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You searched for subject:(EGFR). Showing records 1 – 30 of 486 total matches.

[1] [2] [3] [4] [5] … [17]

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Queens University

1. Gilmour, Alanna. The Role of Neu1 Sialidase in Epidermal Growth Factor Receptor Activation .

Degree: Microbiology and Immunology, 2011, Queens University

 The epidermal growth factor receptor (EGFR) exists as a single, highly glycosylated subunit receptor on the plasma membrane of a cell. Upon ligand binding to… (more)

Subjects/Keywords: EGFR ; Sialidase

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gilmour, A. (2011). The Role of Neu1 Sialidase in Epidermal Growth Factor Receptor Activation . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/6582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gilmour, Alanna. “The Role of Neu1 Sialidase in Epidermal Growth Factor Receptor Activation .” 2011. Thesis, Queens University. Accessed November 26, 2020. http://hdl.handle.net/1974/6582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gilmour, Alanna. “The Role of Neu1 Sialidase in Epidermal Growth Factor Receptor Activation .” 2011. Web. 26 Nov 2020.

Vancouver:

Gilmour A. The Role of Neu1 Sialidase in Epidermal Growth Factor Receptor Activation . [Internet] [Thesis]. Queens University; 2011. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1974/6582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gilmour A. The Role of Neu1 Sialidase in Epidermal Growth Factor Receptor Activation . [Thesis]. Queens University; 2011. Available from: http://hdl.handle.net/1974/6582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Mota, Jorge Alexandre Pereira. Development and optimization of a methodology to synthetize chitosan/cetuximab conjugates for encapsulate siRNA and targeting to EGFR overexpressing cells.

Degree: 2014, RCAAP

 O uso terapêutico de Small Interfering RNA (siRNA) tem sido observado como uma potencial nova abordagem para o tratamento do cancro. O desafio-chave no campo… (more)

Subjects/Keywords: SiRNA; EGFR

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APA (6th Edition):

Mota, J. A. P. (2014). Development and optimization of a methodology to synthetize chitosan/cetuximab conjugates for encapsulate siRNA and targeting to EGFR overexpressing cells. (Thesis). RCAAP. Retrieved from https://www.rcaap.pt/detail.jsp?id=oai:repositorio.cespu.pt:20.500.11816/368

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mota, Jorge Alexandre Pereira. “Development and optimization of a methodology to synthetize chitosan/cetuximab conjugates for encapsulate siRNA and targeting to EGFR overexpressing cells.” 2014. Thesis, RCAAP. Accessed November 26, 2020. https://www.rcaap.pt/detail.jsp?id=oai:repositorio.cespu.pt:20.500.11816/368.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mota, Jorge Alexandre Pereira. “Development and optimization of a methodology to synthetize chitosan/cetuximab conjugates for encapsulate siRNA and targeting to EGFR overexpressing cells.” 2014. Web. 26 Nov 2020.

Vancouver:

Mota JAP. Development and optimization of a methodology to synthetize chitosan/cetuximab conjugates for encapsulate siRNA and targeting to EGFR overexpressing cells. [Internet] [Thesis]. RCAAP; 2014. [cited 2020 Nov 26]. Available from: https://www.rcaap.pt/detail.jsp?id=oai:repositorio.cespu.pt:20.500.11816/368.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mota JAP. Development and optimization of a methodology to synthetize chitosan/cetuximab conjugates for encapsulate siRNA and targeting to EGFR overexpressing cells. [Thesis]. RCAAP; 2014. Available from: https://www.rcaap.pt/detail.jsp?id=oai:repositorio.cespu.pt:20.500.11816/368

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

3. Menezes, Sharleen Valerie. Understanding the molecular mechanisms that underlie the anti-cancer activity of the metastasis suppressor, NDRG1 in the treatment of cancer.

Degree: 2018, University of Sydney

 Pancreatic cancer is an aggressive disease that continues to be associated with low survival rates, therefore there is a need for more targeted therapies. This… (more)

Subjects/Keywords: Cancer; Metastasis; EGFR

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APA (6th Edition):

Menezes, S. V. (2018). Understanding the molecular mechanisms that underlie the anti-cancer activity of the metastasis suppressor, NDRG1 in the treatment of cancer. (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/20193

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Menezes, Sharleen Valerie. “Understanding the molecular mechanisms that underlie the anti-cancer activity of the metastasis suppressor, NDRG1 in the treatment of cancer. ” 2018. Thesis, University of Sydney. Accessed November 26, 2020. http://hdl.handle.net/2123/20193.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Menezes, Sharleen Valerie. “Understanding the molecular mechanisms that underlie the anti-cancer activity of the metastasis suppressor, NDRG1 in the treatment of cancer. ” 2018. Web. 26 Nov 2020.

Vancouver:

Menezes SV. Understanding the molecular mechanisms that underlie the anti-cancer activity of the metastasis suppressor, NDRG1 in the treatment of cancer. [Internet] [Thesis]. University of Sydney; 2018. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2123/20193.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Menezes SV. Understanding the molecular mechanisms that underlie the anti-cancer activity of the metastasis suppressor, NDRG1 in the treatment of cancer. [Thesis]. University of Sydney; 2018. Available from: http://hdl.handle.net/2123/20193

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Keul, Marina. Etablierung von biochemischen und strukturbasierten Systemen zur Charakterisierung wirkstoffresistenter EGFR-Mutanten.

Degree: 2019, Technische Universität Dortmund

 Die Identifikation von EGFR-Mutationen in Exon19 und Exon21 als prädiktive Biomarker, die mit einer erhöhten Sensitivität gegenüber EGFR-Inhibitoren der ersten Generation assoziiert werden konnten, leitete… (more)

Subjects/Keywords: Präzisionsmedizin; NSCLC; EGFR; Kovalente EGFR-Inhibitoren; 570; 540; Biomarker; EGFR

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APA (6th Edition):

Keul, M. (2019). Etablierung von biochemischen und strukturbasierten Systemen zur Charakterisierung wirkstoffresistenter EGFR-Mutanten. (Doctoral Dissertation). Technische Universität Dortmund. Retrieved from http://dx.doi.org/10.17877/DE290R-21096

Chicago Manual of Style (16th Edition):

Keul, Marina. “Etablierung von biochemischen und strukturbasierten Systemen zur Charakterisierung wirkstoffresistenter EGFR-Mutanten.” 2019. Doctoral Dissertation, Technische Universität Dortmund. Accessed November 26, 2020. http://dx.doi.org/10.17877/DE290R-21096.

MLA Handbook (7th Edition):

Keul, Marina. “Etablierung von biochemischen und strukturbasierten Systemen zur Charakterisierung wirkstoffresistenter EGFR-Mutanten.” 2019. Web. 26 Nov 2020.

Vancouver:

Keul M. Etablierung von biochemischen und strukturbasierten Systemen zur Charakterisierung wirkstoffresistenter EGFR-Mutanten. [Internet] [Doctoral dissertation]. Technische Universität Dortmund; 2019. [cited 2020 Nov 26]. Available from: http://dx.doi.org/10.17877/DE290R-21096.

Council of Science Editors:

Keul M. Etablierung von biochemischen und strukturbasierten Systemen zur Charakterisierung wirkstoffresistenter EGFR-Mutanten. [Doctoral Dissertation]. Technische Universität Dortmund; 2019. Available from: http://dx.doi.org/10.17877/DE290R-21096

5. 谷口, 一也. 分子プロファイルによる固形癌の分類と臨床応用への展開 : Tumor classification for clinical application by molecular profiling; ブンシ プロファイル ニヨル コケイガン ノ ブンルイ ト リンショウ オウヨウ エノ テンカイ.

Degree: Nara Institute of Science and Technology / 奈良先端科学技術大学院大学

Subjects/Keywords: ; EGFR

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APA (6th Edition):

谷口, . (n.d.). 分子プロファイルによる固形癌の分類と臨床応用への展開 : Tumor classification for clinical application by molecular profiling; ブンシ プロファイル ニヨル コケイガン ノ ブンルイ ト リンショウ オウヨウ エノ テンカイ. (Thesis). Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Retrieved from http://hdl.handle.net/10061/4075

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

谷口, 一也. “分子プロファイルによる固形癌の分類と臨床応用への展開 : Tumor classification for clinical application by molecular profiling; ブンシ プロファイル ニヨル コケイガン ノ ブンルイ ト リンショウ オウヨウ エノ テンカイ.” Thesis, Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Accessed November 26, 2020. http://hdl.handle.net/10061/4075.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

谷口, 一也. “分子プロファイルによる固形癌の分類と臨床応用への展開 : Tumor classification for clinical application by molecular profiling; ブンシ プロファイル ニヨル コケイガン ノ ブンルイ ト リンショウ オウヨウ エノ テンカイ.” Web. 26 Nov 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

谷口 . 分子プロファイルによる固形癌の分類と臨床応用への展開 : Tumor classification for clinical application by molecular profiling; ブンシ プロファイル ニヨル コケイガン ノ ブンルイ ト リンショウ オウヨウ エノ テンカイ. [Internet] [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; [cited 2020 Nov 26]. Available from: http://hdl.handle.net/10061/4075.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

谷口 . 分子プロファイルによる固形癌の分類と臨床応用への展開 : Tumor classification for clinical application by molecular profiling; ブンシ プロファイル ニヨル コケイガン ノ ブンルイ ト リンショウ オウヨウ エノ テンカイ. [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; Available from: http://hdl.handle.net/10061/4075

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

6. Bedir, Ilgın Gizem. Meningioma doku örneklerinde egfr, 1p36, 14q genlerinin floresan ın situ hibridizasyon (fısh) yöntemiyle değerlendirilmesi .

Degree: ESOGÜ, Tıp Fakültesi, Tıbbi Genetik, 2013, Eskisehir Osmangazi University

 Tüm kanserler içerisinde intrakranial tümörler % 1,5 oranında görülmekle beraber tüm kansere bağlı ölümlerin %2’sinden sorumludur. Meningiomlar primer intrakranial tümörlerin yaklaşık %20’sini oluştururlar. Yavaş büyüyen… (more)

Subjects/Keywords: Meningioma; EGFR; 1p36; 14q; FISH

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APA (6th Edition):

Bedir, I. G. (2013). Meningioma doku örneklerinde egfr, 1p36, 14q genlerinin floresan ın situ hibridizasyon (fısh) yöntemiyle değerlendirilmesi . (Thesis). Eskisehir Osmangazi University. Retrieved from http://hdl.handle.net/11684/984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bedir, Ilgın Gizem. “Meningioma doku örneklerinde egfr, 1p36, 14q genlerinin floresan ın situ hibridizasyon (fısh) yöntemiyle değerlendirilmesi .” 2013. Thesis, Eskisehir Osmangazi University. Accessed November 26, 2020. http://hdl.handle.net/11684/984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bedir, Ilgın Gizem. “Meningioma doku örneklerinde egfr, 1p36, 14q genlerinin floresan ın situ hibridizasyon (fısh) yöntemiyle değerlendirilmesi .” 2013. Web. 26 Nov 2020.

Vancouver:

Bedir IG. Meningioma doku örneklerinde egfr, 1p36, 14q genlerinin floresan ın situ hibridizasyon (fısh) yöntemiyle değerlendirilmesi . [Internet] [Thesis]. Eskisehir Osmangazi University; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/11684/984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bedir IG. Meningioma doku örneklerinde egfr, 1p36, 14q genlerinin floresan ın situ hibridizasyon (fısh) yöntemiyle değerlendirilmesi . [Thesis]. Eskisehir Osmangazi University; 2013. Available from: http://hdl.handle.net/11684/984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

7. Amato, Katherine Renee. Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 Lung cancer remains the leading cause of cancer related deaths in the United States despite a significant number of advancements in the molecular diagnosis and… (more)

Subjects/Keywords: TKI; EPH; KRAS; EGFR

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APA (6th Edition):

Amato, K. R. (2015). Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11130

Chicago Manual of Style (16th Edition):

Amato, Katherine Renee. “Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed November 26, 2020. http://hdl.handle.net/1803/11130.

MLA Handbook (7th Edition):

Amato, Katherine Renee. “Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer.” 2015. Web. 26 Nov 2020.

Vancouver:

Amato KR. Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1803/11130.

Council of Science Editors:

Amato KR. Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/11130


Vanderbilt University

8. Meador, Catherine Belle. Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

EGFR-mutant lung cancers are highly sensitive to EGFR tyrosine kinase inhibitors (TKIs; erlotinib/gefitinib/afatinib), but tumors develop drug resistance within 9-16 months. Resistance to gefitinib/erlotinib commonly… (more)

Subjects/Keywords: targeted therapy; EGFR; lung cancer

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APA (6th Edition):

Meador, C. B. (2015). Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13334

Chicago Manual of Style (16th Edition):

Meador, Catherine Belle. “Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed November 26, 2020. http://hdl.handle.net/1803/13334.

MLA Handbook (7th Edition):

Meador, Catherine Belle. “Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma.” 2015. Web. 26 Nov 2020.

Vancouver:

Meador CB. Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1803/13334.

Council of Science Editors:

Meador CB. Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/13334


Penn State University

9. Wu, Juan. Investigating the roles of beta Heavy-spectrin in epidermal growth factor receptor signaling activity in Drosophila melanogaster .

Degree: 2011, Penn State University

 The spectrin-based membrane skeleton (SBMS) is a flexible and multifunctional scaffold involved in a wide spectrum of cellular processes including the generation of specialized membrane… (more)

Subjects/Keywords: Trafficking; Spectrin; EGFR; Endocytosis

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APA (6th Edition):

Wu, J. (2011). Investigating the roles of beta Heavy-spectrin in epidermal growth factor receptor signaling activity in Drosophila melanogaster . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/11687

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Juan. “Investigating the roles of beta Heavy-spectrin in epidermal growth factor receptor signaling activity in Drosophila melanogaster .” 2011. Thesis, Penn State University. Accessed November 26, 2020. https://submit-etda.libraries.psu.edu/catalog/11687.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Juan. “Investigating the roles of beta Heavy-spectrin in epidermal growth factor receptor signaling activity in Drosophila melanogaster .” 2011. Web. 26 Nov 2020.

Vancouver:

Wu J. Investigating the roles of beta Heavy-spectrin in epidermal growth factor receptor signaling activity in Drosophila melanogaster . [Internet] [Thesis]. Penn State University; 2011. [cited 2020 Nov 26]. Available from: https://submit-etda.libraries.psu.edu/catalog/11687.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu J. Investigating the roles of beta Heavy-spectrin in epidermal growth factor receptor signaling activity in Drosophila melanogaster . [Thesis]. Penn State University; 2011. Available from: https://submit-etda.libraries.psu.edu/catalog/11687

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Debrecen

10. Árvai, Kristóf. Új lehetőségek a valós idejű PCR alkalmazására a molekuláris diagnosztikában .

Degree: DE – TEK – Természettudományi és Technológiai Kar – Biológiai és Ökológiai Intézet, 2011, University of Debrecen

 Kísérleteink célja az volt, hogy megvizsgáljuk a közelmúltban bemutatkozott génszkenneléses eljárás alkalmazhatóságát a laboratóriumunkba érkező klinikai beteg anyagokon, illetve vizsgálataink tárgyát képezte az is, hogy… (more)

Subjects/Keywords: PCR diagnosztika; HRM szekvenálás; EGFR

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APA (6th Edition):

Árvai, K. (2011). Új lehetőségek a valós idejű PCR alkalmazására a molekuláris diagnosztikában . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/107474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Árvai, Kristóf. “Új lehetőségek a valós idejű PCR alkalmazására a molekuláris diagnosztikában .” 2011. Thesis, University of Debrecen. Accessed November 26, 2020. http://hdl.handle.net/2437/107474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Árvai, Kristóf. “Új lehetőségek a valós idejű PCR alkalmazására a molekuláris diagnosztikában .” 2011. Web. 26 Nov 2020.

Vancouver:

Árvai K. Új lehetőségek a valós idejű PCR alkalmazására a molekuláris diagnosztikában . [Internet] [Thesis]. University of Debrecen; 2011. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2437/107474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Árvai K. Új lehetőségek a valós idejű PCR alkalmazására a molekuláris diagnosztikában . [Thesis]. University of Debrecen; 2011. Available from: http://hdl.handle.net/2437/107474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Debrecen

11. Kulcsár, Eszter. Az ErbB1 receptor klaszterizációjának vizsgálata kvantum dot triexciton képalkotás segítségével .

Degree: DE – TEK – Természettudományi és Technológiai Kar – Kémiai Intézet, 2012, University of Debrecen

 Bár a tirozinkináz receptorok aktivációjának jellegzetes molekuláris mechanizmusa szerint a szignalizáció kiváltása receptor-ligand kölcsönhatás indukálta receptordimerizációval történik, az ErbB fehérjék (ErbB1, ErbB2, ErbB3, ErbB4) esetében… (more)

Subjects/Keywords: ErbB; ErbB1; EGFR; QDTI

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APA (6th Edition):

Kulcsár, E. (2012). Az ErbB1 receptor klaszterizációjának vizsgálata kvantum dot triexciton képalkotás segítségével . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/128488

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kulcsár, Eszter. “Az ErbB1 receptor klaszterizációjának vizsgálata kvantum dot triexciton képalkotás segítségével .” 2012. Thesis, University of Debrecen. Accessed November 26, 2020. http://hdl.handle.net/2437/128488.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kulcsár, Eszter. “Az ErbB1 receptor klaszterizációjának vizsgálata kvantum dot triexciton képalkotás segítségével .” 2012. Web. 26 Nov 2020.

Vancouver:

Kulcsár E. Az ErbB1 receptor klaszterizációjának vizsgálata kvantum dot triexciton képalkotás segítségével . [Internet] [Thesis]. University of Debrecen; 2012. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2437/128488.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kulcsár E. Az ErbB1 receptor klaszterizációjának vizsgálata kvantum dot triexciton képalkotás segítségével . [Thesis]. University of Debrecen; 2012. Available from: http://hdl.handle.net/2437/128488

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loughborough University

12. Bu, Yubai. New synthetic routes to nitrogen heterocycles : natural products and novel drug scaffolds.

Degree: PhD, 2017, Loughborough University

This thesis is divided into three main sections. The first chapter contains a brief review of nitrogen heterocyclic chemistry. The second chapter reports the results and their discussion of new heterocyclic chemistry, and the experimental details are provided in the fourth chapter.

Subjects/Keywords: 547; Guanosine; Triazine; EGFR

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APA (6th Edition):

Bu, Y. (2017). New synthetic routes to nitrogen heterocycles : natural products and novel drug scaffolds. (Doctoral Dissertation). Loughborough University. Retrieved from http://hdl.handle.net/2134/27482

Chicago Manual of Style (16th Edition):

Bu, Yubai. “New synthetic routes to nitrogen heterocycles : natural products and novel drug scaffolds.” 2017. Doctoral Dissertation, Loughborough University. Accessed November 26, 2020. http://hdl.handle.net/2134/27482.

MLA Handbook (7th Edition):

Bu, Yubai. “New synthetic routes to nitrogen heterocycles : natural products and novel drug scaffolds.” 2017. Web. 26 Nov 2020.

Vancouver:

Bu Y. New synthetic routes to nitrogen heterocycles : natural products and novel drug scaffolds. [Internet] [Doctoral dissertation]. Loughborough University; 2017. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2134/27482.

Council of Science Editors:

Bu Y. New synthetic routes to nitrogen heterocycles : natural products and novel drug scaffolds. [Doctoral Dissertation]. Loughborough University; 2017. Available from: http://hdl.handle.net/2134/27482


University of Arizona

13. Hart, Matthew Robert. Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer .

Degree: 2013, University of Arizona

 Much of what is known about the role of the ERBB family in cellular biology and in cancer has to do with canonical downstream signaling… (more)

Subjects/Keywords: EGFR; Peptide; Ubiquitin; Genetics; Cancer

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APA (6th Edition):

Hart, M. R. (2013). Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/293476

Chicago Manual of Style (16th Edition):

Hart, Matthew Robert. “Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer .” 2013. Doctoral Dissertation, University of Arizona. Accessed November 26, 2020. http://hdl.handle.net/10150/293476.

MLA Handbook (7th Edition):

Hart, Matthew Robert. “Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer .” 2013. Web. 26 Nov 2020.

Vancouver:

Hart MR. Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/10150/293476.

Council of Science Editors:

Hart MR. Understanding EGFR Modification, Trafficking, and the Importance of its Juxtamembrane Domain in Cancer . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/293476


University of New South Wales

14. Wang, Xiaochun. Overcoming resistance of targeted EGFR/panHER monotherapy by inhibition of STAT3 escape pathway in sarcoma.

Degree: Clinical School - Prince of Wales Hospital, 2014, University of New South Wales

 Sarcoma, a malignancy of mesenchymal origin, is primarily treated by surgery, augmented with chemo- and radiotherapy. The 5-year survival for metastatic sarcoma is 16%. Although… (more)

Subjects/Keywords: EGFR; Sarcoma; STAT3; Targeted therapy

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APA (6th Edition):

Wang, X. (2014). Overcoming resistance of targeted EGFR/panHER monotherapy by inhibition of STAT3 escape pathway in sarcoma. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/54131 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:13255/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Wang, Xiaochun. “Overcoming resistance of targeted EGFR/panHER monotherapy by inhibition of STAT3 escape pathway in sarcoma.” 2014. Doctoral Dissertation, University of New South Wales. Accessed November 26, 2020. http://handle.unsw.edu.au/1959.4/54131 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:13255/SOURCE02?view=true.

MLA Handbook (7th Edition):

Wang, Xiaochun. “Overcoming resistance of targeted EGFR/panHER monotherapy by inhibition of STAT3 escape pathway in sarcoma.” 2014. Web. 26 Nov 2020.

Vancouver:

Wang X. Overcoming resistance of targeted EGFR/panHER monotherapy by inhibition of STAT3 escape pathway in sarcoma. [Internet] [Doctoral dissertation]. University of New South Wales; 2014. [cited 2020 Nov 26]. Available from: http://handle.unsw.edu.au/1959.4/54131 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:13255/SOURCE02?view=true.

Council of Science Editors:

Wang X. Overcoming resistance of targeted EGFR/panHER monotherapy by inhibition of STAT3 escape pathway in sarcoma. [Doctoral Dissertation]. University of New South Wales; 2014. Available from: http://handle.unsw.edu.au/1959.4/54131 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:13255/SOURCE02?view=true


University of New South Wales

15. Luk, Peter Ping-Kit. The influence of the EGFR pathway in modulating the efficacy of gemcitabine.

Degree: Clinical School - St George Hospital, 2011, University of New South Wales

 Multiple anti-cancer drugs are often combined to increase their effectiveness. This study investigates the combination between an epidermal growth factor receptor (EGFR) inhibitor and gemcitabine,… (more)

Subjects/Keywords: Lung cancer; Gemcitabine; EGFR

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APA (6th Edition):

Luk, P. P. (2011). The influence of the EGFR pathway in modulating the efficacy of gemcitabine. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/50384 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9271/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Luk, Peter Ping-Kit. “The influence of the EGFR pathway in modulating the efficacy of gemcitabine.” 2011. Doctoral Dissertation, University of New South Wales. Accessed November 26, 2020. http://handle.unsw.edu.au/1959.4/50384 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9271/SOURCE02?view=true.

MLA Handbook (7th Edition):

Luk, Peter Ping-Kit. “The influence of the EGFR pathway in modulating the efficacy of gemcitabine.” 2011. Web. 26 Nov 2020.

Vancouver:

Luk PP. The influence of the EGFR pathway in modulating the efficacy of gemcitabine. [Internet] [Doctoral dissertation]. University of New South Wales; 2011. [cited 2020 Nov 26]. Available from: http://handle.unsw.edu.au/1959.4/50384 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9271/SOURCE02?view=true.

Council of Science Editors:

Luk PP. The influence of the EGFR pathway in modulating the efficacy of gemcitabine. [Doctoral Dissertation]. University of New South Wales; 2011. Available from: http://handle.unsw.edu.au/1959.4/50384 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9271/SOURCE02?view=true

16. Al-Akhrass, Hussein. Un rôle inédit de la sortiline dans le contrôle du transport rétrograde de l'EGFR pour limiter la croissance tumorale : A new role of sortilin in the control of EGFR retrograde trafficking to limittumour growth.

Degree: Docteur es, Cancérologie, 2017, Limoges

Le cancer du poumon est le troisième cancer le plus fréquent chez les femmes et le deuxième chez les hommes, il est la cause principale… (more)

Subjects/Keywords: Cancer; Signalisation; EGFR; Sortiline; Cancer; Signalling; EGFR; Sortilin; 616.994

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APA (6th Edition):

Al-Akhrass, H. (2017). Un rôle inédit de la sortiline dans le contrôle du transport rétrograde de l'EGFR pour limiter la croissance tumorale : A new role of sortilin in the control of EGFR retrograde trafficking to limittumour growth. (Doctoral Dissertation). Limoges. Retrieved from http://www.theses.fr/2017LIMO0035

Chicago Manual of Style (16th Edition):

Al-Akhrass, Hussein. “Un rôle inédit de la sortiline dans le contrôle du transport rétrograde de l'EGFR pour limiter la croissance tumorale : A new role of sortilin in the control of EGFR retrograde trafficking to limittumour growth.” 2017. Doctoral Dissertation, Limoges. Accessed November 26, 2020. http://www.theses.fr/2017LIMO0035.

MLA Handbook (7th Edition):

Al-Akhrass, Hussein. “Un rôle inédit de la sortiline dans le contrôle du transport rétrograde de l'EGFR pour limiter la croissance tumorale : A new role of sortilin in the control of EGFR retrograde trafficking to limittumour growth.” 2017. Web. 26 Nov 2020.

Vancouver:

Al-Akhrass H. Un rôle inédit de la sortiline dans le contrôle du transport rétrograde de l'EGFR pour limiter la croissance tumorale : A new role of sortilin in the control of EGFR retrograde trafficking to limittumour growth. [Internet] [Doctoral dissertation]. Limoges; 2017. [cited 2020 Nov 26]. Available from: http://www.theses.fr/2017LIMO0035.

Council of Science Editors:

Al-Akhrass H. Un rôle inédit de la sortiline dans le contrôle du transport rétrograde de l'EGFR pour limiter la croissance tumorale : A new role of sortilin in the control of EGFR retrograde trafficking to limittumour growth. [Doctoral Dissertation]. Limoges; 2017. Available from: http://www.theses.fr/2017LIMO0035

17. Yammine, Lucie. La Lipocaline 2 : de son implication dans la régulation du transport endosomal de l'EGFR à la caractérisation fonctionnelle de son interactome : Lipocalin 2 : role in EGFR endosomal trafficking and physiological implications of its interactome.

Degree: Docteur es, Biologie cellulaire, 2018, Sorbonne Paris Cité

La maladie rénale chronique (MRC) se caractérise par des lésions rénales aboutissant à une perte fonctionnelle des néphrons. Une activation du récepteur transmembranaire Epidermal Growth… (more)

Subjects/Keywords: Lipocaline 2; Egfr; Endocytose; Recyclage; Lipocalin 2; Egfr; Endocytosis; Recycling; 616.614

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APA (6th Edition):

Yammine, L. (2018). La Lipocaline 2 : de son implication dans la régulation du transport endosomal de l'EGFR à la caractérisation fonctionnelle de son interactome : Lipocalin 2 : role in EGFR endosomal trafficking and physiological implications of its interactome. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2018USPCB027

Chicago Manual of Style (16th Edition):

Yammine, Lucie. “La Lipocaline 2 : de son implication dans la régulation du transport endosomal de l'EGFR à la caractérisation fonctionnelle de son interactome : Lipocalin 2 : role in EGFR endosomal trafficking and physiological implications of its interactome.” 2018. Doctoral Dissertation, Sorbonne Paris Cité. Accessed November 26, 2020. http://www.theses.fr/2018USPCB027.

MLA Handbook (7th Edition):

Yammine, Lucie. “La Lipocaline 2 : de son implication dans la régulation du transport endosomal de l'EGFR à la caractérisation fonctionnelle de son interactome : Lipocalin 2 : role in EGFR endosomal trafficking and physiological implications of its interactome.” 2018. Web. 26 Nov 2020.

Vancouver:

Yammine L. La Lipocaline 2 : de son implication dans la régulation du transport endosomal de l'EGFR à la caractérisation fonctionnelle de son interactome : Lipocalin 2 : role in EGFR endosomal trafficking and physiological implications of its interactome. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2018. [cited 2020 Nov 26]. Available from: http://www.theses.fr/2018USPCB027.

Council of Science Editors:

Yammine L. La Lipocaline 2 : de son implication dans la régulation du transport endosomal de l'EGFR à la caractérisation fonctionnelle de son interactome : Lipocalin 2 : role in EGFR endosomal trafficking and physiological implications of its interactome. [Doctoral Dissertation]. Sorbonne Paris Cité; 2018. Available from: http://www.theses.fr/2018USPCB027


University of Arizona

18. Su, Hsin-Yuan. Therapeutic Potential of EGFR Derived Peptides in Breast Cancer .

Degree: 2013, University of Arizona

 The epidermal growth factor receptor (EGFR) belongs to the erbB family of receptor tyrosine kinases which consists of four members (EGFR, ErbB2, ErbB3 and ErbB4).… (more)

Subjects/Keywords: EGFR; juxtamembrane domain; MUC1; nuclear EGFR; peptide; Cancer Biology; breast cancer

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APA (6th Edition):

Su, H. (2013). Therapeutic Potential of EGFR Derived Peptides in Breast Cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/293486

Chicago Manual of Style (16th Edition):

Su, Hsin-Yuan. “Therapeutic Potential of EGFR Derived Peptides in Breast Cancer .” 2013. Doctoral Dissertation, University of Arizona. Accessed November 26, 2020. http://hdl.handle.net/10150/293486.

MLA Handbook (7th Edition):

Su, Hsin-Yuan. “Therapeutic Potential of EGFR Derived Peptides in Breast Cancer .” 2013. Web. 26 Nov 2020.

Vancouver:

Su H. Therapeutic Potential of EGFR Derived Peptides in Breast Cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/10150/293486.

Council of Science Editors:

Su H. Therapeutic Potential of EGFR Derived Peptides in Breast Cancer . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/293486


University of Sydney

19. Elmaghrabi, Yasmin Ahmed Mohamed Soliman. The tumor suppressor Annexin A6 increases the sensitivity towards anti-cancer drugs targeting the EGFR/Ras/MAPK pathway .

Degree: 2016, University of Sydney

 Sustained EGFR/Ras/MAPK signaling is associated with various cancers. Hence, blocking EGFR and its downstream effectors has become an established target in anti-cancer therapeutics. However, targeted… (more)

Subjects/Keywords: Annexin A6; Cancer; EGFR; Biomarker; EGFR/Ras/MAPK Pathway

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APA (6th Edition):

Elmaghrabi, Y. A. M. S. (2016). The tumor suppressor Annexin A6 increases the sensitivity towards anti-cancer drugs targeting the EGFR/Ras/MAPK pathway . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/15335

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Elmaghrabi, Yasmin Ahmed Mohamed Soliman. “The tumor suppressor Annexin A6 increases the sensitivity towards anti-cancer drugs targeting the EGFR/Ras/MAPK pathway .” 2016. Thesis, University of Sydney. Accessed November 26, 2020. http://hdl.handle.net/2123/15335.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Elmaghrabi, Yasmin Ahmed Mohamed Soliman. “The tumor suppressor Annexin A6 increases the sensitivity towards anti-cancer drugs targeting the EGFR/Ras/MAPK pathway .” 2016. Web. 26 Nov 2020.

Vancouver:

Elmaghrabi YAMS. The tumor suppressor Annexin A6 increases the sensitivity towards anti-cancer drugs targeting the EGFR/Ras/MAPK pathway . [Internet] [Thesis]. University of Sydney; 2016. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2123/15335.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Elmaghrabi YAMS. The tumor suppressor Annexin A6 increases the sensitivity towards anti-cancer drugs targeting the EGFR/Ras/MAPK pathway . [Thesis]. University of Sydney; 2016. Available from: http://hdl.handle.net/2123/15335

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Zhu, Huijun. A radioiodinated quinazolin derivative can predict radiosensitivity in non-small cell lung cancer : 放射性ヨウ素標識キナゾリン誘導体は非小細胞肺癌において放射線感受性を予測しうる.

Degree: 博士(医学), 2013, Hamamatsu University School of Medicine / 浜松医科大学

ObjectiveThe aim of this study was to determine whether the uptake of radioiodinated6-(3-morpholinopropoxy)-7-ethoxy-4-(3′-iodophenoxy) quinazoline ([125I]PYK) can predict the response to radiotherapy of NSCLC cells in… (more)

Subjects/Keywords: [125I]PYK; irradiation; gefitinib; EGFR; NSCLC

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APA (6th Edition):

Zhu, H. (2013). A radioiodinated quinazolin derivative can predict radiosensitivity in non-small cell lung cancer : 放射性ヨウ素標識キナゾリン誘導体は非小細胞肺癌において放射線感受性を予測しうる. (Thesis). Hamamatsu University School of Medicine / 浜松医科大学. Retrieved from http://hdl.handle.net/10271/2767

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhu, Huijun. “A radioiodinated quinazolin derivative can predict radiosensitivity in non-small cell lung cancer : 放射性ヨウ素標識キナゾリン誘導体は非小細胞肺癌において放射線感受性を予測しうる.” 2013. Thesis, Hamamatsu University School of Medicine / 浜松医科大学. Accessed November 26, 2020. http://hdl.handle.net/10271/2767.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhu, Huijun. “A radioiodinated quinazolin derivative can predict radiosensitivity in non-small cell lung cancer : 放射性ヨウ素標識キナゾリン誘導体は非小細胞肺癌において放射線感受性を予測しうる.” 2013. Web. 26 Nov 2020.

Vancouver:

Zhu H. A radioiodinated quinazolin derivative can predict radiosensitivity in non-small cell lung cancer : 放射性ヨウ素標識キナゾリン誘導体は非小細胞肺癌において放射線感受性を予測しうる. [Internet] [Thesis]. Hamamatsu University School of Medicine / 浜松医科大学; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/10271/2767.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhu H. A radioiodinated quinazolin derivative can predict radiosensitivity in non-small cell lung cancer : 放射性ヨウ素標識キナゾリン誘導体は非小細胞肺癌において放射線感受性を予測しうる. [Thesis]. Hamamatsu University School of Medicine / 浜松医科大学; 2013. Available from: http://hdl.handle.net/10271/2767

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

21. Kim, Christine. Assessing the role of arsenite in disrupting the EGFR signaling axis.

Degree: MS, 2018, University of Louisville

  The epidermal growth factor receptor (EGFR) is a receptor tyrosine kinase localized on the cell surface. Overexpression of EGFR has been used as biomarkers… (more)

Subjects/Keywords: arsenic; EGFR; metal; trafficking; Medical Toxicology

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APA (6th Edition):

Kim, C. (2018). Assessing the role of arsenite in disrupting the EGFR signaling axis. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/3131 ; https://ir.library.louisville.edu/etd/3131

Chicago Manual of Style (16th Edition):

Kim, Christine. “Assessing the role of arsenite in disrupting the EGFR signaling axis.” 2018. Masters Thesis, University of Louisville. Accessed November 26, 2020. 10.18297/etd/3131 ; https://ir.library.louisville.edu/etd/3131.

MLA Handbook (7th Edition):

Kim, Christine. “Assessing the role of arsenite in disrupting the EGFR signaling axis.” 2018. Web. 26 Nov 2020.

Vancouver:

Kim C. Assessing the role of arsenite in disrupting the EGFR signaling axis. [Internet] [Masters thesis]. University of Louisville; 2018. [cited 2020 Nov 26]. Available from: 10.18297/etd/3131 ; https://ir.library.louisville.edu/etd/3131.

Council of Science Editors:

Kim C. Assessing the role of arsenite in disrupting the EGFR signaling axis. [Masters Thesis]. University of Louisville; 2018. Available from: 10.18297/etd/3131 ; https://ir.library.louisville.edu/etd/3131


Vanderbilt University

22. Nebhan, Caroline Amalia. Acquired Resistance to Targeted Therapy in EGFR-Mutant Lung Adenocarcinoma.

Degree: PhD, Cancer Biology, 2014, Vanderbilt University

 Lung cancer is the leading cause of cancer-related death in the United States and worldwide. Lung cancers that are driven by mutations in the Epidermal… (more)

Subjects/Keywords: acquired resistance; EGFR; targeted therapy; lung cancer

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APA (6th Edition):

Nebhan, C. A. (2014). Acquired Resistance to Targeted Therapy in EGFR-Mutant Lung Adenocarcinoma. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14054

Chicago Manual of Style (16th Edition):

Nebhan, Caroline Amalia. “Acquired Resistance to Targeted Therapy in EGFR-Mutant Lung Adenocarcinoma.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed November 26, 2020. http://hdl.handle.net/1803/14054.

MLA Handbook (7th Edition):

Nebhan, Caroline Amalia. “Acquired Resistance to Targeted Therapy in EGFR-Mutant Lung Adenocarcinoma.” 2014. Web. 26 Nov 2020.

Vancouver:

Nebhan CA. Acquired Resistance to Targeted Therapy in EGFR-Mutant Lung Adenocarcinoma. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1803/14054.

Council of Science Editors:

Nebhan CA. Acquired Resistance to Targeted Therapy in EGFR-Mutant Lung Adenocarcinoma. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/14054


Vanderbilt University

23. Hardbower, Dana Michelle. Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis.

Degree: PhD, Microbiology and Immunology, 2017, Vanderbilt University

 Macrophages represent a dynamic and plastic subset of the innate immune system. Macrophage functions include immune surveillance and clearance of pathogens, but they have also… (more)

Subjects/Keywords: Macrophage; EGFR; ODC; Helicobacter pylori; Macrophage Activation

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APA (6th Edition):

Hardbower, D. M. (2017). Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10774

Chicago Manual of Style (16th Edition):

Hardbower, Dana Michelle. “Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed November 26, 2020. http://hdl.handle.net/1803/10774.

MLA Handbook (7th Edition):

Hardbower, Dana Michelle. “Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis.” 2017. Web. 26 Nov 2020.

Vancouver:

Hardbower DM. Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1803/10774.

Council of Science Editors:

Hardbower DM. Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/10774


Stellenbosch University

24. Van der Westhuyzen, Aletta Elizabeth. Synthesis of novel staurosporine analogues as potential kinase inhibitors.

Degree: MSc, Chemistry and Polymer Science, 2015, Stellenbosch University

ENGLISH ABSTRACT: Protein kinases are enzymes that promote phosphorylation – transferring a phosphate group from ATP to a substrate protein. Due to the central involvement… (more)

Subjects/Keywords: Protein kinases; EGFR; Cancer therapy; UCTD; Phosphorylation

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APA (6th Edition):

Van der Westhuyzen, A. E. (2015). Synthesis of novel staurosporine analogues as potential kinase inhibitors. (Masters Thesis). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/96632

Chicago Manual of Style (16th Edition):

Van der Westhuyzen, Aletta Elizabeth. “Synthesis of novel staurosporine analogues as potential kinase inhibitors.” 2015. Masters Thesis, Stellenbosch University. Accessed November 26, 2020. http://hdl.handle.net/10019.1/96632.

MLA Handbook (7th Edition):

Van der Westhuyzen, Aletta Elizabeth. “Synthesis of novel staurosporine analogues as potential kinase inhibitors.” 2015. Web. 26 Nov 2020.

Vancouver:

Van der Westhuyzen AE. Synthesis of novel staurosporine analogues as potential kinase inhibitors. [Internet] [Masters thesis]. Stellenbosch University; 2015. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/10019.1/96632.

Council of Science Editors:

Van der Westhuyzen AE. Synthesis of novel staurosporine analogues as potential kinase inhibitors. [Masters Thesis]. Stellenbosch University; 2015. Available from: http://hdl.handle.net/10019.1/96632

25. Ibach, Jenny. Single molecule imaging of the signaling activity of the epidermal growth factor receptor.

Degree: 2014, Technische Universität Dortmund

 SUMMARY The epidermal growth factor receptor (ErbB1) is a receptor tyrosine kinase involved in various cellular processes, such as growth and differentiation. Its extracellular domains… (more)

Subjects/Keywords: EGFR; Fluoreszenzmikroskopie; Single particle tracking; 570; 540

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APA (6th Edition):

Ibach, J. (2014). Single molecule imaging of the signaling activity of the epidermal growth factor receptor. (Doctoral Dissertation). Technische Universität Dortmund. Retrieved from http://dx.doi.org/10.17877/DE290R-649

Chicago Manual of Style (16th Edition):

Ibach, Jenny. “Single molecule imaging of the signaling activity of the epidermal growth factor receptor.” 2014. Doctoral Dissertation, Technische Universität Dortmund. Accessed November 26, 2020. http://dx.doi.org/10.17877/DE290R-649.

MLA Handbook (7th Edition):

Ibach, Jenny. “Single molecule imaging of the signaling activity of the epidermal growth factor receptor.” 2014. Web. 26 Nov 2020.

Vancouver:

Ibach J. Single molecule imaging of the signaling activity of the epidermal growth factor receptor. [Internet] [Doctoral dissertation]. Technische Universität Dortmund; 2014. [cited 2020 Nov 26]. Available from: http://dx.doi.org/10.17877/DE290R-649.

Council of Science Editors:

Ibach J. Single molecule imaging of the signaling activity of the epidermal growth factor receptor. [Doctoral Dissertation]. Technische Universität Dortmund; 2014. Available from: http://dx.doi.org/10.17877/DE290R-649


Texas Medical Center

26. Lee, Hong-Jen. TYROSINE 370 PHOSPHORYLATION OF ATM POSITIVELY REGULATES DNA DAMAGE RESPONSE.

Degree: PhD, 2014, Texas Medical Center

  Ataxia telangiectasia-mutated (ATM) mediates DNA damage response by controlling irradiation (IR)-induced foci formation, cell cycle checkpoint, and apoptosis. However, how upstream signaling regulates ATM… (more)

Subjects/Keywords: EGFR; ATM; DNA Damage Response; Cancer Biology

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APA (6th Edition):

Lee, H. (2014). TYROSINE 370 PHOSPHORYLATION OF ATM POSITIVELY REGULATES DNA DAMAGE RESPONSE. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/487

Chicago Manual of Style (16th Edition):

Lee, Hong-Jen. “TYROSINE 370 PHOSPHORYLATION OF ATM POSITIVELY REGULATES DNA DAMAGE RESPONSE.” 2014. Doctoral Dissertation, Texas Medical Center. Accessed November 26, 2020. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/487.

MLA Handbook (7th Edition):

Lee, Hong-Jen. “TYROSINE 370 PHOSPHORYLATION OF ATM POSITIVELY REGULATES DNA DAMAGE RESPONSE.” 2014. Web. 26 Nov 2020.

Vancouver:

Lee H. TYROSINE 370 PHOSPHORYLATION OF ATM POSITIVELY REGULATES DNA DAMAGE RESPONSE. [Internet] [Doctoral dissertation]. Texas Medical Center; 2014. [cited 2020 Nov 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/487.

Council of Science Editors:

Lee H. TYROSINE 370 PHOSPHORYLATION OF ATM POSITIVELY REGULATES DNA DAMAGE RESPONSE. [Doctoral Dissertation]. Texas Medical Center; 2014. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/487

27. Chrétien, Anne-Sophie. Fonctionnalité de la signalisation en aval des récepteurs HER : implication dans la réponse cellulaire et tumorale aux thérapies ciblées : Human Epidermal Growth Factor Receptor (HER) downstream signalling functionality : implications in cell and tumour response to targeted therapies.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2011, Université Henri Poincaré – Nancy I

Les thérapies ciblées dirigées contre les récepteurs de la famille des HER représentent un progrès majeur en cancérologie. Leur efficacité repose sur leur capacité à… (more)

Subjects/Keywords: Cancer; Cétuximab; Panitumumab; EGFR; Signalisation cellulaire

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APA (6th Edition):

Chrétien, A. (2011). Fonctionnalité de la signalisation en aval des récepteurs HER : implication dans la réponse cellulaire et tumorale aux thérapies ciblées : Human Epidermal Growth Factor Receptor (HER) downstream signalling functionality : implications in cell and tumour response to targeted therapies. (Doctoral Dissertation). Université Henri Poincaré – Nancy I. Retrieved from http://www.theses.fr/2011NAN10050

Chicago Manual of Style (16th Edition):

Chrétien, Anne-Sophie. “Fonctionnalité de la signalisation en aval des récepteurs HER : implication dans la réponse cellulaire et tumorale aux thérapies ciblées : Human Epidermal Growth Factor Receptor (HER) downstream signalling functionality : implications in cell and tumour response to targeted therapies.” 2011. Doctoral Dissertation, Université Henri Poincaré – Nancy I. Accessed November 26, 2020. http://www.theses.fr/2011NAN10050.

MLA Handbook (7th Edition):

Chrétien, Anne-Sophie. “Fonctionnalité de la signalisation en aval des récepteurs HER : implication dans la réponse cellulaire et tumorale aux thérapies ciblées : Human Epidermal Growth Factor Receptor (HER) downstream signalling functionality : implications in cell and tumour response to targeted therapies.” 2011. Web. 26 Nov 2020.

Vancouver:

Chrétien A. Fonctionnalité de la signalisation en aval des récepteurs HER : implication dans la réponse cellulaire et tumorale aux thérapies ciblées : Human Epidermal Growth Factor Receptor (HER) downstream signalling functionality : implications in cell and tumour response to targeted therapies. [Internet] [Doctoral dissertation]. Université Henri Poincaré – Nancy I; 2011. [cited 2020 Nov 26]. Available from: http://www.theses.fr/2011NAN10050.

Council of Science Editors:

Chrétien A. Fonctionnalité de la signalisation en aval des récepteurs HER : implication dans la réponse cellulaire et tumorale aux thérapies ciblées : Human Epidermal Growth Factor Receptor (HER) downstream signalling functionality : implications in cell and tumour response to targeted therapies. [Doctoral Dissertation]. Université Henri Poincaré – Nancy I; 2011. Available from: http://www.theses.fr/2011NAN10050

28. Fengler, Sven. Spatial-temporal regulation of EGFR phosporylation by protein tyrosine phosphatases.

Degree: 2017, Technische Universität Dortmund

 The activity of the epidermal growth factor receptor (EGFR) and its interactions with protein tyrosine phosphatases (PTPs) is what determines growth factor signaling. Due to… (more)

Subjects/Keywords: Cell-array; EGFR; PTP; CA-FLIM; 570

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fengler, S. (2017). Spatial-temporal regulation of EGFR phosporylation by protein tyrosine phosphatases. (Doctoral Dissertation). Technische Universität Dortmund. Retrieved from http://dx.doi.org/10.17877/DE290R-18025

Chicago Manual of Style (16th Edition):

Fengler, Sven. “Spatial-temporal regulation of EGFR phosporylation by protein tyrosine phosphatases.” 2017. Doctoral Dissertation, Technische Universität Dortmund. Accessed November 26, 2020. http://dx.doi.org/10.17877/DE290R-18025.

MLA Handbook (7th Edition):

Fengler, Sven. “Spatial-temporal regulation of EGFR phosporylation by protein tyrosine phosphatases.” 2017. Web. 26 Nov 2020.

Vancouver:

Fengler S. Spatial-temporal regulation of EGFR phosporylation by protein tyrosine phosphatases. [Internet] [Doctoral dissertation]. Technische Universität Dortmund; 2017. [cited 2020 Nov 26]. Available from: http://dx.doi.org/10.17877/DE290R-18025.

Council of Science Editors:

Fengler S. Spatial-temporal regulation of EGFR phosporylation by protein tyrosine phosphatases. [Doctoral Dissertation]. Technische Universität Dortmund; 2017. Available from: http://dx.doi.org/10.17877/DE290R-18025

29. Wernicke, Julia. Regulation of EGF receptor activation by phosphohistidine phosphatase PHPT1.

Degree: 2017, Technische Universität Dortmund

 Phosphorylation of polybasic stretches in membrane-associated proteins creates an electrostatic switch influencing their interactions with membrane lipids. Such modification affects their localization, conformational fluctuations and… (more)

Subjects/Keywords: EGFR Activation; Phosphohistidine; Phosphatase; PHPT1; 570; 540

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wernicke, J. (2017). Regulation of EGF receptor activation by phosphohistidine phosphatase PHPT1. (Doctoral Dissertation). Technische Universität Dortmund. Retrieved from http://dx.doi.org/10.17877/DE290R-18048

Chicago Manual of Style (16th Edition):

Wernicke, Julia. “Regulation of EGF receptor activation by phosphohistidine phosphatase PHPT1.” 2017. Doctoral Dissertation, Technische Universität Dortmund. Accessed November 26, 2020. http://dx.doi.org/10.17877/DE290R-18048.

MLA Handbook (7th Edition):

Wernicke, Julia. “Regulation of EGF receptor activation by phosphohistidine phosphatase PHPT1.” 2017. Web. 26 Nov 2020.

Vancouver:

Wernicke J. Regulation of EGF receptor activation by phosphohistidine phosphatase PHPT1. [Internet] [Doctoral dissertation]. Technische Universität Dortmund; 2017. [cited 2020 Nov 26]. Available from: http://dx.doi.org/10.17877/DE290R-18048.

Council of Science Editors:

Wernicke J. Regulation of EGF receptor activation by phosphohistidine phosphatase PHPT1. [Doctoral Dissertation]. Technische Universität Dortmund; 2017. Available from: http://dx.doi.org/10.17877/DE290R-18048

30. Garnier, Eugenie. Expression du Facteur XII dans le système nerveux central et son rôle dans l'apoptose neuronale : Expression of Factor XII in the central nervous system and its role in neuronal apoptosis.

Degree: Docteur es, Aspects moleculaires et cellulaires de la biologie, 2018, Normandie

Le facteur XII (FXII) est une sérine protéase de 80 kDa produite et sécrétée par le foie qui initie la voie intrinsèque de la coagulation.… (more)

Subjects/Keywords: Facteur XII; Neurones; EGFR; MET; Apoptosis; Neurons

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Garnier, E. (2018). Expression du Facteur XII dans le système nerveux central et son rôle dans l'apoptose neuronale : Expression of Factor XII in the central nervous system and its role in neuronal apoptosis. (Doctoral Dissertation). Normandie. Retrieved from http://www.theses.fr/2018NORMC413

Chicago Manual of Style (16th Edition):

Garnier, Eugenie. “Expression du Facteur XII dans le système nerveux central et son rôle dans l'apoptose neuronale : Expression of Factor XII in the central nervous system and its role in neuronal apoptosis.” 2018. Doctoral Dissertation, Normandie. Accessed November 26, 2020. http://www.theses.fr/2018NORMC413.

MLA Handbook (7th Edition):

Garnier, Eugenie. “Expression du Facteur XII dans le système nerveux central et son rôle dans l'apoptose neuronale : Expression of Factor XII in the central nervous system and its role in neuronal apoptosis.” 2018. Web. 26 Nov 2020.

Vancouver:

Garnier E. Expression du Facteur XII dans le système nerveux central et son rôle dans l'apoptose neuronale : Expression of Factor XII in the central nervous system and its role in neuronal apoptosis. [Internet] [Doctoral dissertation]. Normandie; 2018. [cited 2020 Nov 26]. Available from: http://www.theses.fr/2018NORMC413.

Council of Science Editors:

Garnier E. Expression du Facteur XII dans le système nerveux central et son rôle dans l'apoptose neuronale : Expression of Factor XII in the central nervous system and its role in neuronal apoptosis. [Doctoral Dissertation]. Normandie; 2018. Available from: http://www.theses.fr/2018NORMC413

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