subject:(FERM)

1. 高井, 友美子. 多様な分子認識に関与するラデキシンFERMドメインの相互作用解析 : Biophysical analyses of the interactions of the radixin FERM domain with CD43 and other intracellular proteins; タヨウナブンシニンシキニカンヨスルラデキシン FERM ドメインノソウゴサヨウカイセキ.

Degree: Nara Institute of Science and Technology / 奈良先端科学技術大学院大学

URL: http://hdl.handle.net/10061/1575

Subjects/Keywords: FERM domain

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APA (6^th Edition):

高井, �. (n.d.). 多様な分子認識に関与するラデキシンFERMドメインの相互作用解析 : Biophysical analyses of the interactions of the radixin FERM domain with CD43 and other intracellular proteins; タヨウナブンシニンシキニカンヨスルラデキシン FERM ドメインノソウゴサヨウカイセキ. (Thesis). Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Retrieved from http://hdl.handle.net/10061/1575

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

高井, 友美子. “多様な分子認識に関与するラデキシンFERMドメインの相互作用解析 : Biophysical analyses of the interactions of the radixin FERM domain with CD43 and other intracellular proteins; タヨウナブンシニンシキニカンヨスルラデキシン FERM ドメインノソウゴサヨウカイセキ.” Thesis, Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Accessed January 19, 2021. http://hdl.handle.net/10061/1575.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

高井, 友美子. “多様な分子認識に関与するラデキシンFERMドメインの相互作用解析 : Biophysical analyses of the interactions of the radixin FERM domain with CD43 and other intracellular proteins; タヨウナブンシニンシキニカンヨスルラデキシン FERM ドメインノソウゴサヨウカイセキ.” Web. 19 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

高井 �. 多様な分子認識に関与するラデキシンFERMドメインの相互作用解析 : Biophysical analyses of the interactions of the radixin FERM domain with CD43 and other intracellular proteins; タヨウナブンシニンシキニカンヨスルラデキシン FERM ドメインノソウゴサヨウカイセキ. [Internet] [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10061/1575.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

高井 �. 多様な分子認識に関与するラデキシンFERMドメインの相互作用解析 : Biophysical analyses of the interactions of the radixin FERM domain with CD43 and other intracellular proteins; タヨウナブンシニンシキニカンヨスルラデキシン FERM ドメインノソウゴサヨウカイセキ. [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; Available from: http://hdl.handle.net/10061/1575

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

University of Toronto

2. Draginov, Arman George. Okapi (Oka), a Novel FERM Domain Protein Essential for Drosophila Oogenesis.

Degree: 2014, University of Toronto

URL: http://hdl.handle.net/1807/82605

►

FERM proteins are a class of important adaptors in animal tissues. They are ubiquitous in the cytocortex, linking the plasma membrane to cytoskeletal components, and… (more)

Subjects/Keywords: Crumbs; Drosophila; Epithelia; FERM; Oogenesis; Ovary; 0307

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APA (6^th Edition):

Draginov, A. G. (2014). Okapi (Oka), a Novel FERM Domain Protein Essential for Drosophila Oogenesis. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/82605

Chicago Manual of Style (16^th Edition):

Draginov, Arman George. “Okapi (Oka), a Novel FERM Domain Protein Essential for Drosophila Oogenesis.” 2014. Masters Thesis, University of Toronto. Accessed January 19, 2021. http://hdl.handle.net/1807/82605.

MLA Handbook (7^th Edition):

Draginov, Arman George. “Okapi (Oka), a Novel FERM Domain Protein Essential for Drosophila Oogenesis.” 2014. Web. 19 Jan 2021.

Vancouver:

Draginov AG. Okapi (Oka), a Novel FERM Domain Protein Essential for Drosophila Oogenesis. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1807/82605.

Council of Science Editors:

Draginov AG. Okapi (Oka), a Novel FERM Domain Protein Essential for Drosophila Oogenesis. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/82605

University of Minnesota

3. Petersen, Karl. Structure-Function Analysis of Motor Proteins: Insights from Conventional and Unconventional Myosins.

Degree: PhD, Biochemistry, Molecular Bio, and Biophysics, 2016, University of Minnesota

URL: http://hdl.handle.net/11299/191470

► Myosin motor proteins play fundamental roles in a multitude of cellular processes. Myosin generates force on cytoskeletal actin filaments to control cell shape, most dramatically… (more)

▼ Myosin motor proteins play fundamental roles in a multitude of cellular processes. Myosin generates force on cytoskeletal actin filaments to control cell shape, most dramatically during cytokinesis, and has a conserved role in defining cell polarity. Myosin contracts the actin cytoskeleton, ensuring prompt turnover of cellular adhesion sites, retracting the cell body during migration and development, and contracting muscle among diverse other functions. How myosins work, and why force generation is essential for their function, is in many cases an open question. Chapter 2 presents a structure-function analysis of the amoebozoan myosin 7 (DdMyo7) in live Dictyostelium discoideum cells. DdMyo7 bears structural resemblance to human Myosin 7 (a protein involved in maintenance of the retina, stereocilia of the ear, and gut microvilli) but has functional similarity to human Myosin 10, a regulator of cell adhesion that is also essential in formation of actin-based structures called filopodia. Phylogenetic analysis of these related proteins shows that DdMyo7 is not directly related to any human myosin but rather represents a molecular ancestor of several vertebrate myosins (Myo7, Myo10 and Myo15). Functional analysis focused on rescue of myo7– cells. The two MyTH4-FERM domains were fully redundant in rescuing formation of filopodia. A conserved Myo7 regulatory motif in the C-terminal FERM domain was found to stimulate filopodia formation when mutated, establishing DdMyo7 as a filopodial motor with features of Myo7 and Myo10. A molecular chimera of DdMyo7 motor/lever arm region fused to the MF domain of human Myo10 partially rescued filopodia formation, suggesting the MF domain plays a similar role in filopodia in divergent organisms. Structural information must be combined with physiological data to understand the mechanism of myosin motor function. Structural studies have long focused on conventional myosin 2 as a model due to ease of protein expression and purification. This approach has yielded considerable data regarding the static structures and in vitro kinetics of the myosin mechanochemical cycle; however, high-resolution methods to observe the dynamics of myosin activation in cells have been lacking. Chapter 4 introduces methods and instrumentation for rapid, precise measurement of fluorescence lifetime. This is a necessary step toward Myo2-based live cell FRET sensors described in Chapter 5. Implications of this work for future studies of myosin physiological function are discussed in Chapter 6.

Subjects/Keywords: actin; cell motility; filopodia; myosin; MyTH4-FERM

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Petersen, K. (2016). Structure-Function Analysis of Motor Proteins: Insights from Conventional and Unconventional Myosins. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/191470

Chicago Manual of Style (16^th Edition):

Petersen, Karl. “Structure-Function Analysis of Motor Proteins: Insights from Conventional and Unconventional Myosins.” 2016. Doctoral Dissertation, University of Minnesota. Accessed January 19, 2021. http://hdl.handle.net/11299/191470.

MLA Handbook (7^th Edition):

Petersen, Karl. “Structure-Function Analysis of Motor Proteins: Insights from Conventional and Unconventional Myosins.” 2016. Web. 19 Jan 2021.

Vancouver:

Petersen K. Structure-Function Analysis of Motor Proteins: Insights from Conventional and Unconventional Myosins. [Internet] [Doctoral dissertation]. University of Minnesota; 2016. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/11299/191470.

Council of Science Editors:

Petersen K. Structure-Function Analysis of Motor Proteins: Insights from Conventional and Unconventional Myosins. [Doctoral Dissertation]. University of Minnesota; 2016. Available from: http://hdl.handle.net/11299/191470

4. Springer, Courtney. Hydroponics and the need for continuous nutrient monitoring .

Degree: 2013, California State University – San Marcos

URL: http://hdl.handle.net/10211.8/537

► This marketing research project was conducted on behalf of Seacoast Science. Seacoast Science is a small, independent, growing company located in Carlsbad, California. Seacoast specializes… (more)

Subjects/Keywords: Hydroponics; Continuous Nutrient Monitoring; FERM Sys

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APA (6^th Edition):

Springer, C. (2013). Hydroponics and the need for continuous nutrient monitoring . (Thesis). California State University – San Marcos. Retrieved from http://hdl.handle.net/10211.8/537

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Springer, Courtney. “Hydroponics and the need for continuous nutrient monitoring .” 2013. Thesis, California State University – San Marcos. Accessed January 19, 2021. http://hdl.handle.net/10211.8/537.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Springer, Courtney. “Hydroponics and the need for continuous nutrient monitoring .” 2013. Web. 19 Jan 2021.

Vancouver:

Springer C. Hydroponics and the need for continuous nutrient monitoring . [Internet] [Thesis]. California State University – San Marcos; 2013. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10211.8/537.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Springer C. Hydroponics and the need for continuous nutrient monitoring . [Thesis]. California State University – San Marcos; 2013. Available from: http://hdl.handle.net/10211.8/537

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of Minnesota

5. Arthur, Ashley. MyTH4-FERM myosin based filopodia initiation.

Degree: PhD, Molecular, Cellular, Developmental Biology and Genetics, 2020, University of Minnesota

URL: http://hdl.handle.net/11299/216420

► Filopodia are thin actin-based structures that cells use to interact with their environments. Filopodia initiation requires a suite of conserved proteins but the mechanism remains… (more)

Subjects/Keywords: actin; filopodia; myosin; MyTH4-FERM; VASP

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Arthur, A. (2020). MyTH4-FERM myosin based filopodia initiation. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/216420

Chicago Manual of Style (16^th Edition):

Arthur, Ashley. “MyTH4-FERM myosin based filopodia initiation.” 2020. Doctoral Dissertation, University of Minnesota. Accessed January 19, 2021. http://hdl.handle.net/11299/216420.

MLA Handbook (7^th Edition):

Arthur, Ashley. “MyTH4-FERM myosin based filopodia initiation.” 2020. Web. 19 Jan 2021.

Vancouver:

Arthur A. MyTH4-FERM myosin based filopodia initiation. [Internet] [Doctoral dissertation]. University of Minnesota; 2020. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/11299/216420.

Council of Science Editors:

Arthur A. MyTH4-FERM myosin based filopodia initiation. [Doctoral Dissertation]. University of Minnesota; 2020. Available from: http://hdl.handle.net/11299/216420

Université de Grenoble

6. Habourdin, Clemence. Caractérisation des membres du clan arrestine chez l'amibe Dictyostelium discoideum : Etude de la protéine AdcA et de son partenaire FrmC : Characterization of arrestin clan members in Dictyostelium discoideum : study of the protein AdcA and its partner FrmC.

Degree: Docteur es, Biologie cellulaire, 2014, Université de Grenoble

URL: http://www.theses.fr/2014GRENV074

►

Le clan « arrestine » est une superfamille de protéines adaptatrices regroupant les arrestines conventionnelles (β-arrestines et arrestines visuelles), bien décrites pour leur rôle dans… (more)

▼

Le clan « arrestine » est une superfamille de protéines adaptatrices regroupant les arrestines conventionnelles (β-arrestines et arrestines visuelles), bien décrites pour leur rôle dans la régulation des récepteurs membranaires couplés aux protéines G hétéro-trimériques et la signalisation associée et des protéines de type arrestin-like identifiées plus récemment, qui semblent partager des fonctions communes dans la régulation et le trafic de cargos membranaires. Ce travail de thèse a porté sur l'étude de la protéine arrestin-like AdcA de Dictyostelium discoideum dans l'objectif d'en définir le rôle fonctionnel. En plus de son module arrestine, caractéristique de cette famille de protéines, AdcA possède un domaine FYVE qui permet son association à la voie endocytaire, un domaine C-terminal riche en tyrosines et un domaine N-terminal riche en histidines impliqué dans son oligomérisation. Mes travaux ont permis d'établir que la protéine AdcA répond à des stress de natures diverses dont l'hyper-osmolarité, par une multi-phosphorylation massive, notamment au niveau du domaine N-terminal. La sensibilité d'AdcA à la dépolymérisation du réseau d'actine suggère un lien fonctionnel entre le cytosquelette d'actine et la cascade de signalisation menant à la phosphorylation de la protéine. En conditions de stress modéré, la phosphorylation d'AdcA est transitoire et sa déphosphorylation, en partie dépendante du facteur de transcription STATc, corrèle avec l'adaptation des cellules aux conditions de stress. Cette modification post-traductionnelle transitoire pourrait permettre de contrôler l'activité d'AdcA et d'optimiser la réponse des cellules au stress. Parallèlement, la caractérisation fonctionnelle d'un partenaire d'AdcA, la protéine FrmC, a été entreprise. FrmC est une nouvelle protéine, encore non caractérisée, présentant plusieurs domaines fonctionnels dont un domaine FERM capable de lier l'actine in vitro et un domaine à motifs LRR. Mes travaux ont notamment mis en évidence que FrmC est recrutée à la membrane plasmique et joue un rôle dans l'adhésion cellulaire. Par ailleurs, l'absence de FrmC affecte l'adaptation des cellules et la réponse d'AdcA en situation de stress hyper-osmotique.

The arrestin clan represents a large group of adaptor proteins which includes the canonical arrestins - β-arrestins and visual arrestins – well described for their role in the regulation of membrane receptors coupled to heterotrimeric G-proteins and associated signaling as well as arrestin-like proteins identified more recently that seem to share functions in the regulation and trafficking of membrane cargoes. This work focused on the study of the arrestin-like protein AdcA of Dictyostelium discoideum, to determine the functional role of this atypical member. In addition to the arrestin module common to all the members of the arrestin family, AdcA harbors a FYVE domain responsible for its association to the endocytic pathway, a C-terminal tyrosine-rich domain and an N-terminal extension rich in histidine residues mediating its…

Advisors/Committee Members: Aubry, Laurence (thesis director).

Subjects/Keywords: Arrestines; Stress hyper-osmotique; Domaine FERM; Adhésion; AdcA; Dictyostelium; Arrestin; Hyeprosmotic stress; FERM domain; Adhesion; AdcA; Dictyostelium; 570

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APA (6^th Edition):

Habourdin, C. (2014). Caractérisation des membres du clan arrestine chez l'amibe Dictyostelium discoideum : Etude de la protéine AdcA et de son partenaire FrmC : Characterization of arrestin clan members in Dictyostelium discoideum : study of the protein AdcA and its partner FrmC. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2014GRENV074

Chicago Manual of Style (16^th Edition):

Habourdin, Clemence. “Caractérisation des membres du clan arrestine chez l'amibe Dictyostelium discoideum : Etude de la protéine AdcA et de son partenaire FrmC : Characterization of arrestin clan members in Dictyostelium discoideum : study of the protein AdcA and its partner FrmC.” 2014. Doctoral Dissertation, Université de Grenoble. Accessed January 19, 2021. http://www.theses.fr/2014GRENV074.

MLA Handbook (7^th Edition):

Habourdin, Clemence. “Caractérisation des membres du clan arrestine chez l'amibe Dictyostelium discoideum : Etude de la protéine AdcA et de son partenaire FrmC : Characterization of arrestin clan members in Dictyostelium discoideum : study of the protein AdcA and its partner FrmC.” 2014. Web. 19 Jan 2021.

Vancouver:

Habourdin C. Caractérisation des membres du clan arrestine chez l'amibe Dictyostelium discoideum : Etude de la protéine AdcA et de son partenaire FrmC : Characterization of arrestin clan members in Dictyostelium discoideum : study of the protein AdcA and its partner FrmC. [Internet] [Doctoral dissertation]. Université de Grenoble; 2014. [cited 2021 Jan 19]. Available from: http://www.theses.fr/2014GRENV074.

Council of Science Editors:

Habourdin C. Caractérisation des membres du clan arrestine chez l'amibe Dictyostelium discoideum : Etude de la protéine AdcA et de son partenaire FrmC : Characterization of arrestin clan members in Dictyostelium discoideum : study of the protein AdcA and its partner FrmC. [Doctoral Dissertation]. Université de Grenoble; 2014. Available from: http://www.theses.fr/2014GRENV074

Vanderbilt University

7. Weck, Meredith Lynn. Mechanisms of myosin-7b function in brush border assembly.

Degree: PhD, Cell and Developmental Biology, 2018, Vanderbilt University

URL: http://hdl.handle.net/1803/11461

► The intestinal brush border serves as the sole site of nutrient absorption within the body, and also acts as an important barrier against luminal pathogens.… (more)

▼ The intestinal brush border serves as the sole site of nutrient absorption within the body, and also acts as an important barrier against luminal pathogens. The brush border is comprised of membrane protrusions called microvilli that are found on the apical surface of enterocytes. These protrusions are supported by a core bundle of 20 to 30 parallel actin filaments with the plus ends oriented towards the lumen. Our laboratory showed that protocadherin-24 and mucin-like protocadherin play a key role in the assembly and organization of the brush border. These protocadherins interact to form a trans-heterophilic adhesion complex that physically connects the distal tips of microvilli and regulates the tight packing. How these complexes are targeted to microvillar tips remains unknown. Microvillar protocadherins interact with several cytoplasmic binding partners, including the actin based motor, myosin-7b (Myo7b). Given the role of other myosin motors in building actin-based protrusions, this work focuses on elucidating the function of Myo7b in brush border assembly. We show that Myo7b is highly enriched at the tips of microvilli in both kidney and intestinal brush borders. Loss of Myo7b results in the mislocalization of the intermicrovillar adhesion links along the microvillar axis, disrupting brush border assembly. We also found that Myo7b motor domains are capable of supporting tip-directed transport. However, motor activity is supplemented by other passive targeting mechanisms, which together drive highly efficient accumulation of the adhesion complex at the tips. Additionally, we have established an in-cell reconstitution assay using filopodial protrusions that can be used to study protein interactions within the adhesion complex. Using this assay, we have begun to characterize the morphological effects of tip-enriched adhesion on actin-based protrusions. Initial experiments suggest adhesion at the distal tips regulates the stability and length of these protrusions. This work provides evidence that the actin-based motor Myo7b promotes the accumulation of adhesion complexes at microvillar tips, which may alter the morphology and dynamics of actin-based protrusions. These findings hold important implications for understanding apical morphogenesis in transporting and sensory epithelial tissues. Advisors/Committee Members: Dylan T. Burnette, Ph.D. (committee member), Matthew J. Lang, Ph.D. (committee member), Ryoma Ohi, Ph.D. (committee member), Matthew J. Tyska, Ph.D. (committee member), Robert J. Coffey, M.D. (Committee Chair).

Subjects/Keywords: brush border; filopodia; intermicrovillar adhesion complex; MyTH4-FERM

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APA (6^th Edition):

Weck, M. L. (2018). Mechanisms of myosin-7b function in brush border assembly. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11461

Chicago Manual of Style (16^th Edition):

Weck, Meredith Lynn. “Mechanisms of myosin-7b function in brush border assembly.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed January 19, 2021. http://hdl.handle.net/1803/11461.

MLA Handbook (7^th Edition):

Weck, Meredith Lynn. “Mechanisms of myosin-7b function in brush border assembly.” 2018. Web. 19 Jan 2021.

Vancouver:

Weck ML. Mechanisms of myosin-7b function in brush border assembly. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1803/11461.

Council of Science Editors:

Weck ML. Mechanisms of myosin-7b function in brush border assembly. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/11461

Iowa State University

8. Loving, Hanna S. Molecular mechanisms of Pyk2 regulation.

Degree: 2020, Iowa State University

URL: https://lib.dr.iastate.edu/etd/17897

► Proline-rich tyrosine kinase 2 also known as Pyk2 is a non-receptor tyrosine kinase that functions various signaling pathways such as cell proliferation and synaptic plasticity.… (more)

▼ Proline-rich tyrosine kinase 2 also known as Pyk2 is a non-receptor tyrosine kinase that functions various signaling pathways such as cell proliferation and synaptic plasticity. Pyk2 expression is predominant in hematopoietic, neuronal, and epithelial cells. Pyk2 is a FAK homolog and functions like FAK in many cell types. However, Pyk2 adopts unique Ca2+-responsiveness in neuronal cells. This research thesis will focus on the structural determination of how purified Pyk2 is auto inhibited and the role a scaffold plays in the activation of Pyk2 at the molecular level. In this thesis, I show that unstimulated Pyk2 is auto-inhibited, and this auto-inhibition is mediated by its N-terminal domain. The N-terminal FERM domain of Pyk2 interacts with the kinase domain resulting in inhibition of kinase activity. Kinase inhibition is due to steric restriction of active site access due to the conformation of the FERM domain. Using a combination of hydrogen/deuterium exchange mass spectrometry (HDX-MS) and site-directed mutagenesis, I mapped functionally important intra-domain interfaces that control the activity and allosteric landscape of Pyk2. Together, these studies underlie the first structural model of the auto-inhibited conformational of Pyk2. This thesis also explores the role of scaffold-induced clustering in Pyk2 activation. Although it is well-established that Ca2+ regulates Pyk2 activation, little is known regarding the molecular mechanisms. I determined that PSD-95 mediates Pyk2 activation through a clustering-based mechanism. PSD-95 binds to auto-inhibited Pyk2 via the first two PDZ domains. HDX-MS implicated the Pyk2 F3 FERM subdomain and kinase C-lobe as the primary scaffolding interfaces. Kinase activity profiling indicates that PSD-95 mediated activation is based on a clustering mechanism. Altogether, using biochemical and biophysical methods, the research reported here determined the auto-inhibited conformation of Pyk2 and elucidated the role of scaffolding in the activation mechanism.

Subjects/Keywords: FERM-kinase; kinase; post-synaptic density; Pyk2; scaffold; synaptic plasticity

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APA (6^th Edition):

Loving, H. S. (2020). Molecular mechanisms of Pyk2 regulation. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/17897

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Loving, Hanna S. “Molecular mechanisms of Pyk2 regulation.” 2020. Thesis, Iowa State University. Accessed January 19, 2021. https://lib.dr.iastate.edu/etd/17897.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Loving, Hanna S. “Molecular mechanisms of Pyk2 regulation.” 2020. Web. 19 Jan 2021.

Vancouver:

Loving HS. Molecular mechanisms of Pyk2 regulation. [Internet] [Thesis]. Iowa State University; 2020. [cited 2021 Jan 19]. Available from: https://lib.dr.iastate.edu/etd/17897.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Loving HS. Molecular mechanisms of Pyk2 regulation. [Thesis]. Iowa State University; 2020. Available from: https://lib.dr.iastate.edu/etd/17897

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of Toronto

9. Vujadinovic, Stefan. The Okapi (Oka) FERM Domain Protein Regulates Somatic Stem Cell Numbers and Cell Intercalation during Drosophila Oogenesis.

Degree: 2017, University of Toronto

URL: http://hdl.handle.net/1807/97275

►

Okapi (Oka) is a Drosophila melanogaster FERM domain protein that is essential for oogenesis. oka (CG34347) mutants are viable but display defects during oogenesis. Mutant… (more)

Subjects/Keywords: Drosophila melanogaster; FERM Domain Protein; Intercalation; Oogenesis; Proliferation; Stem Cells; 0758

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APA (6^th Edition):

Vujadinovic, S. (2017). The Okapi (Oka) FERM Domain Protein Regulates Somatic Stem Cell Numbers and Cell Intercalation during Drosophila Oogenesis. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/97275

Chicago Manual of Style (16^th Edition):

Vujadinovic, Stefan. “The Okapi (Oka) FERM Domain Protein Regulates Somatic Stem Cell Numbers and Cell Intercalation during Drosophila Oogenesis.” 2017. Masters Thesis, University of Toronto. Accessed January 19, 2021. http://hdl.handle.net/1807/97275.

MLA Handbook (7^th Edition):

Vujadinovic, Stefan. “The Okapi (Oka) FERM Domain Protein Regulates Somatic Stem Cell Numbers and Cell Intercalation during Drosophila Oogenesis.” 2017. Web. 19 Jan 2021.

Vancouver:

Vujadinovic S. The Okapi (Oka) FERM Domain Protein Regulates Somatic Stem Cell Numbers and Cell Intercalation during Drosophila Oogenesis. [Internet] [Masters thesis]. University of Toronto; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1807/97275.

Council of Science Editors:

Vujadinovic S. The Okapi (Oka) FERM Domain Protein Regulates Somatic Stem Cell Numbers and Cell Intercalation during Drosophila Oogenesis. [Masters Thesis]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/97275

10. Salo, Jouni-Junior. Näytönohjainarkkitehtuurit.

Degree: 2019, Theseus

URL: http://www.theseus.fi/handle/10024/161683

►

Tässä opinnäytetyössä on perehdytty Yhdysvaltalaisen grafiikkasuorittimien valmistajan Nvidian historiaan ja tuotteisiin. Nvidia on toinen maailman suurimmasta grafiikkasuorittimien valmistajasta. Tässä työssä tutustutaan tarkemmin Nvidian arkkitehtuureihin, Fermiin,… (more)

Subjects/Keywords: grafiikkasuorittimet; Nvidia; Ferm; Kepler; Maxwell; Pascal; Volta; Turing; GPU

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Salo, J. (2019). Näytönohjainarkkitehtuurit. (Thesis). Theseus. Retrieved from http://www.theseus.fi/handle/10024/161683

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Salo, Jouni-Junior. “Näytönohjainarkkitehtuurit.” 2019. Thesis, Theseus. Accessed January 19, 2021. http://www.theseus.fi/handle/10024/161683.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Salo, Jouni-Junior. “Näytönohjainarkkitehtuurit.” 2019. Web. 19 Jan 2021.

Vancouver:

Salo J. Näytönohjainarkkitehtuurit. [Internet] [Thesis]. Theseus; 2019. [cited 2021 Jan 19]. Available from: http://www.theseus.fi/handle/10024/161683.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Salo J. Näytönohjainarkkitehtuurit. [Thesis]. Theseus; 2019. Available from: http://www.theseus.fi/handle/10024/161683

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Vanderbilt University

11. Elliott, Natalina Elizabeth. The role of activating mutations in the FERM domain of janus kinase 3 in the development of adult t-cell leukemia/lymphoma.

Degree: PhD, Cancer Biology, 2013, Vanderbilt University

URL: http://hdl.handle.net/1803/10404

► Adult T-cell leukemia/lymphoma (ATLL) is an incurable peripheral T-cell malignancy where most patients succumb within the first year of diagnosis. Development of ATLL requires human… (more)

▼ Adult T-cell leukemia/lymphoma (ATLL) is an incurable peripheral T-cell malignancy where most patients succumb within the first year of diagnosis. Development of ATLL requires human T-cell lymphotrophic virus type 1 (HTLV-I) retroviral infection followed by accumulation of somatic mutations and changes in gene expression. The discovery of genes and pathways involved in the initiation of ATLL may provide novel therapeutic targets for treatment of this fatal disease. Since the IL-2 signaling pathway plays an important role in ATLL development, mutational analysis of IL-2 signaling pathway components should yield a better understanding of disease progression and outcome. Janus kinase 3 (JAK3), a nonreceptor tyrosine kinase is a key kinase upstream in the IL-2 signaling pathway. Activating somatic mutations in JAK3 have been described in leukemias and lymphomas including acute megakaryoblastic leukemia and natural killer/T-cell lymphoma. Three mutations in the regulatory FERM domain of JAK3 were identified in four of the thirty-six ATLL patients screened and no mutations were found in the twenty-four screened ethnically matched controls. These somatic, missense mutations occurred in the amino terminal regulatory FERM domain. JAK3 FERM domain may play an autoregulatory role by inhibiting kinase activity in the absence of IL-2. In cell culture assays all three FERM domain mutations induce gain of function in JAK3 and can be inhibited with a JAK specific kinase inhibitor, tofacitinib. One of the JAK3 FERM domain mutations (E183G) was characterized in vivo and found to be oncogenic in cooperation with the loss of cell cycle regulatory proteins p16(INK4a) and p14(ARF). These findings emphasize the importance of JAK3 activation in ATLL development and offer a novel therapeutic target for this incurable disease. Advisors/Committee Members: Utpal Davé MD (committee member), Scott Hiebert PhD (committee member), William Pao MD PhD (committee member), Stephen Brandt MD (Committee Chair).

Subjects/Keywords: adult t-cell leukemia/lymphoma; Leukemia; FERM; JAK3; ATLL; HTLV-1; IL-2; mutation; t-cell

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APA (6^th Edition):

Elliott, N. E. (2013). The role of activating mutations in the FERM domain of janus kinase 3 in the development of adult t-cell leukemia/lymphoma. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10404

Chicago Manual of Style (16^th Edition):

Elliott, Natalina Elizabeth. “The role of activating mutations in the FERM domain of janus kinase 3 in the development of adult t-cell leukemia/lymphoma.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed January 19, 2021. http://hdl.handle.net/1803/10404.

MLA Handbook (7^th Edition):

Elliott, Natalina Elizabeth. “The role of activating mutations in the FERM domain of janus kinase 3 in the development of adult t-cell leukemia/lymphoma.” 2013. Web. 19 Jan 2021.

Vancouver:

Elliott NE. The role of activating mutations in the FERM domain of janus kinase 3 in the development of adult t-cell leukemia/lymphoma. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1803/10404.

Council of Science Editors:

Elliott NE. The role of activating mutations in the FERM domain of janus kinase 3 in the development of adult t-cell leukemia/lymphoma. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/10404

University of St. Andrews

12. Herron, Lissa Rocha. A study of the behaviour and interactions of the novel FERM protein Willin .

Degree: 2008, University of St. Andrews

URL: http://hdl.handle.net/10023/418

► Willin is a novel member of the Four-point-one Ezrin Radixin Moesin (FERM) protein superfamily, containing an N-terminal FERM domain most like the Ezrin-Radixin-Moesin (ERM) family… (more)

Subjects/Keywords: Willin; FERM; Merlin; Ezrin; Radixin; Moesin; Neurofascin; L1

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APA (6^th Edition):

Herron, L. R. (2008). A study of the behaviour and interactions of the novel FERM protein Willin . (Thesis). University of St. Andrews. Retrieved from http://hdl.handle.net/10023/418

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Herron, Lissa Rocha. “A study of the behaviour and interactions of the novel FERM protein Willin .” 2008. Thesis, University of St. Andrews. Accessed January 19, 2021. http://hdl.handle.net/10023/418.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Herron, Lissa Rocha. “A study of the behaviour and interactions of the novel FERM protein Willin .” 2008. Web. 19 Jan 2021.

Vancouver:

Herron LR. A study of the behaviour and interactions of the novel FERM protein Willin . [Internet] [Thesis]. University of St. Andrews; 2008. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10023/418.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Herron LR. A study of the behaviour and interactions of the novel FERM protein Willin . [Thesis]. University of St. Andrews; 2008. Available from: http://hdl.handle.net/10023/418

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Texas Medical Center

13. Dimarco Hewitt, Patty. Regulation of Cell Adhesion by the FERM Proteins, PTPN14 and Merlin.

Degree: PhD, 2015, Texas Medical Center

URL: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/588

► Cell-cell adhesion is critical for the control of tissue organization and homeostasis. A family of proteins that regulate cell-cell adhesions is the FERM (4.1… (more)

▼ Cell-cell adhesion is critical for the control of tissue organization and homeostasis. A family of proteins that regulate cell-cell adhesions is the FERM (4.1 protein, Ezrin, Radixin, Moesin) domain-containing proteins.One FERM domain protein, the non-receptor tyrosine phosphatase PTPN14, is mutated or deleted in several human cancers suggesting that it may be involved in tumor development and/or progression. Additionally, the loss of the FERM domain protein Merlin is associated with tumor development and metastasis.Both PTPN14 and Merlin have been shown to localize and possibly regulate adherens junction (AJ) functions. This work sought to determine if PTPN14 is required for the establishment of epithelial adhesion and polarity. Using mammalian epithelial cells as a model to test these hypotheses, we down-regulated the tyrosine phosphatase, PTPN14. Decreased levels of PTPN14 increased E-cadherin membrane movement at cell contacts, disrupting cell contacts and abolishing E-cadherin localization during primordial lumen formation. In cells with established AJs and TJs, decreased levels of PTPN14 led to an increase in β-catenin Y654 phosphorylation, promoting increased membrane mobility of E-cadherin and subsequent destabilization of cell contacts. This was dependent on PTPN14 phosphatase activity at b-cateninY654 opposing Src-mediated disruption of AJs. In cells that lacked PTPN14, for an extended time, I observed alterations in the morphology of the epithelial cells to a spindle morphologycoupled with increased cell migration,transcriptional repression of E-cadherin and activation of Slug. Similar to the regulation of the AJ by PTPN14, we examined another FERM domain protein, Merlin, and found that specific post-translational modifications in its unique N-terminal region regulate distinct AJ-mediated activities. Mutation of a single serine residue in the N-terminal region of Merlin blocked Merlin loading onto the AJ during early cell-cell adhesion formation. Disruption of Merlin AJ loading leads to actin disorganization and loss of contact inhibition of proliferation. Additionally, I found that phosphorylation of a second site in the N-terminal region of Merlin affects the release of the apical proteins, atypical protein kinase C (aPKC) and partitioning defective 3 (Par3), to the apical membrane for the development of functional TJs. Blocking this specific phosphorylation site disrupts the formation of functional polarized epithelia. From this, we see that distinct post-translational modifications of Merlin can alter its interaction with polarity proteins and thus Merlin functionin regulating epithelial cell adhesion and polarity. Overall, these results indicate that mutation or post-translation regulation of FERM proteins could promote increased migration and invasion in cells altering normal epithelial development possibly leading to loss of tissue function and disease. Advisors/Committee Members: Andrew Gladden, Ph.D., Russell Broaddus, M.D., Ph.D., Michael Galko, Ph.D..

Subjects/Keywords: PTPN14; Merlin; FERM; epithelial adhesion; polarity; adherens and tight junctions; aPKC; Biochemistry; Cancer Biology; Cell Biology; Medicine and Health Sciences; Molecular Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Dimarco Hewitt, P. (2015). Regulation of Cell Adhesion by the FERM Proteins, PTPN14 and Merlin. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/588

Chicago Manual of Style (16^th Edition):

Dimarco Hewitt, Patty. “Regulation of Cell Adhesion by the FERM Proteins, PTPN14 and Merlin.” 2015. Doctoral Dissertation, Texas Medical Center. Accessed January 19, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/588.

MLA Handbook (7^th Edition):

Dimarco Hewitt, Patty. “Regulation of Cell Adhesion by the FERM Proteins, PTPN14 and Merlin.” 2015. Web. 19 Jan 2021.

Vancouver:

Dimarco Hewitt P. Regulation of Cell Adhesion by the FERM Proteins, PTPN14 and Merlin. [Internet] [Doctoral dissertation]. Texas Medical Center; 2015. [cited 2021 Jan 19]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/588.

Council of Science Editors:

Dimarco Hewitt P. Regulation of Cell Adhesion by the FERM Proteins, PTPN14 and Merlin. [Doctoral Dissertation]. Texas Medical Center; 2015. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/588

University of Toronto

14. Milic, Milos. Mutational Analysis of FERM Domain Proteins CG34347 and Cdep in Drosophila.

Degree: 2012, University of Toronto

URL: http://hdl.handle.net/1807/32608

►

Crumbs is a transmembrane protein and apical determinant in Drosophila epithelial cells. Its cytoplasmic tail contains a PDZ and a FERM domain-binding site through which… (more)

Subjects/Keywords: Drosophila; Cell Signalling; Crumbs; Polarity; FERM; Cell Signalling; PatJ; Armadillo; Oogenesis; Interfollicular stalk; Notch; Wingless; Hedgehog; Hippo; Follicular epithelium; Cell division; Ovaries; Yurt; 0369; 0307; 0379

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Milic, M. (2012). Mutational Analysis of FERM Domain Proteins CG34347 and Cdep in Drosophila. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/32608

Chicago Manual of Style (16^th Edition):

Milic, Milos. “Mutational Analysis of FERM Domain Proteins CG34347 and Cdep in Drosophila.” 2012. Masters Thesis, University of Toronto. Accessed January 19, 2021. http://hdl.handle.net/1807/32608.

MLA Handbook (7^th Edition):

Milic, Milos. “Mutational Analysis of FERM Domain Proteins CG34347 and Cdep in Drosophila.” 2012. Web. 19 Jan 2021.

Vancouver:

Milic M. Mutational Analysis of FERM Domain Proteins CG34347 and Cdep in Drosophila. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1807/32608.

Council of Science Editors:

Milic M. Mutational Analysis of FERM Domain Proteins CG34347 and Cdep in Drosophila. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/32608

15. PANG JUNXIONG, VINCENT. FRMD4A regulates the entry of West Nile virus into glioblastoma cells.

Degree: 2009, National University of Singapore

URL: http://scholarbank.nus.edu.sg/handle/10635/16285

Subjects/Keywords: West Nile Virus; Ferm Domain; Glioblastoma cells; Differential Permissivity; alphaV Beta3 integrin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

PANG JUNXIONG, V. (2009). FRMD4A regulates the entry of West Nile virus into glioblastoma cells. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/16285

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

PANG JUNXIONG, VINCENT. “FRMD4A regulates the entry of West Nile virus into glioblastoma cells.” 2009. Thesis, National University of Singapore. Accessed January 19, 2021. http://scholarbank.nus.edu.sg/handle/10635/16285.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

PANG JUNXIONG, VINCENT. “FRMD4A regulates the entry of West Nile virus into glioblastoma cells.” 2009. Web. 19 Jan 2021.

Vancouver:

PANG JUNXIONG V. FRMD4A regulates the entry of West Nile virus into glioblastoma cells. [Internet] [Thesis]. National University of Singapore; 2009. [cited 2021 Jan 19]. Available from: http://scholarbank.nus.edu.sg/handle/10635/16285.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

PANG JUNXIONG V. FRMD4A regulates the entry of West Nile virus into glioblastoma cells. [Thesis]. National University of Singapore; 2009. Available from: http://scholarbank.nus.edu.sg/handle/10635/16285

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Universitat Politècnica de Catalunya

16. Izquierdo Ramonet, Maria. Valorització d'escòries d'incineració de residus sòlids urbans en capes granulars del ferm. Aplicació al tram experimental de Tagamanent.

Degree: Departament d'Enginyeria de la Construcció, 2005, Universitat Politècnica de Catalunya

URL: http://hdl.handle.net/10803/6161

► Les escòries són el subproducte més abundant que s'obté en el procés d'incineració de residus sòlids urbans; constitueixen al voltant del 90 % en pes… (more)

▼ Les escòries són el subproducte més abundant que s'obté en el procés d'incineració de residus sòlids urbans; constitueixen al voltant del 90 % en pes del producte sòlid resultant de la combustió. La producció anual a Catalunya és d'unes 150000 tones d'escòries, generades en set plantes incineradores localitzades a Tarragona, Mataró, Sant Adrià de Besòs, Montcada i Reixac, Malla, Girona i Vielha. El seu destí final és l'abocament, juntament amb la utilització puntual en petites obres menors d'àmbit local i de manera poc regulada. Pel seu caràcter a primera vista inert i l'aspecte granular es considera que les escòries es podrien utilitzar en l'àmbit de la construcció. En aquest estudi es pretén avaluar la viabilitat de la valorització de les escòries d'incineració de residus sòlids urbans produïdes a Catalunya en capes granulars de ferms de carreteres, com a substitut d'àrids naturals. La valoració de la idoneïtat de les escòries ha de tenir en compte l'adequació a les normatives tècniques que regeixen aquesta possible aplicació: l'Ordre de 15 de febrer de 1996, sobre valorització d'escòries del Departament de Medi Ambient i l'article 510 sobre tot-u del PG-3 (2004). Més enllà de les prescripcions, tanmateix, s'ha considerat necessària una caracterització de les escòries des de diverses perspectives per tenir un coneixement integral del material que permeti gestionar-lo correctament. Finalment l'estudi es completa amb un tram experimental de camp a Tagamanent, amb l'objectiu de contrastar els resultats obtinguts al laboratori i al camp. A les escòries hi coexisteixen tres paragènesis: els constituents preexistents (preincineració) com el vidre domèstic; els productes de fusió (sinincineració), característics de les escòries i equiparables a una roca ultrabàsica, i la paragènesi postincineració, constituïda per productes de neoformació que es formen a causa de l'elevada reactivitat del residu en sortir del forn i posen de manifest que la mineralogia de les escòries és dinàmica. Les proporcions entre les tipologies de materials són diferents i variables segons la mida de partícula considerada; aquesta particularitat és la clau de l'heterogeneïtat de les escòries. Les escòries tenen una granulometria contínua, poden assolir alts graus de compactació i capacitats portants molt notables, comparables a les que mostren àrids naturals, mentre que són lleugerament menys denses, més poroses i més fràgils que aquests. Cal destacar l'escassa sensibilitat a l'aigua i l'elevada estabilitat volumètrica. Si bé s'han determinat els continguts de més de 50 elements a les escòries, el principal component és el SiO2 (45 %). La resta d'elements majoritaris són Ca, Fe, Al, Na, C, Mg i K, comuns a materials naturals. Entre els minoritaris, alguns també són comuns en àrids naturals com S, Ti, Ba i Mn, però en canvi les escòries estan molt enriquides en Cu, Zn i Pb. Pel seu interès ambiental destaquen els baixos continguts en Ni, Co, As, Se, Mo, Cd i Hg. El comportament ambiental de les escòries… Advisors/Committee Members: [email protected] (authoremail), false (authoremailshow), Vázquez Ramonich, E. (Enric) (director), Querol Carceller, Xavier (codirector).

Subjects/Keywords: escòries; tot-v; lixiviació; ferm; valorització de residus; incineració der esidus sòlids urbans; 3308. Enginyeria i tecnologia del medi ambient - 2506. Geologia - 3305. Tecnologia de la construcció; 504; 55; 624

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Izquierdo Ramonet, M. (2005). Valorització d'escòries d'incineració de residus sòlids urbans en capes granulars del ferm. Aplicació al tram experimental de Tagamanent. (Thesis). Universitat Politècnica de Catalunya. Retrieved from http://hdl.handle.net/10803/6161

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Izquierdo Ramonet, Maria. “Valorització d'escòries d'incineració de residus sòlids urbans en capes granulars del ferm. Aplicació al tram experimental de Tagamanent.” 2005. Thesis, Universitat Politècnica de Catalunya. Accessed January 19, 2021. http://hdl.handle.net/10803/6161.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Izquierdo Ramonet, Maria. “Valorització d'escòries d'incineració de residus sòlids urbans en capes granulars del ferm. Aplicació al tram experimental de Tagamanent.” 2005. Web. 19 Jan 2021.

Vancouver:

Izquierdo Ramonet M. Valorització d'escòries d'incineració de residus sòlids urbans en capes granulars del ferm. Aplicació al tram experimental de Tagamanent. [Internet] [Thesis]. Universitat Politècnica de Catalunya; 2005. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10803/6161.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Izquierdo Ramonet M. Valorització d'escòries d'incineració de residus sòlids urbans en capes granulars del ferm. Aplicació al tram experimental de Tagamanent. [Thesis]. Universitat Politècnica de Catalunya; 2005. Available from: http://hdl.handle.net/10803/6161

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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Open Access Theses and Dissertations