombian universes.” 2009. Web. 19 Apr 2019.

Vancouver:

Papin J. Bases moléculaires des défauts sécrétoires des cellules ß pancréatiques lors de la glucotoxicité : Modelling of a cultural dictionnary for students of French as a foreign language in Colombia : cultural differences between French and Colombian universes. [Internet] [Doctoral dissertation]. Université de Bordeaux I; 2009. [cited 2019 Apr 19]. Available from: http://www.theses.fr/2009BOR13986.

Council of Science Editors:

Papin J. Bases moléculaires des défauts sécrétoires des cellules ß pancréatiques lors de la glucotoxicité : Modelling of a cultural dictionnary for students of French as a foreign language in Colombia : cultural differences between French and Colombian universes. [Doctoral Dissertation]. Université de Bordeaux I; 2009. Available from: http://www.theses.fr/2009BOR13986

5. Βενιεράτος, Παναγιώτης. Μελέτη του αποτελέσματος της γλυκόζης στην σηματοδότηση και την φυσιολογία παγκρεατικών β-κυττάρων.

Degree: 2009, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 Increasing evidence suggest that apart from insulin resistance, type 2 diabetes is also characterized by dysfunction of insulin secretion by the endocrine pancreas. Insulin secretion… (more)

Subjects/Keywords: Γλυκοτοξικότητα β-κυττάρων; Κύτταρα β-TC-6; Σηματοδότηση ινσουλίνης; Απόπτωση β-κυττάρων; Γλυκοτοξικότητα; Διαβήτης τύπου 2; Panvreatic b-cell glucotoxicity; b-TC-6 cells; Insulin signalling; Il-1β; SOCS-1; Beta-cell apoptosis; Glucotoxicity; Type 2 diabetes mellitus

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APA (6th Edition):

Βενιεράτος, . . (2009). Μελέτη του αποτελέσματος της γλυκόζης στην σηματοδότηση και την φυσιολογία παγκρεατικών β-κυττάρων. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/22807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Βενιεράτος, Παναγιώτης. “Μελέτη του αποτελέσματος της γλυκόζης στην σηματοδότηση και την φυσιολογία παγκρεατικών β-κυττάρων.” 2009. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed April 19, 2019. http://hdl.handle.net/10442/hedi/22807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Βενιεράτος, Παναγιώτης. “Μελέτη του αποτελέσματος της γλυκόζης στην σηματοδότηση και την φυσιολογία παγκρεατικών β-κυττάρων.” 2009. Web. 19 Apr 2019.

Vancouver:

Βενιεράτος . Μελέτη του αποτελέσματος της γλυκόζης στην σηματοδότηση και την φυσιολογία παγκρεατικών β-κυττάρων. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2009. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/10442/hedi/22807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Βενιεράτος . Μελέτη του αποτελέσματος της γλυκόζης στην σηματοδότηση και την φυσιολογία παγκρεατικών β-κυττάρων. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2009. Available from: http://hdl.handle.net/10442/hedi/22807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Grenoble

6. Cornali Lablanche, Sandrine. Implication du pore de transition de perméabilité mitochondriale dans l'apoptose de la cellule β pancréatique : Role of PTP in beta cell apoptosis.

Degree: Docteur es, Biologie cellulaire, 2012, Université de Grenoble

Implication du PTP dans la mort cellulaire β pancréatique L'hyperglycémie, l'hyperfructosémie et l'ischémie-reperfusion sont délétères pour la viabilité cellulaire β pancréatique, jouant un rôle majeur… (more)

Subjects/Keywords: Transition de perméabilité; Glucotoxicité; Ischémie-reperfusion; Fructose; INS-1; Îlots pancréatiques humains; Cyclosporine; Metformine; Stress oxydan; Permeability transition; Glucotoxicity; Ischemia-reperfusion injury; Fructose; Human islet; Cyclosporin; Metformin; Oxidative stress

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APA (6th Edition):

Cornali Lablanche, S. (2012). Implication du pore de transition de perméabilité mitochondriale dans l'apoptose de la cellule β pancréatique : Role of PTP in beta cell apoptosis. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2012GRENV009

Chicago Manual of Style (16th Edition):

Cornali Lablanche, Sandrine. “Implication du pore de transition de perméabilité mitochondriale dans l'apoptose de la cellule β pancréatique : Role of PTP in beta cell apoptosis.” 2012. Doctoral Dissertation, Université de Grenoble. Accessed April 19, 2019. http://www.theses.fr/2012GRENV009.

MLA Handbook (7th Edition):

Cornali Lablanche, Sandrine. “Implication du pore de transition de perméabilité mitochondriale dans l'apoptose de la cellule β pancréatique : Role of PTP in beta cell apoptosis.” 2012. Web. 19 Apr 2019.

Vancouver:

Cornali Lablanche S. Implication du pore de transition de perméabilité mitochondriale dans l'apoptose de la cellule β pancréatique : Role of PTP in beta cell apoptosis. [Internet] [Doctoral dissertation]. Université de Grenoble; 2012. [cited 2019 Apr 19]. Available from: http://www.theses.fr/2012GRENV009.

Council of Science Editors:

Cornali Lablanche S. Implication du pore de transition de perméabilité mitochondriale dans l'apoptose de la cellule β pancréatique : Role of PTP in beta cell apoptosis. [Doctoral Dissertation]. Université de Grenoble; 2012. Available from: http://www.theses.fr/2012GRENV009


University of Toronto

7. Lai, Elida Wing Shan. Diabetes and Endoplasmic Reticulum Stress in Pancreatic beta-cells: Effects on Insulin Biosynthesis and beta-cell Apoptosis.

Degree: 2008, University of Toronto

Chronic hyperlipidemia (lipotoxicity) and hyperglycemia (glucotoxicity) have recently been shown to induce Endoplasmic Reticulum (ER) stress, which may contribute to pancreatic beta-cell dysfunction in type… (more)

Subjects/Keywords: pancreatic beta-cells; type 2 diabetes; ER stress; apoptosis; pancreatic beta-cell dysfunction; free fatty acids; lipotoxicity; palmitate; high glucose; hyperglycemia; glucotoxicity; insulin biosynthesis; unfolded protein response; chaperone; GRP78; PDI; INS-1; MIN6; human islets; stable cell line; 0379

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APA (6th Edition):

Lai, E. W. S. (2008). Diabetes and Endoplasmic Reticulum Stress in Pancreatic beta-cells: Effects on Insulin Biosynthesis and beta-cell Apoptosis. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/11149

Chicago Manual of Style (16th Edition):

Lai, Elida Wing Shan. “Diabetes and Endoplasmic Reticulum Stress in Pancreatic beta-cells: Effects on Insulin Biosynthesis and beta-cell Apoptosis.” 2008. Masters Thesis, University of Toronto. Accessed April 19, 2019. http://hdl.handle.net/1807/11149.

MLA Handbook (7th Edition):

Lai, Elida Wing Shan. “Diabetes and Endoplasmic Reticulum Stress in Pancreatic beta-cells: Effects on Insulin Biosynthesis and beta-cell Apoptosis.” 2008. Web. 19 Apr 2019.

Vancouver:

Lai EWS. Diabetes and Endoplasmic Reticulum Stress in Pancreatic beta-cells: Effects on Insulin Biosynthesis and beta-cell Apoptosis. [Internet] [Masters thesis]. University of Toronto; 2008. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1807/11149.

Council of Science Editors:

Lai EWS. Diabetes and Endoplasmic Reticulum Stress in Pancreatic beta-cells: Effects on Insulin Biosynthesis and beta-cell Apoptosis. [Masters Thesis]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/11149

8. ZHANG JIPING. Investigation of the roles of two Rac1 effectors phosphatidylinositol-5 kinase 1a and p21-activated kinase 1 in insulin-secreting cells.

Degree: 2009, National University of Singapore

Subjects/Keywords: phosphatidylinositol-4-phosphate 5-kinase; p21-activated kinase; insulin secretion; glucotoxicity; pancreatic beta cells; Rac

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APA (6th Edition):

JIPING, Z. (2009). Investigation of the roles of two Rac1 effectors phosphatidylinositol-5 kinase 1a and p21-activated kinase 1 in insulin-secreting cells. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/28341 ; http://scholarbank.nus.edu.sg/bitstream/10635%2F28341/1/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F28341/2/bitstream

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

JIPING, ZHANG. “Investigation of the roles of two Rac1 effectors phosphatidylinositol-5 kinase 1a and p21-activated kinase 1 in insulin-secreting cells.” 2009. Thesis, National University of Singapore. Accessed April 19, 2019. http://scholarbank.nus.edu.sg/handle/10635/28341 ; http://scholarbank.nus.edu.sg/bitstream/10635%2F28341/1/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F28341/2/bitstream.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

JIPING, ZHANG. “Investigation of the roles of two Rac1 effectors phosphatidylinositol-5 kinase 1a and p21-activated kinase 1 in insulin-secreting cells.” 2009. Web. 19 Apr 2019.

Vancouver:

JIPING Z. Investigation of the roles of two Rac1 effectors phosphatidylinositol-5 kinase 1a and p21-activated kinase 1 in insulin-secreting cells. [Internet] [Thesis]. National University of Singapore; 2009. [cited 2019 Apr 19]. Available from: http://scholarbank.nus.edu.sg/handle/10635/28341 ; http://scholarbank.nus.edu.sg/bitstream/10635%2F28341/1/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F28341/2/bitstream.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

JIPING Z. Investigation of the roles of two Rac1 effectors phosphatidylinositol-5 kinase 1a and p21-activated kinase 1 in insulin-secreting cells. [Thesis]. National University of Singapore; 2009. Available from: http://scholarbank.nus.edu.sg/handle/10635/28341 ; http://scholarbank.nus.edu.sg/bitstream/10635%2F28341/1/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F28341/2/bitstream

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. RONNE YEO WEE YEH. Mouse Embryonic Stem cell-derived Insulin-producing Cell Line: A Surrogate for the Investigation of Beta Cell Biology and Pathogenesis.

Degree: 2013, National University of Singapore

Subjects/Keywords: Insulin-producing cells; pentose phosphate pathway; clathrin-mediated endocytosis; glucotoxicity; actin dynamics; extracellular signal-regulated kinase

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APA (6th Edition):

YEH, R. Y. W. (2013). Mouse Embryonic Stem cell-derived Insulin-producing Cell Line: A Surrogate for the Investigation of Beta Cell Biology and Pathogenesis. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/48622 ; http://scholarbank.nus.edu.sg/bitstream/10635%2F48622/1/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F48622/2/bitstream

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

YEH, RONNE YEO WEE. “Mouse Embryonic Stem cell-derived Insulin-producing Cell Line: A Surrogate for the Investigation of Beta Cell Biology and Pathogenesis.” 2013. Thesis, National University of Singapore. Accessed April 19, 2019. http://scholarbank.nus.edu.sg/handle/10635/48622 ; http://scholarbank.nus.edu.sg/bitstream/10635%2F48622/1/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F48622/2/bitstream.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

YEH, RONNE YEO WEE. “Mouse Embryonic Stem cell-derived Insulin-producing Cell Line: A Surrogate for the Investigation of Beta Cell Biology and Pathogenesis.” 2013. Web. 19 Apr 2019.

Vancouver:

YEH RYW. Mouse Embryonic Stem cell-derived Insulin-producing Cell Line: A Surrogate for the Investigation of Beta Cell Biology and Pathogenesis. [Internet] [Thesis]. National University of Singapore; 2013. [cited 2019 Apr 19]. Available from: http://scholarbank.nus.edu.sg/handle/10635/48622 ; http://scholarbank.nus.edu.sg/bitstream/10635%2F48622/1/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F48622/2/bitstream.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

YEH RYW. Mouse Embryonic Stem cell-derived Insulin-producing Cell Line: A Surrogate for the Investigation of Beta Cell Biology and Pathogenesis. [Thesis]. National University of Singapore; 2013. Available from: http://scholarbank.nus.edu.sg/handle/10635/48622 ; http://scholarbank.nus.edu.sg/bitstream/10635%2F48622/1/bitstream ; http://scholarbank.nus.edu.sg/bitstream/10635%2F48622/2/bitstream

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Cassel, Roméo. Glucolipotoxicité dans les cellules bêta pancréatiques : Glucotoxicity in pancreatic beta cells.

Degree: Docteur es, Métabolisme, Nutrition, Diabète, 2014, Université Claude Bernard – Lyon I

Le diabète de type 2 est une pathologie chronique complexe associant une altération de sécrétion de l'insuline par le pancréas et une résistance à l'insuline… (more)

Subjects/Keywords: Diabète de type 2; Cellules bêta pancréatiques; Îlots de Langerhans humains; Cellules MIN6B1; Glucotoxicité; Lipotoxicité; Translocon; Système rénine-angiotensine; Type 2 diabetes; Pancreatic beta cells; Human pancreatic islets; MIN6B1 cells; Glucotoxicity; Lipotoxicity; Translocon; Renin-angiotensin system; 616

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APA (6th Edition):

Cassel, R. (2014). Glucolipotoxicité dans les cellules bêta pancréatiques : Glucotoxicity in pancreatic beta cells. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2014LYO10257

Chicago Manual of Style (16th Edition):

Cassel, Roméo. “Glucolipotoxicité dans les cellules bêta pancréatiques : Glucotoxicity in pancreatic beta cells.” 2014. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed April 19, 2019. http://www.theses.fr/2014LYO10257.

MLA Handbook (7th Edition):

Cassel, Roméo. “Glucolipotoxicité dans les cellules bêta pancréatiques : Glucotoxicity in pancreatic beta cells.” 2014. Web. 19 Apr 2019.

Vancouver:

Cassel R. Glucolipotoxicité dans les cellules bêta pancréatiques : Glucotoxicity in pancreatic beta cells. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2014. [cited 2019 Apr 19]. Available from: http://www.theses.fr/2014LYO10257.

Council of Science Editors:

Cassel R. Glucolipotoxicité dans les cellules bêta pancréatiques : Glucotoxicity in pancreatic beta cells. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2014. Available from: http://www.theses.fr/2014LYO10257


Université de Montréal

11. Bédard, Karine. Étude des mécanismes associés aux effets bénéfiques de la restriction calorique sur la fonction somatotrope du rat vieillissant .

Degree: 2012, Université de Montréal

Subjects/Keywords: GHRH-R; Somatotrope; Vieillissement; Restriction calorique modérée de longue durée; Glucotoxicité; Lipotoxicité; Stress oxydant; Somatotroph; Aging; Long-term moderate caloric restriction; Glucotoxicity; Lipotoxicity; Oxidative stress; Growth hormone-releasing hormone receptor

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APA (6th Edition):

Bédard, K. (2012). Étude des mécanismes associés aux effets bénéfiques de la restriction calorique sur la fonction somatotrope du rat vieillissant . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/6265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bédard, Karine. “Étude des mécanismes associés aux effets bénéfiques de la restriction calorique sur la fonction somatotrope du rat vieillissant .” 2012. Thesis, Université de Montréal. Accessed April 19, 2019. http://hdl.handle.net/1866/6265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bédard, Karine. “Étude des mécanismes associés aux effets bénéfiques de la restriction calorique sur la fonction somatotrope du rat vieillissant .” 2012. Web. 19 Apr 2019.

Vancouver:

Bédard K. Étude des mécanismes associés aux effets bénéfiques de la restriction calorique sur la fonction somatotrope du rat vieillissant . [Internet] [Thesis]. Université de Montréal; 2012. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1866/6265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bédard K. Étude des mécanismes associés aux effets bénéfiques de la restriction calorique sur la fonction somatotrope du rat vieillissant . [Thesis]. Université de Montréal; 2012. Available from: http://hdl.handle.net/1866/6265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.