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You searched for subject:(GSK3 ). Showing records 1 – 30 of 57 total matches.

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University of Vermont

1. Bauman, Brittany Nicole. Role of p38 MAPK inhibitor in conditioned fear.

Degree: Biology, 2015, University of Vermont

 <h1>p38 mitogen activated protein kinase (p38) is a kinase that has been implicated in cellular plasticity, stress, and psychiatric disorders and recently in the process… (more)

Subjects/Keywords: p38; conditioned fear; GSK3; inhibition; freezing

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bauman, B. N. (2015). Role of p38 MAPK inhibitor in conditioned fear. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/hcoltheses/186

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bauman, Brittany Nicole. “Role of p38 MAPK inhibitor in conditioned fear.” 2015. Thesis, University of Vermont. Accessed April 11, 2021. https://scholarworks.uvm.edu/hcoltheses/186.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bauman, Brittany Nicole. “Role of p38 MAPK inhibitor in conditioned fear.” 2015. Web. 11 Apr 2021.

Vancouver:

Bauman BN. Role of p38 MAPK inhibitor in conditioned fear. [Internet] [Thesis]. University of Vermont; 2015. [cited 2021 Apr 11]. Available from: https://scholarworks.uvm.edu/hcoltheses/186.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bauman BN. Role of p38 MAPK inhibitor in conditioned fear. [Thesis]. University of Vermont; 2015. Available from: https://scholarworks.uvm.edu/hcoltheses/186

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

2. Dowd, Christopher Colin. Dosage suppression of mutations in casein kinase II by RIM11, a glycogen synthase kinase-3 homolog.

Degree: 2014, University of Georgia

 In this report, a glycogen synthase kinase-3 homolog, RIM11, was isolated in a multicopy screen for suppressors of the salt-sensitive phenotype of casein kinase II… (more)

Subjects/Keywords: S. cerevisiae; CKII; RIM11; GSK3; salt sensitivity

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APA (6th Edition):

Dowd, C. C. (2014). Dosage suppression of mutations in casein kinase II by RIM11, a glycogen synthase kinase-3 homolog. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/20314

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dowd, Christopher Colin. “Dosage suppression of mutations in casein kinase II by RIM11, a glycogen synthase kinase-3 homolog.” 2014. Thesis, University of Georgia. Accessed April 11, 2021. http://hdl.handle.net/10724/20314.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dowd, Christopher Colin. “Dosage suppression of mutations in casein kinase II by RIM11, a glycogen synthase kinase-3 homolog.” 2014. Web. 11 Apr 2021.

Vancouver:

Dowd CC. Dosage suppression of mutations in casein kinase II by RIM11, a glycogen synthase kinase-3 homolog. [Internet] [Thesis]. University of Georgia; 2014. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10724/20314.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dowd CC. Dosage suppression of mutations in casein kinase II by RIM11, a glycogen synthase kinase-3 homolog. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/20314

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Paris-Sud – Paris XI

3. Martel, Cécile. Rôle de la perméabilité membranaire mitochondriale, de la phosphorylation de VDAC et de la signalisation de l’apoptose dans la pathogenèse de la stéatose hépatique : Role of mitochondrial membrane permeability, VDAC phosphorylation and signaling pathway of apoptosis in the pathogenesis of steatosis.

Degree: Docteur es, Cancérologie - Biochimie, Biologie cellulaire et moléculaire, 2011, Université Paris-Sud – Paris XI

La stéatose hépatique non-alcoolique consiste en une accumulation de lipides dans le cytoplasme des hépatocytes. Longtemps considérée comme une pathologie bénigne, elle peut être à… (more)

Subjects/Keywords: Stéatose; Mitochondrie; Apoptose; VDAC; GSK3; Lipotoxicité; Stéatosis; Mitochondria; Apoptosis; VDAC; GSK3; Lipotoxicity

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APA (6th Edition):

Martel, C. (2011). Rôle de la perméabilité membranaire mitochondriale, de la phosphorylation de VDAC et de la signalisation de l’apoptose dans la pathogenèse de la stéatose hépatique : Role of mitochondrial membrane permeability, VDAC phosphorylation and signaling pathway of apoptosis in the pathogenesis of steatosis. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA11T075

Chicago Manual of Style (16th Edition):

Martel, Cécile. “Rôle de la perméabilité membranaire mitochondriale, de la phosphorylation de VDAC et de la signalisation de l’apoptose dans la pathogenèse de la stéatose hépatique : Role of mitochondrial membrane permeability, VDAC phosphorylation and signaling pathway of apoptosis in the pathogenesis of steatosis.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed April 11, 2021. http://www.theses.fr/2011PA11T075.

MLA Handbook (7th Edition):

Martel, Cécile. “Rôle de la perméabilité membranaire mitochondriale, de la phosphorylation de VDAC et de la signalisation de l’apoptose dans la pathogenèse de la stéatose hépatique : Role of mitochondrial membrane permeability, VDAC phosphorylation and signaling pathway of apoptosis in the pathogenesis of steatosis.” 2011. Web. 11 Apr 2021.

Vancouver:

Martel C. Rôle de la perméabilité membranaire mitochondriale, de la phosphorylation de VDAC et de la signalisation de l’apoptose dans la pathogenèse de la stéatose hépatique : Role of mitochondrial membrane permeability, VDAC phosphorylation and signaling pathway of apoptosis in the pathogenesis of steatosis. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2021 Apr 11]. Available from: http://www.theses.fr/2011PA11T075.

Council of Science Editors:

Martel C. Rôle de la perméabilité membranaire mitochondriale, de la phosphorylation de VDAC et de la signalisation de l’apoptose dans la pathogenèse de la stéatose hépatique : Role of mitochondrial membrane permeability, VDAC phosphorylation and signaling pathway of apoptosis in the pathogenesis of steatosis. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA11T075

4. Benajiba, Lina. Identification and Characterization of New Therapeutic Targets in Acute Myeloid Leukemia : Identification et caractérisation de nouvelles cibles thérapeutiques dans les LAM.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2018, Université Paris-Saclay (ComUE)

La leucémie aiguë myéloïde (LAM) est une pathologie hématologique dont le pronostic reste très défavorable, malgré les progrès réalisés dans la compréhension des mécanismes physiopathologiques… (more)

Subjects/Keywords: LAM; Cibles thérapeutiques; GSK3; CKMT1; VCP; AML; Therapeutic targets; GSK3; CKMT1; VCP

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APA (6th Edition):

Benajiba, L. (2018). Identification and Characterization of New Therapeutic Targets in Acute Myeloid Leukemia : Identification et caractérisation de nouvelles cibles thérapeutiques dans les LAM. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2018SACLS173

Chicago Manual of Style (16th Edition):

Benajiba, Lina. “Identification and Characterization of New Therapeutic Targets in Acute Myeloid Leukemia : Identification et caractérisation de nouvelles cibles thérapeutiques dans les LAM.” 2018. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed April 11, 2021. http://www.theses.fr/2018SACLS173.

MLA Handbook (7th Edition):

Benajiba, Lina. “Identification and Characterization of New Therapeutic Targets in Acute Myeloid Leukemia : Identification et caractérisation de nouvelles cibles thérapeutiques dans les LAM.” 2018. Web. 11 Apr 2021.

Vancouver:

Benajiba L. Identification and Characterization of New Therapeutic Targets in Acute Myeloid Leukemia : Identification et caractérisation de nouvelles cibles thérapeutiques dans les LAM. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2018. [cited 2021 Apr 11]. Available from: http://www.theses.fr/2018SACLS173.

Council of Science Editors:

Benajiba L. Identification and Characterization of New Therapeutic Targets in Acute Myeloid Leukemia : Identification et caractérisation de nouvelles cibles thérapeutiques dans les LAM. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2018. Available from: http://www.theses.fr/2018SACLS173


University of Vienna

5. Ukowitz, Michaela. Homology Modelling of Shaggy-like Kinase α in Arabidopsis thaliana (ASKα).

Degree: 2019, University of Vienna

Pflanzen sind kontinuierlich Stress ausgesetzt. Auf der einen Seite ist es biotischer Stress, der unter anderem von Bakterien, Viren, Pilzen oder Insekten ausgelöst wird und… (more)

Subjects/Keywords: 35.76 Aminosäuren, Peptide, Eiweiße; Homology modelling / ASK / GSK3; Homology modelling / ASK / GSK3

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APA (6th Edition):

Ukowitz, M. (2019). Homology Modelling of Shaggy-like Kinase α in Arabidopsis thaliana (ASKα). (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/57692/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ukowitz, Michaela. “Homology Modelling of Shaggy-like Kinase α in Arabidopsis thaliana (ASKα).” 2019. Thesis, University of Vienna. Accessed April 11, 2021. http://othes.univie.ac.at/57692/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ukowitz, Michaela. “Homology Modelling of Shaggy-like Kinase α in Arabidopsis thaliana (ASKα).” 2019. Web. 11 Apr 2021.

Vancouver:

Ukowitz M. Homology Modelling of Shaggy-like Kinase α in Arabidopsis thaliana (ASKα). [Internet] [Thesis]. University of Vienna; 2019. [cited 2021 Apr 11]. Available from: http://othes.univie.ac.at/57692/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ukowitz M. Homology Modelling of Shaggy-like Kinase α in Arabidopsis thaliana (ASKα). [Thesis]. University of Vienna; 2019. Available from: http://othes.univie.ac.at/57692/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Lucas Kuhn Pereira Prado. Exercício físico em idosos: efeito nos biomarcadores periféricos de neuroproteção.

Degree: 2012, University of São Paulo

A atividade física (AF) demonstrou um efeito protetor contra a doença de Alzheimer (DA), mas não se sabe qual a influência da AF sobre o… (more)

Subjects/Keywords: Alzheimer; Atividade física; Exercício; GSK3; Idosos; Aged; Alzheimer; Exercise; GSK3; Physical activity

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APA (6th Edition):

Prado, L. K. P. (2012). Exercício físico em idosos: efeito nos biomarcadores periféricos de neuroproteção. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5140/tde-09082012-141507/

Chicago Manual of Style (16th Edition):

Prado, Lucas Kuhn Pereira. “Exercício físico em idosos: efeito nos biomarcadores periféricos de neuroproteção.” 2012. Masters Thesis, University of São Paulo. Accessed April 11, 2021. http://www.teses.usp.br/teses/disponiveis/5/5140/tde-09082012-141507/.

MLA Handbook (7th Edition):

Prado, Lucas Kuhn Pereira. “Exercício físico em idosos: efeito nos biomarcadores periféricos de neuroproteção.” 2012. Web. 11 Apr 2021.

Vancouver:

Prado LKP. Exercício físico em idosos: efeito nos biomarcadores periféricos de neuroproteção. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2021 Apr 11]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5140/tde-09082012-141507/.

Council of Science Editors:

Prado LKP. Exercício físico em idosos: efeito nos biomarcadores periféricos de neuroproteção. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5140/tde-09082012-141507/

7. Rahal, Pamela. Role of GSK3β - MLK3 - p38γ MAPK Signalling in Satellite Cell Proliferation Regulation : Le rôle de la voie de signalisation GSK3β-MLK3-p38γ MAPK dans la régulation de la prolifération des cellules satellites.

Degree: Docteur es, Sciences de la Vie, 2015, Lyon, École normale supérieure

MLK3 est une ser/thr MAP3K qui active la voie de signalisation des MAPKs dans différents types cellulaires. GSK3β interagit et active MLK3 en la phosphorylant… (more)

Subjects/Keywords: GSK3; MLK3; P38 MAPK; Régénération musculaire; Cellules satellites; Pax7; GSK3; MLK3; P38 MAPK; Muscle regeneration; Satellite cells; Pax7

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APA (6th Edition):

Rahal, P. (2015). Role of GSK3β - MLK3 - p38γ MAPK Signalling in Satellite Cell Proliferation Regulation : Le rôle de la voie de signalisation GSK3β-MLK3-p38γ MAPK dans la régulation de la prolifération des cellules satellites. (Doctoral Dissertation). Lyon, École normale supérieure. Retrieved from http://www.theses.fr/2015ENSL1000

Chicago Manual of Style (16th Edition):

Rahal, Pamela. “Role of GSK3β - MLK3 - p38γ MAPK Signalling in Satellite Cell Proliferation Regulation : Le rôle de la voie de signalisation GSK3β-MLK3-p38γ MAPK dans la régulation de la prolifération des cellules satellites.” 2015. Doctoral Dissertation, Lyon, École normale supérieure. Accessed April 11, 2021. http://www.theses.fr/2015ENSL1000.

MLA Handbook (7th Edition):

Rahal, Pamela. “Role of GSK3β - MLK3 - p38γ MAPK Signalling in Satellite Cell Proliferation Regulation : Le rôle de la voie de signalisation GSK3β-MLK3-p38γ MAPK dans la régulation de la prolifération des cellules satellites.” 2015. Web. 11 Apr 2021.

Vancouver:

Rahal P. Role of GSK3β - MLK3 - p38γ MAPK Signalling in Satellite Cell Proliferation Regulation : Le rôle de la voie de signalisation GSK3β-MLK3-p38γ MAPK dans la régulation de la prolifération des cellules satellites. [Internet] [Doctoral dissertation]. Lyon, École normale supérieure; 2015. [cited 2021 Apr 11]. Available from: http://www.theses.fr/2015ENSL1000.

Council of Science Editors:

Rahal P. Role of GSK3β - MLK3 - p38γ MAPK Signalling in Satellite Cell Proliferation Regulation : Le rôle de la voie de signalisation GSK3β-MLK3-p38γ MAPK dans la régulation de la prolifération des cellules satellites. [Doctoral Dissertation]. Lyon, École normale supérieure; 2015. Available from: http://www.theses.fr/2015ENSL1000


NSYSU

8. Ko, Chiung-Yuan. Phosphorylation and Functional Regulation of Alzheimer's Tau by GSK3-beta and Prolyl Isomerase Pin1.

Degree: Master, Institute of Biomedical Sciences, 2003, NSYSU

 Alzheimerâs disease (AD), one of the most common dementia, is characterized by the formation two types of aggregation in the brain: senile plaques and neurofibrillary… (more)

Subjects/Keywords: phosphorylation; Tau; GSK3-beta; Pin1

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APA (6th Edition):

Ko, C. (2003). Phosphorylation and Functional Regulation of Alzheimer's Tau by GSK3-beta and Prolyl Isomerase Pin1. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0617103-232420

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ko, Chiung-Yuan. “Phosphorylation and Functional Regulation of Alzheimer's Tau by GSK3-beta and Prolyl Isomerase Pin1.” 2003. Thesis, NSYSU. Accessed April 11, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0617103-232420.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ko, Chiung-Yuan. “Phosphorylation and Functional Regulation of Alzheimer's Tau by GSK3-beta and Prolyl Isomerase Pin1.” 2003. Web. 11 Apr 2021.

Vancouver:

Ko C. Phosphorylation and Functional Regulation of Alzheimer's Tau by GSK3-beta and Prolyl Isomerase Pin1. [Internet] [Thesis]. NSYSU; 2003. [cited 2021 Apr 11]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0617103-232420.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ko C. Phosphorylation and Functional Regulation of Alzheimer's Tau by GSK3-beta and Prolyl Isomerase Pin1. [Thesis]. NSYSU; 2003. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0617103-232420

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Marchand, Benoît. Rôle des Glycogène synthase kinases 3 (GSK3) dans la régulation de l’autophagie et du facteur de transcription EB (TFEB) dans les cellules pancréatiques tumorales humaines.

Degree: PhD, Biologie cellulaire, 2016, Université de Sherbrooke

 Plusieurs études ont suggéré une implication des glycogène synthase kinases 3 (GSK3) dans la carcinogenèse, notamment du pancréas. Des études ont rapporté des résultats contradictoires… (more)

Subjects/Keywords: GSK3; Cancer pancréatique; Autophagie; TFEB; mTOR; 14-3-3; JNK

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APA (6th Edition):

Marchand, B. (2016). Rôle des Glycogène synthase kinases 3 (GSK3) dans la régulation de l’autophagie et du facteur de transcription EB (TFEB) dans les cellules pancréatiques tumorales humaines. (Doctoral Dissertation). Université de Sherbrooke. Retrieved from http://www.collectionscanada.gc.ca/obj/thesescanada/vol2/QSHERU/TC-QSHERU-11143_8185.pdf ; http://savoirs.usherbrooke.ca/bitstream/11143/8185/1/Marchand_Benoit_PhD_2016.pdf

Chicago Manual of Style (16th Edition):

Marchand, Benoît. “Rôle des Glycogène synthase kinases 3 (GSK3) dans la régulation de l’autophagie et du facteur de transcription EB (TFEB) dans les cellules pancréatiques tumorales humaines.” 2016. Doctoral Dissertation, Université de Sherbrooke. Accessed April 11, 2021. http://www.collectionscanada.gc.ca/obj/thesescanada/vol2/QSHERU/TC-QSHERU-11143_8185.pdf ; http://savoirs.usherbrooke.ca/bitstream/11143/8185/1/Marchand_Benoit_PhD_2016.pdf.

MLA Handbook (7th Edition):

Marchand, Benoît. “Rôle des Glycogène synthase kinases 3 (GSK3) dans la régulation de l’autophagie et du facteur de transcription EB (TFEB) dans les cellules pancréatiques tumorales humaines.” 2016. Web. 11 Apr 2021.

Vancouver:

Marchand B. Rôle des Glycogène synthase kinases 3 (GSK3) dans la régulation de l’autophagie et du facteur de transcription EB (TFEB) dans les cellules pancréatiques tumorales humaines. [Internet] [Doctoral dissertation]. Université de Sherbrooke; 2016. [cited 2021 Apr 11]. Available from: http://www.collectionscanada.gc.ca/obj/thesescanada/vol2/QSHERU/TC-QSHERU-11143_8185.pdf ; http://savoirs.usherbrooke.ca/bitstream/11143/8185/1/Marchand_Benoit_PhD_2016.pdf.

Council of Science Editors:

Marchand B. Rôle des Glycogène synthase kinases 3 (GSK3) dans la régulation de l’autophagie et du facteur de transcription EB (TFEB) dans les cellules pancréatiques tumorales humaines. [Doctoral Dissertation]. Université de Sherbrooke; 2016. Available from: http://www.collectionscanada.gc.ca/obj/thesescanada/vol2/QSHERU/TC-QSHERU-11143_8185.pdf ; http://savoirs.usherbrooke.ca/bitstream/11143/8185/1/Marchand_Benoit_PhD_2016.pdf


Penn State University

10. Burns, Katherine Anne. REGULATION OF PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR ALPHA ACTIVITY VIA CROSS-TALK WITH GLYCOGEN SYNTHASE KINASE 3 .

Degree: 2008, Penn State University

 Peroxisome proliferator-activated receptor alpha (PPAR alpha) is a nuclear receptor involved in the maintenance of fatty acid homeostasis and is important for mediating the adaptive… (more)

Subjects/Keywords: fasting; phosphorylation; GSK3; PPARalpha

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APA (6th Edition):

Burns, K. A. (2008). REGULATION OF PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR ALPHA ACTIVITY VIA CROSS-TALK WITH GLYCOGEN SYNTHASE KINASE 3 . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/7598

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Burns, Katherine Anne. “REGULATION OF PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR ALPHA ACTIVITY VIA CROSS-TALK WITH GLYCOGEN SYNTHASE KINASE 3 .” 2008. Thesis, Penn State University. Accessed April 11, 2021. https://submit-etda.libraries.psu.edu/catalog/7598.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Burns, Katherine Anne. “REGULATION OF PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR ALPHA ACTIVITY VIA CROSS-TALK WITH GLYCOGEN SYNTHASE KINASE 3 .” 2008. Web. 11 Apr 2021.

Vancouver:

Burns KA. REGULATION OF PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR ALPHA ACTIVITY VIA CROSS-TALK WITH GLYCOGEN SYNTHASE KINASE 3 . [Internet] [Thesis]. Penn State University; 2008. [cited 2021 Apr 11]. Available from: https://submit-etda.libraries.psu.edu/catalog/7598.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Burns KA. REGULATION OF PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR ALPHA ACTIVITY VIA CROSS-TALK WITH GLYCOGEN SYNTHASE KINASE 3 . [Thesis]. Penn State University; 2008. Available from: https://submit-etda.libraries.psu.edu/catalog/7598

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

11. Fagnan, Erin. Mechanisms for Scaffold-Mediated Regulation of Kinase Activity in the Wnt Signaling Pathway.

Degree: PhD, 2019, University of Washington

 Cellular signaling is a complex process involving numerous pathways. Many of these pathways are connected through shared proteins, creating branch points that may result in… (more)

Subjects/Keywords: Axin; Cell Signaling; GSK3; Scaffold Proteins; Wnt pathway; Chemistry; Biochemistry; Chemistry

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APA (6th Edition):

Fagnan, E. (2019). Mechanisms for Scaffold-Mediated Regulation of Kinase Activity in the Wnt Signaling Pathway. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/44746

Chicago Manual of Style (16th Edition):

Fagnan, Erin. “Mechanisms for Scaffold-Mediated Regulation of Kinase Activity in the Wnt Signaling Pathway.” 2019. Doctoral Dissertation, University of Washington. Accessed April 11, 2021. http://hdl.handle.net/1773/44746.

MLA Handbook (7th Edition):

Fagnan, Erin. “Mechanisms for Scaffold-Mediated Regulation of Kinase Activity in the Wnt Signaling Pathway.” 2019. Web. 11 Apr 2021.

Vancouver:

Fagnan E. Mechanisms for Scaffold-Mediated Regulation of Kinase Activity in the Wnt Signaling Pathway. [Internet] [Doctoral dissertation]. University of Washington; 2019. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/1773/44746.

Council of Science Editors:

Fagnan E. Mechanisms for Scaffold-Mediated Regulation of Kinase Activity in the Wnt Signaling Pathway. [Doctoral Dissertation]. University of Washington; 2019. Available from: http://hdl.handle.net/1773/44746


University of Kansas

12. Venugopal, Anand. IDENTIFICATION OF THE RNA BINDING PROTEIN RBM3 AS A NOVEL EFFECTOR OF β-CATENIN SIGNALING AND COLON CANCER STEM CELLS.

Degree: PhD, Molecular & Integrative Physiology, 2014, University of Kansas

 The intestinal epithelium is one of the fastest renewing tissues within the adult. This renewal is primarily driven by the intestinal epithelial stem cell compartment… (more)

Subjects/Keywords: Molecular biology; Oncology; DCLK1; GSK3&beta;

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APA (6th Edition):

Venugopal, A. (2014). IDENTIFICATION OF THE RNA BINDING PROTEIN RBM3 AS A NOVEL EFFECTOR OF β-CATENIN SIGNALING AND COLON CANCER STEM CELLS. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/22535

Chicago Manual of Style (16th Edition):

Venugopal, Anand. “IDENTIFICATION OF THE RNA BINDING PROTEIN RBM3 AS A NOVEL EFFECTOR OF β-CATENIN SIGNALING AND COLON CANCER STEM CELLS.” 2014. Doctoral Dissertation, University of Kansas. Accessed April 11, 2021. http://hdl.handle.net/1808/22535.

MLA Handbook (7th Edition):

Venugopal, Anand. “IDENTIFICATION OF THE RNA BINDING PROTEIN RBM3 AS A NOVEL EFFECTOR OF β-CATENIN SIGNALING AND COLON CANCER STEM CELLS.” 2014. Web. 11 Apr 2021.

Vancouver:

Venugopal A. IDENTIFICATION OF THE RNA BINDING PROTEIN RBM3 AS A NOVEL EFFECTOR OF β-CATENIN SIGNALING AND COLON CANCER STEM CELLS. [Internet] [Doctoral dissertation]. University of Kansas; 2014. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/1808/22535.

Council of Science Editors:

Venugopal A. IDENTIFICATION OF THE RNA BINDING PROTEIN RBM3 AS A NOVEL EFFECTOR OF β-CATENIN SIGNALING AND COLON CANCER STEM CELLS. [Doctoral Dissertation]. University of Kansas; 2014. Available from: http://hdl.handle.net/1808/22535


University of Sydney

13. Duan, Xiaowen. Characterization of Trafficking Regulator of GLUT4-1 (TRARG1) .

Degree: 2020, University of Sydney

 Insulin resistance is a precursor of most metabolic diseases including Type 2 diabetes, and is largely recapitulated by impaired insulin-stimulated glucose transport mediated by the… (more)

Subjects/Keywords: GSK3; GLUT4; TRARG1; Protein phosphorylation; insulin signaling; BCL9L

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APA (6th Edition):

Duan, X. (2020). Characterization of Trafficking Regulator of GLUT4-1 (TRARG1) . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/23722

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Duan, Xiaowen. “Characterization of Trafficking Regulator of GLUT4-1 (TRARG1) .” 2020. Thesis, University of Sydney. Accessed April 11, 2021. http://hdl.handle.net/2123/23722.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Duan, Xiaowen. “Characterization of Trafficking Regulator of GLUT4-1 (TRARG1) .” 2020. Web. 11 Apr 2021.

Vancouver:

Duan X. Characterization of Trafficking Regulator of GLUT4-1 (TRARG1) . [Internet] [Thesis]. University of Sydney; 2020. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/2123/23722.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Duan X. Characterization of Trafficking Regulator of GLUT4-1 (TRARG1) . [Thesis]. University of Sydney; 2020. Available from: http://hdl.handle.net/2123/23722

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Ribeiro, Marco Saleh. Modulação da AKT/GSK3-B e SNAP-25 pela administração de curcumina em modelo de mania induzido por cetamina.

Degree: 2015, Universidade Catolica de Pelotas; Programa de Pos-Graduacao em Saude Comportamento#;-1990782970254042025#; #600; UCPel; Brasil; Centro de Ciencias da Saude#; #-7432574962795991241#; #600

Submitted by Cristiane Chim ([email protected]) on 2016-11-11T11:14:08Z No. of bitstreams: 1 MARCO SALEH RIBEIRO.pdf: 1233321 bytes, checksum: aebd2e7f49d852825cab51345cf5eebc (MD5)

Made available in DSpace on 2016-11-11T11:14:08Z… (more)

Subjects/Keywords: transtorno bipolar; cetamina; GSK3; SNAP-25; FARMACOLOGIA::NEUROPSICOFARMACOLOGIA#; 3291013325247130665#; #600

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ribeiro, M. S. (2015). Modulação da AKT/GSK3-B e SNAP-25 pela administração de curcumina em modelo de mania induzido por cetamina. (Masters Thesis). Universidade Catolica de Pelotas; Programa de Pos-Graduacao em Saude Comportamento#;-1990782970254042025#; #600; UCPel; Brasil; Centro de Ciencias da Saude#; #-7432574962795991241#; #600. Retrieved from http://tede.ucpel.edu.br:8080/jspui/handle/tede/536

Chicago Manual of Style (16th Edition):

Ribeiro, Marco Saleh. “Modulação da AKT/GSK3-B e SNAP-25 pela administração de curcumina em modelo de mania induzido por cetamina.” 2015. Masters Thesis, Universidade Catolica de Pelotas; Programa de Pos-Graduacao em Saude Comportamento#;-1990782970254042025#; #600; UCPel; Brasil; Centro de Ciencias da Saude#; #-7432574962795991241#; #600. Accessed April 11, 2021. http://tede.ucpel.edu.br:8080/jspui/handle/tede/536.

MLA Handbook (7th Edition):

Ribeiro, Marco Saleh. “Modulação da AKT/GSK3-B e SNAP-25 pela administração de curcumina em modelo de mania induzido por cetamina.” 2015. Web. 11 Apr 2021.

Vancouver:

Ribeiro MS. Modulação da AKT/GSK3-B e SNAP-25 pela administração de curcumina em modelo de mania induzido por cetamina. [Internet] [Masters thesis]. Universidade Catolica de Pelotas; Programa de Pos-Graduacao em Saude Comportamento#;-1990782970254042025#; #600; UCPel; Brasil; Centro de Ciencias da Saude#; #-7432574962795991241#; #600; 2015. [cited 2021 Apr 11]. Available from: http://tede.ucpel.edu.br:8080/jspui/handle/tede/536.

Council of Science Editors:

Ribeiro MS. Modulação da AKT/GSK3-B e SNAP-25 pela administração de curcumina em modelo de mania induzido por cetamina. [Masters Thesis]. Universidade Catolica de Pelotas; Programa de Pos-Graduacao em Saude Comportamento#;-1990782970254042025#; #600; UCPel; Brasil; Centro de Ciencias da Saude#; #-7432574962795991241#; #600; 2015. Available from: http://tede.ucpel.edu.br:8080/jspui/handle/tede/536

15. Briant, Rémi. Développement de nouvelles séries d’inhibiteurs hétérocycliques des protéines kinases : synthèse, relations structure-activité et optimisation de leur sélectivité au sein du groupe CMGC : Development of new series of heterocyclic inhibitors of protein kinases : synthesis, structure-activity relationships and optimization of their selectivity within the CMGC group.

Degree: Docteur es, Chimie, 2011, Université Claude Bernard – Lyon I

Depuis la mise sur le marché de l'imatinib (ou Gleevec®), les protéines kinases sont devenues des cibles thérapeutiques privilégiées dans le traitement de multiples pathologies.… (more)

Subjects/Keywords: Inhibiteurs; CMGC; CDK; GSK3; CLK; DYRK; Pyrazolo[1,5-a]triazines; N-&-N1; Inhibitors; CMGC; CDK; GSK3; CLK; DYRK; Pyrazolo[1,5-a]triazines; N-&-N1; 540

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Briant, R. (2011). Développement de nouvelles séries d’inhibiteurs hétérocycliques des protéines kinases : synthèse, relations structure-activité et optimisation de leur sélectivité au sein du groupe CMGC : Development of new series of heterocyclic inhibitors of protein kinases : synthesis, structure-activity relationships and optimization of their selectivity within the CMGC group. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2011LYO10256

Chicago Manual of Style (16th Edition):

Briant, Rémi. “Développement de nouvelles séries d’inhibiteurs hétérocycliques des protéines kinases : synthèse, relations structure-activité et optimisation de leur sélectivité au sein du groupe CMGC : Development of new series of heterocyclic inhibitors of protein kinases : synthesis, structure-activity relationships and optimization of their selectivity within the CMGC group.” 2011. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed April 11, 2021. http://www.theses.fr/2011LYO10256.

MLA Handbook (7th Edition):

Briant, Rémi. “Développement de nouvelles séries d’inhibiteurs hétérocycliques des protéines kinases : synthèse, relations structure-activité et optimisation de leur sélectivité au sein du groupe CMGC : Development of new series of heterocyclic inhibitors of protein kinases : synthesis, structure-activity relationships and optimization of their selectivity within the CMGC group.” 2011. Web. 11 Apr 2021.

Vancouver:

Briant R. Développement de nouvelles séries d’inhibiteurs hétérocycliques des protéines kinases : synthèse, relations structure-activité et optimisation de leur sélectivité au sein du groupe CMGC : Development of new series of heterocyclic inhibitors of protein kinases : synthesis, structure-activity relationships and optimization of their selectivity within the CMGC group. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2011. [cited 2021 Apr 11]. Available from: http://www.theses.fr/2011LYO10256.

Council of Science Editors:

Briant R. Développement de nouvelles séries d’inhibiteurs hétérocycliques des protéines kinases : synthèse, relations structure-activité et optimisation de leur sélectivité au sein du groupe CMGC : Development of new series of heterocyclic inhibitors of protein kinases : synthesis, structure-activity relationships and optimization of their selectivity within the CMGC group. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2011. Available from: http://www.theses.fr/2011LYO10256


Temple University

16. Nwaneshiudu, Chinwe A. Characterization of a functional role of the neurokinin-3 receptor in behavioral effects of cocaine.

Degree: PhD, 2011, Temple University

Pharmacology

The tachykinin NK-3 receptor is a G-protein coupled receptor activated by mammalian tachykinin neuropeptides, which can modulate dopaminergic neurotransmission, and alter dopamine-mediated behaviors. The… (more)

Subjects/Keywords: Health Sciences, Pharmacology; Behavioral Sciences; Biology, Neuroscience; CD-1; cocaine; dopamine; GSK3; NK-3; striatum

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APA (6th Edition):

Nwaneshiudu, C. A. (2011). Characterization of a functional role of the neurokinin-3 receptor in behavioral effects of cocaine. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,62614

Chicago Manual of Style (16th Edition):

Nwaneshiudu, Chinwe A. “Characterization of a functional role of the neurokinin-3 receptor in behavioral effects of cocaine.” 2011. Doctoral Dissertation, Temple University. Accessed April 11, 2021. http://digital.library.temple.edu/u?/p245801coll10,62614.

MLA Handbook (7th Edition):

Nwaneshiudu, Chinwe A. “Characterization of a functional role of the neurokinin-3 receptor in behavioral effects of cocaine.” 2011. Web. 11 Apr 2021.

Vancouver:

Nwaneshiudu CA. Characterization of a functional role of the neurokinin-3 receptor in behavioral effects of cocaine. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2021 Apr 11]. Available from: http://digital.library.temple.edu/u?/p245801coll10,62614.

Council of Science Editors:

Nwaneshiudu CA. Characterization of a functional role of the neurokinin-3 receptor in behavioral effects of cocaine. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,62614


Wayne State University

17. Yu, Wenxi. Novel Regulatory Mechanisms Of Inositol Biosynthesis In Saccharomyces Cerevisiae And Mammalian Cells, And Implications For The Mechanism Underlying Vpa-Induced Glucose 6-Phosphate Depletion.

Degree: PhD, Biological Sciences, 2016, Wayne State University

  Myo-inositol is the precursor of all inositol containing molecules, including inositol phosphates, phosphoinositides and glycosylphosphatidylinositols, which are signaling molecules involved in many critical cellular… (more)

Subjects/Keywords: GSK3; inositol biosynthesis; inositol pyrophosphate; IP6K1; valproic acid; Biochemistry; Genetics; Molecular Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yu, W. (2016). Novel Regulatory Mechanisms Of Inositol Biosynthesis In Saccharomyces Cerevisiae And Mammalian Cells, And Implications For The Mechanism Underlying Vpa-Induced Glucose 6-Phosphate Depletion. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1610

Chicago Manual of Style (16th Edition):

Yu, Wenxi. “Novel Regulatory Mechanisms Of Inositol Biosynthesis In Saccharomyces Cerevisiae And Mammalian Cells, And Implications For The Mechanism Underlying Vpa-Induced Glucose 6-Phosphate Depletion.” 2016. Doctoral Dissertation, Wayne State University. Accessed April 11, 2021. https://digitalcommons.wayne.edu/oa_dissertations/1610.

MLA Handbook (7th Edition):

Yu, Wenxi. “Novel Regulatory Mechanisms Of Inositol Biosynthesis In Saccharomyces Cerevisiae And Mammalian Cells, And Implications For The Mechanism Underlying Vpa-Induced Glucose 6-Phosphate Depletion.” 2016. Web. 11 Apr 2021.

Vancouver:

Yu W. Novel Regulatory Mechanisms Of Inositol Biosynthesis In Saccharomyces Cerevisiae And Mammalian Cells, And Implications For The Mechanism Underlying Vpa-Induced Glucose 6-Phosphate Depletion. [Internet] [Doctoral dissertation]. Wayne State University; 2016. [cited 2021 Apr 11]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1610.

Council of Science Editors:

Yu W. Novel Regulatory Mechanisms Of Inositol Biosynthesis In Saccharomyces Cerevisiae And Mammalian Cells, And Implications For The Mechanism Underlying Vpa-Induced Glucose 6-Phosphate Depletion. [Doctoral Dissertation]. Wayne State University; 2016. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1610


Texas Medical Center

18. Bruno, Debora S. Glycogen Synthase Kinase 3 is Required for Optimal AKT Activation.

Degree: MS, 2011, Texas Medical Center

  The phosphatidylinositol 3-kinase (PI3K) pathway, through its major effector node AKT, is critical for the promotion of cell growth, division, motility and apoptosis evasion.… (more)

Subjects/Keywords: GSK3; AKT; p38; PI3K pathway; signaling; reverse phase protein array; Medicine and Health Sciences

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APA (6th Edition):

Bruno, D. S. (2011). Glycogen Synthase Kinase 3 is Required for Optimal AKT Activation. (Thesis). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/200

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bruno, Debora S. “Glycogen Synthase Kinase 3 is Required for Optimal AKT Activation.” 2011. Thesis, Texas Medical Center. Accessed April 11, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/200.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bruno, Debora S. “Glycogen Synthase Kinase 3 is Required for Optimal AKT Activation.” 2011. Web. 11 Apr 2021.

Vancouver:

Bruno DS. Glycogen Synthase Kinase 3 is Required for Optimal AKT Activation. [Internet] [Thesis]. Texas Medical Center; 2011. [cited 2021 Apr 11]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/200.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bruno DS. Glycogen Synthase Kinase 3 is Required for Optimal AKT Activation. [Thesis]. Texas Medical Center; 2011. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/200

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

19. Ko, How-Wen. GSK3BETA-MEDIATED EZH2 PHOSPHORYLATION SUPPRESSES METHYLATION OF H3K27 AND EZH2’S ONCOGENIC FUNCTIONS.

Degree: PhD, 2016, Texas Medical Center

  During the process of tumorigenesis, inactivation of tumor suppressors is a critical step. Enhancer of zeste homolog 2 (EZH2), a histone methyltransferase and the… (more)

Subjects/Keywords: EZH2; GSK3 beta; H3K27me3; Cancer; Phosphorylation; Cancer Biology; Medicine and Health Sciences

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APA (6th Edition):

Ko, H. (2016). GSK3BETA-MEDIATED EZH2 PHOSPHORYLATION SUPPRESSES METHYLATION OF H3K27 AND EZH2’S ONCOGENIC FUNCTIONS. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/669

Chicago Manual of Style (16th Edition):

Ko, How-Wen. “GSK3BETA-MEDIATED EZH2 PHOSPHORYLATION SUPPRESSES METHYLATION OF H3K27 AND EZH2’S ONCOGENIC FUNCTIONS.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed April 11, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/669.

MLA Handbook (7th Edition):

Ko, How-Wen. “GSK3BETA-MEDIATED EZH2 PHOSPHORYLATION SUPPRESSES METHYLATION OF H3K27 AND EZH2’S ONCOGENIC FUNCTIONS.” 2016. Web. 11 Apr 2021.

Vancouver:

Ko H. GSK3BETA-MEDIATED EZH2 PHOSPHORYLATION SUPPRESSES METHYLATION OF H3K27 AND EZH2’S ONCOGENIC FUNCTIONS. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Apr 11]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/669.

Council of Science Editors:

Ko H. GSK3BETA-MEDIATED EZH2 PHOSPHORYLATION SUPPRESSES METHYLATION OF H3K27 AND EZH2’S ONCOGENIC FUNCTIONS. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/669


University of Southern California

20. Shaw, Jillian Lee Satter. Nebula/DSCR1 upregulation preserves axonal transport and memory function in a Drosophila model for Alzheimer's disease.

Degree: PhD, Neuroscience, 2015, University of Southern California

 The Down syndrome critical region 1 gene (also called regulator of calcineurin and Nebula) is located in the DSCR region of human chromosome 21 and… (more)

Subjects/Keywords: Drosophila; Nebula; DSCR1; Down syndrome; axonal transport; Alzheimer'; s disease; memory; calcineurin; GSK3

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APA (6th Edition):

Shaw, J. L. S. (2015). Nebula/DSCR1 upregulation preserves axonal transport and memory function in a Drosophila model for Alzheimer's disease. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/538930/rec/4349

Chicago Manual of Style (16th Edition):

Shaw, Jillian Lee Satter. “Nebula/DSCR1 upregulation preserves axonal transport and memory function in a Drosophila model for Alzheimer's disease.” 2015. Doctoral Dissertation, University of Southern California. Accessed April 11, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/538930/rec/4349.

MLA Handbook (7th Edition):

Shaw, Jillian Lee Satter. “Nebula/DSCR1 upregulation preserves axonal transport and memory function in a Drosophila model for Alzheimer's disease.” 2015. Web. 11 Apr 2021.

Vancouver:

Shaw JLS. Nebula/DSCR1 upregulation preserves axonal transport and memory function in a Drosophila model for Alzheimer's disease. [Internet] [Doctoral dissertation]. University of Southern California; 2015. [cited 2021 Apr 11]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/538930/rec/4349.

Council of Science Editors:

Shaw JLS. Nebula/DSCR1 upregulation preserves axonal transport and memory function in a Drosophila model for Alzheimer's disease. [Doctoral Dissertation]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/538930/rec/4349


University of Kansas

21. Bhushan, Bharat. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2016, University of Kansas

 Overdose of acetaminophen (APAP) is the major cause of acute liver failure (ALF) in the western world with very limited treatment options. Recent studies suggest… (more)

Subjects/Keywords: Toxicology; Pharmacology; Acetaminophen; EGF receptor; Liver; mice model; Regeneration; Wnt-Beta Catenin-GSK3 Beta pathway

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APA (6th Edition):

Bhushan, B. (2016). MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/25366

Chicago Manual of Style (16th Edition):

Bhushan, Bharat. “MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.” 2016. Doctoral Dissertation, University of Kansas. Accessed April 11, 2021. http://hdl.handle.net/1808/25366.

MLA Handbook (7th Edition):

Bhushan, Bharat. “MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.” 2016. Web. 11 Apr 2021.

Vancouver:

Bhushan B. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/1808/25366.

Council of Science Editors:

Bhushan B. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/25366


University of Western Ontario

22. Jassar, Harman. The Role of GSK3 alpha and beta in Embryonic Craniofacial Development.

Degree: 2021, University of Western Ontario

 Background: The GSK-3 genes (Gsk3a and Gsk3b) have been known to affect many cellular processes and signaling pathways some of which are implicated in the… (more)

Subjects/Keywords: GSK3; craniofacial; mandible; cleft palate; embryonic; intramandibular angle; Genetics; Musculoskeletal System; Orthodontics and Orthodontology

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APA (6th Edition):

Jassar, H. (2021). The Role of GSK3 alpha and beta in Embryonic Craniofacial Development. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/7612

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jassar, Harman. “The Role of GSK3 alpha and beta in Embryonic Craniofacial Development.” 2021. Thesis, University of Western Ontario. Accessed April 11, 2021. https://ir.lib.uwo.ca/etd/7612.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jassar, Harman. “The Role of GSK3 alpha and beta in Embryonic Craniofacial Development.” 2021. Web. 11 Apr 2021.

Vancouver:

Jassar H. The Role of GSK3 alpha and beta in Embryonic Craniofacial Development. [Internet] [Thesis]. University of Western Ontario; 2021. [cited 2021 Apr 11]. Available from: https://ir.lib.uwo.ca/etd/7612.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jassar H. The Role of GSK3 alpha and beta in Embryonic Craniofacial Development. [Thesis]. University of Western Ontario; 2021. Available from: https://ir.lib.uwo.ca/etd/7612

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

23. Kashikar, Nilesh Digvijay. Novel and diverse roles of STRAP in maintenance of mesenchymal morphology and GSK3Beta signaling.

Degree: PhD, Cancer Biology, 2010, Vanderbilt University

 STRAP inhibits transforming growth factor-Beta (TGF-Beta) signaling and enhances tumorigenicity. The aim of our current research project was to identify novel TGF-Beta independent functions of… (more)

Subjects/Keywords: STRAP; EMT; Notch3; Lung cancer; Ubiquitination; GSK3

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APA (6th Edition):

Kashikar, N. D. (2010). Novel and diverse roles of STRAP in maintenance of mesenchymal morphology and GSK3Beta signaling. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12466

Chicago Manual of Style (16th Edition):

Kashikar, Nilesh Digvijay. “Novel and diverse roles of STRAP in maintenance of mesenchymal morphology and GSK3Beta signaling.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed April 11, 2021. http://hdl.handle.net/1803/12466.

MLA Handbook (7th Edition):

Kashikar, Nilesh Digvijay. “Novel and diverse roles of STRAP in maintenance of mesenchymal morphology and GSK3Beta signaling.” 2010. Web. 11 Apr 2021.

Vancouver:

Kashikar ND. Novel and diverse roles of STRAP in maintenance of mesenchymal morphology and GSK3Beta signaling. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/1803/12466.

Council of Science Editors:

Kashikar ND. Novel and diverse roles of STRAP in maintenance of mesenchymal morphology and GSK3Beta signaling. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://hdl.handle.net/1803/12466


Texas Medical Center

24. Smith, Debra L. GSK3 Mediates Signaling Upstream of Akt.

Degree: PhD, 2010, Texas Medical Center

 The phosphatidylinositide 3 kinase (PI3K)/Akt signaling network plays a pivotal role in multiple cellular functions. PI3K links the extracellular growth factor receptors to the serine-threonine… (more)

Subjects/Keywords: GSK3; Akt; PI3K; cancer cell signaling; signal transduction; Biology

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APA (6th Edition):

Smith, D. L. (2010). GSK3 Mediates Signaling Upstream of Akt. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/12

Chicago Manual of Style (16th Edition):

Smith, Debra L. “GSK3 Mediates Signaling Upstream of Akt.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed April 11, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/12.

MLA Handbook (7th Edition):

Smith, Debra L. “GSK3 Mediates Signaling Upstream of Akt.” 2010. Web. 11 Apr 2021.

Vancouver:

Smith DL. GSK3 Mediates Signaling Upstream of Akt. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2021 Apr 11]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/12.

Council of Science Editors:

Smith DL. GSK3 Mediates Signaling Upstream of Akt. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/12

25. Downey, Kimberlee. Antidepressant Interactions of Ketamine and Glycogen Synthase Kinase-3 (GSK3) in Mice.

Degree: MS, Biochemistry and Molecular Biology (Medicine), 2015, University of Miami

 Depression is a prevalent and debilitating mood disorder afflicting nearly one in five people in the United States. Current medications used for treatment of depression… (more)

Subjects/Keywords: ketamine; GSK3; mice; depression; antidepressant effect

…4. Ketamine’s Antidepressant Effect Involves Signaling Between GSK3 and AMPA Receptors… …GSK3 regulation and GSK3 knockin mice .. 13 Figure 3.2.1- Ketamine increases… …inhibitory serine phosphorylation of GSK3 … 24 Figure 3.2.2… …Experimental setup for investigating requirement of GSK3 inhibition in ketamine antidepressant… …response … 25 Figure 3.2.3- GSK3 inhibition is required for the antidepressant effect of… 

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APA (6th Edition):

Downey, K. (2015). Antidepressant Interactions of Ketamine and Glycogen Synthase Kinase-3 (GSK3) in Mice. (Thesis). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_theses/545

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Downey, Kimberlee. “Antidepressant Interactions of Ketamine and Glycogen Synthase Kinase-3 (GSK3) in Mice.” 2015. Thesis, University of Miami. Accessed April 11, 2021. https://scholarlyrepository.miami.edu/oa_theses/545.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Downey, Kimberlee. “Antidepressant Interactions of Ketamine and Glycogen Synthase Kinase-3 (GSK3) in Mice.” 2015. Web. 11 Apr 2021.

Vancouver:

Downey K. Antidepressant Interactions of Ketamine and Glycogen Synthase Kinase-3 (GSK3) in Mice. [Internet] [Thesis]. University of Miami; 2015. [cited 2021 Apr 11]. Available from: https://scholarlyrepository.miami.edu/oa_theses/545.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Downey K. Antidepressant Interactions of Ketamine and Glycogen Synthase Kinase-3 (GSK3) in Mice. [Thesis]. University of Miami; 2015. Available from: https://scholarlyrepository.miami.edu/oa_theses/545

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Lindenberg, J.J. Targeting skin associated DC subsets and conferring resistance to tumor-related immune suppression .

Degree: 2014, Vrije Universiteit Amsterdam

Subjects/Keywords: iimmune suppression; Dendritic cells; CD14; STAT3; GSK3-beta; Targetting

Page 1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lindenberg, J. J. (2014). Targeting skin associated DC subsets and conferring resistance to tumor-related immune suppression . (Doctoral Dissertation). Vrije Universiteit Amsterdam. Retrieved from http://hdl.handle.net/1871/52011

Chicago Manual of Style (16th Edition):

Lindenberg, J J. “Targeting skin associated DC subsets and conferring resistance to tumor-related immune suppression .” 2014. Doctoral Dissertation, Vrije Universiteit Amsterdam. Accessed April 11, 2021. http://hdl.handle.net/1871/52011.

MLA Handbook (7th Edition):

Lindenberg, J J. “Targeting skin associated DC subsets and conferring resistance to tumor-related immune suppression .” 2014. Web. 11 Apr 2021.

Vancouver:

Lindenberg JJ. Targeting skin associated DC subsets and conferring resistance to tumor-related immune suppression . [Internet] [Doctoral dissertation]. Vrije Universiteit Amsterdam; 2014. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/1871/52011.

Council of Science Editors:

Lindenberg JJ. Targeting skin associated DC subsets and conferring resistance to tumor-related immune suppression . [Doctoral Dissertation]. Vrije Universiteit Amsterdam; 2014. Available from: http://hdl.handle.net/1871/52011

27. Ali, Husnain. Exploring the potential of glycogen synthase kinase-3 (GSK3) mediated therapy in castrate-resistant prostate cancer .

Degree: 2016, National University of Ireland – Galway

 Glycogen Synthase Kinase 3 (GSK3) is involved in a myriad of signalling pathways and has recently received considerable interest due to its conflicting roles tumourigenesis.… (more)

Subjects/Keywords: GSK3; Cancer; Apoptosis; NFkB; Medicine; Prostate cancer

…Synthase Kinase 3 (GSK3) is involved in a myriad of signalling pathways and has… …has classically been regarded as a tumour suppressor, emerging evidence suggests that GSK3… …it is also a tumour promoter. One of GSK3’s tumour promoting properties has been linked to… …resistant prostate cancer (CRPC). Targeting GSK3 has been shown to reduce NFκB activity… …levels are increased in CRPC. In this study we sought to determine if GSK3 regulates NFκB… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ali, H. (2016). Exploring the potential of glycogen synthase kinase-3 (GSK3) mediated therapy in castrate-resistant prostate cancer . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/6387

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ali, Husnain. “Exploring the potential of glycogen synthase kinase-3 (GSK3) mediated therapy in castrate-resistant prostate cancer .” 2016. Thesis, National University of Ireland – Galway. Accessed April 11, 2021. http://hdl.handle.net/10379/6387.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ali, Husnain. “Exploring the potential of glycogen synthase kinase-3 (GSK3) mediated therapy in castrate-resistant prostate cancer .” 2016. Web. 11 Apr 2021.

Vancouver:

Ali H. Exploring the potential of glycogen synthase kinase-3 (GSK3) mediated therapy in castrate-resistant prostate cancer . [Internet] [Thesis]. National University of Ireland – Galway; 2016. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10379/6387.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ali H. Exploring the potential of glycogen synthase kinase-3 (GSK3) mediated therapy in castrate-resistant prostate cancer . [Thesis]. National University of Ireland – Galway; 2016. Available from: http://hdl.handle.net/10379/6387

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Sherbrooke

28. Marchand, Benoît. L'activité des glycogènes synthase kinases 3 est essentielle à la survie et à la prolifération des cellules pancréatiques tumorales humaines.

Degree: 2009, Université de Sherbrooke

 Depuis leur découverte, les glycogènes synthase kinases 3 (GSK3) ont été associées à de multiples fonctions cellulaires, incluant notamment la prolifération et la survie cellulaire.… (more)

Subjects/Keywords: Pancréas; BIM; Apoptose; Prolifération; JNK; GSK3; Cancer pancréatique

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APA (6th Edition):

Marchand, B. (2009). L'activité des glycogènes synthase kinases 3 est essentielle à la survie et à la prolifération des cellules pancréatiques tumorales humaines. (Masters Thesis). Université de Sherbrooke. Retrieved from http://savoirs.usherbrooke.ca/handle/11143/4047

Chicago Manual of Style (16th Edition):

Marchand, Benoît. “L'activité des glycogènes synthase kinases 3 est essentielle à la survie et à la prolifération des cellules pancréatiques tumorales humaines.” 2009. Masters Thesis, Université de Sherbrooke. Accessed April 11, 2021. http://savoirs.usherbrooke.ca/handle/11143/4047.

MLA Handbook (7th Edition):

Marchand, Benoît. “L'activité des glycogènes synthase kinases 3 est essentielle à la survie et à la prolifération des cellules pancréatiques tumorales humaines.” 2009. Web. 11 Apr 2021.

Vancouver:

Marchand B. L'activité des glycogènes synthase kinases 3 est essentielle à la survie et à la prolifération des cellules pancréatiques tumorales humaines. [Internet] [Masters thesis]. Université de Sherbrooke; 2009. [cited 2021 Apr 11]. Available from: http://savoirs.usherbrooke.ca/handle/11143/4047.

Council of Science Editors:

Marchand B. L'activité des glycogènes synthase kinases 3 est essentielle à la survie et à la prolifération des cellules pancréatiques tumorales humaines. [Masters Thesis]. Université de Sherbrooke; 2009. Available from: http://savoirs.usherbrooke.ca/handle/11143/4047

29. ANIRUDH GAUTAM MANNAVA. WNT SIGNALLING AT THE MEMBRANE.

Degree: 2016, National University of Singapore

Subjects/Keywords: Wnt signalling; GSK3; Disheveled; membrane activation complex; OTK; neuroblast

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

MANNAVA, A. G. (2016). WNT SIGNALLING AT THE MEMBRANE. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/130175

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

MANNAVA, ANIRUDH GAUTAM. “WNT SIGNALLING AT THE MEMBRANE.” 2016. Thesis, National University of Singapore. Accessed April 11, 2021. http://scholarbank.nus.edu.sg/handle/10635/130175.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

MANNAVA, ANIRUDH GAUTAM. “WNT SIGNALLING AT THE MEMBRANE.” 2016. Web. 11 Apr 2021.

Vancouver:

MANNAVA AG. WNT SIGNALLING AT THE MEMBRANE. [Internet] [Thesis]. National University of Singapore; 2016. [cited 2021 Apr 11]. Available from: http://scholarbank.nus.edu.sg/handle/10635/130175.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

MANNAVA AG. WNT SIGNALLING AT THE MEMBRANE. [Thesis]. National University of Singapore; 2016. Available from: http://scholarbank.nus.edu.sg/handle/10635/130175

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

30. Li, Chen. PI3K/AKT signaling and the regulation of the mitochondrial energy-redox axis.

Degree: PhD, Molecular Pharmacology and Toxicology, 2012, University of Southern California

 The concept of the mitochondrial energy-redox axis integrates the mitochondrial energy-transduction and redox status as a concerted process with the two components inter-linked by the… (more)

Subjects/Keywords: mitochondrial respiration; PI3K; AKT; PTEN; GSK3&beta;

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APA (6th Edition):

Li, C. (2012). PI3K/AKT signaling and the regulation of the mitochondrial energy-redox axis. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/23637/rec/5051

Chicago Manual of Style (16th Edition):

Li, Chen. “PI3K/AKT signaling and the regulation of the mitochondrial energy-redox axis.” 2012. Doctoral Dissertation, University of Southern California. Accessed April 11, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/23637/rec/5051.

MLA Handbook (7th Edition):

Li, Chen. “PI3K/AKT signaling and the regulation of the mitochondrial energy-redox axis.” 2012. Web. 11 Apr 2021.

Vancouver:

Li C. PI3K/AKT signaling and the regulation of the mitochondrial energy-redox axis. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2021 Apr 11]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/23637/rec/5051.

Council of Science Editors:

Li C. PI3K/AKT signaling and the regulation of the mitochondrial energy-redox axis. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/23637/rec/5051

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