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You searched for subject:(Exocytosis). Showing records 1 – 30 of 223 total matches.

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University of Minnesota

1. Finkenstaedt-Quinn, Solaire. A Study of the Regulation Mechanisms of Platelet Activation.

Degree: PhD, Chemistry, 2016, University of Minnesota

 Platelets play an important role in maintaining hemostasis in the body. As they circulate through the blood stream, platelets receive signals from other cells that… (more)

Subjects/Keywords: Exocytosis; Microscopy; Platelet

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APA (6th Edition):

Finkenstaedt-Quinn, S. (2016). A Study of the Regulation Mechanisms of Platelet Activation. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/185613

Chicago Manual of Style (16th Edition):

Finkenstaedt-Quinn, Solaire. “A Study of the Regulation Mechanisms of Platelet Activation.” 2016. Doctoral Dissertation, University of Minnesota. Accessed October 30, 2020. http://hdl.handle.net/11299/185613.

MLA Handbook (7th Edition):

Finkenstaedt-Quinn, Solaire. “A Study of the Regulation Mechanisms of Platelet Activation.” 2016. Web. 30 Oct 2020.

Vancouver:

Finkenstaedt-Quinn S. A Study of the Regulation Mechanisms of Platelet Activation. [Internet] [Doctoral dissertation]. University of Minnesota; 2016. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/11299/185613.

Council of Science Editors:

Finkenstaedt-Quinn S. A Study of the Regulation Mechanisms of Platelet Activation. [Doctoral Dissertation]. University of Minnesota; 2016. Available from: http://hdl.handle.net/11299/185613


Rutgers University

2. Milberg, Oleg, 1983-. The role and regulation of an actomyosin complex in membrane remodeling during regulated exocytosis in a live rodent model system.

Degree: PhD, Chemical and Biochemical Engineering, 2015, Rutgers University

Our lab has developed a subcellular imaging technique based on intravital microscopy (IVM) to study the highly conserved process of regulated exocytosis, as it occurs… (more)

Subjects/Keywords: Exocytosis; Cell membranes

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APA (6th Edition):

Milberg, Oleg, 1. (2015). The role and regulation of an actomyosin complex in membrane remodeling during regulated exocytosis in a live rodent model system. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/48801/

Chicago Manual of Style (16th Edition):

Milberg, Oleg, 1983-. “The role and regulation of an actomyosin complex in membrane remodeling during regulated exocytosis in a live rodent model system.” 2015. Doctoral Dissertation, Rutgers University. Accessed October 30, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/48801/.

MLA Handbook (7th Edition):

Milberg, Oleg, 1983-. “The role and regulation of an actomyosin complex in membrane remodeling during regulated exocytosis in a live rodent model system.” 2015. Web. 30 Oct 2020.

Vancouver:

Milberg, Oleg 1. The role and regulation of an actomyosin complex in membrane remodeling during regulated exocytosis in a live rodent model system. [Internet] [Doctoral dissertation]. Rutgers University; 2015. [cited 2020 Oct 30]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/48801/.

Council of Science Editors:

Milberg, Oleg 1. The role and regulation of an actomyosin complex in membrane remodeling during regulated exocytosis in a live rodent model system. [Doctoral Dissertation]. Rutgers University; 2015. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/48801/


University of Michigan

3. Cazares, Victor A. Molecular Mechanisms Orchestrating the Dynamics of Secretory Vesicle Pools.

Degree: PhD, Neuroscience, 2015, University of Michigan

 The secretion of chemical messengers via Ca2+-dependent exocytosis of vesicles is fundamental to a wide-range of physiological events. Rab GTPases and SNARE proteins govern the… (more)

Subjects/Keywords: Exocytosis; Neurosciences; Health Sciences

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APA (6th Edition):

Cazares, V. A. (2015). Molecular Mechanisms Orchestrating the Dynamics of Secretory Vesicle Pools. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/116755

Chicago Manual of Style (16th Edition):

Cazares, Victor A. “Molecular Mechanisms Orchestrating the Dynamics of Secretory Vesicle Pools.” 2015. Doctoral Dissertation, University of Michigan. Accessed October 30, 2020. http://hdl.handle.net/2027.42/116755.

MLA Handbook (7th Edition):

Cazares, Victor A. “Molecular Mechanisms Orchestrating the Dynamics of Secretory Vesicle Pools.” 2015. Web. 30 Oct 2020.

Vancouver:

Cazares VA. Molecular Mechanisms Orchestrating the Dynamics of Secretory Vesicle Pools. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/2027.42/116755.

Council of Science Editors:

Cazares VA. Molecular Mechanisms Orchestrating the Dynamics of Secretory Vesicle Pools. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/116755


Cornell University

4. Gadi, Deepti. Interplay Of Protein Kinase C, The Marcks Protein, And Phosphoinositides In Regulating Mast Cell Signaling.

Degree: PhD, Chemistry and Chemical Biology, 2012, Cornell University

 Stimulation of immunoglobulin E (IgE)-sensitized mast cells by multivalent antigen triggers a cascade of intracellular signaling events that results in granule exocytosis as a principal… (more)

Subjects/Keywords: Granule exocytosis; marcks; Phosphoinositides

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APA (6th Edition):

Gadi, D. (2012). Interplay Of Protein Kinase C, The Marcks Protein, And Phosphoinositides In Regulating Mast Cell Signaling. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/29275

Chicago Manual of Style (16th Edition):

Gadi, Deepti. “Interplay Of Protein Kinase C, The Marcks Protein, And Phosphoinositides In Regulating Mast Cell Signaling.” 2012. Doctoral Dissertation, Cornell University. Accessed October 30, 2020. http://hdl.handle.net/1813/29275.

MLA Handbook (7th Edition):

Gadi, Deepti. “Interplay Of Protein Kinase C, The Marcks Protein, And Phosphoinositides In Regulating Mast Cell Signaling.” 2012. Web. 30 Oct 2020.

Vancouver:

Gadi D. Interplay Of Protein Kinase C, The Marcks Protein, And Phosphoinositides In Regulating Mast Cell Signaling. [Internet] [Doctoral dissertation]. Cornell University; 2012. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/1813/29275.

Council of Science Editors:

Gadi D. Interplay Of Protein Kinase C, The Marcks Protein, And Phosphoinositides In Regulating Mast Cell Signaling. [Doctoral Dissertation]. Cornell University; 2012. Available from: http://hdl.handle.net/1813/29275


Cornell University

5. Kim, Brian. Microfabricated Devices For Direct Measurements Of Quantal Transmitter Release From Living Cells.

Degree: PhD, Biophysics, 2013, Cornell University

 Neurotransmitters are released in packets or quanta from vesicles that fuse with the cell membrane. The machinery proteins called Soluble N-ethylmaleimide-sensitive factor Attachment Protein Receptor… (more)

Subjects/Keywords: Biosensor; Exocytosis; CMOS Integrated Circuit

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APA (6th Edition):

Kim, B. (2013). Microfabricated Devices For Direct Measurements Of Quantal Transmitter Release From Living Cells. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/33818

Chicago Manual of Style (16th Edition):

Kim, Brian. “Microfabricated Devices For Direct Measurements Of Quantal Transmitter Release From Living Cells.” 2013. Doctoral Dissertation, Cornell University. Accessed October 30, 2020. http://hdl.handle.net/1813/33818.

MLA Handbook (7th Edition):

Kim, Brian. “Microfabricated Devices For Direct Measurements Of Quantal Transmitter Release From Living Cells.” 2013. Web. 30 Oct 2020.

Vancouver:

Kim B. Microfabricated Devices For Direct Measurements Of Quantal Transmitter Release From Living Cells. [Internet] [Doctoral dissertation]. Cornell University; 2013. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/1813/33818.

Council of Science Editors:

Kim B. Microfabricated Devices For Direct Measurements Of Quantal Transmitter Release From Living Cells. [Doctoral Dissertation]. Cornell University; 2013. Available from: http://hdl.handle.net/1813/33818


Cornell University

6. Donovan, Kirk. Coordinating The Transport Cycle Of The Myosin-V Motor Myo2 With Secretory Vesicle Delivery And Exocytosis.

Degree: PhD, Molecular and Cell Biology, 2014, Cornell University

 The polarization of proteins, lipids, and organelles within a eukaryotic cell allows for the spatial regulation of numerous biological processes. Saccharomyces cerevisiae displays exaggerated polarized… (more)

Subjects/Keywords: Myo2; exocytosis; myosin-V

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APA (6th Edition):

Donovan, K. (2014). Coordinating The Transport Cycle Of The Myosin-V Motor Myo2 With Secretory Vesicle Delivery And Exocytosis. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/38860

Chicago Manual of Style (16th Edition):

Donovan, Kirk. “Coordinating The Transport Cycle Of The Myosin-V Motor Myo2 With Secretory Vesicle Delivery And Exocytosis.” 2014. Doctoral Dissertation, Cornell University. Accessed October 30, 2020. http://hdl.handle.net/1813/38860.

MLA Handbook (7th Edition):

Donovan, Kirk. “Coordinating The Transport Cycle Of The Myosin-V Motor Myo2 With Secretory Vesicle Delivery And Exocytosis.” 2014. Web. 30 Oct 2020.

Vancouver:

Donovan K. Coordinating The Transport Cycle Of The Myosin-V Motor Myo2 With Secretory Vesicle Delivery And Exocytosis. [Internet] [Doctoral dissertation]. Cornell University; 2014. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/1813/38860.

Council of Science Editors:

Donovan K. Coordinating The Transport Cycle Of The Myosin-V Motor Myo2 With Secretory Vesicle Delivery And Exocytosis. [Doctoral Dissertation]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/38860


University of Edinburgh

7. Smyth, Annya Mary. Spatial, temporal and functional molecular architecture of the munc18-syntaxin interaction.

Degree: PhD, 2012, University of Edinburgh

 Regulation of soluble N-ethylmaleimide-sensitive fusion protein attachment protein receptors (SNARE) mediated exocytosis is dependent upon four key proteins; the vesicular SNARE synaptobrevin, target SNAREs SNAP-25… (more)

Subjects/Keywords: 572; munc18-1; syntaxin; exocytosis

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APA (6th Edition):

Smyth, A. M. (2012). Spatial, temporal and functional molecular architecture of the munc18-syntaxin interaction. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/9972

Chicago Manual of Style (16th Edition):

Smyth, Annya Mary. “Spatial, temporal and functional molecular architecture of the munc18-syntaxin interaction.” 2012. Doctoral Dissertation, University of Edinburgh. Accessed October 30, 2020. http://hdl.handle.net/1842/9972.

MLA Handbook (7th Edition):

Smyth, Annya Mary. “Spatial, temporal and functional molecular architecture of the munc18-syntaxin interaction.” 2012. Web. 30 Oct 2020.

Vancouver:

Smyth AM. Spatial, temporal and functional molecular architecture of the munc18-syntaxin interaction. [Internet] [Doctoral dissertation]. University of Edinburgh; 2012. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/1842/9972.

Council of Science Editors:

Smyth AM. Spatial, temporal and functional molecular architecture of the munc18-syntaxin interaction. [Doctoral Dissertation]. University of Edinburgh; 2012. Available from: http://hdl.handle.net/1842/9972


University of Minnesota

8. Koseoglu, Secil. Exploring the fundamentals of platelet granular storage and secretion at the single cell level.

Degree: PhD, Chemistry, 2013, University of Minnesota

 Platelets are critical cells in hemostasis and thrombosis, but they are also involved in many important physiological events including inflammation, host defense, wound healing and… (more)

Subjects/Keywords: Exocytosis; Microelectrodes; Platelet; Chemistry

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APA (6th Edition):

Koseoglu, S. (2013). Exploring the fundamentals of platelet granular storage and secretion at the single cell level. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/171414

Chicago Manual of Style (16th Edition):

Koseoglu, Secil. “Exploring the fundamentals of platelet granular storage and secretion at the single cell level.” 2013. Doctoral Dissertation, University of Minnesota. Accessed October 30, 2020. http://hdl.handle.net/11299/171414.

MLA Handbook (7th Edition):

Koseoglu, Secil. “Exploring the fundamentals of platelet granular storage and secretion at the single cell level.” 2013. Web. 30 Oct 2020.

Vancouver:

Koseoglu S. Exploring the fundamentals of platelet granular storage and secretion at the single cell level. [Internet] [Doctoral dissertation]. University of Minnesota; 2013. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/11299/171414.

Council of Science Editors:

Koseoglu S. Exploring the fundamentals of platelet granular storage and secretion at the single cell level. [Doctoral Dissertation]. University of Minnesota; 2013. Available from: http://hdl.handle.net/11299/171414


University of Southern California

9. Cai, Haijiang. Role of complexin in regulated exocytosis.

Degree: PhD, Physiology & Biophysics, 2007, University of Southern California

 Properly regulated exocytosis of neurotransmitters and hormones from membrane-delimited vesicles is essential for normal cellular function. At least 3 distinct steps are involved in regulated… (more)

Subjects/Keywords: complexin; exocytosis

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APA (6th Edition):

Cai, H. (2007). Role of complexin in regulated exocytosis. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/480071/rec/5635

Chicago Manual of Style (16th Edition):

Cai, Haijiang. “Role of complexin in regulated exocytosis.” 2007. Doctoral Dissertation, University of Southern California. Accessed October 30, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/480071/rec/5635.

MLA Handbook (7th Edition):

Cai, Haijiang. “Role of complexin in regulated exocytosis.” 2007. Web. 30 Oct 2020.

Vancouver:

Cai H. Role of complexin in regulated exocytosis. [Internet] [Doctoral dissertation]. University of Southern California; 2007. [cited 2020 Oct 30]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/480071/rec/5635.

Council of Science Editors:

Cai H. Role of complexin in regulated exocytosis. [Doctoral Dissertation]. University of Southern California; 2007. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/480071/rec/5635


University of Alberta

10. Baier, Alicia. The Role of Rho GTPases, Rac1 and Rac2, in Mast Cell Exocytosis.

Degree: MS, Department of Cell Biology, 2012, University of Alberta

 Mast cells are tissue-resident immune cells that undergo exocytosis upon activation, releasing potent immunoregulatory molecules that initiate inflammatory responses. Here, I investigated the role of… (more)

Subjects/Keywords: exocytosis; Rac1 and Rac2 GTPases; mast cell

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APA (6th Edition):

Baier, A. (2012). The Role of Rho GTPases, Rac1 and Rac2, in Mast Cell Exocytosis. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/47429912h

Chicago Manual of Style (16th Edition):

Baier, Alicia. “The Role of Rho GTPases, Rac1 and Rac2, in Mast Cell Exocytosis.” 2012. Masters Thesis, University of Alberta. Accessed October 30, 2020. https://era.library.ualberta.ca/files/47429912h.

MLA Handbook (7th Edition):

Baier, Alicia. “The Role of Rho GTPases, Rac1 and Rac2, in Mast Cell Exocytosis.” 2012. Web. 30 Oct 2020.

Vancouver:

Baier A. The Role of Rho GTPases, Rac1 and Rac2, in Mast Cell Exocytosis. [Internet] [Masters thesis]. University of Alberta; 2012. [cited 2020 Oct 30]. Available from: https://era.library.ualberta.ca/files/47429912h.

Council of Science Editors:

Baier A. The Role of Rho GTPases, Rac1 and Rac2, in Mast Cell Exocytosis. [Masters Thesis]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/47429912h


Cornell University

11. Rahl, Peter. The Elongator Complex Negatively Regulates Polarized Secretion.

Degree: 2007, Cornell University

 Intracellular transport is fundamental for eukaryotic life and the function of regulators of transport is evolutionarily conserved. Rab proteins are a family of key regulators… (more)

Subjects/Keywords: Exocytosis; Rab GTPase

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APA (6th Edition):

Rahl, P. (2007). The Elongator Complex Negatively Regulates Polarized Secretion. (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/8413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rahl, Peter. “The Elongator Complex Negatively Regulates Polarized Secretion.” 2007. Thesis, Cornell University. Accessed October 30, 2020. http://hdl.handle.net/1813/8413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rahl, Peter. “The Elongator Complex Negatively Regulates Polarized Secretion.” 2007. Web. 30 Oct 2020.

Vancouver:

Rahl P. The Elongator Complex Negatively Regulates Polarized Secretion. [Internet] [Thesis]. Cornell University; 2007. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/1813/8413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rahl P. The Elongator Complex Negatively Regulates Polarized Secretion. [Thesis]. Cornell University; 2007. Available from: http://hdl.handle.net/1813/8413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

12. Jewell, Mark Langley. Regulation of Ca2+ channels and exocytosis by receptors for prostaglandin E2.

Degree: PhD, Pharmacology, 2013, Vanderbilt University

 Adrenal chromaffin cells release catecholamines, neuropeptides and other hormones to maintain cardiovascular and metabolic homeostasis, and tune the physiological response to acute stress. As such,… (more)

Subjects/Keywords: amperometry; PGE2; exocytosis; GPCRs; calcium channels

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APA (6th Edition):

Jewell, M. L. (2013). Regulation of Ca2+ channels and exocytosis by receptors for prostaglandin E2. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13495

Chicago Manual of Style (16th Edition):

Jewell, Mark Langley. “Regulation of Ca2+ channels and exocytosis by receptors for prostaglandin E2.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed October 30, 2020. http://hdl.handle.net/1803/13495.

MLA Handbook (7th Edition):

Jewell, Mark Langley. “Regulation of Ca2+ channels and exocytosis by receptors for prostaglandin E2.” 2013. Web. 30 Oct 2020.

Vancouver:

Jewell ML. Regulation of Ca2+ channels and exocytosis by receptors for prostaglandin E2. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/1803/13495.

Council of Science Editors:

Jewell ML. Regulation of Ca2+ channels and exocytosis by receptors for prostaglandin E2. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/13495


Texas A&M University

13. Jaramillo Hernandez, Ana Maria. Molecular and Functional Identification of Two Mucin Secretory Pathways.

Degree: PhD, Medical Sciences, 2018, Texas A&M University

 In airways, secreted mucins absorb large volumes of water to form viscoelastic mucus, which is then propelled proximally by ciliary beating and swallowed. Mucins are… (more)

Subjects/Keywords: Munc18; mucin; exocytosis; asthma; cystic fibrosis; mucus

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APA (6th Edition):

Jaramillo Hernandez, A. M. (2018). Molecular and Functional Identification of Two Mucin Secretory Pathways. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/174327

Chicago Manual of Style (16th Edition):

Jaramillo Hernandez, Ana Maria. “Molecular and Functional Identification of Two Mucin Secretory Pathways.” 2018. Doctoral Dissertation, Texas A&M University. Accessed October 30, 2020. http://hdl.handle.net/1969.1/174327.

MLA Handbook (7th Edition):

Jaramillo Hernandez, Ana Maria. “Molecular and Functional Identification of Two Mucin Secretory Pathways.” 2018. Web. 30 Oct 2020.

Vancouver:

Jaramillo Hernandez AM. Molecular and Functional Identification of Two Mucin Secretory Pathways. [Internet] [Doctoral dissertation]. Texas A&M University; 2018. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/1969.1/174327.

Council of Science Editors:

Jaramillo Hernandez AM. Molecular and Functional Identification of Two Mucin Secretory Pathways. [Doctoral Dissertation]. Texas A&M University; 2018. Available from: http://hdl.handle.net/1969.1/174327


University of Toronto

14. Koo, Ellen. Syntaxin-3 Regulates Biphasic Glucose Stimulated Insulin Secretion in the Pancreatic Beta Cell.

Degree: 2010, University of Toronto

Our study aims to investigate the role of Syntaxin-3 in glucose stimulated insulin secretion (GSIS) and how it regulates the recruitment to plasma membrane and/or… (more)

Subjects/Keywords: Insulin Secretion; Syntaxin; SNAREs; Exocytosis; 0719

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APA (6th Edition):

Koo, E. (2010). Syntaxin-3 Regulates Biphasic Glucose Stimulated Insulin Secretion in the Pancreatic Beta Cell. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25732

Chicago Manual of Style (16th Edition):

Koo, Ellen. “Syntaxin-3 Regulates Biphasic Glucose Stimulated Insulin Secretion in the Pancreatic Beta Cell.” 2010. Masters Thesis, University of Toronto. Accessed October 30, 2020. http://hdl.handle.net/1807/25732.

MLA Handbook (7th Edition):

Koo, Ellen. “Syntaxin-3 Regulates Biphasic Glucose Stimulated Insulin Secretion in the Pancreatic Beta Cell.” 2010. Web. 30 Oct 2020.

Vancouver:

Koo E. Syntaxin-3 Regulates Biphasic Glucose Stimulated Insulin Secretion in the Pancreatic Beta Cell. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/1807/25732.

Council of Science Editors:

Koo E. Syntaxin-3 Regulates Biphasic Glucose Stimulated Insulin Secretion in the Pancreatic Beta Cell. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25732


University of Toronto

15. Fernandez, Nestor Alejandro. Attempts to Elucidate the Role of SNAP23 in Regulated and Pathological Exocytosis in Pancreatic Acinar Cells Using an Inducible SNAP23 Knockout Mouse.

Degree: 2010, University of Toronto

One contentious issue regarding pancreatic acinar exocytosis concerns which SNAP25 isoform (SNAP23/29/47) mediates the various fusion events in this cell type. Based on dominant-negative over-expression… (more)

Subjects/Keywords: pancreatic acinar cell; exocytosis; SNAP23; pHluorin; 0719

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APA (6th Edition):

Fernandez, N. A. (2010). Attempts to Elucidate the Role of SNAP23 in Regulated and Pathological Exocytosis in Pancreatic Acinar Cells Using an Inducible SNAP23 Knockout Mouse. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25576

Chicago Manual of Style (16th Edition):

Fernandez, Nestor Alejandro. “Attempts to Elucidate the Role of SNAP23 in Regulated and Pathological Exocytosis in Pancreatic Acinar Cells Using an Inducible SNAP23 Knockout Mouse.” 2010. Masters Thesis, University of Toronto. Accessed October 30, 2020. http://hdl.handle.net/1807/25576.

MLA Handbook (7th Edition):

Fernandez, Nestor Alejandro. “Attempts to Elucidate the Role of SNAP23 in Regulated and Pathological Exocytosis in Pancreatic Acinar Cells Using an Inducible SNAP23 Knockout Mouse.” 2010. Web. 30 Oct 2020.

Vancouver:

Fernandez NA. Attempts to Elucidate the Role of SNAP23 in Regulated and Pathological Exocytosis in Pancreatic Acinar Cells Using an Inducible SNAP23 Knockout Mouse. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/1807/25576.

Council of Science Editors:

Fernandez NA. Attempts to Elucidate the Role of SNAP23 in Regulated and Pathological Exocytosis in Pancreatic Acinar Cells Using an Inducible SNAP23 Knockout Mouse. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25576

16. Zhang, Yingying. Smcr8 Collaborates With the ALS Linked Gene C9orf72 to Inhibit Autoimmunity and to Regulate Lysosome Exocytosis.

Degree: PhD, 2018, Harvard University

The (GGGGCC)n hexanucleotide repeat expansion in the first intron of C9ORF72 is the most common genetic cause of ALS\FTLD. This mutation causes reduction of C9ORF72… (more)

Subjects/Keywords: ALS; C9orf72; Smcr8; autoimmunity; lysosome exocytosis

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APA (6th Edition):

Zhang, Y. (2018). Smcr8 Collaborates With the ALS Linked Gene C9orf72 to Inhibit Autoimmunity and to Regulate Lysosome Exocytosis. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:42015065

Chicago Manual of Style (16th Edition):

Zhang, Yingying. “Smcr8 Collaborates With the ALS Linked Gene C9orf72 to Inhibit Autoimmunity and to Regulate Lysosome Exocytosis.” 2018. Doctoral Dissertation, Harvard University. Accessed October 30, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:42015065.

MLA Handbook (7th Edition):

Zhang, Yingying. “Smcr8 Collaborates With the ALS Linked Gene C9orf72 to Inhibit Autoimmunity and to Regulate Lysosome Exocytosis.” 2018. Web. 30 Oct 2020.

Vancouver:

Zhang Y. Smcr8 Collaborates With the ALS Linked Gene C9orf72 to Inhibit Autoimmunity and to Regulate Lysosome Exocytosis. [Internet] [Doctoral dissertation]. Harvard University; 2018. [cited 2020 Oct 30]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42015065.

Council of Science Editors:

Zhang Y. Smcr8 Collaborates With the ALS Linked Gene C9orf72 to Inhibit Autoimmunity and to Regulate Lysosome Exocytosis. [Doctoral Dissertation]. Harvard University; 2018. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42015065


Harvard University

17. Wang, Huaiying. Molecular Mechanisms for Active Zone Assembly at Vertebrate Synapses.

Degree: PhD, 2019, Harvard University

Neurons are unique for their ability to transmit a presynaptic signal to postsynaptic target cells on a sub-millisecond timescale. This rapid and precise exchange of… (more)

Subjects/Keywords: Synaptic transmission; Vesicle exocytosis; Presynaptic active zone

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, H. (2019). Molecular Mechanisms for Active Zone Assembly at Vertebrate Synapses. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:42029509

Chicago Manual of Style (16th Edition):

Wang, Huaiying. “Molecular Mechanisms for Active Zone Assembly at Vertebrate Synapses.” 2019. Doctoral Dissertation, Harvard University. Accessed October 30, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:42029509.

MLA Handbook (7th Edition):

Wang, Huaiying. “Molecular Mechanisms for Active Zone Assembly at Vertebrate Synapses.” 2019. Web. 30 Oct 2020.

Vancouver:

Wang H. Molecular Mechanisms for Active Zone Assembly at Vertebrate Synapses. [Internet] [Doctoral dissertation]. Harvard University; 2019. [cited 2020 Oct 30]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42029509.

Council of Science Editors:

Wang H. Molecular Mechanisms for Active Zone Assembly at Vertebrate Synapses. [Doctoral Dissertation]. Harvard University; 2019. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42029509


Hong Kong University of Science and Technology

18. Liu, Yuxin. Pctaire1 phosphorylates N-ethylmaleimide sensitive fusion protein and regulates exocytosis.

Degree: 2006, Hong Kong University of Science and Technology

 Pctaire1, a member of the cyclin-dependent kinase (Cdk)-related kinase family, has recently been identified as a substrate of Cdk5, where Cdk5-mediated phosphorylation enhances Pctaire1 activity.… (more)

Subjects/Keywords: Protein kinases ; Exocytosis

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APA (6th Edition):

Liu, Y. (2006). Pctaire1 phosphorylates N-ethylmaleimide sensitive fusion protein and regulates exocytosis. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-2699 ; https://doi.org/10.14711/thesis-b924233 ; http://repository.ust.hk/ir/bitstream/1783.1-2699/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Yuxin. “Pctaire1 phosphorylates N-ethylmaleimide sensitive fusion protein and regulates exocytosis.” 2006. Thesis, Hong Kong University of Science and Technology. Accessed October 30, 2020. http://repository.ust.hk/ir/Record/1783.1-2699 ; https://doi.org/10.14711/thesis-b924233 ; http://repository.ust.hk/ir/bitstream/1783.1-2699/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Yuxin. “Pctaire1 phosphorylates N-ethylmaleimide sensitive fusion protein and regulates exocytosis.” 2006. Web. 30 Oct 2020.

Vancouver:

Liu Y. Pctaire1 phosphorylates N-ethylmaleimide sensitive fusion protein and regulates exocytosis. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2006. [cited 2020 Oct 30]. Available from: http://repository.ust.hk/ir/Record/1783.1-2699 ; https://doi.org/10.14711/thesis-b924233 ; http://repository.ust.hk/ir/bitstream/1783.1-2699/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu Y. Pctaire1 phosphorylates N-ethylmaleimide sensitive fusion protein and regulates exocytosis. [Thesis]. Hong Kong University of Science and Technology; 2006. Available from: http://repository.ust.hk/ir/Record/1783.1-2699 ; https://doi.org/10.14711/thesis-b924233 ; http://repository.ust.hk/ir/bitstream/1783.1-2699/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Florida International University

19. Pulido, Maria. Mechanism of Superoxide Mediated Regulation of Particle Uptake and Exocytosis by a GPI-anchored Superoxide Dismutase C in Dictyostelium.

Degree: MS, Biology, 2014, Florida International University

  Dictyostelium discoideum is a simple model organism that can be used to study endocytic pathways such as phagocytosis and macropinocytosis because of its homology… (more)

Subjects/Keywords: Dictyostelium discoideum; SodC; endocytosis; exocytosis; RasG

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APA (6th Edition):

Pulido, M. (2014). Mechanism of Superoxide Mediated Regulation of Particle Uptake and Exocytosis by a GPI-anchored Superoxide Dismutase C in Dictyostelium. (Thesis). Florida International University. Retrieved from https://digitalcommons.fiu.edu/etd/1540 ; 10.25148/etd.FI14071177 ; FI14071177

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pulido, Maria. “Mechanism of Superoxide Mediated Regulation of Particle Uptake and Exocytosis by a GPI-anchored Superoxide Dismutase C in Dictyostelium.” 2014. Thesis, Florida International University. Accessed October 30, 2020. https://digitalcommons.fiu.edu/etd/1540 ; 10.25148/etd.FI14071177 ; FI14071177.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pulido, Maria. “Mechanism of Superoxide Mediated Regulation of Particle Uptake and Exocytosis by a GPI-anchored Superoxide Dismutase C in Dictyostelium.” 2014. Web. 30 Oct 2020.

Vancouver:

Pulido M. Mechanism of Superoxide Mediated Regulation of Particle Uptake and Exocytosis by a GPI-anchored Superoxide Dismutase C in Dictyostelium. [Internet] [Thesis]. Florida International University; 2014. [cited 2020 Oct 30]. Available from: https://digitalcommons.fiu.edu/etd/1540 ; 10.25148/etd.FI14071177 ; FI14071177.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pulido M. Mechanism of Superoxide Mediated Regulation of Particle Uptake and Exocytosis by a GPI-anchored Superoxide Dismutase C in Dictyostelium. [Thesis]. Florida International University; 2014. Available from: https://digitalcommons.fiu.edu/etd/1540 ; 10.25148/etd.FI14071177 ; FI14071177

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

20. Bin, Na-Ryum. Unraveling the Molecular Mechanisms of Munc18 and Munc13 in Mast Cell Exocytosis.

Degree: PhD, 2017, University of Toronto

Exocytosis is a fundamental cellular process in which contents stored in vesicles are released out of cells via fusion of membranes. Exocytosis of secretory granules… (more)

Subjects/Keywords: Exocytosis; Mast cell; Munc13; Munc18; SNARE; 0719

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bin, N. (2017). Unraveling the Molecular Mechanisms of Munc18 and Munc13 in Mast Cell Exocytosis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/97097

Chicago Manual of Style (16th Edition):

Bin, Na-Ryum. “Unraveling the Molecular Mechanisms of Munc18 and Munc13 in Mast Cell Exocytosis.” 2017. Doctoral Dissertation, University of Toronto. Accessed October 30, 2020. http://hdl.handle.net/1807/97097.

MLA Handbook (7th Edition):

Bin, Na-Ryum. “Unraveling the Molecular Mechanisms of Munc18 and Munc13 in Mast Cell Exocytosis.” 2017. Web. 30 Oct 2020.

Vancouver:

Bin N. Unraveling the Molecular Mechanisms of Munc18 and Munc13 in Mast Cell Exocytosis. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/1807/97097.

Council of Science Editors:

Bin N. Unraveling the Molecular Mechanisms of Munc18 and Munc13 in Mast Cell Exocytosis. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/97097


University of Toronto

21. Han, Gayoung A. Molecular insights into the mechanisms underlying the pleiotropic functions of Munc18-1 in dense-core vesicle exocytosis.

Degree: 2013, University of Toronto

Exocytosis refers to a cellular process in which an intracellular vesicle fuses its membrane with the plasma membrane to release its contents to the extracellular… (more)

Subjects/Keywords: Munc18-1; exocytosis; dense-core vesicle; 0719

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APA (6th Edition):

Han, G. A. (2013). Molecular insights into the mechanisms underlying the pleiotropic functions of Munc18-1 in dense-core vesicle exocytosis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/70083

Chicago Manual of Style (16th Edition):

Han, Gayoung A. “Molecular insights into the mechanisms underlying the pleiotropic functions of Munc18-1 in dense-core vesicle exocytosis.” 2013. Doctoral Dissertation, University of Toronto. Accessed October 30, 2020. http://hdl.handle.net/1807/70083.

MLA Handbook (7th Edition):

Han, Gayoung A. “Molecular insights into the mechanisms underlying the pleiotropic functions of Munc18-1 in dense-core vesicle exocytosis.” 2013. Web. 30 Oct 2020.

Vancouver:

Han GA. Molecular insights into the mechanisms underlying the pleiotropic functions of Munc18-1 in dense-core vesicle exocytosis. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/1807/70083.

Council of Science Editors:

Han GA. Molecular insights into the mechanisms underlying the pleiotropic functions of Munc18-1 in dense-core vesicle exocytosis. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/70083


University of Minnesota

22. Gruba, Sarah. Exploration of Platelet and Mast Cell Communication: A Study of Membrane Lipids, Asthma, and Inflammation.

Degree: PhD, Chemistry, 2016, University of Minnesota

 This work examines how environmental factors such as lipid membrane concentration, opioid agonist exposure, and inflammatory diseases impact cell communication. It explores the use of… (more)

Subjects/Keywords: asthma; Exocytosis; inflammation; lipids; Mast cells; platelets

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APA (6th Edition):

Gruba, S. (2016). Exploration of Platelet and Mast Cell Communication: A Study of Membrane Lipids, Asthma, and Inflammation. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/199077

Chicago Manual of Style (16th Edition):

Gruba, Sarah. “Exploration of Platelet and Mast Cell Communication: A Study of Membrane Lipids, Asthma, and Inflammation.” 2016. Doctoral Dissertation, University of Minnesota. Accessed October 30, 2020. http://hdl.handle.net/11299/199077.

MLA Handbook (7th Edition):

Gruba, Sarah. “Exploration of Platelet and Mast Cell Communication: A Study of Membrane Lipids, Asthma, and Inflammation.” 2016. Web. 30 Oct 2020.

Vancouver:

Gruba S. Exploration of Platelet and Mast Cell Communication: A Study of Membrane Lipids, Asthma, and Inflammation. [Internet] [Doctoral dissertation]. University of Minnesota; 2016. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/11299/199077.

Council of Science Editors:

Gruba S. Exploration of Platelet and Mast Cell Communication: A Study of Membrane Lipids, Asthma, and Inflammation. [Doctoral Dissertation]. University of Minnesota; 2016. Available from: http://hdl.handle.net/11299/199077


University of Louisville

23. Lominadze, George, 1978-. Defining the mechanisms of neutrophil exocytosis using proteomic techniques.

Degree: PhD, 2005, University of Louisville

Exocytosis of intracellular granules is critical for conversion of inactive, circulating neutrophils to fully activated cells. The p38 MAPK pathway plays a central role in… (more)

Subjects/Keywords: Neutrophil; Exocytosis; Proteomic

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APA (6th Edition):

Lominadze, George, 1. (2005). Defining the mechanisms of neutrophil exocytosis using proteomic techniques. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/852 ; https://ir.library.louisville.edu/etd/852

Chicago Manual of Style (16th Edition):

Lominadze, George, 1978-. “Defining the mechanisms of neutrophil exocytosis using proteomic techniques.” 2005. Doctoral Dissertation, University of Louisville. Accessed October 30, 2020. 10.18297/etd/852 ; https://ir.library.louisville.edu/etd/852.

MLA Handbook (7th Edition):

Lominadze, George, 1978-. “Defining the mechanisms of neutrophil exocytosis using proteomic techniques.” 2005. Web. 30 Oct 2020.

Vancouver:

Lominadze, George 1. Defining the mechanisms of neutrophil exocytosis using proteomic techniques. [Internet] [Doctoral dissertation]. University of Louisville; 2005. [cited 2020 Oct 30]. Available from: 10.18297/etd/852 ; https://ir.library.louisville.edu/etd/852.

Council of Science Editors:

Lominadze, George 1. Defining the mechanisms of neutrophil exocytosis using proteomic techniques. [Doctoral Dissertation]. University of Louisville; 2005. Available from: 10.18297/etd/852 ; https://ir.library.louisville.edu/etd/852


University of Missouri – Columbia

24. Sen, Atanu. Quantification of cell attachment on different materials as candidate electrodes for measurement of quantal exocytosis.

Degree: 2008, University of Missouri – Columbia

 A high throughput lab-on-a-chip device is being developed for single cell capture for the purpose of high time resolution quantal exocytosis measurement with high probability… (more)

Subjects/Keywords: Exocytosis; BioMEMS; Catecholamines

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APA (6th Edition):

Sen, A. (2008). Quantification of cell attachment on different materials as candidate electrodes for measurement of quantal exocytosis. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/5722

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sen, Atanu. “Quantification of cell attachment on different materials as candidate electrodes for measurement of quantal exocytosis.” 2008. Thesis, University of Missouri – Columbia. Accessed October 30, 2020. http://hdl.handle.net/10355/5722.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sen, Atanu. “Quantification of cell attachment on different materials as candidate electrodes for measurement of quantal exocytosis.” 2008. Web. 30 Oct 2020.

Vancouver:

Sen A. Quantification of cell attachment on different materials as candidate electrodes for measurement of quantal exocytosis. [Internet] [Thesis]. University of Missouri – Columbia; 2008. [cited 2020 Oct 30]. Available from: http://hdl.handle.net/10355/5722.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sen A. Quantification of cell attachment on different materials as candidate electrodes for measurement of quantal exocytosis. [Thesis]. University of Missouri – Columbia; 2008. Available from: http://hdl.handle.net/10355/5722

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lund

25. Karanauskaite, Jovita. Nature of Large Vesicle Exocytosis in Pancreatic β-cells: Release of ATP and GABA.

Degree: 2008, University of Lund

 Several high resolution electrophysiological techniques such as measurements of cell membrane capacitance and amperometry are used to detect exocytotic events. A novel method was developed… (more)

Subjects/Keywords: Clinical Medicine; ATP; LDCV; Insulin; exocytosis; Compound Exocytosis; Calcium

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APA (6th Edition):

Karanauskaite, J. (2008). Nature of Large Vesicle Exocytosis in Pancreatic β-cells: Release of ATP and GABA. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/1244545 ; https://portal.research.lu.se/ws/files/3217945/1244547.pdf

Chicago Manual of Style (16th Edition):

Karanauskaite, Jovita. “Nature of Large Vesicle Exocytosis in Pancreatic β-cells: Release of ATP and GABA.” 2008. Doctoral Dissertation, University of Lund. Accessed October 30, 2020. https://lup.lub.lu.se/record/1244545 ; https://portal.research.lu.se/ws/files/3217945/1244547.pdf.

MLA Handbook (7th Edition):

Karanauskaite, Jovita. “Nature of Large Vesicle Exocytosis in Pancreatic β-cells: Release of ATP and GABA.” 2008. Web. 30 Oct 2020.

Vancouver:

Karanauskaite J. Nature of Large Vesicle Exocytosis in Pancreatic β-cells: Release of ATP and GABA. [Internet] [Doctoral dissertation]. University of Lund; 2008. [cited 2020 Oct 30]. Available from: https://lup.lub.lu.se/record/1244545 ; https://portal.research.lu.se/ws/files/3217945/1244547.pdf.

Council of Science Editors:

Karanauskaite J. Nature of Large Vesicle Exocytosis in Pancreatic β-cells: Release of ATP and GABA. [Doctoral Dissertation]. University of Lund; 2008. Available from: https://lup.lub.lu.se/record/1244545 ; https://portal.research.lu.se/ws/files/3217945/1244547.pdf


Universiteit Utrecht

26. Zappelli, C. Ins and outs of platelet α-granule proteins.

Degree: 2014, Universiteit Utrecht

 Although platelets are small anucleate cell fragments, they are highly organized cells rich in different types of organelles. Platelets contain three secretory granules, dense granules,… (more)

Subjects/Keywords: platelets; α-granules; massspectrometry; FactorV; Fibrinogen; Integrin; endocytosis; exocytosis; galectin8; STXBP2

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APA (6th Edition):

Zappelli, C. (2014). Ins and outs of platelet α-granule proteins. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/315468

Chicago Manual of Style (16th Edition):

Zappelli, C. “Ins and outs of platelet α-granule proteins.” 2014. Doctoral Dissertation, Universiteit Utrecht. Accessed October 30, 2020. http://dspace.library.uu.nl:8080/handle/1874/315468.

MLA Handbook (7th Edition):

Zappelli, C. “Ins and outs of platelet α-granule proteins.” 2014. Web. 30 Oct 2020.

Vancouver:

Zappelli C. Ins and outs of platelet α-granule proteins. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2014. [cited 2020 Oct 30]. Available from: http://dspace.library.uu.nl:8080/handle/1874/315468.

Council of Science Editors:

Zappelli C. Ins and outs of platelet α-granule proteins. [Doctoral Dissertation]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/315468

27. Stack, Julianne. The Regulation of voltage-dependent calcium currents and their role in modulation of stimulus-secretion coupling in pituitary.

Degree: PhD, 1990, Oregon Health Sciences University

Subjects/Keywords: Calcium Channels; Exocytosis; Electrophysiology; Endorphins

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APA (6th Edition):

Stack, J. (1990). The Regulation of voltage-dependent calcium currents and their role in modulation of stimulus-secretion coupling in pituitary. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4B27SFM ; http://digitalcommons.ohsu.edu/etd/1824

Chicago Manual of Style (16th Edition):

Stack, Julianne. “The Regulation of voltage-dependent calcium currents and their role in modulation of stimulus-secretion coupling in pituitary.” 1990. Doctoral Dissertation, Oregon Health Sciences University. Accessed October 30, 2020. doi:10.6083/M4B27SFM ; http://digitalcommons.ohsu.edu/etd/1824.

MLA Handbook (7th Edition):

Stack, Julianne. “The Regulation of voltage-dependent calcium currents and their role in modulation of stimulus-secretion coupling in pituitary.” 1990. Web. 30 Oct 2020.

Vancouver:

Stack J. The Regulation of voltage-dependent calcium currents and their role in modulation of stimulus-secretion coupling in pituitary. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 1990. [cited 2020 Oct 30]. Available from: doi:10.6083/M4B27SFM ; http://digitalcommons.ohsu.edu/etd/1824.

Council of Science Editors:

Stack J. The Regulation of voltage-dependent calcium currents and their role in modulation of stimulus-secretion coupling in pituitary. [Doctoral Dissertation]. Oregon Health Sciences University; 1990. Available from: doi:10.6083/M4B27SFM ; http://digitalcommons.ohsu.edu/etd/1824


UCLA

28. Yanes, Rolando Eduardo. Study of Mesoporous Silica Nanoparticles' (MSNs) intracellular trafficking and their application as drug delivery vehicles.

Degree: Microbiology, Immunology, & Molecular Genetics, 2013, UCLA

 Mesoporous silica nanoparticles (MSNs) are attractive drug delivery vehicle candidates due to their biocompatibility, stability, high surface area and efficient cellular uptake. In this dissertation,… (more)

Subjects/Keywords: Nanotechnology; Cellular biology; Exocytosis; Mesoporous Silica Nanoparticles; Targeted drug delivery

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APA (6th Edition):

Yanes, R. E. (2013). Study of Mesoporous Silica Nanoparticles' (MSNs) intracellular trafficking and their application as drug delivery vehicles. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/3zv804km

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yanes, Rolando Eduardo. “Study of Mesoporous Silica Nanoparticles' (MSNs) intracellular trafficking and their application as drug delivery vehicles.” 2013. Thesis, UCLA. Accessed October 30, 2020. http://www.escholarship.org/uc/item/3zv804km.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yanes, Rolando Eduardo. “Study of Mesoporous Silica Nanoparticles' (MSNs) intracellular trafficking and their application as drug delivery vehicles.” 2013. Web. 30 Oct 2020.

Vancouver:

Yanes RE. Study of Mesoporous Silica Nanoparticles' (MSNs) intracellular trafficking and their application as drug delivery vehicles. [Internet] [Thesis]. UCLA; 2013. [cited 2020 Oct 30]. Available from: http://www.escholarship.org/uc/item/3zv804km.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yanes RE. Study of Mesoporous Silica Nanoparticles' (MSNs) intracellular trafficking and their application as drug delivery vehicles. [Thesis]. UCLA; 2013. Available from: http://www.escholarship.org/uc/item/3zv804km

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

29. Kolic, Jelena. Distinct Roles of Class 1 PI3K Isoforms in the Regulation of Beta Cell Exocytosis and Insulin Secretion.

Degree: PhD, Department of Pharmacology, 2014, University of Alberta

 Type 2 diabetes (T2D) is characterized by peripheral insulin resistance and an insufficiency of insulin secretion from the pancreatic beta cell. The incidence of T2D… (more)

Subjects/Keywords: PI3K; Beta cell; Insulin secretion; Exocytosis; GIP; GLP-1

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APA (6th Edition):

Kolic, J. (2014). Distinct Roles of Class 1 PI3K Isoforms in the Regulation of Beta Cell Exocytosis and Insulin Secretion. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/cft848q912

Chicago Manual of Style (16th Edition):

Kolic, Jelena. “Distinct Roles of Class 1 PI3K Isoforms in the Regulation of Beta Cell Exocytosis and Insulin Secretion.” 2014. Doctoral Dissertation, University of Alberta. Accessed October 30, 2020. https://era.library.ualberta.ca/files/cft848q912.

MLA Handbook (7th Edition):

Kolic, Jelena. “Distinct Roles of Class 1 PI3K Isoforms in the Regulation of Beta Cell Exocytosis and Insulin Secretion.” 2014. Web. 30 Oct 2020.

Vancouver:

Kolic J. Distinct Roles of Class 1 PI3K Isoforms in the Regulation of Beta Cell Exocytosis and Insulin Secretion. [Internet] [Doctoral dissertation]. University of Alberta; 2014. [cited 2020 Oct 30]. Available from: https://era.library.ualberta.ca/files/cft848q912.

Council of Science Editors:

Kolic J. Distinct Roles of Class 1 PI3K Isoforms in the Regulation of Beta Cell Exocytosis and Insulin Secretion. [Doctoral Dissertation]. University of Alberta; 2014. Available from: https://era.library.ualberta.ca/files/cft848q912

30. Heider, Margaret R. Investigating the Architecture and Vesicle Tethering Function of the Yeast Exocyst Complex: A Dissertation.

Degree: Biochemistry and Molecular Pharmacology, Biochemistry and Molecular Pharmacology, 2016, U of Massachusetts : Med

  The exocyst is an evolutionarily conserved, hetero-octameric protein complex proposed to serve as a multi-subunit tethering complex for exocytosis, although it remains poorly understood… (more)

Subjects/Keywords: Exocytosis; Extracellular Vesicles; Saccharomyces cerevisiae; Cell Biology; Molecular Biology; Structural Biology

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APA (6th Edition):

Heider, M. R. (2016). Investigating the Architecture and Vesicle Tethering Function of the Yeast Exocyst Complex: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/832

Chicago Manual of Style (16th Edition):

Heider, Margaret R. “Investigating the Architecture and Vesicle Tethering Function of the Yeast Exocyst Complex: A Dissertation.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed October 30, 2020. http://escholarship.umassmed.edu/gsbs_diss/832.

MLA Handbook (7th Edition):

Heider, Margaret R. “Investigating the Architecture and Vesicle Tethering Function of the Yeast Exocyst Complex: A Dissertation.” 2016. Web. 30 Oct 2020.

Vancouver:

Heider MR. Investigating the Architecture and Vesicle Tethering Function of the Yeast Exocyst Complex: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2020 Oct 30]. Available from: http://escholarship.umassmed.edu/gsbs_diss/832.

Council of Science Editors:

Heider MR. Investigating the Architecture and Vesicle Tethering Function of the Yeast Exocyst Complex: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/832

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