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You searched for subject:(Endoplasmic reticulum stress). Showing records 1 – 30 of 198 total matches.

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University of Cambridge

1. Capatina, Nadejda. Role of Endoplasmic Reticulum Stress in Trophoblast Stem Cell Differentiation.

Degree: PhD, 2019, University of Cambridge

 The project aimed to provide a mechanistic understanding of the effect of endoplasmic reticulum (ER) stress on the differentiation of trophoblast stem cells (TSCs), placental… (more)

Subjects/Keywords: endoplasmic reticulum stress; trophoblast; stem cell differentiation

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APA (6th Edition):

Capatina, N. (2019). Role of Endoplasmic Reticulum Stress in Trophoblast Stem Cell Differentiation. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/298767

Chicago Manual of Style (16th Edition):

Capatina, Nadejda. “Role of Endoplasmic Reticulum Stress in Trophoblast Stem Cell Differentiation.” 2019. Doctoral Dissertation, University of Cambridge. Accessed April 21, 2021. https://www.repository.cam.ac.uk/handle/1810/298767.

MLA Handbook (7th Edition):

Capatina, Nadejda. “Role of Endoplasmic Reticulum Stress in Trophoblast Stem Cell Differentiation.” 2019. Web. 21 Apr 2021.

Vancouver:

Capatina N. Role of Endoplasmic Reticulum Stress in Trophoblast Stem Cell Differentiation. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Apr 21]. Available from: https://www.repository.cam.ac.uk/handle/1810/298767.

Council of Science Editors:

Capatina N. Role of Endoplasmic Reticulum Stress in Trophoblast Stem Cell Differentiation. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/298767


Université de Bordeaux I

2. Bouchecareilh, Marion. Régulation de l’activité biologique de la protéine IRE1 : rôle dans le développement des cancers : The effects of exergames training on cognitive aging : a study of learning transfer.

Degree: Docteur es, Biologie cellulaire et physiopathologie, 2008, Université de Bordeaux I

Le Réticulum endoplasmique (RE) est le premier compartiment intracellulaire traversé par les protéines sécrétées. Au sein de cet organite, les protéines acquièrent une conformation native,… (more)

Subjects/Keywords: Réticulum Endoplasmique; Stress; Cancer; Ire1; Endoplasmic Reticulum; Stress; Cancer; Ire1

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APA (6th Edition):

Bouchecareilh, M. (2008). Régulation de l’activité biologique de la protéine IRE1 : rôle dans le développement des cancers : The effects of exergames training on cognitive aging : a study of learning transfer. (Doctoral Dissertation). Université de Bordeaux I. Retrieved from http://www.theses.fr/2008BOR13711

Chicago Manual of Style (16th Edition):

Bouchecareilh, Marion. “Régulation de l’activité biologique de la protéine IRE1 : rôle dans le développement des cancers : The effects of exergames training on cognitive aging : a study of learning transfer.” 2008. Doctoral Dissertation, Université de Bordeaux I. Accessed April 21, 2021. http://www.theses.fr/2008BOR13711.

MLA Handbook (7th Edition):

Bouchecareilh, Marion. “Régulation de l’activité biologique de la protéine IRE1 : rôle dans le développement des cancers : The effects of exergames training on cognitive aging : a study of learning transfer.” 2008. Web. 21 Apr 2021.

Vancouver:

Bouchecareilh M. Régulation de l’activité biologique de la protéine IRE1 : rôle dans le développement des cancers : The effects of exergames training on cognitive aging : a study of learning transfer. [Internet] [Doctoral dissertation]. Université de Bordeaux I; 2008. [cited 2021 Apr 21]. Available from: http://www.theses.fr/2008BOR13711.

Council of Science Editors:

Bouchecareilh M. Régulation de l’activité biologique de la protéine IRE1 : rôle dans le développement des cancers : The effects of exergames training on cognitive aging : a study of learning transfer. [Doctoral Dissertation]. Université de Bordeaux I; 2008. Available from: http://www.theses.fr/2008BOR13711


Colorado State University

3. Estrada, Andrea Lee. Role of fatty acids on endoplasmic reticulum proteostasis in non-alcoholic fatty liver disease, The.

Degree: PhD, Food Science and Human Nutrition, 2017, Colorado State University

 Non-alcoholic fatty liver disease (NAFLD) is currently a significant health concern in both adults and children. NAFLD is a disease characterized by accumulation of fat… (more)

Subjects/Keywords: endoplasmic reticulum stress; protein synthesis; saturated fatty acids; non-alcoholic fatty liver disease; endoplasmic reticulum; proteostasis

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APA (6th Edition):

Estrada, A. L. (2017). Role of fatty acids on endoplasmic reticulum proteostasis in non-alcoholic fatty liver disease, The. (Doctoral Dissertation). Colorado State University. Retrieved from http://hdl.handle.net/10217/185714

Chicago Manual of Style (16th Edition):

Estrada, Andrea Lee. “Role of fatty acids on endoplasmic reticulum proteostasis in non-alcoholic fatty liver disease, The.” 2017. Doctoral Dissertation, Colorado State University. Accessed April 21, 2021. http://hdl.handle.net/10217/185714.

MLA Handbook (7th Edition):

Estrada, Andrea Lee. “Role of fatty acids on endoplasmic reticulum proteostasis in non-alcoholic fatty liver disease, The.” 2017. Web. 21 Apr 2021.

Vancouver:

Estrada AL. Role of fatty acids on endoplasmic reticulum proteostasis in non-alcoholic fatty liver disease, The. [Internet] [Doctoral dissertation]. Colorado State University; 2017. [cited 2021 Apr 21]. Available from: http://hdl.handle.net/10217/185714.

Council of Science Editors:

Estrada AL. Role of fatty acids on endoplasmic reticulum proteostasis in non-alcoholic fatty liver disease, The. [Doctoral Dissertation]. Colorado State University; 2017. Available from: http://hdl.handle.net/10217/185714


University of Otago

4. Seo, Benedict Lloyd. Endoplasmic Reticulum Stress and Russell Bodies: Study of the Relationship Using a Periodontal Inflammation Model .

Degree: 2011, University of Otago

 The endoplasmic reticulum (ER) is an organelle of great importance. It represents the intracellular site for protein synthesis, folding, and post-translational modification. Intricately related processes… (more)

Subjects/Keywords: Endoplasmic reticulum stress; Russell Bodies; Unfolded protein response

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APA (6th Edition):

Seo, B. L. (2011). Endoplasmic Reticulum Stress and Russell Bodies: Study of the Relationship Using a Periodontal Inflammation Model . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/2041

Chicago Manual of Style (16th Edition):

Seo, Benedict Lloyd. “Endoplasmic Reticulum Stress and Russell Bodies: Study of the Relationship Using a Periodontal Inflammation Model .” 2011. Doctoral Dissertation, University of Otago. Accessed April 21, 2021. http://hdl.handle.net/10523/2041.

MLA Handbook (7th Edition):

Seo, Benedict Lloyd. “Endoplasmic Reticulum Stress and Russell Bodies: Study of the Relationship Using a Periodontal Inflammation Model .” 2011. Web. 21 Apr 2021.

Vancouver:

Seo BL. Endoplasmic Reticulum Stress and Russell Bodies: Study of the Relationship Using a Periodontal Inflammation Model . [Internet] [Doctoral dissertation]. University of Otago; 2011. [cited 2021 Apr 21]. Available from: http://hdl.handle.net/10523/2041.

Council of Science Editors:

Seo BL. Endoplasmic Reticulum Stress and Russell Bodies: Study of the Relationship Using a Periodontal Inflammation Model . [Doctoral Dissertation]. University of Otago; 2011. Available from: http://hdl.handle.net/10523/2041


Universiteit Utrecht

5. Renne, M.F. Endoplasmic Reticulum Stress and lipid metabolism.

Degree: 2014, Universiteit Utrecht

 Upon endoplasmic reticulum (ER) stress, the unfolded protein response (UPR) triggers cellular mechanisms to restore ER homeostasis. Aberrancies in lipid homeostasis can cause alterations in… (more)

Subjects/Keywords: Unfolded protein response; lipid metabolism; endoplasmic reticulum stress; membrane fluidity

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APA (6th Edition):

Renne, M. F. (2014). Endoplasmic Reticulum Stress and lipid metabolism. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/296878

Chicago Manual of Style (16th Edition):

Renne, M F. “Endoplasmic Reticulum Stress and lipid metabolism.” 2014. Masters Thesis, Universiteit Utrecht. Accessed April 21, 2021. http://dspace.library.uu.nl:8080/handle/1874/296878.

MLA Handbook (7th Edition):

Renne, M F. “Endoplasmic Reticulum Stress and lipid metabolism.” 2014. Web. 21 Apr 2021.

Vancouver:

Renne MF. Endoplasmic Reticulum Stress and lipid metabolism. [Internet] [Masters thesis]. Universiteit Utrecht; 2014. [cited 2021 Apr 21]. Available from: http://dspace.library.uu.nl:8080/handle/1874/296878.

Council of Science Editors:

Renne MF. Endoplasmic Reticulum Stress and lipid metabolism. [Masters Thesis]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/296878


University of Rochester

6. Syc-Mazurek, Stephanie B. The Molecular Signaling Pathways Controlling Apoptotic Retinal Ganglion Cell Death after Axonal Injury.

Degree: PhD, 2017, University of Rochester

 Vision loss in glaucoma is characterized by the stereotypical death of retinal ganglion cells (RGCs). Ocular hypertension is a major risk factor for glaucoma and… (more)

Subjects/Keywords: Axonopathy; Endoplasmic reticulum stress; Glaucoma; Mitogen activated kinase signaling; Neurodegeneration

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APA (6th Edition):

Syc-Mazurek, S. B. (2017). The Molecular Signaling Pathways Controlling Apoptotic Retinal Ganglion Cell Death after Axonal Injury. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/32918

Chicago Manual of Style (16th Edition):

Syc-Mazurek, Stephanie B. “The Molecular Signaling Pathways Controlling Apoptotic Retinal Ganglion Cell Death after Axonal Injury.” 2017. Doctoral Dissertation, University of Rochester. Accessed April 21, 2021. http://hdl.handle.net/1802/32918.

MLA Handbook (7th Edition):

Syc-Mazurek, Stephanie B. “The Molecular Signaling Pathways Controlling Apoptotic Retinal Ganglion Cell Death after Axonal Injury.” 2017. Web. 21 Apr 2021.

Vancouver:

Syc-Mazurek SB. The Molecular Signaling Pathways Controlling Apoptotic Retinal Ganglion Cell Death after Axonal Injury. [Internet] [Doctoral dissertation]. University of Rochester; 2017. [cited 2021 Apr 21]. Available from: http://hdl.handle.net/1802/32918.

Council of Science Editors:

Syc-Mazurek SB. The Molecular Signaling Pathways Controlling Apoptotic Retinal Ganglion Cell Death after Axonal Injury. [Doctoral Dissertation]. University of Rochester; 2017. Available from: http://hdl.handle.net/1802/32918


University of Alberta

7. Lara, Carlos J. The Herp and HRD1-dependent degradation of TRPP2.

Degree: MS, Department of Physiology, 2012, University of Alberta

 Autosomal Dominant Polycystic Kidney Disease (ADPKD) is a prevalent genetic disorder where multiple fluid-filled cysts destroy kidney architecture, eventually requiring hemodialysis or kidney transplant. Approximately… (more)

Subjects/Keywords: calcium signaling; endoplasmic reticulum stress; polycystic kidney disease; Herp; HRD1; TRPP2

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APA (6th Edition):

Lara, C. J. (2012). The Herp and HRD1-dependent degradation of TRPP2. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/p8418n98g

Chicago Manual of Style (16th Edition):

Lara, Carlos J. “The Herp and HRD1-dependent degradation of TRPP2.” 2012. Masters Thesis, University of Alberta. Accessed April 21, 2021. https://era.library.ualberta.ca/files/p8418n98g.

MLA Handbook (7th Edition):

Lara, Carlos J. “The Herp and HRD1-dependent degradation of TRPP2.” 2012. Web. 21 Apr 2021.

Vancouver:

Lara CJ. The Herp and HRD1-dependent degradation of TRPP2. [Internet] [Masters thesis]. University of Alberta; 2012. [cited 2021 Apr 21]. Available from: https://era.library.ualberta.ca/files/p8418n98g.

Council of Science Editors:

Lara CJ. The Herp and HRD1-dependent degradation of TRPP2. [Masters Thesis]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/p8418n98g


University of Alberta

8. Deslauriers, Andre. Endoplasmic reticulum stress induction by an endogenous retrovirus glycoprotein during neuroinflammation: regulation by a free radical scavenger.

Degree: MS, Department of Medical Microbiology and Immunology, 2010, University of Alberta

Endoplasmic reticulum (ER) stress is a homeostatic mechanism, which is utilized by cells to adapt to inter- and intra-cellular changes. There is a burgeoning literature… (more)

Subjects/Keywords: Human Endougenous Retroviruses; Endoplasmic Reticulum Stress; Multiple Sclerosis

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APA (6th Edition):

Deslauriers, A. (2010). Endoplasmic reticulum stress induction by an endogenous retrovirus glycoprotein during neuroinflammation: regulation by a free radical scavenger. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/bg257g06z

Chicago Manual of Style (16th Edition):

Deslauriers, Andre. “Endoplasmic reticulum stress induction by an endogenous retrovirus glycoprotein during neuroinflammation: regulation by a free radical scavenger.” 2010. Masters Thesis, University of Alberta. Accessed April 21, 2021. https://era.library.ualberta.ca/files/bg257g06z.

MLA Handbook (7th Edition):

Deslauriers, Andre. “Endoplasmic reticulum stress induction by an endogenous retrovirus glycoprotein during neuroinflammation: regulation by a free radical scavenger.” 2010. Web. 21 Apr 2021.

Vancouver:

Deslauriers A. Endoplasmic reticulum stress induction by an endogenous retrovirus glycoprotein during neuroinflammation: regulation by a free radical scavenger. [Internet] [Masters thesis]. University of Alberta; 2010. [cited 2021 Apr 21]. Available from: https://era.library.ualberta.ca/files/bg257g06z.

Council of Science Editors:

Deslauriers A. Endoplasmic reticulum stress induction by an endogenous retrovirus glycoprotein during neuroinflammation: regulation by a free radical scavenger. [Masters Thesis]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/bg257g06z

9. Rafiei, Hossein 1981-. Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease.

Degree: 2017, University of Saskatchewan

 Non-alcoholic fatty liver disease (NAFLD) is a public health burden. Steatosis as the “first hit”, and oxidative stress, inflammation, mitochondrial dysfunction, and endoplasmic reticulum stress(more)

Subjects/Keywords: Non-alcoholic fatty liver disease; Mitochondrial dysfunction; endoplasmic reticulum stress; Steatosis

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APA (6th Edition):

Rafiei, H. 1. (2017). Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/7946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rafiei, Hossein 1981-. “Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease.” 2017. Thesis, University of Saskatchewan. Accessed April 21, 2021. http://hdl.handle.net/10388/7946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rafiei, Hossein 1981-. “Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease.” 2017. Web. 21 Apr 2021.

Vancouver:

Rafiei H1. Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease. [Internet] [Thesis]. University of Saskatchewan; 2017. [cited 2021 Apr 21]. Available from: http://hdl.handle.net/10388/7946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rafiei H1. Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease. [Thesis]. University of Saskatchewan; 2017. Available from: http://hdl.handle.net/10388/7946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

10. Liang, Tian. Molecular Pathogenesis of Dentinogenesis Imperfecta Associated with Dentin Sialophosphoprotein Mutations.

Degree: PhD, Oral Biology, 2018, Texas A&M University

 Dentin sialophosphoprotein (DSPP) is abundantly expressed by odontoblasts, and transiently expressed by ameloblasts. The mutations in DSPP gene can cause dentinogenesis imperfecta (DGI) type II… (more)

Subjects/Keywords: Dentinogenesis Imperfecta; Dentin Sialophosphoprotein; Endoplasmic Reticulum Stress; Unfolded Protein Response

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APA (6th Edition):

Liang, T. (2018). Molecular Pathogenesis of Dentinogenesis Imperfecta Associated with Dentin Sialophosphoprotein Mutations. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/174013

Chicago Manual of Style (16th Edition):

Liang, Tian. “Molecular Pathogenesis of Dentinogenesis Imperfecta Associated with Dentin Sialophosphoprotein Mutations.” 2018. Doctoral Dissertation, Texas A&M University. Accessed April 21, 2021. http://hdl.handle.net/1969.1/174013.

MLA Handbook (7th Edition):

Liang, Tian. “Molecular Pathogenesis of Dentinogenesis Imperfecta Associated with Dentin Sialophosphoprotein Mutations.” 2018. Web. 21 Apr 2021.

Vancouver:

Liang T. Molecular Pathogenesis of Dentinogenesis Imperfecta Associated with Dentin Sialophosphoprotein Mutations. [Internet] [Doctoral dissertation]. Texas A&M University; 2018. [cited 2021 Apr 21]. Available from: http://hdl.handle.net/1969.1/174013.

Council of Science Editors:

Liang T. Molecular Pathogenesis of Dentinogenesis Imperfecta Associated with Dentin Sialophosphoprotein Mutations. [Doctoral Dissertation]. Texas A&M University; 2018. Available from: http://hdl.handle.net/1969.1/174013


McMaster University

11. Tat, Victor. THE ROLE OF THE IRE1α PATHWAY IN VASCULAR STIFFENING AND FIBROSIS.

Degree: MSc, 2017, McMaster University

Background: Vascular stiffening develops with both hypertension and aging, and is a strong predictor of end-organ damage. Excessive deposition of collagen by vascular smooth muscle… (more)

Subjects/Keywords: Hypertension; Unfolded Protein Response; Endoplasmic Reticulum Stress; Vascular Stiffening; Fibrosis

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APA (6th Edition):

Tat, V. (2017). THE ROLE OF THE IRE1α PATHWAY IN VASCULAR STIFFENING AND FIBROSIS. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/21052

Chicago Manual of Style (16th Edition):

Tat, Victor. “THE ROLE OF THE IRE1α PATHWAY IN VASCULAR STIFFENING AND FIBROSIS.” 2017. Masters Thesis, McMaster University. Accessed April 21, 2021. http://hdl.handle.net/11375/21052.

MLA Handbook (7th Edition):

Tat, Victor. “THE ROLE OF THE IRE1α PATHWAY IN VASCULAR STIFFENING AND FIBROSIS.” 2017. Web. 21 Apr 2021.

Vancouver:

Tat V. THE ROLE OF THE IRE1α PATHWAY IN VASCULAR STIFFENING AND FIBROSIS. [Internet] [Masters thesis]. McMaster University; 2017. [cited 2021 Apr 21]. Available from: http://hdl.handle.net/11375/21052.

Council of Science Editors:

Tat V. THE ROLE OF THE IRE1α PATHWAY IN VASCULAR STIFFENING AND FIBROSIS. [Masters Thesis]. McMaster University; 2017. Available from: http://hdl.handle.net/11375/21052

12. Carlisle, Rachel E. Inhibiting endoplasmic reticulum stress prevents the development of hypertensive nephrosclerosis.

Degree: PhD, 2017, McMaster University

Endoplasmic reticulum (ER) stress, which results from the aggregation of misfolded proteins in the ER, has been implicated in many forms of kidney injury, including… (more)

Subjects/Keywords: endoplasmic reticulum stress; chronic kidney disease; 4-phenylbutyrate; hypertension

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APA (6th Edition):

Carlisle, R. E. (2017). Inhibiting endoplasmic reticulum stress prevents the development of hypertensive nephrosclerosis. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/22040

Chicago Manual of Style (16th Edition):

Carlisle, Rachel E. “Inhibiting endoplasmic reticulum stress prevents the development of hypertensive nephrosclerosis.” 2017. Doctoral Dissertation, McMaster University. Accessed April 21, 2021. http://hdl.handle.net/11375/22040.

MLA Handbook (7th Edition):

Carlisle, Rachel E. “Inhibiting endoplasmic reticulum stress prevents the development of hypertensive nephrosclerosis.” 2017. Web. 21 Apr 2021.

Vancouver:

Carlisle RE. Inhibiting endoplasmic reticulum stress prevents the development of hypertensive nephrosclerosis. [Internet] [Doctoral dissertation]. McMaster University; 2017. [cited 2021 Apr 21]. Available from: http://hdl.handle.net/11375/22040.

Council of Science Editors:

Carlisle RE. Inhibiting endoplasmic reticulum stress prevents the development of hypertensive nephrosclerosis. [Doctoral Dissertation]. McMaster University; 2017. Available from: http://hdl.handle.net/11375/22040


NSYSU

13. Tsai, Tsung-Chang. Discovery of New Natural Products from Cultrued Soft Coral Sinularia sandensis and Investigation of Molecular Mechanisms of Apoptosis Induced by 7-Acetylsinumaximol B and Anti-metastasis of Dihydroaustrasulfone Alcohol on Cancer Cells.

Degree: PhD, Institute Of Marine Biotechnology And Resources, 2018, NSYSU

 Cultured soft coral Sinularia sandensis has led to the isolation of elenven natural compounds, including two new cembranoids, 4-carbomethoxyl-10-epigyrosanoldie E (1) and 7-acetylsinumaximol B (2);… (more)

Subjects/Keywords: apoptosis; endoplasmic reticulum stress; configuration; autophagy; migration; invasion

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APA (6th Edition):

Tsai, T. (2018). Discovery of New Natural Products from Cultrued Soft Coral Sinularia sandensis and Investigation of Molecular Mechanisms of Apoptosis Induced by 7-Acetylsinumaximol B and Anti-metastasis of Dihydroaustrasulfone Alcohol on Cancer Cells. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715118-003404

Chicago Manual of Style (16th Edition):

Tsai, Tsung-Chang. “Discovery of New Natural Products from Cultrued Soft Coral Sinularia sandensis and Investigation of Molecular Mechanisms of Apoptosis Induced by 7-Acetylsinumaximol B and Anti-metastasis of Dihydroaustrasulfone Alcohol on Cancer Cells.” 2018. Doctoral Dissertation, NSYSU. Accessed April 21, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715118-003404.

MLA Handbook (7th Edition):

Tsai, Tsung-Chang. “Discovery of New Natural Products from Cultrued Soft Coral Sinularia sandensis and Investigation of Molecular Mechanisms of Apoptosis Induced by 7-Acetylsinumaximol B and Anti-metastasis of Dihydroaustrasulfone Alcohol on Cancer Cells.” 2018. Web. 21 Apr 2021.

Vancouver:

Tsai T. Discovery of New Natural Products from Cultrued Soft Coral Sinularia sandensis and Investigation of Molecular Mechanisms of Apoptosis Induced by 7-Acetylsinumaximol B and Anti-metastasis of Dihydroaustrasulfone Alcohol on Cancer Cells. [Internet] [Doctoral dissertation]. NSYSU; 2018. [cited 2021 Apr 21]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715118-003404.

Council of Science Editors:

Tsai T. Discovery of New Natural Products from Cultrued Soft Coral Sinularia sandensis and Investigation of Molecular Mechanisms of Apoptosis Induced by 7-Acetylsinumaximol B and Anti-metastasis of Dihydroaustrasulfone Alcohol on Cancer Cells. [Doctoral Dissertation]. NSYSU; 2018. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715118-003404


University of California – San Francisco

14. Merksamer, Philip Ian. The Development and Application of Fluorescent Protein Reporters to Measure Endoplasmic Reticulum Stress in Single Cells.

Degree: Cell Biology, 2010, University of California – San Francisco

 In eukaryotic cells, secreted and membrane proteins fold within the endoplasmic reticulum (ER). Various physiological or pathophysiological conditions can disrupt ER protein folding homeostasis and… (more)

Subjects/Keywords: Cellular Biology; endoplasmic reticulum stress; fluorescent protein reporters; unfolded protein response

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APA (6th Edition):

Merksamer, P. I. (2010). The Development and Application of Fluorescent Protein Reporters to Measure Endoplasmic Reticulum Stress in Single Cells. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/1dw4d3hd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Merksamer, Philip Ian. “The Development and Application of Fluorescent Protein Reporters to Measure Endoplasmic Reticulum Stress in Single Cells.” 2010. Thesis, University of California – San Francisco. Accessed April 21, 2021. http://www.escholarship.org/uc/item/1dw4d3hd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Merksamer, Philip Ian. “The Development and Application of Fluorescent Protein Reporters to Measure Endoplasmic Reticulum Stress in Single Cells.” 2010. Web. 21 Apr 2021.

Vancouver:

Merksamer PI. The Development and Application of Fluorescent Protein Reporters to Measure Endoplasmic Reticulum Stress in Single Cells. [Internet] [Thesis]. University of California – San Francisco; 2010. [cited 2021 Apr 21]. Available from: http://www.escholarship.org/uc/item/1dw4d3hd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Merksamer PI. The Development and Application of Fluorescent Protein Reporters to Measure Endoplasmic Reticulum Stress in Single Cells. [Thesis]. University of California – San Francisco; 2010. Available from: http://www.escholarship.org/uc/item/1dw4d3hd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

15. Lerner, Alana Gabrielle. Investigation of apoptosis under endoplasmic reticulum stress during diabetes pathogenesis.

Degree: Biomedical Sciences, 2011, University of California – San Francisco

 Diabetes mellitus is a disease caused by a combination of insulin resistance and decline of &beta-cell function. There is a high demand on the pancreatic… (more)

Subjects/Keywords: Biology; apoptosis; diabetes; endoplasmic reticulum stress; IRE1; TXNIP

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APA (6th Edition):

Lerner, A. G. (2011). Investigation of apoptosis under endoplasmic reticulum stress during diabetes pathogenesis. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/6b79d4gk

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lerner, Alana Gabrielle. “Investigation of apoptosis under endoplasmic reticulum stress during diabetes pathogenesis.” 2011. Thesis, University of California – San Francisco. Accessed April 21, 2021. http://www.escholarship.org/uc/item/6b79d4gk.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lerner, Alana Gabrielle. “Investigation of apoptosis under endoplasmic reticulum stress during diabetes pathogenesis.” 2011. Web. 21 Apr 2021.

Vancouver:

Lerner AG. Investigation of apoptosis under endoplasmic reticulum stress during diabetes pathogenesis. [Internet] [Thesis]. University of California – San Francisco; 2011. [cited 2021 Apr 21]. Available from: http://www.escholarship.org/uc/item/6b79d4gk.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lerner AG. Investigation of apoptosis under endoplasmic reticulum stress during diabetes pathogenesis. [Thesis]. University of California – San Francisco; 2011. Available from: http://www.escholarship.org/uc/item/6b79d4gk

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

16. Gardner, Brooke Meghan. Direct Binding to Unfolded Proteins Activates Ire1 and the Unfolded Protein Response.

Degree: Biochemistry and Molecular Biology, 2012, University of California – San Francisco

 Secreted and transmembrane proteins enter the endoplasmic reticulum (ER) as unfolded, nascent polypeptides. Before they continue along the secretory pathway to their final destination, these… (more)

Subjects/Keywords: Biochemistry; Cellular biology; endoplasmic reticulum; Ire1; stress sensing; Unfolded Protein Response

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APA (6th Edition):

Gardner, B. M. (2012). Direct Binding to Unfolded Proteins Activates Ire1 and the Unfolded Protein Response. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/5vh5b89b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gardner, Brooke Meghan. “Direct Binding to Unfolded Proteins Activates Ire1 and the Unfolded Protein Response.” 2012. Thesis, University of California – San Francisco. Accessed April 21, 2021. http://www.escholarship.org/uc/item/5vh5b89b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gardner, Brooke Meghan. “Direct Binding to Unfolded Proteins Activates Ire1 and the Unfolded Protein Response.” 2012. Web. 21 Apr 2021.

Vancouver:

Gardner BM. Direct Binding to Unfolded Proteins Activates Ire1 and the Unfolded Protein Response. [Internet] [Thesis]. University of California – San Francisco; 2012. [cited 2021 Apr 21]. Available from: http://www.escholarship.org/uc/item/5vh5b89b.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gardner BM. Direct Binding to Unfolded Proteins Activates Ire1 and the Unfolded Protein Response. [Thesis]. University of California – San Francisco; 2012. Available from: http://www.escholarship.org/uc/item/5vh5b89b

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

17. Wang, Eric. The Role of Apoptosis in Differentiation and Disease.

Degree: Biomedical Sciences, 2015, University of California – San Francisco

 Apoptosis is a highly conserved form of programmed cell death in multicellular organisms where a cell activates caspase proteases to trigger its own demise. It… (more)

Subjects/Keywords: Biology; Apoptosis; Embryonic Stem Cell; Endoplasmic reticulum stress; IRE1α; Retinitis pigmentosa

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APA (6th Edition):

Wang, E. (2015). The Role of Apoptosis in Differentiation and Disease. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/3v92x78c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Eric. “The Role of Apoptosis in Differentiation and Disease.” 2015. Thesis, University of California – San Francisco. Accessed April 21, 2021. http://www.escholarship.org/uc/item/3v92x78c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Eric. “The Role of Apoptosis in Differentiation and Disease.” 2015. Web. 21 Apr 2021.

Vancouver:

Wang E. The Role of Apoptosis in Differentiation and Disease. [Internet] [Thesis]. University of California – San Francisco; 2015. [cited 2021 Apr 21]. Available from: http://www.escholarship.org/uc/item/3v92x78c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang E. The Role of Apoptosis in Differentiation and Disease. [Thesis]. University of California – San Francisco; 2015. Available from: http://www.escholarship.org/uc/item/3v92x78c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


National University of Ireland – Galway

18. Saveljeva, Svetlana. Cellular responses to endoplasmic reticulum stress: Role of autophagy and cell death .

Degree: 2014, National University of Ireland – Galway

 This PhD thesis describes cellular responses to the induction of endoplasmic reticulum (ER) stress, focusing on the role of autophagy and cell death. Pro-survival role… (more)

Subjects/Keywords: Endoplasmic reticulum stress; Cell death; Autophagy; Apoptosis; Necroptosis; Biochemistry

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APA (6th Edition):

Saveljeva, S. (2014). Cellular responses to endoplasmic reticulum stress: Role of autophagy and cell death . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/4786

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Saveljeva, Svetlana. “Cellular responses to endoplasmic reticulum stress: Role of autophagy and cell death .” 2014. Thesis, National University of Ireland – Galway. Accessed April 21, 2021. http://hdl.handle.net/10379/4786.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Saveljeva, Svetlana. “Cellular responses to endoplasmic reticulum stress: Role of autophagy and cell death .” 2014. Web. 21 Apr 2021.

Vancouver:

Saveljeva S. Cellular responses to endoplasmic reticulum stress: Role of autophagy and cell death . [Internet] [Thesis]. National University of Ireland – Galway; 2014. [cited 2021 Apr 21]. Available from: http://hdl.handle.net/10379/4786.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Saveljeva S. Cellular responses to endoplasmic reticulum stress: Role of autophagy and cell death . [Thesis]. National University of Ireland – Galway; 2014. Available from: http://hdl.handle.net/10379/4786

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of the Western Cape

19. Pillay, Leeshan. The integrated effects of selected inducers of endoplasmic reticulum stress, the unfolded protein response and apoptosis on P-Glycoprotein mediated drug resistance in MCF-7 breast carcinoma cells .

Degree: 2015, University of the Western Cape

 Purpose: One of the leading causes of death reported in women worldwide is breast cancer. Manytumours, including breast cancer, associated with poor prognosis, have received… (more)

Subjects/Keywords: Breast cancer; P-Glycoprotein; Apoptosis; Endoplasmic reticulum stress; Multidrug resistance

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APA (6th Edition):

Pillay, L. (2015). The integrated effects of selected inducers of endoplasmic reticulum stress, the unfolded protein response and apoptosis on P-Glycoprotein mediated drug resistance in MCF-7 breast carcinoma cells . (Thesis). University of the Western Cape. Retrieved from http://hdl.handle.net/11394/4459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pillay, Leeshan. “The integrated effects of selected inducers of endoplasmic reticulum stress, the unfolded protein response and apoptosis on P-Glycoprotein mediated drug resistance in MCF-7 breast carcinoma cells .” 2015. Thesis, University of the Western Cape. Accessed April 21, 2021. http://hdl.handle.net/11394/4459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pillay, Leeshan. “The integrated effects of selected inducers of endoplasmic reticulum stress, the unfolded protein response and apoptosis on P-Glycoprotein mediated drug resistance in MCF-7 breast carcinoma cells .” 2015. Web. 21 Apr 2021.

Vancouver:

Pillay L. The integrated effects of selected inducers of endoplasmic reticulum stress, the unfolded protein response and apoptosis on P-Glycoprotein mediated drug resistance in MCF-7 breast carcinoma cells . [Internet] [Thesis]. University of the Western Cape; 2015. [cited 2021 Apr 21]. Available from: http://hdl.handle.net/11394/4459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pillay L. The integrated effects of selected inducers of endoplasmic reticulum stress, the unfolded protein response and apoptosis on P-Glycoprotein mediated drug resistance in MCF-7 breast carcinoma cells . [Thesis]. University of the Western Cape; 2015. Available from: http://hdl.handle.net/11394/4459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

20. Dauer, Patricia. Endoplasmic Reticulum Stress-Mediated Signaling in Pancreatic Cancer.

Degree: PhD, Pharmacology, 2018, University of Minnesota

 Pancreatic ductal adenocarcinoma (PDAC) ranks among the poorest prognoses for cancer patients, with an estimated 5-year survival of just 8%. The stagnant survival rates are… (more)

Subjects/Keywords: Chemoresistance; Endoplasmic Reticulum Stress; GRP78; Pancreatic Cancer; SP1; Tumor microenvironment

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APA (6th Edition):

Dauer, P. (2018). Endoplasmic Reticulum Stress-Mediated Signaling in Pancreatic Cancer. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/198397

Chicago Manual of Style (16th Edition):

Dauer, Patricia. “Endoplasmic Reticulum Stress-Mediated Signaling in Pancreatic Cancer.” 2018. Doctoral Dissertation, University of Minnesota. Accessed April 21, 2021. http://hdl.handle.net/11299/198397.

MLA Handbook (7th Edition):

Dauer, Patricia. “Endoplasmic Reticulum Stress-Mediated Signaling in Pancreatic Cancer.” 2018. Web. 21 Apr 2021.

Vancouver:

Dauer P. Endoplasmic Reticulum Stress-Mediated Signaling in Pancreatic Cancer. [Internet] [Doctoral dissertation]. University of Minnesota; 2018. [cited 2021 Apr 21]. Available from: http://hdl.handle.net/11299/198397.

Council of Science Editors:

Dauer P. Endoplasmic Reticulum Stress-Mediated Signaling in Pancreatic Cancer. [Doctoral Dissertation]. University of Minnesota; 2018. Available from: http://hdl.handle.net/11299/198397


University of Southern California

21. Ashish Anshu, Fnu. Anti-cancer effects of novel glidobactin type proteasome inhibitors.

Degree: MS, Molecular Microbiology & Immunology, 2011, University of Southern California

 Proteasome inhibitors are widely used today as an important tool for anti-cancer treatment, which has led to a wide search of novel proteasome inhibitors. Proteasome… (more)

Subjects/Keywords: apoptosis; autophagy; endoplasmic reticulum stress; glidobactin A; hematological malignancy; proteasome

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APA (6th Edition):

Ashish Anshu, F. (2011). Anti-cancer effects of novel glidobactin type proteasome inhibitors. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/456326/rec/839

Chicago Manual of Style (16th Edition):

Ashish Anshu, Fnu. “Anti-cancer effects of novel glidobactin type proteasome inhibitors.” 2011. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/456326/rec/839.

MLA Handbook (7th Edition):

Ashish Anshu, Fnu. “Anti-cancer effects of novel glidobactin type proteasome inhibitors.” 2011. Web. 21 Apr 2021.

Vancouver:

Ashish Anshu F. Anti-cancer effects of novel glidobactin type proteasome inhibitors. [Internet] [Masters thesis]. University of Southern California; 2011. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/456326/rec/839.

Council of Science Editors:

Ashish Anshu F. Anti-cancer effects of novel glidobactin type proteasome inhibitors. [Masters Thesis]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/456326/rec/839

22. Lhomond, Stephanie. Impact fonctionnel de mutations somatiques dans le gène ERN1 (IRE1ΑLPHA) dans les glioblastomes : Impact of functional somatic mutations in the gene ERN1 (IRE1ALPHA) in glioblastomas.

Degree: Docteur es, Biologie Cellulaire et Physiopathologie, 2014, Bordeaux

Dans les cellules eucaryotes, des altérations du microenvironnement cellulaire ou desmutations des protéines de la voie de sécrétion induisent un stress du RE et activent… (more)

Subjects/Keywords: Réticulum endoplasmique; Stress; IRE1α; ERN1; Cancer; Glioblastome; Endoplasmic reticulum; Stress; IRE1α; ERN1; Cancer; Glioblastoma

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APA (6th Edition):

Lhomond, S. (2014). Impact fonctionnel de mutations somatiques dans le gène ERN1 (IRE1ΑLPHA) dans les glioblastomes : Impact of functional somatic mutations in the gene ERN1 (IRE1ALPHA) in glioblastomas. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2014BORD0038

Chicago Manual of Style (16th Edition):

Lhomond, Stephanie. “Impact fonctionnel de mutations somatiques dans le gène ERN1 (IRE1ΑLPHA) dans les glioblastomes : Impact of functional somatic mutations in the gene ERN1 (IRE1ALPHA) in glioblastomas.” 2014. Doctoral Dissertation, Bordeaux. Accessed April 21, 2021. http://www.theses.fr/2014BORD0038.

MLA Handbook (7th Edition):

Lhomond, Stephanie. “Impact fonctionnel de mutations somatiques dans le gène ERN1 (IRE1ΑLPHA) dans les glioblastomes : Impact of functional somatic mutations in the gene ERN1 (IRE1ALPHA) in glioblastomas.” 2014. Web. 21 Apr 2021.

Vancouver:

Lhomond S. Impact fonctionnel de mutations somatiques dans le gène ERN1 (IRE1ΑLPHA) dans les glioblastomes : Impact of functional somatic mutations in the gene ERN1 (IRE1ALPHA) in glioblastomas. [Internet] [Doctoral dissertation]. Bordeaux; 2014. [cited 2021 Apr 21]. Available from: http://www.theses.fr/2014BORD0038.

Council of Science Editors:

Lhomond S. Impact fonctionnel de mutations somatiques dans le gène ERN1 (IRE1ΑLPHA) dans les glioblastomes : Impact of functional somatic mutations in the gene ERN1 (IRE1ALPHA) in glioblastomas. [Doctoral Dissertation]. Bordeaux; 2014. Available from: http://www.theses.fr/2014BORD0038


RMIT University

23. Sun, R. The role of ER stress in lipogenesis and insulin resistance in response to over nutrition.

Degree: 2014, RMIT University

 Insulin resistance is one of the major defect of type 2 diabetes. Excess lipid and endoplasmic reticulum (ER) stress have been suggested to induce hepatic… (more)

Subjects/Keywords: Fields of Research; Hepatic insulin resistance; Endoplasmic reticulum stress; lipid metabolism; Fructose; Liver

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APA (6th Edition):

Sun, R. (2014). The role of ER stress in lipogenesis and insulin resistance in response to over nutrition. (Thesis). RMIT University. Retrieved from http://researchbank.rmit.edu.au/view/rmit:161524

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sun, R. “The role of ER stress in lipogenesis and insulin resistance in response to over nutrition.” 2014. Thesis, RMIT University. Accessed April 21, 2021. http://researchbank.rmit.edu.au/view/rmit:161524.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sun, R. “The role of ER stress in lipogenesis and insulin resistance in response to over nutrition.” 2014. Web. 21 Apr 2021.

Vancouver:

Sun R. The role of ER stress in lipogenesis and insulin resistance in response to over nutrition. [Internet] [Thesis]. RMIT University; 2014. [cited 2021 Apr 21]. Available from: http://researchbank.rmit.edu.au/view/rmit:161524.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sun R. The role of ER stress in lipogenesis and insulin resistance in response to over nutrition. [Thesis]. RMIT University; 2014. Available from: http://researchbank.rmit.edu.au/view/rmit:161524

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Utah

24. Moore, Kristin A. The mechanisms and function of regulated ire1-dependent decay during endoplasmic reticulum stress.

Degree: PhD, Biology, 2016, University of Utah

 The endoplasmic reticulum (ER) is a dynamic organelle that is responsible for the folding and quality control of proteins within the endomembrane system. Both physiological… (more)

Subjects/Keywords: Blos1; Endoplasmic reticulum (ER); ER stress; mRNA degradation; Regulated-Ire1 Dependent Decay; Unfolded Protein Response

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APA (6th Edition):

Moore, K. A. (2016). The mechanisms and function of regulated ire1-dependent decay during endoplasmic reticulum stress. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/4104/rec/2602

Chicago Manual of Style (16th Edition):

Moore, Kristin A. “The mechanisms and function of regulated ire1-dependent decay during endoplasmic reticulum stress.” 2016. Doctoral Dissertation, University of Utah. Accessed April 21, 2021. http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/4104/rec/2602.

MLA Handbook (7th Edition):

Moore, Kristin A. “The mechanisms and function of regulated ire1-dependent decay during endoplasmic reticulum stress.” 2016. Web. 21 Apr 2021.

Vancouver:

Moore KA. The mechanisms and function of regulated ire1-dependent decay during endoplasmic reticulum stress. [Internet] [Doctoral dissertation]. University of Utah; 2016. [cited 2021 Apr 21]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/4104/rec/2602.

Council of Science Editors:

Moore KA. The mechanisms and function of regulated ire1-dependent decay during endoplasmic reticulum stress. [Doctoral Dissertation]. University of Utah; 2016. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/4104/rec/2602


Cornell University

25. Krumm, Christopher Steven. Tales of Metabolic Regulation: Adiponectin in the Cow and Sel1L in the Mouse.

Degree: PhD, Animal Science, 2017, Cornell University

 The first part of this dissertation assessed factors regulating the insulin sensitizing hormone adiponectin in dairy cows. Plasma adiponectin is reduced during the transition from… (more)

Subjects/Keywords: Endocrinology; Biology; Adiponectin; endoplasmic reticulum stress; Metabolism; Sel1L; Transition Dairy Cow; Animal sciences

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APA (6th Edition):

Krumm, C. S. (2017). Tales of Metabolic Regulation: Adiponectin in the Cow and Sel1L in the Mouse. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/47862

Chicago Manual of Style (16th Edition):

Krumm, Christopher Steven. “Tales of Metabolic Regulation: Adiponectin in the Cow and Sel1L in the Mouse.” 2017. Doctoral Dissertation, Cornell University. Accessed April 21, 2021. http://hdl.handle.net/1813/47862.

MLA Handbook (7th Edition):

Krumm, Christopher Steven. “Tales of Metabolic Regulation: Adiponectin in the Cow and Sel1L in the Mouse.” 2017. Web. 21 Apr 2021.

Vancouver:

Krumm CS. Tales of Metabolic Regulation: Adiponectin in the Cow and Sel1L in the Mouse. [Internet] [Doctoral dissertation]. Cornell University; 2017. [cited 2021 Apr 21]. Available from: http://hdl.handle.net/1813/47862.

Council of Science Editors:

Krumm CS. Tales of Metabolic Regulation: Adiponectin in the Cow and Sel1L in the Mouse. [Doctoral Dissertation]. Cornell University; 2017. Available from: http://hdl.handle.net/1813/47862


Vanderbilt University

26. Leamy, Alexandra Kathlene. Role of lipid metabolic pathways in the progression of hepatic lipotoxicity.

Degree: PhD, Chemical Engineering, 2015, Vanderbilt University

 The steady rise in Western obesity rates has been closely linked to significant increases in a multitude of accompanying health problems including obesity, type II… (more)

Subjects/Keywords: phospholipids; triglyceride synthesis; endoplasmic reticulum stress; saturated fatty acids; lipotoxicity; lipid metabolism

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APA (6th Edition):

Leamy, A. K. (2015). Role of lipid metabolic pathways in the progression of hepatic lipotoxicity. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11066

Chicago Manual of Style (16th Edition):

Leamy, Alexandra Kathlene. “Role of lipid metabolic pathways in the progression of hepatic lipotoxicity.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed April 21, 2021. http://hdl.handle.net/1803/11066.

MLA Handbook (7th Edition):

Leamy, Alexandra Kathlene. “Role of lipid metabolic pathways in the progression of hepatic lipotoxicity.” 2015. Web. 21 Apr 2021.

Vancouver:

Leamy AK. Role of lipid metabolic pathways in the progression of hepatic lipotoxicity. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Apr 21]. Available from: http://hdl.handle.net/1803/11066.

Council of Science Editors:

Leamy AK. Role of lipid metabolic pathways in the progression of hepatic lipotoxicity. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/11066


North Carolina State University

27. Kirst, Mariana E. Identification and characterization of maize Derlins in response to endoplasmic reticulum stress.

Degree: PhD, Botany, 2007, North Carolina State University

Subjects/Keywords: Derlin; ERAD; ER stress; endoplasmic reticulum

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APA (6th Edition):

Kirst, M. E. (2007). Identification and characterization of maize Derlins in response to endoplasmic reticulum stress. (Doctoral Dissertation). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/4954

Chicago Manual of Style (16th Edition):

Kirst, Mariana E. “Identification and characterization of maize Derlins in response to endoplasmic reticulum stress.” 2007. Doctoral Dissertation, North Carolina State University. Accessed April 21, 2021. http://www.lib.ncsu.edu/resolver/1840.16/4954.

MLA Handbook (7th Edition):

Kirst, Mariana E. “Identification and characterization of maize Derlins in response to endoplasmic reticulum stress.” 2007. Web. 21 Apr 2021.

Vancouver:

Kirst ME. Identification and characterization of maize Derlins in response to endoplasmic reticulum stress. [Internet] [Doctoral dissertation]. North Carolina State University; 2007. [cited 2021 Apr 21]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/4954.

Council of Science Editors:

Kirst ME. Identification and characterization of maize Derlins in response to endoplasmic reticulum stress. [Doctoral Dissertation]. North Carolina State University; 2007. Available from: http://www.lib.ncsu.edu/resolver/1840.16/4954

28. Aydoğdu, Gülizar. Glutamin taşınması ve metabolizmasının insülin direnci ve endoplazmik retikulum stresi üzerindeki rolü: The role of glutamine transport and metabolism in the insulin resistance and endoplasmic reticulum stress.

Degree: Fen Fakültesi, 2019, University of Ankara

 Obezite, son zamanlardaki önemli sağlık problemlemlerinden biridir. Obezitenin tip-2 diyabet riskini arttırdığı ve tip-2 diyabetteki insülin direncinin ortaya çıkışında da endoplazmik retikulum (ER) stresinin katkısının… (more)

Subjects/Keywords: Endoplazmik retikulum stresi; İnsülin direnci; Glutamin; Endoplasmic reticulum stress; İnsulin resistance; Glutamine

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APA (6th Edition):

Aydoğdu, G. (2019). Glutamin taşınması ve metabolizmasının insülin direnci ve endoplazmik retikulum stresi üzerindeki rolü: The role of glutamine transport and metabolism in the insulin resistance and endoplasmic reticulum stress. (Doctoral Dissertation). University of Ankara. Retrieved from http://hdl.handle.net/20.500.12575/70224

Chicago Manual of Style (16th Edition):

Aydoğdu, Gülizar. “Glutamin taşınması ve metabolizmasının insülin direnci ve endoplazmik retikulum stresi üzerindeki rolü: The role of glutamine transport and metabolism in the insulin resistance and endoplasmic reticulum stress.” 2019. Doctoral Dissertation, University of Ankara. Accessed April 21, 2021. http://hdl.handle.net/20.500.12575/70224.

MLA Handbook (7th Edition):

Aydoğdu, Gülizar. “Glutamin taşınması ve metabolizmasının insülin direnci ve endoplazmik retikulum stresi üzerindeki rolü: The role of glutamine transport and metabolism in the insulin resistance and endoplasmic reticulum stress.” 2019. Web. 21 Apr 2021.

Vancouver:

Aydoğdu G. Glutamin taşınması ve metabolizmasının insülin direnci ve endoplazmik retikulum stresi üzerindeki rolü: The role of glutamine transport and metabolism in the insulin resistance and endoplasmic reticulum stress. [Internet] [Doctoral dissertation]. University of Ankara; 2019. [cited 2021 Apr 21]. Available from: http://hdl.handle.net/20.500.12575/70224.

Council of Science Editors:

Aydoğdu G. Glutamin taşınması ve metabolizmasının insülin direnci ve endoplazmik retikulum stresi üzerindeki rolü: The role of glutamine transport and metabolism in the insulin resistance and endoplasmic reticulum stress. [Doctoral Dissertation]. University of Ankara; 2019. Available from: http://hdl.handle.net/20.500.12575/70224

29. 정, 익락. The effect of iron metabolism on palmitate-induced INS-1 cell death.

Degree: 2015, Ajou University

Ⅰ. INTRODUCTION 1 Ⅱ. MATERIALS AND METHODS 24 A. MATERIALS 24 1. Reagents 24 B. METHODS 25 1. Cell Culture 25 2. Preparation of palmitate… (more)

Subjects/Keywords: Apoptosis; Endoplasmic reticulum(ER) stress; Lipotoxicity; Iron; INS-1 Cell; Palmitate; Transferrin receptor; Soduim fluorocitrate

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

정, . (2015). The effect of iron metabolism on palmitate-induced INS-1 cell death. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/11837 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020143

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

정, 익락. “The effect of iron metabolism on palmitate-induced INS-1 cell death.” 2015. Thesis, Ajou University. Accessed April 21, 2021. http://repository.ajou.ac.kr/handle/201003/11837 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020143.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

정, 익락. “The effect of iron metabolism on palmitate-induced INS-1 cell death.” 2015. Web. 21 Apr 2021.

Vancouver:

정 . The effect of iron metabolism on palmitate-induced INS-1 cell death. [Internet] [Thesis]. Ajou University; 2015. [cited 2021 Apr 21]. Available from: http://repository.ajou.ac.kr/handle/201003/11837 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020143.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

정 . The effect of iron metabolism on palmitate-induced INS-1 cell death. [Thesis]. Ajou University; 2015. Available from: http://repository.ajou.ac.kr/handle/201003/11837 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000020143

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

30. Motomochi, Amanda. Cell stress markers during development of hemolytic uremic syndrome and acute kidney injury.

Degree: MS, Medical Sciences, 2014, Boston University

 Enterohemorrhagic E. coli (EHEC) infections are a leading cause of foodborne illness in the United States. Shiga-like toxins are produced that can cause hemorrhagic colitis… (more)

Subjects/Keywords: Pathology; Enterohemorrhagic Escherichia coli; Endoplasmic reticulum stress; Hemolytic uremic syndrome; Shiga-like toxins

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Motomochi, A. (2014). Cell stress markers during development of hemolytic uremic syndrome and acute kidney injury. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/14397

Chicago Manual of Style (16th Edition):

Motomochi, Amanda. “Cell stress markers during development of hemolytic uremic syndrome and acute kidney injury.” 2014. Masters Thesis, Boston University. Accessed April 21, 2021. http://hdl.handle.net/2144/14397.

MLA Handbook (7th Edition):

Motomochi, Amanda. “Cell stress markers during development of hemolytic uremic syndrome and acute kidney injury.” 2014. Web. 21 Apr 2021.

Vancouver:

Motomochi A. Cell stress markers during development of hemolytic uremic syndrome and acute kidney injury. [Internet] [Masters thesis]. Boston University; 2014. [cited 2021 Apr 21]. Available from: http://hdl.handle.net/2144/14397.

Council of Science Editors:

Motomochi A. Cell stress markers during development of hemolytic uremic syndrome and acute kidney injury. [Masters Thesis]. Boston University; 2014. Available from: http://hdl.handle.net/2144/14397

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