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You searched for subject:(Genetic Phenomena). Showing records 1 – 30 of 262 total matches.

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Drexel University

1. Klee, Nicole Siobhan. Type II diabetes and detrusor muscle function: Compensated and decompensated states.

Degree: 2016, Drexel University

Bladder smooth muscle (detrusor muscle) undergoes temporal changes in response to diabetes, resulting in diabetic bladder dysfunction (DBD). DBD induces a compensated state of bladder… (more)

Subjects/Keywords: Molecular biology; Cytology; Genetics; Biochemical Phenomena; Genetic Phenomena; Genetic Processes

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APA (6th Edition):

Klee, N. S. (2016). Type II diabetes and detrusor muscle function: Compensated and decompensated states. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7159

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Klee, Nicole Siobhan. “Type II diabetes and detrusor muscle function: Compensated and decompensated states.” 2016. Thesis, Drexel University. Accessed July 10, 2020. http://hdl.handle.net/1860/idea:7159.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Klee, Nicole Siobhan. “Type II diabetes and detrusor muscle function: Compensated and decompensated states.” 2016. Web. 10 Jul 2020.

Vancouver:

Klee NS. Type II diabetes and detrusor muscle function: Compensated and decompensated states. [Internet] [Thesis]. Drexel University; 2016. [cited 2020 Jul 10]. Available from: http://hdl.handle.net/1860/idea:7159.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Klee NS. Type II diabetes and detrusor muscle function: Compensated and decompensated states. [Thesis]. Drexel University; 2016. Available from: http://hdl.handle.net/1860/idea:7159

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

2. Sodi, Valerie. Regulation of O-GlcNAc Transferase in breast cancer and its role in regulating lipid metabolism.

Degree: 2016, Drexel University

Cancer cells exhibit altered metabolism characterized by increased glucose and glutamine uptake. Altered utilization of these substrates directly contributes to O-linked-[beta]-N-acetylglucosamine (O-GlcNAc) modifications on intracellular… (more)

Subjects/Keywords: Molecular biology; Cytology; Genetics; Biochemical Phenomena; Genetic Phenomena; Genetic Processes

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APA (6th Edition):

Sodi, V. (2016). Regulation of O-GlcNAc Transferase in breast cancer and its role in regulating lipid metabolism. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7176

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sodi, Valerie. “Regulation of O-GlcNAc Transferase in breast cancer and its role in regulating lipid metabolism.” 2016. Thesis, Drexel University. Accessed July 10, 2020. http://hdl.handle.net/1860/idea:7176.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sodi, Valerie. “Regulation of O-GlcNAc Transferase in breast cancer and its role in regulating lipid metabolism.” 2016. Web. 10 Jul 2020.

Vancouver:

Sodi V. Regulation of O-GlcNAc Transferase in breast cancer and its role in regulating lipid metabolism. [Internet] [Thesis]. Drexel University; 2016. [cited 2020 Jul 10]. Available from: http://hdl.handle.net/1860/idea:7176.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sodi V. Regulation of O-GlcNAc Transferase in breast cancer and its role in regulating lipid metabolism. [Thesis]. Drexel University; 2016. Available from: http://hdl.handle.net/1860/idea:7176

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

3. Tuyishime, Marina. Investigation into the mechanism of a novel HIV-1 fusion inhibitor, SC12.

Degree: 2016, Drexel University

Due to the emergence of drug-resistant viruses and problems with drug side effects, new drugs and drug targets are constantly required in the battle against… (more)

Subjects/Keywords: Molecular biology; Cytology; Genetics; Biochemical Phenomena; Genetic Phenomena; Genetic Processes

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APA (6th Edition):

Tuyishime, M. (2016). Investigation into the mechanism of a novel HIV-1 fusion inhibitor, SC12. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7177

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tuyishime, Marina. “Investigation into the mechanism of a novel HIV-1 fusion inhibitor, SC12.” 2016. Thesis, Drexel University. Accessed July 10, 2020. http://hdl.handle.net/1860/idea:7177.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tuyishime, Marina. “Investigation into the mechanism of a novel HIV-1 fusion inhibitor, SC12.” 2016. Web. 10 Jul 2020.

Vancouver:

Tuyishime M. Investigation into the mechanism of a novel HIV-1 fusion inhibitor, SC12. [Internet] [Thesis]. Drexel University; 2016. [cited 2020 Jul 10]. Available from: http://hdl.handle.net/1860/idea:7177.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tuyishime M. Investigation into the mechanism of a novel HIV-1 fusion inhibitor, SC12. [Thesis]. Drexel University; 2016. Available from: http://hdl.handle.net/1860/idea:7177

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

4. Huang, Yu-Hung. Role of HuR in Mediating Ovarian Cancer Treatment.

Degree: 2016, Drexel University

An mRNA-binding protein, HuR (Human antigen R, aka ELAVL1), is highly elevated in many cancers, and is a master regulator of gene expression. HuR-regulated genes… (more)

Subjects/Keywords: Molecular biology; Cytology; Genetics; Biochemical Phenomena; Genetic Phenomena; Genetic Processes

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APA (6th Edition):

Huang, Y. (2016). Role of HuR in Mediating Ovarian Cancer Treatment. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7154

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Yu-Hung. “Role of HuR in Mediating Ovarian Cancer Treatment.” 2016. Thesis, Drexel University. Accessed July 10, 2020. http://hdl.handle.net/1860/idea:7154.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Yu-Hung. “Role of HuR in Mediating Ovarian Cancer Treatment.” 2016. Web. 10 Jul 2020.

Vancouver:

Huang Y. Role of HuR in Mediating Ovarian Cancer Treatment. [Internet] [Thesis]. Drexel University; 2016. [cited 2020 Jul 10]. Available from: http://hdl.handle.net/1860/idea:7154.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang Y. Role of HuR in Mediating Ovarian Cancer Treatment. [Thesis]. Drexel University; 2016. Available from: http://hdl.handle.net/1860/idea:7154

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

5. Pershing, April M. Genetic exploration of the ATP synthase complex in Plasmodium falciparum.

Degree: 2016, Drexel University

During the blood stages of its life cycle, P. falciparum relies on glycolysis as its major source of ATP rather than oxidative phosphorylation. Yet, we… (more)

Subjects/Keywords: Molecular biology; Cytology; Genetics; Biochemical Phenomena; Genetic Phenomena; Genetic Processes

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APA (6th Edition):

Pershing, A. M. (2016). Genetic exploration of the ATP synthase complex in Plasmodium falciparum. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7148

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pershing, April M. “Genetic exploration of the ATP synthase complex in Plasmodium falciparum.” 2016. Thesis, Drexel University. Accessed July 10, 2020. http://hdl.handle.net/1860/idea:7148.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pershing, April M. “Genetic exploration of the ATP synthase complex in Plasmodium falciparum.” 2016. Web. 10 Jul 2020.

Vancouver:

Pershing AM. Genetic exploration of the ATP synthase complex in Plasmodium falciparum. [Internet] [Thesis]. Drexel University; 2016. [cited 2020 Jul 10]. Available from: http://hdl.handle.net/1860/idea:7148.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pershing AM. Genetic exploration of the ATP synthase complex in Plasmodium falciparum. [Thesis]. Drexel University; 2016. Available from: http://hdl.handle.net/1860/idea:7148

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Burgwin, Chelsea Marie. Characterization of the Cell-Autonomous Traits of Tsk2/+ Mice, a Model of Systemic Sclerosis.

Degree: 2016, Drexel University

Overview: The tight skin 2 (Tsk2/+) mouse is a genetic model of systemic sclerosis (SSc). This model has several similarities to human disease including tight… (more)

Subjects/Keywords: Molecular biology; Cytology; Genetics; Biochemical Phenomena; Genetic Phenomena; Genetic Processes

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APA (6th Edition):

Burgwin, C. M. (2016). Characterization of the Cell-Autonomous Traits of Tsk2/+ Mice, a Model of Systemic Sclerosis. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7149

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Burgwin, Chelsea Marie. “Characterization of the Cell-Autonomous Traits of Tsk2/+ Mice, a Model of Systemic Sclerosis.” 2016. Thesis, Drexel University. Accessed July 10, 2020. http://hdl.handle.net/1860/idea:7149.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Burgwin, Chelsea Marie. “Characterization of the Cell-Autonomous Traits of Tsk2/+ Mice, a Model of Systemic Sclerosis.” 2016. Web. 10 Jul 2020.

Vancouver:

Burgwin CM. Characterization of the Cell-Autonomous Traits of Tsk2/+ Mice, a Model of Systemic Sclerosis. [Internet] [Thesis]. Drexel University; 2016. [cited 2020 Jul 10]. Available from: http://hdl.handle.net/1860/idea:7149.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Burgwin CM. Characterization of the Cell-Autonomous Traits of Tsk2/+ Mice, a Model of Systemic Sclerosis. [Thesis]. Drexel University; 2016. Available from: http://hdl.handle.net/1860/idea:7149

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

7. Sowash Molinari, Aislinn Rebecca. Transcription Factor Specificity Protein 1 (Sp1) Regulates the Centrochromatin Landscape and Centromeric Transcription During Mitosis.

Degree: 2016, Drexel University

Chromosomal instability (CIN) is a dynamic and continual gain or loss of whole chromosomes, or parts of chromosomes, during cell division. It is associated with… (more)

Subjects/Keywords: Molecular biology; Cytology; Genetics; Biochemical Phenomena; Genetic Phenomena; Genetic Processes

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APA (6th Edition):

Sowash Molinari, A. R. (2016). Transcription Factor Specificity Protein 1 (Sp1) Regulates the Centrochromatin Landscape and Centromeric Transcription During Mitosis. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7146

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sowash Molinari, Aislinn Rebecca. “Transcription Factor Specificity Protein 1 (Sp1) Regulates the Centrochromatin Landscape and Centromeric Transcription During Mitosis.” 2016. Thesis, Drexel University. Accessed July 10, 2020. http://hdl.handle.net/1860/idea:7146.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sowash Molinari, Aislinn Rebecca. “Transcription Factor Specificity Protein 1 (Sp1) Regulates the Centrochromatin Landscape and Centromeric Transcription During Mitosis.” 2016. Web. 10 Jul 2020.

Vancouver:

Sowash Molinari AR. Transcription Factor Specificity Protein 1 (Sp1) Regulates the Centrochromatin Landscape and Centromeric Transcription During Mitosis. [Internet] [Thesis]. Drexel University; 2016. [cited 2020 Jul 10]. Available from: http://hdl.handle.net/1860/idea:7146.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sowash Molinari AR. Transcription Factor Specificity Protein 1 (Sp1) Regulates the Centrochromatin Landscape and Centromeric Transcription During Mitosis. [Thesis]. Drexel University; 2016. Available from: http://hdl.handle.net/1860/idea:7146

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Edith Cowan University

8. Rogers, Tamara. A linkage study of autism using multipoint sib-pair analysis.

Degree: 2000, Edith Cowan University

 Autism is a severe developmental disorder that was first described by Kanner in 1943. It is characterised by four major criteria: marked social deficits, delay… (more)

Subjects/Keywords: Autism; Genetic Phenomena

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APA (6th Edition):

Rogers, T. (2000). A linkage study of autism using multipoint sib-pair analysis. (Thesis). Edith Cowan University. Retrieved from https://ro.ecu.edu.au/theses/1536

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rogers, Tamara. “A linkage study of autism using multipoint sib-pair analysis.” 2000. Thesis, Edith Cowan University. Accessed July 10, 2020. https://ro.ecu.edu.au/theses/1536.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rogers, Tamara. “A linkage study of autism using multipoint sib-pair analysis.” 2000. Web. 10 Jul 2020.

Vancouver:

Rogers T. A linkage study of autism using multipoint sib-pair analysis. [Internet] [Thesis]. Edith Cowan University; 2000. [cited 2020 Jul 10]. Available from: https://ro.ecu.edu.au/theses/1536.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rogers T. A linkage study of autism using multipoint sib-pair analysis. [Thesis]. Edith Cowan University; 2000. Available from: https://ro.ecu.edu.au/theses/1536

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

9. Chen, Natalie Cheng. Age-like Phenotype of Microglia During HIV-1 Infection.

Degree: 2016, Drexel University

HIV Associated Neurocognitive Disorder (HAND) persists in 50% of infected patients despite treatments. Moreover, HAND potentiates aging-associated neurocognitive decline as the infected population grow older.… (more)

Subjects/Keywords: Molecular biology; Cytology; Genetics; Biochemical Phenomena; Genetic Phenomena; Genetic Processes; Molecular biology; Cytology; Genetics

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APA (6th Edition):

Chen, N. C. (2016). Age-like Phenotype of Microglia During HIV-1 Infection. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7160

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Natalie Cheng. “Age-like Phenotype of Microglia During HIV-1 Infection.” 2016. Thesis, Drexel University. Accessed July 10, 2020. http://hdl.handle.net/1860/idea:7160.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Natalie Cheng. “Age-like Phenotype of Microglia During HIV-1 Infection.” 2016. Web. 10 Jul 2020.

Vancouver:

Chen NC. Age-like Phenotype of Microglia During HIV-1 Infection. [Internet] [Thesis]. Drexel University; 2016. [cited 2020 Jul 10]. Available from: http://hdl.handle.net/1860/idea:7160.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen NC. Age-like Phenotype of Microglia During HIV-1 Infection. [Thesis]. Drexel University; 2016. Available from: http://hdl.handle.net/1860/idea:7160

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

10. Johansen, Christopher T. Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia.

Degree: 2011, University of Western Ontario

 Cardiovascular disease (CVD) is the leading cause of morbidity and mortality in Canada. Among non-traditional risk factors, plasma triglyceride (TG) concentration is re-emerging as a… (more)

Subjects/Keywords: hypertriglyceridemia; hyperlipoproteinemia; genetic variation; cardiovascular disease; genome-wide association study; resequencing; Biochemical Phenomena, Metabolism, and Nutrition; Cardiovascular Diseases; Genetic Phenomena

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APA (6th Edition):

Johansen, C. T. (2011). Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/219

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Johansen, Christopher T. “Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia.” 2011. Thesis, University of Western Ontario. Accessed July 10, 2020. https://ir.lib.uwo.ca/etd/219.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Johansen, Christopher T. “Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia.” 2011. Web. 10 Jul 2020.

Vancouver:

Johansen CT. Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia. [Internet] [Thesis]. University of Western Ontario; 2011. [cited 2020 Jul 10]. Available from: https://ir.lib.uwo.ca/etd/219.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Johansen CT. Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia. [Thesis]. University of Western Ontario; 2011. Available from: https://ir.lib.uwo.ca/etd/219

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Marshall University

11. Huang, Yue. Pitx2 is Overexpressed in Follicular Cell-Derived Thyroid Cancer and Promotes Thyroid Cancer Proliferation by Regulating Cell Cycle.

Degree: 2011, Marshall University

 Thyroid cancer is the most prevailing malignancy of the endocrine system. Its incidence is rapidly rising at the second fastest rate of all malignancies in… (more)

Subjects/Keywords: Thyroid cancer; PITX2; Gene Transcription; Cyclin D2; Cyclin A1; Cell Cycle; Protein-Protein Interaction; Biological Phenomena, Cell Phenomena, and Immunity; Genetic Phenomena; Genetic Structures; Medicine and Health Sciences; Oncology

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APA (6th Edition):

Huang, Y. (2011). Pitx2 is Overexpressed in Follicular Cell-Derived Thyroid Cancer and Promotes Thyroid Cancer Proliferation by Regulating Cell Cycle. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/87

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Yue. “Pitx2 is Overexpressed in Follicular Cell-Derived Thyroid Cancer and Promotes Thyroid Cancer Proliferation by Regulating Cell Cycle.” 2011. Thesis, Marshall University. Accessed July 10, 2020. http://mds.marshall.edu/etd/87.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Yue. “Pitx2 is Overexpressed in Follicular Cell-Derived Thyroid Cancer and Promotes Thyroid Cancer Proliferation by Regulating Cell Cycle.” 2011. Web. 10 Jul 2020.

Vancouver:

Huang Y. Pitx2 is Overexpressed in Follicular Cell-Derived Thyroid Cancer and Promotes Thyroid Cancer Proliferation by Regulating Cell Cycle. [Internet] [Thesis]. Marshall University; 2011. [cited 2020 Jul 10]. Available from: http://mds.marshall.edu/etd/87.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang Y. Pitx2 is Overexpressed in Follicular Cell-Derived Thyroid Cancer and Promotes Thyroid Cancer Proliferation by Regulating Cell Cycle. [Thesis]. Marshall University; 2011. Available from: http://mds.marshall.edu/etd/87

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kentucky

12. Stults, Dawn Michelle. HUMAN RIBOSOMAL RNA GENE CLUSTERS ARE RECOMBINATIONAL HOTSPOTS IN CANCER.

Degree: 2009, University of Kentucky

 The gene that produces the precursor RNA transcript to the three largest ribosomal RNA molecules (rDNA) is present in multiple copies and organized into gene… (more)

Subjects/Keywords: genomic instability; cancer; gene clusters; DNA repair; recombination; Genetic Phenomena

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APA (6th Edition):

Stults, D. M. (2009). HUMAN RIBOSOMAL RNA GENE CLUSTERS ARE RECOMBINATIONAL HOTSPOTS IN CANCER. (Masters Thesis). University of Kentucky. Retrieved from http://uknowledge.uky.edu/gradschool_theses/626

Chicago Manual of Style (16th Edition):

Stults, Dawn Michelle. “HUMAN RIBOSOMAL RNA GENE CLUSTERS ARE RECOMBINATIONAL HOTSPOTS IN CANCER.” 2009. Masters Thesis, University of Kentucky. Accessed July 10, 2020. http://uknowledge.uky.edu/gradschool_theses/626.

MLA Handbook (7th Edition):

Stults, Dawn Michelle. “HUMAN RIBOSOMAL RNA GENE CLUSTERS ARE RECOMBINATIONAL HOTSPOTS IN CANCER.” 2009. Web. 10 Jul 2020.

Vancouver:

Stults DM. HUMAN RIBOSOMAL RNA GENE CLUSTERS ARE RECOMBINATIONAL HOTSPOTS IN CANCER. [Internet] [Masters thesis]. University of Kentucky; 2009. [cited 2020 Jul 10]. Available from: http://uknowledge.uky.edu/gradschool_theses/626.

Council of Science Editors:

Stults DM. HUMAN RIBOSOMAL RNA GENE CLUSTERS ARE RECOMBINATIONAL HOTSPOTS IN CANCER. [Masters Thesis]. University of Kentucky; 2009. Available from: http://uknowledge.uky.edu/gradschool_theses/626


Marshall University

13. Adkins, Nicholas L. Identification and Characterization of Novel Sir3/MeCP2-Chromatin Interactions.

Degree: 2009, Marshall University

 The eukaryotic genome is packaged into chromosomes that are made up of a highly organized and heavily regulated structure called chromatin. The proteins involved in… (more)

Subjects/Keywords: <; p>; Chromatin.<; /p>; <; p>; DNA-binding proteins.<; /p>;

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APA (6th Edition):

Adkins, N. L. (2009). Identification and Characterization of Novel Sir3/MeCP2-Chromatin Interactions. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/392

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Adkins, Nicholas L. “Identification and Characterization of Novel Sir3/MeCP2-Chromatin Interactions.” 2009. Thesis, Marshall University. Accessed July 10, 2020. http://mds.marshall.edu/etd/392.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Adkins, Nicholas L. “Identification and Characterization of Novel Sir3/MeCP2-Chromatin Interactions.” 2009. Web. 10 Jul 2020.

Vancouver:

Adkins NL. Identification and Characterization of Novel Sir3/MeCP2-Chromatin Interactions. [Internet] [Thesis]. Marshall University; 2009. [cited 2020 Jul 10]. Available from: http://mds.marshall.edu/etd/392.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Adkins NL. Identification and Characterization of Novel Sir3/MeCP2-Chromatin Interactions. [Thesis]. Marshall University; 2009. Available from: http://mds.marshall.edu/etd/392

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Shearstone, Jeffrey R. Global DNA Demethylation During Erythropoiesis: A Dissertation.

Degree: Cancer Biology, Molecular, Cell and Cancer Biology, 2011, U of Massachusetts : Med

  In the mammalian genome, 5‟-CpG-3‟ dinucleotides are frequently methylated, correlating with transcriptional silencing. Genome-wide waves of demethylation are thought to occur only twice during… (more)

Subjects/Keywords: Erythropoiesis; DNA Methylation; Biochemical Phenomena, Metabolism, and Nutrition; Cancer Biology; Cells; Circulatory and Respiratory Physiology; Genetic Phenomena; Genetics and Genomics

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APA (6th Edition):

Shearstone, J. R. (2011). Global DNA Demethylation During Erythropoiesis: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/549

Chicago Manual of Style (16th Edition):

Shearstone, Jeffrey R. “Global DNA Demethylation During Erythropoiesis: A Dissertation.” 2011. Doctoral Dissertation, U of Massachusetts : Med. Accessed July 10, 2020. https://escholarship.umassmed.edu/gsbs_diss/549.

MLA Handbook (7th Edition):

Shearstone, Jeffrey R. “Global DNA Demethylation During Erythropoiesis: A Dissertation.” 2011. Web. 10 Jul 2020.

Vancouver:

Shearstone JR. Global DNA Demethylation During Erythropoiesis: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2011. [cited 2020 Jul 10]. Available from: https://escholarship.umassmed.edu/gsbs_diss/549.

Council of Science Editors:

Shearstone JR. Global DNA Demethylation During Erythropoiesis: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2011. Available from: https://escholarship.umassmed.edu/gsbs_diss/549


Texas Medical Center

15. Cho, Min Soon. ΔNp63 REGULATES A COMPLEX NETWORK OF TARGET GENES IN LIMB AND EPIDERMAL DEVELOPMENT.

Degree: PhD, 2011, Texas Medical Center

  The skin is composed of two major compartments, the dermis and epidermis. The epidermis forms a barrier to protect the body. The stratified epithelium… (more)

Subjects/Keywords: ΔNp63; Epidermis; Limb; Development; Differentiation; Pluripotency; DGCR8; microRNAs; Genetic Phenomena; Medical Sciences

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APA (6th Edition):

Cho, M. S. (2011). ΔNp63 REGULATES A COMPLEX NETWORK OF TARGET GENES IN LIMB AND EPIDERMAL DEVELOPMENT. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/128

Chicago Manual of Style (16th Edition):

Cho, Min Soon. “ΔNp63 REGULATES A COMPLEX NETWORK OF TARGET GENES IN LIMB AND EPIDERMAL DEVELOPMENT.” 2011. Doctoral Dissertation, Texas Medical Center. Accessed July 10, 2020. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/128.

MLA Handbook (7th Edition):

Cho, Min Soon. “ΔNp63 REGULATES A COMPLEX NETWORK OF TARGET GENES IN LIMB AND EPIDERMAL DEVELOPMENT.” 2011. Web. 10 Jul 2020.

Vancouver:

Cho MS. ΔNp63 REGULATES A COMPLEX NETWORK OF TARGET GENES IN LIMB AND EPIDERMAL DEVELOPMENT. [Internet] [Doctoral dissertation]. Texas Medical Center; 2011. [cited 2020 Jul 10]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/128.

Council of Science Editors:

Cho MS. ΔNp63 REGULATES A COMPLEX NETWORK OF TARGET GENES IN LIMB AND EPIDERMAL DEVELOPMENT. [Doctoral Dissertation]. Texas Medical Center; 2011. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/128


University of South Carolina

16. Mittag, Dana Margaret. The Perceived Utility of Personalized Genomic Medicine in Individuals with a Family History of Heart Disease: A Pilot Study.

Degree: MS, Genetic Counseling, 2017, University of South Carolina

  According to the World Health Organization (2005), cardiovascular disease (CVD) is the number one cause of death in most countries. Assessing a patient’s risk… (more)

Subjects/Keywords: Genetic Phenomena; Medical Sciences; Medicine and Health Sciences; Heart Disease; Genomic Medicine

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APA (6th Edition):

Mittag, D. M. (2017). The Perceived Utility of Personalized Genomic Medicine in Individuals with a Family History of Heart Disease: A Pilot Study. (Masters Thesis). University of South Carolina. Retrieved from https://scholarcommons.sc.edu/etd/4180

Chicago Manual of Style (16th Edition):

Mittag, Dana Margaret. “The Perceived Utility of Personalized Genomic Medicine in Individuals with a Family History of Heart Disease: A Pilot Study.” 2017. Masters Thesis, University of South Carolina. Accessed July 10, 2020. https://scholarcommons.sc.edu/etd/4180.

MLA Handbook (7th Edition):

Mittag, Dana Margaret. “The Perceived Utility of Personalized Genomic Medicine in Individuals with a Family History of Heart Disease: A Pilot Study.” 2017. Web. 10 Jul 2020.

Vancouver:

Mittag DM. The Perceived Utility of Personalized Genomic Medicine in Individuals with a Family History of Heart Disease: A Pilot Study. [Internet] [Masters thesis]. University of South Carolina; 2017. [cited 2020 Jul 10]. Available from: https://scholarcommons.sc.edu/etd/4180.

Council of Science Editors:

Mittag DM. The Perceived Utility of Personalized Genomic Medicine in Individuals with a Family History of Heart Disease: A Pilot Study. [Masters Thesis]. University of South Carolina; 2017. Available from: https://scholarcommons.sc.edu/etd/4180

17. Brewer, Daniel Niron. Elucidation of the Role of the Exocyst Subunit Sec6p in Exocytosis: A Dissertation.

Degree: Biochemistry and Molecular Pharmacology, Biochemistry and Molecular Pharmacology, 2009, U of Massachusetts : Med

  Trafficking of protein and lipid cargo through the secretory pathway in eukaryotic cells is mediated by membrane-bound vesicles. Secretory vesicles are targeted to sites… (more)

Subjects/Keywords: SNARE Proteins; Vesicular Transport Proteins; Exocytosis; Amino Acids, Peptides, and Proteins; Cells; Genetic Phenomena

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APA (6th Edition):

Brewer, D. N. (2009). Elucidation of the Role of the Exocyst Subunit Sec6p in Exocytosis: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/446

Chicago Manual of Style (16th Edition):

Brewer, Daniel Niron. “Elucidation of the Role of the Exocyst Subunit Sec6p in Exocytosis: A Dissertation.” 2009. Doctoral Dissertation, U of Massachusetts : Med. Accessed July 10, 2020. https://escholarship.umassmed.edu/gsbs_diss/446.

MLA Handbook (7th Edition):

Brewer, Daniel Niron. “Elucidation of the Role of the Exocyst Subunit Sec6p in Exocytosis: A Dissertation.” 2009. Web. 10 Jul 2020.

Vancouver:

Brewer DN. Elucidation of the Role of the Exocyst Subunit Sec6p in Exocytosis: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2009. [cited 2020 Jul 10]. Available from: https://escholarship.umassmed.edu/gsbs_diss/446.

Council of Science Editors:

Brewer DN. Elucidation of the Role of the Exocyst Subunit Sec6p in Exocytosis: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2009. Available from: https://escholarship.umassmed.edu/gsbs_diss/446


Princeton University

18. Krotov, Dmitry. Strong correlations in gravity and biophysics .

Degree: PhD, 2014, Princeton University

 The unifying theme of this dissertation is the use of correlations. In the first part (chapter 2), we investigate correlations in quantum field theories in… (more)

Subjects/Keywords: critical phenomena; Drosophila embryo; genetic networks; gravity; quantum field theory; Schwinger-Keldysh

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APA (6th Edition):

Krotov, D. (2014). Strong correlations in gravity and biophysics . (Doctoral Dissertation). Princeton University. Retrieved from http://arks.princeton.edu/ark:/88435/dsp01nv935507k

Chicago Manual of Style (16th Edition):

Krotov, Dmitry. “Strong correlations in gravity and biophysics .” 2014. Doctoral Dissertation, Princeton University. Accessed July 10, 2020. http://arks.princeton.edu/ark:/88435/dsp01nv935507k.

MLA Handbook (7th Edition):

Krotov, Dmitry. “Strong correlations in gravity and biophysics .” 2014. Web. 10 Jul 2020.

Vancouver:

Krotov D. Strong correlations in gravity and biophysics . [Internet] [Doctoral dissertation]. Princeton University; 2014. [cited 2020 Jul 10]. Available from: http://arks.princeton.edu/ark:/88435/dsp01nv935507k.

Council of Science Editors:

Krotov D. Strong correlations in gravity and biophysics . [Doctoral Dissertation]. Princeton University; 2014. Available from: http://arks.princeton.edu/ark:/88435/dsp01nv935507k


University of Maine

19. Roy, Tyler A. Discovery of Distinct Mechanisms Underlying the Relationship Between Drug Taking and Predisposing Behaviors.

Degree: MS, Biochemistry, 2020, University of Maine

  Drug addiction is a heritable disease characterized by compulsive drug use. The biological mechanisms driving addiction remain largely unknown.1 Previous studies show shared genetic(more)

Subjects/Keywords: MIce; Drugs; Addiction; Behavior; Gene Knockouts; Animals; Behavior and Behavior Mechanisms; Genetic Phenomena; Mental Disorders

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APA (6th Edition):

Roy, T. A. (2020). Discovery of Distinct Mechanisms Underlying the Relationship Between Drug Taking and Predisposing Behaviors. (Masters Thesis). University of Maine. Retrieved from https://digitalcommons.library.umaine.edu/etd/3168

Chicago Manual of Style (16th Edition):

Roy, Tyler A. “Discovery of Distinct Mechanisms Underlying the Relationship Between Drug Taking and Predisposing Behaviors.” 2020. Masters Thesis, University of Maine. Accessed July 10, 2020. https://digitalcommons.library.umaine.edu/etd/3168.

MLA Handbook (7th Edition):

Roy, Tyler A. “Discovery of Distinct Mechanisms Underlying the Relationship Between Drug Taking and Predisposing Behaviors.” 2020. Web. 10 Jul 2020.

Vancouver:

Roy TA. Discovery of Distinct Mechanisms Underlying the Relationship Between Drug Taking and Predisposing Behaviors. [Internet] [Masters thesis]. University of Maine; 2020. [cited 2020 Jul 10]. Available from: https://digitalcommons.library.umaine.edu/etd/3168.

Council of Science Editors:

Roy TA. Discovery of Distinct Mechanisms Underlying the Relationship Between Drug Taking and Predisposing Behaviors. [Masters Thesis]. University of Maine; 2020. Available from: https://digitalcommons.library.umaine.edu/etd/3168

20. Yildirim, Ozlem. Chromatin Dynamics in Pluripotency and Differentiation: A Dissertation.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2012, U of Massachusetts : Med

  Different cell types in multi-cellular organisms heritably maintain different gene expression patterns despite carrying the same genome; a phenomenon termed epigenetics. It is widely… (more)

Subjects/Keywords: Chromatin; Embryonic Stem Cells; Epigenesis; Genetic; Amino Acids, Peptides, and Proteins; Cell and Developmental Biology; Cells; Genetic Phenomena; Genetics and Genomics

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APA (6th Edition):

Yildirim, O. (2012). Chromatin Dynamics in Pluripotency and Differentiation: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/623

Chicago Manual of Style (16th Edition):

Yildirim, Ozlem. “Chromatin Dynamics in Pluripotency and Differentiation: A Dissertation.” 2012. Doctoral Dissertation, U of Massachusetts : Med. Accessed July 10, 2020. https://escholarship.umassmed.edu/gsbs_diss/623.

MLA Handbook (7th Edition):

Yildirim, Ozlem. “Chromatin Dynamics in Pluripotency and Differentiation: A Dissertation.” 2012. Web. 10 Jul 2020.

Vancouver:

Yildirim O. Chromatin Dynamics in Pluripotency and Differentiation: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2012. [cited 2020 Jul 10]. Available from: https://escholarship.umassmed.edu/gsbs_diss/623.

Council of Science Editors:

Yildirim O. Chromatin Dynamics in Pluripotency and Differentiation: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2012. Available from: https://escholarship.umassmed.edu/gsbs_diss/623


West Virginia University

21. Sundar, Jesse C. Alternative Splicing in Vertebrate Photoreceptors and Mechanisms Underlying Retinitis Pigmentosa.

Degree: PhD, Biochemistry, 2019, West Virginia University

  RNA binding proteins (RBPs) have emerged as important regulators of gene expression. RBPs typically contain RNA binding domains that recognize a specific sequence and/or… (more)

Subjects/Keywords: Alternative Splicing; Musashi; RNA Binding Protein; BBS8; Retina; Photoreceptor; Outer Segment; Rhodopsin; Transducin; Retinitis Pigmentosa; Genetic Phenomena; Genetic Processes

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APA (6th Edition):

Sundar, J. C. (2019). Alternative Splicing in Vertebrate Photoreceptors and Mechanisms Underlying Retinitis Pigmentosa. (Doctoral Dissertation). West Virginia University. Retrieved from https://researchrepository.wvu.edu/etd/7661 ; https://doi.org/10.33915/etd.7661

Chicago Manual of Style (16th Edition):

Sundar, Jesse C. “Alternative Splicing in Vertebrate Photoreceptors and Mechanisms Underlying Retinitis Pigmentosa.” 2019. Doctoral Dissertation, West Virginia University. Accessed July 10, 2020. https://researchrepository.wvu.edu/etd/7661 ; https://doi.org/10.33915/etd.7661.

MLA Handbook (7th Edition):

Sundar, Jesse C. “Alternative Splicing in Vertebrate Photoreceptors and Mechanisms Underlying Retinitis Pigmentosa.” 2019. Web. 10 Jul 2020.

Vancouver:

Sundar JC. Alternative Splicing in Vertebrate Photoreceptors and Mechanisms Underlying Retinitis Pigmentosa. [Internet] [Doctoral dissertation]. West Virginia University; 2019. [cited 2020 Jul 10]. Available from: https://researchrepository.wvu.edu/etd/7661 ; https://doi.org/10.33915/etd.7661.

Council of Science Editors:

Sundar JC. Alternative Splicing in Vertebrate Photoreceptors and Mechanisms Underlying Retinitis Pigmentosa. [Doctoral Dissertation]. West Virginia University; 2019. Available from: https://researchrepository.wvu.edu/etd/7661 ; https://doi.org/10.33915/etd.7661


University of Louisville

22. Carlisle, Samantha Marie. Deciphering the role of human arylamine N-acetyltransferase 1 (NAT1) in breast cancer cell metabolism using a systems biology approach.

Degree: PhD, 2018, University of Louisville

  Background: Human arylamine N-acetyltransferase 1 (NAT1) is a phase II xenobiotic metabolizing enzyme found in almost all tissues. NAT1 can additionally hydrolyze acetyl-coenzyme A… (more)

Subjects/Keywords: NAT1; breast cancer; cellular metabolism; omics; systems biology; bioinformatics; Biochemical Phenomena, Metabolism, and Nutrition; Biochemistry; Bioinformatics; Biological Phenomena, Cell Phenomena, and Immunity; Cancer Biology; Genetic Phenomena; Genetics and Genomics; Molecular Biology; Pharmacology, Toxicology and Environmental Health; Systems Biology

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APA (6th Edition):

Carlisle, S. M. (2018). Deciphering the role of human arylamine N-acetyltransferase 1 (NAT1) in breast cancer cell metabolism using a systems biology approach. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/3022 ; https://ir.library.louisville.edu/etd/3022

Chicago Manual of Style (16th Edition):

Carlisle, Samantha Marie. “Deciphering the role of human arylamine N-acetyltransferase 1 (NAT1) in breast cancer cell metabolism using a systems biology approach.” 2018. Doctoral Dissertation, University of Louisville. Accessed July 10, 2020. 10.18297/etd/3022 ; https://ir.library.louisville.edu/etd/3022.

MLA Handbook (7th Edition):

Carlisle, Samantha Marie. “Deciphering the role of human arylamine N-acetyltransferase 1 (NAT1) in breast cancer cell metabolism using a systems biology approach.” 2018. Web. 10 Jul 2020.

Vancouver:

Carlisle SM. Deciphering the role of human arylamine N-acetyltransferase 1 (NAT1) in breast cancer cell metabolism using a systems biology approach. [Internet] [Doctoral dissertation]. University of Louisville; 2018. [cited 2020 Jul 10]. Available from: 10.18297/etd/3022 ; https://ir.library.louisville.edu/etd/3022.

Council of Science Editors:

Carlisle SM. Deciphering the role of human arylamine N-acetyltransferase 1 (NAT1) in breast cancer cell metabolism using a systems biology approach. [Doctoral Dissertation]. University of Louisville; 2018. Available from: 10.18297/etd/3022 ; https://ir.library.louisville.edu/etd/3022


Texas Medical Center

23. Westin, Jason R. Correlation Matrix Analysis Identifies Gene Signatures of Immune Cell Subsets and Their Interactions in Follicular Lymphoma.

Degree: MS, 2015, Texas Medical Center

  There are important but ill-defined interactions between benign immune cell subsets and neoplastic B cells within follicular lymphoma (FL). Using the novel technique of… (more)

Subjects/Keywords: Follicular lymphoma; lymphoma; correlation matrix; gene signatures; gene expression profiling; immune cells; gene signatures; B-cell lymphoma; Biological Phenomena, Cell Phenomena, and Immunity; Diagnosis; Genetic Phenomena; Genetic Processes; Hematology; Investigative Techniques; Medical Genetics; Medicine and Health Sciences; Oncology; Other Analytical, Diagnostic and Therapeutic Techniques and Equipment; Therapeutics

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APA (6th Edition):

Westin, J. R. (2015). Correlation Matrix Analysis Identifies Gene Signatures of Immune Cell Subsets and Their Interactions in Follicular Lymphoma. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/585

Chicago Manual of Style (16th Edition):

Westin, Jason R. “Correlation Matrix Analysis Identifies Gene Signatures of Immune Cell Subsets and Their Interactions in Follicular Lymphoma.” 2015. Masters Thesis, Texas Medical Center. Accessed July 10, 2020. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/585.

MLA Handbook (7th Edition):

Westin, Jason R. “Correlation Matrix Analysis Identifies Gene Signatures of Immune Cell Subsets and Their Interactions in Follicular Lymphoma.” 2015. Web. 10 Jul 2020.

Vancouver:

Westin JR. Correlation Matrix Analysis Identifies Gene Signatures of Immune Cell Subsets and Their Interactions in Follicular Lymphoma. [Internet] [Masters thesis]. Texas Medical Center; 2015. [cited 2020 Jul 10]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/585.

Council of Science Editors:

Westin JR. Correlation Matrix Analysis Identifies Gene Signatures of Immune Cell Subsets and Their Interactions in Follicular Lymphoma. [Masters Thesis]. Texas Medical Center; 2015. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/585


Texas Medical Center

24. Mayes, Sarah, BA. Obstetrician and Gynecologist Utilization of the NIPT Expanded Testing Option.

Degree: MS, 2015, Texas Medical Center

  Noninvasive prenatal testing (NIPT) enables the detection of common fetal aneuploidies such as trisomy 21, trisomy 18, trisomy 13, and sex chromosome abnormalities via… (more)

Subjects/Keywords: NIPT; noninvasive prenatal testing; maternal serum; obstetrics and gynecology; prenatal screen; genetic counseling; microdeletion; expanded NIPT; trisomy; genetic; Genetic Phenomena; Medical Education; Medical Genetics; Medicine and Health Sciences

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APA (6th Edition):

Mayes, Sarah, B. (2015). Obstetrician and Gynecologist Utilization of the NIPT Expanded Testing Option. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/575

Chicago Manual of Style (16th Edition):

Mayes, Sarah, BA. “Obstetrician and Gynecologist Utilization of the NIPT Expanded Testing Option.” 2015. Masters Thesis, Texas Medical Center. Accessed July 10, 2020. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/575.

MLA Handbook (7th Edition):

Mayes, Sarah, BA. “Obstetrician and Gynecologist Utilization of the NIPT Expanded Testing Option.” 2015. Web. 10 Jul 2020.

Vancouver:

Mayes, Sarah B. Obstetrician and Gynecologist Utilization of the NIPT Expanded Testing Option. [Internet] [Masters thesis]. Texas Medical Center; 2015. [cited 2020 Jul 10]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/575.

Council of Science Editors:

Mayes, Sarah B. Obstetrician and Gynecologist Utilization of the NIPT Expanded Testing Option. [Masters Thesis]. Texas Medical Center; 2015. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/575

25. Yu, Lijian. The Role of PC4 in Oxidative Stress: A Dissertation.

Degree: Molecular Genetics and Microbiology, Microbiology and Physiological Systems, 2011, U of Massachusetts : Med

  Oxidative stress is a cellular condition where cells are challenged by elevated levels of reactive oxygen species (ROS) that are produced endogenously or exogenously.… (more)

Subjects/Keywords: Oxidative Stress; DNA Damage; DNA-Binding Proteins; Subtilisins; Proprotein Convertases; Polyadenylation; Biochemical Phenomena, Metabolism, and Nutrition; Biochemistry, Biophysics, and Structural Biology; Cells; Enzymes and Coenzymes; Genetic Phenomena

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APA (6th Edition):

Yu, L. (2011). The Role of PC4 in Oxidative Stress: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/545

Chicago Manual of Style (16th Edition):

Yu, Lijian. “The Role of PC4 in Oxidative Stress: A Dissertation.” 2011. Doctoral Dissertation, U of Massachusetts : Med. Accessed July 10, 2020. https://escholarship.umassmed.edu/gsbs_diss/545.

MLA Handbook (7th Edition):

Yu, Lijian. “The Role of PC4 in Oxidative Stress: A Dissertation.” 2011. Web. 10 Jul 2020.

Vancouver:

Yu L. The Role of PC4 in Oxidative Stress: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2011. [cited 2020 Jul 10]. Available from: https://escholarship.umassmed.edu/gsbs_diss/545.

Council of Science Editors:

Yu L. The Role of PC4 in Oxidative Stress: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2011. Available from: https://escholarship.umassmed.edu/gsbs_diss/545

26. Sage, Jay M. Support of Mitochondrial DNA Replication by Human Rad51: A Dissertation.

Degree: Biochemistry and Molecular Pharmacology, Program in Biochemistry and Molecular Pharmacology, 2011, U of Massachusetts : Med

  The function of homologous DNA recombination in human mitochondria has been a topic of ongoing debate for many years, with implications for fields ranging… (more)

Subjects/Keywords: Rad51 Recombinase; Protein Transport; DNA; Mitochondrial; Amino Acids, Peptides, and Proteins; Biochemical Phenomena, Metabolism, and Nutrition; Biochemistry, Biophysics, and Structural Biology; Cells; Enzymes and Coenzymes; Genetic Phenomena

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APA (6th Edition):

Sage, J. M. (2011). Support of Mitochondrial DNA Replication by Human Rad51: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/574

Chicago Manual of Style (16th Edition):

Sage, Jay M. “Support of Mitochondrial DNA Replication by Human Rad51: A Dissertation.” 2011. Doctoral Dissertation, U of Massachusetts : Med. Accessed July 10, 2020. https://escholarship.umassmed.edu/gsbs_diss/574.

MLA Handbook (7th Edition):

Sage, Jay M. “Support of Mitochondrial DNA Replication by Human Rad51: A Dissertation.” 2011. Web. 10 Jul 2020.

Vancouver:

Sage JM. Support of Mitochondrial DNA Replication by Human Rad51: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2011. [cited 2020 Jul 10]. Available from: https://escholarship.umassmed.edu/gsbs_diss/574.

Council of Science Editors:

Sage JM. Support of Mitochondrial DNA Replication by Human Rad51: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2011. Available from: https://escholarship.umassmed.edu/gsbs_diss/574

27. Lin, Chien-Ling. Studies on the Regulation of Cytoplasmic Polyadenylation Element-Binding Protein: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2012, U of Massachusetts : Med

  Post-transcriptional regulation of gene expression sits at the core of proteomic complexity; trans-acting factors that regulate RNA localization and translation capacity are thus indispensible.… (more)

Subjects/Keywords: RNA-Binding Proteins; Poly(A)-Binding Proteins; Polyadenylation; Amino Acids, Peptides, and Proteins; Biochemical Phenomena, Metabolism, and Nutrition; Biochemistry, Biophysics, and Structural Biology; Genetic Phenomena; Nucleic Acids, Nucleotides, and Nucleosides

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APA (6th Edition):

Lin, C. (2012). Studies on the Regulation of Cytoplasmic Polyadenylation Element-Binding Protein: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/583

Chicago Manual of Style (16th Edition):

Lin, Chien-Ling. “Studies on the Regulation of Cytoplasmic Polyadenylation Element-Binding Protein: A Dissertation.” 2012. Doctoral Dissertation, U of Massachusetts : Med. Accessed July 10, 2020. https://escholarship.umassmed.edu/gsbs_diss/583.

MLA Handbook (7th Edition):

Lin, Chien-Ling. “Studies on the Regulation of Cytoplasmic Polyadenylation Element-Binding Protein: A Dissertation.” 2012. Web. 10 Jul 2020.

Vancouver:

Lin C. Studies on the Regulation of Cytoplasmic Polyadenylation Element-Binding Protein: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2012. [cited 2020 Jul 10]. Available from: https://escholarship.umassmed.edu/gsbs_diss/583.

Council of Science Editors:

Lin C. Studies on the Regulation of Cytoplasmic Polyadenylation Element-Binding Protein: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2012. Available from: https://escholarship.umassmed.edu/gsbs_diss/583


University of Kentucky

28. Syam, Diana. Immobilizing Mutation in an Unconventional Myosin15a Affects not only the Structure of Mechanosensory Stereocilia in the Inner Ear Hair Cells but also their Ionic Conductances.

Degree: 2014, University of Kentucky

 In the inner and outer hair cells (OHCs) of the inner ear, an unconventional myosin 15a localizes at the tips of mechanosensory stereocilia and plays… (more)

Subjects/Keywords: Organ of Corti; Outer Hair Cells; Myosin15a; Shaker-2 Mice; Patch-Clamp.; Genetic Phenomena; Medical Cell Biology; Other Medical Sciences

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APA (6th Edition):

Syam, D. (2014). Immobilizing Mutation in an Unconventional Myosin15a Affects not only the Structure of Mechanosensory Stereocilia in the Inner Ear Hair Cells but also their Ionic Conductances. (Masters Thesis). University of Kentucky. Retrieved from http://uknowledge.uky.edu/medsci_etds/2

Chicago Manual of Style (16th Edition):

Syam, Diana. “Immobilizing Mutation in an Unconventional Myosin15a Affects not only the Structure of Mechanosensory Stereocilia in the Inner Ear Hair Cells but also their Ionic Conductances.” 2014. Masters Thesis, University of Kentucky. Accessed July 10, 2020. http://uknowledge.uky.edu/medsci_etds/2.

MLA Handbook (7th Edition):

Syam, Diana. “Immobilizing Mutation in an Unconventional Myosin15a Affects not only the Structure of Mechanosensory Stereocilia in the Inner Ear Hair Cells but also their Ionic Conductances.” 2014. Web. 10 Jul 2020.

Vancouver:

Syam D. Immobilizing Mutation in an Unconventional Myosin15a Affects not only the Structure of Mechanosensory Stereocilia in the Inner Ear Hair Cells but also their Ionic Conductances. [Internet] [Masters thesis]. University of Kentucky; 2014. [cited 2020 Jul 10]. Available from: http://uknowledge.uky.edu/medsci_etds/2.

Council of Science Editors:

Syam D. Immobilizing Mutation in an Unconventional Myosin15a Affects not only the Structure of Mechanosensory Stereocilia in the Inner Ear Hair Cells but also their Ionic Conductances. [Masters Thesis]. University of Kentucky; 2014. Available from: http://uknowledge.uky.edu/medsci_etds/2

29. Selva, Erica Marie. Mismatch Repair Acts As a Barrier to Homeologous Recombination in Saccharomyces cerevisiae: A Dissertation.

Degree: Biochemistry and Molecular Pharmacology, Biochemistry & Molecular Biology, 1996, U of Massachusetts : Med

  Homeologous recombination refers to genetic exchanges between DNA partners containing similar but not identical DNA sequences. Heteroduplex intermediates in such exchanges are expected to… (more)

Subjects/Keywords: DNA; Recombinant; Saccharomyces cerevisiae; Fungi; Genetic Phenomena

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APA (6th Edition):

Selva, E. M. (1996). Mismatch Repair Acts As a Barrier to Homeologous Recombination in Saccharomyces cerevisiae: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/61

Chicago Manual of Style (16th Edition):

Selva, Erica Marie. “Mismatch Repair Acts As a Barrier to Homeologous Recombination in Saccharomyces cerevisiae: A Dissertation.” 1996. Doctoral Dissertation, U of Massachusetts : Med. Accessed July 10, 2020. https://escholarship.umassmed.edu/gsbs_diss/61.

MLA Handbook (7th Edition):

Selva, Erica Marie. “Mismatch Repair Acts As a Barrier to Homeologous Recombination in Saccharomyces cerevisiae: A Dissertation.” 1996. Web. 10 Jul 2020.

Vancouver:

Selva EM. Mismatch Repair Acts As a Barrier to Homeologous Recombination in Saccharomyces cerevisiae: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 1996. [cited 2020 Jul 10]. Available from: https://escholarship.umassmed.edu/gsbs_diss/61.

Council of Science Editors:

Selva EM. Mismatch Repair Acts As a Barrier to Homeologous Recombination in Saccharomyces cerevisiae: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 1996. Available from: https://escholarship.umassmed.edu/gsbs_diss/61

30. Ranade, Koustubh. sieB and esc genes of Bacteriophage P22: A Dissertation.

Degree: Molecular Genetics and Microbiology, Molecular Genetics and Microbiology; Program in Molecular Medicine, 1993, U of Massachusetts : Med

  The superinfection exclusion gene (sieB) of Salmonella phage P22 was mapped using phage deletion mutants. The DNA sequence in the region was re-examined in… (more)

Subjects/Keywords: Bacteriophage P22; Chromosome Mapping; Superinfection; Bacteria; Genetic Phenomena; Viruses

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ranade, K. (1993). sieB and esc genes of Bacteriophage P22: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/59

Chicago Manual of Style (16th Edition):

Ranade, Koustubh. “sieB and esc genes of Bacteriophage P22: A Dissertation.” 1993. Doctoral Dissertation, U of Massachusetts : Med. Accessed July 10, 2020. https://escholarship.umassmed.edu/gsbs_diss/59.

MLA Handbook (7th Edition):

Ranade, Koustubh. “sieB and esc genes of Bacteriophage P22: A Dissertation.” 1993. Web. 10 Jul 2020.

Vancouver:

Ranade K. sieB and esc genes of Bacteriophage P22: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 1993. [cited 2020 Jul 10]. Available from: https://escholarship.umassmed.edu/gsbs_diss/59.

Council of Science Editors:

Ranade K. sieB and esc genes of Bacteriophage P22: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 1993. Available from: https://escholarship.umassmed.edu/gsbs_diss/59

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