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You searched for subject:(G protein coupled receptor). Showing records 1 – 30 of 28263 total matches.

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University of Toronto

1. Hamoudi, Zina. Isolation and Characterization of the Corazonin Receptor and Possible Physiological Roles of Corazonin in the Kissing Bug, Rhodnius prolixus.

Degree: 2015, University of Toronto

Neuropeptides control many physiological and endocrinological processes in animals, acting as neuroactive chemicals within the central and peripheral nervous systems. Corazonin (CRZ) is one such… (more)

Subjects/Keywords: G protein-coupled receptor; Insect; Neuropeptide; 0409

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APA (6th Edition):

Hamoudi, Z. (2015). Isolation and Characterization of the Corazonin Receptor and Possible Physiological Roles of Corazonin in the Kissing Bug, Rhodnius prolixus. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70398

Chicago Manual of Style (16th Edition):

Hamoudi, Zina. “Isolation and Characterization of the Corazonin Receptor and Possible Physiological Roles of Corazonin in the Kissing Bug, Rhodnius prolixus.” 2015. Masters Thesis, University of Toronto. Accessed April 09, 2020. http://hdl.handle.net/1807/70398.

MLA Handbook (7th Edition):

Hamoudi, Zina. “Isolation and Characterization of the Corazonin Receptor and Possible Physiological Roles of Corazonin in the Kissing Bug, Rhodnius prolixus.” 2015. Web. 09 Apr 2020.

Vancouver:

Hamoudi Z. Isolation and Characterization of the Corazonin Receptor and Possible Physiological Roles of Corazonin in the Kissing Bug, Rhodnius prolixus. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/1807/70398.

Council of Science Editors:

Hamoudi Z. Isolation and Characterization of the Corazonin Receptor and Possible Physiological Roles of Corazonin in the Kissing Bug, Rhodnius prolixus. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70398


Penn State University

2. Huang, Weishan. IDENTIFICATION AND CHARACTERIZATION OF.

Degree: MS, Cell and Molecular Biology, 2010, Penn State University

 The Tec family kinase, Itk has been shown to be critical in T cell activation and development downstream of the T cell receptor (TCR). Other… (more)

Subjects/Keywords: protein-protein interaction; ; 13; signal transduction; T cell receptor; Itk; G protein coupled receptor

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APA (6th Edition):

Huang, W. (2010). IDENTIFICATION AND CHARACTERIZATION OF. (Masters Thesis). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/11229

Chicago Manual of Style (16th Edition):

Huang, Weishan. “IDENTIFICATION AND CHARACTERIZATION OF.” 2010. Masters Thesis, Penn State University. Accessed April 09, 2020. https://etda.libraries.psu.edu/catalog/11229.

MLA Handbook (7th Edition):

Huang, Weishan. “IDENTIFICATION AND CHARACTERIZATION OF.” 2010. Web. 09 Apr 2020.

Vancouver:

Huang W. IDENTIFICATION AND CHARACTERIZATION OF. [Internet] [Masters thesis]. Penn State University; 2010. [cited 2020 Apr 09]. Available from: https://etda.libraries.psu.edu/catalog/11229.

Council of Science Editors:

Huang W. IDENTIFICATION AND CHARACTERIZATION OF. [Masters Thesis]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/11229


University of Manitoba

3. Sidhu, Crystal. Quinine regulates Rac1 GTPase activity through the bitter taste receptor T2R4 and G-Protein.

Degree: Oral Biology, 2016, University of Manitoba

 Rac1 is a member of the Rho family of low molecular mass GTP binding proteins (GTPases). It regulates the dynamics of actin cytoskeleton by causing… (more)

Subjects/Keywords: G protein-coupled receptor; Rac1 inactivation; Bitter taste receptor; G-alpha protein; Quinine

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APA (6th Edition):

Sidhu, C. (2016). Quinine regulates Rac1 GTPase activity through the bitter taste receptor T2R4 and G-Protein. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31786

Chicago Manual of Style (16th Edition):

Sidhu, Crystal. “Quinine regulates Rac1 GTPase activity through the bitter taste receptor T2R4 and G-Protein.” 2016. Masters Thesis, University of Manitoba. Accessed April 09, 2020. http://hdl.handle.net/1993/31786.

MLA Handbook (7th Edition):

Sidhu, Crystal. “Quinine regulates Rac1 GTPase activity through the bitter taste receptor T2R4 and G-Protein.” 2016. Web. 09 Apr 2020.

Vancouver:

Sidhu C. Quinine regulates Rac1 GTPase activity through the bitter taste receptor T2R4 and G-Protein. [Internet] [Masters thesis]. University of Manitoba; 2016. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/1993/31786.

Council of Science Editors:

Sidhu C. Quinine regulates Rac1 GTPase activity through the bitter taste receptor T2R4 and G-Protein. [Masters Thesis]. University of Manitoba; 2016. Available from: http://hdl.handle.net/1993/31786

4. Lokits, Alyssa Dawn. Nuthin' but a G (protein) thang: Insights into the Mechanics of G protein Signaling from Sequence and Structure.

Degree: PhD, Neuroscience, 2017, Vanderbilt University

G protein-coupled receptors (GPCRs) are a large and diverse group of transmembrane receptors which convert extracellular signals into intracellular responses via coupling to heterotrimeric G(more)

Subjects/Keywords: G protein; G protein Coupled Receptor; GPCR; structure; computation; evolution; phylogenetics; thermodynamics; G alpha; Rosetta

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APA (6th Edition):

Lokits, A. D. (2017). Nuthin' but a G (protein) thang: Insights into the Mechanics of G protein Signaling from Sequence and Structure. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-06132017-085149/ ;

Chicago Manual of Style (16th Edition):

Lokits, Alyssa Dawn. “Nuthin' but a G (protein) thang: Insights into the Mechanics of G protein Signaling from Sequence and Structure.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed April 09, 2020. http://etd.library.vanderbilt.edu/available/etd-06132017-085149/ ;.

MLA Handbook (7th Edition):

Lokits, Alyssa Dawn. “Nuthin' but a G (protein) thang: Insights into the Mechanics of G protein Signaling from Sequence and Structure.” 2017. Web. 09 Apr 2020.

Vancouver:

Lokits AD. Nuthin' but a G (protein) thang: Insights into the Mechanics of G protein Signaling from Sequence and Structure. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2020 Apr 09]. Available from: http://etd.library.vanderbilt.edu/available/etd-06132017-085149/ ;.

Council of Science Editors:

Lokits AD. Nuthin' but a G (protein) thang: Insights into the Mechanics of G protein Signaling from Sequence and Structure. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://etd.library.vanderbilt.edu/available/etd-06132017-085149/ ;


University of Edinburgh

5. White, Colin D. Dissection of GnRH receptor-G protein coupling.

Degree: 2009, University of Edinburgh

 Hypothalamic gonadotropin-releasing hormone (GnRH) (GnRH I) is the central regulator of the mammalian reproductive system. Most vertebrates studied also possess a second form of GnRH,… (more)

Subjects/Keywords: 612.6; GnRH; Hypothalamic gonadotropin-releasing hormone; G-protein; G protein-coupled receptor

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APA (6th Edition):

White, C. D. (2009). Dissection of GnRH receptor-G protein coupling. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/3885

Chicago Manual of Style (16th Edition):

White, Colin D. “Dissection of GnRH receptor-G protein coupling.” 2009. Doctoral Dissertation, University of Edinburgh. Accessed April 09, 2020. http://hdl.handle.net/1842/3885.

MLA Handbook (7th Edition):

White, Colin D. “Dissection of GnRH receptor-G protein coupling.” 2009. Web. 09 Apr 2020.

Vancouver:

White CD. Dissection of GnRH receptor-G protein coupling. [Internet] [Doctoral dissertation]. University of Edinburgh; 2009. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/1842/3885.

Council of Science Editors:

White CD. Dissection of GnRH receptor-G protein coupling. [Doctoral Dissertation]. University of Edinburgh; 2009. Available from: http://hdl.handle.net/1842/3885


Dalhousie University

6. Charette, Nicholle Jeanine. INVOLVEMENT OF DIFFERENT RAB GTPASES IN THE TRAFFICKING OF CXCR4 AND CCR5 HOMO- AND HETERODIMERS BETWEEN THE ENDOPLASMIC RETICULUM AND PLASMA MEMBRANE IN HEK293 AND JURKAT CELLS.

Degree: MS, Department of Pharmacology, 2011, Dalhousie University

 Little is known about the outward trafficking of receptor dimers from the endoplasmic reticulum to the plasma membrane, or the role that trafficking plays in… (more)

Subjects/Keywords: G protein coupled receptor; CXCR4; CCR5; Rab GTPase; Trafficking; Dimerization

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APA (6th Edition):

Charette, N. J. (2011). INVOLVEMENT OF DIFFERENT RAB GTPASES IN THE TRAFFICKING OF CXCR4 AND CCR5 HOMO- AND HETERODIMERS BETWEEN THE ENDOPLASMIC RETICULUM AND PLASMA MEMBRANE IN HEK293 AND JURKAT CELLS. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/14023

Chicago Manual of Style (16th Edition):

Charette, Nicholle Jeanine. “INVOLVEMENT OF DIFFERENT RAB GTPASES IN THE TRAFFICKING OF CXCR4 AND CCR5 HOMO- AND HETERODIMERS BETWEEN THE ENDOPLASMIC RETICULUM AND PLASMA MEMBRANE IN HEK293 AND JURKAT CELLS.” 2011. Masters Thesis, Dalhousie University. Accessed April 09, 2020. http://hdl.handle.net/10222/14023.

MLA Handbook (7th Edition):

Charette, Nicholle Jeanine. “INVOLVEMENT OF DIFFERENT RAB GTPASES IN THE TRAFFICKING OF CXCR4 AND CCR5 HOMO- AND HETERODIMERS BETWEEN THE ENDOPLASMIC RETICULUM AND PLASMA MEMBRANE IN HEK293 AND JURKAT CELLS.” 2011. Web. 09 Apr 2020.

Vancouver:

Charette NJ. INVOLVEMENT OF DIFFERENT RAB GTPASES IN THE TRAFFICKING OF CXCR4 AND CCR5 HOMO- AND HETERODIMERS BETWEEN THE ENDOPLASMIC RETICULUM AND PLASMA MEMBRANE IN HEK293 AND JURKAT CELLS. [Internet] [Masters thesis]. Dalhousie University; 2011. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/10222/14023.

Council of Science Editors:

Charette NJ. INVOLVEMENT OF DIFFERENT RAB GTPASES IN THE TRAFFICKING OF CXCR4 AND CCR5 HOMO- AND HETERODIMERS BETWEEN THE ENDOPLASMIC RETICULUM AND PLASMA MEMBRANE IN HEK293 AND JURKAT CELLS. [Masters Thesis]. Dalhousie University; 2011. Available from: http://hdl.handle.net/10222/14023


Dalhousie University

7. Langelaan, David. Structural Studies Of Apelin And Its Receptor As Well As The Characteristics And Causes Of Membrane Protein Helix Kinks.

Degree: PhD, Department of Biochemistry & Molecular Biology, 2013, Dalhousie University

 Apelin, the endogenous ligand to the apelin receptor, is a small peptide involved with cardiovascular regulation. Using nuclear magnetic resonance (NMR) spectroscopy, I demonstrate that… (more)

Subjects/Keywords: G-protein coupled receptor; Nuclear magnetic resonance spectroscopy; Apelin

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APA (6th Edition):

Langelaan, D. (2013). Structural Studies Of Apelin And Its Receptor As Well As The Characteristics And Causes Of Membrane Protein Helix Kinks. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/22285

Chicago Manual of Style (16th Edition):

Langelaan, David. “Structural Studies Of Apelin And Its Receptor As Well As The Characteristics And Causes Of Membrane Protein Helix Kinks.” 2013. Doctoral Dissertation, Dalhousie University. Accessed April 09, 2020. http://hdl.handle.net/10222/22285.

MLA Handbook (7th Edition):

Langelaan, David. “Structural Studies Of Apelin And Its Receptor As Well As The Characteristics And Causes Of Membrane Protein Helix Kinks.” 2013. Web. 09 Apr 2020.

Vancouver:

Langelaan D. Structural Studies Of Apelin And Its Receptor As Well As The Characteristics And Causes Of Membrane Protein Helix Kinks. [Internet] [Doctoral dissertation]. Dalhousie University; 2013. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/10222/22285.

Council of Science Editors:

Langelaan D. Structural Studies Of Apelin And Its Receptor As Well As The Characteristics And Causes Of Membrane Protein Helix Kinks. [Doctoral Dissertation]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/22285


University of Kansas

8. Shannon, Laura. The Role of the G-Protein Coupled Receptor C-C Chemokine Receptor 7 in T Lymphocyte Migration and Breast Cancer Metastasis.

Degree: PhD, Microbiology, Molecular Genetics & Immunology, 2010, University of Kansas

 C-C Chemokine Receptor 7 (CCR7) promotes migration of T lymphocytes into and throughout lymph nodes via ligands CCL21 and CCL19. The mechanisms by which CCR7… (more)

Subjects/Keywords: Cell biology; Breast cancer; Chemokine; G-protein coupled receptor; Immunology

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APA (6th Edition):

Shannon, L. (2010). The Role of the G-Protein Coupled Receptor C-C Chemokine Receptor 7 in T Lymphocyte Migration and Breast Cancer Metastasis. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/7423

Chicago Manual of Style (16th Edition):

Shannon, Laura. “The Role of the G-Protein Coupled Receptor C-C Chemokine Receptor 7 in T Lymphocyte Migration and Breast Cancer Metastasis.” 2010. Doctoral Dissertation, University of Kansas. Accessed April 09, 2020. http://hdl.handle.net/1808/7423.

MLA Handbook (7th Edition):

Shannon, Laura. “The Role of the G-Protein Coupled Receptor C-C Chemokine Receptor 7 in T Lymphocyte Migration and Breast Cancer Metastasis.” 2010. Web. 09 Apr 2020.

Vancouver:

Shannon L. The Role of the G-Protein Coupled Receptor C-C Chemokine Receptor 7 in T Lymphocyte Migration and Breast Cancer Metastasis. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/1808/7423.

Council of Science Editors:

Shannon L. The Role of the G-Protein Coupled Receptor C-C Chemokine Receptor 7 in T Lymphocyte Migration and Breast Cancer Metastasis. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/7423


Clemson University

9. Agrawal, Vibhor. Understanding Allosteric Modulation of G-Protein Coupled Receptors.

Degree: PhD, Chemistry, 2016, Clemson University

G protein-coupled receptors (GPCRs) which are seven-transmembrane allosteric machine constitutes largest and diverse family of membrane proteins. GPCR participate in activating a diverse range of… (more)

Subjects/Keywords: Allostery; G Protein Coupled Receptor; Ligand biased signalling; Orthosteric ligands

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APA (6th Edition):

Agrawal, V. (2016). Understanding Allosteric Modulation of G-Protein Coupled Receptors. (Doctoral Dissertation). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_dissertations/1630

Chicago Manual of Style (16th Edition):

Agrawal, Vibhor. “Understanding Allosteric Modulation of G-Protein Coupled Receptors.” 2016. Doctoral Dissertation, Clemson University. Accessed April 09, 2020. https://tigerprints.clemson.edu/all_dissertations/1630.

MLA Handbook (7th Edition):

Agrawal, Vibhor. “Understanding Allosteric Modulation of G-Protein Coupled Receptors.” 2016. Web. 09 Apr 2020.

Vancouver:

Agrawal V. Understanding Allosteric Modulation of G-Protein Coupled Receptors. [Internet] [Doctoral dissertation]. Clemson University; 2016. [cited 2020 Apr 09]. Available from: https://tigerprints.clemson.edu/all_dissertations/1630.

Council of Science Editors:

Agrawal V. Understanding Allosteric Modulation of G-Protein Coupled Receptors. [Doctoral Dissertation]. Clemson University; 2016. Available from: https://tigerprints.clemson.edu/all_dissertations/1630


Queen Mary, University of London

10. Sisay, Sofia. GPR55 as a novel target in disease control of multiple sclerosis.

Degree: PhD, 2013, Queen Mary, University of London

 Multiple sclerosis (MS) is a neurodegenerative disease associated with immune attack of the central nervous system (CNS) leading to neuronal and axonal loss. This affects… (more)

Subjects/Keywords: 616.8; Multiple Sclerosis; cannabinoids; autoimmune response; G-protein coupled receptor 55

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APA (6th Edition):

Sisay, S. (2013). GPR55 as a novel target in disease control of multiple sclerosis. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/27210 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.612579

Chicago Manual of Style (16th Edition):

Sisay, Sofia. “GPR55 as a novel target in disease control of multiple sclerosis.” 2013. Doctoral Dissertation, Queen Mary, University of London. Accessed April 09, 2020. http://qmro.qmul.ac.uk/xmlui/handle/123456789/27210 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.612579.

MLA Handbook (7th Edition):

Sisay, Sofia. “GPR55 as a novel target in disease control of multiple sclerosis.” 2013. Web. 09 Apr 2020.

Vancouver:

Sisay S. GPR55 as a novel target in disease control of multiple sclerosis. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2013. [cited 2020 Apr 09]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/27210 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.612579.

Council of Science Editors:

Sisay S. GPR55 as a novel target in disease control of multiple sclerosis. [Doctoral Dissertation]. Queen Mary, University of London; 2013. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/27210 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.612579

11. Bongers, G.M. Signal transduction of the histamine H3 receptor.

Degree: Faculty of Earth and Life Sciences, 2008, NARCIS

Subjects/Keywords: G-protein coupled receptor; Histamine

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APA (6th Edition):

Bongers, G. M. (2008). Signal transduction of the histamine H3 receptor. (Doctoral Dissertation). NARCIS. Retrieved from https://research.vu.nl/en/publications/1384ea18-ab69-403e-a1f7-5f6827762964 ; urn:nbn:nl:ui:31-1871/15988 ; 1384ea18-ab69-403e-a1f7-5f6827762964 ; 1871/15988 ; urn:isbn:9789090237459 ; urn:nbn:nl:ui:31-1871/15988 ; https://research.vu.nl/en/publications/1384ea18-ab69-403e-a1f7-5f6827762964

Chicago Manual of Style (16th Edition):

Bongers, G M. “Signal transduction of the histamine H3 receptor.” 2008. Doctoral Dissertation, NARCIS. Accessed April 09, 2020. https://research.vu.nl/en/publications/1384ea18-ab69-403e-a1f7-5f6827762964 ; urn:nbn:nl:ui:31-1871/15988 ; 1384ea18-ab69-403e-a1f7-5f6827762964 ; 1871/15988 ; urn:isbn:9789090237459 ; urn:nbn:nl:ui:31-1871/15988 ; https://research.vu.nl/en/publications/1384ea18-ab69-403e-a1f7-5f6827762964.

MLA Handbook (7th Edition):

Bongers, G M. “Signal transduction of the histamine H3 receptor.” 2008. Web. 09 Apr 2020.

Vancouver:

Bongers GM. Signal transduction of the histamine H3 receptor. [Internet] [Doctoral dissertation]. NARCIS; 2008. [cited 2020 Apr 09]. Available from: https://research.vu.nl/en/publications/1384ea18-ab69-403e-a1f7-5f6827762964 ; urn:nbn:nl:ui:31-1871/15988 ; 1384ea18-ab69-403e-a1f7-5f6827762964 ; 1871/15988 ; urn:isbn:9789090237459 ; urn:nbn:nl:ui:31-1871/15988 ; https://research.vu.nl/en/publications/1384ea18-ab69-403e-a1f7-5f6827762964.

Council of Science Editors:

Bongers GM. Signal transduction of the histamine H3 receptor. [Doctoral Dissertation]. NARCIS; 2008. Available from: https://research.vu.nl/en/publications/1384ea18-ab69-403e-a1f7-5f6827762964 ; urn:nbn:nl:ui:31-1871/15988 ; 1384ea18-ab69-403e-a1f7-5f6827762964 ; 1871/15988 ; urn:isbn:9789090237459 ; urn:nbn:nl:ui:31-1871/15988 ; https://research.vu.nl/en/publications/1384ea18-ab69-403e-a1f7-5f6827762964


Vanderbilt University

12. Chen, Qiuyan. The structure basis of arrestin mediated GPCR signaling.

Degree: PhD, Pharmacology, 2015, Vanderbilt University

 Arrestin selectively binds the phosphorylated active receptor to either terminate the G protein dependent signaling or initiate G protein independent signaling. Receptor binding induces global… (more)

Subjects/Keywords: signaling; structural basis; arrestin; G protein coupled receptor

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APA (6th Edition):

Chen, Q. (2015). The structure basis of arrestin mediated GPCR signaling. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-11162015-201134/ ;

Chicago Manual of Style (16th Edition):

Chen, Qiuyan. “The structure basis of arrestin mediated GPCR signaling.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed April 09, 2020. http://etd.library.vanderbilt.edu//available/etd-11162015-201134/ ;.

MLA Handbook (7th Edition):

Chen, Qiuyan. “The structure basis of arrestin mediated GPCR signaling.” 2015. Web. 09 Apr 2020.

Vancouver:

Chen Q. The structure basis of arrestin mediated GPCR signaling. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Apr 09]. Available from: http://etd.library.vanderbilt.edu//available/etd-11162015-201134/ ;.

Council of Science Editors:

Chen Q. The structure basis of arrestin mediated GPCR signaling. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu//available/etd-11162015-201134/ ;


University of Iowa

13. Gupte, Raeesa Prashant. Phosphoregulation of somatodendritic voltage-gated potassium channels by pituitary adenylate cyclase-activating polypeptide.

Degree: PhD, Pharmacology, 2015, University of Iowa

  The endogenous neuropeptide pituitary adenylate cyclase-activating polypeptide (PACAP) exerts various neuromodulatory functions in mammalian brain. Enhancement of synaptic activity, mediation of chronic inflammatory and… (more)

Subjects/Keywords: G-protein coupled receptor; Hyperexcitability; Ion channels; Kinase; Neuroprotection; Phosphorylation; Pharmacology

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APA (6th Edition):

Gupte, R. P. (2015). Phosphoregulation of somatodendritic voltage-gated potassium channels by pituitary adenylate cyclase-activating polypeptide. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/5766

Chicago Manual of Style (16th Edition):

Gupte, Raeesa Prashant. “Phosphoregulation of somatodendritic voltage-gated potassium channels by pituitary adenylate cyclase-activating polypeptide.” 2015. Doctoral Dissertation, University of Iowa. Accessed April 09, 2020. https://ir.uiowa.edu/etd/5766.

MLA Handbook (7th Edition):

Gupte, Raeesa Prashant. “Phosphoregulation of somatodendritic voltage-gated potassium channels by pituitary adenylate cyclase-activating polypeptide.” 2015. Web. 09 Apr 2020.

Vancouver:

Gupte RP. Phosphoregulation of somatodendritic voltage-gated potassium channels by pituitary adenylate cyclase-activating polypeptide. [Internet] [Doctoral dissertation]. University of Iowa; 2015. [cited 2020 Apr 09]. Available from: https://ir.uiowa.edu/etd/5766.

Council of Science Editors:

Gupte RP. Phosphoregulation of somatodendritic voltage-gated potassium channels by pituitary adenylate cyclase-activating polypeptide. [Doctoral Dissertation]. University of Iowa; 2015. Available from: https://ir.uiowa.edu/etd/5766


University of Melbourne

14. Shilling, Patrick James. Characterisation of cell-free synthesised GPCRs.

Degree: 2014, University of Melbourne

 The relaxin family peptide receptors (RXFP) belong to the G protein-coupled receptor family (GPCR). This family is comprised of four receptors named RXFP1-4. Each of… (more)

Subjects/Keywords: cell-free; cell-free protein synthesis; G-protein coupled receptor; RXFP; NTS1; membrane protein

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APA (6th Edition):

Shilling, P. J. (2014). Characterisation of cell-free synthesised GPCRs. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/43163

Chicago Manual of Style (16th Edition):

Shilling, Patrick James. “Characterisation of cell-free synthesised GPCRs.” 2014. Doctoral Dissertation, University of Melbourne. Accessed April 09, 2020. http://hdl.handle.net/11343/43163.

MLA Handbook (7th Edition):

Shilling, Patrick James. “Characterisation of cell-free synthesised GPCRs.” 2014. Web. 09 Apr 2020.

Vancouver:

Shilling PJ. Characterisation of cell-free synthesised GPCRs. [Internet] [Doctoral dissertation]. University of Melbourne; 2014. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/11343/43163.

Council of Science Editors:

Shilling PJ. Characterisation of cell-free synthesised GPCRs. [Doctoral Dissertation]. University of Melbourne; 2014. Available from: http://hdl.handle.net/11343/43163


University of Western Ontario

15. Tutunea-Fatan, Elena. Signal Transduction Mechanisms Mediating the Regulation of Vascular G Protein-Coupled Receptors.

Degree: 2016, University of Western Ontario

 Blood pressure homeostasis is controlled via a complex network of cell signaling mechanisms. Among the broad network of receptors and signaling molecules regulating blood vessel… (more)

Subjects/Keywords: G protein-coupled receptors; G protein-coupled receptor kinases; Rab4GTPase; signal transduction; vascular smooth muscle cells; hypertension; Cardiovascular Diseases

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APA (6th Edition):

Tutunea-Fatan, E. (2016). Signal Transduction Mechanisms Mediating the Regulation of Vascular G Protein-Coupled Receptors. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4329

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tutunea-Fatan, Elena. “Signal Transduction Mechanisms Mediating the Regulation of Vascular G Protein-Coupled Receptors.” 2016. Thesis, University of Western Ontario. Accessed April 09, 2020. https://ir.lib.uwo.ca/etd/4329.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tutunea-Fatan, Elena. “Signal Transduction Mechanisms Mediating the Regulation of Vascular G Protein-Coupled Receptors.” 2016. Web. 09 Apr 2020.

Vancouver:

Tutunea-Fatan E. Signal Transduction Mechanisms Mediating the Regulation of Vascular G Protein-Coupled Receptors. [Internet] [Thesis]. University of Western Ontario; 2016. [cited 2020 Apr 09]. Available from: https://ir.lib.uwo.ca/etd/4329.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tutunea-Fatan E. Signal Transduction Mechanisms Mediating the Regulation of Vascular G Protein-Coupled Receptors. [Thesis]. University of Western Ontario; 2016. Available from: https://ir.lib.uwo.ca/etd/4329

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Da Silva, Mélanie. Développement d’essais HTRF® innovants pour détecter l'activation des protéines G natives par leurs récepteurs : Development of HTRF® assays to study G proteins.

Degree: Docteur es, Biologie Santé, 2017, Montpellier

Les récepteurs couplés aux protéines G (RCPG) représentent la plus grande famille de protéines membranaires, et ils sont la cible de plus de 25% des… (more)

Subjects/Keywords: Récepteur-Couplé aux protéines G; Fret; Médicaments; G protein-Coupled receptor; Fret; Drugs

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APA (6th Edition):

Da Silva, M. (2017). Développement d’essais HTRF® innovants pour détecter l'activation des protéines G natives par leurs récepteurs : Development of HTRF® assays to study G proteins. (Doctoral Dissertation). Montpellier. Retrieved from http://www.theses.fr/2017MONTT033

Chicago Manual of Style (16th Edition):

Da Silva, Mélanie. “Développement d’essais HTRF® innovants pour détecter l'activation des protéines G natives par leurs récepteurs : Development of HTRF® assays to study G proteins.” 2017. Doctoral Dissertation, Montpellier. Accessed April 09, 2020. http://www.theses.fr/2017MONTT033.

MLA Handbook (7th Edition):

Da Silva, Mélanie. “Développement d’essais HTRF® innovants pour détecter l'activation des protéines G natives par leurs récepteurs : Development of HTRF® assays to study G proteins.” 2017. Web. 09 Apr 2020.

Vancouver:

Da Silva M. Développement d’essais HTRF® innovants pour détecter l'activation des protéines G natives par leurs récepteurs : Development of HTRF® assays to study G proteins. [Internet] [Doctoral dissertation]. Montpellier; 2017. [cited 2020 Apr 09]. Available from: http://www.theses.fr/2017MONTT033.

Council of Science Editors:

Da Silva M. Développement d’essais HTRF® innovants pour détecter l'activation des protéines G natives par leurs récepteurs : Development of HTRF® assays to study G proteins. [Doctoral Dissertation]. Montpellier; 2017. Available from: http://www.theses.fr/2017MONTT033


The Ohio State University

17. Schmid, Cullen L. Differential regulation of serotonin 2A receptor responsiveness by agonist-directed interactions with beta-arrestin2.

Degree: PhD, Neuroscience Graduate Studies Program, 2011, The Ohio State University

 The G protein-coupled, serotonin 2A (5-HT2A) receptor is a major drug target for the treatment of a number of mental health disorders, including schizophrenia, anxiety… (more)

Subjects/Keywords: Neurosciences; Pharmacology; G protein-coupled receptor; Serotonin 2A receptor; beta-arrestin2; hallucinogens; functional selectivity

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APA (6th Edition):

Schmid, C. L. (2011). Differential regulation of serotonin 2A receptor responsiveness by agonist-directed interactions with beta-arrestin2. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1300287547

Chicago Manual of Style (16th Edition):

Schmid, Cullen L. “Differential regulation of serotonin 2A receptor responsiveness by agonist-directed interactions with beta-arrestin2.” 2011. Doctoral Dissertation, The Ohio State University. Accessed April 09, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1300287547.

MLA Handbook (7th Edition):

Schmid, Cullen L. “Differential regulation of serotonin 2A receptor responsiveness by agonist-directed interactions with beta-arrestin2.” 2011. Web. 09 Apr 2020.

Vancouver:

Schmid CL. Differential regulation of serotonin 2A receptor responsiveness by agonist-directed interactions with beta-arrestin2. [Internet] [Doctoral dissertation]. The Ohio State University; 2011. [cited 2020 Apr 09]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1300287547.

Council of Science Editors:

Schmid CL. Differential regulation of serotonin 2A receptor responsiveness by agonist-directed interactions with beta-arrestin2. [Doctoral Dissertation]. The Ohio State University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1300287547


University of Victoria

18. Churcher, Allison Mary. Evolutionary genomics of odorant receptors: identification and characterization of orthologs in an echinoderm, a cephalochordate and a cnidarian.

Degree: Dept. of Biology, 2011, University of Victoria

 Animal chemosensation involves several families of G protein-coupled receptors (GPCRs) and, though some of these families are well characterized in vertebrates and nematode worms, receptors… (more)

Subjects/Keywords: odorant receptor; G protein-coupled receptor; evolution; animal chemosensory systems; echinoderm; cephalochordate; evolution; cnidarian

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APA (6th Edition):

Churcher, A. M. (2011). Evolutionary genomics of odorant receptors: identification and characterization of orthologs in an echinoderm, a cephalochordate and a cnidarian. (Thesis). University of Victoria. Retrieved from http://hdl.handle.net/1828/3470

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Churcher, Allison Mary. “Evolutionary genomics of odorant receptors: identification and characterization of orthologs in an echinoderm, a cephalochordate and a cnidarian.” 2011. Thesis, University of Victoria. Accessed April 09, 2020. http://hdl.handle.net/1828/3470.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Churcher, Allison Mary. “Evolutionary genomics of odorant receptors: identification and characterization of orthologs in an echinoderm, a cephalochordate and a cnidarian.” 2011. Web. 09 Apr 2020.

Vancouver:

Churcher AM. Evolutionary genomics of odorant receptors: identification and characterization of orthologs in an echinoderm, a cephalochordate and a cnidarian. [Internet] [Thesis]. University of Victoria; 2011. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/1828/3470.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Churcher AM. Evolutionary genomics of odorant receptors: identification and characterization of orthologs in an echinoderm, a cephalochordate and a cnidarian. [Thesis]. University of Victoria; 2011. Available from: http://hdl.handle.net/1828/3470

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

19. Varandas, Katherine Varandas. Retromer endosome exit domains serve multiple trafficking destinations and regulate local G protein activation by GPCRs.

Degree: Cell Biology, 2016, University of California – San Francisco

 This thesis asks (1) whether the divergent trafficking functions of the endosome-associating coat complex retromer require cargos to exit endosomes separately and (2) if distinct… (more)

Subjects/Keywords: Cellular biology; beta-2 adrenergic receptor; G protein coupled receptor; recycling; retrograde transport; Retromer; Wntless

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APA (6th Edition):

Varandas, K. V. (2016). Retromer endosome exit domains serve multiple trafficking destinations and regulate local G protein activation by GPCRs. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/5pn2z1r2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Varandas, Katherine Varandas. “Retromer endosome exit domains serve multiple trafficking destinations and regulate local G protein activation by GPCRs.” 2016. Thesis, University of California – San Francisco. Accessed April 09, 2020. http://www.escholarship.org/uc/item/5pn2z1r2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Varandas, Katherine Varandas. “Retromer endosome exit domains serve multiple trafficking destinations and regulate local G protein activation by GPCRs.” 2016. Web. 09 Apr 2020.

Vancouver:

Varandas KV. Retromer endosome exit domains serve multiple trafficking destinations and regulate local G protein activation by GPCRs. [Internet] [Thesis]. University of California – San Francisco; 2016. [cited 2020 Apr 09]. Available from: http://www.escholarship.org/uc/item/5pn2z1r2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Varandas KV. Retromer endosome exit domains serve multiple trafficking destinations and regulate local G protein activation by GPCRs. [Thesis]. University of California – San Francisco; 2016. Available from: http://www.escholarship.org/uc/item/5pn2z1r2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

20. Zhang, Chen. Integration Of Extracellular And Intracellular Signals Via The Calcium Sensing Receptor (CASR).

Degree: PhD, Chemistry, 2014, Georgia State University

  The Ca2+-sensing receptor (CaSR) regulates the calcium homeostasis in the human body via sensing fluctuations in the extracellular Ca2+ concentration. Naturally occurring mutations in… (more)

Subjects/Keywords: Calcium sensing receptor; Calmodulin; Prediction; Purification; Fluorescence; Ca2+ Signaling; G-protein coupled receptor (GPCR)

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APA (6th Edition):

Zhang, C. (2014). Integration Of Extracellular And Intracellular Signals Via The Calcium Sensing Receptor (CASR). (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/chemistry_diss/91

Chicago Manual of Style (16th Edition):

Zhang, Chen. “Integration Of Extracellular And Intracellular Signals Via The Calcium Sensing Receptor (CASR).” 2014. Doctoral Dissertation, Georgia State University. Accessed April 09, 2020. https://scholarworks.gsu.edu/chemistry_diss/91.

MLA Handbook (7th Edition):

Zhang, Chen. “Integration Of Extracellular And Intracellular Signals Via The Calcium Sensing Receptor (CASR).” 2014. Web. 09 Apr 2020.

Vancouver:

Zhang C. Integration Of Extracellular And Intracellular Signals Via The Calcium Sensing Receptor (CASR). [Internet] [Doctoral dissertation]. Georgia State University; 2014. [cited 2020 Apr 09]. Available from: https://scholarworks.gsu.edu/chemistry_diss/91.

Council of Science Editors:

Zhang C. Integration Of Extracellular And Intracellular Signals Via The Calcium Sensing Receptor (CASR). [Doctoral Dissertation]. Georgia State University; 2014. Available from: https://scholarworks.gsu.edu/chemistry_diss/91


University of Toronto

21. Brisset Di Roberto, RaphaĂŤl. Evolutionary Alternatives on the Road to a New Specificity in a G Protein-Coupled Receptor.

Degree: PhD, 2017, University of Toronto

 Cells sense change in their external environment and react appropriately through the action of signaling pathways. This process is initiated by receptor proteins with high… (more)

Subjects/Keywords: directed evolution; g protein-coupled receptor; ligand efficacy; network evolution; receptor specificity; 0379

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APA (6th Edition):

Brisset Di Roberto, R. (2017). Evolutionary Alternatives on the Road to a New Specificity in a G Protein-Coupled Receptor. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/80849

Chicago Manual of Style (16th Edition):

Brisset Di Roberto, RaphaĂŤl. “Evolutionary Alternatives on the Road to a New Specificity in a G Protein-Coupled Receptor.” 2017. Doctoral Dissertation, University of Toronto. Accessed April 09, 2020. http://hdl.handle.net/1807/80849.

MLA Handbook (7th Edition):

Brisset Di Roberto, RaphaĂŤl. “Evolutionary Alternatives on the Road to a New Specificity in a G Protein-Coupled Receptor.” 2017. Web. 09 Apr 2020.

Vancouver:

Brisset Di Roberto R. Evolutionary Alternatives on the Road to a New Specificity in a G Protein-Coupled Receptor. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/1807/80849.

Council of Science Editors:

Brisset Di Roberto R. Evolutionary Alternatives on the Road to a New Specificity in a G Protein-Coupled Receptor. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/80849


University of California – San Diego

22. Berkamp, Sabrina. Interaction Studies of the Chemokine Interleukin-8 with the G Protein-Coupled Receptor CXCR1 by Nuclear Magnetic Resonance Spectroscopy.

Degree: Chemistry, 2018, University of California – San Diego

 The chemokine Interleukin-8 is a major mediator in inflammation. It interacts with the G Protein-Coupled Receptor CXCR1, which is expressed on neutrophils and other cells.… (more)

Subjects/Keywords: Biophysics; Biochemistry; Molecular biology; chemokine; CXCR1; G Protein-Coupled Receptor; Interleukin-8; NMR; protein structure

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APA (6th Edition):

Berkamp, S. (2018). Interaction Studies of the Chemokine Interleukin-8 with the G Protein-Coupled Receptor CXCR1 by Nuclear Magnetic Resonance Spectroscopy. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/9xh3c406

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Berkamp, Sabrina. “Interaction Studies of the Chemokine Interleukin-8 with the G Protein-Coupled Receptor CXCR1 by Nuclear Magnetic Resonance Spectroscopy.” 2018. Thesis, University of California – San Diego. Accessed April 09, 2020. http://www.escholarship.org/uc/item/9xh3c406.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Berkamp, Sabrina. “Interaction Studies of the Chemokine Interleukin-8 with the G Protein-Coupled Receptor CXCR1 by Nuclear Magnetic Resonance Spectroscopy.” 2018. Web. 09 Apr 2020.

Vancouver:

Berkamp S. Interaction Studies of the Chemokine Interleukin-8 with the G Protein-Coupled Receptor CXCR1 by Nuclear Magnetic Resonance Spectroscopy. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2020 Apr 09]. Available from: http://www.escholarship.org/uc/item/9xh3c406.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Berkamp S. Interaction Studies of the Chemokine Interleukin-8 with the G Protein-Coupled Receptor CXCR1 by Nuclear Magnetic Resonance Spectroscopy. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/9xh3c406

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Johannes Gutenberg Universität Mainz

23. Metz, Verena Vanessa. Untersuchungen zum Ectodomain shedding des Receptor for advanced glycation endproducts.

Degree: 2011, Johannes Gutenberg Universität Mainz

Zu den Liganden des Zelloberflächenrezeptors RAGE gehören AGEs, S100-Proteine, HMGB1 und Aβ. RAGE wird daher eine Rolle bei verschiedenen neurologischen Erkrankungen sowie Diabetes, Arteriosklerose und… (more)

Subjects/Keywords: Ectodomain Shedding; G-Protein gekoppelter Rezeptor; Alzheimer Demenz; Ectodomain shedding; G-protein coupled receptor; Alzheimer´s disease; Life sciences

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APA (6th Edition):

Metz, V. V. (2011). Untersuchungen zum Ectodomain shedding des Receptor for advanced glycation endproducts. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2012/3162/

Chicago Manual of Style (16th Edition):

Metz, Verena Vanessa. “Untersuchungen zum Ectodomain shedding des Receptor for advanced glycation endproducts.” 2011. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed April 09, 2020. http://ubm.opus.hbz-nrw.de/volltexte/2012/3162/.

MLA Handbook (7th Edition):

Metz, Verena Vanessa. “Untersuchungen zum Ectodomain shedding des Receptor for advanced glycation endproducts.” 2011. Web. 09 Apr 2020.

Vancouver:

Metz VV. Untersuchungen zum Ectodomain shedding des Receptor for advanced glycation endproducts. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2011. [cited 2020 Apr 09]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2012/3162/.

Council of Science Editors:

Metz VV. Untersuchungen zum Ectodomain shedding des Receptor for advanced glycation endproducts. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2011. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2012/3162/

24. Gao, Yang. STRUCTURAL BASIS OF VERTEBRATE VISION, A G PROTEIN-COUPLED RECEPTOR SIGNALING CASCADE .

Degree: 2018, Cornell University

 The visual photo-transduction cascade is a prototypical G protein-coupled receptor (GPCR) signaling system, in which light-activated rhodopsin, the GPCR, catalyzes the exchange of GDP for… (more)

Subjects/Keywords: Transducin; Visual phototransduction; Biophysics; Biochemistry; Chemistry; Cryo-EM; GPCR-G protein complex; G protein-coupled receptor; Rhodopsin

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APA (6th Edition):

Gao, Y. (2018). STRUCTURAL BASIS OF VERTEBRATE VISION, A G PROTEIN-COUPLED RECEPTOR SIGNALING CASCADE . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/59736

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gao, Yang. “STRUCTURAL BASIS OF VERTEBRATE VISION, A G PROTEIN-COUPLED RECEPTOR SIGNALING CASCADE .” 2018. Thesis, Cornell University. Accessed April 09, 2020. http://hdl.handle.net/1813/59736.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gao, Yang. “STRUCTURAL BASIS OF VERTEBRATE VISION, A G PROTEIN-COUPLED RECEPTOR SIGNALING CASCADE .” 2018. Web. 09 Apr 2020.

Vancouver:

Gao Y. STRUCTURAL BASIS OF VERTEBRATE VISION, A G PROTEIN-COUPLED RECEPTOR SIGNALING CASCADE . [Internet] [Thesis]. Cornell University; 2018. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/1813/59736.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gao Y. STRUCTURAL BASIS OF VERTEBRATE VISION, A G PROTEIN-COUPLED RECEPTOR SIGNALING CASCADE . [Thesis]. Cornell University; 2018. Available from: http://hdl.handle.net/1813/59736

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

25. Esseltine, Jessica L. Modulation of G Protein-Coupled Receptor Intracellular Trafficking and Signal Transduction.

Degree: 2012, University of Western Ontario

 The seven transmembrane-spanning G Protein-coupled Receptor (GPCR) super family is the largest family of cell-surface receptors, comprising greater than 650 members. GPCRs represent the primary… (more)

Subjects/Keywords: G protein-coupled receptor; Rab GTPase; Receptor trafficking; Signal Transduction; Desensitization; Receptor Carboxyl-Terminal tail domains; Cell Biology

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APA (6th Edition):

Esseltine, J. L. (2012). Modulation of G Protein-Coupled Receptor Intracellular Trafficking and Signal Transduction. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Esseltine, Jessica L. “Modulation of G Protein-Coupled Receptor Intracellular Trafficking and Signal Transduction.” 2012. Thesis, University of Western Ontario. Accessed April 09, 2020. https://ir.lib.uwo.ca/etd/473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Esseltine, Jessica L. “Modulation of G Protein-Coupled Receptor Intracellular Trafficking and Signal Transduction.” 2012. Web. 09 Apr 2020.

Vancouver:

Esseltine JL. Modulation of G Protein-Coupled Receptor Intracellular Trafficking and Signal Transduction. [Internet] [Thesis]. University of Western Ontario; 2012. [cited 2020 Apr 09]. Available from: https://ir.lib.uwo.ca/etd/473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Esseltine JL. Modulation of G Protein-Coupled Receptor Intracellular Trafficking and Signal Transduction. [Thesis]. University of Western Ontario; 2012. Available from: https://ir.lib.uwo.ca/etd/473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

26. Frei, Jan Niclas. Characterisation of the conformational landscape of the β1-adrenergic receptor and development of a strategy towards NMR studies of the protease-activated receptor 1.

Degree: PhD, 2020, University of Cambridge

 The research conducted for this thesis was aimed at providing a comprehensive description of the dynamic and conformational landscape of the minimally thermostabilised avian β1-adrenergic… (more)

Subjects/Keywords: G protein-coupled receptor; Nuclear magnetic resonance spectroscopy; β1-adrenergic receptor; Protease-activated receptor 1; Second harmonic generation

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APA (6th Edition):

Frei, J. N. (2020). Characterisation of the conformational landscape of the β1-adrenergic receptor and development of a strategy towards NMR studies of the protease-activated receptor 1. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/303160

Chicago Manual of Style (16th Edition):

Frei, Jan Niclas. “Characterisation of the conformational landscape of the β1-adrenergic receptor and development of a strategy towards NMR studies of the protease-activated receptor 1.” 2020. Doctoral Dissertation, University of Cambridge. Accessed April 09, 2020. https://www.repository.cam.ac.uk/handle/1810/303160.

MLA Handbook (7th Edition):

Frei, Jan Niclas. “Characterisation of the conformational landscape of the β1-adrenergic receptor and development of a strategy towards NMR studies of the protease-activated receptor 1.” 2020. Web. 09 Apr 2020.

Vancouver:

Frei JN. Characterisation of the conformational landscape of the β1-adrenergic receptor and development of a strategy towards NMR studies of the protease-activated receptor 1. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2020 Apr 09]. Available from: https://www.repository.cam.ac.uk/handle/1810/303160.

Council of Science Editors:

Frei JN. Characterisation of the conformational landscape of the β1-adrenergic receptor and development of a strategy towards NMR studies of the protease-activated receptor 1. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/303160


University of Arizona

27. Navratilova, Edita. Regulation of the human delta opioid receptor .

Degree: 2007, University of Arizona

 Regulation of the human delta opioid receptor (hDOR) is implicated in the development of tolerance to chronic morphine (Zhu et al., 1999). In addition, DORs… (more)

Subjects/Keywords: opioid receptor; G protein-coupled receptor; receptor regulation

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APA (6th Edition):

Navratilova, E. (2007). Regulation of the human delta opioid receptor . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194173

Chicago Manual of Style (16th Edition):

Navratilova, Edita. “Regulation of the human delta opioid receptor .” 2007. Doctoral Dissertation, University of Arizona. Accessed April 09, 2020. http://hdl.handle.net/10150/194173.

MLA Handbook (7th Edition):

Navratilova, Edita. “Regulation of the human delta opioid receptor .” 2007. Web. 09 Apr 2020.

Vancouver:

Navratilova E. Regulation of the human delta opioid receptor . [Internet] [Doctoral dissertation]. University of Arizona; 2007. [cited 2020 Apr 09]. Available from: http://hdl.handle.net/10150/194173.

Council of Science Editors:

Navratilova E. Regulation of the human delta opioid receptor . [Doctoral Dissertation]. University of Arizona; 2007. Available from: http://hdl.handle.net/10150/194173


University of Guelph

28. Ervin, Kelsy. The role of specific estrogen receptors in mediating rapid estrogenic improvements of social learning in female mice .

Degree: 2014, University of Guelph

 Social learning is a process by which an animal gains information from another; however much of the research on estrogens effects on learning focuses on… (more)

Subjects/Keywords: estradiol; social transmission of food preference; estrogen receptor alpha; estrogen receptor beta; G protein-coupled estrogen receptor; GPER; GPR30

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APA (6th Edition):

Ervin, K. (2014). The role of specific estrogen receptors in mediating rapid estrogenic improvements of social learning in female mice . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7818

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ervin, Kelsy. “The role of specific estrogen receptors in mediating rapid estrogenic improvements of social learning in female mice .” 2014. Thesis, University of Guelph. Accessed April 09, 2020. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7818.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ervin, Kelsy. “The role of specific estrogen receptors in mediating rapid estrogenic improvements of social learning in female mice .” 2014. Web. 09 Apr 2020.

Vancouver:

Ervin K. The role of specific estrogen receptors in mediating rapid estrogenic improvements of social learning in female mice . [Internet] [Thesis]. University of Guelph; 2014. [cited 2020 Apr 09]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7818.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ervin K. The role of specific estrogen receptors in mediating rapid estrogenic improvements of social learning in female mice . [Thesis]. University of Guelph; 2014. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7818

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

29. Raehal, Kirsten Michele. Opioid-Induced Side Effects in Beta-arrestin2 adn G Protein-Coupled Receptor Kinase Knockout Mice.

Degree: PhD, Integrated Biomedical Sciences, 2009, The Ohio State University

 Opioid drugs are potent analgesics; however, they also produce several adverse side effects including constipation, antinociceptive tolerance, and physical dependence by activating the mu opioid… (more)

Subjects/Keywords: Pharmacology; opioid; arrestin; G protein-coupled receptor kinase; gastrointestinal; physical dependence; tolerance

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Raehal, K. M. (2009). Opioid-Induced Side Effects in Beta-arrestin2 adn G Protein-Coupled Receptor Kinase Knockout Mice. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1236884585

Chicago Manual of Style (16th Edition):

Raehal, Kirsten Michele. “Opioid-Induced Side Effects in Beta-arrestin2 adn G Protein-Coupled Receptor Kinase Knockout Mice.” 2009. Doctoral Dissertation, The Ohio State University. Accessed April 09, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1236884585.

MLA Handbook (7th Edition):

Raehal, Kirsten Michele. “Opioid-Induced Side Effects in Beta-arrestin2 adn G Protein-Coupled Receptor Kinase Knockout Mice.” 2009. Web. 09 Apr 2020.

Vancouver:

Raehal KM. Opioid-Induced Side Effects in Beta-arrestin2 adn G Protein-Coupled Receptor Kinase Knockout Mice. [Internet] [Doctoral dissertation]. The Ohio State University; 2009. [cited 2020 Apr 09]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1236884585.

Council of Science Editors:

Raehal KM. Opioid-Induced Side Effects in Beta-arrestin2 adn G Protein-Coupled Receptor Kinase Knockout Mice. [Doctoral Dissertation]. The Ohio State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1236884585


University of Cincinnati

30. Wu, Shu-en. Molecular Studies of Host-pathogen Interactions in Human Cytomegalovirus-infected Myeloid Cells.

Degree: PhD, Medicine: Molecular Genetics, Biochemistry, & Microbiology, 2015, University of Cincinnati

 Human cytomegalovirus (HCMV) is a member of the subfamily of Beta-herpesviridae that establishes latency in human hematopoietic progenitor cells, myeloid progenitor cells and monocytes. While… (more)

Subjects/Keywords: Virology; human cytomegalovirus; vitamin D; US28; myeloid cells; YM-254890; G protein-coupled receptor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wu, S. (2015). Molecular Studies of Host-pathogen Interactions in Human Cytomegalovirus-infected Myeloid Cells. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1439308683

Chicago Manual of Style (16th Edition):

Wu, Shu-en. “Molecular Studies of Host-pathogen Interactions in Human Cytomegalovirus-infected Myeloid Cells.” 2015. Doctoral Dissertation, University of Cincinnati. Accessed April 09, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1439308683.

MLA Handbook (7th Edition):

Wu, Shu-en. “Molecular Studies of Host-pathogen Interactions in Human Cytomegalovirus-infected Myeloid Cells.” 2015. Web. 09 Apr 2020.

Vancouver:

Wu S. Molecular Studies of Host-pathogen Interactions in Human Cytomegalovirus-infected Myeloid Cells. [Internet] [Doctoral dissertation]. University of Cincinnati; 2015. [cited 2020 Apr 09]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1439308683.

Council of Science Editors:

Wu S. Molecular Studies of Host-pathogen Interactions in Human Cytomegalovirus-infected Myeloid Cells. [Doctoral Dissertation]. University of Cincinnati; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1439308683

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