subject:(ECT2)

NSYSU

1. Bao, Han-lin. The effects of GTPase related proteins in vascular development in zebrafish.

Degree: Master, Biological Sciences, 2016, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0723116-163853

► Genetic programs and signaling pathways are required for proper growth and patterning of blood vessels. Zebrafish is a powerful vertebrate model organism to study genetic… (more)

Subjects/Keywords: zebrafish; sept8b; gtpbp1l; ISV; CVP; GTPase; rhoub; ect2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Bao, H. (2016). The effects of GTPase related proteins in vascular development in zebrafish. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0723116-163853

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Bao, Han-lin. “The effects of GTPase related proteins in vascular development in zebrafish.” 2016. Thesis, NSYSU. Accessed March 08, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0723116-163853.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Bao, Han-lin. “The effects of GTPase related proteins in vascular development in zebrafish.” 2016. Web. 08 Mar 2021.

Vancouver:

Bao H. The effects of GTPase related proteins in vascular development in zebrafish. [Internet] [Thesis]. NSYSU; 2016. [cited 2021 Mar 08]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0723116-163853.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bao H. The effects of GTPase related proteins in vascular development in zebrafish. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0723116-163853

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

2. Zhou, Jun-Qing. Study the function of GTPase related proteins wasf1 and ect2 in zebrafish vascular development.

Degree: Master, Biological Sciences, 2018, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727118-130736

► Genetic programs and signaling pathways are required for proper growth and patterning of blood vessels. Zebrafish is a powerful vertebrate model organism to study genetic… (more)

Subjects/Keywords: angiogenesis; zebrafish; ect2; wasf1; VEGF; BMP; ISV; CVP

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Zhou, J. (2018). Study the function of GTPase related proteins wasf1 and ect2 in zebrafish vascular development. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727118-130736

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Zhou, Jun-Qing. “Study the function of GTPase related proteins wasf1 and ect2 in zebrafish vascular development.” 2018. Thesis, NSYSU. Accessed March 08, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727118-130736.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Zhou, Jun-Qing. “Study the function of GTPase related proteins wasf1 and ect2 in zebrafish vascular development.” 2018. Web. 08 Mar 2021.

Vancouver:

Zhou J. Study the function of GTPase related proteins wasf1 and ect2 in zebrafish vascular development. [Internet] [Thesis]. NSYSU; 2018. [cited 2021 Mar 08]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727118-130736.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhou J. Study the function of GTPase related proteins wasf1 and ect2 in zebrafish vascular development. [Thesis]. NSYSU; 2018. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727118-130736

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

University of Toronto

3. Weeks, Adrienne Christine. The Role Of ECT2 In Astroctyoma Invasion and Stress Granule Formation.

Degree: PhD, 2014, University of Toronto

URL: http://hdl.handle.net/1807/73778

► In this thesis, the oncogenic role of the cytokinetic RhoA guanine nucleotide exchange factor, ECT2 in astrocytoma is studied. ECT2 is overexpressed in astrocytoma cells,… (more)

▼ In this thesis, the oncogenic role of the cytokinetic RhoA guanine nucleotide exchange factor, ECT2 in astrocytoma is studied. ECT2 is overexpressed in astrocytoma cells, and correlates with poor patient prognosis and increasing astrocytoma grade. ECT2 is a nuclear protein in non- cancerous cells. Here, it is demonstrated that ECT2 is aberrantly located in the cytoplasm of astrocytoma cells at the leading edge of the cell where it activates Rac1 and Cdc42 but not RhoA. Interestingly, ECT2 overexpression results in mesenchymal-amoeboid transition with ECT2 localizing to the retraction bleb in amoeboid cells. Using ECT2 immunoprecipitation followed by mass spectrometry, I identified the novel ECT2 interactor, RASAL2. RASAL2 is a RasGap know to be an inhibitor of breast cancer growth and metastasis. SiRNA-mediated loss of RASAL2 leads to highly invasive astroctyoma cells with an amoeboid phenotype and increased RhoA activity. This suggests that RASAL2 may modulate ECT2 specificity toward critical Rho GTPases. I also demonstrate that ECT2 has a novel role in stress granule formation. Stress granule formation results in translational reorganization that allows cells to survive acute stress such as hypoxia and radiation. ECT2 colocalizes with stress granules and co-immunoprecipites TIAR under stress. SiRNA-mediated silencing of ECT2 results in increased sensitivity to cellular apoptosis and stress granules in a less mature peri-nuclear location, however stress granule number and size are unchanged. The upstream activator of the ECT2 pathway, AurkB, was then examined. I demonstrate for the first time that AurkB and its downstream target phospho- Histone 3 are phosphorylated with cellular stress. Chemical and siRNA mediated interference with AurkB phosphorylation leads to the formation of fewer stress granule of a smaller size and with less TIAR aggregation. Using RNA immunoprecipitation of stress granules, I show that iii mRNA from multiple cellular pathways are contained in astrocytoma stress granules. These pathways include those subserving cellular migration, cellular metabolism, cell death and post translational and transcriptional modifiers. Utilizing a doxycycline-inducible expressing siRNA system, I show that mice injected with ECT2 siRNA live longer, with fewer tumours and decreased invasion than mice injected with NS siRNA controls. Taken together, these data suggest that ECT2 is a putative astrocytoma oncogene that warrants further investigation as a therapeutic target in astrocytoma. Advisors/Committee Members: Rutka, James T, Laboratory Medicine and Pathobiology.

Subjects/Keywords: ECT2; Glioblastoma; Invasion; mesenchymal Amoeboid Transition; Stress granules; 0379

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Weeks, A. C. (2014). The Role Of ECT2 In Astroctyoma Invasion and Stress Granule Formation. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/73778

Chicago Manual of Style (16^th Edition):

Weeks, Adrienne Christine. “The Role Of ECT2 In Astroctyoma Invasion and Stress Granule Formation.” 2014. Doctoral Dissertation, University of Toronto. Accessed March 08, 2021. http://hdl.handle.net/1807/73778.

MLA Handbook (7^th Edition):

Weeks, Adrienne Christine. “The Role Of ECT2 In Astroctyoma Invasion and Stress Granule Formation.” 2014. Web. 08 Mar 2021.

Vancouver:

Weeks AC. The Role Of ECT2 In Astroctyoma Invasion and Stress Granule Formation. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1807/73778.

Council of Science Editors:

Weeks AC. The Role Of ECT2 In Astroctyoma Invasion and Stress Granule Formation. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/73778

University of South Florida

4. Smalley, Tracess B. The Role of aPKCs and aPKC Inhibitors in Cell Proliferation and Invasion in Breast and Ovarian Cancer.

Degree: 2020, University of South Florida

URL: https://scholarcommons.usf.edu/etd/8300

► Research has demonstrated that the atypical protein kinase C-zeta (PKC-ζ) is a component of many dysregulated pathways in breast and ovarian cancer, including cellular proliferation,… (more)

▼ Research has demonstrated that the atypical protein kinase C-zeta (PKC-ζ) is a component of many dysregulated pathways in breast and ovarian cancer, including cellular proliferation, survival, and cell cycle upregulation. Breast and ovarian cancers affect women every day and are second and fifth leading cause of cancer death. Women who seek treatments are commonly met with invasive surgeries or chemotherapy. Protein kinase C (PKC) is a family of serine and threonine phosphorylating kinases that have been shown to modulate and transduce signaling cascades that play roles in the development and survival of cancers. Atypical PKC (aPKC), have been heavily suggested to participate in phosphoinositide 3-kinase (PI3K) misregulated cancers (such as breast and ovarian) and inhibition of these pathway branches have demonstrated decreases in malignant behaviors. In our studies, we investigated (1) PKC-ζ protein in breast tissue to evaluate its potential as a biomarker for breast cancer invasion and (2) the effects of the atypical PKC-ζ inhibitor ζ-Stat on ovarian cell lines (TOV21G and ES-2) to determine the outcomes on proliferation and cellular invasion. In breast cancer tissues, we showed that an overexpression of PKC-ζ protein can be indicative of carcinogenesis. We also evaluated the invasive behavior of MDA-MB-231 breast cancer cells upon the knockdown of PKC-ζ with a Transwell invasion assay and an immunofluorescent probe for filamentous actin (F-actin) organization. In ovarian cancer, we utilized clear cell ovarian carcinoma cells (CCOC) because this subtype of ovarian cancer represents 5% of incidence, presents unique pathological features, has poor prognosis and has a high reoccurrence rate after treatment. Overall, our results showed that PKC-ζ is linked to proliferation and invasion in both breast and ovarian cancer. These results illustrate that PKC-ζ plays a role in invasion pathways through the Ras-related C3 botulinum toxin substrate 1 (Rac1) and Ras homolog gene family member A (RhoA). Additionally, our results showed that TOV21G tumor growth in athymic female mice was decreased when treated with the PKC-ζ inhibitor, ζ-Stat. These data suggests that PKC-ζ is a biomarker and a novel target in the carcinogenesis of breast and CCOC and its inhibition by way of ζ-Stat decreased the rate of proliferation, tumor growth and expression of invasive protein pathways.

Subjects/Keywords: breast cancer; clear cell ovarian cancer; Ect2; invasion; ovarian cancer; PKC-zeta; Rac1; RhoA; Biochemistry; Biology; Cell Biology

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APA (6^th Edition):

Smalley, T. B. (2020). The Role of aPKCs and aPKC Inhibitors in Cell Proliferation and Invasion in Breast and Ovarian Cancer. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/8300

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Smalley, Tracess B. “The Role of aPKCs and aPKC Inhibitors in Cell Proliferation and Invasion in Breast and Ovarian Cancer.” 2020. Thesis, University of South Florida. Accessed March 08, 2021. https://scholarcommons.usf.edu/etd/8300.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Smalley, Tracess B. “The Role of aPKCs and aPKC Inhibitors in Cell Proliferation and Invasion in Breast and Ovarian Cancer.” 2020. Web. 08 Mar 2021.

Vancouver:

Smalley TB. The Role of aPKCs and aPKC Inhibitors in Cell Proliferation and Invasion in Breast and Ovarian Cancer. [Internet] [Thesis]. University of South Florida; 2020. [cited 2021 Mar 08]. Available from: https://scholarcommons.usf.edu/etd/8300.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smalley TB. The Role of aPKCs and aPKC Inhibitors in Cell Proliferation and Invasion in Breast and Ovarian Cancer. [Thesis]. University of South Florida; 2020. Available from: https://scholarcommons.usf.edu/etd/8300

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. OOI YIN YIN. EFFECTS OF MESENCHYMAL STEM CELL-CONDITIONED MEDIUM ON THE ACTIVATION OF MICROGLIA.

Degree: 2014, National University of Singapore

URL: http://scholarbank.nus.edu.sg/handle/10635/118888

Subjects/Keywords: mesenchymal stem cells-conditioned medium; microglia; activation; Ect2; proliferation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

YIN, O. Y. (2014). EFFECTS OF MESENCHYMAL STEM CELL-CONDITIONED MEDIUM ON THE ACTIVATION OF MICROGLIA. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/118888

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

YIN, OOI YIN. “EFFECTS OF MESENCHYMAL STEM CELL-CONDITIONED MEDIUM ON THE ACTIVATION OF MICROGLIA.” 2014. Thesis, National University of Singapore. Accessed March 08, 2021. http://scholarbank.nus.edu.sg/handle/10635/118888.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

YIN, OOI YIN. “EFFECTS OF MESENCHYMAL STEM CELL-CONDITIONED MEDIUM ON THE ACTIVATION OF MICROGLIA.” 2014. Web. 08 Mar 2021.

Vancouver:

YIN OY. EFFECTS OF MESENCHYMAL STEM CELL-CONDITIONED MEDIUM ON THE ACTIVATION OF MICROGLIA. [Internet] [Thesis]. National University of Singapore; 2014. [cited 2021 Mar 08]. Available from: http://scholarbank.nus.edu.sg/handle/10635/118888.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

YIN OY. EFFECTS OF MESENCHYMAL STEM CELL-CONDITIONED MEDIUM ON THE ACTIVATION OF MICROGLIA. [Thesis]. National University of Singapore; 2014. Available from: http://scholarbank.nus.edu.sg/handle/10635/118888

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. CHENG SHI YUAN. Characterisation of the BRCT/DBL Protein ECT2 in cell cycle regulation.

Degree: 2008, National University of Singapore

URL: http://scholarbank.nus.edu.sg/handle/10635/16837

Subjects/Keywords: Ect2; cell cycle; cancer; p27Kip1; RhoGTPase; Rb

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

YUAN, C. S. (2008). Characterisation of the BRCT/DBL Protein ECT2 in cell cycle regulation. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/16837

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

YUAN, CHENG SHI. “Characterisation of the BRCT/DBL Protein ECT2 in cell cycle regulation.” 2008. Thesis, National University of Singapore. Accessed March 08, 2021. http://scholarbank.nus.edu.sg/handle/10635/16837.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

YUAN, CHENG SHI. “Characterisation of the BRCT/DBL Protein ECT2 in cell cycle regulation.” 2008. Web. 08 Mar 2021.

Vancouver:

YUAN CS. Characterisation of the BRCT/DBL Protein ECT2 in cell cycle regulation. [Internet] [Thesis]. National University of Singapore; 2008. [cited 2021 Mar 08]. Available from: http://scholarbank.nus.edu.sg/handle/10635/16837.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

YUAN CS. Characterisation of the BRCT/DBL Protein ECT2 in cell cycle regulation. [Thesis]. National University of Singapore; 2008. Available from: http://scholarbank.nus.edu.sg/handle/10635/16837

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Vanderbilt University

7. Delahanty, Ryan James. Towards an understanding of the role of chromosome 15q11-q13 in idiopathic autism.

Degree: PhD, Human Genetics, 2010, Vanderbilt University

URL: http://hdl.handle.net/1803/10492

► TOWARD AN UNDERSTANDING OF THE ROLE OF CHROMOSOME 15Q11-Q13 IN IDIOPATHIC AUTISM RYAN JAMES DELAHANTY Dissertation under the direction of Dr. James S. Sutcliffe The… (more)

▼ TOWARD AN UNDERSTANDING OF THE ROLE OF CHROMOSOME 15Q11-Q13 IN IDIOPATHIC AUTISM RYAN JAMES DELAHANTY Dissertation under the direction of Dr. James S. Sutcliffe The 15q11-q13 region is a genomic interval involved in a growing number of genomic disorders. The genes in the interval are subject to imprinting and parent-of-origin expression effects. Maternal duplication of the 15q11-q13 region is the most frequent chromosomal abnormality associated with autism. Extensive work has indicated that two genes in this interval, UBE3A and GABRB3, show very strong evidence for association with autism. To examine the extent to which these genes may contribute to autism, family-based association studies of UBE3A and GABRB3 were undertaken. Here we have investigated the role of common variants of UBE3A and GABRB3 in autism as well as the an intense investigation of the association of a rare variant, P11S in GABRB3 and its role in autism. In addition, we have investigated MECP2, a gene which when defective causes Rett syndrome, and potentially regulates gene expression of UBE3A and GABRB3. e have used genetic and biochemical methods to investigate two genes in the UBE3A network, ECT2 and GCH1. Finally, we used genotype data and multiplex ligation probe amplification (MLPA) to determine if copy number variation in the form of deletions and duplications in UBE3A and GABRB3 may play a role in the etiology of autism. Our findings indicated that a common allele of MECP2 is associated with autism, which was replicated by another group. We show association with UBE3A and its associated genes ECT2 and GCH1 as well as a relationship between UBE3A and GCH1 gene and protein expression, observed in a model system, and validated in our samples, which may provide guidance and support for a role of UBE3A and its action at the synapse and potential contribution to autism. We show modest association of GABRB3 with autism and epilepsy, but find a single coding variant, P11S, maternally overtransmitted and in such cases dramatically increasing autism risk. Finally, we found little evidence for microdeletions or microduplications in UBE3A and GABRB3 to contribute to autism pathology. The work presented in this thesis expands on earlier findings with regard to the role of GABRB3 and UBE3A in autism and represents an investigation of variants in these and their related genes spanning the spectrum from common variants of modest effect to rare variants of more profound effect. The availability of new technologies to evaluate copy number variation and next generation sequencing will likely uncover a wider role for 15q11-q13 and related loci in autism. The role of more highly penetrant private mutations of this nature is suggested as an avenue for further investigation. Advisors/Committee Members: Lawrence T. Reiter (committee member), John A. Phillips III (committee member), Chun Li (committee member), James Sutcliffe (committee member), Scott Williams (Committee Chair).

Subjects/Keywords: 15q11-q13; association; autism; GABRB3; UBE3A; ECT2; GCH1; CNV; MECP2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Delahanty, R. J. (2010). Towards an understanding of the role of chromosome 15q11-q13 in idiopathic autism. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10492

Chicago Manual of Style (16^th Edition):

Delahanty, Ryan James. “Towards an understanding of the role of chromosome 15q11-q13 in idiopathic autism.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed March 08, 2021. http://hdl.handle.net/1803/10492.

MLA Handbook (7^th Edition):

Delahanty, Ryan James. “Towards an understanding of the role of chromosome 15q11-q13 in idiopathic autism.” 2010. Web. 08 Mar 2021.

Vancouver:

Delahanty RJ. Towards an understanding of the role of chromosome 15q11-q13 in idiopathic autism. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1803/10492.

Council of Science Editors:

Delahanty RJ. Towards an understanding of the role of chromosome 15q11-q13 in idiopathic autism. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://hdl.handle.net/1803/10492

University of Queensland

8. Priya, Rashmi. Junctional Rho GTPase signalling: molecules and mechanisms.

Degree: Institute for Molecular Bioscience, 2015, University of Queensland

URL: http://espace.library.uq.edu.au/view/UQ:353099

Subjects/Keywords: Rho GTPase; Cadherin; Myosin; ect2; Rock; Feedback signalling; p190b rho gap; Epithelial morphogenesis; Cancer; Bistability; 0601 Biochemistry and Cell Biology; 1112 Oncology and Carcinogenesis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Priya, R. (2015). Junctional Rho GTPase signalling: molecules and mechanisms. (Thesis). University of Queensland. Retrieved from http://espace.library.uq.edu.au/view/UQ:353099

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Priya, Rashmi. “Junctional Rho GTPase signalling: molecules and mechanisms.” 2015. Thesis, University of Queensland. Accessed March 08, 2021. http://espace.library.uq.edu.au/view/UQ:353099.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Priya, Rashmi. “Junctional Rho GTPase signalling: molecules and mechanisms.” 2015. Web. 08 Mar 2021.

Vancouver:

Priya R. Junctional Rho GTPase signalling: molecules and mechanisms. [Internet] [Thesis]. University of Queensland; 2015. [cited 2021 Mar 08]. Available from: http://espace.library.uq.edu.au/view/UQ:353099.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Priya R. Junctional Rho GTPase signalling: molecules and mechanisms. [Thesis]. University of Queensland; 2015. Available from: http://espace.library.uq.edu.au/view/UQ:353099

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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Open Access Theses and Dissertations