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You searched for subject:(Glioblastoma). Showing records 1 – 30 of 811 total matches.

[1] [2] [3] [4] [5] … [28]

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Oregon State University

1. Lal, Sangeet Kumar. Calpain 2 proteolysis regulates glioblastoma cell invasion.

Degree: PhD, Biochemistry and Biophysics, 2011, Oregon State University

Glioblastoma is the most malignant primary brain tumor with the average patients surviving only one year after diagnosis, even with aggressive therapy. The formation of… (more)

Subjects/Keywords: Glioblastoma

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APA (6th Edition):

Lal, S. K. (2011). Calpain 2 proteolysis regulates glioblastoma cell invasion. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/19988

Chicago Manual of Style (16th Edition):

Lal, Sangeet Kumar. “Calpain 2 proteolysis regulates glioblastoma cell invasion.” 2011. Doctoral Dissertation, Oregon State University. Accessed February 26, 2021. http://hdl.handle.net/1957/19988.

MLA Handbook (7th Edition):

Lal, Sangeet Kumar. “Calpain 2 proteolysis regulates glioblastoma cell invasion.” 2011. Web. 26 Feb 2021.

Vancouver:

Lal SK. Calpain 2 proteolysis regulates glioblastoma cell invasion. [Internet] [Doctoral dissertation]. Oregon State University; 2011. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/1957/19988.

Council of Science Editors:

Lal SK. Calpain 2 proteolysis regulates glioblastoma cell invasion. [Doctoral Dissertation]. Oregon State University; 2011. Available from: http://hdl.handle.net/1957/19988


Universidad de Cantabria

2. Fernández Fuente, Gonzalo. Contribución del microentorno a la quimiorresistencia de células iniciadoras de glioblastoma en modelos in vitro.

Degree: 2017, Universidad de Cantabria

 En este trabajo, hemos estudiado la biología de los Glioblastomas, el tumor cerebral primario más frecuente en adultos y que representa uno de los tipos… (more)

Subjects/Keywords: Glioblastoma

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APA (6th Edition):

Fernández Fuente, G. (2017). Contribución del microentorno a la quimiorresistencia de células iniciadoras de glioblastoma en modelos in vitro. (Doctoral Dissertation). Universidad de Cantabria. Retrieved from http://hdl.handle.net/10902/11393

Chicago Manual of Style (16th Edition):

Fernández Fuente, Gonzalo. “Contribución del microentorno a la quimiorresistencia de células iniciadoras de glioblastoma en modelos in vitro.” 2017. Doctoral Dissertation, Universidad de Cantabria. Accessed February 26, 2021. http://hdl.handle.net/10902/11393.

MLA Handbook (7th Edition):

Fernández Fuente, Gonzalo. “Contribución del microentorno a la quimiorresistencia de células iniciadoras de glioblastoma en modelos in vitro.” 2017. Web. 26 Feb 2021.

Vancouver:

Fernández Fuente G. Contribución del microentorno a la quimiorresistencia de células iniciadoras de glioblastoma en modelos in vitro. [Internet] [Doctoral dissertation]. Universidad de Cantabria; 2017. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/10902/11393.

Council of Science Editors:

Fernández Fuente G. Contribución del microentorno a la quimiorresistencia de células iniciadoras de glioblastoma en modelos in vitro. [Doctoral Dissertation]. Universidad de Cantabria; 2017. Available from: http://hdl.handle.net/10902/11393


University of Manitoba

3. Yuan, Haynes (Shek Hei). Counteracting temozolomide resistance in glioblastoma multiforme (GBM) through suppression of the base excision repair (BER) pathway.

Degree: Pharmacology and Therapeutics, 2019, University of Manitoba

 Background Glioblastoma Multiforme (GBM) is an aggressive cerebral cancer. Standard chemotherapeutic of GBM is Temozolomide; a DNA alkylating agent. Patients relapse mostly due to recurrence… (more)

Subjects/Keywords: Glioblastoma Multiforme; Glioblastoma Multiforme

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APA (6th Edition):

Yuan, H. (. H. (2019). Counteracting temozolomide resistance in glioblastoma multiforme (GBM) through suppression of the base excision repair (BER) pathway. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/34166

Chicago Manual of Style (16th Edition):

Yuan, Haynes (Shek Hei). “Counteracting temozolomide resistance in glioblastoma multiforme (GBM) through suppression of the base excision repair (BER) pathway.” 2019. Masters Thesis, University of Manitoba. Accessed February 26, 2021. http://hdl.handle.net/1993/34166.

MLA Handbook (7th Edition):

Yuan, Haynes (Shek Hei). “Counteracting temozolomide resistance in glioblastoma multiforme (GBM) through suppression of the base excision repair (BER) pathway.” 2019. Web. 26 Feb 2021.

Vancouver:

Yuan H(H. Counteracting temozolomide resistance in glioblastoma multiforme (GBM) through suppression of the base excision repair (BER) pathway. [Internet] [Masters thesis]. University of Manitoba; 2019. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/1993/34166.

Council of Science Editors:

Yuan H(H. Counteracting temozolomide resistance in glioblastoma multiforme (GBM) through suppression of the base excision repair (BER) pathway. [Masters Thesis]. University of Manitoba; 2019. Available from: http://hdl.handle.net/1993/34166


Universidade do Rio Grande do Sul

4. Chiela, Eduardo Cremonese Filippi. A autofagia e o ciclo celular desempenham papel central no mecanismo de ação do resveratrol e do co-tratamento com temozolomida em células de linhagens de glioblastoma humano.

Degree: 2011, Universidade do Rio Grande do Sul

Glioblastomas (GBMs) são os tumores primários (gliomas) mais comuns e agressivos do Sistema Nervosos Central, classificados pelos oncologistas como um dos maiores desafios da oncoterapia.… (more)

Subjects/Keywords: Glioblastoma; Resveratrol

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APA (6th Edition):

Chiela, E. C. F. (2011). A autofagia e o ciclo celular desempenham papel central no mecanismo de ação do resveratrol e do co-tratamento com temozolomida em células de linhagens de glioblastoma humano. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/28430

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chiela, Eduardo Cremonese Filippi. “A autofagia e o ciclo celular desempenham papel central no mecanismo de ação do resveratrol e do co-tratamento com temozolomida em células de linhagens de glioblastoma humano.” 2011. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2021. http://hdl.handle.net/10183/28430.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chiela, Eduardo Cremonese Filippi. “A autofagia e o ciclo celular desempenham papel central no mecanismo de ação do resveratrol e do co-tratamento com temozolomida em células de linhagens de glioblastoma humano.” 2011. Web. 26 Feb 2021.

Vancouver:

Chiela ECF. A autofagia e o ciclo celular desempenham papel central no mecanismo de ação do resveratrol e do co-tratamento com temozolomida em células de linhagens de glioblastoma humano. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2011. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/10183/28430.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chiela ECF. A autofagia e o ciclo celular desempenham papel central no mecanismo de ação do resveratrol e do co-tratamento com temozolomida em células de linhagens de glioblastoma humano. [Thesis]. Universidade do Rio Grande do Sul; 2011. Available from: http://hdl.handle.net/10183/28430

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

5. Silva, Mardja Mansur Bueno e. Alterações mitocondriais e reprogramação metabólica em células de glioblastoma após tratamento com temozolomida.

Degree: 2016, Universidade do Rio Grande do Sul

Mitocôndrias desempenham funções celulares vitais. O funcionamento dessas organelas e seu papel no metabolismo celular, não surpreendentemente, têm sido implicados no desenvolvimento de diferentes cânceres.… (more)

Subjects/Keywords: Glioblastoma; Temozolamida

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APA (6th Edition):

Silva, M. M. B. e. (2016). Alterações mitocondriais e reprogramação metabólica em células de glioblastoma após tratamento com temozolomida. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/150664

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Mardja Mansur Bueno e. “Alterações mitocondriais e reprogramação metabólica em células de glioblastoma após tratamento com temozolomida.” 2016. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2021. http://hdl.handle.net/10183/150664.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Mardja Mansur Bueno e. “Alterações mitocondriais e reprogramação metabólica em células de glioblastoma após tratamento com temozolomida.” 2016. Web. 26 Feb 2021.

Vancouver:

Silva MMBe. Alterações mitocondriais e reprogramação metabólica em células de glioblastoma após tratamento com temozolomida. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2016. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/10183/150664.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva MMBe. Alterações mitocondriais e reprogramação metabólica em células de glioblastoma após tratamento com temozolomida. [Thesis]. Universidade do Rio Grande do Sul; 2016. Available from: http://hdl.handle.net/10183/150664

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

6. Folck, Anthony F. A Cell-Based Model to Study Factors that Drive Diffuse Astrocytoma Development.

Degree: 2016, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Secondary gliomas are an incurable form of brain cancer that are diagnosed in people at a median age of 45… (more)

Subjects/Keywords: Astrocytoma; Glioblastoma

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APA (6th Edition):

Folck, A. F. (2016). A Cell-Based Model to Study Factors that Drive Diffuse Astrocytoma Development. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/11053

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Folck, Anthony F. “A Cell-Based Model to Study Factors that Drive Diffuse Astrocytoma Development.” 2016. Thesis, IUPUI. Accessed February 26, 2021. http://hdl.handle.net/1805/11053.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Folck, Anthony F. “A Cell-Based Model to Study Factors that Drive Diffuse Astrocytoma Development.” 2016. Web. 26 Feb 2021.

Vancouver:

Folck AF. A Cell-Based Model to Study Factors that Drive Diffuse Astrocytoma Development. [Internet] [Thesis]. IUPUI; 2016. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/1805/11053.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Folck AF. A Cell-Based Model to Study Factors that Drive Diffuse Astrocytoma Development. [Thesis]. IUPUI; 2016. Available from: http://hdl.handle.net/1805/11053

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidad de Cantabria

7. López López, Carlos. Factores pronósticos clínico-moleculares y modelos predictivos en glioblastoma multiforme a partir de una experiencia intramural : Servicio de Oncología Médica del Hospital Universitario Marqués de Valdecilla (2000-2010).

Degree: 2016, Universidad de Cantabria

 RESUMEN: El glioblastoma multiforme (GBM) es una neoplasia relativamente frecuente entre las del sistema nervioso central, que se caracteriza por su excepcional agresividad y por… (more)

Subjects/Keywords: Glioblastoma multiforme

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

López López, C. (2016). Factores pronósticos clínico-moleculares y modelos predictivos en glioblastoma multiforme a partir de una experiencia intramural : Servicio de Oncología Médica del Hospital Universitario Marqués de Valdecilla (2000-2010). (Doctoral Dissertation). Universidad de Cantabria. Retrieved from http://hdl.handle.net/10902/8433

Chicago Manual of Style (16th Edition):

López López, Carlos. “Factores pronósticos clínico-moleculares y modelos predictivos en glioblastoma multiforme a partir de una experiencia intramural : Servicio de Oncología Médica del Hospital Universitario Marqués de Valdecilla (2000-2010).” 2016. Doctoral Dissertation, Universidad de Cantabria. Accessed February 26, 2021. http://hdl.handle.net/10902/8433.

MLA Handbook (7th Edition):

López López, Carlos. “Factores pronósticos clínico-moleculares y modelos predictivos en glioblastoma multiforme a partir de una experiencia intramural : Servicio de Oncología Médica del Hospital Universitario Marqués de Valdecilla (2000-2010).” 2016. Web. 26 Feb 2021.

Vancouver:

López López C. Factores pronósticos clínico-moleculares y modelos predictivos en glioblastoma multiforme a partir de una experiencia intramural : Servicio de Oncología Médica del Hospital Universitario Marqués de Valdecilla (2000-2010). [Internet] [Doctoral dissertation]. Universidad de Cantabria; 2016. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/10902/8433.

Council of Science Editors:

López López C. Factores pronósticos clínico-moleculares y modelos predictivos en glioblastoma multiforme a partir de una experiencia intramural : Servicio de Oncología Médica del Hospital Universitario Marqués de Valdecilla (2000-2010). [Doctoral Dissertation]. Universidad de Cantabria; 2016. Available from: http://hdl.handle.net/10902/8433


Universidade do Rio Grande do Sul

8. Tamajusuku, Alessandra Sayuri Kikuchi. Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261.

Degree: 2010, Universidade do Rio Grande do Sul

O nucleotídeo de purina ATP, no meio extracelular, participa de diversos processos fisiológicos e patológicos como vasodilatação/constrição, proliferação, diferenciação, modulação sináptica, dor, inflamação e morte… (more)

Subjects/Keywords: Glioma; Glioblastoma; Camundongos

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APA (6th Edition):

Tamajusuku, A. S. K. (2010). Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/30210

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tamajusuku, Alessandra Sayuri Kikuchi. “Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261.” 2010. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2021. http://hdl.handle.net/10183/30210.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tamajusuku, Alessandra Sayuri Kikuchi. “Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261.” 2010. Web. 26 Feb 2021.

Vancouver:

Tamajusuku ASK. Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2010. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/10183/30210.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tamajusuku ASK. Expressão e funcionalidade do receptor P2X7 em linhagem de glioma de camundongo GL261. [Thesis]. Universidade do Rio Grande do Sul; 2010. Available from: http://hdl.handle.net/10183/30210

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

9. Kipper, Franciele Cristina. Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes.

Degree: 2017, Universidade do Rio Grande do Sul

 Gliomas são tumores do sistema nervoso central caracterizados por alta invasibilidade e mortalidade. Inúmeros esforços foram feitos nas últimas décadas para melhorar a sobrevida dos… (more)

Subjects/Keywords: Glioblastoma; Glioma; Temozolomida

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APA (6th Edition):

Kipper, F. C. (2017). Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/170182

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kipper, Franciele Cristina. “Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes.” 2017. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2021. http://hdl.handle.net/10183/170182.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kipper, Franciele Cristina. “Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes.” 2017. Web. 26 Feb 2021.

Vancouver:

Kipper FC. Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2017. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/10183/170182.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kipper FC. Novas abordagens terapêuticas para glioblastoma baseadas no ensaio de resposta a terapias em culturas derivadas de pacientes. [Thesis]. Universidade do Rio Grande do Sul; 2017. Available from: http://hdl.handle.net/10183/170182

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

10. Pereira, Mariana Brutschin. Caracterização transcricional de infiltrados imunológicos e sua relação com a sobrevida de pacientes com glioblastoma.

Degree: 2017, Universidade do Rio Grande do Sul

Introdução: A complexidade das populações de células do sistema imunológico infiltrando tumores humanos com seus efeitos sinérgicos ou antagônicos pode influenciar os tumores de forma… (more)

Subjects/Keywords: Glioblastoma; Sistema imunológico

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pereira, M. B. (2017). Caracterização transcricional de infiltrados imunológicos e sua relação com a sobrevida de pacientes com glioblastoma. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/170192

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pereira, Mariana Brutschin. “Caracterização transcricional de infiltrados imunológicos e sua relação com a sobrevida de pacientes com glioblastoma.” 2017. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2021. http://hdl.handle.net/10183/170192.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pereira, Mariana Brutschin. “Caracterização transcricional de infiltrados imunológicos e sua relação com a sobrevida de pacientes com glioblastoma.” 2017. Web. 26 Feb 2021.

Vancouver:

Pereira MB. Caracterização transcricional de infiltrados imunológicos e sua relação com a sobrevida de pacientes com glioblastoma. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2017. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/10183/170192.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pereira MB. Caracterização transcricional de infiltrados imunológicos e sua relação com a sobrevida de pacientes com glioblastoma. [Thesis]. Universidade do Rio Grande do Sul; 2017. Available from: http://hdl.handle.net/10183/170192

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

11. Silva, Andrew Oliveira. Avaliação dos efeitos antitumorais agudos e crônicos em resposta ao tratamento com temozolomida e sua combinação com vimblastia e mebendazole em células de glioblastoma.

Degree: 2016, Universidade do Rio Grande do Sul

Apesar dos progressos na compreensão da biologia dos Glioblastomas (GBM), poucos avanços terapêuticos foram obtidos, desde que a Temozolomida (TMZ) foi implementada, em 2005, como… (more)

Subjects/Keywords: Glioblastoma; Temozolomida; Vimblastina

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Silva, A. O. (2016). Avaliação dos efeitos antitumorais agudos e crônicos em resposta ao tratamento com temozolomida e sua combinação com vimblastia e mebendazole em células de glioblastoma. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/150589

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Andrew Oliveira. “Avaliação dos efeitos antitumorais agudos e crônicos em resposta ao tratamento com temozolomida e sua combinação com vimblastia e mebendazole em células de glioblastoma.” 2016. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2021. http://hdl.handle.net/10183/150589.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Andrew Oliveira. “Avaliação dos efeitos antitumorais agudos e crônicos em resposta ao tratamento com temozolomida e sua combinação com vimblastia e mebendazole em células de glioblastoma.” 2016. Web. 26 Feb 2021.

Vancouver:

Silva AO. Avaliação dos efeitos antitumorais agudos e crônicos em resposta ao tratamento com temozolomida e sua combinação com vimblastia e mebendazole em células de glioblastoma. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2016. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/10183/150589.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva AO. Avaliação dos efeitos antitumorais agudos e crônicos em resposta ao tratamento com temozolomida e sua combinação com vimblastia e mebendazole em células de glioblastoma. [Thesis]. Universidade do Rio Grande do Sul; 2016. Available from: http://hdl.handle.net/10183/150589

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

12. Finkelberg, Tomer. Investigating the role of cytomegalovirus in the potentiation of glioblastoma growth using multiple murine models.

Degree: MS, Medical Sciences, 2020, Boston University

 OBJECTIVES: To determine the effect of murine cytomegalovirus (MCMV) infection on the growth and proliferation of glioblastoma using murine models of differing genetic backgrounds, as… (more)

Subjects/Keywords: Oncology; Cytomegalovirus; Glioblastoma

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Finkelberg, T. (2020). Investigating the role of cytomegalovirus in the potentiation of glioblastoma growth using multiple murine models. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/41237

Chicago Manual of Style (16th Edition):

Finkelberg, Tomer. “Investigating the role of cytomegalovirus in the potentiation of glioblastoma growth using multiple murine models.” 2020. Masters Thesis, Boston University. Accessed February 26, 2021. http://hdl.handle.net/2144/41237.

MLA Handbook (7th Edition):

Finkelberg, Tomer. “Investigating the role of cytomegalovirus in the potentiation of glioblastoma growth using multiple murine models.” 2020. Web. 26 Feb 2021.

Vancouver:

Finkelberg T. Investigating the role of cytomegalovirus in the potentiation of glioblastoma growth using multiple murine models. [Internet] [Masters thesis]. Boston University; 2020. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/2144/41237.

Council of Science Editors:

Finkelberg T. Investigating the role of cytomegalovirus in the potentiation of glioblastoma growth using multiple murine models. [Masters Thesis]. Boston University; 2020. Available from: http://hdl.handle.net/2144/41237


University of Ottawa

13. Gont, Alexander. Inactivation of Lgl1 in Glioblastoma .

Degree: 2016, University of Ottawa

Glioblastoma is the most aggressive and invasive adult brain cancer. In glioblastoma, the loss of the tumour suppressor PTEN is the most common genetic alteration… (more)

Subjects/Keywords: cancer; glioblastoma; Lgl

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APA (6th Edition):

Gont, A. (2016). Inactivation of Lgl1 in Glioblastoma . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/34965

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gont, Alexander. “Inactivation of Lgl1 in Glioblastoma .” 2016. Thesis, University of Ottawa. Accessed February 26, 2021. http://hdl.handle.net/10393/34965.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gont, Alexander. “Inactivation of Lgl1 in Glioblastoma .” 2016. Web. 26 Feb 2021.

Vancouver:

Gont A. Inactivation of Lgl1 in Glioblastoma . [Internet] [Thesis]. University of Ottawa; 2016. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/10393/34965.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gont A. Inactivation of Lgl1 in Glioblastoma . [Thesis]. University of Ottawa; 2016. Available from: http://hdl.handle.net/10393/34965

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

14. Albert-Vartanian, Alenoush. Role of Hexokinase 2 (HK2) in Modulating Tumor Metabolism and Response to Therapy in Glioblastoma.

Degree: 2013, University of Toronto

Glioblastoma (GBM), similar to many other cancers, exhibits enhanced aerobic glycolysis with concomitant lactate production, a phenomenon known as the Warburg effect. We have demonstrated… (more)

Subjects/Keywords: Metabolism; Glioblastoma; 0992

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APA (6th Edition):

Albert-Vartanian, A. (2013). Role of Hexokinase 2 (HK2) in Modulating Tumor Metabolism and Response to Therapy in Glioblastoma. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42661

Chicago Manual of Style (16th Edition):

Albert-Vartanian, Alenoush. “Role of Hexokinase 2 (HK2) in Modulating Tumor Metabolism and Response to Therapy in Glioblastoma.” 2013. Masters Thesis, University of Toronto. Accessed February 26, 2021. http://hdl.handle.net/1807/42661.

MLA Handbook (7th Edition):

Albert-Vartanian, Alenoush. “Role of Hexokinase 2 (HK2) in Modulating Tumor Metabolism and Response to Therapy in Glioblastoma.” 2013. Web. 26 Feb 2021.

Vancouver:

Albert-Vartanian A. Role of Hexokinase 2 (HK2) in Modulating Tumor Metabolism and Response to Therapy in Glioblastoma. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/1807/42661.

Council of Science Editors:

Albert-Vartanian A. Role of Hexokinase 2 (HK2) in Modulating Tumor Metabolism and Response to Therapy in Glioblastoma. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/42661


University of Houston

15. Avci, Naze Gul. The Influence of Human Umbilical Vein Endothelial Cells in the Formation of Glioblastoma Spheroids in Three-Dimensional Microwells.

Degree: PhD, Biomedical Engineering, 2015, University of Houston

Glioblastoma multiforme (GBM) is the most malignant primary brain tumor with a high infiltrative capacity and increased vascularity. Despite current therapies overall patient survival rate… (more)

Subjects/Keywords: Glioblastoma; 3D spheroids

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APA (6th Edition):

Avci, N. G. (2015). The Influence of Human Umbilical Vein Endothelial Cells in the Formation of Glioblastoma Spheroids in Three-Dimensional Microwells. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/2005

Chicago Manual of Style (16th Edition):

Avci, Naze Gul. “The Influence of Human Umbilical Vein Endothelial Cells in the Formation of Glioblastoma Spheroids in Three-Dimensional Microwells.” 2015. Doctoral Dissertation, University of Houston. Accessed February 26, 2021. http://hdl.handle.net/10657/2005.

MLA Handbook (7th Edition):

Avci, Naze Gul. “The Influence of Human Umbilical Vein Endothelial Cells in the Formation of Glioblastoma Spheroids in Three-Dimensional Microwells.” 2015. Web. 26 Feb 2021.

Vancouver:

Avci NG. The Influence of Human Umbilical Vein Endothelial Cells in the Formation of Glioblastoma Spheroids in Three-Dimensional Microwells. [Internet] [Doctoral dissertation]. University of Houston; 2015. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/10657/2005.

Council of Science Editors:

Avci NG. The Influence of Human Umbilical Vein Endothelial Cells in the Formation of Glioblastoma Spheroids in Three-Dimensional Microwells. [Doctoral Dissertation]. University of Houston; 2015. Available from: http://hdl.handle.net/10657/2005


Boston University

16. Jindani, Rajika. Glioblastoma multiforme: etiology, progression, and treatment.

Degree: MS, Medical Sciences, 2017, Boston University

Glioblastoma multiforme is the most common and malignant brain tumor, accounting for more than 52% of all primary brain tumors. The molecular heterogeneity of the… (more)

Subjects/Keywords: Medicine; GBM; Glioblastoma

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APA (6th Edition):

Jindani, R. (2017). Glioblastoma multiforme: etiology, progression, and treatment. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/26730

Chicago Manual of Style (16th Edition):

Jindani, Rajika. “Glioblastoma multiforme: etiology, progression, and treatment.” 2017. Masters Thesis, Boston University. Accessed February 26, 2021. http://hdl.handle.net/2144/26730.

MLA Handbook (7th Edition):

Jindani, Rajika. “Glioblastoma multiforme: etiology, progression, and treatment.” 2017. Web. 26 Feb 2021.

Vancouver:

Jindani R. Glioblastoma multiforme: etiology, progression, and treatment. [Internet] [Masters thesis]. Boston University; 2017. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/2144/26730.

Council of Science Editors:

Jindani R. Glioblastoma multiforme: etiology, progression, and treatment. [Masters Thesis]. Boston University; 2017. Available from: http://hdl.handle.net/2144/26730


Victoria University of Wellington

17. Brow, Susanna. Investigating the Potential Role of SIRT1 in Glioblastoma Multiforme: A Comparison Between Glioma and Normal Astrocyte Cells in Culture.

Degree: 2015, Victoria University of Wellington

 Objective Glioblastomas (GBMs) are the most prevalent primary brain tumours in adults and the outcome for this disease remains very poor. With treatment options limited,… (more)

Subjects/Keywords: SIRT1; Sirtuin; Glioblastoma

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APA (6th Edition):

Brow, S. (2015). Investigating the Potential Role of SIRT1 in Glioblastoma Multiforme: A Comparison Between Glioma and Normal Astrocyte Cells in Culture. (Masters Thesis). Victoria University of Wellington. Retrieved from http://hdl.handle.net/10063/4879

Chicago Manual of Style (16th Edition):

Brow, Susanna. “Investigating the Potential Role of SIRT1 in Glioblastoma Multiforme: A Comparison Between Glioma and Normal Astrocyte Cells in Culture.” 2015. Masters Thesis, Victoria University of Wellington. Accessed February 26, 2021. http://hdl.handle.net/10063/4879.

MLA Handbook (7th Edition):

Brow, Susanna. “Investigating the Potential Role of SIRT1 in Glioblastoma Multiforme: A Comparison Between Glioma and Normal Astrocyte Cells in Culture.” 2015. Web. 26 Feb 2021.

Vancouver:

Brow S. Investigating the Potential Role of SIRT1 in Glioblastoma Multiforme: A Comparison Between Glioma and Normal Astrocyte Cells in Culture. [Internet] [Masters thesis]. Victoria University of Wellington; 2015. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/10063/4879.

Council of Science Editors:

Brow S. Investigating the Potential Role of SIRT1 in Glioblastoma Multiforme: A Comparison Between Glioma and Normal Astrocyte Cells in Culture. [Masters Thesis]. Victoria University of Wellington; 2015. Available from: http://hdl.handle.net/10063/4879


University of Minnesota

18. Becker, Chani. Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma.

Degree: PhD, Neuroscience, 2016, University of Minnesota

Glioblastoma multiforme (GBM) is a lethal cancer. Without treatment, patients diagnosed with this disease survive nine months. With the best therapeutics science has to offer,… (more)

Subjects/Keywords: BBB; Glioblastoma; Glioma

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APA (6th Edition):

Becker, C. (2016). Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/182319

Chicago Manual of Style (16th Edition):

Becker, Chani. “Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma.” 2016. Doctoral Dissertation, University of Minnesota. Accessed February 26, 2021. http://hdl.handle.net/11299/182319.

MLA Handbook (7th Edition):

Becker, Chani. “Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma.” 2016. Web. 26 Feb 2021.

Vancouver:

Becker C. Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma. [Internet] [Doctoral dissertation]. University of Minnesota; 2016. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/11299/182319.

Council of Science Editors:

Becker C. Molecular Mechanisms Underlying the Failures of Therapeutics in the Treatment of Malignant Glioma. [Doctoral Dissertation]. University of Minnesota; 2016. Available from: http://hdl.handle.net/11299/182319


University of Melbourne

19. Daniel, Paul Marcel. Identification of CREB as a transcriptional regulator of glioblastoma biology.

Degree: 2015, University of Melbourne

Glioblastoma (GBM) is both the most common and malignant type of brain tumour with a median survival of 7.4 months from the date of diagnosis;… (more)

Subjects/Keywords: glioblastoma; CREB; transcription

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APA (6th Edition):

Daniel, P. M. (2015). Identification of CREB as a transcriptional regulator of glioblastoma biology. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/58851

Chicago Manual of Style (16th Edition):

Daniel, Paul Marcel. “Identification of CREB as a transcriptional regulator of glioblastoma biology.” 2015. Doctoral Dissertation, University of Melbourne. Accessed February 26, 2021. http://hdl.handle.net/11343/58851.

MLA Handbook (7th Edition):

Daniel, Paul Marcel. “Identification of CREB as a transcriptional regulator of glioblastoma biology.” 2015. Web. 26 Feb 2021.

Vancouver:

Daniel PM. Identification of CREB as a transcriptional regulator of glioblastoma biology. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/11343/58851.

Council of Science Editors:

Daniel PM. Identification of CREB as a transcriptional regulator of glioblastoma biology. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/58851


University of New South Wales

20. Mutyala, Naga. Evaluating a rodent model and treatments for glioblastoma using gene expression profiling.

Degree: Biotechnology & Biomolecular Sciences, 2019, University of New South Wales

Glioblastoma is the most lethal primary brain tumour in adults, with a poor prognosis and a median survival of less than 15 months. Clearly novel… (more)

Subjects/Keywords: Glioblastoma; Gene expression

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APA (6th Edition):

Mutyala, N. (2019). Evaluating a rodent model and treatments for glioblastoma using gene expression profiling. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/66384

Chicago Manual of Style (16th Edition):

Mutyala, Naga. “Evaluating a rodent model and treatments for glioblastoma using gene expression profiling.” 2019. Doctoral Dissertation, University of New South Wales. Accessed February 26, 2021. http://handle.unsw.edu.au/1959.4/66384.

MLA Handbook (7th Edition):

Mutyala, Naga. “Evaluating a rodent model and treatments for glioblastoma using gene expression profiling.” 2019. Web. 26 Feb 2021.

Vancouver:

Mutyala N. Evaluating a rodent model and treatments for glioblastoma using gene expression profiling. [Internet] [Doctoral dissertation]. University of New South Wales; 2019. [cited 2021 Feb 26]. Available from: http://handle.unsw.edu.au/1959.4/66384.

Council of Science Editors:

Mutyala N. Evaluating a rodent model and treatments for glioblastoma using gene expression profiling. [Doctoral Dissertation]. University of New South Wales; 2019. Available from: http://handle.unsw.edu.au/1959.4/66384


NSYSU

21. Lee, Ya-che. Hexane fraction of Pluchea indica (L.) Less. root extract induce autophagic cell death on glioblastoma multiforme U87 cells.

Degree: Master, Biological Sciences, 2016, NSYSU

 Pluchea indica (L.) Less. is a perennial plant known for its versatile uses in traditional oriental medicine. In our previous study, we demonstrate the in… (more)

Subjects/Keywords: Glioblastoma Multiforme; Autophagy; Pluchea indica

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APA (6th Edition):

Lee, Y. (2016). Hexane fraction of Pluchea indica (L.) Less. root extract induce autophagic cell death on glioblastoma multiforme U87 cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0508116-175738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Ya-che. “Hexane fraction of Pluchea indica (L.) Less. root extract induce autophagic cell death on glioblastoma multiforme U87 cells.” 2016. Thesis, NSYSU. Accessed February 26, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0508116-175738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Ya-che. “Hexane fraction of Pluchea indica (L.) Less. root extract induce autophagic cell death on glioblastoma multiforme U87 cells.” 2016. Web. 26 Feb 2021.

Vancouver:

Lee Y. Hexane fraction of Pluchea indica (L.) Less. root extract induce autophagic cell death on glioblastoma multiforme U87 cells. [Internet] [Thesis]. NSYSU; 2016. [cited 2021 Feb 26]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0508116-175738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee Y. Hexane fraction of Pluchea indica (L.) Less. root extract induce autophagic cell death on glioblastoma multiforme U87 cells. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0508116-175738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

22. Kagami, Luciano Porto. Estudo in silico de novas diidropirimidin-2-tionas com ação inibitória da enzima ecto-5'-nucleotidase.

Degree: 2017, Universidade do Rio Grande do Sul

Os tumores cerebrais primários mais comuns são os gliomas. Os gliomas representam 31% de todos os tumores cerebrais diagnosticados nos Estados Unidos sendo 81% destes… (more)

Subjects/Keywords: Medicamentos; Glioblastoma; Modelagem molecular

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APA (6th Edition):

Kagami, L. P. (2017). Estudo in silico de novas diidropirimidin-2-tionas com ação inibitória da enzima ecto-5'-nucleotidase. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/170643

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kagami, Luciano Porto. “Estudo in silico de novas diidropirimidin-2-tionas com ação inibitória da enzima ecto-5'-nucleotidase.” 2017. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2021. http://hdl.handle.net/10183/170643.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kagami, Luciano Porto. “Estudo in silico de novas diidropirimidin-2-tionas com ação inibitória da enzima ecto-5'-nucleotidase.” 2017. Web. 26 Feb 2021.

Vancouver:

Kagami LP. Estudo in silico de novas diidropirimidin-2-tionas com ação inibitória da enzima ecto-5'-nucleotidase. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2017. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/10183/170643.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kagami LP. Estudo in silico de novas diidropirimidin-2-tionas com ação inibitória da enzima ecto-5'-nucleotidase. [Thesis]. Universidade do Rio Grande do Sul; 2017. Available from: http://hdl.handle.net/10183/170643

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

23. Silva, Andrew Oliveira. Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas.

Degree: 2012, Universidade do Rio Grande do Sul

 Gliomas malignos compreendem o subtipo mais comum e devastador de tumores primários do sistema nervoso central (SNC), sendo o Glioblastoma Multiforme (GBM) a forma mais… (more)

Subjects/Keywords: Glioblastoma; Glioma; Apoptose; Autofagia

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APA (6th Edition):

Silva, A. O. (2012). Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/150622

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Andrew Oliveira. “Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas.” 2012. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2021. http://hdl.handle.net/10183/150622.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Andrew Oliveira. “Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas.” 2012. Web. 26 Feb 2021.

Vancouver:

Silva AO. Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2012. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/10183/150622.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva AO. Silenciamento de XIAP potencializa os efeitos da superexpressão de TP53 na redução da proliferação e aumento da morte celular em gliomas. [Thesis]. Universidade do Rio Grande do Sul; 2012. Available from: http://hdl.handle.net/10183/150622

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

24. Stripay, Jennifer L. Targeting a Network of Cancer Control Nodes through Rescue of c-Cbl; A Novel Therapeutic Approach for Glioblastoma Multiforme.

Degree: PhD, 2016, University of Rochester

Glioblastoma multiforme is the most common primary brain tumor in adults, the most malignant of all intracranial tumors, and is associated with inevitable recurrence and… (more)

Subjects/Keywords: Cancer; cbl; Glioblastoma; Redox; Therapy

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APA (6th Edition):

Stripay, J. L. (2016). Targeting a Network of Cancer Control Nodes through Rescue of c-Cbl; A Novel Therapeutic Approach for Glioblastoma Multiforme. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/31636

Chicago Manual of Style (16th Edition):

Stripay, Jennifer L. “Targeting a Network of Cancer Control Nodes through Rescue of c-Cbl; A Novel Therapeutic Approach for Glioblastoma Multiforme.” 2016. Doctoral Dissertation, University of Rochester. Accessed February 26, 2021. http://hdl.handle.net/1802/31636.

MLA Handbook (7th Edition):

Stripay, Jennifer L. “Targeting a Network of Cancer Control Nodes through Rescue of c-Cbl; A Novel Therapeutic Approach for Glioblastoma Multiforme.” 2016. Web. 26 Feb 2021.

Vancouver:

Stripay JL. Targeting a Network of Cancer Control Nodes through Rescue of c-Cbl; A Novel Therapeutic Approach for Glioblastoma Multiforme. [Internet] [Doctoral dissertation]. University of Rochester; 2016. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/1802/31636.

Council of Science Editors:

Stripay JL. Targeting a Network of Cancer Control Nodes through Rescue of c-Cbl; A Novel Therapeutic Approach for Glioblastoma Multiforme. [Doctoral Dissertation]. University of Rochester; 2016. Available from: http://hdl.handle.net/1802/31636


University of Utah

25. Sankaranarayanan, Preethi. Generalized singular value decomposition (GSVD) comparison of patient-matched normal and tumor aCGH profiles reveals global copy-number alterations predicting glioblastoma multiforme survival.

Degree: MS, Bioengineering, 2013, University of Utah

 Despite recent large-scale profiling efforts, the best prognostic predictor ofglioblastoma multiforme (GBM) remains the patient’s age at diagnosis. Wedescribe a global pattern of tumor-exclusive co-occurring… (more)

Subjects/Keywords: Copy number; Glioblastoma multiforme; GSVD

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APA (6th Edition):

Sankaranarayanan, P. (2013). Generalized singular value decomposition (GSVD) comparison of patient-matched normal and tumor aCGH profiles reveals global copy-number alterations predicting glioblastoma multiforme survival. (Masters Thesis). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2111/rec/1101

Chicago Manual of Style (16th Edition):

Sankaranarayanan, Preethi. “Generalized singular value decomposition (GSVD) comparison of patient-matched normal and tumor aCGH profiles reveals global copy-number alterations predicting glioblastoma multiforme survival.” 2013. Masters Thesis, University of Utah. Accessed February 26, 2021. http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2111/rec/1101.

MLA Handbook (7th Edition):

Sankaranarayanan, Preethi. “Generalized singular value decomposition (GSVD) comparison of patient-matched normal and tumor aCGH profiles reveals global copy-number alterations predicting glioblastoma multiforme survival.” 2013. Web. 26 Feb 2021.

Vancouver:

Sankaranarayanan P. Generalized singular value decomposition (GSVD) comparison of patient-matched normal and tumor aCGH profiles reveals global copy-number alterations predicting glioblastoma multiforme survival. [Internet] [Masters thesis]. University of Utah; 2013. [cited 2021 Feb 26]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2111/rec/1101.

Council of Science Editors:

Sankaranarayanan P. Generalized singular value decomposition (GSVD) comparison of patient-matched normal and tumor aCGH profiles reveals global copy-number alterations predicting glioblastoma multiforme survival. [Masters Thesis]. University of Utah; 2013. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2111/rec/1101


Boston University

26. Handley, Meghan Victoria. GSK-3 inhibitors in glioblastoma therapy: mechanisms of action.

Degree: MS, Medical Sciences, 2015, Boston University

Glioblastoma multiforme (GBM) is the most malignant form of brain cancer. Therapies targeting glioblastoma have not consistently been able to give those diagnosed the best… (more)

Subjects/Keywords: Medicine; Glioblastoma; GSK-3 inhibitors

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APA (6th Edition):

Handley, M. V. (2015). GSK-3 inhibitors in glioblastoma therapy: mechanisms of action. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16063

Chicago Manual of Style (16th Edition):

Handley, Meghan Victoria. “GSK-3 inhibitors in glioblastoma therapy: mechanisms of action.” 2015. Masters Thesis, Boston University. Accessed February 26, 2021. http://hdl.handle.net/2144/16063.

MLA Handbook (7th Edition):

Handley, Meghan Victoria. “GSK-3 inhibitors in glioblastoma therapy: mechanisms of action.” 2015. Web. 26 Feb 2021.

Vancouver:

Handley MV. GSK-3 inhibitors in glioblastoma therapy: mechanisms of action. [Internet] [Masters thesis]. Boston University; 2015. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/2144/16063.

Council of Science Editors:

Handley MV. GSK-3 inhibitors in glioblastoma therapy: mechanisms of action. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/16063


Freie Universität Berlin

27. Onken, Julia Sophie. Experimental, translational and clinical aspects of individualized treatment of glioblastoma.

Degree: 2020, Freie Universität Berlin

 Die Zielsetzung der vorliegenden Arbeit ist es, neue Aspekte der experimentellen, translationalen und klinischen Forschung bei der individualisierten Behandlung des Glioblastoms darzustellen. Die Arbeiten befassen… (more)

Subjects/Keywords: glioblastoma; neuro-oncology; ddc:610

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APA (6th Edition):

Onken, J. S. (2020). Experimental, translational and clinical aspects of individualized treatment of glioblastoma. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-27455

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Onken, Julia Sophie. “Experimental, translational and clinical aspects of individualized treatment of glioblastoma.” 2020. Thesis, Freie Universität Berlin. Accessed February 26, 2021. http://dx.doi.org/10.17169/refubium-27455.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Onken, Julia Sophie. “Experimental, translational and clinical aspects of individualized treatment of glioblastoma.” 2020. Web. 26 Feb 2021.

Vancouver:

Onken JS. Experimental, translational and clinical aspects of individualized treatment of glioblastoma. [Internet] [Thesis]. Freie Universität Berlin; 2020. [cited 2021 Feb 26]. Available from: http://dx.doi.org/10.17169/refubium-27455.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Onken JS. Experimental, translational and clinical aspects of individualized treatment of glioblastoma. [Thesis]. Freie Universität Berlin; 2020. Available from: http://dx.doi.org/10.17169/refubium-27455

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vermont

28. Hermanowski, Henre Michael. Analysis of PARylation and PTEN Mutation Effects on PARP and PARG Inhibition Treatment in Primary Brain Tumors.

Degree: Biochemistry, 2019, University of Vermont

  Abstract: Glioblastoma multiforme (GBM) is the most aggressive primary brain tumor, with a median survival of approximately 15 months. Standard care for GBM has… (more)

Subjects/Keywords: glioblastoma; cancer; PARP; PARG; inhibition

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APA (6th Edition):

Hermanowski, H. M. (2019). Analysis of PARylation and PTEN Mutation Effects on PARP and PARG Inhibition Treatment in Primary Brain Tumors. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/hcoltheses/321

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hermanowski, Henre Michael. “Analysis of PARylation and PTEN Mutation Effects on PARP and PARG Inhibition Treatment in Primary Brain Tumors.” 2019. Thesis, University of Vermont. Accessed February 26, 2021. https://scholarworks.uvm.edu/hcoltheses/321.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hermanowski, Henre Michael. “Analysis of PARylation and PTEN Mutation Effects on PARP and PARG Inhibition Treatment in Primary Brain Tumors.” 2019. Web. 26 Feb 2021.

Vancouver:

Hermanowski HM. Analysis of PARylation and PTEN Mutation Effects on PARP and PARG Inhibition Treatment in Primary Brain Tumors. [Internet] [Thesis]. University of Vermont; 2019. [cited 2021 Feb 26]. Available from: https://scholarworks.uvm.edu/hcoltheses/321.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hermanowski HM. Analysis of PARylation and PTEN Mutation Effects on PARP and PARG Inhibition Treatment in Primary Brain Tumors. [Thesis]. University of Vermont; 2019. Available from: https://scholarworks.uvm.edu/hcoltheses/321

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

29. McEllin, Brian Matthew. Dissecting Molecular Mechanisms of Radioresistance Using in Vitro and in Vivo Brain Tumor Model Systems.

Degree: 2012, University of Texas Southwestern Medical Center

Glioblastoma multiforme (GBM) are deadly brain tumors that are refractory to radiation and chemotherapy. Despite decades of work, little progress has been made in improving… (more)

Subjects/Keywords: PTEN Phosphohydrolase; Brain Neoplasms; Glioblastoma

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APA (6th Edition):

McEllin, B. M. (2012). Dissecting Molecular Mechanisms of Radioresistance Using in Vitro and in Vivo Brain Tumor Model Systems. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1000

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McEllin, Brian Matthew. “Dissecting Molecular Mechanisms of Radioresistance Using in Vitro and in Vivo Brain Tumor Model Systems.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed February 26, 2021. http://hdl.handle.net/2152.5/1000.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McEllin, Brian Matthew. “Dissecting Molecular Mechanisms of Radioresistance Using in Vitro and in Vivo Brain Tumor Model Systems.” 2012. Web. 26 Feb 2021.

Vancouver:

McEllin BM. Dissecting Molecular Mechanisms of Radioresistance Using in Vitro and in Vivo Brain Tumor Model Systems. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/2152.5/1000.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McEllin BM. Dissecting Molecular Mechanisms of Radioresistance Using in Vitro and in Vivo Brain Tumor Model Systems. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1000

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

30. Vigna, Alexandra Souza. Avaliação do papel da proteína ATR na indução de autofagia e senescência por temozolomida em células de glioblastoma.

Degree: 2017, Universidade do Rio Grande do Sul

Autofagia é um processo de degradação fisiológico aumentado em diferentes condições de estresse, em que proteínas e organelas não funcionais são direcionadas ao lisossomo, onde… (more)

Subjects/Keywords: Glioblastoma; Temozolamida; Autofagia; Senescência

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vigna, A. S. (2017). Avaliação do papel da proteína ATR na indução de autofagia e senescência por temozolomida em células de glioblastoma. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/163683

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vigna, Alexandra Souza. “Avaliação do papel da proteína ATR na indução de autofagia e senescência por temozolomida em células de glioblastoma.” 2017. Thesis, Universidade do Rio Grande do Sul. Accessed February 26, 2021. http://hdl.handle.net/10183/163683.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vigna, Alexandra Souza. “Avaliação do papel da proteína ATR na indução de autofagia e senescência por temozolomida em células de glioblastoma.” 2017. Web. 26 Feb 2021.

Vancouver:

Vigna AS. Avaliação do papel da proteína ATR na indução de autofagia e senescência por temozolomida em células de glioblastoma. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2017. [cited 2021 Feb 26]. Available from: http://hdl.handle.net/10183/163683.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vigna AS. Avaliação do papel da proteína ATR na indução de autofagia e senescência por temozolomida em células de glioblastoma. [Thesis]. Universidade do Rio Grande do Sul; 2017. Available from: http://hdl.handle.net/10183/163683

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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