Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Genetics medical sciences ). Showing records 1 – 30 of 188217 total matches.

[1] [2] [3] [4] [5] … [6274]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters


Columbia University

1. Kuo, Chao-Ling. Characterization ofAthsq1, an Atherosclerosis Modifier Locus on Mouse Chromosome 4:.

Degree: 2011, Columbia University

 Atherosclerosis, the primary cause of heart attack, stroke, and peripheral vascular disease, is genetically complex and the genes that confer cardiovascular risk remain largely unknown.… (more)

Subjects/Keywords: Medical sciences; Genetics; Molecular biology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kuo, C. (2011). Characterization ofAthsq1, an Atherosclerosis Modifier Locus on Mouse Chromosome 4:. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8CV4QXC

Chicago Manual of Style (16th Edition):

Kuo, Chao-Ling. “Characterization ofAthsq1, an Atherosclerosis Modifier Locus on Mouse Chromosome 4:.” 2011. Doctoral Dissertation, Columbia University. Accessed January 29, 2020. https://doi.org/10.7916/D8CV4QXC.

MLA Handbook (7th Edition):

Kuo, Chao-Ling. “Characterization ofAthsq1, an Atherosclerosis Modifier Locus on Mouse Chromosome 4:.” 2011. Web. 29 Jan 2020.

Vancouver:

Kuo C. Characterization ofAthsq1, an Atherosclerosis Modifier Locus on Mouse Chromosome 4:. [Internet] [Doctoral dissertation]. Columbia University; 2011. [cited 2020 Jan 29]. Available from: https://doi.org/10.7916/D8CV4QXC.

Council of Science Editors:

Kuo C. Characterization ofAthsq1, an Atherosclerosis Modifier Locus on Mouse Chromosome 4:. [Doctoral Dissertation]. Columbia University; 2011. Available from: https://doi.org/10.7916/D8CV4QXC


University of Oxford

2. Shipman, Lydia. The functional consequences of autoimmune variants in the tyrosine kinase 2 gene region.

Degree: PhD, 2014, University of Oxford

 The tyrosine kinase 2 (TYK2) gene was first implicated in autoimmune disease in 2009 when a nonsynonymous single nucleotide polymorphism (nsSNP) in TYK2 was reported… (more)

Subjects/Keywords: 616.07; Genetics (medical sciences); Immunology; Autoimmunity; genetics

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shipman, L. (2014). The functional consequences of autoimmune variants in the tyrosine kinase 2 gene region. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:d7546fb0-3eb3-459c-867f-6e83d5dc2387 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.644708

Chicago Manual of Style (16th Edition):

Shipman, Lydia. “The functional consequences of autoimmune variants in the tyrosine kinase 2 gene region.” 2014. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:d7546fb0-3eb3-459c-867f-6e83d5dc2387 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.644708.

MLA Handbook (7th Edition):

Shipman, Lydia. “The functional consequences of autoimmune variants in the tyrosine kinase 2 gene region.” 2014. Web. 29 Jan 2020.

Vancouver:

Shipman L. The functional consequences of autoimmune variants in the tyrosine kinase 2 gene region. [Internet] [Doctoral dissertation]. University of Oxford; 2014. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:d7546fb0-3eb3-459c-867f-6e83d5dc2387 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.644708.

Council of Science Editors:

Shipman L. The functional consequences of autoimmune variants in the tyrosine kinase 2 gene region. [Doctoral Dissertation]. University of Oxford; 2014. Available from: http://ora.ox.ac.uk/objects/uuid:d7546fb0-3eb3-459c-867f-6e83d5dc2387 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.644708


University of Oxford

3. Nudel, Ron. Molecular genetics of language impairment.

Degree: PhD, 2015, University of Oxford

 Developmental language impairments are neurodevelopmental disorders in which the acquisition of language, a task which children typically perform with ease, is hindered or fraught with… (more)

Subjects/Keywords: 616.85; Medical Sciences; Clinical genetics; Genetics (medical sciences); Neuroscience; Neurogenetics; Genetics; Communication Disorders; Neurosciences; Genetics,Behavioral; Genetics,Medical

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nudel, R. (2015). Molecular genetics of language impairment. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:70249129-ef2e-4508-b8f6-50d6eae8e78b ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.640099

Chicago Manual of Style (16th Edition):

Nudel, Ron. “Molecular genetics of language impairment.” 2015. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:70249129-ef2e-4508-b8f6-50d6eae8e78b ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.640099.

MLA Handbook (7th Edition):

Nudel, Ron. “Molecular genetics of language impairment.” 2015. Web. 29 Jan 2020.

Vancouver:

Nudel R. Molecular genetics of language impairment. [Internet] [Doctoral dissertation]. University of Oxford; 2015. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:70249129-ef2e-4508-b8f6-50d6eae8e78b ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.640099.

Council of Science Editors:

Nudel R. Molecular genetics of language impairment. [Doctoral Dissertation]. University of Oxford; 2015. Available from: http://ora.ox.ac.uk/objects/uuid:70249129-ef2e-4508-b8f6-50d6eae8e78b ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.640099


University of Oxford

4. Wong, Hei Sunny. Genetic susceptibility to common mycobacterial diseases.

Degree: PhD, 2010, University of Oxford

 Common mycobacterial diseases, including tuberculosis and leprosy, contribute to major mortality and morbidity worldwide. Despite evidence of an important role of host genetic factors in… (more)

Subjects/Keywords: 616.9; Medical Sciences; Clinical genetics; Infectious diseases; Biology (medical sciences); Genetics (medical sciences); Evolution (zoology); genetics; susceptibility; mycobacteria; tuberculosis; leprosy

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wong, H. S. (2010). Genetic susceptibility to common mycobacterial diseases. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:cb4dd818-4693-4168-ad8a-cdeb59e2d5f3 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526415

Chicago Manual of Style (16th Edition):

Wong, Hei Sunny. “Genetic susceptibility to common mycobacterial diseases.” 2010. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:cb4dd818-4693-4168-ad8a-cdeb59e2d5f3 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526415.

MLA Handbook (7th Edition):

Wong, Hei Sunny. “Genetic susceptibility to common mycobacterial diseases.” 2010. Web. 29 Jan 2020.

Vancouver:

Wong HS. Genetic susceptibility to common mycobacterial diseases. [Internet] [Doctoral dissertation]. University of Oxford; 2010. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:cb4dd818-4693-4168-ad8a-cdeb59e2d5f3 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526415.

Council of Science Editors:

Wong HS. Genetic susceptibility to common mycobacterial diseases. [Doctoral Dissertation]. University of Oxford; 2010. Available from: http://ora.ox.ac.uk/objects/uuid:cb4dd818-4693-4168-ad8a-cdeb59e2d5f3 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526415


University of Oxford

5. Javid, Mahsa. Gene modifiers and novel therapies for multiple endocrine neoplasia type 1.

Degree: PhD, 2012, University of Oxford

 Multiple Endocrine Neoplasia Type 1 (MEN1) is an autosomal dominant disorder characterized by the combined occurrence of pituitary, pancreatic islet and parathyroid tumours. MEN1-associated tumours… (more)

Subjects/Keywords: 616.4; Medical Sciences; Biology (medical sciences); Endocrinology; Genetics (life sciences); endocrine; genetics

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Javid, M. (2012). Gene modifiers and novel therapies for multiple endocrine neoplasia type 1. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:baa5b96d-e186-4811-a867-945cee3c85eb ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604373

Chicago Manual of Style (16th Edition):

Javid, Mahsa. “Gene modifiers and novel therapies for multiple endocrine neoplasia type 1.” 2012. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:baa5b96d-e186-4811-a867-945cee3c85eb ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604373.

MLA Handbook (7th Edition):

Javid, Mahsa. “Gene modifiers and novel therapies for multiple endocrine neoplasia type 1.” 2012. Web. 29 Jan 2020.

Vancouver:

Javid M. Gene modifiers and novel therapies for multiple endocrine neoplasia type 1. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:baa5b96d-e186-4811-a867-945cee3c85eb ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604373.

Council of Science Editors:

Javid M. Gene modifiers and novel therapies for multiple endocrine neoplasia type 1. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:baa5b96d-e186-4811-a867-945cee3c85eb ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604373


University of Oxford

6. Boulding, Hannah. Identifying causative elements within structural variants associated with developmental disorders.

Degree: PhD, 2013, University of Oxford

 It has been well established that copy number variation contributes substantially to genetic variation within human populations. However, the extent to which de novo and… (more)

Subjects/Keywords: 616.858; Bioinformatics (life sciences); Genetics (life sciences); Genetics (medical sciences); genetics; genomics; CNVs

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Boulding, H. (2013). Identifying causative elements within structural variants associated with developmental disorders. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:d9af47cc-1c91-4a66-a6ac-86655f1ff375 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581323

Chicago Manual of Style (16th Edition):

Boulding, Hannah. “Identifying causative elements within structural variants associated with developmental disorders.” 2013. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:d9af47cc-1c91-4a66-a6ac-86655f1ff375 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581323.

MLA Handbook (7th Edition):

Boulding, Hannah. “Identifying causative elements within structural variants associated with developmental disorders.” 2013. Web. 29 Jan 2020.

Vancouver:

Boulding H. Identifying causative elements within structural variants associated with developmental disorders. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:d9af47cc-1c91-4a66-a6ac-86655f1ff375 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581323.

Council of Science Editors:

Boulding H. Identifying causative elements within structural variants associated with developmental disorders. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:d9af47cc-1c91-4a66-a6ac-86655f1ff375 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581323


University of Oxford

7. Davenport, Emma Elisabeth. Functional genomics of variation in response to infection : insights into severe sepsis and common variable immune deficiency disorders.

Degree: PhD, 2014, University of Oxford

 Functional genomics uses high throughput genome-wide technologies to investigate the functional consequences of genetic variants on gene expression and protein products. In the context of… (more)

Subjects/Keywords: 616.07; Genetics (medical sciences); Functional genomics; immunology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Davenport, E. E. (2014). Functional genomics of variation in response to infection : insights into severe sepsis and common variable immune deficiency disorders. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:528edf20-f948-4a9c-aa23-1e295b11c8cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.644672

Chicago Manual of Style (16th Edition):

Davenport, Emma Elisabeth. “Functional genomics of variation in response to infection : insights into severe sepsis and common variable immune deficiency disorders.” 2014. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:528edf20-f948-4a9c-aa23-1e295b11c8cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.644672.

MLA Handbook (7th Edition):

Davenport, Emma Elisabeth. “Functional genomics of variation in response to infection : insights into severe sepsis and common variable immune deficiency disorders.” 2014. Web. 29 Jan 2020.

Vancouver:

Davenport EE. Functional genomics of variation in response to infection : insights into severe sepsis and common variable immune deficiency disorders. [Internet] [Doctoral dissertation]. University of Oxford; 2014. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:528edf20-f948-4a9c-aa23-1e295b11c8cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.644672.

Council of Science Editors:

Davenport EE. Functional genomics of variation in response to infection : insights into severe sepsis and common variable immune deficiency disorders. [Doctoral Dissertation]. University of Oxford; 2014. Available from: http://ora.ox.ac.uk/objects/uuid:528edf20-f948-4a9c-aa23-1e295b11c8cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.644672


University of Oxford

8. Miller, Ruth Rosemary. Staphylococcus aureus : the host-organism relationship.

Degree: PhD, 2011, University of Oxford

 Staphylococcus aureus is a worldwide leading cause of skin and soft tissue, bone and joint, and bloodstream infection. Despite this, S. aureus is also a… (more)

Subjects/Keywords: 579.353; Epidemiology; Infectious diseases; Genetics (medical sciences); Medical Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Miller, R. R. (2011). Staphylococcus aureus : the host-organism relationship. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:e87e8eb4-65f4-47f4-a836-c68ba79259df ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555258

Chicago Manual of Style (16th Edition):

Miller, Ruth Rosemary. “Staphylococcus aureus : the host-organism relationship.” 2011. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:e87e8eb4-65f4-47f4-a836-c68ba79259df ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555258.

MLA Handbook (7th Edition):

Miller, Ruth Rosemary. “Staphylococcus aureus : the host-organism relationship.” 2011. Web. 29 Jan 2020.

Vancouver:

Miller RR. Staphylococcus aureus : the host-organism relationship. [Internet] [Doctoral dissertation]. University of Oxford; 2011. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:e87e8eb4-65f4-47f4-a836-c68ba79259df ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555258.

Council of Science Editors:

Miller RR. Staphylococcus aureus : the host-organism relationship. [Doctoral Dissertation]. University of Oxford; 2011. Available from: http://ora.ox.ac.uk/objects/uuid:e87e8eb4-65f4-47f4-a836-c68ba79259df ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555258


University of Oxford

9. Cheng, Timothy. Genetic susceptibility to endometrial cancer.

Degree: PhD, 2015, University of Oxford

 Endometrial cancer (EC) is the fourth most common cancer affecting women in the UK. Those with a family history of EC have an increased risk… (more)

Subjects/Keywords: 618.1; Medical Sciences; Genetics (medical sciences); endometrial cancer

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cheng, T. (2015). Genetic susceptibility to endometrial cancer. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:3a559ae0-156f-48a2-a64e-b03a13c562df ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.658527

Chicago Manual of Style (16th Edition):

Cheng, Timothy. “Genetic susceptibility to endometrial cancer.” 2015. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:3a559ae0-156f-48a2-a64e-b03a13c562df ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.658527.

MLA Handbook (7th Edition):

Cheng, Timothy. “Genetic susceptibility to endometrial cancer.” 2015. Web. 29 Jan 2020.

Vancouver:

Cheng T. Genetic susceptibility to endometrial cancer. [Internet] [Doctoral dissertation]. University of Oxford; 2015. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:3a559ae0-156f-48a2-a64e-b03a13c562df ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.658527.

Council of Science Editors:

Cheng T. Genetic susceptibility to endometrial cancer. [Doctoral Dissertation]. University of Oxford; 2015. Available from: http://ora.ox.ac.uk/objects/uuid:3a559ae0-156f-48a2-a64e-b03a13c562df ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.658527


Virginia Commonwealth University

10. Bigdeli, T. Bernard. Quantitative Genetic Methods to Dissect Heterogeneity in Complex Traits.

Degree: PhD, Human Genetics, 2012, Virginia Commonwealth University

 Etiological models of complex disease are elusive[46, 33, 9], as are consistently replicable findings for major genetic susceptibility loci[54, 14, 15, 24]. Commonly-cited explanations invoke… (more)

Subjects/Keywords: Medical Genetics; Medical Sciences; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bigdeli, T. B. (2012). Quantitative Genetic Methods to Dissect Heterogeneity in Complex Traits. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/2651

Chicago Manual of Style (16th Edition):

Bigdeli, T Bernard. “Quantitative Genetic Methods to Dissect Heterogeneity in Complex Traits.” 2012. Doctoral Dissertation, Virginia Commonwealth University. Accessed January 29, 2020. https://scholarscompass.vcu.edu/etd/2651.

MLA Handbook (7th Edition):

Bigdeli, T Bernard. “Quantitative Genetic Methods to Dissect Heterogeneity in Complex Traits.” 2012. Web. 29 Jan 2020.

Vancouver:

Bigdeli TB. Quantitative Genetic Methods to Dissect Heterogeneity in Complex Traits. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2012. [cited 2020 Jan 29]. Available from: https://scholarscompass.vcu.edu/etd/2651.

Council of Science Editors:

Bigdeli TB. Quantitative Genetic Methods to Dissect Heterogeneity in Complex Traits. [Doctoral Dissertation]. Virginia Commonwealth University; 2012. Available from: https://scholarscompass.vcu.edu/etd/2651


Virginia Commonwealth University

11. Gygax, Derek. Comprehensive Review on the Existence of Genomic Imprinting in Aves.

Degree: MS, Human Genetics, 2014, Virginia Commonwealth University

 Genomic imprinting results in monoallelic parent-of-origin gene expression. Therian mammals show conclusive evidence for imprinting, while the evidence in Aves is conflicting. It’s unclear if… (more)

Subjects/Keywords: Genomic Imprinting; Aves; Medical Genetics; Medical Sciences; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gygax, D. (2014). Comprehensive Review on the Existence of Genomic Imprinting in Aves. (Thesis). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/3375

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gygax, Derek. “Comprehensive Review on the Existence of Genomic Imprinting in Aves.” 2014. Thesis, Virginia Commonwealth University. Accessed January 29, 2020. https://scholarscompass.vcu.edu/etd/3375.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gygax, Derek. “Comprehensive Review on the Existence of Genomic Imprinting in Aves.” 2014. Web. 29 Jan 2020.

Vancouver:

Gygax D. Comprehensive Review on the Existence of Genomic Imprinting in Aves. [Internet] [Thesis]. Virginia Commonwealth University; 2014. [cited 2020 Jan 29]. Available from: https://scholarscompass.vcu.edu/etd/3375.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gygax D. Comprehensive Review on the Existence of Genomic Imprinting in Aves. [Thesis]. Virginia Commonwealth University; 2014. Available from: https://scholarscompass.vcu.edu/etd/3375

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

12. Mohamad, Safa. The Roles of Krüppel-Like Factor 1 (KLF1) in the Human Fetal Erythroid Compartment.

Degree: MS, Human Genetics, 2014, Virginia Commonwealth University

  Erythroid Krüppel-like factor (EKLF or KLF1) is a transcription factor with roles in embryonic and adult erythropoiesis. KLF1 knockout mouse embryos die due to… (more)

Subjects/Keywords: KLF1; erythroid; Medical Genetics; Medical Sciences; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mohamad, S. (2014). The Roles of Krüppel-Like Factor 1 (KLF1) in the Human Fetal Erythroid Compartment. (Thesis). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/587

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mohamad, Safa. “The Roles of Krüppel-Like Factor 1 (KLF1) in the Human Fetal Erythroid Compartment.” 2014. Thesis, Virginia Commonwealth University. Accessed January 29, 2020. https://scholarscompass.vcu.edu/etd/587.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mohamad, Safa. “The Roles of Krüppel-Like Factor 1 (KLF1) in the Human Fetal Erythroid Compartment.” 2014. Web. 29 Jan 2020.

Vancouver:

Mohamad S. The Roles of Krüppel-Like Factor 1 (KLF1) in the Human Fetal Erythroid Compartment. [Internet] [Thesis]. Virginia Commonwealth University; 2014. [cited 2020 Jan 29]. Available from: https://scholarscompass.vcu.edu/etd/587.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mohamad S. The Roles of Krüppel-Like Factor 1 (KLF1) in the Human Fetal Erythroid Compartment. [Thesis]. Virginia Commonwealth University; 2014. Available from: https://scholarscompass.vcu.edu/etd/587

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

13. Sokhi, Upneet K. Analyzing the functions of human polynucleotide phosphorylase (hPNPaseold-35).

Degree: PhD, Human Genetics, 2013, Virginia Commonwealth University

 RNA degradation plays a fundamental role in maintaining cellular homeostasis, along with being a part of normal regulatory mechanisms, whether it occurs as a surveillance… (more)

Subjects/Keywords: ribonuclease; microarray; Medical Genetics; Medical Sciences; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sokhi, U. K. (2013). Analyzing the functions of human polynucleotide phosphorylase (hPNPaseold-35). (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/568

Chicago Manual of Style (16th Edition):

Sokhi, Upneet K. “Analyzing the functions of human polynucleotide phosphorylase (hPNPaseold-35).” 2013. Doctoral Dissertation, Virginia Commonwealth University. Accessed January 29, 2020. https://scholarscompass.vcu.edu/etd/568.

MLA Handbook (7th Edition):

Sokhi, Upneet K. “Analyzing the functions of human polynucleotide phosphorylase (hPNPaseold-35).” 2013. Web. 29 Jan 2020.

Vancouver:

Sokhi UK. Analyzing the functions of human polynucleotide phosphorylase (hPNPaseold-35). [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2013. [cited 2020 Jan 29]. Available from: https://scholarscompass.vcu.edu/etd/568.

Council of Science Editors:

Sokhi UK. Analyzing the functions of human polynucleotide phosphorylase (hPNPaseold-35). [Doctoral Dissertation]. Virginia Commonwealth University; 2013. Available from: https://scholarscompass.vcu.edu/etd/568


Virginia Commonwealth University

14. Alhareeri, Areej. Chromosome-Specific Telomere Length in Women with Breast Cancer: Their Relationship to Chemotherapy and Acquired Psychoneurological Symptoms.

Degree: MS, Human Genetics, 2013, Virginia Commonwealth University

 Breast cancer (BC) is one of the most common diagnosed malignancies in females. Although 90% of early diagnosed women are expected to survive for at… (more)

Subjects/Keywords: Medical Genetics; Medical Sciences; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Alhareeri, A. (2013). Chromosome-Specific Telomere Length in Women with Breast Cancer: Their Relationship to Chemotherapy and Acquired Psychoneurological Symptoms. (Thesis). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/475

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alhareeri, Areej. “Chromosome-Specific Telomere Length in Women with Breast Cancer: Their Relationship to Chemotherapy and Acquired Psychoneurological Symptoms.” 2013. Thesis, Virginia Commonwealth University. Accessed January 29, 2020. https://scholarscompass.vcu.edu/etd/475.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alhareeri, Areej. “Chromosome-Specific Telomere Length in Women with Breast Cancer: Their Relationship to Chemotherapy and Acquired Psychoneurological Symptoms.” 2013. Web. 29 Jan 2020.

Vancouver:

Alhareeri A. Chromosome-Specific Telomere Length in Women with Breast Cancer: Their Relationship to Chemotherapy and Acquired Psychoneurological Symptoms. [Internet] [Thesis]. Virginia Commonwealth University; 2013. [cited 2020 Jan 29]. Available from: https://scholarscompass.vcu.edu/etd/475.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alhareeri A. Chromosome-Specific Telomere Length in Women with Breast Cancer: Their Relationship to Chemotherapy and Acquired Psychoneurological Symptoms. [Thesis]. Virginia Commonwealth University; 2013. Available from: https://scholarscompass.vcu.edu/etd/475

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

15. Yaseen, Alae Abod. THE NATURAL POLYPHENOL RESVERATROL POTENTIATES THE LETHALITY OF HDAC INHIBITORS IN ACUTE MYELOGENOUS LEUKEMIA CELLS THROUGH MULTIPLE MECHANISMS.

Degree: MS, Human Genetics, 2011, Virginia Commonwealth University

 This study examined the mechanisms underlying the interactions between the natural polyphenol Resveratrol and HDAC inhibitors in both U937 myelomonocytic leukemia cell line and blood… (more)

Subjects/Keywords: Resveratrol HDACi Leukemia; Medical Genetics; Medical Sciences; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yaseen, A. A. (2011). THE NATURAL POLYPHENOL RESVERATROL POTENTIATES THE LETHALITY OF HDAC INHIBITORS IN ACUTE MYELOGENOUS LEUKEMIA CELLS THROUGH MULTIPLE MECHANISMS. (Thesis). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/2519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yaseen, Alae Abod. “THE NATURAL POLYPHENOL RESVERATROL POTENTIATES THE LETHALITY OF HDAC INHIBITORS IN ACUTE MYELOGENOUS LEUKEMIA CELLS THROUGH MULTIPLE MECHANISMS.” 2011. Thesis, Virginia Commonwealth University. Accessed January 29, 2020. https://scholarscompass.vcu.edu/etd/2519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yaseen, Alae Abod. “THE NATURAL POLYPHENOL RESVERATROL POTENTIATES THE LETHALITY OF HDAC INHIBITORS IN ACUTE MYELOGENOUS LEUKEMIA CELLS THROUGH MULTIPLE MECHANISMS.” 2011. Web. 29 Jan 2020.

Vancouver:

Yaseen AA. THE NATURAL POLYPHENOL RESVERATROL POTENTIATES THE LETHALITY OF HDAC INHIBITORS IN ACUTE MYELOGENOUS LEUKEMIA CELLS THROUGH MULTIPLE MECHANISMS. [Internet] [Thesis]. Virginia Commonwealth University; 2011. [cited 2020 Jan 29]. Available from: https://scholarscompass.vcu.edu/etd/2519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yaseen AA. THE NATURAL POLYPHENOL RESVERATROL POTENTIATES THE LETHALITY OF HDAC INHIBITORS IN ACUTE MYELOGENOUS LEUKEMIA CELLS THROUGH MULTIPLE MECHANISMS. [Thesis]. Virginia Commonwealth University; 2011. Available from: https://scholarscompass.vcu.edu/etd/2519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

16. Luedders, Jonathan. A Review of Common and Rare Genetic Variants in Schizophrenia.

Degree: MS, Human Genetics, 2011, Virginia Commonwealth University

 Genetic epidemiology has shown a large role for genetic influences on schizophrenia. However, the nature of the variants involved is debated. The common disease-common variant… (more)

Subjects/Keywords: schizophrenia; genetic; Medical Genetics; Medical Sciences; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Luedders, J. (2011). A Review of Common and Rare Genetic Variants in Schizophrenia. (Thesis). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/2540

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Luedders, Jonathan. “A Review of Common and Rare Genetic Variants in Schizophrenia.” 2011. Thesis, Virginia Commonwealth University. Accessed January 29, 2020. https://scholarscompass.vcu.edu/etd/2540.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Luedders, Jonathan. “A Review of Common and Rare Genetic Variants in Schizophrenia.” 2011. Web. 29 Jan 2020.

Vancouver:

Luedders J. A Review of Common and Rare Genetic Variants in Schizophrenia. [Internet] [Thesis]. Virginia Commonwealth University; 2011. [cited 2020 Jan 29]. Available from: https://scholarscompass.vcu.edu/etd/2540.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Luedders J. A Review of Common and Rare Genetic Variants in Schizophrenia. [Thesis]. Virginia Commonwealth University; 2011. Available from: https://scholarscompass.vcu.edu/etd/2540

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

17. Vachtsevanos, Athanasios. Probing the molecular basis of melanopsin induced light sensitivity.

Degree: PhD, 2012, University of Oxford

 It has been demonstrated that retinal photoreception among mammals extends beyond rods and cones to include a small number of intrinsically photosensitive retinal ganglion cells… (more)

Subjects/Keywords: 617.7; Medical Sciences; Clinical laboratory sciences; Clinical genetics; Immunodiagnostics; Biology (medical sciences); Genetics (medical sciences); Ophthamology; ophthalmology; melanospin; siRNA; pupillometry

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vachtsevanos, A. (2012). Probing the molecular basis of melanopsin induced light sensitivity. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:631deeeb-90c0-4e90-b24e-f03e1b318d8b ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627770

Chicago Manual of Style (16th Edition):

Vachtsevanos, Athanasios. “Probing the molecular basis of melanopsin induced light sensitivity.” 2012. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:631deeeb-90c0-4e90-b24e-f03e1b318d8b ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627770.

MLA Handbook (7th Edition):

Vachtsevanos, Athanasios. “Probing the molecular basis of melanopsin induced light sensitivity.” 2012. Web. 29 Jan 2020.

Vancouver:

Vachtsevanos A. Probing the molecular basis of melanopsin induced light sensitivity. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:631deeeb-90c0-4e90-b24e-f03e1b318d8b ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627770.

Council of Science Editors:

Vachtsevanos A. Probing the molecular basis of melanopsin induced light sensitivity. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:631deeeb-90c0-4e90-b24e-f03e1b318d8b ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627770


University of Oxford

18. Chamas, Liliane. The nicotinic acid receptor in human adipose tissue.

Degree: PhD, 2013, University of Oxford

 Nicotinic acid (NA) has been clinically used for over 50 years to regulate lipid plasma levels. It is the only drug in current clinical use… (more)

Subjects/Keywords: 611; Genetics (life sciences); Metabolism; Medical Sciences; Cardiovascular disease; Genetics (medical sciences); niacin; nicotinic acid receptor; genetics; polymorphisms; human; adipose tissue

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chamas, L. (2013). The nicotinic acid receptor in human adipose tissue. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:82650691-be95-4cf7-a3cd-e0821062f49d ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604411

Chicago Manual of Style (16th Edition):

Chamas, Liliane. “The nicotinic acid receptor in human adipose tissue.” 2013. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:82650691-be95-4cf7-a3cd-e0821062f49d ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604411.

MLA Handbook (7th Edition):

Chamas, Liliane. “The nicotinic acid receptor in human adipose tissue.” 2013. Web. 29 Jan 2020.

Vancouver:

Chamas L. The nicotinic acid receptor in human adipose tissue. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:82650691-be95-4cf7-a3cd-e0821062f49d ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604411.

Council of Science Editors:

Chamas L. The nicotinic acid receptor in human adipose tissue. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:82650691-be95-4cf7-a3cd-e0821062f49d ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604411


University of Oxford

19. Shah, Shivang Satish. Examining the relationship between genetic variation at G6PD and severe malaria.

Degree: PhD, 2011, University of Oxford

 Glucose-6-phosphate dehydrogenase (G6PD) deficiency, a common heritable trait whose prevalence mirrors geographic patterns of historic malaria endemicity, is thought to confer a selective advantage owing… (more)

Subjects/Keywords: 614.532; Medical sciences; Biology (medical sciences); Genetics (medical sciences); Malaria; Infectious diseases; Tropical medicine; genetics; glucose-6-phosphate dehydrogenase

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shah, S. S. (2011). Examining the relationship between genetic variation at G6PD and severe malaria. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:933063af-9cd9-4f74-94c9-55ef2db28551 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572473

Chicago Manual of Style (16th Edition):

Shah, Shivang Satish. “Examining the relationship between genetic variation at G6PD and severe malaria.” 2011. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:933063af-9cd9-4f74-94c9-55ef2db28551 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572473.

MLA Handbook (7th Edition):

Shah, Shivang Satish. “Examining the relationship between genetic variation at G6PD and severe malaria.” 2011. Web. 29 Jan 2020.

Vancouver:

Shah SS. Examining the relationship between genetic variation at G6PD and severe malaria. [Internet] [Doctoral dissertation]. University of Oxford; 2011. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:933063af-9cd9-4f74-94c9-55ef2db28551 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572473.

Council of Science Editors:

Shah SS. Examining the relationship between genetic variation at G6PD and severe malaria. [Doctoral Dissertation]. University of Oxford; 2011. Available from: http://ora.ox.ac.uk/objects/uuid:933063af-9cd9-4f74-94c9-55ef2db28551 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572473


Texas Medical Center

20. Penney, Samantha. Evaluation of Knowledge Regarding Diagnostic Strategies for Genetic Diseases in Select Residents.

Degree: MS, 2012, Texas Medical Center

Genetics education for physicians has been a popular publication topic in the United States and in Europe for over 20 years. Decreasing numbers of… (more)

Subjects/Keywords: CMA; resident knowledge; genetics education; medical genetics; microarray; genetic testing; clinical genetics primary care; genetics; pediatrics; physician knowledge; medical education; Medical Education; Medical Genetics; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Penney, S. (2012). Evaluation of Knowledge Regarding Diagnostic Strategies for Genetic Diseases in Select Residents. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/257

Chicago Manual of Style (16th Edition):

Penney, Samantha. “Evaluation of Knowledge Regarding Diagnostic Strategies for Genetic Diseases in Select Residents.” 2012. Masters Thesis, Texas Medical Center. Accessed January 29, 2020. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/257.

MLA Handbook (7th Edition):

Penney, Samantha. “Evaluation of Knowledge Regarding Diagnostic Strategies for Genetic Diseases in Select Residents.” 2012. Web. 29 Jan 2020.

Vancouver:

Penney S. Evaluation of Knowledge Regarding Diagnostic Strategies for Genetic Diseases in Select Residents. [Internet] [Masters thesis]. Texas Medical Center; 2012. [cited 2020 Jan 29]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/257.

Council of Science Editors:

Penney S. Evaluation of Knowledge Regarding Diagnostic Strategies for Genetic Diseases in Select Residents. [Masters Thesis]. Texas Medical Center; 2012. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/257


Virginia Commonwealth University

21. Ray, Christopher. Pediatrician Perceptions of the Patient-Centered Medical Home Model.

Degree: MS, Human Genetics, 2011, Virginia Commonwealth University

  The Patient-Centered Medical Home (PCMH) is an emerging model of health care designed to provide a simpler, more effective health care experience. The model… (more)

Subjects/Keywords: Medical Home; Health Care Policy; Medical Genetics; Medical Sciences; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ray, C. (2011). Pediatrician Perceptions of the Patient-Centered Medical Home Model. (Thesis). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/211

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ray, Christopher. “Pediatrician Perceptions of the Patient-Centered Medical Home Model.” 2011. Thesis, Virginia Commonwealth University. Accessed January 29, 2020. https://scholarscompass.vcu.edu/etd/211.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ray, Christopher. “Pediatrician Perceptions of the Patient-Centered Medical Home Model.” 2011. Web. 29 Jan 2020.

Vancouver:

Ray C. Pediatrician Perceptions of the Patient-Centered Medical Home Model. [Internet] [Thesis]. Virginia Commonwealth University; 2011. [cited 2020 Jan 29]. Available from: https://scholarscompass.vcu.edu/etd/211.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ray C. Pediatrician Perceptions of the Patient-Centered Medical Home Model. [Thesis]. Virginia Commonwealth University; 2011. Available from: https://scholarscompass.vcu.edu/etd/211

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

22. McCulloch, Laura Jade. The molecular genetics of insulin secretion and signalling.

Degree: PhD, 2011, University of Oxford

 Type 2 diabetes (T2D) and fasting plasma glucose (fpg) levels have distinct genetic components which are as yet only modestly understood. Understanding the genetics of… (more)

Subjects/Keywords: 616.4624; Diabetes; Genetics (medical sciences); Type 2 diabetes; monogenic diabetes; genetics

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McCulloch, L. J. (2011). The molecular genetics of insulin secretion and signalling. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:eb170e91-7b3c-453f-af58-7058909de435 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558455

Chicago Manual of Style (16th Edition):

McCulloch, Laura Jade. “The molecular genetics of insulin secretion and signalling.” 2011. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:eb170e91-7b3c-453f-af58-7058909de435 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558455.

MLA Handbook (7th Edition):

McCulloch, Laura Jade. “The molecular genetics of insulin secretion and signalling.” 2011. Web. 29 Jan 2020.

Vancouver:

McCulloch LJ. The molecular genetics of insulin secretion and signalling. [Internet] [Doctoral dissertation]. University of Oxford; 2011. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:eb170e91-7b3c-453f-af58-7058909de435 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558455.

Council of Science Editors:

McCulloch LJ. The molecular genetics of insulin secretion and signalling. [Doctoral Dissertation]. University of Oxford; 2011. Available from: http://ora.ox.ac.uk/objects/uuid:eb170e91-7b3c-453f-af58-7058909de435 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558455


University of Oxford

23. Royrvik, E. C. The peoples of Britain : population genetics, archaeology and linguistics.

Degree: PhD, 2012, University of Oxford

 The history of peoples has always evoked a great deal of both academic and popular interest, and the peoples of Britain, with its island position… (more)

Subjects/Keywords: 304.6072; Genetics (medical sciences); population; genetics; Britain; archaeology; history; demography

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Royrvik, E. C. (2012). The peoples of Britain : population genetics, archaeology and linguistics. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:c25a230b-7cdd-46ab-977c-95a5b81875b4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581106

Chicago Manual of Style (16th Edition):

Royrvik, E C. “The peoples of Britain : population genetics, archaeology and linguistics.” 2012. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:c25a230b-7cdd-46ab-977c-95a5b81875b4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581106.

MLA Handbook (7th Edition):

Royrvik, E C. “The peoples of Britain : population genetics, archaeology and linguistics.” 2012. Web. 29 Jan 2020.

Vancouver:

Royrvik EC. The peoples of Britain : population genetics, archaeology and linguistics. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:c25a230b-7cdd-46ab-977c-95a5b81875b4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581106.

Council of Science Editors:

Royrvik EC. The peoples of Britain : population genetics, archaeology and linguistics. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:c25a230b-7cdd-46ab-977c-95a5b81875b4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581106


University of Oxford

24. Rees, Matthew Geoffrey. Genetic, functional, and phenotypic analysis of human variants in the glucokinase regulatory protein gene.

Degree: PhD, 2012, University of Oxford

 Genome-wide association (GWA) studies have provided significant insight into the underlying genetic components of common human diseases such as type 2 diabetes (T2D). However, the… (more)

Subjects/Keywords: 616.4624; Clinical genetics; Biology (medical sciences); Biochemistry; High-Throughput Screening; Genetics (medical sciences); Metabolism; Molecular genetics; Enzymes; Chemical biology; Diabetes

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rees, M. G. (2012). Genetic, functional, and phenotypic analysis of human variants in the glucokinase regulatory protein gene. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:e1ccbfb3-45bf-4830-85a4-5d7c88599945 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589604

Chicago Manual of Style (16th Edition):

Rees, Matthew Geoffrey. “Genetic, functional, and phenotypic analysis of human variants in the glucokinase regulatory protein gene.” 2012. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:e1ccbfb3-45bf-4830-85a4-5d7c88599945 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589604.

MLA Handbook (7th Edition):

Rees, Matthew Geoffrey. “Genetic, functional, and phenotypic analysis of human variants in the glucokinase regulatory protein gene.” 2012. Web. 29 Jan 2020.

Vancouver:

Rees MG. Genetic, functional, and phenotypic analysis of human variants in the glucokinase regulatory protein gene. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:e1ccbfb3-45bf-4830-85a4-5d7c88599945 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589604.

Council of Science Editors:

Rees MG. Genetic, functional, and phenotypic analysis of human variants in the glucokinase regulatory protein gene. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:e1ccbfb3-45bf-4830-85a4-5d7c88599945 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589604


University of Oxford

25. Beer, Nicola L. The role of genetic variation in glucokinase and glucokinase regulatory protein in diabetes and related traits.

Degree: PhD, 2011, University of Oxford

 The rising prevalence of type 2 diabetes (T2D) is a global problem, and suggests that we need better therapeutic strategies against this disease. The glycolytic… (more)

Subjects/Keywords: 616.3; Endocrinology; Biochemistry; Metabolism; Medical Sciences; Clinical genetics; Diabetes; Genetics (medical sciences); diabetes; metabolism; monogenic disease; genetics; glucokinase; glucokinase regulatory protein

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Beer, N. L. (2011). The role of genetic variation in glucokinase and glucokinase regulatory protein in diabetes and related traits. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:87f8ea0d-9528-49fd-8f01-5f976cf9f210 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.543051

Chicago Manual of Style (16th Edition):

Beer, Nicola L. “The role of genetic variation in glucokinase and glucokinase regulatory protein in diabetes and related traits.” 2011. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:87f8ea0d-9528-49fd-8f01-5f976cf9f210 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.543051.

MLA Handbook (7th Edition):

Beer, Nicola L. “The role of genetic variation in glucokinase and glucokinase regulatory protein in diabetes and related traits.” 2011. Web. 29 Jan 2020.

Vancouver:

Beer NL. The role of genetic variation in glucokinase and glucokinase regulatory protein in diabetes and related traits. [Internet] [Doctoral dissertation]. University of Oxford; 2011. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:87f8ea0d-9528-49fd-8f01-5f976cf9f210 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.543051.

Council of Science Editors:

Beer NL. The role of genetic variation in glucokinase and glucokinase regulatory protein in diabetes and related traits. [Doctoral Dissertation]. University of Oxford; 2011. Available from: http://ora.ox.ac.uk/objects/uuid:87f8ea0d-9528-49fd-8f01-5f976cf9f210 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.543051


University of Oxford

26. Jafar-Mohammadi, Bahram. The evaluation of the contribution of low frequency, intermediate penetrance sequence variants to the pathogenesis of Type 2 Diabetes.

Degree: PhD, 2012, University of Oxford

 Genome wide association studies (GWAS) and their subsequent meta-analysis have identified a large number of susceptibility variants for Type 2 diabetes (T2D) risk. However, the… (more)

Subjects/Keywords: 616.4624; Diabetes; Clinical genetics; Genetics (medical sciences); type 2 diabetes; genetics; low frequency variants

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jafar-Mohammadi, B. (2012). The evaluation of the contribution of low frequency, intermediate penetrance sequence variants to the pathogenesis of Type 2 Diabetes. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:809d4270-3a74-4fea-b696-2f619bfb7100 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581046

Chicago Manual of Style (16th Edition):

Jafar-Mohammadi, Bahram. “The evaluation of the contribution of low frequency, intermediate penetrance sequence variants to the pathogenesis of Type 2 Diabetes.” 2012. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:809d4270-3a74-4fea-b696-2f619bfb7100 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581046.

MLA Handbook (7th Edition):

Jafar-Mohammadi, Bahram. “The evaluation of the contribution of low frequency, intermediate penetrance sequence variants to the pathogenesis of Type 2 Diabetes.” 2012. Web. 29 Jan 2020.

Vancouver:

Jafar-Mohammadi B. The evaluation of the contribution of low frequency, intermediate penetrance sequence variants to the pathogenesis of Type 2 Diabetes. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:809d4270-3a74-4fea-b696-2f619bfb7100 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581046.

Council of Science Editors:

Jafar-Mohammadi B. The evaluation of the contribution of low frequency, intermediate penetrance sequence variants to the pathogenesis of Type 2 Diabetes. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:809d4270-3a74-4fea-b696-2f619bfb7100 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581046


University of Oxford

27. Gregory, Adam Patrick. Functional characterisation of a multiple sclerosis-associated genetic variant.

Degree: PhD, 2012, University of Oxford

 In recent years, Genome-wide association studies (GWAS) have identified several susceptibility-modifying genetic variants to complex diseases such as multiple sclerosis (MS). An association for one… (more)

Subjects/Keywords: 616.834042; Genetics (life sciences); Life Sciences; Immunology; Gene medicine; Genetics (medical sciences); Biology; Medical Sciences; Clinical genetics; Biology (medical sciences); Neuroscience; Multiple Sclerosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gregory, A. P. (2012). Functional characterisation of a multiple sclerosis-associated genetic variant. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:315e39e4-548f-430b-8d54-68094c340a73 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572514

Chicago Manual of Style (16th Edition):

Gregory, Adam Patrick. “Functional characterisation of a multiple sclerosis-associated genetic variant.” 2012. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:315e39e4-548f-430b-8d54-68094c340a73 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572514.

MLA Handbook (7th Edition):

Gregory, Adam Patrick. “Functional characterisation of a multiple sclerosis-associated genetic variant.” 2012. Web. 29 Jan 2020.

Vancouver:

Gregory AP. Functional characterisation of a multiple sclerosis-associated genetic variant. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:315e39e4-548f-430b-8d54-68094c340a73 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572514.

Council of Science Editors:

Gregory AP. Functional characterisation of a multiple sclerosis-associated genetic variant. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:315e39e4-548f-430b-8d54-68094c340a73 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572514


University of Helsinki

28. Vlachopoulou, Efthymia. HLA-DRB1: Haplotype Diversity, Imputation and Association with Acute Coronary Syndrome.

Degree: Haartman Institute, 2015, University of Helsinki

The Human Leukocyte Antigen (HLA) region is located on chromosome 6 (6p21.3) and its main function is to regulate the immune system. This region has… (more)

Subjects/Keywords: Medical Genetics; Medical Genetics

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vlachopoulou, E. (2015). HLA-DRB1: Haplotype Diversity, Imputation and Association with Acute Coronary Syndrome. (Doctoral Dissertation). University of Helsinki. Retrieved from http://hdl.handle.net/10138/156247

Chicago Manual of Style (16th Edition):

Vlachopoulou, Efthymia. “HLA-DRB1: Haplotype Diversity, Imputation and Association with Acute Coronary Syndrome.” 2015. Doctoral Dissertation, University of Helsinki. Accessed January 29, 2020. http://hdl.handle.net/10138/156247.

MLA Handbook (7th Edition):

Vlachopoulou, Efthymia. “HLA-DRB1: Haplotype Diversity, Imputation and Association with Acute Coronary Syndrome.” 2015. Web. 29 Jan 2020.

Vancouver:

Vlachopoulou E. HLA-DRB1: Haplotype Diversity, Imputation and Association with Acute Coronary Syndrome. [Internet] [Doctoral dissertation]. University of Helsinki; 2015. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/10138/156247.

Council of Science Editors:

Vlachopoulou E. HLA-DRB1: Haplotype Diversity, Imputation and Association with Acute Coronary Syndrome. [Doctoral Dissertation]. University of Helsinki; 2015. Available from: http://hdl.handle.net/10138/156247


University of Oxford

29. Travers, Mary E. Mechanisms of Type 2 diabetes susceptibility.

Degree: PhD, 2013, University of Oxford

 Type 2 diabetes (T2D) has a genetic component which is only partially understood. The majority of genetic variance in disease susceptibility is unaccounted for, whilst… (more)

Subjects/Keywords: 616.4; Endocrinology; Genetics (life sciences); Medical Sciences; Biology (medical sciences); Diabetes; Genetics (medical sciences); Type 2 Diabetes; Complex Disease Genetics; Genome-wide association

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Travers, M. E. (2013). Mechanisms of Type 2 diabetes susceptibility. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:d99892d8-534a-4908-b5dc-ab1d8b1cab52 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581219

Chicago Manual of Style (16th Edition):

Travers, Mary E. “Mechanisms of Type 2 diabetes susceptibility.” 2013. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:d99892d8-534a-4908-b5dc-ab1d8b1cab52 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581219.

MLA Handbook (7th Edition):

Travers, Mary E. “Mechanisms of Type 2 diabetes susceptibility.” 2013. Web. 29 Jan 2020.

Vancouver:

Travers ME. Mechanisms of Type 2 diabetes susceptibility. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:d99892d8-534a-4908-b5dc-ab1d8b1cab52 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581219.

Council of Science Editors:

Travers ME. Mechanisms of Type 2 diabetes susceptibility. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:d99892d8-534a-4908-b5dc-ab1d8b1cab52 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581219


University of Oxford

30. Curtis, Helen J. Developing gene knockdown-replacement therapies for spinocerebellar ataxia type 7.

Degree: PhD, 2013, University of Oxford

 For many dominant diseases, conventional treatment options are limited. This makes them attractive candidates for gene therapy, which may be directed to specifically silence a… (more)

Subjects/Keywords: 616.8; Genetics (life sciences); Gene medicine; Genetics (medical sciences); Biology; Biology (medical sciences); Mirtrons; Gene Therapy; Neurodegenerative disease; miRNA; Spinocerebellar Ataxia

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Curtis, H. J. (2013). Developing gene knockdown-replacement therapies for spinocerebellar ataxia type 7. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:9e0570b6-f811-473a-a007-185fc8c77fc6 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.639960

Chicago Manual of Style (16th Edition):

Curtis, Helen J. “Developing gene knockdown-replacement therapies for spinocerebellar ataxia type 7.” 2013. Doctoral Dissertation, University of Oxford. Accessed January 29, 2020. http://ora.ox.ac.uk/objects/uuid:9e0570b6-f811-473a-a007-185fc8c77fc6 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.639960.

MLA Handbook (7th Edition):

Curtis, Helen J. “Developing gene knockdown-replacement therapies for spinocerebellar ataxia type 7.” 2013. Web. 29 Jan 2020.

Vancouver:

Curtis HJ. Developing gene knockdown-replacement therapies for spinocerebellar ataxia type 7. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2020 Jan 29]. Available from: http://ora.ox.ac.uk/objects/uuid:9e0570b6-f811-473a-a007-185fc8c77fc6 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.639960.

Council of Science Editors:

Curtis HJ. Developing gene knockdown-replacement therapies for spinocerebellar ataxia type 7. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:9e0570b6-f811-473a-a007-185fc8c77fc6 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.639960

[1] [2] [3] [4] [5] … [6274]

.