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You searched for subject:(Fibroblast Growth Factor). Showing records 1 – 30 of 203 total matches.

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University of Hawaii – Manoa

1. Ulu, Ferhat. Dose-dependent functions of FGF9 in male primordial germ cell differentiation.

Degree: 2016, University of Hawaii – Manoa

M.S. University of Hawaii at Manoa 2013.

Primordial Germ Cells (PGCs) are the precursors of gametes. For PGC male differentiation, (i) inhibition of meiosis and… (more)

Subjects/Keywords: Fibroblast Growth Factor 9

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APA (6th Edition):

Ulu, F. (2016). Dose-dependent functions of FGF9 in male primordial germ cell differentiation. (Thesis). University of Hawaii – Manoa. Retrieved from http://hdl.handle.net/10125/100649

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ulu, Ferhat. “Dose-dependent functions of FGF9 in male primordial germ cell differentiation.” 2016. Thesis, University of Hawaii – Manoa. Accessed April 18, 2021. http://hdl.handle.net/10125/100649.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ulu, Ferhat. “Dose-dependent functions of FGF9 in male primordial germ cell differentiation.” 2016. Web. 18 Apr 2021.

Vancouver:

Ulu F. Dose-dependent functions of FGF9 in male primordial germ cell differentiation. [Internet] [Thesis]. University of Hawaii – Manoa; 2016. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10125/100649.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ulu F. Dose-dependent functions of FGF9 in male primordial germ cell differentiation. [Thesis]. University of Hawaii – Manoa; 2016. Available from: http://hdl.handle.net/10125/100649

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

2. Kir, Serkan. Regulation of Liver Metabolism by Fibroblast Growth Factor 19.

Degree: 2012, University of Texas Southwestern Medical Center

Fibroblast Growth Factor (FGF) 19 is a postprandial enterokine up-regulated by bile acid receptor FXR upon bile acid uptake into the ileum. FGF19 inhibits hepatic… (more)

Subjects/Keywords: Liver Glycogen; Fibroblast Growth Factor; Protein Biosynthesis

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APA (6th Edition):

Kir, S. (2012). Regulation of Liver Metabolism by Fibroblast Growth Factor 19. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1027

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kir, Serkan. “Regulation of Liver Metabolism by Fibroblast Growth Factor 19.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed April 18, 2021. http://hdl.handle.net/2152.5/1027.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kir, Serkan. “Regulation of Liver Metabolism by Fibroblast Growth Factor 19.” 2012. Web. 18 Apr 2021.

Vancouver:

Kir S. Regulation of Liver Metabolism by Fibroblast Growth Factor 19. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2152.5/1027.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kir S. Regulation of Liver Metabolism by Fibroblast Growth Factor 19. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1027

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

3. Bookout, Angie Lynn. The Liver-Derived Endocrine Hormone FGF21 Alters Metabolism and Diurnal Behavior via the Nervous System.

Degree: 2012, University of Texas Southwestern Medical Center

 Fuel acquisition is essential to survival. During privation, the body protects glucose concentrations acutely by glycogenolysis, and later by gluconeogenesis and ketogenesis. Additionally, animals alter… (more)

Subjects/Keywords: Fibroblast Growth Factor; PPAR gamma; Liver

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APA (6th Edition):

Bookout, A. L. (2012). The Liver-Derived Endocrine Hormone FGF21 Alters Metabolism and Diurnal Behavior via the Nervous System. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/987

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bookout, Angie Lynn. “The Liver-Derived Endocrine Hormone FGF21 Alters Metabolism and Diurnal Behavior via the Nervous System.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed April 18, 2021. http://hdl.handle.net/2152.5/987.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bookout, Angie Lynn. “The Liver-Derived Endocrine Hormone FGF21 Alters Metabolism and Diurnal Behavior via the Nervous System.” 2012. Web. 18 Apr 2021.

Vancouver:

Bookout AL. The Liver-Derived Endocrine Hormone FGF21 Alters Metabolism and Diurnal Behavior via the Nervous System. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2152.5/987.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bookout AL. The Liver-Derived Endocrine Hormone FGF21 Alters Metabolism and Diurnal Behavior via the Nervous System. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/987

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

4. Camero, Corrine Michelle. Investigating fibroblast growth factor and its role in canine osteosarcoma.

Degree: MS, VMS-Veterinary Clinical Medcne, 2017, University of Illinois – Urbana-Champaign

 Canine osteosarcoma (OS) is a malignant neoplasia of the osteoblast, most often identified in the appendicular skeleton, which is both locally aggressive and highly metastatic.… (more)

Subjects/Keywords: fibroblast growth factor; canine osteosarcoma; differentiation

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APA (6th Edition):

Camero, C. M. (2017). Investigating fibroblast growth factor and its role in canine osteosarcoma. (Thesis). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/99310

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Camero, Corrine Michelle. “Investigating fibroblast growth factor and its role in canine osteosarcoma.” 2017. Thesis, University of Illinois – Urbana-Champaign. Accessed April 18, 2021. http://hdl.handle.net/2142/99310.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Camero, Corrine Michelle. “Investigating fibroblast growth factor and its role in canine osteosarcoma.” 2017. Web. 18 Apr 2021.

Vancouver:

Camero CM. Investigating fibroblast growth factor and its role in canine osteosarcoma. [Internet] [Thesis]. University of Illinois – Urbana-Champaign; 2017. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2142/99310.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Camero CM. Investigating fibroblast growth factor and its role in canine osteosarcoma. [Thesis]. University of Illinois – Urbana-Champaign; 2017. Available from: http://hdl.handle.net/2142/99310

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Louisiana State University

5. Sutton, Elizabeth Frost. Fibroblast Growth Factor 21 is a Novel Protein Sensor in Pregnancy.

Degree: PhD, Life Sciences, 2017, Louisiana State University

 The twenty-first century has experienced a shift in cause of death worldwide from communicable diseases to noncommunicable diseases. Interestingly, many of these implicated chronic diseases,… (more)

Subjects/Keywords: Fibroblast growth factor 21; FGF21; pregnancy; protein

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APA (6th Edition):

Sutton, E. F. (2017). Fibroblast Growth Factor 21 is a Novel Protein Sensor in Pregnancy. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-05182017-213436 ; https://digitalcommons.lsu.edu/gradschool_dissertations/4263

Chicago Manual of Style (16th Edition):

Sutton, Elizabeth Frost. “Fibroblast Growth Factor 21 is a Novel Protein Sensor in Pregnancy.” 2017. Doctoral Dissertation, Louisiana State University. Accessed April 18, 2021. etd-05182017-213436 ; https://digitalcommons.lsu.edu/gradschool_dissertations/4263.

MLA Handbook (7th Edition):

Sutton, Elizabeth Frost. “Fibroblast Growth Factor 21 is a Novel Protein Sensor in Pregnancy.” 2017. Web. 18 Apr 2021.

Vancouver:

Sutton EF. Fibroblast Growth Factor 21 is a Novel Protein Sensor in Pregnancy. [Internet] [Doctoral dissertation]. Louisiana State University; 2017. [cited 2021 Apr 18]. Available from: etd-05182017-213436 ; https://digitalcommons.lsu.edu/gradschool_dissertations/4263.

Council of Science Editors:

Sutton EF. Fibroblast Growth Factor 21 is a Novel Protein Sensor in Pregnancy. [Doctoral Dissertation]. Louisiana State University; 2017. Available from: etd-05182017-213436 ; https://digitalcommons.lsu.edu/gradschool_dissertations/4263

6. Kato, Manabu. Activation of FGF2-FGFR Signaling in the Castrated Mouse Prostate Stimulates the Proliferation of Basal Epithelial Cells.

Degree: 博士(医学), 2017, Mie University / 三重大学

The prostate gland is unique in that it undergoes rapid regression following castration but regenerates completely once androgens are replaced. Residual ductal components play an… (more)

Subjects/Keywords: basal epithelial cells; castration; fibroblast growth factor 2; fibroblast growth factor receptor; growth factor; organ culture; prostate

Page 1 Page 2 Page 3

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APA (6th Edition):

Kato, M. (2017). Activation of FGF2-FGFR Signaling in the Castrated Mouse Prostate Stimulates the Proliferation of Basal Epithelial Cells. (Thesis). Mie University / 三重大学. Retrieved from http://hdl.handle.net/10076/00016972

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kato, Manabu. “Activation of FGF2-FGFR Signaling in the Castrated Mouse Prostate Stimulates the Proliferation of Basal Epithelial Cells.” 2017. Thesis, Mie University / 三重大学. Accessed April 18, 2021. http://hdl.handle.net/10076/00016972.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kato, Manabu. “Activation of FGF2-FGFR Signaling in the Castrated Mouse Prostate Stimulates the Proliferation of Basal Epithelial Cells.” 2017. Web. 18 Apr 2021.

Vancouver:

Kato M. Activation of FGF2-FGFR Signaling in the Castrated Mouse Prostate Stimulates the Proliferation of Basal Epithelial Cells. [Internet] [Thesis]. Mie University / 三重大学; 2017. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10076/00016972.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kato M. Activation of FGF2-FGFR Signaling in the Castrated Mouse Prostate Stimulates the Proliferation of Basal Epithelial Cells. [Thesis]. Mie University / 三重大学; 2017. Available from: http://hdl.handle.net/10076/00016972

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Moore, Lisa. FGF signaling in Xenopus laevis lens regeneration.

Degree: PhD, Cell and Developmental Biology, 2015, University of Illinois – Urbana-Champaign

 The larvae of the frog Xenopus laevis is capable of regenerating lenses. In this re- generative process, the corneal tissue is capable of forming a… (more)

Subjects/Keywords: Xenopus laevis; lens; cornea; regeneration; Fibroblast Growth Factors (FGF); Fibroblast Growth Factor Receptors (FGFR)

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APA (6th Edition):

Moore, L. (2015). FGF signaling in Xenopus laevis lens regeneration. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/78759

Chicago Manual of Style (16th Edition):

Moore, Lisa. “FGF signaling in Xenopus laevis lens regeneration.” 2015. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed April 18, 2021. http://hdl.handle.net/2142/78759.

MLA Handbook (7th Edition):

Moore, Lisa. “FGF signaling in Xenopus laevis lens regeneration.” 2015. Web. 18 Apr 2021.

Vancouver:

Moore L. FGF signaling in Xenopus laevis lens regeneration. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2015. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2142/78759.

Council of Science Editors:

Moore L. FGF signaling in Xenopus laevis lens regeneration. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2015. Available from: http://hdl.handle.net/2142/78759


Queen Mary, University of London

8. Mistry, J. N. Defining the role of fibroblast growth factor 21 (FGF21) in the pathogenesis of growth hormone resistance and subsequent growth failure in chronic childhood conditions.

Degree: PhD, 2019, Queen Mary, University of London

Fibroblast growth factor 21 (FGF21) is an essential metabolic regulator, adapting to changes in nutritional status. Excessive undernutrition is suggested to elevate FGF21 levels, developing… (more)

Subjects/Keywords: Fibroblast growth factor 21; FGF21; postnatal growth failure

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APA (6th Edition):

Mistry, J. N. (2019). Defining the role of fibroblast growth factor 21 (FGF21) in the pathogenesis of growth hormone resistance and subsequent growth failure in chronic childhood conditions. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/55457 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786344

Chicago Manual of Style (16th Edition):

Mistry, J N. “Defining the role of fibroblast growth factor 21 (FGF21) in the pathogenesis of growth hormone resistance and subsequent growth failure in chronic childhood conditions.” 2019. Doctoral Dissertation, Queen Mary, University of London. Accessed April 18, 2021. http://qmro.qmul.ac.uk/xmlui/handle/123456789/55457 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786344.

MLA Handbook (7th Edition):

Mistry, J N. “Defining the role of fibroblast growth factor 21 (FGF21) in the pathogenesis of growth hormone resistance and subsequent growth failure in chronic childhood conditions.” 2019. Web. 18 Apr 2021.

Vancouver:

Mistry JN. Defining the role of fibroblast growth factor 21 (FGF21) in the pathogenesis of growth hormone resistance and subsequent growth failure in chronic childhood conditions. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2019. [cited 2021 Apr 18]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/55457 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786344.

Council of Science Editors:

Mistry JN. Defining the role of fibroblast growth factor 21 (FGF21) in the pathogenesis of growth hormone resistance and subsequent growth failure in chronic childhood conditions. [Doctoral Dissertation]. Queen Mary, University of London; 2019. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/55457 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786344

9. Pauley, Sarah. Fibroblast Growth Factor Expression in Inner Correlates with Defects in the Null Mutant.

Degree: M.S. in Biomedical Sciences, Biomedical Sciences (graduate program), 2001, Creighton University

 The inner ear is a multifunctional organ required for hearing, balance, and proprioception of the head. Each area of sensory cells in the ear must… (more)

Subjects/Keywords: Fibroblast Growth Factor 2; Receptors, Fibroblast Growth Factor – genetics

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APA (6th Edition):

Pauley, S. (2001). Fibroblast Growth Factor Expression in Inner Correlates with Defects in the Null Mutant. (Masters Thesis). Creighton University. Retrieved from http://hdl.handle.net/10504/65393

Chicago Manual of Style (16th Edition):

Pauley, Sarah. “Fibroblast Growth Factor Expression in Inner Correlates with Defects in the Null Mutant.” 2001. Masters Thesis, Creighton University. Accessed April 18, 2021. http://hdl.handle.net/10504/65393.

MLA Handbook (7th Edition):

Pauley, Sarah. “Fibroblast Growth Factor Expression in Inner Correlates with Defects in the Null Mutant.” 2001. Web. 18 Apr 2021.

Vancouver:

Pauley S. Fibroblast Growth Factor Expression in Inner Correlates with Defects in the Null Mutant. [Internet] [Masters thesis]. Creighton University; 2001. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10504/65393.

Council of Science Editors:

Pauley S. Fibroblast Growth Factor Expression in Inner Correlates with Defects in the Null Mutant. [Masters Thesis]. Creighton University; 2001. Available from: http://hdl.handle.net/10504/65393

10. Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, Hiroyuki. Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin.

Degree: 博士(歯学), 2014, Fukuoka Dental College / 福岡歯科大学

Transforming growth factor-β1 (TGF-β1) reportedly causes the differentiation of fibroblasts to myofibroblasts during wound healing. We investigated the mechanism underlying the activation of latent TGF-β1… (more)

Subjects/Keywords: keratinocyte; fibroblast; transforming growth factor-β1; αv-integrin; matrix metalloproteinase

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APA (6th Edition):

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, H. (2014). Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. (Thesis). Fukuoka Dental College / 福岡歯科大学. Retrieved from http://id.nii.ac.jp/1167/00000003/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, Hiroyuki. “Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin.” 2014. Thesis, Fukuoka Dental College / 福岡歯科大学. Accessed April 18, 2021. http://id.nii.ac.jp/1167/00000003/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, Hiroyuki. “Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin.” 2014. Web. 18 Apr 2021.

Vancouver:

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa H. Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. [Internet] [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2014. [cited 2021 Apr 18]. Available from: http://id.nii.ac.jp/1167/00000003/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa H. Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2014. Available from: http://id.nii.ac.jp/1167/00000003/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. 小林, 信博. Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる.

Degree: 博士(歯学), 2017, Osaka Dental University / 大阪歯科大学

The effect of porous alpha-tricalcium phosphate (-TCP) with immobilized basic fibroblast growth factor (bFGF) on bone regeneration was evaluated in a canine mandibular bone defect… (more)

Subjects/Keywords: alpha-tricalcium phosphate |basic fibroblast growth factor | bone regeneration

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APA (6th Edition):

小林, . (2017). Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる. (Thesis). Osaka Dental University / 大阪歯科大学. Retrieved from http://id.nii.ac.jp/1392/00000138/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

小林, 信博. “Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる.” 2017. Thesis, Osaka Dental University / 大阪歯科大学. Accessed April 18, 2021. http://id.nii.ac.jp/1392/00000138/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

小林, 信博. “Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる.” 2017. Web. 18 Apr 2021.

Vancouver:

小林 . Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる. [Internet] [Thesis]. Osaka Dental University / 大阪歯科大学; 2017. [cited 2021 Apr 18]. Available from: http://id.nii.ac.jp/1392/00000138/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

小林 . Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる. [Thesis]. Osaka Dental University / 大阪歯科大学; 2017. Available from: http://id.nii.ac.jp/1392/00000138/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

12. Girer, Nathaniel Gabriel. AN EXAMINATION OF THE ARYL HYDROCARBON RECEPTOR IN LIVER METABOLISM.

Degree: 2016, Penn State University

 The aryl hydrocarbon receptor (AHR) is a ligand-activated transcription factor from the basic helix-loop-helix PER/ARNT/SIM family of proteins that is evolutionarily conserved in both vertebrates… (more)

Subjects/Keywords: Aryl Hydrocarbon Receptor; Fibroblast growth factor 21; Liver Metabolism

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APA (6th Edition):

Girer, N. G. (2016). AN EXAMINATION OF THE ARYL HYDROCARBON RECEPTOR IN LIVER METABOLISM. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13610nug128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Girer, Nathaniel Gabriel. “AN EXAMINATION OF THE ARYL HYDROCARBON RECEPTOR IN LIVER METABOLISM.” 2016. Thesis, Penn State University. Accessed April 18, 2021. https://submit-etda.libraries.psu.edu/catalog/13610nug128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Girer, Nathaniel Gabriel. “AN EXAMINATION OF THE ARYL HYDROCARBON RECEPTOR IN LIVER METABOLISM.” 2016. Web. 18 Apr 2021.

Vancouver:

Girer NG. AN EXAMINATION OF THE ARYL HYDROCARBON RECEPTOR IN LIVER METABOLISM. [Internet] [Thesis]. Penn State University; 2016. [cited 2021 Apr 18]. Available from: https://submit-etda.libraries.psu.edu/catalog/13610nug128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Girer NG. AN EXAMINATION OF THE ARYL HYDROCARBON RECEPTOR IN LIVER METABOLISM. [Thesis]. Penn State University; 2016. Available from: https://submit-etda.libraries.psu.edu/catalog/13610nug128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

13. Huang, Yanqing. Fibroblast Growth Factor Signaling in Prostate Stem Cells and Prostate Cancer.

Degree: PhD, Biomedical Sciences, 2015, Texas A&M University

 The prostate is an androgen-dependent male reproductive organ that is comprised of epithelial and stromal compartments. The epithelial compartment contains basal, luminal, and neuroendocrine cells.… (more)

Subjects/Keywords: Fibroblast Growth Factor; Prostate Stem Cells; Prostate Cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Huang, Y. (2015). Fibroblast Growth Factor Signaling in Prostate Stem Cells and Prostate Cancer. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155497

Chicago Manual of Style (16th Edition):

Huang, Yanqing. “Fibroblast Growth Factor Signaling in Prostate Stem Cells and Prostate Cancer.” 2015. Doctoral Dissertation, Texas A&M University. Accessed April 18, 2021. http://hdl.handle.net/1969.1/155497.

MLA Handbook (7th Edition):

Huang, Yanqing. “Fibroblast Growth Factor Signaling in Prostate Stem Cells and Prostate Cancer.” 2015. Web. 18 Apr 2021.

Vancouver:

Huang Y. Fibroblast Growth Factor Signaling in Prostate Stem Cells and Prostate Cancer. [Internet] [Doctoral dissertation]. Texas A&M University; 2015. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1969.1/155497.

Council of Science Editors:

Huang Y. Fibroblast Growth Factor Signaling in Prostate Stem Cells and Prostate Cancer. [Doctoral Dissertation]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155497


University of Texas Southwestern Medical Center

14. Paulson, Vera Ashley. High-Resolution Array Comparative Genomic Hybridization Identifies Common Targets in Rhabdomyosarcoma.

Degree: 2012, University of Texas Southwestern Medical Center

 Rhabdomyosarcoma (RMS) accounts for nearly 50 percent of the soft tissue sarcomas that affect children. There are two major histological variants, alveolar (ARMS) and embryonal… (more)

Subjects/Keywords: Rhabdomyosarcoma; Gene Expression Regulation, Neoplastic; Receptor, Fibroblast Growth Factor, Type 4

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Paulson, V. A. (2012). High-Resolution Array Comparative Genomic Hybridization Identifies Common Targets in Rhabdomyosarcoma. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1112

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Paulson, Vera Ashley. “High-Resolution Array Comparative Genomic Hybridization Identifies Common Targets in Rhabdomyosarcoma.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed April 18, 2021. http://hdl.handle.net/2152.5/1112.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Paulson, Vera Ashley. “High-Resolution Array Comparative Genomic Hybridization Identifies Common Targets in Rhabdomyosarcoma.” 2012. Web. 18 Apr 2021.

Vancouver:

Paulson VA. High-Resolution Array Comparative Genomic Hybridization Identifies Common Targets in Rhabdomyosarcoma. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2152.5/1112.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Paulson VA. High-Resolution Array Comparative Genomic Hybridization Identifies Common Targets in Rhabdomyosarcoma. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1112

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

15. Wei, Wangzhi. The Role of Alternatively spliced Fibroblast Growth Factor Receptor 2 Isoforms in Breast Cancer.

Degree: 2011, University of Toronto

Recent genome-wide association studies identified FGFR2 as one of breast cancer susceptibility genes. FGFR2 expression was down-regulated in breast carcinomas when compared with paired normal… (more)

Subjects/Keywords: Fibroblast Growth Factor Receptor 2; Breast Cancer; NF-κB; 0992

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wei, W. (2011). The Role of Alternatively spliced Fibroblast Growth Factor Receptor 2 Isoforms in Breast Cancer. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/31636

Chicago Manual of Style (16th Edition):

Wei, Wangzhi. “The Role of Alternatively spliced Fibroblast Growth Factor Receptor 2 Isoforms in Breast Cancer.” 2011. Masters Thesis, University of Toronto. Accessed April 18, 2021. http://hdl.handle.net/1807/31636.

MLA Handbook (7th Edition):

Wei, Wangzhi. “The Role of Alternatively spliced Fibroblast Growth Factor Receptor 2 Isoforms in Breast Cancer.” 2011. Web. 18 Apr 2021.

Vancouver:

Wei W. The Role of Alternatively spliced Fibroblast Growth Factor Receptor 2 Isoforms in Breast Cancer. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1807/31636.

Council of Science Editors:

Wei W. The Role of Alternatively spliced Fibroblast Growth Factor Receptor 2 Isoforms in Breast Cancer. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/31636


University of Texas Southwestern Medical Center

16. Boney-Montoya, Jamie. Understanding the Molecular Basis for FGF15/19 and FGF21 Actions on Energy Homeostasis.

Degree: 2012, University of Texas Southwestern Medical Center

 Insulin and glucagon have long been known to play essential roles in controlling energy balance during the fed and fasted states, respectively. Recently, additional metabolic… (more)

Subjects/Keywords: Fibroblast Growth Factor; Glucose; Cyclic AMP Response Element-Binding Protein

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APA (6th Edition):

Boney-Montoya, J. (2012). Understanding the Molecular Basis for FGF15/19 and FGF21 Actions on Energy Homeostasis. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/986

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Boney-Montoya, Jamie. “Understanding the Molecular Basis for FGF15/19 and FGF21 Actions on Energy Homeostasis.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed April 18, 2021. http://hdl.handle.net/2152.5/986.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Boney-Montoya, Jamie. “Understanding the Molecular Basis for FGF15/19 and FGF21 Actions on Energy Homeostasis.” 2012. Web. 18 Apr 2021.

Vancouver:

Boney-Montoya J. Understanding the Molecular Basis for FGF15/19 and FGF21 Actions on Energy Homeostasis. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2152.5/986.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Boney-Montoya J. Understanding the Molecular Basis for FGF15/19 and FGF21 Actions on Energy Homeostasis. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/986

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

17. Dutchak, Paul Anthony. Metabolic Regulation by Fibroblast Growth Factor 21.

Degree: 2011, University of Texas Southwestern Medical Center

Fibroblast growth factor 21 (FGF21) is a secreted hormone that can beneficially regulate glucose and lipid homeostasis. Through a reverse endocrinology approach, we uncovered that… (more)

Subjects/Keywords: Fibroblast Growth Factor; Liver; Peroxisome Proliferator-Activated Receptors

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dutchak, P. A. (2011). Metabolic Regulation by Fibroblast Growth Factor 21. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dutchak, Paul Anthony. “Metabolic Regulation by Fibroblast Growth Factor 21.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed April 18, 2021. http://hdl.handle.net/2152.5/935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dutchak, Paul Anthony. “Metabolic Regulation by Fibroblast Growth Factor 21.” 2011. Web. 18 Apr 2021.

Vancouver:

Dutchak PA. Metabolic Regulation by Fibroblast Growth Factor 21. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2152.5/935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dutchak PA. Metabolic Regulation by Fibroblast Growth Factor 21. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University College London (University of London)

18. Britto, Jonathan Anthony. Syndromic craniofacial dysostosis : from genotype to phenotype : studies of FGFR gene expression in human craniofacial development and craniosynostosis.

Degree: PhD, 2001, University College London (University of London)

Fibroblast growth factor receptors (FGFRs) are a subset of receptor tyrosine kinases. Receptor diversity results from the differential splicing of mRNA to generate membrane bound… (more)

Subjects/Keywords: 612; Fibroblast growth factor receptors

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Britto, J. A. (2001). Syndromic craniofacial dysostosis : from genotype to phenotype : studies of FGFR gene expression in human craniofacial development and craniosynostosis. (Doctoral Dissertation). University College London (University of London). Retrieved from https://discovery.ucl.ac.uk/id/eprint/10100497/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268446

Chicago Manual of Style (16th Edition):

Britto, Jonathan Anthony. “Syndromic craniofacial dysostosis : from genotype to phenotype : studies of FGFR gene expression in human craniofacial development and craniosynostosis.” 2001. Doctoral Dissertation, University College London (University of London). Accessed April 18, 2021. https://discovery.ucl.ac.uk/id/eprint/10100497/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268446.

MLA Handbook (7th Edition):

Britto, Jonathan Anthony. “Syndromic craniofacial dysostosis : from genotype to phenotype : studies of FGFR gene expression in human craniofacial development and craniosynostosis.” 2001. Web. 18 Apr 2021.

Vancouver:

Britto JA. Syndromic craniofacial dysostosis : from genotype to phenotype : studies of FGFR gene expression in human craniofacial development and craniosynostosis. [Internet] [Doctoral dissertation]. University College London (University of London); 2001. [cited 2021 Apr 18]. Available from: https://discovery.ucl.ac.uk/id/eprint/10100497/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268446.

Council of Science Editors:

Britto JA. Syndromic craniofacial dysostosis : from genotype to phenotype : studies of FGFR gene expression in human craniofacial development and craniosynostosis. [Doctoral Dissertation]. University College London (University of London); 2001. Available from: https://discovery.ucl.ac.uk/id/eprint/10100497/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268446


University of Colorado

19. Brooks, Leah Rae. Fibroblast growth factor signaling in the developing serotonergic system and anxiety-related behavior.

Degree: PhD, Integrative Physiology, 2014, University of Colorado

  Anxiety disorders are some of the most commonly diagnosed psychopathologies in both pediatric and adult populations and have been linked to disrupted brain serotonergic… (more)

Subjects/Keywords: Anxiety; Dorsal raphe; Fibroblast growth factor; Serotonin; Neuroscience and Neurobiology

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APA (6th Edition):

Brooks, L. R. (2014). Fibroblast growth factor signaling in the developing serotonergic system and anxiety-related behavior. (Doctoral Dissertation). University of Colorado. Retrieved from https://scholar.colorado.edu/iphy_gradetds/30

Chicago Manual of Style (16th Edition):

Brooks, Leah Rae. “Fibroblast growth factor signaling in the developing serotonergic system and anxiety-related behavior.” 2014. Doctoral Dissertation, University of Colorado. Accessed April 18, 2021. https://scholar.colorado.edu/iphy_gradetds/30.

MLA Handbook (7th Edition):

Brooks, Leah Rae. “Fibroblast growth factor signaling in the developing serotonergic system and anxiety-related behavior.” 2014. Web. 18 Apr 2021.

Vancouver:

Brooks LR. Fibroblast growth factor signaling in the developing serotonergic system and anxiety-related behavior. [Internet] [Doctoral dissertation]. University of Colorado; 2014. [cited 2021 Apr 18]. Available from: https://scholar.colorado.edu/iphy_gradetds/30.

Council of Science Editors:

Brooks LR. Fibroblast growth factor signaling in the developing serotonergic system and anxiety-related behavior. [Doctoral Dissertation]. University of Colorado; 2014. Available from: https://scholar.colorado.edu/iphy_gradetds/30


University of Manitoba

20. Wang, Jie. Fibroblast growth factor-16 and acute doxorubicin cardiotoxicity: a target for early protection.

Degree: Physiology and Pathophysiology, 2018, University of Manitoba

 Background: Doxorubicin is an anti-cancer drug that is widely used in chemotherapy. However, doxorubicin-induced cardiotoxicity is a major risk factor for cancer patients and survivors,… (more)

Subjects/Keywords: Fibroblast growth factor 16; doxorubicin; multidrug resistance protein 1; cardioprotection

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APA (6th Edition):

Wang, J. (2018). Fibroblast growth factor-16 and acute doxorubicin cardiotoxicity: a target for early protection. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/33048

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Jie. “Fibroblast growth factor-16 and acute doxorubicin cardiotoxicity: a target for early protection.” 2018. Thesis, University of Manitoba. Accessed April 18, 2021. http://hdl.handle.net/1993/33048.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Jie. “Fibroblast growth factor-16 and acute doxorubicin cardiotoxicity: a target for early protection.” 2018. Web. 18 Apr 2021.

Vancouver:

Wang J. Fibroblast growth factor-16 and acute doxorubicin cardiotoxicity: a target for early protection. [Internet] [Thesis]. University of Manitoba; 2018. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1993/33048.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang J. Fibroblast growth factor-16 and acute doxorubicin cardiotoxicity: a target for early protection. [Thesis]. University of Manitoba; 2018. Available from: http://hdl.handle.net/1993/33048

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Catholique de Louvain

21. Vietti, Giulia. Molecular and cellular mechanisms of the pro-fibrotic effects of carbon nanotubes.

Degree: 2016, Université Catholique de Louvain

Carbon nanotubes (CNT) are molecular-scale tubes of graphene sheets rolled into cylinders with peculiar characteristics that make them highly attractive for numerous industrial applications. Thus,… (more)

Subjects/Keywords: Proliferation; Lung fibrosis; Fibroblast; Carbon nanotube; Adverse outcome pathway; Growth factor

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APA (6th Edition):

Vietti, G. (2016). Molecular and cellular mechanisms of the pro-fibrotic effects of carbon nanotubes. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/174071

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vietti, Giulia. “Molecular and cellular mechanisms of the pro-fibrotic effects of carbon nanotubes.” 2016. Thesis, Université Catholique de Louvain. Accessed April 18, 2021. http://hdl.handle.net/2078.1/174071.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vietti, Giulia. “Molecular and cellular mechanisms of the pro-fibrotic effects of carbon nanotubes.” 2016. Web. 18 Apr 2021.

Vancouver:

Vietti G. Molecular and cellular mechanisms of the pro-fibrotic effects of carbon nanotubes. [Internet] [Thesis]. Université Catholique de Louvain; 2016. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/2078.1/174071.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vietti G. Molecular and cellular mechanisms of the pro-fibrotic effects of carbon nanotubes. [Thesis]. Université Catholique de Louvain; 2016. Available from: http://hdl.handle.net/2078.1/174071

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Tanner, Yasmine. Drug resistance mechanisms of FGFR-driven cancers.

Degree: PhD, 2019, Queen Mary, University of London

 The fibroblast growth factor (FGF) signalling pathway contributes to the regulation of a variety of cellular functions, affecting differentiation, migration, proliferation, and survival. Unsurprisingly, cancer… (more)

Subjects/Keywords: fibroblast growth factor; cancer; drug resistance; FGFR-driven cancers

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APA (6th Edition):

Tanner, Y. (2019). Drug resistance mechanisms of FGFR-driven cancers. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/56803 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775329

Chicago Manual of Style (16th Edition):

Tanner, Yasmine. “Drug resistance mechanisms of FGFR-driven cancers.” 2019. Doctoral Dissertation, Queen Mary, University of London. Accessed April 18, 2021. http://qmro.qmul.ac.uk/xmlui/handle/123456789/56803 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775329.

MLA Handbook (7th Edition):

Tanner, Yasmine. “Drug resistance mechanisms of FGFR-driven cancers.” 2019. Web. 18 Apr 2021.

Vancouver:

Tanner Y. Drug resistance mechanisms of FGFR-driven cancers. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2019. [cited 2021 Apr 18]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/56803 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775329.

Council of Science Editors:

Tanner Y. Drug resistance mechanisms of FGFR-driven cancers. [Doctoral Dissertation]. Queen Mary, University of London; 2019. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/56803 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775329


Queen Mary, University of London

23. Robbez-Masson, Luisa. Investigating the functional significance of an FGFR2 intronic SNP in breast cancer.

Degree: PhD, 2013, Queen Mary, University of London

 Single nucleotide polymorphisms present in the second intron of the fibroblast growth factor receptor 2 (FGFR2) gene have been linked with increased risk of breast… (more)

Subjects/Keywords: 616.99; Medicine; Cancer; Breast cancer; Fibroblast growth factor receptor 2 (FGFR2)

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APA (6th Edition):

Robbez-Masson, L. (2013). Investigating the functional significance of an FGFR2 intronic SNP in breast cancer. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/8539 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667133

Chicago Manual of Style (16th Edition):

Robbez-Masson, Luisa. “Investigating the functional significance of an FGFR2 intronic SNP in breast cancer.” 2013. Doctoral Dissertation, Queen Mary, University of London. Accessed April 18, 2021. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8539 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667133.

MLA Handbook (7th Edition):

Robbez-Masson, Luisa. “Investigating the functional significance of an FGFR2 intronic SNP in breast cancer.” 2013. Web. 18 Apr 2021.

Vancouver:

Robbez-Masson L. Investigating the functional significance of an FGFR2 intronic SNP in breast cancer. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2013. [cited 2021 Apr 18]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8539 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667133.

Council of Science Editors:

Robbez-Masson L. Investigating the functional significance of an FGFR2 intronic SNP in breast cancer. [Doctoral Dissertation]. Queen Mary, University of London; 2013. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8539 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667133


IUPUI

24. Bonfitto, Anna. Testing bone cell models responsive to a soluble form of klotho.

Degree: 2016, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Fibroblast growth factor-23 (FGF23) is a hormone produced in bone that acts upon the kidney to control blood phosphate and… (more)

Subjects/Keywords: Fibroblast growth factor-23; Klotho; Phosphate metabolism; MC3T3

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APA (6th Edition):

Bonfitto, A. (2016). Testing bone cell models responsive to a soluble form of klotho. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/11872

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bonfitto, Anna. “Testing bone cell models responsive to a soluble form of klotho.” 2016. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/11872.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bonfitto, Anna. “Testing bone cell models responsive to a soluble form of klotho.” 2016. Web. 18 Apr 2021.

Vancouver:

Bonfitto A. Testing bone cell models responsive to a soluble form of klotho. [Internet] [Thesis]. IUPUI; 2016. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/11872.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bonfitto A. Testing bone cell models responsive to a soluble form of klotho. [Thesis]. IUPUI; 2016. Available from: http://hdl.handle.net/1805/11872

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

25. Koleini, Navid. The role of high molecular weight fibroblast growth factor-2 in cardiac response to injury.

Degree: Physiology and Pathophysiology, 2019, University of Manitoba

Fibroblast growth factor-2 (FGF2) is a multifunctional protein expressed as 18 kDa, low molecular weight (Lo-FGF2), and >20 kDa, high molecular weight (Hi-FGF2) isoforms with… (more)

Subjects/Keywords: Fibroblast Growth Factor 2; Doxorubicin cardiotoxicity; Pressure overload cardiac injury

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Koleini, N. (2019). The role of high molecular weight fibroblast growth factor-2 in cardiac response to injury. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/34449

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Koleini, Navid. “The role of high molecular weight fibroblast growth factor-2 in cardiac response to injury.” 2019. Thesis, University of Manitoba. Accessed April 18, 2021. http://hdl.handle.net/1993/34449.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Koleini, Navid. “The role of high molecular weight fibroblast growth factor-2 in cardiac response to injury.” 2019. Web. 18 Apr 2021.

Vancouver:

Koleini N. The role of high molecular weight fibroblast growth factor-2 in cardiac response to injury. [Internet] [Thesis]. University of Manitoba; 2019. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1993/34449.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Koleini N. The role of high molecular weight fibroblast growth factor-2 in cardiac response to injury. [Thesis]. University of Manitoba; 2019. Available from: http://hdl.handle.net/1993/34449

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

26. Utley, Sarah. The role of fibroblast growth factor signaling on postnatal hepatic progenitor cell expansion.

Degree: PhD, Systems Biology and Disease, 2015, University of Southern California

Fibroblast Growth Factor (FGF) signaling is an established regulator of endoderm specification to the hepatic fate. We have previously determined that Fibroblast Growth Factors (FGFs)… (more)

Subjects/Keywords: fibroblast growth factor; hepatic progenitor cells; beta-catenin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Utley, S. (2015). The role of fibroblast growth factor signaling on postnatal hepatic progenitor cell expansion. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/285763/rec/7223

Chicago Manual of Style (16th Edition):

Utley, Sarah. “The role of fibroblast growth factor signaling on postnatal hepatic progenitor cell expansion.” 2015. Doctoral Dissertation, University of Southern California. Accessed April 18, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/285763/rec/7223.

MLA Handbook (7th Edition):

Utley, Sarah. “The role of fibroblast growth factor signaling on postnatal hepatic progenitor cell expansion.” 2015. Web. 18 Apr 2021.

Vancouver:

Utley S. The role of fibroblast growth factor signaling on postnatal hepatic progenitor cell expansion. [Internet] [Doctoral dissertation]. University of Southern California; 2015. [cited 2021 Apr 18]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/285763/rec/7223.

Council of Science Editors:

Utley S. The role of fibroblast growth factor signaling on postnatal hepatic progenitor cell expansion. [Doctoral Dissertation]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/285763/rec/7223


University of Melbourne

27. Tan, Sven Jean. Phosphate handling in kidney disease: the role of klotho and fibroblast growth factor-23.

Degree: 2016, University of Melbourne

 Derangement in phosphate control has long been considered central to the pathophysiological processes leading to chronic kidney disease-mineral bone disorder (CKD-MBD). Discovery of two phosphate… (more)

Subjects/Keywords: klotho; fibroblast growth factor-23; phosphate; kidney disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tan, S. J. (2016). Phosphate handling in kidney disease: the role of klotho and fibroblast growth factor-23. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/123415

Chicago Manual of Style (16th Edition):

Tan, Sven Jean. “Phosphate handling in kidney disease: the role of klotho and fibroblast growth factor-23.” 2016. Doctoral Dissertation, University of Melbourne. Accessed April 18, 2021. http://hdl.handle.net/11343/123415.

MLA Handbook (7th Edition):

Tan, Sven Jean. “Phosphate handling in kidney disease: the role of klotho and fibroblast growth factor-23.” 2016. Web. 18 Apr 2021.

Vancouver:

Tan SJ. Phosphate handling in kidney disease: the role of klotho and fibroblast growth factor-23. [Internet] [Doctoral dissertation]. University of Melbourne; 2016. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/11343/123415.

Council of Science Editors:

Tan SJ. Phosphate handling in kidney disease: the role of klotho and fibroblast growth factor-23. [Doctoral Dissertation]. University of Melbourne; 2016. Available from: http://hdl.handle.net/11343/123415


University of Georgia

28. Esposito, Tina. Placentation in the bovine with specific emphasis on the role of the trophoblast cells and placental abnormalities associated with the nuclear transfer process.

Degree: 2014, University of Georgia

 Placental abnormalities are proving to be a formidable obstacle to overcome in the nuclear transfer process. In the bovine, it is the trophoblast cells which… (more)

Subjects/Keywords: Placenta; Bovine; Trophoblast; Fibroblast growth factor-4; Nuclear transfer; Feeder layers

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APA (6th Edition):

Esposito, T. (2014). Placentation in the bovine with specific emphasis on the role of the trophoblast cells and placental abnormalities associated with the nuclear transfer process. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/20539

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Esposito, Tina. “Placentation in the bovine with specific emphasis on the role of the trophoblast cells and placental abnormalities associated with the nuclear transfer process.” 2014. Thesis, University of Georgia. Accessed April 18, 2021. http://hdl.handle.net/10724/20539.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Esposito, Tina. “Placentation in the bovine with specific emphasis on the role of the trophoblast cells and placental abnormalities associated with the nuclear transfer process.” 2014. Web. 18 Apr 2021.

Vancouver:

Esposito T. Placentation in the bovine with specific emphasis on the role of the trophoblast cells and placental abnormalities associated with the nuclear transfer process. [Internet] [Thesis]. University of Georgia; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10724/20539.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Esposito T. Placentation in the bovine with specific emphasis on the role of the trophoblast cells and placental abnormalities associated with the nuclear transfer process. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/20539

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

29. Damico, Carmen Marie. A Microfludic Assay Device for Study of Cell Migration on ECM-mimicking Suspended Nanofibers in Presence of Biochemical Cues.

Degree: MS, Mechanical Engineering, 2016, Virginia Tech

 Eukaryotic cell chemotaxis, or directed cell migration in response to a chemoeffector gradient, plays a central role in many important biological process such as wound… (more)

Subjects/Keywords: mesenchymal chemotaxis; biophysical-biochemical cues; fibroblast; platelet-derived growth factor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Damico, C. M. (2016). A Microfludic Assay Device for Study of Cell Migration on ECM-mimicking Suspended Nanofibers in Presence of Biochemical Cues. (Masters Thesis). Virginia Tech. Retrieved from http://hdl.handle.net/10919/82003

Chicago Manual of Style (16th Edition):

Damico, Carmen Marie. “A Microfludic Assay Device for Study of Cell Migration on ECM-mimicking Suspended Nanofibers in Presence of Biochemical Cues.” 2016. Masters Thesis, Virginia Tech. Accessed April 18, 2021. http://hdl.handle.net/10919/82003.

MLA Handbook (7th Edition):

Damico, Carmen Marie. “A Microfludic Assay Device for Study of Cell Migration on ECM-mimicking Suspended Nanofibers in Presence of Biochemical Cues.” 2016. Web. 18 Apr 2021.

Vancouver:

Damico CM. A Microfludic Assay Device for Study of Cell Migration on ECM-mimicking Suspended Nanofibers in Presence of Biochemical Cues. [Internet] [Masters thesis]. Virginia Tech; 2016. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10919/82003.

Council of Science Editors:

Damico CM. A Microfludic Assay Device for Study of Cell Migration on ECM-mimicking Suspended Nanofibers in Presence of Biochemical Cues. [Masters Thesis]. Virginia Tech; 2016. Available from: http://hdl.handle.net/10919/82003


Hong Kong University of Science and Technology

30. Kwong, Wai Yeung. Novel recombinant DNA approaches engineered for the production of commercially valuable proteins.

Degree: 2013, Hong Kong University of Science and Technology

 Extracellular production of recombinant proteins using bacterial systems may enable the proteins concerned to retain important properties including structural authenticity and functional activity. By employing… (more)

Subjects/Keywords: Bacillus subtilis ; Escherichia coli ; Secretion ; Fibroblast growth factors ; Epidermal growth factor ; Recombinant proteins ; Therapeutic use

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kwong, W. Y. (2013). Novel recombinant DNA approaches engineered for the production of commercially valuable proteins. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-80935 ; https://doi.org/10.14711/thesis-b1254355 ; http://repository.ust.hk/ir/bitstream/1783.1-80935/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kwong, Wai Yeung. “Novel recombinant DNA approaches engineered for the production of commercially valuable proteins.” 2013. Thesis, Hong Kong University of Science and Technology. Accessed April 18, 2021. http://repository.ust.hk/ir/Record/1783.1-80935 ; https://doi.org/10.14711/thesis-b1254355 ; http://repository.ust.hk/ir/bitstream/1783.1-80935/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kwong, Wai Yeung. “Novel recombinant DNA approaches engineered for the production of commercially valuable proteins.” 2013. Web. 18 Apr 2021.

Vancouver:

Kwong WY. Novel recombinant DNA approaches engineered for the production of commercially valuable proteins. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2013. [cited 2021 Apr 18]. Available from: http://repository.ust.hk/ir/Record/1783.1-80935 ; https://doi.org/10.14711/thesis-b1254355 ; http://repository.ust.hk/ir/bitstream/1783.1-80935/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kwong WY. Novel recombinant DNA approaches engineered for the production of commercially valuable proteins. [Thesis]. Hong Kong University of Science and Technology; 2013. Available from: http://repository.ust.hk/ir/Record/1783.1-80935 ; https://doi.org/10.14711/thesis-b1254355 ; http://repository.ust.hk/ir/bitstream/1783.1-80935/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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