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You searched for subject:(Genetic transcription 60). Showing records 1 – 11 of 11 total matches.

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1. Booker, Betty M. Analyzing DNA topology and transcription in Salmonella enterica serovar Typhimurium during dichotomous growth.

Degree: PhD, 2010, University of Alabama – Birmingham

The bacterial chromosome is dynamic. The principle goal of my research is to understand how DNA topology is altered by transcription in Salmonella enterica serovar… (more)

Subjects/Keywords: Chromosomes<; br>; DNA<; br>; RNA, Ribosomal<; br>; Salmonella typhimurium  – genetics<; br>; Transcription, Genetic

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APA (6th Edition):

Booker, B. M. (2010). Analyzing DNA topology and transcription in Salmonella enterica serovar Typhimurium during dichotomous growth. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,699

Chicago Manual of Style (16th Edition):

Booker, Betty M. “Analyzing DNA topology and transcription in Salmonella enterica serovar Typhimurium during dichotomous growth.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,699.

MLA Handbook (7th Edition):

Booker, Betty M. “Analyzing DNA topology and transcription in Salmonella enterica serovar Typhimurium during dichotomous growth.” 2010. Web. 19 Oct 2019.

Vancouver:

Booker BM. Analyzing DNA topology and transcription in Salmonella enterica serovar Typhimurium during dichotomous growth. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,699.

Council of Science Editors:

Booker BM. Analyzing DNA topology and transcription in Salmonella enterica serovar Typhimurium during dichotomous growth. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,699

2. He, Ti. Molecular regulation of Pax5-mediated biological functions.

Degree: PhD, 2008, University of Alabama – Birmingham

B lineage cells are major players in the adaptive immune system. Pax5 is essential for B lineage cell development and function. Pax5 controls B lineage… (more)

Subjects/Keywords: B-Cell-Specific Activator Protein <; br>; Gene Expression Regulation <; br>; Histone Acetyltransferases  – metabolism <; br>; Transcription Factors <; br>; Transcription, Genetic

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APA (6th Edition):

He, T. (2008). Molecular regulation of Pax5-mediated biological functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,329

Chicago Manual of Style (16th Edition):

He, Ti. “Molecular regulation of Pax5-mediated biological functions.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,329.

MLA Handbook (7th Edition):

He, Ti. “Molecular regulation of Pax5-mediated biological functions.” 2008. Web. 19 Oct 2019.

Vancouver:

He T. Molecular regulation of Pax5-mediated biological functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,329.

Council of Science Editors:

He T. Molecular regulation of Pax5-mediated biological functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,329

3. Ho, Shiuh-Rong. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.

Degree: PhD, 2010, University of Alabama – Birmingham

O-GlcNAcylation is an abundant and dynamic post-translational modification on serine and threonine residues of nuclear and cytoplasmic proteins. O-GlcNAc Transferase (OGT) and Nuclear Cytoplasmic O-GlcNAcase… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism<; br>; Gene Expression Regulation<; br>; Oxidative Stress  – genetics<; br>; Transcription Factors  – metabolism<; br>; Transcription, Genetic

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APA (6th Edition):

Ho, S. (2010). O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1162

Chicago Manual of Style (16th Edition):

Ho, Shiuh-Rong. “O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1162.

MLA Handbook (7th Edition):

Ho, Shiuh-Rong. “O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.” 2010. Web. 19 Oct 2019.

Vancouver:

Ho S. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1162.

Council of Science Editors:

Ho S. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1162

4. Phipps, Sharla Marion Ostein. Genetic and epigenetic modulation of telomerase activity in development and disease.

Degree: PhD, 2007, University of Alabama – Birmingham

The end replication problem of linear chromosomes leads to the erosion of telomeric DNA with each cell division. A mechanism to counteract telomeric attrition involves… (more)

Subjects/Keywords: Telomerase  – Physiological effect <; br>; Telomerase  – Inhibitors <; br>; Genetic regulation <; br>; Enzymes  – Regulation <; br>; Genetic transcription <; br>; Reverse transcriptase <; br>; Breast  – Cancer  – Genetic aspects <; br>; Tretinoin  – Physiological effect <; br>; Epigenesis.

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APA (6th Edition):

Phipps, S. M. O. (2007). Genetic and epigenetic modulation of telomerase activity in development and disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,260

Chicago Manual of Style (16th Edition):

Phipps, Sharla Marion Ostein. “Genetic and epigenetic modulation of telomerase activity in development and disease.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,260.

MLA Handbook (7th Edition):

Phipps, Sharla Marion Ostein. “Genetic and epigenetic modulation of telomerase activity in development and disease.” 2007. Web. 19 Oct 2019.

Vancouver:

Phipps SMO. Genetic and epigenetic modulation of telomerase activity in development and disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,260.

Council of Science Editors:

Phipps SMO. Genetic and epigenetic modulation of telomerase activity in development and disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,260

5. Li, Xingnan. Regulation of [beta]-catenin by Gli1 in epithelial transformation.

Degree: PhD, 2006, University of Alabama – Birmingham

Gli family members-mediated continuous Hedgehog (Hh) pathway activity plays a role in the growth of a number of human cancers, including the common malignancy of… (more)

Subjects/Keywords: beta Catenin  – metabolism <; br>; Cell Transformation, Neoplastic  – genetics <; br>; Oncogene Proteins <; br>; Trans-Activators  – genetics <; br>; Transcription, Genetic

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APA (6th Edition):

Li, X. (2006). Regulation of [beta]-catenin by Gli1 in epithelial transformation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,90

Chicago Manual of Style (16th Edition):

Li, Xingnan. “Regulation of [beta]-catenin by Gli1 in epithelial transformation.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,90.

MLA Handbook (7th Edition):

Li, Xingnan. “Regulation of [beta]-catenin by Gli1 in epithelial transformation.” 2006. Web. 19 Oct 2019.

Vancouver:

Li X. Regulation of [beta]-catenin by Gli1 in epithelial transformation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,90.

Council of Science Editors:

Li X. Regulation of [beta]-catenin by Gli1 in epithelial transformation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,90

6. Luther, Rita Jeanne. Development and implementation of knock-in and BAC-in IL-2 reporter mouse models to characterize IL2 gene regulation in CD4 T cells.

Degree: PhD, 2009, University of Alabama – Birmingham

Maintenance of immune homeostasis involves a balance between T cell effector responses to antigen stimulus and reciprocal downregulation of this response through peripheral tolerance mechanisms.… (more)

Subjects/Keywords: Immune Tolerance<; br>; Interleukin-2<; br>; Mice<; br>; T-Lymphocytes, Regulatory<; br>; Transcription, Genetic

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APA (6th Edition):

Luther, R. J. (2009). Development and implementation of knock-in and BAC-in IL-2 reporter mouse models to characterize IL2 gene regulation in CD4 T cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1097

Chicago Manual of Style (16th Edition):

Luther, Rita Jeanne. “Development and implementation of knock-in and BAC-in IL-2 reporter mouse models to characterize IL2 gene regulation in CD4 T cells.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1097.

MLA Handbook (7th Edition):

Luther, Rita Jeanne. “Development and implementation of knock-in and BAC-in IL-2 reporter mouse models to characterize IL2 gene regulation in CD4 T cells.” 2009. Web. 19 Oct 2019.

Vancouver:

Luther RJ. Development and implementation of knock-in and BAC-in IL-2 reporter mouse models to characterize IL2 gene regulation in CD4 T cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1097.

Council of Science Editors:

Luther RJ. Development and implementation of knock-in and BAC-in IL-2 reporter mouse models to characterize IL2 gene regulation in CD4 T cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1097

7. Jennings, Jessica Amber. Regulation of gene expression in response to continuous low Intensity direct current electrical fields.

Degree: PhD, 2007, University of Alabama – Birmingham

In the normal process of wound healing, small electric fields are generated at the wound site due to a disturbance of the potential difference maintained… (more)

Subjects/Keywords: Skin  – Wounds and injuries  – Treatment <; br>; Genetic regulation <; br>; Genetic transcription <; br>; Electric stimulation  – Therapeutic use <; br>; Electric fields  – Therapeutic use <; br>; Electric currents, Direct  – Therapeutic use <; br>; Vascular endothelial growth factors.

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APA (6th Edition):

Jennings, J. A. (2007). Regulation of gene expression in response to continuous low Intensity direct current electrical fields. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,448

Chicago Manual of Style (16th Edition):

Jennings, Jessica Amber. “Regulation of gene expression in response to continuous low Intensity direct current electrical fields.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,448.

MLA Handbook (7th Edition):

Jennings, Jessica Amber. “Regulation of gene expression in response to continuous low Intensity direct current electrical fields.” 2007. Web. 19 Oct 2019.

Vancouver:

Jennings JA. Regulation of gene expression in response to continuous low Intensity direct current electrical fields. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,448.

Council of Science Editors:

Jennings JA. Regulation of gene expression in response to continuous low Intensity direct current electrical fields. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,448

8. Paik, Jason Chang. A nucleolar specificity factor for E2F1-induced cell death.

Degree: PhD, 2010, University of Alabama – Birmingham

The E2F family of transcription factors are important regulators of cell proliferation, and are often dysregulated in cancers. One member of the E2F family, E2F1,… (more)

Subjects/Keywords: Apoptosis<; br>; Chromosomal Proteins, Non-Histone  – physiology<; br>; E2F1 Transcription Factor  – physiology<; br>; Nuclear Proteins  – physiology<; br>; Transcription, Genetic<; br>; Transcriptional Activation

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APA (6th Edition):

Paik, J. C. (2010). A nucleolar specificity factor for E2F1-induced cell death. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1171

Chicago Manual of Style (16th Edition):

Paik, Jason Chang. “A nucleolar specificity factor for E2F1-induced cell death.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1171.

MLA Handbook (7th Edition):

Paik, Jason Chang. “A nucleolar specificity factor for E2F1-induced cell death.” 2010. Web. 19 Oct 2019.

Vancouver:

Paik JC. A nucleolar specificity factor for E2F1-induced cell death. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1171.

Council of Science Editors:

Paik JC. A nucleolar specificity factor for E2F1-induced cell death. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1171

9. Akinsiku, Olusimidele Tolulope. Qualitative analysis of HIV-1-specific CD8 T cell responses.

Degree: PhD, 2011, University of Alabama – Birmingham

In the absence of antiretroviral therapy (ART), the majority of individuals infected with human immunodeficiency virus-1 (HIV-1) will develop AIDS. HIV-1-infected controllers are exceptions to… (more)

Subjects/Keywords: CD8-Positive T-Lymphocytes  – immunology<; br>; Epitopes, T-Lymphocyte  – immunology<; br>; HIV Infections  – immunology<; br>; HIV-1  – immunology<; br>; HIV-1  – physiology<; br>; Interleukin-2  – metabolism<; br>; RNA, Antisense  – immunology<; br>; RNA, Viral  – immunology<; br>; Transcription, Genetic  – immunology

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APA (6th Edition):

Akinsiku, O. T. (2011). Qualitative analysis of HIV-1-specific CD8 T cell responses. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1148

Chicago Manual of Style (16th Edition):

Akinsiku, Olusimidele Tolulope. “Qualitative analysis of HIV-1-specific CD8 T cell responses.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1148.

MLA Handbook (7th Edition):

Akinsiku, Olusimidele Tolulope. “Qualitative analysis of HIV-1-specific CD8 T cell responses.” 2011. Web. 19 Oct 2019.

Vancouver:

Akinsiku OT. Qualitative analysis of HIV-1-specific CD8 T cell responses. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1148.

Council of Science Editors:

Akinsiku OT. Qualitative analysis of HIV-1-specific CD8 T cell responses. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1148

10. Dodd, Keela. The molecular biology of temperature-dependent sex determination in reptiles.

Degree: PhD, 2007, University of Alabama – Birmingham

Temperature-dependent sex determination (TSD) is a type of environmental sex determination in which the egg’s incubation temperature determines the sex of the embryo. The exact… (more)

Subjects/Keywords: Red-eared slider  – Embryos <; br>; Sex determination, Genetic <; br>; Red-eared slider  – Reproduction  – Effect of temperature on <; br>; Estradiol  – Physiological effect <; br>; Aromatase  – Inhibitors  – Physiological effect <; br>; Transcription factors  – Physiological effect

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APA (6th Edition):

Dodd, K. (2007). The molecular biology of temperature-dependent sex determination in reptiles. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,317

Chicago Manual of Style (16th Edition):

Dodd, Keela. “The molecular biology of temperature-dependent sex determination in reptiles.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,317.

MLA Handbook (7th Edition):

Dodd, Keela. “The molecular biology of temperature-dependent sex determination in reptiles.” 2007. Web. 19 Oct 2019.

Vancouver:

Dodd K. The molecular biology of temperature-dependent sex determination in reptiles. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,317.

Council of Science Editors:

Dodd K. The molecular biology of temperature-dependent sex determination in reptiles. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,317

11. Palmer, Matthew T. The influence of retroviral codon usage on the acquisition of the tRNA used to prime reverse transcription.

Degree: PhD, 2006, University of Alabama – Birmingham

An essential step in retrovirus replication is the conversion of the genomic-length viral RNA into a DNA copy, a reaction which is catalyzed by reverse… (more)

Subjects/Keywords: Leukemia Virus, Murine  – genetics <; br>; Leukemia Virus, Murine  – physiology <; br>; RNA  – genetics <; br>; RNA, Transfer, Lys  – genetics <; br>; RNA, Transfer, Phe  – genetics <; br>; RNA-Directed DNA Polymerase  – genetics <; br>; Transcription, Genetic <; br>; Virus Replication  – genetics

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APA (6th Edition):

Palmer, M. T. (2006). The influence of retroviral codon usage on the acquisition of the tRNA used to prime reverse transcription. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,149

Chicago Manual of Style (16th Edition):

Palmer, Matthew T. “The influence of retroviral codon usage on the acquisition of the tRNA used to prime reverse transcription.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,149.

MLA Handbook (7th Edition):

Palmer, Matthew T. “The influence of retroviral codon usage on the acquisition of the tRNA used to prime reverse transcription.” 2006. Web. 19 Oct 2019.

Vancouver:

Palmer MT. The influence of retroviral codon usage on the acquisition of the tRNA used to prime reverse transcription. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,149.

Council of Science Editors:

Palmer MT. The influence of retroviral codon usage on the acquisition of the tRNA used to prime reverse transcription. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,149

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