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You searched for subject:(Estrogen receptor alpha). Showing records 1 – 30 of 85 total matches.

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Rutgers University

1. Yasrebi, Ali, 1987-. ERE-independent ERα signalling in feeding and exploratory behaviors.

Degree: MS, Endocrinology and Animal Biosciences, 2019, Rutgers University

 The reproductive steroid hormone, 17β-estradiol (E2), controls feeding and exploratory behaviors associated with mood disorders. The loss of circulating E2 puts menopausal women at an… (more)

Subjects/Keywords: Estrogen receptor alpha; Estrogen  – Receptors

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APA (6th Edition):

Yasrebi, Ali, 1. (2019). ERE-independent ERα signalling in feeding and exploratory behaviors. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/62067/

Chicago Manual of Style (16th Edition):

Yasrebi, Ali, 1987-. “ERE-independent ERα signalling in feeding and exploratory behaviors.” 2019. Masters Thesis, Rutgers University. Accessed January 17, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/62067/.

MLA Handbook (7th Edition):

Yasrebi, Ali, 1987-. “ERE-independent ERα signalling in feeding and exploratory behaviors.” 2019. Web. 17 Jan 2021.

Vancouver:

Yasrebi, Ali 1. ERE-independent ERα signalling in feeding and exploratory behaviors. [Internet] [Masters thesis]. Rutgers University; 2019. [cited 2021 Jan 17]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/62067/.

Council of Science Editors:

Yasrebi, Ali 1. ERE-independent ERα signalling in feeding and exploratory behaviors. [Masters Thesis]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/62067/


Cornell University

2. Melville, Katherine. Estrogen Receptor Alpha In Osteoblasts Mediates Bone Mass And Bone’S Response To In Vivo Loading.

Degree: PhD, Biomedical Engineering, 2014, Cornell University

 Decreased bioavailable estrogen levels are a major cause of bone loss in postmenopausal women, but sex hormones are important regulators of bone mass in both… (more)

Subjects/Keywords: estrogen receptor alpha; bone; loading

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APA (6th Edition):

Melville, K. (2014). Estrogen Receptor Alpha In Osteoblasts Mediates Bone Mass And Bone’S Response To In Vivo Loading. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/38857

Chicago Manual of Style (16th Edition):

Melville, Katherine. “Estrogen Receptor Alpha In Osteoblasts Mediates Bone Mass And Bone’S Response To In Vivo Loading.” 2014. Doctoral Dissertation, Cornell University. Accessed January 17, 2021. http://hdl.handle.net/1813/38857.

MLA Handbook (7th Edition):

Melville, Katherine. “Estrogen Receptor Alpha In Osteoblasts Mediates Bone Mass And Bone’S Response To In Vivo Loading.” 2014. Web. 17 Jan 2021.

Vancouver:

Melville K. Estrogen Receptor Alpha In Osteoblasts Mediates Bone Mass And Bone’S Response To In Vivo Loading. [Internet] [Doctoral dissertation]. Cornell University; 2014. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/1813/38857.

Council of Science Editors:

Melville K. Estrogen Receptor Alpha In Osteoblasts Mediates Bone Mass And Bone’S Response To In Vivo Loading. [Doctoral Dissertation]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/38857


University of Houston

3. Son, Jieun 1985-. Mechanism of Estrogen Receptor Alpha in Cervical Carcinogenesis.

Degree: PhD, Cell and Molecular Biology, 2017, University of Houston

 Most cervical cancers are associated with high-risk human papillomaviruses (HPVs). HPVs display tumorigenic functions in host cells mainly through viral oncogenes E6 and E7 that… (more)

Subjects/Keywords: Estrogen receptor alpha; Cervical cancer

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APA (6th Edition):

Son, J. 1. (2017). Mechanism of Estrogen Receptor Alpha in Cervical Carcinogenesis. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/4602

Chicago Manual of Style (16th Edition):

Son, Jieun 1985-. “Mechanism of Estrogen Receptor Alpha in Cervical Carcinogenesis.” 2017. Doctoral Dissertation, University of Houston. Accessed January 17, 2021. http://hdl.handle.net/10657/4602.

MLA Handbook (7th Edition):

Son, Jieun 1985-. “Mechanism of Estrogen Receptor Alpha in Cervical Carcinogenesis.” 2017. Web. 17 Jan 2021.

Vancouver:

Son J1. Mechanism of Estrogen Receptor Alpha in Cervical Carcinogenesis. [Internet] [Doctoral dissertation]. University of Houston; 2017. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/10657/4602.

Council of Science Editors:

Son J1. Mechanism of Estrogen Receptor Alpha in Cervical Carcinogenesis. [Doctoral Dissertation]. University of Houston; 2017. Available from: http://hdl.handle.net/10657/4602


Boston University

4. Koomson, Jacqueline Nyarkoa. The role of estrogen receptors alpha and beta in the development of uterine leiomyomas.

Degree: MS, Medical Sciences, 2017, Boston University

 Uterine leiomyomas are benign tumors within the uterus, where patients present with symptoms such as abnormal bleeding, urinary retention, and pelvic pressure. The exact etiology… (more)

Subjects/Keywords: Medicine; Estrogen; Estrogen receptor alpha; Estrogen receptor beta; Receptor-mediated therapies; Uterine fibroids; Uterine leiomyoma

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APA (6th Edition):

Koomson, J. N. (2017). The role of estrogen receptors alpha and beta in the development of uterine leiomyomas. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/23835

Chicago Manual of Style (16th Edition):

Koomson, Jacqueline Nyarkoa. “The role of estrogen receptors alpha and beta in the development of uterine leiomyomas.” 2017. Masters Thesis, Boston University. Accessed January 17, 2021. http://hdl.handle.net/2144/23835.

MLA Handbook (7th Edition):

Koomson, Jacqueline Nyarkoa. “The role of estrogen receptors alpha and beta in the development of uterine leiomyomas.” 2017. Web. 17 Jan 2021.

Vancouver:

Koomson JN. The role of estrogen receptors alpha and beta in the development of uterine leiomyomas. [Internet] [Masters thesis]. Boston University; 2017. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/2144/23835.

Council of Science Editors:

Koomson JN. The role of estrogen receptors alpha and beta in the development of uterine leiomyomas. [Masters Thesis]. Boston University; 2017. Available from: http://hdl.handle.net/2144/23835


University of Rochester

5. Hsu, Iawen. The Roles of Estrogen Receptors in the Bladder Cancer Development.

Degree: PhD, 2013, University of Rochester

 Early studies documented the existence of sexual dimorphism in bladder cancer occurrence and progression with higher bladder cancer incidence in males than females. However, the… (more)

Subjects/Keywords: Estrogen Receptor Alpha; Estrogen Receptor Beta; MCM5; Bladder Cancer BBN; Inpp4β

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APA (6th Edition):

Hsu, I. (2013). The Roles of Estrogen Receptors in the Bladder Cancer Development. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/27302

Chicago Manual of Style (16th Edition):

Hsu, Iawen. “The Roles of Estrogen Receptors in the Bladder Cancer Development.” 2013. Doctoral Dissertation, University of Rochester. Accessed January 17, 2021. http://hdl.handle.net/1802/27302.

MLA Handbook (7th Edition):

Hsu, Iawen. “The Roles of Estrogen Receptors in the Bladder Cancer Development.” 2013. Web. 17 Jan 2021.

Vancouver:

Hsu I. The Roles of Estrogen Receptors in the Bladder Cancer Development. [Internet] [Doctoral dissertation]. University of Rochester; 2013. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/1802/27302.

Council of Science Editors:

Hsu I. The Roles of Estrogen Receptors in the Bladder Cancer Development. [Doctoral Dissertation]. University of Rochester; 2013. Available from: http://hdl.handle.net/1802/27302

6. Baatarsuren, Batmunkh; Narantsog, Choijookhuu; Naparee, Srisowanna; Uugantsetseg, Byambatsogt; Phyu Synn, Oo; Mohmand Noor, Ali; Yamaguchi, Yuya. Estrogen Accelerates Cell Proliferation through Estrogen Receptor α during Rat Liver Regeneration after Partial Hepatectomy.

Degree: 博士(医学), 2017, University of Miyazaki / 宮崎大学

学位論文の一部を構成しているため、http://hdl.handle.net/10458/5984 に本文を掲載。

Although estrogen is implicated in the regulation of cell growth and differentiation in many organs, the exact mechanism for liver regeneration is not… (more)

Subjects/Keywords: estrogen; estrogen receptor α; liver regeneration; partial hepatectomy; cell proliferation

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APA (6th Edition):

Baatarsuren, Batmunkh; Narantsog, Choijookhuu; Naparee, Srisowanna; Uugantsetseg, Byambatsogt; Phyu Synn, Oo; Mohmand Noor, Ali; Yamaguchi, Y. (2017). Estrogen Accelerates Cell Proliferation through Estrogen Receptor α during Rat Liver Regeneration after Partial Hepatectomy. (Thesis). University of Miyazaki / 宮崎大学. Retrieved from http://hdl.handle.net/10458/5983

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baatarsuren, Batmunkh; Narantsog, Choijookhuu; Naparee, Srisowanna; Uugantsetseg, Byambatsogt; Phyu Synn, Oo; Mohmand Noor, Ali; Yamaguchi, Yuya. “Estrogen Accelerates Cell Proliferation through Estrogen Receptor α during Rat Liver Regeneration after Partial Hepatectomy.” 2017. Thesis, University of Miyazaki / 宮崎大学. Accessed January 17, 2021. http://hdl.handle.net/10458/5983.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baatarsuren, Batmunkh; Narantsog, Choijookhuu; Naparee, Srisowanna; Uugantsetseg, Byambatsogt; Phyu Synn, Oo; Mohmand Noor, Ali; Yamaguchi, Yuya. “Estrogen Accelerates Cell Proliferation through Estrogen Receptor α during Rat Liver Regeneration after Partial Hepatectomy.” 2017. Web. 17 Jan 2021.

Vancouver:

Baatarsuren, Batmunkh; Narantsog, Choijookhuu; Naparee, Srisowanna; Uugantsetseg, Byambatsogt; Phyu Synn, Oo; Mohmand Noor, Ali; Yamaguchi Y. Estrogen Accelerates Cell Proliferation through Estrogen Receptor α during Rat Liver Regeneration after Partial Hepatectomy. [Internet] [Thesis]. University of Miyazaki / 宮崎大学; 2017. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/10458/5983.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baatarsuren, Batmunkh; Narantsog, Choijookhuu; Naparee, Srisowanna; Uugantsetseg, Byambatsogt; Phyu Synn, Oo; Mohmand Noor, Ali; Yamaguchi Y. Estrogen Accelerates Cell Proliferation through Estrogen Receptor α during Rat Liver Regeneration after Partial Hepatectomy. [Thesis]. University of Miyazaki / 宮崎大学; 2017. Available from: http://hdl.handle.net/10458/5983

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Gustafsson, Karin. Estrogen receptor α and Bone – Posttranslational modifications and cell-specific deletion.

Degree: 2019, University of Gothenburg / Göteborgs Universitet

Estrogen is involved in the regulation and development of reproductive organs. In addition, estrogen regulates several other organs including the skeleton, immune system, and adipose… (more)

Subjects/Keywords: estrogen receptor α; bone; osteoporosis; adipose tissue; estrogen; posttranslational modifications

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APA (6th Edition):

Gustafsson, K. (2019). Estrogen receptor α and Bone – Posttranslational modifications and cell-specific deletion. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/60795

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gustafsson, Karin. “Estrogen receptor α and Bone – Posttranslational modifications and cell-specific deletion.” 2019. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed January 17, 2021. http://hdl.handle.net/2077/60795.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gustafsson, Karin. “Estrogen receptor α and Bone – Posttranslational modifications and cell-specific deletion.” 2019. Web. 17 Jan 2021.

Vancouver:

Gustafsson K. Estrogen receptor α and Bone – Posttranslational modifications and cell-specific deletion. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2019. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/2077/60795.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gustafsson K. Estrogen receptor α and Bone – Posttranslational modifications and cell-specific deletion. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2019. Available from: http://hdl.handle.net/2077/60795

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

8. Božović, Ana M., 1977-. Metilovanje promotora i ekspresija estrogenskog receptora beta (ERß) u invazivnim karcinomima dojke.

Degree: Biološki fakultet, 2014, Univerzitet u Beogradu

Biologija - Molekularna genetika kancera / Biology - Molecular genetics of cancer

Invazivni karcinom dojke je najčešći kancer kod žena. Pored genetičkih i epigenetički faktori… (more)

Subjects/Keywords: Breast cancer; estrogen receptor alpha; estrogen receptor beta; progesterone receptor; methylation; quantitative PCR

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APA (6th Edition):

Božović, Ana M., 1. (2014). Metilovanje promotora i ekspresija estrogenskog receptora beta (ERß) u invazivnim karcinomima dojke. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:7126/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Božović, Ana M., 1977-. “Metilovanje promotora i ekspresija estrogenskog receptora beta (ERß) u invazivnim karcinomima dojke.” 2014. Thesis, Univerzitet u Beogradu. Accessed January 17, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:7126/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Božović, Ana M., 1977-. “Metilovanje promotora i ekspresija estrogenskog receptora beta (ERß) u invazivnim karcinomima dojke.” 2014. Web. 17 Jan 2021.

Vancouver:

Božović, Ana M. 1. Metilovanje promotora i ekspresija estrogenskog receptora beta (ERß) u invazivnim karcinomima dojke. [Internet] [Thesis]. Univerzitet u Beogradu; 2014. [cited 2021 Jan 17]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:7126/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Božović, Ana M. 1. Metilovanje promotora i ekspresija estrogenskog receptora beta (ERß) u invazivnim karcinomima dojke. [Thesis]. Univerzitet u Beogradu; 2014. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:7126/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Gagniac, Laurine. Physiologie et physiopathologie des effets membranaires du récepteur des œstrogènes alpha (ERα) dans la glande mammaire : Physiology et physiopathology of membrane estrogen receptor alpha (ERα) in mammary gland.

Degree: Docteur es, Physiopathologie, 2018, Université Toulouse III – Paul Sabatier

It is well established that the 17-estradiol is involved in the development and homeostasis of reproductive and extra-reproductive tissues, particularly the mammary gland. Estradiol classically… (more)

Subjects/Keywords: Glande mammaire; Œstrogènes; Récepteur des œstrogènes alpha; Cellules souches; Cancer du sein; Mammary gland; Estrogens; Estrogen receptor alpha; Estrogen receptor alpha

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APA (6th Edition):

Gagniac, L. (2018). Physiologie et physiopathologie des effets membranaires du récepteur des œstrogènes alpha (ERα) dans la glande mammaire : Physiology et physiopathology of membrane estrogen receptor alpha (ERα) in mammary gland. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2018TOU30101

Chicago Manual of Style (16th Edition):

Gagniac, Laurine. “Physiologie et physiopathologie des effets membranaires du récepteur des œstrogènes alpha (ERα) dans la glande mammaire : Physiology et physiopathology of membrane estrogen receptor alpha (ERα) in mammary gland.” 2018. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed January 17, 2021. http://www.theses.fr/2018TOU30101.

MLA Handbook (7th Edition):

Gagniac, Laurine. “Physiologie et physiopathologie des effets membranaires du récepteur des œstrogènes alpha (ERα) dans la glande mammaire : Physiology et physiopathology of membrane estrogen receptor alpha (ERα) in mammary gland.” 2018. Web. 17 Jan 2021.

Vancouver:

Gagniac L. Physiologie et physiopathologie des effets membranaires du récepteur des œstrogènes alpha (ERα) dans la glande mammaire : Physiology et physiopathology of membrane estrogen receptor alpha (ERα) in mammary gland. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2018. [cited 2021 Jan 17]. Available from: http://www.theses.fr/2018TOU30101.

Council of Science Editors:

Gagniac L. Physiologie et physiopathologie des effets membranaires du récepteur des œstrogènes alpha (ERα) dans la glande mammaire : Physiology et physiopathology of membrane estrogen receptor alpha (ERα) in mammary gland. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2018. Available from: http://www.theses.fr/2018TOU30101


University of Southern California

10. Cho, Tae Hoon. The role of estrogen receptors and nociceptive signaling pathway of primary sensory neurons.

Degree: PhD, Chemical Engineering, 2012, University of Southern California

 Clinical studies suggest the comorbidity of functional pain syndromes such as irritable bowel syndrome (IBS), chronic pelvic pain (CPP), fibromyalgia, and somatoform disorders approaches 40%… (more)

Subjects/Keywords: 17beta-estradiol; DRG; ATP; Capsaicin; calcium; estrogen receptor alpha; estrogen receptor beta

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APA (6th Edition):

Cho, T. H. (2012). The role of estrogen receptors and nociceptive signaling pathway of primary sensory neurons. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/42498/rec/7220

Chicago Manual of Style (16th Edition):

Cho, Tae Hoon. “The role of estrogen receptors and nociceptive signaling pathway of primary sensory neurons.” 2012. Doctoral Dissertation, University of Southern California. Accessed January 17, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/42498/rec/7220.

MLA Handbook (7th Edition):

Cho, Tae Hoon. “The role of estrogen receptors and nociceptive signaling pathway of primary sensory neurons.” 2012. Web. 17 Jan 2021.

Vancouver:

Cho TH. The role of estrogen receptors and nociceptive signaling pathway of primary sensory neurons. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2021 Jan 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/42498/rec/7220.

Council of Science Editors:

Cho TH. The role of estrogen receptors and nociceptive signaling pathway of primary sensory neurons. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/42498/rec/7220


University of Guelph

11. Ervin, Kelsy. The role of specific estrogen receptors in mediating rapid estrogenic improvements of social learning in female mice.

Degree: MS, Department of Psychology, 2014, University of Guelph

 Social learning is a process by which an animal gains information from another; however much of the research on estrogens effects on learning focuses on… (more)

Subjects/Keywords: estradiol; social transmission of food preference; estrogen receptor alpha; estrogen receptor beta; G protein-coupled estrogen receptor; GPER; GPR30

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APA (6th Edition):

Ervin, K. (2014). The role of specific estrogen receptors in mediating rapid estrogenic improvements of social learning in female mice. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7818

Chicago Manual of Style (16th Edition):

Ervin, Kelsy. “The role of specific estrogen receptors in mediating rapid estrogenic improvements of social learning in female mice.” 2014. Masters Thesis, University of Guelph. Accessed January 17, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7818.

MLA Handbook (7th Edition):

Ervin, Kelsy. “The role of specific estrogen receptors in mediating rapid estrogenic improvements of social learning in female mice.” 2014. Web. 17 Jan 2021.

Vancouver:

Ervin K. The role of specific estrogen receptors in mediating rapid estrogenic improvements of social learning in female mice. [Internet] [Masters thesis]. University of Guelph; 2014. [cited 2021 Jan 17]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7818.

Council of Science Editors:

Ervin K. The role of specific estrogen receptors in mediating rapid estrogenic improvements of social learning in female mice. [Masters Thesis]. University of Guelph; 2014. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7818


University of Toronto

12. Rajendra, Sharanya. The Regulation of TiPARP by the Aryl Hydrocarbon Receptor, the Platelet-derived Growth Factor Receptor, and the Estrogen Receptor Alpha.

Degree: 2013, University of Toronto

TiPARP is a PARP-like mART that is induced by and negatively regulates AHR transactivation. Despite these insights, not much is known about TiPARP. This study… (more)

Subjects/Keywords: Aryl Hydrocarbon Receptor; Estrogen Receptor Alpha; TCDD; TiPARP; Gene Regulation; 0419

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APA (6th Edition):

Rajendra, S. (2013). The Regulation of TiPARP by the Aryl Hydrocarbon Receptor, the Platelet-derived Growth Factor Receptor, and the Estrogen Receptor Alpha. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/43310

Chicago Manual of Style (16th Edition):

Rajendra, Sharanya. “The Regulation of TiPARP by the Aryl Hydrocarbon Receptor, the Platelet-derived Growth Factor Receptor, and the Estrogen Receptor Alpha.” 2013. Masters Thesis, University of Toronto. Accessed January 17, 2021. http://hdl.handle.net/1807/43310.

MLA Handbook (7th Edition):

Rajendra, Sharanya. “The Regulation of TiPARP by the Aryl Hydrocarbon Receptor, the Platelet-derived Growth Factor Receptor, and the Estrogen Receptor Alpha.” 2013. Web. 17 Jan 2021.

Vancouver:

Rajendra S. The Regulation of TiPARP by the Aryl Hydrocarbon Receptor, the Platelet-derived Growth Factor Receptor, and the Estrogen Receptor Alpha. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/1807/43310.

Council of Science Editors:

Rajendra S. The Regulation of TiPARP by the Aryl Hydrocarbon Receptor, the Platelet-derived Growth Factor Receptor, and the Estrogen Receptor Alpha. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/43310


University of Illinois – Urbana-Champaign

13. Kaya, Hatice. Identification of molecular signaling pathways underlying human endometrial stromal cell differentiation.

Degree: PhD, 0325, 2014, University of Illinois – Urbana-Champaign

 Decidualization, the differentiation of endometrial stromal cells, is essential for a successful pregnancy. Although the steroid hormones estrogen (E) and progesterone (P) are known to… (more)

Subjects/Keywords: Human endometrial stromal cell differentiation; estrogen receptor alpha; progesterone receptor isoforms

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APA (6th Edition):

Kaya, H. (2014). Identification of molecular signaling pathways underlying human endometrial stromal cell differentiation. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/46949

Chicago Manual of Style (16th Edition):

Kaya, Hatice. “Identification of molecular signaling pathways underlying human endometrial stromal cell differentiation.” 2014. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed January 17, 2021. http://hdl.handle.net/2142/46949.

MLA Handbook (7th Edition):

Kaya, Hatice. “Identification of molecular signaling pathways underlying human endometrial stromal cell differentiation.” 2014. Web. 17 Jan 2021.

Vancouver:

Kaya H. Identification of molecular signaling pathways underlying human endometrial stromal cell differentiation. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2014. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/2142/46949.

Council of Science Editors:

Kaya H. Identification of molecular signaling pathways underlying human endometrial stromal cell differentiation. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2014. Available from: http://hdl.handle.net/2142/46949

14. Farman, Helen. The Role of Estrogen Receptor α Signaling in the Regulation of Bone Mass.

Degree: 2018, University of Gothenburg / Göteborgs Universitet

 Estrogens are major regulators of skeletal growth and maintenance in both females and males. Estrogen receptor α (ERα) is the main mediator of estrogenic effects… (more)

Subjects/Keywords: estrogen receptor α; bone; estrogen

…Lagerquist MK, and Ohlsson C. Female mice lacking estrogen receptor-α in hypothalamic… …Tuukkanen J, Levin ER, Ohlsson C, and Lagerquist MK. Membrane estrogen receptor-α is essential for… …Estrogen dendrimer conjugate ERα Estrogen receptor α ERβ Estrogen receptor β ERs Estrogen… …males (3). Estrogen receptor α (ERα) mediates estrogen effects in bone and… …of the bone tissue and is predominantly found 1 The role of estrogen receptor α in the… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Farman, H. (2018). The Role of Estrogen Receptor α Signaling in the Regulation of Bone Mass. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/57672

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Farman, Helen. “The Role of Estrogen Receptor α Signaling in the Regulation of Bone Mass.” 2018. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed January 17, 2021. http://hdl.handle.net/2077/57672.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Farman, Helen. “The Role of Estrogen Receptor α Signaling in the Regulation of Bone Mass.” 2018. Web. 17 Jan 2021.

Vancouver:

Farman H. The Role of Estrogen Receptor α Signaling in the Regulation of Bone Mass. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2018. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/2077/57672.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Farman H. The Role of Estrogen Receptor α Signaling in the Regulation of Bone Mass. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2018. Available from: http://hdl.handle.net/2077/57672

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

15. Schwanstecher, Annekatrin. Regulation of Adipose Tiglycerid Lipase (ATGL) expression by nuclear estrogen receptors.

Degree: 2015, Freie Universität Berlin

Estrogen Receptors (ERs) are known to play an important role in the metabolic functions of adipose tissue (AT). Gender specific differences concerning lipolysis have been… (more)

Subjects/Keywords: estrogen; estrogen receptor alpha; estrogen receptor beta, ATGL; Adipocyte Triglyceride Lipase; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Schwanstecher, A. (2015). Regulation of Adipose Tiglycerid Lipase (ATGL) expression by nuclear estrogen receptors. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-9781

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schwanstecher, Annekatrin. “Regulation of Adipose Tiglycerid Lipase (ATGL) expression by nuclear estrogen receptors.” 2015. Thesis, Freie Universität Berlin. Accessed January 17, 2021. http://dx.doi.org/10.17169/refubium-9781.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schwanstecher, Annekatrin. “Regulation of Adipose Tiglycerid Lipase (ATGL) expression by nuclear estrogen receptors.” 2015. Web. 17 Jan 2021.

Vancouver:

Schwanstecher A. Regulation of Adipose Tiglycerid Lipase (ATGL) expression by nuclear estrogen receptors. [Internet] [Thesis]. Freie Universität Berlin; 2015. [cited 2021 Jan 17]. Available from: http://dx.doi.org/10.17169/refubium-9781.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schwanstecher A. Regulation of Adipose Tiglycerid Lipase (ATGL) expression by nuclear estrogen receptors. [Thesis]. Freie Universität Berlin; 2015. Available from: http://dx.doi.org/10.17169/refubium-9781

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

16. Nicholson, Tristan Marriner. Estrogen Receptor-alpha is a Key Mediator and Therapeutic Target in Benign Prostatic Hyperplasia.

Degree: PhD, 2015, University of Rochester

 Benign prostatic hyperplasia (BPH) and bladder outlet obstruction (BOO) are common in older men and contribute to lower urinary tract symptoms (LUTS) that significantly impact… (more)

Subjects/Keywords: Estrogen; Testosterone; Benign Prostatic Hyperplasia; Lower Urinary Tract Symptoms; Estrogen Receptor-alpha

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APA (6th Edition):

Nicholson, T. M. (2015). Estrogen Receptor-alpha is a Key Mediator and Therapeutic Target in Benign Prostatic Hyperplasia. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/29781

Chicago Manual of Style (16th Edition):

Nicholson, Tristan Marriner. “Estrogen Receptor-alpha is a Key Mediator and Therapeutic Target in Benign Prostatic Hyperplasia.” 2015. Doctoral Dissertation, University of Rochester. Accessed January 17, 2021. http://hdl.handle.net/1802/29781.

MLA Handbook (7th Edition):

Nicholson, Tristan Marriner. “Estrogen Receptor-alpha is a Key Mediator and Therapeutic Target in Benign Prostatic Hyperplasia.” 2015. Web. 17 Jan 2021.

Vancouver:

Nicholson TM. Estrogen Receptor-alpha is a Key Mediator and Therapeutic Target in Benign Prostatic Hyperplasia. [Internet] [Doctoral dissertation]. University of Rochester; 2015. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/1802/29781.

Council of Science Editors:

Nicholson TM. Estrogen Receptor-alpha is a Key Mediator and Therapeutic Target in Benign Prostatic Hyperplasia. [Doctoral Dissertation]. University of Rochester; 2015. Available from: http://hdl.handle.net/1802/29781


University of the Western Cape

17. Jijana, Abongile Nwabisa. Quantum dot sensitized estrogen receptor alpha-recombinant protein electrochemical biosensor for 17-beta estradiol .

Degree: 2016, University of the Western Cape

 Estrogens play an extraordinary role in the endocrine system regulation through the stimulation and regulation of endocrine pathways. 17β-estradiol is one of the final metabolites… (more)

Subjects/Keywords: Amino acids; Proteins; Fluorescence spectroscopy; Estrogen – Receptors; Estrogen receptor-alpha (ER-𝜶�)

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APA (6th Edition):

Jijana, A. N. (2016). Quantum dot sensitized estrogen receptor alpha-recombinant protein electrochemical biosensor for 17-beta estradiol . (Thesis). University of the Western Cape. Retrieved from http://hdl.handle.net/11394/5330

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jijana, Abongile Nwabisa. “Quantum dot sensitized estrogen receptor alpha-recombinant protein electrochemical biosensor for 17-beta estradiol .” 2016. Thesis, University of the Western Cape. Accessed January 17, 2021. http://hdl.handle.net/11394/5330.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jijana, Abongile Nwabisa. “Quantum dot sensitized estrogen receptor alpha-recombinant protein electrochemical biosensor for 17-beta estradiol .” 2016. Web. 17 Jan 2021.

Vancouver:

Jijana AN. Quantum dot sensitized estrogen receptor alpha-recombinant protein electrochemical biosensor for 17-beta estradiol . [Internet] [Thesis]. University of the Western Cape; 2016. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/11394/5330.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jijana AN. Quantum dot sensitized estrogen receptor alpha-recombinant protein electrochemical biosensor for 17-beta estradiol . [Thesis]. University of the Western Cape; 2016. Available from: http://hdl.handle.net/11394/5330

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Brigham Young University

18. Watson, Kristin Dawn. The Molecular Mechanism of Migraine.

Degree: MS, 2011, Brigham Young University

  Migraine is a common, episodic neurological disorder that includes headache, nausea and hypersensitivity to sensory stimuli. During the headache phase of migraine, migraine patients… (more)

Subjects/Keywords: migraine; casein kinase 1 alpha; estrogen; estrogen receptor β; pain; sex difference; Biochemistry; Chemistry

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APA (6th Edition):

Watson, K. D. (2011). The Molecular Mechanism of Migraine. (Masters Thesis). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4047&context=etd

Chicago Manual of Style (16th Edition):

Watson, Kristin Dawn. “The Molecular Mechanism of Migraine.” 2011. Masters Thesis, Brigham Young University. Accessed January 17, 2021. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4047&context=etd.

MLA Handbook (7th Edition):

Watson, Kristin Dawn. “The Molecular Mechanism of Migraine.” 2011. Web. 17 Jan 2021.

Vancouver:

Watson KD. The Molecular Mechanism of Migraine. [Internet] [Masters thesis]. Brigham Young University; 2011. [cited 2021 Jan 17]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4047&context=etd.

Council of Science Editors:

Watson KD. The Molecular Mechanism of Migraine. [Masters Thesis]. Brigham Young University; 2011. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4047&context=etd

19. Megas, Georgios. Η μελέτη της έκφρασης των οιστρογονικών υποδοχέων ER(α) και ER(β) σε συνάρτηση με τους ανδρογονικούς υποδοχείς στον τοπικά προχωρημένο καρκίνο του προστάτη ως προγνωστικό παράγοντα στην βιοχημική υποτροπή και την πρόοδο της νόσου.

Degree: 2014, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

The objective of this study was to evaluate the expression of estrogen receptors (ER(α) and ER(β)) and androgen receptors (ARs) as prognostic factors for biochemical… (more)

Subjects/Keywords: Ανδρογονικός υποδοχέας; Οιστρογονικός υποδοχέας (α); Οιστρογονικός υποδοχέας (β); Τοπικά προχωρημένος καρκίνος του προστάτη; Ριζική προστατεκτομή; Androgen receptor; Estrogen receptor (α); Estrogen receptor (β); Locally advanced prostate cancer; Radical prostatectomy

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APA (6th Edition):

Megas, G. (2014). Η μελέτη της έκφρασης των οιστρογονικών υποδοχέων ER(α) και ER(β) σε συνάρτηση με τους ανδρογονικούς υποδοχείς στον τοπικά προχωρημένο καρκίνο του προστάτη ως προγνωστικό παράγοντα στην βιοχημική υποτροπή και την πρόοδο της νόσου. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/41641

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Megas, Georgios. “Η μελέτη της έκφρασης των οιστρογονικών υποδοχέων ER(α) και ER(β) σε συνάρτηση με τους ανδρογονικούς υποδοχείς στον τοπικά προχωρημένο καρκίνο του προστάτη ως προγνωστικό παράγοντα στην βιοχημική υποτροπή και την πρόοδο της νόσου.” 2014. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed January 17, 2021. http://hdl.handle.net/10442/hedi/41641.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Megas, Georgios. “Η μελέτη της έκφρασης των οιστρογονικών υποδοχέων ER(α) και ER(β) σε συνάρτηση με τους ανδρογονικούς υποδοχείς στον τοπικά προχωρημένο καρκίνο του προστάτη ως προγνωστικό παράγοντα στην βιοχημική υποτροπή και την πρόοδο της νόσου.” 2014. Web. 17 Jan 2021.

Vancouver:

Megas G. Η μελέτη της έκφρασης των οιστρογονικών υποδοχέων ER(α) και ER(β) σε συνάρτηση με τους ανδρογονικούς υποδοχείς στον τοπικά προχωρημένο καρκίνο του προστάτη ως προγνωστικό παράγοντα στην βιοχημική υποτροπή και την πρόοδο της νόσου. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2014. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/10442/hedi/41641.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Megas G. Η μελέτη της έκφρασης των οιστρογονικών υποδοχέων ER(α) και ER(β) σε συνάρτηση με τους ανδρογονικούς υποδοχείς στον τοπικά προχωρημένο καρκίνο του προστάτη ως προγνωστικό παράγοντα στην βιοχημική υποτροπή και την πρόοδο της νόσου. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2014. Available from: http://hdl.handle.net/10442/hedi/41641

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

20. JavanMoghaddam, Sonia. Cell Cycle Regulatory Roles of Estrogen Receptor alpha (ERα) in Breast Cancer Cells.

Degree: PhD, 2011, Texas Medical Center

  Previous studies have shown that Estrogen Receptor alpha (ERα) is an important indicator for diagnosis, prognosis and treatment of breast cancers. However, the question… (more)

Subjects/Keywords: Estrogen Receptor alpha; Cell cycle; Proliferation; Breast Cancer; Cancer Biology

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APA (6th Edition):

JavanMoghaddam, S. (2011). Cell Cycle Regulatory Roles of Estrogen Receptor alpha (ERα) in Breast Cancer Cells. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/121

Chicago Manual of Style (16th Edition):

JavanMoghaddam, Sonia. “Cell Cycle Regulatory Roles of Estrogen Receptor alpha (ERα) in Breast Cancer Cells.” 2011. Doctoral Dissertation, Texas Medical Center. Accessed January 17, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/121.

MLA Handbook (7th Edition):

JavanMoghaddam, Sonia. “Cell Cycle Regulatory Roles of Estrogen Receptor alpha (ERα) in Breast Cancer Cells.” 2011. Web. 17 Jan 2021.

Vancouver:

JavanMoghaddam S. Cell Cycle Regulatory Roles of Estrogen Receptor alpha (ERα) in Breast Cancer Cells. [Internet] [Doctoral dissertation]. Texas Medical Center; 2011. [cited 2021 Jan 17]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/121.

Council of Science Editors:

JavanMoghaddam S. Cell Cycle Regulatory Roles of Estrogen Receptor alpha (ERα) in Breast Cancer Cells. [Doctoral Dissertation]. Texas Medical Center; 2011. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/121


Georgia State University

21. Bruggeman, Emily C. The Role of Neuronal DNA Methyltransferase 1 in Energy Homeostasis and Obesity.

Degree: PhD, Neuroscience Institute, 2016, Georgia State University

  Obesity is a grave disease that is increasing in global prevalence. Aberrant neuronal DNA methylation patterns have been implicated in the promotion of obesity… (more)

Subjects/Keywords: Epigenetics; Estrogen receptor-alpha; Hypothalamus; Brown adipose tissue

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APA (6th Edition):

Bruggeman, E. C. (2016). The Role of Neuronal DNA Methyltransferase 1 in Energy Homeostasis and Obesity. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/neurosci_diss/24

Chicago Manual of Style (16th Edition):

Bruggeman, Emily C. “The Role of Neuronal DNA Methyltransferase 1 in Energy Homeostasis and Obesity.” 2016. Doctoral Dissertation, Georgia State University. Accessed January 17, 2021. https://scholarworks.gsu.edu/neurosci_diss/24.

MLA Handbook (7th Edition):

Bruggeman, Emily C. “The Role of Neuronal DNA Methyltransferase 1 in Energy Homeostasis and Obesity.” 2016. Web. 17 Jan 2021.

Vancouver:

Bruggeman EC. The Role of Neuronal DNA Methyltransferase 1 in Energy Homeostasis and Obesity. [Internet] [Doctoral dissertation]. Georgia State University; 2016. [cited 2021 Jan 17]. Available from: https://scholarworks.gsu.edu/neurosci_diss/24.

Council of Science Editors:

Bruggeman EC. The Role of Neuronal DNA Methyltransferase 1 in Energy Homeostasis and Obesity. [Doctoral Dissertation]. Georgia State University; 2016. Available from: https://scholarworks.gsu.edu/neurosci_diss/24


Vanderbilt University

22. Fuseini, Hubaida. The Role of Ovarian Hormones and Testosterone on Type 2 and IL-17A-Mediated Airway inflammation.

Degree: PhD, Microbiology and Immunology, 2019, Vanderbilt University

 Severe asthma is a significant health care concern, with patients having poorer asthma control, poorer lung function, and increased health care costs compared to milder… (more)

Subjects/Keywords: type 2; testosterone; ovarian hormones; estrogen receptor alpha; asthma; androgen receptor; IL-17A

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APA (6th Edition):

Fuseini, H. (2019). The Role of Ovarian Hormones and Testosterone on Type 2 and IL-17A-Mediated Airway inflammation. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11949

Chicago Manual of Style (16th Edition):

Fuseini, Hubaida. “The Role of Ovarian Hormones and Testosterone on Type 2 and IL-17A-Mediated Airway inflammation.” 2019. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2021. http://hdl.handle.net/1803/11949.

MLA Handbook (7th Edition):

Fuseini, Hubaida. “The Role of Ovarian Hormones and Testosterone on Type 2 and IL-17A-Mediated Airway inflammation.” 2019. Web. 17 Jan 2021.

Vancouver:

Fuseini H. The Role of Ovarian Hormones and Testosterone on Type 2 and IL-17A-Mediated Airway inflammation. [Internet] [Doctoral dissertation]. Vanderbilt University; 2019. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/1803/11949.

Council of Science Editors:

Fuseini H. The Role of Ovarian Hormones and Testosterone on Type 2 and IL-17A-Mediated Airway inflammation. [Doctoral Dissertation]. Vanderbilt University; 2019. Available from: http://hdl.handle.net/1803/11949

23. Božović Ana. Estrogen receptor beta (ERß) promoter methylation and expression in invasive breast cancers.

Degree: PhD, Biology, 2013, University of Belgrade

Breast cancer is the most common invasive cancer in women. Beside genetic, epigenetic factors influence its initiation and progression. The purpose of this study was… (more)

Subjects/Keywords: Breast cancer; estrogen receptor alpha; estrogen receptor beta; progesterone receptor; methylation; quantitative PCR; Kancer dojke; estrogenski receptor alfa; estrogenski receptor beta; progesteronski receptor; metilacija; kvantitativni PCR

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APA (6th Edition):

Ana, B. (2013). Estrogen receptor beta (ERß) promoter methylation and expression in invasive breast cancers. (Doctoral Dissertation). University of Belgrade. Retrieved from http://dx.doi.org/10.2298/BG20130703BOZOVIC ; http://eteze.bg.ac.rs/application/showtheses?thesesId=799 ; https://fedorabg.bg.ac.rs/fedora/get/o:7126/bdef:Content/get ; http://vbs.rs/scripts/cobiss?command=SEARCH&base=99999&select=ID=44646927

Chicago Manual of Style (16th Edition):

Ana, Božović. “Estrogen receptor beta (ERß) promoter methylation and expression in invasive breast cancers.” 2013. Doctoral Dissertation, University of Belgrade. Accessed January 17, 2021. http://dx.doi.org/10.2298/BG20130703BOZOVIC ; http://eteze.bg.ac.rs/application/showtheses?thesesId=799 ; https://fedorabg.bg.ac.rs/fedora/get/o:7126/bdef:Content/get ; http://vbs.rs/scripts/cobiss?command=SEARCH&base=99999&select=ID=44646927.

MLA Handbook (7th Edition):

Ana, Božović. “Estrogen receptor beta (ERß) promoter methylation and expression in invasive breast cancers.” 2013. Web. 17 Jan 2021.

Vancouver:

Ana B. Estrogen receptor beta (ERß) promoter methylation and expression in invasive breast cancers. [Internet] [Doctoral dissertation]. University of Belgrade; 2013. [cited 2021 Jan 17]. Available from: http://dx.doi.org/10.2298/BG20130703BOZOVIC ; http://eteze.bg.ac.rs/application/showtheses?thesesId=799 ; https://fedorabg.bg.ac.rs/fedora/get/o:7126/bdef:Content/get ; http://vbs.rs/scripts/cobiss?command=SEARCH&base=99999&select=ID=44646927.

Council of Science Editors:

Ana B. Estrogen receptor beta (ERß) promoter methylation and expression in invasive breast cancers. [Doctoral Dissertation]. University of Belgrade; 2013. Available from: http://dx.doi.org/10.2298/BG20130703BOZOVIC ; http://eteze.bg.ac.rs/application/showtheses?thesesId=799 ; https://fedorabg.bg.ac.rs/fedora/get/o:7126/bdef:Content/get ; http://vbs.rs/scripts/cobiss?command=SEARCH&base=99999&select=ID=44646927

24. Börjesson, Anna. The role of estrogen receptor alpha in the regulation of bone and growth plate cartilage.

Degree: 2012, University of Gothenburg / Göteborgs Universitet

 Estrogens are important endocrine regulators of skeletal growth and maintenance in both females and males. Studies have demonstrated that the estrogen receptor (ER)-α is the… (more)

Subjects/Keywords: estrogen receptor α; bone; growth plate; estrogen

…The role of estrogen receptor α in the regulation of bone and growth plate cartilage AF… …growth factor Enzyme-linked immunosorbent assay Estrogen receptor α Total estrogen receptor α… …estrogen receptor modulator Specificity protein 1 Tumor necrosis factor α Micro computed… …estrogen receptor α in the regulation of bone and growth plate cartilage 10 Anna Börjesson… …and secrete matrix proteins which form a 11 The role of estrogen receptor α in the… 

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APA (6th Edition):

Börjesson, A. (2012). The role of estrogen receptor alpha in the regulation of bone and growth plate cartilage. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/30567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Börjesson, Anna. “The role of estrogen receptor alpha in the regulation of bone and growth plate cartilage.” 2012. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed January 17, 2021. http://hdl.handle.net/2077/30567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Börjesson, Anna. “The role of estrogen receptor alpha in the regulation of bone and growth plate cartilage.” 2012. Web. 17 Jan 2021.

Vancouver:

Börjesson A. The role of estrogen receptor alpha in the regulation of bone and growth plate cartilage. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2012. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/2077/30567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Börjesson A. The role of estrogen receptor alpha in the regulation of bone and growth plate cartilage. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2012. Available from: http://hdl.handle.net/2077/30567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

25. Dietrich, Alicia. Role of estrogen receptor α and oxidative stress response proteins in the central nervous system.

Degree: PhD, 0325, 2014, University of Illinois – Urbana-Champaign

 Cellular metabolism results in the production of reactive oxygen species (ROS) as byproducts and can damage proteins, lipids, and DNA. Cells utilize a number of… (more)

Subjects/Keywords: Oxidative stress; Estrogen receptor α; Cu/Zn superoxide dismutase; Amyotrophic lateral sclerosis; Apurinic endonuclease 1; Neuroprotection; Hypoxia; Estrogen

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APA (6th Edition):

Dietrich, A. (2014). Role of estrogen receptor α and oxidative stress response proteins in the central nervous system. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/50567

Chicago Manual of Style (16th Edition):

Dietrich, Alicia. “Role of estrogen receptor α and oxidative stress response proteins in the central nervous system.” 2014. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed January 17, 2021. http://hdl.handle.net/2142/50567.

MLA Handbook (7th Edition):

Dietrich, Alicia. “Role of estrogen receptor α and oxidative stress response proteins in the central nervous system.” 2014. Web. 17 Jan 2021.

Vancouver:

Dietrich A. Role of estrogen receptor α and oxidative stress response proteins in the central nervous system. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2014. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/2142/50567.

Council of Science Editors:

Dietrich A. Role of estrogen receptor α and oxidative stress response proteins in the central nervous system. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2014. Available from: http://hdl.handle.net/2142/50567


Vanderbilt University

26. Palmisano, Brian T. Cholesteryl Ester Transfer Protein Modulates Liver Sex Hormone Signaling to Alter Triglyceride Metabolism in Male and Female Transgenic Mice.

Degree: PhD, Molecular Physiology and Biophysics, 2016, Vanderbilt University

 Elevated plasma triglycerides (TGs) increase risk of cardiovascular disease, especially in women. Estrogen treatment raises plasma TGs in women, but molecular mechanisms remain poorly understood.… (more)

Subjects/Keywords: CETP; beta-oxidation; Low Density Lipoprotein Receptor; LDLR; ER alpha; Estrogen Receptor alpha; SHP; Small Heterodimer Partner; VLDL; Estrogen; TGs; Triglycerides; Cholesteryl Ester Transfer Protein

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APA (6th Edition):

Palmisano, B. T. (2016). Cholesteryl Ester Transfer Protein Modulates Liver Sex Hormone Signaling to Alter Triglyceride Metabolism in Male and Female Transgenic Mice. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13947

Chicago Manual of Style (16th Edition):

Palmisano, Brian T. “Cholesteryl Ester Transfer Protein Modulates Liver Sex Hormone Signaling to Alter Triglyceride Metabolism in Male and Female Transgenic Mice.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2021. http://hdl.handle.net/1803/13947.

MLA Handbook (7th Edition):

Palmisano, Brian T. “Cholesteryl Ester Transfer Protein Modulates Liver Sex Hormone Signaling to Alter Triglyceride Metabolism in Male and Female Transgenic Mice.” 2016. Web. 17 Jan 2021.

Vancouver:

Palmisano BT. Cholesteryl Ester Transfer Protein Modulates Liver Sex Hormone Signaling to Alter Triglyceride Metabolism in Male and Female Transgenic Mice. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/1803/13947.

Council of Science Editors:

Palmisano BT. Cholesteryl Ester Transfer Protein Modulates Liver Sex Hormone Signaling to Alter Triglyceride Metabolism in Male and Female Transgenic Mice. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/13947


East Carolina University

27. Iñigo, Melissa Mae Raval. Induced ablation of skeletal muscle-specific estrogen receptor-alpha in adult female mice increased the susceptibility to develop skeletal muscle inflammation and glucose intolerance under chronic lipid overload.

Degree: PhD, PHD-Physiology, 2018, East Carolina University

 Skeletal muscle-specific ER[alpha] appears to play important roles in regulating skeletal muscle glucose and lipid homeostasis. The overall aim of this dissertation was to determine… (more)

Subjects/Keywords: metabolism; mitochondrial respiration; OXPHOS efficiency; H2O2 emission potential; glucose tolerance; Estrogen Receptor alpha; Mice; Female; Animals; estrogen receptor alpha, human; Glucose Intolerance; Myositis; Muscle, Skeletal; Inflammation; Lipids

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APA (6th Edition):

Iñigo, M. M. R. (2018). Induced ablation of skeletal muscle-specific estrogen receptor-alpha in adult female mice increased the susceptibility to develop skeletal muscle inflammation and glucose intolerance under chronic lipid overload. (Doctoral Dissertation). East Carolina University. Retrieved from http://hdl.handle.net/10342/6785

Chicago Manual of Style (16th Edition):

Iñigo, Melissa Mae Raval. “Induced ablation of skeletal muscle-specific estrogen receptor-alpha in adult female mice increased the susceptibility to develop skeletal muscle inflammation and glucose intolerance under chronic lipid overload.” 2018. Doctoral Dissertation, East Carolina University. Accessed January 17, 2021. http://hdl.handle.net/10342/6785.

MLA Handbook (7th Edition):

Iñigo, Melissa Mae Raval. “Induced ablation of skeletal muscle-specific estrogen receptor-alpha in adult female mice increased the susceptibility to develop skeletal muscle inflammation and glucose intolerance under chronic lipid overload.” 2018. Web. 17 Jan 2021.

Vancouver:

Iñigo MMR. Induced ablation of skeletal muscle-specific estrogen receptor-alpha in adult female mice increased the susceptibility to develop skeletal muscle inflammation and glucose intolerance under chronic lipid overload. [Internet] [Doctoral dissertation]. East Carolina University; 2018. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/10342/6785.

Council of Science Editors:

Iñigo MMR. Induced ablation of skeletal muscle-specific estrogen receptor-alpha in adult female mice increased the susceptibility to develop skeletal muscle inflammation and glucose intolerance under chronic lipid overload. [Doctoral Dissertation]. East Carolina University; 2018. Available from: http://hdl.handle.net/10342/6785


University of Texas Southwestern Medical Center

28. Murakami, Shino. Molecular Mechanisms and Functions of Estrogen Receptor Enhancers in Hormone-Dependent Gene Expression.

Degree: 2017, University of Texas Southwestern Medical Center

 Transcription is a fundamental regulatory mechanism of biological processes in a range of physiological and pathological conditions. Transcription enhancers are DNA regulatory elements that regulate… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing; Enhancer Elements, Genetic; Estrogen Receptor alpha; Estrogens; RNA, Untranslated

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APA (6th Edition):

Murakami, S. (2017). Molecular Mechanisms and Functions of Estrogen Receptor Enhancers in Hormone-Dependent Gene Expression. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7728

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Murakami, Shino. “Molecular Mechanisms and Functions of Estrogen Receptor Enhancers in Hormone-Dependent Gene Expression.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed January 17, 2021. http://hdl.handle.net/2152.5/7728.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Murakami, Shino. “Molecular Mechanisms and Functions of Estrogen Receptor Enhancers in Hormone-Dependent Gene Expression.” 2017. Web. 17 Jan 2021.

Vancouver:

Murakami S. Molecular Mechanisms and Functions of Estrogen Receptor Enhancers in Hormone-Dependent Gene Expression. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/2152.5/7728.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Murakami S. Molecular Mechanisms and Functions of Estrogen Receptor Enhancers in Hormone-Dependent Gene Expression. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7728

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

29. McLachlan, Michael J. Transcription Factor Engineering: Tools and Applications.

Degree: PhD, 0319, 2011, University of Illinois – Urbana-Champaign

 Transcription factors play a vital role in the biology of every organism. By controlling gene expression they regulate growth, development, metabolism, reproduction, signaling, and response… (more)

Subjects/Keywords: Transcription factor; estrogen receptor alpha; protein engineering; biosensor; vascular endothelial growth factor-A (VEGF-A)

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APA (6th Edition):

McLachlan, M. J. (2011). Transcription Factor Engineering: Tools and Applications. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/18624

Chicago Manual of Style (16th Edition):

McLachlan, Michael J. “Transcription Factor Engineering: Tools and Applications.” 2011. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed January 17, 2021. http://hdl.handle.net/2142/18624.

MLA Handbook (7th Edition):

McLachlan, Michael J. “Transcription Factor Engineering: Tools and Applications.” 2011. Web. 17 Jan 2021.

Vancouver:

McLachlan MJ. Transcription Factor Engineering: Tools and Applications. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2011. [cited 2021 Jan 17]. Available from: http://hdl.handle.net/2142/18624.

Council of Science Editors:

McLachlan MJ. Transcription Factor Engineering: Tools and Applications. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2011. Available from: http://hdl.handle.net/2142/18624


University of Guelph

30. Mitchnick, Krista. The Dissociable Involvement of Epigenetic Mechanisms in Hippocampus- and Perirhinal Cortex-mediated Object Memory in Male Rats.

Degree: PhD, Department of Psychology, 2018, University of Guelph

 This thesis investigated the involvement of several epigenetic proteins in hippocampus (HPC)- and perirhinal cortex (PRh)-mediated object recognition. Previous literature has demonstrated the requirement for… (more)

Subjects/Keywords: DNA methylation; DNA demethylation; histone acetylation; DNMT; GADD45; PCAF; CBP; p300; estrogen receptor alpha

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APA (6th Edition):

Mitchnick, K. (2018). The Dissociable Involvement of Epigenetic Mechanisms in Hippocampus- and Perirhinal Cortex-mediated Object Memory in Male Rats. (Doctoral Dissertation). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/13096

Chicago Manual of Style (16th Edition):

Mitchnick, Krista. “The Dissociable Involvement of Epigenetic Mechanisms in Hippocampus- and Perirhinal Cortex-mediated Object Memory in Male Rats.” 2018. Doctoral Dissertation, University of Guelph. Accessed January 17, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/13096.

MLA Handbook (7th Edition):

Mitchnick, Krista. “The Dissociable Involvement of Epigenetic Mechanisms in Hippocampus- and Perirhinal Cortex-mediated Object Memory in Male Rats.” 2018. Web. 17 Jan 2021.

Vancouver:

Mitchnick K. The Dissociable Involvement of Epigenetic Mechanisms in Hippocampus- and Perirhinal Cortex-mediated Object Memory in Male Rats. [Internet] [Doctoral dissertation]. University of Guelph; 2018. [cited 2021 Jan 17]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/13096.

Council of Science Editors:

Mitchnick K. The Dissociable Involvement of Epigenetic Mechanisms in Hippocampus- and Perirhinal Cortex-mediated Object Memory in Male Rats. [Doctoral Dissertation]. University of Guelph; 2018. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/13096

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