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You searched for subject:(ESCC). Showing records 1 – 9 of 9 total matches.

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1. 仲里, 秀次. Early-Stage Induction of SWI/SNF Mutations during Esophageal Squamous Cell Carcinogenesis : SIW/SNF遺伝子異常の食道扁平上皮がん発がんの早期における誘発.

Degree: 博士(医学), 2017, University of the Ryukyus / 琉球大学

 The SWI/SNF chromatin remodeling complex is frequently inactivated by somaticmutations of its various components in various types of cancers, and also by aberrant DNA methylation.… (more)

Subjects/Keywords: Epigenetics; SWI/SNF; mutation; ESCC

Page 1 Page 2 Page 3 Page 4 Page 5

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

仲里, . (2017). Early-Stage Induction of SWI/SNF Mutations during Esophageal Squamous Cell Carcinogenesis : SIW/SNF遺伝子異常の食道扁平上皮がん発がんの早期における誘発. (Thesis). University of the Ryukyus / 琉球大学. Retrieved from http://hdl.handle.net/20.500.12000/36588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

仲里, 秀次. “Early-Stage Induction of SWI/SNF Mutations during Esophageal Squamous Cell Carcinogenesis : SIW/SNF遺伝子異常の食道扁平上皮がん発がんの早期における誘発.” 2017. Thesis, University of the Ryukyus / 琉球大学. Accessed April 18, 2021. http://hdl.handle.net/20.500.12000/36588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

仲里, 秀次. “Early-Stage Induction of SWI/SNF Mutations during Esophageal Squamous Cell Carcinogenesis : SIW/SNF遺伝子異常の食道扁平上皮がん発がんの早期における誘発.” 2017. Web. 18 Apr 2021.

Vancouver:

仲里 . Early-Stage Induction of SWI/SNF Mutations during Esophageal Squamous Cell Carcinogenesis : SIW/SNF遺伝子異常の食道扁平上皮がん発がんの早期における誘発. [Internet] [Thesis]. University of the Ryukyus / 琉球大学; 2017. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/20.500.12000/36588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

仲里 . Early-Stage Induction of SWI/SNF Mutations during Esophageal Squamous Cell Carcinogenesis : SIW/SNF遺伝子異常の食道扁平上皮がん発がんの早期における誘発. [Thesis]. University of the Ryukyus / 琉球大学; 2017. Available from: http://hdl.handle.net/20.500.12000/36588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

2. Mohammed, Muddasir. Influences of porphyromonas gingivalis on the viability of esophageal cancer cells.

Degree: MS, 2018, University of Louisville

  A recent study demonstrated an association between the Porphyromonas gingivalis (Pg)infection and the progression of ESCC (Gao et al., 2016). However, how Pg infection… (more)

Subjects/Keywords: porphyromonas gingivalis; ESCC; esophageal cancer; viability; proliferation; apoptosis; Oral Biology and Oral Pathology

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APA (6th Edition):

Mohammed, M. (2018). Influences of porphyromonas gingivalis on the viability of esophageal cancer cells. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/3101 ; https://ir.library.louisville.edu/etd/3101

Chicago Manual of Style (16th Edition):

Mohammed, Muddasir. “Influences of porphyromonas gingivalis on the viability of esophageal cancer cells.” 2018. Masters Thesis, University of Louisville. Accessed April 18, 2021. 10.18297/etd/3101 ; https://ir.library.louisville.edu/etd/3101.

MLA Handbook (7th Edition):

Mohammed, Muddasir. “Influences of porphyromonas gingivalis on the viability of esophageal cancer cells.” 2018. Web. 18 Apr 2021.

Vancouver:

Mohammed M. Influences of porphyromonas gingivalis on the viability of esophageal cancer cells. [Internet] [Masters thesis]. University of Louisville; 2018. [cited 2021 Apr 18]. Available from: 10.18297/etd/3101 ; https://ir.library.louisville.edu/etd/3101.

Council of Science Editors:

Mohammed M. Influences of porphyromonas gingivalis on the viability of esophageal cancer cells. [Masters Thesis]. University of Louisville; 2018. Available from: 10.18297/etd/3101 ; https://ir.library.louisville.edu/etd/3101


University of California – San Francisco

3. Melton, Collin Alfred. Opposing microRNAs Regulate Mouse Embryonic Stem Cell Self-Renewal.

Degree: Biomedical Sciences, 2010, University of California – San Francisco

 When an embryonic stem cell (ESC) differentiates, it must both silence the ESC self-renewal program as well as activate new tissue-specific programs. In the absence… (more)

Subjects/Keywords: Biology, General; differentiation; embryonic stem cell; ESCC; let-7; microRNA; self-renewal

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APA (6th Edition):

Melton, C. A. (2010). Opposing microRNAs Regulate Mouse Embryonic Stem Cell Self-Renewal. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/9g6288t5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Melton, Collin Alfred. “Opposing microRNAs Regulate Mouse Embryonic Stem Cell Self-Renewal.” 2010. Thesis, University of California – San Francisco. Accessed April 18, 2021. http://www.escholarship.org/uc/item/9g6288t5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Melton, Collin Alfred. “Opposing microRNAs Regulate Mouse Embryonic Stem Cell Self-Renewal.” 2010. Web. 18 Apr 2021.

Vancouver:

Melton CA. Opposing microRNAs Regulate Mouse Embryonic Stem Cell Self-Renewal. [Internet] [Thesis]. University of California – San Francisco; 2010. [cited 2021 Apr 18]. Available from: http://www.escholarship.org/uc/item/9g6288t5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Melton CA. Opposing microRNAs Regulate Mouse Embryonic Stem Cell Self-Renewal. [Thesis]. University of California – San Francisco; 2010. Available from: http://www.escholarship.org/uc/item/9g6288t5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

4. Bolewski, Bianca. Expression and function of the orexin receptor 1 in human gastrointestinal tumors.

Degree: 2012, Freie Universität Berlin

 Early diagnosis and therapy are essential for improving the survival rate of cancer patients. One innovative strategy is demonstrated by targeted tumordiagnosis and -therapy. Here… (more)

Subjects/Keywords: Targeted tumordiagnosis and -therapy; GPCRs; in vivo Imaging; ESCC; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie::572 Biochemie

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APA (6th Edition):

Bolewski, B. (2012). Expression and function of the orexin receptor 1 in human gastrointestinal tumors. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-4662

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bolewski, Bianca. “Expression and function of the orexin receptor 1 in human gastrointestinal tumors.” 2012. Thesis, Freie Universität Berlin. Accessed April 18, 2021. http://dx.doi.org/10.17169/refubium-4662.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bolewski, Bianca. “Expression and function of the orexin receptor 1 in human gastrointestinal tumors.” 2012. Web. 18 Apr 2021.

Vancouver:

Bolewski B. Expression and function of the orexin receptor 1 in human gastrointestinal tumors. [Internet] [Thesis]. Freie Universität Berlin; 2012. [cited 2021 Apr 18]. Available from: http://dx.doi.org/10.17169/refubium-4662.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bolewski B. Expression and function of the orexin receptor 1 in human gastrointestinal tumors. [Thesis]. Freie Universität Berlin; 2012. Available from: http://dx.doi.org/10.17169/refubium-4662

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. LIM KEE SIANG. IMPLICATION OF HIGHLY CYTOTOXIC NATURAL KILLER CELLS FOR ESOPHAGEAL CANCER TREATMENT.

Degree: 2016, National University of Singapore

Subjects/Keywords: NK cell immunotherapy; ESCC; EMT; K562-mb15-41BBL

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APA (6th Edition):

SIANG, L. K. (2016). IMPLICATION OF HIGHLY CYTOTOXIC NATURAL KILLER CELLS FOR ESOPHAGEAL CANCER TREATMENT. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/135257

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

SIANG, LIM KEE. “IMPLICATION OF HIGHLY CYTOTOXIC NATURAL KILLER CELLS FOR ESOPHAGEAL CANCER TREATMENT.” 2016. Thesis, National University of Singapore. Accessed April 18, 2021. http://scholarbank.nus.edu.sg/handle/10635/135257.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

SIANG, LIM KEE. “IMPLICATION OF HIGHLY CYTOTOXIC NATURAL KILLER CELLS FOR ESOPHAGEAL CANCER TREATMENT.” 2016. Web. 18 Apr 2021.

Vancouver:

SIANG LK. IMPLICATION OF HIGHLY CYTOTOXIC NATURAL KILLER CELLS FOR ESOPHAGEAL CANCER TREATMENT. [Internet] [Thesis]. National University of Singapore; 2016. [cited 2021 Apr 18]. Available from: http://scholarbank.nus.edu.sg/handle/10635/135257.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

SIANG LK. IMPLICATION OF HIGHLY CYTOTOXIC NATURAL KILLER CELLS FOR ESOPHAGEAL CANCER TREATMENT. [Thesis]. National University of Singapore; 2016. Available from: http://scholarbank.nus.edu.sg/handle/10635/135257

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Bowling Green State University

6. Pendleton, Emily Grace. The Effect of Curcumin and Tetrahydrocurcumin in Combination with 5-fluorouracil on Esophageal Cancer Cell Lines.

Degree: MS, Biological Sciences, 2015, Bowling Green State University

 Despite our constant efforts to improve upon our current treatments, esophageal cancer does not respond well to traditional therapy. In an effort to better treat… (more)

Subjects/Keywords: Oncology; Biomedical Research; Biology; esophageal squamous cell carcinoma; ESCC; curcumin; tetrahydrocurcumin; 5-fluorouracil; 5-FU; esophageal cancer

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APA (6th Edition):

Pendleton, E. G. (2015). The Effect of Curcumin and Tetrahydrocurcumin in Combination with 5-fluorouracil on Esophageal Cancer Cell Lines. (Masters Thesis). Bowling Green State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1431601435

Chicago Manual of Style (16th Edition):

Pendleton, Emily Grace. “The Effect of Curcumin and Tetrahydrocurcumin in Combination with 5-fluorouracil on Esophageal Cancer Cell Lines.” 2015. Masters Thesis, Bowling Green State University. Accessed April 18, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1431601435.

MLA Handbook (7th Edition):

Pendleton, Emily Grace. “The Effect of Curcumin and Tetrahydrocurcumin in Combination with 5-fluorouracil on Esophageal Cancer Cell Lines.” 2015. Web. 18 Apr 2021.

Vancouver:

Pendleton EG. The Effect of Curcumin and Tetrahydrocurcumin in Combination with 5-fluorouracil on Esophageal Cancer Cell Lines. [Internet] [Masters thesis]. Bowling Green State University; 2015. [cited 2021 Apr 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1431601435.

Council of Science Editors:

Pendleton EG. The Effect of Curcumin and Tetrahydrocurcumin in Combination with 5-fluorouracil on Esophageal Cancer Cell Lines. [Masters Thesis]. Bowling Green State University; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1431601435

7. Iserhard, Ricardo. Avaliação dos níveis de autofagia e morfometria nuclear no carcinoma epidermoide de esôfago.

Degree: 2018, Brazil

O câncer de esôfago é a sexta causa de morte relacionada a neoplasias malignas no mundo. A taxa de sobrevida em 5 anos é inferior… (more)

Subjects/Keywords: Autofagia; Fatores de risco; Prognóstico; Carcinogenese; Carcinoma de células escamosas; Esophageal Squamous Cell Carcinoma (ESCC); Prognostic markers; Nuclear morphometry; Autophagy

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APA (6th Edition):

Iserhard, R. (2018). Avaliação dos níveis de autofagia e morfometria nuclear no carcinoma epidermoide de esôfago. (Masters Thesis). Brazil. Retrieved from http://hdl.handle.net/10183/188943

Chicago Manual of Style (16th Edition):

Iserhard, Ricardo. “Avaliação dos níveis de autofagia e morfometria nuclear no carcinoma epidermoide de esôfago.” 2018. Masters Thesis, Brazil. Accessed April 18, 2021. http://hdl.handle.net/10183/188943.

MLA Handbook (7th Edition):

Iserhard, Ricardo. “Avaliação dos níveis de autofagia e morfometria nuclear no carcinoma epidermoide de esôfago.” 2018. Web. 18 Apr 2021.

Vancouver:

Iserhard R. Avaliação dos níveis de autofagia e morfometria nuclear no carcinoma epidermoide de esôfago. [Internet] [Masters thesis]. Brazil; 2018. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/10183/188943.

Council of Science Editors:

Iserhard R. Avaliação dos níveis de autofagia e morfometria nuclear no carcinoma epidermoide de esôfago. [Masters Thesis]. Brazil; 2018. Available from: http://hdl.handle.net/10183/188943


Freie Universität Berlin

8. Ma, Chenming. Funktionsanalyse des Gens WDR66.

Degree: 2014, Freie Universität Berlin

 Karzinome der Speiseröhre werden schon lange als besonders gefährlich für den Patienten und ihre Behandlung wird weiterhin kontrovers diskutiert. Aufgrund der späten Vorstellung von Patienten… (more)

Subjects/Keywords: WDR66; novel marker; microarray-based gene expression analysis; siRNA; RT-PCR; laser capture microdissection; ESCC; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Ma, C. (2014). Funktionsanalyse des Gens WDR66. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-4857

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ma, Chenming. “Funktionsanalyse des Gens WDR66.” 2014. Thesis, Freie Universität Berlin. Accessed April 18, 2021. http://dx.doi.org/10.17169/refubium-4857.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ma, Chenming. “Funktionsanalyse des Gens WDR66.” 2014. Web. 18 Apr 2021.

Vancouver:

Ma C. Funktionsanalyse des Gens WDR66. [Internet] [Thesis]. Freie Universität Berlin; 2014. [cited 2021 Apr 18]. Available from: http://dx.doi.org/10.17169/refubium-4857.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ma C. Funktionsanalyse des Gens WDR66. [Thesis]. Freie Universität Berlin; 2014. Available from: http://dx.doi.org/10.17169/refubium-4857

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vienna

9. Ehrenhöfer-Wölfer, Katharina. Identification of SMARCA4 as a synthetic lethal dependency in SMARCA2low esophageal squamous cell carcinoma cell lines.

Degree: 2019, University of Vienna

Das Plattenepithelzellkarzinom des Oesophagus (ESCC) ist die dominant auftretende Form des Oesophaguskarzinoms, welches zu den sechst häufigsten durch Krebs hervorgerufenen Todesursachen weltweit zählt. Die Überlebensrate… (more)

Subjects/Keywords: 42.13 Molekularbiologie; Plattenepithelkarzinom des Oesophagus / SMARCA4 / SMARCA2 niedrig exprimiert / CRISPR-Cas9 Domänen-basierte Screens / Synthetische Letalität / PROTAC / Bromodomänen Austausch; ESCC / SMARCA4 / SMARCA2low / CRISPR-Cas9 domain-based screens / synthetic lethality / PROTAC / bromodomain swap

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APA (6th Edition):

Ehrenhöfer-Wölfer, K. (2019). Identification of SMARCA4 as a synthetic lethal dependency in SMARCA2low esophageal squamous cell carcinoma cell lines. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/57580/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ehrenhöfer-Wölfer, Katharina. “Identification of SMARCA4 as a synthetic lethal dependency in SMARCA2low esophageal squamous cell carcinoma cell lines.” 2019. Thesis, University of Vienna. Accessed April 18, 2021. http://othes.univie.ac.at/57580/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ehrenhöfer-Wölfer, Katharina. “Identification of SMARCA4 as a synthetic lethal dependency in SMARCA2low esophageal squamous cell carcinoma cell lines.” 2019. Web. 18 Apr 2021.

Vancouver:

Ehrenhöfer-Wölfer K. Identification of SMARCA4 as a synthetic lethal dependency in SMARCA2low esophageal squamous cell carcinoma cell lines. [Internet] [Thesis]. University of Vienna; 2019. [cited 2021 Apr 18]. Available from: http://othes.univie.ac.at/57580/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ehrenhöfer-Wölfer K. Identification of SMARCA4 as a synthetic lethal dependency in SMARCA2low esophageal squamous cell carcinoma cell lines. [Thesis]. University of Vienna; 2019. Available from: http://othes.univie.ac.at/57580/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.