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You searched for subject:(Gene Expression Regulation Neoplastic 60). Showing records 1 – 30 of 27887 total matches.

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University of Texas Southwestern Medical Center

1. Dang, Tuyen Thi Thanh. Probing the Molecular Requirements for Breast Cancer Cell Motility.

Degree: 2014, University of Texas Southwestern Medical Center

 Invasion of breast cancer cells into the stroma is an early step in metastasis. How invasive ability is conferred in breast cancer cells is poorly… (more)

Subjects/Keywords: Breast Neoplasms; Cell Transformation, Neoplastic; Gene Expression Regulation, Neoplastic; Transcription Factors

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dang, T. T. T. (2014). Probing the Molecular Requirements for Breast Cancer Cell Motility. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3942

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dang, Tuyen Thi Thanh. “Probing the Molecular Requirements for Breast Cancer Cell Motility.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed February 22, 2020. http://hdl.handle.net/2152.5/3942.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dang, Tuyen Thi Thanh. “Probing the Molecular Requirements for Breast Cancer Cell Motility.” 2014. Web. 22 Feb 2020.

Vancouver:

Dang TTT. Probing the Molecular Requirements for Breast Cancer Cell Motility. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Feb 22]. Available from: http://hdl.handle.net/2152.5/3942.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dang TTT. Probing the Molecular Requirements for Breast Cancer Cell Motility. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3942

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Khotskaya, Yekaterina B. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.

Degree: PhD, 2009, University of Alabama – Birmingham

Syndecan-1 (CD138), a transmembrane heparan sulfate-bearing proteoglycan, is expressed at high levels on most myeloma cells and is shed into the microenvironment. In patients, high… (more)

Subjects/Keywords: Gene Expression Regulation, Neoplastic<; br>; Melanoma  – metabolism<; br>; Neoplasm Proteins  – biosynthesis<; br>; Neovascularization, Pathologic  – metabolism<; br>; Syndecan-1  – biosynthesis

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APA (6th Edition):

Khotskaya, Y. B. (2009). Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,766

Chicago Manual of Style (16th Edition):

Khotskaya, Yekaterina B. “Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,766.

MLA Handbook (7th Edition):

Khotskaya, Yekaterina B. “Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.” 2009. Web. 22 Feb 2020.

Vancouver:

Khotskaya YB. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,766.

Council of Science Editors:

Khotskaya YB. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,766

3. Cuddapah, Vishnu Anand. Regulation Of Clc-3 In Human Malignant Glioma.

Degree: PhD, 2012, University of Alabama – Birmingham

Malignant gliomas are the most common and deadly form of primary brain cancer afflicting adults. Current treatment regimens, including surgical debulking, radiotherapy, and chemotherapy, have… (more)

Subjects/Keywords: Brain Neoplasms – metabolism<; br>; Calcium-Calmodulin-Dependent Protein Kinase Type 2 – metabolism.<; br>; Cell Movement – physiology<; br>; Chloride Channels – metabolism.<; br>; Gene Expression Regulation<; br>; Gene Expression Regulation, Enzymologic<; br>; Gene Expression Regulation, Neoplastic<; br>; Glioma – metabolism<; br>; Ion Channels – metabolism<; br>; Membrane Transport Proteins – metabolism.<; br>; Mitosis<; br>; Neoplasms – metabolism<; br>; Neoplasms – pathology

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APA (6th Edition):

Cuddapah, V. A. (2012). Regulation Of Clc-3 In Human Malignant Glioma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1394

Chicago Manual of Style (16th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1394.

MLA Handbook (7th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Web. 22 Feb 2020.

Vancouver:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394.

Council of Science Editors:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394

4. Huffman, Derek M. Calorie restriction, exercise and body fat : effects on cancer and markers of longevity.

Degree: PhD, 2007, University of Alabama – Birmingham

The benefits of calorie restriction (CR) have historically been attributed to a reduction in food intake. More recently, reduced fat stores have been proposed as… (more)

Subjects/Keywords: Adenocarcinoma<; br>; Aging<; br>; Biological Markers  – metabolism<; br>; Caloric Restriction<; br>; Gene Expression Regulation, Neoplastic<; br>; Physical Conditioning, Animal  – physiology<; br>; Prostatic Neoplasms<; br>; Sirtuins  – genetics

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APA (6th Edition):

Huffman, D. M. (2007). Calorie restriction, exercise and body fat : effects on cancer and markers of longevity. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,596

Chicago Manual of Style (16th Edition):

Huffman, Derek M. “Calorie restriction, exercise and body fat : effects on cancer and markers of longevity.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,596.

MLA Handbook (7th Edition):

Huffman, Derek M. “Calorie restriction, exercise and body fat : effects on cancer and markers of longevity.” 2007. Web. 22 Feb 2020.

Vancouver:

Huffman DM. Calorie restriction, exercise and body fat : effects on cancer and markers of longevity. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,596.

Council of Science Editors:

Huffman DM. Calorie restriction, exercise and body fat : effects on cancer and markers of longevity. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,596


University of Texas Southwestern Medical Center

5. Paulson, Vera Ashley. High-Resolution Array Comparative Genomic Hybridization Identifies Common Targets in Rhabdomyosarcoma.

Degree: 2012, University of Texas Southwestern Medical Center

 Rhabdomyosarcoma (RMS) accounts for nearly 50 percent of the soft tissue sarcomas that affect children. There are two major histological variants, alveolar (ARMS) and embryonal… (more)

Subjects/Keywords: Rhabdomyosarcoma; Gene Expression Regulation, Neoplastic; Receptor, Fibroblast Growth Factor, Type 4

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APA (6th Edition):

Paulson, V. A. (2012). High-Resolution Array Comparative Genomic Hybridization Identifies Common Targets in Rhabdomyosarcoma. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1112

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Paulson, Vera Ashley. “High-Resolution Array Comparative Genomic Hybridization Identifies Common Targets in Rhabdomyosarcoma.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed February 22, 2020. http://hdl.handle.net/2152.5/1112.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Paulson, Vera Ashley. “High-Resolution Array Comparative Genomic Hybridization Identifies Common Targets in Rhabdomyosarcoma.” 2012. Web. 22 Feb 2020.

Vancouver:

Paulson VA. High-Resolution Array Comparative Genomic Hybridization Identifies Common Targets in Rhabdomyosarcoma. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Feb 22]. Available from: http://hdl.handle.net/2152.5/1112.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Paulson VA. High-Resolution Array Comparative Genomic Hybridization Identifies Common Targets in Rhabdomyosarcoma. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1112

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

6. Carrasco, Yazmin Paulina. Natural Products as Selective Chemotherapeutic Agents and as Chemical Probes to Understand Biological Processes.

Degree: 2013, University of Texas Southwestern Medical Center

 Aware of the important role that terrestrial microbial natural products play in the discovery of therapeutics and the decrease in rate of discovery of new… (more)

Subjects/Keywords: Gene Expression Regulation, Neoplastic; Fatty Acids, Unsaturated; Receptors, Cytoplasmic and Nuclear

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APA (6th Edition):

Carrasco, Y. P. (2013). Natural Products as Selective Chemotherapeutic Agents and as Chemical Probes to Understand Biological Processes. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/2716

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carrasco, Yazmin Paulina. “Natural Products as Selective Chemotherapeutic Agents and as Chemical Probes to Understand Biological Processes.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed February 22, 2020. http://hdl.handle.net/2152.5/2716.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carrasco, Yazmin Paulina. “Natural Products as Selective Chemotherapeutic Agents and as Chemical Probes to Understand Biological Processes.” 2013. Web. 22 Feb 2020.

Vancouver:

Carrasco YP. Natural Products as Selective Chemotherapeutic Agents and as Chemical Probes to Understand Biological Processes. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Feb 22]. Available from: http://hdl.handle.net/2152.5/2716.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carrasco YP. Natural Products as Selective Chemotherapeutic Agents and as Chemical Probes to Understand Biological Processes. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/2716

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Cody, James Joseph. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.

Degree: PhD, 2008, University of Alabama – Birmingham

Most patients with advanced breast cancer develop osteolytic bone metastases, which have numerous complications. Because current therapies are not curative, new treatments are needed. Conditionally… (more)

Subjects/Keywords: Adenoviridae  – genetics <; br>; Bone Neoplasms  – secondary <; br>; Breast Neoplasms <; br>; Gene Expression Regulation, Neoplastic <; br>; Neoplasm Metastasis  – genetics <; br>; Neoplasm Metastasis  – therapy <; br>; Virus Replication  – genetics

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APA (6th Edition):

Cody, J. J. (2008). A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,320

Chicago Manual of Style (16th Edition):

Cody, James Joseph. “A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,320.

MLA Handbook (7th Edition):

Cody, James Joseph. “A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.” 2008. Web. 22 Feb 2020.

Vancouver:

Cody JJ. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,320.

Council of Science Editors:

Cody JJ. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,320

8. Nash, Kevin T. (Kevin Tyler). KISS1 metastasis suppressor secretion is required for metastasis suppression.

Degree: PhD, 2006, University of Alabama – Birmingham

Failure to reduce the number of cancer deaths over the last 50 years is due to the inability to selectively target metastatic disease. Recently, the… (more)

Subjects/Keywords: Gene Expression Regulation, Neoplastic <; br>; Melanoma  – metabolism <; br>; Melanoma  – secretion <; br>; Neoplasm Metastasis  – prevention & control <; br>; Proteins  – physiology <; br>; Receptors, G-Protein-Coupled  – metabolism <; br>; Tumor Suppressor Proteins  – secretion

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APA (6th Edition):

Nash, K. T. (. T. (2006). KISS1 metastasis suppressor secretion is required for metastasis suppression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,380

Chicago Manual of Style (16th Edition):

Nash, Kevin T (Kevin Tyler). “KISS1 metastasis suppressor secretion is required for metastasis suppression.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,380.

MLA Handbook (7th Edition):

Nash, Kevin T (Kevin Tyler). “KISS1 metastasis suppressor secretion is required for metastasis suppression.” 2006. Web. 22 Feb 2020.

Vancouver:

Nash KT(T. KISS1 metastasis suppressor secretion is required for metastasis suppression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,380.

Council of Science Editors:

Nash KT(T. KISS1 metastasis suppressor secretion is required for metastasis suppression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,380

9. Spratling, Patsy M. Cardiovascular Disease Knowledge And Risk Perception Among Women With Recent Preeclampsia: Interventional Education In Disease Management And Prevention.

Degree: PhD, 2012, University of Alabama – Birmingham

There is a growing body of evidence linking preeclampsia to future development of cardiovascular disease (CVD). Although CVD is well-known as the leading cause of… (more)

Subjects/Keywords: Breast Neoplasms<; br>; Gene Expression Regulation, Neoplastic<; br>; MicroRNAs – genetics.<; br>; Neoplasm Metastasis – prevention & control<; br>; Neoplasm Proteins – physiology.<; br>; RNA, Neoplasm – genetics.

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APA (6th Edition):

Spratling, P. M. (2012). Cardiovascular Disease Knowledge And Risk Perception Among Women With Recent Preeclampsia: Interventional Education In Disease Management And Prevention. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1405

Chicago Manual of Style (16th Edition):

Spratling, Patsy M. “Cardiovascular Disease Knowledge And Risk Perception Among Women With Recent Preeclampsia: Interventional Education In Disease Management And Prevention.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1405.

MLA Handbook (7th Edition):

Spratling, Patsy M. “Cardiovascular Disease Knowledge And Risk Perception Among Women With Recent Preeclampsia: Interventional Education In Disease Management And Prevention.” 2012. Web. 22 Feb 2020.

Vancouver:

Spratling PM. Cardiovascular Disease Knowledge And Risk Perception Among Women With Recent Preeclampsia: Interventional Education In Disease Management And Prevention. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1405.

Council of Science Editors:

Spratling PM. Cardiovascular Disease Knowledge And Risk Perception Among Women With Recent Preeclampsia: Interventional Education In Disease Management And Prevention. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1405


University of Texas Southwestern Medical Center

10. Hight, Suzie K. An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer.

Degree: 2015, University of Texas Southwestern Medical Center

 Cancer cells are characterized by the aberrant regulation of signaling pathways that govern responses to growth stimuli, resulting in dysregulated cellular proliferation. The accumulated genomic… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; Gene Expression Regulation, Neoplastic; Lung Neoplasms; Receptors, Cytoplasmic and Nuclear

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APA (6th Edition):

Hight, S. K. (2015). An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hight, Suzie K. “An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed February 22, 2020. http://hdl.handle.net/2152.5/4462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hight, Suzie K. “An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer.” 2015. Web. 22 Feb 2020.

Vancouver:

Hight SK. An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Feb 22]. Available from: http://hdl.handle.net/2152.5/4462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hight SK. An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Moriarty, Charlotte M. Harwood. Functional Analysis of MicroRNA-10b in Breast Carcinoma: A Dissertation.

Degree: PhD, Molecular, Cell and Cancer Biology, 2009, U of Massachusetts : Med

  MicroRNAs (miRNAs) represent a class of small noncoding RNAs that regulate gene expression. Recent studies have shown that miRNAs are mis-expressed in various human… (more)

Subjects/Keywords: Breast Neoplasms; Gene Expression Regulation; Neoplastic; MicroRNAs; Neoplasm Metastasis; Tumor Markers; Biological; Genetic Phenomena; Neoplasms; Skin and Connective Tissue Diseases

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APA (6th Edition):

Moriarty, C. M. H. (2009). Functional Analysis of MicroRNA-10b in Breast Carcinoma: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/426

Chicago Manual of Style (16th Edition):

Moriarty, Charlotte M Harwood. “Functional Analysis of MicroRNA-10b in Breast Carcinoma: A Dissertation.” 2009. Doctoral Dissertation, U of Massachusetts : Med. Accessed February 22, 2020. https://escholarship.umassmed.edu/gsbs_diss/426.

MLA Handbook (7th Edition):

Moriarty, Charlotte M Harwood. “Functional Analysis of MicroRNA-10b in Breast Carcinoma: A Dissertation.” 2009. Web. 22 Feb 2020.

Vancouver:

Moriarty CMH. Functional Analysis of MicroRNA-10b in Breast Carcinoma: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2009. [cited 2020 Feb 22]. Available from: https://escholarship.umassmed.edu/gsbs_diss/426.

Council of Science Editors:

Moriarty CMH. Functional Analysis of MicroRNA-10b in Breast Carcinoma: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2009. Available from: https://escholarship.umassmed.edu/gsbs_diss/426

12. Guha, Minakshi. Regulation of Cancer Cell Survival Mediated by Endogenous Tumor Suppression: A Dissertation.

Degree: Cancer Biology, Department of Cancer Biology, 2009, U of Massachusetts : Med

  Cancer is the second leading cause of death among men and women after heart disease. Though our knowledge associated with the complexities of the… (more)

Subjects/Keywords: Cell Transformation; Neoplastic; Caspase 2; Microtubule-Associated Proteins; PTEN Phosphohydrolase; Gene Expression Regulation; Neoplastic; Apoptosis; Suppression; Genetic; Amino Acids, Peptides, and Proteins; Cells; Enzymes and Coenzymes; Genetic Phenomena; Neoplasms

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APA (6th Edition):

Guha, M. (2009). Regulation of Cancer Cell Survival Mediated by Endogenous Tumor Suppression: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/431

Chicago Manual of Style (16th Edition):

Guha, Minakshi. “Regulation of Cancer Cell Survival Mediated by Endogenous Tumor Suppression: A Dissertation.” 2009. Doctoral Dissertation, U of Massachusetts : Med. Accessed February 22, 2020. https://escholarship.umassmed.edu/gsbs_diss/431.

MLA Handbook (7th Edition):

Guha, Minakshi. “Regulation of Cancer Cell Survival Mediated by Endogenous Tumor Suppression: A Dissertation.” 2009. Web. 22 Feb 2020.

Vancouver:

Guha M. Regulation of Cancer Cell Survival Mediated by Endogenous Tumor Suppression: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2009. [cited 2020 Feb 22]. Available from: https://escholarship.umassmed.edu/gsbs_diss/431.

Council of Science Editors:

Guha M. Regulation of Cancer Cell Survival Mediated by Endogenous Tumor Suppression: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2009. Available from: https://escholarship.umassmed.edu/gsbs_diss/431


Texas A&M University

13. Samuel, Shaija. Studies on gene expression profiling in JB6 cells susceptible and resistant to tumor promoter induced neoplastic transformation and regulation of gene expression at the AP-1 DNA binding site.

Degree: 2005, Texas A&M University

Gene expression underlies all important biological processes in a cell and mis-regulated gene expression plays a causal or contributory role in several diseases including cancers.… (more)

Subjects/Keywords: gene expression; neoplastic transformation; JB6 cells; AP-1; Regulation of gene; Microarray

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APA (6th Edition):

Samuel, S. (2005). Studies on gene expression profiling in JB6 cells susceptible and resistant to tumor promoter induced neoplastic transformation and regulation of gene expression at the AP-1 DNA binding site. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/2538

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Samuel, Shaija. “Studies on gene expression profiling in JB6 cells susceptible and resistant to tumor promoter induced neoplastic transformation and regulation of gene expression at the AP-1 DNA binding site.” 2005. Thesis, Texas A&M University. Accessed February 22, 2020. http://hdl.handle.net/1969.1/2538.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Samuel, Shaija. “Studies on gene expression profiling in JB6 cells susceptible and resistant to tumor promoter induced neoplastic transformation and regulation of gene expression at the AP-1 DNA binding site.” 2005. Web. 22 Feb 2020.

Vancouver:

Samuel S. Studies on gene expression profiling in JB6 cells susceptible and resistant to tumor promoter induced neoplastic transformation and regulation of gene expression at the AP-1 DNA binding site. [Internet] [Thesis]. Texas A&M University; 2005. [cited 2020 Feb 22]. Available from: http://hdl.handle.net/1969.1/2538.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Samuel S. Studies on gene expression profiling in JB6 cells susceptible and resistant to tumor promoter induced neoplastic transformation and regulation of gene expression at the AP-1 DNA binding site. [Thesis]. Texas A&M University; 2005. Available from: http://hdl.handle.net/1969.1/2538

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. McAlear, Suzanne D. (Suzanne DuChai). Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation.

Degree: PhD, 2007, University of Alabama – Birmingham

Electrogenic Na/bicarbonate cotransporters (NBCes) are important regulators of intracellular pH in many tissues including the kidney, brain, and pancreas, and are members of a superfamily… (more)

Subjects/Keywords: Cysteine  – metabolism<; br>; Gene Expression Regulation  – physiology<; br>; Genetic Variation  – physiology<; br>; Mutagenesis<; br>; Oocytes<; br>; Sodium-Bicarbonate Symporters

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APA (6th Edition):

McAlear, S. D. (. D. (2007). Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,456

Chicago Manual of Style (16th Edition):

McAlear, Suzanne D (Suzanne DuChai). “Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,456.

MLA Handbook (7th Edition):

McAlear, Suzanne D (Suzanne DuChai). “Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation.” 2007. Web. 22 Feb 2020.

Vancouver:

McAlear SD(D. Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,456.

Council of Science Editors:

McAlear SD(D. Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,456

15. Thomas, Holly Reed. Genetic and epigenetic regulation of dihydropyrimidinase and beta-ureidopropionase in individuals with altered uracil catabolism and normal dihydropyrimidine dehydrogenase enzyme activity.

Degree: PhD, 2007, University of Alabama – Birmingham

There remain a number of patients presenting with 5-fluorouracil (5-FU) toxicity despite normal dihydropyrimidine dehydrogenase (DPD) enzyme activity, suggesting possible deficiencies in the two enzymes… (more)

Subjects/Keywords: Amidohydrolases  – genetics <; br>; Antineoplastic Agents  – metabolism <; br>; Dihydrouracil Dehydrogenase (NADP)  – genetics <; br>; Gene Expression Regulation <; br>; Gene Expression Regulation, Enzymologic <; br>; Uracil  – metabolism

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APA (6th Edition):

Thomas, H. R. (2007). Genetic and epigenetic regulation of dihydropyrimidinase and beta-ureidopropionase in individuals with altered uracil catabolism and normal dihydropyrimidine dehydrogenase enzyme activity. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,264

Chicago Manual of Style (16th Edition):

Thomas, Holly Reed. “Genetic and epigenetic regulation of dihydropyrimidinase and beta-ureidopropionase in individuals with altered uracil catabolism and normal dihydropyrimidine dehydrogenase enzyme activity.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,264.

MLA Handbook (7th Edition):

Thomas, Holly Reed. “Genetic and epigenetic regulation of dihydropyrimidinase and beta-ureidopropionase in individuals with altered uracil catabolism and normal dihydropyrimidine dehydrogenase enzyme activity.” 2007. Web. 22 Feb 2020.

Vancouver:

Thomas HR. Genetic and epigenetic regulation of dihydropyrimidinase and beta-ureidopropionase in individuals with altered uracil catabolism and normal dihydropyrimidine dehydrogenase enzyme activity. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,264.

Council of Science Editors:

Thomas HR. Genetic and epigenetic regulation of dihydropyrimidinase and beta-ureidopropionase in individuals with altered uracil catabolism and normal dihydropyrimidine dehydrogenase enzyme activity. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,264

16. He, Ti. Molecular regulation of Pax5-mediated biological functions.

Degree: PhD, 2008, University of Alabama – Birmingham

B lineage cells are major players in the adaptive immune system. Pax5 is essential for B lineage cell development and function. Pax5 controls B lineage… (more)

Subjects/Keywords: B-Cell-Specific Activator Protein <; br>; Gene Expression Regulation <; br>; Histone Acetyltransferases  – metabolism <; br>; Transcription Factors <; br>; Transcription, Genetic

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APA (6th Edition):

He, T. (2008). Molecular regulation of Pax5-mediated biological functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,329

Chicago Manual of Style (16th Edition):

He, Ti. “Molecular regulation of Pax5-mediated biological functions.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,329.

MLA Handbook (7th Edition):

He, Ti. “Molecular regulation of Pax5-mediated biological functions.” 2008. Web. 22 Feb 2020.

Vancouver:

He T. Molecular regulation of Pax5-mediated biological functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,329.

Council of Science Editors:

He T. Molecular regulation of Pax5-mediated biological functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,329

17. Whitsett, Timothy Glynn. Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination.

Degree: PhD, 2007, University of Alabama – Birmingham

It has been established that the environment, including the diet, plays a critical role in a woman’s risk of breast cancer. Two dietary polyphenols that… (more)

Subjects/Keywords: Chemoprevention  – methods<; br>; Gene Expression Regulation  – drug effects<; br>; Genistein  – therapeutic use<; br>; Mammary Neoplasms, Experimental  – prevention & control<; br>; Stilbenes  – therapeutic use

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APA (6th Edition):

Whitsett, T. G. (2007). Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,539

Chicago Manual of Style (16th Edition):

Whitsett, Timothy Glynn. “Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,539.

MLA Handbook (7th Edition):

Whitsett, Timothy Glynn. “Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination.” 2007. Web. 22 Feb 2020.

Vancouver:

Whitsett TG. Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,539.

Council of Science Editors:

Whitsett TG. Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,539

18. Ho, Shiuh-Rong. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.

Degree: PhD, 2010, University of Alabama – Birmingham

O-GlcNAcylation is an abundant and dynamic post-translational modification on serine and threonine residues of nuclear and cytoplasmic proteins. O-GlcNAc Transferase (OGT) and Nuclear Cytoplasmic O-GlcNAcase… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism<; br>; Gene Expression Regulation<; br>; Oxidative Stress  – genetics<; br>; Transcription Factors  – metabolism<; br>; Transcription, Genetic

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APA (6th Edition):

Ho, S. (2010). O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1162

Chicago Manual of Style (16th Edition):

Ho, Shiuh-Rong. “O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1162.

MLA Handbook (7th Edition):

Ho, Shiuh-Rong. “O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.” 2010. Web. 22 Feb 2020.

Vancouver:

Ho S. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1162.

Council of Science Editors:

Ho S. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1162


University of Texas Southwestern Medical Center

19. Topalovski, Mary. Fibulin-5 Promotes Pancreatic Tumor Growth through Inhibition of Integrin-induced ROS: Insights into Tumor-Matrix Signaling.

Degree: 2016, University of Texas Southwestern Medical Center

 Elevated oxidative stress is an aberration seen in many solid tumors, and exploiting this biochemical difference has the potential to enhance the efficacy of anti-cancer… (more)

Subjects/Keywords: Carcinoma, Pancreatic Ductal; Extracellular Matrix Proteins; Gene Expression Regulation, Neoplastic; Neoplasms, Experimental; Pancreatic Neoplasms; Reactive Oxygen Species; Recombinant Proteins; Signal Transduction; Transforming Growth Factor beta

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APA (6th Edition):

Topalovski, M. (2016). Fibulin-5 Promotes Pancreatic Tumor Growth through Inhibition of Integrin-induced ROS: Insights into Tumor-Matrix Signaling. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Topalovski, Mary. “Fibulin-5 Promotes Pancreatic Tumor Growth through Inhibition of Integrin-induced ROS: Insights into Tumor-Matrix Signaling.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed February 22, 2020. http://hdl.handle.net/2152.5/5737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Topalovski, Mary. “Fibulin-5 Promotes Pancreatic Tumor Growth through Inhibition of Integrin-induced ROS: Insights into Tumor-Matrix Signaling.” 2016. Web. 22 Feb 2020.

Vancouver:

Topalovski M. Fibulin-5 Promotes Pancreatic Tumor Growth through Inhibition of Integrin-induced ROS: Insights into Tumor-Matrix Signaling. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Feb 22]. Available from: http://hdl.handle.net/2152.5/5737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Topalovski M. Fibulin-5 Promotes Pancreatic Tumor Growth through Inhibition of Integrin-induced ROS: Insights into Tumor-Matrix Signaling. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

20. Young, Melissa Rasar. Paxillin is a Novel Regulator of Xenopus Oocyte Maturation.

Degree: 2010, University of Texas Southwestern Medical Center

 Oocyte maturation is triggered by steroids in a transcription-independent fashion that involves an unusual positive feedback loop whereby MOS (a germ cell specific Raf) activates… (more)

Subjects/Keywords: Oocytes; Paxillin; Gene Expression Regulation

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APA (6th Edition):

Young, M. R. (2010). Paxillin is a Novel Regulator of Xenopus Oocyte Maturation. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/786

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Young, Melissa Rasar. “Paxillin is a Novel Regulator of Xenopus Oocyte Maturation.” 2010. Thesis, University of Texas Southwestern Medical Center. Accessed February 22, 2020. http://hdl.handle.net/2152.5/786.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Young, Melissa Rasar. “Paxillin is a Novel Regulator of Xenopus Oocyte Maturation.” 2010. Web. 22 Feb 2020.

Vancouver:

Young MR. Paxillin is a Novel Regulator of Xenopus Oocyte Maturation. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2010. [cited 2020 Feb 22]. Available from: http://hdl.handle.net/2152.5/786.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Young MR. Paxillin is a Novel Regulator of Xenopus Oocyte Maturation. [Thesis]. University of Texas Southwestern Medical Center; 2010. Available from: http://hdl.handle.net/2152.5/786

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

21. Caratozzolo, Rose Marie, 1978-. Studies of polyadenylation regulation of U1A mRNA by an RNP complex containing U1A and U1 snRNP.

Degree: PhD, Biochemistry, 2011, Rutgers University

The 3’-end processing of nearly all eukaryotic pre-mRNAs comprises multiple steps which culminate in the addition of a poly(A) tail, which is essential for mRNA… (more)

Subjects/Keywords: Gene expression; Genetic regulation; RNA

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APA (6th Edition):

Caratozzolo, Rose Marie, 1. (2011). Studies of polyadenylation regulation of U1A mRNA by an RNP complex containing U1A and U1 snRNP. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057530

Chicago Manual of Style (16th Edition):

Caratozzolo, Rose Marie, 1978-. “Studies of polyadenylation regulation of U1A mRNA by an RNP complex containing U1A and U1 snRNP.” 2011. Doctoral Dissertation, Rutgers University. Accessed February 22, 2020. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057530.

MLA Handbook (7th Edition):

Caratozzolo, Rose Marie, 1978-. “Studies of polyadenylation regulation of U1A mRNA by an RNP complex containing U1A and U1 snRNP.” 2011. Web. 22 Feb 2020.

Vancouver:

Caratozzolo, Rose Marie 1. Studies of polyadenylation regulation of U1A mRNA by an RNP complex containing U1A and U1 snRNP. [Internet] [Doctoral dissertation]. Rutgers University; 2011. [cited 2020 Feb 22]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057530.

Council of Science Editors:

Caratozzolo, Rose Marie 1. Studies of polyadenylation regulation of U1A mRNA by an RNP complex containing U1A and U1 snRNP. [Doctoral Dissertation]. Rutgers University; 2011. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057530

22. Laver, Travis. Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression.

Degree: PhD, 2008, University of Alabama – Birmingham

Interleukin-8 (IL-8) is a potent chemoattractant of numerous cells, particularly neutrophils, in the innate immune response. In addition to immune functions, IL-8 is known to… (more)

Subjects/Keywords: Astrocytoma <; br>; Gene Expression Regulation <; br>; Interferon-beta  – physiology <; br>; Interferon-Stimulated Gene Factor 3, gamma Subunit  – metabolism <; br>; Interleukin-8 <; br>; STAT1 Transcription Factor  – metabolism <; br>; STAT2 Transcription Factor  – metabolism

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APA (6th Edition):

Laver, T. (2008). Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,204

Chicago Manual of Style (16th Edition):

Laver, Travis. “Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,204.

MLA Handbook (7th Edition):

Laver, Travis. “Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression.” 2008. Web. 22 Feb 2020.

Vancouver:

Laver T. Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,204.

Council of Science Editors:

Laver T. Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,204

23. Faria, André Murad. Papel do LIN28, uma proteína ligadora de RNAs, na tumorigênese adrenocortical.

Degree: PhD, Endocrinologia, 2014, University of São Paulo

 INTRODUÇÃO: O carcinoma adrenocortical é uma neoplasia rara que carreia um prognóstico reservado. Recentemente, uma série de estudos demonstrou o potencial do perfil de miRNAs… (more)

Subjects/Keywords: Adrenocortical carcinoma; Carcinogênese; Carcinogenesis; Carcinoma adrenocortical; Expressão gênica; Gene expression; Marcadores biológicos de tumor; MicroRNAs; MicroRNAs; Neoplasm proteins; Neoplastic regulation of gene expression; Prognosis; Prognóstico; Proteínas de ligação a RNA; Proteínas de neoplasias; Regulação neoplásica da expressão gênica; RNA-binding protein; Sobrevida; Survival; Tumor biologic markers

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APA (6th Edition):

Faria, A. M. (2014). Papel do LIN28, uma proteína ligadora de RNAs, na tumorigênese adrenocortical. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26022015-095301/ ;

Chicago Manual of Style (16th Edition):

Faria, André Murad. “Papel do LIN28, uma proteína ligadora de RNAs, na tumorigênese adrenocortical.” 2014. Doctoral Dissertation, University of São Paulo. Accessed February 22, 2020. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26022015-095301/ ;.

MLA Handbook (7th Edition):

Faria, André Murad. “Papel do LIN28, uma proteína ligadora de RNAs, na tumorigênese adrenocortical.” 2014. Web. 22 Feb 2020.

Vancouver:

Faria AM. Papel do LIN28, uma proteína ligadora de RNAs, na tumorigênese adrenocortical. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2020 Feb 22]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26022015-095301/ ;.

Council of Science Editors:

Faria AM. Papel do LIN28, uma proteína ligadora de RNAs, na tumorigênese adrenocortical. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26022015-095301/ ;


University of Saskatchewan

24. Narayanan, Karthikeyan. SCREENING AND CHARACTERIZATION OF ARABIDOPSIS THALIANA MUTANTS WITH ALTERED CAROTENOID PROFILE.

Degree: 2014, University of Saskatchewan

 Carotenoids are organic pigments that are mainly found in the chloroplasts and chromoplasts of plants and other photosynthetic organisms. Carotenoid molecules containing oxygen, such as… (more)

Subjects/Keywords: Carotneoids; RBP47; KCS19; Gene expression; Gene regulation

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APA (6th Edition):

Narayanan, K. (2014). SCREENING AND CHARACTERIZATION OF ARABIDOPSIS THALIANA MUTANTS WITH ALTERED CAROTENOID PROFILE. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/ETD-2014-06-1601

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Narayanan, Karthikeyan. “SCREENING AND CHARACTERIZATION OF ARABIDOPSIS THALIANA MUTANTS WITH ALTERED CAROTENOID PROFILE.” 2014. Thesis, University of Saskatchewan. Accessed February 22, 2020. http://hdl.handle.net/10388/ETD-2014-06-1601.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Narayanan, Karthikeyan. “SCREENING AND CHARACTERIZATION OF ARABIDOPSIS THALIANA MUTANTS WITH ALTERED CAROTENOID PROFILE.” 2014. Web. 22 Feb 2020.

Vancouver:

Narayanan K. SCREENING AND CHARACTERIZATION OF ARABIDOPSIS THALIANA MUTANTS WITH ALTERED CAROTENOID PROFILE. [Internet] [Thesis]. University of Saskatchewan; 2014. [cited 2020 Feb 22]. Available from: http://hdl.handle.net/10388/ETD-2014-06-1601.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Narayanan K. SCREENING AND CHARACTERIZATION OF ARABIDOPSIS THALIANA MUTANTS WITH ALTERED CAROTENOID PROFILE. [Thesis]. University of Saskatchewan; 2014. Available from: http://hdl.handle.net/10388/ETD-2014-06-1601

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Alasoo, Kaur. Regulation of gene expression in macrophage immune response.

Degree: PhD, 2017, University of Cambridge

Gene expression quantitative trait loci (eQTL) mapping studies can provide mechanistic insights into the functions of disease-associated variants. However, many eQTLs are cell type and… (more)

Subjects/Keywords: Gene expression; Gene regulation; Chromatin; Genetics

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APA (6th Edition):

Alasoo, K. (2017). Regulation of gene expression in macrophage immune response. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/263855

Chicago Manual of Style (16th Edition):

Alasoo, Kaur. “Regulation of gene expression in macrophage immune response.” 2017. Doctoral Dissertation, University of Cambridge. Accessed February 22, 2020. https://www.repository.cam.ac.uk/handle/1810/263855.

MLA Handbook (7th Edition):

Alasoo, Kaur. “Regulation of gene expression in macrophage immune response.” 2017. Web. 22 Feb 2020.

Vancouver:

Alasoo K. Regulation of gene expression in macrophage immune response. [Internet] [Doctoral dissertation]. University of Cambridge; 2017. [cited 2020 Feb 22]. Available from: https://www.repository.cam.ac.uk/handle/1810/263855.

Council of Science Editors:

Alasoo K. Regulation of gene expression in macrophage immune response. [Doctoral Dissertation]. University of Cambridge; 2017. Available from: https://www.repository.cam.ac.uk/handle/1810/263855


University of Cambridge

26. Alasoo, Kaur. Regulation of gene expression in macrophage immune response.

Degree: PhD, 2017, University of Cambridge

Gene expression quantitative trait loci (eQTL) mapping studies can provide mechanistic insights into the functions of disease-associated variants. However, many eQTLs are cell type and… (more)

Subjects/Keywords: 616.07; Gene expression; Gene regulation; Chromatin; Genetics

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APA (6th Edition):

Alasoo, K. (2017). Regulation of gene expression in macrophage immune response. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/263855 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.715945

Chicago Manual of Style (16th Edition):

Alasoo, Kaur. “Regulation of gene expression in macrophage immune response.” 2017. Doctoral Dissertation, University of Cambridge. Accessed February 22, 2020. https://www.repository.cam.ac.uk/handle/1810/263855 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.715945.

MLA Handbook (7th Edition):

Alasoo, Kaur. “Regulation of gene expression in macrophage immune response.” 2017. Web. 22 Feb 2020.

Vancouver:

Alasoo K. Regulation of gene expression in macrophage immune response. [Internet] [Doctoral dissertation]. University of Cambridge; 2017. [cited 2020 Feb 22]. Available from: https://www.repository.cam.ac.uk/handle/1810/263855 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.715945.

Council of Science Editors:

Alasoo K. Regulation of gene expression in macrophage immune response. [Doctoral Dissertation]. University of Cambridge; 2017. Available from: https://www.repository.cam.ac.uk/handle/1810/263855 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.715945

27. Crimmins, Stephen Lewis. Characterization and functional analysis of Usp14.

Degree: PhD, 2007, University of Alabama – Birmingham

The ubiquitin proteasome system (UPS) is essential for regulated protein degrada-tion, a requirement for numerous neuronal process, including vesicle cycling, neuro-transmitter release, spine morphology, and… (more)

Subjects/Keywords: Ataxia  – enzymology <; br>; Ataxia  – genetics <; br>; Gene Expression Regulation, Enzymologic  – physiology <; br>; Neurons  – enzymology <; br>; Ubiquitin  – metabolism <; br>; Ubiquitin Thiolesterase  – biosynthesis <; br>; Ubiquitin Thiolesterase  – genetics

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APA (6th Edition):

Crimmins, S. L. (2007). Characterization and functional analysis of Usp14. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,118

Chicago Manual of Style (16th Edition):

Crimmins, Stephen Lewis. “Characterization and functional analysis of Usp14.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,118.

MLA Handbook (7th Edition):

Crimmins, Stephen Lewis. “Characterization and functional analysis of Usp14.” 2007. Web. 22 Feb 2020.

Vancouver:

Crimmins SL. Characterization and functional analysis of Usp14. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,118.

Council of Science Editors:

Crimmins SL. Characterization and functional analysis of Usp14. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,118

28. Wang, Ying. The role of the hypoxia-inducible factor pathway in bone development and repair.

Degree: PhD, 2007, University of Alabama – Birmingham

Osteogenesis and angiogenesis are tightly coupled during bone formation and repair. Blood vessels not only carry oxygen and nutrients to the developing bone, but also… (more)

Subjects/Keywords: Bone Development  – physiology<; br>; Bone Regeneration  – physiology<; br>; Gene Expression Regulation<; br>; Hypoxia-Inducible Factor 1, alpha Subunit<; br>; Neovascularization, Physiologic<; br>; Osteoblasts  – metabolism<; br>; Osteogenesis  – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, Y. (2007). The role of the hypoxia-inducible factor pathway in bone development and repair. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,618

Chicago Manual of Style (16th Edition):

Wang, Ying. “The role of the hypoxia-inducible factor pathway in bone development and repair.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,618.

MLA Handbook (7th Edition):

Wang, Ying. “The role of the hypoxia-inducible factor pathway in bone development and repair.” 2007. Web. 22 Feb 2020.

Vancouver:

Wang Y. The role of the hypoxia-inducible factor pathway in bone development and repair. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,618.

Council of Science Editors:

Wang Y. The role of the hypoxia-inducible factor pathway in bone development and repair. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,618

29. Hertz, Marla (Marla Ilene). In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation.

Degree: PhD, 2011, University of Alabama – Birmingham

Translation of the majority of eukaryotic mRNAs is initiated upon recognition of its 5′ cap structure by translation initiation factors in so-called cap-dependent translation. Capdependent… (more)

Subjects/Keywords: Dicistroviridae  – metabolism<; br>; Gene Expression Regulation<; br>; Hepacivirus  – metabolism<; br>; Prostatic Neoplasms<; br>; Protein Biosynthesis<; br>; Ribosomal Proteins  – metabolism<; br>; Saccharomyces cerevisiae Proteins  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hertz, M. (. I. (2011). In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,953

Chicago Manual of Style (16th Edition):

Hertz, Marla (Marla Ilene). “In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,953.

MLA Handbook (7th Edition):

Hertz, Marla (Marla Ilene). “In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation.” 2011. Web. 22 Feb 2020.

Vancouver:

Hertz M(I. In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,953.

Council of Science Editors:

Hertz M(I. In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,953

30. Kim, Junghyun. Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells.

Degree: PhD, 2010, University of Alabama – Birmingham

Heme oxygenase-1 (HO-1) is a critical enzyme catalyzing the degradation of heme and generating carbon monoxide, iron, and biliverdin. In addition to heme degradation, HO-1… (more)

Subjects/Keywords: Acute Kidney Injury<; br>; Chromatin  – chemistry<; br>; Gene Expression Regulation, Enzymologic<; br>; Heme Oxygenase-1  – biosynthesis<; br>; Kidney  – enzymology<; br>; Mice<; br>; Sp1 Transcription Factor  – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kim, J. (2010). Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1100

Chicago Manual of Style (16th Edition):

Kim, Junghyun. “Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed February 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1100.

MLA Handbook (7th Edition):

Kim, Junghyun. “Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells.” 2010. Web. 22 Feb 2020.

Vancouver:

Kim J. Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Feb 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1100.

Council of Science Editors:

Kim J. Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1100

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