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You searched for subject:(G Protein Coupled Inwardly Rectifying Potassium Channels). Showing records 1 – 30 of 26287 total matches.

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1. Tchenio, Anna. Reversal strategies within the lateral habenula to ameliorate depressive-like behaviors : Stratégies ciblant l’habénula latérale pour améliorer les symptômes de types dépressifs.

Degree: Docteur es, Neurosciences, 2017, Université Pierre et Marie Curie – Paris VI

 L’agression de l'organisme par un agent physique, psychique ou émotionnel déclenche une réponse physiologique et comportementale qui permet à un individu de se prévenir du… (more)

Subjects/Keywords: Habénula latérale; Dépression; Séparation maternelle; Récepteurs métabotropiques GABABR; G Protein-Coupled Inwardly-Rectifying Potassium Channels; Électrophysiologie; Lateral hebenula; Depression; Maternal separation; 573.8

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tchenio, A. (2017). Reversal strategies within the lateral habenula to ameliorate depressive-like behaviors : Stratégies ciblant l’habénula latérale pour améliorer les symptômes de types dépressifs. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2017PA066301

Chicago Manual of Style (16th Edition):

Tchenio, Anna. “Reversal strategies within the lateral habenula to ameliorate depressive-like behaviors : Stratégies ciblant l’habénula latérale pour améliorer les symptômes de types dépressifs.” 2017. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed June 06, 2020. http://www.theses.fr/2017PA066301.

MLA Handbook (7th Edition):

Tchenio, Anna. “Reversal strategies within the lateral habenula to ameliorate depressive-like behaviors : Stratégies ciblant l’habénula latérale pour améliorer les symptômes de types dépressifs.” 2017. Web. 06 Jun 2020.

Vancouver:

Tchenio A. Reversal strategies within the lateral habenula to ameliorate depressive-like behaviors : Stratégies ciblant l’habénula latérale pour améliorer les symptômes de types dépressifs. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. [cited 2020 Jun 06]. Available from: http://www.theses.fr/2017PA066301.

Council of Science Editors:

Tchenio A. Reversal strategies within the lateral habenula to ameliorate depressive-like behaviors : Stratégies ciblant l’habénula latérale pour améliorer les symptômes de types dépressifs. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. Available from: http://www.theses.fr/2017PA066301


University of Texas Southwestern Medical Center

2. Cheng, Chih-Jen. Understanding Potassium Homeostasis Using Human and Mouse Genetic Models.

Degree: PhD, Integrative Biology, 2013, University of Texas Southwestern Medical Center

Potassium homeostasis is one of the most sophisticated processes involving multiple organs in mammals. Many physiological functions, such as excitability of muscles and neurons, rely… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Potassium Channels, Inwardly Rectifying; Kidney

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APA (6th Edition):

Cheng, C. (2013). Understanding Potassium Homeostasis Using Human and Mouse Genetic Models. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1234

Chicago Manual of Style (16th Edition):

Cheng, Chih-Jen. “Understanding Potassium Homeostasis Using Human and Mouse Genetic Models.” 2013. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed June 06, 2020. http://hdl.handle.net/2152.5/1234.

MLA Handbook (7th Edition):

Cheng, Chih-Jen. “Understanding Potassium Homeostasis Using Human and Mouse Genetic Models.” 2013. Web. 06 Jun 2020.

Vancouver:

Cheng C. Understanding Potassium Homeostasis Using Human and Mouse Genetic Models. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jun 06]. Available from: http://hdl.handle.net/2152.5/1234.

Council of Science Editors:

Cheng C. Understanding Potassium Homeostasis Using Human and Mouse Genetic Models. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1234

3. Tomczyńska, Magdelena. Biologie de l'endothélium vasculaire isolé de souris transgéniques YAC67 et YAC84- modèles murins du syndrome de Down : Biology of vascular endothelium isolated from transgenic mice YAC67 and YAC84 -mouse models for Down syndrome.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2009, Orléans; Académie polonaise des sciences

GIRK2 est situé sur le chromosome 21, dont la trisomie cause le syndrome de Down (DS). Les proportionss des sous-populations de lymphocytes T sont altérées,… (more)

Subjects/Keywords: Girk2; G-protein inwardly rectifying potassium channel 2

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APA (6th Edition):

Tomczyńska, M. (2009). Biologie de l'endothélium vasculaire isolé de souris transgéniques YAC67 et YAC84- modèles murins du syndrome de Down : Biology of vascular endothelium isolated from transgenic mice YAC67 and YAC84 -mouse models for Down syndrome. (Doctoral Dissertation). Orléans; Académie polonaise des sciences. Retrieved from http://www.theses.fr/2009ORLE2067

Chicago Manual of Style (16th Edition):

Tomczyńska, Magdelena. “Biologie de l'endothélium vasculaire isolé de souris transgéniques YAC67 et YAC84- modèles murins du syndrome de Down : Biology of vascular endothelium isolated from transgenic mice YAC67 and YAC84 -mouse models for Down syndrome.” 2009. Doctoral Dissertation, Orléans; Académie polonaise des sciences. Accessed June 06, 2020. http://www.theses.fr/2009ORLE2067.

MLA Handbook (7th Edition):

Tomczyńska, Magdelena. “Biologie de l'endothélium vasculaire isolé de souris transgéniques YAC67 et YAC84- modèles murins du syndrome de Down : Biology of vascular endothelium isolated from transgenic mice YAC67 and YAC84 -mouse models for Down syndrome.” 2009. Web. 06 Jun 2020.

Vancouver:

Tomczyńska M. Biologie de l'endothélium vasculaire isolé de souris transgéniques YAC67 et YAC84- modèles murins du syndrome de Down : Biology of vascular endothelium isolated from transgenic mice YAC67 and YAC84 -mouse models for Down syndrome. [Internet] [Doctoral dissertation]. Orléans; Académie polonaise des sciences; 2009. [cited 2020 Jun 06]. Available from: http://www.theses.fr/2009ORLE2067.

Council of Science Editors:

Tomczyńska M. Biologie de l'endothélium vasculaire isolé de souris transgéniques YAC67 et YAC84- modèles murins du syndrome de Down : Biology of vascular endothelium isolated from transgenic mice YAC67 and YAC84 -mouse models for Down syndrome. [Doctoral Dissertation]. Orléans; Académie polonaise des sciences; 2009. Available from: http://www.theses.fr/2009ORLE2067

4. Milner, Lauren C. The roles of Mpdz and Kcnj9 in ethanal withdrawal : pleiotropic effects and potential mechanisms.

Degree: PhD, 2010, Oregon Health Sciences University

Subjects/Keywords: Alcoholism; Carrier proteins; Potassium channels; Alcohol Withdrawal Seizures; Carrier Proteins; G Protein-Coupled Inwardly-Rectifying Potassium Channels; Quantitative Trait Loci

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APA (6th Edition):

Milner, L. C. (2010). The roles of Mpdz and Kcnj9 in ethanal withdrawal : pleiotropic effects and potential mechanisms. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4JM27MQ ; http://digitalcommons.ohsu.edu/etd/669

Chicago Manual of Style (16th Edition):

Milner, Lauren C. “The roles of Mpdz and Kcnj9 in ethanal withdrawal : pleiotropic effects and potential mechanisms.” 2010. Doctoral Dissertation, Oregon Health Sciences University. Accessed June 06, 2020. doi:10.6083/M4JM27MQ ; http://digitalcommons.ohsu.edu/etd/669.

MLA Handbook (7th Edition):

Milner, Lauren C. “The roles of Mpdz and Kcnj9 in ethanal withdrawal : pleiotropic effects and potential mechanisms.” 2010. Web. 06 Jun 2020.

Vancouver:

Milner LC. The roles of Mpdz and Kcnj9 in ethanal withdrawal : pleiotropic effects and potential mechanisms. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2010. [cited 2020 Jun 06]. Available from: doi:10.6083/M4JM27MQ ; http://digitalcommons.ohsu.edu/etd/669.

Council of Science Editors:

Milner LC. The roles of Mpdz and Kcnj9 in ethanal withdrawal : pleiotropic effects and potential mechanisms. [Doctoral Dissertation]. Oregon Health Sciences University; 2010. Available from: doi:10.6083/M4JM27MQ ; http://digitalcommons.ohsu.edu/etd/669

5. Gantz, Stephanie C. Leveraging the occurrence of spontaneous D2 receptor-mediated IPSCs to understand the dopamine synapse.

Degree: 2015, Oregon Health Sciences University

Subjects/Keywords: Dopamine; Synaptic Potentials; G Protein-Coupled Inwardly-Rectifying Potassium Channels; Synaptic Transmission; Dopamine D2 Receptors; Neuroscience and Neurobiology

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APA (6th Edition):

Gantz, S. C. (2015). Leveraging the occurrence of spontaneous D2 receptor-mediated IPSCs to understand the dopamine synapse. (Thesis). Oregon Health Sciences University. Retrieved from doi:10.6083/M45X27XF ; http://digitalcommons.ohsu.edu/etd/3731

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gantz, Stephanie C. “Leveraging the occurrence of spontaneous D2 receptor-mediated IPSCs to understand the dopamine synapse.” 2015. Thesis, Oregon Health Sciences University. Accessed June 06, 2020. doi:10.6083/M45X27XF ; http://digitalcommons.ohsu.edu/etd/3731.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gantz, Stephanie C. “Leveraging the occurrence of spontaneous D2 receptor-mediated IPSCs to understand the dopamine synapse.” 2015. Web. 06 Jun 2020.

Vancouver:

Gantz SC. Leveraging the occurrence of spontaneous D2 receptor-mediated IPSCs to understand the dopamine synapse. [Internet] [Thesis]. Oregon Health Sciences University; 2015. [cited 2020 Jun 06]. Available from: doi:10.6083/M45X27XF ; http://digitalcommons.ohsu.edu/etd/3731.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gantz SC. Leveraging the occurrence of spontaneous D2 receptor-mediated IPSCs to understand the dopamine synapse. [Thesis]. Oregon Health Sciences University; 2015. Available from: doi:10.6083/M45X27XF ; http://digitalcommons.ohsu.edu/etd/3731

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

6. Wang, Hao-Ran. Regulation of Endocytosis of ROMK Channel by WNK Kinase Family.

Degree: PhD, Integrative Biology, 2009, University of Texas Southwestern Medical Center

 WNK kinases are members of a novel family of serine/threonine kinases with atypical placement of the catalytic lysine. Mutations in WNK1 and WNK4 cause pseudohypoaldosteronism… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Potassium Channels, Inwardly Rectifying; Protein Structure, Tertiary

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APA (6th Edition):

Wang, H. (2009). Regulation of Endocytosis of ROMK Channel by WNK Kinase Family. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/270

Chicago Manual of Style (16th Edition):

Wang, Hao-Ran. “Regulation of Endocytosis of ROMK Channel by WNK Kinase Family.” 2009. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed June 06, 2020. http://hdl.handle.net/2152.5/270.

MLA Handbook (7th Edition):

Wang, Hao-Ran. “Regulation of Endocytosis of ROMK Channel by WNK Kinase Family.” 2009. Web. 06 Jun 2020.

Vancouver:

Wang H. Regulation of Endocytosis of ROMK Channel by WNK Kinase Family. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2009. [cited 2020 Jun 06]. Available from: http://hdl.handle.net/2152.5/270.

Council of Science Editors:

Wang H. Regulation of Endocytosis of ROMK Channel by WNK Kinase Family. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2009. Available from: http://hdl.handle.net/2152.5/270


Univerzitet u Beogradu

7. Bataveljić, Danijela B., 1983-. Neuroinflamatorni procesi mozga u amiotrofičnoj lateralnoj sklerozi : studija na modelu hSOD1 G93A pacova.

Degree: Biološki fakultet, 2013, Univerzitet u Beogradu

Neuronauke - Neurofiziologija sa biofizikom / Neuroscience - Neurophysiology and Biophysics

Amiotrofična lateralna skleroza (ALS) je neurodegenerativno oboljenje koje pogađa motorne neurone u kičmenoj moždini… (more)

Subjects/Keywords: Amyotrophic lateral sclerosis; neuroinflammation; blood-brain barrier; aquaporin-4; inwardly rectifying potassium channel

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APA (6th Edition):

Bataveljić, Danijela B., 1. (2013). Neuroinflamatorni procesi mozga u amiotrofičnoj lateralnoj sklerozi : studija na modelu hSOD1 G93A pacova. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:3046/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bataveljić, Danijela B., 1983-. “Neuroinflamatorni procesi mozga u amiotrofičnoj lateralnoj sklerozi : studija na modelu hSOD1 G93A pacova.” 2013. Thesis, Univerzitet u Beogradu. Accessed June 06, 2020. https://fedorabg.bg.ac.rs/fedora/get/o:3046/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bataveljić, Danijela B., 1983-. “Neuroinflamatorni procesi mozga u amiotrofičnoj lateralnoj sklerozi : studija na modelu hSOD1 G93A pacova.” 2013. Web. 06 Jun 2020.

Vancouver:

Bataveljić, Danijela B. 1. Neuroinflamatorni procesi mozga u amiotrofičnoj lateralnoj sklerozi : studija na modelu hSOD1 G93A pacova. [Internet] [Thesis]. Univerzitet u Beogradu; 2013. [cited 2020 Jun 06]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:3046/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bataveljić, Danijela B. 1. Neuroinflamatorni procesi mozga u amiotrofičnoj lateralnoj sklerozi : studija na modelu hSOD1 G93A pacova. [Thesis]. Univerzitet u Beogradu; 2013. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:3046/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Iowa

8. Gupte, Raeesa Prashant. Phosphoregulation of somatodendritic voltage-gated potassium channels by pituitary adenylate cyclase-activating polypeptide.

Degree: PhD, Pharmacology, 2015, University of Iowa

  The endogenous neuropeptide pituitary adenylate cyclase-activating polypeptide (PACAP) exerts various neuromodulatory functions in mammalian brain. Enhancement of synaptic activity, mediation of chronic inflammatory and… (more)

Subjects/Keywords: G-protein coupled receptor; Hyperexcitability; Ion channels; Kinase; Neuroprotection; Phosphorylation; Pharmacology

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APA (6th Edition):

Gupte, R. P. (2015). Phosphoregulation of somatodendritic voltage-gated potassium channels by pituitary adenylate cyclase-activating polypeptide. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/5766

Chicago Manual of Style (16th Edition):

Gupte, Raeesa Prashant. “Phosphoregulation of somatodendritic voltage-gated potassium channels by pituitary adenylate cyclase-activating polypeptide.” 2015. Doctoral Dissertation, University of Iowa. Accessed June 06, 2020. https://ir.uiowa.edu/etd/5766.

MLA Handbook (7th Edition):

Gupte, Raeesa Prashant. “Phosphoregulation of somatodendritic voltage-gated potassium channels by pituitary adenylate cyclase-activating polypeptide.” 2015. Web. 06 Jun 2020.

Vancouver:

Gupte RP. Phosphoregulation of somatodendritic voltage-gated potassium channels by pituitary adenylate cyclase-activating polypeptide. [Internet] [Doctoral dissertation]. University of Iowa; 2015. [cited 2020 Jun 06]. Available from: https://ir.uiowa.edu/etd/5766.

Council of Science Editors:

Gupte RP. Phosphoregulation of somatodendritic voltage-gated potassium channels by pituitary adenylate cyclase-activating polypeptide. [Doctoral Dissertation]. University of Iowa; 2015. Available from: https://ir.uiowa.edu/etd/5766

9. Kramer, Paul. New roles for metabotropic glutamate receptor signaling in the physiology and pathology of dopamine neurons.

Degree: PhD, 2016, Oregon Health Sciences University

Subjects/Keywords: Neurosciences; Dopaminergic Neurons; Potassium Channels; Inwardly Rectifying; Receptors; Metabotropic Glutamate; Cocaine; Neurophysiology; Neurology; Neurosciences

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APA (6th Edition):

Kramer, P. (2016). New roles for metabotropic glutamate receptor signaling in the physiology and pathology of dopamine neurons. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4PN94Q2 ; http://digitalcommons.ohsu.edu/etd/3872

Chicago Manual of Style (16th Edition):

Kramer, Paul. “New roles for metabotropic glutamate receptor signaling in the physiology and pathology of dopamine neurons.” 2016. Doctoral Dissertation, Oregon Health Sciences University. Accessed June 06, 2020. doi:10.6083/M4PN94Q2 ; http://digitalcommons.ohsu.edu/etd/3872.

MLA Handbook (7th Edition):

Kramer, Paul. “New roles for metabotropic glutamate receptor signaling in the physiology and pathology of dopamine neurons.” 2016. Web. 06 Jun 2020.

Vancouver:

Kramer P. New roles for metabotropic glutamate receptor signaling in the physiology and pathology of dopamine neurons. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2016. [cited 2020 Jun 06]. Available from: doi:10.6083/M4PN94Q2 ; http://digitalcommons.ohsu.edu/etd/3872.

Council of Science Editors:

Kramer P. New roles for metabotropic glutamate receptor signaling in the physiology and pathology of dopamine neurons. [Doctoral Dissertation]. Oregon Health Sciences University; 2016. Available from: doi:10.6083/M4PN94Q2 ; http://digitalcommons.ohsu.edu/etd/3872


University of Manitoba

10. Peterson, Colleen. The regulation of subfornical organ neurons.

Degree: Biological Sciences, 2019, University of Manitoba

 While the central nervous system (CNS) plays a critical role in the regulation of homeostasis, most of the CNS is isolated from constituents of the… (more)

Subjects/Keywords: Subfornical organ; Neurotensin; G-protein coupled receptor; Early postnatal overnutrition; Homeostasis; Ion channels; Endothelin; Substance P; Voltage-gated cation channels

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APA (6th Edition):

Peterson, C. (2019). The regulation of subfornical organ neurons. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/34097

Chicago Manual of Style (16th Edition):

Peterson, Colleen. “The regulation of subfornical organ neurons.” 2019. Masters Thesis, University of Manitoba. Accessed June 06, 2020. http://hdl.handle.net/1993/34097.

MLA Handbook (7th Edition):

Peterson, Colleen. “The regulation of subfornical organ neurons.” 2019. Web. 06 Jun 2020.

Vancouver:

Peterson C. The regulation of subfornical organ neurons. [Internet] [Masters thesis]. University of Manitoba; 2019. [cited 2020 Jun 06]. Available from: http://hdl.handle.net/1993/34097.

Council of Science Editors:

Peterson C. The regulation of subfornical organ neurons. [Masters Thesis]. University of Manitoba; 2019. Available from: http://hdl.handle.net/1993/34097


University of Georgia

11. Ali, Mourad Wagdy Ahmed. Expression and function of regulator of G-protein signaling 10 (RGS10) in ovarian cancer and microglia.

Degree: PhD, Pharmacy, 2014, University of Georgia

G-Protein coupled receptors (GPCRs) mediate a wide array of cellular functions, such as cell proliferation, migration, and survival. Regulators of G-protein signaling (RGS) proteins are… (more)

Subjects/Keywords: G-protein coupled receptors

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APA (6th Edition):

Ali, M. W. A. (2014). Expression and function of regulator of G-protein signaling 10 (RGS10) in ovarian cancer and microglia. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/ali_mourad_w_201405_phd

Chicago Manual of Style (16th Edition):

Ali, Mourad Wagdy Ahmed. “Expression and function of regulator of G-protein signaling 10 (RGS10) in ovarian cancer and microglia.” 2014. Doctoral Dissertation, University of Georgia. Accessed June 06, 2020. http://purl.galileo.usg.edu/uga_etd/ali_mourad_w_201405_phd.

MLA Handbook (7th Edition):

Ali, Mourad Wagdy Ahmed. “Expression and function of regulator of G-protein signaling 10 (RGS10) in ovarian cancer and microglia.” 2014. Web. 06 Jun 2020.

Vancouver:

Ali MWA. Expression and function of regulator of G-protein signaling 10 (RGS10) in ovarian cancer and microglia. [Internet] [Doctoral dissertation]. University of Georgia; 2014. [cited 2020 Jun 06]. Available from: http://purl.galileo.usg.edu/uga_etd/ali_mourad_w_201405_phd.

Council of Science Editors:

Ali MWA. Expression and function of regulator of G-protein signaling 10 (RGS10) in ovarian cancer and microglia. [Doctoral Dissertation]. University of Georgia; 2014. Available from: http://purl.galileo.usg.edu/uga_etd/ali_mourad_w_201405_phd

12. Mailhac, Camille. Développement d’outils pour l'étude de la signalisation médiée par les récepteurs couplés aux protéines G, basés sur l'utilisation d'anticorps à domaine unique de lama : Development of tools for the study of G protein-coupled receptor-mediated signaling based on the use of lama single domain antibodies.

Degree: Docteur es, Biologie. Immunologie, 2017, Aix Marseille Université

L'objectif principal de ma thèse était de développer de nouvelles technologies et des outils pour l’étude de l’activation des récepteurs couplés aux protéines G (GPCR).À… (more)

Subjects/Keywords: RCPG; G protein coupled receptors

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APA (6th Edition):

Mailhac, C. (2017). Développement d’outils pour l'étude de la signalisation médiée par les récepteurs couplés aux protéines G, basés sur l'utilisation d'anticorps à domaine unique de lama : Development of tools for the study of G protein-coupled receptor-mediated signaling based on the use of lama single domain antibodies. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2017AIXM0405

Chicago Manual of Style (16th Edition):

Mailhac, Camille. “Développement d’outils pour l'étude de la signalisation médiée par les récepteurs couplés aux protéines G, basés sur l'utilisation d'anticorps à domaine unique de lama : Development of tools for the study of G protein-coupled receptor-mediated signaling based on the use of lama single domain antibodies.” 2017. Doctoral Dissertation, Aix Marseille Université. Accessed June 06, 2020. http://www.theses.fr/2017AIXM0405.

MLA Handbook (7th Edition):

Mailhac, Camille. “Développement d’outils pour l'étude de la signalisation médiée par les récepteurs couplés aux protéines G, basés sur l'utilisation d'anticorps à domaine unique de lama : Development of tools for the study of G protein-coupled receptor-mediated signaling based on the use of lama single domain antibodies.” 2017. Web. 06 Jun 2020.

Vancouver:

Mailhac C. Développement d’outils pour l'étude de la signalisation médiée par les récepteurs couplés aux protéines G, basés sur l'utilisation d'anticorps à domaine unique de lama : Development of tools for the study of G protein-coupled receptor-mediated signaling based on the use of lama single domain antibodies. [Internet] [Doctoral dissertation]. Aix Marseille Université 2017. [cited 2020 Jun 06]. Available from: http://www.theses.fr/2017AIXM0405.

Council of Science Editors:

Mailhac C. Développement d’outils pour l'étude de la signalisation médiée par les récepteurs couplés aux protéines G, basés sur l'utilisation d'anticorps à domaine unique de lama : Development of tools for the study of G protein-coupled receptor-mediated signaling based on the use of lama single domain antibodies. [Doctoral Dissertation]. Aix Marseille Université 2017. Available from: http://www.theses.fr/2017AIXM0405


University of Georgia

13. Altman, Molly Koertel. G-protein coupled receptor mediated signaling pathways in chemoresistant melanoma and ovarian cancer.

Degree: PhD, Pharmacology, 2014, University of Georgia

 Cellular signaling pathways are involved in numerous physiological processes such as reproduction, growth, and the development of cancer and chemoresistance. G-protein coupled receptors (GPCRs) are… (more)

Subjects/Keywords: G-protein coupled receptors

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APA (6th Edition):

Altman, M. K. (2014). G-protein coupled receptor mediated signaling pathways in chemoresistant melanoma and ovarian cancer. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/altman_molly_k_201408_phd

Chicago Manual of Style (16th Edition):

Altman, Molly Koertel. “G-protein coupled receptor mediated signaling pathways in chemoresistant melanoma and ovarian cancer.” 2014. Doctoral Dissertation, University of Georgia. Accessed June 06, 2020. http://purl.galileo.usg.edu/uga_etd/altman_molly_k_201408_phd.

MLA Handbook (7th Edition):

Altman, Molly Koertel. “G-protein coupled receptor mediated signaling pathways in chemoresistant melanoma and ovarian cancer.” 2014. Web. 06 Jun 2020.

Vancouver:

Altman MK. G-protein coupled receptor mediated signaling pathways in chemoresistant melanoma and ovarian cancer. [Internet] [Doctoral dissertation]. University of Georgia; 2014. [cited 2020 Jun 06]. Available from: http://purl.galileo.usg.edu/uga_etd/altman_molly_k_201408_phd.

Council of Science Editors:

Altman MK. G-protein coupled receptor mediated signaling pathways in chemoresistant melanoma and ovarian cancer. [Doctoral Dissertation]. University of Georgia; 2014. Available from: http://purl.galileo.usg.edu/uga_etd/altman_molly_k_201408_phd

14. Bataveljić Danijela. Neuroinflammatory processes in the amyotrophic latera sclerosis brain : study of hSOD1 G93A rat model.

Degree: PhD, Biology, 2012, University of Belgrade

Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disorder affecting lower and upper motor neurons. The disease shows fast progression and short lifetime following the disease… (more)

Subjects/Keywords: Amyotrophic lateral sclerosis; neuroinflammation; blood-brain barrier; aquaporin-4; inwardly rectifying potassium channel; Amiotrofična lateralna skleroza; neuroinflamacija; krvno-moždana barijera; akvaporin-4; ulazni kalijumski ispravljač

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APA (6th Edition):

Danijela, B. (2012). Neuroinflammatory processes in the amyotrophic latera sclerosis brain : study of hSOD1 G93A rat model. (Doctoral Dissertation). University of Belgrade. Retrieved from http://dx.doi.org/10.2298/BG20121106BATAVELJIC ; http://eteze.bg.ac.rs/application/showtheses?thesesId=51 ; https://fedorabg.bg.ac.rs/fedora/get/o:3046/bdef:Content/get ; http://vbs.rs/scripts/cobiss?command=SEARCH&base=99999&select=ID=41902607

Chicago Manual of Style (16th Edition):

Danijela, Bataveljić. “Neuroinflammatory processes in the amyotrophic latera sclerosis brain : study of hSOD1 G93A rat model.” 2012. Doctoral Dissertation, University of Belgrade. Accessed June 06, 2020. http://dx.doi.org/10.2298/BG20121106BATAVELJIC ; http://eteze.bg.ac.rs/application/showtheses?thesesId=51 ; https://fedorabg.bg.ac.rs/fedora/get/o:3046/bdef:Content/get ; http://vbs.rs/scripts/cobiss?command=SEARCH&base=99999&select=ID=41902607.

MLA Handbook (7th Edition):

Danijela, Bataveljić. “Neuroinflammatory processes in the amyotrophic latera sclerosis brain : study of hSOD1 G93A rat model.” 2012. Web. 06 Jun 2020.

Vancouver:

Danijela B. Neuroinflammatory processes in the amyotrophic latera sclerosis brain : study of hSOD1 G93A rat model. [Internet] [Doctoral dissertation]. University of Belgrade; 2012. [cited 2020 Jun 06]. Available from: http://dx.doi.org/10.2298/BG20121106BATAVELJIC ; http://eteze.bg.ac.rs/application/showtheses?thesesId=51 ; https://fedorabg.bg.ac.rs/fedora/get/o:3046/bdef:Content/get ; http://vbs.rs/scripts/cobiss?command=SEARCH&base=99999&select=ID=41902607.

Council of Science Editors:

Danijela B. Neuroinflammatory processes in the amyotrophic latera sclerosis brain : study of hSOD1 G93A rat model. [Doctoral Dissertation]. University of Belgrade; 2012. Available from: http://dx.doi.org/10.2298/BG20121106BATAVELJIC ; http://eteze.bg.ac.rs/application/showtheses?thesesId=51 ; https://fedorabg.bg.ac.rs/fedora/get/o:3046/bdef:Content/get ; http://vbs.rs/scripts/cobiss?command=SEARCH&base=99999&select=ID=41902607


University of Oxford

15. Kaizik, Stephan Martin. Analysis of mouse models of insulin secretion disorders.

Degree: PhD, 2010, University of Oxford

 In this thesis, three mouse models and cell culture techniques were used to investigate genetic factors leading to glucose intolerance, diabetes or hyperinsulinemia. Loss of… (more)

Subjects/Keywords: 616.4; Life Sciences; Biology; Biochemistry; Molecular Biology; Genetics (life sciences); Endocrinology; Metabolism; Transgenics; Medical Sciences; Diabetes; Genetics (medical sciences); Pharmacology; Physiology; Microscopy; Adenosine Triphosphate; Calcium Signaling; Calcium Oscillations; Insulin Secretion; Glutathione; Disease Models; Islets of Langerhans; Genetic Background; Neonatal diabetes; Hyperinsulinemia; Glutathione Peroxidase; Potassium Channels; Inwardly Rectifying; KATP channels; Transgenic Mice; Nicotinamide Nucleotide Transhydrogenase; Reactive Oxygen Species; RNA Interference; Quantitative PCR; Western Blotting; ROS Measurement; Calcium Imaging; Electrophysiology; Pancreatic Islet Isolation; Sulfonylurea; Glibenclamide; Tolbutamide; Menadione

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APA (6th Edition):

Kaizik, S. M. (2010). Analysis of mouse models of insulin secretion disorders. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:4d44b68a-a0a0-4c92-8809-00ddbfe3e636 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.533854

Chicago Manual of Style (16th Edition):

Kaizik, Stephan Martin. “Analysis of mouse models of insulin secretion disorders.” 2010. Doctoral Dissertation, University of Oxford. Accessed June 06, 2020. http://ora.ox.ac.uk/objects/uuid:4d44b68a-a0a0-4c92-8809-00ddbfe3e636 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.533854.

MLA Handbook (7th Edition):

Kaizik, Stephan Martin. “Analysis of mouse models of insulin secretion disorders.” 2010. Web. 06 Jun 2020.

Vancouver:

Kaizik SM. Analysis of mouse models of insulin secretion disorders. [Internet] [Doctoral dissertation]. University of Oxford; 2010. [cited 2020 Jun 06]. Available from: http://ora.ox.ac.uk/objects/uuid:4d44b68a-a0a0-4c92-8809-00ddbfe3e636 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.533854.

Council of Science Editors:

Kaizik SM. Analysis of mouse models of insulin secretion disorders. [Doctoral Dissertation]. University of Oxford; 2010. Available from: http://ora.ox.ac.uk/objects/uuid:4d44b68a-a0a0-4c92-8809-00ddbfe3e636 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.533854


University of Alberta

16. Lorenz, Chad D. Axon excitability testing shows increased IH activity in populations of slow versus fast motor axons of the rat.

Degree: MS, Physical Education and Recreation Centre for Neuroscience, 2013, University of Alberta

 Despite extensive knowledge of variations in motoneuron (MN) soma and muscle properties across different healthy muscles or motor units, there is comparatively little knowledge about… (more)

Subjects/Keywords: Axon; motor control; IH; axon plasticity; axon ion channels; ionic conductances; ALS; hyperpolarization-activated inwardly rectifying cation conductance; amyotrophic lateral sclerosis; Axon physiology; axon excitability testing; motor axons

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APA (6th Edition):

Lorenz, C. D. (2013). Axon excitability testing shows increased IH activity in populations of slow versus fast motor axons of the rat. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/70795812h

Chicago Manual of Style (16th Edition):

Lorenz, Chad D. “Axon excitability testing shows increased IH activity in populations of slow versus fast motor axons of the rat.” 2013. Masters Thesis, University of Alberta. Accessed June 06, 2020. https://era.library.ualberta.ca/files/70795812h.

MLA Handbook (7th Edition):

Lorenz, Chad D. “Axon excitability testing shows increased IH activity in populations of slow versus fast motor axons of the rat.” 2013. Web. 06 Jun 2020.

Vancouver:

Lorenz CD. Axon excitability testing shows increased IH activity in populations of slow versus fast motor axons of the rat. [Internet] [Masters thesis]. University of Alberta; 2013. [cited 2020 Jun 06]. Available from: https://era.library.ualberta.ca/files/70795812h.

Council of Science Editors:

Lorenz CD. Axon excitability testing shows increased IH activity in populations of slow versus fast motor axons of the rat. [Masters Thesis]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/70795812h

17. Ha, Junghoon. Hydrogen Sulfide Regulation of Kir Channels.

Degree: PhD, Physiology and Biophysics, 2017, Virginia Commonwealth University

Inwardly rectifying potassium (Kir) channels establish and regulate the resting membrane potential of excitable cells in the heart, brain and other peripheral tissues. Phosphatidylinositol-… (more)

Subjects/Keywords: Hydrogen sulfide; potassium channels; PIP2; phosphoinositide; stroke; ischemia; phosphatidylinositol 4; 5-bisphosphate (PIP2); Inwardly rectifying K+ (Kir) Channels; GIRK channels; KATP channels; Gasotransmitters; Cellular and Molecular Physiology; Medicine and Health Sciences; Molecular and Cellular Neuroscience

Inwardly rectifying potassium (Kir) channels establish and regulate the resting… …PIP2 Activation of Kir Channels Inwardly rectifying potassium channels (Fig. 1)… …not only limited to KATP channels, but also applied to other inwardly rectifying potassium… …35 Figure 7. A schematic demonstrating inwardly-rectifying whole-cell potassium currents in… …injury IP3 Inositol 1,4,5-trisphosphate IRK3 Inwardly rectifying potassium channel 3 KATP… 

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APA (6th Edition):

Ha, J. (2017). Hydrogen Sulfide Regulation of Kir Channels. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/KSEY-HY47 ; https://scholarscompass.vcu.edu/etd/5204

Chicago Manual of Style (16th Edition):

Ha, Junghoon. “Hydrogen Sulfide Regulation of Kir Channels.” 2017. Doctoral Dissertation, Virginia Commonwealth University. Accessed June 06, 2020. https://doi.org/10.25772/KSEY-HY47 ; https://scholarscompass.vcu.edu/etd/5204.

MLA Handbook (7th Edition):

Ha, Junghoon. “Hydrogen Sulfide Regulation of Kir Channels.” 2017. Web. 06 Jun 2020.

Vancouver:

Ha J. Hydrogen Sulfide Regulation of Kir Channels. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2017. [cited 2020 Jun 06]. Available from: https://doi.org/10.25772/KSEY-HY47 ; https://scholarscompass.vcu.edu/etd/5204.

Council of Science Editors:

Ha J. Hydrogen Sulfide Regulation of Kir Channels. [Doctoral Dissertation]. Virginia Commonwealth University; 2017. Available from: https://doi.org/10.25772/KSEY-HY47 ; https://scholarscompass.vcu.edu/etd/5204


University of Toronto

18. Hamoudi, Zina. Isolation and Characterization of the Corazonin Receptor and Possible Physiological Roles of Corazonin in the Kissing Bug, Rhodnius prolixus.

Degree: 2015, University of Toronto

Neuropeptides control many physiological and endocrinological processes in animals, acting as neuroactive chemicals within the central and peripheral nervous systems. Corazonin (CRZ) is one such… (more)

Subjects/Keywords: G protein-coupled receptor; Insect; Neuropeptide; 0409

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APA (6th Edition):

Hamoudi, Z. (2015). Isolation and Characterization of the Corazonin Receptor and Possible Physiological Roles of Corazonin in the Kissing Bug, Rhodnius prolixus. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70398

Chicago Manual of Style (16th Edition):

Hamoudi, Zina. “Isolation and Characterization of the Corazonin Receptor and Possible Physiological Roles of Corazonin in the Kissing Bug, Rhodnius prolixus.” 2015. Masters Thesis, University of Toronto. Accessed June 06, 2020. http://hdl.handle.net/1807/70398.

MLA Handbook (7th Edition):

Hamoudi, Zina. “Isolation and Characterization of the Corazonin Receptor and Possible Physiological Roles of Corazonin in the Kissing Bug, Rhodnius prolixus.” 2015. Web. 06 Jun 2020.

Vancouver:

Hamoudi Z. Isolation and Characterization of the Corazonin Receptor and Possible Physiological Roles of Corazonin in the Kissing Bug, Rhodnius prolixus. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2020 Jun 06]. Available from: http://hdl.handle.net/1807/70398.

Council of Science Editors:

Hamoudi Z. Isolation and Characterization of the Corazonin Receptor and Possible Physiological Roles of Corazonin in the Kissing Bug, Rhodnius prolixus. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70398


University of Texas Southwestern Medical Center

19. Li, Tingting. Role of TGR5 in Bile Acid Metabolism.

Degree: PhD, 2011, University of Texas Southwestern Medical Center

 TGR5 is a G protein-coupled bile acid receptor present in various tissues in the body. Its agonism increases energy expenditure and lowers blood glucose. Thus,… (more)

Subjects/Keywords: Receptors, G-Protein-Coupled; Gallbladder; Metabolism

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APA (6th Edition):

Li, T. (2011). Role of TGR5 in Bile Acid Metabolism. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/869

Chicago Manual of Style (16th Edition):

Li, Tingting. “Role of TGR5 in Bile Acid Metabolism.” 2011. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed June 06, 2020. http://hdl.handle.net/2152.5/869.

MLA Handbook (7th Edition):

Li, Tingting. “Role of TGR5 in Bile Acid Metabolism.” 2011. Web. 06 Jun 2020.

Vancouver:

Li T. Role of TGR5 in Bile Acid Metabolism. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Jun 06]. Available from: http://hdl.handle.net/2152.5/869.

Council of Science Editors:

Li T. Role of TGR5 in Bile Acid Metabolism. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/869


University of North Texas

20. Potbhare, Vrinda Nikhil. Expression of G-protein Coupled Receptors in Young and Mature Thrombocytes and Knockdown of Gpr18 in Zebrafish.

Degree: 2013, University of North Texas

 In this study, a novel method based on biotinylated antibodies and streptavidin coated magnetic beads was used to separate the thrombocyte subpopulations from zebrafish whole… (more)

Subjects/Keywords: Thrombocytes; G-protein coupled receptors; knockdown; zebrafish

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APA (6th Edition):

Potbhare, V. N. (2013). Expression of G-protein Coupled Receptors in Young and Mature Thrombocytes and Knockdown of Gpr18 in Zebrafish. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc271881/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Potbhare, Vrinda Nikhil. “Expression of G-protein Coupled Receptors in Young and Mature Thrombocytes and Knockdown of Gpr18 in Zebrafish.” 2013. Thesis, University of North Texas. Accessed June 06, 2020. https://digital.library.unt.edu/ark:/67531/metadc271881/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Potbhare, Vrinda Nikhil. “Expression of G-protein Coupled Receptors in Young and Mature Thrombocytes and Knockdown of Gpr18 in Zebrafish.” 2013. Web. 06 Jun 2020.

Vancouver:

Potbhare VN. Expression of G-protein Coupled Receptors in Young and Mature Thrombocytes and Knockdown of Gpr18 in Zebrafish. [Internet] [Thesis]. University of North Texas; 2013. [cited 2020 Jun 06]. Available from: https://digital.library.unt.edu/ark:/67531/metadc271881/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Potbhare VN. Expression of G-protein Coupled Receptors in Young and Mature Thrombocytes and Knockdown of Gpr18 in Zebrafish. [Thesis]. University of North Texas; 2013. Available from: https://digital.library.unt.edu/ark:/67531/metadc271881/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Lokits, Alyssa Dawn. Nuthin' but a G (protein) thang: Insights into the Mechanics of G protein Signaling from Sequence and Structure.

Degree: PhD, Neuroscience, 2017, Vanderbilt University

G protein-coupled receptors (GPCRs) are a large and diverse group of transmembrane receptors which convert extracellular signals into intracellular responses via coupling to heterotrimeric G(more)

Subjects/Keywords: G protein; G protein Coupled Receptor; GPCR; structure; computation; evolution; phylogenetics; thermodynamics; G alpha; Rosetta

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APA (6th Edition):

Lokits, A. D. (2017). Nuthin' but a G (protein) thang: Insights into the Mechanics of G protein Signaling from Sequence and Structure. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-06132017-085149/ ;

Chicago Manual of Style (16th Edition):

Lokits, Alyssa Dawn. “Nuthin' but a G (protein) thang: Insights into the Mechanics of G protein Signaling from Sequence and Structure.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed June 06, 2020. http://etd.library.vanderbilt.edu/available/etd-06132017-085149/ ;.

MLA Handbook (7th Edition):

Lokits, Alyssa Dawn. “Nuthin' but a G (protein) thang: Insights into the Mechanics of G protein Signaling from Sequence and Structure.” 2017. Web. 06 Jun 2020.

Vancouver:

Lokits AD. Nuthin' but a G (protein) thang: Insights into the Mechanics of G protein Signaling from Sequence and Structure. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2020 Jun 06]. Available from: http://etd.library.vanderbilt.edu/available/etd-06132017-085149/ ;.

Council of Science Editors:

Lokits AD. Nuthin' but a G (protein) thang: Insights into the Mechanics of G protein Signaling from Sequence and Structure. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://etd.library.vanderbilt.edu/available/etd-06132017-085149/ ;

22. Rodzli, Nazahiyah Binti Ahmad. STRUCTURAL AND INTERACTION STUDIES OF PSD95 PDZ DOMAIN-MEDIATED KIR2.1 CLUSTERING MECHANISMS.

Degree: 2017, University of Manchester

 PSD95 is the canonical member of the Membrane Associated Guanylate Kinase class of scaffold proteins. PSD95 is a five-domain major scaffolding protein abundant in the… (more)

Subjects/Keywords: PDZ domain, PSD95; Inwardly rectifying potassium channel, Kir; X-ray crystallography, SAXS, ITC, cryoEM

…calorimetry IPTG Isopropyl-β-thiogalactopyranoside Kir Inwardly rectifying potassium channel LB… …clustering to occur. This thesis focuses on the strong inwardly rectifying potassium channel… …Calcium-calmodulin-dependent serine protein kinase CCD Charge coupled device CCP4… …that membrane protein/ion channels are clustered by head-to-head multimerization of copies of… …70 Figure 2.3 Pipeline of protein crystallographic structure solution post data… 

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APA (6th Edition):

Rodzli, N. B. A. (2017). STRUCTURAL AND INTERACTION STUDIES OF PSD95 PDZ DOMAIN-MEDIATED KIR2.1 CLUSTERING MECHANISMS. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:308198

Chicago Manual of Style (16th Edition):

Rodzli, Nazahiyah Binti Ahmad. “STRUCTURAL AND INTERACTION STUDIES OF PSD95 PDZ DOMAIN-MEDIATED KIR2.1 CLUSTERING MECHANISMS.” 2017. Doctoral Dissertation, University of Manchester. Accessed June 06, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:308198.

MLA Handbook (7th Edition):

Rodzli, Nazahiyah Binti Ahmad. “STRUCTURAL AND INTERACTION STUDIES OF PSD95 PDZ DOMAIN-MEDIATED KIR2.1 CLUSTERING MECHANISMS.” 2017. Web. 06 Jun 2020.

Vancouver:

Rodzli NBA. STRUCTURAL AND INTERACTION STUDIES OF PSD95 PDZ DOMAIN-MEDIATED KIR2.1 CLUSTERING MECHANISMS. [Internet] [Doctoral dissertation]. University of Manchester; 2017. [cited 2020 Jun 06]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:308198.

Council of Science Editors:

Rodzli NBA. STRUCTURAL AND INTERACTION STUDIES OF PSD95 PDZ DOMAIN-MEDIATED KIR2.1 CLUSTERING MECHANISMS. [Doctoral Dissertation]. University of Manchester; 2017. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:308198

23. Rodzli, Nazahiyah. Structural and interaction studies of PSD95 PDZ domain-mediated Kir2.1 clustering mechanisms.

Degree: PhD, 2017, University of Manchester

 PSD95 is the canonical member of the Membrane Associated Guanylate Kinase class of scaffold proteins. PSD95 is a five-domain major scaffolding protein abundant in the… (more)

Subjects/Keywords: 572; X-ray crystallography; SAXS; ITC; cryoEM; Inwardly rectifying potassium channel; Kir; PDZ domain; PSD95

…calorimetry IPTG Isopropyl-β-thiogalactopyranoside Kir Inwardly rectifying potassium channel LB… …clustering to occur. This thesis focuses on the strong inwardly rectifying potassium channel… …Calcium-calmodulin-dependent serine protein kinase CCD Charge coupled device CCP4… …that membrane protein/ion channels are clustered by head-to-head multimerization of copies of… …70 Figure 2.3 Pipeline of protein crystallographic structure solution post data… 

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APA (6th Edition):

Rodzli, N. (2017). Structural and interaction studies of PSD95 PDZ domain-mediated Kir2.1 clustering mechanisms. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/structural-and-interaction-studies-of-psd95-pdz-domainmediated-kir21-clustering-mechanisms(b07686a7-7a91-4673-bed3-16707f502c61).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.713605

Chicago Manual of Style (16th Edition):

Rodzli, Nazahiyah. “Structural and interaction studies of PSD95 PDZ domain-mediated Kir2.1 clustering mechanisms.” 2017. Doctoral Dissertation, University of Manchester. Accessed June 06, 2020. https://www.research.manchester.ac.uk/portal/en/theses/structural-and-interaction-studies-of-psd95-pdz-domainmediated-kir21-clustering-mechanisms(b07686a7-7a91-4673-bed3-16707f502c61).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.713605.

MLA Handbook (7th Edition):

Rodzli, Nazahiyah. “Structural and interaction studies of PSD95 PDZ domain-mediated Kir2.1 clustering mechanisms.” 2017. Web. 06 Jun 2020.

Vancouver:

Rodzli N. Structural and interaction studies of PSD95 PDZ domain-mediated Kir2.1 clustering mechanisms. [Internet] [Doctoral dissertation]. University of Manchester; 2017. [cited 2020 Jun 06]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/structural-and-interaction-studies-of-psd95-pdz-domainmediated-kir21-clustering-mechanisms(b07686a7-7a91-4673-bed3-16707f502c61).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.713605.

Council of Science Editors:

Rodzli N. Structural and interaction studies of PSD95 PDZ domain-mediated Kir2.1 clustering mechanisms. [Doctoral Dissertation]. University of Manchester; 2017. Available from: https://www.research.manchester.ac.uk/portal/en/theses/structural-and-interaction-studies-of-psd95-pdz-domainmediated-kir21-clustering-mechanisms(b07686a7-7a91-4673-bed3-16707f502c61).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.713605


Université de Grenoble

24. Niescierowicz, Katarzyna. Développement de la technologie des récepteurs couplés à un canal ionique pour des études structure-fonction des récepteurs couplés aux protéines G et du canal Kir6.2 : Development of the Ion Channel-Coupled Receptor technology in structure-function studies of G protein-coupled receptors and Kir6.2 channel.

Degree: Docteur es, Biologie structurale et biologie moléculaire, 2013, Université de Grenoble

Les Récepteurs Couplés à un Canal Ionique (ICCRs) sont des canaux ioniques artificielscréés par fusion d'un Récepteur Couplé aux Protéines G (RCPG) au canal ionique… (more)

Subjects/Keywords: GPCR; Canal KATP; Biocapteur; Électrophysiologie (patch–clamp); Double micro–électrodes); Ingénierie moléculaire; Patch clamp; GPCR; Artificial ion channels; G protein-coupled receptor

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APA (6th Edition):

Niescierowicz, K. (2013). Développement de la technologie des récepteurs couplés à un canal ionique pour des études structure-fonction des récepteurs couplés aux protéines G et du canal Kir6.2 : Development of the Ion Channel-Coupled Receptor technology in structure-function studies of G protein-coupled receptors and Kir6.2 channel. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2013GRENV056

Chicago Manual of Style (16th Edition):

Niescierowicz, Katarzyna. “Développement de la technologie des récepteurs couplés à un canal ionique pour des études structure-fonction des récepteurs couplés aux protéines G et du canal Kir6.2 : Development of the Ion Channel-Coupled Receptor technology in structure-function studies of G protein-coupled receptors and Kir6.2 channel.” 2013. Doctoral Dissertation, Université de Grenoble. Accessed June 06, 2020. http://www.theses.fr/2013GRENV056.

MLA Handbook (7th Edition):

Niescierowicz, Katarzyna. “Développement de la technologie des récepteurs couplés à un canal ionique pour des études structure-fonction des récepteurs couplés aux protéines G et du canal Kir6.2 : Development of the Ion Channel-Coupled Receptor technology in structure-function studies of G protein-coupled receptors and Kir6.2 channel.” 2013. Web. 06 Jun 2020.

Vancouver:

Niescierowicz K. Développement de la technologie des récepteurs couplés à un canal ionique pour des études structure-fonction des récepteurs couplés aux protéines G et du canal Kir6.2 : Development of the Ion Channel-Coupled Receptor technology in structure-function studies of G protein-coupled receptors and Kir6.2 channel. [Internet] [Doctoral dissertation]. Université de Grenoble; 2013. [cited 2020 Jun 06]. Available from: http://www.theses.fr/2013GRENV056.

Council of Science Editors:

Niescierowicz K. Développement de la technologie des récepteurs couplés à un canal ionique pour des études structure-fonction des récepteurs couplés aux protéines G et du canal Kir6.2 : Development of the Ion Channel-Coupled Receptor technology in structure-function studies of G protein-coupled receptors and Kir6.2 channel. [Doctoral Dissertation]. Université de Grenoble; 2013. Available from: http://www.theses.fr/2013GRENV056


RMIT University

25. Lawrence, J. The mechanism of action of the neuropeptide galanin in sensory pathways with special reference to nociception.

Degree: 2014, RMIT University

 The neuropeptide galanin is up-regulated after nerve injury in areas involved in nociception, and enhances the analgesic effects of morphine. Galanin binds to G-protein coupled(more)

Subjects/Keywords: Fields of Research; galanin; galanin receptors; GAL1; GAL2; GAL3; g-protein coupled receptors; GPCRs; dorsal root ganglion; DRG; voltage gated calcium channels; VGCC

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APA (6th Edition):

Lawrence, J. (2014). The mechanism of action of the neuropeptide galanin in sensory pathways with special reference to nociception. (Thesis). RMIT University. Retrieved from http://researchbank.rmit.edu.au/view/rmit:160761

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lawrence, J. “The mechanism of action of the neuropeptide galanin in sensory pathways with special reference to nociception.” 2014. Thesis, RMIT University. Accessed June 06, 2020. http://researchbank.rmit.edu.au/view/rmit:160761.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lawrence, J. “The mechanism of action of the neuropeptide galanin in sensory pathways with special reference to nociception.” 2014. Web. 06 Jun 2020.

Vancouver:

Lawrence J. The mechanism of action of the neuropeptide galanin in sensory pathways with special reference to nociception. [Internet] [Thesis]. RMIT University; 2014. [cited 2020 Jun 06]. Available from: http://researchbank.rmit.edu.au/view/rmit:160761.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lawrence J. The mechanism of action of the neuropeptide galanin in sensory pathways with special reference to nociception. [Thesis]. RMIT University; 2014. Available from: http://researchbank.rmit.edu.au/view/rmit:160761

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

26. Huang, Weishan. IDENTIFICATION AND CHARACTERIZATION OF.

Degree: MS, Cell and Molecular Biology, 2010, Penn State University

 The Tec family kinase, Itk has been shown to be critical in T cell activation and development downstream of the T cell receptor (TCR). Other… (more)

Subjects/Keywords: protein-protein interaction; ; 13; signal transduction; T cell receptor; Itk; G protein coupled receptor

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APA (6th Edition):

Huang, W. (2010). IDENTIFICATION AND CHARACTERIZATION OF. (Masters Thesis). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/11229

Chicago Manual of Style (16th Edition):

Huang, Weishan. “IDENTIFICATION AND CHARACTERIZATION OF.” 2010. Masters Thesis, Penn State University. Accessed June 06, 2020. https://etda.libraries.psu.edu/catalog/11229.

MLA Handbook (7th Edition):

Huang, Weishan. “IDENTIFICATION AND CHARACTERIZATION OF.” 2010. Web. 06 Jun 2020.

Vancouver:

Huang W. IDENTIFICATION AND CHARACTERIZATION OF. [Internet] [Masters thesis]. Penn State University; 2010. [cited 2020 Jun 06]. Available from: https://etda.libraries.psu.edu/catalog/11229.

Council of Science Editors:

Huang W. IDENTIFICATION AND CHARACTERIZATION OF. [Masters Thesis]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/11229


Vanderbilt University

27. Bender, Brian Joseph. Knowledge-Based Modeling of G-Protein Coupled Receptors and their Binding Partners.

Degree: PhD, Pharmacology, 2019, Vanderbilt University

G-protein coupled receptors (GPCRs) represent the largest family of membrane proteins and the most heavily targeted classes of proteins for therapeutic intervention. Relatively little is… (more)

Subjects/Keywords: protein modeling; rosetta; peptide docking; g-protein coupled receptors; ligand docking

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bender, B. J. (2019). Knowledge-Based Modeling of G-Protein Coupled Receptors and their Binding Partners. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-01172019-103123/ ;

Chicago Manual of Style (16th Edition):

Bender, Brian Joseph. “Knowledge-Based Modeling of G-Protein Coupled Receptors and their Binding Partners.” 2019. Doctoral Dissertation, Vanderbilt University. Accessed June 06, 2020. http://etd.library.vanderbilt.edu/available/etd-01172019-103123/ ;.

MLA Handbook (7th Edition):

Bender, Brian Joseph. “Knowledge-Based Modeling of G-Protein Coupled Receptors and their Binding Partners.” 2019. Web. 06 Jun 2020.

Vancouver:

Bender BJ. Knowledge-Based Modeling of G-Protein Coupled Receptors and their Binding Partners. [Internet] [Doctoral dissertation]. Vanderbilt University; 2019. [cited 2020 Jun 06]. Available from: http://etd.library.vanderbilt.edu/available/etd-01172019-103123/ ;.

Council of Science Editors:

Bender BJ. Knowledge-Based Modeling of G-Protein Coupled Receptors and their Binding Partners. [Doctoral Dissertation]. Vanderbilt University; 2019. Available from: http://etd.library.vanderbilt.edu/available/etd-01172019-103123/ ;


University of Edinburgh

28. White, Colin D. Dissection of GnRH receptor-G protein coupling.

Degree: 2009, University of Edinburgh

 Hypothalamic gonadotropin-releasing hormone (GnRH) (GnRH I) is the central regulator of the mammalian reproductive system. Most vertebrates studied also possess a second form of GnRH,… (more)

Subjects/Keywords: 612.6; GnRH; Hypothalamic gonadotropin-releasing hormone; G-protein; G protein-coupled receptor

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APA (6th Edition):

White, C. D. (2009). Dissection of GnRH receptor-G protein coupling. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/3885

Chicago Manual of Style (16th Edition):

White, Colin D. “Dissection of GnRH receptor-G protein coupling.” 2009. Doctoral Dissertation, University of Edinburgh. Accessed June 06, 2020. http://hdl.handle.net/1842/3885.

MLA Handbook (7th Edition):

White, Colin D. “Dissection of GnRH receptor-G protein coupling.” 2009. Web. 06 Jun 2020.

Vancouver:

White CD. Dissection of GnRH receptor-G protein coupling. [Internet] [Doctoral dissertation]. University of Edinburgh; 2009. [cited 2020 Jun 06]. Available from: http://hdl.handle.net/1842/3885.

Council of Science Editors:

White CD. Dissection of GnRH receptor-G protein coupling. [Doctoral Dissertation]. University of Edinburgh; 2009. Available from: http://hdl.handle.net/1842/3885


Vanderbilt University

29. Wu, Yuantai. Functional study of g protein signaling during growth and development of the social amoeba Dictyostelium discoideum.

Degree: PhD, Biological Sciences, 2013, Vanderbilt University

 The social amoeba Dictyostelium discoideum senses the bacterial metabolite folic acid to track down bacteria. Previous studies suggested that the folic acid receptor is a… (more)

Subjects/Keywords: Galpha 8; Dictyostelium discoideum; G protein signaling; G protein-coupled receptors; GABA

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wu, Y. (2013). Functional study of g protein signaling during growth and development of the social amoeba Dictyostelium discoideum. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07192013-105419/ ;

Chicago Manual of Style (16th Edition):

Wu, Yuantai. “Functional study of g protein signaling during growth and development of the social amoeba Dictyostelium discoideum.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed June 06, 2020. http://etd.library.vanderbilt.edu/available/etd-07192013-105419/ ;.

MLA Handbook (7th Edition):

Wu, Yuantai. “Functional study of g protein signaling during growth and development of the social amoeba Dictyostelium discoideum.” 2013. Web. 06 Jun 2020.

Vancouver:

Wu Y. Functional study of g protein signaling during growth and development of the social amoeba Dictyostelium discoideum. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2020 Jun 06]. Available from: http://etd.library.vanderbilt.edu/available/etd-07192013-105419/ ;.

Council of Science Editors:

Wu Y. Functional study of g protein signaling during growth and development of the social amoeba Dictyostelium discoideum. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://etd.library.vanderbilt.edu/available/etd-07192013-105419/ ;


University of Manitoba

30. Sidhu, Crystal. Quinine regulates Rac1 GTPase activity through the bitter taste receptor T2R4 and G-Protein.

Degree: Oral Biology, 2016, University of Manitoba

 Rac1 is a member of the Rho family of low molecular mass GTP binding proteins (GTPases). It regulates the dynamics of actin cytoskeleton by causing… (more)

Subjects/Keywords: G protein-coupled receptor; Rac1 inactivation; Bitter taste receptor; G-alpha protein; Quinine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sidhu, C. (2016). Quinine regulates Rac1 GTPase activity through the bitter taste receptor T2R4 and G-Protein. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31786

Chicago Manual of Style (16th Edition):

Sidhu, Crystal. “Quinine regulates Rac1 GTPase activity through the bitter taste receptor T2R4 and G-Protein.” 2016. Masters Thesis, University of Manitoba. Accessed June 06, 2020. http://hdl.handle.net/1993/31786.

MLA Handbook (7th Edition):

Sidhu, Crystal. “Quinine regulates Rac1 GTPase activity through the bitter taste receptor T2R4 and G-Protein.” 2016. Web. 06 Jun 2020.

Vancouver:

Sidhu C. Quinine regulates Rac1 GTPase activity through the bitter taste receptor T2R4 and G-Protein. [Internet] [Masters thesis]. University of Manitoba; 2016. [cited 2020 Jun 06]. Available from: http://hdl.handle.net/1993/31786.

Council of Science Editors:

Sidhu C. Quinine regulates Rac1 GTPase activity through the bitter taste receptor T2R4 and G-Protein. [Masters Thesis]. University of Manitoba; 2016. Available from: http://hdl.handle.net/1993/31786

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