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You searched for subject:(Exon skipping therapeutic strategy). Showing records 1 – 30 of 13414 total matches.

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1. Messaoud Khelifi, Mouna. Etude de séquences cis-régulatrices d'épissage dans le gène DMD : rôle dans la régulation des pseudoexons et intérêt pour le saut d'exon thérapeutique. : Splicing cis-regulatory sequences in the DMD gene : role in pseudoexons regulation and interest for the therapeutic exon skipping strategy.

Degree: Docteur es, Cancérologie, génétique, hématologie, immunologie, 2010, Université Montpellier I

L'épissage des ARN pré-messagers est une étape essentielle pour l'expression des gènes chez les eucaryotes supérieurs. La reconnaissance des exons par la machinerie d'épissage est… (more)

Subjects/Keywords: Gène DMD; Epissage; Saut d'exon thérapeutique; Oligonucléotides antisens; Séquences cis-régulatrices; Pseudoexons; DMD gene; Splicing; Exon skipping therapeutic strategy; Antisense oligonucleotides; Cis-regulatory sequences; Pseudoexons

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Messaoud Khelifi, M. (2010). Etude de séquences cis-régulatrices d'épissage dans le gène DMD : rôle dans la régulation des pseudoexons et intérêt pour le saut d'exon thérapeutique. : Splicing cis-regulatory sequences in the DMD gene : role in pseudoexons regulation and interest for the therapeutic exon skipping strategy. (Doctoral Dissertation). Université Montpellier I. Retrieved from http://www.theses.fr/2010MON13521

Chicago Manual of Style (16th Edition):

Messaoud Khelifi, Mouna. “Etude de séquences cis-régulatrices d'épissage dans le gène DMD : rôle dans la régulation des pseudoexons et intérêt pour le saut d'exon thérapeutique. : Splicing cis-regulatory sequences in the DMD gene : role in pseudoexons regulation and interest for the therapeutic exon skipping strategy.” 2010. Doctoral Dissertation, Université Montpellier I. Accessed December 07, 2019. http://www.theses.fr/2010MON13521.

MLA Handbook (7th Edition):

Messaoud Khelifi, Mouna. “Etude de séquences cis-régulatrices d'épissage dans le gène DMD : rôle dans la régulation des pseudoexons et intérêt pour le saut d'exon thérapeutique. : Splicing cis-regulatory sequences in the DMD gene : role in pseudoexons regulation and interest for the therapeutic exon skipping strategy.” 2010. Web. 07 Dec 2019.

Vancouver:

Messaoud Khelifi M. Etude de séquences cis-régulatrices d'épissage dans le gène DMD : rôle dans la régulation des pseudoexons et intérêt pour le saut d'exon thérapeutique. : Splicing cis-regulatory sequences in the DMD gene : role in pseudoexons regulation and interest for the therapeutic exon skipping strategy. [Internet] [Doctoral dissertation]. Université Montpellier I; 2010. [cited 2019 Dec 07]. Available from: http://www.theses.fr/2010MON13521.

Council of Science Editors:

Messaoud Khelifi M. Etude de séquences cis-régulatrices d'épissage dans le gène DMD : rôle dans la régulation des pseudoexons et intérêt pour le saut d'exon thérapeutique. : Splicing cis-regulatory sequences in the DMD gene : role in pseudoexons regulation and interest for the therapeutic exon skipping strategy. [Doctoral Dissertation]. Université Montpellier I; 2010. Available from: http://www.theses.fr/2010MON13521

2. Turczynski, Sandrine. Stratégie thérapeutique par saut d’exon pour les épidermolyses bulleuses dystrophiques : Exon skipping as a therapeutic approach for Dystrophic Epidermolysis Bullosa.

Degree: Docteur es, Génétique, 2013, Université Paris Descartes – Paris V

 Les Epidermolyses Bulleuses Dystrophiques (EBD) sont des génodermatoses rares et sévères transmises sur un mode autosomique récessif (EBDR) ou dominant (EBDD), dues à une perte… (more)

Subjects/Keywords: Stratégie thérapeutique; Saut d’exon; COL7A1; Epidermolyses bulleuses; Therapeutic approach; Exon skipping; COL7A1; Epidermolysis bullosa; 616.042

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APA (6th Edition):

Turczynski, S. (2013). Stratégie thérapeutique par saut d’exon pour les épidermolyses bulleuses dystrophiques : Exon skipping as a therapeutic approach for Dystrophic Epidermolysis Bullosa. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2013PA05T085

Chicago Manual of Style (16th Edition):

Turczynski, Sandrine. “Stratégie thérapeutique par saut d’exon pour les épidermolyses bulleuses dystrophiques : Exon skipping as a therapeutic approach for Dystrophic Epidermolysis Bullosa.” 2013. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed December 07, 2019. http://www.theses.fr/2013PA05T085.

MLA Handbook (7th Edition):

Turczynski, Sandrine. “Stratégie thérapeutique par saut d’exon pour les épidermolyses bulleuses dystrophiques : Exon skipping as a therapeutic approach for Dystrophic Epidermolysis Bullosa.” 2013. Web. 07 Dec 2019.

Vancouver:

Turczynski S. Stratégie thérapeutique par saut d’exon pour les épidermolyses bulleuses dystrophiques : Exon skipping as a therapeutic approach for Dystrophic Epidermolysis Bullosa. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2013. [cited 2019 Dec 07]. Available from: http://www.theses.fr/2013PA05T085.

Council of Science Editors:

Turczynski S. Stratégie thérapeutique par saut d’exon pour les épidermolyses bulleuses dystrophiques : Exon skipping as a therapeutic approach for Dystrophic Epidermolysis Bullosa. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2013. Available from: http://www.theses.fr/2013PA05T085


University of Debrecen

3. Bilakovics, Noémi. Fungális stwintron (spliceoszómális iker-intron) struktúrák szerepe exon skipping jelenség lejátszódásában .

Degree: DE – Természettudományi és Technológiai Kar – Biotechnológiai Intézet, University of Debrecen

 Szakdolgozatomban két új fonalas gombában, az Apergillus nigerben és Neurospora crassaban, kerül leírásra egy [D5,6] típusú stwintron. Emellett sikerült bizonyítanunk az stwintronok szerepét alternatív splicing… (more)

Subjects/Keywords: stwintron; alternatív splicing; exon skipping

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APA (6th Edition):

Bilakovics, N. (n.d.). Fungális stwintron (spliceoszómális iker-intron) struktúrák szerepe exon skipping jelenség lejátszódásában . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/276231

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bilakovics, Noémi. “Fungális stwintron (spliceoszómális iker-intron) struktúrák szerepe exon skipping jelenség lejátszódásában .” Thesis, University of Debrecen. Accessed December 07, 2019. http://hdl.handle.net/2437/276231.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bilakovics, Noémi. “Fungális stwintron (spliceoszómális iker-intron) struktúrák szerepe exon skipping jelenség lejátszódásában .” Web. 07 Dec 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Bilakovics N. Fungális stwintron (spliceoszómális iker-intron) struktúrák szerepe exon skipping jelenség lejátszódásában . [Internet] [Thesis]. University of Debrecen; [cited 2019 Dec 07]. Available from: http://hdl.handle.net/2437/276231.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Bilakovics N. Fungális stwintron (spliceoszómális iker-intron) struktúrák szerepe exon skipping jelenség lejátszódásában . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/276231

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Freie Universität Berlin

4. Malcher, Jakub. Exon skipping als therapeutische Strategie in Dysferlinopathie.

Degree: 2018, Freie Universität Berlin

 Dysferlinopathie ist eine Muskeldystrophie, die sich in zwei Hauptphänotypen manifestiert: Gliedergürtelmuskeldystrophie Typ 2B type 2B (LGMD2B) oder Miyoshi Myopathie (MM). Sie wird durch Mutationen im… (more)

Subjects/Keywords: dysferlinopathy; LGMD2B; dysferlin; exon skipping; U7 snRNA; rAVV; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie

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APA (6th Edition):

Malcher, J. (2018). Exon skipping als therapeutische Strategie in Dysferlinopathie. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-8037

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Malcher, Jakub. “Exon skipping als therapeutische Strategie in Dysferlinopathie.” 2018. Thesis, Freie Universität Berlin. Accessed December 07, 2019. http://dx.doi.org/10.17169/refubium-8037.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Malcher, Jakub. “Exon skipping als therapeutische Strategie in Dysferlinopathie.” 2018. Web. 07 Dec 2019.

Vancouver:

Malcher J. Exon skipping als therapeutische Strategie in Dysferlinopathie. [Internet] [Thesis]. Freie Universität Berlin; 2018. [cited 2019 Dec 07]. Available from: http://dx.doi.org/10.17169/refubium-8037.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Malcher J. Exon skipping als therapeutische Strategie in Dysferlinopathie. [Thesis]. Freie Universität Berlin; 2018. Available from: http://dx.doi.org/10.17169/refubium-8037

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

5. Chu, Yuan-Ping. Taiwan Banded Krait beta-Bungarotoxins: Novel Isotoxins, Targeting and Gene Organization.

Degree: Master, Institute of Biomedical Sciences, 2002, NSYSU

 beta-Bungarotoxin (beta-Bgt), the presynaptic neurotoxin purified from the venom of Bungarus multicinctus, consists of the A chain and the B chain, cross-linked by an interchain… (more)

Subjects/Keywords: A chain; exon skipping; beta-Bungarotoxin; isotoxin; Bungarus multicinctus

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APA (6th Edition):

Chu, Y. (2002). Taiwan Banded Krait beta-Bungarotoxins: Novel Isotoxins, Targeting and Gene Organization. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0611102-123013

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chu, Yuan-Ping. “Taiwan Banded Krait beta-Bungarotoxins: Novel Isotoxins, Targeting and Gene Organization.” 2002. Thesis, NSYSU. Accessed December 07, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0611102-123013.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chu, Yuan-Ping. “Taiwan Banded Krait beta-Bungarotoxins: Novel Isotoxins, Targeting and Gene Organization.” 2002. Web. 07 Dec 2019.

Vancouver:

Chu Y. Taiwan Banded Krait beta-Bungarotoxins: Novel Isotoxins, Targeting and Gene Organization. [Internet] [Thesis]. NSYSU; 2002. [cited 2019 Dec 07]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0611102-123013.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chu Y. Taiwan Banded Krait beta-Bungarotoxins: Novel Isotoxins, Targeting and Gene Organization. [Thesis]. NSYSU; 2002. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0611102-123013

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Philippi, Susanne. RNA-based therapies for dysferlinopathies : Utilisation d'acide ribonucléique pour le traitement des dysferlinopathies.

Degree: Docteur es, Biologie, 2014, Université Pierre et Marie Curie – Paris VI

L’épissage en cis des précurseurs d’ARN messager (pre-ARNm) est une stratégie intéressante afin de réparer des gènes dont la régulation transcriptionnelle est déterminante pour la… (more)

Subjects/Keywords: Précurseurs d'ARN messager; Signaux d'épissage; Régulation transcriptionnelle; Trans-Épissage; Dysferline; Dystrophies musculaires; Dysferlin; Exon skipping; 573.883 9

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APA (6th Edition):

Philippi, S. (2014). RNA-based therapies for dysferlinopathies : Utilisation d'acide ribonucléique pour le traitement des dysferlinopathies. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2014PA066479

Chicago Manual of Style (16th Edition):

Philippi, Susanne. “RNA-based therapies for dysferlinopathies : Utilisation d'acide ribonucléique pour le traitement des dysferlinopathies.” 2014. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed December 07, 2019. http://www.theses.fr/2014PA066479.

MLA Handbook (7th Edition):

Philippi, Susanne. “RNA-based therapies for dysferlinopathies : Utilisation d'acide ribonucléique pour le traitement des dysferlinopathies.” 2014. Web. 07 Dec 2019.

Vancouver:

Philippi S. RNA-based therapies for dysferlinopathies : Utilisation d'acide ribonucléique pour le traitement des dysferlinopathies. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2014. [cited 2019 Dec 07]. Available from: http://www.theses.fr/2014PA066479.

Council of Science Editors:

Philippi S. RNA-based therapies for dysferlinopathies : Utilisation d'acide ribonucléique pour le traitement des dysferlinopathies. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2014. Available from: http://www.theses.fr/2014PA066479


University of Kentucky

7. Brooks, Samantha Ann. STUDIES OF GENETIC VARIATION AT THE KIT LOCUS AND WHITE SPOTTING PATTERNS IN THE HORSE.

Degree: 2006, University of Kentucky

 There are numerous different white spotting patterns in the horse, including two of particular interest tobiano and sabino. In the mouse, genetic variation in the… (more)

Subjects/Keywords: horse; tobiano; sabino; chromosome inversion; exon skipping

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APA (6th Edition):

Brooks, S. A. (2006). STUDIES OF GENETIC VARIATION AT THE KIT LOCUS AND WHITE SPOTTING PATTERNS IN THE HORSE. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/479

Chicago Manual of Style (16th Edition):

Brooks, Samantha Ann. “STUDIES OF GENETIC VARIATION AT THE KIT LOCUS AND WHITE SPOTTING PATTERNS IN THE HORSE.” 2006. Doctoral Dissertation, University of Kentucky. Accessed December 07, 2019. https://uknowledge.uky.edu/gradschool_diss/479.

MLA Handbook (7th Edition):

Brooks, Samantha Ann. “STUDIES OF GENETIC VARIATION AT THE KIT LOCUS AND WHITE SPOTTING PATTERNS IN THE HORSE.” 2006. Web. 07 Dec 2019.

Vancouver:

Brooks SA. STUDIES OF GENETIC VARIATION AT THE KIT LOCUS AND WHITE SPOTTING PATTERNS IN THE HORSE. [Internet] [Doctoral dissertation]. University of Kentucky; 2006. [cited 2019 Dec 07]. Available from: https://uknowledge.uky.edu/gradschool_diss/479.

Council of Science Editors:

Brooks SA. STUDIES OF GENETIC VARIATION AT THE KIT LOCUS AND WHITE SPOTTING PATTERNS IN THE HORSE. [Doctoral Dissertation]. University of Kentucky; 2006. Available from: https://uknowledge.uky.edu/gradschool_diss/479


Leiden University

8. Rutten, J.W. NOTCH3 cysteine correction: developing a rational therapeutic approach for CADASIL.

Degree: 2016, Leiden University

 The aim of this thesis was to work towards pre-clinical proof-of-concept for NOTCH3 cysteine corrective exon skipping as a rational therapeutic approach for CADASIL. To… (more)

Subjects/Keywords: CADASIL; NOTCH3; Stroke; Dementia; Exon skipping; Therapy; Antisense oligonucleotides; Mutations; CADASIL; NOTCH3; Stroke; Dementia; Exon skipping; Therapy; Antisense oligonucleotides; Mutations

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APA (6th Edition):

Rutten, J. W. (2016). NOTCH3 cysteine correction: developing a rational therapeutic approach for CADASIL. (Doctoral Dissertation). Leiden University. Retrieved from http://hdl.handle.net/1887/43112

Chicago Manual of Style (16th Edition):

Rutten, J W. “NOTCH3 cysteine correction: developing a rational therapeutic approach for CADASIL.” 2016. Doctoral Dissertation, Leiden University. Accessed December 07, 2019. http://hdl.handle.net/1887/43112.

MLA Handbook (7th Edition):

Rutten, J W. “NOTCH3 cysteine correction: developing a rational therapeutic approach for CADASIL.” 2016. Web. 07 Dec 2019.

Vancouver:

Rutten JW. NOTCH3 cysteine correction: developing a rational therapeutic approach for CADASIL. [Internet] [Doctoral dissertation]. Leiden University; 2016. [cited 2019 Dec 07]. Available from: http://hdl.handle.net/1887/43112.

Council of Science Editors:

Rutten JW. NOTCH3 cysteine correction: developing a rational therapeutic approach for CADASIL. [Doctoral Dissertation]. Leiden University; 2016. Available from: http://hdl.handle.net/1887/43112


Leiden University

9. Jirka, S. Developing tissue specific antisense oligonucleotide-delivery to refine treatment for Duchenne muscular dystrophy.

Degree: 2017, Leiden University

 Duchenne muscular dystrophy (DMD) is a severe progressive muscle wasting disorder. DMD is caused by reading frame disrupting mutations in the DMD gene resulting is… (more)

Subjects/Keywords: DMD; AON; Phagedisplay; Peptide; Next generation sequencing; Exon skipping; Muscle; DMD; AON; Phagedisplay; Peptide; Next generation sequencing; Exon skipping; Muscle

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APA (6th Edition):

Jirka, S. (2017). Developing tissue specific antisense oligonucleotide-delivery to refine treatment for Duchenne muscular dystrophy. (Doctoral Dissertation). Leiden University. Retrieved from http://hdl.handle.net/1887/51132

Chicago Manual of Style (16th Edition):

Jirka, S. “Developing tissue specific antisense oligonucleotide-delivery to refine treatment for Duchenne muscular dystrophy.” 2017. Doctoral Dissertation, Leiden University. Accessed December 07, 2019. http://hdl.handle.net/1887/51132.

MLA Handbook (7th Edition):

Jirka, S. “Developing tissue specific antisense oligonucleotide-delivery to refine treatment for Duchenne muscular dystrophy.” 2017. Web. 07 Dec 2019.

Vancouver:

Jirka S. Developing tissue specific antisense oligonucleotide-delivery to refine treatment for Duchenne muscular dystrophy. [Internet] [Doctoral dissertation]. Leiden University; 2017. [cited 2019 Dec 07]. Available from: http://hdl.handle.net/1887/51132.

Council of Science Editors:

Jirka S. Developing tissue specific antisense oligonucleotide-delivery to refine treatment for Duchenne muscular dystrophy. [Doctoral Dissertation]. Leiden University; 2017. Available from: http://hdl.handle.net/1887/51132


Leiden University

10. Toonen, L.J.A. Developing an antisense oligonucleotide treatment for Spinocerebellar Ataxia Type 3.

Degree: 2018, Leiden University

 Spinocerebellar ataxia type 3 (SCA3) is a hereditary neurodegenerative disorder caused by a CAG triplet repeat expansion in the ATXN3 gene. This expanded CAG repeat… (more)

Subjects/Keywords: Spinocerebellar ataxia type 3; Polyglutamine disorder; Antisense oligonucleotides; Exon skipping; Spinocerebellar ataxia type 3; Polyglutamine disorder; Antisense oligonucleotides; Exon skipping

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APA (6th Edition):

Toonen, L. J. A. (2018). Developing an antisense oligonucleotide treatment for Spinocerebellar Ataxia Type 3. (Doctoral Dissertation). Leiden University. Retrieved from http://hdl.handle.net/1887/62616

Chicago Manual of Style (16th Edition):

Toonen, L J A. “Developing an antisense oligonucleotide treatment for Spinocerebellar Ataxia Type 3.” 2018. Doctoral Dissertation, Leiden University. Accessed December 07, 2019. http://hdl.handle.net/1887/62616.

MLA Handbook (7th Edition):

Toonen, L J A. “Developing an antisense oligonucleotide treatment for Spinocerebellar Ataxia Type 3.” 2018. Web. 07 Dec 2019.

Vancouver:

Toonen LJA. Developing an antisense oligonucleotide treatment for Spinocerebellar Ataxia Type 3. [Internet] [Doctoral dissertation]. Leiden University; 2018. [cited 2019 Dec 07]. Available from: http://hdl.handle.net/1887/62616.

Council of Science Editors:

Toonen LJA. Developing an antisense oligonucleotide treatment for Spinocerebellar Ataxia Type 3. [Doctoral Dissertation]. Leiden University; 2018. Available from: http://hdl.handle.net/1887/62616

11. Rai, Ghadi C. Système de connaissance expert dédié à la recherche translationnelle dans les maladies rares : Expert knowledge system dedicated to translational research in rare diseases.

Degree: Docteur es, Biologie. Génomique et bioinformatique, 2016, Aix Marseille Université

Environ 6000 à 8000 maladies rares différentes existent aujourd’hui, affectant environ 6 à 8% de la population mondiale. La grande majorité d’entre elles correspond à… (more)

Subjects/Keywords: Bioinformatique; Maladies rares; Prédiction; Épissage; Substitutions; Brca1; Brca2; Stratégies thérapeutiques; Saut d'exon; Bioinformatics; Rare diseases; Prediction; Splicing; Substitution; Brca1; Brca2; Therapeutics; Exon skipping

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APA (6th Edition):

Rai, G. C. (2016). Système de connaissance expert dédié à la recherche translationnelle dans les maladies rares : Expert knowledge system dedicated to translational research in rare diseases. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2016AIXM5057

Chicago Manual of Style (16th Edition):

Rai, Ghadi C. “Système de connaissance expert dédié à la recherche translationnelle dans les maladies rares : Expert knowledge system dedicated to translational research in rare diseases.” 2016. Doctoral Dissertation, Aix Marseille Université. Accessed December 07, 2019. http://www.theses.fr/2016AIXM5057.

MLA Handbook (7th Edition):

Rai, Ghadi C. “Système de connaissance expert dédié à la recherche translationnelle dans les maladies rares : Expert knowledge system dedicated to translational research in rare diseases.” 2016. Web. 07 Dec 2019.

Vancouver:

Rai GC. Système de connaissance expert dédié à la recherche translationnelle dans les maladies rares : Expert knowledge system dedicated to translational research in rare diseases. [Internet] [Doctoral dissertation]. Aix Marseille Université 2016. [cited 2019 Dec 07]. Available from: http://www.theses.fr/2016AIXM5057.

Council of Science Editors:

Rai GC. Système de connaissance expert dédié à la recherche translationnelle dans les maladies rares : Expert knowledge system dedicated to translational research in rare diseases. [Doctoral Dissertation]. Aix Marseille Université 2016. Available from: http://www.theses.fr/2016AIXM5057


Université de Grenoble

12. Rendu, John. Thérapie génique par saut d'exon : application à une Myopathie à Core et à un cas de syndrome OculoCérébroRénale de Lowe : Exon skipping therapy application to structural myopathy and Lowe syndrome.

Degree: Docteur es, Biotechnologie, 2014, Université de Grenoble

Après la transcription, le pré ARNm subit des étapes de maturation avant de sortir du noyau pour être traduit. Une des étapes de maturation est… (more)

Subjects/Keywords: Épissage; Récepteur de la ryanodine; Saut d'exon; Complexe de mobilisation du Calcium; OCRL; Syndrome de Lowe; Splicing; Ryanodin receptor; Exon skipping; Calcium release complex; OCRL; Lowe syndrome; 610

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APA (6th Edition):

Rendu, J. (2014). Thérapie génique par saut d'exon : application à une Myopathie à Core et à un cas de syndrome OculoCérébroRénale de Lowe : Exon skipping therapy application to structural myopathy and Lowe syndrome. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2014GRENV014

Chicago Manual of Style (16th Edition):

Rendu, John. “Thérapie génique par saut d'exon : application à une Myopathie à Core et à un cas de syndrome OculoCérébroRénale de Lowe : Exon skipping therapy application to structural myopathy and Lowe syndrome.” 2014. Doctoral Dissertation, Université de Grenoble. Accessed December 07, 2019. http://www.theses.fr/2014GRENV014.

MLA Handbook (7th Edition):

Rendu, John. “Thérapie génique par saut d'exon : application à une Myopathie à Core et à un cas de syndrome OculoCérébroRénale de Lowe : Exon skipping therapy application to structural myopathy and Lowe syndrome.” 2014. Web. 07 Dec 2019.

Vancouver:

Rendu J. Thérapie génique par saut d'exon : application à une Myopathie à Core et à un cas de syndrome OculoCérébroRénale de Lowe : Exon skipping therapy application to structural myopathy and Lowe syndrome. [Internet] [Doctoral dissertation]. Université de Grenoble; 2014. [cited 2019 Dec 07]. Available from: http://www.theses.fr/2014GRENV014.

Council of Science Editors:

Rendu J. Thérapie génique par saut d'exon : application à une Myopathie à Core et à un cas de syndrome OculoCérébroRénale de Lowe : Exon skipping therapy application to structural myopathy and Lowe syndrome. [Doctoral Dissertation]. Université de Grenoble; 2014. Available from: http://www.theses.fr/2014GRENV014

13. Simmons, Tabatha Renee. Treatment of DMD 5’ Mutations through Two Different Exon 2 Skipping Strategies: rAAV9.U7snRNA Mediated Skipping and Antisense Morpholino Oligomers.

Degree: PhD, Molecular, Cellular and Developmental Biology, 2016, The Ohio State University

 Duchenne muscular dystrophy (DMD) is an X-linked progressive disease characterized by loss of dystrophin protein as a result of truncating mutations in the DMD gene.… (more)

Subjects/Keywords: Biomedical Research; Duchenne Muscular Dystrophy, Exon Skipping, U7snRNA, Antisense Oligomer

…clinical trial [139]. Exon Skipping As mentioned above, a key strategy to restoration… …U7snRNAmediated DMD exon 2 skipping. Tabatha Simmons, Nicolas Wein, Adeline Vulin-Chaffiol, Felecia… …preparation vii Proof-of-principle study shows efficient exon 2 skipping using antisense… …Hannah Huang, Kevin M. Flanigan. Manuscript in preparation Out-of-frame exon-skipping restores… …13 Exon Skipping… 

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APA (6th Edition):

Simmons, T. R. (2016). Treatment of DMD 5’ Mutations through Two Different Exon 2 Skipping Strategies: rAAV9.U7snRNA Mediated Skipping and Antisense Morpholino Oligomers. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1469122227

Chicago Manual of Style (16th Edition):

Simmons, Tabatha Renee. “Treatment of DMD 5’ Mutations through Two Different Exon 2 Skipping Strategies: rAAV9.U7snRNA Mediated Skipping and Antisense Morpholino Oligomers.” 2016. Doctoral Dissertation, The Ohio State University. Accessed December 07, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1469122227.

MLA Handbook (7th Edition):

Simmons, Tabatha Renee. “Treatment of DMD 5’ Mutations through Two Different Exon 2 Skipping Strategies: rAAV9.U7snRNA Mediated Skipping and Antisense Morpholino Oligomers.” 2016. Web. 07 Dec 2019.

Vancouver:

Simmons TR. Treatment of DMD 5’ Mutations through Two Different Exon 2 Skipping Strategies: rAAV9.U7snRNA Mediated Skipping and Antisense Morpholino Oligomers. [Internet] [Doctoral dissertation]. The Ohio State University; 2016. [cited 2019 Dec 07]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1469122227.

Council of Science Editors:

Simmons TR. Treatment of DMD 5’ Mutations through Two Different Exon 2 Skipping Strategies: rAAV9.U7snRNA Mediated Skipping and Antisense Morpholino Oligomers. [Doctoral Dissertation]. The Ohio State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1469122227

14. Malcher, Jakub. Exon skipping as a therapeutic strategy in dysferlinopathy : Le saut d’exon thérapeutique pour le traitement des dysferlinopathies.

Degree: Docteur es, Physiologie et physiopathologie, 2018, Paris Saclay; Freie Universität (Berlin)

Les dysferlinopathies sont des dystrophies musculaires qui se manifestent par la dystrophie musculaire des ceintures de type 2B (LGMD2B) ou la myopathie de Miyoshi (MM).… (more)

Subjects/Keywords: Dysferlinopathies; Lgmd2b; Dysferline; Saut d’exon; U7 snRNA; Vecteurs AAV; Modulation d'epissage; Dysferlinopathy; Lgmd2b; Dysferlin; Exon skipping; U7 snRNA; AVV vectors; Splice switching; 616.7

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APA (6th Edition):

Malcher, J. (2018). Exon skipping as a therapeutic strategy in dysferlinopathy : Le saut d’exon thérapeutique pour le traitement des dysferlinopathies. (Doctoral Dissertation). Paris Saclay; Freie Universität (Berlin). Retrieved from http://www.theses.fr/2018SACLV007

Chicago Manual of Style (16th Edition):

Malcher, Jakub. “Exon skipping as a therapeutic strategy in dysferlinopathy : Le saut d’exon thérapeutique pour le traitement des dysferlinopathies.” 2018. Doctoral Dissertation, Paris Saclay; Freie Universität (Berlin). Accessed December 07, 2019. http://www.theses.fr/2018SACLV007.

MLA Handbook (7th Edition):

Malcher, Jakub. “Exon skipping as a therapeutic strategy in dysferlinopathy : Le saut d’exon thérapeutique pour le traitement des dysferlinopathies.” 2018. Web. 07 Dec 2019.

Vancouver:

Malcher J. Exon skipping as a therapeutic strategy in dysferlinopathy : Le saut d’exon thérapeutique pour le traitement des dysferlinopathies. [Internet] [Doctoral dissertation]. Paris Saclay; Freie Universität (Berlin); 2018. [cited 2019 Dec 07]. Available from: http://www.theses.fr/2018SACLV007.

Council of Science Editors:

Malcher J. Exon skipping as a therapeutic strategy in dysferlinopathy : Le saut d’exon thérapeutique pour le traitement des dysferlinopathies. [Doctoral Dissertation]. Paris Saclay; Freie Universität (Berlin); 2018. Available from: http://www.theses.fr/2018SACLV007

15. XU HUAN. Application of computational intelligence in biological sciences.

Degree: 2003, National University of Singapore

Subjects/Keywords: DNA microarray; probe; evolutionary strategy; BLAST; premature convergence; Exon

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APA (6th Edition):

HUAN, X. (2003). Application of computational intelligence in biological sciences. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/13516

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

HUAN, XU. “Application of computational intelligence in biological sciences.” 2003. Thesis, National University of Singapore. Accessed December 07, 2019. http://scholarbank.nus.edu.sg/handle/10635/13516.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

HUAN, XU. “Application of computational intelligence in biological sciences.” 2003. Web. 07 Dec 2019.

Vancouver:

HUAN X. Application of computational intelligence in biological sciences. [Internet] [Thesis]. National University of Singapore; 2003. [cited 2019 Dec 07]. Available from: http://scholarbank.nus.edu.sg/handle/10635/13516.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

HUAN X. Application of computational intelligence in biological sciences. [Thesis]. National University of Singapore; 2003. Available from: http://scholarbank.nus.edu.sg/handle/10635/13516

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Shoji, Emi. Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. : デュシェンヌ型筋ジストロフィー患者由来iPS細胞を用いた初期病態再現.

Degree: 博士(医学), 2015, Kyoto University / 京都大学

新制・課程博士

甲第19229号

医博第4028号

Subjects/Keywords: Duchenne muscular dystrophy; Human indcued pluripotent stem cell; Disease modelling; Calcium ion influx; Exon skipping; Dystrophin

Page 1 Page 2

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APA (6th Edition):

Shoji, E. (2015). Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. : デュシェンヌ型筋ジストロフィー患者由来iPS細胞を用いた初期病態再現. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/200495 ; http://dx.doi.org/10.14989/doctor.k19229

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shoji, Emi. “Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. : デュシェンヌ型筋ジストロフィー患者由来iPS細胞を用いた初期病態再現.” 2015. Thesis, Kyoto University / 京都大学. Accessed December 07, 2019. http://hdl.handle.net/2433/200495 ; http://dx.doi.org/10.14989/doctor.k19229.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shoji, Emi. “Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. : デュシェンヌ型筋ジストロフィー患者由来iPS細胞を用いた初期病態再現.” 2015. Web. 07 Dec 2019.

Vancouver:

Shoji E. Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. : デュシェンヌ型筋ジストロフィー患者由来iPS細胞を用いた初期病態再現. [Internet] [Thesis]. Kyoto University / 京都大学; 2015. [cited 2019 Dec 07]. Available from: http://hdl.handle.net/2433/200495 ; http://dx.doi.org/10.14989/doctor.k19229.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shoji E. Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. : デュシェンヌ型筋ジストロフィー患者由来iPS細胞を用いた初期病態再現. [Thesis]. Kyoto University / 京都大学; 2015. Available from: http://hdl.handle.net/2433/200495 ; http://dx.doi.org/10.14989/doctor.k19229

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

17. Shoji, Emi. Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells.

Degree: 2015, Kyoto University

Subjects/Keywords: Duchenne muscular dystrophy; Human indcued pluripotent stem cell; Disease modelling; Calcium ion influx; Exon skipping; Dystrophin

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APA (6th Edition):

Shoji, E. (2015). Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/200495

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shoji, Emi. “Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. ” 2015. Thesis, Kyoto University. Accessed December 07, 2019. http://hdl.handle.net/2433/200495.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shoji, Emi. “Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. ” 2015. Web. 07 Dec 2019.

Vancouver:

Shoji E. Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. [Internet] [Thesis]. Kyoto University; 2015. [cited 2019 Dec 07]. Available from: http://hdl.handle.net/2433/200495.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shoji E. Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells. [Thesis]. Kyoto University; 2015. Available from: http://hdl.handle.net/2433/200495

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

18. Shariat, Nikki. Growth Hormone Splicing and Treatment of Disease Using RNA Interference.

Degree: PhD, Biological Sciences, 2008, Vanderbilt University

 Splicing is the regulated removal of introns and the concurrent ligation of exons to produce mature mRNA transcripts. Variability in this tightly regulated process is… (more)

Subjects/Keywords: Splicing; Exon skipping; Gene therapy  – Methodology; Growth Hormone; RNAi; RNA editing; Somatotropin; Genetic engineering

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APA (6th Edition):

Shariat, N. (2008). Growth Hormone Splicing and Treatment of Disease Using RNA Interference. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-02272008-094210/ ;

Chicago Manual of Style (16th Edition):

Shariat, Nikki. “Growth Hormone Splicing and Treatment of Disease Using RNA Interference.” 2008. Doctoral Dissertation, Vanderbilt University. Accessed December 07, 2019. http://etd.library.vanderbilt.edu/available/etd-02272008-094210/ ;.

MLA Handbook (7th Edition):

Shariat, Nikki. “Growth Hormone Splicing and Treatment of Disease Using RNA Interference.” 2008. Web. 07 Dec 2019.

Vancouver:

Shariat N. Growth Hormone Splicing and Treatment of Disease Using RNA Interference. [Internet] [Doctoral dissertation]. Vanderbilt University; 2008. [cited 2019 Dec 07]. Available from: http://etd.library.vanderbilt.edu/available/etd-02272008-094210/ ;.

Council of Science Editors:

Shariat N. Growth Hormone Splicing and Treatment of Disease Using RNA Interference. [Doctoral Dissertation]. Vanderbilt University; 2008. Available from: http://etd.library.vanderbilt.edu/available/etd-02272008-094210/ ;


Université Paris-Sud – Paris XI

19. Perronnet, Caroline. Etude de thérapies génique et pharmacologique visant à restaurer les capacités cognitives d’un modèle murin de la Dystrophie musculaire de Duchenne : Gene and pharmacological therapies to restore cognitive abilities of a mouse model of Duchenne muscular Dystrophy.

Degree: Docteur es, Neurosciences, 2011, Université Paris-Sud – Paris XI

L’objectif était d’évaluer l’efficacité de thérapies développées pour traiter la dystrophie musculaire de Duchenne (DMD, due à des mutations du gène de la dystrophine) dans… (more)

Subjects/Keywords: Dystrophie Musculaire de Duchenne; Souris mdx; Déficits cognitifs; Comportement; Cytosquelette; Dystrophine; Utrophine; Synapse; Clustering de récepteurs; Thérapie, Saut d’exon, Butyrate; Arginine, Oxyde nitrique.; Duchenne Muscular Dystrophy; Mdx mouse; Cognitive deficits; Behaviour; Cytoskeleton; Dystrophin; Utrophin; Synapse; Receptor clustering; Therapy; Exon skipping

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APA (6th Edition):

Perronnet, C. (2011). Etude de thérapies génique et pharmacologique visant à restaurer les capacités cognitives d’un modèle murin de la Dystrophie musculaire de Duchenne : Gene and pharmacological therapies to restore cognitive abilities of a mouse model of Duchenne muscular Dystrophy. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA112009

Chicago Manual of Style (16th Edition):

Perronnet, Caroline. “Etude de thérapies génique et pharmacologique visant à restaurer les capacités cognitives d’un modèle murin de la Dystrophie musculaire de Duchenne : Gene and pharmacological therapies to restore cognitive abilities of a mouse model of Duchenne muscular Dystrophy.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed December 07, 2019. http://www.theses.fr/2011PA112009.

MLA Handbook (7th Edition):

Perronnet, Caroline. “Etude de thérapies génique et pharmacologique visant à restaurer les capacités cognitives d’un modèle murin de la Dystrophie musculaire de Duchenne : Gene and pharmacological therapies to restore cognitive abilities of a mouse model of Duchenne muscular Dystrophy.” 2011. Web. 07 Dec 2019.

Vancouver:

Perronnet C. Etude de thérapies génique et pharmacologique visant à restaurer les capacités cognitives d’un modèle murin de la Dystrophie musculaire de Duchenne : Gene and pharmacological therapies to restore cognitive abilities of a mouse model of Duchenne muscular Dystrophy. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2019 Dec 07]. Available from: http://www.theses.fr/2011PA112009.

Council of Science Editors:

Perronnet C. Etude de thérapies génique et pharmacologique visant à restaurer les capacités cognitives d’un modèle murin de la Dystrophie musculaire de Duchenne : Gene and pharmacological therapies to restore cognitive abilities of a mouse model of Duchenne muscular Dystrophy. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA112009


University of Lund

20. Zhou, Yuedan. Mechanisms by which variants in the TCF7L2 gene increase the risk of developing Type 2 diabetes.

Degree: 2013, University of Lund

 Type 2 diabetes mellitus (T2DM) is a heterogeneous disease with a multifactorial aetiology comprising of genetic and environmental factors. The common variant most highly associated… (more)

Subjects/Keywords: Endokrinologi och diabetes; TCF7L2; rs7903146; Type 2 diabetes; β cell function; β cell survival; insulin secretion; proinsulin synthesis; proinsulin-to-insulin conversion; insulin maturation; open chromain; exon skipping; p53; TP53INP1; ISL1; MAFA; NEUROD1; PDX1; NKX6.1; PCSK1; PCSK2; SLC30A8; GWAS.

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APA (6th Edition):

Zhou, Y. (2013). Mechanisms by which variants in the TCF7L2 gene increase the risk of developing Type 2 diabetes. (Doctoral Dissertation). University of Lund. Retrieved from http://lup.lub.lu.se/record/4219548 ; http://portal.research.lu.se/ws/files/3847128/4219690.pdf

Chicago Manual of Style (16th Edition):

Zhou, Yuedan. “Mechanisms by which variants in the TCF7L2 gene increase the risk of developing Type 2 diabetes.” 2013. Doctoral Dissertation, University of Lund. Accessed December 07, 2019. http://lup.lub.lu.se/record/4219548 ; http://portal.research.lu.se/ws/files/3847128/4219690.pdf.

MLA Handbook (7th Edition):

Zhou, Yuedan. “Mechanisms by which variants in the TCF7L2 gene increase the risk of developing Type 2 diabetes.” 2013. Web. 07 Dec 2019.

Vancouver:

Zhou Y. Mechanisms by which variants in the TCF7L2 gene increase the risk of developing Type 2 diabetes. [Internet] [Doctoral dissertation]. University of Lund; 2013. [cited 2019 Dec 07]. Available from: http://lup.lub.lu.se/record/4219548 ; http://portal.research.lu.se/ws/files/3847128/4219690.pdf.

Council of Science Editors:

Zhou Y. Mechanisms by which variants in the TCF7L2 gene increase the risk of developing Type 2 diabetes. [Doctoral Dissertation]. University of Lund; 2013. Available from: http://lup.lub.lu.se/record/4219548 ; http://portal.research.lu.se/ws/files/3847128/4219690.pdf


University of Debrecen

21. Bsoul, Ala. Pharmacotherapy of Duchenne Muscular Dystrophy .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

 Duchenne Muscular Dystrophy (DMD) is an X-linked recessive monogenic disease characterized by a rapid lethal course featured by progressive muscle atrophy and weakness. In this… (more)

Subjects/Keywords: DMD Gene Therapy; Exon skipping Therapy; Myostatin Inhibitors in DMD; Corticosteroids in DMD; Stop Codon Read-through Agents

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APA (6th Edition):

Bsoul, A. (n.d.). Pharmacotherapy of Duchenne Muscular Dystrophy . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/273729

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bsoul, Ala. “Pharmacotherapy of Duchenne Muscular Dystrophy .” Thesis, University of Debrecen. Accessed December 07, 2019. http://hdl.handle.net/2437/273729.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bsoul, Ala. “Pharmacotherapy of Duchenne Muscular Dystrophy .” Web. 07 Dec 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Bsoul A. Pharmacotherapy of Duchenne Muscular Dystrophy . [Internet] [Thesis]. University of Debrecen; [cited 2019 Dec 07]. Available from: http://hdl.handle.net/2437/273729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Bsoul A. Pharmacotherapy of Duchenne Muscular Dystrophy . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/273729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Georgia

22. Yang, Lixing. Intrinsic and extrinsic evolution of Helitrons in flowering plant genomes.

Degree: PhD, Genetics, 2009, University of Georgia

 Helitrons are a recently discovered class of eukaryotic transposable elements that are believed to transpose by a rolling circle mechanism. Because Helitrons frequently acquire and… (more)

Subjects/Keywords: exon shuffling

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APA (6th Edition):

Yang, L. (2009). Intrinsic and extrinsic evolution of Helitrons in flowering plant genomes. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/yang_lixing_200912_phd

Chicago Manual of Style (16th Edition):

Yang, Lixing. “Intrinsic and extrinsic evolution of Helitrons in flowering plant genomes.” 2009. Doctoral Dissertation, University of Georgia. Accessed December 07, 2019. http://purl.galileo.usg.edu/uga_etd/yang_lixing_200912_phd.

MLA Handbook (7th Edition):

Yang, Lixing. “Intrinsic and extrinsic evolution of Helitrons in flowering plant genomes.” 2009. Web. 07 Dec 2019.

Vancouver:

Yang L. Intrinsic and extrinsic evolution of Helitrons in flowering plant genomes. [Internet] [Doctoral dissertation]. University of Georgia; 2009. [cited 2019 Dec 07]. Available from: http://purl.galileo.usg.edu/uga_etd/yang_lixing_200912_phd.

Council of Science Editors:

Yang L. Intrinsic and extrinsic evolution of Helitrons in flowering plant genomes. [Doctoral Dissertation]. University of Georgia; 2009. Available from: http://purl.galileo.usg.edu/uga_etd/yang_lixing_200912_phd


Tulane University

23. Zhou, Yu. Gene expression array analysis for female osteoporosis.

Degree: 2018, Tulane University

Osteoporosis is a prevalent bone metabolic disease characterized by bone fragility. As a key pathophysiological mechanism, the disease is caused by excessive bone resorption (by… (more)

Subjects/Keywords: exon array

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APA (6th Edition):

Zhou, Y. (2018). Gene expression array analysis for female osteoporosis. (Thesis). Tulane University. Retrieved from https://digitallibrary.tulane.edu/islandora/object/tulane:79048

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhou, Yu. “Gene expression array analysis for female osteoporosis.” 2018. Thesis, Tulane University. Accessed December 07, 2019. https://digitallibrary.tulane.edu/islandora/object/tulane:79048.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhou, Yu. “Gene expression array analysis for female osteoporosis.” 2018. Web. 07 Dec 2019.

Vancouver:

Zhou Y. Gene expression array analysis for female osteoporosis. [Internet] [Thesis]. Tulane University; 2018. [cited 2019 Dec 07]. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:79048.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhou Y. Gene expression array analysis for female osteoporosis. [Thesis]. Tulane University; 2018. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:79048

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Tessoulin, Benoît. Identification par séquençage de l'exome de la dérégulation des voies de signalisation dans le myélome multiple et leurs conséquences fonctionnelles, notamment sur la voie p53 : Assessment by Whole Exon Sequencing of pathway dysregulations in Multiple Myeloma and their functional impacts, notably on p53 pathway.

Degree: Docteur es, Biologie cellulaire, 2018, Nantes

Au sein des hémopathies malignes B, les plasmocytoses malignes (myélome multiple [MM) et leucémie à plasmocyte [PCLI) occupent une place particulière par leur biologie et… (more)

Subjects/Keywords: Whole Exon Sequencing

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APA (6th Edition):

Tessoulin, B. (2018). Identification par séquençage de l'exome de la dérégulation des voies de signalisation dans le myélome multiple et leurs conséquences fonctionnelles, notamment sur la voie p53 : Assessment by Whole Exon Sequencing of pathway dysregulations in Multiple Myeloma and their functional impacts, notably on p53 pathway. (Doctoral Dissertation). Nantes. Retrieved from http://www.theses.fr/2018NANT1027

Chicago Manual of Style (16th Edition):

Tessoulin, Benoît. “Identification par séquençage de l'exome de la dérégulation des voies de signalisation dans le myélome multiple et leurs conséquences fonctionnelles, notamment sur la voie p53 : Assessment by Whole Exon Sequencing of pathway dysregulations in Multiple Myeloma and their functional impacts, notably on p53 pathway.” 2018. Doctoral Dissertation, Nantes. Accessed December 07, 2019. http://www.theses.fr/2018NANT1027.

MLA Handbook (7th Edition):

Tessoulin, Benoît. “Identification par séquençage de l'exome de la dérégulation des voies de signalisation dans le myélome multiple et leurs conséquences fonctionnelles, notamment sur la voie p53 : Assessment by Whole Exon Sequencing of pathway dysregulations in Multiple Myeloma and their functional impacts, notably on p53 pathway.” 2018. Web. 07 Dec 2019.

Vancouver:

Tessoulin B. Identification par séquençage de l'exome de la dérégulation des voies de signalisation dans le myélome multiple et leurs conséquences fonctionnelles, notamment sur la voie p53 : Assessment by Whole Exon Sequencing of pathway dysregulations in Multiple Myeloma and their functional impacts, notably on p53 pathway. [Internet] [Doctoral dissertation]. Nantes; 2018. [cited 2019 Dec 07]. Available from: http://www.theses.fr/2018NANT1027.

Council of Science Editors:

Tessoulin B. Identification par séquençage de l'exome de la dérégulation des voies de signalisation dans le myélome multiple et leurs conséquences fonctionnelles, notamment sur la voie p53 : Assessment by Whole Exon Sequencing of pathway dysregulations in Multiple Myeloma and their functional impacts, notably on p53 pathway. [Doctoral Dissertation]. Nantes; 2018. Available from: http://www.theses.fr/2018NANT1027

25. Marquette, Amélie. Signalisation et oncogenèse dans le mélanome : Teaching computer knowledge to beginners, in scientific secondary school and university in France : a comparative approach.

Degree: Docteur es, Immunologie, 2009, Université Paris-Est

Le mélanome, la tumeur cutanée la plus agressive, est devenu un problème majeur de santé publique dans de nombreux pays. Diagnostiqué précocement, il peut être… (more)

Subjects/Keywords: Mélanome; Signalisation; Stratégie thérapeutique; Melanoma; Signaling; Therapeutic strategy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Marquette, A. (2009). Signalisation et oncogenèse dans le mélanome : Teaching computer knowledge to beginners, in scientific secondary school and university in France : a comparative approach. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2009PEST0079

Chicago Manual of Style (16th Edition):

Marquette, Amélie. “Signalisation et oncogenèse dans le mélanome : Teaching computer knowledge to beginners, in scientific secondary school and university in France : a comparative approach.” 2009. Doctoral Dissertation, Université Paris-Est. Accessed December 07, 2019. http://www.theses.fr/2009PEST0079.

MLA Handbook (7th Edition):

Marquette, Amélie. “Signalisation et oncogenèse dans le mélanome : Teaching computer knowledge to beginners, in scientific secondary school and university in France : a comparative approach.” 2009. Web. 07 Dec 2019.

Vancouver:

Marquette A. Signalisation et oncogenèse dans le mélanome : Teaching computer knowledge to beginners, in scientific secondary school and university in France : a comparative approach. [Internet] [Doctoral dissertation]. Université Paris-Est; 2009. [cited 2019 Dec 07]. Available from: http://www.theses.fr/2009PEST0079.

Council of Science Editors:

Marquette A. Signalisation et oncogenèse dans le mélanome : Teaching computer knowledge to beginners, in scientific secondary school and university in France : a comparative approach. [Doctoral Dissertation]. Université Paris-Est; 2009. Available from: http://www.theses.fr/2009PEST0079


California State University – Sacramento

26. Shaker, Isaac. Intron mediated enhancement of gene expression in C. elegans.

Degree: MS, Biological Sciences (Molecular and Cellular Biology, 2017, California State University – Sacramento

 Introns can increase cytoplasmic mRNA levels through an as yet poorly understood mechanism known as intron mediated enhancement (IME). IME has been well studied in… (more)

Subjects/Keywords: RNA; EJC; GFP; Exon

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APA (6th Edition):

Shaker, I. (2017). Intron mediated enhancement of gene expression in C. elegans. (Masters Thesis). California State University – Sacramento. Retrieved from http://hdl.handle.net/10211.3/194686

Chicago Manual of Style (16th Edition):

Shaker, Isaac. “Intron mediated enhancement of gene expression in C. elegans.” 2017. Masters Thesis, California State University – Sacramento. Accessed December 07, 2019. http://hdl.handle.net/10211.3/194686.

MLA Handbook (7th Edition):

Shaker, Isaac. “Intron mediated enhancement of gene expression in C. elegans.” 2017. Web. 07 Dec 2019.

Vancouver:

Shaker I. Intron mediated enhancement of gene expression in C. elegans. [Internet] [Masters thesis]. California State University – Sacramento; 2017. [cited 2019 Dec 07]. Available from: http://hdl.handle.net/10211.3/194686.

Council of Science Editors:

Shaker I. Intron mediated enhancement of gene expression in C. elegans. [Masters Thesis]. California State University – Sacramento; 2017. Available from: http://hdl.handle.net/10211.3/194686

27. Termonen, Anniina. Hyppien koulupäivä aktiivisemmaksi : Suomen Voimisteluliiton Rope Skipping kouluille -kehittämisprosessi .

Degree: 2016, Theseus

 Tämän opinnäytetyön tavoitteena oli kehittää Rope Skippingiä alakoulujen käyttöön ja kerätä tietoa sen toimivuudesta ja innostavuudesta alakouluikäisten aktivointikeinona. Muita tavoit-teita oli selvittää millainen materiaali Rope… (more)

Subjects/Keywords: Rope Skipping; lasten liikunta; aktiivisuus; koululiikunta; välituntiliikunta

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Termonen, A. (2016). Hyppien koulupäivä aktiivisemmaksi : Suomen Voimisteluliiton Rope Skipping kouluille -kehittämisprosessi . (Thesis). Theseus. Retrieved from http://www.theseus.fi/handle/10024/117519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Termonen, Anniina. “Hyppien koulupäivä aktiivisemmaksi : Suomen Voimisteluliiton Rope Skipping kouluille -kehittämisprosessi .” 2016. Thesis, Theseus. Accessed December 07, 2019. http://www.theseus.fi/handle/10024/117519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Termonen, Anniina. “Hyppien koulupäivä aktiivisemmaksi : Suomen Voimisteluliiton Rope Skipping kouluille -kehittämisprosessi .” 2016. Web. 07 Dec 2019.

Vancouver:

Termonen A. Hyppien koulupäivä aktiivisemmaksi : Suomen Voimisteluliiton Rope Skipping kouluille -kehittämisprosessi . [Internet] [Thesis]. Theseus; 2016. [cited 2019 Dec 07]. Available from: http://www.theseus.fi/handle/10024/117519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Termonen A. Hyppien koulupäivä aktiivisemmaksi : Suomen Voimisteluliiton Rope Skipping kouluille -kehittämisprosessi . [Thesis]. Theseus; 2016. Available from: http://www.theseus.fi/handle/10024/117519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Azevedo, Regina Maria. O discurso terapêutico de Milton Erickson: uma análise à luz dos padrões da programação neurolinguística.

Degree: PhD, Psicologia Social, 2012, University of São Paulo

Este estudo apresenta o trabalho do psicanalista e hipnoterapeuta americano Milton Hyland Erickson a partir de seus dados biográficos e de sua relevância para a… (more)

Subjects/Keywords: Discourse; Discurso; Hipnose; Hypnosis; Milton Erickson; Milton Erickson; Neuro- Linguistic Programming (NLP); Programação Neurolinguística (PNL); Terapia estratégica; Therapeutic strategy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Azevedo, R. M. (2012). O discurso terapêutico de Milton Erickson: uma análise à luz dos padrões da programação neurolinguística. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/47/47134/tde-05102012-123033/ ;

Chicago Manual of Style (16th Edition):

Azevedo, Regina Maria. “O discurso terapêutico de Milton Erickson: uma análise à luz dos padrões da programação neurolinguística.” 2012. Doctoral Dissertation, University of São Paulo. Accessed December 07, 2019. http://www.teses.usp.br/teses/disponiveis/47/47134/tde-05102012-123033/ ;.

MLA Handbook (7th Edition):

Azevedo, Regina Maria. “O discurso terapêutico de Milton Erickson: uma análise à luz dos padrões da programação neurolinguística.” 2012. Web. 07 Dec 2019.

Vancouver:

Azevedo RM. O discurso terapêutico de Milton Erickson: uma análise à luz dos padrões da programação neurolinguística. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2019 Dec 07]. Available from: http://www.teses.usp.br/teses/disponiveis/47/47134/tde-05102012-123033/ ;.

Council of Science Editors:

Azevedo RM. O discurso terapêutico de Milton Erickson: uma análise à luz dos padrões da programação neurolinguística. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/47/47134/tde-05102012-123033/ ;

29. Tonin, Yann. Développement d'une stratégie thérapeutique anti-réplicative via l'exploitation de la voie d'import des ARN dans les mitochondries humaines : Development of an anti-replicative strategy by exploiting RNA import pathway into human mitochondria.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2013, Université de Strasbourg

Les mitochondries sont impliquées dans de nombreuses voies métaboliques, et des mutations au sein de leur génome (ADNmt) conduisent à l’apparition de nombreuses pathologies. A… (more)

Subjects/Keywords: Mitochondrie; Import d'ARN; Stratégie thérapeutique; Oligonucléotides antiréplicatif; Hétéroplasmie; Mitochondria; RNA import; Oligonucleotides; Therapeutic strategy; Heteroplasmy; Anti-replicative; 572.8

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tonin, Y. (2013). Développement d'une stratégie thérapeutique anti-réplicative via l'exploitation de la voie d'import des ARN dans les mitochondries humaines : Development of an anti-replicative strategy by exploiting RNA import pathway into human mitochondria. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2013STRAJ093

Chicago Manual of Style (16th Edition):

Tonin, Yann. “Développement d'une stratégie thérapeutique anti-réplicative via l'exploitation de la voie d'import des ARN dans les mitochondries humaines : Development of an anti-replicative strategy by exploiting RNA import pathway into human mitochondria.” 2013. Doctoral Dissertation, Université de Strasbourg. Accessed December 07, 2019. http://www.theses.fr/2013STRAJ093.

MLA Handbook (7th Edition):

Tonin, Yann. “Développement d'une stratégie thérapeutique anti-réplicative via l'exploitation de la voie d'import des ARN dans les mitochondries humaines : Development of an anti-replicative strategy by exploiting RNA import pathway into human mitochondria.” 2013. Web. 07 Dec 2019.

Vancouver:

Tonin Y. Développement d'une stratégie thérapeutique anti-réplicative via l'exploitation de la voie d'import des ARN dans les mitochondries humaines : Development of an anti-replicative strategy by exploiting RNA import pathway into human mitochondria. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2013. [cited 2019 Dec 07]. Available from: http://www.theses.fr/2013STRAJ093.

Council of Science Editors:

Tonin Y. Développement d'une stratégie thérapeutique anti-réplicative via l'exploitation de la voie d'import des ARN dans les mitochondries humaines : Development of an anti-replicative strategy by exploiting RNA import pathway into human mitochondria. [Doctoral Dissertation]. Université de Strasbourg; 2013. Available from: http://www.theses.fr/2013STRAJ093

30. Vizcaíno Cela, Leticia. Abordaje de la infección por VIH en personas mayores: efectos, causas y estrategia terapéutica .

Degree: 2015, Universidad da Coruña

 [Resumen] Introducción: Debido a los avances en el tratamiento de la infección por VIH, cada vez son más las personas que envejecen en esta situación.… (more)

Subjects/Keywords: VIH/SIDA; Personas mayores; Efectos negativos; Causa; Estrategia terapéutica; HIV/AIDS; Elderly people; Negative effects; Cause; Therapeutic strategy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vizcaíno Cela, L. (2015). Abordaje de la infección por VIH en personas mayores: efectos, causas y estrategia terapéutica . (Masters Thesis). Universidad da Coruña. Retrieved from http://hdl.handle.net/2183/14593

Chicago Manual of Style (16th Edition):

Vizcaíno Cela, Leticia. “Abordaje de la infección por VIH en personas mayores: efectos, causas y estrategia terapéutica .” 2015. Masters Thesis, Universidad da Coruña. Accessed December 07, 2019. http://hdl.handle.net/2183/14593.

MLA Handbook (7th Edition):

Vizcaíno Cela, Leticia. “Abordaje de la infección por VIH en personas mayores: efectos, causas y estrategia terapéutica .” 2015. Web. 07 Dec 2019.

Vancouver:

Vizcaíno Cela L. Abordaje de la infección por VIH en personas mayores: efectos, causas y estrategia terapéutica . [Internet] [Masters thesis]. Universidad da Coruña; 2015. [cited 2019 Dec 07]. Available from: http://hdl.handle.net/2183/14593.

Council of Science Editors:

Vizcaíno Cela L. Abordaje de la infección por VIH en personas mayores: efectos, causas y estrategia terapéutica . [Masters Thesis]. Universidad da Coruña; 2015. Available from: http://hdl.handle.net/2183/14593

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