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You searched for subject:(Glioma). Showing records 1 – 30 of 473 total matches.

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Universidade do Rio Grande do Sul

1. Lopez, Patrícia Luciana da Costa. Influência das proteínas XIAP e Survivina na resistência de Gliomas.

Degree: 2012, Universidade do Rio Grande do Sul

Glioblastomas (GBM) são tumores de difícil tratamento pela alta resistência a quimioterápicos. Entre os GBMs a presença de células tronco tumorais (CTT) foi associado a… (more)

Subjects/Keywords: Proteínas; Glioma

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APA (6th Edition):

Lopez, P. L. d. C. (2012). Influência das proteínas XIAP e Survivina na resistência de Gliomas. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/142701

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lopez, Patrícia Luciana da Costa. “Influência das proteínas XIAP e Survivina na resistência de Gliomas.” 2012. Thesis, Universidade do Rio Grande do Sul. Accessed October 20, 2019. http://hdl.handle.net/10183/142701.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lopez, Patrícia Luciana da Costa. “Influência das proteínas XIAP e Survivina na resistência de Gliomas.” 2012. Web. 20 Oct 2019.

Vancouver:

Lopez PLdC. Influência das proteínas XIAP e Survivina na resistência de Gliomas. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2012. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10183/142701.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lopez PLdC. Influência das proteínas XIAP e Survivina na resistência de Gliomas. [Thesis]. Universidade do Rio Grande do Sul; 2012. Available from: http://hdl.handle.net/10183/142701

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

2. Flores, Débora Gazzana. Receptor do peptídeo liberador de gastrina (GRPR) em gliomas : expressão, influência na proliferação celular e mecanismos de sinalização.

Degree: 2009, Universidade do Rio Grande do Sul

 Gliomas correspondem à grande maioria de tumores do sistema nervoso central (SNC), sendo que os glioblastomas multiformes são os mais comuns e mais malignos. O… (more)

Subjects/Keywords: Glioma; Gastrinas

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APA (6th Edition):

Flores, D. G. (2009). Receptor do peptídeo liberador de gastrina (GRPR) em gliomas : expressão, influência na proliferação celular e mecanismos de sinalização. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/17341

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Flores, Débora Gazzana. “Receptor do peptídeo liberador de gastrina (GRPR) em gliomas : expressão, influência na proliferação celular e mecanismos de sinalização.” 2009. Thesis, Universidade do Rio Grande do Sul. Accessed October 20, 2019. http://hdl.handle.net/10183/17341.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Flores, Débora Gazzana. “Receptor do peptídeo liberador de gastrina (GRPR) em gliomas : expressão, influência na proliferação celular e mecanismos de sinalização.” 2009. Web. 20 Oct 2019.

Vancouver:

Flores DG. Receptor do peptídeo liberador de gastrina (GRPR) em gliomas : expressão, influência na proliferação celular e mecanismos de sinalização. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2009. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10183/17341.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Flores DG. Receptor do peptídeo liberador de gastrina (GRPR) em gliomas : expressão, influência na proliferação celular e mecanismos de sinalização. [Thesis]. Universidade do Rio Grande do Sul; 2009. Available from: http://hdl.handle.net/10183/17341

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

3. Pyrko, Peter. Targeting ER stress for malignant glioma therapy.

Degree: PhD, Pathobiology, 2007, University of Southern California

 Poor chemosensitivity and the development of chemoresistance remain major obstacles to successful chemotherapy of malignant gliomas. Recently, a new goal has been set: changing malignant… (more)

Subjects/Keywords: glioma

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APA (6th Edition):

Pyrko, P. (2007). Targeting ER stress for malignant glioma therapy. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/324413/rec/6317

Chicago Manual of Style (16th Edition):

Pyrko, Peter. “Targeting ER stress for malignant glioma therapy.” 2007. Doctoral Dissertation, University of Southern California. Accessed October 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/324413/rec/6317.

MLA Handbook (7th Edition):

Pyrko, Peter. “Targeting ER stress for malignant glioma therapy.” 2007. Web. 20 Oct 2019.

Vancouver:

Pyrko P. Targeting ER stress for malignant glioma therapy. [Internet] [Doctoral dissertation]. University of Southern California; 2007. [cited 2019 Oct 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/324413/rec/6317.

Council of Science Editors:

Pyrko P. Targeting ER stress for malignant glioma therapy. [Doctoral Dissertation]. University of Southern California; 2007. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/324413/rec/6317


Kaunas University of Medicine

4. Kaselytė, Agnė. Assay of epigenetics changes in SFRP2 gene in grade II and grade III gliomas able to infleunce drug effects.

Degree: Master, Pharmacy, 2010, Kaunas University of Medicine

Two different mechanisms participate in the loss of SFRPs expression in cancer cells: allelic loss and epigenetic silencing [1]. Methylation status in grade II and… (more)

Subjects/Keywords: Glioma; Methylation; SFRP2 gene; Glioma; SFRP2; Methylation

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APA (6th Edition):

Kaselytė, Agnė. (2010). Assay of epigenetics changes in SFRP2 gene in grade II and grade III gliomas able to infleunce drug effects. (Masters Thesis). Kaunas University of Medicine. Retrieved from http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2010~D_20100621_092807-14356 ;

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

Kaselytė, Agnė. “Assay of epigenetics changes in SFRP2 gene in grade II and grade III gliomas able to infleunce drug effects.” 2010. Masters Thesis, Kaunas University of Medicine. Accessed October 20, 2019. http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2010~D_20100621_092807-14356 ;.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

Kaselytė, Agnė. “Assay of epigenetics changes in SFRP2 gene in grade II and grade III gliomas able to infleunce drug effects.” 2010. Web. 20 Oct 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

Kaselytė, Agnė. Assay of epigenetics changes in SFRP2 gene in grade II and grade III gliomas able to infleunce drug effects. [Internet] [Masters thesis]. Kaunas University of Medicine; 2010. [cited 2019 Oct 20]. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2010~D_20100621_092807-14356 ;.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

Kaselytė, Agnė. Assay of epigenetics changes in SFRP2 gene in grade II and grade III gliomas able to infleunce drug effects. [Masters Thesis]. Kaunas University of Medicine; 2010. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2010~D_20100621_092807-14356 ;

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


Universidade do Rio Grande do Sul

5. Ledur, Pítia Flores. Prevalência de OPCS (células precursoras de oligodendrócitos) em gliomas é determinante para o estabelecimento de condições autênticas de cultivo e para a identificação de alvos terapêuticos.

Degree: 2015, Universidade do Rio Grande do Sul

Glioblastomas Multiformes (GBM) são tumores do Sistema Nervoso Central com altas taxas de invasibilidade e grande resistência a quimio e radioterapias, cuja origem foi inicialmente… (more)

Subjects/Keywords: Glioma; Cérebro; Oligodendroglioma

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APA (6th Edition):

Ledur, P. F. (2015). Prevalência de OPCS (células precursoras de oligodendrócitos) em gliomas é determinante para o estabelecimento de condições autênticas de cultivo e para a identificação de alvos terapêuticos. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/141886

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ledur, Pítia Flores. “Prevalência de OPCS (células precursoras de oligodendrócitos) em gliomas é determinante para o estabelecimento de condições autênticas de cultivo e para a identificação de alvos terapêuticos.” 2015. Thesis, Universidade do Rio Grande do Sul. Accessed October 20, 2019. http://hdl.handle.net/10183/141886.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ledur, Pítia Flores. “Prevalência de OPCS (células precursoras de oligodendrócitos) em gliomas é determinante para o estabelecimento de condições autênticas de cultivo e para a identificação de alvos terapêuticos.” 2015. Web. 20 Oct 2019.

Vancouver:

Ledur PF. Prevalência de OPCS (células precursoras de oligodendrócitos) em gliomas é determinante para o estabelecimento de condições autênticas de cultivo e para a identificação de alvos terapêuticos. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2015. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10183/141886.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ledur PF. Prevalência de OPCS (células precursoras de oligodendrócitos) em gliomas é determinante para o estabelecimento de condições autênticas de cultivo e para a identificação de alvos terapêuticos. [Thesis]. Universidade do Rio Grande do Sul; 2015. Available from: http://hdl.handle.net/10183/141886

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

6. Tamajusuku, Alessandra Sayuri Kikuchi. Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261.

Degree: 2010, Universidade do Rio Grande do Sul

O nucleotídeo de purina ATP, no meio extracelular, participa de diversos processos fisiológicos e patológicos como vasodilatação/constrição, proliferação, diferenciação, modulação sináptica, dor, inflamação e morte… (more)

Subjects/Keywords: Glioma; Glioblastoma; Camundongos

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APA (6th Edition):

Tamajusuku, A. S. K. (2010). Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/30210

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tamajusuku, Alessandra Sayuri Kikuchi. “Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261.” 2010. Thesis, Universidade do Rio Grande do Sul. Accessed October 20, 2019. http://hdl.handle.net/10183/30210.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tamajusuku, Alessandra Sayuri Kikuchi. “Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261.” 2010. Web. 20 Oct 2019.

Vancouver:

Tamajusuku ASK. Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2010. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10183/30210.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tamajusuku ASK. Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261. [Thesis]. Universidade do Rio Grande do Sul; 2010. Available from: http://hdl.handle.net/10183/30210

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

7. Kipper, Franciele Cristina. Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes.

Degree: 2017, Universidade do Rio Grande do Sul

 Gliomas são tumores do sistema nervoso central caracterizados por alta invasibilidade e mortalidade. Inúmeros esforços foram feitos nas últimas décadas para melhorar a sobrevida dos… (more)

Subjects/Keywords: Glioblastoma; Glioma; Temozolomida

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APA (6th Edition):

Kipper, F. C. (2017). Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/170182

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kipper, Franciele Cristina. “Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes.” 2017. Thesis, Universidade do Rio Grande do Sul. Accessed October 20, 2019. http://hdl.handle.net/10183/170182.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kipper, Franciele Cristina. “Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes.” 2017. Web. 20 Oct 2019.

Vancouver:

Kipper FC. Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2017. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10183/170182.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kipper FC. Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes. [Thesis]. Universidade do Rio Grande do Sul; 2017. Available from: http://hdl.handle.net/10183/170182

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

8. Mistry, Matthew. BRAF Mutation and CDKN2A Deletion Define a Clinically Distinct Subgroup of Childhood Secondary High-Grade Glioma.

Degree: 2014, University of Toronto

This thesis sought to uncover genetic events leading to transformation of pediatric low-grade glioma (PLGG) to secondary high-grade glioma (sHGG) and to define transformation risk… (more)

Subjects/Keywords: Glioma; Pediatric; 0306

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APA (6th Edition):

Mistry, M. (2014). BRAF Mutation and CDKN2A Deletion Define a Clinically Distinct Subgroup of Childhood Secondary High-Grade Glioma. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/68561

Chicago Manual of Style (16th Edition):

Mistry, Matthew. “BRAF Mutation and CDKN2A Deletion Define a Clinically Distinct Subgroup of Childhood Secondary High-Grade Glioma.” 2014. Masters Thesis, University of Toronto. Accessed October 20, 2019. http://hdl.handle.net/1807/68561.

MLA Handbook (7th Edition):

Mistry, Matthew. “BRAF Mutation and CDKN2A Deletion Define a Clinically Distinct Subgroup of Childhood Secondary High-Grade Glioma.” 2014. Web. 20 Oct 2019.

Vancouver:

Mistry M. BRAF Mutation and CDKN2A Deletion Define a Clinically Distinct Subgroup of Childhood Secondary High-Grade Glioma. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/1807/68561.

Council of Science Editors:

Mistry M. BRAF Mutation and CDKN2A Deletion Define a Clinically Distinct Subgroup of Childhood Secondary High-Grade Glioma. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68561


Victoria University of Wellington

9. Hunn, Martin Kent. Improving immunotherapy for high grade glioma.

Degree: 2015, Victoria University of Wellington

 Glioblastoma multiforme (GBM) is a malignant primary brain tumour that is almost always fatal. Conventional treatment modalities are limited by toxicity. T cell-based immunotherapy is… (more)

Subjects/Keywords: Glioma; Immunotherapy; Vaccine

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APA (6th Edition):

Hunn, M. K. (2015). Improving immunotherapy for high grade glioma. (Doctoral Dissertation). Victoria University of Wellington. Retrieved from http://hdl.handle.net/10063/4869

Chicago Manual of Style (16th Edition):

Hunn, Martin Kent. “Improving immunotherapy for high grade glioma.” 2015. Doctoral Dissertation, Victoria University of Wellington. Accessed October 20, 2019. http://hdl.handle.net/10063/4869.

MLA Handbook (7th Edition):

Hunn, Martin Kent. “Improving immunotherapy for high grade glioma.” 2015. Web. 20 Oct 2019.

Vancouver:

Hunn MK. Improving immunotherapy for high grade glioma. [Internet] [Doctoral dissertation]. Victoria University of Wellington; 2015. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10063/4869.

Council of Science Editors:

Hunn MK. Improving immunotherapy for high grade glioma. [Doctoral Dissertation]. Victoria University of Wellington; 2015. Available from: http://hdl.handle.net/10063/4869


University of Minnesota

10. Becker, Chani. Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma.

Degree: PhD, Neuroscience, 2016, University of Minnesota

 Glioblastoma multiforme (GBM) is a lethal cancer. Without treatment, patients diagnosed with this disease survive nine months. With the best therapeutics science has to offer,… (more)

Subjects/Keywords: BBB; Glioblastoma; Glioma

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Becker, C. (2016). Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/182319

Chicago Manual of Style (16th Edition):

Becker, Chani. “Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma.” 2016. Doctoral Dissertation, University of Minnesota. Accessed October 20, 2019. http://hdl.handle.net/11299/182319.

MLA Handbook (7th Edition):

Becker, Chani. “Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma.” 2016. Web. 20 Oct 2019.

Vancouver:

Becker C. Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma. [Internet] [Doctoral dissertation]. University of Minnesota; 2016. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/11299/182319.

Council of Science Editors:

Becker C. Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma. [Doctoral Dissertation]. University of Minnesota; 2016. Available from: http://hdl.handle.net/11299/182319


University of Manchester

11. Coope, David John. Use of [11C]-methionine PET and diffusion-/perfusion-weighted MR imaging in gliomas.

Degree: PhD, 2010, University of Manchester

 Introduction: Low-grade gliomas are a sub-group of primary brain tumours that typically affect young adults and which present specific challenges to conventional diagnostic imaging. They… (more)

Subjects/Keywords: 615.84; Methionine PET Glioma

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APA (6th Edition):

Coope, D. J. (2010). Use of [11C]-methionine PET and diffusion-/perfusion-weighted MR imaging in gliomas. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:207525 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.521589

Chicago Manual of Style (16th Edition):

Coope, David John. “Use of [11C]-methionine PET and diffusion-/perfusion-weighted MR imaging in gliomas.” 2010. Doctoral Dissertation, University of Manchester. Accessed October 20, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:207525 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.521589.

MLA Handbook (7th Edition):

Coope, David John. “Use of [11C]-methionine PET and diffusion-/perfusion-weighted MR imaging in gliomas.” 2010. Web. 20 Oct 2019.

Vancouver:

Coope DJ. Use of [11C]-methionine PET and diffusion-/perfusion-weighted MR imaging in gliomas. [Internet] [Doctoral dissertation]. University of Manchester; 2010. [cited 2019 Oct 20]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:207525 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.521589.

Council of Science Editors:

Coope DJ. Use of [11C]-methionine PET and diffusion-/perfusion-weighted MR imaging in gliomas. [Doctoral Dissertation]. University of Manchester; 2010. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:207525 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.521589


Universidade do Rio Grande do Sul

12. Zamin, Lauren Lúcia. Resveratrol e quercetina : avaliação da atividade antitumoral e dos mecanismos de ação em linhagens de gliomas in vitro e em um modelo de implante de gliomas in vivo.

Degree: 2010, Universidade do Rio Grande do Sul

Dentre os tumores cerebrais primários, o glioblastoma (GBM) é o mais comum, apresentando alta taxa de mortalidade e morbidade. O tratamento convencional desses tumores tem… (more)

Subjects/Keywords: Resveratrol; Quercetina; Atividade antitumoral; Glioma

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APA (6th Edition):

Zamin, L. L. (2010). Resveratrol e quercetina : avaliação da atividade antitumoral e dos mecanismos de ação em linhagens de gliomas in vitro e em um modelo de implante de gliomas in vivo. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/25145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zamin, Lauren Lúcia. “Resveratrol e quercetina : avaliação da atividade antitumoral e dos mecanismos de ação em linhagens de gliomas in vitro e em um modelo de implante de gliomas in vivo.” 2010. Thesis, Universidade do Rio Grande do Sul. Accessed October 20, 2019. http://hdl.handle.net/10183/25145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zamin, Lauren Lúcia. “Resveratrol e quercetina : avaliação da atividade antitumoral e dos mecanismos de ação em linhagens de gliomas in vitro e em um modelo de implante de gliomas in vivo.” 2010. Web. 20 Oct 2019.

Vancouver:

Zamin LL. Resveratrol e quercetina : avaliação da atividade antitumoral e dos mecanismos de ação em linhagens de gliomas in vitro e em um modelo de implante de gliomas in vivo. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2010. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10183/25145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zamin LL. Resveratrol e quercetina : avaliação da atividade antitumoral e dos mecanismos de ação em linhagens de gliomas in vitro e em um modelo de implante de gliomas in vivo. [Thesis]. Universidade do Rio Grande do Sul; 2010. Available from: http://hdl.handle.net/10183/25145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

13. Braganhol, Elizandra. Sistema purinérgico e a progressão dos gliomas : avaliação de parâmetros proliferativos e inflamatórios.

Degree: 2010, Universidade do Rio Grande do Sul

Os nucleotídeos extracelulares modulam uma variedade de ações biológicas via ativação de receptores purinérgicos. Esses efeitos são controlados pela ação de ectonucleotidases, tais como as… (more)

Subjects/Keywords: Glioma; Receptores purinérgicos; Ectonucleotidases

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Braganhol, E. (2010). Sistema purinérgico e a progressão dos gliomas : avaliação de parâmetros proliferativos e inflamatórios. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/26487

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Braganhol, Elizandra. “Sistema purinérgico e a progressão dos gliomas : avaliação de parâmetros proliferativos e inflamatórios.” 2010. Thesis, Universidade do Rio Grande do Sul. Accessed October 20, 2019. http://hdl.handle.net/10183/26487.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Braganhol, Elizandra. “Sistema purinérgico e a progressão dos gliomas : avaliação de parâmetros proliferativos e inflamatórios.” 2010. Web. 20 Oct 2019.

Vancouver:

Braganhol E. Sistema purinérgico e a progressão dos gliomas : avaliação de parâmetros proliferativos e inflamatórios. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2010. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10183/26487.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Braganhol E. Sistema purinérgico e a progressão dos gliomas : avaliação de parâmetros proliferativos e inflamatórios. [Thesis]. Universidade do Rio Grande do Sul; 2010. Available from: http://hdl.handle.net/10183/26487

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade Estadual de Campinas

14. Ikemori, Rafael Yamashita. Analise de possiveis mecanismos e consequencias funcionais da expressão de galectina-3 em celulas de glioma expostas a condições hipoxicas .

Degree: 2009, Universidade Estadual de Campinas

 Resumo: Gliomas são tumores primários do sistema nervoso central e o glioblastoma multiforme é sua forma clínica mais comum e de pior prognóstico. Na tentativa… (more)

Subjects/Keywords: Glioma; NG97ht; Galectina 3; Pseudopaliçada

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APA (6th Edition):

Ikemori, R. Y. (2009). Analise de possiveis mecanismos e consequencias funcionais da expressão de galectina-3 em celulas de glioma expostas a condições hipoxicas . (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/316961

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ikemori, Rafael Yamashita. “Analise de possiveis mecanismos e consequencias funcionais da expressão de galectina-3 em celulas de glioma expostas a condições hipoxicas .” 2009. Thesis, Universidade Estadual de Campinas. Accessed October 20, 2019. http://repositorio.unicamp.br/jspui/handle/REPOSIP/316961.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ikemori, Rafael Yamashita. “Analise de possiveis mecanismos e consequencias funcionais da expressão de galectina-3 em celulas de glioma expostas a condições hipoxicas .” 2009. Web. 20 Oct 2019.

Vancouver:

Ikemori RY. Analise de possiveis mecanismos e consequencias funcionais da expressão de galectina-3 em celulas de glioma expostas a condições hipoxicas . [Internet] [Thesis]. Universidade Estadual de Campinas; 2009. [cited 2019 Oct 20]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/316961.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ikemori RY. Analise de possiveis mecanismos e consequencias funcionais da expressão de galectina-3 em celulas de glioma expostas a condições hipoxicas . [Thesis]. Universidade Estadual de Campinas; 2009. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/316961

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

15. Mita, Raja. Role of brain FABP and its ligands in malignant glioma cell migration.

Degree: PhD, Department of Oncology, 2010, University of Alberta

 Patients diagnosed with malignant glioma tumours have median survivals of 1.6 yrs and 5 months, respectively, highlighting the deadly nature of this disease. Despite aggressive… (more)

Subjects/Keywords: fatty acids; glioma; fabp7

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mita, R. (2010). Role of brain FABP and its ligands in malignant glioma cell migration. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/8w32r7236

Chicago Manual of Style (16th Edition):

Mita, Raja. “Role of brain FABP and its ligands in malignant glioma cell migration.” 2010. Doctoral Dissertation, University of Alberta. Accessed October 20, 2019. https://era.library.ualberta.ca/files/8w32r7236.

MLA Handbook (7th Edition):

Mita, Raja. “Role of brain FABP and its ligands in malignant glioma cell migration.” 2010. Web. 20 Oct 2019.

Vancouver:

Mita R. Role of brain FABP and its ligands in malignant glioma cell migration. [Internet] [Doctoral dissertation]. University of Alberta; 2010. [cited 2019 Oct 20]. Available from: https://era.library.ualberta.ca/files/8w32r7236.

Council of Science Editors:

Mita R. Role of brain FABP and its ligands in malignant glioma cell migration. [Doctoral Dissertation]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/8w32r7236


University of Alberta

16. Swan, Amanda C. An Anisotropic Diffusion Model for Brain Tumour Spread.

Degree: PhD, Department of Mathematical and Statistical Sciences, 2016, University of Alberta

 Gliomas, tumours arising from the glial cells of the nervous system, are some of the most difficult tumours to treat. In particular, glioblastoma are a… (more)

Subjects/Keywords: Anisotropic Diffusion; Glioma; Mathematical Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Swan, A. C. (2016). An Anisotropic Diffusion Model for Brain Tumour Spread. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/cpr76f371p

Chicago Manual of Style (16th Edition):

Swan, Amanda C. “An Anisotropic Diffusion Model for Brain Tumour Spread.” 2016. Doctoral Dissertation, University of Alberta. Accessed October 20, 2019. https://era.library.ualberta.ca/files/cpr76f371p.

MLA Handbook (7th Edition):

Swan, Amanda C. “An Anisotropic Diffusion Model for Brain Tumour Spread.” 2016. Web. 20 Oct 2019.

Vancouver:

Swan AC. An Anisotropic Diffusion Model for Brain Tumour Spread. [Internet] [Doctoral dissertation]. University of Alberta; 2016. [cited 2019 Oct 20]. Available from: https://era.library.ualberta.ca/files/cpr76f371p.

Council of Science Editors:

Swan AC. An Anisotropic Diffusion Model for Brain Tumour Spread. [Doctoral Dissertation]. University of Alberta; 2016. Available from: https://era.library.ualberta.ca/files/cpr76f371p


Texas A&M University

17. Cherry, Evan Michael. Noncanonical NF-?B Signaling in Glioma is Activated by TWEAK and Promotes Invasion.

Degree: 2017, Texas A&M University

 High-grade gliomas are an invasive and deadly brain cancer. We have shown that noncanonical NF-?B/RelB signaling can drive invasion in the aggressive mesenchymal subtype of… (more)

Subjects/Keywords: Glioma; TWEAK; Noncanonical NF-?B

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APA (6th Edition):

Cherry, E. M. (2017). Noncanonical NF-?B Signaling in Glioma is Activated by TWEAK and Promotes Invasion. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/161267

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cherry, Evan Michael. “Noncanonical NF-?B Signaling in Glioma is Activated by TWEAK and Promotes Invasion.” 2017. Thesis, Texas A&M University. Accessed October 20, 2019. http://hdl.handle.net/1969.1/161267.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cherry, Evan Michael. “Noncanonical NF-?B Signaling in Glioma is Activated by TWEAK and Promotes Invasion.” 2017. Web. 20 Oct 2019.

Vancouver:

Cherry EM. Noncanonical NF-?B Signaling in Glioma is Activated by TWEAK and Promotes Invasion. [Internet] [Thesis]. Texas A&M University; 2017. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/1969.1/161267.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cherry EM. Noncanonical NF-?B Signaling in Glioma is Activated by TWEAK and Promotes Invasion. [Thesis]. Texas A&M University; 2017. Available from: http://hdl.handle.net/1969.1/161267

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

18. Villodre, Emilly Schlee. Estratégias farmacológicas para a redução da população de células tronco tumorais em glioblastoma.

Degree: 2012, Universidade do Rio Grande do Sul

Glioblastomas são os tumores mais agressivos do Sistema Nervoso Central (SNC). Caracterizam-se por sua alta invasibilidade, proliferação, altos índices de recorrência e morte, assim como… (more)

Subjects/Keywords: Glioma; Temozolamida; Resveratrol; Doxorrubicina

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Villodre, E. S. (2012). Estratégias farmacológicas para a redução da população de células tronco tumorais em glioblastoma. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/72311

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Villodre, Emilly Schlee. “Estratégias farmacológicas para a redução da população de células tronco tumorais em glioblastoma.” 2012. Thesis, Universidade do Rio Grande do Sul. Accessed October 20, 2019. http://hdl.handle.net/10183/72311.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Villodre, Emilly Schlee. “Estratégias farmacológicas para a redução da população de células tronco tumorais em glioblastoma.” 2012. Web. 20 Oct 2019.

Vancouver:

Villodre ES. Estratégias farmacológicas para a redução da população de células tronco tumorais em glioblastoma. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2012. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10183/72311.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Villodre ES. Estratégias farmacológicas para a redução da população de células tronco tumorais em glioblastoma. [Thesis]. Universidade do Rio Grande do Sul; 2012. Available from: http://hdl.handle.net/10183/72311

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

19. Vargas, José Eduardo. Estudos in vitro e in silico dos mecanismos moleculares da senescência celular em glioblastomas.

Degree: 2013, Universidade do Rio Grande do Sul

 Os gliomas representam a maioria dos tumores do sistema nervoso central, sendo o glioblastoma o mais maligno entre eles. O tratamento destes tumores ainda é… (more)

Subjects/Keywords: Glioma; Temozolamida; Resveratrol; Senescência

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APA (6th Edition):

Vargas, J. E. (2013). Estudos in vitro e in silico dos mecanismos moleculares da senescência celular em glioblastomas. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/78129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vargas, José Eduardo. “Estudos in vitro e in silico dos mecanismos moleculares da senescência celular em glioblastomas.” 2013. Thesis, Universidade do Rio Grande do Sul. Accessed October 20, 2019. http://hdl.handle.net/10183/78129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vargas, José Eduardo. “Estudos in vitro e in silico dos mecanismos moleculares da senescência celular em glioblastomas.” 2013. Web. 20 Oct 2019.

Vancouver:

Vargas JE. Estudos in vitro e in silico dos mecanismos moleculares da senescência celular em glioblastomas. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2013. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10183/78129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vargas JE. Estudos in vitro e in silico dos mecanismos moleculares da senescência celular em glioblastomas. [Thesis]. Universidade do Rio Grande do Sul; 2013. Available from: http://hdl.handle.net/10183/78129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

20. Junior, Mário Luiz Conte da Frota. A esponja marinha Polymastia janeirensis como fonte de novos fármacos contra o câncer.

Degree: 2008, Universidade do Rio Grande do Sul

Nos últimos anos, o ambiente marinho tem sido pesquisado para uma variedade de compostos com diferentes atividades biológicas. Entre todos os organismos marinhos, as esponjas… (more)

Subjects/Keywords: Polymastia janeirensis; Farmacologia; Glioma; Câncer

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APA (6th Edition):

Junior, M. L. C. d. F. (2008). A esponja marinha Polymastia janeirensis como fonte de novos fármacos contra o câncer. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/13832

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Junior, Mário Luiz Conte da Frota. “A esponja marinha Polymastia janeirensis como fonte de novos fármacos contra o câncer.” 2008. Thesis, Universidade do Rio Grande do Sul. Accessed October 20, 2019. http://hdl.handle.net/10183/13832.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Junior, Mário Luiz Conte da Frota. “A esponja marinha Polymastia janeirensis como fonte de novos fármacos contra o câncer.” 2008. Web. 20 Oct 2019.

Vancouver:

Junior MLCdF. A esponja marinha Polymastia janeirensis como fonte de novos fármacos contra o câncer. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2008. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10183/13832.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Junior MLCdF. A esponja marinha Polymastia janeirensis como fonte de novos fármacos contra o câncer. [Thesis]. Universidade do Rio Grande do Sul; 2008. Available from: http://hdl.handle.net/10183/13832

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

21. Ledur, Pítia Flores. Células tronco tumorais e o sistema purinérgico.

Degree: 2009, Universidade do Rio Grande do Sul

Gliomas são os tumores mais comuns no SNC, apresentando altas taxas de invasibilidade e proliferação, resistência à quimio e radioterapias, e elevados índices de recorrência… (more)

Subjects/Keywords: Glioma; Biologia molecular; Biologia celular

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APA (6th Edition):

Ledur, P. F. (2009). Células tronco tumorais e o sistema purinérgico. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/17066

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ledur, Pítia Flores. “Células tronco tumorais e o sistema purinérgico.” 2009. Thesis, Universidade do Rio Grande do Sul. Accessed October 20, 2019. http://hdl.handle.net/10183/17066.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ledur, Pítia Flores. “Células tronco tumorais e o sistema purinérgico.” 2009. Web. 20 Oct 2019.

Vancouver:

Ledur PF. Células tronco tumorais e o sistema purinérgico. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2009. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10183/17066.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ledur PF. Células tronco tumorais e o sistema purinérgico. [Thesis]. Universidade do Rio Grande do Sul; 2009. Available from: http://hdl.handle.net/10183/17066

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

22. Brennan, Paul Martin. Using novel models of glioma for cancer discovery science.

Degree: PhD, 2014, University of Edinburgh

 The prognosis for patients diagnosed with glioma has changed little over the past two decades. Many therapies that appeared promising in preclinical studies have been… (more)

Subjects/Keywords: 616.99; glioma; stem cells

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APA (6th Edition):

Brennan, P. M. (2014). Using novel models of glioma for cancer discovery science. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/9613

Chicago Manual of Style (16th Edition):

Brennan, Paul Martin. “Using novel models of glioma for cancer discovery science.” 2014. Doctoral Dissertation, University of Edinburgh. Accessed October 20, 2019. http://hdl.handle.net/1842/9613.

MLA Handbook (7th Edition):

Brennan, Paul Martin. “Using novel models of glioma for cancer discovery science.” 2014. Web. 20 Oct 2019.

Vancouver:

Brennan PM. Using novel models of glioma for cancer discovery science. [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/1842/9613.

Council of Science Editors:

Brennan PM. Using novel models of glioma for cancer discovery science. [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/9613


Universidade do Rio Grande do Sul

23. Silva, Andrew Oliveira. Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas.

Degree: 2012, Universidade do Rio Grande do Sul

 Gliomas malignos compreendem o subtipo mais comum e devastador de tumores primários do sistema nervoso central (SNC), sendo o Glioblastoma Multiforme (GBM) a forma mais… (more)

Subjects/Keywords: Glioblastoma; Glioma; Apoptose; Autofagia

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APA (6th Edition):

Silva, A. O. (2012). Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/150622

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Andrew Oliveira. “Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas.” 2012. Thesis, Universidade do Rio Grande do Sul. Accessed October 20, 2019. http://hdl.handle.net/10183/150622.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Andrew Oliveira. “Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas.” 2012. Web. 20 Oct 2019.

Vancouver:

Silva AO. Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2012. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10183/150622.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva AO. Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas. [Thesis]. Universidade do Rio Grande do Sul; 2012. Available from: http://hdl.handle.net/10183/150622

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

24. Killela, Patrick J. Genetic Studies Identify Critical Biomarkers and Refine the Classification of Malignant Gliomas .

Degree: 2014, Duke University

  Gliomagenesis is driven by a complex network of genetic alterations and while the glioma genome has been a focus of investigation for many years;… (more)

Subjects/Keywords: Oncology; Medicine; Pathology; Glioma; Oncogenomics

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APA (6th Edition):

Killela, P. J. (2014). Genetic Studies Identify Critical Biomarkers and Refine the Classification of Malignant Gliomas . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/9080

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Killela, Patrick J. “Genetic Studies Identify Critical Biomarkers and Refine the Classification of Malignant Gliomas .” 2014. Thesis, Duke University. Accessed October 20, 2019. http://hdl.handle.net/10161/9080.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Killela, Patrick J. “Genetic Studies Identify Critical Biomarkers and Refine the Classification of Malignant Gliomas .” 2014. Web. 20 Oct 2019.

Vancouver:

Killela PJ. Genetic Studies Identify Critical Biomarkers and Refine the Classification of Malignant Gliomas . [Internet] [Thesis]. Duke University; 2014. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10161/9080.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Killela PJ. Genetic Studies Identify Critical Biomarkers and Refine the Classification of Malignant Gliomas . [Thesis]. Duke University; 2014. Available from: http://hdl.handle.net/10161/9080

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

25. Gonçalves, Rosângela Mayer. O papel da autofagia na resistência de gliomas ao tratamento com temozolomida e inibidor de histonas desacetilases.

Degree: 2017, Universidade do Rio Grande do Sul

Glioblastoma multiforme é o tipo mais frequente de tumor cerebral primário, sendo caracterizado por uma alta agressividade e um prognóstico bastante limitado. O ácido hidroxâmico… (more)

Subjects/Keywords: Autofagia; Glioma; Morte celular; Temozolamida

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APA (6th Edition):

Gonçalves, R. M. (2017). O papel da autofagia na resistência de gliomas ao tratamento com temozolomida e inibidor de histonas desacetilases. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/153333

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gonçalves, Rosângela Mayer. “O papel da autofagia na resistência de gliomas ao tratamento com temozolomida e inibidor de histonas desacetilases.” 2017. Thesis, Universidade do Rio Grande do Sul. Accessed October 20, 2019. http://hdl.handle.net/10183/153333.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gonçalves, Rosângela Mayer. “O papel da autofagia na resistência de gliomas ao tratamento com temozolomida e inibidor de histonas desacetilases.” 2017. Web. 20 Oct 2019.

Vancouver:

Gonçalves RM. O papel da autofagia na resistência de gliomas ao tratamento com temozolomida e inibidor de histonas desacetilases. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2017. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10183/153333.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gonçalves RM. O papel da autofagia na resistência de gliomas ao tratamento com temozolomida e inibidor de histonas desacetilases. [Thesis]. Universidade do Rio Grande do Sul; 2017. Available from: http://hdl.handle.net/10183/153333

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

26. Gascon, Marc-Andre. The prognostic value of biomarkers in the evaluation of glioblastoma multiforme.

Degree: MS, Physician Assistant Program, 2017, Boston University

 BACKGROUND: Glioblastoma multiforme (GBM) is a highly heterogeneous tumor of the central nervous system (CNS) that exhibits considerable variation in its clinical course. Recently, the… (more)

Subjects/Keywords: Neurosciences; Biomarker; Glioblastoma; Glioma; Prognostic

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APA (6th Edition):

Gascon, M. (2017). The prognostic value of biomarkers in the evaluation of glioblastoma multiforme. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/26587

Chicago Manual of Style (16th Edition):

Gascon, Marc-Andre. “The prognostic value of biomarkers in the evaluation of glioblastoma multiforme.” 2017. Masters Thesis, Boston University. Accessed October 20, 2019. http://hdl.handle.net/2144/26587.

MLA Handbook (7th Edition):

Gascon, Marc-Andre. “The prognostic value of biomarkers in the evaluation of glioblastoma multiforme.” 2017. Web. 20 Oct 2019.

Vancouver:

Gascon M. The prognostic value of biomarkers in the evaluation of glioblastoma multiforme. [Internet] [Masters thesis]. Boston University; 2017. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/2144/26587.

Council of Science Editors:

Gascon M. The prognostic value of biomarkers in the evaluation of glioblastoma multiforme. [Masters Thesis]. Boston University; 2017. Available from: http://hdl.handle.net/2144/26587


University of Melbourne

27. Liubinas, Simon Vincent. Radiological and molecular factors associated with seizures in patients with supratentorial gliomas.

Degree: 2013, University of Melbourne

 Tumour associated epilepsy (TAE) is a common and disabling symptom experienced by patients with supratentorial gliomas. The pathogenesis of TAE is likely to involve a… (more)

Subjects/Keywords: glioma; seizures; epilepsy; IDH1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Liubinas, S. V. (2013). Radiological and molecular factors associated with seizures in patients with supratentorial gliomas. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/38036

Chicago Manual of Style (16th Edition):

Liubinas, Simon Vincent. “Radiological and molecular factors associated with seizures in patients with supratentorial gliomas.” 2013. Doctoral Dissertation, University of Melbourne. Accessed October 20, 2019. http://hdl.handle.net/11343/38036.

MLA Handbook (7th Edition):

Liubinas, Simon Vincent. “Radiological and molecular factors associated with seizures in patients with supratentorial gliomas.” 2013. Web. 20 Oct 2019.

Vancouver:

Liubinas SV. Radiological and molecular factors associated with seizures in patients with supratentorial gliomas. [Internet] [Doctoral dissertation]. University of Melbourne; 2013. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/11343/38036.

Council of Science Editors:

Liubinas SV. Radiological and molecular factors associated with seizures in patients with supratentorial gliomas. [Doctoral Dissertation]. University of Melbourne; 2013. Available from: http://hdl.handle.net/11343/38036

28. Ríos García, Marcos. Papel de AMPK en la regulación del metabolismo y proliferación celular durante el proceso de tumorogénesis.

Degree: 2018, Universidad de Santiago de Compostela

Subjects/Keywords: Glioma; AMPK

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APA (6th Edition):

Ríos García, M. (2018). Papel de AMPK en la regulación del metabolismo y proliferación celular durante el proceso de tumorogénesis. (Thesis). Universidad de Santiago de Compostela. Retrieved from http://hdl.handle.net/10347/3711

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ríos García, Marcos. “Papel de AMPK en la regulación del metabolismo y proliferación celular durante el proceso de tumorogénesis.” 2018. Thesis, Universidad de Santiago de Compostela. Accessed October 20, 2019. http://hdl.handle.net/10347/3711.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ríos García, Marcos. “Papel de AMPK en la regulación del metabolismo y proliferación celular durante el proceso de tumorogénesis.” 2018. Web. 20 Oct 2019.

Vancouver:

Ríos García M. Papel de AMPK en la regulación del metabolismo y proliferación celular durante el proceso de tumorogénesis. [Internet] [Thesis]. Universidad de Santiago de Compostela; 2018. [cited 2019 Oct 20]. Available from: http://hdl.handle.net/10347/3711.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ríos García M. Papel de AMPK en la regulación del metabolismo y proliferación celular durante el proceso de tumorogénesis. [Thesis]. Universidad de Santiago de Compostela; 2018. Available from: http://hdl.handle.net/10347/3711

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

29. WU, HUI-CHUN. Autophagy inducers modulated ATG4B expression in human brain tumor cells.

Degree: Master, Biological Sciences, 2018, NSYSU

 Autophagy is a self-eating mechanism in cells through which damaged proteins and organelles are recruited to autophagosomes and fused with lysosome for their bulk degradation… (more)

Subjects/Keywords: Rapamycin; Chloroquine; Glioma; Autophagy; ATG4B

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

WU, H. (2018). Autophagy inducers modulated ATG4B expression in human brain tumor cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0605118-111049

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

WU, HUI-CHUN. “Autophagy inducers modulated ATG4B expression in human brain tumor cells.” 2018. Thesis, NSYSU. Accessed October 20, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0605118-111049.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

WU, HUI-CHUN. “Autophagy inducers modulated ATG4B expression in human brain tumor cells.” 2018. Web. 20 Oct 2019.

Vancouver:

WU H. Autophagy inducers modulated ATG4B expression in human brain tumor cells. [Internet] [Thesis]. NSYSU; 2018. [cited 2019 Oct 20]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0605118-111049.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

WU H. Autophagy inducers modulated ATG4B expression in human brain tumor cells. [Thesis]. NSYSU; 2018. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0605118-111049

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

30. Decker, Stacy Ann. A spontaneous murine model for the study of CD44 In glioma progression.

Degree: PhD, Neuroscience, 2012, University of Minnesota

 Mouse models of malignant glioma that accurately recapitulate the genetic and phenotypic heterogeneity are essential for advancing brain tumor therapeutics. We have developed a novel… (more)

Subjects/Keywords: CD44; Glioma; Invasion; Tumor development

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Decker, S. A. (2012). A spontaneous murine model for the study of CD44 In glioma progression. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/136786

Chicago Manual of Style (16th Edition):

Decker, Stacy Ann. “A spontaneous murine model for the study of CD44 In glioma progression.” 2012. Doctoral Dissertation, University of Minnesota. Accessed October 20, 2019. http://purl.umn.edu/136786.

MLA Handbook (7th Edition):

Decker, Stacy Ann. “A spontaneous murine model for the study of CD44 In glioma progression.” 2012. Web. 20 Oct 2019.

Vancouver:

Decker SA. A spontaneous murine model for the study of CD44 In glioma progression. [Internet] [Doctoral dissertation]. University of Minnesota; 2012. [cited 2019 Oct 20]. Available from: http://purl.umn.edu/136786.

Council of Science Editors:

Decker SA. A spontaneous murine model for the study of CD44 In glioma progression. [Doctoral Dissertation]. University of Minnesota; 2012. Available from: http://purl.umn.edu/136786

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