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You searched for subject:(GPVI). Showing records 1 – 5 of 5 total matches.

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University College Cork

1. Stack, John. Rheumatoid arthritis and atherosclerosis: common pathogenic mechanisms explored.

Degree: 2020, University College Cork

 This body of work draws on parallels between the pathogenesis of rheumatoid arthritis (RA) and atherosclerosis. The pathogenesis and genetic basis of both diseases are… (more)

Subjects/Keywords: Rheumatoid arthritis; CX3CR1; GPVI; Atherosclerosis; Soluble GPVI; Genetic case control study; Biomarker

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stack, J. (2020). Rheumatoid arthritis and atherosclerosis: common pathogenic mechanisms explored. (Thesis). University College Cork. Retrieved from http://hdl.handle.net/10468/9939

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stack, John. “Rheumatoid arthritis and atherosclerosis: common pathogenic mechanisms explored.” 2020. Thesis, University College Cork. Accessed October 28, 2020. http://hdl.handle.net/10468/9939.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stack, John. “Rheumatoid arthritis and atherosclerosis: common pathogenic mechanisms explored.” 2020. Web. 28 Oct 2020.

Vancouver:

Stack J. Rheumatoid arthritis and atherosclerosis: common pathogenic mechanisms explored. [Internet] [Thesis]. University College Cork; 2020. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10468/9939.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stack J. Rheumatoid arthritis and atherosclerosis: common pathogenic mechanisms explored. [Thesis]. University College Cork; 2020. Available from: http://hdl.handle.net/10468/9939

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

2. Thomas, Dafydd Huw. Regulation of Syk activity in GPVI-mediated platelet activation.

Degree: PhD, 2010, Temple University

Pharmacology

Activation of platelets is essential for hemostasis. Following damage to the vascular endothelium collagen is exposed, to which platelets stably adhere. After adhesion on… (more)

Subjects/Keywords: Health Sciences, Pharmacology; Collagen; GPVI; Phosphatase; Platelets; Syk; TULA

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Thomas, D. H. (2010). Regulation of Syk activity in GPVI-mediated platelet activation. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,92028

Chicago Manual of Style (16th Edition):

Thomas, Dafydd Huw. “Regulation of Syk activity in GPVI-mediated platelet activation.” 2010. Doctoral Dissertation, Temple University. Accessed October 28, 2020. http://digital.library.temple.edu/u?/p245801coll10,92028.

MLA Handbook (7th Edition):

Thomas, Dafydd Huw. “Regulation of Syk activity in GPVI-mediated platelet activation.” 2010. Web. 28 Oct 2020.

Vancouver:

Thomas DH. Regulation of Syk activity in GPVI-mediated platelet activation. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Oct 28]. Available from: http://digital.library.temple.edu/u?/p245801coll10,92028.

Council of Science Editors:

Thomas DH. Regulation of Syk activity in GPVI-mediated platelet activation. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,92028


University of Pennsylvania

3. Lee, Mei Yan. Systems Biology of Platelet Activation.

Degree: 2016, University of Pennsylvania

 Platelet intracellular calcium mobilization [Ca(t)]i is a measure of platelet activation and controls important events downstream that contribute to hemostasis such as granule release, cyclooxygenase-1… (more)

Subjects/Keywords: Calcium; Fibrin; GPVI; Machine learning; Neural Networks; Platelet; Bioinformatics; Biology; Chemical Engineering

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, M. Y. (2016). Systems Biology of Platelet Activation. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1835

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Mei Yan. “Systems Biology of Platelet Activation.” 2016. Thesis, University of Pennsylvania. Accessed October 28, 2020. https://repository.upenn.edu/edissertations/1835.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Mei Yan. “Systems Biology of Platelet Activation.” 2016. Web. 28 Oct 2020.

Vancouver:

Lee MY. Systems Biology of Platelet Activation. [Internet] [Thesis]. University of Pennsylvania; 2016. [cited 2020 Oct 28]. Available from: https://repository.upenn.edu/edissertations/1835.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee MY. Systems Biology of Platelet Activation. [Thesis]. University of Pennsylvania; 2016. Available from: https://repository.upenn.edu/edissertations/1835

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Roweth, Harvey George. Mechanisms of platelet inhibition by the selective serotonin reuptake inhibitor citalopram.

Degree: PhD, 2018, University of Cambridge

 Background: Selective serotonin reuptake inhibitor (SSRI) antidepressants prevent serotonin (5-HT) uptake by the serotonin transporter (SERT). Since blood platelets express SERT, SSRIs may modify platelet… (more)

Subjects/Keywords: 616.1; Platelet; Citalopram; Selective serotonin reuptake inhibitor; CalDAG-GEFI; GPVI; Aggregation; Rap1; Calcium; Serotonin; Serotonin transporter; Neutrophil; Thrombosis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Roweth, H. G. (2018). Mechanisms of platelet inhibition by the selective serotonin reuptake inhibitor citalopram. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.22708 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744799

Chicago Manual of Style (16th Edition):

Roweth, Harvey George. “Mechanisms of platelet inhibition by the selective serotonin reuptake inhibitor citalopram.” 2018. Doctoral Dissertation, University of Cambridge. Accessed October 28, 2020. https://doi.org/10.17863/CAM.22708 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744799.

MLA Handbook (7th Edition):

Roweth, Harvey George. “Mechanisms of platelet inhibition by the selective serotonin reuptake inhibitor citalopram.” 2018. Web. 28 Oct 2020.

Vancouver:

Roweth HG. Mechanisms of platelet inhibition by the selective serotonin reuptake inhibitor citalopram. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2020 Oct 28]. Available from: https://doi.org/10.17863/CAM.22708 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744799.

Council of Science Editors:

Roweth HG. Mechanisms of platelet inhibition by the selective serotonin reuptake inhibitor citalopram. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://doi.org/10.17863/CAM.22708 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744799


University of Pennsylvania

5. Schmaier, Alec A. The Evolution of Platelet Responses to Collagen Under Conditions of Hemodynamic Flow.

Degree: 2010, University of Pennsylvania

 Collagen activates platelets through the immune-type GPVI receptor, but the mechanism by which this receptor has adapted to function in platelets under conditions of rapid… (more)

Subjects/Keywords: platelet; collagen; GPVI; immune; hemostasis; thrombosis; Biochemistry; Cardiology; Cellular and Molecular Physiology; Comparative and Evolutionary Physiology; Other Immunology and Infectious Disease; Poultry or Avian Science

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schmaier, A. A. (2010). The Evolution of Platelet Responses to Collagen Under Conditions of Hemodynamic Flow. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/203

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schmaier, Alec A. “The Evolution of Platelet Responses to Collagen Under Conditions of Hemodynamic Flow.” 2010. Thesis, University of Pennsylvania. Accessed October 28, 2020. https://repository.upenn.edu/edissertations/203.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schmaier, Alec A. “The Evolution of Platelet Responses to Collagen Under Conditions of Hemodynamic Flow.” 2010. Web. 28 Oct 2020.

Vancouver:

Schmaier AA. The Evolution of Platelet Responses to Collagen Under Conditions of Hemodynamic Flow. [Internet] [Thesis]. University of Pennsylvania; 2010. [cited 2020 Oct 28]. Available from: https://repository.upenn.edu/edissertations/203.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schmaier AA. The Evolution of Platelet Responses to Collagen Under Conditions of Hemodynamic Flow. [Thesis]. University of Pennsylvania; 2010. Available from: https://repository.upenn.edu/edissertations/203

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.