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You searched for subject:(Genetic regulation 60). Showing records 1 – 9 of 9 total matches.

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1. Phipps, Sharla Marion Ostein. Genetic and epigenetic modulation of telomerase activity in development and disease.

Degree: PhD, 2007, University of Alabama – Birmingham

The end replication problem of linear chromosomes leads to the erosion of telomeric DNA with each cell division. A mechanism to counteract telomeric attrition involves… (more)

Subjects/Keywords: Telomerase  – Physiological effect <; br>; Telomerase  – Inhibitors <; br>; Genetic regulation <; br>; Enzymes  – Regulation <; br>; Genetic transcription <; br>; Reverse transcriptase <; br>; Breast  – Cancer  – Genetic aspects <; br>; Tretinoin  – Physiological effect <; br>; Epigenesis.

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APA (6th Edition):

Phipps, S. M. O. (2007). Genetic and epigenetic modulation of telomerase activity in development and disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,260

Chicago Manual of Style (16th Edition):

Phipps, Sharla Marion Ostein. “Genetic and epigenetic modulation of telomerase activity in development and disease.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,260.

MLA Handbook (7th Edition):

Phipps, Sharla Marion Ostein. “Genetic and epigenetic modulation of telomerase activity in development and disease.” 2007. Web. 22 Sep 2019.

Vancouver:

Phipps SMO. Genetic and epigenetic modulation of telomerase activity in development and disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,260.

Council of Science Editors:

Phipps SMO. Genetic and epigenetic modulation of telomerase activity in development and disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,260

2. McAlear, Suzanne D. (Suzanne DuChai). Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation.

Degree: PhD, 2007, University of Alabama – Birmingham

Electrogenic Na/bicarbonate cotransporters (NBCes) are important regulators of intracellular pH in many tissues including the kidney, brain, and pancreas, and are members of a superfamily… (more)

Subjects/Keywords: Cysteine  – metabolism<; br>; Gene Expression Regulation  – physiology<; br>; Genetic Variation  – physiology<; br>; Mutagenesis<; br>; Oocytes<; br>; Sodium-Bicarbonate Symporters

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APA (6th Edition):

McAlear, S. D. (. D. (2007). Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,456

Chicago Manual of Style (16th Edition):

McAlear, Suzanne D (Suzanne DuChai). “Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,456.

MLA Handbook (7th Edition):

McAlear, Suzanne D (Suzanne DuChai). “Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation.” 2007. Web. 22 Sep 2019.

Vancouver:

McAlear SD(D. Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,456.

Council of Science Editors:

McAlear SD(D. Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,456

3. He, Ti. Molecular regulation of Pax5-mediated biological functions.

Degree: PhD, 2008, University of Alabama – Birmingham

B lineage cells are major players in the adaptive immune system. Pax5 is essential for B lineage cell development and function. Pax5 controls B lineage… (more)

Subjects/Keywords: B-Cell-Specific Activator Protein <; br>; Gene Expression Regulation <; br>; Histone Acetyltransferases  – metabolism <; br>; Transcription Factors <; br>; Transcription, Genetic

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APA (6th Edition):

He, T. (2008). Molecular regulation of Pax5-mediated biological functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,329

Chicago Manual of Style (16th Edition):

He, Ti. “Molecular regulation of Pax5-mediated biological functions.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,329.

MLA Handbook (7th Edition):

He, Ti. “Molecular regulation of Pax5-mediated biological functions.” 2008. Web. 22 Sep 2019.

Vancouver:

He T. Molecular regulation of Pax5-mediated biological functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,329.

Council of Science Editors:

He T. Molecular regulation of Pax5-mediated biological functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,329

4. Ho, Shiuh-Rong. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.

Degree: PhD, 2010, University of Alabama – Birmingham

O-GlcNAcylation is an abundant and dynamic post-translational modification on serine and threonine residues of nuclear and cytoplasmic proteins. O-GlcNAc Transferase (OGT) and Nuclear Cytoplasmic O-GlcNAcase… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism<; br>; Gene Expression Regulation<; br>; Oxidative Stress  – genetics<; br>; Transcription Factors  – metabolism<; br>; Transcription, Genetic

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APA (6th Edition):

Ho, S. (2010). O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1162

Chicago Manual of Style (16th Edition):

Ho, Shiuh-Rong. “O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1162.

MLA Handbook (7th Edition):

Ho, Shiuh-Rong. “O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.” 2010. Web. 22 Sep 2019.

Vancouver:

Ho S. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1162.

Council of Science Editors:

Ho S. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1162

5. Jennings, Jessica Amber. Regulation of gene expression in response to continuous low Intensity direct current electrical fields.

Degree: PhD, 2007, University of Alabama – Birmingham

In the normal process of wound healing, small electric fields are generated at the wound site due to a disturbance of the potential difference maintained… (more)

Subjects/Keywords: Skin  – Wounds and injuries  – Treatment <; br>; Genetic regulation <; br>; Genetic transcription <; br>; Electric stimulation  – Therapeutic use <; br>; Electric fields  – Therapeutic use <; br>; Electric currents, Direct  – Therapeutic use <; br>; Vascular endothelial growth factors.

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APA (6th Edition):

Jennings, J. A. (2007). Regulation of gene expression in response to continuous low Intensity direct current electrical fields. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,448

Chicago Manual of Style (16th Edition):

Jennings, Jessica Amber. “Regulation of gene expression in response to continuous low Intensity direct current electrical fields.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,448.

MLA Handbook (7th Edition):

Jennings, Jessica Amber. “Regulation of gene expression in response to continuous low Intensity direct current electrical fields.” 2007. Web. 22 Sep 2019.

Vancouver:

Jennings JA. Regulation of gene expression in response to continuous low Intensity direct current electrical fields. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,448.

Council of Science Editors:

Jennings JA. Regulation of gene expression in response to continuous low Intensity direct current electrical fields. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,448

6. Saini, Reshu. Model system using controlled receptor expression for molecular ultrasound imaging applications.

Degree: MS, 2012, University of Alabama – Birmingham

Ultrasound is a noninvasive, inexpensive imaging modality used to characterize biological tissues. Targeted contrast-enhanced ultrasound imaging is an emerging preclinical strategy which uses contrast agents… (more)

Subjects/Keywords: Contrast-enhanced ultrasound<; br>; Microbubbles – Diagnostic use<; br>; Ultrasound contrast media<; br>; Adenoviruses<; br>; Genetic vectors<; br>; Viruses – Receptors<; br>; Genetic regulation

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APA (6th Edition):

Saini, R. (2012). Model system using controlled receptor expression for molecular ultrasound imaging applications. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1327

Chicago Manual of Style (16th Edition):

Saini, Reshu. “Model system using controlled receptor expression for molecular ultrasound imaging applications.” 2012. Masters Thesis, University of Alabama – Birmingham. Accessed September 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1327.

MLA Handbook (7th Edition):

Saini, Reshu. “Model system using controlled receptor expression for molecular ultrasound imaging applications.” 2012. Web. 22 Sep 2019.

Vancouver:

Saini R. Model system using controlled receptor expression for molecular ultrasound imaging applications. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2012. [cited 2019 Sep 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1327.

Council of Science Editors:

Saini R. Model system using controlled receptor expression for molecular ultrasound imaging applications. [Masters Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1327

7. Berletch, Joel Bradford. Green tea polyphenols are associated with changes in genetic and epigenetic anti-cancer mechanisms in vitro and in vivo.

Degree: PhD, 2007, University of Alabama – Birmingham

Human chromosomes are protected from degradation associated with cell division by hexameric repeats of 5’-TTAGGG-3’ termed telomeres. During cellular replication human chromosomes undergo the loss… (more)

Subjects/Keywords: Antineoplastic agents <; br>; Cancer  – Genetic aspects <; br>; Gene targeting <; br>; Cancer cells  – Growth  – Regulation <; br>; Telomerase  – Inhibitors <; br>; Catechin  – Physiological effect <; br>; Green tea  – Physiological effect.

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APA (6th Edition):

Berletch, J. B. (2007). Green tea polyphenols are associated with changes in genetic and epigenetic anti-cancer mechanisms in vitro and in vivo. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,174

Chicago Manual of Style (16th Edition):

Berletch, Joel Bradford. “Green tea polyphenols are associated with changes in genetic and epigenetic anti-cancer mechanisms in vitro and in vivo.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,174.

MLA Handbook (7th Edition):

Berletch, Joel Bradford. “Green tea polyphenols are associated with changes in genetic and epigenetic anti-cancer mechanisms in vitro and in vivo.” 2007. Web. 22 Sep 2019.

Vancouver:

Berletch JB. Green tea polyphenols are associated with changes in genetic and epigenetic anti-cancer mechanisms in vitro and in vivo. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,174.

Council of Science Editors:

Berletch JB. Green tea polyphenols are associated with changes in genetic and epigenetic anti-cancer mechanisms in vitro and in vivo. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,174

8. Ramaswamy, Girish. Mechanical and geometric characterization of mouse cortical bone with osteoblast-specific knockout of insulin-like growth factor receptor gene.

Degree: MS, 2007, University of Alabama – Birmingham

The primary purpose of this study was to characterize the mechanical properties of long bones in mice with osteoblast-specific knockout of the gene encoding the… (more)

Subjects/Keywords: Tibia  – Mechanical properties <; br>; Flexure <; br>; Somatomedin  – Secretion  – Regulation <; br>; Somatomedin  – Receptors <; br>; Gene silencing <; br>; Genetic recombnation <; br>; Transgenic mice  – Physiology

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APA (6th Edition):

Ramaswamy, G. (2007). Mechanical and geometric characterization of mouse cortical bone with osteoblast-specific knockout of insulin-like growth factor receptor gene. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,535

Chicago Manual of Style (16th Edition):

Ramaswamy, Girish. “Mechanical and geometric characterization of mouse cortical bone with osteoblast-specific knockout of insulin-like growth factor receptor gene.” 2007. Masters Thesis, University of Alabama – Birmingham. Accessed September 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,535.

MLA Handbook (7th Edition):

Ramaswamy, Girish. “Mechanical and geometric characterization of mouse cortical bone with osteoblast-specific knockout of insulin-like growth factor receptor gene.” 2007. Web. 22 Sep 2019.

Vancouver:

Ramaswamy G. Mechanical and geometric characterization of mouse cortical bone with osteoblast-specific knockout of insulin-like growth factor receptor gene. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,535.

Council of Science Editors:

Ramaswamy G. Mechanical and geometric characterization of mouse cortical bone with osteoblast-specific knockout of insulin-like growth factor receptor gene. [Masters Thesis]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,535

9. DeAngelis, Joseph Tyson. An investigation of the molecular mechanisms involved in the generation of breast cancer.

Degree: PhD, 2010, University of Alabama – Birmingham

Cell culture models of oncogenesis that use cellular reprogramming to generate a neoplastic cell from a normal cell provide one of the few opportunities to… (more)

Subjects/Keywords: Breast  – Cancer  – Molecular aspects <; br>; Breast  – Cancer  – Genetic aspects <; br>; Cancer cells  – Growth  – Regulation <; br>; Nuclear receptors (Biochemistry) <; br>; Metastasis <; br>; Brain  – Cancer

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APA (6th Edition):

DeAngelis, J. T. (2010). An investigation of the molecular mechanisms involved in the generation of breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,841

Chicago Manual of Style (16th Edition):

DeAngelis, Joseph Tyson. “An investigation of the molecular mechanisms involved in the generation of breast cancer.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,841.

MLA Handbook (7th Edition):

DeAngelis, Joseph Tyson. “An investigation of the molecular mechanisms involved in the generation of breast cancer.” 2010. Web. 22 Sep 2019.

Vancouver:

DeAngelis JT. An investigation of the molecular mechanisms involved in the generation of breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,841.

Council of Science Editors:

DeAngelis JT. An investigation of the molecular mechanisms involved in the generation of breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,841

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