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You searched for subject:(EMT). Showing records 1 – 30 of 392 total matches.

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University of Florida

1. Rosati, Lindsey Pikos. The Role of Epithelial Mesenchymal Transition in Periodontal Disease.

Degree: MS, Dental Sciences - Dentistry, 2017, University of Florida

Subjects/Keywords: emt

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APA (6th Edition):

Rosati, L. P. (2017). The Role of Epithelial Mesenchymal Transition in Periodontal Disease. (Masters Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/UFE0051062

Chicago Manual of Style (16th Edition):

Rosati, Lindsey Pikos. “The Role of Epithelial Mesenchymal Transition in Periodontal Disease.” 2017. Masters Thesis, University of Florida. Accessed April 14, 2021. https://ufdc.ufl.edu/UFE0051062.

MLA Handbook (7th Edition):

Rosati, Lindsey Pikos. “The Role of Epithelial Mesenchymal Transition in Periodontal Disease.” 2017. Web. 14 Apr 2021.

Vancouver:

Rosati LP. The Role of Epithelial Mesenchymal Transition in Periodontal Disease. [Internet] [Masters thesis]. University of Florida; 2017. [cited 2021 Apr 14]. Available from: https://ufdc.ufl.edu/UFE0051062.

Council of Science Editors:

Rosati LP. The Role of Epithelial Mesenchymal Transition in Periodontal Disease. [Masters Thesis]. University of Florida; 2017. Available from: https://ufdc.ufl.edu/UFE0051062

2. Lortal Canguilhem, Barbara. «Caractérisation de sept lignées cellulaires humaines de cancer de vessie pour les principaux marqueurs de la transition épithélio-mésenchymateuse, Twist1 et E-cadhérine, et pour une nouvelle drogue, le saracatinib. » : «Caracterisation of 7 human cells lines of bladder cancer for the main markers of EMT, Twist1 and E-cadherin, and for a new drug, Saracatinib».

Degree: Docteur es, Sciences, technologie, santé. Biologie cellulaire et physiopathologie, 2012, Université de Bordeaux Segalen

Le cancer de la vessie représente le 4ième cancer en terme d’incidence. La mortalité de ce cancer est principalement due à la formation de métastases… (more)

Subjects/Keywords: EMT; Saracatinib; Vessie; EMT; Saracatinib; Bladder

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APA (6th Edition):

Lortal Canguilhem, B. (2012). «Caractérisation de sept lignées cellulaires humaines de cancer de vessie pour les principaux marqueurs de la transition épithélio-mésenchymateuse, Twist1 et E-cadhérine, et pour une nouvelle drogue, le saracatinib. » : «Caracterisation of 7 human cells lines of bladder cancer for the main markers of EMT, Twist1 and E-cadherin, and for a new drug, Saracatinib». (Doctoral Dissertation). Université de Bordeaux Segalen. Retrieved from http://www.theses.fr/2012BOR22026

Chicago Manual of Style (16th Edition):

Lortal Canguilhem, Barbara. “«Caractérisation de sept lignées cellulaires humaines de cancer de vessie pour les principaux marqueurs de la transition épithélio-mésenchymateuse, Twist1 et E-cadhérine, et pour une nouvelle drogue, le saracatinib. » : «Caracterisation of 7 human cells lines of bladder cancer for the main markers of EMT, Twist1 and E-cadherin, and for a new drug, Saracatinib».” 2012. Doctoral Dissertation, Université de Bordeaux Segalen. Accessed April 14, 2021. http://www.theses.fr/2012BOR22026.

MLA Handbook (7th Edition):

Lortal Canguilhem, Barbara. “«Caractérisation de sept lignées cellulaires humaines de cancer de vessie pour les principaux marqueurs de la transition épithélio-mésenchymateuse, Twist1 et E-cadhérine, et pour une nouvelle drogue, le saracatinib. » : «Caracterisation of 7 human cells lines of bladder cancer for the main markers of EMT, Twist1 and E-cadherin, and for a new drug, Saracatinib».” 2012. Web. 14 Apr 2021.

Vancouver:

Lortal Canguilhem B. «Caractérisation de sept lignées cellulaires humaines de cancer de vessie pour les principaux marqueurs de la transition épithélio-mésenchymateuse, Twist1 et E-cadhérine, et pour une nouvelle drogue, le saracatinib. » : «Caracterisation of 7 human cells lines of bladder cancer for the main markers of EMT, Twist1 and E-cadherin, and for a new drug, Saracatinib». [Internet] [Doctoral dissertation]. Université de Bordeaux Segalen; 2012. [cited 2021 Apr 14]. Available from: http://www.theses.fr/2012BOR22026.

Council of Science Editors:

Lortal Canguilhem B. «Caractérisation de sept lignées cellulaires humaines de cancer de vessie pour les principaux marqueurs de la transition épithélio-mésenchymateuse, Twist1 et E-cadhérine, et pour une nouvelle drogue, le saracatinib. » : «Caracterisation of 7 human cells lines of bladder cancer for the main markers of EMT, Twist1 and E-cadherin, and for a new drug, Saracatinib». [Doctoral Dissertation]. Université de Bordeaux Segalen; 2012. Available from: http://www.theses.fr/2012BOR22026


Universitat de Valencia

3. Pulido Endrino, Inés. Análisis genómico funcional de la resistencia a las terapias anti-EGFR asociada al fenotipo mesenquimal en el cáncer pulmonar.

Degree: 2017, Universitat de Valencia

 Lung cancer is the leading cause of cancer death in western countries. It is highly resistant to the conventional therapy, metastatic and with a 5-years… (more)

Subjects/Keywords: CPNM; EMT; ITQ

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APA (6th Edition):

Pulido Endrino, I. (2017). Análisis genómico funcional de la resistencia a las terapias anti-EGFR asociada al fenotipo mesenquimal en el cáncer pulmonar. (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/60462

Chicago Manual of Style (16th Edition):

Pulido Endrino, Inés. “Análisis genómico funcional de la resistencia a las terapias anti-EGFR asociada al fenotipo mesenquimal en el cáncer pulmonar. ” 2017. Doctoral Dissertation, Universitat de Valencia. Accessed April 14, 2021. http://hdl.handle.net/10550/60462.

MLA Handbook (7th Edition):

Pulido Endrino, Inés. “Análisis genómico funcional de la resistencia a las terapias anti-EGFR asociada al fenotipo mesenquimal en el cáncer pulmonar. ” 2017. Web. 14 Apr 2021.

Vancouver:

Pulido Endrino I. Análisis genómico funcional de la resistencia a las terapias anti-EGFR asociada al fenotipo mesenquimal en el cáncer pulmonar. [Internet] [Doctoral dissertation]. Universitat de Valencia; 2017. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10550/60462.

Council of Science Editors:

Pulido Endrino I. Análisis genómico funcional de la resistencia a las terapias anti-EGFR asociada al fenotipo mesenquimal en el cáncer pulmonar. [Doctoral Dissertation]. Universitat de Valencia; 2017. Available from: http://hdl.handle.net/10550/60462


Universiteit Utrecht

4. Wiebrands, K. A role of epithelial-mesenchymal transitions in carcinogenic progression.

Degree: 2010, Universiteit Utrecht

 The epithelial-mesenchymal transition plays an important role in several developmental processes, tissue repair, but is also associated with fibrosis and cancer. During tumorigenic progression, EMT(more)

Subjects/Keywords: epithelial-mesenchymal transition; EMT; metastasis

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APA (6th Edition):

Wiebrands, K. (2010). A role of epithelial-mesenchymal transitions in carcinogenic progression. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/190315

Chicago Manual of Style (16th Edition):

Wiebrands, K. “A role of epithelial-mesenchymal transitions in carcinogenic progression.” 2010. Masters Thesis, Universiteit Utrecht. Accessed April 14, 2021. http://dspace.library.uu.nl:8080/handle/1874/190315.

MLA Handbook (7th Edition):

Wiebrands, K. “A role of epithelial-mesenchymal transitions in carcinogenic progression.” 2010. Web. 14 Apr 2021.

Vancouver:

Wiebrands K. A role of epithelial-mesenchymal transitions in carcinogenic progression. [Internet] [Masters thesis]. Universiteit Utrecht; 2010. [cited 2021 Apr 14]. Available from: http://dspace.library.uu.nl:8080/handle/1874/190315.

Council of Science Editors:

Wiebrands K. A role of epithelial-mesenchymal transitions in carcinogenic progression. [Masters Thesis]. Universiteit Utrecht; 2010. Available from: http://dspace.library.uu.nl:8080/handle/1874/190315


Texas A&M University

5. Bai, Yan. BMPs Regulate the Oft Development via miRNAs.

Degree: PhD, Medical Sciences, 2014, Texas A&M University

 Congenital heart diseases (CHD) are the leading cause of infant mortality and morbidity. Defects of the outflow tract (OFT) make up a large percentage of… (more)

Subjects/Keywords: BMP; OFT; EMT; miRNA; aneurysm

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APA (6th Edition):

Bai, Y. (2014). BMPs Regulate the Oft Development via miRNAs. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/153514

Chicago Manual of Style (16th Edition):

Bai, Yan. “BMPs Regulate the Oft Development via miRNAs.” 2014. Doctoral Dissertation, Texas A&M University. Accessed April 14, 2021. http://hdl.handle.net/1969.1/153514.

MLA Handbook (7th Edition):

Bai, Yan. “BMPs Regulate the Oft Development via miRNAs.” 2014. Web. 14 Apr 2021.

Vancouver:

Bai Y. BMPs Regulate the Oft Development via miRNAs. [Internet] [Doctoral dissertation]. Texas A&M University; 2014. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1969.1/153514.

Council of Science Editors:

Bai Y. BMPs Regulate the Oft Development via miRNAs. [Doctoral Dissertation]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/153514


University of Tasmania

6. Nowrin, K. Drivers of epithelial-mesenchymal transition in chronic obstructive pulmonary disease.

Degree: 2015, University of Tasmania

 COPD is a chronic disease with a complex condition that involves a frequent mix of airway and lung parenchymal damage. The Global Initiative for Chronic… (more)

Subjects/Keywords: COPD; smoking; EMT; pSmad2/3

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APA (6th Edition):

Nowrin, K. (2015). Drivers of epithelial-mesenchymal transition in chronic obstructive pulmonary disease. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/23228/1/Norwin_whole_thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nowrin, K. “Drivers of epithelial-mesenchymal transition in chronic obstructive pulmonary disease.” 2015. Thesis, University of Tasmania. Accessed April 14, 2021. https://eprints.utas.edu.au/23228/1/Norwin_whole_thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nowrin, K. “Drivers of epithelial-mesenchymal transition in chronic obstructive pulmonary disease.” 2015. Web. 14 Apr 2021.

Vancouver:

Nowrin K. Drivers of epithelial-mesenchymal transition in chronic obstructive pulmonary disease. [Internet] [Thesis]. University of Tasmania; 2015. [cited 2021 Apr 14]. Available from: https://eprints.utas.edu.au/23228/1/Norwin_whole_thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nowrin K. Drivers of epithelial-mesenchymal transition in chronic obstructive pulmonary disease. [Thesis]. University of Tasmania; 2015. Available from: https://eprints.utas.edu.au/23228/1/Norwin_whole_thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

7. Bommi, Prashant V. DDB2 Regulates EMT in Oral SCC's and Transcriptionally Regulates HIF1A.

Degree: 2017, University of Illinois – Chicago

 An underlying hallmark of cancers is their ability to survive in hostile microenvironment marked by unrestrained proliferation acquired through genomic instability, which is often associated… (more)

Subjects/Keywords: DNA Damage; Hypoxia; EMT

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APA (6th Edition):

Bommi, P. V. (2017). DDB2 Regulates EMT in Oral SCC's and Transcriptionally Regulates HIF1A. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/22007

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bommi, Prashant V. “DDB2 Regulates EMT in Oral SCC's and Transcriptionally Regulates HIF1A.” 2017. Thesis, University of Illinois – Chicago. Accessed April 14, 2021. http://hdl.handle.net/10027/22007.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bommi, Prashant V. “DDB2 Regulates EMT in Oral SCC's and Transcriptionally Regulates HIF1A.” 2017. Web. 14 Apr 2021.

Vancouver:

Bommi PV. DDB2 Regulates EMT in Oral SCC's and Transcriptionally Regulates HIF1A. [Internet] [Thesis]. University of Illinois – Chicago; 2017. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10027/22007.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bommi PV. DDB2 Regulates EMT in Oral SCC's and Transcriptionally Regulates HIF1A. [Thesis]. University of Illinois – Chicago; 2017. Available from: http://hdl.handle.net/10027/22007

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

8. Lagunas, Angelica Maria. Telomere DNA Damage Response Regulates Cancer Stem Cell Phenotype.

Degree: 2018, University of Illinois – Chicago

 Chromosome ends are protected by telomeres that prevent DNA damage response and degradation. When telomeres become critically short, the DNA damage response is activated at… (more)

Subjects/Keywords: telomeres; EMT; cancer stem cells

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APA (6th Edition):

Lagunas, A. M. (2018). Telomere DNA Damage Response Regulates Cancer Stem Cell Phenotype. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/22726

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lagunas, Angelica Maria. “Telomere DNA Damage Response Regulates Cancer Stem Cell Phenotype.” 2018. Thesis, University of Illinois – Chicago. Accessed April 14, 2021. http://hdl.handle.net/10027/22726.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lagunas, Angelica Maria. “Telomere DNA Damage Response Regulates Cancer Stem Cell Phenotype.” 2018. Web. 14 Apr 2021.

Vancouver:

Lagunas AM. Telomere DNA Damage Response Regulates Cancer Stem Cell Phenotype. [Internet] [Thesis]. University of Illinois – Chicago; 2018. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10027/22726.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lagunas AM. Telomere DNA Damage Response Regulates Cancer Stem Cell Phenotype. [Thesis]. University of Illinois – Chicago; 2018. Available from: http://hdl.handle.net/10027/22726

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Leicester

9. Hill, Louise Anne. The master-regulators of EMT and E-cadherin constitute a novel pathway in malignant melanoma.

Degree: PhD, 2013, University of Leicester

 The master-regulators of an epithelial-mesenchymal transition (MR-EMT) have a pivotal role in the regulation of carcinoma development, promoting transformation and generating a migratory and invasive… (more)

Subjects/Keywords: 616.99477; EMT; Cancer; Melanoma

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APA (6th Edition):

Hill, L. A. (2013). The master-regulators of EMT and E-cadherin constitute a novel pathway in malignant melanoma. (Doctoral Dissertation). University of Leicester. Retrieved from https://figshare.com/articles/The_master-regulators_of_EMT_and_E-cadherin_constitute_a_novel_pathway_in_malignant_melanoma/10158425 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.593680

Chicago Manual of Style (16th Edition):

Hill, Louise Anne. “The master-regulators of EMT and E-cadherin constitute a novel pathway in malignant melanoma.” 2013. Doctoral Dissertation, University of Leicester. Accessed April 14, 2021. https://figshare.com/articles/The_master-regulators_of_EMT_and_E-cadherin_constitute_a_novel_pathway_in_malignant_melanoma/10158425 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.593680.

MLA Handbook (7th Edition):

Hill, Louise Anne. “The master-regulators of EMT and E-cadherin constitute a novel pathway in malignant melanoma.” 2013. Web. 14 Apr 2021.

Vancouver:

Hill LA. The master-regulators of EMT and E-cadherin constitute a novel pathway in malignant melanoma. [Internet] [Doctoral dissertation]. University of Leicester; 2013. [cited 2021 Apr 14]. Available from: https://figshare.com/articles/The_master-regulators_of_EMT_and_E-cadherin_constitute_a_novel_pathway_in_malignant_melanoma/10158425 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.593680.

Council of Science Editors:

Hill LA. The master-regulators of EMT and E-cadherin constitute a novel pathway in malignant melanoma. [Doctoral Dissertation]. University of Leicester; 2013. Available from: https://figshare.com/articles/The_master-regulators_of_EMT_and_E-cadherin_constitute_a_novel_pathway_in_malignant_melanoma/10158425 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.593680


University of Florida

10. Wang, Bihan. Role of LSD 1 in Chemoresistant Colon Cancer.

Degree: MS, Biochemistry and Molecular Biology, 2019, University of Florida

 Colorectal cancer is the third leading cause of cancer-related deaths in United States. Currently, 5-fluorouracil (5-FU) is usually used as the backbone of treatment for… (more)

Subjects/Keywords: cancer  – emt  – epigenetic  – lsd1

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APA (6th Edition):

Wang, B. (2019). Role of LSD 1 in Chemoresistant Colon Cancer. (Masters Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/UFE0054107

Chicago Manual of Style (16th Edition):

Wang, Bihan. “Role of LSD 1 in Chemoresistant Colon Cancer.” 2019. Masters Thesis, University of Florida. Accessed April 14, 2021. https://ufdc.ufl.edu/UFE0054107.

MLA Handbook (7th Edition):

Wang, Bihan. “Role of LSD 1 in Chemoresistant Colon Cancer.” 2019. Web. 14 Apr 2021.

Vancouver:

Wang B. Role of LSD 1 in Chemoresistant Colon Cancer. [Internet] [Masters thesis]. University of Florida; 2019. [cited 2021 Apr 14]. Available from: https://ufdc.ufl.edu/UFE0054107.

Council of Science Editors:

Wang B. Role of LSD 1 in Chemoresistant Colon Cancer. [Masters Thesis]. University of Florida; 2019. Available from: https://ufdc.ufl.edu/UFE0054107


University of Melbourne

11. Golenkina, Sofya. NetrinA and Frazzled in regulation of wing disc epithelia.

Degree: 2017, University of Melbourne

 The chemotrophic factor Netrin and its receptors play a critical role during axon outgrowth, organogenesis and cancer progression. In my PhD I have found that… (more)

Subjects/Keywords: EMT; Netrin; Frazzled; Drosophila

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APA (6th Edition):

Golenkina, S. (2017). NetrinA and Frazzled in regulation of wing disc epithelia. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/192658

Chicago Manual of Style (16th Edition):

Golenkina, Sofya. “NetrinA and Frazzled in regulation of wing disc epithelia.” 2017. Doctoral Dissertation, University of Melbourne. Accessed April 14, 2021. http://hdl.handle.net/11343/192658.

MLA Handbook (7th Edition):

Golenkina, Sofya. “NetrinA and Frazzled in regulation of wing disc epithelia.” 2017. Web. 14 Apr 2021.

Vancouver:

Golenkina S. NetrinA and Frazzled in regulation of wing disc epithelia. [Internet] [Doctoral dissertation]. University of Melbourne; 2017. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/11343/192658.

Council of Science Editors:

Golenkina S. NetrinA and Frazzled in regulation of wing disc epithelia. [Doctoral Dissertation]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/192658


University of Melbourne

12. Soo, Eliza Ting Li. Epithelial-to-Mesenchymal Transition (EMT) in human breast cancer: investigating microRNAs in breast cancer EMT.

Degree: 2015, University of Melbourne

 Breast cancer is the most common malignancy among women worldwide, with mortality primarily associated with metastasis. In recent years, microRNAs (miRNAs) have emerged as a… (more)

Subjects/Keywords: breast cancer; EMT; microRNA; miRNA

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APA (6th Edition):

Soo, E. T. L. (2015). Epithelial-to-Mesenchymal Transition (EMT) in human breast cancer: investigating microRNAs in breast cancer EMT. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/55572

Chicago Manual of Style (16th Edition):

Soo, Eliza Ting Li. “Epithelial-to-Mesenchymal Transition (EMT) in human breast cancer: investigating microRNAs in breast cancer EMT.” 2015. Doctoral Dissertation, University of Melbourne. Accessed April 14, 2021. http://hdl.handle.net/11343/55572.

MLA Handbook (7th Edition):

Soo, Eliza Ting Li. “Epithelial-to-Mesenchymal Transition (EMT) in human breast cancer: investigating microRNAs in breast cancer EMT.” 2015. Web. 14 Apr 2021.

Vancouver:

Soo ETL. Epithelial-to-Mesenchymal Transition (EMT) in human breast cancer: investigating microRNAs in breast cancer EMT. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/11343/55572.

Council of Science Editors:

Soo ETL. Epithelial-to-Mesenchymal Transition (EMT) in human breast cancer: investigating microRNAs in breast cancer EMT. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/55572


Univerzitet u Beogradu

13. Filipović, Jelena M., 1985-, 59504137. Ekspresija protein arginin metil transferaze 1 (PRMT1) i njenih ko-efektora u tumorima bubrežnih ćelija kod odraslih.

Degree: Medicinski fakultet, 2020, Univerzitet u Beogradu

Medicina - Molekularna medicina / Medicine - Molecular medicine

Tumori bubrežnih ćelija (engl Renal cell tumors, RCT) čine 80% primarnih tumora bubrega. Najučestaliji RCT su:… (more)

Subjects/Keywords: PRMT1; EMT; renal cell tumors

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APA (6th Edition):

Filipović, Jelena M., 1985-, 5. (2020). Ekspresija protein arginin metil transferaze 1 (PRMT1) i njenih ko-efektora u tumorima bubrežnih ćelija kod odraslih. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:23022/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Filipović, Jelena M., 1985-, 59504137. “Ekspresija protein arginin metil transferaze 1 (PRMT1) i njenih ko-efektora u tumorima bubrežnih ćelija kod odraslih.” 2020. Thesis, Univerzitet u Beogradu. Accessed April 14, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:23022/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Filipović, Jelena M., 1985-, 59504137. “Ekspresija protein arginin metil transferaze 1 (PRMT1) i njenih ko-efektora u tumorima bubrežnih ćelija kod odraslih.” 2020. Web. 14 Apr 2021.

Vancouver:

Filipović, Jelena M., 1985- 5. Ekspresija protein arginin metil transferaze 1 (PRMT1) i njenih ko-efektora u tumorima bubrežnih ćelija kod odraslih. [Internet] [Thesis]. Univerzitet u Beogradu; 2020. [cited 2021 Apr 14]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:23022/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Filipović, Jelena M., 1985- 5. Ekspresija protein arginin metil transferaze 1 (PRMT1) i njenih ko-efektora u tumorima bubrežnih ćelija kod odraslih. [Thesis]. Univerzitet u Beogradu; 2020. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:23022/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


King Abdullah University of Science and Technology

14. AbuElela, Ayman. Regulation of the O-glycan-type Sialyl-Lewis X (sLex) Bio-synthesis Pathway during Cell Transformation Programs: Epithelial-Mesenchymal Transition (EMT) and Molecular Subtypes in Breast Carcinoma and Human T Cell Activation.

Degree: Biological and Environmental Sciences and Engineering (BESE) Division, 2017, King Abdullah University of Science and Technology

 During tumor progression and development of distant metastases, a subset of cancer cells undergoes transformation programs, such as epithelial-mesenchymal transition (EMT), to acquire enhanced migratory… (more)

Subjects/Keywords: Glycosyltransferases; EMT; Metastasis; E-Selectin

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APA (6th Edition):

AbuElela, A. (2017). Regulation of the O-glycan-type Sialyl-Lewis X (sLex) Bio-synthesis Pathway during Cell Transformation Programs: Epithelial-Mesenchymal Transition (EMT) and Molecular Subtypes in Breast Carcinoma and Human T Cell Activation. (Thesis). King Abdullah University of Science and Technology. Retrieved from http://hdl.handle.net/10754/626306

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

AbuElela, Ayman. “Regulation of the O-glycan-type Sialyl-Lewis X (sLex) Bio-synthesis Pathway during Cell Transformation Programs: Epithelial-Mesenchymal Transition (EMT) and Molecular Subtypes in Breast Carcinoma and Human T Cell Activation.” 2017. Thesis, King Abdullah University of Science and Technology. Accessed April 14, 2021. http://hdl.handle.net/10754/626306.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

AbuElela, Ayman. “Regulation of the O-glycan-type Sialyl-Lewis X (sLex) Bio-synthesis Pathway during Cell Transformation Programs: Epithelial-Mesenchymal Transition (EMT) and Molecular Subtypes in Breast Carcinoma and Human T Cell Activation.” 2017. Web. 14 Apr 2021.

Vancouver:

AbuElela A. Regulation of the O-glycan-type Sialyl-Lewis X (sLex) Bio-synthesis Pathway during Cell Transformation Programs: Epithelial-Mesenchymal Transition (EMT) and Molecular Subtypes in Breast Carcinoma and Human T Cell Activation. [Internet] [Thesis]. King Abdullah University of Science and Technology; 2017. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10754/626306.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

AbuElela A. Regulation of the O-glycan-type Sialyl-Lewis X (sLex) Bio-synthesis Pathway during Cell Transformation Programs: Epithelial-Mesenchymal Transition (EMT) and Molecular Subtypes in Breast Carcinoma and Human T Cell Activation. [Thesis]. King Abdullah University of Science and Technology; 2017. Available from: http://hdl.handle.net/10754/626306

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Jacqueroud, Laurent. Identification et caractérisation des complexes transcriptionnels de la protéine TWIST1 essentiels à la progression tumorale : The heterodimeric TWIST1-E12 complex drives the oncogenic potential of TWIST1 in human mammary epithelial cells.

Degree: Docteur es, Biologie moléculaire et cellulaire, 2015, Université Claude Bernard – Lyon I

Dans ce manuscrit, nous démontrons par le biais de dimères forcés que toutes les propriétés oncogéniques de la protéine TWIST1, telles qu'évaluées par le biais… (more)

Subjects/Keywords: TWIST1; Hétérodimère; Sénescence; EMT; TWIST1; Heterodimer; Senescence; EMT; 572.8

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APA (6th Edition):

Jacqueroud, L. (2015). Identification et caractérisation des complexes transcriptionnels de la protéine TWIST1 essentiels à la progression tumorale : The heterodimeric TWIST1-E12 complex drives the oncogenic potential of TWIST1 in human mammary epithelial cells. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2015LYO10054

Chicago Manual of Style (16th Edition):

Jacqueroud, Laurent. “Identification et caractérisation des complexes transcriptionnels de la protéine TWIST1 essentiels à la progression tumorale : The heterodimeric TWIST1-E12 complex drives the oncogenic potential of TWIST1 in human mammary epithelial cells.” 2015. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed April 14, 2021. http://www.theses.fr/2015LYO10054.

MLA Handbook (7th Edition):

Jacqueroud, Laurent. “Identification et caractérisation des complexes transcriptionnels de la protéine TWIST1 essentiels à la progression tumorale : The heterodimeric TWIST1-E12 complex drives the oncogenic potential of TWIST1 in human mammary epithelial cells.” 2015. Web. 14 Apr 2021.

Vancouver:

Jacqueroud L. Identification et caractérisation des complexes transcriptionnels de la protéine TWIST1 essentiels à la progression tumorale : The heterodimeric TWIST1-E12 complex drives the oncogenic potential of TWIST1 in human mammary epithelial cells. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2015. [cited 2021 Apr 14]. Available from: http://www.theses.fr/2015LYO10054.

Council of Science Editors:

Jacqueroud L. Identification et caractérisation des complexes transcriptionnels de la protéine TWIST1 essentiels à la progression tumorale : The heterodimeric TWIST1-E12 complex drives the oncogenic potential of TWIST1 in human mammary epithelial cells. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2015. Available from: http://www.theses.fr/2015LYO10054


University of Helsinki

16. Godbole, Nimish. MicroRNAs affecting epithelial-mesenchymal transition pathway in malignant mesothelioma.

Degree: Medicinska fakulteten, 2018, University of Helsinki

 Background: Malignant mesothelioma is a fatal cancer of the mesothelial cells characterized by previous exposure to asbestos, long latency period and shorter survival time thereafter.… (more)

Subjects/Keywords: MicroRNA; miRNA; mesothelioma; EMT; MicroRNA; miRNA; mesothelioma; EMT

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APA (6th Edition):

Godbole, N. (2018). MicroRNAs affecting epithelial-mesenchymal transition pathway in malignant mesothelioma. (Masters Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/236388

Chicago Manual of Style (16th Edition):

Godbole, Nimish. “MicroRNAs affecting epithelial-mesenchymal transition pathway in malignant mesothelioma.” 2018. Masters Thesis, University of Helsinki. Accessed April 14, 2021. http://hdl.handle.net/10138/236388.

MLA Handbook (7th Edition):

Godbole, Nimish. “MicroRNAs affecting epithelial-mesenchymal transition pathway in malignant mesothelioma.” 2018. Web. 14 Apr 2021.

Vancouver:

Godbole N. MicroRNAs affecting epithelial-mesenchymal transition pathway in malignant mesothelioma. [Internet] [Masters thesis]. University of Helsinki; 2018. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10138/236388.

Council of Science Editors:

Godbole N. MicroRNAs affecting epithelial-mesenchymal transition pathway in malignant mesothelioma. [Masters Thesis]. University of Helsinki; 2018. Available from: http://hdl.handle.net/10138/236388


NSYSU

17. Su, Huei-Ting. The Stem Cell Marker Nestin is Critical for TGF beta1- Mediated Tumor Progression in Pancreatic Cancer.

Degree: Master, Institute of Biomedical Sciences, 2012, NSYSU

 Stem cell marker Nestin is an intermediate filament protein that plays an important role in cell integrity, migration and differentiation. Nestin expression occurs in approximately… (more)

Subjects/Keywords: EMT; Intermediate filament proteins; Nestin; PDAC

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APA (6th Edition):

Su, H. (2012). The Stem Cell Marker Nestin is Critical for TGF beta1- Mediated Tumor Progression in Pancreatic Cancer. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0625112-152043

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Su, Huei-Ting. “The Stem Cell Marker Nestin is Critical for TGF beta1- Mediated Tumor Progression in Pancreatic Cancer.” 2012. Thesis, NSYSU. Accessed April 14, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0625112-152043.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Su, Huei-Ting. “The Stem Cell Marker Nestin is Critical for TGF beta1- Mediated Tumor Progression in Pancreatic Cancer.” 2012. Web. 14 Apr 2021.

Vancouver:

Su H. The Stem Cell Marker Nestin is Critical for TGF beta1- Mediated Tumor Progression in Pancreatic Cancer. [Internet] [Thesis]. NSYSU; 2012. [cited 2021 Apr 14]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0625112-152043.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Su H. The Stem Cell Marker Nestin is Critical for TGF beta1- Mediated Tumor Progression in Pancreatic Cancer. [Thesis]. NSYSU; 2012. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0625112-152043

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. 佐藤, 竜太. Angiopoietin-like protein 2 induces androgen-independent and malignant behavior in human prostate cancer cells.

Degree: 博士(医学), 2016, Oita University / 大分大学

 Angiopoietin-like proteins (ANGPTLs), which comprise 7 members (ANGPTL1-ANGPTL7), structurally resemble angiopoietins. We investigated the roles of ANGPTLs in the acquisition of androgen independence and the… (more)

Subjects/Keywords: ANGPTL2; prostate cancer; EMT; apoptosis; integrin α5β1

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APA (6th Edition):

佐藤, . (2016). Angiopoietin-like protein 2 induces androgen-independent and malignant behavior in human prostate cancer cells. (Thesis). Oita University / 大分大学. Retrieved from http://hdl.handle.net/10559/15619

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

佐藤, 竜太. “Angiopoietin-like protein 2 induces androgen-independent and malignant behavior in human prostate cancer cells.” 2016. Thesis, Oita University / 大分大学. Accessed April 14, 2021. http://hdl.handle.net/10559/15619.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

佐藤, 竜太. “Angiopoietin-like protein 2 induces androgen-independent and malignant behavior in human prostate cancer cells.” 2016. Web. 14 Apr 2021.

Vancouver:

佐藤 . Angiopoietin-like protein 2 induces androgen-independent and malignant behavior in human prostate cancer cells. [Internet] [Thesis]. Oita University / 大分大学; 2016. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10559/15619.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

佐藤 . Angiopoietin-like protein 2 induces androgen-independent and malignant behavior in human prostate cancer cells. [Thesis]. Oita University / 大分大学; 2016. Available from: http://hdl.handle.net/10559/15619

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

19. Welch-Reardon, Katrina Marie. Defining a role for Snail family transcription factors during angiogenesis.

Degree: Biological Sciences, 2014, University of California – Irvine

 Angiogenesis is a tightly regulated multi-step process in which new blood vessels form from the preexisting vasculature. Due to extensive research in the field of… (more)

Subjects/Keywords: Biology; Angiogenesis; EMT; EndMT; Slug; Snail

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APA (6th Edition):

Welch-Reardon, K. M. (2014). Defining a role for Snail family transcription factors during angiogenesis. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/91r2t8z5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Welch-Reardon, Katrina Marie. “Defining a role for Snail family transcription factors during angiogenesis.” 2014. Thesis, University of California – Irvine. Accessed April 14, 2021. http://www.escholarship.org/uc/item/91r2t8z5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Welch-Reardon, Katrina Marie. “Defining a role for Snail family transcription factors during angiogenesis.” 2014. Web. 14 Apr 2021.

Vancouver:

Welch-Reardon KM. Defining a role for Snail family transcription factors during angiogenesis. [Internet] [Thesis]. University of California – Irvine; 2014. [cited 2021 Apr 14]. Available from: http://www.escholarship.org/uc/item/91r2t8z5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Welch-Reardon KM. Defining a role for Snail family transcription factors during angiogenesis. [Thesis]. University of California – Irvine; 2014. Available from: http://www.escholarship.org/uc/item/91r2t8z5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

20. Farina, Mirko. Slaying the chimera: a complementarity approach to the extended mind thesis.

Degree: 2012, University of Edinburgh

 Much of the literature directed at the Extended Mind Thesis (EMT) has revolved around parity issues, focussing on the problem of how to individuate the… (more)

Subjects/Keywords: extended mind thesis; EMT; sensory substitution

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APA (6th Edition):

Farina, M. (2012). Slaying the chimera: a complementarity approach to the extended mind thesis. (Thesis). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/6294

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Farina, Mirko. “Slaying the chimera: a complementarity approach to the extended mind thesis.” 2012. Thesis, University of Edinburgh. Accessed April 14, 2021. http://hdl.handle.net/1842/6294.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Farina, Mirko. “Slaying the chimera: a complementarity approach to the extended mind thesis.” 2012. Web. 14 Apr 2021.

Vancouver:

Farina M. Slaying the chimera: a complementarity approach to the extended mind thesis. [Internet] [Thesis]. University of Edinburgh; 2012. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1842/6294.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Farina M. Slaying the chimera: a complementarity approach to the extended mind thesis. [Thesis]. University of Edinburgh; 2012. Available from: http://hdl.handle.net/1842/6294

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

21. Yang, Pei-hua. Study the mechanisms of Pentabromophenol in the modulation of TGF-β signaling.

Degree: Master, Biological Sciences, 2017, NSYSU

 Pentabromophenol (PBP), a brominated flame retardant (BFR), is widely used in various consumer products. BFRs exert adverse health effects such as neurotoxic and endocrine-disrupting effects.… (more)

Subjects/Keywords: BFR; EMT; Smad; TGF-β; PBP

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APA (6th Edition):

Yang, P. (2017). Study the mechanisms of Pentabromophenol in the modulation of TGF-β signaling. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715117-084334

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yang, Pei-hua. “Study the mechanisms of Pentabromophenol in the modulation of TGF-β signaling.” 2017. Thesis, NSYSU. Accessed April 14, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715117-084334.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yang, Pei-hua. “Study the mechanisms of Pentabromophenol in the modulation of TGF-β signaling.” 2017. Web. 14 Apr 2021.

Vancouver:

Yang P. Study the mechanisms of Pentabromophenol in the modulation of TGF-β signaling. [Internet] [Thesis]. NSYSU; 2017. [cited 2021 Apr 14]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715117-084334.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yang P. Study the mechanisms of Pentabromophenol in the modulation of TGF-β signaling. [Thesis]. NSYSU; 2017. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715117-084334

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

22. DeLaughter, Daniel Morris. Novel Regulators of Epithelial-to-Mesenchymal Transformation in Cardiogenesis are Identified Through Next-Generation Sequencing.

Degree: PhD, Cell and Developmental Biology, 2013, Vanderbilt University

 Epithelial-to-Mesenchymal Transformation (EMT) is an important process in development, and occurs during key steps in both valvular and coronary vessel development. This dissertation uses transcriptional… (more)

Subjects/Keywords: EMT; Endocardial Cushion; Heart Valve; Development

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APA (6th Edition):

DeLaughter, D. M. (2013). Novel Regulators of Epithelial-to-Mesenchymal Transformation in Cardiogenesis are Identified Through Next-Generation Sequencing. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12416

Chicago Manual of Style (16th Edition):

DeLaughter, Daniel Morris. “Novel Regulators of Epithelial-to-Mesenchymal Transformation in Cardiogenesis are Identified Through Next-Generation Sequencing.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed April 14, 2021. http://hdl.handle.net/1803/12416.

MLA Handbook (7th Edition):

DeLaughter, Daniel Morris. “Novel Regulators of Epithelial-to-Mesenchymal Transformation in Cardiogenesis are Identified Through Next-Generation Sequencing.” 2013. Web. 14 Apr 2021.

Vancouver:

DeLaughter DM. Novel Regulators of Epithelial-to-Mesenchymal Transformation in Cardiogenesis are Identified Through Next-Generation Sequencing. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1803/12416.

Council of Science Editors:

DeLaughter DM. Novel Regulators of Epithelial-to-Mesenchymal Transformation in Cardiogenesis are Identified Through Next-Generation Sequencing. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/12416


Vanderbilt University

23. Stoops, Sydney Lear. Discovery, Optimization, and Understanding the Mechanism of Action of Small Molecules that Restore E-cadherin Expression.

Degree: PhD, Pharmacology, 2012, Vanderbilt University

 E-cadherin is a transmembrane protein that maintains intercellular contacts and cellular polarity in epithelial tissues. The down-regulation of E-cadherin is thought to aid in the… (more)

Subjects/Keywords: EMT; E-Cadherin; Epithelial-Mesenchymal Transition

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APA (6th Edition):

Stoops, S. L. (2012). Discovery, Optimization, and Understanding the Mechanism of Action of Small Molecules that Restore E-cadherin Expression. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11403

Chicago Manual of Style (16th Edition):

Stoops, Sydney Lear. “Discovery, Optimization, and Understanding the Mechanism of Action of Small Molecules that Restore E-cadherin Expression.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed April 14, 2021. http://hdl.handle.net/1803/11403.

MLA Handbook (7th Edition):

Stoops, Sydney Lear. “Discovery, Optimization, and Understanding the Mechanism of Action of Small Molecules that Restore E-cadherin Expression.” 2012. Web. 14 Apr 2021.

Vancouver:

Stoops SL. Discovery, Optimization, and Understanding the Mechanism of Action of Small Molecules that Restore E-cadherin Expression. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1803/11403.

Council of Science Editors:

Stoops SL. Discovery, Optimization, and Understanding the Mechanism of Action of Small Molecules that Restore E-cadherin Expression. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/11403


Texas Tech University

24. -0519-8840. Studying the anticancer properties of Parthenolide (PTL) in MCF-7 breast cancer cells.

Degree: MS, Nutritional Sciences, 2018, Texas Tech University

 Background: Breast cancer is considered as the most prevalent cancer in women world-wide. The average risk for a woman developing breast cancer in her life… (more)

Subjects/Keywords: Breast cancer; Parthenolide; Apoptosis; EMT; Angiogenesis

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APA (6th Edition):

-0519-8840. (2018). Studying the anticancer properties of Parthenolide (PTL) in MCF-7 breast cancer cells. (Masters Thesis). Texas Tech University. Retrieved from http://hdl.handle.net/2346/74400

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-0519-8840. “Studying the anticancer properties of Parthenolide (PTL) in MCF-7 breast cancer cells.” 2018. Masters Thesis, Texas Tech University. Accessed April 14, 2021. http://hdl.handle.net/2346/74400.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-0519-8840. “Studying the anticancer properties of Parthenolide (PTL) in MCF-7 breast cancer cells.” 2018. Web. 14 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-0519-8840. Studying the anticancer properties of Parthenolide (PTL) in MCF-7 breast cancer cells. [Internet] [Masters thesis]. Texas Tech University; 2018. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2346/74400.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-0519-8840. Studying the anticancer properties of Parthenolide (PTL) in MCF-7 breast cancer cells. [Masters Thesis]. Texas Tech University; 2018. Available from: http://hdl.handle.net/2346/74400

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


University of Adelaide

25. Paterson, Emily Louise. Downregulation of the MicroRNA-200 family induces epithelial to mesenchymal transition.

Degree: 2010, University of Adelaide

 Epithelial to mesenchymal transition (EMT) is a reversible developmental morphogenesis associated with cancer invasion and metastasis. Essential for the transition is the downregulation of E-cadherin,… (more)

Subjects/Keywords: EMT; microRNA; colon cancer; in situ hybridisation

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APA (6th Edition):

Paterson, E. L. (2010). Downregulation of the MicroRNA-200 family induces epithelial to mesenchymal transition. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/65625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Paterson, Emily Louise. “Downregulation of the MicroRNA-200 family induces epithelial to mesenchymal transition.” 2010. Thesis, University of Adelaide. Accessed April 14, 2021. http://hdl.handle.net/2440/65625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Paterson, Emily Louise. “Downregulation of the MicroRNA-200 family induces epithelial to mesenchymal transition.” 2010. Web. 14 Apr 2021.

Vancouver:

Paterson EL. Downregulation of the MicroRNA-200 family induces epithelial to mesenchymal transition. [Internet] [Thesis]. University of Adelaide; 2010. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2440/65625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Paterson EL. Downregulation of the MicroRNA-200 family induces epithelial to mesenchymal transition. [Thesis]. University of Adelaide; 2010. Available from: http://hdl.handle.net/2440/65625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Miami

26. Besser, Alexandra. p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program.

Degree: PhD, Cancer Biology (Medicine), 2015, University of Miami

  In normal cells, p27 regulates cell cycle and functions as an atypical tumor suppressor. Unlike typical tumor suppressors such as p16 and pRb, p27… (more)

Subjects/Keywords: p27; Metastasis; EMT; PI3K; STAT3; TGF-B2

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APA (6th Edition):

Besser, A. (2015). p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/1545

Chicago Manual of Style (16th Edition):

Besser, Alexandra. “p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program.” 2015. Doctoral Dissertation, University of Miami. Accessed April 14, 2021. https://scholarlyrepository.miami.edu/oa_dissertations/1545.

MLA Handbook (7th Edition):

Besser, Alexandra. “p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program.” 2015. Web. 14 Apr 2021.

Vancouver:

Besser A. p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program. [Internet] [Doctoral dissertation]. University of Miami; 2015. [cited 2021 Apr 14]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1545.

Council of Science Editors:

Besser A. p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program. [Doctoral Dissertation]. University of Miami; 2015. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1545

27. Spafford, PL. Mechanisms and drivers of epithelial to mesenchymal transition in COPD.

Degree: 2018, University of Tasmania

 Chronic obstructive pulmonary disease (COPD) is a progressive and irreversible limitation of airflow which predominantly affectsa smokers. One of the mechanisms behind the development and… (more)

Subjects/Keywords: EMT; COPD; salmeterol; tiotropium; fluticasone; BEAS-2B

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APA (6th Edition):

Spafford, P. (2018). Mechanisms and drivers of epithelial to mesenchymal transition in COPD. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/30032/1/Spafford_whole_thesis.pdf ; Spafford, PL ORCID: 0000-0002-6990-9680 <https://orcid.org/0000-0002-6990-9680> 2018 , 'Mechanisms and drivers of epithelial to mesenchymal transition in COPD', PhD thesis, University of Tasmania.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Spafford, PL. “Mechanisms and drivers of epithelial to mesenchymal transition in COPD.” 2018. Thesis, University of Tasmania. Accessed April 14, 2021. https://eprints.utas.edu.au/30032/1/Spafford_whole_thesis.pdf ; Spafford, PL ORCID: 0000-0002-6990-9680 <https://orcid.org/0000-0002-6990-9680> 2018 , 'Mechanisms and drivers of epithelial to mesenchymal transition in COPD', PhD thesis, University of Tasmania..

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Spafford, PL. “Mechanisms and drivers of epithelial to mesenchymal transition in COPD.” 2018. Web. 14 Apr 2021.

Vancouver:

Spafford P. Mechanisms and drivers of epithelial to mesenchymal transition in COPD. [Internet] [Thesis]. University of Tasmania; 2018. [cited 2021 Apr 14]. Available from: https://eprints.utas.edu.au/30032/1/Spafford_whole_thesis.pdf ; Spafford, PL ORCID: 0000-0002-6990-9680 <https://orcid.org/0000-0002-6990-9680> 2018 , 'Mechanisms and drivers of epithelial to mesenchymal transition in COPD', PhD thesis, University of Tasmania..

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Spafford P. Mechanisms and drivers of epithelial to mesenchymal transition in COPD. [Thesis]. University of Tasmania; 2018. Available from: https://eprints.utas.edu.au/30032/1/Spafford_whole_thesis.pdf ; Spafford, PL ORCID: 0000-0002-6990-9680 <https://orcid.org/0000-0002-6990-9680> 2018 , 'Mechanisms and drivers of epithelial to mesenchymal transition in COPD', PhD thesis, University of Tasmania.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

28. Salt, Megan. Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival.

Degree: Biomedical Sciences, 2014, University of California – San Francisco

 Tumors showing evidence of epithelial to mesenchymal transition (EMT) have been associated with metastasis, drug resistance, and poor prognosis. Heterogeneity along the EMT spectrum is… (more)

Subjects/Keywords: Cellular biology; Biology; EMT; ERBB3; PI3K

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Salt, M. (2014). Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/52s838hv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Salt, Megan. “Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival.” 2014. Thesis, University of California – San Francisco. Accessed April 14, 2021. http://www.escholarship.org/uc/item/52s838hv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Salt, Megan. “Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival.” 2014. Web. 14 Apr 2021.

Vancouver:

Salt M. Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival. [Internet] [Thesis]. University of California – San Francisco; 2014. [cited 2021 Apr 14]. Available from: http://www.escholarship.org/uc/item/52s838hv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Salt M. Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival. [Thesis]. University of California – San Francisco; 2014. Available from: http://www.escholarship.org/uc/item/52s838hv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Bristol

29. Marcolino De Assis Junior, Eudmar. The regulation of PRH/HHEX by transforming growth factor β.

Degree: PhD, 2019, University of Bristol

 The transcription factor PRH/HHEX (Proline-Rich Homeodomain/Haematopoietically Expressed Homeobox) controls cell proliferation, cell differentiation and cell migration/invasion in a diverse range of cell types. Here I… (more)

Subjects/Keywords: Prostate Cancer; PRH/HHEX; TGF-ß; EMT

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Marcolino De Assis Junior, E. (2019). The regulation of PRH/HHEX by transforming growth factor β. (Doctoral Dissertation). University of Bristol. Retrieved from http://hdl.handle.net/1983/f66f42ba-a735-4397-9367-bdd63e2e519d

Chicago Manual of Style (16th Edition):

Marcolino De Assis Junior, Eudmar. “The regulation of PRH/HHEX by transforming growth factor β.” 2019. Doctoral Dissertation, University of Bristol. Accessed April 14, 2021. http://hdl.handle.net/1983/f66f42ba-a735-4397-9367-bdd63e2e519d.

MLA Handbook (7th Edition):

Marcolino De Assis Junior, Eudmar. “The regulation of PRH/HHEX by transforming growth factor β.” 2019. Web. 14 Apr 2021.

Vancouver:

Marcolino De Assis Junior E. The regulation of PRH/HHEX by transforming growth factor β. [Internet] [Doctoral dissertation]. University of Bristol; 2019. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1983/f66f42ba-a735-4397-9367-bdd63e2e519d.

Council of Science Editors:

Marcolino De Assis Junior E. The regulation of PRH/HHEX by transforming growth factor β. [Doctoral Dissertation]. University of Bristol; 2019. Available from: http://hdl.handle.net/1983/f66f42ba-a735-4397-9367-bdd63e2e519d


University of Leicester

30. Papadogeorgakis, Eftychios. Development of malignant melanoma is dependent on a switch in Embryonic Transcription Factors orchestrated by the BRAF-MAPK pathway.

Degree: PhD, 2013, University of Leicester

 Reactivation of master regulators of epithelial to mesenchymal transition (MR-EMT) represents the molecular basis for tumour cell plasticity, malignant transformation and metastases. However, the current… (more)

Subjects/Keywords: 616.99; Melanoma; EMT Transcription Factors; BRAF

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Papadogeorgakis, E. (2013). Development of malignant melanoma is dependent on a switch in Embryonic Transcription Factors orchestrated by the BRAF-MAPK pathway. (Doctoral Dissertation). University of Leicester. Retrieved from https://figshare.com/articles/Development_of_malignant_melanoma_is_dependent_on_a_switch_in_Embryonic_Transcription_Factors_orchestrated_by_the_BRAF-MAPK_pathway/10131503 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.579207

Chicago Manual of Style (16th Edition):

Papadogeorgakis, Eftychios. “Development of malignant melanoma is dependent on a switch in Embryonic Transcription Factors orchestrated by the BRAF-MAPK pathway.” 2013. Doctoral Dissertation, University of Leicester. Accessed April 14, 2021. https://figshare.com/articles/Development_of_malignant_melanoma_is_dependent_on_a_switch_in_Embryonic_Transcription_Factors_orchestrated_by_the_BRAF-MAPK_pathway/10131503 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.579207.

MLA Handbook (7th Edition):

Papadogeorgakis, Eftychios. “Development of malignant melanoma is dependent on a switch in Embryonic Transcription Factors orchestrated by the BRAF-MAPK pathway.” 2013. Web. 14 Apr 2021.

Vancouver:

Papadogeorgakis E. Development of malignant melanoma is dependent on a switch in Embryonic Transcription Factors orchestrated by the BRAF-MAPK pathway. [Internet] [Doctoral dissertation]. University of Leicester; 2013. [cited 2021 Apr 14]. Available from: https://figshare.com/articles/Development_of_malignant_melanoma_is_dependent_on_a_switch_in_Embryonic_Transcription_Factors_orchestrated_by_the_BRAF-MAPK_pathway/10131503 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.579207.

Council of Science Editors:

Papadogeorgakis E. Development of malignant melanoma is dependent on a switch in Embryonic Transcription Factors orchestrated by the BRAF-MAPK pathway. [Doctoral Dissertation]. University of Leicester; 2013. Available from: https://figshare.com/articles/Development_of_malignant_melanoma_is_dependent_on_a_switch_in_Embryonic_Transcription_Factors_orchestrated_by_the_BRAF-MAPK_pathway/10131503 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.579207

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