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You searched for subject:(Epithelial to mesenchymal transition). Showing records 1 – 30 of 32080 total matches.

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1. Sultana, Shamima. Overexpression of microRNA-542-3p attenuates the differentiating capacity of endometriotic stromal cells. : miR-542-3p の過剰発現は子宮内膜症間質細胞の脱落膜化能力を低下させる.

Degree: 博士(医学), 2018, Saitama Medical University / 埼玉医科大学

Endometriosis is defined as the presence of endometrial glandular and stromal cells outside of the uterine cavity. A previous study reported that microRNA (miR)-542-3p plays… (more)

Subjects/Keywords: decidualization; endometriosis; mesenchymal‐to‐epithelial transition; microRNA; migration

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sultana, S. (2018). Overexpression of microRNA-542-3p attenuates the differentiating capacity of endometriotic stromal cells. : miR-542-3p の過剰発現は子宮内膜症間質細胞の脱落膜化能力を低下させる. (Thesis). Saitama Medical University / 埼玉医科大学. Retrieved from http://id.nii.ac.jp/1386/00000611/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sultana, Shamima. “Overexpression of microRNA-542-3p attenuates the differentiating capacity of endometriotic stromal cells. : miR-542-3p の過剰発現は子宮内膜症間質細胞の脱落膜化能力を低下させる.” 2018. Thesis, Saitama Medical University / 埼玉医科大学. Accessed July 23, 2019. http://id.nii.ac.jp/1386/00000611/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sultana, Shamima. “Overexpression of microRNA-542-3p attenuates the differentiating capacity of endometriotic stromal cells. : miR-542-3p の過剰発現は子宮内膜症間質細胞の脱落膜化能力を低下させる.” 2018. Web. 23 Jul 2019.

Vancouver:

Sultana S. Overexpression of microRNA-542-3p attenuates the differentiating capacity of endometriotic stromal cells. : miR-542-3p の過剰発現は子宮内膜症間質細胞の脱落膜化能力を低下させる. [Internet] [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. [cited 2019 Jul 23]. Available from: http://id.nii.ac.jp/1386/00000611/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sultana S. Overexpression of microRNA-542-3p attenuates the differentiating capacity of endometriotic stromal cells. : miR-542-3p の過剰発現は子宮内膜症間質細胞の脱落膜化能力を低下させる. [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. Available from: http://id.nii.ac.jp/1386/00000611/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

2. Kao, Yu-Chen. Study the effects of Dibromotyrosine Derivative in TGF-β responsiveness.

Degree: Master, Biological Sciences, 2017, NSYSU

 The Transforming growth factor β1 (TGF-β1) is belong to transforming growth factor superfamily. Many tumor lesions process are related to TGF-β1, such as: cell proliferation,… (more)

Subjects/Keywords: TGF-β; bromotyrosine derivative; small molecular inhibitors; epithelial-to-mesenchymal transition

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APA (6th Edition):

Kao, Y. (2017). Study the effects of Dibromotyrosine Derivative in TGF-β responsiveness. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0718117-194840

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kao, Yu-Chen. “Study the effects of Dibromotyrosine Derivative in TGF-β responsiveness.” 2017. Thesis, NSYSU. Accessed July 23, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0718117-194840.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kao, Yu-Chen. “Study the effects of Dibromotyrosine Derivative in TGF-β responsiveness.” 2017. Web. 23 Jul 2019.

Vancouver:

Kao Y. Study the effects of Dibromotyrosine Derivative in TGF-β responsiveness. [Internet] [Thesis]. NSYSU; 2017. [cited 2019 Jul 23]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0718117-194840.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kao Y. Study the effects of Dibromotyrosine Derivative in TGF-β responsiveness. [Thesis]. NSYSU; 2017. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0718117-194840

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

3. Li, Yanyang. Proteomic Characterization of Embryonic Epicardial-Myocardial Signaling.

Degree: 2017, University of Illinois – Chicago

 The communication between the epicardium and the underlying myocardium is crucial not only for proper heart development but also for homeostasis and response to injury… (more)

Subjects/Keywords: Cardiovascular development; NF-κB; Epithelial-to-Mesenchymal Transition; Mass spectrometry

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APA (6th Edition):

Li, Y. (2017). Proteomic Characterization of Embryonic Epicardial-Myocardial Signaling. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21930

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Yanyang. “Proteomic Characterization of Embryonic Epicardial-Myocardial Signaling.” 2017. Thesis, University of Illinois – Chicago. Accessed July 23, 2019. http://hdl.handle.net/10027/21930.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Yanyang. “Proteomic Characterization of Embryonic Epicardial-Myocardial Signaling.” 2017. Web. 23 Jul 2019.

Vancouver:

Li Y. Proteomic Characterization of Embryonic Epicardial-Myocardial Signaling. [Internet] [Thesis]. University of Illinois – Chicago; 2017. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/10027/21930.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li Y. Proteomic Characterization of Embryonic Epicardial-Myocardial Signaling. [Thesis]. University of Illinois – Chicago; 2017. Available from: http://hdl.handle.net/10027/21930

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

4. Pilzecker, B. The E- and N-cadherin switch in Epithelial to Mesenchymal Transition and metastasis. Potential drug targets?.

Degree: 2013, Universiteit Utrecht

 Carcinomas are most prevalent type of cancers and arise from an epithelial layer. Epithelial layers have a strict organization; cells are tightly linked through different… (more)

Subjects/Keywords: Carcinomas; Epithelial to Mesenchymal Transition (EMT); E-cadherin; N-cadherin

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APA (6th Edition):

Pilzecker, B. (2013). The E- and N-cadherin switch in Epithelial to Mesenchymal Transition and metastasis. Potential drug targets?. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/287101

Chicago Manual of Style (16th Edition):

Pilzecker, B. “The E- and N-cadherin switch in Epithelial to Mesenchymal Transition and metastasis. Potential drug targets?.” 2013. Masters Thesis, Universiteit Utrecht. Accessed July 23, 2019. http://dspace.library.uu.nl:8080/handle/1874/287101.

MLA Handbook (7th Edition):

Pilzecker, B. “The E- and N-cadherin switch in Epithelial to Mesenchymal Transition and metastasis. Potential drug targets?.” 2013. Web. 23 Jul 2019.

Vancouver:

Pilzecker B. The E- and N-cadherin switch in Epithelial to Mesenchymal Transition and metastasis. Potential drug targets?. [Internet] [Masters thesis]. Universiteit Utrecht; 2013. [cited 2019 Jul 23]. Available from: http://dspace.library.uu.nl:8080/handle/1874/287101.

Council of Science Editors:

Pilzecker B. The E- and N-cadherin switch in Epithelial to Mesenchymal Transition and metastasis. Potential drug targets?. [Masters Thesis]. Universiteit Utrecht; 2013. Available from: http://dspace.library.uu.nl:8080/handle/1874/287101


Princeton University

5. Ell, Brian James. Tumor-Intrinsic and -Extrinsic Mechanisms in Breast Cancer Metastasis .

Degree: PhD, 2013, Princeton University

 In breast cancer, mortality is predominantly associated with metastasis, cancerous spread to distant organs. Understanding tumor dissemination is vitally important to continued therapeutic advancement, but… (more)

Subjects/Keywords: Breast Cancer; Epithelial-to-mesenchymal transition; Metastasis; MicroRNA; Osteoclast

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APA (6th Edition):

Ell, B. J. (2013). Tumor-Intrinsic and -Extrinsic Mechanisms in Breast Cancer Metastasis . (Doctoral Dissertation). Princeton University. Retrieved from http://arks.princeton.edu/ark:/88435/dsp015x21tf54k

Chicago Manual of Style (16th Edition):

Ell, Brian James. “Tumor-Intrinsic and -Extrinsic Mechanisms in Breast Cancer Metastasis .” 2013. Doctoral Dissertation, Princeton University. Accessed July 23, 2019. http://arks.princeton.edu/ark:/88435/dsp015x21tf54k.

MLA Handbook (7th Edition):

Ell, Brian James. “Tumor-Intrinsic and -Extrinsic Mechanisms in Breast Cancer Metastasis .” 2013. Web. 23 Jul 2019.

Vancouver:

Ell BJ. Tumor-Intrinsic and -Extrinsic Mechanisms in Breast Cancer Metastasis . [Internet] [Doctoral dissertation]. Princeton University; 2013. [cited 2019 Jul 23]. Available from: http://arks.princeton.edu/ark:/88435/dsp015x21tf54k.

Council of Science Editors:

Ell BJ. Tumor-Intrinsic and -Extrinsic Mechanisms in Breast Cancer Metastasis . [Doctoral Dissertation]. Princeton University; 2013. Available from: http://arks.princeton.edu/ark:/88435/dsp015x21tf54k


University of Melbourne

6. Nguyen, Linh My. Effects of chemotherapy on colorectal liver metastases.

Degree: 2012, University of Melbourne

 Background: Colorectal cancer (CRC) is the fourth most frequently occurring cancer in the world. Despite optimum surgical endeavours, many patients will develop disease recurrence. Treatments… (more)

Subjects/Keywords: OXi4503; Sunitinib; epithelial to mesenchymal transition; colorectal liver metastases

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APA (6th Edition):

Nguyen, L. M. (2012). Effects of chemotherapy on colorectal liver metastases. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37303

Chicago Manual of Style (16th Edition):

Nguyen, Linh My. “Effects of chemotherapy on colorectal liver metastases.” 2012. Doctoral Dissertation, University of Melbourne. Accessed July 23, 2019. http://hdl.handle.net/11343/37303.

MLA Handbook (7th Edition):

Nguyen, Linh My. “Effects of chemotherapy on colorectal liver metastases.” 2012. Web. 23 Jul 2019.

Vancouver:

Nguyen LM. Effects of chemotherapy on colorectal liver metastases. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/11343/37303.

Council of Science Editors:

Nguyen LM. Effects of chemotherapy on colorectal liver metastases. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37303


University of Ottawa

7. Conway, Jillian. A Novel Role for SLK in Transforming Growth Factor-Beta-Mediated Epithelial-to-Mesenchymal Transition .

Degree: 2017, University of Ottawa

 In the late stages of cancer, tumors acquire the ability to spread throughout the body and invade distant tissues in a process called metastasis. Studies… (more)

Subjects/Keywords: Epithelial-to-mesenchymal transition; Ste20-like kinase; Transforming Growth Factor-Beta

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APA (6th Edition):

Conway, J. (2017). A Novel Role for SLK in Transforming Growth Factor-Beta-Mediated Epithelial-to-Mesenchymal Transition . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/35699

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Conway, Jillian. “A Novel Role for SLK in Transforming Growth Factor-Beta-Mediated Epithelial-to-Mesenchymal Transition .” 2017. Thesis, University of Ottawa. Accessed July 23, 2019. http://hdl.handle.net/10393/35699.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Conway, Jillian. “A Novel Role for SLK in Transforming Growth Factor-Beta-Mediated Epithelial-to-Mesenchymal Transition .” 2017. Web. 23 Jul 2019.

Vancouver:

Conway J. A Novel Role for SLK in Transforming Growth Factor-Beta-Mediated Epithelial-to-Mesenchymal Transition . [Internet] [Thesis]. University of Ottawa; 2017. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/10393/35699.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Conway J. A Novel Role for SLK in Transforming Growth Factor-Beta-Mediated Epithelial-to-Mesenchymal Transition . [Thesis]. University of Ottawa; 2017. Available from: http://hdl.handle.net/10393/35699

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

8. Petrovich, Giulia. Investigating the differential instructive roles of WT1's isoforms.

Degree: PhD, 2016, University of Edinburgh

 The Wilms' tumour suppressor gene Wt1 is a key regulator of embryonic development and tissue homeostasis. In humans, mutation in the gene may lead to… (more)

Subjects/Keywords: 616.99; roles of WT1 isoforms; WT1; epithelial to mesenchymal transition; EMT

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APA (6th Edition):

Petrovich, G. (2016). Investigating the differential instructive roles of WT1's isoforms. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/23423

Chicago Manual of Style (16th Edition):

Petrovich, Giulia. “Investigating the differential instructive roles of WT1's isoforms.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed July 23, 2019. http://hdl.handle.net/1842/23423.

MLA Handbook (7th Edition):

Petrovich, Giulia. “Investigating the differential instructive roles of WT1's isoforms.” 2016. Web. 23 Jul 2019.

Vancouver:

Petrovich G. Investigating the differential instructive roles of WT1's isoforms. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1842/23423.

Council of Science Editors:

Petrovich G. Investigating the differential instructive roles of WT1's isoforms. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/23423


University of South Florida

9. Richards, Edward J. Function of Long Noncoding RNAs in Breast Cancer.

Degree: 2015, University of South Florida

 Breast cancer is a disease that will be diagnosed in about 1 in 10 women throughout their lifetime. The majority of breast cancers are originated… (more)

Subjects/Keywords: TGFβ; epithelial to mesenchymal transition; metastasis; Cell Biology; Molecular Biology

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APA (6th Edition):

Richards, E. J. (2015). Function of Long Noncoding RNAs in Breast Cancer. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/5767

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Richards, Edward J. “Function of Long Noncoding RNAs in Breast Cancer.” 2015. Thesis, University of South Florida. Accessed July 23, 2019. https://scholarcommons.usf.edu/etd/5767.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Richards, Edward J. “Function of Long Noncoding RNAs in Breast Cancer.” 2015. Web. 23 Jul 2019.

Vancouver:

Richards EJ. Function of Long Noncoding RNAs in Breast Cancer. [Internet] [Thesis]. University of South Florida; 2015. [cited 2019 Jul 23]. Available from: https://scholarcommons.usf.edu/etd/5767.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Richards EJ. Function of Long Noncoding RNAs in Breast Cancer. [Thesis]. University of South Florida; 2015. Available from: https://scholarcommons.usf.edu/etd/5767

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

10. Fairchild, Corinne Leigh Alinea. Restricting cell movement: the role of Tspan18 in neural crest migration.

Degree: PhD, 2013, University of Minnesota

 The neural crest is a unique population of stem cells that arise from the developing central nervous system of vertebrate embryos. Unlike surrounding neuroepithelial cells,… (more)

Subjects/Keywords: Cadherin; Epithelial-to-mesenchymal transition (EMT); FoxD3; Neural crest; Tetraspanin

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APA (6th Edition):

Fairchild, C. L. A. (2013). Restricting cell movement: the role of Tspan18 in neural crest migration. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/157977

Chicago Manual of Style (16th Edition):

Fairchild, Corinne Leigh Alinea. “Restricting cell movement: the role of Tspan18 in neural crest migration.” 2013. Doctoral Dissertation, University of Minnesota. Accessed July 23, 2019. http://purl.umn.edu/157977.

MLA Handbook (7th Edition):

Fairchild, Corinne Leigh Alinea. “Restricting cell movement: the role of Tspan18 in neural crest migration.” 2013. Web. 23 Jul 2019.

Vancouver:

Fairchild CLA. Restricting cell movement: the role of Tspan18 in neural crest migration. [Internet] [Doctoral dissertation]. University of Minnesota; 2013. [cited 2019 Jul 23]. Available from: http://purl.umn.edu/157977.

Council of Science Editors:

Fairchild CLA. Restricting cell movement: the role of Tspan18 in neural crest migration. [Doctoral Dissertation]. University of Minnesota; 2013. Available from: http://purl.umn.edu/157977


University of Iowa

11. Nauseef, Jones Trevor. An investigation of the molecular and biophysical properties of metastatic cells.

Degree: PhD, Molecular Physiology and Biophysics, 2015, University of Iowa

  Prostate cancer presents a significant paradox: it is very common, yet rarely fatal. To wit, the prostate is the most common non-skin tissue for… (more)

Subjects/Keywords: publicabstract; Biophysics; Cancer; Epithelial to mesenchymal transition; Metastasis; Microfluidics; Biophysics

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APA (6th Edition):

Nauseef, J. T. (2015). An investigation of the molecular and biophysical properties of metastatic cells. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/3150

Chicago Manual of Style (16th Edition):

Nauseef, Jones Trevor. “An investigation of the molecular and biophysical properties of metastatic cells.” 2015. Doctoral Dissertation, University of Iowa. Accessed July 23, 2019. https://ir.uiowa.edu/etd/3150.

MLA Handbook (7th Edition):

Nauseef, Jones Trevor. “An investigation of the molecular and biophysical properties of metastatic cells.” 2015. Web. 23 Jul 2019.

Vancouver:

Nauseef JT. An investigation of the molecular and biophysical properties of metastatic cells. [Internet] [Doctoral dissertation]. University of Iowa; 2015. [cited 2019 Jul 23]. Available from: https://ir.uiowa.edu/etd/3150.

Council of Science Editors:

Nauseef JT. An investigation of the molecular and biophysical properties of metastatic cells. [Doctoral Dissertation]. University of Iowa; 2015. Available from: https://ir.uiowa.edu/etd/3150


University of Toronto

12. Brethour, Dylan Edward. PrP and its Ancestral Relatives ZIP6 and ZIP10 Interact with NCAM1, Altering its Molecular Environment and Post-translational Modifications during Epithelial-to-mesenchymal Transition.

Degree: 2016, University of Toronto

The prion protein (PrP) was recently found to be evolutionarily linked to a subfamily of ZIP transporters which possess a PrP-like domain. A member of… (more)

Subjects/Keywords: epithelial-to-mesenchymal transition; mass spectrometry; ncam; prion protein; ZIP6; 0317

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APA (6th Edition):

Brethour, D. E. (2016). PrP and its Ancestral Relatives ZIP6 and ZIP10 Interact with NCAM1, Altering its Molecular Environment and Post-translational Modifications during Epithelial-to-mesenchymal Transition. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/80225

Chicago Manual of Style (16th Edition):

Brethour, Dylan Edward. “PrP and its Ancestral Relatives ZIP6 and ZIP10 Interact with NCAM1, Altering its Molecular Environment and Post-translational Modifications during Epithelial-to-mesenchymal Transition.” 2016. Masters Thesis, University of Toronto. Accessed July 23, 2019. http://hdl.handle.net/1807/80225.

MLA Handbook (7th Edition):

Brethour, Dylan Edward. “PrP and its Ancestral Relatives ZIP6 and ZIP10 Interact with NCAM1, Altering its Molecular Environment and Post-translational Modifications during Epithelial-to-mesenchymal Transition.” 2016. Web. 23 Jul 2019.

Vancouver:

Brethour DE. PrP and its Ancestral Relatives ZIP6 and ZIP10 Interact with NCAM1, Altering its Molecular Environment and Post-translational Modifications during Epithelial-to-mesenchymal Transition. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1807/80225.

Council of Science Editors:

Brethour DE. PrP and its Ancestral Relatives ZIP6 and ZIP10 Interact with NCAM1, Altering its Molecular Environment and Post-translational Modifications during Epithelial-to-mesenchymal Transition. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/80225


University of California – Irvine

13. Villarreal Ponce, Alvaro P. An Ovol2-Zeb1 EMT-Regulatory Circuit Governs Mammary Basal-Luminal Binary Differentiation.

Degree: Biomedical Sciences, 2017, University of California – Irvine

 The capacity of epithelial cells to acquire enhanced lineage plasticity could depend on their ability to undergo EMT. Investigations performed on cultured epithelial cells support… (more)

Subjects/Keywords: Biology; Biochemistry; EMT; Epithelial Stem Cells; Epithelial-to-Mesenchymal Transition; Mammary gland; Ovol2; Zeb1

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APA (6th Edition):

Villarreal Ponce, A. P. (2017). An Ovol2-Zeb1 EMT-Regulatory Circuit Governs Mammary Basal-Luminal Binary Differentiation. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/5b8693wg

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Villarreal Ponce, Alvaro P. “An Ovol2-Zeb1 EMT-Regulatory Circuit Governs Mammary Basal-Luminal Binary Differentiation.” 2017. Thesis, University of California – Irvine. Accessed July 23, 2019. http://www.escholarship.org/uc/item/5b8693wg.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Villarreal Ponce, Alvaro P. “An Ovol2-Zeb1 EMT-Regulatory Circuit Governs Mammary Basal-Luminal Binary Differentiation.” 2017. Web. 23 Jul 2019.

Vancouver:

Villarreal Ponce AP. An Ovol2-Zeb1 EMT-Regulatory Circuit Governs Mammary Basal-Luminal Binary Differentiation. [Internet] [Thesis]. University of California – Irvine; 2017. [cited 2019 Jul 23]. Available from: http://www.escholarship.org/uc/item/5b8693wg.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Villarreal Ponce AP. An Ovol2-Zeb1 EMT-Regulatory Circuit Governs Mammary Basal-Luminal Binary Differentiation. [Thesis]. University of California – Irvine; 2017. Available from: http://www.escholarship.org/uc/item/5b8693wg

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

14. Rasmussen, Jeffrey Philip. Epithelial morphogenesis of the Caenorhabditis elegans pharynx.

Degree: PhD, 2012, University of Washington

 The assembly of cells into functional organs requires the coordination of cell shape and polarity with organ architecture. Although defects in cell shape and polarity… (more)

Subjects/Keywords: Caenorhabditis elegans; epithelial polarity; fusogen; laminin; mesenchymal to epithelial transition; tubulogenesis; Molecular and cellular biology

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APA (6th Edition):

Rasmussen, J. P. (2012). Epithelial morphogenesis of the Caenorhabditis elegans pharynx. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/19742

Chicago Manual of Style (16th Edition):

Rasmussen, Jeffrey Philip. “Epithelial morphogenesis of the Caenorhabditis elegans pharynx.” 2012. Doctoral Dissertation, University of Washington. Accessed July 23, 2019. http://hdl.handle.net/1773/19742.

MLA Handbook (7th Edition):

Rasmussen, Jeffrey Philip. “Epithelial morphogenesis of the Caenorhabditis elegans pharynx.” 2012. Web. 23 Jul 2019.

Vancouver:

Rasmussen JP. Epithelial morphogenesis of the Caenorhabditis elegans pharynx. [Internet] [Doctoral dissertation]. University of Washington; 2012. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1773/19742.

Council of Science Editors:

Rasmussen JP. Epithelial morphogenesis of the Caenorhabditis elegans pharynx. [Doctoral Dissertation]. University of Washington; 2012. Available from: http://hdl.handle.net/1773/19742


Universiteit Utrecht

15. Wiebrands, K. A role of epithelial-mesenchymal transitions in carcinogenic progression.

Degree: 2010, Universiteit Utrecht

 The epithelial-mesenchymal transition plays an important role in several developmental processes, tissue repair, but is also associated with fibrosis and cancer. During tumorigenic progression, EMT… (more)

Subjects/Keywords: epithelial-mesenchymal transition; EMT; metastasis

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APA (6th Edition):

Wiebrands, K. (2010). A role of epithelial-mesenchymal transitions in carcinogenic progression. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/190315

Chicago Manual of Style (16th Edition):

Wiebrands, K. “A role of epithelial-mesenchymal transitions in carcinogenic progression.” 2010. Masters Thesis, Universiteit Utrecht. Accessed July 23, 2019. http://dspace.library.uu.nl:8080/handle/1874/190315.

MLA Handbook (7th Edition):

Wiebrands, K. “A role of epithelial-mesenchymal transitions in carcinogenic progression.” 2010. Web. 23 Jul 2019.

Vancouver:

Wiebrands K. A role of epithelial-mesenchymal transitions in carcinogenic progression. [Internet] [Masters thesis]. Universiteit Utrecht; 2010. [cited 2019 Jul 23]. Available from: http://dspace.library.uu.nl:8080/handle/1874/190315.

Council of Science Editors:

Wiebrands K. A role of epithelial-mesenchymal transitions in carcinogenic progression. [Masters Thesis]. Universiteit Utrecht; 2010. Available from: http://dspace.library.uu.nl:8080/handle/1874/190315


University of Manitoba

16. Al Hattab, Danah. Role of scleraxis in regulating snai1 gene expression and its effect on epithelialmesenchymal transition.

Degree: Physiology and Pathophysiology, 2017, University of Manitoba

Epithelial-mesenchymal transition (EMT) is a process in which epithelial cells lose their ability for cell to cell adhesion, causing a pheno-conversion of epithelial cells into… (more)

Subjects/Keywords: Scleraxis; Snai1; Epithelial- mesenchymal transition

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APA (6th Edition):

Al Hattab, D. (2017). Role of scleraxis in regulating snai1 gene expression and its effect on epithelial –mesenchymal transition. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/32750

Chicago Manual of Style (16th Edition):

Al Hattab, Danah. “Role of scleraxis in regulating snai1 gene expression and its effect on epithelial –mesenchymal transition.” 2017. Masters Thesis, University of Manitoba. Accessed July 23, 2019. http://hdl.handle.net/1993/32750.

MLA Handbook (7th Edition):

Al Hattab, Danah. “Role of scleraxis in regulating snai1 gene expression and its effect on epithelial –mesenchymal transition.” 2017. Web. 23 Jul 2019.

Vancouver:

Al Hattab D. Role of scleraxis in regulating snai1 gene expression and its effect on epithelial –mesenchymal transition. [Internet] [Masters thesis]. University of Manitoba; 2017. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1993/32750.

Council of Science Editors:

Al Hattab D. Role of scleraxis in regulating snai1 gene expression and its effect on epithelial –mesenchymal transition. [Masters Thesis]. University of Manitoba; 2017. Available from: http://hdl.handle.net/1993/32750

17. Mourareau, Céline. Bio-CAD - Etude de biomarqueurs de progression tumorale dans les cancers des voies aéro-digestives supérieures en fonction de leur statut HPV. : Bio-CAD - Study of tumor progression biomarkers in upper aerodigestive tract cancers according to their HPV status.

Degree: Docteur es, Biologie céllulaire, 2016, Reims

Chaque année 610 000 cancers sont diagnostiqués dans le monde induits par une infection à papillomavirus humains à haut-risque (HPV-HR). Bien que les carcinomes des… (more)

Subjects/Keywords: Carcinome; Oropharynx; Papillomavirus Humain; Transition Epithélio-Mésenchymateuse; Carcinoma; Oropharynx; Human Papillomavirus; Epithelial-To-Mesenchymal Transition

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APA (6th Edition):

Mourareau, C. (2016). Bio-CAD - Etude de biomarqueurs de progression tumorale dans les cancers des voies aéro-digestives supérieures en fonction de leur statut HPV. : Bio-CAD - Study of tumor progression biomarkers in upper aerodigestive tract cancers according to their HPV status. (Doctoral Dissertation). Reims. Retrieved from http://www.theses.fr/2016REIMS029

Chicago Manual of Style (16th Edition):

Mourareau, Céline. “Bio-CAD - Etude de biomarqueurs de progression tumorale dans les cancers des voies aéro-digestives supérieures en fonction de leur statut HPV. : Bio-CAD - Study of tumor progression biomarkers in upper aerodigestive tract cancers according to their HPV status.” 2016. Doctoral Dissertation, Reims. Accessed July 23, 2019. http://www.theses.fr/2016REIMS029.

MLA Handbook (7th Edition):

Mourareau, Céline. “Bio-CAD - Etude de biomarqueurs de progression tumorale dans les cancers des voies aéro-digestives supérieures en fonction de leur statut HPV. : Bio-CAD - Study of tumor progression biomarkers in upper aerodigestive tract cancers according to their HPV status.” 2016. Web. 23 Jul 2019.

Vancouver:

Mourareau C. Bio-CAD - Etude de biomarqueurs de progression tumorale dans les cancers des voies aéro-digestives supérieures en fonction de leur statut HPV. : Bio-CAD - Study of tumor progression biomarkers in upper aerodigestive tract cancers according to their HPV status. [Internet] [Doctoral dissertation]. Reims; 2016. [cited 2019 Jul 23]. Available from: http://www.theses.fr/2016REIMS029.

Council of Science Editors:

Mourareau C. Bio-CAD - Etude de biomarqueurs de progression tumorale dans les cancers des voies aéro-digestives supérieures en fonction de leur statut HPV. : Bio-CAD - Study of tumor progression biomarkers in upper aerodigestive tract cancers according to their HPV status. [Doctoral Dissertation]. Reims; 2016. Available from: http://www.theses.fr/2016REIMS029

18. Molina-Castro, Silvia. Study of the Hippo/YAP1 signaling pathway in gastric carcinogenesis induced by Helicobacter pylori : Etude de la voie de signalisation HIPPO/YAP dans la carcinogenèse gastrique induite par l'infection à Helicobacter pylori.

Degree: Docteur es, Microbiologie Immunologie, 2017, Bordeaux

 Le cancer gastrique (CG) est une maladie multifactorielle, fréquemment associée à l’infection chronique par des souches CagA+ d’Helicobacter pylori. La transition épithélio-mésenchymateuse (EMT) est un… (more)

Subjects/Keywords: Cellules souches gastriques; Transition épithélio-mésenchymateuse; CD44; Cancer stem cells; Epithelial to mesenchymal transition; CD44

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APA (6th Edition):

Molina-Castro, S. (2017). Study of the Hippo/YAP1 signaling pathway in gastric carcinogenesis induced by Helicobacter pylori : Etude de la voie de signalisation HIPPO/YAP dans la carcinogenèse gastrique induite par l'infection à Helicobacter pylori. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2017BORD0623

Chicago Manual of Style (16th Edition):

Molina-Castro, Silvia. “Study of the Hippo/YAP1 signaling pathway in gastric carcinogenesis induced by Helicobacter pylori : Etude de la voie de signalisation HIPPO/YAP dans la carcinogenèse gastrique induite par l'infection à Helicobacter pylori.” 2017. Doctoral Dissertation, Bordeaux. Accessed July 23, 2019. http://www.theses.fr/2017BORD0623.

MLA Handbook (7th Edition):

Molina-Castro, Silvia. “Study of the Hippo/YAP1 signaling pathway in gastric carcinogenesis induced by Helicobacter pylori : Etude de la voie de signalisation HIPPO/YAP dans la carcinogenèse gastrique induite par l'infection à Helicobacter pylori.” 2017. Web. 23 Jul 2019.

Vancouver:

Molina-Castro S. Study of the Hippo/YAP1 signaling pathway in gastric carcinogenesis induced by Helicobacter pylori : Etude de la voie de signalisation HIPPO/YAP dans la carcinogenèse gastrique induite par l'infection à Helicobacter pylori. [Internet] [Doctoral dissertation]. Bordeaux; 2017. [cited 2019 Jul 23]. Available from: http://www.theses.fr/2017BORD0623.

Council of Science Editors:

Molina-Castro S. Study of the Hippo/YAP1 signaling pathway in gastric carcinogenesis induced by Helicobacter pylori : Etude de la voie de signalisation HIPPO/YAP dans la carcinogenèse gastrique induite par l'infection à Helicobacter pylori. [Doctoral Dissertation]. Bordeaux; 2017. Available from: http://www.theses.fr/2017BORD0623


University of Vienna

19. Martinez Turtos, Adriana. Extracellular vesicles and their impact on the epithelial plasticity of hepatocellular carcinoma cells.

Degree: 2018, University of Vienna

 Leberkrebs ist der weltweit sechsthäufigste maligne Tumor, der zur zweithäufigsten Mortalität aller Krebserkrankten führt. Das hepatozelluläre Karzinom (HCC) ist der vorherrschende primäre Leberkrebs. Die schlechte… (more)

Subjects/Keywords: 42.13 Molekularbiologie; hepatozelluläre Karzinom / epithelial zu mesenchymalen Transition / extrazelluläre Vesikel; hepatocellular carcinoma / epithelial-to-mesenchymal transition / extracellular vesicles

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APA (6th Edition):

Martinez Turtos, A. (2018). Extracellular vesicles and their impact on the epithelial plasticity of hepatocellular carcinoma cells. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/51143/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Martinez Turtos, Adriana. “Extracellular vesicles and their impact on the epithelial plasticity of hepatocellular carcinoma cells.” 2018. Thesis, University of Vienna. Accessed July 23, 2019. http://othes.univie.ac.at/51143/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Martinez Turtos, Adriana. “Extracellular vesicles and their impact on the epithelial plasticity of hepatocellular carcinoma cells.” 2018. Web. 23 Jul 2019.

Vancouver:

Martinez Turtos A. Extracellular vesicles and their impact on the epithelial plasticity of hepatocellular carcinoma cells. [Internet] [Thesis]. University of Vienna; 2018. [cited 2019 Jul 23]. Available from: http://othes.univie.ac.at/51143/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Martinez Turtos A. Extracellular vesicles and their impact on the epithelial plasticity of hepatocellular carcinoma cells. [Thesis]. University of Vienna; 2018. Available from: http://othes.univie.ac.at/51143/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Princeton University

20. Li, Wenyang. Identification and Characterization of Deubiquitinases and Long Non-coding RNAs as Novel Regulators of Breast Cancer .

Degree: PhD, 2017, Princeton University

 Abstract Cancer initiation and progression is orchestrated by a complicated web of signaling pathways with numerous effector proteins and RNAs. My studies focus on identifying… (more)

Subjects/Keywords: Cancer; Deubiquitinase; Epithelial to mesenchymal transition; Long non-coding RNA; SLUG; TGF beta

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APA (6th Edition):

Li, W. (2017). Identification and Characterization of Deubiquitinases and Long Non-coding RNAs as Novel Regulators of Breast Cancer . (Doctoral Dissertation). Princeton University. Retrieved from http://arks.princeton.edu/ark:/88435/dsp01st74ct124

Chicago Manual of Style (16th Edition):

Li, Wenyang. “Identification and Characterization of Deubiquitinases and Long Non-coding RNAs as Novel Regulators of Breast Cancer .” 2017. Doctoral Dissertation, Princeton University. Accessed July 23, 2019. http://arks.princeton.edu/ark:/88435/dsp01st74ct124.

MLA Handbook (7th Edition):

Li, Wenyang. “Identification and Characterization of Deubiquitinases and Long Non-coding RNAs as Novel Regulators of Breast Cancer .” 2017. Web. 23 Jul 2019.

Vancouver:

Li W. Identification and Characterization of Deubiquitinases and Long Non-coding RNAs as Novel Regulators of Breast Cancer . [Internet] [Doctoral dissertation]. Princeton University; 2017. [cited 2019 Jul 23]. Available from: http://arks.princeton.edu/ark:/88435/dsp01st74ct124.

Council of Science Editors:

Li W. Identification and Characterization of Deubiquitinases and Long Non-coding RNAs as Novel Regulators of Breast Cancer . [Doctoral Dissertation]. Princeton University; 2017. Available from: http://arks.princeton.edu/ark:/88435/dsp01st74ct124

21. Zakopoulou, Roubini. Μελέτη σταδίων καρκινογένεσης που σχετίζονται με επιθηλιο - μεσεγχυματική μετατροπή σε κυτταρικές σειρές και αξιολόγηση των καρκινικών δεικτών που σχετίζονται με τον παραπάνω φαινότυπο.

Degree: 2014, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

E-cadherin (CDH1) loss occurs frequently in carcinogenesis contributing to invasionand metastasis. We observed that mouse and human epithelial cell linesoverexpressing the replication licensing factor Cdc6… (more)

Subjects/Keywords: Ε-καδερίνη; Επιθηλιο-μεσεγχυματική Μετατροπή; E-cadherin; Cdc6; Epithelial to mesenchymal transition (EMT)

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APA (6th Edition):

Zakopoulou, R. (2014). Μελέτη σταδίων καρκινογένεσης που σχετίζονται με επιθηλιο - μεσεγχυματική μετατροπή σε κυτταρικές σειρές και αξιολόγηση των καρκινικών δεικτών που σχετίζονται με τον παραπάνω φαινότυπο. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/38425

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zakopoulou, Roubini. “Μελέτη σταδίων καρκινογένεσης που σχετίζονται με επιθηλιο - μεσεγχυματική μετατροπή σε κυτταρικές σειρές και αξιολόγηση των καρκινικών δεικτών που σχετίζονται με τον παραπάνω φαινότυπο.” 2014. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed July 23, 2019. http://hdl.handle.net/10442/hedi/38425.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zakopoulou, Roubini. “Μελέτη σταδίων καρκινογένεσης που σχετίζονται με επιθηλιο - μεσεγχυματική μετατροπή σε κυτταρικές σειρές και αξιολόγηση των καρκινικών δεικτών που σχετίζονται με τον παραπάνω φαινότυπο.” 2014. Web. 23 Jul 2019.

Vancouver:

Zakopoulou R. Μελέτη σταδίων καρκινογένεσης που σχετίζονται με επιθηλιο - μεσεγχυματική μετατροπή σε κυτταρικές σειρές και αξιολόγηση των καρκινικών δεικτών που σχετίζονται με τον παραπάνω φαινότυπο. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2014. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/10442/hedi/38425.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zakopoulou R. Μελέτη σταδίων καρκινογένεσης που σχετίζονται με επιθηλιο - μεσεγχυματική μετατροπή σε κυτταρικές σειρές και αξιολόγηση των καρκινικών δεικτών που σχετίζονται με τον παραπάνω φαινότυπο. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2014. Available from: http://hdl.handle.net/10442/hedi/38425

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kansas

22. Crow, Jennifer M. Tumor-Derived Exosomes Mediate Platinum Resistance in Ovarian Cancer Through Regulation of EMT.

Degree: PhD, Pathology & Laboratory Medicine, 2016, University of Kansas

 Nano-sized vesicles, termed exosomes, have been implicated in the transfer of oncogenic proteins and genetic material from one cell to another. We speculated this may… (more)

Subjects/Keywords: Pathology; Genetics; Oncology; Carboplatin; Epithelial to Mesenchymal Transition; Ovarian Cancer; Platinum; SMAD; TGF-beta

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APA (6th Edition):

Crow, J. M. (2016). Tumor-Derived Exosomes Mediate Platinum Resistance in Ovarian Cancer Through Regulation of EMT. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/25365

Chicago Manual of Style (16th Edition):

Crow, Jennifer M. “Tumor-Derived Exosomes Mediate Platinum Resistance in Ovarian Cancer Through Regulation of EMT.” 2016. Doctoral Dissertation, University of Kansas. Accessed July 23, 2019. http://hdl.handle.net/1808/25365.

MLA Handbook (7th Edition):

Crow, Jennifer M. “Tumor-Derived Exosomes Mediate Platinum Resistance in Ovarian Cancer Through Regulation of EMT.” 2016. Web. 23 Jul 2019.

Vancouver:

Crow JM. Tumor-Derived Exosomes Mediate Platinum Resistance in Ovarian Cancer Through Regulation of EMT. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1808/25365.

Council of Science Editors:

Crow JM. Tumor-Derived Exosomes Mediate Platinum Resistance in Ovarian Cancer Through Regulation of EMT. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/25365


Case Western Reserve University

23. Yori, Jennifer L. Discerning The Role Of Krüppel-Like Factor 4 In Breast Cancer.

Degree: PhD, Pharmacology, 2011, Case Western Reserve University

  Krüppel-like factor 4 (KLF4) is a zinc-finger transcription factor which plays an integral role in a myriad of cellular processes, ranging from regulation of… (more)

Subjects/Keywords: Biomedical Research; Cellular Biology; Pharmacology; KLF4; E-cadherin; Epithelial-to-Mesenchymal Transition; breast cancer; metastasis

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APA (6th Edition):

Yori, J. L. (2011). Discerning The Role Of Krüppel-Like Factor 4 In Breast Cancer. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1295636538

Chicago Manual of Style (16th Edition):

Yori, Jennifer L. “Discerning The Role Of Krüppel-Like Factor 4 In Breast Cancer.” 2011. Doctoral Dissertation, Case Western Reserve University. Accessed July 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1295636538.

MLA Handbook (7th Edition):

Yori, Jennifer L. “Discerning The Role Of Krüppel-Like Factor 4 In Breast Cancer.” 2011. Web. 23 Jul 2019.

Vancouver:

Yori JL. Discerning The Role Of Krüppel-Like Factor 4 In Breast Cancer. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2011. [cited 2019 Jul 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1295636538.

Council of Science Editors:

Yori JL. Discerning The Role Of Krüppel-Like Factor 4 In Breast Cancer. [Doctoral Dissertation]. Case Western Reserve University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1295636538


Penn State University

24. Wokpetah, Joseph Mawumenyo. Regulation of cancer stem cells by the tumor microenvironment.

Degree: MS, Chemical Engineering, 2014, Penn State University

 Cancer is the abnormal growth of cells that tend to multiply in an uncontrolled way and, in some cases, metastasize to other locations in the… (more)

Subjects/Keywords: Epithelial to mesenchymal transition; metastasis; transforming growth factor beta; Young's modulus; upregulation; downregulation

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APA (6th Edition):

Wokpetah, J. M. (2014). Regulation of cancer stem cells by the tumor microenvironment. (Masters Thesis). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/21336

Chicago Manual of Style (16th Edition):

Wokpetah, Joseph Mawumenyo. “Regulation of cancer stem cells by the tumor microenvironment.” 2014. Masters Thesis, Penn State University. Accessed July 23, 2019. https://etda.libraries.psu.edu/catalog/21336.

MLA Handbook (7th Edition):

Wokpetah, Joseph Mawumenyo. “Regulation of cancer stem cells by the tumor microenvironment.” 2014. Web. 23 Jul 2019.

Vancouver:

Wokpetah JM. Regulation of cancer stem cells by the tumor microenvironment. [Internet] [Masters thesis]. Penn State University; 2014. [cited 2019 Jul 23]. Available from: https://etda.libraries.psu.edu/catalog/21336.

Council of Science Editors:

Wokpetah JM. Regulation of cancer stem cells by the tumor microenvironment. [Masters Thesis]. Penn State University; 2014. Available from: https://etda.libraries.psu.edu/catalog/21336


University of Ottawa

25. Carter, Lauren. Defining the Epithelial-to-Mesenchymal Transition and Regulation of Stemness in the Ovarian Surface Epithelium .

Degree: 2018, University of Ottawa

 The ovarian surface epithelium (OSE) is a monolayer of cells surrounding the ovary that is ruptured during ovulation. After ovulation the wound is repaired, however… (more)

Subjects/Keywords: ovarian surface epithelium; ovulation; stemness; epithelial-to-mesenchymal transition; wound repair; Cox2; Brca1; Snai1

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APA (6th Edition):

Carter, L. (2018). Defining the Epithelial-to-Mesenchymal Transition and Regulation of Stemness in the Ovarian Surface Epithelium . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/38491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carter, Lauren. “Defining the Epithelial-to-Mesenchymal Transition and Regulation of Stemness in the Ovarian Surface Epithelium .” 2018. Thesis, University of Ottawa. Accessed July 23, 2019. http://hdl.handle.net/10393/38491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carter, Lauren. “Defining the Epithelial-to-Mesenchymal Transition and Regulation of Stemness in the Ovarian Surface Epithelium .” 2018. Web. 23 Jul 2019.

Vancouver:

Carter L. Defining the Epithelial-to-Mesenchymal Transition and Regulation of Stemness in the Ovarian Surface Epithelium . [Internet] [Thesis]. University of Ottawa; 2018. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/10393/38491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carter L. Defining the Epithelial-to-Mesenchymal Transition and Regulation of Stemness in the Ovarian Surface Epithelium . [Thesis]. University of Ottawa; 2018. Available from: http://hdl.handle.net/10393/38491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Ohio University

26. Carlson, Grady E. The Epithelial-to-Mesenchymal Transition Regulates the E-selectin Ligand Activities of Breast Cancer Cells.

Degree: PhD, Chemical Engineering (Engineering and Technology), 2016, Ohio University

 During metastasis the epithelial-to-mesenchymal transition (EMT) increases the motility and invasiveness of breast cancer cells (BCs). However, it has yet to be determined whether the… (more)

Subjects/Keywords: Chemical Engineering; Biomedical Engineering; Breast cancer; metastasis; epithelial-to-mesenchymal transition; E-selectin; adhesion

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APA (6th Edition):

Carlson, G. E. (2016). The Epithelial-to-Mesenchymal Transition Regulates the E-selectin Ligand Activities of Breast Cancer Cells. (Doctoral Dissertation). Ohio University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1477670221426081

Chicago Manual of Style (16th Edition):

Carlson, Grady E. “The Epithelial-to-Mesenchymal Transition Regulates the E-selectin Ligand Activities of Breast Cancer Cells.” 2016. Doctoral Dissertation, Ohio University. Accessed July 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1477670221426081.

MLA Handbook (7th Edition):

Carlson, Grady E. “The Epithelial-to-Mesenchymal Transition Regulates the E-selectin Ligand Activities of Breast Cancer Cells.” 2016. Web. 23 Jul 2019.

Vancouver:

Carlson GE. The Epithelial-to-Mesenchymal Transition Regulates the E-selectin Ligand Activities of Breast Cancer Cells. [Internet] [Doctoral dissertation]. Ohio University; 2016. [cited 2019 Jul 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1477670221426081.

Council of Science Editors:

Carlson GE. The Epithelial-to-Mesenchymal Transition Regulates the E-selectin Ligand Activities of Breast Cancer Cells. [Doctoral Dissertation]. Ohio University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1477670221426081


University of Western Ontario

27. Blom, Jessica N. Cardiac repair post-myocardial infarction: Roles of the primary cilium and the long non-coding RNA Malat1.

Degree: 2017, University of Western Ontario

 Complete occlusion of a coronary artery causes myocardial infarction (MI), resulting in cardiomyocyte cell death. The surviving myocardium undergoes a deleterious remodeling process which causes… (more)

Subjects/Keywords: Myocardial infarction; epicardium-derived cells; epithelial-to-mesenchymal transition; Wt1; primary cilium; Malat1; Medical Physiology

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APA (6th Edition):

Blom, J. N. (2017). Cardiac repair post-myocardial infarction: Roles of the primary cilium and the long non-coding RNA Malat1. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4897

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Blom, Jessica N. “Cardiac repair post-myocardial infarction: Roles of the primary cilium and the long non-coding RNA Malat1.” 2017. Thesis, University of Western Ontario. Accessed July 23, 2019. https://ir.lib.uwo.ca/etd/4897.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Blom, Jessica N. “Cardiac repair post-myocardial infarction: Roles of the primary cilium and the long non-coding RNA Malat1.” 2017. Web. 23 Jul 2019.

Vancouver:

Blom JN. Cardiac repair post-myocardial infarction: Roles of the primary cilium and the long non-coding RNA Malat1. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2019 Jul 23]. Available from: https://ir.lib.uwo.ca/etd/4897.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Blom JN. Cardiac repair post-myocardial infarction: Roles of the primary cilium and the long non-coding RNA Malat1. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4897

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. G. Celesti. MESENCHYMAL FEATURES MEDIATED BY TWIST1 IN COLORECTAL CANCER CELLS AND MICROENVIRONMENT.

Degree: 2011, Università degli Studi di Milano

 Background. Colorectal cancer (CRC) is a major cause of death for cancer in western countries, ranking third in both sexes. Therapeutic developments in the past… (more)

Subjects/Keywords: epithelial to mesenchymal transition; colorectal cancer; metastasis; twist1; Settore BIO/10 - Biochimica

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APA (6th Edition):

Celesti, G. (2011). MESENCHYMAL FEATURES MEDIATED BY TWIST1 IN COLORECTAL CANCER CELLS AND MICROENVIRONMENT. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/158084

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Celesti, G.. “MESENCHYMAL FEATURES MEDIATED BY TWIST1 IN COLORECTAL CANCER CELLS AND MICROENVIRONMENT.” 2011. Thesis, Università degli Studi di Milano. Accessed July 23, 2019. http://hdl.handle.net/2434/158084.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Celesti, G.. “MESENCHYMAL FEATURES MEDIATED BY TWIST1 IN COLORECTAL CANCER CELLS AND MICROENVIRONMENT.” 2011. Web. 23 Jul 2019.

Vancouver:

Celesti G. MESENCHYMAL FEATURES MEDIATED BY TWIST1 IN COLORECTAL CANCER CELLS AND MICROENVIRONMENT. [Internet] [Thesis]. Università degli Studi di Milano; 2011. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/2434/158084.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Celesti G. MESENCHYMAL FEATURES MEDIATED BY TWIST1 IN COLORECTAL CANCER CELLS AND MICROENVIRONMENT. [Thesis]. Università degli Studi di Milano; 2011. Available from: http://hdl.handle.net/2434/158084

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

29. Zhang, Hang. Theoretical and Computational Studies on the Dynamics and Regulation of Cell Phenotypic Transitions.

Degree: PhD, Animal and Poultry Sciences, 2016, Virginia Tech

 Cell phenotypic transitions, or cell fate decision making processes, are regulated by complex regulatory networks composed of genes, RNAs, proteins and metabolites. The regulation can… (more)

Subjects/Keywords: Mathematical Modeling; Epigenetics; Cell Differentiation; Mono-allelic expression; Epithelial-to-Mesenchymal-Transition

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, H. (2016). Theoretical and Computational Studies on the Dynamics and Regulation of Cell Phenotypic Transitions. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/65159

Chicago Manual of Style (16th Edition):

Zhang, Hang. “Theoretical and Computational Studies on the Dynamics and Regulation of Cell Phenotypic Transitions.” 2016. Doctoral Dissertation, Virginia Tech. Accessed July 23, 2019. http://hdl.handle.net/10919/65159.

MLA Handbook (7th Edition):

Zhang, Hang. “Theoretical and Computational Studies on the Dynamics and Regulation of Cell Phenotypic Transitions.” 2016. Web. 23 Jul 2019.

Vancouver:

Zhang H. Theoretical and Computational Studies on the Dynamics and Regulation of Cell Phenotypic Transitions. [Internet] [Doctoral dissertation]. Virginia Tech; 2016. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/10919/65159.

Council of Science Editors:

Zhang H. Theoretical and Computational Studies on the Dynamics and Regulation of Cell Phenotypic Transitions. [Doctoral Dissertation]. Virginia Tech; 2016. Available from: http://hdl.handle.net/10919/65159


University of Edinburgh

30. Cervantes Arias, Alejandro. Epithelial to Mesenchymal Transition and the generation of stem-like cells in companion animal breast cancer.

Degree: PhD, 2016, University of Edinburgh

 Breast cancer is the most common cancer in women and unspayed female dogs. The Epithelial to Mesenchymal Transition (EMT) is a process involved in embryogenesis,… (more)

Subjects/Keywords: 636.089; epithelial to mesenchymal transition; EMT; cancer; stem cell; companion animals; pets

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cervantes Arias, A. (2016). Epithelial to Mesenchymal Transition and the generation of stem-like cells in companion animal breast cancer. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/23556

Chicago Manual of Style (16th Edition):

Cervantes Arias, Alejandro. “Epithelial to Mesenchymal Transition and the generation of stem-like cells in companion animal breast cancer.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed July 23, 2019. http://hdl.handle.net/1842/23556.

MLA Handbook (7th Edition):

Cervantes Arias, Alejandro. “Epithelial to Mesenchymal Transition and the generation of stem-like cells in companion animal breast cancer.” 2016. Web. 23 Jul 2019.

Vancouver:

Cervantes Arias A. Epithelial to Mesenchymal Transition and the generation of stem-like cells in companion animal breast cancer. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2019 Jul 23]. Available from: http://hdl.handle.net/1842/23556.

Council of Science Editors:

Cervantes Arias A. Epithelial to Mesenchymal Transition and the generation of stem-like cells in companion animal breast cancer. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/23556

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